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HEY GUYS! SO EPISODE 45 AUDIO FILES GOT DELETED, WE HAVE TO USE EPISODE 46 :( SO TECHNICALLY, THIS IS EPISODE 46 BUT WE HAVE TO UPLOAD IT AS EPISODE 45 *CRIES* Listen to Cheli open up about some raunchy stories that were sent in! The new patreon minisode is up! Thank you to everyone who has submitted a story! We all love you. Want to show some support? We are now on Patreon! https://www.patreon.com/LivingWithanEmpathOn There we will be doing live streams, answering questions, releasing never heard episodes, giving Patreons first access to live readings and bookings and SOOOOOO much more!! Do not forget to leave a review on apple podcast! Do you have any questions or just want to send in a story you feel like sharing? Email livingwithanempath@gmail.com Social media: https://www.instagram.com/miayeun/ https://www.instagram.com/livingwithanempath/ https://www.instagram.com/light_goddess_4angels/ https://www.instagram.com/beautifulsoul_bjj_/ Darkest Child by Kevin MacLeod Link: https://incompetech.filmmusic.io/song/3615-darkest-child License: http://creativecommons.org/licenses/by/4.0/ --- Support this podcast: https://anchor.fm/livingwithanempath/support
This episode is a milestone for several reasons. One, it's the 50th episode and, for a weekly podcast that means it has been about a year, which is also a not insignificant milestone. For a one-man-band hobby podcaster, that's a pretty good run. When I started KEW I wasn't sure where I was going or what I was trying to accomplish over the long term.And, really, I began asking myself that question around Episode 41 when I announced the 'state of the podcast', which was mostly about changing up a few mechanical aspects like social media and shortening the episodes from an hour or more to around 20 minutes.Prior to that I was on a roll of episodes that reflected my personal growth (Ep. 33: Soul, Ep. 34 Self, Ep. 36: Narcissism and Codependency) and things I have learned in counseling, therapy, and coaching.The rest of the Episodes tended to address concepts I have thought about for years or longer. I learned a lot by sitting down and talking these concepts through in podcast form. Then I did an interview series about Curiosity that gave me some experience networking and interviewing but also expounded on a key element of what KEW was all about. And I didn't realize this when I started.In fact, I didn't now much when I started, but I did know I wanted to develop some cohesion across my varied thoughts. I wanted to find a theme in this work that could be organized into some sort of cohesive document, series, or work. I wanted to find the patterns in my ideas and figure out how to put them all together to be presented as an integrated whole.And Episode 50 seemed like the right time to so.I have spent the past few weeks figuring out what this integrative device would be and I have found a place to start.The reason I was compelled to do this podcast/blog/vlog is to address a single theme:The human condition is a struggle between who we ARE and who we think we SHOULD BEThough I'm sure this isn't entirely universal, it's close enough for me. And it IS me. And I want to share my thoughts about the WHAT, WHY, and HOW of this struggle with the world. Because I bring a unique perspective, and I will argue that UNIQUENESS is supposed to be expressed. SO, inherent to my argument, basically Episode 50 forward, is me expressing my unique offering to the world about what, why, and how we deal with this struggle. The 'Are vs. Should' Problem, if you will.And I will do this in real time. I have a rough 'table of contents' for two products. One about the What and the Why, and one about the How. Basically, I will develop, in conjunction with weekly podcast topics, this table of contents into two cohesive manuscripts to be released at some later date. The first will explain WHAT *I* think this dilemma IS and WHY *I* think this struggle exists. In the second book I will share HOW *I* think we can deal with or address this struggle.These works will build upon my SCIENCE knowledge and PERSONAL GROWTH experience. My credentials or credibility, if you will. It's what I have, after all. And who I am. And that's all I got.So Episode 50 explains all that in a bit more detail. I hope you enjoy and will help me complete these works.
So Episode 23 is a bit of a special one. We wanted to do a Geekachu Watch Along episode so here it is! Ger is a massive John Carpenter fan but has never seen 'They Live'. So four your delight and delectation we give you the Geekachu 'They Live' commentary track (sponsored by Pringles) Come and have a listen... what's the worst that can happen?
Eric Rieger 0:00 Gut Check project fans and KBMD Health family. How are you doing today? It's your host, Eric Rieger, soon to be joined by my co host, Dr. Kenneth Brown. This is episode number 56. And if you've heard 54 and 55, which we've gotten plenty of email about, thank you for all of that. You know that 56 is kind of the combination here. So we're talking about issues around erectile dysfunction, and more or less kind of the catalyst that causes it as it pertains to inflammation and systemic disease and certainly virus infections. So that being said, go ahead and dive in here to 56 I think that there's a lot of hope here. There's a lot of things that people can actively do to stave off some of these manifestations because that's what erectile dysfunction is it's it's a signal that something else is happening. So without further ado, let's get to our sponsors. Of course there is artron to get your daily poly females that are trying to go to love my tummy calm again. It's love my tummy, calm us, slash KB MD and get your daily poly phenol today developed by my co host, Dr. Kenneth Brown. Dr. Ron teal is chock full of polyphenols does not matter if you have gut issues if you suffer from IBS or related symptoms or you're just a a an active athlete and you want to optimize your daily health so that you can be your best you need polyphenols, there's no way around it. So are trying to go to love my tummy.com slash KB MD. You got great tasting food, unrefined bakery go to unrefined bakery.com because it does not matter. If you have a specialty diet or not. unrefined bakery can make things available to you that maybe you thought were now impossible because you are celiac, or because you're paleo, or because you're keto? No, they have cracked the code using all natural ingredients. Go to unrefined bakery.com Get yourself some bread, get some tasty, tasty bread, knowing that you're both celiac and paleo. You can do it there. And it's gonna taste great, so unrefined. bakery.com use code, gut check and save 20% off of your very first order. They delivered to all of the lower 48 states even though they're based here in the DFW Metroplex of North Texas, so unrefined bakery.com Last but not least, KB and D health.com. kb MD health calm is the home of the gut check project. That's where Dr. Kenneth brown makes available his own CBD as well as broccoli and artron. teal, so check out KB Md health.com. Use code GCP and save 20% off of any order at any time. Okay, without wasting any more of your time. Let's get to episode number 56.Hello, Gut project fans and KB MD Health family hope you having a great day. It's me Eric Rieger here with the awesome co host, Dr. Kenneth Brown. It's Episode 56, a continuation of 54 and 55. So, Dr. Brown, why don't you kind of set the stage for us?Ken Brown 3:32 Yeah, so this is part three of what we didn't really realize it would turn into a part three series, right? That part one of this particular series. So Episode 34, was that I was seeing so many patients that were coming in. And because of the job that I have being a physician against neurologists, they're willing to talk about certain things. They're young men that had never had issues were showing up with erectile dysfunction. months, like eight months after having COVID. They want to know if there was a correlation. Well, because of that, I started looking into things. And then we actually did find that there's some studies going on with this. And so it's going to be much bigger than what people think we're just a little ahead of the curve on this. So Episode 54 was about how it's doing it. Episode 55 is a natural ways to try and protect yourself and Episode 56. It's a continuation of that. As it turns out, there's tons of science on this. And if you just look and you know where to look and you have access to people that can get these articles. It's fascinating. fascinating, because what we're talking about is not just a reptile health, we're talking about overall health. Every one of these topics that affect Well, if something affects your penis, it's probably affecting other parts of your body.Eric Rieger 4:44 Yeah. If that were to happen, it would probably affect your mentation, your mood, your drive to do a lot of things. So yeah, it definitely it's everything is universal. Everything that seems local is really universal. Yeah, vice versa.Ken Brown 4:58 Yeah. So it's We're gonna go over some really cool stuff today.Eric Rieger 5:02 So just briefly personal Eric anything cool going on man we're still remodeling kids are fine. Marie's on a, she took a trip to Montana she's on her. They were hired her social media conglomerate that she built is the Montana filming awesome stuff that probably looks different than a GI sweet I kind of wish I was looking at it.Ken Brown 5:23 That's awesome. For me. Same stuff we're remodeling, no big deal only issue is is that with some countries in some states having different COVID stuff, my kids want to start traveling more, but I don't know if that particular country would is going to cancel the tournament. And I'm trying to rearrange my schedule to go and book flights. It's just so annoying. And what a privilege to say that the what not, I mean, honestly a privilege to say it's annoying that I'm trying to plan a trip, although I'll be extremely important for my kids. We're in a position here in Texas where most of the economy is opened up. Oh, yeah, yeah, there's still places where everything shut down. So if you're a restaurant owner in New York or California, it's still super difficult. And I'm kind of complaining about the fact that I can't book a trip because I don't know if that country is going to, you know, do some blocking haveEric Rieger 6:18 been talking about this today. But before you walked into the room with one we had a patient from Canada, originally from Canada, so as a sister who lives there, and she was sharing with us that they are still you it's almost like textile blue laws out there every day. So you can't go into a Walmart and shop on the side of their textileKen Brown 6:38 blue laws. Me. Ah,Eric Rieger 6:41 so then in the old days, and I think even a New Jersey, in this, it's changed, it may still be the same. On Sunday. You can't buy certain textiles. Really? Yeah, it's just a blue law. I mean, I don't I don't know. I don't I don't want to talk out of turn there. But back in when I was really, really young, you couldn't couldn't go into a mall and shop at like a dillards was closed on Sunday. So yeah, I we don't do that anymore. No,Ken Brown 7:07 we don't cuz that's like the only time I'd be willing to go to a mall, like, really early. But, but really, Mother's Day anniversaries come on? Yeah, I have the moles that are, youEric Rieger 7:20 know, no one in my household. But what was interesting is she said that in the Walmart, for instance, is kind of a universal way to break it down, that they have blocked off or quarantined off, anything that's not food related. So you go into the store, and you'll see everything rubbed off, you cannot go over there to make a purchase or anything like that. And that's it. That was in Ontario, Canada. So I don't know if that's universal for the entire country. She just knew what her sister was relaying. And so if you're watching this, this is recorded. May 19 2021. It's starkly different here in Texas, where our governor released here recently that we're at the at the low, and everything's pretty wide open,Ken Brown 8:06 it's pretty wide open mass are no longer require they're suggested. And I don't think we're seeing a big bump. I talked to the doctors at my house. And they're like, no, we're not seeing a big bump COVID. Now, of course, IEric Rieger 8:17 live in a more rural setting in you do. And as far as what people say, is wide open, quite frankly, 1010 miles west of me is, I think has been wide open since a year ago.Ken Brown 8:30 It's just it's fascinating. Because we're gonna look back at these things. We're gonna look back at our first COVID episodes. Oh, yeah, we were with everybody else. But I felt like we were looking at the data ahead of the data that was being discussed on national news in Fauci and all these other people, because, shockingly, we were pretty spot on the whole time. And I think I hope that people when they walk away from this podcast can realize that this is not a politically motivated, or really, I think that a lot of things that we come to we don't know what the answer is going to be, or are certainly open to the we're going to change our mind.Eric Rieger 9:05 I think in a moment, you're gonna ask me a little bit about cbds, kind of the heads of he gave me but I'm not really sure where we're going. But years ago, you and I both had doubts if it did anything. And now I would say that we were open enough to try and you've got to be able, I think, to be intellectually honest with yourself when you have something new or information to make sure that you're receiving it, and you're evaluating it and not just putting up a wall because it disagrees with where you are currently.Ken Brown 9:34 100% I mean, I think that that is when you talk about the cognitive dissonance, or Oh, yeah, how's your views are different than mine? I'm going to shut you off before you're we did a whole episode on anger or agreement and how much of your brain gets occupied when you disagree with somebody and you're trying to combat it? Because you one of the things I notice, in fact, I noticed this with this podcast, because I heard I heard Joe Rogan talking about it, you know joselyn To be a really good listener. I mean, one of the flaws of what we do here is that I have all this science, I want to make sure that we get through in a timely fashion. And sometimes when we're talking and we can have like, great conversation, I'm kind of looking at my notes going, Okay, well, we need to make sure that we cover this because this is this was on my agenda. Sure. I'm listening to you. And now just realize that I do that I'm going to work on that I'm gonna work on listening, as his most recent guest said, We teach in school. reading, writing, arithmetic, and speaking and listening, but not listening. Yeah.Eric Rieger 10:37 All right, I certainly younger version of me definitely could have been a much better listener. And I think it's active. I mean, I'm not even trying to be funny. It just, as I've grown older, I've learned that listening is a skill, listening to someone tell their story. And even though you feel like you have a story that relates to them, if they're really airing out something, don't try to match their story, just listen to them. And that's, that's I mean, quite honestly, it's been difficult for me growing up because you feel like you're building a common bond. Oh, that happened to you. This happened to me. But you can see where that goes. When you're really young. It turns into a one upsmanship. And then eventually, you're like, well, I don't want to tell them anything. They just tell me what they did was 10 times better than what I did.Ken Brown 11:21 Yeah, I'm really, really guilty of this, as pointed out when on my 50th birthday met Jr. and Brian, and it was the running joke was me storytelling. It's just like, oh, you went 10 minutes without talking to your boss. Like, oh, yeah, I'mEric Rieger 11:40 gonna work on that for now. Hey, man, but the greatest thing is, it's not that you did or didn't do it in the past, it's in everybody can do it. And it doesn't have to be about storytelling. It's, what is it? Who do I want to be? And how do I get there? And what things can I improve to make it easier?Ken Brown 11:54 And as we're always doing the show, all these things are mental improvements that people do. But there's so many times when people have health hindrances that have pain, or they're not sleeping well, or that is the least of their concerns. Definitely, you know, I mean, I try and control my diet, I try and meditate, I try and do some breathing exercises on a daily basis. But it's because I have the luxury of not having severe health issues. That is not the primary concern, right, because I went for about two months, as we discussed last time, where my primary health concern was my severe neck pain. And that just shut everything down. And the only thing that I could really wrap my brain around was okay, you need to figure out how to not panic, right. And by practicing those things leading up to this, when that happened, I felt like it kind of helped me get over the acute phase of it. And now I'm into this kind of new aspect of now we're rehabbing soEric Rieger 12:55 and I don't want to derail that. analogies are just something that I've always liked. And we You and I both build on these throughout every single episode, we find an analogy, but something that was realized that occurred, I can remember being young, and we were complaining in elementary school, it was it was something really silly had to do with participant participation, and how you could earn to be the first one out at PE or something like this. And there was someone in our class who I didn't feel like it, several of us didn't feel like it earned the right to do it. And then quietly, our teacher pulled us aside and said, this is the best thing that's happened to her because she doesn't have a home. Like what you have, and I started thinking a lot was wanting to get to PE first she just wanted a place to sleep. And when you're young, that kind of context is important because I mean, yeah, it was kind of shoved in our face, but it shows you that your I don't know I don't know how to put it the immediacy of what you think that you need or want. Sometimes somebody else may need or want that same thing but at the same time they don't even have you said your health so that you could do the exercises to feel good sometimes you don't have the base things that allow you to Oh, Peter, just do whatever. Oh, let'sKen Brown 14:17 talk about I mean generosity feeds right there. 100% I mean, generosity feeds Ron Kobani feeds kids that normally only eat at school, those are the only meals that they get when they go home on Friday. They don't eat until Monday. And recognizing that need and spending his life to correct that one. small niche is just amazing to me. 100%Eric Rieger 14:43 and shout out to Ron generosity feedsKen Brown 14:46 Yeah, and we're we're avid sponsors and have been for the last couple of years and I'm just so blessed to have met people like that, that inspire me to be a better person.Eric Rieger 14:54 Yeah. Now didn't want to derail But anyway, that just popped in my mind. Yeah. You know, you got That's what this that's what I think that we're talking about though how do you get to the point where you can make better decisions for who you want to be and how you want to be there and a lot of that comes from take care of your if your physical health and it will allow you to do that.Ken Brown 15:14 Yeah, that and you know, be open minded that the maybe your initial ideas as you grow and experience them What ifEric Rieger 15:24 when they can't be open minded because your physical health is bad. AndKen Brown 15:27 one of them that's today was today was open minded lesson I came across it You and I were we're scoping today and I came across an article, which I thought was entertaining. And then you became academic on me, which then now I'm like, Oh, I just want to gloss over right now. As as, as a fellow there's, you know, you go to residency and then you go to fellowship. So you do three as a residency three is a fellowship, depending on what you do. And it became a running joke between all these people that I basically did. I don't even want to compare it. But as we've discussed before, when you rush or go through basic training, or you do residency, you get very close to these people, because you're all have this common bond. Oh, yeah, you're kind of getting beat down a little bit. But you're doing it together, but you're doing it together. Yeah. So in residency, I had friends that became cardiologists nephrologist. I'm a gastroenterologist. And there would always be this running joke about who has the smartest organ, silly, nerdy arguments, the kidneys smarter than the GI tract, and so on. And I would always go will the anus is the smartest thing, it could actually determine, can your organ determine the consistency between a gas liquid or solid? No other organ can do that? No. So I've always said the innocent, smartest. And I was gonna joke around about how a new study just came out that basically makes my system the winner. And it starts with, I kid you not, it's a bunch of new sites that have come out citing a scientific study out of Japan, saying how mammals, mice and pigs at least have the capability of breathing. They said breathing through their butts was the title. I had to stop and looked at it. And it was an Associated Press article, or a BBC or a New York Times, they're all jumping on this, because it's a bit of clickbait. And you and I were talking about it today. And I said, Yeah, basically, the rectum, or the distal rectum absorbs is anything that's put in there so readily, that this, the scientists figured out that they could hyper oxygenate the rectum, while not allowing the animal to breathe. And the animal stayed alive and corrected, its hypoxia. And then you started asking all these scientific questions as an anesthesiologist should?Eric Rieger 17:51 I didn't know, I was just curious if oxygen going in is a very, very important part of the process. But at the same time, what's the vehicle or the means for the waste gas, co2 and other things to be blown off? Because as co2 builds up, you become more acidotic? And that's where organ systems can, you know, this is a gastroenterologist, they can fail. So they become too acidotic. And that becomes a problem. However, it turns into, I think that that question we were like, like, this isn't just a joke topic. This is something that Yeah, soKen Brown 18:25 I came in with a joke, I was gonna make fun of it. And they asked me questions. So then I had to do the thing that we do. And I tracked down the original article. And it's thick, and it's beautiful as the scientists should not be made a joke of which is what their bit what's being done. I guess any press is better than no press. But it's quite elegant. And I would like to discuss this in the future where you walk through this article as a, a expert in the acid base physiology due to your anesthesia training into how you can oxygenate, but it is kind of an interesting phenomenon where maybe we could use something like this instead of making fun of it and look at that and say, Look, in certain situations, maybe some form of rectal oxygenation hunter says, You and I talked about, because we're really into rectal delivery of other things. So which, which, honestly, decades or centuries ago would have been odd to think that you can actually give a medicine through the rectum. That doesn't make sense, right? Yeah. SoEric Rieger 19:31 someone did it. They demonstrated that it works. So obviously, they may be getting made fun of today. But who knows, if somebody has an injured lung? Maybe this is a way that you can help someone who's in distress. And I can't imagine one's going to do it voluntarily.Ken Brown 19:46 Why No, I mean, like, I mean, who knows? 