Podcasts about Huntington

This week on the Long Island Tea Podcast, Sharon and Stacy are spilling all the tea—from Sharon's weekend getaway in Fire Island to Stacy's unforgettable night at the VMAs! Plus, we're celebrating National Guacamole and Cheeseburger Days with the best local spots, spotlighting filmmaker Doug Gallo and his award-winning doc WEST LANDING, and sharing exciting updates like Adventureland's record-breaking charity night, LI siblings chasing Olympic dreams, and how to plan for the 2025 Ryder Cup at Bethpage Black. Don't miss the #ThisWeekendOnLongIsland lineup, ChariTEA news from the Pink Promises Luncheon, and a CelebriTEA alert: Cardi B is coming to West Babylon!#ShowUsYourLongIslander This week, we're spotlighting Doug Gallo, a Hampton Bays-based director debuting his short documentary WEST LANDING, which captures the deep bond among senior open-water swimmers in his hometown. The film has already screened at the Long Island International Film Expo, Chain NYC Film Festival, and internationally at events like Garda Cinema in Italy, with more stops ahead this fall, including the Offshore Art & Film Festival in Montauk. Know someone doing something amazing? DM us or email spillthetea@discoverlongisland.com to share YOUR Long Islander!#LongIslandLife National Guacamole Day – September 16Celebrate at top spots like Verde Kitchen and Cocktails (Bay Shore), Besito (West Islip & Huntington), and Ruta Oaxaca (Patchogue).National Cheeseburger Day – September 18Grab a bite at The Halston (Melville), Mirabelle Tavern (Stony Brook), Tellers (Islip), The James (Babylon), or The Local (Babylon).Adventureland Raises $400K for CharityAdventureland's VIP Night in Farmingdale broke records with over 2,000 guests raising $400K for the Helping Hands Foundation.LI Sibling Skaters Eye 2026 OlympicsOona and Gage Brown, World Junior Champions from Long Island, are training hard to represent Team USA at the 2026 Winter Olympics.LI Aquarium Offers $25 Days + Fall FunEnjoy $25 admission on Sept 25, Oct 25, and Nov 25, plus seasonal events like a Halloween Party, Fish & Sips, Santa Brunch, and more.Ryder Cup 2025 Guide Now LiveHeading to Bethpage Black? Discover Long Island's all-inclusive Ryder Cup guide covers travel tips, local attractions, shuttle info, and more. Check it out at discoverlongisland.com/ryder-cup#ThisWeekendOnLongIslandFriday, Sept 19: Babylon Pumpkin Fest at Cedar Beach – live music, vendors, and fall fun.Saturday, Sept 20: LI Cares 5K & Paws Walk at Belmont Lake State Park – family-friendly run with pets, raffles, and breakfast.Sept 20–21: Outdoor Art Show & Music Fest at Gallery North – 60+ artists, music, and food.Sunday, Sept 21: Cow Harbor Day in Northport – parade, vendors, boat races, and more.#ChariTEAThe Pink Promises Luncheon is Sept 16 at Crest Hollow, benefiting the Breast Cancer Research Foundation. Luxury auction goes live Sept 15 at pinkpromises.org#CelebriTEACardi B is coming to Looney Tunes in West Babylon on Sept 19 at 1 p.m. for a CD promo event. Photo op only with wristband from in-store purchase. Doors open at 11 a.m.CONNECT WITH US:Instagram: https://www.instagram.com/longislandteapodcast/YouTube: https://www.youtube.com/@DiscoverLongIslandNYTikTok: https://www.tiktok.com/@longislandteapodcastX(Twitter): https://x.com/liteapodcastFacebook: https://www.facebook.com/longislandteapodcast/DM us on any of our social channels or email spillthetea@discoverlongisland.com to tell us what you want to hear! Whether it is Long Island related or not, we are here to spill some tea with you!Shop Long Island Apparel!shop.discoverlongisland.com Hosted on Acast. See acast.com/privacy for more information.

In this episode of Hope for the Caregiver, I open with the story of a 1950s chemist who failed 39 times before discovering something simple yet world-changing. His quiet persistence became a picture of what caregivers need when our hearts feel rusted and stuck. We can't muscle our way through this life; we need grace that loosens what strain has tightened. That theme of letting go of force carries through the whole show. I share a personal story from the 2004 Republican National Convention, when Gracie sang at Madison Square Garden and the White House unexpectedly asked us to stay for President Bush's speech. While I was panicked and trying to control everything, my father told me, “Sit down and be still.” It became a defining moment—reminding me how often our Heavenly Father speaks the same words into our chaos. I also reflect on my recent Blaze Media article, “School Shootings and the Street Called Straight,” exploring how Paul faced the full measure of suffering ahead of him and still pressed on. That same Spirit sustains us when the caregiving road looks dark and unending. Then I share part of my conversation with DJ and Jessica Rowland, a couple navigating Huntington's disease. Their love, resilience, and commitment to live fully—despite grief and fear—show how loosening our grip can make space for grace. I close at the Caregiver Keyboard with a hymn every caregiver should know: “Turn Your Eyes Upon Jesus.” Written by Helen Lemmel at age 55, this hymn calls us to lift our gaze from what we can't fix and rest it on the One who carries us.   ORDER THIS BOOK TODAY!    

Amanda Staveley is one of the most influential figures in modern football, best known for helping steer the takeover that transformed Newcastle United. In this episode, Amanda opens up about what it takes to build something lasting in football and in life. From being underestimated in high finance to proving herself in one of the toughest industries, she reflects on the quiet power of persistence and the importance of surrounding yourself with people you can trust. She talks candidly about what it means to keep going in the face of uncertainty, how her Huntington's disease has sharpened her priorities, and why empathy and vulnerability have become such central parts of her journey.This episode is about more than football, it's about life, hope, and the meaning of legacy.

The Drive with Paul Swann for September 15, 2025. 

What happens when a teenage Eagle Scout and his father go looking for Bigfoot in the piney woods of East Texas — and actually find it? In this intense and unforgettable episode, we hear from Ryan, a Boykin Springs native who has spent years interacting with something wild, powerful, and very real in the forests of Angelina County. From roadside sightings and campfire wood knocks to police dashcam videos and quiet tracking moments near Aldridge Sawmill, Ryan's encounters have left a mark — not just on him, but on an entire community. You'll hear about his early sightings, how curiosity turned into weekend investigations with friends, and why so many members of his local church refuse to return to the woods. If you've ever wondered what it's like to be watched, followed, or even protected by a creature no one believes in — this is your episode. Featuring stories from Boykin Springs, Crockett, Huntington, and beyond, this is East Texas like you've never heard it before.

Jen and Frank chat about their first trips to Walt Disney World; Frank's in 1981 and Jen's in 1985. On this episode…

 On today's episode of The Drive with Paul Swann, Paul welcomes Marshall women's soccer announcer Bobby Iddings. He has the call as Marshall's women host WVU in Huntington 

On today's edition of The Drive with Paul Swann, Paul welcomes Jarrod Hunley, Director of Marketing and Fan Engagement at Marshall University, to discuss the latest in Herd promotions and how fans can get involved this season. 

Huntington is one of ten towns in Suffolk County, New York, United States. The town's population was 204,127 at the time of the 2020 census, making it the 11th most populous city/town in the state.[1]Founded in 1653, the Town of Huntington is located on the North Shore of Long Island in northwestern Suffolk County, with the Long Island Sound to its north and Nassau County adjacent to the west.[2] It is part of the New York metropolitan area.PICTURE: By Town of Huntington, uploaded by Hayden Soloviev - https://northportjournal.com/people/the-town-of-huntington-gets-new-seal-with-official-town-colors-and-longstanding-motto, Public Domain, https://commons.wikimedia.org/w/index.php?curid=165322660

Drew has a lot to say about the NFL week 1.  Starting with the Broncos and that there's no need to overreact to the ugly win.  The Buffs need to start the guy that helps you win now, and that's Ryan Staub.  The CSU Rams escape a bad upset but need to get things right quickly.  And Kyle Freeland sits down with Drew to talk about the tumultuous last week.  

