Podcasts about cholelithiasis

In this episode, we review the high-yield topic of⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Cholelithiasis⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠from the Gastrointestinal section. Follow ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Medbullets⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets

In this episode, Prof Clare Collins and Ilyse Jones talk about gallstones, their causes, and management strategies. The conversation highlights the increased risk of gallstones in women due to hormonal factors, the role of bile in fat digestion, and what happens once your gallbladder is removed. Dietary recommendations are provided to help reduce the risk of gallstones and also to manage symptoms both before and after surgery to remove the gallbladder.Here's a link to more information on gallstones at The Conversation https://theconversation.com/got-gallstones-heres-what-to-eat-and-avoid-53229#Gallstones, #Gallbladder, #Cholelithiasis, #diet, #nutrition, #health, #women, #estrogen, #bile, #DigestiveHealth, #malabsorption Hosted on Acast. See acast.com/privacy for more information.

Get ahead with VETAHEAD and join Dr. Proença on 15 minutes of ZooMed (exotic animal medicine) content. Today, Dr. Proença Dr. Proença dives into the world of sodium urate cholelithiasis in iguanas—and it's more common than you might think! Discover the clinical and pathologic findings from this eye-opening study, featuring five iguanas diagnosed with gallstones. Learn about the symptoms (or lack thereof), the link to hepatic disease, and why ultrasounds and CTs are now a must for diagnosing these tricky cases. Whether you're dealing with incidental findings or biliary obstruction, this episode will change how you approach reptile care. Tune in for tips on medical management, surgery, and everything in between ⁠Click here to get your VETAHEAD E-Magazine!⁠⁠⁠⁠⁠ ⁠ ⁠⁠⁠⁠⁠⁠Click here to receive a VETAHEAD Gift!⁠⁠⁠⁠⁠⁠⁠⁠ Do you want to access more ZooMed (exotics) knowledge directly from specialists? Come with us and #jointhemovement #nospeciesleftbehind ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Head to VETAHEAD Website⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Join our VETAHEAD Community⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Follow @the_vetahead on Instagram⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Subscribe to @vetahead channel on YouTube⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Follow @vetahead on Facebook⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Follow @vetahead on TikTok

In this episode, we review the high-yield topic of ⁠⁠Cholelithiasis and biliary colic⁠⁠⁠ ⁠from the Gastrointestinal section. Follow ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Medbullets⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets Linkedin: https://www.linkedin.com/company/medbullets

Gallstones are the hardened precipitates—“stones”—of the substrates found in bile. The liver makes bile to help digest fats, and the bile is stored in the gallbladder. When there is an excess of a particular substance in the bile (eg, cholesterol or unconjugated bilirubin), gallstones form in the gallbladder. Gallstones can be as small as a grain of sand, as large as a golf ball, or any size in between. Gallstones are very common and usually do not cause any symptoms. But not all gallstones stay in the gallbladder. After listening to this AudioBrick, you should be able to: Define, compare, and contrast cholelithiasis, cholangitis, choledocholithiasis, and cholecystitis, listing the most common causes of each. Describe the structure and pathogenesis of cholesterol and pigment gallstones. List the major risk factors for development of gallstones, describe the typical clinical course, and list the potential complications. Distinguish acute and chronic cholecystitis, and describe the clinical presentation, signs, and lab abnormalities. Describe the treatment of gallstones and acute cholecystitis. You can also check out the original brick on Cholelithiasis and Cholecystitis from our Gastrointestinal collection, which is available for free. Learn more about Rx Bricks by signing up for a free USMLE-Rx account: www.usmle-rx.com You will get 5 days of full access to our Rx360+ program, including nearly 800 Rx Bricks.  After the 5-day period, you will still be able to access over 150 free bricks, including the entire collections for General Microbiology and Cellular and Molecular Biology. *** If you enjoyed this episode, we'd love for you to leave a review on Apple Podcasts.  It helps with our visibility, and the more med students (or future med students) listen to the podcast, the more we can provide to the future physicians of the world. Follow USMLE-Rx at: Facebook: www.facebook.com/usmlerx Blog: www.firstaidteam.com Twitter: https://twitter.com/firstaidteam Instagram: https://www.instagram.com/firstaidteam/ YouTube: www.youtube.com/USMLERX Learn how you can access over 150 of our bricks for FREE: https://usmlerx.wpengine.com/free-bricks/

