Podcasts about kbq

Bobby starts an eyebrow war. Yung Gravy takes a fall. Khalyla loves Arby's. We chat tall bones, congressman feet, height manifestation, flute delivery guy, and KBQ. TigerBelly Live Tickets! www.tigerbellylive.com Learn more about your ad choices. Visit megaphone.fm/adchoices

On 26 April 1986, one of the reactors at the Chernobyl Nuclear Power Plant exploded after unsanctioned experiments on the reactor by plant operators were done improperly. The resulting loss of control was due to design flaws of the RBMK reactor, which made it unstable when operated at low power, and prone to thermal runaway where increases in temperature increase reactor power output.[24][25] Chernobyl city was evacuated nine days after the disaster. The level of contamination with caesium-137 was around 555 kBq/m2 (surface ground deposition in 1986).[26][27] Later analyses concluded that, even with very conservative estimates, relocation of the city (or of any area below 1500 kBq/m2) could not be justified on the grounds of radiological health.[28][29][30] This however does not account for the uncertainty in the first few days of the accident about further depositions and weather patterns. Moreover, an earlier short-term evacuation could have averted more significant doses from short-lived isotope radiation (specifically iodine-131, which has a half-life of about eight days). Estimates of health effects are a subject of some controversy, see Effects of the Chernobyl disaster. Read About the Chernobyl Disaster Chernobyl - Wikipedia Follow Us @s3podcast_ on instagram --- Send in a voice message: https://anchor.fm/thes3podcast/message

On May 14, 1945, Albert Stevens was injected with 131 kBq (3.55 µCi) of plutonium - the highest known radiation dose in any human - without his knowledge or informed consent. This is his story. The Wholesome Show is Dr Rod Lamberts and Dr Will Grant, proudly brought to you by The Australian National Centre for the Public Awareness of Science!

Tonight on Back In The Game we will be joined by Lya and Shana Wesley. co-owners of the Washington DC area's  favorite BBQ restaurant, KBQ Real Barbeque.  The sisters merged their marketing and culinary skills in 2007 to found KBQ.  Every year since opening, KBQ has been voted best ribs by the Washingtonian magazine and maintains a strong following due to its outstanding products and welcoming dining experience.  They are in the process of opening a second location in DC this month which will be run by Shana's daughter, Justice Nelson.  Lya and Shana were raised in Los Angeles, California and migrated to the DC area separately after college.  They both worked more than 10 years in corporate America before deciding to venture into the entrepreneurial arena.  There are many challenges as well as rewards associated in running a business, but the sisters are committed to working together while creating a lasting legacy and generational wealth for their family. KBQ Real Barbeque at 9101 Woodmore Centre Drive, Lanham, MD 20706  phone # 301-322-1527 www.KBQBBQ.com

