Podcasts about Albin

Label: Eigenvertrieb

Fluent Fiction - Swedish: Rekindling Family Ties Among Stockholm's Masterpieces Find the full episode transcript, vocabulary words, and more:fluentfiction.com/sv/episode/2025-10-25-22-34-02-sv Story Transcript:Sv: Det var en krispig höstdag i Stockholm.En: It was a crisp autumn day in Stockholm.Sv: Träden utanför Nationalmuseum skiftade i gyllene och röda färger, som om de själva var konstverk.En: The trees outside the Nationalmuseum were changing to golden and red hues, as if they themselves were artworks.Sv: Sofia och Linnea gick långsamt genom museets magnifika salar.En: Sofia and Linnea walked slowly through the museum's magnificent halls.Sv: De skulle på en efterlängtad konstutställning.En: They were going to a long-awaited art exhibition.Sv: Sofia älskade konst.En: Sofia loved art.Sv: Det gav henne en känsla av ro och inspiration.En: It gave her a sense of peace and inspiration.Sv: Men den här dagen grumlades hennes glädje av oro.En: But this day, her joy was clouded by worry.Sv: "Linnea, tänk om vi stöter på Albin idag?"En: "Linnea, what if we run into Albin today?"Sv: viskade Sofia.En: whispered Sofia.Sv: Hennes blick flackade i oroliga mönster, speglande de intrikata målningarna omkring dem.En: Her glance flickered in worried patterns, mirroring the intricate paintings around them.Sv: "Du har velat återförenas med honom länge nu, Sofia," svarade Linnea mjukt och la en hand på Sofias axel.En: "You've been wanting to reunite with him for a long time now, Sofia," Linnea replied softly, placing a hand on Sofia's shoulder.Sv: "Kanske är det ett tecken."En: "Maybe it's a sign."Sv: Sofia nickade, men en tyngd vilade kvar i hennes bröst.En: Sofia nodded, but a weight lingered in her chest.Sv: Hennes bror Albin, som hon inte hade träffat på flera år, hade plötsligt dykt upp i hennes tankar denna dag.En: Her brother Albin, whom she had not seen for several years, had suddenly appeared in her thoughts today.Sv: Han bodde utomlands och deras relation hade varit kylig.En: He lived abroad and their relationship had been chilly.Sv: Gamla konflikter låg fortfarande och pyrde under ytan.En: Old conflicts were still simmering beneath the surface.Sv: Som de långsamt vandrade genom utställningen, de njöt av varje konstverk.En: As they slowly wandered through the exhibition, they enjoyed each artwork.Sv: Mitt bland färger och former såg de en bekant gestalt.En: Among the colors and shapes, they saw a familiar figure.Sv: Där stod Albin.En: There stood Albin.Sv: Han såg äldre ut, men de där igenkännande dragen fanns kvar.En: He looked older, but those recognizable features remained.Sv: "Sofia", uppmanade Linnea tyst och knuffade henne försiktigt framåt.En: "Sofia," Linnea urged quietly and nudged her gently forward.Sv: Sofia kände en hemsk blandning av förväntan och rädsla.En: Sofia felt a dreadful mix of anticipation and fear.Sv: Skulle hon säga hej?En: Should she say hello?Sv: Vad skulle hon säga?En: What would she say?Sv: Om han bara ignorerade henne?En: What if he just ignored her?Sv: Men denna plats, dessa konstverk, gav henne mod.En: But this place, these artworks, gave her courage.Sv: Hon satte kurs mot Albin.En: She set a course toward Albin.Sv: "Hej, Albin," sa hon, rösten skakig men tydlig.En: "Hello, Albin," she said, her voice shaky but clear.Sv: Albin vände sig långsamt och såg på henne.En: Albin turned slowly and looked at her.Sv: Blicken mjuknade något, men han såg också förbluffad ut.En: His gaze softened somewhat, but he also appeared astonished.Sv: "Sofia," svarade han till sist.En: "Sofia," he finally replied.Sv: De stod där mitt bland mästerverken, två syskon som delade mer än bara gener.En: They stood there among the masterpieces, two siblings sharing more than just genes.Sv: Orden kom först långsamt.En: The words came slowly at first.Sv: Samtalet blev snabbt uppriktigt och känsloladdat.En: The conversation quickly became sincere and emotional.Sv: Sofia talade om sina känslor, om saknaden och ängsligheten.En: Sofia talked about her feelings, about the longing and anxiety.Sv: Albin berättade om sitt liv utomlands, om varför han hållit sig undan.En: Albin spoke about his life abroad, about why he had stayed away.Sv: Det var svårt.En: It was difficult.Sv: För gamla sår läkte inte omedelbart.En: For old wounds do not heal immediately.Sv: Men där, bland konsten, hittade de en sällsynt gemensam mark.En: But there, among the art, they found a rare common ground.Sv: Insikten om att det som en gång verkade omöjligt att reparera kanske kunde bli helt.En: The realization that what once seemed impossible to repair could perhaps become whole.Sv: När Sofia och Linnea senare den kvällen lämnade museet, vilade ett lugn över Sofia.En: When Sofia and Linnea left the museum later that evening, a calm rested over Sofia.Sv: Hon hade gått igenom en storm av känslor, men hittade ett frö av hopp.En: She had gone through a storm of emotions, but found a seed of hope.Sv: Hennes mod belönades och hon kände sig starkare.En: Her courage was rewarded and she felt stronger.Sv: Människor förändras.En: People change.Sv: Förbindelser kan återuppbyggas.En: Connections can be rebuilt.Sv: Höstens färgglada löv viskade om nya början, och mitt i allt detta, fann Sofia en ny början med sin bror.En: The colorful autumn leaves whispered of new beginnings, and in the midst of it all, Sofia found a new beginning with her brother. Vocabulary Words:crisp: krispigautumn: hösthues: färgermagnificent: magnifikaclouded: grumladesflickered: flackadeintricate: intrikatalingered: vilade kvarabroad: utomlandschilly: kyligsimmering: pyrdewandered: vandradefamiliar: bekanturged: uppmanadedreadful: hemskanticipation: förväntanastonished: förbluffadmasterpieces: mästerverkensincere: uppriktigtanxiety: ängslighetheal: läkterare: sällsyntrealization: insiktenimpossible: omöjligtrepair: repareracalm: lugnstorm: stormseed: fröcourage: modrewarded: belönades

Dans la sélection musicale de Brice Albin : Pepe Oleka, Nikanor, Fanicko, First King feat Fanicko, OPA, OPA feat T.Gang, Vano Baby, Bobo Wé, Axel Merryl et Credo. Pour visionner les clips, cliquez sur les titres des chansons :  Pepe Oleka - Sêbla koko Nikanor - Veux-tu m'épouser ? Fanicko - Origine First King feat Fanicko - Jago life OPA - Che Che Che OPA feat T.Gang le technicien - Pause Vano Baby - Fitè Bobo Wé - Mea culpa Axel Merryl feat Mia Guisse et Bass Thioung - Titulaire Credo - Dudedji Retrouvez la playlist officielle de RFI Musique.

Dans la sélection musicale de Brice Albin : Pepe Oleka, Nikanor, Fanicko, First King feat Fanicko, OPA, OPA feat T.Gang, Vano Baby, Bobo Wé, Axel Merryl et Credo. Pour visionner les clips, cliquez sur les titres des chansons :  Pepe Oleka - Sêbla koko Nikanor - Veux-tu m'épouser ? Fanicko - Origine First King feat Fanicko - Jago life OPA - Che Che Che OPA feat T.Gang le technicien - Pause Vano Baby - Fitè Bobo Wé - Mea culpa Axel Merryl feat Mia Guisse et Bass Thioung - Titulaire Credo - Dudedji Retrouvez la playlist officielle de RFI Musique.

Le film d'animation «La vie de château», réalisé par Clémence Madeleine-Perdrillat nous plonge dans les coulisses de ce monument. C'est l'histoire de Violette, 8 ans, qui doit aller vivre chez son oncle qui s'occupe de l'entretien du château. Avec Violette, on découvre un monde que les touristes ne voient jamais, celui d'une multitude de jardiniers, de fontainiers, d'agent d'entretien, de conservateurs qui s'affairent tous les jours pour préserver le patrimoine et l'histoire de ce château. Clémence Madeleine-Perdrillat, réalisatrice ; Gilles Bultez, chef du service des eaux et fontaines du château de Versailles et Pierre-Hippolyte Pénet, conservateur en chef du patrimoine au château de Versailles, étaient les invités de Nathalie Amar.   ► Chronique Le hit de la semaine Kayz Loum nous fait découvrir «Aje» du duo Trinix qui a fait appel à la star française d'origine camerounaise Tayc et à l'artiste nigérian dont tout le monde parle Khaid !    ► Playlist du jour - Albin de la Simone et Alice on the Roof - Pourquoi on pleure. - Thylacine - Versailles.

Le film d'animation «La vie de château», réalisé par Clémence Madeleine-Perdrillat nous plonge dans les coulisses de ce monument. C'est l'histoire de Violette, 8 ans, qui doit aller vivre chez son oncle qui s'occupe de l'entretien du château. Avec Violette, on découvre un monde que les touristes ne voient jamais, celui d'une multitude de jardiniers, de fontainiers, d'agent d'entretien, de conservateurs qui s'affairent tous les jours pour préserver le patrimoine et l'histoire de ce château. Clémence Madeleine-Perdrillat, réalisatrice ; Gilles Bultez, chef du service des eaux et fontaines du château de Versailles et Pierre-Hippolyte Pénet, conservateur en chef du patrimoine au château de Versailles, étaient les invités de Nathalie Amar.   ► Chronique Le hit de la semaine Kayz Loum nous fait découvrir «Aje» du duo Trinix qui a fait appel à la star française d'origine camerounaise Tayc et à l'artiste nigérian dont tout le monde parle Khaid !    ► Playlist du jour - Albin de la Simone et Alice on the Roof - Pourquoi on pleure. - Thylacine - Versailles.

Andakterna den här veckan tar avstamp i de fem världsreligionerna och har Helgon som tema. Idag hör du Albin Tanke som utgår från sin kristna tro. Lyssna på alla avsnitt i Sveriges Radio Play. Ur Andakten:”Att inte vara ett helgon precis” eller att ”inte vara Guds bästa barn”, kan vara sätt att beskriva när vi halkat snett och inte gjort rätt för oss. Men framför allt kanske det säger något om hur vår värld är beskaffad och det beskriver våra mänskliga villkor. Det uttrycker brist, att vi är indragna i en sprucken och ofullständig värld.För även den som inte är ett helgon precis, är ändå älskad av Gud till 100 %. Liksom vi alla är bristfälliga och underkastade synden, så är vi samtidigt genomälskade. Människan är Simul iustus et peccator som Martin Luther uttryckte det på latin och som betyder att människan på en och samma gång är både rättfärdig och syndare.Text:Gal 5:22Musik:Prague's Ballet av Marius Neset med Marius Neset, Leif Ove Andsnes och Louisa TuckProducent:Susanna Némethliv@sverigesradio.se

Veckans andakter hålls av Albin Tanke, präst och psykoterapeut i Malmö. Temat för veckan är Att omdanas. Lyssna på alla avsnitt i Sveriges Radio Play. Ur andakten:Jag sitter och tittar på mitt tummade exemplar av filosofen Martin Bubers bok ”Jag och du” och tänker på freden. Hur fred börjar inom var och en av oss – genom hur vi ser på oss själva och hur vi möter varandra. För att ett verkligt möte ska kunna äga rum behöver det finnas ett mellanrum mellan oss. Ett mellanrum som skapar liv bortom vår kontroll.Mellanrum ger möjlighet att se annorlunda, det öppnar för respekt. Där ingen på förhand har bestämt vem vi är, eller beslutat vad mötet ska vara bra för eller vad vi ska användas till. När vi backar tillbaka lite och verkligen kan se varandra, kan liv fyllas mellan oss. Då kan vi få syn på livet i blicken på ett nytt sätt. Vi får möjlighet att söka freden i den andres blick.Andakten sändes första gången 2024.Text:Filipperbrevet 4:7Musik:Pianosonat nr 16 av WA Mozart, Andante med Maria João PiresProducent:Susanna Némethliv@sverigesradio.se

Veckans andakter hålls av Albin Tanke, präst och psykoterapeut i Malmö. Temat för veckan är Att omdanas. Lyssna på alla avsnitt i Sveriges Radio Play. Ur andakten:Det finns nog få saker som är så viktiga som att låta hoppet leva i våra liv. Hoppet är livsgnista – och den bygger på en grundläggande, djup tillit som jag tror att vi alla bär på nånstans inom oss.Hoppet är inte enbart en gåva som räcks oss. Det finns också ett egenansvar i att försöka skapa hopp. Som Guds skapade medskapare har vi ansvar i världen att söka efter hoppet och hålla det vid liv så gott vi kan. Hoppet är alltså inte enbart något som kommer utifrån – vi är med och bygger det och är medansvariga för det och vi kan hjälpa varandra med hoppet.Andakten sändes första gången 2024.Text:Matt 28:20Musik:Pianosonat nr 16 av WA Mozart, Andante med Maria João PiresProducent:Susanna Némethliv@sverigesradio.se

Veckans andakter hålls av Albin Tanke, präst och psykoterapeut i Malmö. Temat för veckan är Att omdanas. Lyssna på alla avsnitt i Sveriges Radio Play. Ur andakten:På skrivbordet framför mig ligger alla mina prydliga ”att göra-listor”. De är mycket omsorgsfullt skrivna. Jag lägger ofta ganska mycket tid på att skriva dem.Men behöver vi egentligen skriva det som är allra viktigast på en ”komma ihåg-lapp”? Jag märker att mina ”att göra-listor” blir mer och mer översvallande. De börjar handla om större projekt, olika självförverkligande-idéer som nog egentligen handlar om något helt annat, en djupare önskan.Andakten sändes första gången 2024.Text:Matt 6:25-28, 31-34Musik:Pianosonat nr 16 av WA Mozart, Andante med Maria João PiresProducent:Susanna Némethliv@sverigesradio.se

Ce soir, un comédien humoriste et désormais chanteur avec un premier album solo. Vincent Dedienne revient avec “Un lendemain soir de gala”. 14 chansons toutes inspirées de personnages ou d'extraits de son spectacle qu'il prolonge donc en musique. Vincent Dedienne sur la scène de CAVOUS pour nous interpréter Y'en a encore en duo avec Albin de la Simone qui signe le texte et la mélodie de ce titre. A signaler, la réédition de l'album : « Toi là-bas » d'Albin de la Simonequi sera disponible le 30 octobre prochain avec deux titres inédits. Albin qui sera du 12 au 15 Novembre en concert au studio104 à Paris. Vincent Dedienne "Un lendemain soir de gala" est sorti le 10 octobre, Vincent Dedienne est en tournée dans toute la France et au Trianon le 20 décembre.Tous les soirs, du lundi au vendredi à 20h sur France 5, Anne-Elisabeth Lemoine et toute son équipe accueillent les personnalités et artistes qui font l'actualité.

Veckans andakter hålls av Albin Tanke, präst och psykoterapeut i Malmö. Temat för veckan är Att omdanas. Lyssna på alla avsnitt i Sveriges Radio Play. Ur andakten:Jag bor ganska nära en stor och vacker kyrkogård. Det är en begravningsplats som också är som en vacker park – med rik växtlighet, många stora träd, slingrande stigar och blommande buskar. Här brukar jag springa min morgonmotionsrunda. Jag älskar kyrkogårdens lugn. Det är så härligt att få röra på sig just på en sådan vacker plats – som trots att den ligger mitt i stan – är helt skyddad. Skyddad mot stress och krav och med en alldeles särskild atmosfär – fylld av rikedom, historia och frid. Stillheten och den fantastiska landskapsarkitekturen är själavårdande.Men så mitt när jag springer där på kyrkogården så drabbas jag plötsligt av tvivel: ”Är det verkligen okej att springa här? På en kyrkogård? Detta är ju de dödas vilorum… Vanhelgar jag inte de döda nu?”Andakten sändes första gången 2024.Text:Jes 40:31Musik:Pianosonat nr 16 av WA Mozart, Andante med Maria João PiresProducent:Susanna Némethliv@sverigesradio.se

