Podcasts about disorders

In this episode of the Experience Miracles podcast, Dr. Tony Ebel addresses a critical and misunderstood topic: chiropractic care for children with hypermobility conditions like EDS, Down syndrome, and Chiari malformations. He explains why these children not only can receive chiropractic care safely, but actually need it more than typical children. Dr. Ebel breaks down how hypermobile joints create compensatory fixated segments that become subluxated, requiring specialized mixed-technique approaches. This episode is essential for parents who have been incorrectly advised by other healthcare providers to avoid chiropractic care for their hypermobile children.Key Topics & Timestamps([00:01:00]) - Why Hypermobile Children NEED Chiropractic Care More Than Others([00:03:00]) - Common Conditions: EDS, Down Syndrome, and Chiari Malformations([00:05:00]) - How the Brain Creates Compensatory Fixation in Response to Instability([00:07:00]) - Specialized Techniques Required: Tonal vs. Manual Approaches([00:11:00]) - Why This Care is Essential, Not Optional, for Genetic Hypermobility([00:12:00]) - Medical Doctors' Misunderstanding and the Harm of Avoiding Care-- Follow us on Socials: Instagram: @pxdocs Facebook: Dr. Tony Ebel & The PX Docs Network Youtube: The PX Docs For more information, visit PXDocs.com to read informative articles about the power of Neurologically-Focused Chiropractic Care. Find a PX Doc Office near me: PX DOCS DirectoryTo watch Dr. Tony's 30 min Perfect Storm Webinar: Click HereSubscribe, share, and stay tuned for more incredible episodes unpacking the power of Nervous System focused care for children!

Still struggling with thyroid symptoms even though your labs look “normal” and you're on medication? There might be a deeper reason—and it's often overlooked.In this episode, Dr. Anshul Gupta explores one of the most underdiagnosed root causes of thyroid dysfunction. Through powerful patient stories and deep insights from his functional medicine practice, you'll understand why standard treatments often fall short—and what might really be going on beneath the surface.In this episode, you'll explore: - A commonly missed environmental trigger affecting thyroid health - The hidden genetic factor that makes healing harder for some people - Why your home might be making you sick—even if it looks perfectly clean - The real reason thyroid medication alone might not be enough - A deeper look at how root-cause healing actually works - What to consider if you're dealing with brain fog, fatigue, or stubborn antibodies If you've been feeling stuck, frustrated, or like your thyroid treatment just isn't working… this episode might hold the missing piece of your healing journey. Connect With Me -Instagram - https://www.instagram.com/anshulguptamd/Twitter - https://www.twitter.com/anshulguptamdFacebook - https://www.facebook.com/drguptafunctPinterest - https://www.pinterest.com/anshulguptamdTo Buy Good Quality Supplements Goto -https://functionalwellbeingshop.com/Work With Me -https://www.anshulguptamd.com/work-with-me/Take The Thyroid Quiz & Evaluate Your Thyroid Health -https://www.anshulguptamd.com/thyroid-quiz/About Dr.Anshul Gupta MD -Dr. Anshul Gupta Md Is a Board-certified Family Medicine Physician, With Advanced Certification In Functional Medicine, Peptide Therapy, And Also Fellowship training in Integrative Medicine. He Has Worked At The Prestigious Cleveland Clinic Department Of Functional Medicine As Staff Physician Alongside Dr. Mark Hyman. He Believes In Empowering His Patients To Take Control Of Their Health And Partners With Them In Their Healing Journey.He Now Specializes As A Thyroid Functional Medicine Doctor, And Help People Reverse Their Unresolved Symptoms Of Thyroid Dysfunction.

Guest: Jen Mackie, owner of Kelly O' Bryans in New Westminster Learn more about your ad choices. Visit megaphone.fm/adchoices

We are still learning about all the effects of cannabis use. Raphael Cuomo, professor of medicine at the University of California, San Diego, explores how it may tie in with one affliction. Raphael E. Cuomo, PhD, is a globally recognized authority in biomedical science and non-communicable disease epidemiology. His research has shaped how scientists and […]

Tucked deep within the brain and no larger than a pea, the pituitary gland is often called the body's “master gland.” It oversees the endocrine system—a network of glands and organs that produce hormones regulating everything from growth and metabolism to reproduction and mood. At Pacific Neuroscience Institute's Pituitary Disorders Center, endocrinologist Dr. Noa Tal specializes in identifying and treating hormone and endocrine imbalances. Patients may present with a wide range of symptoms—unexplained weight gain, irregular menstrual cycles, hot flashes, or mood changes—that often seem unrelated. In cases like Cushing's disease, a pituitary tumor triggers excess cortisol production, resulting in a puzzling array of symptoms. Sometimes the cause is even more elusive, such as side effects from medications. That's where careful clinical investigation comes in. Dr. Tal and her interdisciplinary team work closely to uncover the root of hormonal issues and guide patients toward not just better outcomes, but better quality of life. Listen in!

