Podcasts about patani

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2022.10.28.514238v1?rss=1 Authors: Virdi, G. S., Choi, M. L., Evans, J. R., Yao, Z., Athauda, D., Strohbuecker, S., Wernick, A. I., Alrashidi, H., Melandri, D., Perez-lloret, J., Stroh, P. R., Sylantyev, S., Eaton, S., Heales, S., Kunath, T., Horrocks, M. H., Abramov, A. Y., Patani, R., Gandhi, S. Abstract: Mutations in the SNCA gene cause autosomal dominant Parkinsons disease (PD), with progressive loss of dopaminergic neurons in the substantia nigra, and accumulation of aggregates of alpha-synuclein. However, the sequence of molecular events that proceed from the SNCA mutation during development, to its end stage pathology is unknown. Utilising human induced pluripotent stem cells (hiPSCs) with SNCA mutations, we resolved the temporal sequence of pathophysiological events that occur during neuronal differentiation in order to discover the early, and likely causative, events in synucleinopathies. We adapted a small molecule-based protocol that generates highly enriched midbrain dopaminergic (mDA) neurons ( greater than 80%). We characterised their molecular identity using single-cell RNA sequencing and their functional identity through the synthesis and secretion of dopamine, the ability to generate action potentials, and form functional synapses and networks. RNA velocity analyses confirmed the developmental transcriptomic trajectory of midbrain neural precursors into mDA neurons using our approach, and identified key driver genes in mDA neuronal development. To characterise the synucleinopathy, we adopted super-resolution methods to determine the number, size and structure of aggregates in SNCA-mutant mDA neurons. At one week of differentiation, prior to maturation to mDA neurons of molecular and functional identity, we demonstrate the formation of small aggregates; specifically, beta-sheet rich oligomeric aggregates, in SNCA-mutant midbrain immature neurons. The aggregation progresses over time to accumulate phosphorylated aggregates, and later fibrillar aggregates. When the midbrain neurons were functional, we observed evidence of impaired physiological calcium signalling, with raised basal calcium, and impairments in cytosolic and mitochondrial calcium efflux. Once midbrain identity fully developed, SNCA-mutant neurons exhibited bioenergetic impairments, mitochondrial dysfunction and oxidative stress. During the maturation of mDA neurons, upregulation of mitophagy and autophagy occured, and ultimately these multiple cellular stresses lead to an increase in cell death by six weeks post-differentiation. Our differentiation paradigm generates an efficient model for studying disease mechanisms in PD, and highlights that protein misfolding to generate intraneuronal oligomers is one of the earliest critical events driving disease in human neurons, rather than a late-stage hallmark of the disease. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2022.10.10.511318v1?rss=1 Authors: Balendra, R., Ruiz de los Mozos, I., Glaria, I., Milioto, C., Odeh, H. M., Wilson, K. M., Ule, A. M., Hallegger, M., Masino, L., Martin, S., Patani, R., Shorter, J., Ule, J., Isaacs, A. Abstract: An intronic GGGGCC repeat expansion in C9orf72 is a common genetic cause of amyotrophic lateral sclerosis and frontotemporal dementia. The repeats are transcribed in both sense and antisense directions to generate distinct dipeptide repeat proteins, of which poly(GA), poly(GR) and poly(PR) have been implicated in contributing to neurodegeneration. Poly(PR) binding to RNA may contribute to toxicity, but analysis of poly(PR)-RNA binding on a genome-wide scale has not yet been carried out. We therefore performed crosslinking and immunoprecipitation (CLIP) analysis in human cells to identify the RNA binding sites of poly(PR). We found that poly(PR) binds to nearly 600 RNAs, with the sequence GAAGA enriched at the binding sites. In vitro experiments showed that polyGAAGA RNA binds poly(PR) with higher affinity than control RNA and induces phase-separation of poly(PR) into condensates. These data indicate that poly(PR) preferentially binds to polyGAAGA-containing RNAs, which may have physiological consequences. Copy rights belong to original authors. Visit the link for more info Podcast created by PaperPlayer

