Podcasts about Imaging

Breast cancer screening is often treated as a given. Mammograms are framed as routine, early detection as unquestionably life-saving, and following guidelines as the responsible choice. But what if the full picture is more complicated?In this episode of hol+, Dr. Taz sits down with integrative oncologist and breast surgeon Dr. Jenn Simmons, author of The Smart Woman's Guide to Breast Cancer, to explore what breast cancer screening data actually shows, where common narratives may oversimplify reality, and how statistics can sometimes be misunderstood by both patients and providers.Together, they discuss the difference between screening and diagnostic imaging, why detecting more cancers does not always mean fewer deaths, and how concepts like overdiagnosis and lead-time bias shape our interpretation of outcomes. Dr. Jenn also explains how breast cancer behaves differently from many other cancers, why progression is not always linear, and what tumor markers like ER, PR, HER2, and triple-negative really indicate.The conversation expands beyond imaging into a whole-body view of breast health, touching on inflammation, immune function, metabolic health, toxic burden, stress, and lifestyle as factors that shape cancer risk and recovery. Rather than promoting fear or urgency, this episode focuses on helping listeners understand their bodies, ask better questions, and make informed decisions with clarity.This episode is for anyone who wants a deeper understanding of breast cancer screening, feels overwhelmed by conflicting guidance, or wants to approach breast health with more nuance and less panic.About Dr. Jenn SimmonsDr. Jenn Simmons is an integrative oncologist, breast surgeon, and founder of Real Health MD. She was Philadelphia's first fellowship-trained breast surgeon and spent nearly two decades leading one of the region's top breast programs before transitioning into integrative oncology.Drawing from her experience in conventional cancer care and her own health journey, Dr. Simmons now focuses on whole-body approaches to breast health, cancer prevention, and recovery, including metabolic health, inflammation, immune function, lifestyle medicine, and root-cause healing. She is the author of The Smart Woman's Guide to Breast Cancer, a patient-centered resource designed to help women better understand their diagnosis, ask informed questions, and navigate treatment decisions with clarity rather than fear.Order the BookThe Smart Woman's Guide to Breast Cancer Stay Connected:Connect further to Hol+ at https://holplus.co/- Don't forget to like, subscribe, and hit the notification bell to stay updated on future episodes of hol+.Follow Dr. Jenn SimmonsInstagram: https://www.instagram.com/drjennsimmonsYouTube: https://www.youtube.com/@dr.jennsimmonsWebsite: https://www.jennsimmonsmd.com/Follow Dr. Taz on Instagram: https://www.instagram.com/drtazmd/https://www.instagram.com/liveholplus/Subscribe to the audio podcast: https://holplus.transistor.fm/subscribeSubscribe to the video podcast: https://www.youtube.com/@DrTazMD/podcastsGet your copy of The Hormone Shift: Balance Your Body and Thrive Through Midlife and MenopauseHost & Production TeamHost: Dr. Taz; Produced by ClipGrowth.com (Producer: Pat Gostek)00:00 Swedish trial claim and overdiagnosis framing 00:52 Why this became a part two conversation 03:25 Reframing screening narratives and medical training gaps 04:39 Why Dr. Simmons questions mammograms as a screening tool 06:06 Origins of screening programs and “invitation to screen” bias 07:12 Relative risk vs absolute numbers example (4 vs 5 per 1,000) 08:49 Overdiagnosis explained with a vivid analogy 09:50 Autopsy-study claim and the “microscopic cancer” idea 12:11 Swedish trial claim revisited: more diagnoses, same deaths 13:38 Downstream harms: callbacks, biopsies, overtreatment 15:04 Lead-time bias and survival statistics explained 16:44 Dr. Simmons' view on the founder's regret narrative 18:16 Switzerland headline clarified and what actually changed 20:10 Cautionary stories and aggressive cancers discussion 22:07 Why breast cancer does not always progress linearly 24:21 Buckets: DCIS, invasive, inflammatory, receptor types 26:15 Clinical vs subclinical disease approach 28:25 Long-term tradeoffs and “forgotten woman” after treatment 32:15 What ER PR HER2 mean biologically and system incentives 35:33 Testosterone discussion and prevention claim presented 42:15 Hormones after breast cancer and the 4-year “reintroduction” idea 44:29 Triple negative: environment, toxicity, immune system focus 49:19 What to do next: pause, exceptions, whole-body workup 52:32 Prevention and breast health approach begins 53:24 At-home tears test explanation (as discussed) 56:24 Detox basics and why sweating is emphasized 59:34 Imaging preferences for screening and what to do if limited access

Heart disease risk isn't just about cholesterol. In this episode of A Whole New Level, Dr. Matthew Budoff explains why coronary artery calcium (CAC) scoring may be the most important test most people aren't getting—and why imaging your arteries directly can reveal risk that blood tests alone can miss.Drawing on decades of research and data from the landmark MESA study, Dr. Budoff explains how calcium scoring predicts real cardiovascular events, how plaque actually forms and progresses, and why some people with high cholesterol never develop plaque—while others with “normal” labs do.This episode focuses on how to measure your actual cardiovascular risk, not just estimate it.Sign Up to Get Your Free Ultimate Guide to Glucose: ⁠https://levels.link/wnlIn this episode, we cover:Why CAC scoring is one of the strongest predictors of future heart eventsWhy cholesterol is critical—but only explains about half of heart disease riskWhy some people with very high LDL have zero plaque—and others with normal labs have dangerous plaqueWhy CAC is best understood as the “tip of the iceberg” of total plaque burdenWhen to escalate to CT angiography and advanced imagingHow plaque regression is possible—and what interventions actually drive itThe future of cardiac risk prediction: Lp(a), inflammation, and AI-driven plaque analysisThis conversation reframes heart risk around what's actually happening inside your arteries—not just what shows up in bloodwork.

Hip Replacement surgeries are growing at a rate of 10% per year. But Dr. Prather says that proper Structure-Function Care can help people avoid Hip Surgery, delay surgery for years, and even help those who have already had surgery. In this episode, you'll learn:—How Osteoarthritis is the #1 cause of Hip Replacements. And 40 million Americans have Osteoarthritis, including 80% of those over the age of 50. —How NonSteroidal Anti-Inflammatory Drugs (NSAIDs) can temporarily help the pain, but will make the problem worse by dissolving the cartilage in the joint. —How Hip Surgeries have "dramatically" improved over the years with increased patient satisfaction rates. But, you will still be considered permanently disabled after a Hip Replacement.—The natural products that work better than drugs to help with joint pain, including Glucosamine and Chondroitin.—The Vitamins, Minerals, and Herbals that are beneficial for joint health.—Why a combination of Chiropractic and Physical Therapy makes things go so much better for a patient. —The difference that treatments like Orthotics, Acupuncture, Dry Needling, Decompression, Ultrasound, Electrical Stimulation, and Diathermy can make for patients with Hip problems.—Why Dr. Prather calls the Rapid Release treatment "absolutely wonderful for Trigger Points" and has "been like a miracle" for patients. And the Chinese Liniments that are "highly recommended" to relieve pain.—What an exam for Hip Problems should include. And why good Imaging is important to figure out exactly what is going on, so the right treatment plan can be made. —The surprising connection between gut issues and hip degeneration.http://www.TheVoiceOfHealthRadio.com*Receive exclusive bonus content as a member of our Voice Of Health Patreon Community:https://www.patreon.com/cw/VoiceofHealthPodcast

When does a metatarsal stress reaction actually show up on imaging? In this episode of the Doc On The Run Podcast, Dr. Christopher Segler explains the difference between a stress response, stress reaction, and true stress fracture—and why timing matters when choosing X-rays, MRI, ultrasound, or CT scans. Learn how early imaging can help you make smarter race decisions, avoid false reassurance from a “normal” X-ray, and protect your fitness without turning a minor stress reaction into a full fracture.