VISIT US AT ⁠⁠⁠⁠⁠NCLEXHIGHYIELD.COM⁠⁠⁠⁠⁠ No matter where you are in the world, or what your schedule is like, access the entire course at ⁠⁠⁠⁠⁠⁠⁠www.NCLEXHighYieldCourse.com⁠⁠⁠⁠⁠⁠⁠  The NCLEX High Yield Podcast was featured on ⁠⁠⁠⁠⁠⁠⁠Top 15 NCLEX Podcasts⁠⁠⁠⁠⁠⁠⁠! Make sure you ⁠⁠⁠⁠⁠⁠⁠JOIN OUR NEW VIP FACEBOOK GROUP!⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠https://nclexhighyield.com/blogs/news/nclex-high-yield-quick-links⁠⁠⁠⁠⁠⁠⁠ A topic that confuses many, but listen to how Dr. Zeeshan breaks this bad boy down! Many people get overwhelmed with all the information that's out there, we keep it simple! Join us weekly for FREE Zoom Sessions and be one of the many REPEAT test takers that passed the exam by spending NO MONEY with NCLEX High Yield! NCLEX High Yield is a Prep Course and Tutoring Company started by Dr. Zeeshan in order to help people pass the NCLEX, whether it's the first time , or like the majority of our students, it's NOT their first time. We keep things simple, show you trends and tips that no one has discovered, and help you on all levels of the exam! Follow us on Instagram: ⁠⁠⁠⁠@NCLEXHighYield ⁠⁠⁠⁠or check out our website www.NCLEXHighYield.com Make sure you join us for our FREE Weekly Zoom Sessions! Every Wednesday 3PM PST / 6PM EST. Subscribe to our newsletter at ⁠⁠⁠⁠⁠⁠nclexhighyield.com⁠⁠⁠⁠⁠⁠ --- Support this podcast: https://podcasters.spotify.com/pod/show/nclexhighyield/support

Thank you for listening to this episode of "Health and Fitness" from the Nezpod Studios! Enjoy your night or the start of your day, spiced by our top-notch health and fitness/wellness updates coined from the best sources around the globe: made only for your utmost enjoyment and enlightenment… Click on subscribe to get more spicy episodes for free! See you again soon on the next episode of Health and Fitness Updates! Learn more about your ad choices. Visit megaphone.fm/adchoices

Thank you for listening to this episode of "Health and Fitness" from the Nezpod Studios! Enjoy your night or the start of your day, spiced by our top-notch health and fitness/wellness updates coined from the best sources around the globe: made only for your utmost enjoyment and enlightenment… Click on subscribe to get more spicy episodes for free! See you again soon on the next episode of Health and Fitness Updates! Learn more about your ad choices. Visit megaphone.fm/adchoices

Thank you for listening to this episode of "Health and Fitness" from the Nezpod Studios! Enjoy your night or the start of your day, spiced by our top-notch health and fitness/wellness updates coined from the best sources around the globe: made only for your utmost enjoyment and enlightenment… Click on subscribe to get more spicy episodes for free! See you again soon on the next episode of Health and Fitness Updates! Learn more about your ad choices. Visit megaphone.fm/adchoices

Both Pathophysiology and Clinical Aspects of gallstone disorders are discussed followed by important hints on differential diagnosis of gallstone disease from one another and from their complications, most specifically from cholecystitis and cholangitis.

In this episode, we review the high-yield topic of Cholelithiasis from the Gastrointestinal section. Follow Medbullets on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets --- Send in a voice message: https://anchor.fm/medbulletsstep1/message

Another "bread and butter" area of general surgery is the gallbladder. Listen along as Dr. Oli tells you all about cholelithiasis and acute cholecystitis: what they are, how to make the diagnosis, and all the treatment options. Happy learning! Don't forget to check out our Facebook page www.facebook.com/drolimedonthego

In this episode, we review the high-yield topic of Cholelithiasis and Biliary Colic from the Gastrointestinal section.