50 years from maybe they will 50 years from now you may walk into a hospital code saying where the patient's crashing, and it's just like we need mouth to buy Right nowEric Rieger 20:00 I won't work. I will not work there. I've been called things where maybe people have thought that I must breathe through. NowKen Brown 20:10 that is, so that is an example of just being I came here to make fun of it. And then you started questioning the science. And I went, you're right. Whoa. So we'll talk about that some other time. I changed my mind, I'm I am accepting that. I should not hang on to certain ideas, because our knowledge changes our experience change. And if we as everyone should not just completely hang on to idea because the idea is not you. It's just an idea.Eric Rieger 20:42 Yeah, that's, that's a really good point. And I would also add that if in 10 years, I have all of the same beliefs and whatever that I do today, that I'm not grown. I'veKen Brown 20:55 learned nothing. That's a Mohammed Ali quote. I wasn't really Yeah, it really is. It was he? He's, he's a boxer. No, I think he was actually quoted as saying, Do you feel like you're the same man now than you were whatever, there were there. Were kind of implying that you're, you're 40. And it was 30. And his response was, if I'm the same man, I was then then I just wasted 10 years.Eric Rieger 21:17 Oh, yeah. Oh, I've heard that. Yeah. But I'm sure that's subconsciously in there somewhere. But no, I feel that way. Because I'm not the same person that I was 10 years ago, or 20 year old me, probably and I hate I wouldn't be free. Anyway, so what's up?Ken Brown 21:35 Alright, so let's get into some other cool stuff. We're gonna, we're, you know, this is the part three. It's there's a little bit of science here, but it all makes sense. And I want if you're listening to this, when I keep talking about the different aspects of either reptile dysfunction, or the CNS, or the endocannabinoid system, picture, how it works everywhere, it's not selective. That's the key to this. People are not putting this together, they're not realizing that you can take control of your health in a natural way. And it's not conjecture, it's based on science. So that's what this is. Okay, so to recap, in part one, erectile dysfunction, we showed that inflammatory damage from COVID actually causes erectile dysfunction by affecting the endothelial cells, which is the lining of the blood vessels. Part Two was how effective poly phenol zz are in working as a potent anti oxidant and blocking inflammatory pathways, which ultimately increases nitric oxide, and also activating the cert one pathway. So after we get done with that episode, okay, you and I went to the next room, and you started asking me all these questions about nitric oxide?Eric Rieger 22:55 Yeah, I did. Because you had, you had initiated, something I didn't. I didn't know. And I don't even know that I fully understand it. But that nitric oxide can exist in different forms just as the compound itself.Ken Brown 23:08 So as it is, it's like, shit I didn't have to answer. And so I'm like, Oh, my gosh, now I got to go down this, this, you have to lay the foundation to be able to understand everything else.Eric Rieger 23:17 Yeah. And just to add, you had labeled nitric oxide, and essentially, there was neuronal nitric oxide. I've already forgotten the other two thatKen Brown 23:27 I'll go through right now for you don't even worry about it. Because I think that's what that is the really cool thing. So when people talk about nitric oxide, one of the things that I have heard before and in our adaptive or in our innate immune lecture, we talked about how nitric oxide can actually be a pro inflammatory. And so meaning you can cause inflammation. So now I figured some stuff out. To make nitric oxide, there has to be an enzyme called nitric oxide synthase. These are a family of enzymes that catalyze what they do is they're the thing that makes the reaction happen to produce nitric oxide from an amino acid called l arginine. So nitric oxide is a cellular signaling molecule and it helps modulate tons of stuff like vascular tone, insulin secretion, airway tone, peristalsis, which is gut movement, and it's involved in angiogenesis, which is blood vessel growth, in addition to that neuronal development, meaning your neurons, and shockingly I did not notice it works as a neurotransmitter as well. So basically, we're talking the whole body. It does unique things in unique places. So as Eric was mentioning, there's three main types. There's inducible, nitric oxide, I nos. So I nos is a pro inflammatory nitric oxide synthase enzyme. So what it does, it helps acute insults so if you get infected by a virus infected by bacteria, your ionos will go up. And during an acute infection, it helps get rid of everything thereEric Rieger 25:09 does want to clarify, you're saying that's the enzyme not nitric oxide, I nos is the enzyme,Ken Brown 25:14 yes or no. So as it turns out, an imbalance of nitric oxide will result in oxidation. But we're going to get into this. And this is where it gets really neat. The AI nos has a specific role in making sure that there's an inflammatory process that happens. Okay, so I'm just saying that nitric oxide synthase. So if you're out there buying supplements, and you're like, oh, man, it's increases nitric oxide. You want to know, how is it? Yeah, arginine is at the substrate. Yeah, because if you're somebody with chronic inflammation, tons of inflammation that's going on, and you start beating yourself, Argentine, theoretically, you could be feeding your ionos. Because, like an acute infection with a bacteria or virus, you want this to happen, because you want to squash that. But if you have chronic inflammatory cytokines going on, it will actually induce more eyeness. So in other words, if you're chronically inflamed, yeah, taking Argentine could be fueling the fire that's doing it. So if you have chronic inflammation, I nos has been been associated with creating more of the inflammatory cascade, which results in more ionos, which results in a vicious circle. Ultimately, I nos is been implicated in being very high in autoimmune disease, Parkinson's, Alzheimer's, multiple sclerosis, and even strokes. So when we talk about nitric oxide, it is all about the balance and about where it's happening. And Angie sent me a really cool article explaining specifically about how certain cytokines will induce ionos. But that's irrelevant. Bottom line is, if you are chronically inflamed, and your eye nos is using this against you, so it's using your fuel against you, you have to stop the chronic inflammation first.Eric Rieger 27:05 That makes sense, though, because the probably the most notable activity of nitric oxide on its surface, regardless of where it comes from, is basal dilation and a key part or key component of an inflammatory process just like carrying away anything else is recruitment, recruitment of cytokines or whatever needs to be so it makes it makes sense.Ken Brown 27:26 Yeah, that that actually makes sense. And that was surprising to me, because I did not know about this until I started researching for this. The second type that we talked about on the second episode is endothelial nitric oxide, e nos. This is found in the lining of blood vessels. This aids in tissue repair, regeneration. And the key here is it enhances blood flow, and I did not know this. It dissolves plaques. Hmm. And it will dilate the blood vessel. Yeah, so we talked about this during the poly phenol episode. And it does this by several different ways, but it dramatically activates endothelial endothelial, nitric oxide synthase in us. And so that's one of the ways that does it. And another really simple way to do it is to have is to go exercise when you exercise, you release enas. And that's what allows for the blood vessel dilation and everything. Okay, I bring up the plaques because as we sit there and talk about if you have coronary artery disease, and you're put on Plavix and things like that, nobody's discussing this. Now. How do you turn on your emails, it gets rid of plaques increase the blood vessels. It's doing what these pharmacologic agents are hoping to do without the side effects of a cerebral bleed are these different issues? Interesting something to think about? If you can turn on the enzyme, which does this, then you can do it through exercise and or through various things. We're going to get into all of them.Eric Rieger 28:58 Something just popped in my head. Of course, we've talked previously, you've probably year and a half ago, about different diet intervals, fasting and intermittent fasting, and how those have been shown especially like water fasting for over 72 hours, I think it is, can demonstrate or they've shown that you can I think valter Longo talked about you could initiate the decrease in plaques and stuff like me. Is that related, do you think to enough? Actually,Ken Brown 29:30 I absolutely think that there is something about that. I think that there's some other components to it. Dr. William Davis discussed how as a cardiothoracic surgeon or cardiologist remember what he is. But he said that he's taking people that had 90% plaques and put them on a five day fast and then redid their AMG grant angiogram. Yeah. And it was clean. So there is something to this. So if you're going to do this, and if you have fast you turn on, I mean, I'm speculating. Now if you turn on your stem cells and you increase The nos, you're going to you need something to clear the plaques. Yeah, dilate the blood vessels. So you don't cause a clot. Think about that. It dilates. And if you've got plaques there, it shifts them off, and it's in a big pipe, so it doesn't stick. Yeah. If those plaques shear off into what we discussed in the last episode, where it creates the bumps, hung up is the thumb over the hose thing. It's the thumb over the hose. It'll just block it. So enough, very important. Interesting. Now, you and I are always about the brain. So now let's talk about neuronal nitric oxide and nos. This acts on the brain in the nervous system enhances the ability of the brain and nervous system to adapt to change, and enhance the communication between neurons and to recover from insult. So enmasse modulates neuronal function through release of neuro transmitters, when you said earlier, is that the nitric oxide, they have actually proven that and nos can do things directly. Okay, I nos can do things directly. He nos can do things directly independent of nitric oxide. Wow. Okay. And they get turned on for different reasons. So this has been shown to be involved in long term prevention of depression, and improved memory formation. So when you have en nos going on, and it's also part of the neural plasticity and regeneration. I'm a big fan of the Huberman podcast and he, his whole, his whole theme is neural plasticity. And this guy hasn't even talked about this yet. talks about everything else, but I'm like, calm and start talking some and so if you if you know him, or whatever it's yell, you know, you're not even talking in nice, man, you're just scratching the surface. But um, you could say it like that also. Alright, so here's the part that we were confused by. Okay. So your question to me was, I don't get how nitric oxide functions differently in different places. And I just stared at you blankly after our episode notEric Rieger 32:06 clarified not just as a molecule unless it's being contained, or specifically only releases certain time that that was that was our discussion.Ken Brown 32:13 Yes. So as it turns out, nitric oxide is nitric oxide. Okay. So it's not like there's different nitric oxides that we know of right now. But the different nitric oxide synthase is caused different responses. So first would be the amount of nitric oxide produced too much, and you can't clear it, it becomes a pro inflammatory,Eric Rieger 32:36 oxidized, right?Ken Brown 32:36 It'll oxidize, okay. That's the first thing and the second that each type of these enzymes can actually have a direct effect on the cells in the local area. So like, in the case of ionos, it can actually increase oxidative stress through superoxide production, or en nos can have direct neurotransmitter capabilities. So a lot of science just there. It's allEric Rieger 33:02 brand new, but that that brings some clarity thatKen Brown 33:05 it brings some clarity, which which will bring clarity to all these other studies I'm going to bring up and we're going to go through really quickly and by all I just made a few, but it it reiterates that what we're talking about and what you can do for your health is based on scienceEric Rieger 33:18 now that's fantastic.Ken Brown 33:20 Okay, so while I was figuring out all this nitric oxide stuff, this is actually where this went down a rabbit hole. I kept finding studies that they kept discussing the endocannabinoid system and nitric oxide. And I ended up going down some rabbit holes and discovered some studies on how cannabidiol CBD and the endocannabinoid system is involved in this whole process. And ultimately, how it relates to erectile dysfunction because if you're like this is part three of erectile dysfunction. I haven't heard anything about that yet.Eric Rieger 33:52 Yeah, well, it is where we talk to them as part of an inflammatory process check jack and is the ECS and have a problem with with too much inflammatory process? Yes, yes,Ken Brown 34:03 sir. It does. Alright, so ECS? endocannabinoid system. Eric, can you just recap what it is? Oh,Eric Rieger 34:10 no cannabinoid system we have it all throughout our entire body. It basically is the copyright strikes the balance between reacting to something but not overreacting. Let's see here. You got to get some some cannabis. Or I'm sorry, I'm gonna say the right the neurotransmitters that we have or the cannabinoids, endogenous Canada in the largestKen Brown 34:31 categoryEric Rieger 34:33 was tripping over my own words but so you've had an and amide into ag. There's the two main ones and then to augment that activity, you can take exogenous which means you consume it. cannabinoids, THC and CBD with CBD being the one that we legally use to attenuate or lessen issues of inflammatory process, absolutelyKen Brown 34:56 by increasing your own endocannabinoids and probably a little bit of direct stimulation as the science becomes more but yeah,Eric Rieger 35:03 see it so and like everything in the body, you know, neurotransmitters are consumed by enzymes. And so what CBD they think does it doesn't necessarily replace an end of mind, for instance, but I think what it does or it can do is it will stop some of the hyperactivity of the enzyme that gobbles up an animal.Ken Brown 35:24 Correct. So I think that's pretty much what you I think that's exactly what I want to hear. Yeah, that's, that's exactly. Alright. So knowing that the endocannabinoid system, if you've not read about that, or if you've heard some things about CBD, that it's shocking how much science there is on all of this, and how nobody really talks about this. And it changes and it changes. And it changes when scientists do stuff. But let's talk about this. Alright, so I found some really cool stuff going down this rabbit hole. At first, they were talking about three studies briefly out of the same university in Egypt, because I think that they knocked over the first domino and went, well, what happens when this happens? What happens when this happens? And it's actually really cool because it applies to humans directly. So the key to all these studies is how the nitric oxide synthase pathway was regulated, and it's regulated independent of the colon nergic pathway, which in the episode 54, we went into great detail about Yeah, so in other words, it's another way to stimulate nitric oxide synthase these guys figured this out. Okay, so the first paper that they published, looked at how the CB one receptor interacts with enough endothelial nitric oxide synthase, they were able to show that the how the effect by increasing a Nandha mind, the endocannabinoid that Eric just talked about caused relaxation in the corpus cavernosa. corpus cavernosum are the spongy, blood filled vessels in your penis that allows for interaction. It was not through us through acetylcholine, which is how every other urology book describes erection. Yeah, now one medical book that I've come across actually talks about CB one doing this. So this is fascinating about this. So and it is doing this by actually increasing inas, the endothelial nitric oxide synthase. So by stimulating CB one through an and amide, they could actually show an increase of the e nos. And this resulted in increased corpus cavernosum blood flow.Eric Rieger 37:33 That's impressive. For a lot of different reasons. Number one, that no one's really glommed on to it or captured it. It's understandable that while you were in training that it hadn't been discussed at all, because I don't think that the the idea that an ECS existed until the 90s, so that that's okay. And so now you've got to figure out where all these functions are. But what what I found already and what you said is a change in science and really changing the way that I'm approaching it is I thought that for the most part, cb ones were in the CNS, or just the, you know, the brain and spinal cord.Ken Brown 38:08 That's a really good point. Yeah, because we've always been taught that they say that CB one is or I'm sorry, we haven't been taught in the most recent books have been published that discuss how CB one is more related to the neurologic aspects. These guys are showing that now send your penis also.Eric Rieger 38:21 Yeah, I don't even just started. Sometimes what we get accused of thinking withKen Brown 38:27 anyway, I gotta study for that also. So then the same group said, Well, wait a minute, we've just discovered something really cool. So they said, Well, what happens in disease? So they took some rats and they gave them cirrhosis? I'm not really to really get into the details, whatever, get into the, you know, like the methods of the study. Okay, so I picture a bunch of really cool rats hanging out at a bar for hours on end. They're like, Hey, man, we're just talking about we got this great lab gig and they didn't know that there that they were causing cirrhosis. I think it was done in a very different way. But I don't think I imagined it that way. Yeah, that you can imagine that way. I'm gonna imagine it that way. So what they did is they cause cirrhosis in rats, and then they were able to demonstrate enhanced relaxation of the corpus cavernosum those spongy tissues. In other words, they showed increased blood flow, when the CB one system was activated once again using an and amide then they wanted to have the cause and effect. And what they did is they blocked the CB one receptor, and there was nothing happened. So it was definitely related to the