Ta emot - Albin Karlsson by Pingstkyrkan Arken, Värnamo

While genetic testing has replaced muscle biopsy in the diagnosis of many genetic myopathies, clinical assessment and the integration of clinical and laboratory findings remain key elements for the diagnosis and treatment of muscle diseases. In this episode, Casey Albin, MD, speaks with Margherita Milone, MD, PhD, FAAN, FANA, author of the article “A Pattern Recognition Approach to Myopathy” in the Continuum® October 2025 Muscle and Neuromuscular Junction Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Milone is a professor of neurology and the director of the Muscle Pathology Laboratory at Mayo Clinic College of Medicine and Science in Rochester, Minnesota. Additional Resources Read the article: A Pattern Recognition Approach to Myopathy Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hello, this is Dr Casey Albin. Today I'm interviewing Dr Margherita Milone on her article on a pattern recognition approach to myopathy, which appears in the October 2025 Continuum issue on muscle and neuromuscular junction disorders. Welcome to the podcast, Dr Milone. Thank you so much for joining us. I'll start off by having you introduce yourself to our listeners. Dr Milone: Hello Casey, thank you so much for this interview and for bringing the attention to the article on muscle diseases. So, I'm Margherita Milone. I'm one of the neuromuscular neurologists at Mayo Clinic in Rochester. I have been interested in muscle disorders since I was a neurology resident many years ago. Muscle diseases are the focus of my clinical practice and research interest. Dr Albin: Wonderful. Thank you so much. When I think about myopathies, I generally tend to think of three large buckets: the genetic myopathy, the inflammatory myopathies, and then the necrotizing myopathies. Is that a reasonable approach to conceptualizing these myopathies? Dr Milone: Yeah, the ideology of the myopathies can be quite broad. And yes, we have a large group of genetic muscle diseases, which are the most common. And then we have immune-mediated muscle diseases, which include inflammatory myopathies as well as some form of necrotizing myopathies. Then we have some metabolic myopathies, which could be acquired or could be genetic. And then there are muscle diseases that are due to toxins as well as to infection. Dr Albin: Wow. So, lots of different etiologies. And that really struck me about your article, is that these can present in really heterogeneous ways, and some of them don't really read the rule book. So, we have to have a really high level of suspicion, for someone who's coming in with weakness, to remember to think about a myopathy. One of the things that I like to do is try to take us through a little bit of a case to sort of walk us through how you would approach if someone comes in. So, let's say you get, you know, a forty-year-old woman, and she's presenting with several months of progressive weakness. And she says that even recently she's noted just a little bit of difficulty swallowing. It feels to her like things are getting stuck. What are some of the things when you are approaching the history that would help you tease this to a myopathy instead of so many other things that can cause a patient to be weak? Dr Milone: Yes. So, as you mentioned, people who have a muscle disease have the muscle weakness often, but the muscle weakness is not just specific for a muscle disease. Because you can have a mass weakness in somebody who has a neurogenic paralysis. The problem with diagnosis of muscle diseases is that patients with these disorders have a limited number of symptom and sign that does not match the large heterogeneity of the etiology. So, in someone who has weakness, that weakness could represent a muscle disease, could represent an anterior horn cell disease, could represent a defect of neuromuscular junction. The clinical history of weakness is not sufficient by itself to make you think about a muscle disease. You have to keep that in the differential diagnosis. But your examination will help in corroborating your suspicion of a muscle disease. Let's say if you have a patient, the patient that you described, with six months' history of progressive weakness, dysphagia, and that patient has normal reflexes, and the patient has no clinical evidence for muscle fatigability and no sensory loss, then the probability that that patient has a myopathy increases. Dr Albin: Ah, that's really helpful. I'm hearing a lot of it is actually the lack of other findings. In some ways it's asking, you know, have you experienced numbness and tingling? And if not, that's sort of eliminating that this might not be a neuropathy problem. And then again, that fatigability- obviously fatigability is not specific to a neuromuscular junction, but knowing that is a hallmark of myasthenia, the most common of neuromuscular disorders. Getting that off the table helps you say, okay, well, it's not a neuromuscular junction problem, perhaps. Now we have to think more about, is this a muscle problem itself? Are there any patterns that the patients describe? I have difficulty getting up from a chair, or I have difficulty brushing my hair. When I think of myopathies, I historically have thought of, sort of, more proximal weakness. Is that always true, or not so much? Dr Milone: Yeah. So, there are muscle diseases that involve predominantly proximal weakness. For example, the patient you mentioned earlier could have, for example, an autoimmune muscle disease, a necrotizing autoimmune myopathy; could have, perhaps, dermatomyositis if there are skin changes. But a patient with muscle disease can also present with a different pattern of weakness. So, myopathies can lead to this weakness, and foot drop myopathies can cause- can manifest with the weakness of the calf muscles. So, you may have a patient presenting to the clinic who has no the inability to stand on tiptoes, or you may have a patient who has just facial weakness, who has noted the difficulty sealing their lips on the glasses when they drink and experiencing some drooling in that setting, plus some hand weakness. So, the muscle involved in muscle diseases can vary depending on the underlying cause of the muscle disease. Dr Albin: That's really helpful. So, it really is really keeping an open mind and looking for some supporting features, whether it's bulbar involvement, extraocular eye muscle involvement; looking, you know, is it proximal, is it distal? And then remembering that any of those patterns can also be a muscle problem, even if sometimes we think of distal being more neuropathy and proximal myopathy. Really, there's a host of ranges for this. I really took that away from your article. This is, unfortunately, not just a neat way to box these. We really have to have that broad differential. Let me ask another question about your history. How often do you find that patients complain of, sort of, muscular cramping or muscle pain? And does that help you in terms of deciding what type of myopathy they may have? Dr Milone: Many patients with muscle disease have muscle pain. The muscle pain could signal a presence of inflammation in skeletal muscle, could be the result of overuse from a muscle that is not functioning normally. People who have myotonia experience muscle stiffness and muscle pain. Patients who have a metabolic myopathy usually have exercise-induced muscle pain. But, as we know, muscle pain is also very nonspecific, so we have to try to find out from the patient in what setting the pain specifically occurs. Dr Albin: That's really helpful. So, it's asking a little bit more details about the type of cramping that they have, the type of pain they may be experiencing, to help you refine that differential. Similarly, one of the things that I historically have always associated with myopathies is an elevation in the CK, or the creatinine kinase. How sensitive and specific is that, and how do you as the expert sort of take into account, you know, what their CK may be? Dr Milone: So, this is a very good point. And the elevation of creatine kinase can provide a clue that the patient has a muscle disease, but it is nonspecific for muscle disease because we know that elevation of creatine kinase can occur in the setting of a neurogenic process. For example, we can see elevation of the creatine kinase in patients who have ALS or in patients who have spinal muscular atrophy. And in these patients---for example, those with spinal muscular atrophy---the CK elevation can be also of significantly elevated up to a couple of thousand. Conversely, we can have muscle diseases where the CK elevation does not occur. Examples of these are some genetic muscle disease, but also some acquired muscle diseases. If we think of, for example, cases where inflammation in the muscle occurs in between muscle fibers, more in the interstitium of the muscle, that disease may not lead to significant elevation of the CK. Dr Albin: That's super helpful. So, I'm hearing you say CK may be helpful, but it's neither completely sensitive nor completely specific when we're thinking about myopathic disorders. Dr Milone: You are correct. Dr Albin: Great. So, coming back to our patients, you know, she says that she has this dysphasia. How do bulbar involvement or extraocular eye movement involvement, how do those help narrow your differential? And what sort of disorders are you thinking of for patients who may have that bulbar or extraocular muscle involvement? Dr Milone: Regarding dysphagia, that can occur in the setting of acquired myopathies relatively frequent; for example, in inclusion body myositis or in other forms of inflammatory myopathy. Your patient, I believe, was in their forties, so it's a little bit too young for inclusion body myositis. Involvement of the extraocular muscles is usually much more common in genetic muscle diseases and much less frequent in hereditary muscle disease. So, if there is involvement of the extraocular muscles, and if there is a dysphagia, and if there is a proximal weakness, you may think about oculopharyngeal muscular dystrophy, for example. But obviously, in a patient who has only six months of history, we have to pay attention of the degree of weakness the patient has developed since the symptom onset. Because if the degree of weakness is mild, yes, it could still be a genetic or could be an acquired disease. But if we have a patient who, in six months, from being normal became unable to climb stairs, then we worry much more about an acquired muscle disease. Dr Albin: That's really helpful. So, the time force of this is really important. And when you're trying to think about, do I put this in sort of a hereditary form of muscle disease, thinking more of an indolent core, something that's going to be slowly progressive versus one of those inflammatory or necrotizing pathologies, that's going to be a much more quick onset, rapidly progressive, Do I have that right? Dr Milone: In general, the statement is correct. They tend, acquired muscle disease, to have a faster course compared to a muscular dystrophy. But there are exceptions. There have been patients with immune mediated necrotizing myopathy who have been misdiagnosed as having limb-girdle muscular dystrophy just because the disease has been very slowly progressive, and vice versa. There may be some genetic muscle diseases that can present in a relatively fast way. And one of these is a lipid storage myopathy, where some patients may develop subacutely weakness, dysphagia, and even respiratory difficulties. Dr Albin: Again, I'm hearing you say that we really have to have an open mind that myopathies can present in a whole bunch of different ways with a bunch of different phenotypes. And so, keeping that in mind, once you suspect someone has a myopathy, looking at the testing from the EMG perspective and then maybe laboratory testing, how do you use that information to guide your work up? Dr Milone: The EMG has a crucial role in the diagnosis of muscle diseases. Because, as we said earlier, weakness could be the result of muscle disease or other form of neuromuscular disease. If the EMG study will show evidence of muscle disease supporting your diagnostic hypothesis, now you have to decide, is this an acquired muscle disease or is this a genetic muscle disease? If you think that, based on clinical history of, perhaps, subacute pores, it is more likely that the patient has an acquired muscle disease, then I would request a muscle biopsy. The muscle biopsy will look for structural abnormalities that could help in narrowing down the type of muscle disease that the patient has. Dr Albin: That's really helpful. When we're sending people to get muscle biopsies, are there any tips that you would give the listeners in terms of what site to biopsy or what site, maybe, not to biopsy? Dr Milone: This is a very important point. A muscle biopsy has the highest diagnostic yield if it's done in a muscle that is weak. And because muscle diseases can result in proximal or distal weakness, if your patient has distal weakness, you should really biopsy a distal muscle. However, we do not wish to biopsy a muscle that is too weak, because otherwise the biopsy sample will result just in fibrous and fatty connected tissue. So, we want to biopsy a muscle that has mild to moderate weakness. Dr Albin: Great. So, a little Goldilocks phenomenon: has to be some weak, but not too weak. You got to get just the right feature there. I love that. That's a really good pearl for our listeners to take. What about on the flip side? Let's say you don't think it's an acquired a muscular disease. How are you handling testing in that situation? Dr Milone: If you think the patient has a genetic muscle disease, you pay a lot of attention to the distribution of the weakness. Ask yourself, what is the best pattern that represent the patient's weakness? So, if I have a patient who has facial weakness, dysphagia, muscle cramping, and then on examination represent myotonia, then at that point we can go straight to a genetic test for myotonic dystrophy type one. Dr Albin: That's super helpful. Dr Milone: So, you request directly that generic test and wait for the result. If positive, you will have proof that your diagnostic hypothesis was correct. Dr Albin: You're using the genetic testing to confirm your hypothesis, not just sending a whole panel of them. You're really informing that testing based on the patient's pattern of weakness and the exam findings, and sometimes even the EMG findings as well. Is that correct? Dr Milone: You are correct, and ideally, yes. And this is true for certain muscle diseases. In addition to myotonic dystrophy type one, for example, if you have a patient who has fascial scapulohumeral muscular weakness, you can directly request a test for FSHD. So, the characterization of the clinical phenotype is crucial before selecting the genetic test for diagnosis. Dr Albin: Wonderful. Dr Milone: However, this is not always possible, because you may have a patient who has just a limb-girdle weakness, and the limb-girdle weakness can be limb-girdle muscular dystrophy. But we know that there are many, many types of limb-girdle muscular dystrophies. Therefore, the phenotype is not sufficient to request specific genetic tests for one specific form of a limb-girdle muscular dystrophy. And in those cases, more complex next-generation sequencing panels have a higher chance of providing the answer. Dr Albin: Got it, that makes sense. So, sometimes we're using a specific genetic test; sometimes, it is unfortunate that we just cannot narrow down to one disease that we might be looking for, and we may need a panel in that situation. Dr Milone: You are correct. Dr Albin: Fantastic. Well, as we wrap up, is there anything on the horizon for muscular disorders that you're really excited about? Dr Milone: Yes, there are a lot of exciting studies ongoing for gene therapy, gene editing. So, these studies are very promising for the treatment of genetic muscle disease, and I'm sure there will be therapists that will improve the patient's quality of life and the disease outcome. Dr Albin: It's really exciting. Well, thank you again. Today I've been interviewing Dr Margarita Malone on her article on a pattern recognition approach to myopathy, which appears in the October 2025 Continuum issue on muscle and neuromuscular junction disorders. Be sure to check out Continuum Audio episodes from this and other issues, and thank you to our listeners for joining us today. And thank you, Dr Milone. Dr Milone: Thank you, Casey. Very nice chatting with you about this. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

durée : 00:58:47 - Le 13/14 - par : Bruno Duvic - Basé sur des faits réels peu connus, La Conspiration raconte les débuts d'un coup d'État qui a failli renverser la démocratie américaine. Vous aimez ce podcast ? Pour écouter tous les autres épisodes sans limite, rendez-vous sur Radio France.

Il pli gugent chanta Maria Catrina Albin-Caduff en il ravugl da «La Compagnia Rossini»: «Là ma sent 'jau purtada da mia segunda famiglia», di Maria Catrina. Tar il nov musical «Plaun Salter», s'engascha la sopranista davant e davos las culissas per il bainreussir da l'ovra da ses bab Armin Caduff.

In this episode, host Bill Calkins is joined by industry strategist and co-founder of A24 Consulting, Clint Albin, to discuss the artificial intelligence (AI) in the greenhouse and garden center industry. Bill and Clint explore the challenges faced by green industry businesses, including seasonal fluctuations and changing consumer behaviors and how AI can provide solutions to enhance customer engagement and streamline operations—what Clint refers to as “the front door and the overhead door”. The conversation emphasizes the importance of integrating AI thoughtfully and creatively into existing processes, the need for immediate customer feedback and some potential risks associated with AI implementation. Clint shares insights on future Green Profit article and podcast topics related to procurement and local marketing strategies, highlighting the significance of innovation in the horticulture sector. Some key takeaways from this episode: AI can empower businesses of all shapes and sizes. Our industry faces challenges due to seasonal business cycles. Customer visits to garden centers need to be increased. Immediate customer feedback is crucial for improvement. AI should not be applied to broken systems. The essence of abundance in AI can save time or make money. A policy on AI usage is necessary for each business. Understanding customer needs is always a key to success. This podcast supplements an article series in Green Profit magazine. Why You Need AI, No Matter Your Size (September 2025)

Patty discusses the 33rd Annual Indian Taco Supper at the Heritage Bible Church on the WRAM Morning Show.

Fluent Fiction - Swedish: Rekindling Connections: A Stockholm Autumn Tale Find the full episode transcript, vocabulary words, and more:fluentfiction.com/sv/episode/2025-09-28-22-34-01-sv Story Transcript:Sv: I Gamla Stan, den charmiga delen av Stockholm, var höstmarknaden i full gång.En: In Gamla Stan, the charming part of Stockholm, the autumn market was in full swing.Sv: De smala gatorna var fyllda med folk som ville fynda bland stånden med handgjorda varor och traditionella svenska godsaker.En: The narrow streets were filled with people eager to bargain among the stalls with handmade goods and traditional Swedish delicacies.Sv: Lukten av brända mandlar och varm glögg hängde i luften.En: The scent of roasted almonds and warm glögg hung in the air.Sv: Mitt i allt detta, i ett litet kafé med mjuka ljus från stearinljus och doften av kanel i luften, satt Albin.En: Amidst all this, in a small café with soft candlelight and the aroma of cinnamon in the air, sat Albin.Sv: Han stirrade på sin kopp kaffe och tänkte på gamla tider.En: He stared at his cup of coffee and thought about old times.Sv: Albin hade alltid trivts här.En: Albin had always felt at home here.Sv: Kaféet var en plats fylld av minnen, särskilt de med Lina.En: The café was a place filled with memories, especially those with Lina.Sv: Han hade inte sett henne på evigheter, ändå fanns hon alltid i tankarna.En: He hadn't seen her in ages, yet she was always in his thoughts.Sv: Han visste inte om han skulle våga hoppas eller om det var för sent.En: He didn't know if he dared to hope or if it was too late.Sv: Plötsligt öppnades dörren, och där stod Lina.En: Suddenly, the door opened, and there stood Lina.Sv: Hennes leende var varmt men det fanns ett stänk av överraskning i hennes ögon när hon fick syn på Albin.En: Her smile was warm, but there was a hint of surprise in her eyes when she spotted Albin.Sv: Efter en kort tvekan gick hon fram till hans bord.En: After a brief hesitation, she approached his table.Sv: "Hej Albin", sade Lina och satte sig.En: "Hi Albin," said Lina and sat down.Sv: Hennes röst var lugn, avslappnad.En: Her voice was calm, relaxed.Sv: "Hej Lina", svarade Albin och försökte dölja sitt hjärtas acceleration.En: "Hi Lina," replied Albin, trying to conceal his heart's acceleration.Sv: "Det var länge sedan."En: "It's been a long time."Sv: Lina log.En: Lina smiled.Sv: "Ja, det var det.En: "Yes, it has.Sv: Jag är glad att se dig."En: I'm glad to see you."Sv: De pratade om vädret, höstens färger och minnen från deras tid tillsammans.En: They talked about the weather, the colors of fall, and memories from their time together.Sv: Men under de lätta samtalsämnena kände Albin tyngden av det som verkligen behövde sägas.En: But beneath the light conversation topics, Albin felt the weight of what truly needed to be said.Sv: "Lina", började Albin med en osäkerhet i rösten.En: "Lina," Albin began with uncertainty in his voice.Sv: "Jag har tänkt mycket på oss.En: "I've thought a lot about us.Sv: Jag saknar...En: I miss...Sv: oss."En: us."Sv: Lina betraktade honom med tankfulla ögon.En: Lina regarded him with thoughtful eyes.Sv: Hon var tyst en stund och Albin kände att det var nu eller aldrig.En: She was silent for a moment, and Albin felt that it was now or never.Sv: "Jag gjorde misstag", fortsatte han.En: "I made mistakes," he continued.Sv: "Och jag vill att du ska veta att jag alltid har ångrat att jag lät det ta slut så."En: "And I want you to know that I've always regretted letting it end that way."Sv: Lina lyssnade, hennes leende var nu mildare, nästan sorgset.En: Lina listened, her smile now gentler, almost sorrowful.Sv: "Albin", sade hon försiktigt.En: "Albin," she said cautiously.Sv: "Jag har gått vidare.En: "I've moved on.Sv: Jag är lycklig där jag är nu.En: I'm happy where I am now.Sv: Men jag håller inte fast vid gammal smärta längre."En: But I'm no longer holding onto old pain."Sv: Albin nickade med en viss besvikelse men också insikt.En: Albin nodded with a certain disappointment but also understanding.Sv: "Jag förstår, jag vill bara säga förlåt.En: "I understand, I just wanted to say sorry.Sv: Och kanske, om möjligt, att vi kan vara vänner igen?"En: And maybe, if possible, that we could be friends again?"Sv: Lina tog hans hand.En: Lina took his hand.Sv: Kaféet omkring dem var till brädden fyllt med ljudet av marknadens glädje.En: The café around them was filled to the brim with the sounds of the market's joy.Sv: "Ja, jag skulle gilla det.En: "Yes, I would like that.Sv: Vänner är vi alltid."En: Friends we are, always."Sv: De satt där en stund till, pratade och skrattade precis som gamla vänner.En: They sat there for a while longer, talking and laughing just like old friends.Sv: Albin kände en vikt lätta från hans hjärta.En: Albin felt a weight lift from his heart.Sv: Han visste att han kunde gå vidare nu, precis som Lina hade gjort.En: He knew he could move on now, just like Lina had.Sv: När de lämnade kaféet, skildes de åt på gatan.En: As they left the café, they parted ways on the street.Sv: Höstsolen kastade långa skuggor på de gamla kullerstensgatorna.En: The autumn sun cast long shadows on the old cobblestone streets.Sv: Livet rörde sig vidare, och det gjorde de också, fri från det förflutnas kedjor, redo att omfamna sina egna nya äventyr.En: Life moved on, and so did they, free from the chains of the past, ready to embrace their own new adventures. Vocabulary Words:charming: charmigaautumn: höstbargain: fyndadelicacies: godsakeraroma: doftenmemories: minnensurprise: överraskninghesitation: tvekanconceal: döljaacceleration: accelerationregret: ångratpain: smärtasorrowful: sorgsetembrace: omfamnacandles: stearinljusregarded: betraktadethoughtful: tankfullchains: kedjorcobblestone: kullerstensgatorbrim: bräddenfill: fyllteager: ivrigstall: ståndglimpse: blickscent: luktenwarm: varmtdelicacies: godsakeralmonds: mandlarhesitant: tvekanderelaxed: avslappnad

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I detta avsnitt pratar vi om den nuvarande marknaden för klockindustrin och hur podden kommer att utvecklas framöver. Vi gör också en kort sammanfattning av året som gått och Albin delar med sig av sina favoritklockor för 2025.Alla artiklar hittar ni på Eqotime.comReferenser i avsnittet Tissot PR 516, Chronographhttps://eqotime.com/hands-on-tissot-pr516-chronograph-valjoux-klassisk-racingdesign-i-blatt-och-vitt/ Certina DS-X GMThttps://eqotime.com/certina-ds-x-gmt-aventyrlig-nyhet-for-resenaren/ Intervju med Niels Eggerding, Frederique Constanthttps://poddtoppen.se/podcast/1588926450/eqotime-podcast/eqotime-moter-niels-eggerding-vd-pa-frederique-constant Hosted on Acast. See acast.com/privacy for more information.