Inflammatory Bowel Disorders, Pancreatitis, Cholecystitis, Cholelithiasis, Hapatitis, Liver Cirrhosis and many others

Gallstones are the hardened precipitates—“stones”—of the substrates found in bile. The liver makes bile to help digest fats, and the bile is stored in the gallbladder. When there is an excess of a particular substance in the bile (eg, cholesterol or unconjugated bilirubin), gallstones form in the gallbladder. Gallstones can be as small as a grain of sand, as large as a golf ball, or any size in between. Gallstones are very common and usually do not cause any symptoms. But not all gallstones stay in the gallbladder. After listening to this AudioBrick, you should be able to: Define, compare, and contrast cholelithiasis, cholangitis, choledocholithiasis, and cholecystitis, listing the most common causes of each. Describe the structure and pathogenesis of cholesterol and pigment gallstones. List the major risk factors for development of gallstones, describe the typical clinical course, and list the potential complications. Distinguish acute and chronic cholecystitis, and describe the clinical presentation, signs, and lab abnormalities. Describe the treatment of gallstones and acute cholecystitis. You can also check out the original brick on Cholelithiasis and Cholecystitis from our Gastrointestinal collection, which is available for free. Learn more about Rx Bricks by signing up for a free USMLE-Rx account: www.usmle-rx.com You will get 5 days of full access to our Rx360+ program, including nearly 800 Rx Bricks.  After the 5-day period, you will still be able to access over 150 free bricks, including the entire collections for General Microbiology and Cellular and Molecular Biology. *** If you enjoyed this episode, we'd love for you to leave a review on Apple Podcasts.  It helps with our visibility, and the more med students (or future med students) listen to the podcast, the more we can provide to the future physicians of the world. Follow USMLE-Rx at: Facebook: www.facebook.com/usmlerx Blog: www.firstaidteam.com Twitter: https://twitter.com/firstaidteam Instagram: https://www.instagram.com/firstaidteam/ YouTube: www.youtube.com/USMLERX Learn how you can access over 150 of our bricks for FREE: https://usmlerx.wpengine.com/free-bricks/

Cholecystitis, Cholangitis, Choledocholithiasis, Cholelithiasis review for your PANCE, PANRE, and EOR’s. --- Support this podcast: https://anchor.fm/scott--shapiro/support

So we have covered appendicitis, SBO, cholangitis, and postop fevers so far with the podcast.  In trying to hit the big ticket conditions for surgery, it is time for a review of acute cholecystitis and cholelithiasis. 

Cholelithiasis, Cholezystitis und Cholangitis Gallengangskarzinom Hepatozelluläres Karzinom Pankreas- und Leberchirurgie Anal- und Rektumprolaps Hämorrhoiden und Hämorrhoidalleiden Analvenenthrombose Analfissur Analabszess und Analfistel Analkarzinom Sinus pilonidali Äußere Hernien Leistenhernie Schenkelhernie Innere Hernien

There are 5 key diagnoses classically associated with right upper quadrant (RUQ) abdominal pain. Cholelithiasis and Biliary Colic Cholelithiasis = Gallstones in the gallbladder Frequently seen on CT scan or RUQ ultrasound Present in 15% of the population Biliary colic = Intermittent episodes of pain if stone passes Classically colicky/crampy/spasmy pain in RUQ Frequently radiates […]

This episode covers Chapter 80 of Rosen's Emergency Medicine 9th edition. (Yes the new edition). Building on previous episodes (see fever, Jaundice and abdo pain), today we take a look at all things right upper quadrant badness.   1) List 8 ddx for hepatitis   2) Complete the following table for Hepatitis A, B & C: Transmission, Risk Factors, Carrier State, Acute Infection, Previous Infection, Chronic Infection, Prev Vaccine, Transmission Risk, Vaccine.   (show notes: What is hepatitis E? Where is it commonly found (geographically)? What is the significance of hepatitis D?)   3) Describe the post-exposure prophylaxis for exposure to HepA, HepB, HepC   4) Compare the expected lab work in acute viral hepatitis vs EtOH hepatitis   5) What liver diseases are associated with alcohol abuse? What non-hepatic conditions are associated with alcohol abuse? Describe the management of EtOH hepatitis   6) List 6 stigmata of chronic liver dz and list 3 complications   7) How is are chronic cirrhosis and ascites managed in the ER?   8) Describe a grading scale for hepatic encephalopathy and list 5 management considerations   9) Describe the ER diagnosis and management of SBP.   10) List 3 types of drug-induced liver disease.   11) What are two types of hepatic abscesses? How are they diagnosed and treated?   12) What is budd-chiari syndrome? How is it managed?   13) What is primary sclerosing cholangitis (PSC)? What is primary biliary cirrhosis? What is PSC associated with?   14) List 6 RFs for Cholelithiasis   15) Describe the clinical presentation of cholecystitis. List Lab, Xray (3) and US (4) findings   16) List 4 patients that get acalculous cholecystitis   17) List 4 considerations in the management of acute cholecystitis. When is surgery performed early?   18) What is the classic presentation of ascending cholangitis? What two clinical eponyms are described? How is ascending cholangitis managed? Wisecracks:   1.. Which conditions are associated with transaminases in the 10000s? How do you approach a patient with a needlestick injury? What is the risk of transmission following a needlestick? What are underlying causes of hepatic encephalopathy in patients with known liver disease? What are the typical investigations performed on ascites fluid? What is the SAAG and how is it interpreted?  What is the significance of a calcified gallbladder?

