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I säsongens tredje avsnitt är virkesköparen Albin Jönsson och SCA Skogs marknads- och försäljningsdirektör Viktoria Lindstrand Munkby gäster. Albin berättar om kontakter med skogsägare och om stöttning till dem med bland annat tillsyn, planering och avverkning. Viktoria tar upp olika delar för att utveckla både medarbetare och marknadsverksamheten. Hon berättar också om viktiga marknadsfrågor för tillfället. Gästerna pratar även med våra poddvärdar om smidiga tjänster som SCA kan erbjuda för att underlätta skogsägandet och vid virkesaffärer.
Au programme de l'émission du 18 février : avec Julie Fette, universitaire, pour la seconde partie de son interviewJE VEUX UNE BD ET PAS N'IMPORTE LAQUELLE - chronique de Julia Segui - c'est à 09 min
El è cussegliader da facultad tar la Banca Raiffeisen Cadi. En ses temp liber viva Donat Albin per ses hobis e per il sport: «La fermezza da Trun èn las uniuns», di il president da l'Uniun da scursalets Trun. El raquinta da sias visiuns per la patinera, dal plaschair dal hockey e da ses viadis. En ses temp liber viva il cussegliader da facultad per sias uniuns: «La fermezza da Trun èn las uniuns. I sa mussa adina puspè che las uniuns tegnan ensemen per realisar projects en vischnanca», di il president da l'Uniun da scursalets Trun. La stagiun dal glatsch natiral a Pustget saja bain curta, dentant fitg intensiva - cun bels puncts culminants sportivs che gartegian grazia a bler idealissem dals commembers e da las commembras. La passiun per il hockey sin glatsch è vegnida messa en tgina a Donat Albin: «Cura ch'jau era pitschen, passentava mes bab praticamain tut l'enviern sin il glatsch. Oz è il hockey era mia gronda passiun, ed jau hai plaschair che nossa uniun giauda buna accoglientscha tar la populaziun». Donat Albin raschuna da sia passiun per il sport, da viadis, da sia musica, ma era dal plaschair vid sia lavur sco cussegliader da facultad ed insumma da sia vita privata e professiunala.
Many serious medical illnesses are associated with some degree of serum electrolyte abnormality, renal impairment, or both. The neurologist must determine if the patient's neurologic symptoms are related to the renal and electrolyte disturbances or whether a concurrent primary neurologic process is at play. In this episode, Casey Albin, MD, speaks with Eelco F. M. Wijdicks, MD, PhD, FAAN, FACP, FNCS, author of the article "Neurologic Manifestations of Renal and Electrolyte Disorders" in the Continuum® February 2026 Neurology of Systemic Disease issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Wijdicks is a professor of neurology and attending neurointensivist for the Neurosciences Intensive Care Unit at Mayo Clinic in Rochester, Minnesota. Additional Resources Read the article: Neurologic Manifestations of Renal and Electrolyte Disorders Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Guest: @EWijdicks Full episode transcript available here
Här kommer ett kärleksbrev till dig, min kära lyssnare. En påminnelse om att romantik behövs och att den gör livet lite härligare. Och kanske viktigast av allt: romantik är något vi kan öva på, lite mer, varje dag.I det här avsnittet hyllar jag de små gesterna som gör störst skillnad och hur vi uppvaktar varandra i vardagen. Med mig har jag några personer jag tycker väldigt mycket om som delar sina tankar om romantik, kärleksbrev, presenter och romantisk mat.Du hör Ebba Kleberg von Sydow, Hanaw Rashid, Martin Hansson och Adam & Albin. Ett avsnitt om pirr, värme och valet att om och om igen bjuda in mer romantik i livet.
Why are Americans getting sicker, heavier, and more stressed? In this episode of The Ana Cruz Show, Ana Cruz talks with Dr. Jaclyn Albin, a pediatrician and lifestyle medicine expert, about how food impacts our body, mind, and overall health. They discuss what's missing from the average American plate, how processed foods fuel chronic disease and depression, and practical tips to eat smarter every day. Follow @anacruzshow on Instagram and TikTok for more, and visit www.rollosdemujeres.com for updates and resources! Check out more of Dr. Albin's work on her LinkedIn page. Here are some delicious recipes from the culinarymedicine.org and a podcast on culinary medicine to learn more! Learn more about your ad choices. Visit megaphone.fm/adchoices
Margaux de Radio Campus Amiens à la rencontre de Louis Aguilar, un artiste musicien d'origine Lilloise, aujourd'hui implanté à Amiens. Il pioche son inspiration et ses créations de ses voyages aux Etats-Unis, où il a pris goût pour la musique country et folk. Il se fait appeler l'Americana du Nord de la France, crooner au coeur tendre. Il a été finaliste du Printemps de Bourges en 2015, iNOUÏS du Printemps de Bourges en 2016 et a collaboré avec Julien Doré, Pomme, Yuksek ou encore Albin de la Simone. Il fait également partie du duo Weekend Affair. Le 28 novembre 2025, il sort son 5ème album folk « First Impressions », dont on découvre les premiers titres avec Margaux dans ce nouvel épisode de Starting Block. https://linktr.ee/louisaguilarProduction: Radio Campus AmiensJanvier 2026———————Starting Block l'émissionLe rendez-vous hebdomadaire dédié aux musiques émergentes, à retrouver chaque vendredi sur les radios Campus de FranceChaque semaine, une Radio Campus vous invite à découvrir les groupes, les salles, évènements et projets musicaux émergents de son territoire à travers une exploration radiophonique de 30 minutes chrono. Du groove phocéen au punk orléanais via le rock ‘n roll de Rennes, prenez le bon départ à l'écoute de Radio Campus pour cette course d'orientation inédite dans le son d'aujourd'hui!Infos & podcasts www.radiocampus.fr/emission/starting-block------------------------------------------------------RADIO CAMPUS FRANCERadio Campus France est le réseau des radios associatives, libres, étudiantes et locales fédérant 30 radios partout en France.NOUS SUIVRE | FOLLOW USwww.radiocampus.frInsta @radio_campusNOUS ÉCOUTER | LISTENSite, webradios et podcastswww.radiocampus.frHébergé par Ausha. Visitez ausha.co/politique-de-confidentialite pour plus d'informations.
Margaux de Radio Campus Amiens à la rencontre de Louis Aguilar, un artiste musicien d'origine Lilloise, aujourd'hui implanté à Amiens. Il pioche son inspiration et ses créations de ses voyages aux Etats-Unis, où il a pris goût pour la musique country et folk. Il se fait appeler l'Americana du Nord de la France, crooner au coeur tendre. Il a été finaliste du Printemps de Bourges en 2015, iNOUÏS du Printemps de Bourges en 2016 et a collaboré avec Julien Doré, Pomme, Yuksek ou encore Albin de la Simone. Il fait également partie du duo Weekend Affair. Le 28 novembre 2025, il sort son 5ème album folk « First Impressions », dont on découvre les premiers titres avec Margaux dans ce nouvel épisode de Starting Block. https://linktr.ee/louisaguilar Production: Radio Campus Amiens Janvier 2026 ——————— Starting Block l'émission Le rendez-vous hebdomadaire dédié aux musiques émergentes, à retrouver chaque vendredi sur les radios Campus de France Chaque semaine, une Radio Campus vous invite à découvrir les groupes, les salles, évènements et projets musicaux émergents de son territoire à travers une exploration radiophonique de 30 minutes chrono. Du groove phocéen au punk orléanais via le rock ‘n roll de Rennes, prenez le bon départ à l'écoute de Radio Campus pour cette course d'orientation inédite dans le son d'aujourd'hui! Infos & podcasts www.radiocampus.fr/emission/starting-block ------------------------------------------------------ RADIO CAMPUS FRANCE Radio Campus France est le réseau des radios associatives, libres, étudiantes et locales fédérant 30 radios partout en France. NOUS SUIVRE | FOLLOW US www.radiocampus.fr Insta @radio_campus NOUS ÉCOUTER | LISTEN Site, webradios et podcasts www.radiocampus.fr
Andakterna den här veckan tar avstamp i de fem världsreligionerna på temat Kyla. Idag med prästen och psykoterapeuten Albin Tanke som utgår från sin kristna tro. Lyssna på alla avsnitt i Sveriges Radios app. Ur andakten:Det biter i kinderna. Hu, vad det är kallt. Jag vet inte hur det är hos dig, men här i Malmö där jag befinner mig blåser det iskalla vindar. Det gör det ofta denna tid på året. Det kan kännas som flera minusgrader trots att termometern står på plus. Och det värsta, det är nog när kylan når in innanför och det svårt att bli varm igen. Då gäller det att försöka hålla igång, att inte stelna till. Ja, jag försöker verkligen göra det jag kan för att hålla kylan stången och inte låta den ta över på olika sätt.När vinterns kyla tränger djupt in i märgen, då påminns jag om hur skört livet är. Visst är vintern en vacker tid, det tycker jag verkligen, men jag vet också att kylan är svår för många, för alla som inte kan stänga den ute, för alla som saknar möjligheter att hitta in i den trygga värmen. Jag är så tacksam för kyrkans olika verksamheter med t ex värmestugor som på många håll görs i samverkan med t ex Stadsmissionen; för att ge värme på olika sätt.Text:Joh 1:5Musik:Prague's Ballet av Marius Neset med Marius Neset, Leif Ove Andsnes och Louisa TuckProducent:Susanna Némethliv@sverigesradio.se
Clinicians and patients are in a state of prognostic uncertainty when they are unsure about the future course of an illness. By embracing uncertainty while cultivating prognostic awareness, neurologists can serve the critical role of supporting patients and families through the living and dying process. In this episode, Casey Albin, MD, speaks with Robert G. Holloway, MD, MPH, FAAN, author of the article "Managing Prognostic Uncertainty in Neurologic Disease" in the Continuum® December 2025 Neuropalliative Care issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Holloway is the Edward and Alma Vollertsen Rykenboer Chair and a professor of neurology in the department of neurology at the University of Rochester School of Medicine and Dentistry in Rochester, New York. Additional Resources Read the article: Managing Prognostic Uncertainty in Neurologic Disease Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hello, this is Dr Casey Albin. Today I'm interviewing Dr Bob Holloway about his article on managing prognostic uncertainty in neurologic disease, which appears in the December 2025 Continuum issue on neuropalliative care. Welcome to the podcast, and please introduce yourself to our audience. Dr Holloway: Hi, Casey, and thank you. Again, my name is Bob Holloway. I'm a clinician and neurologist up in Rochester, New York, and I've been doing both neurology and palliative care for many years. Dr Albin: Well, that's fantastic. And I really wanted to emphasize how much I really enjoyed reading this article. I know that we're going to get into some of the pearls that you offer, but I really want to tell the listeners, like, this is a great one to read because not only does it have sort of a philosophical take, but you also really provide some pragmatic tips of how we can help our patients manage this prognostic uncertainty. But maybe just tell us a little bit, what is prognostic uncertainty? Dr Holloway: Yes, thank you. Well, I think everyone has a sense of what prognostic uncertainty is. And it's just the uncertain futures that we as clinicians and our patients face. And I would just say that a way to summarize it is just, how do we manage the "not yet" of neurologic illness? Dr Albin: I love that. In neurologic illness, there is so much "not yet" and there are so many unknowns. And what I thought was really helpful about your article is you kind of give us three buckets in which we can think about the different types of uncertainty our patients are facing. What are those? Dr Holloway: This is, I think, an area that really is of interest to me, thinking about how to organize the prognostic "not yet" or that landscape. And one way I've tried to simplify it is to think about it as data-centered. And that's the world that we mostly live in as neurologists. That's the probability distributions. We also have kind of system-level uncertainties, and that's the uncertainties that our health system affords for our patients. And then we have, also, the patient-centered uncertainties and the uncertainties that those two prior categories cause for our patients. And that's a big uncertainty that we often don't address. Dr Albin: In reading the article, I was really struck by, we spend a lot of time thinking about data uncertainty. Can we get population-based research? Can we sort of look at prognostication scoring? I live in the ICU, and so we think a lot about these, like, scoring metrics and putting patients into buckets and helping us derive their care based on where their severity index is. And I'm sure that is true in many of the divisions of neurology. But what I did not really appreciate---and I thought you did a really fantastic job of kind of drawing our attention to---is there's a lot of system-centered uncertainty. Can you give us a little bit of examples, like, what is system-based uncertainty? Dr Holloway: I think system-level uncertainties just encompass the practical information gaps that may arise during our healthcare encounter. And a lot of, I think, the uncertainty that our patients face and families, they actually describe it as they feel captive by the uncertainty. And it's just the unknowns, not just what affords from the actual information about the disease and its prognosis in the future, but actually the level of the system, like, who's going to take care of them? How do you manage arranging for nurses to come into the home or all those practical-level uncertainties that the system provides that sometimes we don't do a good job of road-mapping for patients. Dr Albin: Absolutely. Because I feel like we have a little bit of a gap in that often as physicians. Like, the family asks, what will hospice at home look like? Well, you know, that's a question for case management. I think they'll come in and they'll tell you. But it strikes me that that's a real gap of my being able to walk patients through. Will they get home health care? Will they have transportation set up? Will there be a nurse who comes in to check? How often are they available? What's the cost going to be? All of these practical aspects of dealing with an illness that are beyond sort of our scope of knowledge, but probably have a huge practical impact to the patient. Dr Holloway: Without question, every encounter patients wonder about, that kind of future wish landscape that we- all our future-oriented desires and hopes. And so much of that is the practical aspects of our health system, which is often fragmented, kind of unknown, uncertain. And that's a huge source of uncertainty for our patients and families. And then that leads to many other uncertainties that we need to address. Dr Albin: Absolutely. I think another one that we, again, maybe don't spend quite as much time thinking about is this patient-level uncertainty. What's going on there? Dr Holloway: Yeah. So, I think patient-level uncertainty is that uncertainty that they experience when confronted with the two other types of uncertainty: the actual data-centered uncertainty and the system-level uncertainty. And that's that, kind of, very huge kind of uncertainty about what it means for them and their family and their future futures. And that's a source of huge stress and anxiety, and often frankly bordering on dread and fear for our patients and families. That actually gets into very levels of uncertainty that I would call maybe over even in the existential realm. Patient-level uncertainty in the actual existential questions or the fear and the dread or the kind of just unnerving aspect of it is actually even more important to patients than the scientific or data-centered uncertainty that we focus most of our attention on. Dr Albin: Yeah, I think this is, to me, was getting towards that, like, what does the patient care about and how are they coping with what is in many times a really dramatic shift in their life expectancy or morbidity expectations and this sort of radical renegotiation about what it means to have a neurologic illness? And how does that shift their thinking about who they are and their priorities in the world? Is that right? Dr Holloway: One thousand percent, and in fact, I will say---and I think is one of the main take home messages is that, you know, managing prognostic certainty is not an end in itself. It really is to help patients and families adaptively cope to their new and often harsh new reality, that we could help them adapt to their new normal. I think that is one of our main tasks as neurologists in our care teams is to help patients find and ultimately maybe achieve existential or spiritual or well-being even in their new health states. You know, that you certainly often see in the intensive care unit, but we often always see in the outpatient realm as well, and all our other diseases. Dr Albin: I think that's really hard to do. I think those conversations are incredibly difficult and trying to navigate where patients want to be, what would bring meaning, what would bring value. I think many of us struggle to have these pretty real and intense conversations with families about what really is important. And one of the things I really liked about this article is you kind of walk us through some steps that we as clinicians can take to get a little bit more comfortable. Maybe just walk us through, what are some of the things that you have found most helpful in trying to get families and patients to open up about what brings them meaning? How are they navigating this new, really uncertain time in their life? Dr Holloway: Yeah, so I do kind of have a ten-point recommendations of how to help cultivate a more integrated awareness of an uncertain future. I mean, I think the most important thing is actually just recognizing that embracing uncertainty as an amazingly remarkable cognitive tool. I mean, let's face it, uncertainty, when it happens with neurologic illness and disease, is often fearful. It's scary. It kind of changes our world. But on the flip side of it, it's a remarkable cognitive tool that actually can help us find new ways and new paths and new creativity. And I think we can use that kind of opposites to help our patients find new meaning in very difficult situations. So, thinking about uncertainty, kind of being courageous, leaning into it and recognizing that it does create anxieties and fear, but it also can kind of help create new solutions and new ideas to help people navigate. Dr Albin: I was hoping that maybe you could give us an example of, like, how would you do that? If a patient comes in and they're dealing with, you know, a new diagnosis and they're navigating this new uncertainty, what are