This week on the Long Island Tea Podcast, Sharon and Stacy are recapping another busy week repping Discover Long Island and dropping some exciting updates — including record-breaking tourism numbers that prove Long Island is shining brighter than ever. We're celebrating three years of growth, nearly $8 billion in visitor spending in 2024, and a massive economic boost that's helping keep our region thriving year-round. This week's Show Us Your Long Islander spotlight shines on Jack Kennedy, a 17-year-old tennis phenom from Huntington who's now the #1 junior player in the U.S. and competing in the U.S. Open Juniors — just miles from where he first picked up a racquet. Plus — big wins for Fire Island's shoreline, Suffolk's working waterfronts, a new raw bar for LIRR riders, and yes… an actual gator in a Long Island pond. (Only on Long Island, right?)Don't miss this episode — it's full of pride, power moves, and positivity for our region.#ShowUsYourLongIslander At just 17, Jack Kennedy from Huntington is the #1 U.S. junior tennis player — and he's now competing in the U.S. Open Juniors, just miles from the Melville courts where he grew up playing."I've been watching this tournament since I was five. Playing here now feels like a dream come true," says Jack, who's also the top college recruit in the nation and has committed to the University of Virginia for 2026.With big goals to go pro and a love for the game that started right here on Long Island, Jack's journey is one to watch.Show us YOUR Long Islander by sending us a DM, give us a call and leave a voicemail at 877-386-6654 x 400 or email us at spillthetea@discoverlongisland.com#LongIslandLifeWe're celebrating three straight years of record-breaking tourism on Long Island — and we're just getting started. In 2024 alone, visitors spent an incredible $7.9 billion across our region, a 3.8% jump from last year, generating $945 million in state and local taxes and supporting over 78,000 local jobs. At Discover Long Island, we're proud to lead the charge in putting our destination on the map with smart, data-driven marketing and unforgettable experiences. From world-class events like the upcoming 2025 Ryder Cup to year-round promotions that support our downtowns and small businesses, we're making sure Long Island shines in every season. Suffolk County saw the biggest gains, with a 7% increase in visitor spending — and thanks to tourism, local households saved nearly $1,000 in taxes. We're proud to keep Long Island front and center as one of the top travel spots in the country. Let's keep the momentum going!#ThisWeekendOnLongIslandFriday, September 12thEnergy Medicine with Nicholas Pratley at Shou Sugi Ban HouseSaturday, September 13th Deco in Style: 100 Years at Long Island MuseumPatty Larkin & Lucy Kalansky Songbird Sessions at Long Island Game FarmSunday, September 14thLong Island Explorium's Maker FaireArts on Terry (AOT) 2025For more events to check out and detailed info please visit discoverlongisland.com or download our mobile app!CONNECT WITH US:Instagram: https://www.instagram.com/longislandteapodcast/YouTube: https://www.youtube.com/@DiscoverLongIslandNYTikTok: https://www.tiktok.com/@longislandteapodcastX (Twitter): https://x.com/liteapodcastFacebook: https://www.facebook.com/longislandteapodcast/Shop Long Island Apparel:shop.discoverlongisland.comBe sure to leave us a 5-star rating and review wherever you're listening, and screenshot your review for $5 off our Merch (Please email us to confirm) Hosted on Acast. See acast.com/privacy for more information.

On today's edition of The Drive with Paul Swann, Paul speaks with Greg Stotelmyer, longtime voice of Eastern Kentucky University. 

Neoborn Caveman unleashes satire, truth, and green-tea-fueled rants, exposing techno-feudalism, organ harvesting, and the fight for sovereignty in this radio show replay.NC starts with a shout to his Purple Rabbits, tearing into social media as a surveillance beast training AI for a matrix-style takeover. From historical communism to modern techno-feudalism, he exposes control systems that thrive on fear and conformity, urging listeners to reclaim sovereignty through community and simple joys: think barefoot walks, purring cats, and kids' laughter. No social contract? No problem—you never signed it.The show weaves in NC's music musings, mentioning his unreleased song “You Deserve to Be Heard” and reflecting on hits like “The Breakup Therapy” (1M+ Spotify plays). He critiques consumerism and calls for real community over compliance. Neoborn then spotlights NYC model John Rudat's bravery in defending assault victims in Germany, slamming ideological abuse, failing law enforcement, and UKSSR dystopias. A teaser for Cynthia Sun's harrowing Falun Dafa interview exposes the Chinese Communist Party's organ harvesting, funded by Westerners—yet hope shines through. The public half closes with NC's Rocky-inspired pep talk: You're amazing, unique, and capable of rising above any low. Ignore the inner naysayer, find your true North, and keep fighting.The subscriber's edition (Patreon exclusive) features rising star Shelby Lentz, sharing her journey through music, marriage, and Huntington's disease. From childhood trauma to founding Champions for HD, Shelby's resilience echoes Rocky Balboa's grit. Her country-rock songs, inspired by Avril Lavigne and Stallone's films, uplift as she dreams of a Mars festival and a Stallone-signed tattoo. It's raw, real, and relentlessly positive.Musical GuestsSami Chohfi, Inoxidables, Van Hechter & Chauncey Dandridge, The Revolt, Shelby LentzKey TakeawaysQuestion the System: Social media surveils, techno-feudalism looms—reject unsigned contracts and reclaim “We the people.”Community Over Consumerism: Build real connections, not endless consumption. Join the campfire.Courage Defines Us: From Rudat's heroism to Shelby's grit, stand up for what's right, no matter the odds.Hope Amid Horror: Organ harvesting exposes dark truths, but action and awareness light the way.Be Your Own Rocky: Life's lows are temporary—rise, trust your potential, and live vibrantly.Support the ShowPatreon: Join for free at patreon.com/TheNeobornCavemanShow for exclusives like Shelby's interview and more.Reviews: Drop 5 stars on Apple Podcasts, Spotify, or your platform to fight the algorithm.Connect: DM @NeobornCaveman on X, hit up Trans Radio UK, or email for song requests and rants.Books: Grab Canada's Mirage or The Digital Trap on Amazon for more NC wisdom.Tune in for satire, truth, and tunes that slap harder than a techno-feudal tax. Slurp your green tea, question everything, and remember: You're a Purple Rabbit in a world of brutes—keep shining! See you next time on The Neoborn Caveman Show!Humanity centered satirical takes on the world & news + music - with a marble mouthed host.Free speech marinated in comedy.Supporting Purple Rabbits. Hosted on Acast. See acast.com/privacy for more information.

On today's edition of The Drive with Paul Swann, Paul welcomes Rafa Simoes, head coach of Marshall University's women's soccer team. Marshall hosts WVU in Huntington this Friday, marking a rare in‑state showdown.

First comes love, then comes marriage, then comes … divorce? Award-winning television host, actress and playwright Joy Behar goes from screen to stage in her new play, “My First Ex-Husband," premiering at the Huntington on September 12. We speak with Joy Behar and fellow iconic funny lady and cast member Jackie Hoffman about the show. Then, Olivia Blount squeaked through her employment interview to land the job of her dreams: working with the renowned Aubrey Merritt, a detective with a global reputation for solving impossible murder cases. Aubrey and Olivia's Boomer - Gen Z relationship is at the heart of Liza Tully's new novel, “The World's Greatest Detective and Her Just Okay Assistant" -- our September selection for Bookmarked: The Under the Radar Book Club.  RSVP to our Bookmarked LIVE! event at Parkside Bookshop: bit.ly/UTRParkside

Cuenta Paracelso, afamado médico y alquimista, que la epidemia bailable pudo empezar en 1518, diagnosticada como exceso de "sangre caliente”. Hay quien la bautizó como baile de S.Vito, otros como enfermedad de Huntington pero la solución continua sin encontrarse. El twist, el madison, infinidad, -uno por semana en los 60- y después la Disco o fenómenos tan exóticos como la lambada, de parecidos efectos. La “Macarena", convenientemente coreografiada traspasó fronteras y la yenka, con su manual de instrucciones incluido, perfecta para diletantes. Todo un abanico de posibilidades en esta edicion para perder la cabeza non stop ¡Pista!.Para bailadores sin complejos. Puedes hacerte socio del Club Babel y apoyar este podcast: mundobabel.com/club Si te gusta Mundo Babel puedes colaborar a que llegue a más oyentes compartiendo en tus redes sociales y dejar una valoración de 5 estrellas en Apple Podcast o un comentario en Ivoox. Para anunciarte en este podcast, ponte en contacto con: mundobabelpodcast@gmail.com.

