Podcasts about frizzled

It's no secret that Simon Davis LOVES Greek yoghurt as an ingredient. But has he ever been this excited about Greek yoghurt as he is today?Listen for a fantastic rave about Anna Jones's new cookbook 'Easy Wins' - an exasperated take on mayo - and this fantastic recipe:Frizzled Spring Onion and Olive Oil Dip150g good Greek yoghurt, strained if you can be bothered1 bunch spring onions, finely chopped100ml olive oil1 tsp pul biber Turkish chilli flakes 1 tsp sugar1/2 tsp ground turmericzest and juice of 2 limessalt and pepperOver a low-medium heat, fry the spring onion and pul biber chilli flakes in the olive oil until crisping and charring on the edges, veritably sizzling and frizzling - around 10 minutes.At the 9th minute, add the sugar and turmeric.Remove from heat, add salt and pepper then set aside and allow to coolJuice and zest the limes into the yoghurt, mix through, add salt to taste. Opulently dress the yoghurt with the yellow onion and oil mixture. Mix through or leave it on top. Serve with bread, chips, roast chicken, lamb chops, whatever you want! It's amazing! Hosted on Acast. See acast.com/privacy for more information.

In Episode 30, host David Crowley from Cooking Chat talks with Mike Cleavenger from The Frizzled Leek about his ideas for cherry tomatoes, scallions and delicata squash. We also hear about his ideas for using appliances such as an air fryer for convenient preparation of local food. In this episode, we cover: How Mike got interested in cooking at an early age. Gaining a foundation of classic cooking techniques at culinary school. How to use an air fryer for slow roasted tomatoes. Ways to prepare a delicata squash. Mike's motivation for starting The Frizzled Leek website and what it offers home cooks. How an air fryer and sous vide preparations can help with dinner prep. His tips for grilling scallions.

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2023.08.02.551383v1?rss=1 Authors: Cohen, M. L., Brumwell, A. N., Ho, T. C., Montas, G., Golden, J. A., Jones, K. D., Wolters, P. J., Wei, Y., Chapman, H. A., Le Saux, C. J. Abstract: Reciprocal interactions between alveolar fibroblasts and epithelial cells are crucial for lung homeostasis, injury repair, and fibrogenesis, but underlying mechanisms remain unclear. To investigate this, we administered the fibroblast-selective TGF{beta}1 signaling inhibitor, epigallocatechin gallate (EGCG), to Interstitial Lung Disease (ILD) patients undergoing diagnostic lung biopsy and conducted single-cell RNA sequencing on spare tissue. Unexposed biopsy samples showed higher fibroblast TGF{beta}1 signaling compared to non-disease donor or end-stage ILD tissues. In vivo, EGCG significantly downregulated TGF{beta}1 signaling and several pro-inflammatory and stress pathways in biopsy samples. Notably, EGCG reduced fibroblast secreted Frizzle-like Receptor Protein 2 (sFRP2), an unrecognized TGF{beta}1 fibroblast target gene induced near type II alveolar epithelial cells (AEC2s). In human AEC2-fibroblast coculture organoids, sFRP2 was essential for AEC2 trans-differentiation to basal cells. Precision cut lung slices (PCLS) from normal donors demonstrated that TGF{beta}1 promoted KRT17 expression and AEC2 morphological change, while sFRP2 was necessary for KRT5 expression in AEC2-derived basaloid cells. Wnt-receptor Frizzled 5 (Fzd5) expression and downstream calcineurin-related signaling in AEC2s were required for sFRP2-induced KRT5 expression. These findings highlight stage-specific TGF{beta}1 signaling in ILD, the therapeutic potential of EGCG in reducing IPF-related transcriptional changes, and identify the TGF{beta}1-non-canonical Wnt pathway crosstalk via sFRP2 as a novel mechanism for dysfunctional epithelial signaling in Idiopathic Pulmonary Fibrosis/ILD. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC

Annie is deze week in sublieme vorm. Met licht gerechten met veel groenten, perfect voor het warme weer. Met een klein stukje brood erbij voor de klein eters en een groot stuk voor de grote eters. Het leven is niet zo ingewikkeld.Het menu van deze weekKikkererwten salade met feta/halloumi van AllisonSpaghetti met romige bloemkoolsaus en krokant broodkruimSalmorejoSpinaziesalade met pitacroutons, dadels en amandelenMosselen met erwtjes en dragonBestellen Kan Altijd Nog is een productie van Wat Schaft de Podcast. Muziek van Mell & Vintage Future. Adverteren? adverteren@watschaftdepodcast.nlZie het privacybeleid op https://art19.com/privacy en de privacyverklaring van Californië op https://art19.com/privacy#do-not-sell-my-info.