Show Notes 2/13/2026AI model from Google's DeepMind reads recipe for life in DNA Source: BBC Link: https://www.bbc.com/news/articles/c39428dv18yoCarbon Robotics Launches the World's First-Ever Large Plant ModelSource: BusinessWire.com Link: https://www.businesswire.com/news/home/20260202630325/en/Carbon-Robotics-Launches-the-Worlds-First-Ever-Large-Plant-ModelYour future home might be framed with printed plasticSource: MIT News Link: https://news.mit.edu/2026/your-future-home-might-be-framed-with-printed-plastic-0203A new scan lets scientists see inside the human body in 3D color Source: ScienceDaily.comLink: https://www.sciencedaily.com/releases/2026/02/260204121550.htm3D-printed passive cooling system cools data centers without fans or pumps Source: Interesting EngineeringLink: https://interestingengineering.com/ai-robotics/3d-printed-passive-cooling-data-centersHow we're helping preserve the genetic information of endangered species with AI Source: Google's The Keyword BlogLink: https://blog.google/innovation-and-ai/technology/ai/ai-to-preserve-endangered-species/The Navy's Batwing Fighter Jet Promises Mach 4 Speed… But It's Still Just a ConceptSource: YD Design Link: https://www.yankodesign.com/2026/02/06/the-navys-batwing-fighter-jet-promises-mach-4-speed-but-its-still-just-a-concept/New study of chemical reactions in space 'could impact the [theories of the] origin of life in ways we hadn't thought of'Source: LiveScience.com Link: https://www.livescience.com/chemistry/complex-building-blocks-of-life-can-form-on-space-dust-offering-new-clues-to-the-origins-of-life

In the final episode, Vidur Mahajan looks ahead to the future of radiology. From AI-enabled education to convergence with genomics and pathology, this discussion explores how imaging and AI will reshape diagnostics over the next decade.   Timestamps:  00:00 – Introduction  00:51 – Exciting innovations  03:47 – Role of radiologists  05:59 – AI and education  08:40 – AI and genomics  10:55 – What's next? 

In this episode, we sit with Dr. Tarik Alkasab, Heather Chase and Dr. C. Michael Hood to talk about recent developments in the worlds of Common Data Elements (CDS, see https://www.rsna.org/practice-tools/data-tools-and-standards/radelement-common-data-elements), Open Imaging Data Model (OIDM, see https://www.openimagingdata.org/) and other developments such as the Imaging Problem List (IPL).

  Snoreo... 10 years ago, back at the place we called our

In this episode of the Heart podcast, Digital Media Editor, Professor James Rudd, is joined by Professor Kazuaki Negishi from Sydney, Australia. They discuss the optimal use of imaging to detect cardiac effects of cancer therapies. If you enjoy the show, please leave us a positive review wherever you get your podcasts. It helps us to reach more people - thanks! Link to published paper: https://heart.bmj.com/content/111/22/1057

Imaging With Intention: Optimizing Care Through Collaboration in Diagnostic Radiology Link for CME Credit: https://cmetracker.net/UTHSCSA/Publisher?page=pubOpen#/getCertificate/10101474 Host and Executive Producer Holly Wayment and UT Health San Antonio's Department of Pediatrics bring us this  Grand Rounds episode with Desi Schiess, MD,  pediatric radiologist. This episode reviews evidence-based imaging choices, radiation considerations, and practical tips for ordering X‑ray, ultrasound, CT, MRI, fluoroscopy, and nuclear medicine in children. It includes case examples, a quiz, and guidance on when to consult a radiologist to ensure safe, effective pediatric imaging.

CMS permanently authorized virtual direct supervision for contrast-enhanced imaging starting January 2026. But state laws, ACR standards, and technologist training create a complex compliance environment. What does your facility need to know right now? ContrastConnect City: Las Vegas Address: Las vegas Website: https://www.contrast-connect.com/

In this episode of The Heart of Innovation, cohosts Kym McNicholas and Dr. John Phillips feature multiple patients courageously sharing their lived experiences with Peripheral Artery Disease, including rare and often misunderstood cases that challenge what patients are told is "possible." Pamela's story highlights a critical gap in PAD care. After being told by a large hospital system that amputation was inevitable, she reached out to the Global PAD Association's Leg Saver Hotline. Through patient advocacy and care coordination, the underlying contributors to her disease were identified, including the role climate played in worsening her symptoms. Her decision to relocate to a warmer environment helped stabilize her condition and avoid limb loss, proving that earlier intervention and individualized care matter. Francine's journey offers another rare perspective. Diagnosed at just 48 despite being a runner, personal trainer, and fitness instructor, she was found to have a full occlusion in her left leg and intermittent blockages in her right. After undergoing a femoral-popliteal bypass, multiple stents, and angioplasty procedures, Francine transformed her lifestyle through the Dean Ornish program and a low-fat vegan diet. She has since completed four half marathons and continues teaching group fitness, yoga, and Reiki. Theresa's story exposes how often PAD is dismissed, even when the warning signs are clear. With a family history of PAD, Theresa recognized the symptoms early. At 46, she sought help after developing walking pain, only to be told repeatedly that it was a back problem and that she was "too young" to have PAD. A Doppler study was performed but interpreted as normal. For five years, as her symptoms worsened and her walking distance shrank to less than ten metres, she continued to advocate for herself before finally insisting on a vascular referral. Within minutes of meeting a vascular consultant, Theresa was diagnosed with severe PAD. Imaging revealed a 100 percent blockage in her right leg and 80 percent in her left. Angioplasty provided temporary relief, but restenosis occurred quickly. A second procedure resulted in arterial injury, requiring placement of a 30-centimetre stent in her right thigh. Despite ongoing pain, Theresa developed remarkable collateral circulation, so robust that it complicated intervention attempts. Today, she remains closely monitored by a responsive vascular specialist and manages her condition with vigilance, pacing, and rest. She also notes a meaningful improvement in walking pain after starting Wegovy, an observation she continues to discuss with her care team. Equally important, Theresa speaks candidly about the emotional toll of PAD. A special education teacher who loves to travel, she feared the disease would take away the life she loved, as it had for her father. After a period of isolation, she made a conscious decision that PAD would be part of her story, but not the author of it. She now works full time, travels when she can, adapts when needed, and lives by a powerful truth: she controls PAD, not the other way around. Together, these stories reinforce a message too many patients never hear in time: Leg pain, cramping, and difficulty walking are not normal aging. They are warnings.