Tics are movements or sounds that are quick, recurrent, and nonrhythmic. They fluctuate over time and can be involuntary or semivoluntary. Although behavioral therapy remains the first-line treatment, modifications to comprehensive behavioral intervention have been developed to make treatment more accessible. In this episode, Casey Albin, MD, speaks with Jessica Frey, MD, author of the article “Tourette Syndrome and Tic Disorders” in the Continuum® August 2025 Movement Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Frey is an assistant professor of neurology, Movement Disorders Fellowship Program Director, and Neurology Student Clerkship Director at the Rockefeller Neuroscience Institute in the department of neurology at West Virginia University in Morgantown, West Virginia. Additional Resources Read the article: Tourette Syndrome and Tic Disorders Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Transcript Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hi all, this is Dr Casey Albin. Today I'm interviewing Dr Jessica Frey about her article Tourette Syndrome and Tic Disorders, which appears in the August 2025 Continuum issue on movement disorders. Dr Frey, thank you so much for being here, and welcome to the podcast. I'd love for you to briefly introduce yourself to our audience. Dr Frey: Thank you for having me here today. My name is Jessica Frey, and I am a movement disorder specialist at West Virginia University. I'm also the movement disorder fellowship director, as well as the neurology clerkship student director. Dr Albin: Dr. Frey, I feel like this was one of the things I actually had no exposure to as a resident. For trainees that kind of want to get a better understanding of how these are managed, what kind of counseling you do, what kind of interventions you're using, how can they get a little bit more exposure? Dr Frey: That's a great question, and I actually had a similar experience to you. I did not see that many patients with Tourette syndrome while I was in my residency training. I got a lot more exposure during my fellowship training, and that's when I actually fell in love with that patient population, caring for them, seeing them be successful. I think it depends on the program that you're in. During the pediatric neurology rotation might be your best bet to getting exposure to patients with Tourette syndrome, since a lot of them are going to be diagnosed when they're quite young, and sometimes they'll even continue to follow through young adulthood in the pediatric neurology clinic. However, up to 20% of patients with Tourette syndrome will have persistent tics during adulthood. And so, I think it is important for neurology trainees to understand how to manage them, understand what resources are out there. So, if you have an interest in that, absolutely try to follow either in the pediatric neurology department, or if you have a movement disorder program that has a Tourette clinic or has a movement disorder specialist who has an interest in Tourette syndrome, definitely try to hang out with them. Get to know that patient population, and educate yourself as much as you're able to educate the patients as well. Dr Albin: Yeah, I think that's fantastic advice. You wrote a fantastic article, and it covers a lot of ground. And I think let's start at some of the basics. When I think of Tourette syndrome and tics, I think of Tourette syndrome having tics, but maybe not all patients who have tics have Tourette syndrome. And so, I was wondering, A, if you could confirm that's true; and then could you tell us a little bit about some of the diagnostic criteria for each of these conditions? Dr Frey: Sure. So, a tic is a phenomenological description. So basically, what you're seeing is a description of a motor or phonic tic, which is a particular type of movement disorder. Tourette syndrome is a very specific diagnosis, and the diagnostic criteria for Tourette syndrome at this point in time is that you need to have had at least one phonic tic and two or more motor tics over the course of at least a year before the age of eighteen. Dr Albin: Got it. So, there's certainly more specific and a lot more criteria for having Tourette syndrome. I was struck in reading your article how many myths there are surrounding Tourette syndrome and tic disorders kind of in general. What's known about the pathophysiology of Tourette syndrome, and what are some common misconceptions about patients who have this disorder? Dr Frey: Yeah, so I think that's a really excellent question because for so many years, Tourette syndrome and tic disorders in general were thought to be psychogenic in origin, even dating back to when they were first described. The history of Tourette syndrome is quite interesting in that, when Tourette---who, you know, it's named after---was working with Charcot, a lot of the initial descriptors were of actual case reports of patients who had more psychogenic descriptions, and eventually they became known as tic disorders as well. It wasn't until the discovery of Haldol and using Haldol as a treatment for tic disorders that people started to change their perception and say, okay, maybe there is actually a neurologic basis for Tourette syndrome. So, in terms of the pathophysiology, it's not completely known, but what we do know about it, we think that there is some sort of hyperactivity in the corticostriatal-thalamocortical circuits. And we think that because of this hyperactivity, it leads to the hyperactive movement disorder. We think similar circuitry is involved in conditions like OCD, or obsessive compulsive disorder; as well as ADHD, or attention deficit hyperactivity disorder. And because of that, we actually do tend to see an overlap between all three of these conditions in both individuals and families. Dr Albin: And hearing all of that, does this all come back to, sort of, dopamine and, sort of, behavioral motivation, or is it different than that? Dr Frey: It's probably more complex than just dopamine, but there is the thought that dopamine does play a role. And even one of the hypotheses regarding the pathophysiology is actually that these tics might start as habits, and then when the habits become more common, they actually reshape the dopaminergic pathways. And each time a tic occurs, there's a little bit of a dopaminergic reward. And so over time, that reshapes those hyperactive pathways and changes the actual circuitry of the brain, leading it to be not just a habit but part of their neurologic makeup. Dr Albin: It's fascinating to hear how that actually might play into our neural circuitry and, over time, rewire our brain. Fascinating. I mean, this is just so interesting how movement disorders play into such behavioral regulation and some comorbid conditions like ADHD and OCD. I thought it would be really helpful, maybe, to our listeners to kind of think through a case that I suspect is becoming more common. So, if it's okay with you, I'll present sort of a hypothetical. Dr Frey: Absolutely. Dr Albin: This is a father bringing in his seventeen-year-old daughter. She's coming into the clinic because she's been demonstrating, over the past four to six weeks, some jerking movement in her right arm. And it's happened multiple times a day. And it was a pretty sudden onset. She had not had any movement like this before, and then several weeks ago, started moving the right hand. And then it became even more disruptive: her right leg was involved, she had some scrunching her face. This is all happening at a time where she was dealing with some stress, maybe a little bit of applications around college that she was having a lot of anxiety about. How do you sort of approach this case if this is someone who comes to your office? Dr Frey: Sure. So, I think the first thing that you want to get is a good solid history, trying to understand, what is the origin of these abnormal movements and what led to the abnormal movements. Now, a key thing here is that in Tourette syndrome, and most physiologic tic syndromes, there's a pretty early onset. So, in Tourette syndrome, the expected age of onset is between the ages of five and seven years old. So, to have kind of acute new abnormal movements as a seventeen-year-old would be very unusual for a new-onset diagnosis of Tourette syndrome. However, there's a couple of things from the history that could help you. One would be, were there ever tics in the past? Because sometimes, when you think retrospectively, a lot of these patients might have had a simple eye-blinking tic or a coughing tic when they were a child. And perhaps they did have Tourette syndrome, a very mild case of it. But because the tics were never that pronounced, they never went to see anyone about it and it was never known that they had Tourette syndrome in the first place. If there is no history like that and the movements are completely new, out of the blue, of course you want to rule out anything acute that could be going on that could be causing that. Looking at the phenomenology of the movements can also be very helpful. When you're looking at abnormal tic movements, you would expect most cases of something like Tourette syndrome to occur first in the midline and go in a rostrocoidal distribution. So, you mostly see things happening with eye blinking, throat clearing, sniffling, neck snapping. These are some of the immediate tics that start to happen. We also usually start to see simple tics, as opposed to complex tics, at the beginning. Now, over the course of time, many patients do develop more complex tics that might involve the arms or the extremities, but that would be unusual to see this as a presenting feature of new-onset Tourette syndrome. Dr Albin: Got it. So, I'm hearing that the history really matters and that sometimes, like those, like, first-onset seizures, I imagine as a neurointensivist, we see a lot of patients who've had seizures who think that they're presenting the first time. And then we go back and we say, well, actually they have had some abnormal movements at night. Sounds like it's very similar with these movement disorders where you have to really go back and ask, well, was there some sniffling? Did they go through a phase where they were grunting frequently? Because I can imagine that many children make those behaviors, and that it may not have registered as something that was cause for concern. Dr Frey: Absolutely. Dr Albin: And then the other thing I heard from you was that the phenomenology really matters and that there is a typical presentation, starting from sort of the face and working the way down. And that can be really helpful. But in this case, the family is quite clear. No, no, no. She's never had movements like this before. This is- nothing like this. We promise you, did not go through a phase where she was coughing or blinking, or, this is all totally new. And the phenomenology, they say, no, no, she did not start with blinking. It definitely started in the arm and then progressed in its complex movements. So, knowing that about her, how does that sort of shape how you move forward with the diagnosis? Dr Frey: Yeah. So, really good question. And this is something that I think really peaked during the Covid-19 pandemic. We saw an influx of patients, especially teenage girls or young adult girls, who basically would come in and have these new, acute-onset, abnormal movements. We weren't sure what to call them initially. There was some discussion of calling them “explosive tic disorder” and things like that. A lot of these actually looked very similar to psychogenic nonepileptic seizures, where they would come into the emergency department and have many abnormal movements that were so severe, that they were having a “tic attack” and couldn't stop the abnormal movements from occurring. And we saw so many of these cases during the Covid-19 pandemic that it eventually became known as a distinctive diagnostic criteria with the name of “functional ticlike behavior”, or FTLB. When we think about functional ticlike behavior, we think that these tics are driven more by anxiety and stress. A lot of times, the backstory of these patients, they were in a very stressful situation, and that's when the abnormal movement started. So, a very similar kind of backstory to patients that might develop psychogenic nonepileptic seizures. These tics were popularized, for lack of a better term, via social media during the Covid-19 pandemic. One article is out there that even has called these functional ticlike behaviors as “a pandemic within a pandemic”, because there was such a strong showing of ticlike behavior in the clinics during the Covid-19 pandemic. Although social media was thought to play a big role in these functional ticlike behaviors, we think that there's probably a little bit more complexity and nuance to why these functional ticlike behaviors develop. There is probably a little bit of a genetic predisposition. There's probably some other psychosocial factors at play. And when we see cases like this, the best thing that you can do is educate your patients about the differences between functional ticlike behaviors and tics that we see associated with conditions like Tourette syndrome. And then the best types of treatments that we have seen thus far are treating any underlying stressors, if any of those exist, as well as cognitive behavioral therapy has been shown to be somewhat helpful. As the Covid-19 pandemic has wound down, we have actually seen a lot less cases in our clinic. And one reason we think is less stressors, less uncertainty for the future, which we think was a driving precipitant of some of these cases. But it also is not as popularized in the media as well. There were a lot of TikTok users in particular, which lent itself to the name “TikTok tic”. These videos are not as viewed or not as popular as they were during the Covid-19 pandemic. One reason being that because we are not all relegated to our homes, constantly looking to online sources of information---just in general, we have kind of not been on the Internet as much as we were during the Covid-19 pandemic---as a society as a whole. Dr Albin: This is really fascinating how the environmental milieu, for lack of a better word, like, really influenced how patients were experiencing, sort of, functional neurologic disorders. In your article you describe really these three baskets of primary tic---which can then be a part of Tourette syndrome---,functional ticlike behaviors---which really were a unique manifestation of stress and anxiety specifically during the Covid-19 pandemic---, and then tics as a manifestation of some either different underlying etiology or medication side effect. So, when do you get concerned about that secondary etiology? Dr Frey: So secondary tics can occur in a variety of instances. I think some of the more common examples would be in genetic disorders. So, Huntington's disease is a really good example. I think we all associate chorea with Huntington's disease. That's probably the most commonly associated phenomenology that we see with Huntington's disease. But we can see a variety of movement disorders in Huntington's, and one of them is tics. So, when we see tics in association with other types of movement disorders, we should be thinking about a possible genetic etiology. If we see tics in association with other neurologic symptoms, such as seizures or cognitive changes, we should be thinking that this is something besides a primary tic disorder. You also mentioned medication use, and it's really important to think about tardive tics. I know we often think about tardive dyskinesia, and the first kind of phenomenology that jumps into our brain is usually chorea because it's those abnormal lip movements, finger movements, toe movements that we see after a patient has been on, for example, an antipsychotic or an antiemetic that has antidopaminergic properties. However, we can see a variety of abnormal movement disorders that occur secondary to antidopaminergic medications, especially after abrupt withdrawal of these antidopaminergic medications. And tics are one of them. There have been cases reported where people that have tardive tics will still report that they have a premonitory urge, as well as a sense of relief after their tics. So, it actually can seem very similar to Tourette syndrome and the tics that people with Tourette syndrome experience on a regular basis. The key here is that the treatment might differ because if it's due to an antidopaminergic medication or abrupt withdrawal of that antidopaminergic medication, you might need to treat it a little bit differently than you would otherwise. Dr Albin: I love that you bring in, it's not just looking at their specific movement disorder that they may be coming to clinic with, that tic disorder, but are there other movement disorders? Has there been a change in their medication history? Have they had cognitive changes? So really emphasizing the importance of that complete and comprehensive neurologic history, neurologic physical exam, to really get the complete picture so that it's not honing in on, oh, this is a primary tic. That's all there is to it, because it could be so much more. I know we're getting close to sort of the end of our time together, but I really wanted to switch to end on talking about treatment. And your article does such a beautiful job of talking about behavioral interventions and really exciting new medical interventions. But I would like to, if you don't mind, have you focus on, what behavioral counseling and what education do you provide for patients and their families? Because I imagine that the neurologist plays a really important role in educating the patient and their family about these disorders. Dr Frey: Absolutely. When we think about treatment, one of the most important things you can do for patients with Tourette syndrome or other primary tic disorders is educate them. This remains true whether it's a primary tic disorder that we see in Tourette syndrome or the functional ticlike behavior that we've discussed here. A lot of times, because there is such a stigma against people with tic disorders and Tourette syndrome, when they hear that they have Tourette syndrome or they are diagnosed with that, sometimes that can be an upsetting diagnosis. And sometimes you have to take time explaining what exactly that means and debunking a lot of the myths that go along with the stigmas associated with Tourette syndrome. I think a lot of times people are under the false assumption that people with Tourette syndrome cannot lead normal lives and cannot hold down jobs and cannot be productive members of society. None of that is true. Most of my patients have great lives, good quality of life, and are able to go about their day-to-day life without any major issues. And one of the reasons for that is we do have a lot of great treatment options available. Another important stigma to break down is that people with tic disorders are doing this for attention or doing this because they are trying to get something from someone else. That is absolutely false. We do think that the tics themselves are semivolitional because people with Tourette syndrome have some degree of control over their tics. They can suppress them for a period of time. But a lot of people with tic disorders and Tourette syndrome will describe their tics as if you're trying to hold onto a sneeze. And you can imagine how uncomfortable it is to hold in a sneeze. We're all able to do it for a period of time, but it's much easier to just allow that sneeze to occur. And a lot of times that's what they are experiencing, too. So, although there is some degree of control, it's not complete control, and they're certainly not doing these tics on purpose or for attention. So that's another important myth to debunk when you're counseling patients and their families. I think the dynamic between young patients that are presenting with their parents or guardians, sometimes that dynamic is a little bit challenging because another faulty assumption is that parents feel they are responsible for having this happen to their child. There used to be a really strong sense that parents were responsible for the tics that occurred in their children, and that is also absolutely not true. Parenting has nothing to do with having the tics or not. We know that this is a neurodevelopmental disorder. The brain is indeed wired differently and it's important to counsel that with the parents, too, so that they understand what tools they need to be successful for their children as well. Dr Albin: I love that. So, it's a lot of partnership with patients and their families. I really like that this is just a wire different, and I hope over time that working together we as neurologists can help break down some of that stigmatization for these patients. This has been an absolutely phenomenal discussion. I have so enjoyed learning from your article. For the listeners out there, there are some really phenomenal tables that go into sort of how to approach this from the office perspective, how to approach it from the treatment perspective. So, thank you again, Dr Jessica Frey, for your article on Tourette syndrome and tic disorders, which appears in the August 2025 Continuum issue on movement disorders. Be sure to check out Continuum Audio episodes from this and other issues, and thank you so much to our listeners for joining us today. Dr Frey: Thank you for having me. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