endocannabinoid system, not some other weird thing that we haven't figured out. This is the cause and effect of this receptor stimulation. So the unique thing about this is even in a disease state like cirrhosis, it helped these little rats to have directions directions. Yeah. Which is interesting because they're talking about cirrhosis. And remember the episode that we actually debunked how the FDA came out and said that CBD causes cirrhosis? Oh,Eric Rieger 40:04 yeah, I recall that. And it was completely erroneous.Ken Brown 40:08 Yeah, it's completely erroneous. And it didn't make any sense at all. Where is that we got one of those apps. I don't even know what episode it was. But we actually talked about that. And this is an example of Look, this is a way to take a certain disease, which generally has severe erectile dysfunction and they're like, now if you stimulate this looks like you can overcome it in different way. So that's cirrhosis, but you're like, ah, only a small percentage of drunken rats gets a very small percentage.Eric Rieger 40:40 But unfortunately, that's not true either.Ken Brown 40:44 But there's a there's a much higher percentage of donut loving rats that have Yeah, so they got so excited after that first study that they did a third study where they looked at the endocannabinoid system in diabetic rats Okay, then they showed on this study that the first thing is once they made the the rats diabetic, their baseline endothelial nitric oxide synthase, Enos was lower than all the non diabetic rats Think about that for a moment, okay, by having diabetes, your e nos is lower. In other words, your ability to produce nitric oxide becomes lessEric Rieger 41:25 Yeah, and and all the ramifications that you talked about what he nos actually does vascular, you're losing that ability, you'reKen Brown 41:32 losing it, it's not just in your penis, you're losing it everywhere, and nobody's talking about that aspect, which is, why don't we focus on something to increase the e nos. Alright, so by adding a Nanda amide, directly, they showed increased vascular activity in the corpus callosum and increased nitric oxide consistent, proportionally to the increase in nitric oxide synthase or enas. Now this part is really cool. When they added Argentine l arginine, which is a substrate to make nitric oxide, it synergistically went up. Make sense? So if you have a proper diet, and you are have a balanced endocannabinoid system, even if you have diabetes, your ability to have an erection is should go up based on these animal studies. And you know, we're not making any claims here. I'm just saying, based on these studies, it's super interesting to talk about.Eric Rieger 42:26 Yeah, no, it's it's, it's fascinating. I mean, and let's, let's call it what it is the the meds that have been approved to treat erectile dysfunction, they essentially they got here, we talked about the five different ways or the five different things that have to happen for an erection to occur. Essentially, what viagara is doing is it's stopping the phosphate arrays from breaking down. So this is what that that was a valid mechanism to address. This is yet another, in my opinion, valid mechanism to addressKen Brown 42:57 Yeah, so viagara doesn't work until you've arched actually formed the nitric oxide, right. So once you form it, you can have a longer sustained direction due to not breaking down the nitric oxide. man you got to get it started someplace. Well, one great way to do it looks like is to make sure that you have a balanced endocannabinoid system.Eric Rieger 43:16 Yeah, no, that makes sense. And I've never heard that before. And right now,Ken Brown 43:20 all of this is, all of this is new to me. I've got to be totally honest. And it was all sparked off of you asking you a question. I don't really get the whole, why you can act differently. And then this led to these things. Thank goodness. And that's what I love about hanging out with you is that you ask questions. You got to I mean, there's times where you're not been there and you're just like, Why is the sky blue? And why is why do my feet feel so heavy and I just have to look up everything.Eric Rieger 43:48 I can't even come up with something ridiculous.Ken Brown 43:52 Alright, so then, here's another interesting study by a totally different group. They were looking at it in a more indirect way. So your response of which head is actually responsible for the sexual function. These guys took some of this information and I bring this up, because this is looking at the end neurone the end nos process or the neuronal nitric oxide synthase. They looked at how the endocannabinoid system helps improve neuronal and Nos. and in prior studies, it was shown that a NAND amide helps not only form and nos, but it also works to drive the nitric oxide into neuronal cells. So and amide goes, Hey, and nos we need some more nitric oxide. They produce it and then of my grabs, it shoves it in a neuron.Eric Rieger 44:47 Maybe I'm leaving too far ahead, but this sounds like neuroplasticity could be enhanced simply with a healthy ECSKen Brown 44:56 100%. So in this study, what they did is they took neuronal cells. And they showed how stimulation of the CB one leads to a massive increase in neuronal nitric oxide. Now, if you say massive, Well, isn't that inflammatory? If you have something that puts it in a cell and puts it to using us, it's been you. Yeah, it's not accumulating. Yeah. So the CB one receptor and the N nos protein have been shown to be in close proximity. So, and randomize here, cb one receptors here, I'm sorry, the nitric oxide synthase stimulation is here, a Nana Midas or the CB one receptors here. And when they're together, this is where nitric oxide will help not only the neuron that they live in, but since it can move over, and it is nitric oxide, which is a gas I guess. It moves over and it can work in what's called a parrot grind function. In other words, it can help its neighbors. So if a cell gets injured, yeah, and sends a signal and your Endocannabinoid is healthy, it'll stimulate en nos and say, let's get some nitric oxide over here Help Help my friend who's just been injured. Fascinating,Eric Rieger 46:13 very, and that's the first time that there's been any of this is very superficial, but it's the first time I've ever felt like there's context or who was it that was talking to us about CBD, and that they were studying it for Ms stuff. Because if you're thinking about how ms functions and the breakdown, a lot of that comes down to the schwann cells file and and she's breaking my own sheath. And so you're you're losing transmission, you're you're leaking it's there's no insulation on the long nerve. So what if that role of CBD in part is that not only is it staving off inflammation, but it's actually allowing other neurons in the area to donate help to one that is distressed? I mean, we're leaving here. We don't have that in front of us. But I'm curious.Ken Brown 47:05 That's pretty much what they're saying though. Yeah, that's pretty much what they're saying, at least in this animal model that interesting. So yeah, so this is where nitric oxide can help the neuron that it's in, in the neurons that it's nearby. So because the two receptors are nearby, that's how they actually do this. And then this is the leap for the indirect erectile dysfunction. This kind of surprised me also, they actually act on the so this is we're talking about the endocannabinoid system, cb one receptors and act on the gonadotropin releasing hormone. So the endocannabinoid system can help with both the neuro component of erectile dysfunction as well as the endothelial component. So by releasing gonadotropin releasing a by stimulating gonadotropin releasing hormone that results in an increased testosterone.Eric Rieger 47:57 That's fantasticKen Brown 47:58 or estrogen because it's again, a trouble releasing Yeah, everything's in balance. Everything is not black and white is the yin and yang of the body and you need enough of everything. That's the beauty of the endocannabinoid system. It's our great balancer. Like you said it was the it's the traffic cop that says more or lessEric Rieger 48:15 Yeah, as fans. Fascinating I've never heard this stuff.Ken Brown 48:20 It's I was pretty shot. This is this is what's fun about doing this podcast is that you pose a question and then I'm like, dang it. I'm definitely not staying up because sleep is super important. If you don't sleep, you screw it up. But I will wake up early and go, Well, I'm not gonna work out today, I'm going to go down these rabbit holes. All right. And so oh my gosh, there's so much more research out there that we can't even get into today. But like, like this title of this article that was published recently, cannabis dial induces on top of G to protect neuro neural cells from mitochondrial dysfunction by up regulating cert one to inhibit NF Kappa beta and notch pathways. That's the title. So we've talked about all these things, and it sounds super complex, and nobody would probably ever read this, but it's actually a really good read because what they showed is that CBD protects neurons by turning on the cert one pathway, which we talked about last episode, causing a toughie G, which is programmed cell death in old and sick cells, while down regulating inflammation through the NF Kappa beta pathway. Sounds freakley familiar to how polyphenols do it.Eric Rieger 49:34 Yeah, it really does. I mean, and you can throw in there the cuz I think that last week, we labeled NF Kappa beta as the black hat cowboy. Oh, yeah. And then we said that the white hat cowboy would be a nerf to NRF two. And so it, it seems to me like these are simply complements that we are uncovering that could Be a far more natural way for your body to determine. Look, just give me the tools for the tool chest. I'll figure out what things goKen Brown 50:08 100% another article I gotta read this title because these scientists need always in the titlesEric Rieger 50:17 thatKen Brown 50:18 they need a marketing agent. Let's get this know that that breathe through your bot is all over the news.Eric Rieger 50:27 Yeah, that's a click. That's a clickbait clickbaitKen Brown 50:29 and it's way more than that these guys put a title that says CBG CBD Delta nine THC CBN CB g a CB, d A and delta nine t. h ca as antioxidant agents, and they're in intervention abilities in antioxidant action.Eric Rieger 50:48 It's important stuff and no one's gonna read that. Bob, doKen Brown 50:51 you want all that in there? Yeah, I want it all. We're all out of seas. Yeah, we're all out of seeds. Alright, so the title is too much. But basically what they presented was that they reported the antioxidant activity of CBG, CBD and all those other ones. And they present a data to prove that all the examine cannabinoids named exhibit potent anti oxidant activity in their ability to scavenge to scavenge free radicals to prevent the oxidative process. So very last episode, we're talking about how polyphenols are potent antioxidants. nobody's really discussing how CBD and THC and CBG are potent antioxidants. But they the whole study is how they actually do it, how they scavenge free radicals.Eric Rieger 51:44 And it's brilliant, honestly. And it just, it just lends more credence to why it's important to have a healthy ECS. I mean, I know that those are all exogamous that you read off, I think for the most part, but they from what I understand that ECS to date, they really serve complement to your Endocannabinoid. So exactlyKen Brown 52:03 an exhaust meaning these are the ones that we take in through our diet, or through smoking or whatever if, if it's that, but these are the ones that we ingest that actually do that.Eric Rieger 52:13 Yeah. Fascinating.Ken Brown 52:14 It's totally fascinating. And then oh, my gosh, one last one, because this one blew my mind, as I'm sitting there looking at these different articles, and you're reading through them. And it's the sad part is, as we're preparing for this, there's only so much we can actually talk about right here. But like, I want to call people I'm reading I'm like, Are you kidding me? This is nuts. Like I want to call the guy that made that title and be like, here's the deal. Next time you do a paper, let me write the title for you. And it'll be clickbait all the way his scientific colleagues will just be like, what kind of title is that? And it's just like, CBD causes you know, decreased baldness and it's like, but that's not really what it's about. It's about the telomeres and it's about you know, the the flu avian cells and he's like, Yeah, but and this guy said baldness, and I just want to Pulitzer. Yeah.Eric Rieger 53:03 I mean, that's really what it comes down to. You've got to get it language that someone's gonna read 100Ken Brown 53:07 Exactly. Alright, so we did a whole episode on Brock elite. We did. So love broccoli. It's on the KVM the health store. And broccoli is the really world's only stable sulfur fame product. We did this with David Roberts and dr. john Gill de wickedly smart people, both of them. That's Episode 44. highly suggest that you go take a look at that. Because of that knowledge that we learned on that show. I was I came across an interview quite a while ago actually with a doctor Jed Fahy who, what's her name, Rhonda Patrick. And he was discussing how his research was showing that a there was some similarities between a isocyanate called more engine is similar to sulforaphane not quite but similar. And his reasoning for this is that it's actually it's a tree that's very abundant in subtropical regions. And they should be looking at this for adding it to diets of certain things. Then remembering that I came across this article, which led me to start looking at various things. And this thing popped up anti inflammatory and antioxidant effects of CBD and more engine and lipid polysaccharides stimulated macrophages. So lipid polysaccharide is a protein on the outer. It makes up the outer shell of a bacteria. It's what our bodies react to. And it's also what causes inflammation. So what they did is they looked at how do macrophages, part of our innate immune system respond to stimulation when they're exposed to these lipid polysaccharides? In other words, when you feed a macrophage a piece of bacteria, what does it do? And they wanted to see how it reacts to pro inflammatory markers like TNF alpha interleukin. Sex etc. And they wanted to know how exposing the macrophages to CBD in one arm Morrigan in one arm, and then what happens if you put both of them together and see what happens. So remember that CBD has already been shown to have anti inflammatory anxiolytic anti cancer capabilities. More ringan is under this class of glucosinolates, like sulforaphane, and these molecules, these molecules are potent anti inflammatory regulators as well, in this case, it's called an isotherm cyanate meridian from the poly phenol Moringa. Okay, you see how it's all coming together here. Alright, so bottom line is the results are pretty cool. The pretreatment with either CBD or meridian resulted in significant decrease in anti inflammatory and antioxidant effects from CBD and more ringan separately, with decreases in TNF alpha, interleukin six and all the other inflammatory cytokines that we talked about. But when combined, they showed exponential decrease in inflammatory markers, while increasing NRF to white hat and good cytokines like interleukin 10. So the combination also enhance the nuclear intracellular level of antioxidants. So it KVM D on our store, we had the biohack combo, which is atrantil, CBD and broccoli. They did a study on the biohack combo. Yeah, they did. This is crazy. And it doesn't mean that you're not gonna be able to mount an anti inflammatory response. It's that living in the Western world, you're exposed to chronic inflammation. Yeah, chronic inflammation leads to all these things. So lots of articles, lots of rabbit holes to wrap up. We started with COVID, causing a reptile dysfunction and affecting endothelial cells. Then we went to how do you protect that by increasing e nos and decreasing inflammation with polyphenols? And now we've wrapped up with how CBD can help with this. And ultimately, the combination of sulforaphane plus the polyphenols and atrantil plus CBD, according to these studies, likely protect both your brain and your endothelial cells, ultimately, protecting your penis.Eric Rieger 57:22 Yeah, ultimately,Ken Brown 57:23 that's a lot of science to say, don't let COVID take your penis. Yeah, it'sEric Rieger 57:29 a lot of science to say exactly that we probably didn't even need to have you science to say just that.Ken Brown 57:36 Probably that one statement alone. Be like a T shirt.Eric Rieger 57:40 Yeah, we could have just said that, and probably called it a day. But now that, honestly, all of that is fascinating. It's informative. But it makes it makes sense. And even though it feels like that we go to like a local focal point to john Miller gets address interest. It just, it forced us to find out, like you said about nitric oxide and the different uses. And really, it's it's still universal. It's still universal throughout the entire human body. These are things that we need to be concerned about. And yes, we're talking about a penis today. But that process is happening there. It's the same thing that can be used to protect your brain, protect your heart, protect the rest of your organ systems, so that you can do what we were talking about the very beginning of the show, which is what kind of life would you like to live? How do you get there?Ken Brown 58:28 So I guess if we were to talk about the mission of this podcast, I would say that, you know, we've always talked about healthspan. But now I've got this whole new process, you cannot truly develop as an individual if you're being hindered by day to day aspects that prevent you from leading a normal life. Right? So it's really hard to say you should meditate when you don't have food on the table. Yeah. It's really hard to say you should go exercise when you have heart disease, and you can't, you can't exercise. I mean, there's all these aspects. So I guess the goal of the gut check project is to improve the health span.Eric Rieger 59:13 Now how 100% I mean, yeah, it's You're right. I like those examples telling, telling a homeless person to take in more fruits and vegetables. That's exactly a weird thing toKen Brown 59:24 Yes, they need those but there are other pressing needs so that they'll need to address so that they can tell you a parent that was two jobs to the weekend that they can't be home and they don't have food to give their kids to make sure that their kids don't eat cheap food. That's whatever it it. Just it doesn't happen that way now, and if we can, if we can do something, something becauseEric Rieger 59:49 maybe maybe people who watch this show or share the show. It's not necessarily that we're talking only to you but if you share with someone else And you are on your pathway to becoming healthier so that you're more vibrant than maybe that's kind of where it starts. I'm talking about myself too. I mean, I need to be healthy so that I can help take care of people that I'll take care of same for you. And same for anybody who's watching or listening. So you can't help somebody if you can't help yourself.Ken Brown 1:00:18 Yeah. And this is a good example that if you follow the show, or if you have a question, the questions turn into. In my mind, all of this is new to me. If it's not new to you, then you're way ahead of the game. As a guest, right now,Eric Rieger 1:00:37 yeah, yesterday.Ken Brown 1:00:39 But if this is new to you, as well, then please like share, we will always do the disclaimer that everything I've talked about today I am a doctor Eric is a certified crna. But we are not giving medical advice, while all we're doing is trying to show that there is some science behind all of this. And we are passionate about trying to make people healthier, ultimately, not just physically healthy. We're not talking about looking good. We want you to feel good. And we want you to be mentally healthy as well, because all of it is tied in together. And that's what we keep doing almost every single time is we're like wow, every time we want to separate. We want to do a three part episode on the penis, it ends up being the bodyEric Rieger 1:01:23 100% brain, we want you to stay healthy, mostly so that you can stay around long enough to ventilate through your anus. So we'reKen Brown 1:01:31 gonna get to that one. I want to make fun of it. I'm just gonna Heckle you the whole time. You have to show me the real science about why it's valid.Eric Rieger 1:01:40 Okay, that sounds great. I hope it's in there. But no, I'm excited that they're gonna be a good episode.Ken Brown 1:01:45 SoEric Rieger 1:01:46 that's Episode 56. Thank you all so much for hanging with us from 54 to 56 on this particular topic, it's very interesting. Hopefully, it's helpful. We've gotten not surprisingly, lots of feedback on anything discussing erectile dysfunction. Now I know why Pfizer cares so much about war. So anyway, thank you all for writing in and we'll see you Episode 57. I don't think it'll be a continuationKen Brown 1:02:11 conspiracy theory. Pfizer also came out with the first vaccine knowing that COVID can cause erectile dysfunction is the vaccine only partially is not where we need to buy. Bye, everybody.