The Grand High Witch stands before all the witches. Never before has anything so scary been seen. Listen but only if you're not having nightmares! --- Support this podcast: https://podcasters.spotify.com/pod/show/sarah25628/support

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2023.03.31.535068v1?rss=1 Authors: Shi, Q., Zheng, L. Abstract: Long-term space flight will lead to bone loss and osteoblasts dysfunction. The underlying mechanism is still far to reveal. Frizzled-9 (Fzd9) is a Wnt receptor which is essential to osteoblasts differentiation and bone formation. Here we investigate whether Fzd9 plays a role in simulated microgravity (SMG) induced osteoblasts dysfunction. After 1-3 days of SMG, the osteogenic markers were decreased which accompanied the decline of Fzd9 expression. Fzd9 also decreased in the femur of the rats after 3 weeks of hindlimb unloading. Overexpression of Fzd9 will counteract SMG-induced osteoblasts dysfunction. However, Fzd9 overexpression did not affect SMG induced pGSK3 and -catenin expression or sublocalization. Overexpression of Fzd9 regulates the phosphorylation of Akt and ERK, as well as induces F-actin polymerization to form the actin cap, presses the nuclei, and increases the nuclear pore size, which promotes nuclear translocation of YAP. Our study provides mechanistic insights into the role of Fzd9 regulates YAP in SMG-mediated osteoblasts dysfunction and indicates Fzd9 as a potential target to restore osteoblast function in bone diseases and space flight. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC

Big bold flavors and a big bold personality — Alison Roman brings it all in this book about “having people over” and not “entertaining,” thank you very much. Renee thinks Roman shows her immature side and we look past the very 1970s photography style to get to the meat of this popular cookbook. Resources mentioned in this episode: Alison Roman Small Kitchen, Big Thanksgiving video Sweet Enough Recipes mentioned in this episode: Creamy goat cheese with lemony za'atar (page 53) Seeded breadsticks with parmesan (page 66) Labne with sizzled scallions and chile (almost ranch) (page 22) A better garlic bread/Caramelized garlic on toast with anchovies (page 73) Frizzled chickpeas and onions with feta and oregano (page 168) Roasted squash with yogurt and spiced, buttered pistachios (page 148) The perfect herby salad (page 86) Celery salad with cilantro and sesame (page 100) Summer squash with basil, parmesan and toasted buckwheat (page 108) Smashed cucumbers with sizzled turmeric and garlic (page 107) Spicy caramelized leeks with fresh lemon (page 140) Buttered turmeric rice with crushed almonds and herbs (page 173) A very good lasagna (page 256) One-pot chicken with dates and caramelized lemon (page 194) Buttered salmon with red onion and dill (page 247) Salmon with soy and citrusy charred scallions (page 238) Lemony turmeric tea cake (page 309) Crushed blackberry and cornmeal cake (page 272) The only pie crust (page 311) Casual apple tart with caramelized buttermilk (page 284) Sweet and salty cream cheese tart (page 292) Join our Cookbook Club! Our Instagram, @cookbookclubshow E-mail us: cookbookclubshow@gmail.com Find Renee and Sara on Instagram: @hipchickdigs and @realtor_saragray Cook along with us! Next cookbook episode (releasing 12/28/2022): Super Natural Every Day, by Heidi Swanson