Normal pressure hydrocephalus (NPH) is a pathologic condition whereby excess CSF is retained in and around the brain despite normal intracranial pressure. MRI-safe programmable shunt valves allow for fluid drainage adjustment based on patients' symptoms and radiographic images. Approximately 75% of patients with NPH improve after shunt surgery regardless of shunt type or location. In this episode, Aaron Berkowitz, MD, PhD, FAAN, speaks with Kaisorn L. Chaichana, MD, author of the article “Management of Normal Pressure Hydrocephalus” in the Continuum® June 2025 Disorders of CSF Dynamics issue. Dr. Berkowitz is a Continuum® Audio interviewer and a professor of neurology at the University of California San Francisco in the Department of Neurology in San Francisco, California. Dr. Chaichana is a professor of neurology in the department of neurological surgery at the Mayo Clinic in Jacksonville, Florida. Additional Resources Read the article: Management of Normal Pressure Hydrocephalus Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @LyellJ Guest: @kchaichanamd Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Berkowitz: This is Dr Aaron Berkowitz, and today I'm interviewing Dr Kaisorn Chaichana about his article on management of normal pressure hydrocephalus, which he wrote with Dr Jeremy Cutsforth-Gregory. The article appears in the June 2025 Continuum issue on disorders of CSF dynamics. Welcome to the podcast, and please introduce yourself to our audience. Dr Chaichana: Yeah, thank you for having me. I'm Kaisorn Chaichana. I'm a neurosurgeon at Mayo Clinic in Jacksonville, Florida. Part of my practice is doing hydrocephalus care, which includes shunts for patients with normal pressure hydrocephalus. Dr Berkowitz: Fantastic. Well, before we get into shunt considerations and NPH specifically, which I know is the focus of your article, I thought it would be a great opportunity for a neurologist to pick a neurosurgeon's brain a bit about shunts. So, to start, can you lay out for us the different types of shunts and shunt procedures, the advantages, disadvantages of each type of shunt, how you think about which shunt procedure should be used for which patient, that type of thing? Dr Chaichana: Yeah. So, there are different types of shunts, and the most common one that is used is called a ventricular peritoneal shunt. So, it has a ventricular catheter, it has a catheter that tunnels underneath the skin and it goes into the peritoneum where the fluid goes from the ventricular system into the peritoneum. Typically, the shunts are in the ventricle because that is the largest fluid-filled space in the brain. Other terminal areas include the atrium, which is really the jugular vein, and those are called ventricular atrial shunts. You can also have ventricular pleural shunts, which end in the pleural space and drain flui into the pleural space. Those are pretty much the most common ventricular shunts. There's also a lumboperitoneal shunt that drains from the lumbar spine, similar to a lumbar drain into the peritoneum. For the lumbar shunts, we don't typically have a lumbar pleural or lumbar atrial shunt just because of the pressure dynamics, because the lumbar spine is below the lung and as well as the atrium. And so, the drainage pattern is very different than ventricular peritoneal which is top to bottom. The most common shunt, why we use the ventricular peritoneal shunt the most, is because it has the most control. So, the peritoneum is set at a standard pressure in the intraabdominal pressure, whereas the ventricular atrial shunt depends on your venous return or venous pressure and your ventricular pleural shunt varies with inspiration and expiration. So, the easiest way for us to control the fluid, the ventricular system is through the ventricular peritoneal shunt. And that's why that's our most common shunt that we use. Dr Berkowitz: Fantastic. So, as you mention in the article, neurologists may be reluctant to offer a shunt to patients with NPH because many patients may not improve, or they improve only transiently; and out of fear of shunt complications. So, of course, as neurologists, we often only hear about a patient's shunt when there is a problem. So, we have this sort of biased view of seeing a lot of shunt malfunction and shunt infection. Of course, we might not see the patient if their shunt is working just fine. How common are these complications in practice, and how do you as a neurosurgeon weigh the risks against the often uncertain or transient benefits of a shunt in a patient with NPH who may be older and multiple medical comorbidities? How do you think about that and talk about it with patients? Dr Chaichana: When you hear about shunt complications, most of the shunt complications you hear about are typically in patients with congenital hydrocephalus. Those patients often require several shunt revisions just from either growing or the shunt stays in for a long time or the ventricular caliber is a lot less than some with normal pressure hydrocephalus. So, we don't really see a lot of complications with normal pressure hydrocephalus. So that shunt placement in these patients is typically pretty safe. The procedure's a relatively short procedure, around 30 minutes to 45 minutes to place a shunt, and we can control the pressure within the shunt setting so that we don't overdrain---which means too much fluid drains from the ventricular system---which can cause things like a subdural, which is probably the most common complication associated with normal pressure hydrocephalus. So, to obviate those risks, what we do is typically insert the shunt and then keep the shunt setting at a high setting. The higher the setting, the less it drains, and then we bring it slowly down based on the patient's symptoms to try to minimize the risk of this over drainage in the subdural hematoma while at the same time benefiting the patient. So, there's a concern for shunt in patients with normal pressure hydrocephalus. The concern or the complication risks are very low. The problem with normal pressure hydrocephalus, though, is that over time these patients benefit less and less from drainage or their disease process takes over. So, I do recommend placing this shunt as soon as possible just so that we can maximize their quality of life for that period of time. Dr Berkowitz: So, if I'm understanding you, then the risk of complication is more sort of due to the mechanical factors in patients with congenital hydrocephalus or sort of outgrowing the shunt, their pressure dynamics may be changing over time. And in your experience, an older patient with NPH, although they may have more medical comorbidities, the procedure itself is relatively quick and low-risk. And the actual complications due to mechanical factors, my understanding, are just much less common because the patient is obviously fully grown and they're getting one sort of procedure at one point in time and tend to need less revision, have less complication. Is that right? Dr Chaichana: Yeah, that's correct. The complication risk for normal hydrocephalus is a lot less than other types of hydrocephalus. Dr Berkowitz: That's helpful to know. While we're talking about some of these complications, let's say we're following a patient in neurology with NPH who has a shunt. What are some of the symptoms and signs of shunt malfunction or shunt infection? And what are the best studies to order to evaluate for these if we're concerned about them? Dr Chaichana: Yeah. So basically, for shunt malfunction, it's basically broken down into two categories. It's either overdrainage or underdrainage. So, underdrainage is where the shunt doesn't function enough. And so basically, they return to their state before the shunt was placed. So that could be worsening gait function, memory function, urinary incontinence are the typical symptoms we look for in patients with normal pressure hydrocephalus and underdrainage, or the shunt is not working. For patients that are having overdrainage, which is draining too much, the classic sign is typically headaches when they stand up. And the reason behind that is when there's overdrainage, there's less cerebrospinal fluid in their ventricular system, which means less intracranial pressure. So that when they stand up, the pressure differential between their head and the ground is more than when they're lying down. And because of that pressure differential, they usually have worsening headaches when standing up or sitting up. The other thing are severe headaches, which would be a sign of a subdural hematoma or focality in their neurological symptoms that could point to a subdural hematoma, such as weakness, numbness, speaking problems, depending on the hemisphere. How we work this up is, regardless if you're concerned about overdrainage or underdrainage, we usually start with a CAT scan or an MRI scan. Typically, we prefer a CAT scan because it's quicker, but the CAT scan will show us if the ventricular caliber is the same and/or the placement of the proximal catheter. So, what we look for when we see that CAT scan or that MRI to see the location of the proximal catheter to make sure it hasn't changed from any previous settings. And then we see the caliber of the ventricles. If the caliber of the ventricles is smaller, that could be a sign of overdrainage. If the caliber of the ventricles are larger, it could be a sign of underdrainage. The other thing we look for are subdural fluid collections or hydromas or subdural hematomas, which would be another sign of lower endocranial pressure, which would be a sign of overdrainage. So those are the biggest signs we look for, for underdrainage and overdrainage. Other things we can look for if we're concerned of the shunt is fractured, we do a shunt X-ray and what a shunt x-ray is is x-rays of the skull, the neck and the abdomen to see the catheter to make sure it's not kinked or fractured. If you're really concerned, you can't tell from the x-ray, another scan to order is a CT of the chest and abdomen and pelvis to look at the location of the catheter to make sure there's no brakes in the catheter, there's no fluid collections on the distal portion of the catheter, which would be a sign of shunt malfunction as well. Other tests that you can do to really exclude shunt malfunction is a shunt patency test, and what that is a nuclear medicine test where radionucleotide is injected into the valve and then the radionucleotide is traced over time or imaged through time to make sure that it's draining appropriately from the valve into the distal catheter into the peritoneum or the distal site. If there's a shunt malfunction that's not drainage, that radioisotope would remain stagnant either in the valve or in the catheter. There's overdrainage, we can't really tell, but there will be a quick drainage of the radioisotope. For shunt infection, we start with an imaging just to make sure there's not a shunt malfunction, and that usually requires cerebrospinal fluid to test. The cerebrospinal fluid can come from the valve itself, or it can come from other areas like the lumbar spine. If the lumbar spine is showing signs of shunt infection, then that usually means the shunt is infected. If the valve is aspirated with- at the bedside with a butterfly needle into the valve and that shows signs of shunt infection, that also could be a sign of infection. Dr Berkowitz: That's very helpful. You mentioned CT and shunt series. One question that often comes up when obtaining neuroimaging in patients with a shunt, who have NPH or otherwise, is whether we need to call you when we're doing an MRI to reprogram the shunt before or after. Is there a way we can know as a neurologists at the bedside or as patients carry a card, like with some devices where we know whether we have to call and bother our neurosurgery colleagues to get this MRI? Or if the radiology techs ask us, is this safe? And is the patient's shunt going to get turned off? How do we go about determining this? Dr Chaichana: Yeah, so unfortunately, a lot of patients don't carry a card. We typically offer a card when we do the shunt, but that card, there's two problems with it. One is it tells the model, but the second thing is it has to be updated any time the shunt is changed to a different setting. Oftentimes patients don't know that shunt setting, and often times they don't know that company brand that they use. There are different types of shunts with different types of settings. If there's ever concern as to what type of shunt they have, an x-ray is usually the best bet to see with a shunt series, or a skull x-ray. A lateral skull x-ray usually looks at the valve, and the valve has certain radio-dense markers that indicate what type of shunt it is. And that way you can call neurosurgery and we can always tell you what the shunt setting is before the MRI is done. Problem with an MRI scan if you do it without a shunt x-ray before is that you don't know the setting before unless the patient really knows or it's in the patient chart, and the MRI can need to change the setting. It doesn't usually turn it off, but it would change the setting, which would change the fluid dynamics within their ventricular system, which could lead to overdrainage or underdrainage. So, any time a patient needs MRI imaging, whether it's even the brain MRI, a spine MRI, or even abdominal MRI, really a shunt x-ray should be done just to see the shunt setting so that it could be returned to that setting after the MRI is done. Dr Berkowitz: So, the only way to know sort of what type of shunt it would be short of the patient knowing or the patient getting care at the same hospital where the shunt was placed and looking it up in the operative reports would be a skull film. That would then tell us what type of shunt is there and then the marking of the setting. And then we would be able to call our colleagues in neurosurgery and say, this patient is getting an MRI this is the setting, this is the type of shunt. And do we need to call you afterwards to come by and reprogram it? Is that right? Dr Chaichana: That's correct, yeah. Dr Berkowitz: Is there anything we would be able to see on there, or it's best we just- best we just call you and clarify? Dr Chaichana: The easiest thing to do is, when you get the skull x-ray, you can Google different types of shunts or search for different shunts, and they'll have markers that show the type of shunt it is as well as the setting that it's at. And just match it up with the picture. Dr Berkowitz: And as long as it's not a programmable shunt, there's no concern about doing the MRI. Is that right? Dr Chaichana: Correct. So, if it's a programmable shunt, even if it's MRI-compatible, we still like to get the setting before and make sure the setting after the MRI is the same. Nonprogrammable shunts can't be changed with MRI scans, and those don't need neurosurgery after the MRI scan, but it should be confirmed before the scan is done. Dr Berkowitz: Very helpful. Okay, so let's turn to NPH specifically. As you know, there's a lot of debate in the literature, some arguing, even, NPH might not even exist, some saying it's underdiagnosed. I think. I don't know if it was last year at our American Academy of Neurology conference or certainly in recent years, there was a pro and con debate of “we are underdiagnosing NPH” versus “we are overdiagnosing NPH.” What's your perspective as a neurosurgeon? What's the perspective in neurosurgery? Is this something we're underdiagnosing, and the times you shunt these patients you see miraculous results? Is this something that we're overdiagnosing, you get a lot of patients sent to that you think maybe won't benefit from a shunt? Or is it just really hard to say and some patients have shunt-responsive noncommunicating hydrocephalus of unclear etiology and either concurrent Parkinson's disease, Alzheimer's, cervical lumbar stenosis, neuropathy, vestibular problems, and all these other issues that play into multifactorial gait to sort of display a certain amount of the percentage of problem in a given patient or take overtime? What's your perspective if you're open to sharing it, or what's the perspective of neurosurgery? Is this debated as it is in neurology or this is just a standard thing you see and patients respond to shunt to some degree in some proportion of the time? And what are the sort of predictors you see in your experience? Dr Chaichana: Yeah, so, for me, I'd say it's too complicated for a neurosurgeon to evaluate. We rely on neurology to tell us whether or not they need a shunt. But I think the problem is, obviously, a part of the workout for at least the ones that I like to do, is that I want them to have a high-volume lumbar puncture with pre- and postgait analysis to see if there's really an objective measure of them improving. If they have an objective measure of improvement---and what's even better is that they have a subjective measure of improvement on top of the objective measure of improvement---then they benefit from a shunt. The problem is, some patients do benefit even though they don't have objective performance increases after a high-volume shunt. And those are the ones that make me the most worrisome to do the shunt, just because I don't like to do a procedure where there's no benefit for the patient. I do see, according to the literature as well, that there's around a 30 to 40%, even 50%, increase in gait function, even in patients that don't have large improvements following the high-volume lumbar puncture. And those are the most challenging patients for us as neurosurgeons because we'll put the shunt in, they say we're no better in terms of their gait, no better in terms of their urinary incontinence. We try to lower their shunt down to a certain setting and we're kind of stuck after that point. The good thing about NPH, though, is that, from the neurosurgery side, the shunt, like I said, is a pretty benign, low-risk procedure. So, we're not putting the patient through a very severe procedure to see if there's any benefit. So, in cases where we try to improve their quality of life in patients that don't have a benefit from high-volume lumbar puncture, we give them the odds of whether or not it's improving and say it might not improve. But because the procedure's minimally invasive, I think it's a good way to see if we can benefit their quality of life. Dr Berkowitz: Yeah, it's a very helpful perspective. Yeah, those are the most challenging cases on our side as well, right. If the patient- we think they may have NPH, or their gait and/or urinary and/or cognitive problems are- at least have a component of NPH that could be reversible, we certainly want to do the large volume lumbar puncture and/or consider a lumbar drain trial, all discussed in other articles and interviews for this issue of Continuum, But the really tough ones, as you said, there is this literature on patients who don't respond to the large-volume lumbar puncture for some reason but still may be shunt responsive. And despite all the imaging predictors and all the other ways we try to think about this, it's hard to know who's going to benefit. I think that's really a helpful perspective from your end that, as you say in the very beginning of your article, right, maybe there's a little bit too much fear of shunting on the neurology side because when we hear about shunts, it's often in the setting of complication. And so, we're not sort of getting the full spectrum of all the patients you shunt and you see who are doing just fine. They might not improve---the question is related to NPH---but at least they're not harmed by the shunt, and we're maybe overbiased and/or seeing a overly representative sample of negative shunt outcomes when they're actually not that common in practice. Is that a fair summary of your perspective? Dr Chaichana: Yeah, that's correct. So, I mean, complications can occur---and anytime you do a surgery, there are risks of complications---but I think they're relatively low for the benefit that we can help their quality of life. And the procedure's pretty short. So, the risk, it mostly outweighs the benefits in cases with normal pressure hydrocephalus. Dr Berkowitz: Very helpful perspective. So, well, thanks so much again. Today I've been interviewing Dr Kaisorn Chaichana about his article on management of normal pressure hydrocephalus, which he wrote with Dr Jeremy Cutsforth-Gregory. This article appears in the most recent issue of Continuum on disorders of CSF dynamics. Be sure to check out Continuum Audio episodes from this and other issues, and thank you to our listeners for joining us today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

