Podcasts about Interruption

People Are Marrying AI Now (And Your Business Should Be Worried). New Episode

In the opening message of the Sightline series, Pastor Nick Newman taught that many people can be near Jesus yet still disconnected from Him, often preoccupied with “cleaning their nets” instead of surrendering their lives. Through Luke 5:1–4, he showed that Jesus steps into our mess before we clean it up, offering a divine interruption that redirects our future, invites us deeper, and calls us to trust His voice over our frustration. This message was recorded live on February 15th, 2026.

The biblical account of Moses and the burning bush exemplifies how God can interrupt us right in the middle of our routines. Before Moses ever heard the voice of God, he had to decide to "turn aside" to pursue "God's Interruption." - When God Interrupts Your Program.

Subscribe for more Videos: http://www.youtube.com/c/PlantationSDAChurchTV Theme: God restores our identity and heals the heart by making love safe again. Speaker: Pastor Latoya Smythe-Forbes Title: Where Did Love Go? Key text: https://www.bible.com/bible/59/GEN.3.10.esv Bulletin/Notes: http://bible.com/events/49562996 Date: February 14, 2026 Tags: #psdatv #love #fear #God #heart #safe #safety #intimacy #interruption #restore #restoration #WhereDidLoveGo #LoveDidntLeave #FromFearToHealing #RestoringTheHeart #SafeInGodsLove For more life lessons and inspirational content, please visit us at http://www.plantationsda.tv. Church Copyright License (CCLI): 1659090 CCLI Streaming Plus License: 21338439Support the show: https://adventistgiving.org/#/org/ANTBMV/envelope/startSee omnystudio.com/listener for privacy information.

