Podcasts about Polycystic kidney disease

In this episode, hosts Dr. Abby Eblen from Nashville Fertility Center, Dr. Susan Hudson from Texas Fertility Center, and Dr. Carrie Bedient from the Fertility Center of Las Vegas speak with Richard Kellner, founder of the Polycystic Kidney Disease-Free Alliance, and Natasha Rogina, the organization's chief spokesperson. Together, they discuss polycystic kidney disease (PKD) and how pre-implantation genetic testing-monogenic (PGT-M) can help families prevent passing this genetic condition to their children. Natasha, who has PKD herself, shares her personal decision to pursue IVF with PGT-M to ensure her children would not inherit the disease. PKD is a progressive condition that typically leads to kidney failure by midlife, often requiring dialysis or a transplant. Because many insurers do not cover genetic testing, the PKD-Free Alliance offers grants to help families access PGT-M to prevent passing the trait to their children. The PKD-Free Alliance's goal is to raise awareness and let couples know this option exists. This insightful conversation explores genetic inheritance, reproductive choices, and the future of PKD prevention, offering hope to families facing this challenging condition.

Joe speaks to siblings William and Noreen O'Halloran, who both suffer from Polycystic Kidney Disease, about the importance of organ donation Hosted on Acast. See acast.com/privacy for more information.

This episode covers autosomal recessive polycystic kidney disease.Written notes can be found at https://zerotofinals.com/paediatrics/renal/arpkd/Questions can be found at https://members.zerotofinals.com/Books can be found at https://zerotofinals.com/books/The audio in the episode was expertly edited by Harry Watchman.

Polycystic kidney disease is the most common genetic kidney disease worldwide and is characterized by progressive development of kidney cysts. Author Nerra Dahl, MD, PhD, Mayo Clinic, discusses the pathophysiology, clinical presentation, diagnosis, prognosis, and treatment of ADPKD in adults, with JAMA Associate Editor David Simel, MD, MHS. Related Content: Autosomal Dominant Polycystic Kidney Disease

"Only move from love, don't move from fear." That's the mantra guiding Mark Brand, a world-class chef who went from spiraling into self-destruction to serving over 2,200 meals a day to Vancouver's most vulnerable. At 27, Mark was diagnosed with Polycystic Kidney Disease—a degenerative condition that would eventually shut down his organs. Doctors warned him he'd be on dialysis by 35 and told him to slow down. Instead, he went harder—booze, drugs, chaos. But rock bottom isn't always the end. For Mark, it was the beginning of something bigger. In this raw and powerful episode, Mark opens up about his journey from near-death to radical purpose, the moment that changed everything, and how food became his weapon in the fight against hunger, inequality, and despair.Be sure to catch the full video version of this episode on YouTube.Follow Sickboy on Instagram, TikTok and Discord!

"Only move from love, don't move from fear." That's the mantra guiding Mark Brand, a world-class chef who went from spiraling into self-destruction to serving over 2,200 meals a day to Vancouver's most vulnerable. At 27, Mark was diagnosed with Polycystic Kidney Disease—a degenerative condition that would eventually shut down his organs. Doctors warned him he'd be on dialysis by 35 and told him to slow down. Instead, he went harder—booze, drugs, chaos. But rock bottom isn't always the end. For Mark, it was the beginning of something bigger. In this raw and powerful episode, Mark opens up about his journey from near-death to radical purpose, the moment that changed everything, and how food became his weapon in the fight against hunger, inequality, and despair.Be sure to catch the full video version of this episode on YouTube.Follow Sickboy on Instagram, TikTok and Discord!

In today's episode, we're finishing up our series on kidney failure treatment options with a discussion on conservative care. What exactly is conservative care and who can benefit from it? Christine Corbett, Associate Chief Nursing Officer and nurse practitioner specializing in palliative care is here to answer that question and more. Sara Hicklin and Bobbie Reed who have firsthand experience with conservative care in their families are also here to share their perspectives on this treatment option. Christine Corbett, DNP, APRN, FNP-BC, CNN-NP, FNKF is the newly appointed Assistant Research Professor at George Washington University and Executive Director for the Coalition for Supportive Care of Kidney Patients. She is a practicing Nurse Practitioner specializing in kidney palliative care, and a Trauma-Sensitive HeartMath Certified Practitioner. Dr. Corbett has over 25 years of nephrology experience and four years of experience in Palliative Care. Her doctoral focus was shared decision-making, advance care planning and appropriate palliative care referral for select patients with chronic kidney disease. In the past four years she developed, implemented, and managed the Comprehensive Conservative Kidney Care Clinic for patients who choose to forego dialysis at an academic safety net hospital in Kansas City. Bobbie Reed is on a mission for her son and for the millions of others living with chronic kidney disease. A graduate of Indiana University of Pennsylvania with a Bachelor of Family Consumer Science degree, Bobbie is the office manager at her family's insurance business. She wants to pay it forward by helping others with their struggles in dealing with kidney disease. As part of the NKF's Kidney Advocacy Committee, Bobbie is not only the Pennsylvania Liaison, but is the immediate past Region 2 leader for the group.  She continues to help her son navigate his kidney disease journey and, in addition, use her experience to benefit and advocate for many others afflicted with kidney disease. Sara Hicklin and her family has a genetic kidney disease called Polycystic Kidney Disease or PKD. With this family and personal history, Sara is passionate about educating others about kidney disease, along with recruiting and educating potential organ donors. She is also fascinated by the advances in treatment options in three generations and wants to contribute in a small way to what may become available for treatment of kidney disease in generations to come.   Additional Resources Coalition for Supportive Care Palliative Care Resources Palliative Care Resources Advanced Directives The Patient Will See You Now by Eric Topol Palliative Care in Nephrology Definitions from the episode: Durable Power of Attorney (DPOA) Portable Medical Orders (POLST): https://polst.org/   Do you have comments, questions, or suggestions? Email us at NKFpodcast@kidney.org. Also, make sure to rate and review us wherever you listen to podcasts.

Episode 48 of the PKD Dietitian Podcast kicks off the first "Fact vs. Fiction" segment tackling myths about meat consumption for those with Polycystic Kidney Disease. Is it necessary to eliminate meat from your diet if you have PKD? The PKD Dietitian will clarify that while a PKD diagnosis doesn't mean you must stop eating meat, paying attention to the type and amount of protein you consume is important. Diana will break down the three primary protein sources—plant protein, animal meat, and animal-derived products—and highlight the benefits of incorporating more plant-based proteins into your diet. With practical tips like Meatless Mondays and understanding plant-forward eating, she aims to clear up misconceptions and offer actionable advice to support your PKD kidney health through mindful protein consumption. SCHEDULE YOUR PKD CONNECT CALL: https://p.bttr.to/3yDTdkE REFERENCES https://www.clinicalnutritionopenscience.com/article/S2667-2685(23)00047-5/fulltext https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7460905/ https://pubmed.ncbi.nlm.nih.gov/23302719/ https://journals.lww.com/jasn/fulltext/1999/11000/dietary_protein_restriction_and_the_progression_of.21.aspx https://www.sciencedirect.com/science/article/abs/pii/S0899900717301971 https://www.sciencedirect.com/science/article/abs/pii/S1051227619302651 https://journals.lww.com/cjasn/pages/articleviewer.aspx?year=2019&issue=05000&article=00009&type=Fulltext DISCLAIMER The PKD Dietitian Podcast is meant for educational purposes only. It is not a substitute for professional medical advice, diagnosis, or treatment. Always consult your healthcare provider with any medical questions or concerns.