Le guitariste Thomas Naïm est le seul guitariste au monde à proposer un album solo sur Hendrix May This Be Love avec la participation du spécialiste français de Jimi Hendrix : Yazid Manou. (Rediffusion) La #SessionLive reçoit Thomas Naïm pour la sortie de l'album May This Be Love et le spécialiste français de Jimi Hendrix : Yazid Manou ! C'est après un stage d'été au Berklee College of Music de Boston et des cours à l'American School of Modern Music à Paris, que Thomas Naïm fonde avec la chanteuse Joyce Hozé le duo Tom & Joyce. Influencé par la MPB, la bossa nova des 60's et le jazz, le groupe enregistre deux albums : Tom & Joyce sorti en 2002 (Yellow Productions/East West) et Antigua en 2005 (Yellow Productions/Tommy Boy) sur lequel participe le légendaire batteur Tony Allen et dont certains morceaux seront remixés par des producteurs de dance music parmi les plus reconnus (Masters At Work, François Kevorkian ou Bob Sinclar). Par la suite, Thomas Naïm sera sollicité pour accompagner sur scène ou en studio de nombreux artistes venant d'horizons parfois très différents parmi lesquels Youn Sun Nah, Hugh Coltman, Hindi Zahra, Sébastien Tellier, Ala.ni, Mayra Andrade, Tiken Jah Fakoly, Blick Bassy, Idrissa Diop, Bob Sinclar, Salomé de Bahia, Brisa Roché, Bernard Lavilliers, Albin de la Simone, Michel Fugain, Claire Diterzi, Renan Luce… Il mène en parallèle depuis plusieurs années une carrière solo à la tête d'un trio avec le bassiste Marcello Giuliani et le batteur Raphaël Chassin avec lesquels il enregistre en 2018 l'album Desert Highway. En 2020, Thomas Naïm va confronter son trio aux compositions du maître Jimi Hendrix et enregistrer l'album Sounds of Jimi. L'idée de départ n'est pas de reprendre les titres d'Hendrix tel quel mais de trouver pour chaque morceau une esthétique plus personnelle, et à l'exception de trois titres chantés par Hugh Coltman et Célia Kameni, de privilégier l'approche instrumentale. L'album accueillera également en invités Erik Truffaz à la trompette et Camille Bazbaz à l'orgue. En 2022, il retourne en studio avec son trio augmenté de l'organiste/pianiste de Marc Benham pour enregistrer, sous la houlette du réalisateur grand angle Daniel Yvinec, On the Far Side, un album consacré à ses propres compositions dont la sortie est prévue pour février 2023. Thomas Naïm y retrouve ses premières amours, le Jazz, qu'il mêle à ses influences de toujours la pop, le rock psychédélique, les bandes originales de films et mille autres choses. Laurent Bardainne (saxophone) souffle sur trois titres un vent créatif qui donne à cet album unique une force et une couleur presque mystique. Avec May This Be Love, Thomas Naïm devient le 1er guitariste au monde, à reprendre Hendrix en guitare solo. Titres interprétés au grand studio - Hey Joe Live RFI - The Wind Cries Mary, extrait de l'album - Purple Haze Live RFI Line Up : Thomas Naïm, guitare. Son : Benoît Le Tirant, Camille Roch, Mathias Taylor. Album May This Be Love (Rootless Blues 2025). Site -  YouTube - Bandcamp.   Yazid Manou est journaliste, relations presse et spécialiste de Jimi Hendrix en France. en 1990, il organise un concert à l'Olympia pour les 20 ans de la disparition du guitariste américain. Il est le héros du roman « Blues pour Jimi Hendrix » écrit par Stéphane Koechlin. Yazid Manou est le gardien d'un mort comme Anubis dans la mythologie égyptienne, Jimi Hendrix. Il porte des fleurs sur sa tombe, soigne sa postérité, veille à sa gloire... En 1990, vingt ans après la mort de Jimi le 18 septembre 1970, il a organisé le festival « Jimi's Back » à Paris pendant une semaine dont une soirée à l'Olympia qui a bouleversé sa vie. Il y a invité de nombreux artistes pour reprendre les thèmes du bluesman. Il a convié bien sûr en premier lieu l'ex-bassiste de Jimi, Noel Redding, personnage douloureux et attachant, frappé quelques mois plus tôt par un deuil terrible, la mort de sa fiancée Carol Appleby dans un accident de voiture. Depuis, la vie de Yazid - devenu par la suite attaché de presse indépendant - est rythmée par les nouvelles de la « famille Hendrix », disputes avec la soeur adoptive Janie, rencontre avec ceux qui l'ont connu (B. B. King, Eric Clapton, Paul McCartney, Johnny Hallyday, Miles Davis, Taj Mahal), sorties d'albums (Jimi a davantage publié de disques une fois mort que vivant), disparition des témoins, Noel, Buddy Miles, Monika Dannemann (dernière compagne), Al Hendrix (son père) puis Mitch Mitchell. Il a construit sa vie autour d'un mort, de la mort en général. Yazid est un infatigable marcheur africain arpentant le pavé parisien en compagnie de son fantôme. Playlist Jimi Hendrix par Yazid Manou 51st Anniversary, Wait Until Tomorrow, Red House & All Along the Watchtower. Concert du 20 mars @ Le 360 Paris. Réalisation : Hadrien Touraud.

Le guitariste Thomas Naïm est le seul guitariste au monde à proposer un album solo sur Hendrix May This Be Love avec la participation du spécialiste français de Jimi Hendrix : Yazid Manou. (Rediffusion) La #SessionLive reçoit Thomas Naïm pour la sortie de l'album May This Be Love et le spécialiste français de Jimi Hendrix : Yazid Manou ! C'est après un stage d'été au Berklee College of Music de Boston et des cours à l'American School of Modern Music à Paris, que Thomas Naïm fonde avec la chanteuse Joyce Hozé le duo Tom & Joyce. Influencé par la MPB, la bossa nova des 60's et le jazz, le groupe enregistre deux albums : Tom & Joyce sorti en 2002 (Yellow Productions/East West) et Antigua en 2005 (Yellow Productions/Tommy Boy) sur lequel participe le légendaire batteur Tony Allen et dont certains morceaux seront remixés par des producteurs de dance music parmi les plus reconnus (Masters At Work, François Kevorkian ou Bob Sinclar). Par la suite, Thomas Naïm sera sollicité pour accompagner sur scène ou en studio de nombreux artistes venant d'horizons parfois très différents parmi lesquels Youn Sun Nah, Hugh Coltman, Hindi Zahra, Sébastien Tellier, Ala.ni, Mayra Andrade, Tiken Jah Fakoly, Blick Bassy, Idrissa Diop, Bob Sinclar, Salomé de Bahia, Brisa Roché, Bernard Lavilliers, Albin de la Simone, Michel Fugain, Claire Diterzi, Renan Luce… Il mène en parallèle depuis plusieurs années une carrière solo à la tête d'un trio avec le bassiste Marcello Giuliani et le batteur Raphaël Chassin avec lesquels il enregistre en 2018 l'album Desert Highway. En 2020, Thomas Naïm va confronter son trio aux compositions du maître Jimi Hendrix et enregistrer l'album Sounds of Jimi. L'idée de départ n'est pas de reprendre les titres d'Hendrix tel quel mais de trouver pour chaque morceau une esthétique plus personnelle, et à l'exception de trois titres chantés par Hugh Coltman et Célia Kameni, de privilégier l'approche instrumentale. L'album accueillera également en invités Erik Truffaz à la trompette et Camille Bazbaz à l'orgue. En 2022, il retourne en studio avec son trio augmenté de l'organiste/pianiste de Marc Benham pour enregistrer, sous la houlette du réalisateur grand angle Daniel Yvinec, On the Far Side, un album consacré à ses propres compositions dont la sortie est prévue pour février 2023. Thomas Naïm y retrouve ses premières amours, le Jazz, qu'il mêle à ses influences de toujours la pop, le rock psychédélique, les bandes originales de films et mille autres choses. Laurent Bardainne (saxophone) souffle sur trois titres un vent créatif qui donne à cet album unique une force et une couleur presque mystique. Avec May This Be Love, Thomas Naïm devient le 1er guitariste au monde, à reprendre Hendrix en guitare solo. Titres interprétés au grand studio - Hey Joe Live RFI - The Wind Cries Mary, extrait de l'album - Purple Haze Live RFI Line Up : Thomas Naïm, guitare. Son : Benoît Le Tirant, Camille Roch, Mathias Taylor. Album May This Be Love (Rootless Blues 2025). Site -  YouTube - Bandcamp.   Yazid Manou est journaliste, relations presse et spécialiste de Jimi Hendrix en France. en 1990, il organise un concert à l'Olympia pour les 20 ans de la disparition du guitariste américain. Il est le héros du roman « Blues pour Jimi Hendrix » écrit par Stéphane Koechlin. Yazid Manou est le gardien d'un mort comme Anubis dans la mythologie égyptienne, Jimi Hendrix. Il porte des fleurs sur sa tombe, soigne sa postérité, veille à sa gloire... En 1990, vingt ans après la mort de Jimi le 18 septembre 1970, il a organisé le festival « Jimi's Back » à Paris pendant une semaine dont une soirée à l'Olympia qui a bouleversé sa vie. Il y a invité de nombreux artistes pour reprendre les thèmes du bluesman. Il a convié bien sûr en premier lieu l'ex-bassiste de Jimi, Noel Redding, personnage douloureux et attachant, frappé quelques mois plus tôt par un deuil terrible, la mort de sa fiancée Carol Appleby dans un accident de voiture. Depuis, la vie de Yazid - devenu par la suite attaché de presse indépendant - est rythmée par les nouvelles de la « famille Hendrix », disputes avec la soeur adoptive Janie, rencontre avec ceux qui l'ont connu (B. B. King, Eric Clapton, Paul McCartney, Johnny Hallyday, Miles Davis, Taj Mahal), sorties d'albums (Jimi a davantage publié de disques une fois mort que vivant), disparition des témoins, Noel, Buddy Miles, Monika Dannemann (dernière compagne), Al Hendrix (son père) puis Mitch Mitchell. Il a construit sa vie autour d'un mort, de la mort en général. Yazid est un infatigable marcheur africain arpentant le pavé parisien en compagnie de son fantôme. Playlist Jimi Hendrix par Yazid Manou 51st Anniversary, Wait Until Tomorrow, Red House & All Along the Watchtower. Concert du 20 mars @ Le 360 Paris. Réalisation : Hadrien Touraud.

Le roman est un récit puissant pour sensibiliser aux enjeux écologiques et à l'urgence climatique. Daouda Ba, Marion Lagardère, Nathan Devers et Jean-Pierre Goux l'ont bien compris. Leurs livres font partie de cette rentrée littéraire et ils sont nos invités.   « Le roman est une machine inventée par l'homme pour l'appréhension du réel dans sa complexité », aimait dire le romancier et poète Louis Aragon. Or, l'urgence climatique et la crise écologique auxquelles nous sommes confrontés sont en effet bien complexes à appréhender. Il y a, bien sûr, toutes les approches en-ique : les rapports scientifiques, les analyses sociologiques, philosophiques ou économiques…  Mais pour convaincre et inciter à l'action ou au moins susciter une réaction, il faut parvenir à toucher le cœur et l'imaginaire, et cela seule la fiction peut le faire. Car «l'espèce humaine est fabulatrice», comme le décrit si bien Nancy Huston. Nous aimons que l'on nous raconte des histoires. Et la très bonne nouvelle, c'est qu'il y a de plus en plus d'écrivains qui s'emparent de ces questions... Avec - Nathan Devers pour son roman Surchauffe (Albin Michel) - Marion Lagardère pour son ouvrage La nuit des temps futurs (Librinova) - Jean-Pierre Goux, spécialiste en transition écologique pour son roman Révolution Bleue, la clé des songes (Eyrolles Éditions) - Daouda Ba en direct de Dakar pour son livre Les rêves brisés (L'Harmattan).  Musiques diffusées dans l'émission The Beatles - Paperback writer Albin de la Simone - J'aime lire.

Äntligen är det ordning på torpet, synd bara att kåken brinner. Kim, Per och Pest faller snabbt ner i brunpölen, bakdelen, röven och den eventuella bögen. Frågorna är som best pubertala men åh så viktiga! Lyssna nu, du kan vara bög! Finger eller ljus till alla er därute! Kärlek! /Imperiet podcast

#SessionLive avec deux poètes de la chanson française : Mathieu Boogaerts pour Grand piano et Albin de la Simone pour Toi là-bas. (Rediffusion) Notre premier invité est Mathieu Boogaerts pour la sortie de Grand Piano. Note d'intention par Mathieu Boogaerts : Mathieu Boogaerts, « Grand piano » ?  C'est le titre de mon neuvième album, et c'est un oxymore : « Figure de style qui vise à rapprocher deux termes que leurs sens devraient éloigner ». « Grand » car je l'ai voulu ainsi : franc, épais, puissant, plus de matière, de volume que ses prédécesseurs... Plus âgé ? Une batterie, une basse électrique, une basse synthétique, une guitare électrique, une guitare acoustique, un synthétiseur, un saxophone, une flûte, un accordéon, un chœur, des percussions, un piano électrique et un piano droit : la gamme de couleurs qu'il m'a fallu pour dépeindre en détails les sentiments que je déploie dans mes douze nouvelles chansons. Mais « Piano », car toujours sur le ton de la confidence, léger, fragile, doux, nuancé. Comment ? J'ai écrit et composé ce répertoire entre septembre 2020 et mars 2023 dans de nombreux lieux dont Londres, Paris, Istanbul, Amsterdam, Budapest, Plaisians, Risoul et les Landes. Le disque a été enregistré « de manière classique » entre 2023 et 2024 à La Frette Studios en région parisienne. Comme pour chacun de mes disques, Renaud Letang a ensuite élaboré le mixage, puis le duo de graphistes M/M (Paris) a conçu la pochette : ici une nature morte illustrant la facture, le geste, l'intention poétique de l'album. Avant ? Né en 1970 à Fontenay-sous-Bois. J'ai sorti mon premier « Ondulé » de clip-vidéo en 1995, suivi de l'album Super. Depuis : huit albums studio, trois albums live, plus de mille concerts à travers le monde, des chansons pour Camélia Jordana, Luce, Zaz, Vanessa Paradis... D'innombrables collaborations. En 2023, en pleine fabrication du disque, j'ai donné vingt concerts à Paris lors desquels je tirai au sort les chansons de ma discographie. Après ? Joie : l'écriture et la réalisation des clips-vidéo des morceaux. Les interpréter sur scène accompagné par mon tout nouveau groupe... Le Grand piano ! Mon meilleur disque ? Oui ! Titres interprétés dans le grand studio - Ma Jeunesse Live RFI  - Faut toujours écouter son corps, extrait de l'album - Dans une case Live RFI.  Line Up : Mathieu Boogaerts, guitare voix. Son : Camille Roch, Jérémie Besset. ► Album Grand Piano (Tôt ou Tard 2025). Site internet - YouTube - Facebook À lire aussiMathieu Boogaerts: «Grand piano», entre intimité et ambition musicale   Puis, nous recevons Albin de la Simone pour la sortie de Toi là-bas (disque) et Mes Battements (livre avec dessins chez Actes Sud). ► Teaser MdM. Le Livre Mes battements. Rome, 30 septembre 2024. Il est 11h du matin, partie à 4h30 chez moi, j'arrive à la Villa Médicis qui me fait le beau cadeau de m'inviter en courte résidence pour finir le livre que vous tenez entre les mains. Donc, à l'heure où j'écris ces lignes, ce n'est encore qu'un tas de dessins et de textes plus ou moins ordonnés. J'ai du pain sur la planche. Car un premier livre de ce type, comme un premier disque, est un peu constitué d'une vie entière, et quand on a 50 ans passés, il y a du tri à faire. À partir du deuxième, si on a bien fait son boulot dans le premier, on part d'une page blanche ou, au pire, d'une page beige. Nous verrons. L'album Toi là-bas. Paris, le 1er décembre 2024. Après quelques saisons très denses, j'ai ressenti à l'automne dernier le besoin imparable de me retrouver seul. Du moins face à moi-même. Je suis parti m'enfermer à la Villa Médicis où j'ai terminé d'écrire et de dessiner mon premier livre (Mes battements, paru en mars 2025 chez Actes Sud), un voyage intérieur qui, depuis des mois, me baladait loin dans le passé, mon village, mon enfance, mon adolescence. Je me suis amusé à reprendre quelques chansons de mes débuts comme Je te manque, Avril 4000 ou Non merci (2005), et j'ai remarqué que je leur donnais quelque chose que je ne voyais pas à l'époque. Je les habitais d'une nouvelle façon. J'ai tiré le fil, et d'autres chansons comme J'aime lire (2008) et enfin Pourquoi on pleure (2017) sont sorties naturellement de la pelote. Alors j'ai eu envie de les enregistrer à nouveau, comme de les photographier dans leur nouveau costume. Je me suis laissé aller sans faire de plans, sans pression, juste pour le jeu de la réinterprétation, pour le plaisir. Beaucoup de plaisir, musical autant que vocal. Les chansons en sont sorties plus sereines et plus sensuelles aussi, je crois. Boîtes à rythmes et basses profondes, synthétiseurs en halos suaves autour de mes instruments acoustiques chéris, comme mon piano Una Corda, et aussi cette incroyable Fender VI chère à The Cure ou Richard Hawley, une guitare électrique des années 60 accordée une octave plus grave, dont le son me retourne (écoutez le solo dans La valse des lilas). J'ai invité Alice on the Roof – avec qui je travaille pour son prochain album et que j'adore – à partager Pourquoi on pleure. Alice a accepté. La chanson en duo a pris un sens nouveau. Je pensais faire un petit EP à sortir à l'occasion de la parution du livre, mais comme je fouillais dans le rétroviseur depuis des mois, j'ai croisé quelques autres chansons importantes dans ma vie. La très souchonesque C'est bien moi que j'ai chantée l'an passé en hommage à Françoise Hardy avec Sage. La sublime Valse des lilas de Michel Legrand dont j'ai tant aimé la version américaine Once Upon a Summertime par Blossom Dearie ou Miles Davis. Et le temps s'arrêtait, d'Adamo, que nous avions arrangée avec Julien Chirol et Renaud Létang en 2003 pour le grand Salvatore lui-même. Et surtout, Ma gueule, lourdeur de Johnny qui me renvoyait au pire de la préadolescence brutale et masculiniste, jusqu'à ce que je découvre en la chantant qu'elle pouvait m'aller comme un gant et m'émouvoir même. Puisque mon livre et mon disque sortent en même temps, puisque leurs visuels et leurs thématiques sont cousins, j'ai quand même eu envie d'écrire une chanson nouvelle pour faire le pont entre les deux. En laissant errer mon regard dans les arbres de la Villa Médicis, j'ai revu Laurence, Natalia, Maud, Sidonie, Ouria… premières amoureuses qui ne m'ont pas connu. Que j'ai aimées, follement, mais de loin. Que j'ai regardées, sans relâche, dont j'ai étudié les gestes, les habitudes, les vêtements, ne détournant le regard que si je sentais le leur se tourner vers moi. J'avais trop peur. Peur de quoi. J'ai aussi entendu dans les arbres les échos de Charlotte Sometimes des Cure, sur laquelle je pleurais ces amours à distance. Trois jours plus tard, j'ai terminé la première maquette de Toi là-bas. Ça n'est donc pas un EP, mais bien un album. Mon huitième. Post-scriptum : Je ne résiste pas à repartir sur la route pour promener mes chansons et mon livre. Cette fois, je serai seul sur scène. Je chanterai et jouerai, je parlerai un peu, mais aussi je dessinerai. À moins d'un miracle anatomique, il est peu probable que je parvienne à faire tout cela en même temps. Titres interprétés dans le grand studio - Pourquoi on pleure Live RFI  - Toi là-bas, extrait de l'album - Quoi ma gueule Live RFI.  Line Up : Albin de la Simone, piano, voix. Son : Benoît Letirant, Mathias Taylor, Camille Roch. ► Album Toi là-bas (Tôt ou Tard). Site internet - YouTube - Facebook   Réalisation : Hadrien Touraud.