Episode 211: Understanding HFpEF.  Hyo Mun and Jordan Redden (medical students) explain the pathophysiology of heart failure with preserved ejection fraction (HFpEF) and how it differentiates from HFrEF. Dr. Arreaza asks insightful questions and summarizes some key elements of HFpEF. Written by Hyo Mun, MS4, American University of the Caribbean; and Jordan Redden, MS4, Ross University School of Medicine. Comments and edits by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice.What is EF? Just imagine, the heart is a pump, blood gets into the heart through the veins, the ventricles fill up and then squeeze the blood out. So, the percent of blood that is pumped out is the EF. Let's start at the beginning. What is HFpEF?Mike: HFpEF stands for heart failure with preserved ejection fraction. Basically, these patients squeeze normally—their ejection fraction is 50% or higher—but here's the thing: the heart can't relax and fill the way it should. The muscle gets stiff, almost like a thick leather boot that just won't stretch. And because the ventricle can't fill properly, pressure starts backing up into the lungs and the rest of the body. That's when patients start experiencing shortness of breath, leg swelling, fatigue—all those classic symptoms.Dr. Arreaza: And this is where people get fooled by the ejection fraction.Mike: Exactly. The ejectionfraction tells you total left ventricular emptying, not just forward flow.Jordan: The classic example is severe mitral regurgitation. You can eject 60% of your blood volume and still be in cardiogenic shock because most of that blood is leaking backward into the left atrium instead of going into the aorta. So, you get pulmonary edema, hypotension, fatigue, all with a “normal” EF. Which is honestly terrifying if you're over-relying on echo reports without thinking clinically.Dr. Arreaza: And in HFpEF, functional mitral regurgitation often shows up later in the disease. It's not usually the primary cause; it's more of a marker of advanced disease. Moderate to severe MR in HFpEF independently predicts worse outcomes, including a higher risk of mortality or heart failure hospitalization. So, let's contrast this with HFrEF. How are these two different?Mike: HFrEF—heart failure with reduced ejection fraction—is a pumping problem. The heart muscle is weak and can't contracteffectively. Ejection fraction drops below 40%, and this is your classic systolic dysfunction.Jordan: HFpEF, on the other hand, is diastolic dysfunction. The heart muscle is thick, fibrotic, and noncompliant. It squeezes fine, but it just doesn't relax, even though the EF looks reassuring on paper.Mike: I like to explain it this way: HFrEF is a weak heart that can't squeeze. HFpEF is a stiff heart that can't relax. Totally different problems.Dr. Arreaza: And then there's the gray zone: heart failure with mildly reduced EF, or HFmrEF. That's an EF between 41 and 49% with evidence of elevated filling pressures. It really shares the features of both worlds. So, what actually causes HFpEF versus HFrEF?Jordan: HFpEF is basically what happens when all the problems of modern living catch up with you. You've got chronic hypertension, obesity, diabetes, metabolic syndrome, aging, systemic inflammation—all of these things slowly remodel the heart over years. The muscle gets thick and stiff, and eventually the ventricle just loses its ability to relax. So, HFpEF is really a disease of metabolic dysfunction and chronic stress in the heart. Mike: HFrEF is more about direct injury. Think about myocardial infarctions, ischemic cardiomyopathy, viral myocarditis, alcohol toxicity, chemotherapy like doxorubicin, genetic cardiomyopathies, or chronic uncontrolled tachycardia. These insults actually damage or kill heart muscle cells, leading to a dilated, weak ventricle that can't pump effectively.Dr. Arreaza: So the short version: HFpEF is caused by chronic metabolic and hypertensive stress, while HFrEF is caused mainly by myocardial damage. A question we get a lot: does HFpEF eventually turn into HFrEF? What do you guys think?Mike: In most cases, no. HFpEF patients usually stay HFpEF throughout their disease course. They don't just “burn out” and turn into HFrEF.Jordan: They're generally separate disease entities with different pathophysiology. A patient with HFpEF can develop HFrEF if they have a big myocardial infarction or ongoing ischemia that damages the muscle, but that's not the natural progression.Mike: Interestingly though, the opposite can happen. Some HFrEF patients actually improve their ejection fraction with good medical therapy—that's called HF with improved EF—and it's a great sign that treatment is working.Dr. Arreaza: Another question. How do HFpEF and HFrEF compare to restrictive cardiomyopathy and constrictive pericarditis?Jordan: Clinically, they can all look very similar: dyspnea, edema, fatigue, but the underlying mechanisms are completely different.Mike: In HFpEF, the myocardium itself is stiff from hypertrophy and fibrosis. The problem is intrinsic to the heart muscle, and EF stays preserved. Echoshows diastolic dysfunction with elevated filling pressures.Jordan: In HFrEF, the myocardium is weak. The ventricle is often dilated and contracts poorly, with a reduced EF.Mike: Restrictive cardiomyopathy is different. Here, the myocardium gets infiltrated by abnormal stuff—amyloid, iron, sarcoid—and that makes it extremely stiff. It can look like HFpEF on the surface, but it's usually more severe. On Echo You'll see biatrial enlargement, small ventricles, and preserved EF. And importantly, it's a pathologic diagnosis, so you need advanced imaging or biopsy to confirm it.Jordan: Constrictive pericarditis is another mimic, but here the myocardium is usually normal. The problem is that the pericardium is thickened, calcified, and rigid. This will physically prevent the heart from being filled. Imaging shows pericardial thickening, septal bounce, and respiratory variation in flow, and cath shows equalization of diastolic pressures, which is the hallmark of constrictive pericarditis.Dr. Arreaza: So the takeaway is: HFpEF is a clinical syndrome driven by common metabolic and hypertensive causes, while restrictive and constrictive diseases are specific pathologic entities. If “HFpEF” is unusually severe or not responding to treatment, you need to think beyond HFpEF. Which type of heart failure is more common right now?Mike: Good question, the answer is: HFpEF. It now accounts for up to 60% of all heart failure cases, and it's still rising.Dr. Arreaza: Why is that?Jordan: Because people are living longer, gaining weight, and developing more metabolic syndrome. HFpEF thrives in older, or people with obesity, hypertension, or diabetes: basically, the modern American population. At the same time, better treatment of acute MIs means fewer people are developing HFrEF from massive heart attacks.Mike: HFpEF is the heart failure epidemic of the 21st century. It's honestly the cardiology equivalent of type 2 diabetes.Dr. Arreaza: Let's talk aboutCOVID-19. (2025 and still talking about it) Does it actually increase heart failure risk?Mike: Yes, absolutely. COVID increases both acute and long-term heart failure risk.Jordan: During acute infection, COVID can cause myocarditis, trigger massive inflammation, and precipitate acute decompensated heart failure, especially in patients with pre-existing disease. It also causes microthrombi, which can injure the myocardium.Mike: And after infection, even mild cases are linked to a significantly higher risk of developing new heart failure within the following year. Both HFpEF and HFrEF rates go up.Dr. Arreaza: I remember seeing this in 2021, we had a patient with acute COVID and HFrEF, her EF was about 10%, I lost contact with the patient and at the end I don't know what happened to her. What's the pathophysiology of COVID and heart failure?Mike: COVID causes direct viral injury through ACE2 receptors, triggers massive inflammation that damages the endothelium and heart muscle, leads to microvascular clotting and fibrosis—all mechanisms that promote HFpEF.Jordan: Add autonomic dysfunction, persistent low-grade inflammation, and worsening metabolic syndrome, and you've got a perfect storm for heart failure.Dr. Arreaza: Bottom line: COVID is a cardiovascular disease as much as a respiratory one. If someone had COVID and now has unexplained dyspnea or fatigue, think about heart failure. Get an echo, get a BNP, start treatment. Last big question: why did we have so many therapies for HFrEF but essentially none for HFpEF for years?Mike: HFrEF is mechanistically straightforward. You've got a weak heart with excessive neurohormonal activation going on — so you block RAAS, block the sympathetic system, drop the afterload. The drugs make sense.Jordan: HFpEF is messy. It's not one disease. It's stiffness, fibrosis, inflammation, microvascular dysfunction, metabolic disease, atrial fibrillation, all overlapping. One drug can't fix all of that.Mike: And some drugs that worked beautifully in HFrEF actually made HFpEF worse. Take Beta blockers, for example.  They slow heart rate, which is a problem because HFpEF patients rely on heart rate to maintain their cardiac output.Jordan: The breakthrough came with SGLT-2 inhibitors: diabetes drugs that unexpectedly addressed multiple HFpEF mechanisms at once: volume, metabolism, inflammation, and myocardial energetics.Dr. Arreaza: The miracle drug for HFpEF! Alright, let's wrap up.Mike: Bottom line: HFpEF is common, complex, and dangerous: even if the EF looks “normal.”Jordan: And if you're relying on ejection fraction alone, HFpEF will humble you every time.Dr. Arreaza: If you liked this episode, share it with a friend or a colleague and rate us wherever you listen. This is Dr. Arreaza, signing off.Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! _____________________References:Barzin A, Barnhouse KK, Kane SF. Heart Failure With Preserved Ejection Fraction. Am Fam Physician. 2025;112(4):435-440.Heidenreich PA, Bozkurt B, Aguilar D, et al. 2022 AHA/ACC/HFSA guideline for the management of heart failure. Circulation. 2022;145(18):e895-e1032.Kittleson MM, Panjrath GS, Amancherla K, et al. 2023 ACC expert consensus decision pathway on management of heart failure with preserved ejection fraction. J Am Coll Cardiol. 2023;81(18):1835-1878.Anker SD, Butler J, Filippatos G, et al. Empagliflozin in heart failure with a preserved ejection fraction. N Engl J Med. 2021;385(16):1451-1461.Solomon SD, McMurray JJV, Claggett B, et al. Dapagliflozin in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2022;387(12):1089-1098.Pitt B, Pfeffer MA, Assmann SF, et al. Spironolactone for heart failure with preserved ejection fraction. N Engl J Med. 2014;370(15):1383-1392.Yusuf S, Pfeffer MA, Swedberg K, et al. Effects of candesartan in patients with chronic heart failure and preserved left-ventricular ejection fraction. Lancet. 2003;362(9386):777-781.Solomon SD, McMurray JJV, Anand IS, et al. Angiotensin-neprilysin inhibition in heart failure with preserved ejection fraction. N Engl J Med. 2019;381(17):1609-1620.Kosiborod MN, Abildstrøm SZ, Borlaug BA, et al. Semaglutide in patients with heart failure with preserved ejection fraction and obesity. N Engl J Med. 2023;389(12):1069-1084.Xie Y, Xu E, Bowe B, Al-Aly Z. Long-term cardiovascular outcomes of COVID-19. Nat Med. 2022;28(3):583-590.Puntmann VO, Carerj ML, Wieters I, et al. Outcomes of cardiovascular magnetic resonance imaging in patients recently recovered from COVID-19. JAMA Cardiol. 2020;5(11):1265-1273.Basso C, Leone O, Rizzo S, et al. Pathological features of COVID-19-associated myocardial injury. Eur Heart J. 2020;41(39):3827-3835.Nalbandian A, Sehgal K, Gupta A, et al. Post-acute COVID-19 syndrome. Nat Med. 2021;27(4):601-615.Badve SV, Roberts MA, Hawley CM, et al. Effects of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers in adults with estimated GFR less than 60 mL/min per 1.73 m². Ann Intern Med. 2024;177(8):953-963.Navis G, Faber HJ, de Zeeuw D, de Jong PE. ACE inhibitors and the kidney: a risk-benefit assessment. Drug Saf. 1996;15(3):200-211.Textor SC, Novick AC, Tarazi RC, et al. Critical perfusion pressure for renal function in patients with bilateral atherosclerotic renal vascular disease. Ann Intern Med. 1985;102(3):308-314.Hackam DG, Spence JD, Garg AX, Textor SC. Role of renin-angiotensin system blockade in atherosclerotic renal artery stenosis and renovascular hypertension. Hypertension. 2007;50(6):998-1003.Ronco C, Haapio M, House AA, et al. Cardiorenal syndrome. J Am Coll Cardiol. 2008;52(19):1527-1539.Prins KW, Neill JM, Tyler JO, et al. Effects of beta-blocker withdrawal in acute decompensated heart failure. JACC Heart Fail. 2015;3(8):647-653.Jondeau G, Neuder Y, Eicher JC, et al. B-CONVINCED: Beta-blocker CONtinuation Vs. INterruption in patients with Congestive heart failure hospitalizED for a decompensation episode. Eur Heart J. 2009;30(18):2186-2192.Theme song, Works All The Time by Dominik Schwarzer, YouTube ID: CUBDNERZU8HXUHBS, purchased from https://www.premiumbeat.com/. 