In this episode of Longwood GradCast: Beyond the Degree, host Dr. Sarah Tanner-Anderson sits down with Daija Fowler, a speech-language pathologist whose journey from psychology major to Communication Sciences and Disorders leader highlights the power of purpose, mentorship, and professional reinvention.Daija traces her path back to her undergraduate days at UNC Greensboro, where a shift in academic focus—and a few pivotal conversations with her mom and a childhood friend—led her to discover a calling in Communication Sciences and Disorders. That moment of clarity set her on a path toward Longwood University, where she pursued her Master's degree in speech-language pathology.A first-generation college student, Daija speaks openly about the challenges she faced navigating graduate school applications, tuition costs, and the weight of doing it all without a roadmap. But it was at Longwood where she found her footing—and her voice. As a graduate assistant in the CSD department, Daija stepped into roles that demanded precision, organization, and adaptability. With the mentorship of faculty like Dr. King, she not only helped run the behind-the-scenes operations of the clinic but also developed a deeper appreciation for the profession she was preparing to enter.Now working full-time in the school system, Daija reflects on her transition from private practice to public education while maintaining her original clients in an after-school capacity. From the nerves of her first interviews to the satisfaction of helping students meet their communication goals, Daija shares how each stage of her journey has deepened her confidence and commitment. Her passion for augmentative and alternative communication (AAC) and fluency—specialties she didn't expect to fall in love with—demonstrates the value of staying open to new possibilities.Throughout the episode, Daija shines a light on the often-overlooked complexities of the SLP role. From diagnostics to collaboration with classroom teachers, she makes it clear that speech-language pathology is about far more than “teaching kids to talk.” It's about connection, growth, and giving students the tools to be heard.A standout moment in the conversation is Daija's tribute to the mentors who helped shape her: Dr. King, Dr. Wallace, and Dr. Walker. Their belief in her potential fueled her drive and underscored the lifelong impact of supportive academic relationships.As she looks ahead, Daija's considering ways to share her story more broadly—possibly through a YouTube channel—to support others navigating the early stages of their SLP careers. Describing her journey in three words—authentic, adaptable, creative—Daija's story is one of resilience, resourcefulness, and the courage to grow beyond expectations.Subscribe now to hear more powerful stories from Lancers making a difference beyond the degree—and follow us to stay connected: Facebook: @longwoodgraduatestudies Instagram: @longwoodgradstudies Twitter: @longwoodcgps YouTube: @longwoodgradstudies Website: www.Longwood.edu/Gradprograms

Trying to get back into the flow. I still don't have a desk or recording equipment. My phone will have to do the job. This is a quick pebble in the road on what is a mental health disorder.   Mental health disorders can affect a person's mood, feelings or become intrusive thoughts.   Most of us have experienced high stress periods or feeling like it's too much or way too much, or you really can't stand it anymore.    For those of us with continuous anxiety symptoms, it leans more toward being a disorder.  Or a condition.   Now, it's really important that you don't self-diagnose. You really should have a thorough workup by your doctor because there are so many physical health problems that have the exact same symptoms as an anxiety symptoms.    I just want you to have a better understanding of what those terms mean.    Bottom line, you have anxiety, you have anxiety symptoms, and they seem to be going on for a long time. It could be a condition. It could be a disorder.    Or it could just be something that you're dealing with and you're learning how to handle.  If you need support contact the National Suicide Prevention Lifeline at 988 or 1-800-273-8255, the Trevor Project at 1-866-488-7386 or text “START” to 741-741. Resources Mentioned:  The World Health Organization has an information page on mental disorders.    The American Psychiatric Association has page on What is Mental Illness?. The site uses condition and disorder.  Disclaimer:  Links to other sites are provided for information purposes only and do not constitute endorsements.  Always seek the advice of a qualified health provider with questions you may have regarding a medical or mental health disorder. This blog and podcast is intended for informational and educational purposes only. Nothing in this program is intended to be a substitute for professional psychological, psychiatric or medical advice, diagnosis, or treatment.    

So... my little blue vibrator did me dirty.

《生技來一刻》即將滿五歲了。眼看我們一起走過了五個年頭。你是否曾經想過，這些 podcast 故事是怎麼製作的呢？又需要什麼樣的魔法才能讓這些故事完美呈現呢？而，又是誰在施展這樣的魔法呢？ 在第五季的尾聲，讓我們一起來認識《生技來一刻》的幕後團隊和 podcast 製作的故事。我們會分享是什麼讓後製疲於奔命，又是什麼讓製作人頭痛欲裂，關掉麥克風後又有什麼秘辛。讓我們一起來聽聽生技來一刻團隊的背後故事，認識施展魔法的小精靈們吧！ 因為小精靈人數眾多，幕後故事會分成上下集。沒有聽到你心心念念的那個工作人員的心聲嗎？那就千萬不要錯過兩週後的下集。   《生技來一刻》感謝國科會與駐波士頓辦事處科技組贊助我們製作節目。我們也歡迎聽眾的小額捐款，您的支持能幫助我們製作更優質的節目。https://www.paypal.com/US/fundraiser/charity/2101877  

with Charlie GammellCheck out our Bookshop.org affiliate site behindthelines and please sign up for my substack at arthursnell.substack.com and/or follow me on Bluesky@snellarthur.bsky.social. You can sometimes find me on other podcasts - most often Disorder which I am involved with in partnership with RUSI, the Royal United Services Institute, the world's oldest think tank. Hosted on Acast. See acast.com/privacy for more information.

The UN is demonstrably powerless in Gaza; unable to facilitate elections in Libya; and blocked by Russia from doing anything meaningful on major issues, like Ukraine, Climate Change, Tarriffs, or the Arctic. Is International Diplomacy Dead? Where is the UN in pushing back against Trump? Is there any way that countries can finally put their differences aside on major divisive issues -- like Ukraine and Gaza -- but work together on other more unifying ones – like kleptocracy and climate change – and finally Order the Disorder? To find out, Jason is joined by Stephanie Williams – Former top United Nations mediator and a senior American diplomat. Stephanie served as the Special Adviser to the Secretary General on Libya, as well as acting UN SRSG/Special Envoy to Libya. She is the author of an excellent and highly detailed new book on international diplomacy, ‘Libya Since Qaddafi: Chaos and the Search for Peace'. As with Jason's own book, Stephanie's is with Hurst/OUP, by far the best publishers of cutting edge books on Libya.  The duo delve into the effectiveness of the UN and the international community in addressing global conflicts, particularly focusing on Libya. Plus they tackle: the complexities of state building, the Disordering influence of Russia on the operations of the UN, the impact of US foreign policy on Libya, and why incoherence and coordination complexities within the West makes it nearly impossible to do diplomacy anymore. And as they Order the Disorder, Jason and Stephanie propose genuine things Steve Witkoff and others can do to formulate a more unified approach to major issues of the day. Producer: George McDonagh Subscribe to our Substack - https://natoandtheged.substack.com/ Disorder on YouTube - https://www.youtube.com/@DisorderShow  Show Notes Links: Get Stephanie Williams' book, Libya Since Qaddafi: Chaos and the Search for Peace - https://www.hurstpublishers.com/book/libya-since-qaddafi/  Get Jason's book Libya and the Global Enduring Disorder - https://www.hurstpublishers.com/book/libya-and-the-global-enduring-disorder/  Watch Why Regime Change in Iran is Impossible - https://www.youtube.com/watch?v=UlX5_mp1JWc   Listen back to Ep4. Who is leading the disorder? https://pod.link/1706818264/episode/9ebdc18174d18cb019d148053a98e287  Learn more about your ad choices. Visit megaphone.fm/adchoices

In this episode, Dr Joost Groen, a clinical biochemist at the University Medical Center Groningen, and Dr Matt Gentry, Professor & Chair of Biochemistry & Molecular Biology in the College of Medicine at University of Florida, join Rodrigo and Silvia to discuss new insights, AI, cancer metabolism and some of their favourite papers on Glycogen Storage Disorders. Authors opinions are their own and do not represent their institutions. GSD episode papers: Brain glycogen serves as a critical glucosamine cache required for protein glycosylation. Sun et al A machine learning model accurately identifies glycogen storage disease Ia patients based on plasma acylcarnitine profiles. Groen et al Small-molecule inhibition of glycogen synthase 1 for the treatment of Pompe disease and other glycogen storage disorders. Ullman et al Repurposing SGLT2 inhibitors: Treatment of renal proximal tubulopathy in Fanconi-Bickel syndrome with empagliflozin. Overduin et al Gross-Valle The relation between dietary polysaccharide intake and urinary excretion of tetraglucoside. Gross-Valle et al Glycogen drives tumour initiation and progression in lung adenocarcinoma. Clarke HA et al Spatial metabolomics reveals glycogen as an actionable target for pulmonary fibrosis. Conroy et al In situ mass spectrometry imaging reveals heterogeneous glycogen stores in human normal and cancerous tissues. Young et al Glycogen accumulation modulates life span in a mouse model of amyotrophic lateral sclerosis. Brewer et al Dynamics of cognitive variability with age and its genetic underpinning in NIHR BioResource Genes and Cognition cohort participants. Rahman MS et al Neurological glycogen storage diseases and emerging therapeutics Colpaert et al

Clarence Ford spoke to Dr Themba Hadebe, Clinical Executive at Bonitas on Seasonal Affective Disorder (SAD) and how it often gets dismissed as “winter blues” Views and News with Clarence Ford is the mid-morning show on CapeTalk. This 3-hour long programme shares and reflects a broad array of perspectives. It is inspirational, passionate and positive. Host Clarence Ford’s gentle curiosity and dapper demeanour leave listeners feeling motivated and empowered. Known for his love of jazz and golf, Clarrie covers a range of themes including relationships, heritage and philosophy. Popular segments include Barbs’ Wire at 9:30am (Mon-Thurs) and The Naked Scientist at 9:30 on Fridays. Thank you for listening to a podcast from Views & News with Clarence Ford Listen live on Primedia+ weekdays between 09:00 and 12:00 (SA Time) to Views and News with Clarence Ford broadcast on CapeTalk https://buff.ly/NnFM3Nk For more from the show go to https://buff.ly/erjiQj2 or find all the catch-up podcasts here https://buff.ly/BdpaXRn Subscribe to the CapeTalk Daily and Weekly Newsletters https://buff.ly/sbvVZD5 Follow us on social media: CapeTalk on Facebook: https://www.facebook.com/CapeTalk CapeTalk on TikTok: https://www.tiktok.com/@capetalk CapeTalk on Instagram: https://www.instagram.com/ CapeTalk on X: https://x.com/CapeTalk CapeTalk on YouTube: https://www.youtube.com/@CapeTalkSee omnystudio.com/listener for privacy information.