#SessionLive avec deux poètes de la chanson française : Mathieu Boogaerts pour Grand piano et Albin de la Simone pour Toi là-bas. (Rediffusion) Notre premier invité est Mathieu Boogaerts pour la sortie de Grand Piano. Note d'intention par Mathieu Boogaerts : Mathieu Boogaerts, « Grand piano » ?  C'est le titre de mon neuvième album, et c'est un oxymore : « Figure de style qui vise à rapprocher deux termes que leurs sens devraient éloigner ». « Grand » car je l'ai voulu ainsi : franc, épais, puissant, plus de matière, de volume que ses prédécesseurs... Plus âgé ? Une batterie, une basse électrique, une basse synthétique, une guitare électrique, une guitare acoustique, un synthétiseur, un saxophone, une flûte, un accordéon, un chœur, des percussions, un piano électrique et un piano droit : la gamme de couleurs qu'il m'a fallu pour dépeindre en détails les sentiments que je déploie dans mes douze nouvelles chansons. Mais « Piano », car toujours sur le ton de la confidence, léger, fragile, doux, nuancé. Comment ? J'ai écrit et composé ce répertoire entre septembre 2020 et mars 2023 dans de nombreux lieux dont Londres, Paris, Istanbul, Amsterdam, Budapest, Plaisians, Risoul et les Landes. Le disque a été enregistré « de manière classique » entre 2023 et 2024 à La Frette Studios en région parisienne. Comme pour chacun de mes disques, Renaud Letang a ensuite élaboré le mixage, puis le duo de graphistes M/M (Paris) a conçu la pochette : ici une nature morte illustrant la facture, le geste, l'intention poétique de l'album. Avant ? Né en 1970 à Fontenay-sous-Bois. J'ai sorti mon premier « Ondulé » de clip-vidéo en 1995, suivi de l'album Super. Depuis : huit albums studio, trois albums live, plus de mille concerts à travers le monde, des chansons pour Camélia Jordana, Luce, Zaz, Vanessa Paradis... D'innombrables collaborations. En 2023, en pleine fabrication du disque, j'ai donné vingt concerts à Paris lors desquels je tirai au sort les chansons de ma discographie. Après ? Joie : l'écriture et la réalisation des clips-vidéo des morceaux. Les interpréter sur scène accompagné par mon tout nouveau groupe... Le Grand piano ! Mon meilleur disque ? Oui ! Titres interprétés dans le grand studio - Ma Jeunesse Live RFI  - Faut toujours écouter son corps, extrait de l'album - Dans une case Live RFI.  Line Up : Mathieu Boogaerts, guitare voix. Son : Camille Roch, Jérémie Besset. ► Album Grand Piano (Tôt ou Tard 2025). Site internet - YouTube - Facebook À lire aussiMathieu Boogaerts: «Grand piano», entre intimité et ambition musicale   Puis, nous recevons Albin de la Simone pour la sortie de Toi là-bas (disque) et Mes Battements (livre avec dessins chez Actes Sud). ► Teaser MdM. Le Livre Mes battements. Rome, 30 septembre 2024. Il est 11h du matin, partie à 4h30 chez moi, j'arrive à la Villa Médicis qui me fait le beau cadeau de m'inviter en courte résidence pour finir le livre que vous tenez entre les mains. Donc, à l'heure où j'écris ces lignes, ce n'est encore qu'un tas de dessins et de textes plus ou moins ordonnés. J'ai du pain sur la planche. Car un premier livre de ce type, comme un premier disque, est un peu constitué d'une vie entière, et quand on a 50 ans passés, il y a du tri à faire. À partir du deuxième, si on a bien fait son boulot dans le premier, on part d'une page blanche ou, au pire, d'une page beige. Nous verrons. L'album Toi là-bas. Paris, le 1er décembre 2024. Après quelques saisons très denses, j'ai ressenti à l'automne dernier le besoin imparable de me retrouver seul. Du moins face à moi-même. Je suis parti m'enfermer à la Villa Médicis où j'ai terminé d'écrire et de dessiner mon premier livre (Mes battements, paru en mars 2025 chez Actes Sud), un voyage intérieur qui, depuis des mois, me baladait loin dans le passé, mon village, mon enfance, mon adolescence. Je me suis amusé à reprendre quelques chansons de mes débuts comme Je te manque, Avril 4000 ou Non merci (2005), et j'ai remarqué que je leur donnais quelque chose que je ne voyais pas à l'époque. Je les habitais d'une nouvelle façon. J'ai tiré le fil, et d'autres chansons comme J'aime lire (2008) et enfin Pourquoi on pleure (2017) sont sorties naturellement de la pelote. Alors j'ai eu envie de les enregistrer à nouveau, comme de les photographier dans leur nouveau costume. Je me suis laissé aller sans faire de plans, sans pression, juste pour le jeu de la réinterprétation, pour le plaisir. Beaucoup de plaisir, musical autant que vocal. Les chansons en sont sorties plus sereines et plus sensuelles aussi, je crois. Boîtes à rythmes et basses profondes, synthétiseurs en halos suaves autour de mes instruments acoustiques chéris, comme mon piano Una Corda, et aussi cette incroyable Fender VI chère à The Cure ou Richard Hawley, une guitare électrique des années 60 accordée une octave plus grave, dont le son me retourne (écoutez le solo dans La valse des lilas). J'ai invité Alice on the Roof – avec qui je travaille pour son prochain album et que j'adore – à partager Pourquoi on pleure. Alice a accepté. La chanson en duo a pris un sens nouveau. Je pensais faire un petit EP à sortir à l'occasion de la parution du livre, mais comme je fouillais dans le rétroviseur depuis des mois, j'ai croisé quelques autres chansons importantes dans ma vie. La très souchonesque C'est bien moi que j'ai chantée l'an passé en hommage à Françoise Hardy avec Sage. La sublime Valse des lilas de Michel Legrand dont j'ai tant aimé la version américaine Once Upon a Summertime par Blossom Dearie ou Miles Davis. Et le temps s'arrêtait, d'Adamo, que nous avions arrangée avec Julien Chirol et Renaud Létang en 2003 pour le grand Salvatore lui-même. Et surtout, Ma gueule, lourdeur de Johnny qui me renvoyait au pire de la préadolescence brutale et masculiniste, jusqu'à ce que je découvre en la chantant qu'elle pouvait m'aller comme un gant et m'émouvoir même. Puisque mon livre et mon disque sortent en même temps, puisque leurs visuels et leurs thématiques sont cousins, j'ai quand même eu envie d'écrire une chanson nouvelle pour faire le pont entre les deux. En laissant errer mon regard dans les arbres de la Villa Médicis, j'ai revu Laurence, Natalia, Maud, Sidonie, Ouria… premières amoureuses qui ne m'ont pas connu. Que j'ai aimées, follement, mais de loin. Que j'ai regardées, sans relâche, dont j'ai étudié les gestes, les habitudes, les vêtements, ne détournant le regard que si je sentais le leur se tourner vers moi. J'avais trop peur. Peur de quoi. J'ai aussi entendu dans les arbres les échos de Charlotte Sometimes des Cure, sur laquelle je pleurais ces amours à distance. Trois jours plus tard, j'ai terminé la première maquette de Toi là-bas. Ça n'est donc pas un EP, mais bien un album. Mon huitième. Post-scriptum : Je ne résiste pas à repartir sur la route pour promener mes chansons et mon livre. Cette fois, je serai seul sur scène. Je chanterai et jouerai, je parlerai un peu, mais aussi je dessinerai. À moins d'un miracle anatomique, il est peu probable que je parvienne à faire tout cela en même temps. Titres interprétés dans le grand studio - Pourquoi on pleure Live RFI  - Toi là-bas, extrait de l'album - Quoi ma gueule Live RFI.  Line Up : Albin de la Simone, piano, voix. Son : Benoît Letirant, Mathias Taylor, Camille Roch. ► Album Toi là-bas (Tôt ou Tard). Site internet - YouTube - Facebook   Réalisation : Hadrien Touraud.

Albin Gerhardsson kommer precis ifrån en helg där han vunnit årets första deltävling i MXSM. En serie där han ligger på en total tredje plats inför det avslutande racet i Vimmerby. I dagarna också uttagen till årets MXoN lag som kommer att ta sig över till USA för att försvara Sveriges färger. Vi pratar om allt detta och även Gotland Grand National.

Il se lance dans la musique : l'humoriste et comédien Vincent Dedienne dévoile vendredi 5 septembre deux extraits de son premier album, qui s'intitulera "Un lendemain soir de Gala", avec des chansons écrites par des copains chanteurs, Vincent Delerm, Albin de la Simone ou encore Ben Mazué, à partir de son précédent spectacle comique. Marie Gicquel l'a rencontré pour RTL. Autre actualité musicale, le retour du chanteur à l'accordéon Claudio Capéo. Ecoutez Laissez-vous tenter - Première avec Marie Gicquel du 05 septembre 2025.Hébergé par Audiomeans. Visitez audiomeans.fr/politique-de-confidentialite pour plus d'informations.

Tisdag! Albin må vara på flängande fot, men inte ska väl det få stå i vägen för ett gäng kakor? Det blir vikingar, midsommar och Telia. Har du ett skvaller som fler borde få höra? Maila det till kafferepetpod@gmail.comMissa inte vår månatliga systerpodd Cigarrummet. Bli prenumerant på www.underproduktion.se/cigarrummet1:50 - Livförsäkring på jobbet4:30 - Midsommarnatts(mar)dröm10:52 - Farfar och internet Hosted on Acast. See acast.com/privacy for more information.

Multiple system atrophy is a rare, sporadic, adult-onset, progressive, and fatal neurodegenerative disease. Accurate and early diagnosis remains challenging because it presents with a variable combination of symptoms across the autonomic, extrapyramidal, cerebellar, and pyramidal systems. Advances in brain imaging, molecular biomarker research, and efforts to develop disease-modifying agents have shown promise to improve diagnosis and treatment. In this episode, Casey Albin, MD speaks with Tao Xie, MD, PhD, author of the article “Multiple System Atrophy” in the Continuum® August 2025 Movement Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Xie is director of the Movement Disorder Program, chief of the Neurodegenerative Disease Section in the department of neurology at the University of Chicago Medicine in Chicago, Illinois. Additional Resources Read the article: Multiple System Atrophy Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr. Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hello everyone, this is Dr Casey Albin. Today I'm interviewing Dr Tao Xie about his article on diagnosis and management of multiple system atrophy, which appears in the August 2025 Continuum issue on movement disorders. Welcome to the podcast, and please introduce yourself to our audience. Dr Xie: Thank you so much, Dr Albin. My name is Tao Xie, and sometimes people also call me Tao Z. I'm a mood disorder neurologist, professor of neurology at the University of Chicago. I'm also in charge of the mood disorder program here, and I'm the section chief in the neurodegenerative disease in the Department of Neurology at the University of Chicago Medicine. Thank you for having me, Dr Albin and Dr Okun and the American Academy of Neurology. This is a great honor and pleasure to be involved in this education session. Dr Albin: We are delighted to have you, and thank you so much for the thoughtful approach to the diagnosis and management. I really want to encourage our listeners to check out this article. You know, one of the things that you emphasize is multiple system atrophy is a fairly rare condition. And I suspect that clinicians and trainees who even have a fair amount of exposure to movement disorders may not have encountered that many cases. And so, I was hoping that you could just start us off and walk us through what defines multiple system atrophy, and then maybe a little bit about how it's different from some of the more commonly encountered movement disorders. Dr Xie: This is a really good question, Dr Albin. Indeed, MSA---multisystem atrophy----is a rare disease. It is sporadic, adult-onset, progressive, fatal neurodegenerative disease. By the name MSA, multisystem atrophy. Clinically, it will present with multiple symptoms and signs involving multiple systems, including symptoms of autonomic dysfunction and symptoms of parkinsonism, which is polyresponsive to the levodopa treatment; and the symptom of cerebellar ataxia, and symptom of spasticity and other motor and nonmotor symptoms. And you may be wondering, what is the cause- underlying cause of these symptoms? Anatomically, we can find the area in the basal ganglia striatonigral system, particularly in the putamen and also in the cerebellar pontine inferior, all of the nuclear area and the specific area involved in the autonomic system in the brain stem and spinal cord: all become smaller. We call it atrophy. Because of the atrophy in this area, they are responsible for the symptom of parkinsonism if it is involved in the putamen and the cerebral ataxia, if it's involved in the pons and cerebral peduncle and the cerebellum. And all other area, if it's involved in the autonomic system can cause autonomic symptoms as well. So that's why we call it multisystem atrophy. And then what's the underlying cellular and subcellular pathological, a hallmark that is in fact caused by misfolded alpha-synuclein aggregate in the oligodontia site known as GCI---glial cytoplasmic increasing bodies---in the cells, and sometimes it can also be found in the neuronal cell as well in those areas, as mentioned, which causes the symptom. But clinically, the patient may not present all the symptoms at the same time. So, based on the predominant clinical symptom, if it's mainly levodopa, polyresponsive parkinsonism, then we call it MSAP. If it's mainly cerebellar ataxia, then we call it MSAC. But whether we call it MSP or MSC, they all got to have autonomic dysfunction. And also as the disease progresses, they can also present both phenotypes together. We call that mixed cerebellar ataxia and parkinsonism in the advanced stage of the disease. So, it is really a complicated disease. The complexity and the similarity to other mood disorders, including parkinsonism and the cerebellar ataxia, make it really difficult sometimes, particularly at the early stages of disease, to differentiate one from the other. So, that was challenging not only for other professionals, general neurologists and even for some movement disorder specialists, that could be difficult particularly if you aim to make an accurate and early diagnosis. Dr Albin: Absolutely. That is such a wealth of knowledge here. And I'm going to distill it just a little bit just to make sure that I understand this right. There is alpha-synuclein depositions, and it's really more widespread than one would see maybe in just Parkinson's disease. And with this, you are having patients present with maybe one of two subtypes of their clinical manifestations, either with a Parkinson's-predominant movement disorder pattern or a cerebellar ataxia type movement disorder pattern. Or maybe even mixed, which really, you know, we have to make things quite complicated, but they are all unified and having this shared importance of autonomic features to the diagnosis. Have I got that all sort of correct? Dr Xie: Correct. You really summarize well. Dr Albin: Fantastic. I mean, this is quite a complicated disease. I would pose to you sort of a case, and I imagine this is quite common to what you see in your clinic. And let's say, you know, a seventy-year-old woman comes to your clinic because she has had rigidity and poor balance. And she's had several falls already, almost always from ground level. And her family tells you she's quite woozy whenever she gets up from the chair and she tends to kind of fall over. But they noticed that she's been stiff,and they've actually brought her to their primary care doctor and he thought that she had Parkinson's disease. So, she started levodopa, but they're coming to you because they think that she probably needs a higher dose. It's just not working out very well for her. So how would you sort of take that history and sort of comb through some of the features that might make you more concerned that the patient actually has undiagnosed multiple systems atrophy? Dr Xie: This is a great case, because we oftentimes can encounter similar cases like this in the clinic. First of all, based on the history you described, it sounds like an atypical parkinsonism based on the slowness, rigidity, stiffness; and particularly the early onset of falls, which is very unusual for typical Parkinson disease. It occurs too early. If its loss of balance, postural instability, and fall occurred within three years of disease onset---usually the motor symptom onset---then it raises a red flag to suspect this must be some atypical Parkinson disorders, including multiple system atrophy. Particularly, pou also mentioned that the patient is poorly responsive to their levodopa therapy, which is very unusual because for Parkinson disease, idiopathic Parkinson disease, we typically expect patients would have a great response to the levodopa, particularly in the first 5 to 7 years. So to put it all together, this could be atypical parkinsonism, and I could not rule out the possibility of MSA. Then I need to check more about other symptoms including autonomic dysfunction, such as orthostatic hypertension, which is a blood pressure drop when the patient stands up from a lying-down position, or other autonomic dysfunctions such as urinary incontinence or severe urinary retention. So, in the meantime, I also have to put the other atypical Parkinson disorder on the differential diagnosis, such as PSP---progressive supranuclear palsy---and the DLBD---dementia with Lewy body disease.---Bear this in mind. So, I want to get more history and more thorough bedside assessment to rule in or rule out my diagnosis and differential diagnosis. Dr Albin: That's super helpful. So, looking for early falls, the prominence of autonomic dysfunction, and then that poor levodopa responsiveness while continuing to sort of keep a very broad differential diagnosis? Dr Xie: Correct. Dr Albin: One of the things that I just have to ask, because I so taken by this, is that you say in the article that some of these patients actually have preservation of smell. In medical school, we always learn that our Parkinson's disease patients kind of had that early loss of smell. Do you find that to be clinically relevant? Is that- does that anecdotally help? Dr Xie: This is a very interesting point because we know that the loss of smelling function is a risk effect, a prodromal effect, for the future development of Parkinson disease. But it is not the case for MSA. Strange enough, based on the literature and the studies, it is not common for the patient with MSA to present with anosmia. Some of the patients may have mild to moderate hyposmia, but not to the degree of anosmia. So, this is why even in the more recent diagnosis criteria, the MDS criteria published 2022, it even put the presence of anosmia in the exclusion criteria. So, highlight the importance of the smell function, which is well-preserved for the majority in MSA, into that category. So, this is a really interesting point and very important for us, particularly clinicians, to know the difference in the hyposmia, anosmia between the- we call it the PD, and the dementia Lewy bodies versus MSA. Dr Albin: Fascinating. And just such a cool little tidbit to take with us. So, the family, you know, you're talking to them and they say, oh yes, she has had several fainting episodes and we keep taking her to the primary care doctor because she's had urinary incontinence, and they thought maybe she had urinary tract infections. We've been dealing with that. And you're sort of thinking, hm, this is all kind of coming together, but I imagine it is still quite difficult to make this diagnosis based on history and physical alone. Walk our listeners through sort of how you're using MRI and DAT scan and maybe even some other biomarkers to help sort of solidify that diagnosis. Dr Xie: Yeah, that's a wonderful question. Yeah. First of all, UTI is very common for patients with MSA because of urinary retention, which puts them into a high risk of developing frequent UTI. That, for some patients, could be the very initial presentation of symptoms. In this case, if we check, we say UTI is not present or UTI is present but we treat it, then we check the blood pressure and we do find also hypertension---according to new diagnosis criteria, starting drop is 20mm mercury, but that's- the blood pressure drop is ten within three minutes. And also, in the meantime the patients present persistent urinary incontinence even after UTI was treated. And then the suspicion for MS is really high right at this point. But if you want increased certainty and a comfortable level on your diagnosis, then we also need to look at the brain MRI mark. This is a required according to the most recent MDS diagnosis criteria. The presence of the MRI marker typical for MSA is needed for the diagnosis of clinically established MSA, which holds the highest specificity in the clinical diagnosis. So then, we have- we're back to your question. We do need to look at the brain MRI to see whether evidence suggestive of atrophy around the putamen area, around the cerebellar pontine inferior olive area, is present or not. Dr Albin: Absolutely. That's super helpful. And I think clinicians will really take that to sort of helping to build a case and maybe recognizing some of this atypical Parkinson's disease as a different disease entity. Are there any other biomarkers in the pipeline that you're excited about that may give us even more clarity on this diagnosis? Dr Xie: Oh, yeah. This is a very exciting area. In terms of biomarker for the brain imaging, particularly brain MRI, in fact, today there's a landmark paper just published in the Java Neurology using AI, artificial intelligence or machine learning aid, diagnoses a patient with parkinsonism including Parkinson's disease, MSA, and PSP, with very high diagnostic accuracy ranging from 96% to 98%. And some of the cases even were standard for autopsy, with pathological verification at a very high accurate rate of 93.9%. This is quite amazing and can really open new diagnosis tools for us to diagnose this difficult disease; not only in an area with a bunch of mood disorder experts, but also in the rural area, in the area really in need of mood disorder experts. They can provide tremendous help to provide accurate, early diagnosis. Dr Albin: That's fantastic and I love that, increasing the access to this accurate diagnosis. What can't artificial intelligence do for us? That's just incredible. Dr Xie: And also, you know, this is just one example of how the brain biomarker can help us. Theres other---a fluid biomarker, molecular diagnostic tools, is also available. Just to give you an example, one thing we know over the past couple years is skin biopsy. Through the immunofluorescent reaction, we can detect whether the hallmark of abnormally folded, misfolded, and the phosphorate, the alpha-synuclein aggregate can be found just by this little pinch of skin biopsy. Even more advanced, there's another diagnosis tool we call the SAA, we call the seizure amplification assay, that can even help us to differentiate MSA from other alpha-synucleinopathy, including Parkinson disease and dementia with Lewy bodies. If we get a little sample from CSF, spinal cerebral fluids, even though this is probably still at the early stage, a lot of developments still ongoing, but this, this really shows you how exciting this area is now. We're really in a fast forward-moving path now. Dr Albin: It's really incredible. So, lots coming down the track in, sort of, MRI, but also with CSF diagnosis and skin biopsies. Really hoping that we can hone in some of those tools as they become more and more validated to make this diagnosis. Is that right? Dr Xie: Correct. Dr Albin: Amazing. We can talk all day about how you manage these in the clinic, and I really am going to direct our listeners to go and read your fantastic article, because you do such an elegant job talking about how this takes place in a multidisciplinary setting, if at all possible. But as a neurointensivist, I was telling you, we have so much trouble in the hospital. We have A-lines, and we have the ability to get rapid KUBs to look at Ilias, and we can have many people as lots of diagnosis, and we still have a lot of trouble treating autonomiclike symptoms. Really, really difficult. And so, I just wanted to kind of pick your brain, and I'll start with just the one of orthostatic hypotension. What are some of the tips that you have for, you know, clinicians that are dealing with this? Because I imagine that this is quite difficult to do without patients. Dr Xie: Exactly. This is indeed a very difficult symptom to deal with, particularly at an outpatient setting. But nowadays with the availability of more medication---to give an example, to treat patients with orthostatic hypertension, we have not only midodrine for the cortisol, we also have droxidopa and several others as well. And so, we have more tools at hand to treat the patient with orthostatic hypertension. But I think the key thing here, particularly for us to the patient at the outpatient setting: we need to educate the patient's family well about the natural history of the disease course. And we also need to tell them what's the indication and the potential side effect profile of any medication we prescribe to them so that they can understand what to expect and what to watch for. And in the meantime, we also need to keep really effective and timely communication channels, make sure that the treating physician and our team can be reached at any time when the patient and family need us so that we can be closely monitoring, their response, and also monitoring potential side effects as well to keep up the quality of care in that way. Dr Albin: Yeah, I imagine that that open communication plays a huge role in just making sure that patients are adapting to their symptoms, understanding that they can reach out if they have refractory symptoms, and that- I imagine this takes a lot of fine tuning over time. Dr Xie: Correct. Dr Albin: Well, this has just been such a delight to get to talk to you. I really feel like we could dive even deeper, but I know for the sake of time we have to kind of close out. Are there any final points that you wanted to share with our listeners before we end the interview? Dr Xie: I think for the patients, I want them to know that nowadays with advances in science and technology, particularly given a sample of rapid development in the diagnostic tools and the multidisciplinary and multisystemic approach to treatment, nowadays we can make an early and accurate diagnosis of the MSA, and also, we can provide better treatment. Even though so far it is still symptomatically, mainly, but in the near future we hope we can also discover disease-modifying treatment which can slow down, even pause or prevent the disease from happening. And for the treating physician and care team professionals, I just want them to know that you can make a difference and greatly help the patient and the family through your dedicated care and also through your active learning and innovative research. You can make a difference. Dr Albin: That's amazing and lots of hope for these patients. Right now, you can provide really great care to take care of them, make an early and accurate diagnosis; but on the horizon, there are really several things that are going to move the field forward, which is just so exciting. Again today, I've been really greatly honored and privileged to be able to talk to Dr Tao Xie about his article on diagnosis and management of multiple system atrophy, which appears in the August 2025 Continuum issue on movement disorders. Be sure to check out Continuum Audio episodes for this and other issues. And thank you again to our listeners for joining us today. Dr Xie: Thank you so much for having me. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

