Podcasts about th17 cells

Ozone therapy is the latest in treatments that produces multiple benefits - skin, gut, immune health and more! But how does it work and is it right for you? I'll answer these questions and more in this episode. Watch now!  Simply O3 At-Home Therapy: https://www.simplyo3.com/collections/3-0-home-kits/products/stratus-3-0-gold-kit

References Immunity. 2021 Feb 9; 54(2): 308–323.e6. J Endocrinol. 2003 Sep;178(3):479-89. --- Send in a voice message: https://anchor.fm/dr-daniel-j-guerra/message

At Cornell University in Ithaca, NY, Mandy joins Immune to discuss her career and her research on understanding how Th17 cells develop and are influenced to be ‘good' or ‘bad' in their function. Hosts: Vincent Racaniello, Cynthia Leifer, Steph Langel, and Brianne Barker Guest: Mandy McGeachy Subscribe (free): Apple Podcasts, Google Podcasts. RSS, email Become a patron of Immune! Links for this episode Th17 cytokines in health and disease (Immunity) Time stamps by Jolene. Thanks! Music by Steve Neal. Immune logo image by Blausen Medical Send your immunology questions and comments to immune@microbe.tv

T-helper 17 (Th17) cells and regulatory T cells (Tregs) play a role in developing autoimmune conditions like Graves' disease and Hashimoto's.Today I'm sharing everything you need to know about these cells since you'll hear me refer to them a lot in future podcast episodes.In this episode, you'll learn:Where Th17 and Treg cells originateThe role of Th17 and Treg cellsHow Th17 and Treg cells are connected to autoimmune conditionsNutrients, supplements, and natural agents that have been shown to increase regulatory T cells and decrease Th17 cellsTune in for next week's Q&A episode, where I'll discuss something many people commonly use in their homes that can increase Th17 cells.I hope you found this episode valuable, and I look forward to catching you in the next episode!To learn more, visit the show notes at https://savemythyroid.com/podcast/what-are-th17-and-treg-cells/.

The challenge of living with MS often begins with the challenge of getting a timely and accurate diagnosis. There are no symptoms, physical findings, or laboratory tests that can, by themselves, determine if someone has MS, and the most common symptoms of MS can often resemble symptoms of other conditions. All of these variables can sometimes lead to patients getting the wrong diagnosis. Dr. Andrew Solomon, Associate Professor of Neurological Sciences and Division Chief of Multiple Sclerosis at the University of Vermont Larner College of Medicine, is joining me to discuss diagnosing and misdiagnosing MS. We'll also fill you in on the details of the upcoming European MS Platform Annual Conference (it's free to register!). We'll tell you about a cannabis-based treatment that's been shown to improve spasticity without causing weakness in people with MS. We'll share the results of a study that looked at how a COVID-19 infection affects MS. You'll hear about newly published research that may have isolated the gene that causes male immune cells to drive more severe MS. And we'll give you the rundown of this month's Can Do MS programs. We have a lot to talk about! Are you ready for RealTalk MS??! European MS Platform Annual Conference  1:38 Cannabis extract improves spasticity without causing weakness  2:44 How COVID-19 may affect MS  4:43 A word about our show notes  6:49 Study shows male immune cells drive more severe MS  8:18 This month's Can Do MS programs  11:38 Dr. Andrew Solomon discusses diagnosing MS and misdiagnosing MS  13:13 Share this episode  25:54 Have a minute? Leave a rating & review for the podcast  26:14 SHARE THIS EPISODE OF REALTALK MS Just copy this link & paste it into your text or email: https://realtalkms.com/196 ADD YOUR VOICE TO THE CONVERSATION I've always thought about the RealTalk MS podcast as a conversation. And this is your opportunity to join the conversation by sharing your feedback, questions, and suggestions for topics that we can discuss in future podcast episodes. Please shoot me an email or call the RealTalk MS Listener Hotline and share your thoughts! Email: jon@realtalkms.comPhone: (310) 526-2283 And don't forget to join us in the RealTalk MS Facebook group! LINKS If your podcast app doesn't allow you to click on these links, you'll find them in the show notes in the RealTalk MS app or at www.RealTalkMS.com National MS Society COVID-19 Vaccine Guidance for People Living with MS European MS Platform Annual Conference STUDY: COVID-19 is Associated with New Symptoms of Multiple Sclerosis That are Prevented by Disease-Modifying Therapies STUDY: Male Sex Chromosomal Complement Exacerbates the Pathogenicity of Th17 Cells in a Chronic Model of Central Nervous System Autoimmunity Can Do MS Program Information and Registration Join the RealTalk MS Facebook Group Download the RealTalk MS App for iOS Download the RealTalk MS App for Android Give RealTalk MS a Rating and Review Follow RealTalk MS on Twitter, @RealTalkMS_jon, and subscribe to our newsletter at our website, RealTalkMS.com. RealTalk MS Episode 196 Guests: Dr. Andrew Solomon Tags: MS, MultipleSclerosis, MSResearch, MSSociety, RealTalkMS Privacy Policy

John Alcorn, PhD is a scientist at UPMC Children's Hospital of Pittsburgh and an associate professor in the University of Pittsburgh School of Medicine. The Alcorn lab is focused on T cell immunity, host defense, epithelial cell biology, and lung physiology. A primary lab focus is on Influenza infection and the host defense mechanisms of T helper 17 cells. His lab has recently shown that the TH17 effector cytokines IL-17 and IL-22 are required for host defense against a variety of extracellular pathogens. 