China Government Turns APPS into SPY Tools | Ex FBI Agent Explainsew Episode

Episode 212: Managing HFpEFHyo Mun and Jordan Redden (medical students) explain how to manage HFpEF with medications and touch some basics about nonpharmacologic treatments. Dr. Arreaza asks insightful questions to guide the discussion. Written by Hyo Mun, MSIV, American University of the Caribbean; and Jordan Redden, MSIV, Ross University School of Medicine. Comments by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice.Treatment of HFpEFArreaza: Mike, if you had to name the one therapy everyone with HFpEF should be on, what is it?Mike: That's easy! SGLT-2 inhibitors. This is the one slam-dunk we have in HFpEF. Empagliflozin (Jardiance) or dapagliflozin (Farxiga) should be started in essentially every patient with HFpEF, and it doesn't matter if they have diabetes or not.Jordan: And that's worth repeating, because people still think of these as “diabetes drugs.” They're not anymore. In HFpEF, SGLT-2 inhibitors reduce heart-failure hospitalizations, improve symptoms, improve quality of life, and even reduce cardiovascular death.Dr. Arreaza: They're also simple. Empagliflozin 10 mg daily or dapagliflozin 10 mg daily. No titration, no drama. The effectiveness of these meds was established around 2019 with DAPA-HF and later with DELIVER. These were trials thatdemonstrated that dapagliflozin reduces worsening heart failure and cardiovascular events across the full spectrum of heart failure, from reduced to preserved ejection fraction, independent of diabetes status.Mike: And the number needed to treat is about 28 to prevent one heart-failure hospitalization. That's excellent for a disease where we historically had almost nothing that worked.Jordan: They're also safe in chronic kidney disease down to an eGFR of about 25, which makes them even more useful in this population.Dr. Arreaza: Alright. We got SGLT-2 inhibitor, what's next?Mike: Volume management. Loop diuretics are still the backbone of symptom control in HFpEF. If the patient is volume overloaded, you diurese, and you diurese aggressively.Jordan: The goal is euvolemia. Dry weight, no edema, no orthopnea, no waking up gasping for air. A lot of these patients end up needing chronic oral loop diuretics to stay there.Dr. Arreaza: Something to remember: HFpEF patients don't tolerate congestion well, and being “a little wet” is not benign. Let's move into RAAS inhibition. Where do ARBs and ACE inhibitors fit in?Mike: Between ARBs and ACE inhibitors, ARBs are the winners in HFpEF. They actually reduce heart failure hospitalizations—drugs like candesartan, losartan, valsartan. ACE inhibitors? Not so much. They showed minimal benefit in older HFpEF patients, which is why we go with ARBs instead.Jordan: But a lot of clinicians get nervous about ACE inhibitors and ARBs because of kidney function, so it's worth talking through how these drugs actually work in the kidney.Dr. Arreaza: Yes, misunderstanding may lead to unnecessary drug discontinuation.Jordan: Under normal conditions, the afferent arteriole brings blood into the glomerulus, and the efferent arteriole is constricted by angiotensin II. That constriction keeps pressure high in the glomerulus and maintains filtration.Mike: Here's what happens with an ACE inhibitor: you block angiotensin II, the efferent arteriole relaxes, glomerular pressure drops, and GFR dips slightly. Creatinine bumps up a little, and that scares people, but that's actually the whole point—that's how you get kidney protection long-term.Jordan: High intraglomerular pressure causes hyperfiltration injury and scarring over time. Lowering that pressure protects the kidney long-term. The short-term GFR drop is the price you pay for long-term benefits.Dr. Arreaza: So let's talk about CKD, because this is where people panic.Mike: Right. ACE inhibitors and ARBs are not contraindicated in chronic kidney disease. In fact, they're recommended even in advanced stages. They reduce progression to kidney failure by about a third.Jordan: The key is how you use them. Start low. Check creatinine and potassium one to two weeks after starting, then periodically. A creatinine rise up to 30% from baseline is acceptable. That's not kidney injury, that's physiology.Dr. Arreaza: And what about potassium creeping up?Mike: You adjust the dose or add a potassium binder. You don't just automatically stop the drug.Dr. Arreaza: Now there is one absolute contraindication everyone needs to know about! (board exam test)Jordan: Bilateral renal artery stenosis. This is the big one. In these patients, the kidneys are completely dependent on angiotensin II–mediated efferent constriction to maintain GFR. Take that away, and GFR collapses.Mike: Creatinine can jump dramatically within days. If you see a creatinine rise of 20% or more shortly after starting an ACE inhibitor, you should be thinking about bilateral renal artery stenosis and stopping the drug immediately.Dr. Arreaza: After revascularization, though, many patients can tolerate ACE inhibitors again, so this isn't always permanent. What about cardiorenal syndrome? That's where things get uncomfortable.Mike: It is uncomfortable, but cardiorenal syndrome isn't a contraindication. These patients have severe heart failure and kidney disease, and their mortality is actually higher than patients with heart failure alone.Jordan: ACE inhibitors still reduce mortality and slow kidney disease progression in this group. Studies show that stopping ACE inhibitors during acute heart-failure admissions increases in-hospital mortality three- to four-fold.Dr. Arreaza: So we are cautious, but we don't avoid it.Mike: Exactly. Start low, titrate slowly, monitor labs closely, accept up to a 30% creatinine rise. You only stop if kidney function keeps worsening, or potassium gets dangerously high.Dr. Arreaza: Alright. Let's move on. What about mineralocorticoid receptor antagonists… MRA?Jordan: Spironolactone or eplerenone might reduce hospitalizations in HFpEF, but the data is mixed. This is more of a “select patients” situation.Mike: And you have to watch potassium and kidney function carefully, especially if they're already on an ACE inhibitor or ARB.Dr. Arreaza: What about sacubitril-valsartan, also known as Entresto®?Mike: Entresto may help patients with mildly reduced EF roughly in the 45 to 57% range. It's not first-line for HFpEF, but in select patients, it's reasonable.Dr. Arreaza: Now let's clarify one of the biggest sources of confusion: beta blockers.Jordan: Beta blockers are not a treatment for HFpEF itself. They're only indicated if the patient has another reason to be on them, like coronary disease or atrial fibrillation.Mike: And timing really matters here. You absolutely do not start beta blockers during acute decompensated heart failure. Their negative inotropic effects can make things worse when patients are volume overloaded.Jordan: But, and this is critical, you also don't stop them if the patient is already taking one. Abrupt withdrawal causes a sympathetic surge and dramatically increases mortality.Dr. Arreaza: If a patient is admitted on a beta blocker, what do we do?Mike: Continue it at the same dose or reduce it slightly if they're really unstable. Once they're euvolemic and stable, you can carefully titrate up.Jordan: And watch for chronotropic incompetence. HFpEF patients often rely on heart-rate response to exercise, and beta blockers can worsen exercise intolerance.Dr. Arreaza: Beyond medications, HFpEF is really about treating comorbidities. Aerobic activity can be an initial strategy to improve exercise intolerance and has evidence of improving aerobic function and quality of life. Sodium restriction: improves symptoms, does not decrease risk of death or hospitalizations.Mike: Hypertension control is huge. For diabetes, the SGLT-2 inhibitors will perform double duty. For obesity, weight loss improves symptoms, and GLP-1 agonists like semaglutide are absolute gamechangers.Jordan: Don't forget sleep apnea, atrial fibrillation, and lifestyle. Exercise improves the quality of life, even if it doesn't change hard outcomes. Lifestyle is the main treatment. Dr. Arreaza: And when should you refer to cardiology?Mike: You should refer when the diagnosis isn't clear; symptoms are not responding to treatment, difficult volume management, end-organ dysfunction, or if you are concerned about advanced heart failure.Dr. Arreaza: So, it has been a great discussion. What is the takeaway?Mike: HFpEF treatment isn't about one magic drug -- it's about volume control, SGLT2 inhibitors, smart use of RAAS blockade, and aggressive management of comorbidities.Jordan: And it's understanding the physiology, so you don't withhold life-saving therapies out of fear.Dr. Arreaza: Well said. If you found this helpful, share it with a friend or colleague and rate us wherever you listen. This is Dr. Arreaza, signing off.Jordan/Mike: Thanks! Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! _____________________References:Barzin A, Barnhouse KK, Kane SF. Heart Failure With Preserved Ejection Fraction. Am Fam Physician. 2025;112(4):435-440.Heidenreich PA, Bozkurt B, Aguilar D, et al. 2022 AHA/ACC/HFSA guideline for the management of heart failure. Circulation. 2022;145(18):e895-e1032.Kittleson MM, Panjrath GS, Amancherla K, et al. 2023 ACC expert consensus decision pathway on management of heart failure with preserved ejection fraction. J Am Coll Cardiol. 2023;81(18):1835-1878.Anker SD, Butler J, Filippatos G, et al. Empagliflozin in heart failure with a preserved ejection fraction. N Engl J Med. 2021;385(16):1451-1461.Solomon SD, McMurray JJV, Claggett B, et al. Dapagliflozin in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2022;387(12):1089-1098.Pitt B, Pfeffer MA, Assmann SF, et al. Spironolactone for heart failure with preserved ejection fraction. N Engl J Med. 2014;370(15):1383-1392.Yusuf S, Pfeffer MA, Swedberg K, et al. Effects of candesartan in patients with chronic heart failure and preserved left-ventricular ejection fraction. Lancet. 2003;362(9386):777-781.Solomon SD, McMurray JJV, Anand IS, et al. Angiotensin-neprilysin inhibition in heart failure with preserved ejection fraction. N Engl J Med. 2019;381(17):1609-1620.Kosiborod MN, Abildstrøm SZ, Borlaug BA, et al. Semaglutide in patients with heart failure with preserved ejection fraction and obesity. N Engl J Med. 2023;389(12):1069-1084.Xie Y, Xu E, Bowe B, Al-Aly Z. Long-term cardiovascular outcomes of COVID-19. Nat Med. 2022;28(3):583-590.Puntmann VO, Carerj ML, Wieters I, et al. Outcomes of cardiovascular magnetic resonance imaging in patients recently recovered from COVID-19. JAMA Cardiol. 2020;5(11):1265-1273.Basso C, Leone O, Rizzo S, et al. Pathological features of COVID-19-associated myocardial injury. Eur Heart J. 2020;41(39):3827-3835.Nalbandian A, Sehgal K, Gupta A, et al. Post-acute COVID-19 syndrome. Nat Med. 2021;27(4):601-615.Badve SV, Roberts MA, Hawley CM, et al. Effects of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers in adults with estimated GFR less than 60 mL/min per 1.73 m². Ann Intern Med. 2024;177(8):953-963.Navis G, Faber HJ, de Zeeuw D, de Jong PE. ACE inhibitors and the kidney: a risk-benefit assessment. Drug Saf. 1996;15(3):200-211.Textor SC, Novick AC, Tarazi RC, et al. Critical perfusion pressure for renal function in patients with bilateral atherosclerotic renal vascular disease. Ann Intern Med. 1985;102(3):308-314.Hackam DG, Spence JD, Garg AX, Textor SC. Role of renin-angiotensin system blockade in atherosclerotic renal artery stenosis and renovascular hypertension. Hypertension. 2007;50(6):998-1003.Ronco C, Haapio M, House AA, et al. Cardiorenal syndrome. J Am Coll Cardiol. 2008;52(19):1527-1539.Prins KW, Neill JM, Tyler JO, et al. Effects of beta-blocker withdrawal in acute decompensated heart failure. JACC Heart Fail. 2015;3(8):647-653.Jondeau G, Neuder Y, Eicher JC, et al. B-CONVINCED: Beta-blocker CONtinuation Vs. INterruption in patients with Congestive heart failure hospitalizED for a decompensation episode. Eur Heart J. 2009;30(18):2186-2192.Theme song, Works All The Time by Dominik Schwarzer, YouTube ID: CUBDNERZU8HXUHBS, purchased from https://www.premiumbeat.com/.