Albin Brun mag es, Naturerfahrungen und Landschaftsstimmungen in Klänge zu fassen. Beim Festival «Alpentöne» gehört er zu den Stammgästen. Im Theater Uri erzählt er von seiner Faszination für Volksmusik aus verschiedenen Kulturen. Und er verrät, weshalb er seine Konzerte in Gedichtform ankündigt. In seiner Kindheit in Luzern hätte wohl niemand vermutet, dass Albin Brun einmal Musiker werden würde. Sieben Jahre lang ging er lustlos in den Flötenunterricht, bis er in den 1970er Jahren erstmals das Folk-Festival Lenzburg besuchte: „Dort entdeckte ich den Geist des Einfachen, Unverfälschten.“ Albin Brun fing Feuer und begann, autodidaktisch mehrere Instrumente zu lernen. Nach der Schule tingelte er eine Weile als Strassenmusiker durch die Lande, bevor er sich an der Jazzschule einschrieb. Mittlerweile hat ihn seine Leidenschaft um die ganze Welt geführt. Diese Erfahrung kommt in seiner Musik zum Ausdruck, in der Heimisches und Fremdes ganz selbstverständlich zusammenfinden. Die Musiktitel: 1. Urs Hostettler: Outobahn 2. Bobby Jones: Bringin' in the sheep 3. Didier Squiban: Ledenez Kurzer Einspieler: Ala Fekra - Longa Yamen 4. Singfrauen Winterthur: Aslanuri Mravaljamier Albin Brun Quartett / Franziska Welti, Leitung 5. Mikhail Alperin & Arkady Shilkloper: Unisons 6. Paul Giger: Zäuerli - Pt. 1 7. Albin Brun Quartett – Himbrimi (Album: «Pas de quatre»)

Join Nick Lamagna on The A Game Podcast with our guest Nick Albin, aka "Chewy," one of the most well known Brazilian Jiu Jitsu Black Belts and entrepreneurs on the internet today!  He is an entrepreneur, business owner, podcast host and coach fighting out of Louisville, Kentucky!  After a traumatic experience where he was jumped in high school, he lacked self confidence and a sense of safety and self worth.  He was eventually brought back out of his shell and gained his confidence back through wrestling, weight lifting and martial arts eventually earning a black belt in Brazilian Jiu Jitsu in 2011. This self proclaimed meathead at heart has an extensive background in martial arts, health, nutrition and fitness.  He has a gift for explaining things in an entertaining and relatable way which has caught on like wild fire where he has earned a blackbelt in branding business and marketing.  He continually finds ways to grow his academy and podcast sharing his love of Brazilian jiu jitsu, health and fitness with the world through his academy, Derby City Mixed Martial Arts and his wildly popular Youtube Channel and social media, where athletes and business owners can relate to the parallels of the stress of humanity and he is connecting the bridges of the brains of the civilian and the combat sport competitor to show us that we're all the same and can help each other do better He also has a popular tournament, The Chewjitsu Open he puts on and is gaining a lot of traction in the competition circles.   You know him through his Chewjitsu brand,The Chewjitsu Podcast and his successful Chew Crew Newsletter, welcome to the A Game Podcast, BJJ Royalty Nick Albin! Topics for this episode include: ✅ An important question you must answer before you go into business ✅ The answer to successful branding that no one wants to hear ✅ How to deal with fear and anxiety in life and business ✅ Learn the superpower to embrace the things you don't enjoy ✅ What to look for when you're trying to find a great mentor + more! Check the show notes to connect with all things Chewy!   Connect with Nick "Chewy" Albin: www.chewjitsu.net Nick Albin on Facebook Nick Albin on Instagram Nick Albin on Twitter Nick Albin on LinkedIn Nick Albin on TikTok Nick Albin on Youtube Free Focused BJJ Ebook Subscribe to the Chew Crew Newsletter!   Connect with ChewJitsu Podcast: ChewJitsu Podcast Chewjitsu Podcast on Facebook Chewjitsu Podcast on Instagram Chewjitsu Podcast on Threads Chewjitsu Podcast on Youtube   Connect with Derby City: Derby City Mixed Martial Arts Derby City Mixed Martial Arts on Facebook Derby City MMA on Instagram Derby City MMA on Threads Derby City Mixed Martial Arts on Youtube     Connect with other Chewy Jitsu Open Tournament: The Chewjitsu Open On Facebook Chewjitsu Open on Instagram Chewjistu Open on Threads --- Connect with Nick Lamagna www.nicknicknick.com Text Nick (516)540-5733 Connect on ALL Social Media and Podcast Platforms Here FREE Checklist on how to bring more value to your buyers  

Ep 465, pt 3: Guest Ryan Lambert gives his thoughts on Maine goalie Albin Boija, his potential and whether or not the Bruins have a real chance to sign him after college. To learn more about listener data and our privacy practices visit: https://www.audacyinc.com/privacy-policy Learn more about your ad choices. Visit https://podcastchoices.com/adchoices

With the increase in the public's attention to all aspects of brain health, neurologists need to understand their role in raising awareness, advocating for preventive strategies, and promoting brain health for all. To achieve brain health equity, neurologists must integrate culturally sensitive care approaches, develop adapted assessment tools, improve professional and public educational materials, and continually innovate interventions to meet the diverse needs of our communities. In this BONUS episode, Casey Albin, MD, speaks with Daniel José Correa, MD, MSc, FAAN and Rana R. Said, MD, FAAN, coauthors of the article “Bridging the Gap Between Brain Health Guidelines and Real-world Implementation” in the Continuum® June 2025 Disorders of CSF Dynamics issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Correa is the associate dean for community engagement and outreach and an associate professor of neurology at the Albert Einstein College of Medicine Division of Clinical Neurophysiology in the Saul Korey Department of Neurology at the Montefiore Medical Center, New York, New York. Dr. Said is a professor of pediatrics and neurology, the director of education, and an associate clinical chief in the division of pediatric neurology at the University of Texas Southwest Medical Center in Dallas, Texas. Additional Resources Read the article: Bridging the Gap Between Brain Health Guidelines and Real-world Implementation Subscribe to Continuum®: shop.lww.com/Continuum Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Guests: @NeuroDrCorrea, @RanaSaidMD Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. This exclusive Continuum Audio interview is available only to you, our subscribers. We hope you enjoy it. Thank you for listening. Dr Albin: Hi all, this is Dr Casey Albin. Today I'm interviewing Dr Daniel Correa and Dr Rana Said about their article on bridging the gap between brain health guidelines and real-world implementation, which they wrote with Dr Justin Jordan. This article appears in the June 2025 Continuum issue on disorders of CSF dynamics. Thank you both so much for joining us. I'd love to just start by having you guys introduce yourselves to our listeners. Rana, do you mind going first? Dr Said: Yeah, sure. Thanks, Casey. So, my name is Rana Said. I'm a professor of pediatrics and neurology at the University of Texas Southwestern Medical Center in Dallas. Most of my practice is pediatric epilepsy. I'm also the associate clinical chief and the director of education for our division. And in my newer role, I am the vice chair of the Brain Health Committee for the American Academy of Neurology. Dr Albin: Absolutely. So just the right person to talk about this. And Daniel, some of our listeners may know you already from the Brain and Life podcast, but please introduce yourself again. Dr Correa: Thank you so much, Casey for including us and then highlighting this article. So yes, as you said, I'm the editor and the cohost for the Brain and Life podcast. I do also work with Rana and all the great members of the Brain Health Initiative and committee within the AAN, but in my day-to-day at my institution, I'm an associate professor of neurology at the Albert Einstein College of Medicine in the Montefiore Health System. I do a mix of general neurology and epilepsy and with a portion of my time, I also work as an associate Dean at the Albert Einstein College of Medicine, supporting students and trainees with community engagement and outreach activities. Dr Albin: Excellent. Thank you guys both so much for taking the time to be here. You know, brain health has really become this core mission of the AAN. Many listeners probably know that it's actually even part of the AAN's mission statement, which is to enhance member career fulfillment and promote brain health for all. And I think a lot of us have this kind of, like, vague idea about what brain health is, but I'd love to just start by having a shared mental model. So, Rana, can you tell us what do you mean when you talk about brain health? Dr Said: Yeah, thanks for asking that question. And, you know, even as a group, we really took quite a while to solidify, like, what does that even mean? Really, the concept is that we're shifting from a disease-focused model, which we see whatever disorder comes in our doors, to a preventative approach, recognizing that there's a tremendous interconnectedness between our physical health, our mental health, cognitive and social health, you know, maintaining our optimal brain function. And another very important part of this is that it's across the entire lifespan. So hopefully that sort of solidifies how we are thinking about brain health. Dr Albin: Right. Daniel, anything else to add to that? Dr Correa: One thing I've really liked about this, you know, the evolution of the 2023 definition from the AAN is its highlight on it being a continuous state. We're not only just talking about prevention of injury and a neurologic condition, but then really optimizing our own health and our ability to engage in our communities afterwards, and that there's always an opportunity for improvement of our brain health. Dr Albin: I love that. And I really felt like in this article, you walked us through some tangible pillars that support the development and maintenance of this lifelong process of maintaining and developing brain health. And so, Daniel, I was wondering, you know, we could take probably the entire time just to talk about the five pillars that support brain health. But can you give us a pretty brief overview of what those are that you outlined in this article? Dr Correa: I mean, this was one of the biggest challenges and really bundling all the possibilities and the evidence that's out there and just getting a sense of practical movement forward. So, there are many organizations and groups out there that have formed pillars, whether we're calling them seven or eight, you know, the exact number can vary, but just to have something to stand on and move forward. We've bundled one of them as physical and sleep health. So really encouraging towards levels of activity and not taking it as, oh, that there's a set- you know, there are recommendations out there for amount of activity, but really looking at, can we challenge people to just start growing and moving forward at their current ability? Can we challenge people to look at their sleep health, see if there's an aspect to improve, and then reassess with time? We particularly highlight the importance of mental health, whether it's before a neurologic condition or a brain injury occurs or addressing the mental health comorbidities that may come along with neurologic conditions. Then there's of course the thing that everyone thinks about, I think, with brain health in terms of is cognitive health. And you know, I think that's the first place that really enters either our own minds or as we are observers of our elder individuals in our family. And more and more there has been the highlight on the need for social interconnectedness, community purpose. And this is what we include as a pillar of social health. And then across all types of neurologic potential injuries is really focusing on the area of brain injury. And so, I think the area that we've often been focused as neurologists, but also thinking of both the prevention along with the management of the condition or the injury after it occurs. Dr Albin: Rana, anything else to add to that? That's a fantastic overview. Dr Said: Daniel, thank you for- I mean, you just set it up so beautifully. I think the other thing that maybe would be important for people to understand is that as we're talking through a lot of these, these are individual. These sound like very individual-basis factors. But as part of the full conversation, we also have to understand that there are some factors that are not based on the individual, and then that leads to some of the other initiatives that we'll be talking about at the community and policy levels. So, for example, if an individual is living in an area with high air pollution. Yes, we want them to be healthy and exercise and sleep, but how do we modify those factors? What about lead leaching from our aging pipes or even infectious diseases? So, I think that outside of our pillars, this is sort of the next step is to understand what is also at large in our communities. Dr Albin: That's a really awesome point. I love that the article really does shine through and that there are these individual factors, and then there there's social factors, there's policy factors. I want to start just with that individual because I think so many of our patients probably know, like, stress management, exercise, sleep, all of that stuff is really important. But when I was reading your article, what was not so obvious to me was, what's the role that we as neurologists should play in advocating? And really more importantly, like, how should we do that? And again, it struck me that there are these kind of two issues at play. And one is that what Daniel was saying that, you know, a lot of our patients are coming because they have a problem, right? We are used to operating in this disease-based care, and there's just limited time, competing clinical demands. If they're not coming to talk about prevention, how do we bring that in? And so Rana, maybe I'll start with you just for that question, you know, for the patients who are seeing us with a disease complaint or they're coming for the management of a problem, how are you organizing this at the bedside to kind of factor in a little bit about that preventative brain health? Dr Said: You know, I think the most important thing at the bedside is, one, really identifying the modifiable risk factors. These have been well studied, we understand them. Hypertension, diabetes, smoking, weight management. And we know that these definitely are correlative. So is it our role just to talk about stroke, or should we talk about, how are you managing your blood pressure? Health education, if there was one major cornerstone, is elevating health literacy for everyone and understanding that patients value clear and concise information about brain health, about modifiable risk factors. And the corollary to that, of course, are what are the resources and services? I completely understand---I'm a practicing clinician---the constraints that we have at the bedside, be it in the hospital or in our clinics. And so being the source of information, how are we referring our families and individuals to social workers, community health worker support, and really partnering with them, food banks, injury prevention programs, patient advocacy organizations? I think those are really ways that we can meet the impacts that we're looking at the bedside that can feel very tangible and practical. Dr Albin: That's really excellent advice. And so, I'd like to ask a follow-up question. With your knowledge of this, trying to get more multidisciplinary buy-in from your clinic so that you really have the support to get these services that are so critically important. And how do you do that? Dr Said: Yeah, I think it's, one, being a champion. So, what does a champion mean? It means that somebody has to decide this is really important. And I think we all realize that we're not the only ones in the room who care about this. We're all in this, and we all care about it. But how do we champion it and carry it through? And so that's the first. Second you find your partnerships: your social workers, your case managers, your other colleagues. And then what is the first-level entry thing that you can do? So for example, I'm a pediatric epileptologist. One of the things we know is that in pediatric epilepsy, depression and anxiety are very strong comorbidities. So, before we get to the point where a child is in distress, every single one of our epilepsy patients who walks in the door over the age of twelve has an age-appropriate screener that is given to them in both English and Spanish. And we assess it and we determine stratifying risk. And then we have our social workers on the back end and we decide, is this a child who needs resources? Is this a child who needs to be walked to the emergency room, escorted? And anything in between. And I think that that was a just a very tangible example of, every single person can do this and ask about it. And through the development of dot phrases and clear protocols, it works really well. Dr Albin: I love that, the way that you're just being mindful. At every step of the way, we can help people towards this lifelong brain health. And Daniel, you work with an adult population. So I wonder, what are your tips for bringing this to a different patient population? Dr Correa: Well, I think---adult or child---one thing that we often are aware of with so many of the other things that we're doing in bedside or clinic room counseling, but we don't necessarily think of in this context of brain health, is, remember all the people in the room. So, at the bedside, whether it's in the ICU, discharge counseling, the initial admission, the whole family is often involved and really concerned about the active issue. But you can look for opportunities- we often try to counsel and support families about the importance of their own sleep and rest and highlighting it not just as being there for their family member, but highlighting it to them as a measure of their own improvement of their brain health. So, looking at ways where, one, I try to find, is there something I can do to support and educate the whole family about their brain health? And then- and with an epilepsy, or in many other situations, I try to look for one comorbidity that might be a pillar of brain health to address that maybe I wasn't already thinking. And then I consider, is there an additional thing that they wouldn't naturally connect to their epilepsy or their headaches that I can bring in for them to work on? You know, we can't often give people twelve different things to work on, and they'd just feel like, okay like, you have no realistic understanding of my life. But if we can just highlight on one, and remind them that there can be many more ways to improve their health and to follow up either with us as their neurologist or their future primary care doctors to address those additional needs. Again, I would really highlight the importance of a multidisciplinary approach and looking for opportunities. We've too often, I feel, relied on primary care as being the first line for addressing unmet social health needs. We know that so many people, once they have a neurologic condition or the potential, even, of a neurologic condition, they're concerned about dementia or something, they may view us, as their neurologist, as their most important provider. And if they don't have the resource of time and money to show up at other doctors, we may be the first one they're coming to. And so, tapping into your institution's resources and finding out, are there things that are available to the primary care services that for some reason we're not able to get on the inpatient side or the outpatient side? Referring to social workers and care workers and showing that our patients have an independent need, that they're not somehow getting captured by the primary care doctors. Dr Albin: I really love that. I think that we- just being more invested and just being ready to step into that role is really important. I was noticing in this article, you really call that being a brain health ambassador, being really mindful, and I will direct all of our listeners to Figure 3, which really captures what practitioners can do both at the bedside, within their local community, and even at the professional society level, to really advocate for policies that promote brain wellness. Rana, at the very beginning of this conversation, you noted, you know, this is not just an individual problem. This really is something that is a component of our policy and the structure of our local communities. I really loved in the article, there's a humility that this cannot be just a person-by-person bedside approach, that this is a little bit determined by the social determinants of health. And so, Rana, can you walk us through a little bit of what are the social determinants of health, and why are these so crucially important when we think about brain health for all? Dr Said: Yeah, social determinants of health are a really key factor that it looks at, what are the health factors that are environmental; for example, that are not directly like what your blood pressure is, what, you know, what your BMI is, that definitely impact our health outcomes. So, these include environmental things like where people are born, where they live, where they learn, work, play, worship, and age. It encompasses factors like your socioeconomic status, your education, the neighborhoods where you are living, definitely healthcare access. And then all of this is in a social and community context. We know that the impact of social determinants of health on brain health are profound for the entire lifespan and that- so, for example, if someone is from a disadvantaged background or that leads to chronic stress, they can have limited access to healthcare. They can have greater risk of exposure to, let's say, environmental toxins, and all of that will shape how their brain health is. Violence, for example. And so, as we think about how we're going to target and enhance brain health, we really have to understand that these are vulnerable populations, special high-risk populations, that often have a disproportionate burden of neurologic disorders. And by identifying them and then developing targeted interventions, it promotes health equity. And it really has to be done in looking at culturally- ethnocultural-sensitive healthcare education resources, thinking about culturally sensitive or adaptive assessment tools that work for different populations so that these guidelines that we have, that we've already identified as being so valuable, can be equitably applied, which is one crucial component of reducing brain health risk factors. And lastly, at the neighborhood level, this is where we really rely on our partnerships with community partners who really understand their constituents and they understand how to have the special conversations, how to enhance brain health through resource utilization. And so, this is another plug for policy and resources. Dr Albin: I love that. And thinking about the neighborhood and the policy levels and all the things that we have to do. Daniel, I'd like to ask you, is there anything else you would add? Dr Correa: Yeah, you know, so I really wanted to come back to this thing is that often and unfortunately, in the beginning understanding of social determinants of health, they're thought of as a positive or a negative factor, and often really negative. These are just facts. They're aspects about our community, our society, and some of them may be at the individual level. They're not at fault of any individual or community, or even our society. They're just the realities. And when someone has a factor that may predict a health disparity or an unmet social need---I wanted to come back to that concept and that term---one or two positive factors that are social determinants of health for that individual are unmet social needs. It's a point of promise. It's a potential to be addressed. And seeking ways to connect them with community services, social work, caregivers, these are ways where- that we can remove a barrier to, so that the possibility of the recommendations that we're used to doing, giving recommendations about medications and management, can be fully appreciated for that person. And the other aspect is, like brain health, this is a continuous state. The social determinants of health may be different for the child, the parent, and the elderly family member in the household, and there might be some that are shared across them. And when one of those individuals has a new medical illness or a new condition, a stroke, and now has a mobility limitation, that may change a social determinant of health for that person or for anyone else in the family, the other people now becoming caregivers. We're used to this. And for someone after a stroke or traumatic brain injury, now they have mobility changes. And so, we work on addressing those. But thinking on how those things now become a barrier for engaging with community and accessing things, something as simple as their pharmacy. Dr Albin: I hear a lot of “this is a fluid situation,” but there's hope here because these are places that we can intervene and that we can really champion brain health throughout this fluid situation. Which kind of brings me to what we're going to close out with, which is, I'm going to have you do a little thought exercise, which is that you find a magic lamp and a genie comes out. And we'll call this the brain health genie. The genie says that they are going to grant you one wish for the betterment of brain health. Daniel, I'll start with you. What is the one thing that you think could really move the needle on promoting and maintaining brain health? Dr Correa: I will jump on nutrition and food access. If we could somehow get rid of food insecurity and have access to whole and fresh foods for everyone, and people could go back to looking at opportunities from their ancestral and cultural experiences to cook and make whole-food recipes from their own cultures. Using something like the Mediterranean diet and the mind diet as a framework, but not looking at those as cultural barriers that we somehow all have to eat a certain way. So, I think that would really be the place I would go to first that would improve all of our brain health. Dr Albin: I love that. So, wholesome eating. Rana, how about you? One magic wish. Dr Said: I think traumatic brain injury prevention. I think it's so- it feels so within our reach, and it just always is so heart-hurting when you think that wearing helmets, using seatbelts, practicing safety in sports, gun safety---because we know unfortunately that in pediatric patients, firearm injury is the leading cause of traumatic brain injury. In our older patients, fall reduction. If we could figure out how to really disseminate the need for preventative measures, get everyone really on board, I think this is- the genie wouldn't have to work too hard to make that one come true. Dr Albin: I love that. As a neurointensivist, I definitely feel that TBI prevention. We could talk about this all day long. I really wish we had a longer bit of time, but I really would direct all of our listeners to this fantastic article where you give really practical advice. And so again, today I've been interviewing Drs Daniel Correa and Rana Said about their article on bridging the gap between brain health guidelines and real-world implementation, written with Dr Justin Jordan. This article appears in the most recent issue of Continuum on the disorders of CSF dynamics. Be sure to check out Continuum Audio episodes from this and other issues. And thank you so much for our listeners for joining us today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. We hope you've enjoyed this subscriber-exclusive interview. Thank you for listening.