The carcinogenic effect of the radio isotope Rn-222 of the noble gas radon and its progeny, as well as its residential distribution, are well studied. In contrast, the knowledge about the effects and average dwelling concentration levels of its radio isotope Rn-220 (thoron) is still limited. Generally, this isotope has been assumed to be a negligible contributor to the effective annual dose. However, only recently it has been pointed out in several international studies, that the dose due to thoron exceeds the one from Rn-222 under certain conditions. Additionally, radon monitors may show a considerable sensitivity towards thoron which was also not accounted for in general. Therefore a reliable, inexpensive exposimeter, which allows to distinguish between decays of either radon and thoron, is required to conduct further studies. The scope of this thesis was to develop an electronic radon/thoron exposimeter which features small size, low weight and minimal power consumption. The design is based on the diffusion chamber principle and employs state-of-the-art alpha particle spectroscopy to measure activity concentrations. The device was optimized via inlet layout and filter selection for high thoron diffusion. Calibration measurements showed a similar sensitivity of the monitor towards radon and thoron, with a calibration factor of cfRn-222 = 16.2±0.9 Bq×m-3/cph and cfRn-220 = 14.4±0.8 Bq×m-3/cph, respectively. Thus, the radon sensitivity of the device was enhanced by a factor two compared to a previous prototype. The evaluation method developed in this work, in accordance with ISO 11665 standards, was validated by intercomparison measurements. The detection limits for radon and thoron were determined to be C#Rn-222 = 44.0 Bq/m3 and C#Rn-220 = 40.0 Bq/m3, respectively, in case of a low radon environment, a one-hour measurement interval, and a background count rate of zero. In contrast, in mixed radon/thoron concentrations where the Po-212 peak must be used for thoron concentration determination, a calibration factor of cfRn-220 = 100±10 Bq×m-3/cph was measured, yielding a detection limit of C#Rn-220 = 280 Bq/m3. Further, Monte Carlo (MC) simulations were performed by means of various codes including Geant4, to study the effect of the variation of parameters influencing the calibration factors. The results showed reasonable agreement between simulated and acquired spectra, with differences being below 8%, thus validating the employed simulation model. The simulations indicated a significant impact of environmental parameters, such as temperature and pressure, on the measured spectra and accordingly on the calibration factor. Therefore the calibration factor was quantified as a function of temperature, relative humidity and pressure as well as chamber volume. For devices with increased detection volume a considerable influence of air density changes, corresponding to altitudes from 0-5,000 m, and temperatures from -25 to 35 °C, on the calibration factor of up to 32% was observed. In contrast, for devices with standard housing the calibration factor changed only up to 4%. When increasing the detection volume compared to the employed standard housing by at least a factor of four, a maximum increase of the sensitivity of about 20% was found, at the expense of device portability. On the contrary, when reducing the height of the housing by 10~$mm$, which yields 40% less volume, a decrease of sensitivity by 30% and 41% for radon and thoron was observed, respectively. Finally, devices were used and tested at different realistic conditions, such as mines, radon spas, and dwellings with mixed Rn-222 and Rn-220 environments. Measurements in a salt mine with the device developed within the framework of this thesis revealed maximum radon concentrations of up to 1.0 kBq/m3. In the Bad Gastein Heilstollen, Rn-222 concentrations up to 24.3 kBq/m3 were found, in agreement with an AlphaGuard reference device. First measurements in radon/thoron environments of about 200 Bq/m3 each, in a clay model house at the Helmholtz Center Munich, showed reasonable agreement with reference devices, thus validating the introduced evaluation method. First measurements in a private Bavarian clay house revealed a low thoron concentration of about CRn-220 = 13.0±3.0 Bq/m3, in comparison to a high radon concentration of CRn-222 = 200±70 Bq/m3.

Thyroid cancer derived from follicular epithelial cells is the most common endocrine malignancy in man. An increased incidence of predominantly papillary thyroid carcinomas (PTC) was found in children exposed to radiation after the Chernobyl nuclear accident in 1986. Therefore, in this study, the goal was to establish a mouse model of thyroid carcinogenesis, based on a standardized histological classification scheme for the murine thyroid tumors, and complemented by molecular genetic analyses. In previous studies, radioiodine (I131, 111 kBq) was injected into iodine deficient fed mothers of various mouse strains (F1-hybrids and backcrosses of C57/BL6, C3H, BALB/c, and JF1). The first injection was applied during gestation and the second during lactation. The necropsy tissue was submitted for the analysis in this study. A set of 365 thyroid glands (203 irradiated and 162 control mice) was histological examined following the current WHO classification of human thyroid tumors (2004) for comparative purposes. The irradiated mice showed 24 % of cases with simple hyperplasia (SH), 20 % with nodular hyperplasia (NH), 7 % with follicular thyroid adenoma (FTA), and 5 % with follicular thyroid carcinoma (FTC) whereas in the control group only 3 % SH, 3 % of NH, 1 % of FTA, and 1 % of FTC were observed. Interestingly, no PTC was diagnosed in the mice, which is the most frequent irradiation-related type of thyroid cancer in human. Therefore, the histological type of the radiation-associated thyroid tumors in mice differs from that in human. However, some cases of murine FTC presented PTC-like biological behavior. In addition to the significant increase of hyperplasias in irradiated mice, most of the FTC (82 %) arose amongst a background of hyperplastic nodules. Therefore, a progression from NH to FTC, based on genetic instability, cannot be ruled out. The following molecular methods were used: PCR- (polymerase chain reaction-) based loss of heterozygosity (LOH), comparative genomic hybridization (CGH), and fluorescence-in-situ-hybridization (FISH). Since the CGH-study in mice using formalin-fixed paraffin-embedded tissue (FFPE) is not yet established, an important part of the study was dedicated to evaluate this methodology. The LOH-study was performed with thyroid gland tissue from 40 mice (seven normal thyroid glands, 12 SH, 10 NH, 10 FTA, and one FTC) using 36 microsatellites for nine different loci. With the exception of an LOH with a single microsatellite on chromosome 14 in 40 % of NH, LOH was found in 75 % of the irradiated male mice with H6F1-background on chromosomes 4, 5, 6, 11, 14, and / or 19. This suggests the existence of a mouse strain specific genetic predisposition, which influence on the genetic stability. One of the FTA (an atypical FTA) was highly suspicious for a deletion of the tumorsuppressorgene Rb1 (supported by intragenic FISH-analysis), which could play an important role in the thyroid carcinogenesis. For the CGH-study, thyroid tissue derived from 21 different mice (F2-hybrids) was analyzed (two normal thyroid glands, one SH, 12 NH, two FTA, and eight FTC). In 46 % of the hyperplasias, small chromosomal gains and losses located on different chromosomes were observed; suggesting that there exists a genetic instability, which may lead eventually to malignant progression. Regional polyploidies on chromosomes 4 and 5 were demonstrated in one of the FTAs, which could be a hint for the location of oncogenes. Taken together, in FTA development there is a broad spectrum of genetic alteration, and by inference mechanisms. In contrast, the FTC exhibited a significant increase of specific aneuploidies, mainly deletions of the chromosomes 4 (88 %), 9 (50 %), and 14 (38 %). Identical alterations of chromosomes 4 and 9 were also observed in the one case of an FTC from a non-irradiated mouse. These data indicate that irradiation, most probably, increases the frequency of genetic changes, but does not change the type of genetic alterations, which play a crucial role in thyroid carcinogenesis in mice. A better understanding of molecular genetics involved in thyroid tumorigenesis in standardized mouse models may give insight into the pathogenesis of the various tumor types. Together with the results from human pathology and in vitro studies, this may lead to a better knowledge about the molecular pathways with diagnostic, prognostic, and therapeutic relevance. The results of this study demonstrate a morphological and genetical difference between human (PTC) and murine (FTC) radiation-associated thyroid tumors, but a strong similarity to the human follicular tumors. Therefore, this mouse model serves as a good model of carcinogenetic mechanisms, tumor induction, and progression in the human follicular tumors FTA and FTC, resulting from the cooperative effect of radioiodine exposition and iodine deficiency.

