Podcasts about Encephalopathy

Send us a textShifting outlooks after neonatal encephalopathy in the era of therapeutic hypothermia.Christoffel K, Mulkey SB.Pediatr Res. 2025 Jun 4. doi: 10.1038/s41390-025-04156-0. Online ahead of print.PMID: 40467976 Review.As always, feel free to send us questions, comments, or suggestions to our email: nicupodcast@gmail.com. You can also contact the show through Instagram or Twitter, @nicupodcast. Or contact Ben and Daphna directly via their Twitter profiles: @drnicu and @doctordaphnamd. The papers discussed in today's episode are listed and timestamped on the webpage linked below. Enjoy!

Dr. Greg Cooper talks with Dr. David A. Wolk about limbic predominant age-related TDP-43 encephalopathy (LATE) discussing its clinical features, diagnostic criteria, and the importance of recognizing this condition in the context of Alzheimer disease. Read the related article.  Disclosures can be found at Neurology.org.   

This episode covers hypoxic-ischaemic encephalopathy.Written notes can be found at https://zerotofinals.com/paediatrics/neonatology/hie/Questions can be found at https://members.zerotofinals.com/Books can be found at https://zerotofinals.com/books/The audio in the episode was expertly edited by Harry Watchman.

In this powerful episode honoring HIE Awareness Month, Neonatal Nurse Practitioner Jessica Fulton shares the raw and emotional story of her son Bo's birth — a delivery marked by unexpected complications that led to a diagnosis of Hypoxic-Ischemic Encephalopathy (HIE). Jessica opens up about what it was like to watch her own son become a critical patient in the very NICU where she had worked for years — as her personal and professional worlds suddenly collided. She speaks candidly about the shocking helplessness she felt as a parent, despite her medical background, and the lifelong challenges that continue to follow: a cerebral palsy diagnosis, fierce advocacy, intensive therapies, emotional isolation, and the realities of parenting a child with complex needs. This episode is a must-listen for NICU parents, healthcare providers, and anyone supporting a family navigating the lifelong impact of HIE. Dr. Brown's Medical: https://www.drbrownsmedical.com Our NICU Roadmap: A Comprehensive NICU Journal: https://empoweringnicuparents.com/nicujournal/ NICU Mama Hats: https://empoweringnicuparents.com/hats/ NICU Milestone Cards: https://empoweringnicuparents.com/nicuproducts/ Newborn Holiday Cards: https://empoweringnicuparents.com/shop/ Empowering NICU Parents Show Notes: https://empoweringnicuparents.com/shownotes/ Episode 68 Show Notes: https://empoweringnicuparents.com/episode68 Empowering NICU Parents Instagram: https://www.instagram.com/empoweringnicuparents/ Empowering NICU Parents FB Group: https://www.facebook.com/groups/empoweringnicuparents Pinterest Page: https://pin.it/36MJjmH

Conditions Covered • Wernicke's Encephalopathy • Hepatic Encephalopathy • Toxic & Metabolic Encephalopathy • Uremic Encephalopathy • Hypertensive Encephalopathy ⸻ Encephalopathy = Global brain dysfunction Encephalitis = Brain inflammation ⸻ Wernicke's Encephalopathy Acute, reversible encephalopathy caused by thiamine (B1) deficiency. Key Differentiator: Confusion + Ataxia + Ophthalmoplegia in a malnourished or alcoholic patient. Essentials: • […] The post 129b: Quick Encephalopathy Review for the PANCE appeared first on Physician Assistant Exam Review.

Send us a textWhole-Body Hypothermia for Neonatal Encephalopathy in Preterm Infants 33 to 35 Weeks' Gestation: A Randomized Clinical Trial.Faix RG, Laptook AR, Shankaran S, Eggleston B, Chowdhury D, Heyne RJ, Das A, Pedroza C, Tyson JE, Wusthoff C, Bonifacio SL, Sánchez PJ, Yoder BA, Laughon MM, Vasil DM, Van Meurs KP, Crawford MM, Higgins RD, Poindexter BB, Colaizy TT, Hamrick SEG, Chalak LF, Ohls RK, Hartley-McAndrew ME, Dysart K, D'Angio CT, Guillet R, Kicklighter SD, Carlo WA, Sokol GM, DeMauro SB, Hibbs AM, Cotten CM, Merhar SL, Bapat RV, Harmon HM, Sewell E, Winter S, Natarajan G, Mosquera R, Hintz SR, Maitre NL, Benninger KL, Peralta-Carcelen M, Hines AC, Duncan AF, Wilson-Costello DE, Trembath A, Malcolm WF, Walsh MC; Eunice Kennedy Shriver National Institute of Child Health and Human Development Neonatal Research Network.JAMA Pediatr. 2025 Feb 24:e246613. doi: 10.1001/jamapediatrics.2024.6613. Online ahead of print.PMID: 39992674As always, feel free to send us questions, comments, or suggestions to our email: nicupodcast@gmail.com. You can also contact the show through Instagram or Twitter, @nicupodcast. Or contact Ben and Daphna directly via their Twitter profiles: @drnicu and @doctordaphnamd. The papers discussed in today's episode are listed and timestamped on the webpage linked below. Enjoy!