This episode covers Chapter 80 of Rosen's Emergency Medicine 9th edition. (Yes the new edition). Building on previous episodes (see fever, Jaundice and abdo pain), today we take a look at all things right upper quadrant badness.   1) List 8 ddx for hepatitis   2) Complete the following table for Hepatitis A, B & C: Transmission, Risk Factors, Carrier State, Acute Infection, Previous Infection, Chronic Infection, Prev Vaccine, Transmission Risk, Vaccine.   (show notes: What is hepatitis E? Where is it commonly found (geographically)? What is the significance of hepatitis D?)   3) Describe the post-exposure prophylaxis for exposure to HepA, HepB, HepC   4) Compare the expected lab work in acute viral hepatitis vs EtOH hepatitis   5) What liver diseases are associated with alcohol abuse? What non-hepatic conditions are associated with alcohol abuse? Describe the management of EtOH hepatitis   6) List 6 stigmata of chronic liver dz and list 3 complications   7) How is are chronic cirrhosis and ascites managed in the ER?   8) Describe a grading scale for hepatic encephalopathy and list 5 management considerations   9) Describe the ER diagnosis and management of SBP.   10) List 3 types of drug-induced liver disease.   11) What are two types of hepatic abscesses? How are they diagnosed and treated?   12) What is budd-chiari syndrome? How is it managed?   13) What is primary sclerosing cholangitis (PSC)? What is primary biliary cirrhosis? What is PSC associated with?   14) List 6 RFs for Cholelithiasis   15) Describe the clinical presentation of cholecystitis. List Lab, Xray (3) and US (4) findings   16) List 4 patients that get acalculous cholecystitis   17) List 4 considerations in the management of acute cholecystitis. When is surgery performed early?   18) What is the classic presentation of ascending cholangitis? What two clinical eponyms are described? How is ascending cholangitis managed? Wisecracks:   1.. Which conditions are associated with transaminases in the 10000s? How do you approach a patient with a needlestick injury? What is the risk of transmission following a needlestick? What are underlying causes of hepatic encephalopathy in patients with known liver disease? What are the typical investigations performed on ascites fluid? What is the SAAG and how is it interpreted?  What is the significance of a calcified gallbladder?

PhysicianAssistantBoards.com - Today were going to tackle three topics: cholecystitis, cholangitis, and cholelithiasis.  Well go over cause, symptoms, signs, diagnostic testing, and treatment. 

Melissa Bagloo, MD, a surgeon at the Center for Metabolic and Weight Loss Surgery at NewYork-Presbyterian Hospital/Columbia University Medical Center, discusses diseases of the gallbladder, specifically gallstones or Cholelithiasis, their management and treatment options.

Melissa Bagloo, MD, a surgeon at the Center for Metabolic and Weight Loss Surgery at NewYork-Presbyterian Hospital/Columbia University Medical Center, talks about diseases of the gallbladder such as disorders that accompany Cholelithiasis or gallstones, Choledocholithiasis, Gallstone Illeus, and Gallbladder Cancer.