some of the things that you ask to help them reframe that, to kind of take some of the good about that uncertainty? How do you navigate that? Dr Holloway: One of the other recommendations is actually just resetting the timeline and expectations for these conversations. That it shouldn't be expected that patients should accept their harsh new reality immediately, that it takes time in a trusted environment. And that there's this, like, oscillating nature of hopes and fears and dread, and you've just got to work with them over time. And with time, and once you understand who the patient and family are and understand where they find meaning and where they find, actually, joy in their life, or what actually brings them meaning, you can start recasting their futures into credible narratives in their kind of future landscape in ways that I think can help them enter into their new realities within the, you know, framework of disease management that you can offer them within your healthcare team or your healthcare system or wherever you are in the world and the available resources that you have to offer patients and families. Dr Albin: So, this sounds like a lot to me like active listening and really trying to get to know what is important to the family, what is important to the patient. And I guess probably just creating that space even in that busy clinical environment. Do I have that right? Dr Holloway: You can absolutely do that, right. You know, and honestly, active listening, we are challenged in our busy healthcare system to do this, but I think with the right listening skills and the appropriate ways of paying attention, you can definitely illuminate these possible, kind of future-oriented worlds for patients and help them navigate those new terrains with them. Frankly, I think that's a real new space for us in neurology. We don't think about and train how to create credible narratives for patients and families. We do it on the fly, but I think there's so much more work to do. How do you actually keep, you know, that best-case, worst-case, most likely credible narratives for patients that can help them adapt to their new realities and support them on their new journeys? Dr Albin: I love that best-case, worst-case, most likely case. I find that framework really helpful. But you talk in your article, it's not just about using that best case or worst case or most likely, but it's actually building some forecasting into that and having some real data to kind of support what you're saying. And there's a lot of growth towards actually becoming good as a medical forecaster. Can you describe a little bit, what did you mean by that? Dr Holloway: You're absolutely right. I think, actually, one of the skillsets of becoming and managing prognostic uncertainty is actually becoming a skilled medical forecaster. And it's a really tall order. So, we've got to be both good medical forecasters as well as helping patients adaptively cope to their new reality. But the good medical forecasting is actually now going more quantitative in thinking about the data that's available to help think about the important outcomes for patients and families and then predicting what their probabilities are so you can shape those futures around. So, yes, we do have to have an open mindset. We do have to actually look at the data that's available and actually think about, what are those long-term probabilities and outcomes? And we can be honest about those and even communicate them with families. But it's a really good skill set to have. Dr Albin: Yeah. This to me was a little bit about, how do you bring in the data knowledge that we try to get over time as we develop our expertise? You're developing not just a reliance on population-based data, but in my experience, I have seen this. And that sort of ability to kind of look at the patient in front of you, think about the big picture, but also a little bit about their unique medical comorbidities or prior life experiences. So, some of that database knowledge, and then bringing in and getting to know what is important to the patient. And so, sort of marrying that data-centric/patient-centric mindset. Dr Holloway: I love it. I guess the other way of saying that, too, is we need to think with precision, but communicate in narratives. And it's okay to gently put more precise estimates on our probability predictions with patients and families, what we think is the most likely case, best and worst case. Because patients and families want us to be more precise. We often shy away from it, but- so, it's okay to think in precisions, but we've got to put those in narratives in the most likely, best-, and worst-case scenarios. And don't be afraid if you think in terms of ninety percents, ten percents, fifty percents; most patients and families don't mind that. And what they're telling us is they actually want to hear that, if you are comfortable talking in those terms. Dr Albin: Yeah, absolutely. And giving a sense of the humility to say, like, this is my best guess based on medical data and my experience, I would say, but again, none of us have a crystal ball. And I do think families, as long as you're sort of couching your expectations into the sort of imperfect, but I'm doing my best, really appreciate that. Dr Holloway: They totally do all the time. Just say, I simply don't know for certain, but these are my best estimates. That's a good way of just phrasing that. Dr Albin: Yeah. So powerful. I don't know for certain. And then I wanted to just kind of close out, because there's this one term that you use that I thought was so interesting. And I wanted you to kind of tell our listeners a little bit about what you mean here, which is that, when you're actively open-minded, you're using this, quote, "dragonfly eyes." What do you mean by that? Dr Holloway: So, the dragonfly eyes, as you know, they can look at three sixty around them and they just, they move in all directions. Being actively open minded, I guess the biggest example I would say is, I don't like the term prognostic discordance, which means that there's a difference of subjective estimates of prognosis between patients and families. Being openly minded is actually embracing the potential information that the family has about prognosis and incorporating that into your estimates. So, I wouldn't say it's discordances, per se; I think being really actively open-minded is taking that all in and utilizing that as, you know what, they know more than you do about the patient and their loved ones, and they may have insights that can inform your best estimates of prognosis. So, the true dragonfly prognosticator actually is one who embraces and doesn't consider it discord, but considers it kind of new, useful information that I just need to weigh in so I can help the family in my best professional way in terms of developing a prognosis, whatever the condition may be. Dr Albin: I can imagine this is just so challenging and something that takes a long time to sort of perfect all of this. I think you say right below that, you need a growth mindset to do this because it is hard, and it's going to take an active participation and an active desire to get better at these conversations with our families. Dr Holloway: One thousand percent. You are so right that it takes time, effort, and not feeling like you're being challenged, but that actually you are including them in your entire body of knowledge, that you're just- it's part of all you're collecting. And even, I was on service last week, and I talked to residents and students about that very issue. It's like take their prognosis. And someone who came in, we thought CJB, very sad, tragic case, but we were thinking about what the future may look like and how do we actually work with the family who had very what we thought was unrealistic expectations. I said, well, no, this is not discordance. This is just useful information that we can take understand where they're coming from and incorporate that into the ways we want to build relationships, build trust, and over time we'll get to a point where we hopefully can work with them and have them have that fully integrated awareness of their future. Dr Albin: Yeah, that's beautiful. It really is this ongoing negotiation that really requires so much listening, understanding, and then obviously information and expertise about the data that we're presenting and the likelihood outcome, recognizing that there's a lot of uncertainty in all of this. Which, you know, again, this is kind of a 360 talk. At every level there is uncertainty, and that's what makes it so hard. Dr Holloway: Yeah, you're absolutely right. And actually, even in the article I kind of used the term radical uncertainty as that, no matter how resolvable all this uncertainty is, there will always still remain that radical element of our existence which we have to actually incorporate and be prepared for. And actually, not only of ourselves, but actually for patients and families and helping manage that. Using narratives and credible narratives and kind of ranges of possibilities is the best way to do that in a personalized way. Dr Albin: Well, this has been a fantastic conversation, and I know that we are running a bit short on time. So, as we wrap up and you think about this topic, are there any key take-home messages that you hope our listeners will walk away with? Dr Holloway: I think one main emphasis is that despite all the successes we feel we have in neurology, is that we all have to recognize that prognostic uncertainty is just going to increase in the future. But this is going to be for several reasons. One is that, just, the illness uncertainty of all of our great therapies are just going to be creating more uncertainty for the future. And precision medicine is paradoxical, and that actually it creates more uncertainty. So, I think we need to be prepared that we have to manage prognostic uncertainty better, because it's definitely going to increase. And two, it's what I said earlier, is that actually managing prognostic uncertainty is not an end to itself. It's actually helping patients and families adapt to their new and sometimes harsh new reality and actually help them to ultimately get to a place where maybe either their condition is neither dreaded, but actually they can accept it as their new reality and actually achieve some sort of existential well-being and existential health. I think that we have a lot more to emphasize in this area. And for far too long, we've focused on the certainty aspect of our field and not enough on the uncertainty in the world of medicine to help our patients and families. Dr Albin: And gosh, isn't there just so much uncertainty? And I think this has been beautiful. So, thank you again for coming and sharing your expertise. Dr Holloway: Thank you very much. It's been a pleasure. Dr Albin: For all of our listeners out there, this is a truly fantastic article, and I would just like to direct you to going to read the cases because not only do the cases offer a little bit of practical advice, but there's one that's actually sort of a philosophical discussion about, what does it mean to be alive and confront death? There's some beautiful artwork that's featured as well. So this is just a really unique article, and I'm excited for our listeners to have a chance to check it out. So again, today I've been interviewing Dr Bob Holloway about his article on managing prognostic uncertainty in neurologic disease, which appears in the December 2025 Continuum issue on neuropalliative care. Be sure to check out Continuum Audio episodes from this and other issues. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.
Andakterna den här veckan hålls av prästen och psykoterapeuten Albin Tanke från Malmö. Veckans tema är Livets källa. Lyssna på alla avsnitt i Sveriges Radios app. Ur andakten:”Var kommer jag ifrån och vart är jag på väg?” Sådana tankar har jag grubblat mycket på genom åren. ”Varför finns universum? Vem är Gud? Varför finns det överhuvudtaget liv?”Vissa tider har grubblerierna varit mer intensiva, andra tider har de knappt funnits alls. Men visst ligger de där i bakgrunden och utgör en slags fond till livet. Plötsligt så snurrar de upp för mig på nytt: ”Varför finns just jag? Varför blev jag inte någon annan?” Och så vidare.Någon gång har jag delat några av de här tankarna och frågeställningarna med mina vänner. Och då har de sagt: ”Men sluta grubbla sådär”, ”slappna av och lev”. Kanske har de rätt? Ibland låter jag tankarna slå knut på mig själv. Det är lite grann som att inte se skogen för alla träd. Att konstruera grubblerier som inte leder till nyfikenhet, kunskap eller kreativitet utan istället håller tillbaka och kanske till och med ger bränsle åt rädslan – nä, det vill jag ju inte fastna i.Såklart avspeglar dessa tankar också hur jag ibland tänker om Gud. Tankar som gör Gud till något främmande. Som om Gud är ett objekt som jag på distans måste förhålla mig till. Något som jag måste parera eller på olika sätt hitta till eller kanske till och med försöka uppnå? Som om jag är här och Gud är där – och kontakten mellan oss sval och kravfylld, tyngd, distanserad eller helt enkelt bara väldigt svåruppkopplad.Text:Apostlagärningarna 17:27-28Musik:The day before you came av Björn Ulvaeus och Benny Andersson med Benny AnderssonProducent:Susanna Némethliv@sverigesradio.se
Andakterna den här veckan hålls av prästen och psykoterapeuten Albin Tanke från Malmö. Veckans tema är Livets källa. Lyssna på alla avsnitt i Sveriges Radios app. Ur andakten:”Berätta för mig hur du har det?” – Denna mening kan öppna liv. Att någon av hjärtat vill höra vad jag har att säga, särskilt när livet är svårt – att få uttrycka sig och bli lyssnad på –det är något av det största.Jag vet inte hur många gånger jag har burit på svåra tankar och gått ältat på jobbiga problem. Och när jag har fått möjlighet att berätta – att beskriva vad jag tänker på – så kommer en befrielses kraft över mig. Det tar kanske inte bort själva problemet, det ger inte några snabba lösningar. Men själva delandet avlastar. Formulerandet skapar någon slags ordning som ger perspektiv. Det är något i berättandet som öppnar sig. Och det sker emellan oss, mellan berättaren och lyssnaren. Det öppnar för Guds skapande kraft.Text:Romarbrevet 12:15Musik:The day before you came av Björn Ulvaeus och Benny Andersson med Benny AnderssonProducent:Susanna Némethliv@sverigesradio.se
Andakterna den här veckan hålls av prästen och psykoterapeuten Albin Tanke från Malmö. Veckans tema är Livets källa. Lyssna på alla avsnitt i Sveriges Radios app. Ur andakten:Precis som våra kroppar behöver fysiskt vatten för att överleva, behöver våra själar en andlig källa för att finna mening, riktning och levande liv. Gud beskrivs ofta som denna urkälla, alltings ursprung och mål, ja källan till allt liv. I en värld fylld av stress, osäkerhet och strävan efter snabb tillfredsställelse på olika sätt kan vi lätt känna oss uttorkade och trötta. Kanske sätter jag hela min lit till att söka lyckan istället för att öppna för mening? Kanske får jag för mig att det går att söka tröst genom jobb och karriär eller genom materiella ting, istället för gemenskap och att låta relationer djupna? När tillvaron instrumentaliseras för mina begär istället för att se mina verkliga behov urholkas själen lätt. Och det lämnar mig mycket törstigare än förut. Jesus använde bilden av levande vatten när han talade med kvinnan vid Sykars brunn. Han erbjöd henne ett vatten som skulle bli en källa inom henne och som aldrig skulle göra henne törstig igen. Detta ”levande vatten” är en bild av den Heliga Andens kraft i våra liv. Text:Joh 4:10-15Musik:The day before you came av Björn Ulvaeus och Benny Andersson med Benny AnderssonProducent:Susanna Némethliv@sverigesradio.se
Andakterna den här veckan hålls av prästen och psykoterapeuten Albin Tanke från Malmö. Veckans tema är Livets källa. Lyssna på alla avsnitt i Sveriges Radios app. Ur andakten:Livet är inte alltid fyllt av glädje, kreativitet och skaparkraft. Plötsligt känner jag hur tomheten har blivit allt större och min glädje och längtan, ja, jag vet inte riktigt vart den har tagit vägen? Jag gör ungefär det jag brukar, vardagen rullar på, men det känns stumt på något sätt – som om riktningen inte riktigt vet vart den ska vända sig i mitt inre.Kanske känner du det också så ibland? Jag funderar på om min själ kanske inte riktigt har fått tillräckligt med näring på det senaste? Kanske ligger utmattningen där och lurar om hörnet?Våra själar är som en jordmån. Precis som fysisk jord behöver vatten för att ge liv och ge möjlighet för kornen att gro, träden att skjuta sina skott och öppna för möjlighet till frukt, så behöver också våra inre liv näring för att vi ska kunna växa i tro och kärlek. Bibeln använder ofta bilden av vatten som en symbol för liv, växtkraft och för Guds Ande. Kanske har jag glömt att tillåta min själs jord att bli vattnad med Guds levande vatten?Text:Jesaja 58:11Musik:The day before you came av Björn Ulvaeus och Benny Andersson med Benny AnderssonProducent:Susanna Némethliv@sverigesradio.se
Andakterna den här veckan hålls av prästen och psykoterapeuten Albin Tanke i Malmö. Temat är livets källa. Lyssna på alla avsnitt i Sveriges Radios app. Ur andakten:För en tid sedan blev jag inbjuden till en skola. Lärarna sa att eleverna hade så många frågor om Gud och att det nog var bäst att de bjöd in mig så att de fick ställa alla sina frågor till en präst. Det första jag tänkte i mitt stilla sinne var, men hur i all världen ska jag kunna svara på det? Hur ska jag med ett enkelt svar säga något om det största – det som inte med ord går att uttrycka? Men så tänkte jag, att visst, jag har ju en massa erfarenheter av det jag kallar gudsrelation och jag har många år bakom mig av möten med olika människor som på livets väg använt olika språkbruk, bilder och andra uttryck för att kunna tala, beskriva och fördjupa detta mysterium som vi kallar Gud – Så jag bestämde mig för att göra som lärarna föreslog – jag gick till skolan och beredde mig på att möta mellanstadieklassen alla frågor.Text:Jesaja 41:10Musik:The day before you came av Björn Ulvaeus och Benny Andersson med Benny AnderssonProducent:Susanna Némethliv@sverigesradio.se
Albin Ljudén startade bivaxfabriken i Gymnasiet ihop med en kompis på en budget som understiger många ehandlares kaffe-kassa. Men, rätt snart snurrade man 10 miljoner i omsättning. I dagens avsnitt pratar vi om hur man kom på idén med en bivaxduk, hur man gått på svårigheter i expansionen, hur Albin skalat upp och ned personalstyrkan samt hur man jobbar med expansion av produktsortimentet. Det är en äkta entreprenörsresa från en generation företagare som kommer spöa min egen inom några år. Enjoy!