The Sultanate of Patani - now part of modern day Thailand - enjoyed a golden age during the reign of four successive queens, which commenced in 1584. Under their rule, the kingdom's economic and military strength greatly increased to the point that it was able to fight off four major Siamese invasions.In this episode of Not Just the Tudors, Professor Suzannah Lipscomb discovers more about these extraordinary rulers, their power and their influence, to Professor Stefan Amirell, President of the Swedish Historical Association and an expert in female political leadership in world history.The Senior Producer was Elena Guthrie. It was edited by Thomas Ntinas and produced by Rob Weinberg.For more Not Just The Tudors content, subscribe to our Tudor Tuesday newsletter here >For your chance to win five non-fiction history books - including a signed copy of Dan Snow's On This Day in History - please fill out this short survey: https://www.surveymonkey.co.uk/survey-taken/?sm=IthGeoCcJUiKNx0R8Pv7Ogn50xYWgriQdyDMjMZwy8jmNE1jQh63NtWjK1DQdAssMjnsuFzX5eJOGw0w3NS4sgHthi59y72wWjesdfmNxyU_3DIf you'd like to learn even more, we have hundreds of history documentaries, ad free podcasts and audiobooks at History Hit - subscribe today!To download, go to Android > or Apple store > Hosted on Acast. See acast.com/privacy for more information.

PATREON EXCLUSIVE. Extremely excited to share this bonus episode with you on the 4 queens of Aceh who ruled from 1641 to 1699, beginning with Sultanah Safiatuddin or Taj al-Alam Safiatuddin Syah. Unlike the four queens of Patani, this was not an unbroken lineage. EXCERPT: Khan argues that maintaining peace and stability so commerce could thrive was an even bigger challenge than the ones faced by the male predecessors of the Acehnese queens. During the time of the queens, although the VOC might have controlled a larger share of the international trade in this region by the end of the century, Aceh's regional trade continued to thrive and, as a trading port which served private traders from all over the world, Aceh's international commercial networks continued to be resilient. By the end of the reigns of these women sovereigns, Malay writing and literature in Aceh had developed to a height unrivalled till today: this could be said to constitute the real golden age in Acehnese history. Sorry, Iskandar Muda. The book really goes into detail so I recommend reading it, the text is already up in our resource library under Episode 19.

Welcome to TalkBricks Masters - A LEGO Masters Weekly Recap Podcast! This week Michael (from the LEGO YouTube channel TalkBricks) is joined by LEGO Masters contestants Paras Patani & Michael "Moto" Kanemoto!! We talk through everything from the harshest feedback Jamie every gave them to what song Paras sang for over 7 hours straight during a challenge!! Check out our social media to send along questions for our future guests - @TalkBricks on all social media platforms!

I spoke to Dr. Duncan McCargo, Director at the Nordic Institute of Asia Studies and the author of " Tearing Apart The Land: Islam and Legitimacy in Southern Thailand" Our discussion is on Patani, located in Southern Thailand it's one of the most misunderstood regions in all of Southeast Asia. Dr. McCargo and I discuss how Patani's fate was decided by the Kingdom of Siam & The British Empire, why the region of Patani has been so resistant to being part of Thailand, what that resistance has looked like, and what larger questions Patani raises both for Thailand and globally. For more I can't recommend Dr. McCargo's book enough, it's fascinating not just for those interested in Thailand but for those interested in larger philosophical questions on the Nation-State, social movements and identity. You can find it here: https://www.cornellpress.cornell.edu/book/9781501702914/tearing-apart-the-land/ Dr. McCargo also recommended the Nordic Institute's own podcast on Asia: https://podcasts.apple.com/dk/podcast/the-nordic-asia-podcast/id1509921432 Music is by H3 Beats: https://www.youtube.com/watch?v=rut35JzzGVY

Patani joins us to talk about his work and his experience on the show. 

On this weeks Analysand [English] we examine the conflict in Patani or ‘The Three Southern Provinces'. Joined by out guest K, we explore the history of the insurgency, all the way to it's present day, as well as the view from Bangkok. The insurgency often goes underreported in Thailand, even by the Thai press. So we do our best to shine a light on the conflict. . The book that Samai recommended is: Moore Phd, Dr Jeff M. 2014. The Thai Way of Counterinsurgency (North Charleston, SC: Createspace Independent Publishing Platform). . About Haji Sulong: (English) http://www.dindeng.com/haji-sulong/ (Thai) https://www.bbc.com/thai/thailand-53761549 . About Thai Imperialism and Colonisation: (Thai) http://www.dindeng.com/thai-imperialism-and-colonisation/

We've hit 25 episodes and this one is an incredibly interesting interview on the insurgency in the south of Thailand! Mr. Shintaro Hara is an analyst and a translator who lives in Patani and has been interacting with people and communities that are affected daily by the insurgency in Thailand. The conversation talks about the concerns of individuals who inhabit the region and the importance of self-determination. We also talk about the various groups that were created to represent the interest of the Muslim population in Patani, why the struggle became armed, and how these groups might earn money to fund their struggle. We finally touch on why Thaification has adversely harmed the region and how the region is coping economically given the current pandemic.