Richard Kellner is on a mission to end polycystic kidney disease (PKD), a leading cause of kidney failure in younger people. The key is combining  pre-implantation genetic testing with in vitro fertilization to prevent the disease from being passed from parent to child. The PKD Outreach Foundation, run by Richard is raising awareness so that physicians and parents-to-be know about this option. PKD Outreach is also making testing and IVF more affordable and accessible through a partnership with US Fertility and by making cash grants to families.In addition, PKD Outreach is  publicizing ways to slowing the progression in young adults with lifestyle and drugs, and supporting live kidney donation.Richard has a compelling personal story. His late wife passed away from PKD, even after receiving a kidney donation from Richard. His adult kids have it, too.I like the organization's mission and determination so I decided to donate. You might want to do the same.Host David E. Williams is president of healthcare strategy consulting firm Health Business Group. Produced by Dafna Williams.

Polycystic Kidney Disease is a genetic condition you may not have heard of, but it affects over 600,000 people in the U.S. and is one of the leading causes of kidney failure. Our guest is Richard Kellner, who founded The PKD Outreach Foundation, after his wife passed away, and who has been working with over 100 families across the country and currently has 10 families who have had or are expecting PKD-free babies. For more, visit pkdo.org.

Send us a textPolycystic Kidney Disease is consider to be an uncurable and irreversible disease.But what if this long-thought unstoppable disease could, in fact, be slowed or even prevented from being passed on to future generations?In this week's episode of HealthBiz Briefs, guest Richard Kellner, founder of PKD Outreach, shares his connection to the disease and how his company is working to end this debilitating condition for future generations.This episode is brought to you by BetterHelp. Give online therapy a try at https://betterhelp.com/caretalk and get on your way to being your best self.As a BetterHelp affiliate, we may receive compensation from BetterHelp if you purchase products or services through the links provided.

Listen as Dr. London Smith (.com) and his producer Cameron discuss Autosomal Recessive Polycystic Kidney Disease with special guest Skunky Maltodextress (Victor Olivas). Not so boring! https://www.patreon.com/join/jockdocpodcast Hosts: London Smith, Cameron Clark. Guest: Victor Olivas. Produced by: Dylan Walker Created by: London Smith

Listen as Dr. London Smith (.com) and his producer Cameron discuss Autosomal Dominant Polycystic Kidney Disease with special guest Dr. Liverpool Smythe (Jennifer Prescott). Not so boring! https://www.patreon.com/join/jockdocpodcast Hosts: London Smith, Cameron Clark. Guest: Jennifer Prescott. Produced by: Dylan Walker Created by: London Smith

In this heartfelt episode of Kent's Kidney Stories, host Kent Bresler reconnects with Ali Dawson and Sonia, two inspiring individuals from London. Their story is a testament to the power of friendship, kindness, and the life-changing impact of kidney donation. We hear about the incredible journey of Sonia, who suffers from polycystic kidney disease, and her miraculous connection with Ali, a former student who selflessly stepped forward to donate his kidney. Sonia shares the emotional rollercoaster of her diagnosis, the challenges of dialysis, and the overwhelming gratitude she feels for Ali's altruistic act. Ali, in turn, discusses his motivations and the unwavering determination that led him to become a living donor. The episode delves into the differences in kidney donation practices between the UK and the US, highlighting the need for greater awareness and support for living donors. Kent, Ali, and Sonia explore the importance of preemptive transplants, the emotional and physical toll of dialysis, and the profound bond that forms between donors and recipients. Join us for this moving conversation that underscores the significance of organ donation and the enduring impact it has on the lives of recipients and their families. Whether you're a patient, a potential donor, or simply someone interested in the transformative power of organ donation, this episode offers valuable insights and inspiration. For more information on Kidney Solutions and to join their supportive community, visit kidneysolutions.org. Host: Kent Bresler Producer: Jason Nunez Remember to keep breathing, and don't miss the next amazing episodes of Kent's Kidney Stories!  

In this episode we are joined by Nephrology Specialist Registrar Dr Sarah Cormican to discuss polycystic kidney disease as potential station in the MRCPI Part II clinical exam. Link to Pastest MRCP Clinical revision resource: https://bit.ly/468j0lT Consultant supervisor Professor Declan Byrne, consultant physician St James's Hospital, Dublin Show music by Stepping on Lego - email: steppingonlegoband@gmail.com - socials: @steppingonlegoGet in touch with us by email mrcpibedside@gmail.com or social media @mrcpibedsideGood luck!