ALBINÃO e GORILÃO são podcasters, RICARDINHO ACF é engenheiro mecânico e TIAGO TCAR é empresário. Eles vão bater um papo sobre tudo e sobre nada ao mesmo tempo. O Vilela teve que voltar das férias pra fazer esse episódio e provavelmente vai dormir o tempo todo.
When we talk about it, it makes you want to smile, but at the same time your eyes can fill with tears. And, speaking about saudade, I have a lot. Sunday is a saudade called Minas Gerais. Of the dreams I dreamed, of others that I didn't even allow myself to dream. In any corner of the world, saudade follows me, but especially in autumn. This sensation of things lost, past and future, grips me in the present. Sometimes it flows through my fingers, run over by the rush of everyday life. Saudade is what proves to us that we love, that we lived moments that were worthwhile, and that we have stories that deserve to be remembered. It's what connects us with what we were and who we were. Sunday is a day to remember everything you had, have and could have had. It's a time to be grateful, despite anything else. To me, this mix conveys a feeling not only of nostalgia but also the alchemy of saudade into a deep moment of rest and introspection. Watch the sun's rays come in through the window and smell the lush birth of a new day. I hope this selection brings warmth and spurs energy for your day. Words by @albindj Tracklist: U.e. - Froggy Explorer (28912, 2025) Ross Alexander - Fresh Ice Cream (Sucata Tapes, 2017) Stone - Achey (3 X L, 2025) Black Sabbath - Planet Caravan (Vertigo Records, 1970) Lars Barkuhn - Massai (Utopia Records, 2017) Ezra Feinberg - Soft Power ft. David Lackner (Tonal Union, 2024) Deux Filles - The Letter (Humbug, 1993) Joanne Robertson and Oliver Coates - Gown (AD 93, 2025) Unchained - Largo (A Colourful Storm, 2024) Purelink - First Iota ft. Angelina Nonaj (Peak Oil, 2025) Ark Noir - Anthocyanin (Molten Moods, 2023) Milton Nascimento e Jobim Trio - Dias Azuis (Blue Note, 2008) Skeleten - E Tusk (Self Release, 2025) Robert Rich - Whispers Of Eden (Hearts Of Space, 1994) Ludvig Cimbrelius - A Summer Yet To Come (Stereoscenic Records, 2024) LAVURN - Kill +1 (Self Release, 2025) Officer John - Handle (Wah Wah Wino, 2025) Skeleten - World Facing (Self Release, 2025) Sir Was - In The Midst (City Slang, 2017) conforter2 - How To Arrive (Nous'klaer Audio, 2023) Qur'an Shaheed - Dreams (Leaving Records, 2025) Herbert & Momoko - Need To Run (Accidental Records, 2025) Tom Skinner - The Maxim feat. Meshell Ndegeocello (International Anthem, 2025) Follow us on social media: @itsdelayed linktr.ee/delayed www.delayed.nyc www.facebook.com/itsdelayed www.instagram.com/_____delayed www.youtube.com/@_____delayed Contact us: info@delayed.nyc
Le film d'animation «La vie de château», réalisé par Clémence Madeleine-Perdrillat nous plonge dans les coulisses de ce monument. C'est l'histoire de Violette, 8 ans, qui doit aller vivre chez son oncle qui s'occupe de l'entretien du château. Avec Violette, on découvre un monde que les touristes ne voient jamais, celui d'une multitude de jardiniers, de fontainiers, d'agent d'entretien, de conservateurs qui s'affairent tous les jours pour préserver le patrimoine et l'histoire de ce château. - Rediffusion - Clémence Madeleine-Perdrillat, réalisatrice ; Gilles Bultez, chef du service des eaux et fontaines du château de Versailles et Pierre-Hippolyte Pénet, conservateur en chef du patrimoine au château de Versailles, étaient les invités de Nathalie Amar. ► Chronique Le hit de la semaine Kayz Loum nous fait découvrir «Aje» du duo Trinix qui a fait appel à la star française d'origine camerounaise Tayc et à l'artiste nigérian dont tout le monde parle Khaid ! ► Playlist du jour - Albin de la Simone et Alice on the Roof - Pourquoi on pleure. - Thylacine - Versailles.
Ville Tolvanen oli ja on digitalisaation edelläkävijä, hallitustyön kehittäjä, sijoittaja, omistaja, yrittäjä ja montaa muutakin. Kasvuyrittämistarinastaja asuntojen flippaustaustasta Taloudellisesta riippumattomuudesta, ajan jakamisesta Suomen ja Italian välillä Kuluttamisen tarpeista ja ihmiselämän sykleistä Etätyömahdollisuuden lisääntymisestä Lomataloidean syntymisestä Italian talon pohdituista hankintakriteereistä 5 vuotta tyhjänä olleen talon hankkimisesta ja remontoimisesta Talon Airbnb -vuokraamisesta; vuokrauskausi, asiakkaat Pankkiasioista ja rahoituksesta Toisen kohteen hankkimisesta Italiasta omaa elämäntapaa täydentämään Miten lähestyä kannattavuusajattelua, ansaintalogiikkaa, tuoton lähteistä Kolmannen, konseptia täydentävän kohteen tulosta mukaan Italiasta, riskeistä, kulttuurista, taloudesta Mahdollisista seuraavista askelista Insta agnes_and_albin, villetolvanen Nettisivut Ville Tolvanen Kiitos Ostan Asuntoja -sisällön mahdollistavalle sponsorille: Sijoitusasunnot.comostaa huolellisesti tutkimiaan kokonaisia kerrostaloja ja myy niistä valmiiksi vuokrattuja sijoitusasuntoja alle markkinahinnan. Mikäli etsit kassavirtapositiivista sijoitusasuntoa, liity sijoitusasunnot.cominsijoittajalistalle. Saat uusista kohteista kattavat myyntimateriaalit, joissa on asuntosijoittajan keskeisimmät tunnusluvut ja taloyhtiöiden tiedot selkeässä muodossa. Tarjolla on myös monimuotoista opastusta asuntosijoittamisen saloihin ja sisältöä Instassa, Facebookissa ja YouTubessa. Tutustu maksuttomaan Asuntosijoituskouluun Lue Sijoitusasunnot.com Blogista: Täydellinen vuokrailmoitus Kolmen kivijalan kasvattamisesta fokus yhteen Henri Neuvonen – Ostan Asuntoja Podcast #378 Kiitos Ostan Asuntoja -sisällön mahdollistavalle sponsorille: Vuokraustoiminta on tärkeä osa asuntosijoittamista. Ajan tai taitojen puuttuessa sen voi ulkoistaa OVV:n ammattilaiselle oli kyseessä sitten yksittäinen vuokra-asunto tai suursijoittajan asuntosalkku. Mikäli haluat vain keskittyä nauttimaan sijoitusasuntosi tuotosta, OVV Kokonaispalveluon sopiva vaihtoehto. Se on kaiken kattava "avaimet käteen" -konsepti, jossa OVV hoitaa kaiken vuokrasuhteen alusta loppuun myöntäen vuokratakuun koko vuokrasuhteen ajalle. Jos vuokralainen ei maksaisi vuokraansa, paikallinen OVV:n toimisto maksaa sen. Mikäli haluat apua hyvän vuokralaisen löytämisessä mutta hoidat vuokrasuhteen hallinnan itse, OVV Vuokravälityspalveluon oikea vaihtoehto. OVV palvelee ja neuvoo koko vuokrasuhteen ajan. OVV.com OVV Asuntopalvelut Instagram, Facebook- Marko Kaarton Blogi Marko Kaarto – Ostan Asuntoja Podcast #362 Jyväskylässä 250 asuntoa vuodessa Kim Kähkönen Osa 1 – Ostan Asuntoja Podcast #381 Kiitos Ostan Asuntoja -sisällön mahdollistavalle sponsorille: On kuulopuheita ja luulopuheita – ja sitten on oikeaa tietoa. Tätä tietoa ja tukea tarjoaa yli 34 000 jäsenen Suomen Vuokranantajat. Jäsenenä saat pääsyn muun muassa vuokranantajan työkaluihin, markkinatietoon, kattavaan tietopankkiin ja maksuttomaan lakineuvontaan. Luotettavaa osaamista. Liity jäseneksi ja suosittele kaverillesi: vuokranantajat.fi/liity Työpaikalle on kiva mennä Eemeli Karlsson Osa 1 – Ostan Asuntoja Podcast #310 Karlsson, Grahn, Parviainen, Huru – Kuukausipalaveri 26 – Ostan Asuntoja Podcast #384 Naapurikodit ostaa kokonaisia kiinteistöjä, jalostaa niistä viihtyisiä koteja ja jää omistajaksi nostamaan taloyhtiön arvoa. Osa asunnoista tarjotaan ostettavaksi. Haluatko asuntosijoittajana mukaan? Lue lisää naapurikodit .fi Naapurikoteja Anniina ja Jesse Parviainen Osa 1 – Ostan Asuntoja Podcast #283 Taloyhtiösäästö etsii säästö- ja lisätulokohteita taloyhtiöllesi. Palvelulla on tulostakuu. Maksat vain toteutuneesta taloudellisesta tuloksesta. Katso lisää taloyhtiosaasto.fi Uudistunut Vuokraovi on entistä houkuttelevampi vuokra-asuntojen markkinapaikka, jossa hyvää vuokrakotia etsivät vuokralaiset ja niitä tarjoavat vuokranantajat onnistuvat löytämään toisensa vieläkin helpommin. Vuokraovi.com Oletko ostamassa sijoitusasuntoa ja haluaisit markkinahinta-arvion ostettavasta asunnosta? Pankkiarvio.fi on edullinen ja helppo palvelu tähän tarpeeseen. Täytät vain omat tietosi ja lähetät sinulla olevat dokumentit. Markkinahinta-arvion avulla saat näkemystä siihen, ettet maksa asunnosta liikaa. Pankkiarvio.fi. Roni Arvonen – Asunnoista, Podcastista, X:stä, elämästä – Ostan Asuntoja Podcast #341 "Haluaisitko olla itsevarma asuntosijoittaja? Jo yli sata asuntosijoittajaa on käynyt Ostan Asuntoja Podistakin tutun Suvi Schwabin Asuntoasiaa -valmennuksen, jossa teoria muuttuu käytännön teoiksi henkilökohtaisen sparrauksen avulla. Lisätietoa viestimällä suvi@asuntoasiaa.fi tai Instassa Asuntoasiaa. Hoida kohteidesi ja vuokrasuhteidesi elinkaaren hallinta kasvavan asiakasjoukon tarpeiden pohjalta jatkuvasti kehittyvän Flatcon avulla. Yksityinen vuokranantaja voi käyttää Flatcon perustoimintoja maksutta ja lisäpalveluja joustavasti tarpeen mukaan. Johtava vuokranantajan verkkopalvelu Flatco.fi Seuraava Marko Kaarton järjestämä Ammattimainen asuntosijoittaminen -valmennus pidetään 7. helmikuuta 26 Valmentajina: Terhi Majasalmi, Mikko Sjögren, Mika Sucksdorff, Hannu Liitsola ja Marko Kaarto. Aika: 7.2.2026 klo 9.30–19.00 Paikka Teknopolis Ruoholahti Helsinki tai etäosallistuen Koodilla HURU Ostan Asuntoja seuraajista 10 nopeinta saa valmennuksen ennakkotarjoushintaan 339 €. asuntosijoituskirja.fi/valmennukset Marko Kaarto järjesti 13.9.25 Ammattimainen asuntosijoittaminen -valmennuksen. Osallistujia oli yli 220. Valmentajina Tellu Uotila, Joonas Orava, Heikki Pajunen, Heikki Karu ja Marko Kaarto. Tilaa noin 6,5 tunnin taltiointi hintaan 247 € koodilla HURU25. Linkki tässä tai Ostan Asuntoja Blogissa. Asuntosijoittamisen lumipalloefekti -kirjan tilauspaikka on asuntosijoituskirja.fi. Alekoodi TAVOITE. Sinustako flippaaja? -kirjan alekoodi on FLIPPAA. E-kirjat saa luettavaksi heti. Marko Kaarton esikoiskirja "Sijoita Asuntoihin! Aloita, kehity, vaurastu" löytyy samalta sivustolta. Koodilla ALOITA saat lisäalennuksen. Kirjat ja valmennukset ovat vuokratuloista verovähennyskelpoisia kuluja. Samalta sivustolta löytyy "Harrin privaattisparraus" -kuvaus ostanasuntoja@NostrVerified.com, Ostan Asuntoja X, Ostan Asuntoja Insta, Ostan Asuntoja FB, Ostan Asuntoja TikTok
Albin Berglund avslutar säsongen med att bli tvåa totalt på Novemberkåsan och det i hans första Kåsa. Samt två i juniorklassen på Gotland Grand National och fyra totalt. En otrolig säsongsavslutning efter en lite upp och ner SM-säsong. Vi pratar så klart om allt detta och mycket mer.Podcastvärd: Tomas Persson NordahlFoto: Kalle Lundstedt, Race Magazine
Tisdag! Albin har viktigare saker för sig, men kakorna är inte mindre brända för det. Det blir dejting, fyrverkerier och äldrevård.Har du ett skvaller som fler borde få höra? Maila det till kafferepetpod@gmail.comMissa inte vår månatliga systerpodd Cigarrummet. Bli prenumerant på www.underproduktion.se/cigarrummet2:45 - Dålig dejt i Surte7:40 - Kaninen och raketen 9:40 - Det är Karin Hosted on Acast. See acast.com/privacy for more information.
durée : 01:29:10 - Cabaret 42e Rue : Laurent Laffite et la troupe de " La Cage Aux Folles " - par : Laurent Valière - Laurent Lafitte incarnera Albin et Zaza, rôle créé par Michel Serrault, dans l'adaptation française de "La Cage aux Folles" au théâtre du Châtelet en décembre, mise en scène par Olivier Py. Aux côtés de la troupe, il nous présente en avant première une sélection d'extraits en version acoustique. - réalisé par : Céline Parfenoff Vous aimez ce podcast ? Pour écouter tous les autres épisodes sans limite, rendez-vous sur Radio France.