The Drive with Paul Swann for Friday, September 5, 2025

Corduroy Brown was so ill with COVID during the pandemic, he was airlifted from Huntington, W.Va., to more advanced medical facilities in Morgantown. During the helicopter ride, he died.  Fortunately, quick work from the EMTs revived him. He survived and experienced something most people don't: A glimpse of what's on the other side. It changed his outlook on life and his music, which has taken off since. Brown released a gripping and powerful solo album called Let Me Know in 2021 after he fully recovered. It has propelled him from small gigs to opening for headline acts like Everclear and Hoobastank, as well as some headline and featured spots at some nice music festivals throughout the region. Brown joined us on Roots Music Rambler to talk about his path to music, his actual death experience, how that changed his music and what's next. (Hint: New album news nested in this episodes!) Needless to say, we all left the conversation excited about Corduroy Brown, what's to come, and life in general. Frank also grilled Falls on his lucky selection to Tyler Childers' secret pop-up concert at Dinosaur World in July, and both share their pics for new music recommendations in the Pickin' the Grinnin' segment.  Download the episode and subscribe at rootsmusicrambler.com or wherever you get your podcasts.  Also be sure to help spread the love of the show with Roots Music Rambler's new merch, now available at rootsmusicrambler.com/store. Authentic t-shirts, hats and stickers are now available.  Buckle up for The Hoe-Down and the Throw-Down! It's a new episode of Roots Music Rambler. Notes and links:  Corduroy Brown online Corduroy Brown on Spotify Corduroy Brown on Instagram Falls's viral video from Tyler Childers' pop-up The Roots Music Rambler Store Roots Music Rambler on Instagram Roots Music Rambler on TikTok  Roots Music Rambler on Facebook Jason Falls on Instagram Francesca Folinazzo on Instagram Pickin' the Grinnin' Recommendations Stephanie Anne Johnson Deer Tick Subscribe to Roots Music Rambler on Spotify, Apple Podcasts, GoodPods or wherever you get your podcasts. Theme Music: Sheepskin & Beeswax by Genticorum; Copyright 2025 - Falls+Partners. All music on the program is licensed by ASCAP, BMI and SESAC. Roots Music Rambler is a member of the Americana Music Association. Learn more about your ad choices. Visit megaphone.fm/adchoices

Jen an Frank record the 400th episode of the most listened to and downloaded siblings podcast saluting all theme parks but mostly Walt Disney World dropping Thursday from that magical place we call Long Island... Theme Park Thursday with Dillo's Diz! On this episode…

DJ Mo Radio - Mo Music. Mo Variety. In The Mix. - WPRK 91.5 Winter Park-Orlando, FL Fridays 5-7pm EST - Super Station 104.5 Huntington, WV - Mondays-Thursdays 9-11pm EST & Fridays 5-7pm EST - Sundays 8pm-12am EST - 93.3 Tiger FM Fort Myers, FL - Website: http://www.djmoradio.com Facebook: https://www.facebook.com/djmoradio Instagram: https://www.instagram.com/djmoradio DJ Mo Booking: 407-501-6345 Email: djmoradio@gmail.com 

On today's show, Paul welcomes two guests. Niesha Campbell, the new deputy athletic director at Marshall, joins the program. Later, Corey Riggs from the Missouri State University Bears Radio Network checks in to preview the Bears. 