Hello Gut check project fans and KB MD Health family I hope you're having a great day This is your host Eric Rieger soon to be joined on my awesome co host Dr. Kenneth Brown. If the audio sounds kind of funny on this intro, I'm just having to record it while doing a little traveling and regardless, it will not take away today's episode will not disappoint. This is about erectile dysfunction. What can be done about it? Who wants to deal with it doesn't matter if it's male or female. You don't want to have to be dealing with erectile dysfunction it you know the inappropriate time. So let's not waste any more of your time and get straight to the podcast. But first, our awesome sponsors artron to get your daily poly finos and ultra and to go to love my tummy.com slash KB MD. Love My tummy.com get your poly phenol calls today and of course unrefined bakery incredible food, go to unrefined bakery.com if you are a keto eater, a paleo eater, a gluten free eater or you just like great food that happens to be healthy for you. Go to unrefined bakery.com felt like you had to avoid good tasting cookies simply because you have celiac disease. Well, don't worry, they are certified gluten free and all of their products and get 20% off your entire first order with code. gut check that's unrefined bakery.com use code gut check and save 20% off your entire first order. And last but not least, go to KB Md health.com. If you'd like to check out the podcast and all of our products that we offer, then you can do so just head to KB Md health.com. You can find the home of the podcast all of our previous recordings from gut check project and you know get to know Dr. Brown and myself a little better and shoot us a message. All right. On to episode number 54 the reptile dysfunction Hello gut check project fans and KB MD health family. How are you doing today? I hope it's great. I am here for Episode 54 with my awesome co host Dr. Kenneth brown and today's topic. I think it's actually pretty popular even though when people have it. They don't like it. I was gonna say it's not popular. It's common. Oh yeah. Popular. Yes. No one really? Yeah. No. Today we're talking about erectile dysfunction Ed, and I'm doing this as gastroenterologist because we're seeing that the pandemic may have a strong effect on this but probably not in the way that you're thinking. So today's episode is going to really look at the rise in erectile dysfunction in younger men that are otherwise healthy. That's what we're going to talk about the rise in erectile dysfunction pun intended. Yes, apparently that is funny wording for you. But nice. So erectile dysfunction Episode 54. I don't you told me there's going to be surprised. Yeah. Yeah. So lately what I've been doing when I get Latin when I latch on to a topic, I pretty much don't tell Eric what we're gonna do. So just kind of see him. And so I thought today would be really funny to see how many times he giggles at the word reptile. So if we count it, it could it could turn into a drinking game. I'm not sure. Yeah. Okay. Well, fortunately, all natural, sparkling wine. So that's what we're gonna talk about today, reptile dysfunction. So before we jump into that, I have an ask of anybody who's listening to this. Okay. All right. So this is my ask for the public. I have an amazing team at my office and one of my amazing team members. It gave her two week notice and she's leaving for reasons like all people do, she's getting better opportunities. And I'm proud of her to do this. She helps her deal who is my scheduler work with my patients, and she helps these patients and it's been working really great. So what I'm asking is if anybody listening happens to be super cool, nice, loyal and wants to work with a great team. Or if you know anybody who happens to be super cool, loyal and wants work with a great team. I would like to find somebody to fill this role. There is no medical experience necessary. I'm just looking to hire the personality. So we're just trying to find these cool loyal people go to KB Md health.com kB Md health.com and just hit the Contact Us button. If you know of anybody. Sign up your friends family, we just I really am very proud of my team. I'm very proud of the way that my team takes care of my patients and I'm really going to fill this pretty tough void so I'm reaching out to everybody. Yeah, and if you're watching this show, from far away from Plano does not include commute travel expenses or per diems. So you're probably going to want to be in the area of the clip probably want to be local. Yeah. On your way. And if you're thinking of moving the plane over for Go anyways, I would go ahead and do that. And so, all right, let's do the usual Eric, what's going on with your life? We're about to renovate our house we moved into about a year ago. And actually today after we finish filming, I've got to go to a builders meeting. And so this is a this is a first a true renovation of a home that we that we live in and feeling that anxious but gonna be displaced for a little bit so I'm not super excited about it, but at the same time, we'll be okay. Yeah, I mean, I would just to ease your anxiety if you learn more about homebuilding Oh, then you'll feel more comfortable. So I'm gonna recommend a movie called the money pit with Tom Hanks. That is not the movie. Showing long and Tom, you'll learn a lot about that. Yeah, I'd say never heard of this movie. Doesn't he say home crap home. As far as me, I've got Lucas in Costa Rica. Today's international tournament right now is in the quarters currently playing. Lucas, let's give it to him. And Carla will be playing out of town this weekend for a large tournament as well. So we got the usual thing going on. And I'm getting my stem cells next week. You are you're getting stem cells. In fact, he even came down with a little bit of extra pain yesterday in your neck. I did. Yeah. So it's interesting. I'm really excited to see what this will do. I hurt my neck. surgery was recommended. I've been really trying to avoid surgery. Dr. Wade McKenna was a guest on our show early on. And he's really one of the world's leading stem cells. Definitely. I just happen to be fortunate enough that he's in the DFW Metroplex. So, him and Dr. Jerry Lewis are going to work their magic and I'm going to get a stem cell injection plus a stem cell infusion. And what you and I were talking and yeah, so everyone. So as I do scopes, everyone saw you a little pain, I'll be like, it's I'm still gonna do it. We were talking, you came across some literature, which we will do a whole show on after I do this. All right, about how polyphenols can augment the stem cell stuff, right? Yeah. So there's a handful of them and kind of on a whim, just knowing that we're research based and wait is certainly research based. And we've talked many times with him about how can we complement each other? Because that way, it's very altruistic. And if you have, if you don't know that, just listen to the episode. And you can learn a lot about what he's given up just to give his patients the best service. But knowing that I was really curious on what do polyphenols do, they could augment what it is that Wade McKenna does at his office. And I mean, it's a simple Google search turned up a lot of articles about, well, I don't want to get into it and give it all away. But yeah, it's it's there. Well, it's interesting, because you and I both do intermittent fasting. And we know that intermittent fasting causes a tapa G and fasting long enough turns on stem cells, and we know the polyphenols are fasting the medic molecule, correct. So that at least we know that way. But we're gonna do a whole show on that. And we might even be able to get one of these cool scientists come in and talk about it, you know, like we did with Charlene and Sylvia, which I still get a lot of feedback from patients about that. No kidding, is deep, but I was I'm really proud of our audience that we have. Because y'all, you stick to it, and you really dig in deep to the to the material. And I mean, how many times do we in the show going, man, I hope that wasn't too thick. And the next thing you know, we get email and patients coming in saying that they love the episode had no idea that such and such worked. And sometimes I'm like, did we say that? No. Is that what they told us? Yeah, we're getting emails from really all over the world, which is really cool that we got people listening in all different kinds of places. Sure. Um, last episode, we talked about what month is this what day it is, and as it turns out, April right now is stress Awareness Month. Oh, nice stress Awareness Month. And interestingly, we just got our paper published, we just got our study published in the clinical case reports and reviews at the beginning of COVID. To try and help the frontline workers manage it. The title is gastrointestinal symptom and stress recall survey in frontline health care, healthcare workers after consumption of combined quebracho conquer tree embell Salma a wild extract, what that is, is that's the generic term for atrantil. So this will talk about this study on a different date, but I'm very happy that it got published because it was really good results. And as we're gonna get into further into this episode, and then further into the next episode, right, we're gonna get into how maybe polyphenols are really affecting your stress level and different things. Yeah. So and I know what you're gonna do before you interrupt me, I know what you're gonna do. You're like, hey, it's not just stress Awareness Month in April, I realized that April is actually national Awareness Month of a whole bunch of things, like a whole bunch of things like Autism Awareness Month, which is very important, very important. And then and I'm not kidding here. It is. National safe. Digging month is that we have to call test dig before you hit a line. I have no idea but I know you dug deep on that. I mean, I can just imagine this, you know, I mean, like, for God's sakes, Bill, you're just such a reckless digger. It's April, this is the one month where you got to not do that. It's April. This is not reckless digging. It's safe digging bill. So I just imagine conversations like that happening. I, I'm not kidding, I think you dial 311 and you find out if they can send someone out to your property and tell you if there's a line or a pipe or something like that before you dig. They must get inundated in April. Well, I must have to hire like they couldn't have done it. late February because we were iced over. That's true. So now now's when people I remember being snowed over and you are like, don't you just wish this was all thought? Dig? I thought for sure you'd at least wonder who bill is and why such a reckless digger. You know? Well, I thought everybody knows who bill is. I know Bill's my pet badger. So Bill, and I've been at odds for a long time about his digging habits at my house. So I'm just hoping that Well, hopefully Bill's calling 311 I don't know. I'm not. I don't know, Bill. I got him because he was supposed to be the sweet badger. It's called a honey badger. But he's not sweet at all. Oh, not even Nice. Yeah, no, he's not they never are. Did you have skunks are a badger is a form of badger. I didn't know that. Yeah, well, not honey badger. They do not smell like honey whatsoever. So Bill causes a lot of stress in my life digging everywhere without calling 311. Before doing this a national safe digging month. Why do we even care about stress? Stress affects so many things? Oh, wow. Yeah, super important. Because stress can actually those headaches, you're having the insomnia that's going on this severe fatigue that you're having, you're going to these doctors seeing stuff when actually it could be stress. So it's important to pay attention. And I got to add another layer of stress here. Okay, hate to do this. This is already a very, very stressful time in the pandemic, everything's going on. There is new evidence coming out that COVID is now causing chronic erectile dysfunction in younger men who were completely otherwise healthy. That's terrible. I know. It's like it's not a bad enough virus already. But so do do the link this back to stress. And where we're going, we're gonna get into this. So actually, this has actually come up in my clinic several times. So you know, we did the episode about hemorrhoids last time, I do treat a lot of hemorrhoids. And once you're doing that with somebody, I think that they feel comfortable enough bringing up other potentially difficult topics like erectile dysfunction. So I've been seeing a trend of younger men asking me questions about erectile dysfunction. And after the first one mentioned that this kind of all started after they had COVID. I started asking, and most of these people have had COVID. Interesting. Yeah. So this is why we're doing this episode, I went down a proverbial badger hole. And we're gonna try and solve this just trying to sort kind of our categories here are these same men? I guess you mentioned hemorrhoids. Are these same people also afflicted with hemorrhoids? If I do hemorrhoids, what's going on there? Well, it's this is an interesting topic also, because that's something else that I want to look into in the future, there is some correlation with large hemorrhoids leading to what's called venous Steele syndrome is a whole separate topic. It just happens to be that the people I treat are also the same ones that are coming to me for hemorrhoids, right? So if all you have is a hammer that everything's a nail, Cheryl, I'm trying to think outside the box and go, Why in the world would this do this? And so I started researching it. And lo and behold, a new report was just published, which confirmed my suspicion. So a study was just recently published out of Italy, showing that COVID-19 increases the risk of developing chronic erectile dysfunction, nearly six fold. I must say that again, if you get COVID, and you're a young male, you may have thought that it was perfectly smooth and easy. And you got through it. You have a six fold increase in developing erectile dysfunction. It's terrible. And then they backtracked with the data a little bit. And then they realize that those that were already diagnosed with erectile dysfunction had a five fold increase of contracting the virus. That works now both ways. The second part makes a little bit of sense, and we'll get into that, but this study was led by Dr. Giannini, who is a professor of endocrinology and medical sexology at the University of Rome. It was published in andrology. I just liked the idea of being double board certified and sexology and endocrinology. Yeah, I didn't know that was an option. I didn't know it was either. I'm gonna go get double board. That's it. I mean, it doesn't sound like it wouldn't be entertaining. This was just published just last month. In this paper, they explained that ultimately, the underlying issue and erectile dysfunction is endothelial dysfunction attorneys gonna keep coming up over and over. Okay? Now this is a super complex process. And I'm going to go into this in in detail in just a few minutes. But basically in their study design, they did an online survey early on in the pandemic. So they discovered this very early. We don't know what these numbers are now that we're a year into the pandemic. And they compare to erectile dysfunction. People that fill out the survey versus non erectile dysfunction groups EDI for short, and accounted for other variabilities like psychological stress locked down financial stress, they took those out, they're looking strictly at COVID at COVID on this one. So they were able to get 100 adults, and they use what's called a logistic regression model, which is why you hire statisticians to do these kind of papers for you. Yes. And they came up with those numbers, that after getting COVID, the likelihood of developing EDI was six fold higher than those that were not infected. Wow, I crazy, right? And their conclusion is that erectile dysfunction is all about the endo phillium. And those are the layers that line the blood vessels right now it's starting to go, that makes sense. Because we know that COVID creates a lot of different problems. And so that's when I went, Oh, my gosh, this makes sense. So, in there, Dr. Giannini must have a sense of humor, because in the paper, they actually said you should, it gives a whole new meaning to wearing the mask mask up to keep it up is what the doctor said. So I guess, somebody that's double board and endocrinology and sexology can make jokes like that and still get published. Yeah, respected journals. I tried that there's no way to be accepted. And so what he's saying is wearing the mask could potentially prevent you from getting COVID and also protect your prevent you from getting sexual dysfunction. And they're plausible mechanism by which COVID-19 may impact directly on the endothelial cells, leading to a reptile dysfunction. So in the paper, they go on to describe we're now seeing all these COVID long holders, right seeing patients that show up with neurologic complications, cardiac complications, pulmonary complications, we also know that people were having cardiac events, and they're having strokes. It all comes down to the inflammation in the endothelial lining the endothelial lines, all these origins, but more importantly, it lines the blood vessels supplying the organs. So the concern specifically is that the virus is directly damaging these endothelial cells. And this is how some people feel the virus causes long term damage, not just to those origins, but if you're otherwise healthy, it could hurt it could cause long term damage to your penis. I don't want that. No, this is I mean, this is kind of shocking stuff to think about it, but it makes sense because we're gonna get into the physiology of the penis. Here. Oh, by the way, I'm just got my master's in psychology. I'm not a doctorate yet. Not double board certified, but I just got my master's. I don't know when you had the time an hour ago, when I was preparing for this podcast. That does not sound like an accredited school. I is one of those online things. And that's one of the really reputable hard to get into, like University of Phoenix. I mean, it was, you know, Danna college, online school. So interesting. Yeah, we'll see what it we'll see what you learned. What I thought was interesting that somebody for 400 bucks, they gave me the title and then I Danna is where I went to undergrad. They close, they are not around anymore. So then another study. So then I started going down, and I looked at this and I was like, Okay, we got one study that looked at the actual a pool of people that had this. And then another study recently conducted, what they did is they made the correlation between COVID and erectile dysfunction. And their conclusion was that they assumed that the increase in cardiovascular and pulmonary complications is what is driving the erectile dysfunction. I mean, it makes sense it, I guess, I guess what I'm what I think you're leaning to or that this information is leading us towards is that COVID basically kicks off this systemic inflammation problem. And that being the case. inflammation, once again, is the root of long term and chronic disease. And knowing that erectile dysfunction happens to be one of the manifestations of and so that's what I want to get out here. You're exactly right, local or systemic inflammation, because all inflammation starts local and when it leads to a cytokine release on whatever episode that was that we did the innate immune system. Do you have any idea of why we believe that the end endothelial cells with a blood vessel cells are so ravaged by SARS cov, two or COVID, probably because of angiotensin converting enzyme receptors, Estill receptors. That's exactly it. So in this particular study and another one in the Journal of endocrinology investigation, what they said was that we had to be very careful because we know that SARS cov, two binds to the h2 receptor to get into the cell is access. That's its access. So the final conclusion of all these articles is that we now know that COVID can have both a direct and indirect effect on your penis. It's terrible. nobody's talking about that now, and this could just be the beginning. Like we don't really know like this. The data they published six fold increase was a very small number of people because it was early on in the pandemic, right? I mean, kudos to Dr. Giannini, who was thinking about erectile dysfunction when we didn't even know that SARS cov. Two was, yeah, I mean, I'm amazed continually who hopped on board early to know that these were things to be testing for so early in the game. Yeah. So the so the study that we just got published, we were working on this way back in April. I know we're working on a Yeah, working on in April, launching in May. Yeah. Working on an April launching in May, finalizing June, July, August. And then you go through all these revisions and so that, I mean, he really, he really worked hard to get this out. So more stress for stress Awareness Month. So I'm gonna, I'm throwing a lot at you. Let's just recap. Because right now, this is stuff that nobody's talking about. Yeah, no, right. I mean, I don't know where to begin with the recap. But I do believe that what we need to talk about is how can you prepare your body to successfully harness and Okay, let's just say that someone gets COVID. Yeah, I don't want them to worry perpetually, that they won't ever be able to imagine a life without EDI. And in sorry, for the moment, I'm gonna give you all play by play. Ken is on the ground. He's looking for a cable. Yes. And he just found it. And he wants, he wants me to keep talking. But what he's doing is