The Brian Bailey Show 11-04-25 Dwayne Harris, Charlotte Coach Tim Albin by Pirate Radio
EPISODE 235 Charlotte Head Coach Tim Albin previews the Charlotte-ECU game on Saturday by Pirate Radio 92.7FM Greenville
Fluent Fiction - Swedish: Rekindling Family Ties Among Stockholm's Masterpieces Find the full episode transcript, vocabulary words, and more:fluentfiction.com/sv/episode/2025-10-25-22-34-02-sv Story Transcript:Sv: Det var en krispig höstdag i Stockholm.En: It was a crisp autumn day in Stockholm.Sv: Träden utanför Nationalmuseum skiftade i gyllene och röda färger, som om de själva var konstverk.En: The trees outside the Nationalmuseum were changing to golden and red hues, as if they themselves were artworks.Sv: Sofia och Linnea gick långsamt genom museets magnifika salar.En: Sofia and Linnea walked slowly through the museum's magnificent halls.Sv: De skulle på en efterlängtad konstutställning.En: They were going to a long-awaited art exhibition.Sv: Sofia älskade konst.En: Sofia loved art.Sv: Det gav henne en känsla av ro och inspiration.En: It gave her a sense of peace and inspiration.Sv: Men den här dagen grumlades hennes glädje av oro.En: But this day, her joy was clouded by worry.Sv: "Linnea, tänk om vi stöter på Albin idag?"En: "Linnea, what if we run into Albin today?"Sv: viskade Sofia.En: whispered Sofia.Sv: Hennes blick flackade i oroliga mönster, speglande de intrikata målningarna omkring dem.En: Her glance flickered in worried patterns, mirroring the intricate paintings around them.Sv: "Du har velat återförenas med honom länge nu, Sofia," svarade Linnea mjukt och la en hand på Sofias axel.En: "You've been wanting to reunite with him for a long time now, Sofia," Linnea replied softly, placing a hand on Sofia's shoulder.Sv: "Kanske är det ett tecken."En: "Maybe it's a sign."Sv: Sofia nickade, men en tyngd vilade kvar i hennes bröst.En: Sofia nodded, but a weight lingered in her chest.Sv: Hennes bror Albin, som hon inte hade träffat på flera år, hade plötsligt dykt upp i hennes tankar denna dag.En: Her brother Albin, whom she had not seen for several years, had suddenly appeared in her thoughts today.Sv: Han bodde utomlands och deras relation hade varit kylig.En: He lived abroad and their relationship had been chilly.Sv: Gamla konflikter låg fortfarande och pyrde under ytan.En: Old conflicts were still simmering beneath the surface.Sv: Som de långsamt vandrade genom utställningen, de njöt av varje konstverk.En: As they slowly wandered through the exhibition, they enjoyed each artwork.Sv: Mitt bland färger och former såg de en bekant gestalt.En: Among the colors and shapes, they saw a familiar figure.Sv: Där stod Albin.En: There stood Albin.Sv: Han såg äldre ut, men de där igenkännande dragen fanns kvar.En: He looked older, but those recognizable features remained.Sv: "Sofia", uppmanade Linnea tyst och knuffade henne försiktigt framåt.En: "Sofia," Linnea urged quietly and nudged her gently forward.Sv: Sofia kände en hemsk blandning av förväntan och rädsla.En: Sofia felt a dreadful mix of anticipation and fear.Sv: Skulle hon säga hej?En: Should she say hello?Sv: Vad skulle hon säga?En: What would she say?Sv: Om han bara ignorerade henne?En: What if he just ignored her?Sv: Men denna plats, dessa konstverk, gav henne mod.En: But this place, these artworks, gave her courage.Sv: Hon satte kurs mot Albin.En: She set a course toward Albin.Sv: "Hej, Albin," sa hon, rösten skakig men tydlig.En: "Hello, Albin," she said, her voice shaky but clear.Sv: Albin vände sig långsamt och såg på henne.En: Albin turned slowly and looked at her.Sv: Blicken mjuknade något, men han såg också förbluffad ut.En: His gaze softened somewhat, but he also appeared astonished.Sv: "Sofia," svarade han till sist.En: "Sofia," he finally replied.Sv: De stod där mitt bland mästerverken, två syskon som delade mer än bara gener.En: They stood there among the masterpieces, two siblings sharing more than just genes.Sv: Orden kom först långsamt.En: The words came slowly at first.Sv: Samtalet blev snabbt uppriktigt och känsloladdat.En: The conversation quickly became sincere and emotional.Sv: Sofia talade om sina känslor, om saknaden och ängsligheten.En: Sofia talked about her feelings, about the longing and anxiety.Sv: Albin berättade om sitt liv utomlands, om varför han hållit sig undan.En: Albin spoke about his life abroad, about why he had stayed away.Sv: Det var svårt.En: It was difficult.Sv: För gamla sår läkte inte omedelbart.En: For old wounds do not heal immediately.Sv: Men där, bland konsten, hittade de en sällsynt gemensam mark.En: But there, among the art, they found a rare common ground.Sv: Insikten om att det som en gång verkade omöjligt att reparera kanske kunde bli helt.En: The realization that what once seemed impossible to repair could perhaps become whole.Sv: När Sofia och Linnea senare den kvällen lämnade museet, vilade ett lugn över Sofia.En: When Sofia and Linnea left the museum later that evening, a calm rested over Sofia.Sv: Hon hade gått igenom en storm av känslor, men hittade ett frö av hopp.En: She had gone through a storm of emotions, but found a seed of hope.Sv: Hennes mod belönades och hon kände sig starkare.En: Her courage was rewarded and she felt stronger.Sv: Människor förändras.En: People change.Sv: Förbindelser kan återuppbyggas.En: Connections can be rebuilt.Sv: Höstens färgglada löv viskade om nya början, och mitt i allt detta, fann Sofia en ny början med sin bror.En: The colorful autumn leaves whispered of new beginnings, and in the midst of it all, Sofia found a new beginning with her brother. Vocabulary Words:crisp: krispigautumn: hösthues: färgermagnificent: magnifikaclouded: grumladesflickered: flackadeintricate: intrikatalingered: vilade kvarabroad: utomlandschilly: kyligsimmering: pyrdewandered: vandradefamiliar: bekanturged: uppmanadedreadful: hemskanticipation: förväntanastonished: förbluffadmasterpieces: mästerverkensincere: uppriktigtanxiety: ängslighetheal: läkterare: sällsyntrealization: insiktenimpossible: omöjligtrepair: repareracalm: lugnstorm: stormseed: fröcourage: modrewarded: belönades
Dans la sélection musicale de Brice Albin : Pepe Oleka, Nikanor, Fanicko, First King feat Fanicko, OPA, OPA feat T.Gang, Vano Baby, Bobo Wé, Axel Merryl et Credo. Pour visionner les clips, cliquez sur les titres des chansons : Pepe Oleka - Sêbla koko Nikanor - Veux-tu m'épouser ? Fanicko - Origine First King feat Fanicko - Jago life OPA - Che Che Che OPA feat T.Gang le technicien - Pause Vano Baby - Fitè Bobo Wé - Mea culpa Axel Merryl feat Mia Guisse et Bass Thioung - Titulaire Credo - Dudedji Retrouvez la playlist officielle de RFI Musique.
Dans la sélection musicale de Brice Albin : Pepe Oleka, Nikanor, Fanicko, First King feat Fanicko, OPA, OPA feat T.Gang, Vano Baby, Bobo Wé, Axel Merryl et Credo. Pour visionner les clips, cliquez sur les titres des chansons : Pepe Oleka - Sêbla koko Nikanor - Veux-tu m'épouser ? Fanicko - Origine First King feat Fanicko - Jago life OPA - Che Che Che OPA feat T.Gang le technicien - Pause Vano Baby - Fitè Bobo Wé - Mea culpa Axel Merryl feat Mia Guisse et Bass Thioung - Titulaire Credo - Dudedji Retrouvez la playlist officielle de RFI Musique.
While genetic testing has replaced muscle biopsy in the diagnosis of many genetic myopathies, clinical assessment and the integration of clinical and laboratory findings remain key elements for the diagnosis and treatment of muscle diseases. In this episode, Casey Albin, MD, speaks with Margherita Milone, MD, PhD, FAAN, FANA, author of the article “A Pattern Recognition Approach to Myopathy” in the Continuum® October 2025 Muscle and Neuromuscular Junction Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Milone is a professor of neurology and the director of the Muscle Pathology Laboratory at Mayo Clinic College of Medicine and Science in Rochester, Minnesota. Additional Resources Read the article: A Pattern Recognition Approach to Myopathy Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hello, this is Dr Casey Albin. Today I'm interviewing Dr Margherita Milone on her article on a pattern recognition approach to myopathy, which appears in the October 2025 Continuum issue on muscle and neuromuscular junction disorders. Welcome to the podcast, Dr Milone. Thank you so much for joining us. I'll start off by having you introduce yourself to our listeners. Dr Milone: Hello Casey, thank you so much for this interview and for bringing the attention to the article on muscle diseases. So, I'm Margherita Milone. I'm one of the neuromuscular neurologists at Mayo Clinic in Rochester. I have been interested in muscle disorders since I was a neurology resident many years ago. Muscle diseases are the focus of my clinical practice and research interest. Dr Albin: Wonderful. Thank you so much. When I think about myopathies, I generally tend to think of three large buckets: the genetic myopathy, the inflammatory myopathies, and then the necrotizing myopathies. Is that a reasonable approach to conceptualizing these myopathies? Dr Milone: Yeah, the ideology of the myopathies can be quite broad. And yes, we have a large group of genetic muscle diseases, which are the most common. And then we have immune-mediated muscle diseases, which include inflammatory myopathies as well as some form of necrotizing myopathies. Then we have some metabolic myopathies, which could be acquired or could be genetic. And then there are muscle diseases that are due to toxins as well as to infection. Dr Albin: Wow. So, lots of different etiologies. And that really struck me about your article, is that these can present in really heterogeneous ways, and some of them don't really read the rule book. So, we have to have a really high level of suspicion, for someone who's coming in with weakness, to remember to think about a myopathy. One of the things that I like to do is try to take us through a little bit of a case to sort of walk us through how you would approach if someone comes in. So, let's say you get, you know, a forty-year-old woman, and she's presenting with several months of progressive weakness. And she says that even recently she's noted just a little bit of difficulty swallowing. It feels to her like things are getting stuck. What are some of the things when you are approaching the history that would help you tease this to a myopathy instead of so many other things that can cause a patient to be weak? Dr Milone: Yes. So, as you mentioned, people who have a muscle disease have the muscle weakness often, but the muscle weakness is not just specific for a muscle disease. Because you can have a mass weakness in somebody who has a neurogenic paralysis. The problem with diagnosis of muscle diseases is that patients with these disorders have a limited number of symptom and sign that does not match the large heterogeneity of the etiology. So, in someone who has weakness, that weakness could represent a muscle disease, could represent an anterior horn cell disease, could represent a defect of neuromuscular junction. The clinical history of weakness is not sufficient by itself to make you think about a muscle disease. You have to keep that in the differential diagnosis. But your examination will help in corroborating your suspicion of a muscle disease. Let's say if you have a patient, the patient that you described, with six months' history of progressive weakness, dysphagia, and that patient has normal reflexes, and the patient has no clinical evidence for muscle fatigability and no sensory loss, then the probability that that patient has a myopathy increases. Dr Albin: Ah, that's really helpful. I'm hearing a lot of it is actually the lack of other findings. In some ways it's asking, you know, have you experienced numbness and tingling? And if not, that's sort of eliminating that this might not be a neuropathy problem. And then again, that fatigability- obviously fatigability is not specific to a neuromuscular junction, but knowing that is a hallmark of myasthenia, the most common of neuromuscular disorders. Getting that off the table helps you say, okay, well, it's not a neuromuscular junction problem, perhaps. Now we have to think more about, is this a muscle problem itself? Are there any patterns that the patients describe? I have difficulty getting up from a chair, or I have difficulty brushing my hair. When I think of myopathies, I historically have thought of, sort of, more proximal weakness. Is that always true, or not so much? Dr Milone: Yeah. So, there are muscle diseases that involve predominantly proximal weakness. For example, the patient you mentioned earlier could have, for example, an autoimmune muscle disease, a necrotizing autoimmune myopathy; could have, perhaps, dermatomyositis if there are skin changes. But a patient with muscle disease can also present with a different pattern of weakness. So, myopathies can lead to this weakness, and foot drop myopathies can cause- can manifest with the weakness of the calf muscles. So, you may have a patient presenting to the clinic who has no the inability to stand on tiptoes, or you may have a patient who has just facial weakness, who has noted the difficulty sealing their lips on the glasses when they drink and experiencing some drooling in that setting, plus some hand weakness. So, the muscle involved in muscle diseases can vary depending on the underlying cause of the muscle disease. Dr Albin: That's really helpful. So, it really is really keeping an open mind and looking for some supporting features, whether it's bulbar involvement, extraocular eye muscle involvement; looking, you know, is it proximal, is it distal? And then remembering that any of those patterns can also be a muscle problem, even if sometimes we think of distal being more neuropathy and proximal myopathy. Really, there's a host of ranges for this. I really took that away from your article. This is, unfortunately, not just a neat way to box these. We really have to have that broad differential. Let me ask another question about your history. How often do you find that patients complain of, sort of, muscular cramping or muscle pain? And does that help you in terms of deciding what type of myopathy they may have? Dr Milone: Many patients with muscle disease have muscle pain. The muscle pain could signal a presence of inflammation in skeletal muscle, could be the result of overuse from a muscle that is not functioning normally. People who have myotonia experience muscle stiffness and muscle pain. Patients who have a metabolic myopathy usually have exercise-induced muscle pain. But, as we know, muscle pain is also very nonspecific, so we have to try to find out from the patient in what setting the pain specifically occurs. Dr Albin: That's really helpful. So, it's asking a little bit more details about the type of cramping that they have, the type of pain they may be experiencing, to help you refine that differential. Similarly, one of the things that I historically have always associated with myopathies is an elevation in the CK, or the creatinine kinase. How sensitive and specific is that, and how do you as the expert sort of take into account, you know, what their CK may be? Dr Milone: So, this is a very good point. And the elevation of creatine kinase can provide a clue that the patient has a muscle disease, but it is nonspecific for muscle disease because we know that elevation of creatine kinase can occur in the setting of a neurogenic process. For example, we can see elevation of the creatine kinase in patients who have ALS or in patients who have spinal muscular atrophy. And in these patients---for example, those with spinal muscular atrophy---the CK elevation can be also of significantly elevated up to a couple of thousand. Conversely, we can have muscle diseases where the CK elevation does not occur. Examples of these are some genetic muscle disease, but also some acquired muscle diseases. If we think of, for example, cases where inflammation in the muscle occurs in between muscle fibers, more in the interstitium of the muscle, that disease may not lead to significant elevation of the CK. Dr Albin: That's super helpful. So, I'm hearing you say CK may be helpful, but it's neither completely sensitive nor completely specific when we're thinking about myopathic disorders. Dr Milone: You are correct. Dr Albin: Great. So, coming back to our patients, you know, she says that she has this dysphasia. How do bulbar involvement or extraocular eye movement involvement, how do those help narrow your differential? And what sort of disorders are you thinking of for patients who may have that bulbar or extraocular muscle involvement? Dr Milone: Regarding dysphagia, that can occur in the setting of acquired myopathies relatively frequent; for example, in inclusion body myositis or in other forms of inflammatory myopathy. Your patient, I believe, was in their forties, so it's a little bit too young for inclusion body myositis. Involvement of the extraocular muscles is usually much more common in genetic muscle diseases and much less frequent in hereditary muscle disease. So, if there is involvement of the extraocular muscles, and if there is a dysphagia, and if there is a proximal weakness, you may think about oculopharyngeal muscular dystrophy, for example. But obviously, in a patient who has only six months of history, we have to pay attention of the degree of weakness the patient has developed since the symptom onset. Because if the degree of weakness is mild, yes, it could still be a genetic or could be an acquired disease. But if we have a patient who, in six months, from being normal became unable to climb stairs, then we worry much more about an acquired muscle disease. Dr Albin: That's really helpful. So, the time force of this is really important. And when you're trying to think about, do I put this in sort of a hereditary form of muscle disease, thinking more of an indolent core, something that's going to be slowly progressive versus one of those inflammatory or necrotizing pathologies, that's going to be a much more quick onset, rapidly progressive, Do I have that right? Dr Milone: In general, the statement is correct. They tend, acquired muscle disease, to have a faster course compared to a muscular dystrophy. But there are exceptions. There have been patients with immune mediated necrotizing myopathy who have been misdiagnosed as having limb-girdle muscular dystrophy just because the disease has been very slowly progressive, and vice versa. There may be some genetic muscle diseases that can present in a relatively fast way. And one of these is a lipid storage myopathy, where some patients may develop subacutely weakness, dysphagia, and even respiratory difficulties. Dr Albin: Again, I'm hearing you say that we really have to have an open mind that myopathies can present in a whole bunch of different ways with a bunch of different phenotypes. And so, keeping that in mind, once you suspect someone has a myopathy, looking at the testing from the EMG perspective and then maybe laboratory testing, how do you use that information to guide your work up? Dr Milone: The EMG has a crucial role in the diagnosis of muscle diseases. Because, as we said earlier, weakness could be the result of muscle disease or other form of neuromuscular disease. If the EMG