Chorea describes involuntary movements that are random, abrupt, and unpredictable, flowing from one body part to another. The most common cause of genetic chorea in adults is Huntington disease, which requires comprehensive, multidisciplinary care as well as support for care partners, who may themselves be diagnosed with the disease. In this episode, Aaron Berkowitz, MD, PhD FAAN speaks with Kathryn P. L. Moore, MD, MSc, author of the article “Huntington Disease and Chorea” in the Continuum® August 2025 Movement Disorders issue. Dr. Berkowitz is a Continuum® Audio interviewer and a professor of neurology at the University of California San Francisco in the Department of Neurology in San Francisco, California. Dr. Moore is an assistant professor and director of the Parkinson's Disease and Movement Disorders Fellowship in the department of neurology at Duke University in Durham, North Carolina. Additional Resources Read the article: Huntington Disease and Chorea Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @AaronLBerkowitz Guest: @KatiePMooreMD Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Berkowitz: This is Dr Aaron Berkowitz with Continuum Audio, and today I'm interviewing Dr Kathryn Moore about her article on diagnosis and management of Huntington disease and chorea, which appears in the August 2025 Continuum issue on movement disorders. Welcome to the podcast, Dr Moore. Could you please introduce yourself to our audience? Dr Moore: Yeah, thank you so much. I'm so excited to be here. I'm Dr Moore. I'm an assistant professor of neurology at Duke University, where I work as a movement disorder specialist. I run our fellowship there and help with our residency program as well. So, I'm excited to speak with our listeners about chorea today. Dr Berkowitz: Fantastic. And we're excited to talk to you about chorea. So, as a general neurologist myself, I only see chorea pretty rarely compared to other movement disorders like tremor, myoclonus, maybe the occasional tic disorder. And like anything I don't see very often, I always have to look up the differential diagnosis and how to evaluate a patient with chorea. So, I was so glad to read your article. And next time I see a patient with chorea, I know I'll be referring to your article as a great reference to have a framework for how to approach it. I hope our readers will look at all these helpful tables on differential diagnosis based on distribution of chorea in the body, potential etiologies, time course of onset and evolution, associated drug-induced causes, what tests to send. So, I highly recommend our listeners read the article. Keep those tables handy for when a patient comes in with chorea. I'm excited to pick your brain about some of these topics today. First, how do you go about distinguishing chorea from other hyperkinetic movement disorders when you see a patient that you think might have chorea? Dr Moore: One of the wonderful things about being a movement disorder specialist is we spend a lot of time looking at movements and training our brain to make these distinctions. The things that I would be looking out for chorea is involuntary, uncontrolled movements that appear to be brief and flowing from one part of the body to another. So, if you can watch a patient and predict what movements they're going to do, this probably isn't chorea. And it should be flowing from one part of the body to another. So, not staying just in one part of the body or having sustained movements. It can be difficult to distinguish between a tic or dystonia or myoclonus. Those things tend to be more predictable and repetitive than the chorea, which tends to be really random and can look like dancing. Dr Berkowitz: That's very helpful. So, once you've decided the patient has chorea, what's your framework for thinking about the differential diagnosis of the cause of the patient's chorea? Dr Moore: Well, that could be really challenging. The differential for chorea is very broad, and so the two things that I tend to use are age of the patient and acuity of onset. And so, if you're thinking about acute onset of chorea, you're really looking at a structural lesion like a stroke or a systemic issue like infection, hyperglycemia, etc. Where a gradually progressive chorea tends to be genetic in nature. When you're thinking about the difference between a child and an adult, the most common cause of chorea in a child is Sydenham's chorea. And actually, the most common cause of chorea that I tend to see is Parkinson's disease medication. So, if anybody's seen dyskinesia in Parkinson's disease, you've seen chorea. But it's those two things that I'm using, the age of the patient and the acuity. Somewhere in the middle, though---so, if you have subacute onset of chorea---it's important to remember to think about autoimmune conditions or paraneoplastic conditions because these are treatable. Dr Berkowitz: That's very helpful. So, like in any chief concern in neurology, we're using the context like the age and then the time course. And then a number of other helpful points in your article about the distribution of chorea in the body. Any comments you'd like to make about- we have this very helpful table that I thought was very interesting. So, you really get deep into the nuances of chorea and the movement disorder specialist expert level. Are there any aspects of parts of the body affected by chorea or distribution of chorea across the body that help you hone your differential diagnosis? Dr Moore: Certainly. I think where the chorea is located in the body can be helpful, but not as helpful as other conditions where you're localizing a lesion or that sort of thing. Because you can have a systemic cause of chorea that causes a hemichorea; that you can have hyperglycemia causing a hemichorea, or even Sydenham's chorea being a hemichorea. But things that we think about, if the forehead is involved, I would think about Huntington's disease, although this is not pathognomonic. And if it's involving the face or the mouth, you can think about neuroacanthocytosis or, more commonly, tardive dyskinesia. Hemichorea would make me think about some of those systemic issues like hyperglycemia, Sydenham's chorea, those sorts of things, but I would rely more on the historical context and the acuity of presentation than the distribution itself. Dr Berkowitz: Got it. That's very helpful. So those can be helpful features, but not sort of specific for any particular condition. Dr Moore: Exactly. Dr Berkowitz: Yeah, I often see forehead chorea mentioned as sort of specific to Huntington's disease. Since I don't see much Huntington's disease myself, what does forehead chorea look like? What is the forehead doing? How do you recognize that there is chorea of the forehead? It's just sort of hard for me to imagine what it would look like. Dr Moore: It's really tricky. I think seeing the eyebrows go up and down or the brows furrow in an unpredictable way is really what we're looking for. And that can be hard if you're having a conversation. My forehead is certainly animated as we're talking about one of my favorite topics here. One of the tricks that I use with the fellows is to observe the forehead from the side, and there you can see the undulation of the forehead muscles. And that can be helpful as you're looking for these things. I think where it's most helpful to use the forehead is if you're trying to determine if someone with a psychiatric history has tardive dyskinesia or Huntington's disease, because there can be quite a lot of overlap there. And unfortunately, patients can have both conditions. And so, using the forehead movement can be helpful to maybe direct further testing for Huntington's disease. Dr Berkowitz: Oh, wow, that's a very helpful pearl. So, if you see, sort of, diffuse chorea throughout the body and the forehead is involved, to my understanding it may be less specific. But in the context of wondering, is the neuropsychiatric condition and movement disorder related by an underlying cause in the case of seeing orofacial dyskinesias, is the relationship a drug having caused a tardive dyskinesia or is the whole underlying process Huntington's, the absence of forehead might push you a little more towards tardive dyskinesia, presuming there is an appropriate implicated drug and the presence of forehead chorea would really clue you in more to Huntington's. Did I understand that pearl? Dr Moore: That's exactly right, and I'm glad you brought up the point about making sure, if you're considering tardive dyskinesia, that there has been an appropriate drug exposure. Because without that you can't make that diagnosis. Dr Berkowitz: That's a very helpful and interesting pearl, looking at the forehead from the side. That is a movement disorders pearl for sure. Sort of not just looking at the forehead from one angle and trying to figure out what it's doing, but going to look at the patient in profile and trying to sort it out. I love that. Okay. So, based on the differential diagnosis you would have crafted based on whether this is sort of acute, subacute, chronic, the age of the patient, whether it's unilateral, bilateral, which parts of the body. How do you go about the initial evaluation in terms of laboratory testing, imaging, etc.? Dr Moore: Well, certainly in an acute-onset patient, you're going to get a number of labs---and that's listed out for you in the paper---and consider imaging as well, looking for an infarct. One thing our learners will know is that sort of the typical answer to what's the infarct causing hemichorea would be the subthalamic nucleus. But really, those infarcts can be almost anywhere. There are case reports for infarcts in a wide variety of places in the brain leading to hemichorea. So, I think some general blood work and an MRI of the brain is a good place to start. For someone who has a more chronic course of the development of chorea, there are certain labs that I would get---and an MRI, because if you get an MRI and there's heavy metal deposition or other disease, structurally, that indicates a certain condition, that can help you pretty considerably. But otherwise, I'm looking for inflammatory markers, heavy metals, HIV, some general other things that are outlined, to help make sure that I'm not missing something that's treatable before I go down the route of genetic testing. And we may talk about this in a little bit, but if you start out with genetic testing and then you sort of have to back up and do more systemic testing, that can be very disjointed when it comes to good patient care. Dr Berkowitz: That's very helpful. So yeah, if it's acute, obviously this is the most straightforward scenario, acute and unilateral. We're imagining something lesional, as you said, either a stroke or---not sort of sudden, but fast, but not sudden---you might think of another structural lesion. Toxoplasmosis, right, has an affinity for the basal ganglia if you were seeing this in a patient who is immunocompromised. But in a case that, probably as you alluded to, sort of what we would see most commonly in practice, those still relatively rare, sort of subacute to chronic symmetric chorea. There's a long list of tests that are recommended. In your article and in other texts, I've read lupus testing, anti-phospholipid antibodies… but the list is long. I'll refer readers to your article. Out of curiosity as a specialist, how often do you see any of these labs come back revealing any underlying diagnosis in a patient who's otherwise healthy and just has developed chorea and comes to you with that chief concern? I feel like I've sent that mega-workup a few times; I'm obviously a general neurologist, but not nearly as many times as you have been. It's- I can't remember a time where something has come up, maybe an ANA one to forty or something like this that we don't think is relevant. But in your practice, how often do you end up finding a reversible cause in the laboratory testing versus ending up starting to go down the genetic testing route, which we'll talk about in a moment? Dr Moore: It's not common, but it is important that we capture these things. Because for a lot of those laboratory tests, there are treatments that are available, or other health implications if those come back positive. So, the case I think of is a polycythemia vera patient who had diffused subacute onset chorea and was able to be treated, was temporarily managed with medication for her chorea, and as her PV improved, she was able to come off those medications. As I was alluding to before---and I'm sure we'll talk about genetic testing---if you test for HD and it's negative, do you go down the route of additional expensive genetic testing, or do you then circle back and go, oops, I missed this treatable condition? As we talk about genetic testing as well, getting HD testing is a pretty involved process. And so, we want to make sure we are checking all those boxes before we move forward. So, it's not common, but we do catch some treatable conditions, and that's really important not to miss. Dr Berkowitz: That's very interesting. So, you diagnosed that polycythemia vera by blood smear, is that how you make the diagnosis? Dr Moore: Yes. Dr Berkowitz: And is that a once-in-a-career-so-far type of thing, or does that happen time to time? Dr Moore: For me, that's a once-so-far, but I don't doubt that I'll see it again. Dr Berkowitz: Great. And how about lupus and some of these other things we look for in the absence of other systemic features? Have you picked up any of these or heard of colleagues picking up something on laboratory testing? They said, oh, this patient came in for a referral for genetic testing, negative Huntington's disease. And good news, we found polycythemia vera; good news, we found undiagnosed lupus and we reversed it. I'm just curious, epidemiologically, seeing these long lists and not having the subspecialty practice that you do, how often you find a reversible cause like we do for neuropathy all the time, right? Oh, it's diabetes, it's B12---maybe not reversible, but preventing progression---or reversible dementia work up. You get so excited when you find low B12 and you replete the patient's B12, and they get better when they had been concerned they were developing an irreversible condition. How often does one in your subspecialty find a reversible cause on that initial mega-lab screen? Dr Moore: I think it's really uncommon, and maybe the folks that do are caught by someone else that never make it to Huntington's clinic, but I don't tend to see those cases. There are, of course, case reports and well-described in the literature about lupus and movement disorders and things of that nature, but that doesn't come to our clinic on a regular basis for sure. Dr Berkowitz: Got it. That's helpful to hear. Well, we've alluded to genetic testing a number of times now, so let's go ahead and talk about it. A lot of your article focuses on Huntington disease, and I was thinking about---in the course of our medical training in medical school, and then neurology residency, for those of us who don't become movement disorder experts like yourself---we learn a lot about Huntington disease. That's sort of the disease that causes chorea, until we later learned there are a whole number of diseases, not just the reversible causes we've been talking about, but a number of genetic diseases which you expertly reviewing your article. So, what are some of the red flags that suggest to you that a patient with chronically progressive chorea---and whom you're concerned for Huntington's or another genetic cause---what are some things you notice about the history, about the exam, the symptoms, the signs, the syndrome, that suggest to you that, actually, this one looks like it might not turn out to be HD. I think this patient might have something else. And as you have alluded to, how do you approach this? Do you send HD testing, wait for it to come back, and then go forward? Are there genetic panels for certain genetic causes of chorea? Do you skip just a whole exome sequencing, or will you miss some of the trinucleotide repeat conditions? How do you approach this in practice? Dr Moore: I'll try to tackle all that. One thing I will say is that a lot of patients with chorea, regardless of the cause, can look very similar to one another. So, if you're looking at chronic onset chorea, perhaps with some neuropsychiatric features, I'm going to most often think about HD because that's the most common cause. Certainly, as we mentioned before, if there's a lot of tongue protrusion, I would think about the acanthocytic conditions, neurocanthocytosis and McCloud syndrome. But generally in those conditions, we're looking at HD as the most likely cause. Certainly, if there is epilepsy or some other syndromic types of things going on, I may think more broadly. But it's important to know that while HD, as you mentioned, is the cause of chorea, many of our patients will have parkinsonism, tics, dystonia, a whole host of other movement phenomenologies. So, that wouldn't dissuade me from thinking about HD. When we think about the kind of patients that you're describing, upwards of 95% of those people will have Huntington's disease. And the process for genetic testing is fairly involved. The Huntington's Disease Society of America has organized a set of recommendations for providers to go about the process of genetic testing in a safe and supportive way for patients and their families. And so that's referred to in the article because it really is important and was devised by patients and families that are affected by this disease. And so, when we're thinking about genetic testing for HD, if I reveal that you have HD, this potentially affects your children and your parents and your siblings. You can have a lot of implications for the lives and health and finances of your family members. We also know that there is high suicidality in patients with HD, in patients who are at risk for HD; and there's even a higher risk of suicidality in patients who are at risk but test negative for HD. So, we do recommend a supportive environment for these patients and their families. And so, for presymptomatic patients or patients who are at risk and don't have chorea, this involves making sure we have, sort of, our ducks in a row, as it were, when we think about life insurance, and, do you have somebody supportive to be with you through this journey of genetic testing, no matter what the results are? So, oftentimes I'll say to folks, you know, there's this 20-page policy that I encourage you to look at, but there are Huntington's Disease Centers of Excellence across the country that are happy to help you with that process, to make sure that the patients are well supported. This is an individual genetic test because, as you mentioned, it is a CAG repeat disorder. And unfortunately, there is no chorea panel. So, if an HD test comes back negative, what we'll do then is think about what's called the HD phenocopies. As I mentioned before, some of these patients who look like they have HD will have a negative HD test. And so, what do you do then? Well, there's a handful of phenocopies---so, other genetic mutations that cause a very similar presentation. And so, we try to be smart, since there's not a panel, we try to be smart about how we choose which test to do next. So, for instance, there's a condition called DRPLA that is present in an African-American family here in my area, in North Carolina, as well as in Japan. And so, if someone comes from those backgrounds, we may decide that that's the next test that we're going to do. If they are white European descent, we may consider a different genetic test; or if they're sub-Saharan African, we may choose a different one from that. However, even if you do a really thorough job, all those blood tests, all those genetic tests, you will occasionally get patients that you can't find a diagnosis for. And so, it's important to know even when you do a good job, you may still not find the answer. And so, I think trying to do things with this complex of the presentation in a systematic way for yourself so you're not missing something. So, going back to our answer about, how do I look at lupus and polycythemia vera and all of that, to think about it in a systematic way. That when you get to the end and you say, well, I don't have an answer, you know you've tried. Dr Berkowitz: That's very helpful to hear your approach to these challenging scenarios, and also how to approach the potential challenging diagnosis for patients and their families getting this diagnosis, particularly in the presymptomatic phase. And your article touches on this with a lot of nuance and thoughtfulness. So, I encourage our listeners to have a read of that section as well. So, last here, just briefly in our final moments, you discuss in your article the various symptomatic treatments for chorea. We won't have time to go into all the details of all the many treatments you discussed, but just briefly, how do you decide which medication to start in an individual patient with chorea for symptomatic management? What are some of the considerations related to the underlying condition, potential side effect profiles of the particular medications, or any other considerations just broadly, generally, as you think about choosing one of the many medications that can be used to treat chorea? Dr Moore: Certainly. So, there is a group of FDA-approved medications, VMAT2 inhibitors, that we can choose from, or the off-label use of neuroleptics. And so, there's a lot of things that go into that. Some of that is insurance and cost and that sort of thing, and that can play a role. Others are side effects. So, for the VMAT2 inhibitors, they all do have a black box warning from the FDA about suicidality. And so, if a patient does struggle with mental health, has a history of suicidality, psychiatric admissions for that sort of thing, then I would be more cautious about using that medication. All patients are counseled about that, as are their families, to help us give them good support. So, the neuroleptics do not tend to have that side effect and can help with mood as well as the chorea and can be helpful in that way. And some of them, of course, will have beneficial side effects. So, olanzapine may help with appetite, which can be important in this disease. So, the big considerations would be the black box warning and suicidality, as well as, are we trying to just treat chorea or are we treating chorea and neuropsychiatric issues? Dr Berkowitz: Fantastic. Thank you for that overview. And again, for our listeners, there's a lot more detail about all of these medications, how they work, how they're used in different patient populations, their side effects, etc, to be reviewed in your excellent article. Again, today, I've been interviewing Dr Kathryn Moore about her article on diagnosis and management of Huntington's disease in chorea, which appears in the August 2025 Continuum issue on movement disorders. Be sure to check out Continuum Audio episodes from this and other issues. And thank you so much to our listeners for joining today. And thank you again, Dr Moore. Dr Moore: Thanks for having me. Dr Monteith: This is Dr Teshamae Monteith, associate editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