quite dangerous. He's touching things, he's unplugging things. And I can guarantee you he did not call 311 before he started to dig down there, and then that mess of wires. Alright, however, he's back, I think you're muted. So that was really bad. So what I was saying is, is I think that regardless of what causes or triggers a cascade of inflammation, even if it happens to be COVID-19, you want to be certain that your body is prepared to not allow inflammation to run out of control. Absolutely. and protect your endothelial lining everywhere, right? And long term inflammation will be destroying that your healthy cells will be competing with out of control inflammation to 100%. So we're gonna eventually talk about how to protect yourself. Okay, but not on this episode. This is part one, part two, we're gonna be getting some cool science. Yeah, I just learned that. I know. Okay, but you're but you're on the right track your your level two Polly phonologists. So we're gonna be talking about that. But to actually understand this, I don't think many people really know what erectile dysfunction is. I mean, it gets talked about all the time. There's commercials and all this stuff. Yeah. Well, okay, where do you Where do you want to go? Let's start. Let's start with the definition. Okay. By definition, erectile dysfunction is defined in the medical literature as the inability to achieve or maintain an erection sufficient for satisfactory sexual performance. Yeah, that sounds awful. Yeah. very vague from medical definition also. Yeah, because that's how medical things are written. I just imagine this, this process with your with your partner and cheer he or she goes, you know, really should go see somebody about that. And then your responses, why we just had sex for two hours. And then shear your partner goes, That's not enough. I need at least three hours and by the medical definition, I'm not fully satisfied, therefore you have erectile dysfunction. You got Ed. I don't think that we're a good match. The definition was sufficient for satisfactory sexual performance. What is your satisfactory 100% not everything written in the medical journal is tested for for common communication I had and I'm not making So when I did residency, I worked in the geriatric clinic so you do internal medicine, you get assigned to clinic. I love doing the geriatric clinic, as I spent most of the time taking drugs away. 85 year old male 83 year old female, super smart. They came to the clinic. Because her complaint was, Oh, yes. Her complaint was that something is wrong, because they're only intimate three times a week. And for the past 60 years, good. There was awesome. I'm like, Oh, my. And so I went and when when you're in residency, you have to present it to the attendings. And every attending was like a maintenance couple. No kidding. Yeah. So they're apparently at 85, three times a week was considered EDI, in this case, because they had a different expectation, then everyone. Come on, man. Post stemcell. We don't even go to Padre anymore. I'm freaking 90. It's like, you know, 14 days always going to bed at two three in the morning. He won't even stay up and dance. Is this. I'm gonna I'm sorry, ma'am. Why are you here? He's like, Bill just doesn't want to rave anymore. It's because bills and bills shows up a lot. Bill. So anyways, I just think it's funny because whatever your definition of EDI, it's kind of like what your definition is. But I think most people can agree that the inability to achieve interaction or lose interaction is a big deal, right. And prior to COVID, it was projected that 322 million men worldwide will have significant erectile dysfunction by 2025. My prediction is that post COVID this may be a much, much bigger deal. We may be seeing a ton more. That worldwide, worldwide. Okay. That's a lot. It's still quite a bit, but just south of 8 billion people. 322 million seeking care. Oh, seeking care. Wow. That's good point. Yeah. Wow. That's awful. Yeah. A lot of people. That's a lot of unhappy couples, for sure. Yeah, for sure. Alright, so you're ready to talk some sexology it's good. All right, we're gonna talk about the process of an erection. Okay, so it sounds to me like we're gonna be talking about blood flow and what stimulates that and how those things work. This is kind of a PG episode. Yeah, because we got it because it's just off the topic, but we're gonna be science is the topic. Alright. Remember that? The process of an erection is very complex. Actually. I forgot how complex this was. It's multifactorial. You need to have a balance of psychological hormonal, neurological, vascular and anatomic factors. any alteration anywhere can actually affect the whole process. Yes, no penis, no erection. This is true. No. Unless you're like, mentally on a different level. No blood. No. I mean, we're picking up what you're putting down what's going on down here. I have a penile gland erection. I've achieved enlightenment. I can make my pineal gland. your pineal gland is in your brain. I'm sorry. I'm assuming Everybody knows that. Yeah, maybe you do gland is also pronounced but yeah, that's that's honestly, I just nodded. I didn't know what the hell you're talking. I was trying to make a pun. Yeah. It's called the third eye. But it's up here. Yeah. So anyways, a lots going on. This is super complex. Alright, I'm going to go Bill Nye science guy on you. Okay. And so I built a prop before you got here. This will be interesting. I hope that if you're not, if you're one of the 1000s that are online, you just go back to this part of the show and look at whatever the heck is planed. Everyone what's going on here and we will walk through a reaction. I thought we would do. Yeah, it looks like that's what we're gonna do. But can you describe what I just put on the table there It looks to me like to dish sponges with a two retractable those little squishy key ring holders holding the two sponges together. And on one end, you have a long hot dog balloon filled with water. And I can only imagine that what we're going to do here is show what happens when blood flow arrives in a typical man's penis. You are exactly right. So what we're gonna do here is Oh, yes. So let's walk through the process. Okay. So first step, your brain, which is probably on this end, somewhere way on the other end of that way over here, okay? So your brain find something arousing They could be anything for anybody, okay? And that arousing that arousal without knowing it, a signal is sent through your nerves, okay? To a nerve in the endothelial cells of your penis, and we're going to talk specifically about that. And what it does is it releases a chemical that we're all that we talked about all the time. acetylcholine, acetylcholine Yeah. And then what acetylcholine does is it actually hops onto the endothelial cells, the lining of the sponge hops there. And the acetylcholine actually creates this calcium mediated conversion of Argentine to nitric oxide. And people talk about nitric oxide all the time about vezo dilation. We've talked about it before on different episodes for sports using polyphenols for sports, right? So they see the Coleen calcium mediated channel opens up arginine gets converted to nitric oxide, and then nitric oxide is the key to this whole process. Okay. So nitric oxide, what it does is, it goes through this very interesting process of turning on something called the cgmp, the cyclic guanosine monophosphate, which opens up potassium and calcium channels. And what that does, is that stimulates increased blood flow represented by the balloon, notice that my hand is trapping the blood flow there. So right now the nitric oxide stimulated by acetylcholine turned on by the brain has opened up blood flow to these spongy areas here, known as the corpus cavernosum. The corpus cavernosum is filled with blood vessels and so blood rushes in. And then these swell up and it actually traps the blood, right? And this is the actual process of having an erection. So you need the mental component. You need the proper amino acids you need the proper health and the nitric oxide. Why is the nitric oxide so important? Because in the next episode, we're gonna talk about what COVID does to this whole process, okay, and as it turns out, affecting the endothelial cells is the key. Because when the endothelial cells don't do this process, you cannot achieve this, okay? So, when you are done being aroused, the cgmp is then broken down by an enzyme called phospho. diaster, Ace, five, PD five. And then the blood is slowly allowed to leave the cleaners, and you go back to its natural state. And so that fossil diaster race is important also, because once you achieve arousal, and once you're no longer aroused, the penis goes back to its normal state. That's the thing that created that made Pfizer into a multi multi billion dollar business, some scientist was trying to work on a high blood pressure medicine, to increase nitric oxide to vezo dilate the blood so that you drop your blood pressure. And if you remember the history of that one, they were doing a clinical study and none of the men returned their unused samples unused. And then that's when, you know, some poor scientists is like my life's work is ruined, I'll never fix high blood pressure, nobody ate something much better here again. And that is what is now known as Viagra, it actually blocks that enzyme that allows this to be released. And so this is all happening on the endothelial cells. And so once that enzyme gets blocked, then you can actually maintain an erection longer and all the other things and all the other TV commercials regarding those whole class of medicines. Now, there are phosphodiesterase five inhibitors, one little enzyme is what MIT turned it into a billion dollar industry. And so this is all happening right there to endothelial cells. So if there's any inflammation or damage to those cells, this whole process can't be achieved. Yeah. No matter how much you want to will it right, like there's more going on than that. So would you like to recap the process? Wow, that is a lot. And you don't really have to say the sciency words I just want to see you squeezing the balloon I can see. So essentially arousal, if endothelial cells are now stimulated, so that they can be dilated and that occurs because arginine is then converted into nitric oxide, which then of course has cgmp. And suddenly now we have a place for this high pressure blood flow to go. And I see I'm having problems. Probably more the reason why you want to be so anxiety can also I'm very nervous isn't working. But anyway, so now we have an erection because what we have here is the positive. Well all four all four measures were met. And our endothelial cells are allowed to becoming gorged, and then suddenly now phospho diaster race arrives, because we're finished, and blood flow returns back to normal, and you're good to go. 100%. So let's look at a couple health issues that tend to have more reptile dysfunction. So diabetes, definitely Oh man, poor vasculature, poor vasculature. You can have neuropathy, you don't have the nerves traveling secreting the acetylcholine, you have some vasculopathy. So your arteries look like the tail end of this in the middle. So there's things that can happen with that. hypertension is another one chronic hypertension. Definitely. So chronic hypertension, it can lead to issues with erectile dysfunction. Both of those have increased h2 receptors in the endothelial cells. Yeah. So you got better access for that COVID if you happen to be infected, so now you've got it's kind of like, multiplying the damage. Yes, exactly. And which also makes sense why people with prior reptile dysfunction had a five fold increase in developing significant COVID. Kind of makes sense. Yeah, it does. It's just more Ace to receptors present. Yeah. So you know, it's a very crude model, Bill Nye science guy kind of thing here. Oh, I think you really hit it on the park. I imagine walking around the studio trying to find things that I can like, you know, what I really want to do I want to do, I want to find something that that signifies the corpus cavernosum. It's a spongy, like smooth muscle sponge. Yes. Yeah, like that. I don't remember anybody talking about the key ring holder. But I mean, regardless, it works. Trust me. I thought of a lot of different ways how to make sure that the sponge is bad, though. I mean, that's kind of how tissues functions. Yeah. Yeah. So you have this. This is a smooth muscle. So anything that affects smooth muscle can actually affect this. Yeah. So that's how come we always talk about increasing sports performance, increasing nitric oxide, having the proper diet, all these things play into a role. So having the proper diet, maintaining healthy lifestyle, exercising increases? Oh, here's a great example. I was thinking about this. I was like, why is exercise so important? Well, exercise increases BDNF in your brain, oh, decreased neuro inflammation, allowing for more sensitivity allowing for more acetylcholine to go through. We know from Angie's Angie being on a guest. On the episode, she discussed how TNF an inflammatory marker decreases acetylcholine. So the more chronic inflammation you have, the higher likelihood you will not have the proper amount acetylcholine. So there's something else there, then we get into the vascular aspect of this, with regular exercise in good sleep, you have increased or improved vascular tone. So it all kind of plays into this is something that we didn't talk about in the breakdown on on the stimuli for where acetylcholine is coming from. And just a brief thing, but if I remember correctly, I think that the parasympathetic nervous system is actually the catalyst right for the release of acetylcholine. And if you can't rest, and you don't have good vagal tone, then you then just like you said earlier, you won't have the correct neurotransmitters to make this happen. That comes with great rest, which helps lower your anxiety. How can you get great rest you exercise is a cyclical thing. You're You know what? I'm so glad you brought that up, because this is something we learned in med school. And I forgot to because it wasn't discussed in any of these articles, and I kind of stopped at that. But you're exactly right. To achieve an orgasm. I need the parasympathetic system to get that acetylcholine to start on sorry, to achieve an erection to achieve an orgasm, you need your sympathetic. Yeah. And so that sympathetic drops all these things maintaining this, which is why you go back to your normal state post or in your phospho das races now present. Yes, yeah. phosphodiesterase is released, you break down cgmp blood flow is allowed to leave. And that's a really cool, fascinating thing, which is that people who have great vagal tone generally are exercising and don't have chronic anxiety. They usually have great rest, they typically eat well. Good. When we have patients who show up in the clinic or at the procedure, the procedure suite, they'll have a low heart rate. They generally have good physique and are healthy. They're either runner, cyclers lifters, crossfitters, whatever happens to be but today they're active, they vigorous lots, lots and lots and lots of diggers. But But I guess I'm just saying it augments in order to prepare for something like, like COVID you really just want to be in shape. You just want to take care of yourself. You want to give yourself that health span. We've we Sitting on services. Yeah and this this can be done through a lot of different ways. Just getting up and getting some. Oh my gosh. So while I was waiting for you to come over I get done with clinic a little early turned on. Brian Reagan has special at Red Rocks. Oh nice. I didn't know there was one. Oh, it's he's just I just you know he's just good. We're both big fans of stand up comedy huge big fan of stand up comedy. It's called Reagan on the rocks. And I'm I'm didn't look it up. But it the way you describe it. I think it's red rocks in Colorado. Oh, it's gotta be it was outdoor. Yeah, yeah. And it's like this amphitheater type style, outdoor rocks. And he was like, he was Yeah, I'm at that age. I'm going to doctors all the time. My doctor said, Brian, you need to quit being so sedentary. As he goes, right? Then I promised myself I need to get a dictionary. And never got around to it. I just lay around the house all day. So but I thought that was funny because he's never I just get out and do something, just get moving a little bit. you increase the blood flow, increase your body's responsiveness, your vagal tone. And then other issues that you may have problems with. The interesting thing is how many people how many men will avoid other health issues? Until erectile dysfunction comes up? Yeah. Which is, which I think is you ask a lot of people yeah, that that'll be the thing. They'll drive into a doctor? Yeah, I think guys are just generally just I don't know what it is. But guys are just generally that way about health care in, in total. Agreed? Yeah. So this is part one, because I didn't want to go on for way too long. This is still I think we're, we're having a decent time here. We're a little bit shorter than we normally do. But that's probably Yeah. But that's probably a good thing, because maybe we try and pack too much in all the time. So part two is going to be what can you do to make sure that you protect your penis, okay? How polyphenols on a molecular level work to take care of it, prevent it. And it all comes down to everything you're talking about inflammation and how it does it the amount that you need to take, how do you increase your nitric oxide all of the above. And spoiler, it is going to be all about inflammation. And I'm just I'm looking at the products that that are near and dear to my heart. I tried to polyphenol blend CBD didn't find anything directly related to nitric oxide. But there's a lot of evidence that CBD decreasing anxiety definitely allows a greater vagal tone and acetylcholine release. And of course, brockagh lead with sulforaphane in it decreasing NF Kappa beta, which we'll get into, which is all about inflammation. So that awesome episode, great introduction to what people struggle with and shouldn't be ashamed to go and seek help now. And I think that we'll be able to explain how to protect yourself from this, which ultimately is protecting your overall health, which ultimately protects you from COVID. And if you've been a maybe a little bit Cavalier towards COVID. Yeah, this is one of those things that kind of gets you thinking twice a little bit. Yeah, definitely no awesome episode. Yeah. Hey, this is I mean, I don't know if you want to try this at all, but it is a little bit soothing to move the water back and forth. Do you remember those those, those little things that used to have the inverted water below the water? Snake? Water? Snake? Yeah, you try to hang on to it and go all over the place? Yeah, that's similar to that. It is similar to that. But so anyways, I've submitted a patent as a teaching tool once I get my psychology degree. So I was told to tell you, you're not going to get that. That is going to be Episode 54. Be certain to turn in our are we going to do the follow up on 55? We're going to develop on 55. Yes. So 55 is going to be all about the science and what you can do to protect your penis, protect your health. And we're going to continue with, you know, battling COVID by prevention, not just when this happens, you know, it's this thing that we talked about what we've done, I don't remember how long that was vitamin D and all the usual stuff. I don't know either. But it's all it's funny how this is just becoming this multi circle Venn diagram and everything's landing right in the middle. And that middle is inflammation all the time. Definitely. Oh, almost forgot, this show is not meant for medical advice. So if you have any issues with erectile dysfunction, or any other health concern, please consult the doctors that you know, and this is technically just a show that we just talked about things that happen to be in health. It is it's just a show. But the other thing is that the whole point of the show is to also try and help one person so if somebody is having an issue, and they agree checked out because of this because they found the weird sponge thing, entertaining or not entertaining, then we've done our job. Absolutely. So Episode 54. No joke. Not one from you about car 54 I'm shocked with all of your your lame dad jokes. I honestly expected you to show up with like a cop hat and a spinning red light or something like that. I took my because my employer was put it two weeks and I took the staff out. Yeah, we are at a restaurant. And of course I'm throwing out and the waitress, same age as all my employees. they impart that my employees apologize, so he's gonna throw these out all night. She's like, Oh, don't worry, I'm totally down with that jokes. I'm like my boys have it doesn't matter how funny I think a meme is. I send it to them and they but they just type back seven years ago. It's all I get back. It's all you buy for both Mac engaged. So tune in next week for more dad jokes, I guess tons of dad jokes. Alright, that's gonna be Episode 54. Thank you so much for liking shared, certainly if you have a concern, rewatch and then tune in for Episode 55 or get some follow up on what can be done to naturally protect yourself from hopefully, yeah, and and and in all seriousness, if you have a friend or anybody that has discussed this with you or something like that, make sure that they listen to this because it could be an early sign of something else. Definitely that could that could save their life. Thank you all very much. We'll see you next time. Bye bye.