study will show evidence of muscle disease supporting your diagnostic hypothesis, now you have to decide, is this an acquired muscle disease or is this a genetic muscle disease? If you think that, based on clinical history of, perhaps, subacute pores, it is more likely that the patient has an acquired muscle disease, then I would request a muscle biopsy. The muscle biopsy will look for structural abnormalities that could help in narrowing down the type of muscle disease that the patient has. Dr Albin: That's really helpful. When we're sending people to get muscle biopsies, are there any tips that you would give the listeners in terms of what site to biopsy or what site, maybe, not to biopsy? Dr Milone: This is a very important point. A muscle biopsy has the highest diagnostic yield if it's done in a muscle that is weak. And because muscle diseases can result in proximal or distal weakness, if your patient has distal weakness, you should really biopsy a distal muscle. However, we do not wish to biopsy a muscle that is too weak, because otherwise the biopsy sample will result just in fibrous and fatty connected tissue. So, we want to biopsy a muscle that has mild to moderate weakness. Dr Albin: Great. So, a little Goldilocks phenomenon: has to be some weak, but not too weak. You got to get just the right feature there. I love that. That's a really good pearl for our listeners to take. What about on the flip side? Let's say you don't think it's an acquired a muscular disease. How are you handling testing in that situation? Dr Milone: If you think the patient has a genetic muscle disease, you pay a lot of attention to the distribution of the weakness. Ask yourself, what is the best pattern that represent the patient's weakness? So, if I have a patient who has facial weakness, dysphagia, muscle cramping, and then on examination represent myotonia, then at that point we can go straight to a genetic test for myotonic dystrophy type one. Dr Albin: That's super helpful. Dr Milone: So, you request directly that generic test and wait for the result. If positive, you will have proof that your diagnostic hypothesis was correct. Dr Albin: You're using the genetic testing to confirm your hypothesis, not just sending a whole panel of them. You're really informing that testing based on the patient's pattern of weakness and the exam findings, and sometimes even the EMG findings as well. Is that correct? Dr Milone: You are correct, and ideally, yes. And this is true for certain muscle diseases. In addition to myotonic dystrophy type one, for example, if you have a patient who has fascial scapulohumeral muscular weakness, you can directly request a test for FSHD. So, the characterization of the clinical phenotype is crucial before selecting the genetic test for diagnosis. Dr Albin: Wonderful. Dr Milone: However, this is not always possible, because you may have a patient who has just a limb-girdle weakness, and the limb-girdle weakness can be limb-girdle muscular dystrophy. But we know that there are many, many types of limb-girdle muscular dystrophies. Therefore, the phenotype is not sufficient to request specific genetic tests for one specific form of a limb-girdle muscular dystrophy. And in those cases, more complex next-generation sequencing panels have a higher chance of providing the answer. Dr Albin: Got it, that makes sense. So, sometimes we're using a specific genetic test; sometimes, it is unfortunate that we just cannot narrow down to one disease that we might be looking for, and we may need a panel in that situation. Dr Milone: You are correct. Dr Albin: Fantastic. Well, as we wrap up, is there anything on the horizon for muscular disorders that you're really excited about? Dr Milone: Yes, there are a lot of exciting studies ongoing for gene therapy, gene editing. So, these studies are very promising for the treatment of genetic muscle disease, and I'm sure there will be therapists that will improve the patient's quality of life and the disease outcome. Dr Albin: It's really exciting. Well, thank you again. Today I've been interviewing Dr Margarita Malone on her article on a pattern recognition approach to myopathy, which appears in the October 2025 Continuum issue on muscle and neuromuscular junction disorders. Be sure to check out Continuum Audio episodes from this and other issues, and thank you to our listeners for joining us today. And thank you, Dr Milone. Dr Milone: Thank you, Casey. Very nice chatting with you about this. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.
durée : 00:58:47 - Le 13/14 - par : Bruno Duvic - Basé sur des faits réels peu connus, La Conspiration raconte les débuts d'un coup d'État qui a failli renverser la démocratie américaine. Vous aimez ce podcast ? Pour écouter tous les autres épisodes sans limite, rendez-vous sur Radio France.
In this episode, host Bill Calkins is joined by industry strategist and co-founder of A24 Consulting, Clint Albin, to discuss the artificial intelligence (AI) in the greenhouse and garden center industry. Bill and Clint explore the challenges faced by green industry businesses, including seasonal fluctuations and changing consumer behaviors and how AI can provide solutions to enhance customer engagement and streamline operations—what Clint refers to as “the front door and the overhead door”. The conversation emphasizes the importance of integrating AI thoughtfully and creatively into existing processes, the need for immediate customer feedback and some potential risks associated with AI implementation. Clint shares insights on future Green Profit article and podcast topics related to procurement and local marketing strategies, highlighting the significance of innovation in the horticulture sector. Some key takeaways from this episode: AI can empower businesses of all shapes and sizes. Our industry faces challenges due to seasonal business cycles. Customer visits to garden centers need to be increased. Immediate customer feedback is crucial for improvement. AI should not be applied to broken systems. The essence of abundance in AI can save time or make money. A policy on AI usage is necessary for each business. Understanding customer needs is always a key to success. This podcast supplements an article series in Green Profit magazine. Why You Need AI, No Matter Your Size (September 2025)
Fluent Fiction - Swedish: Rekindling Connections: A Stockholm Autumn Tale Find the full episode transcript, vocabulary words, and more:fluentfiction.com/sv/episode/2025-09-28-22-34-01-sv Story Transcript:Sv: I Gamla Stan, den charmiga delen av Stockholm, var höstmarknaden i full gång.En: In Gamla Stan, the charming part of Stockholm, the autumn market was in full swing.Sv: De smala gatorna var fyllda med folk som ville fynda bland stånden med handgjorda varor och traditionella svenska godsaker.En: The narrow streets were filled with people eager to bargain among the stalls with handmade goods and traditional Swedish delicacies.Sv: Lukten av brända mandlar och varm glögg hängde i luften.En: The scent of roasted almonds and warm glögg hung in the air.Sv: Mitt i allt detta, i ett litet kafé med mjuka ljus från stearinljus och doften av kanel i luften, satt Albin.En: Amidst all this, in a small café with soft candlelight and the aroma of cinnamon in the air, sat Albin.Sv: Han stirrade på sin kopp kaffe och tänkte på gamla tider.En: He stared at his cup of coffee and thought about old times.Sv: Albin hade alltid trivts här.En: Albin had always felt at home here.Sv: Kaféet var en plats fylld av minnen, särskilt de med Lina.En: The café was a place filled with memories, especially those with Lina.Sv: Han hade inte sett henne på evigheter, ändå fanns hon alltid i tankarna.En: He hadn't seen her in ages, yet she was always in his thoughts.Sv: Han visste inte om han skulle våga hoppas eller om det var för sent.En: He didn't know if he dared to hope or if it was too late.Sv: Plötsligt öppnades dörren, och där stod Lina.En: Suddenly, the door opened, and there stood Lina.Sv: Hennes leende var varmt men det fanns ett stänk av överraskning i hennes ögon när hon fick syn på Albin.En: Her smile was warm, but there was a hint of surprise in her eyes when she spotted Albin.Sv: Efter en kort tvekan gick hon fram till hans bord.En: After a brief hesitation, she approached his table.Sv: "Hej Albin", sade Lina och satte sig.En: "Hi Albin," said Lina and sat down.Sv: Hennes röst var lugn, avslappnad.En: Her voice was calm, relaxed.Sv: "Hej Lina", svarade Albin och försökte dölja sitt hjärtas acceleration.En: "Hi Lina," replied Albin, trying to conceal his heart's acceleration.Sv: "Det var länge sedan."En: "It's been a long time."Sv: Lina log.En: Lina smiled.Sv: "Ja, det var det.En: "Yes, it has.Sv: Jag är glad att se dig."En: I'm glad to see you."Sv: De pratade om vädret, höstens färger och minnen från deras tid tillsammans.En: They talked about the weather, the colors of fall, and memories from their time together.Sv: Men under de lätta samtalsämnena kände Albin tyngden av det som verkligen behövde sägas.En: But beneath the light conversation topics, Albin felt the weight of what truly needed to be said.Sv: "Lina", började Albin med en osäkerhet i rösten.En: "Lina," Albin began with uncertainty in his voice.Sv: "Jag har tänkt mycket på oss.En: "I've thought a lot about us.Sv: Jag saknar...En: I miss...Sv: oss."En: us."Sv: Lina betraktade honom med tankfulla ögon.En: Lina regarded him with thoughtful eyes.Sv: Hon var tyst en stund och Albin kände att det var nu eller aldrig.En: She was silent for a moment, and Albin felt that it was now or never.Sv: "Jag gjorde misstag", fortsatte han.En: "I made mistakes," he continued.Sv: "Och jag vill att du ska veta att jag alltid har ångrat att jag lät det ta slut så."En: "And I want you to know that I've always regretted letting it end that way."Sv: Lina lyssnade, hennes leende var nu mildare, nästan sorgset.En: Lina listened, her smile now gentler, almost sorrowful.Sv: "Albin", sade hon försiktigt.En: "Albin," she said cautiously.Sv: "Jag har gått vidare.En: "I've moved on.Sv: Jag är lycklig där jag är nu.En: I'm happy where I am now.Sv: Men jag håller inte fast vid gammal smärta längre."En: But I'm no longer holding onto old pain."Sv: Albin nickade med en viss besvikelse men också insikt.En: Albin nodded with a certain disappointment but also understanding.Sv: "Jag förstår, jag vill bara säga förlåt.En: "I understand, I just wanted to say sorry.Sv: Och kanske, om möjligt, att vi kan vara vänner igen?"En: And maybe, if possible, that we could be friends again?"Sv: Lina tog hans hand.En: Lina took his hand.Sv: Kaféet omkring dem var till brädden fyllt med ljudet av marknadens glädje.En: The café around them was filled to the brim with the sounds of the market's joy.Sv: "Ja, jag skulle gilla det.En: "Yes, I would like that.Sv: Vänner är vi alltid."En: Friends we are, always."Sv: De satt där en stund till, pratade och skrattade precis som gamla vänner.En: They sat there for a while longer, talking and laughing just like old friends.Sv: Albin kände en vikt lätta från hans hjärta.En: Albin felt a weight lift from his heart.Sv: Han visste att han kunde gå vidare nu, precis som Lina hade gjort.En: He knew he could move on now, just like Lina had.Sv: När de lämnade kaféet, skildes de åt på gatan.En: As they left the café, they parted ways on the street.Sv: Höstsolen kastade långa skuggor på de gamla kullerstensgatorna.En: The autumn sun cast long shadows on the old cobblestone streets.Sv: Livet rörde sig vidare, och det gjorde de också, fri från det förflutnas kedjor, redo att omfamna sina egna nya äventyr.En: Life moved on, and so did they, free from the chains of the past, ready to embrace their own new adventures. Vocabulary Words:charming: charmigaautumn: höstbargain: fyndadelicacies: godsakeraroma: doftenmemories: minnensurprise: överraskninghesitation: tvekanconceal: döljaacceleration: accelerationregret: ångratpain: smärtasorrowful: sorgsetembrace: omfamnacandles: stearinljusregarded: betraktadethoughtful: tankfullchains: kedjorcobblestone: kullerstensgatorbrim: bräddenfill: fyllteager: ivrigstall: ståndglimpse: blickscent: luktenwarm: varmtdelicacies: godsakeralmonds: mandlarhesitant: tvekanderelaxed: avslappnad
Tics are movements or sounds that are quick, recurrent, and nonrhythmic. They fluctuate over time and can be involuntary or semivoluntary. Although behavioral therapy remains the first-line treatment, modifications to comprehensive behavioral intervention have been developed to make treatment more accessible. In this episode, Casey Albin, MD, speaks with Jessica Frey, MD, author of the article “Tourette Syndrome and Tic Disorders” in the Continuum® August 2025 Movement Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Frey is an assistant professor of neurology, Movement Disorders Fellowship Program Director, and Neurology Student Clerkship Director at the Rockefeller Neuroscience Institute in the department of neurology at West Virginia University in Morgantown, West Virginia. Additional Resources Read the article: Tourette Syndrome and Tic Disorders Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Transcript Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hi all, this is Dr Casey Albin. Today I'm interviewing Dr Jessica Frey about her article Tourette Syndrome and Tic Disorders, which appears in the August 2025 Continuum issue on movement disorders. Dr Frey, thank you so much for being here, and welcome to the podcast. I'd love for you to briefly introduce yourself to our audience. Dr Frey: Thank you for having me here today. My name is Jessica Frey, and I am a movement disorder specialist at West Virginia University. I'm also the movement disorder fellowship director, as well as the neurology clerkship student director. Dr Albin: Dr. Frey, I feel like this was one of the things I actually had no exposure to as a resident. For trainees that kind of want to get a better understanding of how these are managed, what kind of counseling you do, what kind of interventions you're using, how can they get a little bit more exposure? Dr Frey: That's a great question, and I actually had a similar experience to you. I did not see that many patients with Tourette syndrome while I was in my residency training. I got a lot more exposure during my fellowship training, and that's when I actually fell in love with that patient population, caring for them, seeing them be successful. I think it depends on the program that you're in. During the pediatric neurology rotation might be your best bet to getting exposure to patients with Tourette syndrome, since a lot of them are going to be diagnosed when they're quite young, and sometimes they'll even continue to follow through young adulthood in the pediatric neurology clinic. However, up to 20% of patients with Tourette syndrome will have persistent tics during adulthood. And so, I think it is important for neurology trainees to understand how to manage them, understand what resources are out there. So, if you have an interest in that, absolutely try to follow either in the pediatric neurology department, or if you have a movement disorder program that has a Tourette clinic or has a movement disorder specialist who has an interest in Tourette syndrome, definitely try to hang out with them. Get to know that patient population, and educate yourself as much as you're able to educate the patients as well. Dr Albin: Yeah, I think that's fantastic advice. You wrote a fantastic article, and it covers a lot of ground. And I think let's start at some of the basics. When I think of Tourette syndrome and tics, I think of Tourette syndrome having tics, but maybe not all patients who have tics have Tourette syndrome. And so, I was wondering, A, if you could confirm that's true; and then could you tell us a little bit about some of the diagnostic criteria for each of these conditions? Dr Frey: Sure. So, a tic is a phenomenological description. So basically, what you're seeing is a description of a motor or phonic tic, which is a particular type of movement disorder. Tourette syndrome is a very specific diagnosis, and the diagnostic criteria for Tourette syndrome at this point in time is that you need to have had at least one phonic tic and two or more motor tics over the course of at least a year before the age of eighteen. Dr Albin: Got it. So, there's certainly more specific and a lot more criteria for having Tourette syndrome. I was struck in reading your article how many myths there are surrounding Tourette syndrome and tic disorders kind of in general. What's known about the pathophysiology of Tourette syndrome, and what are some common misconceptions about patients who have this disorder? Dr Frey: Yeah, so I think that's a really excellent question because for so many years, Tourette syndrome and tic disorders in general were thought to be psychogenic in origin, even dating back to when they were first described. The history of Tourette syndrome is quite interesting in that, when Tourette---who, you know, it's named after---was working with Charcot, a lot of the initial descriptors were of actual case reports of patients who had more psychogenic descriptions, and eventually they became known as tic disorders as well. It wasn't until the discovery of Haldol and using Haldol as a treatment for tic disorders that people started to change their perception and say, okay, maybe there is actually a neurologic basis for Tourette syndrome. So, in terms of the pathophysiology, it's not completely known, but what we do know about it, we think that there is some sort of hyperactivity in the corticostriatal-thalamocortical circuits. And we think that because of this hyperactivity, it leads to the hyperactive movement disorder. We think similar circuitry is involved in conditions like OCD, or obsessive compulsive disorder; as well as ADHD, or attention deficit hyperactivity disorder. And because of that, we actually do tend to see an overlap between all three of these conditions in both individuals and families. Dr Albin: And hearing all of that, does this all come back to, sort of, dopamine and, sort of, behavioral motivation, or is it different than that? Dr Frey: It's probably more complex than just dopamine, but there is the thought that dopamine does play a role. And even one of the hypotheses regarding the pathophysiology is actually that these tics might start as habits, and then when the habits become more common, they actually reshape the dopaminergic pathways. And each time a tic occurs, there's a little bit of a dopaminergic reward. And so over time, that reshapes those hyperactive pathways and changes the actual circuitry of the brain, leading it to be not just a habit but part of their neurologic makeup. Dr Albin: It's fascinating to hear how that actually might play into our neural circuitry and, over time, rewire our brain. Fascinating. I mean, this is just so interesting how movement disorders play into such behavioral regulation and some comorbid conditions like ADHD and OCD. I thought it would be really helpful, maybe, to our listeners to kind of think through a case that I suspect is becoming more common. So, if it's okay with you, I'll present sort of a hypothetical. Dr Frey: Absolutely. Dr Albin: This is a father bringing