We would like to thank the sponsor of this episode, BTG Speciality Pharmaceuticals. BTG provides rescue medicines typically used in emergency rooms and intensive care units to treat patients for whom there are limited treatment options. They are dedicated to delivering quality medicines that make a real difference to patients and their families through the development, manufacture, and commercialization of pharmaceutical products. Their current portfolio of antidotes counteracts certain snake venoms and the toxicity associated with some heart and cancer medications. Their drug, Voraxaze, is for high-dose methotrexate toxicity. --- Dr. Nancy DeMore is a board-certified breast surgical oncologist and is the BMW endowed chair in cancer research at MUSC Health. Dr. DeMore obtained her medical degree from Chicago Medical School. She completed her residency at Boston University Medical Center, a cancer research fellowship at Harvard Medical School, and fellowship training at Memorial Sloan Kettering Cancer Center. Dr. DeMore practiced as a professor of surgery at the University of North Carolina-Chapel Hill for 13 years. --- What We Do at MIB Agents: PROGRAMS: ✨ End-of-Life MISSIONS ✨ Gamer Agents ✨ Agent Writers ✨ Prayer Agents ✨ Healing Hearts - Bereaved Parent Support ✨ Ambassador Agents - Peer Support ✨ Warrior Mail ✨ Young Adult Survivorship Support Group ✨ EDUCATION for physicians, researchers and families: ✨ OsteoBites, weekly webinar & podcast with thought leaders and innovators in Osteosarcoma ✨ MIB Book: Osteosarcoma: From our Families to Yours ✨ RESEARCH: Annual MIB FACTOR Research Conference ✨ Funding $100,000 annually for OS research ✨ MIB Testing & Research Directory ✨ The Osteosarcoma Project partner with Broad Institute of MIT and Harvard ... Kids are still dying with 40+ year old treatments. Help us MakeItBetter.

It's Time to GET FRizled! what does that mean? What a great question! we don't know! We also don't know what "Bussin" means. We also aren't sure how to make money on making video games but we're sure you all know that by now. This week the boys get frizzled, Redefine RPG, and peel some fighters! Patreon - https://www.patreon.com/Spiritualsuccessorpodcast?fan_landing=true Merch - https://streamlabs.com/spiritualsuccessorpodcast/merch Operation Tango Let's Play! - https://youtu.be/KX-a4yn37eo Cheap Shop by Anamanaguchi - https://open.spotify.com/album/3DNHW2ckMRr3v0f7JF3Hc Find out more at https://spiritual-successor.pinecast.co

The crew is back and they are going to discuss a lot. Day 2 of the tampering period has added 1 player to the 49ers and a draft pick has left. The team is now at 9 picks and a familiar face has been added to the mix. After the dust has settled from Day 1, how do the guys feel with the moves made and much much more!

Building on our discussion of both signalling pathways and G-protein coupled receptors from a few weeks ago, this week we're going to discuss the Wnt signalling pathway- a pathway which is crucial in aspects of development and can have widespread impacts on the cell. It all sounds rather intimidating, but there's actually surprisingly few steps involved in this pathway- and we're only going to be focusing on one part of the signalling network, which makes life a lot less complicated! Sources for this episode: 1) Alberts, Johnson, Lewis, Raff, Roberts, and Walter (2008), Molecular Biology of the Cell, Fifth Edition, p.948- 950. Abingdon: Garland Science, Taylor and Francis Group LLC. 2) Janda, C. Y., Waghray, D., Levin, A. M., Thomas, C. and Garcia, K. C. (2012), Structural basis of Wnt recognition by Frizzled. Science 337: 59- 63. 3) Author unknown, National Center for Biotechnology Information (2020), PubChem Compound Summary for CID 445638, Palmitoleic acid (online) [Accessed 29/11/2020].

Twenty years ago, Professor Elizabeth Vincan set out to understand how cancer cells ‘talk' to each other and the cells around them. Her research group was among a number to realise that some cancer cells always ‘switched on' specific genes that function in an ancient form of cell-to-cell communication. And the idea was that if you could find out what these genes did, and block them, it could provide a new way to treat cancer. “At that time I was a young post doc mum and working where I was working was just too difficult because I had to go over the West Gate Bridge,” she says. So when a position came up at Western Hospital that suited her, she fell into cancer research. “The good thing about that is that I don't actually have any formal cancer training, so when I address a question I come at it from a completely different tack. So that has been instrumental in the path that my career has taken,” Professor Vincan says. “What I realised way back in the late 1990s is that growing – a solid tumour, for example, is very similar to growing an organ in the body. The same pathways are involved.” Since then Professor Vincan, her lab and collaborators have been recognised for their work on cell to cell communication in cancer. “We've shown that the genetic mutations that initiate cancer form the platform of disease. On top of that there's the genes my lab work on, involved in a particular communication pathway. When we block a gene called Frizzled-7, we can actually hit cancer on the head,” she says. Now, that to me is – has just been wonderful.” Episode recorded: November 12, 2019. Interviewer: Dr Andi Horvath. Producer, editor and audio engineer: Chris Hatzis. Co-producers: Silvi Vann-Wall and Dr Andi Horvath. Image: Lorna McInroy/ Wellcome Trust.