Dan Littman discusses the opposing roles of Th17 cells. They protect mucosal surfaces from infection with bacteria and fungi, but they can also cause autoimmune inflammation.

Dan Littman discusses the opposing roles of Th17 cells. They protect mucosal surfaces from infection with bacteria and fungi, but they can also cause autoimmune inflammation.

Apparently chewing stimulates the production of Th17 cells in the mouth, which could help fight disease in some parts of the body.

Apparently chewing stimulates the production of Th17 cells in the mouth, which could help fight disease in some parts of the body.

Good dental hygiene is crucial in the fight against gum disease, which can lead to a common condition called periodontitis. It comes about when changes to the bacteria in the mouth cause a reaction called inflammation in areas around the teeth, potentially destroying tissue and bone and causing tooth loss. But quite why an altered microbiome causes this inflammation isn't known, so there are no targeted treatments, and surgery is the main option for severe cases. But now scientists have spotted that periodontitis patients have higher numbers of immune cells called TH17 cells in their mouths,... Like this podcast? Please help us by supporting the Naked Scientists

Good dental hygiene is crucial in the fight against gum disease, which can lead to a common condition called periodontitis. It comes about when changes to the bacteria in the mouth cause a reaction called inflammation in areas around the teeth, potentially destroying tissue and bone and causing tooth loss. But quite why an altered microbiome causes this inflammation isn't known, so there are no targeted treatments, and surgery is the main option for severe cases. But now scientists have spotted that periodontitis patients have higher numbers of immune cells called TH17 cells in their mouths,... Like this podcast? Please help us by supporting the Naked Scientists

Wed, 23 Jan 2013 12:00:00 +0100 https://edoc.ub.uni-muenchen.de/15274/ https://edoc.ub.uni-muenchen.de/15274/1/Lluis_Cano_Anna_Maria.pdf Lluis Cano, Anna Maria

Fri, 1 Jan 2010 12:00:00 +0100 https://epub.ub.uni-muenchen.de/18366/1/TH17_Cells_in_Autoimmune_Arthritis.pdf Skapenko, A.; Schulze-Koops, Hendrik; Kerzendorf, U.; Reindl, C.; Dechant, C.; Grunke, M.; Leipe, Jan ddc:610, Medizin

Learn about a microRNA that confers robustness on the pathways regulating fly development (start 0:50), the cytokine interleukin-1 which acts as a key player in the production of Th17 cells (start 10:23) and using stem cell therapy to treat heart disease (start 15:22).

Traditionally, Crohn's disease has been associated with a Th1 cytokine profile, while Th2 cytokines are modulators of ulcerative colitis. This concept has been challenged by the description of tolerising regulatory T cells (Treg) and by proinflammatory Th17 cells, a novel T cell population characterised by the master transcription factor RORtextgreekgt, the surface markers IL23R and CCR6, and by production of the proinflammatory cytokines IL17A, IL17F, IL21, IL22 and IL26, and the chemokine CCL20. Th17 cells differentiate under the influence of IL1textgreekb, IL6, IL21 and IL23. Recent studies indicate that TGFtextgreekb is essential not only for the development of murine Th17 cells but also for differentiation of human Th17 cells. TGFtextgreekb reciprocally regulates the differentiation of inflammatory Th17 cells and suppressive Treg subsets, with the concomitant presence of proinflammatory cytokines favouring Th17 cell differentiation. Several studies demonstrated an important role of Th17 cells in intestinal inflammation, particularly in Crohn's disease. Genome-wide association studies indicate that IL23R and five additional genes involved in Th17 differentiation (IL12B, JAK2, STAT3, CCR6 and TNFSF15) are associated with susceptibility to Crohn's disease and partly also to ulcerative colitis. Taken together, both Th1 and Th17 cells are important mediators of inflammation in Crohn's disease, although activities previously ascribed to IL12 may be mediated by IL23. Anti-IL12/IL23p40 antibody therapy, which targets both Th1 and Th17 cells, is effective in Crohn's disease. However, the complex relationship between Th1 and Th17 cells has not been completely analysed. This will be of great importance to delineate the specific contributions of these cells to Crohn's disease and other autoimmune diseases.