In this episode, we welcome Skip Lievsay, CAS. Skip is an Oscar-winning Sound Editor and Re-Recording Mixer who has worked on films including Blood Simple, The Color of Money, Raising Arizona, Goodfellas, The Silence of the Lambs, Cape Fear, Malcolm X, Casino, Fargo, The Big Lebowski, No Country for Old Men, I Am Legend, Gravity, The Hunger Games, Birdman, Roma, Uncut Gems, A Quiet Place: Day One, and Marty Supreme. In our chat, he shares on his early days, about working with the Coen Brothers, and his creative approach to crafting sound in post. In March, Skip is being honored with the Cinema Audio Society Career Achievement Award in Los Angeles, CA. The Making Of is presented by AJA:From cinema to proAV: gaining a competitive edge with streaming knowledgeThe worlds of cinema production and proAV are converging. Cinema-grade equipment is making its way into more stadiums, houses of worship, and concert venues. Because of this, professionals that understand the tools and disciplines powering both will stand out. Get ahead of the curve with the latest streaming insights and gear from AJA.Sony's Breakthrough Audio, Imaging and Sports Technologies Raise the Bar for the NFL's Super Bowl LX in San FranciscoSony's New Coach's Headsets, 175+ Cameras and Hawk-Eye Tracking and Video Technologies Capture Every Angle, Split‑Second Decision and High‑Speed Moment of the NFL's Biggest GameSony, an official technology partner of the NFL, announced today that its technology ecosystem will play an expanded role in Super Bowl LX, marking the company's most advanced presence at the NFL's championship game to date.Read more hereNow with Massive 8TB Capacity—Thunderbolt 5 SpeedThe OWC Envoy Pro Ultra now comes in a new 8TB capacity, pairing enormous space with next‑generation Thunderbolt 5 performance. With real‑world speeds over 6000 MB/s and a rugged, bus‑powered design, it's perfect for 4K/8K workflows, on‑location shoots, and fast media offloads. High‑speed, high‑capacity, and ready for serious creative work.Browse hereDonate to Help The Digital Cinema Society SurviveDCS, a 501(c)(3) nonprofit dedicated to keeping filmmakers current on motion picture technology is currently in dire financial straights. We are trying to make the best of a tough situation and asking those who can afford it to help keep us afloat.Please consider donating to DCS hereMeet LiveU Solo PRO:Deliver an outstanding live video experience with the LiveU Solo PRO. Designed for creators and professionals alike, the Solo PRO video encoder provides one-touch, wireless live streaming directly from your camera to popular online platforms and any web destination. Stream with confidence using bonded 4G/5G connectivity and enjoy superior reliability and image quality up to 4K resolution. To learn more, contact Videoguys at 800-323-2325.Learn more herePodcast Rewind:Feb. 2026 - Ep. 116.Feature your products or services in this newsletter and reach 250K+ film and TV industry professionals. To learn more, please email mvalinsky@me.com Get full access to The Making Of at themakingof.substack.com/subscribe

Today, I am thrilled to connect with nurse practitioner, Dr. Lisa Chism. Lisa is the Clinical Director of the Oakland Macomb Center for Breast Health in Michigan. She has over 25 years of experience, specializing in breast health, menopause, sexual health, and breast cancer survivorship. She is also an author and a faculty member at a local university. In our conversation, we discuss the breast cancer risk for women with dense breasts, family history, or prior biopsy, diving into supplemental imaging, lifestyle modifications, HRT, and breast risk, and the changes that occur after a breast cancer diagnosis. We also explore the genitourinary syndrome of menopause and screening, permanent versus non-permanent changes occurring in the genital urinary area, and anticipatory informed care guidance for patients with a history of trauma. This is one of those conversations you will definitely want to revisit. With Lisa's thoughtful advocacy, deep commitment to patient care, and powerful insights, it is clear why sharing her message is so critically important. IN THIS EPISODE, YOU WILL LEARN: The different levels of breast density and their implications for cancer detection Various risk assessment tools used to determine breast cancer risk  How alcohol impacts the risk of breast cancer  Why weight management essential for post-menopausal women The importance of having detailed conversations with providers about menopause symptoms and the available treatment options Lisa shares her approach to evaluating and educating patients  How trauma impacts women's sexual health Permanent and non-permanent changes that occur in the vaginal area during menopause Can older women still do HRT? A simple breast-examination habit for all women Connect with Cynthia Thurlow   Follow on X, Instagram & LinkedIn Check out Cynthia's website Submit your questions to support@cynthiathurlow.com Join other like-minded women in a supportive, nurturing community: The Midlife Pause/Cynthia Thurlow  Cynthia's Menopause Gut Book is on presale now! Cynthia's Intermittent Fasting Transformation Book The Midlife Pause Supplement Line Connect with Dr. Lisa Chism Instagram (@DrMommyPoppins) Instagram (@TheAdoptedNurse)  Oakland Macomb OBGYN Center for Breast Health-Rochester Hills