in his seventeen-year-old daughter. She's coming into the clinic because she's been demonstrating, over the past four to six weeks, some jerking movement in her right arm. And it's happened multiple times a day. And it was a pretty sudden onset. She had not had any movement like this before, and then several weeks ago, started moving the right hand. And then it became even more disruptive: her right leg was involved, she had some scrunching her face. This is all happening at a time where she was dealing with some stress, maybe a little bit of applications around college that she was having a lot of anxiety about. How do you sort of approach this case if this is someone who comes to your office? Dr Frey: Sure. So, I think the first thing that you want to get is a good solid history, trying to understand, what is the origin of these abnormal movements and what led to the abnormal movements. Now, a key thing here is that in Tourette syndrome, and most physiologic tic syndromes, there's a pretty early onset. So, in Tourette syndrome, the expected age of onset is between the ages of five and seven years old. So, to have kind of acute new abnormal movements as a seventeen-year-old would be very unusual for a new-onset diagnosis of Tourette syndrome. However, there's a couple of things from the history that could help you. One would be, were there ever tics in the past? Because sometimes, when you think retrospectively, a lot of these patients might have had a simple eye-blinking tic or a coughing tic when they were a child. And perhaps they did have Tourette syndrome, a very mild case of it. But because the tics were never that pronounced, they never went to see anyone about it and it was never known that they had Tourette syndrome in the first place. If there is no history like that and the movements are completely new, out of the blue, of course you want to rule out anything acute that could be going on that could be causing that. Looking at the phenomenology of the movements can also be very helpful. When you're looking at abnormal tic movements, you would expect most cases of something like Tourette syndrome to occur first in the midline and go in a rostrocoidal distribution. So, you mostly see things happening with eye blinking, throat clearing, sniffling, neck snapping. These are some of the immediate tics that start to happen. We also usually start to see simple tics, as opposed to complex tics, at the beginning. Now, over the course of time, many patients do develop more complex tics that might involve the arms or the extremities, but that would be unusual to see this as a presenting feature of new-onset Tourette syndrome. Dr Albin: Got it. So, I'm hearing that the history really matters and that sometimes, like those, like, first-onset seizures, I imagine as a neurointensivist, we see a lot of patients who've had seizures who think that they're presenting the first time. And then we go back and we say, well, actually they have had some abnormal movements at night. Sounds like it's very similar with these movement disorders where you have to really go back and ask, well, was there some sniffling? Did they go through a phase where they were grunting frequently? Because I can imagine that many children make those behaviors, and that it may not have registered as something that was cause for concern. Dr Frey: Absolutely. Dr Albin: And then the other thing I heard from you was that the phenomenology really matters and that there is a typical presentation, starting from sort of the face and working the way down. And that can be really helpful. But in this case, the family is quite clear. No, no, no. She's never had movements like this before. This is- nothing like this. We promise you, did not go through a phase where she was coughing or blinking, or, this is all totally new. And the phenomenology, they say, no, no, she did not start with blinking. It definitely started in the arm and then progressed in its complex movements. So, knowing that about her, how does that sort of shape how you move forward with the diagnosis? Dr Frey: Yeah. So, really good question. And this is something that I think really peaked during the Covid-19 pandemic. We saw an influx of patients, especially teenage girls or young adult girls, who basically would come in and have these new, acute-onset, abnormal movements. We weren't sure what to call them initially. There was some discussion of calling them “explosive tic disorder” and things like that. A lot of these actually looked very similar to psychogenic nonepileptic seizures, where they would come into the emergency department and have many abnormal movements that were so severe, that they were having a “tic attack” and couldn't stop the abnormal movements from occurring. And we saw so many of these cases during the Covid-19 pandemic that it eventually became known as a distinctive diagnostic criteria with the name of “functional ticlike behavior”, or FTLB. When we think about functional ticlike behavior, we think that these tics are driven more by anxiety and stress. A lot of times, the backstory of these patients, they were in a very stressful situation, and that's when the abnormal movement started. So, a very similar kind of backstory to patients that might develop psychogenic nonepileptic seizures. These tics were popularized, for lack of a better term, via social media during the Covid-19 pandemic. One article is out there that even has called these functional ticlike behaviors as “a pandemic within a pandemic”, because there was such a strong showing of ticlike behavior in the clinics during the Covid-19 pandemic. Although social media was thought to play a big role in these functional ticlike behaviors, we think that there's probably a little bit more complexity and nuance to why these functional ticlike behaviors develop. There is probably a little bit of a genetic predisposition. There's probably some other psychosocial factors at play. And when we see cases like this, the best thing that you can do is educate your patients about the differences between functional ticlike behaviors and tics that we see associated with conditions like Tourette syndrome. And then the best types of treatments that we have seen thus far are treating any underlying stressors, if any of those exist, as well as cognitive behavioral therapy has been shown to be somewhat helpful. As the Covid-19 pandemic has wound down, we have actually seen a lot less cases in our clinic. And one reason we think is less stressors, less uncertainty for the future, which we think was a driving precipitant of some of these cases. But it also is not as popularized in the media as well. There were a lot of TikTok users in particular, which lent itself to the name “TikTok tic”. These videos are not as viewed or not as popular as they were during the Covid-19 pandemic. One reason being that because we are not all relegated to our homes, constantly looking to online sources of information---just in general, we have kind of not been on the Internet as much as we were during the Covid-19 pandemic---as a society as a whole. Dr Albin: This is really fascinating how the environmental milieu, for lack of a better word, like, really influenced how patients were experiencing, sort of, functional neurologic disorders. In your article you describe really these three baskets of primary tic---which can then be a part of Tourette syndrome---,functional ticlike behaviors---which really were a unique manifestation of stress and anxiety specifically during the Covid-19 pandemic---, and then tics as a manifestation of some either different underlying etiology or medication side effect. So, when do you get concerned about that secondary etiology? Dr Frey: So secondary tics can occur in a variety of instances. I think some of the more common examples would be in genetic disorders. So, Huntington's disease is a really good example. I think we all associate chorea with Huntington's disease. That's probably the most commonly associated phenomenology that we see with Huntington's disease. But we can see a variety of movement disorders in Huntington's, and one of them is tics. So, when we see tics in association with other types of movement disorders, we should be thinking about a possible genetic etiology. If we see tics in association with other neurologic symptoms, such as seizures or cognitive changes, we should be thinking that this is something besides a primary tic disorder. You also mentioned medication use, and it's really important to think about tardive tics. I know we often think about tardive dyskinesia, and the first kind of phenomenology that jumps into our brain is usually chorea because it's those abnormal lip movements, finger movements, toe movements that we see after a patient has been on, for example, an antipsychotic or an antiemetic that has antidopaminergic properties. However, we can see a variety of abnormal movement disorders that occur secondary to antidopaminergic medications, especially after abrupt withdrawal of these antidopaminergic medications. And tics are one of them. There have been cases reported where people that have tardive tics will still report that they have a premonitory urge, as well as a sense of relief after their tics. So, it actually can seem very similar to Tourette syndrome and the tics that people with Tourette syndrome experience on a regular basis. The key here is that the treatment might differ because if it's due to an antidopaminergic medication or abrupt withdrawal of that antidopaminergic medication, you might need to treat it a little bit differently than you would otherwise. Dr Albin: I love that you bring in, it's not just looking at their specific movement disorder that they may be coming to clinic with, that tic disorder, but are there other movement disorders? Has there been a change in their medication history? Have they had cognitive changes? So really emphasizing the importance of that complete and comprehensive neurologic history, neurologic physical exam, to really get the complete picture so that it's not honing in on, oh, this is a primary tic. That's all there is to it, because it could be so much more. I know we're getting close to sort of the end of our time together, but I really wanted to switch to end on talking about treatment. And your article does such a beautiful job of talking about behavioral interventions and really exciting new medical interventions. But I would like to, if you don't mind, have you focus on, what behavioral counseling and what education do you provide for patients and their families? Because I imagine that the neurologist plays a really important role in educating the patient and their family about these disorders. Dr Frey: Absolutely. When we think about treatment, one of the most important things you can do for patients with Tourette syndrome or other primary tic disorders is educate them. This remains true whether it's a primary tic disorder that we see in Tourette syndrome or the functional ticlike behavior that we've discussed here. A lot of times, because there is such a stigma against people with tic disorders and Tourette syndrome, when they hear that they have Tourette syndrome or they are diagnosed with that, sometimes that can be an upsetting diagnosis. And sometimes you have to take time explaining what exactly that means and debunking a lot of the myths that go along with the stigmas associated with Tourette syndrome. I think a lot of times people are under the false assumption that people with Tourette syndrome cannot lead normal lives and cannot hold down jobs and cannot be productive members of society. None of that is true. Most of my patients have great lives, good quality of life, and are able to go about their day-to-day life without any major issues. And one of the reasons for that is we do have a lot of great treatment options available. Another important stigma to break down is that people with tic disorders are doing this for attention or doing this because they are trying to get something from someone else. That is absolutely false. We do think that the tics themselves are semivolitional because people with Tourette syndrome have some degree of control over their tics. They can suppress them for a period of time. But a lot of people with tic disorders and Tourette syndrome will describe their tics as if you're trying to hold onto a sneeze. And you can imagine how uncomfortable it is to hold in a sneeze. We're all able to do it for a period of time, but it's much easier to just allow that sneeze to occur. And a lot of times that's what they are experiencing, too. So, although there is some degree of control, it's not complete control, and they're certainly not doing these tics on purpose or for attention. So that's another important myth to debunk when you're counseling patients and their families. I think the dynamic between young patients that are presenting with their parents or guardians, sometimes that dynamic is a little bit challenging because another faulty assumption is that parents feel they are responsible for having this happen to their child. There used to be a really strong sense that parents were responsible for the tics that occurred in their children, and that is also absolutely not true. Parenting has nothing to do with having the tics or not. We know that this is a neurodevelopmental disorder. The brain is indeed wired differently and it's important to counsel that with the parents, too, so that they understand what tools they need to be successful for their children as well. Dr Albin: I love that. So, it's a lot of partnership with patients and their families. I really like that this is just a wire different, and I hope over time that working together we as neurologists can help break down some of that stigmatization for these patients. This has been an absolutely phenomenal discussion. I have so enjoyed learning from your article. For the listeners out there, there are some really phenomenal tables that go into sort of how to approach this from the office perspective, how to approach it from the treatment perspective. So, thank you again, Dr Jessica Frey, for your article on Tourette syndrome and tic disorders, which appears in the August 2025 Continuum issue on movement disorders. Be sure to check out Continuum Audio episodes from this and other issues, and thank you so much to our listeners for joining us today. Dr Frey: Thank you for having me. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.
Le guitariste Thomas Naïm est le seul guitariste au monde à proposer un album solo sur Hendrix May This Be Love avec la participation du spécialiste français de Jimi Hendrix : Yazid Manou. (Rediffusion) La #SessionLive reçoit Thomas Naïm pour la sortie de l'album May This Be Love et le spécialiste français de Jimi Hendrix : Yazid Manou ! C'est après un stage d'été au Berklee College of Music de Boston et des cours à l'American School of Modern Music à Paris, que Thomas Naïm fonde avec la chanteuse Joyce Hozé le duo Tom & Joyce. Influencé par la MPB, la bossa nova des 60's et le jazz, le groupe enregistre deux albums : Tom & Joyce sorti en 2002 (Yellow Productions/East West) et Antigua en 2005 (Yellow Productions/Tommy Boy) sur lequel participe le légendaire batteur Tony Allen et dont certains morceaux seront remixés par des producteurs de dance music parmi les plus reconnus (Masters At Work, François Kevorkian ou Bob Sinclar). Par la suite, Thomas Naïm sera sollicité pour accompagner sur scène ou en studio de nombreux artistes venant d'horizons parfois très différents parmi lesquels Youn Sun Nah, Hugh Coltman, Hindi Zahra, Sébastien Tellier, Ala.ni, Mayra Andrade, Tiken Jah Fakoly, Blick Bassy, Idrissa Diop, Bob Sinclar, Salomé de Bahia, Brisa Roché, Bernard Lavilliers, Albin de la Simone, Michel Fugain, Claire Diterzi, Renan Luce… Il mène en parallèle depuis plusieurs années une carrière solo à la tête d'un trio avec le bassiste Marcello Giuliani et le batteur Raphaël Chassin avec lesquels il enregistre en 2018 l'album Desert Highway. En 2020, Thomas Naïm va confronter son trio aux compositions du maître Jimi Hendrix et enregistrer l'album Sounds of Jimi. L'idée de départ n'est pas de reprendre les titres d'Hendrix tel quel mais de trouver pour chaque morceau une esthétique plus personnelle, et à l'exception de trois titres chantés par Hugh Coltman et Célia Kameni, de privilégier l'approche instrumentale. L'album accueillera également en invités Erik Truffaz à la trompette et Camille Bazbaz à l'orgue. En 2022, il retourne en studio avec son trio augmenté de l'organiste/pianiste de Marc Benham pour enregistrer, sous la houlette du réalisateur grand angle Daniel Yvinec, On the Far Side, un album consacré à ses propres compositions dont la sortie est prévue pour février 2023. Thomas Naïm y retrouve ses premières amours, le Jazz, qu'il mêle à ses influences de toujours la pop, le rock psychédélique, les bandes originales de films et mille autres choses. Laurent Bardainne (saxophone) souffle sur trois titres un vent créatif qui donne à cet album unique une force et une couleur presque mystique. Avec May This Be Love, Thomas Naïm devient le 1er guitariste au monde, à reprendre Hendrix en guitare solo. Titres interprétés au grand studio - Hey Joe Live RFI - The Wind Cries Mary, extrait de l'album - Purple Haze Live RFI Line Up : Thomas Naïm, guitare. Son : Benoît Le Tirant, Camille Roch, Mathias Taylor. Album May This Be Love (Rootless Blues 2025). Site - YouTube - Bandcamp. Yazid Manou est journaliste, relations presse et spécialiste de Jimi Hendrix en France. en 1990, il organise un concert à l'Olympia pour les 20 ans de la disparition du guitariste américain. Il est le héros du roman « Blues pour Jimi Hendrix » écrit par Stéphane Koechlin. Yazid Manou est le gardien d'un mort comme Anubis dans la mythologie égyptienne, Jimi Hendrix. Il porte des fleurs sur sa tombe, soigne sa postérité, veille à sa gloire... En 1990, vingt ans après la mort de Jimi le 18 septembre 1970, il a organisé le festival « Jimi's Back » à Paris pendant une semaine dont une soirée à l'Olympia qui a bouleversé sa vie. Il y a invité de nombreux artistes pour reprendre les thèmes du bluesman. Il a convié bien sûr en premier lieu l'ex-bassiste de Jimi, Noel Redding, personnage douloureux et attachant, frappé quelques mois plus tôt par un deuil terrible, la mort de sa fiancée Carol Appleby dans un accident de voiture. Depuis, la vie de Yazid - devenu par la suite attaché de presse indépendant - est rythmée par les nouvelles de la « famille Hendrix », disputes avec la soeur adoptive Janie, rencontre avec ceux qui l'ont connu (B. B. King, Eric Clapton, Paul McCartney, Johnny Hallyday, Miles Davis, Taj Mahal), sorties d'albums (Jimi a davantage publié de disques une fois mort que vivant), disparition des témoins, Noel, Buddy Miles, Monika Dannemann (dernière compagne), Al Hendrix (son père) puis Mitch Mitchell. Il a construit sa vie autour d'un mort, de la mort en général. Yazid est un infatigable marcheur africain arpentant le pavé parisien en compagnie de son fantôme. Playlist Jimi Hendrix par Yazid Manou 51st Anniversary, Wait Until Tomorrow, Red House & All Along the Watchtower. Concert du 20 mars @ Le 360 Paris. Réalisation : Hadrien Touraud.