What is vasopressin and how is it connected to Polycystic Kidney Disease? In this episode, the PKD Dietitian is going to: Break down exactly what vasopressin is. Explain how it functions in the body and how it affects your kidneys. Highlight how it impacts PKD cyst growth. PLUS share dietary and lifestyle factors that impact vasopressin levels...for better or worse. RESOURCES BLOG: What is a Good Fluid Goal for Polycystic Kidney Disease? EPISODE 3: Listener Q - How Much Water Should I Drink? REFERENCES Torres VE. Salt, water, and vasopressin in polycystic kidney disease. Kidney Int. 2020;98(4):831-834. doi:10.1016/j.kint.2020.06.001 ARTICLE LINK Van Gastel MDA, Torres VE. Polycystic Kidney Disease and the Vasopressin Pathway. Ann Nutr Metab. 2017;70 Suppl 1:43-50. doi:10.1159/000463063 ARTICLE LINK Taylor JM, Hamilton-Reeves JM, Sullivan DK, et al. Diet and polycystic kidney disease: A pilot intervention study. Clin Nutr. 2017;36(2):458-466. doi:10.1016/j.clnu.2016.01.003 ARTICLE LINK Amro OW, Paulus JK, Noubary F, Perrone RD. Low-Osmolar Diet and Adjusted Water Intake for Vasopressin Reduction in Autosomal Dominant Polycystic Kidney Disease: A Pilot Randomized Controlled Trial. Am J Kidney Dis. 2016;68(6):882-891. doi:10.1053/j.ajkd.2016.07.023 ARTICLE LINK Maatje D.A. van Gastel, Vicente E. Torres; Polycystic Kidney Disease and the Vasopressin Pathway. Ann Nutr Metab 15 June 2017; 70 (Suppl. 1): 43–50. ARTICLE LINK Torres, Vicente E.; Harris, Peter C. Strategies Targeting cAMP Signaling in the Treatment of Polycystic Kidney Disease. Journal of the American Society of Nephrology 25(1):p 18-32, January 2014. | DOI: 10.1681/ASN.2013040398 ARTICLE LINK Wang, Xiaofang; Wu, Yanhong; Ward, Christopher J.; Harris, Peter C.; Torres, Vicente E.. Vasopressin Directly Regulates Cyst Growth in Polycystic Kidney Disease. Journal of the American Society of Nephrology 19(1):p 102-108, January 2008. | DOI: 10.1681/ASN.2007060688 ARTICLE LINK Tasneem M, Mannix C, Wong A, Zhang J, Rangan G. Is serum copeptin a modifiable biomarker in autosomal dominant polycystic kidney disease?. World J Nephrol. 2018;7(2):51-57. doi:10.5527/wjn.v7.i2.51 ARTICLE LINK Bankir L, Roussel R, Bouby N. Protein- and diabetes-induced glomerular hyperfiltration: role of glucagon, vasopressin, and urea. Am J Physiol Renal Physiol. 2015;309(1):F2-F23. doi:10.1152/ajprenal.00614.2014 ARTICLE LINK Alwis US, Delanghe J, Dossche L, et al. Could Evening Dietary Protein Intake Play a Role in Nocturnal Polyuria?. J Clin Med. 2020;9(8):2532. Published 2020 Aug 5. doi:10.3390/jcm9082532 ARTICLE LINK Grantham, Jared J.. Therapy for Polycystic Kidney Disease? It's Water, Stupid!. Journal of the American Society of Nephrology 19(1):p 1-7, January 2008. | DOI: 10.1681/ASN.2007101100 ARTICLE LINK Yamada Y, Fujiki H, Mizuguchi H, et al. Nihon Yakurigaku Zasshi. 2022;157(4):254-260. doi:10.1254/fpj.22006 ARTICLE LINK Rangan GK, Wong ATY, Munt A, et al. Prescribed Water Intake in Autosomal Dominant Polycystic Kidney Disease. NEJM Evid. 2022;1(1):EVIDoa2100021. doi:10.1056/EVIDoa2100021 ARTICLE LINK Sparapani S, Millet-Boureima C, Oliver J, et al. The Biology of Vasopressin. Biomedicines. 2021;9(1):89. Published 2021 Jan 18. doi:10.3390/biomedicines9010089 ARTICLE LINK DISCLAIMER The PKD Dietitian Podcast is meant for educational purposes only. Information presented should not be used in place of advice from a medical professional.

Lindsay struggled with infertility and went through several rounds of failed medicated cycles, with no clear answer as to why she couldn't get pregnant. She had one living child, followed by the neonatal loss of her son, Gus, due to polycystic kidney disease. Lindsay shares her experience finding out the diagnosis, going through the pregnancy, and the time following her loss. --- Support this podcast: https://podcasters.spotify.com/pod/show/findinghopeafterloss/support

Are you tired of being confused about how much protein you can, and should have with Polycystic Kidney Disease? The PROTEIN & PKD workshop is just for you. Learn how much dietary protein is good for your stage of PKD plus start making nutrition changes to support slower PKD progression. Your invitation to a live workshop with Diana, The PKD Dietitian. WORKSHOP: PKD & Protein: How To Navigate Your Needs for Kidney Health ENROLL: Early Bird Discount COUPON CODE: Protein DETAILS: March 27th, 6 pm EST Printable workbook + guides 75 min Workshop + 15 min Q&A Replay provided QUESTIONS: reach out to Diana, The PKD Dietitian at diana@thepkddietitian.com

Listener Q Episode + FAQs Q: "I occasionally fast but am considering doing a long fast, like the 3-day water fast that was done in the study. What do you think of extended fasting? Is it good for PKD?" A: Tune in and find out: If there are benefits for PKD with an extended water fast The PKD Dietitians' thoughts on if you should try a water fast FAQs on whether you should do keto FAQs on how much fluid you should drink If labs are a good marker of sodium intake PLUS ways to work with the PKD Dietitian RESOURCES MENTIONED Episode 14: How I Became The PKD Dietitian Episode 19: Ketogenic Diets: Are They Good for PKD? Episode 34: KETO-ADPKD: What Were The Study's Results? Blog: What is a Good Fluid Goal for Polycystic Kidney Disease? Info: PKD Nutrition Academy QUESTIONS? Do you have a question for The PKD Dietitian or a topic you would like covered? Contact The PKD Dietitian REFERENCES https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8369953 https://pubmed.ncbi.nlm.nih.gov/37377031/ https://pubmed.ncbi.nlm.nih.gov/29455546/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7929006/ DISCLAIMER The PKD Dietitian Podcast is meant for educational purposes only. Information presented should not be used in place of advice from a medical professional.

In this shorty episode, The PKD DIetitian tackles the Carnivore diet, what it really is, why folks with PKD are doing it, and provides her thoughts (scientifically backed of course) on whether the meat-based dietary craze is good for Polycystic Kidney Disease. RESOURCES: PKD KETO PRO - more program info CONNECT WITH THE PKD DIETITIAN INSTAGRAM: https://www.instagram.com/the.pkd.dietitian/ FACEBOOK: https://www.facebook.com/PKDdietitian PINTEREST: https://www.pinterest.com/PKDDietitian REFERENCES Can a carnivore diet provide all essential nutrients? Micronutrient Gaps in Three Commercial Weight-Loss Diet Plans Behavioral Characteristics and Self-Reported Health Status among 2029 Adults Consuming a "Carnivore Diet" Limitations of Self-reported Health Status and Metabolic Markers among Adults Consuming a "Carnivore Diet" DISCLAIMER: The PKD Dietitian Podcast is meant for educational purposes. The information provided shouldn't be considered a substitute for professional medical advice, diagnosis, or treatment.

This episode covers polycystic kidney disease.Written notes can be found at https://zerotofinals.com/medicine/renal/pkd/ or in the renal medicine section of the 2nd edition of the Zero to Finals medicine book.The audio in the episode was expertly edited by Harry Watchman.

It's been another week of nonsense reporting regarding Yoshinobu Yamamoto and we're bringing in Tony Mazz to help us filter through the bullshit. We talk about the myriad of inaccurate reports, actual reasons why he might want to sign with the Red Sox, the communication between the front office and ownership, why Mazz doesn't want the Red Sox to suck, what plan B might be after Yamamoto, and more. Jared and Tyler also go in on a certain individual who has been tweeting fake scoops for over a decade. We also welcome Sharon and Anna-Lee Boerner, the masterminds behind the “what happened to Bullet” soundbite. Sharon shares her story of battling Polycystic Kidney Disease with the help of her daughter Anna-Lee, who is recovering from surgery to remove a rare tumor. Sharon and Anna-Lee have become a huge part of the Name Redacted family and any support that our community can give to them would be greatly appreciated. The link to their gofundme is below! Thanks for listening and we'll see you next week! https://www.gofundme.com/f/sharon-and-annalee-boerner-medical-bills?utm_campaign=p_lico+share-sheet-first-launch&utm_medium=copy_link&utm_source=customer Learn more about your ad choices. Visit megaphone.fm/adchoices