Einleitung: Im Klinikum Innenstadt der Ludwig-Maximilians-Universität München werden herztransplantierte Patienten im Rahmen der nachsorglichen Betreuung in regelmäßigen Abständen sonographisch untersucht. Ziel dieser Arbeit ist es, bei einem dort regelmäßig betreuten Patientengut Infektionen, Neoplasien und andere unerwünschte Wirkungen der Immunsuppression wie vor allem die anhand der routinemäßigen Abdominalsonographien erhobenen pathologischen Befunde und morphologischen Organveränderungen deskriptiv zu dokumentieren. Dadurch soll ein Eindruck von der klinischen Relevanz der routinemäßigen Abdominalsonographie als Screening-Methode in der Nachsorge bei herztransplantierten Patienten zur frühzeitigen Erfassung pathologischer Befunde infolge immunsuppressiver Medikation geschaffen werden. Methode: Das Patientengut der vorliegenden Studie bestand ausschließlich aus herztransplantierten Patienten, die zur Vermeidung einer Transplantatabstoßung eine immunsuppressive Medikation erhielten und zwar als Kombination aus den immunsuppressiven Substanzen Cyclosporin A oder Tacrolimus, Azathioprin, Mycophenolat mofetil und Prednisolon. Insgesamt handelte es sich um 122 Patienten, die im Zeitraum von 1983 bis 1999 ein Spenderherz erhalten haben und von denen 85 Cyclosporin und 37 Tacrolimus als Basisimmunsuppressivum bekamen. Dazu wurden die pathologischen Befunde des Gesamtkollektivs ein Jahr, drei Jahre und fünf Jahre nach Herztransplantation des jeweiligen Patienten graphisch dargestellt. Zusätzlich zu den pathologisch-morphologischen sonographischen Befunden sollten auch die Häufigkeitsverteilungen der Neoplasien, opportunistischen Infektionen, das Neuauftreten von Diabetes mellitus, arterieller Hypertonie, Hyperlipidämie, Hyperurikämie, koronarer Herzkrankheit, peripherer arterieller Verschlusskrankheit sowie das Auftreten einer transitorischen ischämischen Attacke nach Herztransplantation dokumentiert werden. Abgesehen von den Häufigkeitsverteilungen im Gesamtkollektiv sollten auch die Häufigkeitsverteilungen in der Cyclosporin-Gruppe und der Tacrolimus-Gruppe angegeben werden. Von einem Vergleich dieser beiden Gruppen mit Signifikanzberechnungen sollte jedoch aufgrund mannigfaltiger Einflussgrößen abgesehen werden. Ergebnisse: Fünf Jahre nach Transplantation ergaben sich im Gesamtkollektiv unter anderem folgende Befunde: • 38,5 % Cholelithiasis • 9,0 % Cholezystitis • 21,3 % Hepatomegalie • 51,6 % Echogenitätserhöhung der Leber (Fettleber) • 36,1 % Größenabnahmen der Nieren • 86,9 % Echogenitätserhöhung der Nieren • 76,2 % Verminderung der Parenchymbreite der Nieren • 77,0 % Niereninsuffizienz im Stadium der kompensierten Retention • 9,8 % Niereninsuffizienz im präterminalen bzw. terminalen Stadium • 17,2 % sekundärer Hyperparathyreoidismus • 27,0 % Osteoporose • 19,7 % Pankreaslipomatose • 23,8 % Splenomegalie • 38,5 % Aortensklerose • 27,0 % maligne Entartungen • 52,5 % opportunistische Infektionen • 70,5 % Neuauftreten von arterieller Hypertonie • 65,6 % Neuauftreten von Hyperlipidämie • 16,4 % Neuauftreten der koronaren Herzkrankheit (KHK) • 18,0 % Neuauftreten der peripheren arteriellen Verschlusskrankheit (pAVK) • 5,7 % Erstmanifestation einer transitorischen ischämischen Attacke (TIA) Schlussfolgerung: In Anbetracht der Häufigkeit der anhand serieller Sonographien frühzeitig erfassbaren pathologisch-morphologischen Veränderungen der Abdominalorgane ist zu schließen, dass die in regelmäßigen zeitlichen Intervallen durchgeführte Sonographie, eine kostengünstige und nicht-invasive Untersuchung, fester Bestandteil der nachsorglichen Betreuung (herz-) transplantierter Patienten sein sollte. Die möglichst frühzeitige Detektion pathologisch-morphologischer Veränderungen der Abdominalorgane ermöglicht dem behandelnden Arzt, rasch entsprechende Maßnahmen zu ergreifen, um eine weitere Verschlechterung der krankhaften Befunde zu verhindern bzw. wenigstens zu verzögern. So kann beispielsweise nach sonographischer Erfassung einer pathologisch verringerten Parenchymbreite der Nieren, die mit der Nephrotoxizität des verabreichten Calcineurin-Inhibitors Cyclosporin oder Tacrolimus im Zusammenhang stehen kann, das nicht nephrotoxische Mycophenolat mofetil (eventuell im Austausch gegen Azathioprin) in die immunsuppressive Kombinationstherapie integriert werden, was eine schrittweise Reduzierung der Erhaltungsdosis des nephrotoxischen Calcineurin-Inhibitors ermöglicht.