In this episode of Focal Point, Sam, Hannah and Amy from the IMV team discuss AI and its impact on the veterinary industry with Dr. Adele Williams-Xavier.Adele is a specialist in equine internal medicine and an AI and clinical data expert within the veterinary industry. She has been overseeing clinical AI tool creation and getting data to sufficient quality for AI builds to produce high quality AI tools for the past 7 years. Adele runs her own AI and clinical data consultancy business, Ai-WX, where she advises veterinary staff, veterinary business and veterinary educators on AI literacy, AI implementation, ethical and responsible use of AI, as well as advising tech startups on AI tool product improvements and how to get the most value from clinical data. She works part-time with CoVet, as a veterinary AI expert, helping to improve the AI scribe vet co-pilot product, and is a board advisor for PetScribers and Prim Veterinary. Adele advocates for ethical and responsible AI adoption within the veterinary industry, has a column on AI in the Veterinary Record, and is a recognised international speaker on AI in veterinary medicine. If you are curious about AI and want practical advice on how to consider the adoption of AI tool, then listen in as we discuss where AI is used, the risks, how to approach AI tools for veterinary medicine, and the future. Got a question or suggestion? We would love to hear from you, and you can contact us using learning@imv-imaging.com. You can also check out our other learning resources on our Website, and our CPD courses via our Academy education platform. If you want more information on our in practice training, you can find it here.

Imagine being able to see the invisible—amyloid plaques, tau tangles, and metabolic changes in the living brain. This is no longer science fiction; it's reality at the University of Kentucky. Dr. Greg discusses this new cutting edge diagnostic tool with UK Sanders Brown Center on Aging researcher Dr. Gregory Jicha.

A structure to the acute abdomen and acute abdominal pain - what is acute abdomen, what are the causes of acute abdomen and the differential diagnosis of acute abdominal pain? Acute abdomen examination (causes by location) as well as history and acute abdomen signs and symptoms. We also take a look at how an acute abdomen is managed. PDFs available here: https://rhesusmedicine.com/pages/general-surgeryConsider subscribing (if you found any of the info useful!): https://www.youtube.com/channel/UCRks8wB6vgz0E7buP0L_5RQ?sub_confirmation=1Buy Us A Coffee!: https://www.buymeacoffee.com/rhesusmedicineTimestamps:0:00 What is an acute abdomen? 0:23 Causes of acute abdomen / Causes of acute abdominal pain2:40 Diagnosis of acute abdomen / Diagnosis of acute abdominal pain - Causes of acute abdomen by location 4:35 Diagnosis of acute abdomen / Diagnosis of acute abdominal pain - History6:40 Acute abdomen physical exam / Acute abdominal pain physical exam8:17 Diagnosis of acute abdomen / Diagnosis of acute abdominal pain - Labs and Imaging10:01 Treatment of acute abdomen / Treatment of acute abdominal painLINK TO SOCIAL MEDIA: https://www.instagram.com/rhesusmedicine/Disclaimer: Please remember this video and all content from Rhesus Medicine is for educational and entertainment purposes only and is not a guide to diagnose or to treat any form of condition. The content is not to be used to guide clinical practice and is not medical advice. Please consult a healthcare professional for medical advice.ReferencesMSD Manuals Professional Edition (2025) Acute abdominal pain. Reviewed/Revised Jul 2024; Modified Jul 2024. Available at: https://www.msdmanuals.com/en-gb/professional/gastrointestinal-disorders/acute-abdomen-and-surgical-gastroenterology/acute-abdominal-pain BMJ Best Practice (2025) Evaluation of acute abdomen. Last reviewed: 23 Dec 2025; Last updated: 22 Jul 2025. Available at: https://bestpractice.bmj.com/topics/en-us/503 BMJ Best Practice (2025) Acute appendicitis: symptoms, diagnosis and treatment. Last reviewed: 27 Dec 2025; Last updated: 17 Apr 2025. Available at: https://bestpractice.bmj.com/topics/en-gb/290 BMJ Best Practice (2025) Imaging and investigations for abdominal pain. Available at: https://bestpractice.bmj.com/topics/en-gb/787/diagnosis-approach NICE Guidelines NG156 (2025) Abdominal aortic aneurysm: diagnosis and management. Available at: https://www.nice.org.uk/guidance/ng156/chapter/recommendations

When should you attempt to save the root filled molar that everyone else thinks is doomed? What are the key steps to safely remove, treat, and replant a tooth without causing fractures or resorption? And how do you manage patient expectations and post-op care to maximize success? In this episode, Dr. Samuel Kratchman and Dr. Shivakar join Jaz to explore intentional tooth replantation—a procedure that rarely gets the spotlight but can completely change treatment options for challenging cases. They cover everything from case selection and imaging, to managing crowns and fragile teeth, to simple tools and techniques that make this procedure predictable and accessible. They also dive into patient communication, consent, and how to include this procedure as part of your everyday dental armamentarium, giving you the confidence to consider it when the right case comes along. https://youtu.be/SjJTzbJ_AXs Watch PDP256 on YouTube Key Takeaways: Intentional replantation is a viable alternative to extraction. The success rate of intentional replantation is documented at 88-89%. Patient education is crucial for successful treatment outcomes. The periodontal ligament must be kept moist during the procedure. Imaging is essential for understanding tooth anatomy before replantation. The procedure can be performed atraumatically with proper technique. Replantation can be a last chance for teeth that are difficult to replace with implants. A mindset shift is needed in dentistry to prioritize saving natural teeth. Apical infections are often linked to the root tip and surrounding tissue. A good coronal seal is essential before any restorative work. Common complications include ankylosis and resorption. Inflammation can aid in the extraction process by serving the ligament. Post-operative care is vital for successful recovery. Highlights: 00:00 Teaser 00:48 Introduction 03:27 Pearl: PDL is everything  04:54 Interview with Dr. Shivakar Mehrotra 07:03 Interview with Dr. Samuel Kratchman 11:01 Terminologies and Success Rates of Replantation 16:03 Indications of Replantation 22:29 Evaluating Radiographs and Clinical Factors 28:48 Case Studies and Practical Applications 30:51 Midroll 34:12 Case Studies and Practical Applications 38:08 Management of Apical Infection 40:35 Curveball Scenario: Combined Endodontic and Restorative Challenge 45:57 Replantation Success Rates and Complications 51:06 Radiographic Signs and Extraction Techniques 56:03 Postoperative Care and Instructions 59:49 Final Thoughts and Resources 01:02:14 Outro