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2020.09.08.287797v1?rss=1 Authors: Virdi, G. S., Choi, M. L., Yao, Z., Evans, J. R., Athauda, D., Melandri, D., Sylantyev, S., Abramov, A. Y., Patani, R., Gandhi, S. Abstract: The development of human induced pluripotent stem cells (hiPSC) has greatly aided our ability to model neurodegenerative diseases. However, generation of midbrain dopaminergic (mDA) neurons is a major challenge and protocols are variable. Here, we developed a method to differentiate hiPSCs into enriched populations (>80%) of mDA neurons using only small molecules. We confirmed the identity of the mDA neurons using single-cell RNA-sequencing and detection of classical markers. Single-cell live imaging demonstrated neuronal calcium signalling and functional dopamine transport. Electrophysiology measures highlighted the ability to form synapses and networks in culture. Patient-specific hiPSC lines differentiated to produce functional mDA neurons that exhibit the hallmarks of synucleinopathy including: aggregate formation, oxidative stress as well as mitochondrial dysfunction and impaired lysosomal dynamics. In summary, we establish a robust differentiation paradigm to generate enriched mDA neurons from hiPSCs, which can be used to faithfully model key aspects of Parkinson's disease (PD), providing the potential to further elucidate molecular mechanisms contributing to disease development. Copy rights belong to original authors. Visit the link for more info

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2020.08.17.253773v1?rss=1 Authors: Luisier, R., Serio, A., Patani, R., Newcombe, J., Greensmith, L., Devine, H., Taha, D. M., Tyzack, G. E., Hagemann, C. Abstract: Histopathological analysis of tissue sections is an invaluable resource in neurodegeneration research. Importantly, cell-to-cell variation in both the presence and severity of a given phenotype is however a key limitation of this approach, reducing the signal to noise ratio and leaving unresolved the potential of single-cell scoring for a given disease attribute. Here, we developed an image processing pipeline for automated identification and profiling of motor neurons (MNs) in amyotrophic lateral sclerosis (ALS) pathological tissue sections. This approach enabled unbiased analysis of hundreds of cells, from which hundreds of features were readily extracted. Next by testing different machine learning methods, we automated the identification of phenotypically distinct MN subpopulations in VCP- and SOD1-mutant transgenic mice, revealing common aberrant phenotypes in cellular shape. Additionally we established scoring metrics to rank cells and tissue samples for both disease probability and severity. Finally, by adapting this methodology to human post-mortem tissue analysis, we validated our core finding that morphological descriptors strongly discriminate ALS from control healthy tissue at the single cell level. In summary, we show that combining automated image processing with machine learning methods substantially improves the speed and reliability of identifying phenotypically diverse MN populations. Determining disease presence, severity and unbiased phenotypes at single cell resolution might prove transformational in our understanding of ALS and neurodegenerative diseases more broadly. Copy rights belong to original authors. Visit the link for more info

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2020.07.20.211557v1?rss=1 Authors: Tyzack, G. E., Neeves, J., Klein, P., Crerar, H., Ziff, O., Taha, D. M., Luisier, R., Luscombe, N. M., Patani, R. Abstract: Intron retention is known to regulate gene expression. We recently described intron retention as the predominant splicing programme characterizing early stages of motor neurogenesis from human induced pluripotent stem cells (hiPSCs) and it's perturbation in familial amyotrophic lateral sclerosis (ALS). Here, we sought to gain more insight into the nucleocytoplasmic distribution of aberrant intron-retaining transcripts (IRTs) and to identify their discriminating molecular features. We combined cellular fractionation with hiPSCs undergoing motor neurogenesis and deep-sequenced 95 samples: ALS vs control hiPSCs and nuclear vs cytoplasmic compartments across six timepoints, which represents a rich transcriptomic resource for basic and applied neuroscientists. Using this resource, we identified >100 aberrant cytoplasmic IRTs in cultures carrying ALS-causing VCP gene mutations. We taxonomized aberrant IRTs by their nucleocytoplasmic distribution and demonstrate that these classes exhibit sequence-specific attributes and differential predicted binding affinity to ALS-related RNA binding proteins. In summary we uncover a distinct class of cytoplasmic IRTs that serve as blueprints for established molecular hallmarks of ALS (nuclear-to-cytoplasmic mislocalisation of TDP-43, SFPQ and FUS) and therefore may also represent therapeutic targets. Copy rights belong to original authors. Visit the link for more info