Next dates: July 19 - Balearic London @ 17 Little Portland Street, London | Aug 16 - Balearic London x Love Dancing @ We Out Here, Dorset Follow me on Instagram Turned On is supported by my Patreon followers. If you want to show your love for my podcast and what I do, you can subscribe to my Patreon for £2 a month to support me and in return you can enjoy perks like guestlist benefits for my gigs, free downloads of my edits before anyone else, full tracklists for live recordings, exclusive previews of my tracks and feedback on your tracks if you're a producer. Or turn a friend on to Turned On by giving this podcast a 5-star review, reposting it on Mixcloud or SoundCloud or sending it to a friend. Follow me on Songkick to receive alerts when I'm playing near you  Bookings: info@bengomori.com Discover more new music + exclusive premieres on our SoundCloud  Follow the Turned On Spotify playlist, with 1000s of tracks played on this show and in my sets. Turned On is powered by Inflyte – the world's fastest growing music promo platform. Orbita - Batuki [Tiff's Joints] Cesare & Disorder & Weg - Always Here [Serialism Records]  Tiffany – In The Dark (Nofraje Edit) [Bandcamp] Roman Truth - Rock Of Truth (Roman Truth Edit) [?] Munk & Kapote feat. George Kranz - La Musica (Din Daa Daa Version) [Toy Tonics] Luca Olivotto - Passion [803 Crystal Grooves]  Gary's Garage - Gary Loves Garage [Gary's Garage] Louie Vega feat. Moodymann - Seven Mile (Charles Levine Wild Version) [Nervous Records] The Cure - A Fragile Thing (Sally C Remix) [Universal] La La x Ilario Alicante - Patched Up [Nervous Records]  Klubbheads - Klubhopping [Armada Music] Kwench - The Right Size (Techno Mix) [DJ World] Future Classic: Strath - Julia [HE.SHE.THEY]

Pastor Micah Klutinoty brings a message from Psalm 115:1-13. What we worship shapes who we become. If we give glory to lifeless idols, we become like them—empty and powerless—but if we worship the living God, we reflect His life, blessing, and glory. True worship rightly assigns glory to God alone and transforms us to be more like Him.

In episode 62 we discuss the article “Oral vs Extended-Release Injectable Naltrexone for Hospitalized Patients with Alcohol Use Disorder.” Magane KM, et al.Oral vs Extended-Release Injectable Naltrexone for Hospitalized Patients With Alcohol Use Disorder: A Randomized Clinical Trial. JAMA Intern Med. 2025 Jun 1;185(6):635-645. We also discuss reduced overdose deaths and changes in buprenorphine labelling to include higher doses. NPR:Drug deaths plummet among young Americans as fentanyl carnage eases Federal Register:Modifications to Labeling of Buprenorphine-Containing Transmucosal Products for the Treatment of Opioid Dependence --- This podcast offers category 1 and MATE-ACT CME credits through MI CARES and Michigan State University. To get credit for this episode and others, go tothis link to make your account, take a brief quiz, and claim your credit. To learn more about opportunities in addiction medicine, visitMI CARES. CME: https://micaresed.org/courses/podcast-addiction-medicine-journal-club/ --- Original theme music:composed and performed by Benjamin Kennedy Audio editing: Michael Bonanno Executive producer:Dr. Patrick Beeman A podcast fromArs Longa Media --- This is Addiction Medicine Journal Club with Dr. Sonya Del Tredici and Dr. John Keenan. We practice addiction medicine and primary care, and we believe that addiction is a disease that can be treated. This podcast reviews current articles to help you stay up to date with research that you can use in your addiction medicine practice. Learn more about your ad choices. Visit megaphone.fm/adchoices

Dan Jones, retired EPS member (spent time with the gang's unit and homicide team), chair of justice studies, NorQuest College Learn more about your ad choices. Visit megaphone.fm/adchoices

Contact Welcomed HereMOD episodes have no shelf-life or expiration date.  Our focus is principled so each episode reflects the immediate peace and stability of our Ever-Present Knowing Awareness that clarifies conflicted impressions manifest by the instability and hyper-reactivity of obsessive thoughts purely induced, “imagined reality”.While the Absolute nature of Life is an unadulterated gift - we earn our human experience. Good or bad, like it or not, it is how and what we think of Life, Reality, and Truth that complicate all the consequential forms of conflicted dysfunctional incapacities incorrectly blamed on life and human nature. It is not Life that is impossible, hard, or a struggle but imagining that our thoughts about it are omnipotent and as a result unchangeable. This addictive mental trend has the world of things and people seem in chronic need of change while we choose to inflict others with our thoughts to pretend we are not capable of causing such conflicts. This induced form of insanity once seen clearly is laughable. The only thoughts induced insanity seemingly overlook are the mistaken beliefs of their perpetrator. Acting like we're in a coma to maintain unnatural conditions does not affect our essence but causes over-dramatic mental and inter-reactive consequences. We choose how and what we think - and this news is good or bad dependent on how accurately, or honestly, our choices are conceded.  We Know We Know. We Are Aware We Are Aware. We Are as We Are. Reality is unlimited and never changes. The idea that how and what we think creates reality suggests otherwise. Acting on backward thoughts leads to behaviors that are out of order reflecting a reversal of our natural fortune. Anxious, nervous and systemic disorders reflect this impossible attempt to reverse the Law and order of the universe. Dis-ease is the lack of ease created and maintained by such twisted mental acrobatics. Stress and Anxiety inhibit healing and so contribute to any ailments. Mentality is a bodily function. Mental disease is a physcial ailment leading to others as long as it is improperly diagnosed - any treatment will perpetuate its contagion. Principles affirm our indivisible nature. Sharing principles affirms and confirms our indivisibility as our nature. Inspiration is natural while desperation, depression, degradation and acting oblivious to what is obvious is a choice. Absolute Intelligence, Peace and Silence is Nature's nature since It does not change. The nature of nature is naturally our nature. Ignoring what is happening, acting as though it shouldn't be or isn't happening, produces the unintelligible jibbersh of ignorance - not reality.

COMPUTER

No guest this week, just a lot of news to talk about. Like what games does CMON have left? And why is Asmodee hiring their crowdfunding “expert”? Even I can read the tea leaves on why thats a bad idea. Also, would you pay $165 for a Magic the Gathering event? I don't think I would.