Le guitariste Thomas Naïm est le seul guitariste au monde à proposer un album solo sur Hendrix May This Be Love avec la participation du spécialiste français de Jimi Hendrix : Yazid Manou. (Rediffusion) La #SessionLive reçoit Thomas Naïm pour la sortie de l'album May This Be Love et le spécialiste français de Jimi Hendrix : Yazid Manou ! C'est après un stage d'été au Berklee College of Music de Boston et des cours à l'American School of Modern Music à Paris, que Thomas Naïm fonde avec la chanteuse Joyce Hozé le duo Tom & Joyce. Influencé par la MPB, la bossa nova des 60's et le jazz, le groupe enregistre deux albums : Tom & Joyce sorti en 2002 (Yellow Productions/East West) et Antigua en 2005 (Yellow Productions/Tommy Boy) sur lequel participe le légendaire batteur Tony Allen et dont certains morceaux seront remixés par des producteurs de dance music parmi les plus reconnus (Masters At Work, François Kevorkian ou Bob Sinclar). Par la suite, Thomas Naïm sera sollicité pour accompagner sur scène ou en studio de nombreux artistes venant d'horizons parfois très différents parmi lesquels Youn Sun Nah, Hugh Coltman, Hindi Zahra, Sébastien Tellier, Ala.ni, Mayra Andrade, Tiken Jah Fakoly, Blick Bassy, Idrissa Diop, Bob Sinclar, Salomé de Bahia, Brisa Roché, Bernard Lavilliers, Albin de la Simone, Michel Fugain, Claire Diterzi, Renan Luce… Il mène en parallèle depuis plusieurs années une carrière solo à la tête d'un trio avec le bassiste Marcello Giuliani et le batteur Raphaël Chassin avec lesquels il enregistre en 2018 l'album Desert Highway. En 2020, Thomas Naïm va confronter son trio aux compositions du maître Jimi Hendrix et enregistrer l'album Sounds of Jimi. L'idée de départ n'est pas de reprendre les titres d'Hendrix tel quel mais de trouver pour chaque morceau une esthétique plus personnelle, et à l'exception de trois titres chantés par Hugh Coltman et Célia Kameni, de privilégier l'approche instrumentale. L'album accueillera également en invités Erik Truffaz à la trompette et Camille Bazbaz à l'orgue. En 2022, il retourne en studio avec son trio augmenté de l'organiste/pianiste de Marc Benham pour enregistrer, sous la houlette du réalisateur grand angle Daniel Yvinec, On the Far Side, un album consacré à ses propres compositions dont la sortie est prévue pour février 2023. Thomas Naïm y retrouve ses premières amours, le Jazz, qu'il mêle à ses influences de toujours la pop, le rock psychédélique, les bandes originales de films et mille autres choses. Laurent Bardainne (saxophone) souffle sur trois titres un vent créatif qui donne à cet album unique une force et une couleur presque mystique. Avec May This Be Love, Thomas Naïm devient le 1er guitariste au monde, à reprendre Hendrix en guitare solo. Titres interprétés au grand studio - Hey Joe Live RFI - The Wind Cries Mary, extrait de l'album - Purple Haze Live RFI Line Up : Thomas Naïm, guitare. Son : Benoît Le Tirant, Camille Roch, Mathias Taylor. Album May This Be Love (Rootless Blues 2025). Site - YouTube - Bandcamp. Yazid Manou est journaliste, relations presse et spécialiste de Jimi Hendrix en France. en 1990, il organise un concert à l'Olympia pour les 20 ans de la disparition du guitariste américain. Il est le héros du roman « Blues pour Jimi Hendrix » écrit par Stéphane Koechlin. Yazid Manou est le gardien d'un mort comme Anubis dans la mythologie égyptienne, Jimi Hendrix. Il porte des fleurs sur sa tombe, soigne sa postérité, veille à sa gloire... En 1990, vingt ans après la mort de Jimi le 18 septembre 1970, il a organisé le festival « Jimi's Back » à Paris pendant une semaine dont une soirée à l'Olympia qui a bouleversé sa vie. Il y a invité de nombreux artistes pour reprendre les thèmes du bluesman. Il a convié bien sûr en premier lieu l'ex-bassiste de Jimi, Noel Redding, personnage douloureux et attachant, frappé quelques mois plus tôt par un deuil terrible, la mort de sa fiancée Carol Appleby dans un accident de voiture. Depuis, la vie de Yazid - devenu par la suite attaché de presse indépendant - est rythmée par les nouvelles de la « famille Hendrix », disputes avec la soeur adoptive Janie, rencontre avec ceux qui l'ont connu (B. B. King, Eric Clapton, Paul McCartney, Johnny Hallyday, Miles Davis, Taj Mahal), sorties d'albums (Jimi a davantage publié de disques une fois mort que vivant), disparition des témoins, Noel, Buddy Miles, Monika Dannemann (dernière compagne), Al Hendrix (son père) puis Mitch Mitchell. Il a construit sa vie autour d'un mort, de la mort en général. Yazid est un infatigable marcheur africain arpentant le pavé parisien en compagnie de son fantôme. Playlist Jimi Hendrix par Yazid Manou 51st Anniversary, Wait Until Tomorrow, Red House & All Along the Watchtower. Concert du 20 mars @ Le 360 Paris. Réalisation : Hadrien Touraud.
Multiple system atrophy is a rare, sporadic, adult-onset, progressive, and fatal neurodegenerative disease. Accurate and early diagnosis remains challenging because it presents with a variable combination of symptoms across the autonomic, extrapyramidal, cerebellar, and pyramidal systems. Advances in brain imaging, molecular biomarker research, and efforts to develop disease-modifying agents have shown promise to improve diagnosis and treatment. In this episode, Casey Albin, MD speaks with Tao Xie, MD, PhD, author of the article “Multiple System Atrophy” in the Continuum® August 2025 Movement Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Xie is director of the Movement Disorder Program, chief of the Neurodegenerative Disease Section in the department of neurology at the University of Chicago Medicine in Chicago, Illinois. Additional Resources Read the article: Multiple System Atrophy Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr. Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hello everyone, this is Dr Casey Albin. Today I'm interviewing Dr Tao Xie about his article on diagnosis and management of multiple system atrophy, which appears in the August 2025 Continuum issue on movement disorders. Welcome to the podcast, and please introduce yourself to our audience. Dr Xie: Thank you so much, Dr Albin. My name is Tao Xie, and sometimes people also call me Tao Z. I'm a mood disorder neurologist, professor of neurology at the University of Chicago. I'm also in charge of the mood disorder program here, and I'm the section chief in the neurodegenerative disease in the Department of Neurology at the University of Chicago Medicine. Thank you for having me, Dr Albin and Dr Okun and the American Academy of Neurology. This is a great honor and pleasure to be involved in this education session. Dr Albin: We are delighted to have you, and thank you so much for the thoughtful approach to the diagnosis and management. I really want to encourage our listeners to check out this article. You know, one of the things that you emphasize is multiple system atrophy is a fairly rare condition. And I suspect that clinicians and trainees who even have a fair amount of exposure to movement disorders may not have encountered that many cases. And so, I was hoping that you could just start us off and walk us through what defines multiple system atrophy, and then maybe a little bit about how it's different from some of the more commonly encountered movement disorders. Dr Xie: This is a really good question, Dr Albin. Indeed, MSA---multisystem atrophy----is a rare disease. It is sporadic, adult-onset, progressive, fatal neurodegenerative disease. By the name MSA, multisystem atrophy. Clinically, it will present with multiple symptoms and signs involving multiple systems, including symptoms of autonomic dysfunction and symptoms of parkinsonism, which is polyresponsive to the levodopa treatment; and the symptom of cerebellar ataxia, and symptom of spasticity and other motor and nonmotor symptoms. And you may be wondering, what is the cause- underlying cause of these symptoms? Anatomically, we can find the area in the basal ganglia striatonigral system, particularly in the putamen and also in the cerebellar pontine inferior, all of the nuclear area and the specific area involved in the autonomic system in the brain stem and spinal cord: all become smaller. We call it atrophy. Because of the atrophy in this area, they are responsible for the symptom of parkinsonism if it is involved in the putamen and the cerebral ataxia, if it's involved in the pons and cerebral peduncle and the cerebellum. And all other area, if it's involved in the autonomic system can cause autonomic symptoms as well. So that's why we call it multisystem atrophy. And then what's the underlying cellular and subcellular pathological, a hallmark that is in fact caused by misfolded alpha-synuclein aggregate in the oligodontia site known as GCI---glial cytoplasmic increasing bodies---in the cells, and sometimes it can also be found in the neuronal cell as well in those areas, as mentioned, which causes the symptom. But clinically, the patient may not present all the symptoms at the same time. So, based on the predominant clinical symptom, if it's mainly levodopa, polyresponsive parkinsonism, then we call it MSAP. If it's mainly cerebellar ataxia, then we call it MSAC. But whether we call it MSP or MSC, they all got to have autonomic dysfunction. And also as the disease progresses, they can also present both phenotypes together. We call that mixed cerebellar ataxia and parkinsonism in the advanced stage of the disease. So, it is really a complicated disease. The complexity and the similarity to other mood disorders, including parkinsonism and the cerebellar ataxia, make it really difficult sometimes, particularly at the early stages of disease, to differentiate one from the other. So, that was challenging not only for other professionals, general neurologists and even for some movement disorder specialists, that could be difficult particularly if you aim to make an accurate and early diagnosis. Dr Albin: Absolutely. That is such a wealth of knowledge here. And I'm going to distill it just a little bit just to make sure that I understand this right. There is alpha-synuclein depositions, and it's really more widespread than one would see maybe in just Parkinson's disease. And with this, you are having patients present with maybe one of two subtypes of their clinical manifestations, either with a Parkinson's-predominant movement disorder pattern or a cerebellar ataxia type movement disorder pattern. Or maybe even mixed, which really, you know, we have to make things quite complicated, but they are all unified and having this shared importance of autonomic features to the diagnosis. Have I got that all sort of correct? Dr Xie: Correct. You really summarize well. Dr Albin: Fantastic. I mean, this is quite a complicated disease. I would pose to you sort of a case, and I imagine this is quite common to what you see in your clinic. And let's say, you know, a seventy-year-old woman comes to your clinic because she has had rigidity and poor balance. And she's had several falls already, almost always from ground level. And her family tells you she's quite woozy whenever she gets up from the chair and she tends to kind of fall over. But they noticed that she's been stiff,and they've actually brought her to their primary care doctor and he thought that she had Parkinson's disease. So, she started levodopa, but they're coming to you because they think that she probably needs a higher dose. It's just not working out very well for her. So how would you sort of take that history and sort of comb through some of the features that might make you more concerned that the patient actually has undiagnosed multiple systems atrophy? Dr Xie: This is a great case, because we oftentimes can encounter similar cases like this in the clinic. First of all, based on the history you described, it sounds like an atypical parkinsonism based on the slowness, rigidity, stiffness; and particularly the early onset of falls, which is very unusual for typical Parkinson disease. It occurs too early. If its loss of balance, postural instability, and fall occurred within three years of disease onset---usually the motor symptom onset---then it raises a red flag to suspect this must be some atypical Parkinson disorders, including multiple system atrophy. Particularly, pou also mentioned that the patient is poorly responsive to their levodopa therapy, which is very unusual because for Parkinson disease, idiopathic Parkinson disease, we typically expect patients would have a great response to the levodopa, particularly in the first 5 to 7 years. So to put it all together, this could be atypical parkinsonism, and I could not rule out the possibility of MSA. Then I need to check more about other symptoms including autonomic dysfunction, such as orthostatic hypertension, which is a blood pressure drop when the patient stands up from a lying-down position, or other autonomic dysfunctions such as urinary incontinence or severe urinary retention. So, in the meantime, I also have to put the other atypical Parkinson disorder on the differential diagnosis, such as PSP---progressive supranuclear palsy---and the DLBD---dementia with Lewy body disease.---Bear this in mind. So, I want to get more history and more thorough bedside assessment to rule in or rule out my diagnosis and differential diagnosis. Dr Albin: That's super helpful. So, looking for early falls, the prominence of autonomic dysfunction, and then that poor levodopa responsiveness while continuing to sort of keep a very broad differential diagnosis? Dr Xie: Correct. Dr Albin: One of the things that I just have to ask, because I so taken by this, is that you say in the article that some of these patients actually have preservation of smell. In medical school, we always learn that our Parkinson's disease patients kind of had that early loss of smell. Do you find that to be clinically relevant? Is that- does that anecdotally help? Dr Xie: This is a very interesting point because we know that the loss of smelling function is a risk effect, a prodromal effect, for the future development of Parkinson disease. But it is not the case for MSA. Strange enough, based on the literature and the studies, it is not common for the patient with MSA to present with anosmia. Some of the patients may have mild to moderate hyposmia, but not to the degree of anosmia. So, this is why even in the more recent diagnosis criteria, the MDS criteria published 2022, it even put the presence of anosmia in the exclusion criteria. So, highlight the importance of the smell function, which is well-preserved for the majority in MSA, into that category. So, this is a really interesting point and very important for us, particularly clinicians, to know the difference in the hyposmia, anosmia between the- we call it the PD, and the dementia Lewy bodies versus MSA. Dr Albin: Fascinating. And just such a cool little tidbit to take with us. So, the family, you know, you're talking to them and they say, oh yes, she has had several fainting episodes and we keep taking her to the primary care doctor because she's had urinary incontinence, and they thought maybe she had urinary tract infections. We've been dealing with that. And you're sort of thinking, hm, this is all kind of coming together, but I imagine it is still quite difficult to make this diagnosis based on history and physical alone. Walk our listeners through sort of how you're using MRI and DAT scan and maybe even some other biomarkers to help sort of solidify that diagnosis. Dr Xie: Yeah, that's a wonderful question. Yeah. First of all, UTI is very common for patients with MSA because of urinary retention, which puts them into a high risk of developing frequent UTI. That, for some patients, could be the very initial presentation of symptoms. In this case, if we check, we say UTI is not present or UTI is present but we treat it, then we check the blood pressure and we do find also hypertension---according to new diagnosis criteria, starting drop is 20mm mercury, but that's- the blood pressure drop is ten within three minutes. And also, in the meantime the patients present persistent urinary incontinence even after UTI was treated. And then the suspicion for MS is really high right at this point. But if you want increased certainty and a comfortable level on your diagnosis, then we also need to look at the brain MRI mark. This is a required according to the most recent MDS diagnosis criteria. The presence of the MRI marker typical for MSA is needed for the diagnosis of clinically established MSA, which holds the highest