workpointTODAY ชวนพูดคุยกับ ผศ.เจะอับดุลเลาะ เจ๊ะสอเหาะ อาจารย์คณะศิลปกรรมศาสตร์ ม.อ.ปัตตานี และผู้ก่อตั้งผู้ก่อตั้ง Patani Artspace ถึงสถานการณ์ใน 3 จังหวัดชายแดนใต้ช่วงโควิด-19 และการบอกเล่า"ความจริง"ในพื้นที่ผ่านงานศิลปะ

People Group Summary https://joshuaproject.net/people_groups/14343/TH Listen to the "Gateway to the Unreached" with Greg Kelley, produced by the Alliance for the Unreached: https://alliancefortheunreached.org/podcast/

In this episode, we'll talk about Raja Hijau's rise to power, and the Malay Sultanate of Patani's nearly unbroken line of queens from 1584 to 1718. This episode features a music clip, "Bangkit Pemuda Pemudi - Patriot Patani" by Fai Kencrut. Don't forget to follow us on Twitter and Instagram, @herstoryseapod! For a copy of the show notes with all the references, access to the close friends Instagram stories, and a shout out at the end of the next episode, join us on Patreon at https://bit.ly/herstoryseapatreon. Maraming salamat!

“ถ้าผู้คนสามารถบอกเล่าเรื่องราวของตัวเองได้อย่างเป็นระบบ น่าสนใจ มีเนื้อหาสาระ มีประเด็น ต่อให้เป็นเรื่องราวที่เรียบง่ายที่สุด ก็ยังน่าสนใจ และสิ่งเหล่านั้นจะเป็นการเพิ่มอำนาจต่อรองให้กับตัวเอง” - นวลน้อย ธรรมเสถียร ธิติ มีแต้ม สนทนากับ ‘นวลน้อย’ ที่ปัตตานี เมืองแห่งความสวยงามทางพหุวัฒนธรรม แต่ก็เต็มไปด้วยเหตุการณ์ความรุนแรง อย่างน้อยก็กินเวลานานถึง 15 ปี ตั้งแต่ พ.ศ.2547 ปัจจุบัน นวลน้อยเป็นสื่อมวลชนอิสระ ที่กำลังปลุกปั้นวัฒนธรรมการอ่านการเขียนให้กับคนในพื้นที่สามจังหวัดชายแดนภาคใต้ ขณะเดียวกันก็ยังติดตามสถานการณ์การเมืองไทยไปด้วยพร้อมๆ กัน แต่สำหรับแวดวงสื่อมวลชน นวลน้อยวิ่งอยู่ในสนามข่าวมาตั้งแต่ทศวรรษที่ 80 ทำข่าวในนามสำนักข่าว BBC ที่สมัยนั้นยังออกอากาศด้วยวิทยุ พูดอย่างรวบรัดที่สุด ขณะที่แวดวงนิเทศศาสตร์ในปัจจุบันกำลังสั่นคลอนมากที่สุดสาขาหนึ่ง อะไรคือหลักคิดของนวลน้อยในการรับมือกับความเปลี่ยนแปลงครั้งใหญ่นี้, BBC สอนอะไรแก่เธอ และถึงที่สุดการได้ผูกพันกับสามจังหวัดชายแดนภาคใต้ กระทั่งได้เปิดเพจ Patani NOTES ขึ้นมา เธอกำลังคิดอะไร กำลังทำอะไร เราจะคุยกับเธอยาวๆ