Welcome or welcome back to Authentically ADHD, the podcast where we embrace the chaos and magic of the ADHD brain. Im carmen and today we're diving into a topic that's as complex as my filing system (which is to say, very): ADHD and its common co-occurring mood and learning disorders. Fasten your seatbelts (and if you're like me, try not to get distracted by the shiny window view) – we're talking anxiety, depression, OCD, dyslexia, dyscalculia, and bipolar disorder, all hanging out with ADHD.Why cover this? Because ADHD rarely rides solo. In fact, research compiled by Dr. Russell Barkley finds that over 80% of children and adults with ADHD have at least one other psychiatric disorder, and more than half have two or more coexisting conditions. Two-thirds of folks with ADHD have at least one coexisting condition, and often the classic ADHD symptoms (you know, fidgeting, daydreaming, “Did I leave the stove on?” moments) can overshadow those other disorders. It's like ADHD is the friend who talks so loud at the party that you don't notice the quieter buddies (like anxiety or dyslexia) tagging along in the background.But we're going to notice them today. With a blend of humor, sass, and solid neuroscience (yes, we can be funny and scientific – ask me how I know!), we'll explore how each of these conditions shows up alongside ADHD. We'll talk about how they can be misdiagnosed or missed entirely, and—most importantly—we'll dish out strategies to tell them apart and tackle both. Knowledge is power and self-awareness is the key, especially when it comes to untangling ADHD's web of quirks and comrades in chaos. So, let's get into it!ADHD and Anxiety: Double Trouble in OverdriveLet's start with anxiety, ADHD's frequent (and frantic) companion. Ever had your brain ping-pong between “I can't focus on this work” and “I'm so worried I'll mess it up”? That's ADHD and anxiety playing tango in your head. It's a double whammy: ADHD makes it hard to concentrate, and anxiety cranks up the worry about consequences. As one study notes, about 2 in 5 children with ADHD have significant problems with anxiety, and over half of adults with ADHD do as well. In other words, if you have ADHD and feel like a nervous wreck half the time, you're not alone – you're in very good (and jittery) company.ADHD and anxiety can look a lot alike on the surface. Both can make you restless, unfocused, and irritable. I mean, is it ADHD distractibility or am I just too busy worrying about everything to pay attention? (Hint: it can be both.) Especially for women, ADHD is often overlooked and mislabeled as anxiety. Picture a girl who can't concentrate in class: if she's constantly daydreaming and fidgety, one teacher calls it ADHD. Another sees a quiet, overwhelmed student and calls it anxiety. Same behavior, different labels. Women in particular have had their ADHD misdiagnosed as anxiety or mood issues for years, partly because anxious females tend to internalize symptoms (less hyperactive, more “worrier”), and that masks the ADHD beneath.So how do we tell ADHD and anxiety apart? One clue is where the distraction comes from. ADHD is like having 100 TV channels in your brain and someone else is holding the remote – your attention just flips on its own. Anxiety, on the other hand, is like one channel stuck on a horror movie; you can't focus on other things because a worry (or ten) is running on repeat. An adult with ADHD might forget a work deadline because, well, ADHD. An adult with anxiety might miss the deadline because they were paralyzed worrying about being perfect. Both end up missing the deadline (relatable – ask me how I know), but for different reasons.Neuroscience is starting to unravel this knot. There's evidence of a genetic link between ADHD and anxiety – the two often run in the family together. In brain studies, both conditions involve irregularities in the prefrontal cortex (the brain's command center for focus and planning) and the limbic system (emotion center). Essentially, if your brain were a car, ADHD means the brakes (inhibition) are a bit loose, and anxiety means the alarm system is hyper-sensitive. Combine loose brakes with a blaring alarm and you get… well, us. Fun times, right?Here's an interesting tidbit: Females with ADHD are more likely to report anxiety than males. Some experts think this is partly due to underdiagnosed ADHD – many girls grew up being told they were just “worrywarts” when in fact ADHD was lurking underneath, making everyday life more overwhelming and thus feeding anxiety. As Dr. Thomas Brown (a top ADHD expert) points out, emotional regulation difficulties (like chronic stress or worry) are characteristic of ADHD, even though they're not in the official DSM checklist. Our ADHD brains can amplify emotions – so a normal worry for someone else becomes a five-alarm fire for us.Now, action time: How do we manage this dynamic duo? The first step is getting the right diagnosis. A clinician should untangle whether symptoms like trouble concentrating are from anxiety, ADHD, or both. They might ask: Have you always had concentration issues (pointing to ADHD), or did they start when your anxiety kicked into high gear? Also, consider context – ADHD symptoms occur in most settings (school, work, home), while pure anxiety might spike in specific situations (say, social anxiety in crowds, or panic attacks only under stress).Treatment has to tackle both. Therapy – especially Cognitive Behavioral Therapy (CBT) – is a rockstar here. CBT can teach you skills to manage worry (hello, deep breathing and logical rebuttals to “what if” thoughts) and also help with ADHD organization hacks (like breaking tasks down, creating routines). Many find that medication is needed for one or both conditions. Stimulant meds (like methylphenidate or amphetamines) treat ADHD, but in someone with severe anxiety, a stimulant alone can sometimes ramp up the jitters. In fact, children (and adults) with ADHD + anxiety often don't respond as well to ADHD meds unless the anxiety is also addressed. Doctors might add an SSRI or other anti-anxiety medication to the mix, or choose a non-stimulant ADHD med if stimulants prove too anxiety-provoking.Let me share a quick personal strategy (with a dash of humor): I have ADHD and anxiety, so my brain is basically an internet browser with 50 tabs open – and 10 of them are frozen on a spinning “wheel of doom” (those are the anxieties). One practical tip that helps me distinguish the two is to write down my racing thoughts. If I see worries like “I'll probably get fired for sending that email typo” dominating the page, I know anxiety is flaring. If the page is blank because I got distracted after one sentence... well, hello ADHD! This silly little exercise helps me decide: do I need to do some calming techniques, or do I need to buckle down and use an ADHD strategy like the Pomodoro method? Try it out: Knowledge is power, and self-awareness is the key.Quick Tips – ADHD vs Anxiety: When in doubt, ask what's driving the chaos.* Content of Thoughts: Racing mind full of specific worries (anxiety) vs. racing mind full of everything except what you want to focus on (ADHD).* Physical Symptoms: Anxiety often brings friends like sweaty palms, racing heart, and tummy trouble. ADHD's restlessness isn't usually accompanied by fear, just boredom or impulsivity.* Treatment Approaches: For co-occurring cases, consider therapy and possibly a combo of medications. Experts often treat the most impairing symptom first – if panic attacks keep you homebound, address that alongside ADHD. Conversely, untreated ADHD can actually fuel anxiety (ever notice how missing deadlines and forgetfulness make you more anxious? Ask me how I know!). A balanced plan might be, say, stimulant medication + talk therapy for anxiety, or an SSRI combined with ADHD coaching. Work closely with a professional to fine-tune this.Alright, take a breath (seriously, if you've been holding it – breathing is good!). We've tackled anxiety; now let's talk about the dark cloud that can sometimes follow ADHD: depression.ADHD and Depression: When the Chaos Brings a CloudADHD is often associated with being energetic, spontaneous, even optimistic (“Sure, I can start a new project at 2 AM!”). So why do so many of us also struggle with depression? The reality is, living with unmanaged ADHD can be tough. Imagine years of what Dr. Russell Barkley calls “developmental delay” in executive function – always feeling one step behind in managing life, despite trying so hard. It's no surprise that about 1 in 5 kids with ADHD also has a diagnosable depression, and studies show anywhere from 8% to 55% of adults with ADHD have experienced a depressive disorder in their lifetime. (Yes, that range is huge – it depends how you define “depression” – but even on the low end it's a lot.) Dr. Barkley himself notes that roughly 25% of people with ADHD will develop significant depression by adulthood. In short, ADHD can come with a case of the blues (not the fun rhythm-and-blues kind, unfortunately).So what does ADHD + depression look like? Picture this: You've got a pile of unfinished projects, bills, laundry – the ADHD “trail of crumbs.” Initially, you shrug it off or maybe crack a joke (“organizational skills, who's she?”). But over time, the failures and frustrations can chip away at your self-esteem. You start feeling helpless or hopeless: “Why bother trying if I'm just going to screw it up or forget again?” That right there is the voice of depression sneaking in. ADHD's impulsivity might also lead to regrettable decisions or conflicts that you later brood over, another pathway to depressed mood.In fact, the Attention Deficit Disorder Association points out that ADHD's impact on our lives – trouble with self-esteem, work or school difficulties, and strained relationships – can contribute to depression. It's like a one-two punch: ADHD creates problems; those problems make you sad or defeated, which then makes it even harder to deal with ADHD. Fun cycle, huh?Now, depression itself can mask as ADHD in some cases, especially in adults. Poor concentration, low motivation, fatigue, social withdrawal – these can appear in major depression and look a lot like ADHD symptoms. If an adult walks into a doctor's office saying “I can't focus and I'm procrastinating a ton,” a cursory eval might yield an ADHD diagnosis. But if that focus problem started only after they, say, lost a loved one or fell into a deep funk, and they also feel worthless or have big sleep/appetite changes, depression may be the primary culprit. On the flip side, a person with lifelong ADHD might be misdiagnosed as just depressed, because they seem down or overwhelmed. As always, timeline is key: ADHD usually starts early (childhood), whereas depression often has a more defined onset. Also, ask: Is the inability to focus present even when life's going okay? If yes, ADHD is likely in the mix. If the focus issues wax and wane with mood, depression might be the driver.There's also a nuance: ADHD mood issues vs. clinical depression. People with ADHD can have intense emotions and feel demoralized after a bad day, but often these feelings can lift if something positive happens (say, an exciting new interest appears – suddenly we have energy!). Clinical depression is more persistent – even good news might not cheer you up much. As Dr. Thomas Brown emphasizes, ADHD includes difficulty regulating emotion; an ADHD-er might feel sudden anger or sadness that's intense but then dissipates . By contrast, depression is a consistent low mood or loss of pleasure in things over weeks or months. Knowing this difference can be huge in sorting out what's going on.Now, how do we deal with this combo? The good news: many treatments for depression also help ADHD and vice versa. Therapy is a prime example. Cognitive Behavioral Therapy and related approaches can address negative thought patterns (“I'm just a failure”) and also help with practical skills for ADHD (like scheduling, or as I call it, tricking my brain into doing stuff on time). There are even specialized therapies for adults with ADHD that blend mood and attention strategies. On the medication front, sometimes a single med can pull double duty. One interesting option is bupropion (Wellbutrin) – an antidepressant that affects dopamine and norepinephrine, which can improve both depression and ADHD symptoms in some people. There's also evidence that stimulant medications plus an antidepressant can be a powerful combo: stimulants to improve concentration and energy, antidepressant to lift mood. Psychiatrists will tailor this to the individual – for instance, if someone is severely depressed (can't get out of bed), treating depression first may be priority. If the depression seems secondary to ADHD struggles, improving the ADHD could automatically boost mood. Often, it's a balancing act of treating both concurrently – maybe starting an antidepressant and an ADHD med around the same time, or ensuring therapy covers both bases.Let's not forget lifestyle: exercise, sleep, nutrition – these affect both ADHD and mood. Regular exercise, for example, can increase BDNF (a brain growth factor) and neurotransmitters that help both attention and mood. Personally, I found that when I (finally) started a simple exercise routine, my mood swings evened out a bit and my brain felt a tad less foggy. (Of course, starting that routine required overcoming my ADHD inertia – ask me how I know that took a few tries... or twenty.)Quick Tips – ADHD vs Depression:* Check Your Joy Meter: With ADHD alone, you can still feel happy/excited when something engaging happens (ADHD folks light up for interesting tasks!). With depression, even things you normally love barely register. If your favorite hobbies no longer spark any joy, that's a red flag for depression.* All in Your Head? ADHD negative thoughts sound like “Ugh, I forgot again, I need a better system.” Depression thoughts sound like “I forgot again because I'm useless and nothing will ever change.” Listen to that self-talk; depression is a sneaky bully.* Professional Help: A thorough evaluation can include psychological tests or questionnaires to measure attention and mood separately. For treatment, consider a combined approach: therapy (like CBT or coaching) plus meds as needed. According to research, a mix of stimulant medication and therapy (especially CBT) can help treat both conditions. And remember, addressing one can often relieve the other: improve your ADHD coping skills, and you might start seeing hope instead of disappointment (boosting mood); treat your depression, and suddenly you have the energy to tackle that ADHD to-do list.Before we move on, one more important note: if you ever have thoughts of self-harm or suicide, please reach out to a professional immediately. Depression is serious, and when compounded with ADHD impulsivity, it can be dangerous. There is help, and you're not alone – so many of us have been in that dark place, and it can get better with the right support. Knowledge is power and self-awareness is the key, yes, but sometimes you also need a good therapist, maybe a support group, and possibly medication to truly turn things around. There's no shame in that game.Alright, deep breath. It's getting a bit heavy in here, so let's pivot to something different: a condition that seems like the opposite of ADHD in some ways, yet can co-occur – OCD. And don't worry, we'll crank the sass back up a notch.ADHD and OCD: The Odd Couple of AttentionWhen you think of Obsessive-Compulsive Disorder (OCD), you might picture someone extremely organized, checking the stove 10 times, everything neat and controlled. When you think ADHD… well, “organized” isn't the first word that comes to mind, right?

Why is cannabis illegal in the UK? This is a vexed question that dates back to colonial times but continues to be a hot button issue in the present day. Indeed, Charlie Falconer recently chaired a report for the London Drugs Commission on this very topic, recommending that cannabis possession be decriminalised but dealing remain outlawed. Is this an elegant solution or a mealy-mouthed fudge? To debate the issue, Charlie is joined by his regular companions – Nicholas Mostyn and Helena Kennedy – as well as Professor Mike Barnes, a renowned neurologist with a particular interest in medical cannabis, and Peter Reynolds, president of the cannabis campaign group CLEAR UK.If you have questions, criticisms, praise or other feedback, please do send your thoughts to us via lawanddisorderfeedback@gmail.com!Law and Disorder is a Podot podcast.Hosted by: Charlie Falconer, Helena Kennedy, Nicholas Mostyn.Executive Producer and Editor: Nick Hilton.Associate Producer: Ewan Cameron.Music by Richard Strauss, arranged and performed by Anthony Willis & Brett Bailey. Hosted on Acast. See acast.com/privacy for more information.