Today I'm going to share some important information related to chronic kidney disease. 10% of cases of end-stage renal disease are related to polycystic kidney disease. Polycystic kidney disease has mainly been thought of as a genetic problem, and until fairly recently, many people thought you couldn't do much about genetic diseases. But it turns out that you can. Some of the top warning signs of polycystic kidney disease are increased risk of infection, urinary bleeding, reduced function of the kidneys, and high blood pressure. A recent study done on mice found that the ketogenic diet halted and reversed polycystic kidney disease in the mice. They also found that time-restricted eating created a similar effect. Another study found that the keto diet may reduce the risk of kidney stones as long as the person is also on a low-oxalate diet. Along with keto and consuming low-oxalate foods, consuming enough water may help reduce the risk of kidney stones. In the study on polycystic kidney disease, they found that the cysts absorb glucose and are dependent on glucose. Glucose can contribute to other kidney issues as well. A low-carb diet and fasting are both great ways to get your body to start making ketones. You can also take ketones. The best natural remedies for kidney problems: • Curcumin • Resveratrol • EGCG (in green tea) • Quercetin • Berberine What I would do for chronic kidney disease: 1. Take the above natural remedies 2. Go on a low-oxalate keto diet 3. Do intermittent fasting

Today I'm going to share some important information related to chronic kidney disease. 10% of cases of end-stage renal disease are related to polycystic kidney disease. Polycystic kidney disease has mainly been thought of as a genetic problem, and until fairly recently, many people thought you couldn't do much about genetic diseases. But it turns out that you can. Some of the top warning signs of polycystic kidney disease are increased risk of infection, urinary bleeding, reduced function of the kidneys, and high blood pressure. A recent study done on mice found that the ketogenic diet halted and reversed polycystic kidney disease in the mice. They also found that time-restricted eating created a similar effect. Another study found that the keto diet may reduce the risk of kidney stones as long as the person is also on a low-oxalate diet. Along with keto and consuming low-oxalate foods, consuming enough water may help reduce the risk of kidney stones. In the study on polycystic kidney disease, they found that the cysts absorb glucose and are dependent on glucose. Glucose can contribute to other kidney issues as well. A low-carb diet and fasting are both great ways to get your body to start making ketones. You can also take ketones. The best natural remedies for kidney problems: • Curcumin • Resveratrol • EGCG (in green tea) • Quercetin • Berberine What I would do for chronic kidney disease: 1. Take the above natural remedies 2. Go on a low-oxalate keto diet 3. Do intermittent fasting DATA: https://journals.physiology.org/doi/f... https://www.nih.gov/news-events/news-... https://fmcna.com/insights/articles/a... https://www.sciencedirect.com/science...

Richard Kellner, the founder of the Polycystic Kidney Disease Outreach Foundation joins us to talk about his mission for ending PKD in future generations. Emily talks about her fertility journey and why she has decided to do PGT-M (pre-genetic implantation testing). Learn more about PKDo: https://www.pkdo.org/pkd-free-babies Want to learn how to find freedom from chronic pain and fatigue? Register for my free training Flare-Up Freedom: https://www.thespooniementor.com/free/  Earn with Ease (Business Coaching): https://www.emilyfraser.ca/earnwithease Thriving Spoonie Pathway (Live Group Program for Spoonies): https://www.emilyfraser.ca/tsp Instagram: https://instagram.com/thespooniementor Website: https://www.emilyfraser.ca/  --- Send in a voice message: https://podcasters.spotify.com/pod/show/thespooniepodcast/message

Experts Drs Matthew A. Sparks and Dana V. Rizk discuss the pathology, presentation, and management of IgA nephropathy. Looking for the latest on clinical trials and approved treatments? Tune in! Relevant disclosures can be found with the episode show notes on Medscape (https://www.medscape.com/viewarticle/991603). The topics and discussions are planned, produced, and reviewed independently of advertisers. This podcast is intended only for US healthcare professionals. Resources Chronic Kidney Disease (CKD) https://emedicine.medscape.com/article/238798-overview IgA Nephropathy https://emedicine.medscape.com/article/239927-overview Polycystic Kidney Disease https://emedicine.medscape.com/article/244907-overview Biomarkers for IgA Nephropathy on the Basis of Multi-Hit Pathogenesis https://pubmed.ncbi.nlm.nih.gov/29740706/ Galactose-Deficient IgA1 as a Candidate Urinary Marker of IgA Nephropathy https://pubmed.ncbi.nlm.nih.gov/35683557/ Immunological Drivers of IgA Nephropathy: Exploring the Mucosa-Kidney Link https://pubmed.ncbi.nlm.nih.gov/34821031/ Nephrotic Syndrome https://emedicine.medscape.com/article/244631-overview Podocytopathies https://pubmed.ncbi.nlm.nih.gov/32792490/ Minimal-Change Disease https://emedicine.medscape.com/article/243348-overview IgA Vasculitis (Henoch-Schönlein Purpura) https://emedicine.medscape.com/article/984105-overview Sodium-Glucose Transport Protein 2 (SGLT2) Inhibitors https://www.ncbi.nlm.nih.gov/books/NBK576405/ Dapagliflozin in Patients With Chronic Kidney Disease https://pubmed.ncbi.nlm.nih.gov/32970396/ Empagliflozin in Patients With Chronic Kidney Disease https://pubmed.ncbi.nlm.nih.gov/36331190/ A Controlled Trial of Fish Oil in IgA Nephropathy. Mayo Nephrology Collaborative Group https://pubmed.ncbi.nlm.nih.gov/7935657/ Fish Consumption, Omega 3 Fatty Acids and Cardiovascular Disease. The Science and the Clinical Trials https://pubmed.ncbi.nlm.nih.gov/19326716/ Tonsillectomy in a European Cohort of 1,147 Patients With IgA Nephropathy https://pubmed.ncbi.nlm.nih.gov/26586175/ Sparsentan in Patients With IgA Nephropathy: A Prespecified Interim Analysis From a Randomised, Double-Blind, Active-Controlled Clinical Trial https://pubmed.ncbi.nlm.nih.gov/37015244/ Intensive Supportive Care Plus Immunosuppression in IgA Nephropathy https://pubmed.ncbi.nlm.nih.gov/26962737/ Effect of Oral Methylprednisolone on Clinical Outcomes in Patients With IgA Nephropathy: The TESTING Randomized Clinical Trial https://pubmed.ncbi.nlm.nih.gov/28763548/ Targeted-Release Budesonide Versus Placebo in Patients With IgA Nephropathy (NEFIGAN): A Double-Blind, Randomised, Placebo-Controlled Phase 2b Trial https://pubmed.ncbi.nlm.nih.gov/28363480/ Results From Part A of the Multi-Center, Double-Blind, Randomized, Placebo-Controlled NefIgArd Trial, Which Evaluated Targeted-Release Formulation of Budesonide for the Treatment of Primary Immunoglobulin A Nephropathy https://pubmed.ncbi.nlm.nih.gov/36270561/ Crescents and IgA Nephropathy: A Delicate Marriage https://pubmed.ncbi.nlm.nih.gov/35806856/ Crescentic, Proliferative IgA Nephropathy: Clinical and Histological Response to Methylprednisolone and Intravenous Cyclophosphamide https://pubmed.ncbi.nlm.nih.gov/12808169/ Hydroxychloroquine Inhibits Macrophage Activation and Attenuates Renal Fibrosis After Ischemia-Reperfusion Injury https://pubmed.ncbi.nlm.nih.gov/33936063/ New Treatment Strategies for IgA Nephropathy: Targeting Plasma Cells as the Main Source of Pathogenic Antibodies https://pubmed.ncbi.nlm.nih.gov/35628935/