In this Episode, we tackle Huntington's latest Comic Con. The Pullman GeekFest. Let us know if you attended GeekFest and give us your opinion on the Comic-Con/Festival!! Make sure to follow us on all SOCIAL MEDIA @FrankOpinionswithAdamandTaskerhttps://www.instagram.com/frank_opinions_podcast/?hl=en

Comments from Marshall head football coach Tony Gibson. 

On this West Virginia Morning, the immigration crackdown is creating concerns for the specialized workforce of Kentucky's billion-dollar horse racing industry. And a Huntington music fest celebrates the diverse sounds of a troubled teen from the early 1900s. The post ICE Raids Affect Horse Racing And Diamond Teeth Mary Still Sparkles, This West Virginia Morning appeared first on West Virginia Public Broadcasting.

Connecticut's AG will defend Revolution Wind in court this week. Early voting for a few primaries begins today in a handful of Connecticut towns. An effort in Huntington is already gearing up for America's 250th anniversary. Plus, the latest from WSHU's Long Story Short.

Q-BANK: https://www.patreon.com/highyieldfamilymedicineIntro - (0:35),Normal aging (1:37),Mild cognitive impairment (2:37),Reversible causes of dementia (4:50),Vitamin B12 deficiency (5:09),Hypothyroidism (6:25),Pseudo-dementia (6:52),Normal pressure hydrocephalus (7:42),Neurosyphilis (8:57),HIV-associated dementia (10:05),Delerium (11:04),Alzheimer's disease (12:38),Vascular dementia (17:25),Lewy body dementia (18:58),Frontotemporal dementia (20:38),Huntington's disease (21:55),Creutzfeld-Jakob disease (22:54),Practice questions (24:03)

Young leaders are born in the show rings of 4-H fairs and Cora Rodibaugh knows that story well. This week, the 10-year 4-H'er sits down with Indiana Farm Bureau's Annie Romine to recap her career, talk about her love of pigs and the skills she's acquired along the way that have her equipped her for the future. She also shares what's ahead for her as she takes off for Purdue University to start the fall semester. Indiana Farm Bureau is dedicated to supporting youth in agriculture. There are several opportunities for youth to be involved in INFB, with scholarships for graduating seniors and college students; Collegiate Farm Bureau chapters at Purdue, Huntington and Vincennes universities; FFA advocacy days and trainings; grants for FFA chapters and 4-H clubs; and leadership development events, competitions, awards and conferences. INFB also serves as presenting sponsor of the Exhibitor Experience for livestock exhibitors and all showmanship contests at the Indiana State Fair.  You can learn more at infb.org/YFAP. 

America's Prom Queen and King might be our only hope... Swift x Kelcie = trU Luv 4evA Tickets for The Big Baby Tour https://www.whitneycummings.com 09/05 – Ridgefield, CT0 9/06 – Huntington, NY 09/12 – Vancouver, BC 09/19 – Richmond, VA 09/20 – Norfolk, VA 09/26 -- Riyadh, Saudi Arabia 10/03 – Toronto, ON 10/04 – Baltimore, MD 10/24 – Fayetteville, AR 10/25 – Hot Springs, AR1 1/21 – Reading, PA 11/22 – Philadelphia, PA 12/05 – Fort Lauderdale, FL 12/06 – New Orleans, LA SHOP: https://whitneycummings.com/index.html#store Thank you to our sponsors! Quit with Jones Quit with Jones combines discreet, FDA-approved nicotine mints with a behavioral-support app to help you quit or cut back.Take the free quiz and get matched to the right strength mint, plus track your progress and get daily support with the Jones app.

Lee Corso steps down after a legendary career.  Drew pays tribute.  Week 1 of college football had lots of theater but Drew says calm down about Arch Manning.  The Buffs had a bad first outing.  Drew is weighing in on the Micah Parsons trade.  We've got an NFL preview and the Rockies struggles continue.

On this episode, Jen and Frank chat with author Neil O'Brien! Neil's book, 'After Disney: Toil, Trouble, and the Transformation of America's Favorite Media Company' tells the untold succession struggle at Walt Disney Productions following the death of its founder. Neil O'Brien is an award-winning journalist and producer who has worked at NBC News for more than two decades. He has produced hundreds of hours of television over the course of his career and his work with NBC News Specials has been honored with four Emmy Awards, a Peabody, and an Edward R. Murrow Award. *** Dillo's Diz 55 Gerard St. #987. Huntington, NY 11743 Affiliate Links Music & Themes produced by Matt Harvey. Feedspot's Top 25 Siblings Podcasts You Must Follow AND Top 100 Disney Podcasts You Must Follow. ONE STOP SHOP ALL THE @DillosDiz LINKS! DIllo's Diz Resort Guests: Theme Park Rob, Jeffers, Skipper Bob, Nathaniel Hardy, Louis and Dr. Val of #FigmentsInTime, Lee Taylor, Maz, Troy with the Disney Assembled Podcast, Judy Van Cleef, Ryan Alexander, PixieDustPhD, Jason Romans, Holly Maddock, Lexi Andrea, Adam Elmers, DCLDuo, Disney Assembled Question or Comment? We LOVE interacting with listeners! FOLLOW Dillo's Diz on INSTAGRAM: https://www.instagram.com/dillosdiz/ FOLLOW Dillo's Diz on YOUTUBE: https://www.youtube.com/dillosdiz FOLLOW Dillo's Diz on TWITTER: https://twitter.com/dillosdiz