If there’s one thing I love
Hi Audio Verse Award voters, I'm Emily VanDerWerff, and I'm the co-creator and co-executive producer of Arden, which is... kind of sort of a fake true crime show, but also about two women who just can't stop arguing with each other because, I mean, they should get together, and it's also kind of a Shakespeare adaptation and also about generational trauma. It's a comedy! Anyway, this is Episode #203, the third episode of our second season, and it's called The True Crime Podcast's The Thing. This is the episode where we kick off our big mystery for Season 2, which is looking into a young woman who believes her father was murdered, and she believes it was done to achieve the sale of a ranch she believes to be rightfully hers. If you think this sounds a little bit like Hamlet, that's entirely coincidental! We had no idea that was happening! We did not mean to do that at all! Anyway, if you've never listened to our show before, Bea Casely is a journalist, Brenda Bentley is a detective, they attempt to solve cold cases, and in this episode they meet Dana Hamill, who is our analogue for... Simba, from The Lion King, and not Hamlet, from the Shakespeare play Hamlet, at all. Thank you so much for listening. ...So Episode 203 features most of the people we have up for nominations this year, but it misses one of our performers, who joins the show in Episode 204: Saoirse Ó Súilleabháin as Olivia Breckenridge Here is a clip from the fifth episode of our second season, "More Kin, Less Kind," featuring Saoirse, and Libby Woodbridge, who plays Dana Hamill, having a conversation about their failed marriage. Please enjoy! Doesn't that sound fun? The transcript for this episode can be found here. http://ardenpodcast.com Twitter: @ArdenPod
Shannon and Janine recorded this episode after experiencing some pretty significant unanticipated events on our September trips. Shannon's eastern Oregon RV trip was disrupted by heavy smoke from the wildfires and Janine, whose stay in Walla Walla was also affected by the smoke, had to grapple with her computer becoming inoperable during her big driving trip. This week we discuss how our careful preparation--and knowing what's important to us--allowed us to adapt with ease when faced with the unexpected. Discussion topics include: • The high levels of wildfire-related smoke that Shannon and Janine were experiencing at the time we recorded this episode • How Shannon’s trip to eastern Oregon was affected by the smoke and the ways she and her husband adapted their itinerary to accommodate it • The considerations they made to allow them to have a successful trip despite the disruptions • Once again, the importance of knowing what is important to you • Janine’s computer malfunction that threw her for a big loop • The 90th birthday video gift for her father that was imperiled by her computer meltdown • How Janine had to embrace good enough to be able to present her gift to her father (it turned out to be a wild success!) • The hand-made thank-you note Shannon left for park rangers to thank them for the cleanliness of the pit toilets • The silver linings of the adaptations Janine’s decision not to visit Mount Rushmore, despite spending the night in Rapid City Note: We delayed publishing this episode by a week because the Get Out the Vote episode felt more urgent. So Episode 123 was actually recorded before Episode 122. Go to www.gettingtogoodenough.com for links.
So Episode 3 was great, in terms of the relationship between the main leads! I hope there won't be a love triangle in the drama, so I hope Hae-hyo won't fall for Jeong-ha. We will see how it will turn out. I am really excited about the Episode 4 which I will watch tomorrow. Look forward for the Review tomorrow.
Our regularly scheduled programming got derailed by both life and work this past week. So Episode 21 of Lawyerpreneur is a short episode with updates about various projects I'm working on both on my own and with other lawyers, and how you can interact with them. My "What I'm Doing Now" page: jeremywrichter.com/now/Do You Draw Pictures? by Becki C. Lee and Walter Jaczkowski.Articles about the business and craft of writing at expectantwriter.com.Support the show (https://www.patreon.com/lawyerpreneur)
Show Resources: Episode 25 - How to Optimize Your LinkedIn Ads For Better Performance Episode 06 - LinkedIn Ads Bidding & Budgeting Strategies Episode 15 - Benchmarking Your LinkedIn Ads LinkedIn Learning course about LinkedIn Ads by AJ Wilcox: LinkedIn Advertising Course Contact us at Podcast@B2Linked.com with ideas for what you'd like AJ to cover. Show Transcript: You've heard of agile development. But have you heard of Agile LinkedIn Ads management? Strap in, I'm gonna show you how it's done. Welcome to the LinkedIn Ads Show. Here's your host, AJ Wilcox. 0:21 Hey there LinkedIn Ads fanatics. So Episode 25 was all about the types of optimizations that you can make in your account to show improvement. But you've all been asking me how often you should be doing this optimization and how much data you need and how fast you can go. So I'm going to break down exactly how you can go and launch, test, and pivot with your LinkedIn Ads in a very agile way so you can move faster and conquer your competition. In the news, LinkedIn did a survey where they had just over 800 marketers across the globe kind of self report on how they'd been disrupted during the whole COVID situation and the results were quite interesting. About 10% of respondents said that their business had been totally disrupted. So this is a group who are trying to save their marketing budgets, and basically try to stay in business and not lay anyone off. Then about 70% responded that it was business, but unusual. So they're spending time on strategic planning. They've been mildly disrupted, and they're trying to figure out ways to get around and get through. But business is still going. And then you had 20%, who really said their business had been evolving. These were companies in a category that are really thriving right now. They're in a growing market. They're finding new customers, they're taking advantage of these new opportunities that the post COVID world is providing from those that I've spoken to I would say that these percentages are probably about accurate. In the survey results, they linked to a document that they call Driving ROI. And it's a set of best practice resources that LinkedIn compiled to help marketers. So I clicked on it, I read it, it was from 2019. And it is something that I'd read before but had kind of forgotten about. And I want to point out just some interesting findings here. And I've linked to this in the show notes down below. But it's called the long and short of ROI. As you consume this, you can just hear LinkedIn ranting, during the whole piece about the complaints that they hear so often from advertisers. They really come to three points here. Number one, marketers are measuring ROI too quickly. Number two, when they say they're measuring ROI, they're not really measuring ROI. And number three, internal pressures are causing marketers to move too fast and make actually poor decisions. And although it does sound like a bit of a rant, I totally understand where they're coming from. I'm sure they've had many advertisers quit where they say, you know what, we had to quit, we're just not seeing the return on our investment here. So this is LinkedIn reminding us if your sales cycle is six months long, quit complaining when you haven't seen ROI after four months of advertising. We talked about this in Episode 24, all about funnels. But LinkedIn is going to look really expensive if you're just tracking to the cost per lead. And if you're not measuring past that, if you're not measuring to cost per sales qualified lead, or cost per proposal, or cost per closed deal. Those are the points in your sales process where LinkedIn is gonna start looking really good. So if you're not tracking all the way to that, you probably won't have enough faith in the platform to give it a real shot, or even keep going. We're also planning a Q and A episode here in the next few episodes. This is our first of hopefully many. And I want to get our q&a questions from you. So any question you have about LinkedIn, I mean, we'll make it a total potpourri. Email them over to us at Podcast@B2Linked.com and we'll do our best to include it. I want to highlight a couple new reviews on our iTunes page Naira Perez, who is the founder of SpringHill Digital up in Portland. She is a LinkedIn Ads and a social ads expert. I've gotten to talk to you on many occasions. She's amazing. Okay, so her review says two words "amazing and useful". "AJ gives you actionable lessons. He doesn't keep secrets when it comes to optimizing ads on LinkedIn. He shares what has worked for him and what hasn't. If you run LinkedIn ads, listen to AJ, you will learn something in every episode, he is the gift that keeps on giving". Naira, thank you so much for the kind words, I'm so glad that you're getting a lot of use out of this. The next one comes from Mark Gustafson, who's the founder of 900Kings, and actually a close friend. He's actually the inspiration and my fact checker for episodes eight and nine, all about Facebook ads and Google ads. He's a fantastic paid search and social marketer. So Mark says "best B2B Advertising resource". "AJ is the best in the business. There isn't anyone else I turned to with B2B questions. He's easily the most knowledgeable about the LinkedIn ads platform. The podcast is pure value and perfect for the newest B2B marketer all the way to the most senior. Also, can we talk about that dreamy voice? I could listen to those dulcet tones for days." Mark, thanks for the kind words and thanks for turning a bright red behind the mic. I'm so glad this isn't a video podcast. And seriously, listen to my voice. There is nothing sweet about this. I'll rant for a second. I've got this crazy accent from growing up in both Utah and Arizona and learning Spanish in high school learning Russian after high school and before college. Basically, I don't even know how to describe my own accent. I sound real weird and I fully admit it. Okay, cool. Now I want to feature you so please do make sure you review on whatever podcast player you listen to this on. Leave a review. I'd love to shout you out on air. Thanks in advance for that. Okay, with that being said, let's hit it. 5:59 Agile Testing We're gonna talk about agile testing. So what is agile? Well, agile methodology is really started out in project management as a way for cross functional teams to get to move quickly and build collaboratively through continuous improvement. Now, you may have heard of development teams working in two weeks sprints, or doing daily stand up meetings. This all comes from the whole agile movement. We've adapted this to LinkedIn Ads management because it's a process that really requires continuous improvement, just like project management. So what is agile management of LinkedIn Ads? Well, to me, it's it's really making quick decisions on results from your LinkedIn Ads, so that you can learn more and test faster, find out what performs and then you can do more of that and have success for longer. So I'm going to share the agile process that we follow. And it all starts the moment we launch new ads. So when we launch new ads, we try to launch on either a Monday or a Tuesday whenever possible. And that's because those two days are the days where LinkedIn traffic is the strongest. We try to stay in the morning because morning tends to be the strongest traffic times for LinkedIn. We're always going to launch two ads, an AB test, where we are varying something so that we have something to compare against. Because if you just launch one ad, whether it performs well or whether it performs poorly, you don't know what caused that. But if you launch two at a time, you're giving yourself a better chance of having something that's going to be successful and getting to compare against what wasn't successful. Now if our Monday or a Tuesday happens to land on a holiday or the next few days our holiday we try to postpone either launched the week earlier or a week later. Again, when possible. Sometimes you've got a gun to your head and you just have to get ads launched. I'm sure you've listened to episode six because it was one of our most popular episodes. It's all about bidding and budgeting and it's the strategy we use to get the lowest cost from LinkedIn, no matter what your budget and what your performance. So if you've listened to that, you know that you're going to start with cost per click bidding. And you're going to bid really low to keep your risk low as you're testing. Right after your ads go live for the first day or day and a half, LinkedIn is testing your ads to try to figure out what the relevancy score is going to be. In order for them to test, they seem to give you pretty prime placement for your ads. And they're going to show quite a few impressions to your audience. And really, they're going to give you the benefit of the doubt in most situations. Regardless of how your bidding, chances are, they'll probably show you towards the top of the rankings. So you're probably getting impressions that are worth more than what you're paying, especially if you're bidding low. But once a day to a day and a half has passed. LinkedIn has shown your ad enough times they've given it enough impressions or given them enough impressions, that they can give you this relevancy score. And let's say it's a number between 0 and 10. Based off of that relevancy score, your next few days are going to become very evident how you're performing. So days two and three, we're watching to see what happens. LinkedIn has given you the relevancy score that they think you deserve. I think they make the decision a little bit quick, but so do all the other platforms. But you're really on your own now. And it could result in three possible outcomes here. So the first outcome is, you have a really high click through rate right out of the gate. And so LinkedIn gives you a great relevancy score, you immediately start spending everything you want to. And if you click performance chart inside of campaign manager, and look at these campaigns, by impressions, it will look like a couple of flat days where LinkedIn was giving you the benefit of the doubt. And then a spike upward when you actually outperformed their expectations. That's fantastic. On the opposite side of the spectrum, you could also come out of the gate with a low click through rate and a poor relevancy score. You'll know this happened when your impressions really fall off a cliff. So if you go to again, performance chart inside of campaign manager, and you look by the impressions by day, your first day, LinkedIn gave you a bunch of impressions. The second day, it was kind of halfway through where they decided you were a poor performer. And then by the third day, they just didn't deliver much. It looks like the downslope of a mountain. And the third potential outcome is really you did okay, you got an okay click through rate and a decent relevancy score. And things might continue the way that LinkedIn kind of predicted they were. So now we'll dive into what you can do, based off of which outcome you really landed in. 10:40 Outcome Number 1 So outcome number one, you did great, your ads are attractive enough to get traffic, but don't rest on your laurels yet it's not over. Once you get people to click, now you need to convert them. So assuming things are looking good to the click through rate, people actually care about your ads. Now you're going to go into data gaps. mode, let your ads run for the next $300 to $1,000 and get a feel for the conversion rate. If you're happy with your conversion rate and your cost per lead, just let it ride. Go back into data gathering mode, you're gonna hands off, leave it alone. So you can accumulate enough data to analyze, and use to optimize later. Go listen to Episode 15, if you haven't already, because it's all about benchmarks. And we go super deep into how you can tell what's working and what's not. So you can focus in the right area. And you want to make sure that you're watching this performance over time, because we have this thing happen in social advertising, especially called ad saturation, or audience fatigue. And what that is, is you are showing your same ads and offers to the same people over and over and on a pretty good performing ad, still only about 1% of people who see it will end up clicking on it. So that means 99% of people potentially see your ad and go, nah, I don't want to click on it. Even the people who might want to click on it, if it's the fourth, fifth, eighth time they've seen this exact ad, they're going to take a mental note of it, and then just skip it next time they become banner blind. And the way this will manifest itself in your account, if you go back into performance chart in campaign manager, and switch to looking by average click through rate, you can see over time that your click through rate is starting to drop. We found this period to be about 27 to 33 days on average, which is about a month. So what that tells us is if things are going pretty well, we might check on it two weeks from now three weeks from now and just see our our click through rates decreasing significantly. Does it feel like these ads have lost steam? Have they dropped in relevancy score leading to higher cost per click or lower delivery? And we know because the average is about a month. That means once a month, we're going to plan on refreshing our ad creative or testing a new offer, even even if it was a fantastically powerful high performing offer to begin with, because over time, anything will become a low performer, if you've saturated your audience hard enough, and that's mostly ad saturation, people getting sick of seeing the same ads. And you can relieve them of this by simply just changing the image. You may also want to change your ad copy in case they've already clicked or maybe even converted. But what's most important here is the ad needs to look different and stand out. 13:35 Audience Fatigue Now audience fatigue is something that's a little bit different. Let's say you've been advertising heavily for the last four or five years. Or maybe we'll simplify it, let's say for the last one year, you've been going heavy on an audience. You've been religiously changing your ad copy, keeping things fresh, trying new offers entirely, you may find that your performance decreases over time because the that audience has already heard of your company and is now starting to ignore anything from your company. This isn't a great place to be in because if you've been advertising heavily for years and years, chances are you are really relying on the leads that this platform pushes. But it could be a sign that you either need to really vary things up, or even try going on pause for a little bit, giving your audience a rest, and then see if they come back after a little while. So that's how I handle it. If you launch and the launch was very successful, your ads did great. 