Anne and Mike continue their conversation with David Rintell, Head of Patient Advocacy at BridgeBio, in part two of our autosomal dominant polycystic kidney disease (ADPKD) episode. Anne describes her recovery from the first kidney transplant along with the complications she attributes to her two native kidneys which were not removed during the initial surgery and issues with polycystic liver disease. In the 11 years following, Anne endured an operation to remove her native kidneys, followed by a double organ transplant, giving her both a new liver and kidney. Her first kidney transplant was generously gifted by a friend. However, Anne received her new liver and second kidney from a deceased donor, a distinctly different process. Anne and Mike express their gratitude for their good fortune while acknowledging the courage of the donor family to make the decision to give an extension on life to another human, when their loved one could no longer live.   Check out Anne and Mike's journey in their video, "The Search and the Gift" https://vimeo.com/33405248 . For more information about organ donation, visit http://donatelife.net/    To learn more about ADPKD, listen to Part 1 to hear Rachel Groth, Ph.D., Vice President of Research at BridgeBio X, explain why ADPKD develops, how it affects the kidneys, what complications may arise from ADPKD and why individuals with end-stage kidney disease receive dialysis or kidney transplants.

Title: “We named the new kidney - bean.” Anne is living with autosomal dominant polycystic kidney disease, (ADPKD)   Anne joins David Rintell, Head of Patient Advocacy at BridgeBio to talk about how autosomal dominant polycystic kidney disease (ADPKD) has impacted her and the lives of her family members. ADPKD weaves through the branches of Anne's family tree for generations. When Anne was only six years old, her father died at 46 of ADPKD related kidney failure, dialysis, and kidney transplantation. Two of Anne's five siblings have ADPKD. One had a successful kidney transplant, but another sibling died while on the waiting list for a new kidney. Anne wasn't surprised by her ADPKD diagnosis at age 25 and was hopeful for a better outcome than experienced by her father and her sibling since she received the diagnosis at a younger age. Progress was not reassuring to Mike, Anne's husband, when Anne's kidney function declined to less than 30%. Anne needed a kidney transplant, and soon. Motivated to find a donor and save her life, Mike created a robust campaign to locate a kidney for Anne. After many months of social posts, connections, conversations, prayers, and a documentary, a donor was discovered – a perfect match. The surgery was much quicker than the campaign. Anne left the hospital with her new kidney, appropriately named “bean,” just three days after her surgery. Rachel Groth, Ph.D., Vice President of Research at BridgeBio X, the company's dedicated lab space focused on cutting-edge early research discovery, provides an overview of ADPKD, a genetic condition that causes small fluid-filled sacs called cysts to develop in the kidneys. She explains why ADPKD develops, how it affects the kidneys, what complications may arise from ADPKD and why individuals with end-stage kidney disease receive dialysis or kidney transplants.

polycystic kidney disease is a condition characterized by the development of multiple cysts within the renal tubules of the kidney and is the most common hereditary renal disease the normal functional unit of the kidney is the nephron made up of a glomerulus and Bowman's capsule proximal convoluted tubule Loop of henle distal convoluted tubule and the collecting duct blood filters through the glomerulus forming filtrate which then passes through the tubules undergoing see and reabsorption eventually forming urine and passing into the ureter and bladder overall there are around 1 million of these nephrons in each kid in polycystic kidney disease these tubules develop into cysts which become filled with fluid and can range in size from being microscopic to several centimeters in size the process can start even in utero meaning in the womb as the tubules become cysts they cannot carry the normal filtering function therefore the number of functioning nephrons begins to decrease this is worsened by the fact that as the cysts grow they compress nearby nephrons also making them dysfunctional initially this may not be seen to have any clinical impact as the remaining number of nephrons may increase there to maintain the glomerular filtration rate but as the disease progresses and more nephrons are affected the remaining ones cannot make up the difference overall renal function then deteriorates eventually reaching end-stage renal disease by definition the presence of structural injury makes polycystic kidney disease a form of chronic kidney disease with end-stage renal disease being defined as needing renal replacement therapy like dialysis or transplant or a GF far below 15 ml per minute there are two main types of polycystic kidney disease both caused by genetics the first is autosomal dominant with two main types one coming from mutations in P KD 1 on a chromosome 16 which codes for the protein polycystic in this is affected in around 85% of cases and is involved in cell to cell or cell Matrix interaction cell cycle regulation and calcium transport the second coming from mutations in PKD to on chromosome for coding for Polly system to which codes for Learn more about your ad choices. Visit megaphone.fm/adchoices

polycystic kidney disease is a condition characterized by the development of multiple cysts within the renal tubules of the kidney and is the most common hereditary renal disease the normal functional unit of the kidney is the nephron made up of a glomerulus and Bowman's capsule proximal convoluted tubule Loop of henle distal convoluted tubule and the collecting duct blood filters through the glomerulus forming filtrate which then passes through the tubules undergoing see and reabsorption eventually forming urine and passing into the ureter and bladder overall there are around 1 million of these nephrons in each kid in polycystic kidney disease these tubules develop into cysts which become filled with fluid and can range in size from being microscopic to several centimeters in size the process can start even in utero meaning in the womb as the tubules become cysts they cannot carry the normal filtering function therefore the number of functioning nephrons begins to decrease this is worsened by the fact that as the cysts grow they compress nearby nephrons also making them dysfunctional initially this may not be seen to have any clinical impact as the remaining number of nephrons may increase there to maintain the glomerular filtration rate but as the disease progresses and more nephrons are affected the remaining ones cannot make up the difference overall renal function then deteriorates eventually reaching end-stage renal disease by definition the presence of structural injury makes polycystic kidney disease a form of chronic kidney disease with end-stage renal disease being defined as needing renal replacement therapy like dialysis or transplant or a GF far below 15 ml per minute there are two main types of polycystic kidney disease both caused by genetics the first is autosomal dominant with two main types one coming from mutations in P KD 1 on a chromosome 16 which codes for the protein polycystic in this is affected in around 85% of cases and is involved in cell to cell or cell Matrix interaction cell cycle regulation and calcium transport the second coming from mutations in PKD to on chromosome for coding for Polly system to which codes for Learn more about your ad choices. Visit megaphone.fm/adchoices