DJ Mo Radio - Mo Music. Mo Variety. In The Mix. - WPRK 91.5 Winter Park-Orlando, FL Fridays 5-7pm EST - Super Station 104.5 Huntington, WV - Mondays-Thursdays 9-11pm EST & Fridays 5-7pm EST - Sundays 8pm-12am EST - 93.3 Tiger FM Fort Myers, FL - Website: http://www.djmoradio.com Facebook: https://www.facebook.com/djmoradio Instagram: https://www.instagram.com/djmoradio DJ Mo Booking: 407-501-6345 Email: djmoradio@gmail.com

EPISODE #116 - Heard you missed him well he's back! Diamond David Lee Roth has hit the road with his solo band playing a solidly packed all classic Van Halen set list. Loud Dave has a spirited discussion with special guest Darren Paltrowitz (author of “DLR Book: How David Lee Roth Changed the World,” co-host of The DLR Cast) about Roth's August 10th concert at The Paramount in Huntington, NY, which they both attended. The Daves breakdown the new “Balance” (Expanded Edition) box set plus more VH News and a new mailbag segment complete this August episode.Download the podcast for free on Spreaker, iHeartRadio, Spotify, Google podcasts, Amazon Music, Podvine or iTunes. Connect with the Daves on Twitter: @ddunchained, Facebook: Dave & Dave Unchained – A Van Halen podcast, Instagram: ddunchainedpodcast or via email: ddunchainedpodcast@gmail.com

Nick and Jonathan give each other quick stories around sports and give their instant reaction to them.

On today's episode of The Drive, Paul welcomes Ryan Crisp, Associate Athletics Director for Annual Giving, Ticket Sales & Fan Engagement at Marshall, and Jeff Dantzler from the Bulldogs Sports Network. Get the latest on Herd athletics and hear insider perspectives straight from the source. 