Our friend Albin Sadar joins us to discuss his recent article "Some People Are Still Oblivious to the Obvious".The article criticizes modern gender norms as rejecting self-evident truths and urges Christians to actively defend traditional values.See omnystudio.com/listener for privacy information.

Childhood-onset hydrocephalus encompasses a wide range of disorders with varying clinical implications. There are numerous causes of symptomatic hydrocephalus in neonates, infants, and children, and each predicts the typical clinical course across the lifespan. Etiology and age of onset impact the lifelong management of individuals living with childhood-onset hydrocephalus. In this episode, Casey Albin, MD, speaks with Shenandoah Robinson, MD, FAANS, FAAP, FACS, author of the article “Childhood-onset Hydrocephalus” in the Continuum® June 2025 Disorders of CSF Dynamics issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Robinson is a professor of neurosurgery, neurology, and pediatrics at Johns Hopkins University School of Medicine in Baltimore, Maryland. Additional Resources Read the article: Childhood-onset Hydrocephalus Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hi, this is Dr Casey Albin. Today I'm interviewing Dr Shenandoah Robinson about her article on childhood onset hydrocephalus, which appears in the June 2025 Continuum issue on disorders of CSF dynamics. Dr Robinson, thank you so much for being here. Welcome to the podcast. I'd love to start by just having you briefly introduce yourself to our audience. Dr Robinson: I'm a pediatric neurosurgeon at Johns Hopkins, and I'm very fortunate to care for kids and children from the neonatal intensive care unit all the way up through young adulthood. And I have a strong interest in developing better treatments for hydrocephalus. Dr Albin: Absolutely. And this was a great article because I really do think that understanding how children with hydrocephalus are treated really does inform how we can care for them throughout the continuum of their lifespan. You know, I was shocked in reading your article about the scope of the problem for childhood onset hydrocephalus. Can you walk our listeners through what are the most common reasons why CSF diversion is needed in the pediatric population? Dr Robinson: For the United States, and Canada too, the most common reasons are spina bifida---so, a baby that's born with a myelomeningocele and then develops associated hydrocephalus---and then about equally as common is posthemorrhagic hydrocephalus of prematurity, congenital causes such as from aquaductal stenosis, and other genetic causes are less common. And then we also have kids that develop hydrocephalus after trauma or meningitis or tumors or other sort of acquired problems during childhood. Dr Albin: So, it's a really diverse and sort of heterogeneous causes that across sort of the, you know, the neonatal period all the way to, you know, young adulthood. And I'm sure that those etiologies really shift based on sort of the subgroup population that you're talking about. Dr Robinson: Yes, they definitely shift over time. Fortunately for our kids that are born with problems that raise concerns, such as myelomeningocele or if they're born preterm, they sort of declare themselves by the time they're a year old. So, if you're an adult provider, they should have defined themselves and it's unlikely that they will suddenly develop hydrocephalus as a teenager or older adult. Dr Albin: Totally makes sense. I think many of the listeners to this podcast are adult neurologists who are probably very familiar with external ventriculostomies for temporary CSF diversion, and with the more permanent ventricular peritoneal shines or ventricular atrial or plural shines that are needed when there's the need for permanent diversion. But you described in your article two procedures that provide temporary CSF diversion that I think many of our listeners are probably not as familiar with, which is the ventricular access devices and ventriculosubgaleal shunts. Can you briefly describe what those procedures provide? Who are the candidates for them? And then what complications neurologists may need to think about if they're consulted for comanagement in one of these complex patients? Dr Robinson: Well, the good thing is that if as an adult neurologist you encounter someone with, you know, residual tubing from one of these procedures, you are unlikely to need to do anything about it. So, we put in ventricular access device or ventriculosubgaleal shunts, usually in newborns or infants. And sometimes when they no longer need the device, we just leave it in because that saves them an extra surgery. So, if you encounter one later on, it's most likely you won't need to do anything. Often if the baby goes on to show that they need a permanent shunt, we go ahead and put in that permanent shunt. We may or may not go back and take out the reservoir or the subgaleal shunt. The reservoir and subgaleal shunts are often put in the frontal location. Sometimes we'll put the permanent shunt in the occipital location and just leave the residual tubing there. So, you're very unlikely to need to intervene with a reservoir or subgaleal shunt if you encounter an older child or adult with that left in. We use these in the small babies because the external ventricular drains that we're very familiar with have a very high complication rate in this population. In the adult ICU, you often see these, and maybe there's, you know, a few percent risk of infection. It actually heads into 20 to 25% in our preterm infants and other newborns that require one of these devices for drainage. So, we try not to use external ventricular drains like we use in older patients. We use the internalized device: either the ventricular reservoir with a little area for us to tap every day, every other day; or the ventriculosubgaleal shunt, which diverts the spinal fluid to a pocket in the scalp. So, we use these in preterm infants that are too tiny for a permanent shunt. And for some of our babies that are born, for example, with an omphalocele, that we can't use their peritoneal cavity and so we need some temporizing device to manage their CSF. Dr Albin: Totally makes sense. And so just to clarify, I mean, this is a tube that's placed into the ventricles of the brain and then it's tunneled into the subgaleal space and the collection, the CSF, just builds up there, like? Dr Robinson: Yeah. Dr Albin: And over time either, you know, the baby will learn how to account for that extra CSF, and then I guess it's just reabsorbed? Dr Robinson: Yeah. When it's present, though, it looks like maybe, I don't know if you're familiar with like a tissue expander. There is this bubble of fluid under the scalp, but it's prominent, it can be several centimeters in diameter. Dr Albin: Wow, that's just absolutely fascinating. And I don't think I've ever had the opportunity to see this in clinical practice. I've really learned quite a bit about this. I assume that these children are going to go on to get some sort of permanent diversion. And then, you know, over time, those permanent shunts do create a lot of problems. And so, I was hoping you could kind of walk us through, you know, what are some of the things that you're seeing that you're concerned about? And then if you've just inherited a patient who had a shunt placed at, say, a different institution, how do you go about figuring out what kind of shunt it is and if they're still dependent on it? Dr Robinson: There's a few things that, fortunately, technology is helping with. So, it is much easier now for patients to get their images uploaded to image-sharing software, and then we can download their images into our institutional software, which is very helpful. Another option is that we are strongly encouraging our families to use a app such as HydroAssist that's available from the Hydrocephalus Association. So that's an app that goes on your phone, and you can upload the images from an MRI or a CT scan or x-rays from a shunt series. And then that you can take if you're traveling and you have to go to emergency department or you're establishing care with a new provider, you can have your information right there and not be under stress to remember it. It also has areas so you can record the type of valve. And all of our valves have pluses and minuses, they all tend to malfunction a little bit. And they can be particularly helpful with different types of hydrocephalus. I really doubt that we're going to narrow down from the fifteen or so valves we have access to now. And so, recording your valve type, the manufacturer as well as the setting, is very helpful when you're transferring care or if you're traveling and then have to, unfortunately, stop in the emergency department. Dr Albin: Yeah, I thought that was a really great pearl that, like, families now are empowered to sort of take control of understanding sort of the devices that they have, the settings that they're using. And what an incredible thing for providers who are going to care for these patients who, you know, unfortunately do end up in centers that are not their primary center. The other challenge that I find… I practice as a neurointensivist, and sometimes patients come in and they have a history of being shunt dependent and they present with a neurologic change. And I think that we as neurologists can be a little quick to blame the shunt and want the shunt to be tapped. And I was really struck in reading this article about the complexity of shunt taps. And I was hoping, you know, can you kind of walk us through what's involved and maybe why we should have a little bit of a higher threshold before just saying, ah, just have the neurosurgeons tap the shunt. Like, it's not that straightforward. Dr Robinson: And it may depend on the population you're caring for. So, when I was at a different institution, we actually published that there's about a 5% complication rate from shunt taps. And that may be- that was in pediatric patients. And again, that may be population dependent, but you can introduce infection to a perfectly clean shunt by doing a shunt tap. You can also cause an acute shunt malfunction. So that's why we tend to prefer that only neurosurgeons are doing shunt taps for evaluation of a shunt malfunction. There are times that, for example, our patients who are getting intrathecal chemotherapy or something have a CSF access device like an Ommaya reservoir, and other providers may tap that reservoir to instill medicine. But that's different than an evaluation, like, you're talking about somebody with a neurological change. And so, it is possible that if somebody has small ventricles or something, if you tap that shunt, you can take a marginally functioning shunt and turn it into an acute proximal malfunction, which is an emergency. Dr Albin: Absolutely. I think that's a fantastic pearl for us to take away from this. It's just that heightened level. And kind of on the flip side of that, you know, and I really- I do feel for us when we're trying to kind of, you know, make a case that it's, it's not the shunt. Many of our shunted patients also have a lot of neurologic complexity, which I think you really talked upon in this article. I mean, these are patients who have developmental cognitive delays and that they have epilepsy and that they're at risk for, you know, complications from prematurity, since that's a very common reason that patients are getting shunts. But from your experience as a neurosurgeon, what are some of the features that make you particularly concerned about shnut malfunction? And how do you sort of evaluate these patients when they come in with that altered mental status? Dr Robinson: It is challenging, especially for our patients that have, you know, some intellectual delay or other difficulties that make it hard for them to give an accurate history. Problem is, if they're sick and lethargic, they may not remember the symptoms that they had when they were sick. But sometimes there's hopefully there's a family member present that does remember and can say, oh, no, this is what they look like when they have a viral illness. And this is different from when they have the shot malfunction, which was projectile emesis, not associated with a fever. It's rare to have a fever with a shunt malfunction, although shunt infection often presents with malfunction. So, it's not completely exclusionary. We often look at the imaging, but it's taking the whole picture together. Some of the common other diagnoses we see are severe constipation that can decrease the drainage from the shunt and even cause papilledema in some people. So, we look at that as well on the shunt series. It's very important to have the shunt series if you're concerned about shunt malfunction or- the shunt tubing is good. It tends to last maybe 20to 25 years before it starts to degrade. And so, you may have had a functioning shunt for decades and it worked well and you're very dependent on it, and then it breaks and you become ill. But on the flip side, we have patients that have had a broken shunt for years, they just didn't know about it. And we don't want to jump in and operate on them and then cause complexities. And so, it is a challenge to sort out. The simplest thing is obviously if they come in and their ventricles are significantly larger, and that goes along with a several-hour or a couple-day deterioration, that's a little more clear-cut. Dr Albin: Absolutely. And you talked about this shunt series. What other imaging- and, sort of maybe walk us through, what's involved in a shunt series, what are you looking at? And then what other imaging is sort of your preferred method for evaluating these patients? Dr Robinson: In adult patients, the shunt series is the x-ray from the entire shunt. And so, if they have an atrial shunt, that would be skull x-ray plus a chest x-ray; or the shunt ends in the perineal cavity, it goes to the perineum. And we're looking for continuity. We're looking for the- sometimes as people grow and age, the ventricular catheter can pull out of the ventricle. So, we're looking to make sure that the ventricular catheter is in an optimal position relative to the skull. We can also look at the valve setting to see the type of valve. So, that can also be helpful as well. And then in terms of additional imaging, a CT scan or an MRI is helpful. If you don't know what type of valve they have, they should not, ideally, go in the MRI scanner. We like to know what their setting is before they go in the MRI because we're going to have to reset the valve after they come out of the MRI if it's a programmable valve. Dr Albin: This is fantastic. I've heard several pearls. So, one is that with the shunt series, which, am I correct in understanding those are just plain X-rays? Dr Robinson: Yes. Dr Albin: Right. Then we can look for constipation, and that might be actually something really serious in a pediatric patient that could clue us in that they could actually be developing hydrocephalus or increased ICP just because of the abdominal pressure. And then that we need to be mindful of what are the stunt settings before we expose anyone to the MRI machine. Is that two good takeaways from all of this? Dr Robinson: Yes. And it's very rare that there'll be an MRI tech that will allow a patient with a valve in the MRI without knowing what it is. So, they have their job security that way. But yeah, if you're not sure, just go ahead and get the CT. Obviously, in our younger kids, we're trying to avoid CT scans. But if you're weighing off trying to decide if somebody has a shunt malfunction versus, you know, waiting 12 or 24 hours for an MRI, go ahead and get the CT. Dr Albin: Absolutely. I love it. Those are things I'm going to take with me for this. I have one more question about these shunts. So, every now and then, and I think you started to touch on this, we will get a shunt series and we'll see that the catheter is fractured. Do the patients develop little- like, a tract that continues to allow diversion even though the catheter is fractured? Dr Robinson: Yes. So, they can develop scar tissue around, and some people have more scar tissue than others. You'll even see that sometimes, say, the catheter has fractured and we'll take out that old fractured tubing and put in new tubing on the other side. But if you go and palpate their neck or chest, you'll still feel that tract is there because it calcifies along the tract. Some patients drain through that calcified tract for weeks or months without symptoms, and then it can occlude off. So, we don't consider it a reliable pathway. It's also not a reliable pathway if you're positioned prone in the OR. So some of our orthopedic colleagues, for example, if they go to do a spine fusion, we like to confirm that the shunt is working before you undergo that long anesthesia, but also that you're going to be positioned prone and you could potentially- you know, the pressure could occlude that track that normally is open. Dr Albin: This is fantastic. I feel like I've gotten everything I've ever wanted to know about shunts and all of their complications in this, which is, you know, this is really difficult. And I think that because we are not trained to put these in, sometimes we see them and we just say, oh, it's fractured that must be a malfunction. But it's good to know that sometimes those patients can drain through, you know, a sort of scarred-down tract, but that it may not be nearly as reliable as when they have the tubing in place. Another really good thing that I'm going to put in my back pocket for the next time I see a patient with a potential shunt malfunction. Dr Robinson: And we do have some patients that the tubing is fractured years ago and they don't need it repaired, and that totally can be challenging when they then transfer to your practice for follow-up care. We tend to follow those patients very closely, both our clinic visits as well as having them seen by ophthalmology. So, there are teenagers and young adults out there that have… their own system has recovered and they are no longer shunt-dependent; and they may have a broken shunt and not actually be using that track, but they usually have had fairly intensive follow up to prove that they're not shunt-dependent. And we still have a healthy respect there that, you know, if they start to get a headache, we're going to take that quite seriously as opposed to, you know, some of our shunt patients, about 10 to 20%, have chronic headaches that are not shunt-related. So, not everybody who has a headache and has a shunt has a shunt malfunction. It's tough. Dr Albin: This is really tough. That actually brings me to sort of the last clinical scenario that I was hoping we could get your perspective on. And I think this would be of great interest to neurologists, especially in the context that these children may develop headaches that have nothing to do with the shunt. I'd like to sort of give you this hypothetical case that I'm a neurologist seeing a patient in clinic and it's a teenager, maybe a young adult, and they had a shunt placed early in childhood. They've done really well. And they've come to me for management of a new headache. And, you know, as part of this workup, their primary care provider had ordered an MRI. And, you know, I look at the MRI, and I don't think that the ventricles look really enlarged. They don't look overdrained. Is having an MRI that looks pretty okay, is that enough to exonerate the shunt in this situation? Dr Robinson: In most cases it is. The one time that we don't see a substantial change in the ventricles is if we have a pseudocyst in the abdomen. The ventricles cannot enlarge initially, and then later on they might enlarge. So, we see that sometimes that somebody will come in and their ventricles will be stable in size, but we're still a little bit suspicious. They've got this persistent headache. They may have, you know, some emesis or loss of appetite, loss of activity, and a slower presentation than you would get with an acute proximal malfunction. We can check an abdominal ultrasound for them. And sometimes, even though the ventricles haven't changed in size, they still have a malfunction because they have that distal pseudocyst. One of the questions that we ask our patients when we're establishing care, in addition to what valve type they have and what sort of their shunt history or other interventions such as endoscopic third ventriculostomy, is to ask if their ventricles enlarge when they have a shunt malfunction. There is a small fraction where they do not. They kind of have a stiff brain, if you will. And so, it's good to know that. That's one of the key factors is asking somebody, do the ventricles enlarge when they have a malfunction? If they have enlarged in the past, they're likely to enlarge again if they have a malfunction. But again, it's not 100%. So, in peds, 20% of the time the ventricles don't enlarge. So, in adults, I'm not that- you know, I don't know what percentage it is, but it's something to consider that you can have a stable ventricular size and still have a shunt malfunction. So, if your clinical judgment, you're just kind of, like, still uneasy, you know, respect that and maybe do a little more workup. That's why we so much want patients to establish care with somebody, whether it's a neurologist or a neurosurgeon or other provider in some areas that have fewer neurospecialists, but to establish care so that you all know what a change is for that patient. That's really important. Dr Albin: That's fantastic. So, to summarize that, it's really important to understand the patient's baseline and how they presented with prior shunt complications, if they've had some. That if they're coming in with a new headache that we don't have a baseline, so, we should just have a heightened level of awareness that, like, the shunt has a start and it has an end. And even if the start of the shunt in the brain looks okay, there still could be the potential for complications in the abdomen. And maybe the third thing I heard from that is that we should look for GI symptoms and sort of be aware of when there could be a complication in the abdomen as well. Does that all sound about right? Dr Robinson: And especially for our kids with spina bifida and for posthemorrhagic hydrocephalus are now adults, because the preterm infants are prone to necrotizing enterocolitis. And they may not have had surgery for it, but they still may have adhesions and other things that predispose them to develop pseudocysts over time. And then our individuals with spina bifida often have various abdominal surgeries and other procedures to help them manage their bowel and bladder function. And so that can also create adhesions that then predisposes to pseudocysts. So, we do have a healthy respect for that. In addition, it used to be---because we have gotten a little better with shunts over time---it used to be, like, when I was in training that you heard, you know, if you haven't had a shunt malfunction for 10 or 15 years, you must- you may no longer be dependent. And that's not really true. There are some people who outgrow their need for shunt dependence, but not everyone does outgrow it. And so, you can be 15, 20 years without a shunt revision and still be shunt-dependent. Dr Albin: Those are fantastic pearls. I think most of them, walking away with this, like, a very healthy respect for the fact that these are complex patients, which the shunt is one component of sort of the things that can go wrong and that we have to have a really healthy respect and really detailed investigation and sort of take the big picture. I really like that. Dr Robinson: Yeah, I know. I think it's- there's a very strong push amongst pediatric neurosurgery and a lot of the related, our colleagues in other areas, to develop multidisciplinary transition clinics and lifespan programs for these patients to help keep everything else optimized so that they're not coming in, for example, with seizures. But then you have to figure out if this is a seizure or a shunt; you know, if we can keep them on track, if we can keep them healthy in all their other dimensions, it makes it safer for them in terms of their shunt malfunction. Dr Albin: Absolutely. I love that, and just the multidisciplinary preventative aspect of trying to keep these patients well. So important. Dr Robinson, I really would like to thank you for your time. We're getting towards the end of our time together. Are there any other points about the article that you just are anxious that leave the readers with, or should I just direct them back to the fantastic review that you've put together on this topic? Dr Robinson: No, I think that we covered a lot of the high points. I think one of the really exciting things for hydrocephalus is that there's a lot of investigations into other options besides shunts for certain populations. We are seeing less hydrocephalus now with the fetal repair of the myelomeningocele, which is great. And we're trying to make inroads into posthemorrhagic hydrocephalus as well. So, there are a lot of great things on the horizon and, you know, hopefully someday we won't have the need to have these discussions so much for shunts. Dr Albin: I love it. I think that's really important. And all of those points were touched on the article. And so, I really invite our listeners to go and check out the article, where you can see sort of, like, how this is evolving in real time. Thank you, Dr Robinson. Please go and check out the childhood-onset hydrocephalus article, which appears in the most recent issue of Continuum on the disorders of CSF dynamics. And be sure to check out Continuum Audio episodes from this and other issues. Thank you again to our listeners for joining us today. And thank you, Dr Robinson. Dr Robinson: Thanks for having me. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