A breakthrough in understanding chronic traumatic encephalopathy (CTE), a disease linked to repeated head blows, has been made at the University of Auckland. Neuroscientists at the University of Auckland's Centre for Brain Research examined brain tissue, primarily from former rugby players, with the research shedding new light on how specific cells respond to damage from repeated head knocks. Producer Amani spoke with Senior Author of the research Dr Helen Murray about how the findings open the way for a deeper understanding of the causes of CTE and about what the research process involved.

In this episode of Autism for Badass Moms, we sit down with Allysa Parker, a dedicated Certified Dental Assistant and fierce advocate for her 6-year-old son, Julian. Julian was diagnosed with Hypoxic Ischemic Encephalopathy (H.I.E.) just two days after birth and later received an Autism diagnosis at 4.5 years old. Navigating these diagnoses has been an emotional rollercoaster for Allysa, bringing moments of helplessness and anxiety. But through it all, she has remained committed to ensuring Julian experiences the world to the fullest. Now, Allysa is using her journey to empower other moms, sharing her insights on H.I.E. and Autism while building a community of support and encouragement. Tune in to hear Allysa's incredible journey. Follow her journey on Instagram: https://www.instagram.com/lovely.loveable.leo/ Don't forget to subscribe and stay tuned for more empowering stories like Allysa's. Follow Us: Instagram: https://www.instagram.com/theabmpodcast/ Facebook: https://www.facebook.com/profile.php?id=100095054651586

In this episode, we review the high-yield topic ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Hepatic Encephalopathy ⁠⁠⁠⁠⁠⁠⁠⁠from the Gastrointestinal section at ⁠⁠⁠⁠⁠⁠Medbullets.com⁠⁠⁠⁠⁠⁠ Follow ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Medbullets⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets Linkedin: https://www.linkedin.com/company/medbullets

In this Pulmonary & Critical Care episode, Dr. Meghan Nothem presents an unusual case of acute encephalopathy and unresponsiveness to Dr. Paul A. Bergl. Stay tuned to found out the final diagnosis! Session facilitator: Dr. Hui Ting Ruan Case discussant:  Dr. Paul A. Bergl is an intensivist at Gundersen Lutheran Medical Center in La Crosse, WI and… Read More »Episode 374 – Rafael Medina Subspecialty Episode – Acute encephalopathy and unresponsiveness

While there are many kinds of dementia, like Alzheimer's disease and Lewy body dementia, there's one that researchers have only recently identified. LATE, or Limbic-predominant Age-related TDP-43 Encephalopathy, is a newly-characterized type of dementia associated with abnormal clumps of a protein called TDP-43. So, what exactly do we know about LATE? Dr. David Wolk joins the podcast to share what key features of LATE are, how it compares to Alzheimer's disease and impacts treatment, and what next steps are needed to better understand this neurodegenerative disease. Guest: David Wolk, MD, director, Penn Alzheimer's Disease Research Center, co-director, Penn Memory Center, co-director, Penn Institute on Aging, chief, Division of Cognitive Neurology, professor of neurology, University of Pennsylvania Perelman School of Medicine Show Notes Learn more about LATE on the National Institute on Aging's website and on Penn Memory Center's website. Read Dr. Wolk's article, "Clinical criteria for limbic-predominant age-related TDP-43 encephalopathy," on the journal Alzheimer's & Dementia's website. Learn more about Dr. Wolk in his profile on the Penn Memory Center website. Connect with us Find transcripts and more at our website. Email Dementia Matters: dementiamatters@medicine.wisc.edu Follow us on Facebook and Twitter. Subscribe to the Wisconsin Alzheimer's Disease Research Center's e-newsletter. Enjoy Dementia Matters? Consider making a gift to the Dementia Matters fund through the UW Initiative to End Alzheimer's. All donations go toward outreach and production.

Host: Charles Turck, PharmD, BCPS, BCCCP Guest: Jasmohan Bajaj, MD Around 50 percent of people with cirrhosis have hepatic encephalopathy, which means some kind of brain dysfunction. As these patients age, it gets more and more difficult to differentiate between hepatic encephalopathy and dementia, which is why two recent studies sought to uncover how many patients with dementia have undiagnosed cirrhosis. According to the findings, the rate of undiagnosed cirrhosis and hepatic encephalopathy could be as high as 10‒13 percent. Joining Dr. Charles Turck to share further details about the studies, findings, and implications is Dr. Jasmohan Bajaj, Professor of Medicine in the Division of Gastroenterology, Hepatology, and Nutrition at Virginia Commonwealth University.