14:30 Outcome Number 2 But what if you had a failed launch? This is outcome number two, it means your ads really didn't land with your audience. There could be a lot of reasons for that. You might have done a poor job of actually defining who that audience was. Your ad might not have looked attractive enough for them to stop scrolling. And maybe most often, your ad copy didn't do a good job of pressing on a pain point, or calling out to them in a way that they cared about. But for whatever reason, your ads came out of the gate with a poor, click through rate and you've got a bad relevancy score. LinkedIn gave you a small sample of impressions for a day to a day and a half. And it means your ads didn't get enough clicks to qualify for a high relevancy score. So performance is likely going to be terrible. You can force it, you can specifically go in and bid higher or do automated bidding. But if you do this, you will pay way too much per click, and you will just be getting robbed by the platform. When ads come out of the gate performing poorly, sometimes it's really unfair. Sometimes LinkedIn unfairly awarded you a poor relevancy score because in the sample, they showed they didn't see enough clicks. And so they assumed you had a poor relevancy score. But you can resurrect this by just launching the same ads again. So if we launch on a Friday or a Saturday, and our ads just die, most of the time, what we'll try doing is just go and relaunch exactly the same ads on a Monday or a Tuesday, and just see. We're giving LinkedIn just one more chance with these ads to see if they unfairly awarded a poor relevancy score. If they fail for a second time, though, especially on a Monday or a Tuesday morning launch, then we know something's wrong with that ad creative. And we need to go back to the drawing board, or continue to suffer the most expensive cost per click you've ever seen from any channel. If you're having a hard time getting people to click on your ads or getting LinkedIn to even serve them. This point, you probably have not gotten enough traffic to find out how it's going to convert on your offer. Or maybe you did get a decent conversion rate. But since the clicks are costing so much, your cost per lead wouldn't be worthwhile in the end anyway. So at this point, things are going poorly and you should plan on just pausing these ads and relaunching an entirely new test. Episode 24 was all about funnels, so make sure you're paying attention to the right thing. If your click through rates are doing great, don't go and test new ads copy. And if your conversion rates are great, don't go and make major landing page changes. First start with the lowest hanging fruit, the parts of the account that are having the most trouble. If what you're trying isn't working, you can really try something radically different. And like I mentioned earlier, an AB test is going to give you a better chance of finding success with at least one of your variations. So try launching two ads where you vary the pain point you're pressing on. Or maybe you're touching on a different motivation. Or maybe you're even testing different calls to action or offers. You never know how something is going to perform until you test it. So don't be afraid to launch new ads and quickly retire them or pause them. If they're not living up to your standards. Then we have the third outcome, which is kind of like it's okay performance. There are certainly things you can do to try to increase performance. But if things are just going okay, I would say just like option number one where we had a success campaign go into data gathering mode. And then once you have enough data, evaluate to see if it's worth continuing or if there's something in the account that needs to be improved. Here's a quick sponsor break, and then we'll dive into maintaining an account once it's doing well. 18:15 The LinkedIn Ads Show is proudly brought to you by B2Linked.com, the LinkedIn Ads Experts. 18:25 If the performance of your LinkedIn Ads is important to you, B2Linked is the agency you'll want to work with. We manage LinkedIn's's largest accounts and we are the only media buying agency to be official LinkedIn partners. And performance to your goals is our only priority. Fill out the contact form on any page of B2Linked.com to get in touch, and we'd love to help you absolutely demolish your goals. 18:47 Continued Maintenance Alright, let's jump into continued maintenance. We talked about Ad saturation, how about once a month, your ads aren't going to be fresh anymore and you've got to change them up. So plan on doing that at least once per month. And follow the same agile testing steps that I mentioned before. Every new ad launch, you're going to take a look at it and say, okay, for the first three days, I'm going to watch and see, do these perform well? Are they getting a good click through rate? Are we getting a good cost per click? And then over the next week or two, you're saying, okay, is this leading to the conversions at the right costs that I want. You'll also want to keep your offers or your calls to action fresh, because you can change the image you can change the ad copy of your ads, a ton of times, but eventually people are going to catch on if you've been advertising exactly the same offer for the last six months. Every offer really has its own life. We've seen some that after a month that audience has just done and then we had one account where we had a winning offer that we couldn't dethrone for like seven months. No matter what other offers we threw at this audience, they kept preferring the one from seven months ago, and it was still converting even though it wasn't converting at the same rate that it was at the beginning, so we were trying to get it off its throne. So if you've been running an offer for about a month, chances are you can refresh your ad creative, use a new visual, and you can get that offer to live for another month, maybe even two. So watch that performance. Specifically watch your cost per lead, and your conversion rate as you go. As soon as you see that conversion rates start to slip, that's probably a great sign that you need to change up your call to action, give them a different kind of offer something that's new, that they will actually consider if they've already seen the other one several times and have decided, oh, I've already converted, or no, I'm not interested in that. And then keep that up, rotate through new ad creative and new offers as needed as your performance starts to decrease. And if you do this, congratulations, your lead generation machine is complete. To maintain it, what you need to do is Just keep feeding it new ads, new headlines, new intros, and new offers only when they're needed. And then this entire time you're gaining knowledge, you're learning about your audience along the way, you're finding out what they like and what they don't. 21:14 Pain Points Here at B2Linked, we do a lot of ads troubleshooting. And so I thought it would be helpful to at least share with you how we think about finding pain points and what potential solutions are. So let's say your ads aren't performing well. That means either you're getting a high cost per click, or a low click through rate, or even both, they oftentimes go together. What you can do is try new ad copy, new imagery. If it's a video ad, try new video creative. And after two or three different tests of messaging, or visuals, if it's still not getting clicked, chances are the problem is your offer. You're probably asking people to do something that they are either unwilling to do and it scares them away, or they just don't see value in what about your conversion rates. Let's say you're not happy with how much you're paying per conversion, or your conversion rate is low. There are two things that we like to test here. And the first is evaluate your landing page. It's possible that your offer itself is really attractive, but maybe the way that your landing page is laid out, or the elements on it are getting in the way or distracting, and it's decreasing your conversion rate. One easy way to test if it's your landing page that's getting in the way, or the offer is you can test the same exact ad, but run it as a LinkedIn lead gen form. So you're asking people with the same form, but you're skipping the landing page, your website visit all together. Now lead gen forms, as I'm sure you know, tend to convert significantly higher. So we expect that when we do this test, the lead gen form is probably going to convert let's say 10 to 50% higher. But if it is significantly more than that, let's say conversion rate doubles or triples, that's my first clue that something on the landing page was getting in the way and you need to do some testing there. If it's not the landing page, though, it's likely the offer itself. Listen to Episode 10, where we go really deep into offers. And that'll give you some great ideas on how to try out new offers ideate, formulate, and create new offers. What if your ads and conversions are all going really well, but sales reports back that they're not closing these deals? Well, there's a lot of different things that are possibly out of your control here as a marketer, but maybe your sales team isn't nurturing right? Or, and this is a hard realization. You might not have the right product market fit, maybe your product or service that you're selling, maybe it doesn't solve a significant pain point enough that people want to buy. And no amount of snazzy marketing can fix that. Those are obviously much deeper problems, but see if you can isolate where they are and take off your marketer hat and put on your sales enablement hat and go and try to solve that problem, and that will earn you some significant quarterly bonuses. 24:06 Goal of agile management The goal of agile management of LinkedIn is really threefold. Number one, you want confidence that what you're doing is working and will lead to business. And this is hard, because in B2B, we oftentimes have these long sales cycles. And it doesn't make sense to keep advertising for a year and a half if you don't know for sure that there's going to be some revenue that comes from that. You'll want to have stopped advertising much earlier. So as a marketer, you're looking for shorter term clues that the traffic will convert into profitable sales. Goal number two is you want to keep fresh with your audience so that they don't get sick of your ads, your offers, or even your company. If you can stay fresh, your performance won't decline significantly. And this is fantastic because social ads of any kind really are a moving target because the same thing that worked two months ago may totally fail today. And that's just because of ad saturation. So do your part to keep things looking fresh so that you never get stale to that audience. And then Goal number three here, you want to identify the problems and inefficiencies of your account so that you can fix them early and they don't turn into something terrible later on. Okay, I've got the episode resources for you coming up, so stick around. 25:28 Thank you for listening to the LinkedIn Ads Show. Hungry for more? AJ Wilcox, take it away. 25:39 References I referenced quite a few other episodes in here. So check those out in the show notes if you haven't listened to them already. Episode 25 is all about optimization of your account, making it better when it's already doing well. Episode Six is on bidding and budgeting. Episode 10 is all about offers. And Episode 15 is all about benchmarking. To see where you stand if you're performing well, or if you're performing poorly. If you're new to LinkedIn ads, or if you have a colleague you're trying to train, check out the course, I actually did the LinkedIn Ads course on LinkedIn Learning. There's a link for that down below. But because it's LinkedIn Learning, it is insanely inexpensive, and it's a great training. It's the same thing that I charge $500 an hour for and would take me an hour and a half to train you and your team. And through LinkedIn, you can get it for 25 bucks. Or if you have a LinkedIn premium subscription, it's free. Take a look at your podcast player right now, especially if you're new, if this is your first episode. If so, congratulations. Welcome! And hit that subscribe button. We want to make sure you stick around and hear more awesome LinkedIn Ads strategies. Please do rate and review us on whatever podcast player you lean on. We're especially looking for stitcher right now. So if you happen to be a stitcher user, I would love it if you go and review us there because we only have one lonely one there. And of course, I'll shout you out for your review. As long as I know that it's you. So give me something good to say about you there. And like I mentioned at the beginning of the show, get your QA questions in, email us at Podcast@B2Linked.com. And actually, feel free to email us with ideas or topics you'd like to see us cover or questions, anything that you'd like. But especially I'm looking for good Q&A questions to cover or a whole Q&A episode. Hopefully, it's the first of many. So I'll see you back here next week, cheering you on in your LinkedIn Ads initiatives.
Blockchain and games is a topic that I have come across many times in the last years. Many people talk about it and it is said, it is a game-changer. In this episode I wanted to learn more how blockchain can work for game developers. A company that is quite often in the news, when it is about blockchain and games is Animoca Brands. So who better could I talk to then the CEO himself; Robby Yung. Robby started several businesses in China and Hong Kong, including those in mobile telecoms and software applications, cable television, internet services, outdoor advertising, and publishing. He has raised many rounds of private and institutional venture capital for these businesses as well as done four IPO's, several trade sales, follow on financings, and lots of M&A. I would say he is super experienced. What is Animoca Brands? Animoca Brands leverages gamification, blockchain, and artificial intelligence technologies to develop and publish a broad portfolio of mobile products including games such as The Sandbox, Crazy Kings, and Crazy Defense Heroes as well as products based on popular intellectual properties such as Formula 1, Garfield, Snoopy, Thomas & Friends, Ever After High and Doraemon. Animoca Brands’ portfolio of blockchain investments and partnerships includes Lucid Sight, Dapper Labs (creators of CryptoKitties), WAX, Harmony, and Decentraland. The Company is based in Hong Kong, Canada, Finland, and Argentina. Blockchain-enabled games Animoca Brands develops blockchain-enabled games, which facilitates a genuine form of digital ownership of in-game content and collectables in its games. The digitised approach allows players to maintain ownership features across games and platforms, compared to the traditional game industry, where content is owned by the developer/publisher and rented by the player. The mission of Animoca; They leverage their unique capabilities to develop compelling consumer applications with focus on: Motivation Ensuring applications are fun and engaging: Games, Gamification Technology Foundational: Mobile, Blockchain and AI Innovation Staying ahead of the curve by strategic and technological means It was a very learningful interview for myself and I do hope for my listeners too. In this article I did link to Robby's linkedin and to the corporate website. So if you want to learn more, feel free to reach out to either Robby or the company. The interview is done in two parts, since it was quite long. two times 25 minutes. So Episode 38, which will be released next week, will have part two!
Moses walked right into an ambush. After the 10 plagues, a short exodus, and crossing the Red Sea, Moses and Company are attacked by the armies of Amalek. Who? That's a good question, and to defeat this guy with 600,000 Hebrews, you even need supernatural help from God (the staff). Even though Moses and Joshua win the day, God promises that the "War with Amalek" will continue through the generations. So Episode 10 looks at some possibilities for this faceless enemy.
So Episode 204 was a catalyst to this Episode! Getting down to the grit and and ideology of what’s taking place within our lives. Embrace change or get left behind. --- Support this podcast: https://anchor.fm/maul-daniels/support
So Episode 6 i talk about how things are most likely going to be for a while moving forward. Pay cuts for sure are going to happen for a lot of DJs! Will you shut up and take it or will you hold your ground and value ?
So Episode 2 of the Spiritual Gas Mixshow aired on 29th of September 2007 on Gospel Impact Radio. A Christian rapper from the U.S. named President Chell wanted to do an intro tune for the mixshow and it was used for the first time in this episode. It turned to be intro tune for the mixshow even till the last show in 2012. This episode is a nice mixture of Christian Hip Hop and Christian R&B (R&P) and features a lot of classic songs by Da' T.R.U.T.H., Corey Red & Precise, Flame, Claude Deuce, Canton Jones, Deitrick Haddon, Cross Movement, Braille and many more! A true classic Spiritual Gas mix. God Bless you...