Join us on "Mental Matters" as we explore a personal story of transformation. Our guest opens up about their early struggles with addiction and the pivotal moments that led to a life of purpose and empowerment. Through candid conversations, we uncover the courage it takes to share one's story and the profound impact of communities like Alcoholics Anonymous. This episode is a testament to the power of personal growth and the incredible changes that happen when we face our fears head-on. Tune in for an inspiring journey of hope and resilience.Sponsors and important linksSubscribe to my newsletter: Mental Matters: Self-Improvement Edition | Asekho Toto | SubstackWant to be a guest on Mental Matters Hosted By Asekho Toto? Send Asekho Toto a message on PodMatch, here: www.podmatch.com/hostdetailpreview/1604880114184x746605277921114400You can buy my book on Stan Store: https://stan.store/Asekhototo/p/get-my-book-at-the-end-i-found-healingDonations are welcomed at paypal.me/asekhototoFDN Course www.anw5astrk.com/3DDP436/2CTPL/for a $650 off use this code aff1485fdn1

Albin shares a 4th of July Fun Facts Friday!See omnystudio.com/listener for privacy information.

Az év legnagyobb hazai irodalmi történése Tandori Dezső első, de egészen idáig kiadatlan verseskötetének, az Egyetlennek a megjelenése. A kötetet 1964-ben visszadobta a Magvető, Tandori pedig egy noteszba írta le kézírással a verseket, és küldte el Párizsba barátjának, Márffy Albinnak. Sok évtizeddel később és pár évvel Tandori halála után Márffy Albintól került vissza a kötet Magyarországra, hogy aztán végre megjelenhessen. Ebben hatalmas érdemei voltak a zenész Pándi Balázsnak, aki évek óta szenvedélyesen kutatja a Tandori-hagyatékot, és aki megkereste a kötet tervével Simon Mártont, aki éppen egy évvel ezelőtt indított el saját mikrokiadóját, az Okapi Presst. Velük beszélgettünk többek között Tandori alakjáról és életművéről, a kötet kalandos történetéről, hogy mit érdemes olvasni Tandoritól, és hogy mi értelme van mikrokiadót indítani. A tartalomból: 00:00 Pár verseskötet, amit említünk: a Szép versek 2025 a Magvetőtől és Csordás Kata kötete, A tékozlás öröme. És lesz élő podcastfelvételünk a Művészetek völgyében, hamarosan kiderülnek a részletek, például a vendégünk kiléte is! 3:15 A mai témánk Tandori Dezső első, de csak most megjelent kötete, az Egyetlen. És két vendégünk Pándi Balázs és Simon Márton. Rögtön az első kérdés: hogyan lett számukra ennyire fontos Tandori? És mi van a „sapkás, medvés, verebes” Tandorin túl? Nincs olyan típusú vers, amit el lehet képzelni, és Tandori ne írta volna már meg régen. Simon Márton emlegetett könyvesmagazinos cikke itt, a hivatkozott telexes cikk pedig itt olvasható. 9:36 Tandori lemezgyűjteményének története, az emlegetett Instagram, és a 444-es film Pándi Balázsról. És az összefüggés az absztinencia beállta és a Tandori-kutatás felerősödése között. 14:50 Márffy Albin és a véletlenek szerepe, és miért dobta vissza a Magvető 1964-ben a kéziratot? 24:00 Hogyan kerül egy ennyire kis kiadóhoz ez a kötet? 27:10 És hogyan lehet ennyit írni? Ez nem munka, hanem létezési forma. A rengeteg még nem ismert mű és a váratlan Tandori-Prince párhuzam. 35:36 Tandorit akkor érdemes olvasni, ha vágysz arra az elveszettségre, hogy csak visz a szöveg. Olyan szerző, aki alkalmas arra, hogy kimozdítson a valóságból. 42:57 Az Okapi Press története és az első év tapasztalatai. További tervek elfeledett művek kiadására, még ha ennél durvább valószínűleg már nem lesz. 60:50 Könyvek vendégeink ajánlásában: magyar festészeti kötetek, Kiss Tibor Noé - Olvadás, Roberto Bolaño - Vad nyomozók, Kemény István - Lovag Dulcinea, Fancsali Kinga - Nem a haláltól és Tandori Dezső - Még így sem. See omnystudio.com/listener for privacy information.

Albin Sadar Recaps the High Seas Hijinks on the Greek Cruise with EricSee omnystudio.com/listener for privacy information.

Albin joins us again this week for a Fun Facts Friday specialSee omnystudio.com/listener for privacy information.

Eric is interviews by Albin about his book Fish out of Water See omnystudio.com/listener for privacy information.

As part of the work of a behavior analyst, our ethical code requires us to explain our services in language that can be understood by clients and stakeholders. That holds doubly true when our clients speak a primary language other than English. And just busting out Google Translate isn't going to cut it! In the next entry in our Tutorial series, we break out the “how to” basics behind following best practices when working with an interpreter in delivering ABA services. From meaningful feedback and collaboration with an interpreter to ensuring a respectful flow of communication between all parties, we'll get your practice up to speed in no time. Well, at least in the time it takes to listen to this episode. This episode is available for 1.0 CULTURAL (ETHICS) CEU. Articles discussed this episode: Dowdy, A., Obidimalor, K.C., Tinanci, M., & Travers, J.C. (2021). Delivering culturally sound and high-quality behavior analytic services when working with an interpreter. Behavior Analysis: Research and Practice, 21, 51-64. doi: 10.1037/bar0000206 Vasquez, D.J., Lechago, S.A., & McCarville, M.J. (2024). Training behavior analysis graduate students to work with an interpreter. Behavior Analysis in Practice, 17, 1160-1174. doi: 10.1007/s40617-024-00938-w Hadziabdic, E., Albin, B., Heikkila, K., & Hjelm, K. (2014). Family members' experiences of the use of interpreters in healthcare. Primary Health Care Research and Development, 15, 156-169. doi: 10.1017/S1463423612000680 If you're interested in ordering CEs for listening to this episode, click here to go to the store page. You'll need to enter your name, BCBA #, and the two episode secret code words to complete the purchase. Email us at abainsidetrack@gmail.com for further assistance.

André Saint‑Albin celebrates "Looks Good On Me," a bold, club‑ready track released on Fedde Le Grand's Darklight imprint. Merging his contrasting Paris and Miami roots, his sound blends electro, progressive, big room, tech, and tribal elements. His mix is a feel good journey featuring original IDs, mashups, remixes, and cuts from Chris Lorenzo, Cloonee, FISHER, Green Velvet, James Hype, Martin Garrix, Steve Aoki and more!

Albin has left the stage, and the Dondins are on their way!! What can Georges and Jean-Michel do to patch this up and get Anne's family to love them? Well, a lot of shenanigans and the realization Jean-Michel is a terrible person. We're finishing up La Cage! Strap on your highest heels and join us! We are what we are!!

From the Archives The Atlantic Group Albin Z November 5, 2013  Welcome to the Atlantic Group Podcast. Our 7th  tradition states, “Every A.A. group ought to be fully self-supporting declining outside contributions.” Your contribution covers the expenses of our group, this podcast, and also that of our AA service structure. If you are an AA member who finds this podcast helpful, you can contribute using Venmo @AGTradition7 or Zelle at AGNYCINFO@gmail.com. Under what's it for, please write AG Podcast. Thank you for your support. For any questions, please e-mail: TuesdayAGNYC@gmail.com

Now that we've got some vacation time coming up in our near futures, we can sit back, relax…and make our listener's choose our episodes for us! That's right. We've got not one but TWO polls for listeners this month that will determine our summer Listener Choice topic as well as the next year's worth of Book Clubs. Excited to vote? Well, only patrons (at ANY level) will have access to the LC and Book Club polls so, if you haven't signed up yet, better get on it before everyone else chooses your books for you. Oh, and we have a full month of episodes all about IOA, working with an interpreter, and a whole grab bag of ethics! So, by vacation, we meant podcasting wearing sunglasses and shorts. Listener Choice Poll Book Club Poll Join the Patreon! Articles for June 2025 IOA Hausman, N.L., Javed, N., Bednar, M.K., Guell, M., Schaller, E., Nevill, R.E., & Kahng, S. (2022). Interobserver agreement: A preliminary investigation into how much is enough? Journal of Applied Behavior Analysis, 55, 357-368. doi: 10.1002/jaba.811 Garrity, M.L., Luiselli, J.K., & McCollum, S.A. (2008). Effects of a supervisory intervention on assessment of interobserver agreement by educational service providers. Behavioral Interventions, 23, 105-112. doi: 10.1002/bin.258 Hartz, R.M., Gould, K., Harper, J.M., & Luiselli, J.K. (2020). Assessing interobserver agreement (IOA) with procedural integrity: Evaluation of training methods among classroom instructors. Child and Family Behavior Therapy, 43, 1-12. doi: 10.1080/00168890.2020.1848404   Ethicstime! (Summer 2025 Grab Bag) Henderson, T.B., Ludden, B.J., & Romero, R.A. (2023). The ethical obligations, barriers, and solutions for interprofessional collaboration in the treatment of autistic individuals. Behavior Analysis in Practice, 16, 963-976. doi: 10.1007/s40617-023-00787-z Shreck, K., Ivy, J. W., & Zane, T. (2023). Teaching behavior analysts to address unethical behavior: Developing evidence based ethics instructional methods. Behavior Analysis in Practice. doi: 10.1007/s40617-023-00845-6 Logue, J. J., Hustyi, K. M., Toby, L M., & Outlaw, E. E. (2023). Promoting ethical and evidence-based practice through a panel review process: A case study in implementation research. Behavior Analysis in Practice. doi: 10.1007/s40617-023-00807-y   Tutorial: Working With an Interpreter Dowdy, A., Obidimalor, K.C., Tinanci, M., & Travers, J.C. (2021). Delivering culturally sound and high-quality behavior analytic services when working with an interpreter. Behavior Analysis: Research and Practice, 21, 51-64. doi: 10.1037/bar0000206 Vasquez, D.J., Lechago, S.A., & McCarville, M.J. (2024). Training behavior analysis graduate students to work with an interpreter. Behavior Analysis in Practice, 17, 1160-1174. doi: 10.1007/s40617-024-00938-w Hadziabdic, E., Albin, B., Heikkila, K., & Hjelm, K. (2014). Family members' experiences of the use of interpreters in healthcare. Primary Health Care Research and Development, 15, 156-169. doi: 10.1017/S1463423612000680

Albin Sadar returns with his latest articles including "Silence isn’t peace — it’s just surrender in slow motion." https://www.theblaze.com/columns/opinion/silence-isnt-peace-its-just-surrender-in-slow-motionSee omnystudio.com/listener for privacy information.