Today's guest is Kelly Sutton, MHL, BSN, RN, CCDS, CCS, CDI education specialist for ACDIS/HCPro. Today's show is part of the “Talking CDI” series. In every episode of this series, Director of Programming Rebecca Hendren is joined by a special guest, such as a member of the ACDIS Advisory Board or one of our expert Boot Camp instructors to discuss a topic pertinent to today's CDI profession. Our intro and outro music for the ACDIS Podcast is “medianoche” by Dee Yan-Kay and our ad music is “Take Me Higher” by Jahzzar, both obtained from the Free Music Archive. Have questions about today's show or ideas for a future episode? Contact the ACDIS team at info@acdis.org. Want to submit a question for a future "listener questions" episode? Fill out this brief form!  CEU info: Each ACDIS Podcast episode now offers 0.5 ACDIS CEUs which can be used toward recertifying your CCDS or CCDS-O credential for those who listen to the show in the first two days from the time of publication. To receive your 0.5 CEUs, go to the show page on acdis.org, by clicking on the “ACDIS Podcast” link located under the “Resources” tab. To take the evaluation, click the most recent episode from the list on the podcast homepage, view the podcast recording at the bottom of that show page, and click the live link at the very end after the music has ended. Your certificate will be automatically emailed to you upon submitting the brief evaluation. (Note: If you are listening via a podcast app, click this link to go directly to the show page on acdis.org: https://acdis.org/acdis-podcast/listener-questions-aki-encephalopathy-case-management-collaboration-and-more) Note: To ensure your certificate reaches you and does not get trapped in your organization's spam filters, please use a personal email address when completing the CEU evaluation form. The cut-off for today's episode CEU is Friday, July 19, at 11:00 p.m. eastern. After that point, the CEU period will close, and you will not be eligible for the 0.5 CEUs for this week's episode. Today's sponsor: Today's show is brought to you by ACDIS Encore: Clinical & Coding Online, August 13-15, featuring sessions recorded live during the 2024 ACDIS national conference! Click here to learn more: https://bit.ly/3yv2P5X. ACDIS update: Respond to the 2024 CDI Week Industry Survey by July 19! (https://www.surveymonkey.com/r/2024-CDI-Week-Industry-Survey) Apply to speak at the 2025 ACDIS conference, Physician Advisor Forum, or ACDIS Symposium: Outpatient CDI by July 26! (https://acdis.org/25speaker)  

When the diseased liver is unable to filter out neurotoxins like ammonia, we can see a spectrum of the neuropsychiatric symptoms of hepatic encephalopathy. For the final installment in our liver failure series, host Sarah Lorenzini highlights a complicated case of hepatic encephalopathy where critical thinking and interdisciplinary teamwork were key to preventing further complications.She examines the common signs and symptoms of hepatic encephalopathy, factors that trigger or exacerbate the condition, and challenges nurses face in the diagnostic process. This episode also dives into treatment strategies and how to differentiate between hepatic encephalopathy and alcohol withdrawal.Tune in to learn how cirrhosis can lead to hepatic encephalopathy, the effect of neurotoxins in the brain, and how you, as a nurse, can manage patient care!Topics discussed in this episode:Assessment of Sarah's rapid response consult patientThe team's interventions and patient outcomePathophysiology of hepatic encephalopathySigns and symptoms of hepatic encephalopathyDiagnostic challenges and aggravating factorsStrategies to manage hepatic encephalopathyHepatic encephalopathy versus alcohol withdrawalMentioned in this episode:AND If you are planning to sit for your CCRN and would like to take the Critical Care Academy CCRN prep course you can visit https://www.ccrnacademy.com and use coupon code RAPID10 to get 10% off the cost of the course!Rapid Response and Rescue Intro CourseCONNECT

In this episode, listen to our editorial apprentice, Dr. Eric Peeples describe the scope and importance of our collection on neonatal encephalopathy and hypoxic-ischemic encephalopathy.Visit the collection here: Neonatal Encephalopathy and Hypoxic Ischemic Encephalopathy (nature.com) Hosted on Acast. See acast.com/privacy for more information.

Today's guest is Sharme Brodie, RN, CCDS, CCDS-O, CDI education specialist for HCPro/ACDIS. Today's show is hosted by ACDIS Associate Editorial Director Linnea Archibald, Director of Programming Rebecca Hendren, Editor and Product Coordinator Karla Kozak, and Associate Editor Jess Fluegel. The show provides a glimpse behind the scenes of all things ACDIS in a more casual, conversational format than our other podcast series.  Click here to listen to the episode on AKI that was referenced on the show: https://bit.ly/3Q8lWZI Our intro and outro music for the ACDIS Podcast is “medianoche” by Dee Yan-Kay and our ad music is “Take Me Higher” by Jahzzar, both obtained from the Free Music Archive. Have questions about today's show or ideas for a future episode? Contact the ACDIS team at info@acdis.org. Want to submit a question for a future "listener questions" episode? Fill out this brief form!  CEU info: Each ACDIS Podcast episode now offers 0.5 ACDIS CEUs which can be used toward recertifying your CCDS or CCDS-O credential for those who listen to the show in the first two days from the time of publication. To receive your 0.5 CEUs, go to the show page on acdis.org, by clicking on the “ACDIS Podcast” link located under the “Resources” tab. To take the evaluation, click the most recent episode from the list on the podcast homepage, view the podcast recording at the bottom of that show page, and click the live link at the very end after the music has ended. Your certificate will be automatically emailed to you upon submitting the brief evaluation. (Note: If you are listening via a podcast app, click this link to go directly to the show page on acdis.org: https://acdis.org/acdis-podcast/listener-questions-aki-mi-encephalopathy-provider-engagement-and-more) Note: To ensure your certificate reaches you and does not get trapped in your organization's spam filters, please use a personal email address when completing the CEU evaluation form. The cut-off for today's episode CEU is Friday, April 26, at 11:00 p.m. eastern. After that point, the CEU period will close, and you will not be eligible for the 0.5 CEUs for this week's episode. ACDIS update: Apply to speak at our 2025 ACDIS events, including the national conference in May 2025, by Friday, July 26! (https://acdis.org/25speaker)   Members are encouraged to vote in the 2024 ACDIS Advisory Board election by Wednesday, May 1! (https://acdis.org/2024-acdis-advisory-board) Send your denials management articles to Jess Fluegel at jess.fluegel@hcpro.com by June 1 for consideration for the summer edition of the CDI Journal!