Bongani Bingwa sits down with Jonathan “Khabazela” Fairban to talk about the moments when conversations get unexpectedly interrupted. 702 Breakfast with Bongani Bingwa is broadcast on 702, a Johannesburg based talk radio station. Bongani makes sense of the news, interviews the key newsmakers of the day, and holds those in power to account on your behalf. The team bring you all you need to know to start your day Thank you for listening to a podcast from 702 Breakfast with Bongani Bingwa Listen live on Primedia+ weekdays from 06:00 and 09:00 (SA Time) to Breakfast with Bongani Bingwa broadcast on 702: https://buff.ly/gk3y0Kj For more from the show go to https://buff.ly/36edSLV or find all the catch-up podcasts here https://buff.ly/zEcM35T Subscribe to the 702 Daily and Weekly Newsletters https://buff.ly/v5mfetc Follow us on social media: 702 on Facebook: https://www.facebook.com/TalkRadio702 702 on TikTok: https://www.tiktok.com/@talkradio702 702 on Instagram: https://www.instagram.com/talkradio702/ 702 on X: https://x.com/Radio702 702 on YouTube: https://www.youtube.com/@radio7See omnystudio.com/listener for privacy information.

For more coverage on the issues that matter to you, download the WMAL app, visit WMAL.com or tune in live on WMAL-FM 105.9 from 9:00am-12:00pm Monday-Friday  To join the conversation, check us out on Twitter @WMAL and @ChrisPlanteShow Learn more about your ad choices. Visit podcastchoices.com/adchoices

This week! We're kicking off the season of thirst with a film that Michael Harriet has called a documentary, "House Party" (1990)! Listen in as Ryan shares his love for the reality this film displays, Kayleigh shares the time she saw a possum in the wild streets of Richmond, and they both discuss the subtle science of Kool-Aid mixology. Interruption: "Whatcha Say" by Jason Derulo *Please enjoy this explicit content responsibly*Six Packs Count: 1 Abs, 0 Bevs

New Year New Me Week 3:"A Good Interruption" Pastor Sebastian BareSee omnystudio.com/listener for privacy information.