Send us a textIn this episode, Dr. Matthew R. Hitchcock, MD, MBA, shares his journey as a direct primary care physician and healthcare entrepreneur. Frustrated by long waits and high costs in traditional imaging, he founded Hitchcock Direct Imaging, a cash-only, insurance-free imaging center in Chattanooga, Tennessee. Dr. Hitchcock discusses how direct care models can improve patient access, the regulatory hurdles he navigated, and the importance of transparency in healthcare pricing. Learn how collaboration, innovation, and patient-focused care are reshaping the healthcare experience. Connect with him:Website: Hitchcock Family MedicineHitchcock Direct Imaging Support the show*Next Coaching Cohort Starts Feb 25, 2026. Launch it in 8 weeks and apply here --> Record your question easily here --> Join the Direct Care Society private Facebook group here. --> Own a DSC practice and want to share your story? Apply here EMR I'm currently using in my Direct Care practice Simple Practice HIPAA Compliant Email with Paubox Get $250 Credit Here Find me on LinkedIn https://linkedin.com/in/teadpm More resources teadpm.com

Send us a textIn this episode, Dr. Matthew R. Hitchcock, MD, MBA, shares his journey as a direct primary care physician and healthcare entrepreneur. Frustrated by long waits and high costs in traditional imaging, he founded Hitchcock Direct Imaging, a cash-only, insurance-free imaging center in Chattanooga, Tennessee. Dr. Hitchcock discusses how direct care models can improve patient access, the regulatory hurdles he navigated, and the importance of transparency in healthcare pricing. Learn how collaboration, innovation, and patient-focused care are reshaping the healthcare experience. Connect with him:Website: Hitchcock Family MedicineHitchcock Direct Imaging Support the show*Next Coaching Cohort Starts Feb 25, 2026. Launch it in 8 weeks and apply here --> Record your question easily here --> Join the Direct Care Society private Facebook group here. --> Own a DSC practice and want to share your story? Apply here EMR I'm currently using in my Direct Care practice Simple Practice HIPAA Compliant Email with Paubox Get $250 Credit Here Find me on LinkedIn https://linkedin.com/in/teadpm More resources teadpm.com

The Final March of the Floppies Episode: A Visit with John Morris, The Applesauce, The Imaging of Floppies, An Office Becomes a Battlefield, The Forever Trickle, Images and Analytics, The Final Boss of the Floppies.A celebration of what may turn out to be one of the most definite major projects in the big-office era of my work - taking down the thousands of floppy disks people have sent me over the years.

Show Notes 23 January 2026Story 1: MIT just made aluminum 5x stronger with 3D printing - it is stronger, lighter, and tough enough to reshape the future of flightSource: ScienceDaily Based on MIT news announcementLink: https://www.sciencedaily.com/releases/2025/12/251226045316.htmSee also: https://news.mit.edu/2025/printable-aluminum-alloy-sets-strength-records-may-enable-lighter-aircraft-parts-1007Story 2: New 'physics shortcut' lets laptops tackle quantum problems once reserved for supercomputers and AISource: LiveScience.com Story by Owen HughesLink: https://www.livescience.com/technology/computing/new-physics-shortcut-lets-laptops-tackle-quantum-problems-once-reserved-for-supercomputers-and-aiStory 3: Hyperloop shock: China's maglev hits 435 mph in 2 seconds, sets world recordSource: Interesting Engineering Story by Sujita SinhaLink: https://interestingengineering.com/transportation/china-maglev-hits-435-mph-in-2-secondsHonorable MentionsStory: This 3D-printed, carbon-absorbing bridge is inspired by bonesSource: CNN Story by Rebecca CalmsLink: https://www.cnn.com/science/diamanti-3d-printed-concrete-sustainable-bridge-hnk-spc-intlStory: New drug stalls Alzheimer's development in breakthrough trialSource: ScienceAlert.com Story by David NieldLink: https://www.sciencealert.com/new-drug-stalls-alzheimers-development-in-breakthrough-trialStory: Scientists build a quantum computer that can repair itself using recycled atomsSource: Phys.org Story by Robert EganLink: https://phys.org/news/2025-12-scientists-quantum-recycled-atoms.htmlStory: New Image Sensor Breaks Optical Limits – My subhead: a lens‑free system that achieves sub‑micron, 3D resolution across a wide field of view, something previously considered impossible with optical wavelengthsSource: University of Connecticut Story by Sarah RedmondLink: https://today.uconn.edu/2025/12/new-image-sensor-breaks-optical-limits/

The medical system expects you to chase 33 different screening appointments across multiple specialists and check your organs one at a time. But there's a smarter way that scans your entire body in under an hour to catch cancer, aneurysms, fatty liver, and other serious health issues before they become life-threatening. In today's episode, I sit down with Dr. Daniel Durand, Chief Medical Officer of Prenuvo, to talk about the real-world power (and limitations) of proactive whole-body MRI screening. Dr. Durand walks us through how this advanced screening method detects over 500 conditions, many of which traditional screenings miss. We talk about the importance of early detection for cancers and neurodegenerative diseases, and how this proactive approach to health can save lives.   "It's better to see things early when you can intervene, and see them in a controlled context when you're healthy." ~ Dr. Daniel Durand   In This Episode: -  Introduction to Dr. Daniel Durand and his background - Conventional vs whole body scans - Conditions that whole-body MRI can detect - Bringing scans to underserved populations - Imaging for risk identification vs. diagnosis - How often you should rescan and what to expect - EMF exposure concerns and MRI safety parameters - How consumer demand is driving change in medicine   Products & Resources Mentioned: Prenuvo Whole-Body MRI: My listeners get a special discount when you book at https://prenuvo.com/wendymyers  Bon Charge Blue Light Blockers: Get 15% off with code WENDY at https://boncharge.com  Organifi Happy Drops: Save 20% with code MYERSDETOX at https://organifi.com/myersdetox  Organifi Collagen: Use code MYERSDETOX for 20% off at https://organifi.com/myersdetox  Chef's Foundry P600 Ceramic Cookware: Get 20% off with code WENDY20 at https://chefsfoundry.com  Heavy Metals Quiz: Take it for free at https://heavymetalsquiz.com    About Dr. Daniel Durand: Dr. Daniel Durand is a dual board-certified adult & pediatric radiologist and Chief Medical Officer at Prenuvo, where he leads clinical operations, research, and the medical group for the world's largest network of proactive whole-body MRI clinics. Previously, he served as Chief Clinical Officer & Chief Innovation Officer at LifeBridge Health and held leadership roles in accountable care at Johns Hopkins. He is passionate about empowering primary care and shifting medicine toward true prevention through advanced imaging. Learn more at: https://prenuvo.com/wendymyers    Disclaimer The Myers Detox Podcast was created and hosted by Dr. Wendy Myers. This podcast is for information purposes only. Statements and views expressed on this podcast are not medical advice. This podcast, including Wendy Myers and the producers, disclaims responsibility for any possible adverse effects from using the information contained herein. The opinions of guests are their own, and this podcast does not endorse or accept responsibility for statements made by guests. This podcast does not make any representations or warranties about guests' qualifications or credibility. Individuals on this podcast may have a direct or indirect financial interest in products or services referred to herein. If you think you have a medical problem, consult a licensed physician.

Success stories on the global stage! Kassa Darge, MD PhD, speaks with host Raisa Amiruddin, MBBS, on the training and educational needs to improve pediatric radiology in resource-limited settings, while sharing inspirational anecdotes from his career and strategies to get involved in outreach initiatives.