Listen as Dr. London Smith (.com) and his producer Cameron discuss Bovine Spongiform Encephalopathy (Mad Cow Disease) as they share about their Dr. Uden's cures.  Sponsored by Caldera + Lab (use code "jockdoc" to get 20% off!). Not so boring! https://calderalab.com/pages/podcast-special-offer?show=Jock+Doc&utm_medium=podcast&utm_source=JocDoc https://www.patreon.com/join/jockdocpodcast Hosts: London Smith, Cameron Clark. Produced by: Dylan Walker Created by: London Smith

On this episode, we continue our examination of cardiac surgery with its unique challenges and risks for patients, with a  focus on accurate and thorough documentation of these procedures.   Moderator: Tomas Villanueva, DO, MBA, FACPE, SFHM Senior Principal Clinical Operations and Quality Vizient Guest: Rachel Mack, MSN, RN, CCDS, CDIP,  CCS, CRC Consulting Director Clinical Documentation Improvement Vizient   Show Notes: [00:31] Cardiac surgery patients: post-op and readmissions [01:24] “Volume overload,” i.e., heart failure [01:47] Five-day readmission stays! [03:34] Story – cardiac patient on the move [04:49] Common cardiac surgeries [08:22] Vasopressors do not always indicate shock [09:24] Encephalopathy and dementia   Resources: To contact Modern Practice: modernpracticepodcast@vizientinc.com Rachel's email: rachel.mack@vizientinc.com   Subscribe Today! Apple Podcasts Amazon Podcasts Android Google Podcasts Spotify RSS Feed

Hypoxic Ischaemic Encephalopathy (HIE), a subset of neonatal encephalopathy, is the most common neurological condition in term born infants. It is known that a range of acute and chronic placental pathologies are more common in infants with HIE. However little is known about how differences in utero-placental function might contribute to varied outcomes in these infants.In this episode of Pediapod, we speak to Early Career Investigator Dr. Jeffrey Russ from Duke University Medical Center, who retrospectively analyzed whether acute versus chronic placental pathology were differentially associated with outcomes in patients with presumed HIE.Read the full study here: The association of placental pathology and neurodevelopmental outcomes in patients with neonatal encephalopathy | Pediatric Research (nature.com) Hosted on Acast. See acast.com/privacy for more information.

Event Objectives:Identify metabolic causes of encephalopathy.List the tests that should be performed when considering a metabolic cause of encephalopathy.Describe the obstacles that metabolic patients face in their diagnostic odyssey.Claim CME credit here!

Dr. Alexander Menze talks with Dr. Brooke Conway Kleven about the association between a clinical diagnosis of traumatic encephalopathy syndrome and subsequent temporal decline in cognitive or MRI volumetric measures. Read the related article in Neurology.  Visit NPUb.org/Podcast for associated article links. This episode was sponsored by the ExTINGUISH Trial for NMDAR Encephalitis: Call 844-4BRAIN5 to refer patients.

This case covers the pathophysiology, diagnosis, and management of hepatic encephalopathy with a focus on pharmacologic treatments. The corresponding video is on YouTube (https://youtu.be/bcLjGR3-gY4).   **Thank you to our sponsor Doc2Doc Lending, the Personal Lending platform designed for Doctors, by Doctors. Check out https://doc2doclending.com/davinci to learn more today.   DaVinci Academy YouTube Channel: https://www.youtube.com/@DaVinciAcademyMed Website: https://www.dviacademy.com/ Instagram: https://www.instagram.com/davinci_academy1/ DaVinci Academy Merch - Coffee mugs, T-shirts, hoodies and more: https://my-store-d90f46.creator-spring.com

Question-based review of Acute liver failure, staging of hepatic encephalopathy (per clinical & EEG criteria), management of ALF with understanding of the severity criteria, Stage-based management of hepatic encephalopathy, Indications & Eligibility requirement for Liver transplantation, and a brief discussion on acetaminophen toxicity at the end.

Dr. Halley Alexander discusses the paper, "Traumatic Encephalopathy Syndrome: Can It Stand the Test of Time?" Show references: https://n.neurology.org/content/early/2023/06/28/WNL.0000000000207711 This episode was sponsored by the ExTINGUISH Trial for NMDAR Encephalitis: Call 844-4BRAIN5 to refer patients.

In this episode we will be covering Facebook Live Questions 7/ 3-7/9 free Facebook Group Registered Dietitian Exam Study Group with Dana RD! Don't forget to check out my recorded courses here.Looking for additional tutoring service? Visit my website! Interested in monthly group tutoring? Click here to learn more and apply.