Twenty years ago, Professor Elizabeth Vincan set out to understand how cancer cells ‘talk’ to each other and the cells around them. Her research group was among a number to realise that some cancer cells always ‘switched on’ specific genes that function in an ancient form of cell-to-cell communication. And the idea was that if you could find out what these genes did, and block them, it could provide a new way to treat cancer. “At that time I was a young post doc mum and working where I was working was just too difficult because I had to go over the West Gate Bridge,” she says. So when a position came up at Western Hospital that suited her, she fell into cancer research. “The good thing about that is that I don’t actually have any formal cancer training, so when I address a question I come at it from a completely different tack. So that has been instrumental in the path that my career has taken,” Professor Vincan says. “What I realised way back in the late 1990s is that growing – a solid tumour, for example, is very similar to growing an organ in the body. The same pathways are involved.” Since then Professor Vincan, her lab and collaborators have been recognised for their work on cell to cell communication in cancer. “We’ve shown that the genetic mutations that initiate cancer form the platform of disease. On top of that there’s the genes my lab work on, involved in a particular communication pathway. When we block a gene called Frizzled-7, we can actually hit cancer on the head,” she says. Now, that to me is – has just been wonderful.” Episode recorded: November 12, 2019. Interviewer: Dr Andi Horvath. Producer, editor and audio engineer: Chris Hatzis. Co-producers: Silvi Vann-Wall and Dr Andi Horvath. Image: Lorna McInroy/ Wellcome Trust.

Join Corrie Perkin and Caroline Wilson for Ep 110 'Frizzled and Grizzled'. It certainly feels like we're on the down hill run to Christmas - for our Melbourne and Victorian listeners we'd love you to join us at our Live Christmas Podcast Event - there's still plenty of tickets available, so make a night of it! WHERE – the rooftop at Bells in South Melbourne.  WHEN – Tuesday December the 10th, 6.30pm – 8.30pm HOW TO BOOK – Email Tara via events@crocmedia.com or phone (03) 8825 6605.   This week Corrie and Caro discuss a recent incident in sleepy Woodend, where Nazi symbols were paraded around a supermarket and they ponder the importance of keeping the truth of the Holocaust alive, especially through screen and literature.  Prince Andrew's been sacked by his Mum, his birthday party's been called off and we can't help but see the similarities in storylines playing out on the new season of The Crown.  Caro takes us inside AFL HQ with a couple of theories about a succession play should Gillon McLachlan step down at the end of next year.  Corrie's 'Crush Of the Week' is Pete Buttigieg who's a candidate for the Democratic Party nomination in 2020. You can listen to his interview on The Daily HERE.  We've got plenty of great recommendations in BSF, Corrie's been reading Damascus by Christos Tsiolkas (available HERE) - a confronting read not for the faint hearted. Caro's been to the cinema to see Knives Out and Corrie shares a great recipe from Alison Roman's Nothing Fancy cookbook. . .Frizzled Chickpeas and Onions with Oregano and Mint. (see recipe below).  Caro's Grumpy that Marie Kondo first urged us to de clutter but has now launched an online store to sell us back the clutter of the future!  OTHER FILMS AND BOOKS MENTIONED IN THIS EPISODE Movies: Hope Gap and The Good Liar.  Books: Rebel Prince: The Power, Passion and Defiance of Prince Charles by Tom Bower.   For videos and pics make sure you follow us on Instagram @DontShootPod. Like our Facebook page and hit 'Sign Up' to receive weekly updates HERE.  Email the show via feedback@dontshootpod.com.au 'Don't Shoot The Messenger' is produced, engineered and edited by Jane Nield for Crocmedia.   Frizzled Chickpeas with onion, oregano and fetta. From the cookbook Nothing Fancy by Alison Roman Ingredients 1/2 cup olive oil 1 large red or yellow onion, thinly sliced 2x 445g cans chickpeas, drained and rinsed 4 garlic loves, smashed Pinch of crushed red pepper flakes 4 sprigs marjoram or oregano, plus more leaves for garnish 60 g feta, thinly sliced or crumbled Steps Heat the olive oil in a large skillet over medium-high heat. Add the onion and season with salt and pepper. Cook, stirring occasionally, until the onion has softened, and is just starting to brown, 5 to 8 minutes. Add the chickpeas, garlic, red pepper flakes, and half the marjoram or oregano leaves. Season with salt and pepper, and toss to coat. Continue to cook, shaking the skillet occasionally to make sure nothing is stick and that the chickpeas are getting equal attention from the oil and heat, until the chickpeas are golden brown and appear fried around the edges and the onion is a deep golden brown and looks somewhere between fried and caramelized. Season to taste. Remove from the heat and transfer to a large serving bowl. Top with the feta and remaining marjoram or oregano.          