specificity in the clinical diagnosis. So then, we have- we're back to your question. We do need to look at the brain MRI to see whether evidence suggestive of atrophy around the putamen area, around the cerebellar pontine inferior olive area, is present or not. Dr Albin: Absolutely. That's super helpful. And I think clinicians will really take that to sort of helping to build a case and maybe recognizing some of this atypical Parkinson's disease as a different disease entity. Are there any other biomarkers in the pipeline that you're excited about that may give us even more clarity on this diagnosis? Dr Xie: Oh, yeah. This is a very exciting area. In terms of biomarker for the brain imaging, particularly brain MRI, in fact, today there's a landmark paper just published in the Java Neurology using AI, artificial intelligence or machine learning aid, diagnoses a patient with parkinsonism including Parkinson's disease, MSA, and PSP, with very high diagnostic accuracy ranging from 96% to 98%. And some of the cases even were standard for autopsy, with pathological verification at a very high accurate rate of 93.9%. This is quite amazing and can really open new diagnosis tools for us to diagnose this difficult disease; not only in an area with a bunch of mood disorder experts, but also in the rural area, in the area really in need of mood disorder experts. They can provide tremendous help to provide accurate, early diagnosis. Dr Albin: That's fantastic and I love that, increasing the access to this accurate diagnosis. What can't artificial intelligence do for us? That's just incredible. Dr Xie: And also, you know, this is just one example of how the brain biomarker can help us. Theres other---a fluid biomarker, molecular diagnostic tools, is also available. Just to give you an example, one thing we know over the past couple years is skin biopsy. Through the immunofluorescent reaction, we can detect whether the hallmark of abnormally folded, misfolded, and the phosphorate, the alpha-synuclein aggregate can be found just by this little pinch of skin biopsy. Even more advanced, there's another diagnosis tool we call the SAA, we call the seizure amplification assay, that can even help us to differentiate MSA from other alpha-synucleinopathy, including Parkinson disease and dementia with Lewy bodies. If we get a little sample from CSF, spinal cerebral fluids, even though this is probably still at the early stage, a lot of developments still ongoing, but this, this really shows you how exciting this area is now. We're really in a fast forward-moving path now. Dr Albin: It's really incredible. So, lots coming down the track in, sort of, MRI, but also with CSF diagnosis and skin biopsies. Really hoping that we can hone in some of those tools as they become more and more validated to make this diagnosis. Is that right? Dr Xie: Correct. Dr Albin: Amazing. We can talk all day about how you manage these in the clinic, and I really am going to direct our listeners to go and read your fantastic article, because you do such an elegant job talking about how this takes place in a multidisciplinary setting, if at all possible. But as a neurointensivist, I was telling you, we have so much trouble in the hospital. We have A-lines, and we have the ability to get rapid KUBs to look at Ilias, and we can have many people as lots of diagnosis, and we still have a lot of trouble treating autonomiclike symptoms. Really, really difficult. And so, I just wanted to kind of pick your brain, and I'll start with just the one of orthostatic hypotension. What are some of the tips that you have for, you know, clinicians that are dealing with this? Because I imagine that this is quite difficult to do without patients. Dr Xie: Exactly. This is indeed a very difficult symptom to deal with, particularly at an outpatient setting. But nowadays with the availability of more medication---to give an example, to treat patients with orthostatic hypertension, we have not only midodrine for the cortisol, we also have droxidopa and several others as well. And so, we have more tools at hand to treat the patient with orthostatic hypertension. But I think the key thing here, particularly for us to the patient at the outpatient setting: we need to educate the patient's family well about the natural history of the disease course. And we also need to tell them what's the indication and the potential side effect profile of any medication we prescribe to them so that they can understand what to expect and what to watch for. And in the meantime, we also need to keep really effective and timely communication channels, make sure that the treating physician and our team can be reached at any time when the patient and family need us so that we can be closely monitoring, their response, and also monitoring potential side effects as well to keep up the quality of care in that way. Dr Albin: Yeah, I imagine that that open communication plays a huge role in just making sure that patients are adapting to their symptoms, understanding that they can reach out if they have refractory symptoms, and that- I imagine this takes a lot of fine tuning over time. Dr Xie: Correct. Dr Albin: Well, this has just been such a delight to get to talk to you. I really feel like we could dive even deeper, but I know for the sake of time we have to kind of close out. Are there any final points that you wanted to share with our listeners before we end the interview? Dr Xie: I think for the patients, I want them to know that nowadays with advances in science and technology, particularly given a sample of rapid development in the diagnostic tools and the multidisciplinary and multisystemic approach to treatment, nowadays we can make an early and accurate diagnosis of the MSA, and also, we can provide better treatment. Even though so far it is still symptomatically, mainly, but in the near future we hope we can also discover disease-modifying treatment which can slow down, even pause or prevent the disease from happening. And for the treating physician and care team professionals, I just want them to know that you can make a difference and greatly help the patient and the family through your dedicated care and also through your active learning and innovative research. You can make a difference. Dr Albin: That's amazing and lots of hope for these patients. Right now, you can provide really great care to take care of them, make an early and accurate diagnosis; but on the horizon, there are really several things that are going to move the field forward, which is just so exciting. Again today, I've been really greatly honored and privileged to be able to talk to Dr Tao Xie about his article on diagnosis and management of multiple system atrophy, which appears in the August 2025 Continuum issue on movement disorders. Be sure to check out Continuum Audio episodes for this and other issues. And thank you again to our listeners for joining us today. Dr Xie: Thank you so much for having me. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.
Join Nick Lamagna on The A Game Podcast with our guest Nick Albin, aka "Chewy," one of the most well known Brazilian Jiu Jitsu Black Belts and entrepreneurs on the internet today! He is an entrepreneur, business owner, podcast host and coach fighting out of Louisville, Kentucky! After a traumatic experience where he was jumped in high school, he lacked self confidence and a sense of safety and self worth. He was eventually brought back out of his shell and gained his confidence back through wrestling, weight lifting and martial arts eventually earning a black belt in Brazilian Jiu Jitsu in 2011. This self proclaimed meathead at heart has an extensive background in martial arts, health, nutrition and fitness. He has a gift for explaining things in an entertaining and relatable way which has caught on like wild fire where he has earned a blackbelt in branding business and marketing. He continually finds ways to grow his academy and podcast sharing his love of Brazilian jiu jitsu, health and fitness with the world through his academy, Derby City Mixed Martial Arts and his wildly popular Youtube Channel and social media, where athletes and business owners can relate to the parallels of the stress of humanity and he is connecting the bridges of the brains of the civilian and the combat sport competitor to show us that we're all the same and can help each other do better He also has a popular tournament, The Chewjitsu Open he puts on and is gaining a lot of traction in the competition circles. You know him through his Chewjitsu brand,The Chewjitsu Podcast and his successful Chew Crew Newsletter, welcome to the A Game Podcast, BJJ Royalty Nick Albin! Topics for this episode include: ✅ An important question you must answer before you go into business ✅ The answer to successful branding that no one wants to hear ✅ How to deal with fear and anxiety in life and business ✅ Learn the superpower to embrace the things you don't enjoy ✅ What to look for when you're trying to find a great mentor + more! Check the show notes to connect with all things Chewy! Connect with Nick "Chewy" Albin: www.chewjitsu.net Nick Albin on Facebook Nick Albin on Instagram Nick Albin on Twitter Nick Albin on LinkedIn Nick Albin on TikTok Nick Albin on Youtube Free Focused BJJ Ebook Subscribe to the Chew Crew Newsletter! Connect with ChewJitsu Podcast: ChewJitsu Podcast Chewjitsu Podcast on Facebook Chewjitsu Podcast on Instagram Chewjitsu Podcast on Threads Chewjitsu Podcast on Youtube Connect with Derby City: Derby City Mixed Martial Arts Derby City Mixed Martial Arts on Facebook Derby City MMA on Instagram Derby City MMA on Threads Derby City Mixed Martial Arts on Youtube Connect with other Chewy Jitsu Open Tournament: The Chewjitsu Open On Facebook Chewjitsu Open on Instagram Chewjistu Open on Threads --- Connect with Nick Lamagna www.nicknicknick.com Text Nick (516)540-5733 Connect on ALL Social Media and Podcast Platforms Here FREE Checklist on how to bring more value to your buyers
Ep 465, pt 3: Guest Ryan Lambert gives his thoughts on Maine goalie Albin Boija, his potential and whether or not the Bruins have a real chance to sign him after college. To learn more about listener data and our privacy practices visit: https://www.audacyinc.com/privacy-policy Learn more about your ad choices. Visit https://podcastchoices.com/adchoices
Ep 465, pt 3: Guest Ryan Lambert gives his thoughts on Maine goalie Albin Boija, his potential and whether or not the Bruins have a real chance to sign him after college. To learn more about listener data and our privacy practices visit: https://www.audacyinc.com/privacy-policy Learn more about your ad choices. Visit https://podcastchoices.com/adchoices
Our friend Albin Sadar joins us to discuss his recent article "Some People Are Still Oblivious to the Obvious".The article criticizes modern gender norms as rejecting self-evident truths and urges Christians to actively defend traditional values.See omnystudio.com/listener for privacy information.
Albin shares a 4th of July Fun Facts Friday!See omnystudio.com/listener for privacy information.
Albin Sadar Recaps the High Seas Hijinks on the Greek Cruise with EricSee omnystudio.com/listener for privacy information.
Albin joins us again this week for a Fun Facts Friday specialSee omnystudio.com/listener for privacy information.
Eric is interviews by Albin about his book Fish out of Water See omnystudio.com/listener for privacy information.
As part of the work of a behavior analyst, our ethical code requires us to explain our services in language that can be understood by clients and stakeholders. That holds doubly true when our clients speak a primary language other than English. And just busting out Google Translate isn't going to cut it! In the next entry in our Tutorial series, we break out the “how to” basics behind following best practices when working with an interpreter in delivering ABA services. From meaningful feedback and collaboration with an interpreter to ensuring a respectful flow of communication between all parties, we'll get your practice up to speed in no time. Well, at least in the time it takes to listen to this episode. This episode is available for 1.0 CULTURAL (ETHICS) CEU. Articles discussed this episode: Dowdy, A., Obidimalor, K.C., Tinanci, M., & Travers, J.C. (2021). Delivering culturally sound and high-quality behavior analytic services when working with an interpreter. Behavior Analysis: Research and Practice, 21, 51-64. doi: 10.1037/bar0000206 Vasquez, D.J., Lechago, S.A., & McCarville, M.J. (2024). Training behavior analysis graduate students to work with an interpreter. Behavior Analysis in Practice, 17, 1160-1174. doi: 10.1007/s40617-024-00938-w Hadziabdic, E., Albin, B., Heikkila, K., & Hjelm, K. (2014). Family members' experiences of the use of interpreters in healthcare. Primary Health Care Research and Development, 15, 156-169. doi: 10.1017/S1463423612000680 If you're interested in ordering CEs for listening to this episode, click here to go to the store page. You'll need to enter your name, BCBA #, and the two episode secret code words to complete the purchase. Email us at abainsidetrack@gmail.com for further assistance.
Now that we've got some vacation time coming up in our near futures, we can sit back, relax…and make our listener's choose our episodes for us! That's right. We've got not one but TWO polls for listeners this month that will determine our summer Listener Choice topic as well as the next year's worth of Book Clubs. Excited to vote? Well, only patrons (at ANY level) will have access to the LC and Book Club polls so, if you haven't signed up yet, better get on it before everyone else chooses your books for you. Oh, and we have a full month of episodes all about IOA, working with an interpreter, and a whole grab bag of ethics! So, by vacation, we meant podcasting wearing sunglasses and shorts. Listener Choice Poll Book Club Poll Join the Patreon! Articles for June 2025 IOA Hausman, N.L., Javed, N., Bednar, M.K., Guell, M., Schaller, E., Nevill, R.E., & Kahng, S. (2022). Interobserver agreement: A preliminary investigation into how much is enough? Journal of Applied Behavior Analysis, 55, 357-368. doi: 10.1002/jaba.811 Garrity, M.L., Luiselli, J.K., & McCollum, S.A. (2008). Effects of a supervisory intervention on assessment of interobserver agreement by educational service providers. Behavioral Interventions, 23, 105-112. doi: 10.1002/bin.258 Hartz, R.M., Gould, K., Harper, J.M., & Luiselli, J.K. (2020). Assessing interobserver agreement (IOA) with procedural integrity: Evaluation of training methods among classroom instructors. Child and Family Behavior Therapy, 43, 1-12. doi: 10.1080/00168890.2020.1848404 Ethicstime! (Summer 2025 Grab Bag) Henderson, T.B., Ludden, B.J., & Romero, R.A. (2023). The ethical obligations, barriers, and solutions for interprofessional collaboration in the treatment of autistic individuals. Behavior Analysis in Practice, 16, 963-976. doi: 10.1007/s40617-023-00787-z Shreck, K., Ivy, J. W., & Zane, T. (2023). Teaching behavior analysts to address unethical behavior: Developing evidence based ethics instructional methods. Behavior Analysis in Practice. doi: 10.1007/s40617-023-00845-6 Logue, J. J., Hustyi, K. M., Toby, L M., & Outlaw, E. E. (2023). Promoting ethical and evidence-based practice through a panel review process: A case study in implementation research. Behavior Analysis in Practice. doi: 10.1007/s40617-023-00807-y Tutorial: Working With an Interpreter Dowdy, A., Obidimalor, K.C., Tinanci, M., & Travers, J.C. (2021). Delivering culturally sound and high-quality behavior analytic services when working with an interpreter. Behavior Analysis: Research and Practice, 21, 51-64. doi: 10.1037/bar0000206 Vasquez, D.J., Lechago, S.A., & McCarville, M.J. (2024). Training behavior analysis graduate students to work with an interpreter. Behavior Analysis in Practice, 17, 1160-1174. doi: 10.1007/s40617-024-00938-w Hadziabdic, E., Albin, B., Heikkila, K., & Hjelm, K. (2014). Family members' experiences of the use of interpreters in healthcare. Primary Health Care Research and Development, 15, 156-169. doi: 10.1017/S1463423612000680
Albin Sadar returns with his latest articles including "Silence isn’t peace — it’s just surrender in slow motion." https://www.theblaze.com/columns/opinion/silence-isnt-peace-its-just-surrender-in-slow-motionSee omnystudio.com/listener for privacy information.
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