This is the March episode of 3-Minute 3Rs, brought to you by the North American 3Rs Collaborative (www.na3rsc.org, the NC3Rs (www.nc3rs.org.uk), and Lab Animal (www.nature.com/laban) The papers behind the pod: 1. Progressive Motor Neuron Pathology and the Role of Astrocytes in a Human Stem Cell Model of VCP-Related ALS. https://bit.ly/2uiF64X 2. A critical evaluation of TRPA1-mediated locomotor behavior in zebrafish as a screening tool for novel anti-nociceptive drug discovery. https://go.nature.com/2Cu7u8C 3. No-touch measurements of vital signs in small conscious animals. https://bit.ly/2WdN5Mw [NC3Rs] A team led by Dr Rickie Patani has developed a human-derived model of ALS that could bring us a step closer to treating the disease effectively while avoiding the use of animals altogether. Animal models are widely used to study ALS, also known as motor neurone disease, but current therapies can only slow its progression – and even then, the effect is modest. Instead, Dr Patani's team, based at UCL and the Francis Crick Institute, used human induced pluripotent stem cells to study how ALS causes motor neurones to degenerate. They investigated the molecular processes that lead to the death of motor neurones, which are kickstarted by the loss of a protein called TDP-43 from the cell nucleus. They also discovered that ALS makes astrocytes degenerate too, so they can't play their usual role in helping motor neurones survive, compounding the effects of the disease. For this work, Dr Patani was awarded the NC3Rs' International 3Rs Prize earlier this month. The prize is sponsored by GSK and celebrates outstanding 3Rs science every year. Read the paper in Cell Reports or visit the NC3Rs website to learn more about the 3Rs Prize. [LA] Rodents remain popular for in vivo validation of novel drugs. But screening candidates is costly to do in rodents, which has researchers looking for alternatives to evaluate compounds in a higher throughput manner. Zebrafish are increasingly used for such screening purposes. A new paper from Richard van Rijn's lab at Purdue published in the journal Scientific Reports evaluates a zebrafish screen for drugs that Transient Receptor Potential A1, or TRPA1. TRPA1 encodes a calcium ion channel and has been shown to be involved in pain perception in rodent models. In zebrafish, activating TRPA1 causes hyperlocomotion, which the researchers hypothesized could be a useful phenotypic readout of drug efficacy. They tested compounds known to activate and inactivate TRPA1 in human cells, mice, and zebrafish larvae and found that the compounds affect all three models in a dose-dependent manner. Evaluation can be somewhat tricky in the zebrafish because they have a second copy of TRPA1 to contend with, but the fish could still help screen initial compounds before researchers take them onward. [LA] What if you could measure the vital signs of your animals without having to prep or handle them? Engineers at Cornell recently described the use of radio frequency near-field coherent sensing to do just that in a paper published in Science Advances. They developed the technology first for humans, but have now shown its potential for use with small animals in real-time. The technology uses radio waves. These penetrate the body and can be used to detect the motion of internal organs. When the signal is processed, parameters like heart rate and respiration rate can be captured. The system can be wired or wireless, and was shown to work with an anesthetized rat and freely moving hamster, Russian tortoise, and betta fish. There was some variability and further comparisons with existing methods would help clarify the accuracy and robustness of the new system, but near-field coherent sensing could be a promising new way to keep an eye on animal vital signs. See acast.com/privacy for privacy and opt-out information.