polycystic kidney disease is a condition characterized by the development of multiple cysts within the renal tubules of the kidney and is the most common hereditary renal disease the normal functional unit of the kidney is the nephron made up of a glomerulus and Bowman's capsule proximal convoluted tubule Loop of henle distal convoluted tubule and the collecting duct blood filters through the glomerulus forming filtrate which then passes through the tubules undergoing see and reabsorption eventually forming urine and passing into the ureter and bladder overall there are around 1 million of these nephrons in each kid in polycystic kidney disease these tubules develop into cysts which become filled with fluid and can range in size from being microscopic to several centimeters in size the process can start even in utero meaning in the womb as the tubules become cysts they cannot carry the normal filtering function therefore the number of functioning nephrons begins to decrease this is worsened by the fact that as the cysts grow they compress nearby nephrons also making them dysfunctional initially this may not be seen to have any clinical impact as the remaining number of nephrons may increase there to maintain the glomerular filtration rate but as the disease progresses and more nephrons are affected the remaining ones cannot make up the difference overall renal function then deteriorates eventually reaching end-stage renal disease by definition the presence of structural injury makes polycystic kidney disease a form of chronic kidney disease with end-stage renal disease being defined as needing renal replacement therapy like dialysis or transplant or a GF far below 15 ml per minute there are two main types of polycystic kidney disease both caused by genetics the first is autosomal dominant with two main types one coming from mutations in P KD 1 on a chromosome 16 which codes for the protein polycystic in this is affected in around 85% of cases and is involved in cell to cell or cell Matrix interaction cell cycle regulation and calcium transport the second coming from mutations in PKD to on chromosome for coding for Polly system to which codes for Learn more about your ad choices. Visit megaphone.fm/adchoices

Join Diana, The PKD Dietitian, as she provides an in-depth look at the steps you need to consider and take when starting ketogenic therapy safely for PKD. This is the second episode of a 3-part series on Keto and Polycystic Kidney Disease. RESOURCES Grab your *FREE* Starter Grocery Guide to get an idea of all the food you can, and should, include! Check out The pkDO Foundation Listen to Episode 19: Ketogenic Diets: Are They Good for PKD? WORK WITH DIANA Kidney Jumpstart PKD Kidney Health Pro *this is the 90-day program You can contact Diana at diana@thepkddietitian.com All information provided on this podcast is meant for educational purposes, not in place of medical advice. Always consult your doctor before starting any treatment.

Dive into all things Ketogenic Therapy and Polycystic Kidney Disease with The PKD Dietitian. This is the first episode of a 3-part series! IN THIS EPISODE: You will learn about the history of ketogenic diet therapy in medicine Finally know, and understand what ketones, ketosis, and keto are Get an answer to the million-dollar question of why were are even talking about keto & PKD Expert answers to some of your top questions, like "Can I do keto for PKD?" and "Should I do keto" Make sure to hit that SUBSCRIBE button and never miss an episode! REFERENCES: History of Keto https://onlinelibrary.wiley.com/doi/10.1111/j.1528-1167.2008.01821.x Weimb's Research Ketosis Slows the Progression of PKD https://www.nature.com/articles/s41581-019-0226-4 Ketosis Ameliorates Renal Cyst Growth in Polycystic Kidney Disease https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6904245/ Ren.Nu Paper Ren.Nu, a Dietary Program for Individuals with Autosomal-Dominant Polycystic Kidney Disease Implementing a Sustainable, Plant-Focused, Kidney-Safe, Ketogenic Approach with Avoidance of Renal Stressors https://www.mdpi.com/2673-8236/2/2/20 Charlie Foundation https://charliefoundation.org/about-us/ Keto & PKD: Two Myths and one truth https://www.thepkddietitian.com/blog/keto-and-PKD What is sugar and does it affect PKD? https://www.thepkddietitian.com/blog/sugarandPKD

The Chronic Kidney Disease Advocate Our Featured guest is Uncle Jim Myers! Jim is an extraordinary advocate for chronic kidney disease! Jim is an kidney transplant recipient after suffering with Polycystic Kidney Disease and needing dialysis following kidney failure. Jim has been awarded for his contributions to the kidney community and for advocating for many with CKD! Jim is not only a mentor but a friend to many and he also has an amazing kidney story! Jim hosts his own podcast called “Kidney Stories 2” @ https://youtube.com/@UncleJim1954 Find out more about Jim Myers here: https://aakp.org/about/board-of-directors/ We Share Stories of Hope @ https://www.hopewithjonathan.com/ Help Support HWJ by Subscribing (it's Free) @ https://youtube.com/@HopewithJonathan #chronickidneydisease #polycystickidneydisease #kidneyadvocate --- Support this podcast: https://podcasters.spotify.com/pod/show/hope-with-jonathan/support

In this episode, Diana is sharing her 4 Question Smart Guide to help you walk through if and how you should start a new supplement. Plus how to make a plan for your supplementation routine! Join as The PKD Dietitian gives you examples, walks you through how to use the questions for yourself, and provides you will some great tips on taking supplements with Polycystic Kidney Disease! 4 QUESTIONS TO ASK YOURSELF 1. Could it hurt? 2. How will it help? 3. How much? 4. How long? RESOURCES Connect with the PKD Dietitian's email, newsletter, and updates Book a Lab & Kidney Health Review

Dr. Allen Davidoff is the Founder and CEO of XORTX Therapeutics, testing the benefit of a therapeutic for autosomal dominant polycystic kidney disease, a rare progressive kidney disease with a clear genetic connection. Evidence suggests that lowering the activity of the xanthine oxidase enzyme in the kidney and lowering uric acid production via this enzyme is critical to developing a therapy that can benefit these patients. Allen explains, "In terms of real therapies that slow the progression of kidney disease, there are very few. And so, to take the example of the rare disease, autosomal dominant polycystic kidney disease, one drug is currently approved. It was approved in 2018, that's a drug called tolvaptan, but it's only really used in about 5% of individuals. So that leaves this rare disease population largely underserved despite the fact that there's an approved drug." "The drug that we're developing is oxypurinol. It is a xanthine oxidase inhibitor that has been studied extensively over a number of years. It is arguably the single best-tolerated xanthine oxidase inhibitor that's never been to market. We've started with that drug and reformulated it in a proprietary way to deliver it so that it will reach circulating blood concentrations that can inhibit the xanthine oxidase enzyme by 90% or more." @XORTXinc #ADPKD #PKD #ProgressiveKidneyDisease #KidneyHealth #RareDisease XORTX.com Download the transcript here

Dr. Allen Davidoff is the Founder and CEO of XORTX Therapeutics, testing the benefit of a therapeutic for autosomal dominant polycystic kidney disease, a rare progressive kidney disease with a clear genetic connection. Evidence suggests that lowering the activity of the xanthine oxidase enzyme in the kidney and lowering uric acid production via this enzyme is critical to developing a therapy that can benefit these patients. Allen explains, "In terms of real therapies that slow the progression of kidney disease, there are very few. And so, to take the example of the rare disease, autosomal dominant polycystic kidney disease, one drug is currently approved. It was approved in 2018, that's a drug called tolvaptan, but it's only really used in about 5% of individuals. So that leaves this rare disease population largely underserved despite the fact that there's an approved drug." "The drug that we're developing is oxypurinol. It is a xanthine oxidase inhibitor that has been studied extensively over a number of years. It is arguably the single best-tolerated xanthine oxidase inhibitor that's never been to market. We've started with that drug and reformulated it in a proprietary way to deliver it so that it will reach circulating blood concentrations that can inhibit the xanthine oxidase enzyme by 90% or more." @XORTXinc #ADPKD #PKD #ProgressiveKidneyDisease #KidneyHealth #RareDisease XORTX.com Listen to the podcast here