Chief Medical Officer at Northwestern Medicine Dr. Kevin Most joins the Steve Cochran Show to discuss the gradual shift towards non-contact football summer camps, the new at-home lymes disease test kit, and he eases your COVID-19 vaccine confusion.  Dr. Kevin Most's notes from the Steve Cochran Show: CTE - Chronic traumatic Encephalopathy- new study- not the number of concussions, more important is the cumulative impact to the brain. New study released last Tuesday showed that the chances of CTE are not only related to the number of head impacts but also to the cumulative impact of all the hits. This was a study that reviewed 34 previous studies that looked at the number of hits and the impact of those hits measured by helmet sensors This looked back over 20 years and studied the 631 players who had donated their brain to these studies that were overseen by Boston University This study looked at what aspects of head hits most commonly contributed most to CTE progression They reviewed number of hits to the head, number of years played, and force of the hits Study found that the best predictor was the cumulative force of the head hits over the course of their career, not the number of concussions (which are very under reported or identified) The study reviewed 631 brains, of those 451 or 71%, had  CTE The players who were estimated to have absorbed the greatest cumulative force had the most advanced CTE This reinforced that the concept of the impact of subconcussive  hits, rather than number of concussions was the driving force behind the diagnosis and cognitive decline. The data collected was from college players as the NFL has not released its helmet sensor data The study looks at the number of hits and the force of the hits and them tracks the cumulative data Different players get hit in different areas of the brain, lineman take hits to the front of their heads, where quarterbacks take hits to the back of their head most often. This is leading to research on different helmets for different positions Future studies will look at the location of the hits and the impact More data will come out as helmet sensors become universally used. Many high schools going to low contact practice and well controlled contact practice. Helmet technology has certainly advanced to safer helmets, however players are bigger and faster and technology cannot keep up with protection from the force generated. Helmets have sensors for the force of the hit and some actually have EEG monitors, both are used to be more objective when a player needs to be removed form a game. Covid Vaccines- Confused? You are not alone… Think back over the past 3 years- one shot or 2, age 16 or 18? Boosters for which strain? Who gets boosters, new booster this fall The Covid virus is first noted in a patient in  December of 2019 In January of 2020 the first case is found in Seattle, Washington, a few days later the first case was noted in Illinois The Covid Virus was genetically sequenced shortly after it was identified, this allowed for vaccine research and trials to begin The original vaccine was produced in record time using newer technology, and approved for individuals over the age of 18. Confusion/concern- made too fast Original Covid Vaccine was produced using the original strain of Covid ( SARS-VoV-2)- released December 11, 2020, Pfizer and Moderna This was a 2 shot series, released under an emergency authorization A month later J and J Covid vaccine gets released as a single shot for those over the age of 16, some skeptics about this, but shortages promoted it Confusion- why only one shot Shortly after that Pfizer vaccine is approved for children aged 12 and over, then follows up with an approval for 6 months to age 12. Confusion/ concern- enough research done on kids with new vaccine? Moderna vaccine gets approved for 6 months to age 11 Fast forward to August of 2022, Moderna and Pfizer get approval for new booster based on BA4 and BA5 strains, the predominant strains at that time. Confusion/Concern- will we need boosters for all variants Booster is approved as a single shot for those 18 and older to be given at least 2 months following the completion of the primary series. UK and Canada approve 2nd booster for those over age 65, XXXXX Date, US is silent. Confusion/concern- why are they doing a second booster and we are not US approves 2nd dose of Covid booster April, 2023, for those over 65 or immunocompromised. Booster is still BA4 BA5 based US no longer gives original Covid vaccine to those who have not been vaccinated, now move to “Booster” vaccine as the standard News out now, this fall there will be a new Covid Vaccine using the predominant strain at this time XBB This will be recommended for all this fall along with your annual flu vaccine. Confusion/Concern- will insurance cover the cost as it is not government stock Will we need another booster in the spring?  Confusion/Concern- we seem to need a booster 6-8 months after our last shot, will this happen again? Home test for Lyme disease One great thing that came out of covid was the advancement of testing in the home for the disease. We saw earlier diagnosis made with less hassle and cost Now we are seeing how that is being advanced Home test for Lyme Disease is in the research phase. Hopefully faster identification will lead to earlier treatment , thus preventing any long term consequences from the illness Currently we use blood tests looking for the bodies response to the infection, we look for antibodies to the bacteria. Unfortunately the body may take a couple weeks to make enough antibodies  to be at a level that we can identify them Now we have home tests where a patient can prick their finger and send a drop of blood and sent that to a lab, this still may not be positive for weeks and has a turnaround time of 3-5 days This delay in antibody detection allows the bacteria to spread throughout the body Test #1-Work is now being done with a simple urine test that will identify small portions of the bacteria within hours after the infection begins.  Virginia Tech Test #2- is even faster. It is a sticker that is applied to the skin immediately after the tick is removed, and if the tick was infected with the bacteria for Lyme disease, the sticker will change colors Both of these are a couple years away, however it is showing that we will be doing more in home testing and hopefully quicker diagnosis at a lower cost See omnystudio.com/listener for privacy information.

As always, feel free to send us questions, comments, or suggestions to our email: nicupodcast@gmail.com. You can also contact the show through Instagram or Twitter, @nicupodcast. Or contact Ben and Daphna directly via their Twitter profiles: @drnicu and @doctordaphnamd. The papers discussed in today's episode are listed and timestamped on the webpage linked below.Enjoy!_____________________________________________________________________________________Show notes, articles, and CME form can be found on our website: http://www.the-incubator.org/128/

In this episode, we review the high-yield topic of Spongiform Encephalopathy / Creutzfeldt-Jakob Disease ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠from the Neurology section. Follow ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Medbullets⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets --- Send in a voice message: https://podcasters.spotify.com/pod/show/medbulletsstep1/message

Review the current status of hypothermia treatment for hypoxic-ischemic encephalopathy (HIE).Review other trials of therapeutic hypothermia.Review the results of combining hypothermia with other neuroprotective treatments.Discuss the considerations of therapeutic hypothermia for infants with mild HIE.