Episode 211: Understanding HFpEF.  Hyo Mun and Jordan Redden (medical students) explain the pathophysiology of heart failure with preserved ejection fraction (HFpEF) and how it differentiates from HFrEF. Dr. Arreaza asks insightful questions and summarizes some key elements of HFpEF. Written by Hyo Mun, MS4, American University of the Caribbean; and Jordan Redden, MS4, Ross University School of Medicine. Comments and edits by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice.What is EF? Just imagine, the heart is a pump, blood gets into the heart through the veins, the ventricles fill up and then squeeze the blood out. So, the percent of blood that is pumped out is the EF. Let's start at the beginning. What is HFpEF?Mike: HFpEF stands for heart failure with preserved ejection fraction. Basically, these patients squeeze normally—their ejection fraction is 50% or higher—but here's the thing: the heart can't relax and fill the way it should. The muscle gets stiff, almost like a thick leather boot that just won't stretch. And because the ventricle can't fill properly, pressure starts backing up into the lungs and the rest of the body. That's when patients start experiencing shortness of breath, leg swelling, fatigue—all those classic symptoms.Dr. Arreaza: And this is where people get fooled by the ejection fraction.Mike: Exactly. The ejectionfraction tells you total left ventricular emptying, not just forward flow.Jordan: The classic example is severe mitral regurgitation. You can eject 60% of your blood volume and still be in cardiogenic shock because most of that blood is leaking backward into the left atrium instead of going into the aorta. So, you get pulmonary edema, hypotension, fatigue, all with a “normal” EF. Which is honestly terrifying if you're over-relying on echo reports without thinking clinically.Dr. Arreaza: And in HFpEF, functional mitral regurgitation often shows up later in the disease. It's not usually the primary cause; it's more of a marker of advanced disease. Moderate to severe MR in HFpEF independently predicts worse outcomes, including a higher risk of mortality or heart failure hospitalization. So, let's contrast this with HFrEF. How are these two different?Mike: HFrEF—heart failure with reduced ejection fraction—is a pumping problem. The heart muscle is weak and can't contracteffectively. Ejection fraction drops below 40%, and this is your classic systolic dysfunction.Jordan: HFpEF, on the other hand, is diastolic dysfunction. The heart muscle is thick, fibrotic, and noncompliant. It squeezes fine, but it just doesn't relax, even though the EF looks reassuring on paper.Mike: I like to explain it this way: HFrEF is a weak heart that can't squeeze. HFpEF is a stiff heart that can't relax. Totally different problems.Dr. Arreaza: And then there's the gray zone: heart failure with mildly reduced EF, or HFmrEF. That's an EF between 41 and 49% with evidence of elevated filling pressures. It really shares the features of both worlds. So, what actually causes HFpEF versus HFrEF?Jordan: HFpEF is basically what happens when all the problems of modern living catch up with you. You've got chronic hypertension, obesity, diabetes, metabolic syndrome, aging, systemic inflammation—all of these things slowly remodel the heart over years. The muscle gets thick and stiff, and eventually the ventricle just loses its ability to relax. So, HFpEF is really a disease of metabolic dysfunction and chronic stress in the heart. Mike: HFrEF is more about direct injury. Think about myocardial infarctions, ischemic cardiomyopathy, viral myocarditis, alcohol toxicity, chemotherapy like doxorubicin, genetic cardiomyopathies, or chronic uncontrolled tachycardia. These insults actually damage or kill heart muscle cells, leading to a dilated, weak ventricle that can't pump effectively.Dr. Arreaza: So the short version: HFpEF is caused by chronic metabolic and hypertensive stress, while HFrEF is caused mainly by myocardial damage. A question we get a lot: does HFpEF eventually turn into HFrEF? What do you guys think?Mike: In most cases, no. HFpEF patients usually stay HFpEF throughout their disease course. They don't just “burn out” and turn into HFrEF.Jordan: They're generally separate disease entities with different pathophysiology. A patient with HFpEF can develop HFrEF if they have a big myocardial infarction or ongoing ischemia that damages the muscle, but that's not the natural progression.Mike: Interestingly though, the opposite can happen. Some HFrEF patients actually improve their ejection fraction with good medical therapy—that's called HF with improved EF—and it's a great sign that treatment is working.Dr. Arreaza: Another question. How do HFpEF and HFrEF compare to restrictive cardiomyopathy and constrictive pericarditis?Jordan: Clinically, they can all look very similar: dyspnea, edema, fatigue, but the underlying mechanisms are completely different.Mike: In HFpEF, the myocardium itself is stiff from hypertrophy and fibrosis. The problem is intrinsic to the heart muscle, and EF stays preserved. Echoshows diastolic dysfunction with elevated filling pressures.Jordan: In HFrEF, the myocardium is weak. The ventricle is often dilated and contracts poorly, with a reduced EF.Mike: Restrictive cardiomyopathy is different. Here, the myocardium gets infiltrated by abnormal stuff—amyloid, iron, sarcoid—and that makes it extremely stiff. It can look like HFpEF on the surface, but it's usually more severe. On Echo You'll see biatrial enlargement, small ventricles, and preserved EF. And importantly, it's a pathologic diagnosis, so you need advanced imaging or biopsy to confirm it.Jordan: Constrictive pericarditis is another mimic, but here the myocardium is usually normal. The problem is that the pericardium is thickened, calcified, and rigid. This will physically prevent the heart from being filled. Imaging shows pericardial thickening, septal bounce, and respiratory variation in flow, and cath shows equalization of diastolic pressures, which is the hallmark of constrictive pericarditis.Dr. Arreaza: So the takeaway is: HFpEF is a clinical syndrome driven by common metabolic and hypertensive causes, while restrictive and constrictive diseases are specific pathologic entities. If “HFpEF” is unusually severe or not responding to treatment, you need to think beyond HFpEF. Which type of heart failure is more common right now?Mike: Good question, the answer is: HFpEF. It now accounts for up to 60% of all heart failure cases, and it's still rising.Dr. Arreaza: Why is that?Jordan: Because people are living longer, gaining weight, and developing more metabolic syndrome. HFpEF thrives in older, or people with obesity, hypertension, or diabetes: basically, the modern American population. At the same time, better treatment of acute MIs means fewer people are developing HFrEF from massive heart attacks.Mike: HFpEF is the heart failure epidemic of the 21st century. It's honestly the cardiology equivalent of type 2 diabetes.Dr. Arreaza: Let's talk aboutCOVID-19. (2025 and still talking about it) Does it actually increase heart failure risk?Mike: Yes, absolutely. COVID increases both acute and long-term heart failure risk.Jordan: During acute infection, COVID can cause myocarditis, trigger massive inflammation, and precipitate acute decompensated heart failure, especially in patients with pre-existing disease. It also causes microthrombi, which can injure the myocardium.Mike: And after infection, even mild cases are linked to a significantly higher risk of developing new heart failure within the following year. Both HFpEF and HFrEF rates go up.Dr. Arreaza: I remember seeing this in 2021, we had a patient with acute COVID and HFrEF, her EF was about 10%, I lost contact with the patient and at the end I don't know what happened to her. What's the pathophysiology of COVID and heart failure?Mike: COVID causes direct viral injury through ACE2 receptors, triggers massive inflammation that damages the endothelium and heart muscle, leads to microvascular clotting and fibrosis—all mechanisms that promote HFpEF.Jordan: Add autonomic dysfunction, persistent low-grade inflammation, and worsening metabolic syndrome, and you've got a perfect storm for heart failure.Dr. Arreaza: Bottom line: COVID is a cardiovascular disease as much as a respiratory one. If someone had COVID and now has unexplained dyspnea or fatigue, think about heart failure. Get an echo, get a BNP, start treatment. Last big question: why did we have so many therapies for HFrEF but essentially none for HFpEF for years?Mike: HFrEF is mechanistically straightforward. You've got a weak heart with excessive neurohormonal activation going on — so you block RAAS, block the sympathetic system, drop the afterload. The drugs make sense.Jordan: HFpEF is messy. It's not one disease. It's stiffness, fibrosis, inflammation, microvascular dysfunction, metabolic disease, atrial fibrillation, all overlapping. One drug can't fix all of that.Mike: And some drugs that worked beautifully in HFrEF actually made HFpEF worse. Take Beta blockers, for example.  They slow heart rate, which is a problem because HFpEF patients rely on heart rate to maintain their cardiac output.Jordan: The breakthrough came with SGLT-2 inhibitors: diabetes drugs that unexpectedly addressed multiple HFpEF mechanisms at once: volume, metabolism, inflammation, and myocardial energetics.Dr. Arreaza: The miracle drug for HFpEF! Alright, let's wrap up.Mike: Bottom line: HFpEF is common, complex, and dangerous: even if the EF looks “normal.”Jordan: And if you're relying on ejection fraction alone, HFpEF will humble you every time.Dr. Arreaza: If you liked this episode, share it with a friend or a colleague and rate us wherever you listen. This is Dr. Arreaza, signing off.Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! _____________________References:Barzin A, Barnhouse KK, Kane SF. Heart Failure With Preserved Ejection Fraction. Am Fam Physician. 2025;112(4):435-440.Heidenreich PA, Bozkurt B, Aguilar D, et al. 2022 AHA/ACC/HFSA guideline for the management of heart failure. Circulation. 2022;145(18):e895-e1032.Kittleson MM, Panjrath GS, Amancherla K, et al. 2023 ACC expert consensus decision pathway on management of heart failure with preserved ejection fraction. J Am Coll Cardiol. 2023;81(18):1835-1878.Anker SD, Butler J, Filippatos G, et al. Empagliflozin in heart failure with a preserved ejection fraction. N Engl J Med. 2021;385(16):1451-1461.Solomon SD, McMurray JJV, Claggett B, et al. Dapagliflozin in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2022;387(12):1089-1098.Pitt B, Pfeffer MA, Assmann SF, et al. Spironolactone for heart failure with preserved ejection fraction. N Engl J Med. 2014;370(15):1383-1392.Yusuf S, Pfeffer MA, Swedberg K, et al. Effects of candesartan in patients with chronic heart failure and preserved left-ventricular ejection fraction. Lancet. 2003;362(9386):777-781.Solomon SD, McMurray JJV, Anand IS, et al. Angiotensin-neprilysin inhibition in heart failure with preserved ejection fraction. N Engl J Med. 2019;381(17):1609-1620.Kosiborod MN, Abildstrøm SZ, Borlaug BA, et al. Semaglutide in patients with heart failure with preserved ejection fraction and obesity. N Engl J Med. 2023;389(12):1069-1084.Xie Y, Xu E, Bowe B, Al-Aly Z. Long-term cardiovascular outcomes of COVID-19. Nat Med. 2022;28(3):583-590.Puntmann VO, Carerj ML, Wieters I, et al. Outcomes of cardiovascular magnetic resonance imaging in patients recently recovered from COVID-19. JAMA Cardiol. 2020;5(11):1265-1273.Basso C, Leone O, Rizzo S, et al. Pathological features of COVID-19-associated myocardial injury. Eur Heart J. 2020;41(39):3827-3835.Nalbandian A, Sehgal K, Gupta A, et al. Post-acute COVID-19 syndrome. Nat Med. 2021;27(4):601-615.Badve SV, Roberts MA, Hawley CM, et al. Effects of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers in adults with estimated GFR less than 60 mL/min per 1.73 m². Ann Intern Med. 2024;177(8):953-963.Navis G, Faber HJ, de Zeeuw D, de Jong PE. ACE inhibitors and the kidney: a risk-benefit assessment. Drug Saf. 1996;15(3):200-211.Textor SC, Novick AC, Tarazi RC, et al. Critical perfusion pressure for renal function in patients with bilateral atherosclerotic renal vascular disease. Ann Intern Med. 1985;102(3):308-314.Hackam DG, Spence JD, Garg AX, Textor SC. Role of renin-angiotensin system blockade in atherosclerotic renal artery stenosis and renovascular hypertension. Hypertension. 2007;50(6):998-1003.Ronco C, Haapio M, House AA, et al. Cardiorenal syndrome. J Am Coll Cardiol. 2008;52(19):1527-1539.Prins KW, Neill JM, Tyler JO, et al. Effects of beta-blocker withdrawal in acute decompensated heart failure. JACC Heart Fail. 2015;3(8):647-653.Jondeau G, Neuder Y, Eicher JC, et al. B-CONVINCED: Beta-blocker CONtinuation Vs. INterruption in patients with Congestive heart failure hospitalizED for a decompensation episode. Eur Heart J. 2009;30(18):2186-2192.Theme song, Works All The Time by Dominik Schwarzer, YouTube ID: CUBDNERZU8HXUHBS, purchased from https://www.premiumbeat.com/. 