What do you get when you... Take a scientist who researches PKD + discoveries related to PKD growth pathways + realization the pathways depend on nutrients? You get a medical food - KetoCitra - the first, and only, medical food for Polycystic Kidney Disease. Today I'm joined by Dr. Thomas Weimbs, and we are going to dive into what this means for you! A glance at the episode: How the Weimbs Lab, which studies PKD, came about Medical food vs. Supplement A look at Ketocitra and what's inside How Ketocitra is specifically formulated for PKD Opportunities you have to impact PKD with diet and nutrition RESOURCES Santa Barbara Nutrients KetoCitra The Weimbs Laboratory >>> connect on Facebook Connect on Youtube REFERENCES Ketosis Ameliorates Renal Cyst Growth in Polycystic Kidney Disease Crystal deposition triggers tubule dilation that accelerates cystogenesis in polycystic kidney disease Have a question for the PKD Dietitian or a topic you would like covered? >>> Contact The PKD Dietitian

Been told to avoid NSAIDs? That's good advice for Polycystic Kidney Disease. In this episode, I am going to get into what NSAIDs are, why they aren't kidney-friendly, and specifically why they aren't PKD-friendly. A big thanks to the pkDO foundation! Connect with pkDO at www.pkDO.org Learn more about PGT-M, IVF, PKD, and pkDO >>> www.pkDO.org RESOURCES Lab and Kidney Health Review https://www.thepkddietitian.com/labandkidneyhealth REFERENCES National Kidney Foundation https://bpspubs.onlinelibrary.wiley.com/doi/full/10.1002/prp2.817 https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4144785/ https://cjasn.asnjournals.org/content/10/3/435.short https://www.sciencedirect.com/science/article/pii/S0085253815480433

 Resources We Mentioned in the Show  UC Davis Laboratory:   https://vgl.ucdavis.edu/test/pkd1-cat VCA: Polycystic Kidney Disease in Dogs: https://vcahospitals.com/know-your-pet/polycystic-kidney-disease-in-dogs#:~:text=What%20is%20polycystic%20kidney%20disease,in%20both%20size%20and%20number. VCA: Polycystic Kidney Disease in Cats: https://vcahospitals.com/know-your-pet/kidney-disease-polycystic-kidney-disease-in-cats Cornell: Polycystic Kidney Disease in Cats: https://www.vet.cornell.edu/departments-centers-and-institutes/cornell-feline-health-center/health-information/feline-health-topics/polycystic-kidney-disease    Thanks so much for tuning in. Join us again next week for another episode!  Want to earn some RACE approved CE credits for listening to the podcast? You can earn between 0.5-1.0  hour of RACE approved CE credit for each podcast episode you listen to.    Join the Internal Medicine For Vet Techs Membership to earn and keep track of your continuing education hours as you get your learn on!   Join now! http://internalmedicineforvettechsmembership.com/   Get Access to the Membership Site for your RACE approved CE certificates Sign up at https://internalmedicineforvettechsmembership.com  Get Access to the Technician Treasure Trove  Sign up at https://imfpp.org/treasuretrove    Thanks for listening!  – Yvonne and Jordan     

3 easy swaps you can make in your day-to-day diet that have a positive impact on Polycystic Kidney Disease. Learn what to swap out and what to swap in with this Take Action Episode! Resources: PKD Dietitian Blog >>> https://www.thepkddietitian.com/blog/ Olive Oil Blog >>> https://www.thepkddietitian.com/blog/healthy-fat-PKD-olive-oil Blackberry & Coconut Flax Parfait >>> https://www.thepkddietitian.com/blog/Blackberry-parfait Have a question for the PKD Dietitian or a topic you would like covered? Send Diana a Request! References:USDA's Foodkeeper App USDA FoodData Central https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4375225/ https://pubmed.ncbi.nlm.nih.gov/26071633/ https://pubmed.ncbi.nlm.nih.gov/26378571/ https://pubmed.ncbi.nlm.nih.gov/26378571/

A look at the myths, research, and recommendations around coffee consumption and Polycystic Kidney Disease. Plus 3 Questions to ask yourself to help determine your best amount! Episode Q&A at a glance: Is coffee safe to drink with Polycystic Kidney Disease? Does caffeine cause cyst to grow faster? What is a recommended "safe" intake of coffee that won't hurt the kidneys? Stay in the know! Sign up for the PKD Dietitan's Newsletter References: The Effect of Caffeine on Renal Epithelial Cells from Patients with Autosomal Dominant Polycystic Kidney Disease (2002) Learn more about PKD and vasopressin Caffeine Accelerates Cystic Kidney Disease in a Pkd1-Deficient Mouse Model (2019) Caffeine intake by patients with autosomal dominant polycystic kidney disease (2012) Chronic caffeine consumption exacerbates hypertension in rats with polycystic kidney disease (1998) Relationship between caffeine intake and autosomal dominant polycystic kidney disease progression: a retrospective analysis using the CRISP cohort (2018) ​​Long-term effect of coffee consumption on autosomal dominant polycystic kidneys disease progression: results from the Suisse ADPKD, a Prospective Longitudinal Cohort Study (2018) Genetic determinants of blood pressure responses to caffeine drinking (2012) Cardiovascular effects of caffeinated beverages (2019)

You can reduce your kidney stone risk with Polycystic Kidney Disease, and it's all about nutrition. This episode is all about picking the step that will serve you best and taking action! Here's a glance at the episode: Top 6 things you can start right now specifically for ADPKD and stone prevention Including my 3 core pillars of PKD Nutrition If you have had a stone → grab my freebie roadmap on what to do next Resources: Blog: Kidney Stones Risk & PKD, Are They Connected? Freebie: PKD & Kidney Stones - A Roadmap To Dietary Prevention Tracking: Cronometer Have a question for the PKD Dietitian or a topic you would like covered? >>> Contact The PKD Dietitian

Kidney stones and Polycystic Kidney Disease are connected. But why and how? Tune in and find out! The questions I am going to answer for you: What are kidney stones? Why & How do they form? Why is PKD a risk factor for stones? Plus flushing out the 2 most common stones with PKD and what that tells us about diet! This is the first part of a 2-part series on Stones! Resources: Kidney Stone Risk & PKD, are they connected? Finding the PKD Dietitian Podcast valuable? Make sure to rate and review it wherever you listen to podcasts. This helps other PKD Warriors find it! Connect with The PKD Dietitian! INSTAGRAM: https://www.instagram.com/the.pkd.dietitian/ FACEBOOK: https://www.facebook.com/PKDdietitian WEBSITE: https://www.thepkddietitian.com/

Guest: Fouad T. Chebib, M.D. Host: Denise M. Dupras, M.D., Ph.D. What is polycystic kidney disease? Is it all genetic? As providers, what should we be considering and what patients should we think about screening with genetic testing? Join this episode of our Genes & Your Health edition from the Center for Individualized Medicine to learn more about the role of genes in polycystic kidney disease. This episode features Fouad T. Chebib, M.D., a Mayo Clinic nephrologist from Jacksonville, Florida, whose specialty is in polycystic kidney disease. Connect with the Mayo Clinic's School of Continuous Professional Development online at https://ce.mayo.edu/ or on Twitter @MayoMedEd.