MedLink Neurology Podcast is delighted to feature selected episodes from BrainWaves, courtesy of James E Siegler MD, its originator and host. BrainWaves is an academic audio podcast whose mission is to educate medical providers through clinical cases and topical reviews in neurology, medicine, and the humanities, and episodes originally aired from 2016 to 2021. Originally released: July 18, 2017 The human body only has enough thiamine to last 2-3 weeks, so what happens when we run out? In this BrainWaves brief, we review the consequences of acute thiamine deficiency on the nervous system. BrainWaves podcasts and online content are intended for medical education only and should not be used to guide medical decision-making in routine clinical practice. Any cases discussed in this episode are fictional and do not contain any patient health-identifying information. The content in this episode was approved and vetted by Michael Rubenstein. REFERENCES Caine D, Halliday GM, Kril JJ, Harper CG. Operational criteria for the classification of chronic alcoholics: identification of Wernicke's encephalopathy. J Neurol Neurosurg Psychiatry 1997;62(1):51-60. PMID 9010400Galvin R, Bråthen G, Ivashynka A, et al. EFNS guidelines for diagnosis, therapy and prevention of Wernicke encephalopathy. Eur J Neurol 2010;17(12):1408-18. PMID 20642790Sechi G, Serra A. Wernicke's encephalopathy: new clinical settings and recent advances in diagnosis and management. Lancet Neurol 2007;6(5):442-55. PMID 17434099Zuccoli G, Pipitone N. Neuroimaging findings in acute Wernicke's encephalopathy: review of the literature. AJR Am J Roentgenol 2009;192(2):501-8. PMID 19155417We believe that the principles expressed or implied in the podcast remain valid, but certain details may be superseded by evolving knowledge since the episode's original release date. 

Show notes at pharmacyjoe.com/episode783. In this episode, I’ll discuss why the administration of glucose without thiamine might precipitate or worsen Wernicke’s encephalopathy. The post 783: Why Might the Administration of Glucose Without Thiamine Precipitate or Worsen Wernicke’s Encephalopathy? appeared first on Pharmacy Joe.

Show notes at pharmacyjoe.com/episode783. In this episode, I’ll discuss why the administration of glucose without thiamine might precipitate or worsen Wernicke’s encephalopathy. The post 783: Why Might the Administration of Glucose Without Thiamine Precipitate or Worsen Wernicke’s Encephalopathy? appeared first on Pharmacy Joe.

Show notes at pharmacyjoe.com/episode782. In this episode, I will discuss how I would treat Wernicke's Encephalopathy if I were completely out of IV thiamine. The post 782: Wernicke's Encephalopathy Treatment During an IV Thiamine Shortage appeared first on Pharmacy Joe.

Show notes at pharmacyjoe.com/episode782. In this episode, I will discuss how I would treat Wernicke s Encephalopathy if I were completely out of IV thiamine. The post 782: Wernicke s Encephalopathy Treatment During an IV Thiamine Shortage appeared first on Pharmacy Joe.

How to evaluate the patient with unexplained encephalopathy, and a practical approach to diagnosing autoimmune encephalitis with an emphasis on anti-NMDA receptor encephalitis—with Dr. Casey Albin (@CaseyAlbin), neurologist and neurointensivist, assistant professor of Neurology and Neurosurgery at Emory, and part of the NeuroEmcrit team. Claim your CME credit here! Find us on Patreon here! Buy … Continue reading "Episode 55: Undifferentiated encephalopathy and autoimmune encephalitis, with Casey Albin"