Send us a textJoin Kasey on the PT Snacks podcast as we delve into lumbar spinal stenosis, perfect for physical therapists and students eager to enhance their fundamentals. Understand the basics of stenosis, its impact on patients, and effective treatment approaches. Learn how to differentiate between types of stenosis, evaluate symptoms, and integrate conservative treatments with manual therapy and exercise for optimal patient outcomes. Stay informed on imaging techniques and when surgery might be necessary. Tune in for this bite-size segment and boost your practice skills!00:00 Introduction to PT Snacks Podcast00:48 Understanding Lumbar Spinal Stenosis02:47 Symptoms and Causes of Lumbar Spinal Stenosis07:57 Diagnosis and Imaging of Spinal Stenosis10:45 Conservative Treatments for Spinal Stenosis12:08 Surgical Options and Post-Op Care12:49 Conclusion and Additional ResourcesSupport the showNeed CEUs? Unlock unlimited online courses, live webinars, and certification-prep programs with MedBridge. You'll get: Thousands of accredited, evidence-based courses across multiple specialties (PT, OT, AT, SLP) that count for state-license CEUs. Access anytime, from your office, phone, or home—perfect for busy clinicians. One annual subscription, no per-course fee. Special offers: Use code PTSNACKSPODCAST at checkout and save over $100. Students use code PTSNACKSPODCASTSTUDENT for a discounted annual plan. Studying for the NPTE? Check out PT Final Exam — they've helped thousands of students pass with confidence. Use code PTSnacks at checkout for a discount. Stay Connected! Follow so you never miss an episode. Send your questions via email to ptsnackspodcast@gmail.com Join the email list ...

Stem cell science is reshaping treatment for complex disorders of the brain and spinal cord. Researchers develop cell based therapies to replace lost dopaminergic neurons in Parkinson's disease, fill gaps across spinal cord injuries, and calm seizure networks in refractory epilepsy by restoring the balance of inhibition and excitation. Teams test immune cell therapies against Epstein Barr virus infected B cells in multiple sclerosis and collaborate to move treatments across the blood brain barrier to reach diseased cells. Clinicians combine imaging in the operating room, surgical tools, and research on biological age and the pace of aging to understand disease and guide treatment. Patient advocates describe challenges and hopes for better options, and contemporary art reflects on perception in digital and physical worlds. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40992]

Stem cell science is reshaping treatment for complex disorders of the brain and spinal cord. Researchers develop cell based therapies to replace lost dopaminergic neurons in Parkinson's disease, fill gaps across spinal cord injuries, and calm seizure networks in refractory epilepsy by restoring the balance of inhibition and excitation. Teams test immune cell therapies against Epstein Barr virus infected B cells in multiple sclerosis and collaborate to move treatments across the blood brain barrier to reach diseased cells. Clinicians combine imaging in the operating room, surgical tools, and research on biological age and the pace of aging to understand disease and guide treatment. Patient advocates describe challenges and hopes for better options, and contemporary art reflects on perception in digital and physical worlds. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40992]

Stem cell science is reshaping treatment for complex disorders of the brain and spinal cord. Researchers develop cell based therapies to replace lost dopaminergic neurons in Parkinson's disease, fill gaps across spinal cord injuries, and calm seizure networks in refractory epilepsy by restoring the balance of inhibition and excitation. Teams test immune cell therapies against Epstein Barr virus infected B cells in multiple sclerosis and collaborate to move treatments across the blood brain barrier to reach diseased cells. Clinicians combine imaging in the operating room, surgical tools, and research on biological age and the pace of aging to understand disease and guide treatment. Patient advocates describe challenges and hopes for better options, and contemporary art reflects on perception in digital and physical worlds. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40992]

Stem cell science is reshaping treatment for complex disorders of the brain and spinal cord. Researchers develop cell based therapies to replace lost dopaminergic neurons in Parkinson's disease, fill gaps across spinal cord injuries, and calm seizure networks in refractory epilepsy by restoring the balance of inhibition and excitation. Teams test immune cell therapies against Epstein Barr virus infected B cells in multiple sclerosis and collaborate to move treatments across the blood brain barrier to reach diseased cells. Clinicians combine imaging in the operating room, surgical tools, and research on biological age and the pace of aging to understand disease and guide treatment. Patient advocates describe challenges and hopes for better options, and contemporary art reflects on perception in digital and physical worlds. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40992]

Stem cell science is reshaping treatment for complex disorders of the brain and spinal cord. Researchers develop cell based therapies to replace lost dopaminergic neurons in Parkinson's disease, fill gaps across spinal cord injuries, and calm seizure networks in refractory epilepsy by restoring the balance of inhibition and excitation. Teams test immune cell therapies against Epstein Barr virus infected B cells in multiple sclerosis and collaborate to move treatments across the blood brain barrier to reach diseased cells. Clinicians combine imaging in the operating room, surgical tools, and research on biological age and the pace of aging to understand disease and guide treatment. Patient advocates describe challenges and hopes for better options, and contemporary art reflects on perception in digital and physical worlds. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40992]

Stem cell science is reshaping treatment for complex disorders of the brain and spinal cord. Researchers develop cell based therapies to replace lost dopaminergic neurons in Parkinson's disease, fill gaps across spinal cord injuries, and calm seizure networks in refractory epilepsy by restoring the balance of inhibition and excitation. Teams test immune cell therapies against Epstein Barr virus infected B cells in multiple sclerosis and collaborate to move treatments across the blood brain barrier to reach diseased cells. Clinicians combine imaging in the operating room, surgical tools, and research on biological age and the pace of aging to understand disease and guide treatment. Patient advocates describe challenges and hopes for better options, and contemporary art reflects on perception in digital and physical worlds. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40992]

CMS has made virtual contrast supervision permanent in 2026, changing how imaging centers operate. Discover what compliance, patient safety, and weekend coverage look like under the new rules—and why preparation starts now. More at https://www.contrast-connect.com/blog-post/2026-the-year-of-virtual-supervision ContrastConnect City: Las Vegas Address: 309 Queens Gate Ct Website: https://www.contrast-connect.com/

The 2026 CMS rule change permanently authorizes virtual contrast supervision—but are imaging centers ready? We tackle how CT and MRI facilities can scale, stay compliant, and convert staffing challenges into operational advantages under the new guidelines. Learn more at https://www.contrast-connect.com/ ContrastConnect City: Las Vegas Address: 309 Queens Gate Ct Website: https://www.contrast-connect.com/

Lance Mabry, PT, DPT, OCS, FAAOMPT, an assistant professor at High Point University and a seasoned imaging educator, joins host Phil Plisky for a conversation that challenges the status quo around imaging in rehab. Together, they explore how clinicians like you can step into a more informed, active role when it comes to referring, reviewing, and communicating about diagnostic imaging. You'll hear practical insights that help you avoid common missteps, reduce patient harm, and advocate for evidence-based, ethical decision-making. Whether you're a PT, AT, OT, or SLP, this episode equips you to better navigate the gray areas of imaging with confidence.Learning ObjectivesAnalyze the evidence on asymptomatic findings in diagnostic imaging to support ethical and informed decision-makingApply evidence-based, practical strategies to actionably address and communicate missed or overlooked radiologic findings in a way that enhances interprofessional collaborationSolve patient case scenarios involving signs of cervical myelopathy by identifying implications for performance and participation in meaningful daily activitiesTimestamps(00:00:00) Welcome(00:00:05) The importance of imaging in rehabilitation(00:00:22) Introducing Dr. Lace Mabry(00:08:20) Understanding when to refer for imaging(00:15:05) The role of rehabilitation providers in imaging decisions(00:18:12) Effective communication with radiologists(00:29:00) Interpreting imaging findings and patient communication(00:45:30) Case study: successful imaging referral and outcomes(00:50:36) Three important takeawaysRehab and Performance Lab is brought to you by Medbridge. If you'd like to earn continuing education credit for listening to this episode and access bonus takeaway handouts, log in to your Medbridge account and navigate to the course where you'll find accreditation details. If applicable, complete the post-course assessment and survey to be eligible for credit. The takeaway handout on Medbridge gives you the key points mentioned in this episode, along with additional resources you can implement into your practice right away.To hear more episodes of Rehab and Performance Lab, visit ⁠⁠⁠⁠⁠⁠⁠⁠https://www.medbridge.com/rehab-and-performance-lab⁠⁠⁠⁠⁠⁠⁠⁠If you'd like to subscribe to Medbridge, visit ⁠⁠⁠⁠⁠⁠⁠⁠https://www.medbridge.com/pricing/