Progressive supranuclear palsy and corticobasal syndrome are closely related neurodegenerative disorders that present with progressive parkinsonism and multiple other features that overlap clinically and neuropathologically. Early recognition is critical to provide appropriate treatment and supportive care. In this episode, Teshamae Monteith, MD, FAAN speaks with Nikolaus R. McFarland, MD, PhD, FAAN, author of the article “Progressive Supranuclear Palsy and Corticobasal Syndrome” in the Continuum® August 2025 Movement Disorders issue. Dr. Monteith is the associate editor of Continuum® Audio and an associate professor of clinical neurology at the University of Miami Miller School of Medicine in Miami, Florida. Dr. McFarland is an associate professor of neurology at the University of Florida College of Medicine at the Norman Fixel Institute for Neurological Diseases in Gainesville, Florida. Additional Resources  Read the article: Progressive Supranuclear Palsy and Corticobasal Syndrome Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @headacheMD Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Monteith: Hi, this is Dr Teshamae Monteith. Today I'm interviewing Dr Nikolaus McFarland about his article on progressive supranuclear palsy and cortical basilar syndrome, which appears in the August 2025 Continuum issue on movement disorders. Welcome, how are you? Dr Farland: I'm great. Thank you for inviting me to do this. This is a great opportunity. I had fun putting this article together, and it's part of my passion. Dr Monteith: Yes, I know that. You sit on the board with me in the Florida Society of Neurology and I've seen your lectures. You're very passionate about this. And so why don't you first start off with introducing yourself, and then tell us just a little bit about what got you interested in this field. Dr Farland: I'm Dr Nicholas McFarlane. I'm an associate professor at the University of Florida, and I work at the Norman Fixel Institute for Neurological Diseases. I am a director of a number of different centers. So, I actually direct the cure PSP Center of Care and the MSA Center of Excellence at the University of Florida; I also direct the Huntington's clinic there as well. But for many years my focus has been on atypical parkinsonisms. And, you know, I've treated these patients for years, and one of my focuses is actually these patients who suffer from progressive supranuclear palsy and corticobasal syndrome. So that's kind of what this review is all about. Dr Monteith: You probably were born excited, but I want to know what got you interested in this in particular? Dr Farland: So, what got me interested in this in particular was really the disease and the challenges that's involved in it. So, Parkinson's disease is pretty common, and we see a lot of that in our clinic. Yet many times, roughly about 10 to 15% of my patients present with these atypical disorders. And they're quite fascinating. They present in different ways. They're fairly uncommon. They're complex disorders that progress fairly rapidly, and they have multiple different features. They're sort of exciting to see clinically as a neurologist. I think they're really interesting from an academic standpoint, but also in the standpoint of really trying to bring together sort of a team. We have built a multidisciplinary team here at the University of Florida to take care of these patients. They require a number of folks on that team to take care of them. And so, what's exciting, really, is the challenge of treating these patients. There are very limited numbers of therapies that are available, and the current therapies that we have often really aren't great and over time they fail. And so, part of the challenge is actually doing research. And so, there's actually a lot of new research that's been going on in this field. Recently, there's been some revisions to the clinical criteria to help diagnose these disorders. So, that's really what's exciting. The field is really moving forward fairly rapidly with a number of new diagnostics, therapeutics coming out. And hopefully we can make a real difference for these patients. And so that's what really got me into this field, the challenge of trying to treat these patients, help them, advocate for them and make them better. Dr Monteith: And so, tell me what the essential points of this article. Dr Farland: So, the essential points, really, of this article is: number one, you know, just to recognize the new clinical criteria for both PSP and corticobasal syndrome, the diagnosis for these disorders or the phenotypic spectrum has really expanded over the years. So, we now recognize many different phenotypes of these disorders, and the diagnosis has gotten fairly complicated. And so, one of the goals of this article was to review those new diagnostic criteria and the different phenotypic ways these diseases present. I wanted to discuss, also, some of the neuropathology and clinicopathological overlap that's occurred in these diseases as well as some of the new diagnostic tests that are available. That's definitely growing. Some of the new studies that are out, in terms of research and clinical trials. And then wanted to review some of the approaches for treatment for neurologists. Particularly, we're hoping that, you know, this article educates folks. If you're a general neurologist, we're hoping that recognizing these diseases early on will prompt you to refer these patients to specialty clinics or movement disorder specialists early on so they can get appropriate care, confirm your diagnosis, as well as get them involved in trials if they are available. Dr Monteith: And how has the clinical criteria for PSP and cortical basilar syndrome changed? Dr Farland: I think I already mentioned there's been an evolution of the clinical criteria for PSP. There's new diagnostic criteria that were recently published, and it recognizes the multiple clinical phenotypes and the spectrum of the disease that's out there, which is much broader than we thought about. Corticobasal clinical criteria are the Dr Armstrong criteria from 2013. They have not been updated, but they are in the works of being updated. But it does recognize the classic presentation of corticobasal syndrome, plus a frontal executive predominant and then a variant that actually overlaps with PSP. So, there's a lot more overlap in these two diseases than we originally recognized. Dr Monteith: And so, you spoke a bit about FTD spectrum. So why don't you tell us a little bit about what that is? I know you mentioned multiple phenotypes. Dr Farland: What I really want to say is that both PSP and corticobasal syndrome, they're relatively rare, and what- sort of as to common features, they both are progressive Parkinson disorders, but they have variable features. While they're commonly associated with Parkinson's, they also fit within this frontotemporal lobar spectrum, having features that overlap both clinically and neuropathologically. I just want folks to understand that overlap. One of this pathological overlap here is the predominant Tau pathology in the brain, an increasing recognology- recognition of sort of the pathological heterogeneity within these disorders. So, there's an initial description, a classic of PSP, as Richardson syndrome. But now we recognize there are lots of different features to it and there are different ways it presents, and there's definitely a lot of clinical pathological overlap. Dr Monteith: Why don't we just talk about some red flags for PSP? Dr Farland: Yeah, sure. So, some of the red flags for PSP and even corticobasal syndrome are: number one is rapid progression with early onset of falls, gait difficulty, falling typically backwards, early speech and swallow problems that are more prominent than you see in Parkinson's disease, as well as eye gaze issues. So, ocular motor features, particularly vertical gaze palsy. In particular what we talk about is the supranuclear gaze palsy, and one of the most sensitive features that we've seen with these is downgaze limitation or slowed downgaze, and eventually a full vertical gaze palsy and followed supranuclear gaze palsy. So, there's some of the red flags that we see. So, while we think about the lack of response to levodopa frequently as something that's a red flag for Parkinson's, there are many times that we see Parkinson's patients, and about a quarter of them don't really respond. There's some features that don't respond to levodopa that may not be so specific, but also can be helpful in this disease. Dr Monteith: And what about the red flags for cortical basilar syndrome? Dr Farland: So, for cortical basilar syndrome, some of the red flags again are this rapidly depressive syndrome tends to be, at least in its classical present presentation, more asymmetric in its presentation of parkinsonism, with features including things like dystonic features, okay? For limb dystonia and apraxias---so, inability to do a learned behavior. One of those red flags is a patient who comes in and says, my hand doesn't work anymore, which is something extremely uncommon that you hear in Parkinson's disease. Most of those patients will present, say, I might have a tremor, but they very rarely will tell you that I can't use my hand. So look out for that sign. Dr Monteith: And let's talk a little bit about some of the advances in the fields you mentioned, evolving biomarker and imaging capacities. So, how are these advances useful in helping us understand these conditions, especially when there's so much heterogeneity? Dr Farland: I might start by talking a little bit about some of the clinical criteria that have advanced. Why don't we start there and just discuss some of the advances? I think in PSP, I think, originally we had both probable and possible diagnoses of PSP, and the diagnostic criteria were basically focused on what was what's called “classical PSP” or “Richardson syndrome”. But now we recognize that there are multiple phenotypes. There's an overlap with Parkinsonism that's slower in progression and morphs into PSP, the classical form. There's a frontal behavioral variant where patients present with that frontal behavioral kind of thing. There's a speech-language variant that can overlap with PSP. So they have prominent speech language, potentially even apraxia speech. So, recognition of these different phenotypes is sort of a new thing in this field. There's even overlap with cortical basal syndrome and PSP, and we note that the pathology can overlap as well. So, I think that's one of the things that have changed over time. And these were- recently came out in 2017 in a new publication in the Movement Disorders Society. So, in terms of diagnostic tests as well---and there's been quite a bit of evolution---really still to date, our best diagnostic test is imaging. MRI is really one of our best tests currently. Currently blood tests, spinal fluid, there's new biomarkers in terms of skin… they're still in the research phase and not necessarily very specific yet. So, we rely heavily on imaging still; and for PSP, what we're looking for largely are changes in the brain stem, and particularly focused on the midbrain. So disproportionate midbrain atrophy compared to the pons and the rest of the midbrain is a fairly specific intensive sign for PSP. Whereas in MSA we see more of a pontine atrophy compared to the midbrain. So that can be really helpful, and there are lots of different new measurements that can be done. PET scans are also being used as well. And there are new PET markers, but they still remain kind of research-based, but are becoming more and more prevalent and may be available soon for potential use. Although there's some overlap with PET tracers with Alzheimer's disease and different Tau isoforms. So, something to be wary about, but we will be seeing some of these soon coming out as well. More kind of up-to-date things include things like the spinal fluid as well as even some of the skin biopsies. And I think we've heard some word of recent studies that have come out that potentially in the very near future we might actually have some Tau protein tests that we can look at Tau either in spinal fluid or even in a skin biopsy. But again, still remains research-based and, we still need more information as to whether these tests can be reproducible and how sensitive or specific they are. Dr Monteith: It sounds like, when really approaching these patients, still, it's a lot of back to the history, back to the clinical and some basic imaging that we should be able to identify to distinguish these types of patients, and we're not quite where we need to be yet for biomarker. Dr Farland: I totally agree with you. I think it starts, really, with the clinical exam and that's our main focus here; and understanding some of the new clinical criteria which are more sensitive, but also specific, too. And they're really useful to look at. So, I think reviewing those; patients do progress, following them over time can be really useful. And then for diagnosis, getting imaging if you suspect a patient has an atypical presentation of parkinsonism, to look for signs or features that might be specific for these different disorders. Dr Monteith: Why don't we take a typical case, a typical patient that you would see in clinic, and walk us through the thought process---especially, maybe they presented somewhat early---and the different treatment approaches to helping the patient, and of course their family. Dr Farland: Yeah, sure. So, a typical patient might be someone who comes in with, like, a three year history of progressive gait problems and falling. And let's say the patient says, I'm falling backwards frequently. They may have had, like, a rib fracture, or they hit their head once, and they're describing some speech issues as well. Now they're relying on a walker and family members saying they rarely let them be by themselves. And there may be some slowing of their cognitive function and maybe a bit of withdrawal. So that's a typical patient. So, the approach here is really, what are some of the red flags? I think already you hear a red flag of a rapidly progressive disease. So, Parkinson's disease patients rarely have frequent falls within the first five years. So, this is within three years or less. You're already hearing early onset of gait problems and falling, and particularly falling backwards rather than forwards as often Parkinson's disease patients do. You're hearing early speech problems and maybe a subtle hint of cognitive slowing and some withdrawal. So, a lot of things that sort of are red flags. So, our approach really would be examining this patient really closely. Okay? We'd be listening to the history, looking at the patient. One thing is that some of these patients come in, they may be in a wheelchair already. That's a red flag for us. If they're wearing sunglasses---sometimes we see that patients, they have photosensitivity and they're in a chair and they're wearing sunglasses---you take the glasses off and you look at their face and they have that sort of a facial stare to them---not just the masked face, but the stare---and their eyes really aren't moving. So, another kind of clue, maybe this is probably something atypical, particularly PSP is what I'm thinking about. So, the approach is really, do a thorough exam. I always recommend looking at eye movements and starting with volitional saccades, not giving them a target necessarily, but asking them to look up and then look down. And then particularly look at the speed of downgaze and whether they actually have full versions down, are able to do that. That's probably your most sensitive test for a patient who has PSP. Not the upgaze, which can be- upgaze impairment in older patients can be nonspecific. So, look for that down gaze. So, if I can get out one message, that's one thing that can be easily done and examined fairly quickly for diagnosis of these patients. And then just look for signs of rigidity, bradykinesia, maybe even some myelopraxia, and then look at their gait carefully so that there's a high suspicion. Again, if there's some atypical features, imaging is really important. So, my next step would be probably getting an MRI to evaluate whether- do they have brain somatrophy or other widespread atrophy or other signs? You need to think about your differential diagnosis for some of these patients as well. So, common things are common; vascular disease, you can't have vascular parkinsonism or even signs of NPH. Both of those can present with progressive gait difficulty and falls. So, the gait may look more like Parkinson's rather than ataxic gait that we see in classic PSP, but still they have early gait issues, and that can be a mimicker of PSP, So looking for both of those things in your imaging. Think about sort of autoimmune potentially causes. So, if they have a really rapid progressive cause, there are some rare autoimmune things. There have been recent reports of things like IgLON5, although there's limited cases, but we're doing more screening for some of those autoimmune causes. And then even some infectious causes like Whipples, that are rarely present like this. Okay? And have other signs and features. Dr Monteith: So, let's say you diagnose this patient with PSP and you're assessing the patients to see how you can improve their quality of life. So, what are some potential symptomatic managements that will help our patient? Dr Farland: I recommend for most all of these patients… while the literature indicates that many patients with PSP, and especially corticobasal syndrome, don't respond well to levodopa. So, the classic treatment for parkinsonism. However, we all recommend a trial of levodopa. These patients may respond partially to doses of levodopa, and we try to push the doses a bit higher. So, the recommended trial is usually a dose up to roughly 1000 milligrams of levodopa per day. And give it some time, at least two, if not actually three months of a trial. If not well-tolerated, you can back off. If there's no response at all or no improvement, then slowly back off and taper patients off and ask them to tell you whether they feel like they're actually worsening. So, many patients, sometimes, don't recognize the improvements, or family members don't recognize it until we actually taper them back off. And they may end up saying there are some other things that even recognize. Even some nonmotor benefits can be seen with levodopa. In some cases, we do keep them on levodopa, but levodopa's our best therapy for this. Dopamine agonists, MAO inhibitors, have all been sort of tried and they've been studied, but often don't really help or fail to help benefit these patients and could be fraught with some other side effects. I think many people do also turn to Amantadine as a treatment for Parkinson's, gait problems, freezing, if you see it in these disorders. Yet Amantadine is fraught with issues of side effects, including cognitive issues, and I think is not well-tolerated. But there are the rare patient who actually does respond to this or claims they respond to this. By and large, these patients relentlessly progress, unfortunately. So, beside treatment of other symptoms, I think it's really important to recognize that they require supportive cares and therapy. So, starting those early on and getting your allied healthcares kind of involved. So that includes people like physical, occupational therapy for the gait issues, the falls, occupational therapy for doing daily activities. Speech language pathology can be really a critical player for these because of the early speech and language issues, as well as swallow difficulties. Swallow is compared quickly in these patients. And so, we do recommend the screening evaluation, then often following patients either every six- or even annually, at least, with a swallow evaluation. And we recommend the fluoroscopic-guided kind of modified barium swallow for these patients.  Dr Monteith: And how does that differ if, let's say, the patient had cortical basilar syndrome? What are some of the symptomatic treatments that would be high on your consideration? Dr Farland: So actually, these patients also have a very similar approach, and they often have some overlapping features. Maybe a little bit of difference in terms of the level of apraxia and some dystonic features that you see in corticobasal syndrome. So, as I mentioned earlier that these patients have a more typ- when they present, typically have a more asymmetric presentation. And one of the biggest issues is this limb apraxia. They may have abnormal movements as well as, like, the alien limb-type phenomena as well. So, the focus of therapy, while similar in the sense we focus on the parkinsonism, I do always try levodopa and try to ramp up the doses to see if it benefits. It does often fail, but it's definitely worth trying. The other focus of these patients is trying to treat symptoms. Dystonia, those features… in some cases, we can help; if it's painful or uncomfortable, muscle relaxants can be used. If it's vocal, things like Botox can be really helpful. Often times it is more palliative than actually restorative in terms of function, but still can be really helpful for patients who ask about pain and discomfort and trying to treat. And then of course, again, the focus on our supportive care. We need to build that network and build that team of folks, the therapists, the physical, occupational, and the speech therapist to help them. If they have language problems---like either in PSP or corticobasal---I'll also include my request to a speech language pathologist to work on cognitive function. That's a special, additional thing you have to ask for and then specifically request when you make a referral to a speech language pathologist. Dr Monteith: That is so important. I think keeping the simulation, keeping the social support, and I would probably guess that you would also include screening for sleep and mood disorder. Dr Farland: Absolutely. Mood disorders are really big in these diseases. Patients are suffering terribly. You do hear about labile mood in both of these diseases, particularly PSP; and even what's called pseudobulbar palsy, where the mood is not always congruent with the affect. So they may laugh or cry inappropriately, and particularly the crying can be very disturbing to family and caregivers to see that. And so, treating those things can be really important. So always asking about the mood issues. Depression in particular is something that we're very sensitive about, and there is a higher incidence of suicidal ideations. Asking about that and feeling and making sure that they are in a safe environment can be really important. Dr Monteith: Thank you so much. Dr Farland: Thank you. Dr Monteith: Today I've been interviewing Dr Nikolaus McFarland about his article on progressive supranuclear palsy and cortical basilar syndrome, which appears in the August 2025 Continuum issue on movement disorders. Be sure to check out Continuum Audio episodes from this and other issues, and thank you to our listeners for joining today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