A peaceful fishing excursion at Restrepum Inlet turns into a murder mystery when the gang finds a dead body. However, who he was and what he was doing on private property are not the only reasons for concern... Featuring stories on Tamam Shud and the Sommerset Man, Gloria Ramirez, Roland T. Owen and Room 1046. Special thanks to Sabrina Ambra!

Join us this week as we talk with Ansley Davies, Associate Curator for the Los Angeles County Department of Parks & Recreation about the otherworldlyContinue Reading

PAPC separates tissues at a Snail's pace Brachet's cleft separates the ectoderm and mesoderm of early Xenopus and zebrafish embryos. Luu et al. reveal that, in conjunction with the transcription factor Snail1, the protocadherin PAPC promotes tissue separation by down-regulating planar cell polarity proteins at the ectoderm-mesoderm boundary. This biosights episode presents the paper by Luu et al. from the March 16, 2015, issue of The Journal of Cell Biology and includes an interview with one of the paper's senior authors, Rudolf Winklbauer (University of Toronto, Toronto, Canada). Produced by Caitlin Sedwick and Ben Short. See the associated paper in JCB for details on the funding provided to support this original research. Subscribe to biosights via iTunes or RSS View biosights archive The Rockefeller University Press biosights@rockefeller.edu

Codit gets his big chance when Detective Dendro lets him take the lead investigating palms with withered fronds standing alongside other healthy palms of the same species. Can Codit make his mentor proud? Find out in the Case of the Frizzled Fronds.(A,U,T,M,L,Bs)