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Is the selling in tech overblown? Investors are looking for some clarity and pivoting their attention to Alphabet which reports in Overtime. We run through what to expect with shareholder Doug Clinton from Intelligent Alpha and Oppenheimer analyst Jason Helfstein. Plus, former St. Louis Fed President Jim Bullard weighs in on what could be next for the Fed under Kevin Warsh. And, the big battle between Anthropic and OpenAI is headed to the Superbowl. We explain. Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Send us a textThinking about running paid ads in 2026? Pump the brakes.Frazier and Michael are breaking down the truth about paid advertising — what works, what doesn't, and where most loan officers go wrong. Whether you're sitting on a 30K marketing budget or barely spending $300 a month, this episode is a tactical deep dive into interruption vs. intent, social vs. search, and why Google still wins if you're serious about ROI.No fluff. No theory. Just straight talk on what actually drives deals — and what will drain your wallet.What You'll LearnWhy social media ads aren't dead — but most of them are dumbThe difference between passive brand amplification and transactional lead genWhy low-cost leads are usually garbage (and what actually matters)How intent changes everything — and why interruption-based ads have a short fuseWhat to realistically budget for Google or social ads in 2026Real Talk Quotes“You don't own the customer. You're just renting attention.”“Lead cost means nothing if the leads suck.”“You're never gonna get a CTC on your feelings.”“Intent-based leads wait for you. Interruption leads forget you.”“We can all get 50-cent leads. That's not the flex you think it is.”Tactical Takeaways✅ Expect to spend $1,500/month minimum if you want transactional ROI✅ Social media ads work best to amplify brand — not chase cold leads✅ Google Search ads are still the king of high-intent lead generation✅ YouTube & TikTok are the only social platforms with real search intent✅ If you're not following up with automation, you're burning money✅ Don't run your own ads unless you want to waste time and budgetWhy This Episode MattersMost LOs are playing checkers when it comes to paid ads — this episode helps you play chess. Whether you're doing consumer direct or trying to convert your social audience, you'll learn where to invest, what to expect, and how to actually get deals from your dollars.Want Help Running Ads That Work?This episode is powered by Empower LO, the trusted team behind hundreds of top-producing LOs running scalable Google ad campaigns.Learn more at empowerlo.com