[Rerun] Dr Kirk Honda talks with Dr Jennifer Sampson about hoarding disorder.This episode is sponsored by BetterHelp. Give online therapy a try at betterhelp.com/KIRK to get 10% off your first month.Become a member: https://www.youtube.com/channel/UCOUZWV1DRtHtpP2H48S7iiw/joinBecome a patron: https://www.patreon.com/PsychologyInSeattleEmail: https://www.psychologyinseattle.com/contactWebsite: https://www.psychologyinseattle.comMerch: https://psychologyinseattle-shop.fourthwall.com/Instagram: https://www.instagram.com/psychologyinseattle/Facebook Official Page: https://www.facebook.com/PsychologyInSeattle/TikTok: https://www.tiktok.com/@kirk.hondaJanuary 25, 2016The Psychology In Seattle Podcast ®Trigger Warning: This episode may include topics such as assault, trauma, and discrimination. If necessary, listeners are encouraged to refrain from listening and care for their safety and well-being.Disclaimer: The content provided is for educational, informational, and entertainment purposes only. Nothing here constitutes personal or professional consultation, therapy, diagnosis, or creates a counselor-client relationship. Topics discussed may generate differing points of view. If you participate (by being a guest, submitting a question, or commenting) you must do so with the knowledge that we cannot control reactions or responses from others, which may not agree with you or feel unfair. Your participation on this site is at your own risk, accepting full responsibility for any liability or harm that may result. Anything you write here may be used for discussion or endorsement of the podcast. Opinions and views expressed by the host and guest hosts are personal views. Although, we take precautions and fact check, they should not be considered facts and the opinions may change. Opinions posted by participants (such as comments) are not those of the hosts. Readers should not rely on any information found here and should perform due diligence before taking any action. For a more extensive description of factors for you to consider, please see www.psychologyinseattle.com

In this episode, we explore methadone maintenance therapy for opioid use disorder, covering dosing strategies, recent regulatory changes, and safety considerations. Why do so many patients fail on methadone despite its proven effectiveness, and how can proper dosing make the difference between recovery and relapse? Faculty: Smita Das, M.D. Host: Richard Seeber, M.D. Learn more about our memberships here Earn 1 CME: Pharmacologic Management of Opioid Use Disorder Methadone for Managing OUD

Recorded 2025-07-10 02:58:08

I spoke to Dutch defence journalist and Russia expert Steven Derix about an important new revelation of Russia's use of chemical weapons in the war in Ukraine, happening as part of direct orders and thousands of times. You can find the original report herehttps://www.nrc.nl/nieuws/2025/07/04/nederlandse-inlichtingendiensten-russen-zetten-in-oekraine-op-grote-schaal-chemische-wapens-in-a4899319Check out our Bookshop.org affiliate site behindthelines and please sign up for my substack at arthursnell.substack.com and/or follow me on Bluesky@snellarthur.bsky.social. You can sometimes find me on other podcasts - most often Disorder which I am involved with in partnership with RUSI, the Royal United Services Institute, the world's oldest think tank. Hosted on Acast. See acast.com/privacy for more information.

Learn the warning signs of AUD, from drinking more than intended to neglecting responsibilities. This medical condition exists on a spectrum from mild to severe, but treatment works at any stage—despite the stigma that prevents many from seeking help. Learn more at https://sayarc.com Addiction Resource Center LLC. City: Yuba City Address: 1002 Live Oak Blvd. Website: https://sayarc.com

Bill O'Reilly reacts to a viral video of good samaritans saving a woman on a New York subway from a crazed maniac, and New York releasing the criminal. Learn more about your ad choices. Visit megaphone.fm/adchoices

 Normal pressure hydrocephalus (NPH) is a clinical syndrome of gait abnormality, cognitive impairment, and urinary incontinence. Evaluation of CSF dynamics, patterns of fludeoxyglucose (FDG) uptake, and patterns of brain stiffness may aid in the evaluation of challenging cases that lack typical clinical and structural radiographic features. In this episode, Katie Grouse, MD, FAAN, speaks with Aaron Switzer, MD, MSc, author of the article “Radiographic Evaluation of Normal Pressure Hydrocephalus” in the Continuum® June 2025 Disorders of CSF Dynamics issue. Dr. Grouse is a Continuum® Audio interviewer and a clinical assistant professor at the University of California San Francisco in San Francisco, California. Dr. Switzer is a clinical assistant professor of neurology in the department of clinical neurosciences at the University of Calgary in Calgary, Alberta, Canada. Additional Resources Read the article: Radiographic Evaluation of Normal Pressure Hydrocephalus Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Grouse: This is Dr Katie Grouse. Today I'm interviewing Dr Aaron Switzer about his article on radiographic evaluation of normal pressure hydrocephalus, which he wrote with Dr Patrice Cogswell. This article appears in the June 2025 Continuum issue on disorders of CSF dynamics. Welcome to the podcast, and please introduce yourself to our audience. Dr. Switzer: Thanks so much for having me, Katie. I'm a neurologist that's working up in Calgary, Alberta, Canada, and I have a special interest in normal pressure hydrocephalus. So, I'm very happy to be here today to talk about the radiographic evaluation of NPH. Dr Grouse: I'm so excited to have you here today. It was really wonderful to read your article. I learned a lot on a topic that is not something that I frequently evaluate in my clinic. So, it's really just a pleasure to have you here to talk about this topic. So, I'd love to start by asking, what is the key message that you hope for neurologists who read your article to take away from it? Dr. Switzer: The diagnosis of NPH can be very difficult, just given the clinical heterogeneity in terms of how people present and what their images look like. And so, I'd like readers to know that detailed review of the patient's imaging can be very helpful to identify those that will clinically improve with shunt surgery. Dr Grouse: There's another really great article in this edition of Continuum that does a really great job delving into the clinical history and exam findings of NPH. So, I don't want to get into that topic necessarily today. However, I'd love to hear how you approach a case of a hypothetical patient, say, where you're suspicious of NPH based on the history and exam. I'd love to talk over how you approach the imaging findings when you obtain an MRI of the brain, as well as any follow-up imaging or testing that you generally recommend. Dr. Switzer: So, I break my approach down into three parts. First, I want to try to identify ventriculomegaly and any signs that would support that, and specifically those that are found in NPH. Secondly, I want to look for any alternative pathology or evidence of alternative pathology to explain the patient's symptoms. And then also evaluate any contraindications for shunt surgery. For the first one, usually I start with measuring Evans index to make sure that it's elevated, but then I want to measure one of the other four measurements that are described in the article, such as posterior colossal angle zed-Evans index---or z-Evans index for the American listeners---to see if there's any other features that can support normal pressure hydrocephalus. It's very important to identify whether there are features of disproportionately enlarged subarachnoid space hydrocephalus, or DESH, which can help identify patients who may respond to shunt surgery. And then if it's really a cloudy clinical picture, it's complicated, it's difficult to know, I would usually go through the full evaluation of the iNPH radscale to calculate a score in order to determine the likelihood that this patient has NPH. So, the second part of my evaluation is to rule out evidence of any alternative pathology to suggest another cause for the patient's symptoms, such as neurodegeneration or cerebrovascular disease. And then the third part of my evaluation is to look for any potential contraindications for shunt surgery, the main one being cerebral microbleed count, as a very high count has been associated with the hemorrhagic complications following shunt surgery. Dr Grouse: You mentioned about your use of the various scales to calculate for NPH, and your article does a great job laying them out and where they can be helpful. Are there any of these scales that can be reasonably relied on to predict the presence of NPH and responsiveness to shunt placement? Dr. Switzer: I think the first thing to acknowledge is that predicting shunt response is still a big problem that is not fully solved in NPH. So, there is not one single imaging feature, or even combination of imaging features, that can reliably predict shunt response. But in my view and in my practice, it's identifying DESH, I think, is really important---so, the disproportionately enlarged subarachnoid space hydrocephalus---as well as measuring the posterior colossal angle. I find those two features to be the most specific. Dr Grouse: Now you mentioned the concept of the NPH subtypes, and while this may be something that many of our listeners are familiar with, I suspect that, like myself when I was reading this article, there are many who maybe have not been keeping up to date on these various subtypes. Could you briefly tell us more about these NPH subtypes? Dr. Switzer: Sure. The Japanese guidelines for NPH have subdivided NPH into three different main categories. So that would be idiopathic, delayed onset congenital, and secondary normal pressure hydrocephalus. And so, I think the first to talk about would be the secondary NPH. We're probably all more familiar with that. That's any sort of pathology that could lead to disruption in CSF dynamics. These are things like, you know, a slow-growing tumor that is obstructing CSF flow or a widespread meningeal process that's reducing absorption of CSF, for instance. So, identifying these can be important because it may offer an alternative treatment for what you're seeing in the patient. The second important one is delayed onset congenital. And when you see an image of one of these subtypes, it's going to be pretty different than the NPH because the ventricles are going to be much larger, the sulcal enfacement is going to be more diffuse. Clinically, you may see that the patients have a higher head circumference. So, the second subtype to know about would be the delayed onset congenital normal pressure hydrocephalus. And when you see an image of one of these subtypes, it's going to be a little different than the imaging of NPH because the ventricles are going to be much larger, the sulcal enfacement is going to be more diffuse. And there are two specific subtypes that I'd like you to know about. The first would be long-standing overt ventriculomegaly of adulthood, or LOVA. And the second would be panventriculomegaly with a wide foramen of magendie and large discernomagna, which is quite a mouthful, so we just call it PAVUM. The importance of identifying these subtypes is that they may be amenable to different types of treatment. For instance, LOVA can be associated with aqueductal stenosis. So, these patients can get better when you treat them with an endoscopic third ventriculostomy, and then you don't need to move ahead with a shunt surgery. And then finally with idiopathic, that's mainly what we're talking about in this article with all of the imaging features. I think the important part about this is that you can have the features of DESH, or you can not have the features of DESH. The way to really define that would be how the patient would respond to a large-volume tap or a lumbar drain in order to define whether they have this idiopathic NPH. Dr Grouse: That's really helpful. And for those of our listeners who are so inclined, there is a wonderful diagram that lays out all these subtypes that you can take a look at. I encourage you to familiarize yourself with these different subtypes. Now it was really interesting to read in your article about some of the older techniques that we used quite some time ago for diagnosing normal pressure hydrocephalus that thankfully we're no longer using, including isotope encephalography and radionuclide cisternography. It certainly made me grateful for how we've come in our diagnostic tools for NPH. What do you think the biggest breakthrough in diagnostic tools that are now clinically available are? Dr. Switzer: You know, definitely the advent of structural imaging was very important for the evaluation of NPH, and specifically the identification of disproportionately enlarged subarachnoid space hydrocephalus, or DESH, in the late nineties has been very helpful for increasing the specificity of diagnosis in NPH. But some of the newer technologies that have become available would be phase-contrast MRI to measure the CSF flow rate through the aqueduct has been very helpful, as well as high spatial resolution T2 imaging to actually image the ventricular system and look for any evidence of expansion of the ventricles or obstruction of CSF flow. Dr Grouse: Regarding the scales that you had referenced earlier, do you think that we can look forward to more of these scales being automatically calculated and reported by various software techniques and radiographic interpretation techniques that are available or going to be available? Dr. Switzer: Definitely yes. And some of these techniques are already in development and used in research settings, and most of them are directed towards automatically detecting the features of DESH. So, that's the high convexity tight sulci, the focally enlarged sulci, and the enlarged Sylvian fissures. And separating the CSF from the brain tissue can help you determine where CSF flow is abnormal throughout the brain and give you a more accurate picture of CSF dynamics. And this, of course, is all automated. So, I do think that's something to keep an eye out for in the future. Dr Grouse: I wanted to ask a little more about the CSF flow dynamics, which I think may be new to a lot of our listeners, or certainly something that we've only more recently become familiar with. Can you tell us more about these advances and how we can apply this information to our evaluations for NPH? Dr. Switzer: So currently, only the two-dimensional phase contrast MRI technique is available on a clinical basis in most centers. This will measure the actual flow rate through the cerebral aqueduct. And so, in NPH, this can be elevated. So that can be a good supporting marker for NPH. In the future, we can look forward to other techniques that will actually look at three-dimensional or volume changes over time and this could give us a more accurate picture of aberrations and CSF dynamics. Dr Grouse: Well, definitely something to look forward to. And on the topic of other sort of more cutting-edge or, I think, less commonly-used technologies, you also mentioned some other imaging modalities, including diffusion imaging, intrathecal gadolinium imaging, nuclear medicine studies, MR elastography, for example. Are any of these modalities particularly promising for NPH evaluations, in your opinion? Do you think any of these will become more popularly used? Dr. Switzer: Yes, I think that diffusion tract imaging and MR elastography are probably the ones to keep your eye out for. They're a little more widely applicable because you just need an MR scanner to acquire the images. It's not invasive like the other techniques mentioned. So, I think it's going to be a lot easier to implement into clinical practice on a wide scale. So, those would be the ones that I would look out for in the future. Dr Grouse: Well, that's really exciting to hear about some of these techniques that are coming that may help us even more with our evaluation. Now on that note, I want to talk a little bit more about how we approach the evaluation and, in your opinion, some of the biggest pitfalls in the evaluation of NPH that you've found in your career. Dr. Switzer: I think there are three of note that I'd like to mention. The first would be overinterpreting the Evans index. So, just because an image shows that there's an elevated Evans index does not necessarily mean that NPH is present. So that's where looking for other corroborating evidence and looking for the clinical features is really important in the evaluation. Second would be misidentifying the focally enlarged sulci as atrophy because when you're looking at a brain with these blebs of CSF space in different parts of the brain, you may want to associate that to neurodegeneration, but that's not necessarily the case. And there are ways to distinguish between the two, and I think that's another common pitfall. And then third would be in regards to the CSF flow rate through the aqueduct. And so, an elevated CSF flow is suggestive of NPH, but the absence of that does not necessarily rule NPH out. So that's another one to be mindful of. Dr Grouse: That's really helpful. And then on the flip side, any tips or tricks or clinical pearls you can share with us that you found to be really helpful for the evaluation of NPH? Dr. Switzer: One thing that I found really helpful is to look for previous imaging, to look if there were features of NPH at that time, and if so, have they evolved over time; because we know that in idiopathic normal pressure hydrocephalus, especially in the dash phenotype, the ventricles can become larger and the effacement of the sulci at the convexity can become more striking over time. And this could be a helpful tool to identify how long that's been there and if it fits with the clinical history. So that's something that I find very helpful. Dr Grouse: Absolutely. When I read that point in your article, I thought that was really helpful and, in fact, I'm guessing something that a lot of us probably aren't doing. And yet many of our patients for one reason or other, probably have had imaging five, ten years prior to their time of evaluation that could be really helpful to look back at to see that evolution. Dr. Switzer: Yes, absolutely. Dr Grouse: It's been such a pleasure to read your article and talk with you about this today. Certainly a very important and helpful topic for, I'm sure, many of our listeners. Dr. Switzer: Thank you so much for having me. Dr Grouse: Again, today I've been interviewing Dr Aaron Switzer about his article on radiographic evaluation of normal pressure hydrocephalus, which he wrote with Dr Patrice Cogswell. This article appears in the most recent issue of Continuum on disorders of CSF dynamics. Be sure to check out Continuum Audio episodes from this and other issues, and thank you to our listeners for joining today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