Approach to Acute Encephalopathy - with Dr. Jakel by Dr. Laura Caputo

Episode 118: Wernicke's Encephalopathy Dr. Malave explains the diagnosis and treatment of Wernicke's encephalopathy. Editing and comments by Hector Arreaza.  Written by Maria Fernanda Malave, edited by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice.Definition.As a reminder for everyone, vitamin B1 and thiamine are the same substance with different names. Wernicke's encephalopathy (WE) is a neurological syndrome secondary to severe, short-term B1 deficiency. In the past, but less frequently nowadays, it was more commonly associated with alcohol use disorder. However, today we know that any condition that decreases dietary intake and increases thiamine use, or its elimination, puts patients at risk of developing this encephalopathy. Causes:Chronic alcoholism is the most important cause of WE. Around 70-80% of the causes are associated with chronic alcohol consumption. Non-alcoholic WE may be caused by Decreased intake: Some types of WE may be caused by a psychiatric illness that decreases the dietary intake of B1, such as anorexia nervosa, schizophrenia, or dementia. Arreaza: Also, prolonged fasting or starvation.Lack of absorption of B1: Other causes might be related to malabsorptive syndromes, bariatric surgeries, or hyperemesis gravidarumIncreased use of B1: Any disease that increases the use of B1 and, therefore, low levels of thiamine, such as cancer, thyrotoxicosis, and systemic illnesses like infections. High-carb diets are associated with high thiamine use. Also, patients who receive IV glucose w/o supplements are at risk of developing Wernicke's encephalopathy.Increased elimination of B1: Other causes are related to increased elimination of B1, such as dialysis.Immunodeficiencies: Immunodeficiency syndromes and transplantation also cause WE.Why is thiamine important?Thiamine is one of the main cofactors in three key enzymes for energy metabolism: alpha-ketoglutarate dehydrogenase, pyruvate dehydrogenase, and transketolase. If we go back to biochemistry in med school, we can remember these enzymes play a significant role in the Krebs cycle and pentose phosphate pathways. Thiamine uses Mg+2 as a cofactor, so a magnesium deficiency can mimic WE.Pathophysiology.B1 deficiency causes lactic acid accumulation due to anaerobic glycolysis, leading to neuronal cytotoxic edema and vasogenic edema with petechial hemorrhages. MRI of the brain shows symmetrical hyperintensities, most commonly in the thalamus, mammillary bodies, cerebellum, and the periaqueductal area surrounding the third and fourth ventricles. The diagnosis of WE is made clinically, even though the MRI is a useful complementary tool to the clinical diagnosis. Diagnosis.WE presentation has always been described as the classic triad of ophthalmoplegia (or nystagmus), encephalopathy (confusion or memory impairment), and gait ataxia. However, this presentation is present only in less than 20% of patients, and most of the patients present with a neurologic syndrome that includes 2 out of the classic triad plus nonspecific symptoms such as hallucinations, hypothermia, hypotension, indifference or inattentiveness, seizures, behavioral disturbances, and bilateral lower extremity weakness. In 1997, Caine et al. suggested that a diagnosis of WE can be made if 2 out of 4 of these criteria were present in a patient with ophthalmoplegia/nystagmus + ataxia + memory impairment or confusion and clinical evidence of malnutrition or from laboratory data. Thiamine levels can be normal in patients with WE, so thiamine level is not a requirement for diagnosis.Almost 80% of WE cases are diagnosed on autopsy, which means this disease goes undiagnosed most of the time. The diagnosis is clinical, and MRI can assist in cases that are uncertain. Treatment.Thiamine supplementation is inexpensive, accessible, and easy to administer, so if we have a patient with a suspicious neurologic syndrome that could be WE, B1 must be given as soon as possible. Treatment should not be delayed while waiting for MRI results, The treatment consists of IV thiamine 500mg TID for 2-3 days, followed by 250 IM or IV for additional 5 days, in combination with other B vitamins. Because GI absorption of thiamine is impaired in alcoholics and malnourished patients, oral administration is contraindicated during initial treatment for WE. Thiamine 100 mg PO should be continued after the completion of parenteral treatment and after discharge from the hospital until patients are no longer considered at risk. Magnesium and other vitamins are replaced as well, along with other nutritional deficits if present.B1 blood levels or erythrocyte transketolase activity (which is a way to evaluate thiamine deficiency) are measured before and after thiamine pyrophosphate supplementation. A low transketolase and a more than 25 percent stimulation are positive for thiamine deficiency. This test is often not readily available, especially at the ED. High-quality liquid chromatography can also measure serum thiamine or TPP level in serum or whole blood.For practical purposes, and given high undiagnosed rates and mortality, IV thiamine should be given to all patients with malnutrition admitted to the hospital or seen in clinic. __________________________Conclusion: Now we conclude episode number 117 “Wernicke's Encephalopathy.” Dr. Malave explained that a low thiamine level is not required for the diagnosis of Wernicke's encephalopathy. Start supplementation if you have a high suspicion of thiamine deficiency, especially before giving IV glucose to a malnourished patient. We were reminded that GI absorption of thiamine is impaired in patients with chronic alcohol use, so make sure you give thiamine IV or IM. This week we thank Hector Arreaza and Maria Fernanda Malave. Audio edition by Adrianne Silva.Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! _____________________Links:He Jingqi, Li Jinguang, Li Zhijun, Ren Honghong, Chen Xiaogang, Tang Jinsong. A Case Report of Wernicke's Encephalopathy Associated with Schizophrenia. Frontiers in Psychiatry. Vol 12. Year 2021. DOI=10.3389/fpsyt.2021.657649. https://www.frontiersin.org/articles/10.3389/fpsyt.2021.657649/fullOta, Y., Capizzano, A.A., Moritani, T. et al. Comprehensive review of Wernicke encephalopathy: pathophysiology, clinical symptoms and imaging findings. Jpn J Radiol 38, 809–820 (2020). https://doi.org/10.1007/s11604-020-00989-3.Yuen T So, MD, PhD, Wernicke encephalopathy, last updated: Feb 11, 2020, UpToDate. Retrieved October 25, 2022.https://www.uptodate.com/contents/wernicke-encephalopathy.Patel S, Topiwala K, Hudson L. Wernicke's Encephalopathy. Cureus. 2018 Aug 22;10(8):e3187. doi: 10.7759/cureus.3187. PMID: 30364782; PMCID: PMC6199146. https://pubmed.ncbi.nlm.nih.gov/30364782/Royalty-free music used for this episode: Simon Pettersson – good vibes_ Fashionista, downloaded on October 1, 2022, from https://www.videvo.net 

Guest: Betsy Pilon - In this episode, Michelle is joined by Betsy, the Executive Director of “Hope for Acquired Hypoxic Ischemic Encephalopathy (HIE).” This hour is a deeply profound conversation as one mom shares her journey of advocacy and empowerment, inspiring and educating us along the way. Join in as the topics of causes and current treatment for HIE. are presented. Potential long-term impacts and barriers to continued treatment after NICU discharge will be discussed.