Let us be and have fun with this music

Im Glauben nach Venedig zu fahren, steuerte das Schiff, welches er nach kurzer Kriegsgefangenschaft in Amsterdam bestieg, auf direktem Wege Westafrika an. Krankheit, Tod und Schiffbruch begleiteten diese Reise, die 7 Jahre dauert und ihn quer über den Erdball bis nach Südostasien (und wieder zurück) führen sollte.Vor kurzem hat sich zu dieser Einzigartigkeit noch eine weitere hinzugesellt. Fernbergers Raißbuch enthält die einzigen Aufzeichnungen über einen kurzen Krieg zwischen den Königreichen Ayudhya und Patani (beide im heutigen Thailand) in den Jahren 1624/25.In dieser Sendung erzählt Helmut Lukas über Christoph Carl Fernbergers erstaunliche Reise, über Reisen im 17. Jahrhundert generell, über Verständigungsmöglichkeiten und -schwierigkeiten, über die kriegerische Auseinandersetzung zwischen Raja Ungus Königreich Patani und Songtham, dem König von Siam/Ayudhya, aber auch über das damalige Frauenbild da Patani mehr als 125 Jahre lang von Frauen regiert wurde.Gestaltung: Doris Bauer (Verantwortlich für den Sendungsinhalt)Im Interview: Univ. Doz. Dr. Helmut Lukas, Institut für Sozialanthropologie an der Österreichischen Akademie der Wissenschaften.Er forscht seit Jahrzehnten in Südostasien und ist Herausgeber der in Kürze erscheinenden Publikation:Christoph Carl Fernberger: The First Austrian in Patani and Ayudhya (1624-1625). Tri-lingual (German-English-Thai) annotated edition of Fernberger’s diary concerning his stays in Ayudhya & Patani in the years 1624 & 1625. Centre for European Studies at Chulalongkorn University, Bangkok. Co-editor: Charit Tingsabadh.Sprecher: Walther MoserWeitere Publikation: Wernhart, Karl Rudolf: Christoph Carl Fernberger, Der erste österreichische Weltreisende 1621-1628. Völlig überarbeitete und neu kommentierte Ausgabe. Mit ergänzendem Kommentar für Indonesien und Südostasien von Helmut Lukas; Reihe: Reiseforschung, Bd. 2, 1. Auflage 2011, 2. Auflage, 2012, 168 S.Musik: Krayne - Dance of the Zen Master; Zaire - Isla de Java; Senyawa - Warna; SaReGaMa - One Thousand And One Nights. Nachzuhören auf Free Music Archive und Jamendo, einer Community für freie, legale und unlimitierte Musik, die unter Creative Commons Lizenzen veröffentlicht wurde.

My first short film score https://www.youtube.com/watch?v=fpyFw_B8xv8

Ayeuna Abah seja ngadongéng lalakon Sakadang Kuya anu rék di kawinkeun ka anakna Patani. Mudah-mudahan hikmah tina téma Dongéng ieu tiasa di lenyepan ku urang saréréa. Wilujeng ngadangukeun. Kaparayuuuunn Sadayanaaaaaa.

Jacques IVANOFF, CNRS - IRASEC. Depuis l'intégration du sultanat de Patani dans la nation thaïlandaiseau début du siècle, la résistance des millions de Malais musulmans de Thaïlande du Sud-Est ne s'est jamais démentie. Celle-ci s'appuie sur une représentation de l'environnement qui considère la nature comme fragmentée et coupée de l’homme. Cette parcellisation et cette rupture de liens sont à l'origine d’une quête épique au cœur de la tradition orale locale, une quête de l’union de l’homme et de son environnement, jugée comme un préalable incontournable à l’union des territoires éclatés autrefois entre chefferies belliqueuses et aujourd’hui entre la Malaysia et la Thaïlande. La nécessité de recomposer un lien avec la nature se double donc de la volonté de recomposer une unité territoriale malmenée par la colonisation thaïlandaise et par le désir de retrouver une identité perdue. Les données imaginaires à l’origine des expressions culturelles de la société malaise se sont accordées avec les revendications politiques contemporaines. La quête des héros malais pour rapprocher l’homme de la nature se confond maintenant avec la quête des rebelles musulmans et dans ce rapprochement objectif entre la culture, l’identité et la revendication politique réside la force de la rébellion du sud mais aussi ses limites.

Jacques IVANOFF, CNRS - IRASEC. Depuis l'intégration du sultanat de Patani dans la nation thaïlandaiseau début du siècle, la résistance des millions de Malais musulmans de Thaïlande du Sud-Est ne s'est jamais démentie. Celle-ci s'appuie sur une représentation de l'environnement qui considère la nature comme fragmentée et coupée de l’homme. Cette parcellisation et cette rupture de liens sont à l'origine d’une quête épique au cœur de la tradition orale locale, une quête de l’union de l’homme et de son environnement, jugée comme un préalable incontournable à l’union des territoires éclatés autrefois entre chefferies belliqueuses et aujourd’hui entre la Malaysia et la Thaïlande. La nécessité de recomposer un lien avec la nature se double donc de la volonté de recomposer une unité territoriale malmenée par la colonisation thaïlandaise et par le désir de retrouver une identité perdue. Les données imaginaires à l’origine des expressions culturelles de la société malaise se sont accordées avec les revendications politiques contemporaines. La quête des héros malais pour rapprocher l’homme de la nature se confond maintenant avec la quête des rebelles musulmans et dans ce rapprochement objectif entre la culture, l’identité et la revendication politique réside la force de la rébellion du sud mais aussi ses limites.