Claudio Milstein, PhD joins the podcast to discuss the concept of unified care in managing voice, swallowing, and upper airway disorders. Dr. Milstein explains the importance of interdisciplinary collaboration in optimizing patient outcomes and shares details about an exciting upcoming CME on unified care.

Send us a textScott and John hang with Troll for Trout (and many others) bassist and friend of the NMD Podcast, John Connors,  for a round of "Pick 5". Youtube: https://www.youtube.com/@narcissisticmusicdisorderJoin us on Facebook at NMD podcast group.nmdpodcast@gmail.com to contact us.Tell your friends!!Be sure to Like and Subscribe. Thanks for listening!

The verdict is in and let's be honest, the justice system is a hot mess. Also, we are tired of the N-Word Pass!! Huge shoutout to Chris Laflare and from Was It Good Though Podcast, Jazz and Jason for joining us. Become a Habitual Ish Talker and follow us on The App Formally Known As Twitter: twitter.com/TalkinIsh_PodJoin in on the conversation! E-Mail us at ⁠talkinishpod@gmail.com⁠Listen to the audio version: https://linktr.ee/TalkinIshPod00:00​​​ - Intro/Idle Chit Chat08:50 - Viewer Comments 26:24 - RTD 1: Diddy and the Verdict of Discontent 1:17:11 - RTD 2: Can We Stop With Giving The N-Word Pass?!2:09:46 - Wrap It Up, YO!!! (Closing)

Welcome to Part 3 of my mini-series answering your burning questions! Today, we're diving into the topic of verbal imitation.When it comes to verbal imitation and echoic goals, context is everything. These goals must be functional and meaningful to the individual child in order to support communication development.As an SLP collaborating with a BCBA, and vice versa, rely on assessment tools and an SLP's robust training in speech to identify and shape functional goals. It's crucial that we plan and program with intention—otherwise, we risk discouraging a child from verbalizingThe key? Collaborate early and often. Assess together. Plan together. Use resources like the Autism IEP Goal Bank (don't miss the freebie!). Then, have the SLP on the team focus on those targeted words and move into collaboratively supporting generalization into the natural environment for a robust bank of words.Looking ahead to 2026, we're excited to explore communication disorders more deeply over at ABA Speech Connection. Stay tuned—because sometimes, you don't know what you don't know. #autism #speechtherapy What's Inside:Summer mini-seriesStrategy to work collaboratively as BCBA and SLPsVerbal imitation goals Communication developmentMentioned In This Episode:Verbal Imitation Guide (Hack #19) Join our ethics course Speech Membership - ABA Speech  ABA Speech: Home

Today on Grounded: The Vestibular Podcast, we are talking all about CGRP medications. These are a class of drugs used to manage migraine attacks. Interestingly, people with migraine have a higher level of CGRP during migraine attacks, but also have a harder time breaking them down for whatever reason. In this episode, we'll dig into: What CGRP medications are How CGRP medications work Generic names of CGRP medications How effective CGRPs are How CGRPs interact with Botox Will insurance cover CGRP medications What step therapy is and how to navigate it If a multi-pronged approach is still worth it Whether you're newly diagnosed, still searching for answers, or supporting someone with VM, this episode was created as a resource for you. Tune in and discover if we're fans of CGRPs over here… or not—and everything you need to know about these medications. Links/Resources Mentioned: Vestibular Group Fit (code GROUNDED at checkout!)  More Links/Resources: ⁠The 4 Steps to Managing Vestibular Migraine ⁠The PPPD Management Masterclass⁠ ⁠What your Partner Should Know About Living with Dizziness⁠ ⁠The FREE Mini VGFit Workout⁠ ⁠The FREE POTS - safe Workouts⁠ ⁠Vestibular Group Fit (code GROUNDED at checkout for 15% off your first subscription cycle!) ⁠ Connect with Dr. Madison: ⁠@⁠⁠TheVertigoDoctor ⁠ ⁠@TheOakMethod⁠ ⁠@VestibularGroupFit⁠ Connect with Dr. Jenna @dizzy.rehab.therapist  Work with Dr. Madison 1:1, Vestibular Rehabilitation Therapy Vestibular Group Fit Small Group Coaching (offered throughout the year, sign up for our email list to learn when!) Why The Oak Method? Learn about it here! Love what you heard? Reviews really help us out! Please consider leaving one for us.  This podcast is for informational purposes only and may not be the best fit for you and your personal situation. It shall not be construed as medical advice. The information and education provided here is not intended or implied to supplement or replace professional medical treatment, advice, and/or diagnosis. Always check with your own physician or medical professional before trying or implementing any information read here. ————————————— vestibular migraine, VM, CGRP medications, CGRPs for migraine, calcitonin gene-related peptides, migraine disorder, migraine attacks, preventative CGRP, acute CGRP, Aimovig, Ajovy, CGRP injections, Vyepti, Ubrelvy, oral CGRP, are CGRPs covered by insurance, step therapy, comprehensive migraine treatment

In his roll as UK Foreign Secretary, David Lammy has often talked of his concept of progressive realism, but what does that mean? And how has that impacted British foreign policy over the past 12 months? In this episode of Disorder, Alex Hall Hall and Arthur Snell delve into the complexities of British diplomacy over the past year – offering a grade point for each area of British foreign policy. They assess the UK's relationships with the US and EU, the implications of NATO and defense strategies, their dealings with Ukraine amidst the ongoing conflict with Russia and the government's response to the humanitarian crisis in the Middle East.  To close – and Order the Disorder – the pair express the need for a clearer vision in foreign policy, greater engagement with the Global South, and a clearer moral backbone from Starmer and co. Producer: George McDonagh Subscribe to our Substack - https://natoandtheged.substack.com/ Disorder on YouTube - https://www.youtube.com/@DisorderShow  Show Notes Links: Read ‘Keir Starmer Has Missed His Chance to Make a Bold Break With the Past on Foreign Policy' by Alex Hall Hall: https://bylinetimes.com/2025/06/27/labour-foreign-policy-report-card/  Learn more about your ad choices. Visit megaphone.fm/adchoices