What is sugar? Complex vs. Simple vs. Refined vs. Whole, and where do carbs fit in…“Sugar” can be confusing, to say the least. Tune in for a look at nutritional differences between sugars and their impact on PKD progression. In this episode you will learn: What “sugar” really is 3 main categories of carbohydrates Refined vs. natural sugar How sugar can impact Polycystic Kidney Disease (PKD) My top 3 tips to scale back refined sugar intake Related Resources: What is Sugar & Does it Affect Polycystic Kidney Disease? Time Restricted Feeding in Autosomal Dominant Polycystic Kidney Disease Ketogenic Dietary Interventions in ADPKD (Keto-ADPKD) Daily Caloric Restriction in ADPKD Connect with Diana, The PKD Dietitian! INSTAGRAM: https://www.instagram.com/the.pkd.dietitian/ FACEBOOK: https://www.facebook.com/PKDdietitian WEBSITE: https://www.thepkddietitian.com/ Have a question for the PKD Dietitian or a topic you would like covered? Contact The PKD Dietitian

What is PKD? #pkd #polycystickidneydisease #kidneydisease Kidney Conversations takes a look into Polycystic Kidney Disease, which is a chronic kidney disease that is also known as PKD. Polycystic Kidney Disease is genetic and is passed from parent to child. Polycystic Kidney Disease although genetic has also been known to skip a generation, leaving some wondering how they contracted the disease. Research has been on going and also increased in many countries in the world. With an increase in research Polycystic Kidney Disease has come a long way. New treatments have been and are currently being devolved to help battle Polycystic Kidney Disease. By watching this video you will be educated and learn the main topics and have a better understanding of PKD. You should also take the advice of your doctor, nurses and medical team when dealing with Polycystic Kidney Disease and any other issues regarding your health and wellbeing. Some of the key moments in this video are: 0:00 Introduction 0:54 Intro to polycystic kidney disease 1:18 Causes of polycystic kidney disease 2:01 How many people have polycystic kidney disease 2:36 Closing Remarks

Polycystic kidney disease is an inherited disorder where clusters of cysts develop within the kidneys, causing the kidneys to enlarge and lose function over time. The cysts, which are noncancerous sacs containing fluid, vary in size, and they can grow to be large. This disorder can occur in children and adults.The two main types of polycystic kidney disease, caused by different genetic flaws, are: Autosomal dominant polycystic kidney disease (ADPKD). Signs and symptoms of ADPKD often develop between the ages of 30 and 40. In the past, this type was called adult polycystic kidney disease, but children can develop the disorder.Only one parent needs to have the disease for it to pass to the children. If one parent has ADPKD, each child has a 50% chance of getting the disease. This form accounts for most of the cases of polycystic kidney disease.  Autosomal recessive polycystic kidney disease (ARPKD). This type is far less common than is ADPKD. The signs and symptoms often appear shortly after birth. Sometimes, symptoms don't appear until later in childhood or during adolescence.Both parents must have abnormal genes to pass on this form of the disease. If both parents carry a gene for this disorder, each child has a 25% chance of getting the disease. Polycystic kidney disease also can cause cysts to develop in the liver and elsewhere in the body. The disease can cause serious complications, including high blood pressure and kidney failure.The disease varies greatly in its severity, and some complications from polycystic kidney disease are preventable. Lifestyle changes and treatments might help reduce damage to the kidneys from complications, but long-term interventions, including dialysis or kidney transplant, are sometimes needed.On this special Ask the Mayo Mom edition of the Mayo Clinic Q&A podcast, host Dr. Angela Mattke is joined by Dr. Christian Hanna, a pediatric nephrologist with Mayo Clinic Children's Center to discuss PKD in children.

Jim needs to find a living kidney donor! In this Special Edition of West Orange on the Go, Austin Arthur of Winter Garden sits down with Jim and Alisa Cochrane in this special edition of West Orange on the Go. Jim is a 50-year-old veteran who enlisted straight after graduation from high school. He was deployed to the Persian Gulf on aircraft carrier USS America and participated in operations Desert Shield, Desert Storm and, Provide Comfort. Jim became one of the youngest enlisted aviation air wing warfare specialists. Jim has a genetic Disorder called Polycystic Kidney Disease that has attacked and permanently damaged his kidney function. Jim needs to find a living kidney donor! This West Orange Veteran needs a kidney, please spread the word! Visit https://dovetransplant.org/ to learn more or to place an application to become a donor! This episode is brought to you by Gymnastics USA and Horizon West Neighbors Helping Neighbors #veteran #livingdonor #livingkidneydonor #polycystickidneydisease #kidneydisease #dovedonor #altruisticdonor #shareyourspare #vetshelpingvets #westorange #wintergarden #WestOrangeOnTheGo

In most cases, when someone steps forward as a living donor, nobody actually asks them to donate a kidney.  Usually, the donor hears the story of someone needing a kidney and makes the decision to donate without being asked.  The Living Kidney Donors Network (LKDN) is a non-profit 501(c)3 organization established in 2007 by Harvey Mysel whose primary mission is to educate people in need of a kidney transplant about living kidney donation and help them communicate their need to family members and friends. LKDN also has a program that educates the public about the need for living kidney donors.Harvey is a 2-time kidney transplant recipient. After proactively managing an inherited kidney condition, PKD (Polycystic Kidney Disease) for over 20 years he had his first kidney transplant in February 2007. He was fortunate that his wife Amy was a compatible donor. Harvey contracted a virus, (BK Virus,) 2 years after his transplant and after years of battling this virus, in August 2012 he had his second kidney transplant. Harvey and his donor, Stephen Liegghio, who was introduced to Harvey by an advocate, were involved in a 3-way kidney paired exchange.Most kidney donors donate to someone they know and patients need to first have the knowledge about living kidney transplants and learn how to develop their kidney campaign so they could be effective in communicating their need. The FREE online program Having Your Donor Find YOU! does just that. In addition to the work he does at the LKDN, Harvey is a tri-athlete, competing in a triathlon 5 months after his first transplant. He is a 3-time Gold Medal winner and Silver medalist in the Transplant Games. Harvey is an accomplished bicycle rider, tennis player, platform tennis player, bowler, hiker and rock climber.Episode LinksDonor DiariesLiving Kidney Donors NetworkNorthwestern Kidney Champion ProgramNational Kidney Donor OrganizationJohns Hopkins COVID/kidney Recipient Study