Hackers aren't stealing passwords anymore. They're stealing what makes companies valuable.We cover: • The European Space Agency breach and why even elite organizations aren't safe • Pig butchering romance scams and how social engineering targets human psychology • The Nike data breach where attackers stole intellectual property, not customer data • AI voice cloning, deepfake scams, and real-world fraud attempts • Why modern cybercrime looks more like organized crime than traditional hacking.If you care about cybersecurity threats, ransomware groups, AI scams, social engineering attacks, or how hackers actually operate today, this episode will change how you think about risk.Cybercrime is evolving fast. Most defenses aren't.CHAPTERS00:00 – Hackers stopped stealing passwords and started stealing everything 01:50 – Welcome to Chaos + panel intros 03:40 – Meme culture, cold weather, and why Dino has too much free time 04:25 – European Space Agency hacked twice in one month 06:00 – If rocket scientists can't keep hackers out… 07:10 – Space nerds, Carl Sagan, and stardust detour 08:15 – Romance scams and pig butchering explained 09:20 – “Wrong number” texts and how the scam really starts 10:50 – How victims are slowly manipulated into crypto scams 12:25 – The 764 gang and psychological weaponization 13:40 – The dark twist: scammers as human trafficking victims 16:40 – Luxury lifestyles, urgency, and fake success 18:20 – Why victims don't report scams 23:30 – Fake job offers and too-good-to-be-true salaries 24:20 – Nike breach isn't what you think 25:45 – Why hackers skipped customer data entirely 27:10 – WorldLeaks, ransomware evolution, and pure extortion 28:35 – 1.4TB of stolen intellectual property 30:30 – Stealing what actually makes Nike… NikeQuestions? Text our Studio direct. We read these and when helpful we give a special shout out for those to contact us.Growth without Interruption. Get peace of mind. Stay Competitive-Get NetGain. Contact NetGain today at 844-777-6278 or reach out online at www.NETGAINIT.com Support the show

Come journey with me to the cosmos and see God

How are your “artful interruption” skills? This is a challenging yet essential skill for countering advice-giving patterns when facilitating coaching circles, peer learning groups, or team meetings. Inspired by a former student's question about redirecting well-intentioned advice-givers without disrupting group dynamics, Meg provides practical strategies for facilitators who want to cultivate curiosity over knowing in group settings. By introducing the “two muscle framework,” along with specific techniques, visual cues, and verbal redirects, Meg demonstrates how to protect the coaching space and help people discover their own solutions. Learn how you can help safeguard space for learning and discovery! Join us to learn more!Show Highlights:The challenge of incorporating polite, thoughtful interruptions into a coaching culture: Why is this an essential and highly relevant topic in today's coaching space?Understanding the two-muscle framework: the “knowing muscle” and the “curiosity muscle” (We short-circuit the creative process when we rush in with answers and advice)Advice-giving in groups is problematic in several specific ways. Setting the stage before the first session with non-negotiables:Pre-frame the purpose and agreements. Explain the “muscle” metaphor.Model what “good” looks like.Create psychological safety around interruption.Address the elephant in the room by naming what makes this challenging to do.The Facilitator's Toolkit, Six Specific Strategies for Interrupting Skillfully:The Appreciative Redirect (Acknowledge, redirect, and teach)The Pause and Pivot (Put your idea in the “parking lot” to give them time to think through and explore.)Visual Cues and Signals (These can be fun with card signals, the “muscle-flex” gesture, and a “parking lot” for ideas. Meg even has suggestions for using these with virtual meetings.)The Pattern Interrupt with Choice (Give agency while redirecting behavior.)Reflection Break (This is where the deepest learning happens.)The Question Upgrade (This is teaching in real-time.)Understanding when stepping in with advice and expertise is valuable (Use both/and thinking about coaching vs. advising.)A measuring stick question: “What creates more lasting impact, being told or discovering?”Practical tips for implementation:Start with shorter rounds.

It's our second time doing this, but we hit all the top stories in DC sports... This is Grant & Danny the Interruption.

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While we discuss the scholarship fund with Libby and Maureen from LESA, we get a little creative with incoming donations which only seem to interrupt when Ryan speaks.

durée : 00:11:55 - Les Nuits de France Culture - par : Albane Penaranda, Mathias Le Gargasson, Antoine Dhulster - Dans cet entretien de Max Frisch, au micro de Marie-Hélène Baconnet en janvier 1976, l'écrivain suisse de langue allemande évoque la présence de la révolte et le désir de révolution qui transparaissent dans ses pièces et romans. - réalisation : Virginie Mourthé - invités : Max Frisch

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Peter is newly famous. His first sermon went viral and he now helps lead a church of thousands. On his way to church, he runs into a guy who adds nothing to his growing resume. Instead of ignoring, he slows down to see and care for this forgotten man, finally embracing the servant-leadership mindset of Jesus. What happens next can only be described as a divine appointment. Let's explore how small interruptions can become divine interactions

Have you ever noticed how we keep telling ourselves cyber risk is a “future problem” right up until it walks in the front door wearing a name badge?Because in this episode, we're not talking about Hollywood hackers or hoodie clichés. We're talking about how attacks actually happen now. We interview global vCISO Dan Elliott from Recorded Future all about it.  Question? Text our Studio direct.Growth without Interruption. Get peace of mind. Stay Competitive-Get NetGain. Contact NetGain today at 844-777-6278 or reach out online at www.NETGAINIT.com Support the show

We're joined by Walter Eppich to talk about how learning happens in conversations. Specifically, Walter discusses how he watched a surprisingly successful call by a junior doctor that brought a surgeon running from the OR down to the ED to see their patient. Doctors in the early stages of their career tend to ramble when giving reports—including every piece of information that they know in the hopes of including something relevant. How do we learn to communicate with other healthcare professionals in a way that makes your current problem their most important problem? Why do we have every junior doctor go through phone call failure rather than explicitly teaching the structure of talk that we know works, and that they'll be steered towards by explicit feedback in the calls? Apple Podcasts: https://podcasts.apple.com/us/podcast/the-center-for-medical-simulation/id1279266822 Leadership Coaching from Jenny Rudolph: https://harvardmedsim.org/personal-leadership-coaching-with-jenny-rudolph/ #healthcaresimulation #nursing #medicine #debriefing