Could anxiety just be energy misinterpreted? Kate Mason sits down with clinical hypnotherapist and bestselling author Jake Yanitz Rubin to explore a radically new understanding of anxiety—not as a mental illness, but as a misunderstood part of the human experience. They dive deep into how reframing anxiety as energy can unlock personal growth, self-awareness, and even spiritual awakening. Jake shares powerful insights from his book From Anxiety to Awakening, practical mindset shifts, and life-changing tools rooted in psychology, ancient philosophy, and consciousness work. Listen For05:02 Panic, Purpose, and the Pivot Within10:12 What's Really Happening in Hypnosis?14:59 Anxiety Isn't a Disorder It's Energy19:08 You Are Not Your Thoughts26:50 The Present Moment is All There Is36:37 Be Your Own Frequency Live by Your Truth Leave a rating/review for this podcast with one click Connect with guest: Jake Yanitz Rubin, Author | Transformational Mentor | Spiritual Teacher | Clinical Hypnotherapist | 25+ Years Guiding Personal AwakeningWebsite | From Anxiety to Awakening Book | LinkedIn| InstagramContact Kate:Email | Website | Kate's Book on Amazon | LinkedIn | Facebook | X

Today I'm happy to chat with our patient Leslie who shares her successful journey with histamine intolerance healing. We'll go through how SIBO led to her histamine issues, the connection to low vagus nerve tone, as well as the low histamine diet and treatments such as dao enzymes. Tune in to learn how you can reduce histamine in the body naturally.    Start healing with us! Learn more about our virtual clinic:  https://drruscio.com/virtual-clinic/ Histamine Intolerance and Diet Guide: https://drruscio.com/guides/get-histamine-intolerance-guide/  

ADHD isn't a defect— it's a brilliant survival strategy.Heather McKean and co-host Kent dive into the neuroscience (dopamine, DMN, hyper-vigilance), childhood ACEs, and school-system pressures that wire an “ADHD brain.” Then they show how Mind Change tools re-route those patterns—no shame, no labels.Inside this conversationWhy a hyperactive brain is often a hyper-vigilant brainDopamine “hunger” vs. true connectionHow compliance-based classrooms amplify symptomsRewiring steps that turn coping skills into super-powersHit ▶︎ to rethink everything you were told about ADHD—and grab the free resources at mindchange.com.Support this podcast at — https://redcircle.com/the-mind-change-podcast/donationsAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

Dr. Wendy is talking to Dr. Eva Ritvo about AI and mental health. We are also talking to Dr. Timothy Fong about cannabis use disorder and what we can do. It's all on KFIAM-640!

In this episode, host Alyssa Watson, DVM, is joined by John M. Thomason, DVM, MS, DACVIM (SAIM), to talk about his recent Clinician's Brief article, “Top 4 Primary Immune-Mediated Disorders in Dogs.” In part 1 of this 2-part conversation, Dr. Thomason focuses on the diagnosis and management of IMHA and IMTP. You'll hear vital details for both conditions including the right way to handle blood smears and slide agglutination, which IMHA cases are hypercoagulable (spoiler: all of them), and if vincristine actually helps in IMTP (spoiler again: it does).Resources:https://www.cliniciansbrief.com/article/anemia-thrombocytopenia-immune-disorder-dogshttps://www.zoetisus.com/products/dogs/librelaContact:podcast@instinct.vetWhere To Find Us:Website: CliniciansBrief.com/PodcastsYouTube: Youtube.com/@clinicians_briefFacebook: Facebook.com/CliniciansBriefLinkedIn: LinkedIn.com/showcase/CliniciansBrief/Instagram: @Clinicians.BriefX: @CliniciansBriefThe Team:Alyssa Watson, DVM - HostAlexis Ussery - Producer & Multimedia Specialist

Is it healthy to dwell in the past? Before the turn of the last century, nostalgia was still considered by some mental health professionals as a psychological disorder. So then…why have numerous storied food and beverage CPG brands more recently leaned on their decades of history to redesign packaging with elements of their past? Obviously, the understanding of nostalgia evolved over time…now being viewed less as a disorder and more as a natural human emotion. But with nostalgia being perhaps the most active and useful during uncomfortable (or transitionary) states, it's no wonder why many legacy CPG brands have recently leveraged it when seeking to elevate connectedness towards a simpler era of life. Yet, deploying a “blast from the past” strategy isn't universally impactful, and I believe today's consumers are typically more engaged by the future that brand is creating compared to what happened in the past.

Dr. Timothy Fong is a Professor of Psychiatry at the Jane and Terry Semel Institute for Neuroscience and Human Behavior at UCLA. He is board certified in adult and addiction psychiatry.Dr. Fong is also a member of the Steering Committee of the UCLA Center for Cannabis andCannabinoids whose mission is to address the most pressing questions related to the impact ofcannabis legalization through rigorous scientific study and discourse across disciplines.Take a listen to his take on usage.

This content has been developed for healthcare professionals only. Patients who seek health information should consult with their physician or relevant patient advocacy groups.For the full presentation, downloadable Practice Aids, slides, and complete CME/MOC/AAPA/IPCE information, and to apply for credit, please visit us at PeerView.com/GWQ865. CME/MOC/AAPA/IPCE credit will be available until June 29, 2026.Cracking the Code of GBA1-Associated Parkinson's Disease and Lysosomal Storage Disorders: Transforming Diagnosis and Management In support of improving patient care, this activity has been planned and implemented by PVI, PeerView Institute for Medical Education, and Gaucher Community Alliance. PVI, PeerView Institute for Medical Education, is jointly accredited by the Accreditation Council for Continuing Medical Education (ACCME), the Accreditation Council for Pharmacy Education (ACPE), and the American Nurses Credentialing Center (ANCC), to provide continuing education for the healthcare team.SupportThis activity is supported by an educational grant from Lilly.Disclosure information is available at the beginning of the video presentation.

This content has been developed for healthcare professionals only. Patients who seek health information should consult with their physician or relevant patient advocacy groups.For the full presentation, downloadable Practice Aids, slides, and complete CME/MOC/AAPA/IPCE information, and to apply for credit, please visit us at PeerView.com/GWQ865. CME/MOC/AAPA/IPCE credit will be available until June 29, 2026.Cracking the Code of GBA1-Associated Parkinson's Disease and Lysosomal Storage Disorders: Transforming Diagnosis and Management In support of improving patient care, this activity has been planned and implemented by PVI, PeerView Institute for Medical Education, and Gaucher Community Alliance. PVI, PeerView Institute for Medical Education, is jointly accredited by the Accreditation Council for Continuing Medical Education (ACCME), the Accreditation Council for Pharmacy Education (ACPE), and the American Nurses Credentialing Center (ANCC), to provide continuing education for the healthcare team.SupportThis activity is supported by an educational grant from Lilly.Disclosure information is available at the beginning of the video presentation.

We hear much in Unity about Divine Order, which simply means Spirit guides your lives and circumstances by Her orderly process. Did you know God also guides you through disorder, stressful times when challenges abound. Can you allow yourself to be subject to divine guidance during those inevitable times of chaos and disorder? Let's talk about this on Sunday.

I see a lot of health educators throw their weight around saying that they have more then a few decades of experience working one on one with clients, and then selling you the idea that acting based on your symptoms is just mimicking the allopathic pharma way of thinking. But what if we acknowledged our symptoms and used them as guiding lights instead of lab tests? Or you could do both. Natasha Snoeijer focuses on thyroid, hormone and metabolic optimization. She joins me to bring that "what about both?" energy to the health discussion. What if high dose supplementation can create long term healing? One vitamin did that for her. She shares her thoughts on the sugar diet, how to use your bowel movements to assess your health status, how to supplement thyroid properly, why she isn't a fan of hair tissue mineral analysis tests and what she likes instead, why candida cleanses don't work, and lots more. Work with Natasha: https://www.natashabwellness.com My website: www.matt-blackburn.com Mitolife products: www.mitolife.co Music by Nicholas Jimenez: https://spoti.fi/4cte2nD

REGIME CHANGE AND DISORDER. GREGORY COPLEY, DEFENSE & FOREIGN AFFAIRS 1870 SIEGE OF PARIS

On this Out of the Loop, Jason Pack helps us understand what the conflict between Iran and Israel signifies, and where we can expect it to go from here.Welcome to what we're calling our "Out of the Loop" episodes, where we dig a little deeper into fascinating current events that may only register as a blip on the media's news cycle and have conversations with the people who find themselves immersed in them. Disorder podcast host Jason Pack is here to help us make sense of the recent escalation in conflict between Iran and Israel — how we got here, the dangers and opportunities of the moment, and what we need from world leadership to keep the problem contained.Full show notes and resources can be found here: jordanharbinger.com/1177On This Episode of Out of the Loop:Israel launched surprise attacks on Iran's nuclear program and leadership, setting back its nuclear capabilities by months to years while demonstrating complete intelligence penetration.The US brokered a ceasefire between Iran and Israel, but this only addresses symptoms — the underlying regional conflicts and proxy wars remain unresolved.Iran announced it's accelerating its nuclear program in response to the attacks, following the "Libya lesson" that nuclear weapons provide protection from regime change.The current moment presents a unique opportunity for comprehensive Middle East peace due to weakened Iranian proxies and shifting regional power dynamics.Success requires multilateral diplomacy involving Qatar, Europe, Gulf states, and addressing root causes — not just ceasefire management but genuine conflict resolution through shared interests.And much more!Connect with Jordan on Twitter, on Instagram, and on YouTube. If you have something you'd like us to tackle here on an Out of the Loop episode, drop Jordan a line at jordan@jordanharbinger.com and let him know!Connect with Jason Pack on Twitter or on LinkedIn, and be sure to subscribe to his newsletter and check out his Disorder podcast!And if you're still game to support us, please leave a review here — even one sentence helps! Sign up for Six-Minute Networking — our free networking and relationship development mini course — at jordanharbinger.com/course!Subscribe to our once-a-week Wee Bit Wiser newsletter today and start filling your Wednesdays with wisdom!Do you even Reddit, bro? Join us at r/JordanHarbinger!This Episode Is Brought To You By Our Fine Sponsors:Boulevard: 10% off first year: joinblvd.com/jordanIDEO U: 15% off: ideou.com/jordanOpenPhone: 20% off 1st 6 months: openphone.com/jordanAirbnb: airbnb.com/hostHomes.com: Find your home: homes.comSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.