New white paper from ACDIS regarding Case Mix IndexLink to white paperHepatic Encephalopathy code discussion with Dr. KennedyNew codes in Final Rule 2023 (Dementia, SDOH, Hepatic Encephalopathy) Support the show

On this episode, we are joined by Jessa Mae Sabate and Nichole Arroyo (4th year MUSC PharmD candidates) to discuss a patient case looking into the management of advanced liver disease. We discuss hepatic encephalopathy and treatment options for hepatitis C. We also review the management of cholestatic pruritus, insomnia, and some other behavioral health issues in the patient's history.  Thanks for listening! We want to give a big thanks to our main sponsor Pyrls. Try out their drug information app today. Visit the website below for a free trial: www.pyrls.com/corconsultrx If you want to support the podcast, check out our Patreon account. Subscribers will have access to all previous and new pharmacotherapy lectures as well as downloadable PowerPoint slides for each lecture. You can find our account at the website below:  www.patreon.com/corconsultrx If you have any questions for Cole or me, reach out to us on any of the following: Text - 415-943-6116 Mike - mcorvino@corconsultrx.com Cole - cswanson@corconsultrx.com Instagram and other social media platforms - @corconsultrx This podcast reviews current evidence-based medicine and pharmacy treatment options. This podcast is intended to be used for educational purposes only and is intended for healthcare professionals and students. This podcast is not for patients and not intended as advice or treatment.

Do you know someone that is an athlete with a concussion? Tune if for a podcast with a special host Dr. Matthews, surgeon and vitamin D specialist. Take advantage of an exclusive podcast offer today by visiting http://www.invitehealth.com/podcast. For more information on the products or studies mentioned in this episode, as well as a complete transcript of the audio, click here

Guest: Raquel Garcia, SLPD, CCC-SLP, CNT, BCS-S - In this course, the presenter discusses hypoxic-ischemic encephalopathy (HIE) and how this devastating diagnosis can result in pediatric dysphagia. We learn about new resources and organizations, such as “Hope for HIE”, that are educating and advocating interprofessional practice partners and caregivers about the most current research to advance the quality of care for even our littlest patients.

Support the show

In this episode, we review the high-yield topic of Hepatic Encephalopathy from the Gastrointestinal section. Follow Medbullets on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbulletsIn this episode --- Send in a voice message: https://anchor.fm/medbulletsstep1/message

For our 29th Podcast episode and to close out HIE Awareness month, I sat down with Brigid Janousek. She bravely shares the story of her son William's traumatic delivery and his subsequent treatment with therapeutic hypothermia due to his diagnosis of Hypoxic Ischemic Encephalopathy or HIE. Infants with HIE have experienced a lack of oxygen to the brain either prior to birth, during the birth process, after birth, or during childhood. Infants with suspected HIE are treated in the NICU with therapeutic hypothermia or cooling that needs to be initiated within the first 6 hours of life. The cooling therapy reduces the rate of brain cell death and the risk of reperfusion injury which commonly occurs due to the release of toxins from the injured brain cells. The cooling process occurs for 72 hours post-delivery and has been shown to minimize the extent of death and disability in children. Brigid shares a more in depth look into William's time in the NICU, and how they first learned about his diagnosis. She explains why they felt like they were outcasts in the NICU due to William being a term infant. Brigid shares the struggles they endured once they were discharged home and how she unknowingly was suffering from postpartum depression, perinatal mood disorder, and PTSD. We discuss ways it was helpful for her to work through her trauma and why she is so passionate about bringing awareness to the misconception that all NICU babies are preterm as well as the importance of focusing on maternal mental health. We thank Brigid for sharing her family's very personal story. It is our goal to help bring awareness to HIE and to help break down some of the barriers and social disparities that exist with HIE infants and full-term NICU babies and strive for inclusive NICU messaging.Stay tuned, you will not want to miss their story…Our NICU Roadmap: https://empoweringnicuparents.com/nicujournal/Website: https://empoweringnicuparents.comEmpowering NICU Parents Show Notes: https://empoweringnicuparents.com/episode29/Empowering NICU Parents Instagram: https://www.instagram.com/empoweringnicuparents/Empowering NICU Parents FB Group: https://www.facebook.com/groups/empoweringnicuparentsPinterest Page: https://pin.it/36MJjmH

In this episode, we review the high-yield topic of Hepatic Encephalopathy from the Gastrointestinal section. Follow Medbullets on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets

Today we are joined by Dr. Brian Locke to talk about the newest big trial on IV fluids in critically ill patients and a meta-analysis that looks at all the similar trials. Is this question finally settled?We also look at PICC lines vs midlines, IV L-ornithine L-aspartate in acute treatment of hepatic encephalopathy, a new ward-based program for delirium prevention in older inpatients, post-discharge thromboprophylaxis for high risk COVID patients, and whether mindfulness training can help burnout in residents.Balanced Multielectrolyte Solution vs Normal Saline in Critically Ill Patients (PLUS)Balanced Crystalloid vs Normal Saline Meta-analysisPICC lines vs MidlinesApixaban vs Rivaroxaban for Recurrent VTE LOLA for Hepatic EncephalopathyEat-Walk-Engage in Older Inpatients (CHERISH)Rivaroxaban for Post-discharge Prophylaxis in High Risk COVID patients (MICHELLE)Mindfulness Training and BurnoutMusic from Uppbeat (free for Creators!):https://uppbeat.io/t/soundroll/dopeLicense code: NP8HLP5WKGKXFW2R