In this episode, we're joined by Madhav Bhandari, VP Marketing at Storylane, the demo automation platform helping B2B teams show their product before prospects ever talk to sales. Storylane is a profitable, fully remote SaaS with 50 employees, and a clear path toward $20M+. Madhav shares how Storylane broke out of the “race to average” in an insanely crowded martech and sales tech market, not by spending more, but by building a repeatable system for pattern interruption. We unpack how they used unconventional SEO, brand plays, and GTM experiments to grow brand searches from 1,500 to 12,000 per month, drive massive inbound, and turn marketing into a true growth engine. We spoke with Madhav about why great products aren't enough anymore, how to become the first vendor prospects discover, and how to build a marketing portfolio that consistently produces breakout ideas instead of chasing one-hit wonders. Here are some of the key questions we address: Why visibility, not budget, is the real advantage in crowded SaaS markets What pattern interruption actually means and how to systematize it How Storylane grew brand searches 8x and created an unfair inbound advantage Why traditional SEO and blog strategies are too slow, and what replaced them How to build a marketing portfolio instead of chasing viral one-offs Where pattern interruption should live and why leadership must own it

He didn't start playing golf until he was 37, and has the game ever impacted who Michael Wilbon of ESPN has met, where he has traveled, and how he lives his life.Join Michael on a delicious, story-filled journey that includes playing golf with a U.S. President, on-course trash talking with Tony Kornheiser, his co-host on "Pardon the Interruption," and watching his son blow by the old man off the tee.Please enjoy this terrific interview with ESPN's Michael Wilbon and discover how golf has truly changed his life.

Hi folks, due to unforseen circumstances we were unable to record our latest episode this weekend, so we'll only be recording agaon on the 1st of Feb and publishing the episode a week later.Apologies for the inconvenience but we'll be back with you soon!

This week! We watched our most heartfelt film to date, "Mask" (1985), starring Cher AND Sam Elliott! Listen in as Ryan shares how he viewed this film as a kid versus now, Kayleigh shares how she discovered Cher, and they both celebrate "Little Egypt."Interruption: "Naive" by The Kooks*Please enjoy this explicit content responsibly*Trigger warning: there is a brief mention of pedophilia in this episode 

Today I am delighted to talk with Roderick Ferguson about his provocative and much-needed intervention, “An Interruption in Our Cowardice.”  Initially driven by his deep disappointment in some Black intellectuals' compliance and even assistance with reactionary forces, this essay opens onto profound issues of institutionalization, professionalization, and the deadening and repressive mental, social, and intellectual habits being “accepted” create. In our conversation we spend some time talking about alternative, and very real counterexamples to cowardice, such as the fearless examples of the encampments of the Student Intifada. We note that such alternative sites have always been there historically, and that it is crucial to turn our eyes to those spaces, if we are going to preserve the promise of liberatory education.Roderick A. Ferguson is the William Robertson Coe Professor of Women's, Gender, and Sexuality Studies and American Studies at Yale University. He is also faculty in the Yale Prison Education Initiative as well as the Yale-New Haven Teachers Institute/Yale National Initiative. He is the author of One-Dimensional Queer (Polity, 2019), We Demand: The University and Student Protests (University of California, 2017), The Reorder of Things: The University and Its Pedagogies of Minority Difference (University of Minnesota, 2012), and Aberrations in Black: Toward a Queer of Color Critique(University of Minnesota, 2004). He is the co-editor with Grace Hong of the anthology Strange Affinities: The Gender and Sexual Politics of Comparative Racialization (Duke University, 2011). He is also co-editor with Erica Edwards and Jeffrey Ogbar of Keywords of African American Studies (NYU, 2018). He is the 2020 recipient of the Kessler Award from the Center for LGBTQ Studies (CLAGS). His book In View of the Tradition: Black Art and Radical Thought will be released Fall 2026. 

It's 2026! Time to get your laugh on. Host Clinton brings us Madison on the Air #51, Comedy4Cast: Just a Click Away, and Tek Diff #202- Interruption! Learn more about your ad choices. Visit megaphone.fm/adchoices

Question? Text our Studio direct.Growth without Interruption. Get peace of mind. Stay Competitive-Get NetGain. Contact NetGain today at 844-777-6278 or reach out online at www.NETGAINIT.com Support the show

Why modern cybercrime targets trust, urgency, and decision-making instead of systemsDeepfake Attacks, Voice Cloning, and Why AI Social Engineering WorksTraditional fraud used to feel obvious: misspellings, odd links, weird emails. Now? Deepfakes embed perfectly familiar voices and faces into your feed — or your inbox.Listen to Perry Carpenter on this. If you love this topic as much as we do grab Perry's incredible book FAIK available everywhere. Here's a non-affiliated link: https://www.barnesandnoble.com/w/faik-perry-carpenter/1145888787?ean=2940190971293Don't Miss the Deepfake Webinar coming up! You WIll See how you can test out your own deepfake to better understand them.  https://info.knowbe4.com/new-deepfake-training-na?partnerref=blogChapters 00:00 Why the Next Breach Won't Look Like the Last One01:44 Welcome to America in 202603:00 Why Deepfakes Are Exploding Right Now05:20 Yes — A Voice Can Be Cloned in Seconds07:30 What Deepfakes Actually Are (No Hype)09:35 Legitimate Uses vs Weaponized Intent12:45 Why Deepfake Companies Stay Quiet17:05 Faces and Voices Are the New Attack Surface18:30 Stop Asking “Is This Real?” Ask This Instead20:55 Why Spotting Artifacts No Longer Works23:45 The One Question That Cuts Through Deepfakes25:50 What Leaders Should Actually Do Today27:45 Old-School Security Still Wins30:05 Why Detection Tools Are Losing the Race33:10 Romance Scams, HR Fraud, and Deepfake Hiring35:45 10 of 15 Job Candidates Were Fake — Here's Why38:40 Fake Workers, Real Access, Real Damage42:20 Deepfakes as Multi-Stage AttackQuestion? Text our Studio direct.Growth without Interruption. Get peace of mind. Stay Competitive-Get NetGain. Contact NetGain today at 844-777-6278 or reach out online at www.NETGAINIT.com Support the show

The schedule beats me again, but I rise with a short dash of comedy and a look at my ambitious 2009 plans! Learn more about your ad choices. Visit megaphone.fm/adchoices