Der Erfolg chirurgischer Eingriffe wird nicht selten durch überschießende Wundheilung zunichte gemacht, so daß ein erneuter Eingriff notwendig wird. Am Wundort lokal eingesetzte Radionuklide mit kurzreichweitiger Strahlung können solche gutartigen Wucherungen verhindern. Das Radionuklid P-32 eignet sich als reiner Elektronenemitter mit einer Halbwertszeit von 14,3 Tagen und einer mittleren Energie von 694,9 keV (Emax=1710,48 keV) für diese Aufgabe und kann durch den Einfang thermischer Neutronen (1 · 10^14 /s/cm^2) im Kernreaktor aus dem stabilen P-31 hergestellt werden. Nach einer typischen Bestrahlungszeit (14 Tage) beträgt der P-32–Anteil 1,4 · 10^-5. Implantate aus Polymer bzw. bioresorbierbarem Material als Träger des radioaktiven Strahlers ermöglichen gegenüber metallischen Implantaten neue Anwendungen für diese Art der Strahlentherapie. In dieser Arbeit wurde eine Herstellungsmethode für bisher nicht verfügbare organische radioaktive Implantate entwickelt und ein dazugehöriges Dosimetriesystem aufgebaut. Mittels Ionenimplantation können P-32–Ionen mit bis zu 180 keV einige 100 nm tief in organische Implantatmaterialien eingeschossen werden. Für eine typische Dosis (15 Gy in 7 Tagen in 1 mm Abstand zum Implantat) wird eine Aktivität von 75 kBq benötigt, dies entspricht 1,3 · 10^11 P-32–Ionen. Die dafür optimierte Zerstäubungsionenquelle ermöglicht einen Ionenstrahl mit hohem Strahlstrom (>14 µA P–) und geringer Emittanz (

Radon decay product activity was measured in saliva of 10 male patients 20-30 min after a 1-hour radon exposure in the gallery of the Gasteiner Heilstollen (radon activity 36.2 kBq/m(3), radon progeny activity 20.3 kBq/m(3)), in 1 patient showing relatively high activity (75th percentile) measurements were continued until 65 min after exposure. Patients were asked to collect about 2 mi of saliva in the mouth and produce it on a filter. After drying the filter at 300 degrees C, radon progeny activity was measured. Activity (median) at 20-30 min after leaving the treatment area was 4.5 Bq (25th percentile 1 Bq; 75th percentile 21 Bq). In the patient who underwent additional measurements the activity showed a further increase up to 29 Bq (35 min after radon exposure) before it continuously decreased to a very low activity (1-3 Bq) at 65 min after exposure. The results show that a significantly increased radon decay product activity is found in saliva after speleotherapeutic radon exposure. Maximum values were observed 35 min after radon exposure. Radon decay product activity almost disappeared after about 1 h.