After starting podcasting several years ago, I had no idea where my podcasting journey would take me. And it's still an excellent opportunity to refine and grow my process as I explore podcasting further in 2020. All that being said, you're tuning into the 100th episode of this podcast. I've gone through some changes since I launched, such as niching down even further to content that will specifically help freelancers. And I took an 18 month break in between ending that sort of first season of my podcast and rebooting in 2019. That being said, if you're a new listener to this show, I wanted to do a recap for this 100th episode of the top 10 episodes that are the most downloaded, the most talked about, the ones that I feel are the best and can be the most helpful for freelancers. So I'm going to recap these 10 episodes for you to give you a brief introduction about what that episode is about and why it's important. This would not be a good episode to listen to if you are driving, exercising, or not able to easily write things down. If you are able to write things down, you can grab these episode numbers and make note of the ones that you want to go and take a listen to. 10. Episode 91 A relatively recent episode that is all about the freelancers guide to raising your rates. I get questions about people raising their rates all the time. It does not have to be that complicated. It's important to raise your rates on a regular basis. But a lot of freelancers seem to get stumped with this idea of how they should do this. I get questions like: Do I do this across my entire business? Do I keep my current clients at the rates they have? How do I break this news to current clients that I'm thinking about having a rate increase? How often should I do it? How much should that increase be? This is an area where a lot of freelancers tend to overthink. So if you're confused about some of my recommended approaches, check out “Episode 91: The Freelancers Guide to Raising Your Rates”. 9. Episode 10 This is one of my favorite time management and productivity tips, the Pomodoro Technique. I use the Pomodoro Technique every single day. And Episode 10 is called “Pump Up Your Business with the Pomodoro Technique” because it really has the potential to be a game changer and help you lay out your dates more effectively. One of the biggest mistakes that I see a lot of people make is trying to work in really long uninterrupted stretches and thinking, “Well, you know, if I spend six hours on this project, I can knock it out from beginning to end.” What tends to happen for most people is that that's too overwhelming and too long. So thinking about how you can chunk your work into smaller segments, and remain hyper focused during that period, is really what the Pomodoro Technique is all about. In this episode, I gave you some ideas for how to get started. And then some of the different timers and tools that I recommend or have used with the Pomodoro Technique. Now, there's been a lot of really good research about how many pomodoros, which typically means 25 minute work segments, but can also refer to 50 minute work segments, are optimal in a day? This isn't a situation where you want to take that eight hour work day and say that you're going to have 16 pomodoro in that period with no breaks beyond five minutes in between each one. There's definitely a sweet spot to hit there with several focused work periods per day. But not overloading yourself, because your brain really has a hard time keeping up with that. 8. Episode 17 I would love for you to go back and listen to Episode 17, where I had guest Catherine Morehouse talking about the power of niching down. Now a lot of freelancers and freelance coaches will tell you that you should never niche down. That is something I do not agree with. Because I think that niching down has the potential for you to start charging as an expert and really be a specialty provider. If you are just a writer, there are so many writers that you have no way to distinguish yourself. And niching down doesn't have to mean that you claim one particular industry or one type of project and you do that forever. With freelancing you have a tremendous amount of flexibility. But we talk in this episode about how focusing on the clients you like to serve best makes you become the go to person for that service. So go listen to Episode 17 if you're curious about whether or not you might want to niche down in 2020. 7. Episode 23 Switching back over to these time management and productivity tips, This one is called “Stop Changing Lanes in Your Brain”. This is another thing that I coach freelancers about a lot. And it works hand in hand with the Pomodoro techniques that I covered in Episode 10. Changing lanes in your brain by constantly switching between different types of tasks is not just exhausting, it's really inefficient. And yet, it's the way that 90% of freelancers run their business. Choosing instead to batch your work and to focus on particular tasks during certain blocks of time is much more likely to make you feel successful and not as exhausted at the end of the day. So check out Episode 23 if you want to learn a little bit more about what I mean by changing lanes in your brain and how you can kind of break out of some of those bad habits. 6. Episode 26 Another challenge into that basis, a lot of freelancers, especially those who are scaling, is shiny object syndrome. This is the idea that you see a new project or idea and you run with it all the way before fully evaluating it. And that takes your focus away from some of the activities that you really need to be doing to grow your business. So Episode 26 with guest Rita Morales is perfect if you're thinking about how to cope with shiny object syndrome. How much is enough? When is an idea just an idea that you should store as a potential future thing to explore? And when is it something you need to take action on right away? 5. Episode 94 I just recently recorded this episode. It is a must must must listen to episode. This was with guest, Mariam Tsaturyan. And we were talking about freelance contracts. What goes into a contract? What mistakes do freelancers make when putting together contracts? What clauses are Must have, or clauses that you should be aware of when they come to you and a client provided contract? Mariam is not just an attorney, she is a freelancer herself. And she sells some amazing templates to help you get started so you don't have to pay hundreds of dollars to an attorney. So that's Episode 94. Any Freelancer in business for themselves has to know how to use contracts. So I strongly recommend that episode. 4. Episode 54 This episode is all about choosing the right clients. I had a guest on the show who was an editor and we talked a lot about what it really means to define who your ideal client is and how to work specifically and mostly with those clients. If you've been listening to this podcast for any period of time, you know that I am a big advocate of only working with the right clients. The right client means your ideal clients, the rock stars that you want to build your business around. But so many freelancers get tied into this idea of wanting to work with everyone and making themselves a little too available to those potential clients. In this episode, you'll hear from both me and my guest Elizabeth, what it means to choose the right clients and what that looks like for us. Because even though it's important that everyone should implement only working with their ideal clients, that's going to look different from one freelancer to another. And it's up to you to determine what your ideal client avatar is. A lot of freelancers get hung up on, “Well, should I only work with one particular type of client? Should I only work with one type of industry? Is that what it means to say that I have an ideal client?” Sometimes you can go too far with that and you limit yourself as far as what opportunities are coming to you. So it's important to think about what's that perfect balance that I can implement in my business that is going to be really successful for me to attract the right people and also repel the wrong people. Because you definitely want to make sure that you have a nice balance between those two things. And I've got a great freebie that goes along really well with that, and it is called “Creating an Ideal Client Avatar”. You can visit https://www.betterbizacademy.com/creating-an-ideal-client-avatar/. There is a PDF there that can help you walk through figuring out who your ideal client is. 3. Episode 71 I believe this episode is a must listen. It is all about toxic freelance clients. And it's interesting because since I recorded this episode, I've seen so many people own that term of toxic freelance clients and use it in their own way. So it's been really interesting to see how that has kind of spread from what I define a toxic freelance client as working with the wrong person or the wrong team can be really detrimental to your mental and physical health and also the way that you feel about your business every day. So listen in on some examples of what toxic freelance clients can be, and how to figure out if you are currently working with one. 2. Episode 75 Another must listen is the episode that comes in the number two spot on this top 10 list. That's “Episode 75: What to Do When Nothing is Converting with Your Clients”. Nothing drives me crazier than someone who says, “I've been pitching for two years and haven't had any results.” Never wait until the point where it's been six months, a year, or even two years before you ask questions about what you could be doing more effectively and figuring out why nothing is landing. When I've worked one on one with freelancers in this situation, 9 of 10 times there's something wrong in their process. That's pretty easily fixed. It could be that their pitches are terrible or their work samples don't speak to what they're claiming in their pitch. They're targeting the wrong clients or they're not pitching enough. And so all of those things are really within your control as a freelancer and business owner. So listen in on what I recommend you do, based on where you're at in your business and some of the challenges that you're having if nothing is working. 1. Episode 80 And number one on the list of Top 10 episodes is “Episode 80: 10 Habits of Successful Six Figure Freelancers”. At the time I'm recording this episode, I am working on the final draft for my second book, which will come out in October 2020, “The Six Figure Freelancer”. I've done a lot of interviews with other six figure freelancers. And I've worked with quite a few aspiring and current six figure freelancers in a coaching capacity. I've taken some of the things that they all have in common, or some of the habits they tend to most frequently have or work towards, to be successful. Even if your goal isn't to have a six figure or multi six figure business, it's very important to think about the mindsets and the habits that other people who run a business at that level have. Because even adopting some of those could help you with your time management, your client selection, or with the way that you attract clients to you. You may be thinking, “I don't have the time or the interest to build a six figure freelance business.” It's still valuable to listen in to those different habits and workflows that six figure freelancers adopt because it can really make a difference in your business, even if you are only a side hustler. That's my top 10 favorite episodes of this podcast since I launched in 2017. Remember, if you have an episode idea, you can submit that to info@betterbizacademy.com. For those of you who have been tuning in since the beginning, thanks for hanging around until Episode 100. And I hope the future episodes continue to serve you just as well and help you really scale your freelance business. Thanks for tuning in for another episode of the advanced freelancing podcast. For more freelance advice, get a copy of my book Start Your Own Freelance Writing Business—available now! Buy it from Amazon, Barnes & Noble, Apple Books, and more.
First, we want to apologize due to the fact that this episode was supposed to drop on Monday but was not recorded until late in the week. We know, we got to be better for ya. But since Vermont is having a huge snowstorm, retrieving the podcast equipment is type different. So Episode 23 will be this week's episode and Bright joins us. The introduction of Uncle Ruckus takes on a talk about race (4:50). Then the question of one's body count came in (39:47) which takes the pod in a nice direction. Then we touch on some anime towards the end (74:42).
So Episode 20 was recorded in the summer but has been lost in our archives and never got posted... UNTIL TODAY! On this episode we talk about social media and look at different platforms being a better fit for certain outcomes. We also talk a little about weed... That just kinda happened.
So Episode 28 is my official last episode before I leave for Basic Training in South Carolina for 9 weeks. I should be back from 2nd or 3rd week of November. My mom has requested to be on the podcast since the beginning of the podcast. Now we are finally here. She is talking about the 4 most hatred sports figures in Sports World. Plus I tested her to see how well she knows me. --- Send in a voice message: https://anchor.fm/primetimethoughts/message
This episode features a special guest my girlfriend (at the time of recording anyway) introducing her to the show formally & diving into a heavy topic of finance. Building off episode 6's conversation and bringing in some dialogue. So Episode 6 part 2? or whatever you want to call it. Needless to say a look at what I hope to be a more common format of the show open & transparent conversation.
So Episode 1 didn't kick off the way that I expected. I had a pretty good topic, I had potential guests but in the end nobody came through. I was texting people down to the last hour but couldn't confirm anybody so I ended up going to go pick up my big sis Dominique and we just kinda talked about random stuff honestly. It still came out funny but I think/hope that after people hear what these sound like and the potential in them that they'll be more serious about accepting an invitation to talk. And I sure hope so man cuz if not eff yall! Lol naw but anyway Ima wrap this up and dip, shoutout to Roy at @Japangeles, he gave me a hoodie to rock that'll be in a future episode that I film for YouTube, shoutout to Dominique for being funny and coming through and shoutout to ya'll listening/reading this now. I'm gonna try to record these Tuesday nights and put them out on Fridays so the flakes have a chance to make it up in a few days. Alright peace out for now!
Sprachnachrichten - Der "Hey, ich brauche ein Gesprächsthema"-Podcast
Ein frisches Jahr, ein frischer Podcast! Um den Beginn von 2018 angemessen zu zelebrieren, kommen wir nach drölftausend Spezial-Ausgaben mal wieder mit einer News und So-Episode um die Ecke. Die Themen findet ihr wie immer in unseren fein säuberlich herausgesuchten Zeitstempeln: 02:43 - Cleveres Silvester in Neuseeland 08:46 - Breakfast with Dads in Dallas, Texas 21:22 - Google und seine hyperintelligenten AI's/künstlichen Intelligenzen 24:33 - Logan Paul im Suicide Forest: Warum er ein Arschloch ist 39:13 - Sonntagsfrage: Warum ist Alkohol kulturell akzeptierter, als Cannabis? #podcast #news #nachrichten #silvester #neuseeland #clever #smart #alkohol #drogen #breakfast #frühstück #dads #väter #papa #dallas #texas #google #ai #künstliche #intelligenzen #loganpaul #logan #paul #suicideforest #suicide #forest #arschloch #sonntagsfrage #cannabis #kultur #kirche
Hey, there CNFers, Happy New Year. It's 2018 and we're gettin' rollin' here for the biggest, baddest year for The Creative Nonfiction Podcast. And what is the Creative Nonfiction Podcast? It's the show where I speak to the world's best artists about creating works of nonfiction: leaders in the worlds of narrative journalism, documentary film, radio, essay and memoir, and tease out the habits and routines so that you can apply their tools of mastery to your own work. I'm Brendan O'Meara. And to kick off the New Year, I'm actually not interviewing anyone because I've been traveling around creation at significant personal cost to see family and friends on the East Coast. So Episode 82 is me reading “The Language of the Gods,” my essay for Chris Arvidson's and Diana Nelson Jones' collection of baseball essays in “The Love of Baseball: Essays by Lifelong Fans.” The essay does have some footnotes, something I used to love, but am starting to have mixed feelings about, so when the footnotes appear, you'll hear me say FOOTNOTE and I'll read it followed by END FOOTNOTE. They're not too disruptive. This is gonna be a big year, so if you dig the show, subscribe wherever you get your podcasts, share it with a friend, and leave an honest review on iTunes. I'm extending my offer to edit a piece of your work up to 2,000 words and an hour of my time just for leaving a candid review. Just send me a screenshot of the review when it posts and I'll reach out.
We've been doing this for a while, both on this podcast and on others. We've had all kinds of technical issues over the years, but this is probably the first time we've ever failed to actually save a recorded segment after completion. So Episode 75 of For All You Kids Out There is only two halves, which makes more mathematical sense we guess. There's some outdated playoff thoughts, some outdated USMNT discussion, early thoughts on the Mets managerial search, road trips plans to Syracuse and then we chat with Erik Malinowski about the Golden State Warriors and baseball murder mysteries.
So Episode 23 of our podcast was brought up on Talks Machina this week, and we wanted to talk a bit about what happened. Here's the bottom line: We were wrong. We're sorry.
[School of Everything Else 2016] Alex's 500th Podcast. I've been at this game for almost a decade and produced and hosted four media discussion shows, Digital Cowboys, Digital Gonzo, Digital Drift and School of Movies. What we have here is a collection of highlights from the first few years. This celebratory episode ended up as two, simply because there was so much to choose from. So Episode 501 will be released next week. Consider them like Kill Bill Volumes 1 & 2. This is significant interviews, first meetings, moments of particular insight and of course the absolute funniest bits, many of which were suggested by you guys while I was putting this together. Volume 1. Digital Cowboys Another Day at the Office [Joinee Radio, Early 2007 - 0.01.22] Curse of the Golden Flower [Digital Cowboys Episode 01 - 0.06.36] An Important Announcement [DC48, 2008 - 0.08.58] First Meeting With Daniel Floyd [DC10, 2009 - 0.12.13] Red vs Blue Interview [DC126 - 0.15.24] Giant Bomb Snackbox [DC104 - 0.19.40] Jonathan Coulton Interview [DC121 - 0.22.23] Dominik Diamond Interview [DC133 - 0.27.07] The Wii - First Meeting With James Batchelor [DC130 - 0.32.51] First Meeting With Neil Taylor [DC167, 2010 - 0.34.37] Duke Nukem Kicked Out [DC180 - 0.35.18] Legends of Zelda - Opening Montage [DC164 - 0.38.41] Paul & Storm Interview [DC163 - 0.42.53] Digital Cowboys - The Last One [DC109, 2011 - 0.49.10] Volume 2: Digital Gonzo New Solo Show [Digital Gonzo Episode 42, 2011 - 0.55.23] Thor - Alex Doing Mr Plinkett Doing Thor [DG70, 2012 - 0059.44] Never Mind the BuzzGeeks: Cosmocats [DG33 - 1.02.20] Back to the Future - Time Travel [DG19 - 1.05.00] Fire [The Internet News - 1.09.42] Never Mind the BuzzGeeks: Coffee Commercials [DG39 - 1.13.06] Movie Clichés - The Wilhelm Scream [DG34 - 1.15.43] Schools [The Internet News - 1.17.44] Blue Sun Audio Drama [DG35 - 1.22.25] James Bond - The Henchman Agent #1 [DG43 - 1.29.30] James Bond - Henchman Agent #2/Herr Stamper [DG45 - 1.33.51] Firefly - The Henchman Agent #4 [DG106 - 1.33.52] Batman Returns - The Henchman Agent #3/The Finale [DG77 - 1.36.51] Alien - Shotgun Penis [DG74 - 1.41.36] Alien Resurrection - Alien DNA/Executives [DG82 - 1.45.29] Superman IV - Physics! [DG133, 2013 - 1.51.56] Sound of Gonzo - Snowboarding Spy [DG102 - 1.57.00] The Last Airbender - Exposition Scrolls [DG95 - 1.59.26] Sound of Gonzo - Vice City/Dance Hall Deadlock [DG142 - 2.00.47] Iron Man 3 Avengers in Advertising [DG154 - 2.02.51] Return of the King - Lyra on LOTR [DG112 - 2.06.40] The Two Towers - Gollum [DG111 - 2.07.36] An Unexpected Journey - Gollum-Off [DG113 - 2.11.29] Fan Response - First Meeting With Bob Chipman [DG149 - 2.12.25] Pacific Rim - Signing Off [DG161 - 2.21.30] Tower of the Sorcerer - "Can I Ride It?" [DG150 - 2.32.15]
In episode 10 of the CTGY podcast, we get inside the box and tear apart Hellraiser! We have a conversation about the first film and then Hellraiser II, when we first saw the films, how we feel about them and if Kelly will ever bother to see any more after this.. So Episode 10! We are SO thankful to everyone that has actually bothered to take the time our of their day to listen to our episodes - especially to those that have stuck with us throughout all the awful audio issues, the terrible shark puns, and now.. tonight DEAL OR NO KILL, a new gameshow that we.. may never do again.
This episode of Dragnet first aired on September 20, 1955. This broadcast contains the final original script for the series. Though this Dragnet series would continue to air until February 26, 1957, the remaining broadcasts would be all re-runs. So Episode 318, "The Big Close," concludes this podcast. Thanks for listening. Contact bonadonna@sxu.edu with questions or comments.
[ download lowfi version ] [ download hifi version ] Facebook changed! Lutherans don’t like change. So Episode 153 of HT-Radio begins with Pr. Borghardt and Jon Kohlmeier talking a little bit about the changes made to Facebook and more so about changes in the church. Then, Pr. Kuhlman joins them to talk about the […] Copyright Higher Things®, Higher Things - Dare to be Lutheran. Support the work of Higher Things.
Welcome back and thanks to everyone who has been leaving us messages and downloading the episodes we are both overwhelmed by the support we have been receiving. So Episode 5 is a little different, instead of being a discussion on Episode 4 as you may expect this one is all about the music used in … Continue reading "BHC005 – The Music of Being Human" The post BHC005 – The Music of Being Human first appeared on Being Human Cast.
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