Der in der Evolution hochkonservierte Wnt-Signalweg spielt in der Embryonalentwicklung, der Stammzellbiologie sowie in der Entstehung von Tumoren eine tragende Rolle. Es werden dabei verschiedene Arten der Wnt-vermittelten Signalübertragung unterschieden: Einerseits der sogenannte kanonische, über β-Catenin vermittelte Wnt-Signalweg und andererseits der Wnt/Ca2+- sowie der planar cell polarity (PCP)-like-Weg, die beide die Signaltransduktion unabhängig von β-Catenin übermitteln. Bislang sind die Mechanismen des Wnt/β-Catenin-Signalweges nur teilweise aufgeklärt, wobei insbesondere das Zusammenspiel von Wnts, Frizzleds und deren Korezeptoren auf funktioneller und struktureller Ebene bis heute nur rudimentär beschrieben wurden. In unserer Arbeitsgruppe konnte in vorangegangenen Arbeiten gezeigt werden, dass der Frizzled-8-Rezeptor (Fzd8) in HT1080-Fibrosarkomzellen im Verhältnis zu den übrigen Fzds sehr stark exprimiert wird und seine erhöhte Expression mit einer Aktivierung des Wnt/β-Catenin-Signalweges sowie mit einer erhöhten Invasivität und Proliferation assoziiert ist (Leitenstern et al., mündliche Mitteilung). Ferner wurde gezeigt, dass die Expression von Fzd8 durch Wnt3a bzw. den kanonischen Wnt-Signalweg negativ reguliert wird (Karow 2008; Kolben, Perobner et al. 2012). Vor diesem Hintergrund sollte nun im Rahmen dieser Arbeit mit Hilfe von Reportergenexperimenten die transkriptionelle Regulation von Fzd8 im Detail untersucht werden. Hierzu wurden die Fzd8-Promotorregion und verschiedene 5’- und 3’-terminale Verkürzungsvarianten in den Vektor pGLuc-Basic kloniert, der eine Quantifizierung der Promotoraktivität mittels des Reporterproteins Gaussia Luciferase erlaubt. Der Fzd8-Promotor konnte durch dieses Verfahren in aktivierende und repressive Elemente unterteilt werden, wobei auch ein putatives Enhancer-Element identifiziert werden konnte. Unter Wnt3a-Stimulation zeigten alle untersuchten Promotorkonstrukte eine verringerte Reporterproteinaktivität, was auf eine negative Regulation von Fzd8 durch Wnt3a hinweist. Interessanterweise aber führten Manipulationen am Wnt/β-Catenin-Signalweg weiter downstream zu entgegengesetzten Ergebnissen. So führte ein APC-Knockdown, der mit einer starken Aktivitätszunahme des Wnt/β-Catenin-Signalweges einhergeht, zu einer Zunahme der Fzd8-Promotoraktivität. Im Gegensatz hierzu ging ein Knockdown von β-Catenin, der einer Inhibierung des Wnt/β-Catenin-Signalweges gleichkommt, mit einer verminderten Fzd8-Promotoraktivität einher. Diese Ergebnisse deuten darauf hin, dass die Wnt3a-vermittelte Repression von Fzd8 auch unabhängig vom kanonischen Wnt/β-Catenin-Signalweg erfolgen könnte. Des Weiteren wurde die Fzd8-Promotorregion einer in silico-Analyse mit der Software MatInspector unterzogen, um mögliche Transkriptionsfaktoren zu identifizieren, die für die Fzd8-Regulation maßgeblich sein könnten. Dabei zeigte sich, dass der Transkriptionsfaktor ZF5, der sowohl aktivierend als auch reprimierend an verschiedenen Promotoren wirken kann, aufgrund des Verteilungsmusters seiner Bindungsstellen am Fzd8-Promotor als möglicher Regulator fungieren könnte. ZF5 konnte dabei als putativ positives Wnt/β-Catenin-Zielgen charakterisiert werden. Es konnte ferner gezeigt werden, dass Fzd8 indirekt über ZF5 durch den Wnt/β-Catenin-Signalweg induziert werden kann. Da ZF5 wie auch Fzd8 in HT1080-Fibrosarkomzellen stark exprimiert wird und die ZF5-Überexpression mit einer Zunahme des Wnt/β-Catenin-Signals einhergeht, könnte hier ein positiver feedback loop vorliegen. Somit könnte diese Amplifikation des Wnt/β-Catenin-Signals von maßgeblicher Bedeutung für die verstärkte Proliferation und Invasivität von mesodermalen Tumoren sein.

Structural analysis reveals how a Wnt binds to its receptor.

Wnt/β-catenin signaling is of fundamental importance in the regulation of self-renewal, migration/invasion, and differentiation of human mesenchymal stem cells (hMSCs). Because little information is available about the function of Frizzled receptors (Fzds) as the main receptors of Wnt proteins in hMSCs, we first performed comparative Fzd mRNA expression profiling. Fzd9 and Fzd10 were not expressed in hMSCs. While Fzd3 was expressed at low levels in hMSCs, the other Fzds exhibited high expression rates. Activation and repression of Wnt signaling in hMSCs revealed that the expression levels of Fzd1, Fzd6, and Fzd7 are positively correlated with the Wnt/β-catenin activation status, whereas Fzd8 exhibited an inverse relation. For studying the functional relevance of Fzds in Wnt/β-catenin signaling, RNA interference, ectopic expression studies, and rescue approaches were performed in hMSCs carrying a highly sensitive TCF/LEF reporter gene system (Gaussia luciferase). We found that, Fzd1, Fzd5, Fzd7, and Fzd8 are largely involved in Wnt/β-catenin signaling of hMSCs. Moreover, the knockdown of Fzd5 can be compensated by the ectopic expression of Fzd7. Conversely, the ectopic expression of Fzd5 in Fzd7-knockdown hMSCs resulted in a rescue of Wnt/β-catenin signaling, pointing to a functional redundancy of Fzd5 and Fzd7.

Thu, 30 Jun 2011 12:00:00 +0100 https://edoc.ub.uni-muenchen.de/13188/ https://edoc.ub.uni-muenchen.de/13188/1/Kolben_Thomas.pdf Kolben, Thomas

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