It's 2026! Time to get your laugh on. Host Clinton brings us Madison on the Air #51, Comedy4Cast: Just a Click Away, and Tek Diff #202- Interruption! Learn more about your ad choices. Visit megaphone.fm/adchoices

В данном подкасте мы обсудим что такое trip cancellation and trip interruption insurance. Чем они отличаются друг от друга и чем отличаются подобные страховки на кредитных карточках и если покупать напрямую у страховой/страхового агента. При каких условиях не действуют эти страховки. Michael Modelevsky Insurance and Financial Specialist. Tel., WhatsApp, Viber: (647) 284-7016 mljmod@rogers.com (далее…) ————————————————- The content of this site and our podcasts are for information only. Everybody's financial situation is different and the thoughts we provide here may not be applicable to you. We can't be held responsible for the consequences if you pursue an unsuitable course of action. Сообщение Что такое Trip Cancellation and Trip Interruption insurance | 317 появились сначала на Moneyinside.ca-самый популярный финансовый подкаст в Канаде!.

Sometimes the biggest breakthroughs come in the form of interruptions. Today, we revisit a powerful moment in Acts 3, when a man who had been paralyzed since birth asked for a few coins, but instead received a life-altering miracle through Jesus. He didn't expect anything more than a handout, but what he got was healing, freedom, and the ability to walk, leap, and praise God for the first time in his life. As we step into this new year, maybe what we need most isn't what we've been asking for, but what God is already preparing to give us. Don't miss the joy that can come from a holy interruption. It might be exactly what your soul needs today.Pursuing God with Gene Appel is designed to help you pursue God, build community, and unleash compassion. Grounded in Scripture and shaped by Eastside's conviction that God's grace is for everyone, each episode invites you to discover God's presence and activity in your life.

God often meets us when we least expect it. In Week 1 of our ENCOUNTER series, we explore Moses' burning bush moment and discover how a true encounter with God interrupts the ordinary, reveals His holiness, and invites us into His mission. Come ready to listen, respond, and say yes to what God may be calling you to do.

Title: God's Beautiful InterruptionSpeaker: Nate HoldridgeScripture: 1 Peter 2:9-10

What was supposed to be a wild night out turned into a story so uncomfortable, it still lives rent-free in this listener’s head. On Jubile’s Dirty Little Secret, a casual confession takes a jaw-dropping turn when an unexpected person walks in at the absolute worst possible moment — and makes an offer no one was prepared for. The juiciest, most outrageous confession podcast from The Jubal Show! It's the Jubal Show's Dirty Little Secret! Listeners spill their wildest, weirdest, and most scandalous secrets anonymously—no judgment, just pure entertainment. From shocking revelations to hilarious mishaps, you never know what you'll hear next! Hosted by Jubal Fresh and the team, every episode is packed with jaw-dropping confessions, witty reactions, and unexpected twists. Got a secret? Share it with us… we promise we won’t tell!➡︎ Get on The Jubal Show with your story - https://thejubalshow.com This is just a tiny piece of The Jubal Show. You can find every podcast we have, including the full show every weekday right here…➡︎ https://thejubalshow.com/podcasts The Jubal Show is everywhere, and also these places: Website ➡︎ https://thejubalshow.com Instagram ➡︎ https://instagram.com/thejubalshow X/Twitter ➡︎ https://twitter.com/thejubalshow Tiktok ➡︎ https://www.tiktok.com/@the.jubal.show Facebook ➡︎ https://facebook.com/thejubalshow YouTube ➡︎ https://www.youtube.com/@JubalFresh Support the show: https://the-jubal-show.beehiiv.com/subscribeSee omnystudio.com/listener for privacy information.

What was supposed to be a wild night out turned into a story so uncomfortable, it still lives rent-free in this listener’s head. On Jubile’s Dirty Little Secret, a casual confession takes a jaw-dropping turn when an unexpected person walks in at the absolute worst possible moment — and makes an offer no one was prepared for. The juiciest, most outrageous confession podcast from The Jubal Show! It's the Jubal Show's Dirty Little Secret! Listeners spill their wildest, weirdest, and most scandalous secrets anonymously—no judgment, just pure entertainment. From shocking revelations to hilarious mishaps, you never know what you'll hear next! Hosted by Jubal Fresh and the team, every episode is packed with jaw-dropping confessions, witty reactions, and unexpected twists. Got a secret? Share it with us… we promise we won’t tell!➡︎ Get on The Jubal Show with your story - https://thejubalshow.com This is just a tiny piece of The Jubal Show. You can find every podcast we have, including the full show every weekday right here…➡︎ https://thejubalshow.com/podcasts The Jubal Show is everywhere, and also these places: Website ➡︎ https://thejubalshow.com Instagram ➡︎ https://instagram.com/thejubalshow X/Twitter ➡︎ https://twitter.com/thejubalshow Tiktok ➡︎ https://www.tiktok.com/@the.jubal.show Facebook ➡︎ https://facebook.com/thejubalshow YouTube ➡︎ https://www.youtube.com/@JubalFresh Support the show: https://the-jubal-show.beehiiv.com/subscribeSee omnystudio.com/listener for privacy information.

Question? Text our Studio direct.New Episode

Another year gone! With 2025 rapidly bidding us adieu, Dennis, Tom, and their good pal Debbie Foster send us off with their annual Kennedy-Mighell edition of Pardon the Interruption. They argue different sides of popular tech topics in “Toss-Up”,  play legal tech Mad Libs in “What's the Word?”, and take the temp on 2025 trends in “Hot or Not?”. And, we know the question on everyone's minds—Can Tom keep up his yearslong winning streak? Listen in to find out.  As always, stay tuned for the parting shots, that one tip, website, or observation that you can use the second the podcast ends. Have a technology question for Dennis and Tom? Call their Tech Question Hotline at 720-441-6820 for the answers to your most burning tech questions. Learn more about your ad choices. Visit megaphone.fm/adchoices

Pardon the Interruption hosts, Tony Kornheiser and Michael Wilbon, react to the 49ers victory over the colts and more. Also, the hosts debate coaching decisions and highlight upcoming playoff contenders. Learn more about your ad choices. Visit podcastchoices.com/adchoices

Jesus' arrival is a picture of vulnerability and weakness, because that's where He finds us, and rescues us.