What downtown have you explored the most this summer? We're sharing a charming new spot you'll want to visit year-round — hint: it looks like it was pulled straight from a Hallmark movie! This week, Sharon and Stacy recap their time representing Discover Long Island at some exciting partner activations, including the First Annual Oyster Jamboree at Smith Point County Park and Tyler Hubbard live at the Catholic Health Amphitheater at Bald Hill! They also dive into some current hot topics, including controversial school bans, and share a heartwarming story about a local Long Islander who turned a difficult moment into something special for his daughter as she heads off to school each day. Plus — did someone say PIZZA FESTIVAL coming to Long Island?!

Today on The Drive, Paul shares comments from Marshall head coach Tony Gibson as the Herd prepares for Georgia. Plus, an interview with Marshall cross country runner Braden Elswich on the upcoming season and his goals for the year. 

Radiate The Word with Rev. Lynn Ann LaBorde. Rev. LaBorde is a gifted psychic medium and the Founder of Sohmalife Sanctuary, where she serves as a Transformational Alchemist. With over 40 years of experience in metaphysical practices—including advanced Pranic healing, hypnotherapy, and Reiki—Lynn helps individuals transmute struggles into spiritual growth and enlightenment. She is the author of Channeling Magdalene, A Year of Love and has trained with renowned mentors in the healing arts. Through Sohmalife Sanctuary in Huntington, New York, she has hosted over 500 events and reached more than 25,000 people worldwide, both online and in person. Her mission is to guide others back to their divine essence and natural, healthy state as sacred beings. ✨ Ready to live more authentically—in your emotions, your energy, and your relationships? Book a session with our expert practitioners at radiatewellnesscommunity.com/appointment.

Marshall heads to Athens, and The Drive has your preview. Paul Swann breaks down the matchup with comments from Marshall head coach Tony Gibson and Georgia head coach Kirby Smart. Hear what both coaches are saying ahead of Saturday's showdown. 

"The Blue Within" dives into the surprising potential of methylene blue—a simple dye with extraordinary promise—in the fight against Huntington's disease and other neurodegenerative disorders. Blending science, human stories, and community voices, this episode asks: could one drop of blue hold the key to clarity, hope, and change? 

HC Kevin Stefanski and DL Adin Huntington join Nathan Zegura and Je'Rod Cherry to preview the final preseason game of the year on this week's episode of The Kevin Stefanski Show!See omnystudio.com/listener for privacy information.

In this 2023 episode, Huntington, West Virginia, is an idyllic college town on the Ohio River, but it's also the epicenter of a massive international internet crime ring. Federal prosecutor Katie Robeson hasn't been on the job long when she learns that a woman in Huntington is acting as money mule, transferring millions of dollars overseas. Following the money, Katie and her team uncover an elaborate network of criminals, based in Huntington, targeting people all over the country.

In this episode, noted Marshall soccer broadcaster Bobby Iddings joins Paul Swann to preview the kickoff of the Marshall University Men's Soccer season. 

On this episode, Jen and Frank chat with DillosDizResort.com guest Ryan Alexander about his incredible trip to Alaska on Disney Cruise Line! *** Dillo's Diz 55 Gerard St. #987. Huntington, NY 11743 Affiliate Links Music & Themes produced by Matt Harvey. Feedspot's Top 25 Siblings Podcasts You Must Follow AND Top 100 Disney Podcasts You Must Follow. ONE STOP SHOP ALL THE @DillosDiz LINKS! DIllo's Diz Resort Guests: Theme Park Rob, Jeffers, Skipper Bob, Nathaniel Hardy, Louis and Dr. Val of #FigmentsInTime, Lee Taylor, Maz, Troy with the Disney Assembled Podcast, Judy Van Cleef, Ryan Alexander, PixieDustPhD, Jason Romans, Holly Maddock, Lexi Andrea, Adam Elmers, DCLDuo, Disney Assembled Question or Comment? We LOVE interacting with listeners! FOLLOW Dillo's Diz on INSTAGRAM: https://www.instagram.com/dillosdiz/ FOLLOW Dillo's Diz on YOUTUBE: https://www.youtube.com/dillosdiz FOLLOW Dillo's Diz on TWITTER: https://twitter.com/dillosdiz

Today on The Drive with Paul Swann: Marshall women's basketball coach Juli Fulks joins the show. Plus, Marshall announces a future series with Kentucky.

The Rockies are beginning to turn some heads with their much imporved play.  Newcommer Kyle Karros joins the show to talk about his ascent to the big leagues and his first big league hit and RBI.

Today on The Drive with Paul Swann: Gerald Harrison, Marshall's new Director of Athletics, joins the show as he begins his first week on the job.

In today's episode of The Drive with Paul Swann, hear comments from Marshall football head coach Tony Gibson as he evaluates the Thundering Herd's readiness for their tough season opener at Georgia.

It's a bouquet of trivia on Go Fact Yourself!Mimi Rogers is an actor – best known these days for her role in the Amazon series “Bosch.” She's acted in that family of shows for ten years, but she'll tell us about how her part was never supposed to last that long. Plus, Mimi reveals some secrets about her time on the set of Austin Powers.  Matt Rogers is one of the hosts of the podcast Las Culturistas. He'll tell us about how he started the show with his co-host Bowen Yang, his Christmas themed album and tour that he manifested, and what it was like to actually meet the Queen of Christmas: Mariah Carey. You can stream “The Las Culturistas Culture Awards” on Peacock now.Areas of Expertise:Mimi: Types of flowers, Italian cooking, and the TV show “Sex and the City.”Matt: Mariah Carey, the TV show “The Real Housewives of New York” (pre-reboot), and Long Island. What's the Difference: Boring!What's the difference between drilling and boring?What's the difference between a hog and a boar?Guest Experts:Dr. Nicole Cavender: Director of the Botanical Gardens at The Huntington.Rep. Laura Gillen: United States Congresswoman serving New York's fourth district. Hosts: J. Keith van StraatenHelen HongCredits:Theme Song by Jonathan Green.Maximum Fun's Senior Producer is Laura Swisher.Co-Producer and Editor is Julian Burrell.Seeing our next live-audience shows by YOU!