Send us a textIn this episode of the PT Snacks podcast, your host Kasey dives into the often-overlooked topic of rib stress fractures. Aimed at physical therapists and PT students, this segment focuses on identifying, understanding, and managing these injuries, particularly in rowers and overhead athletes. Learn about the prevalence, symptoms, risk factors, and treatment strategies for rib stress fractures, along with expert tips for differential diagnosis and patient management. Whether you're prepping for your SCS or looking to improve patient outcomes, this episode offers valuable insights delivered in an engaging, bite-sized format.00:00 Introduction to PT Snacks Podcast00:17 Understanding Rib Stress Fractures01:32 Prevalence and Affected Populations03:26 Symptoms and Diagnosis08:42 Differential Diagnoses10:45 Management and Treatment14:05 Imaging and Final Thoughts15:14 Conclusion and Additional ResourcesSupport the showNeed CEUs? Unlock unlimited online courses, live webinars, and certification-prep programs with MedBridge. You'll get: Thousands of accredited, evidence-based courses across multiple specialties (PT, OT, AT, SLP) that count for state-license CEUs. Access anytime, from your office, phone, or home—perfect for busy clinicians. One annual subscription, no per-course fee. Special offers: Use code PTSNACKSPODCAST at checkout and save over $100. Students use code PTSNACKSPODCASTSTUDENT for a discounted annual plan. Studying for the NPTE? Check out PT Final Exam — they've helped thousands of students pass with confidence. Use code PTSnacks at checkout for a discount. Stay Connected! Follow so you never miss an episode. Send your questions via email to ptsnackspodcast@gmail.com Join the email list ...

Part 2 goes from definitions to implications.Bottom-up pain does not mean “it's in the tissues.”This episode is a recording of a live interview with pain researcher and clinician Asaf Weissman. If you haven't watched Part 1, start there—we laid the foundation: why pain semantics matter, how mixed messages harm patients, and why “nothing is wrong with your body” is an overreach.In Part 2, we dig into:What “always bottom up” actually means (and what it doesn't)Structural paradigm vs pathophysiology: why imaging often fails usWhy stress, fear, and emotions are usually modulators, not causesThreshold models: when trajectories may (and may not) be changeableThe case for neuroimmune mechanisms in chronic pain statesWhere diagnostics and biologics may take chronic pain care nextWhat role physios may play as case managers and guidesThis is the second half of a two-part series. Part 1 builds the framework. Part 2 challenges how we interpret evidence, scope, and clinical uncertainty—while staying anchored to what helps the patient in front of you.*********************************************************************

Develop a clear approach to undifferentiated gait problems, from first hallway impressions to exam maneuvers that actually change decisions. Dr. Maebe O'Hare joins us to separate neuropathy, radiculopathy, and orthopedic causes, and to show where EMG, imaging, PT, and devices fit into the management of gait disorders.• Watching the walk for rhythm, cadence, symmetry, stance and swing• Using video to isolate limb and phase changes• History clues that localize head versus legs• Distinguishing neuropathic pain from radicular patterns• Non-neurologic causes including osteoarthritis and deconditioning• Sensory testing that matters for gait, including proprioception• Romberg done right and when to stress it• When EMG clarifies neuropathy versus nerve root disease• Imaging for neurogenic claudication and focal deficits• PT as diagnostic and therapeutic partner• Choosing assistive devices and AFOs to reduce falls• Setting expectations for neuropathic pain medsFind all of our prior podcasts on your favorite podcast app, and you can always check out our website at theneurotransmitters.comSend us a textUnderstanding Hypophosphatemia: Recognition, Diagnosis, and TreatmentEndocrine experts distinguish Hypophosphatemia from osteoporosis & osteomalaciaListen on: Apple Podcasts SpotifySupport the show Check out our website at www.theneurotransmitters.com to sign up for emails, classes, and quizzes! Would you like to be a guest or suggest a topic? Email us at contact@theneurotransmitters.com Follow our podcast channel on

What if we could precisely measure a cell at its most fundamental level? In this episode, we talk with the University of Chicago scientist Peter Maurer about how he and his colleagues made the breakthrough discovery of turning a protein found in living cells into the first biological quantum bit, also known as a qubit.Maurer explains how quantum systems—once thought to be too fragile for real-world use—are becoming some of the most powerful sensors ever built, and what they could teach us about the brain, the body and more. Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Ben & Woods listen back to some flubs from our imaging voice and react to breaking NFL news.

In this episode of Longevity by Design, host Dr. Gil Blander sits down with Dr. Wei-Wu, Executive Chairman at Human Longevity, Inc. Together, they explore how advances in genome sequencing, AI, and multi-layered diagnostics are changing the fight against age-related diseases. Wei-Wu shares why understanding your own genetic risks and combining them with other health data leads to better prevention and a longer healthspan.Wei-Wu explains the value of integrating genome sequencing, advanced imaging, and liquid biopsy to catch diseases like cancer early, before symptoms appear. He draws on real-world examples, including how combining different tests can spot cancers that single methods might miss. The conversation highlights how technology brings down costs, making once-rare insights widely available, and how each person stands to benefit from personalized risk profiles.The episode closes with practical advice: use today's tools to become the CEO of your own health. Wei-Wu urges listeners to embrace data-driven, individualized care and stresses that no single tool or habit holds all the answers. Instead, true longevity comes from a holistic, ongoing approach, one that uses all available knowledge to prevent disease and extend both life and health.Guest-at-a-Glance

Certified Thermal Electrician™ is the most complete thermal imaging certification program built specifically for electricians, electrical inspectors, and electrical contractors. This video is a sample from our program lesson on Understanding Severity in Electrical Thermal Imaging.This professional thermal imaging training teaches you how to safely perform infrared inspections, interpret thermal images using ΔT analysis, apply NFPA 70B & NFPA 70E standards, and write defensible inspection reports that protect both your customer and your license. Whether you are an electrician, master electrician, electrical contractor, facility maintenance technician, or electrical inspector, this course gives you real-world field skills you can apply immediately. 

Certified Thermal Electrician™ is the most complete thermal imaging certification program built specifically for electricians, electrical inspectors, and electrical contractors. This video is a sample from our program lesson on Understanding Severity in Electrical Thermal Imaging.This professional thermal imaging training teaches you how to safely perform infrared inspections, interpret thermal images using ΔT analysis, apply NFPA 70B & NFPA 70E standards, and write defensible inspection reports that protect both your customer and your license. Whether you are an electrician, master electrician, electrical contractor, facility maintenance technician, or electrical inspector, this course gives you real-world field skills you can apply immediately. 

Certified Thermal Electrician™ is the most complete thermal imaging certification program built specifically for electricians, electrical inspectors, and electrical contractors. This video is a sample from our program lesson on Understanding Severity in Electrical Thermal Imaging.This professional thermal imaging training teaches you how to safely perform infrared inspections, interpret thermal images using ΔT analysis, apply NFPA 70B & NFPA 70E standards, and write defensible inspection reports that protect both your customer and your license. Whether you are an electrician, master electrician, electrical contractor, facility maintenance technician, or electrical inspector, this course gives you real-world field skills you can apply immediately. 

Global Practices in Cardiac Imaging for Cardiac Sarcoidosis   Guest: Tahir Kafil, M.D. Host: Sharonne Hayes, M.D.   Cardiac imaging is a cornerstone in the diagnostic work-up and management of cardiac sarcoidosis. However, indications and use of advanced cardiac imaging vary from institution to institution, and even between providers at the same institution. We conducted an international Delphi consensus study of 89 global experts in cardiac sarcoidosis to evaluate real-world clinical practices and use of advanced cardiac imaging. We developed consensus for use of advanced cardiac imaging in cardiac sarcoidosis. Areas lacking consensus were noted as priority areas for research.   Topics Discussed: From a big picture perspective, how is cardiac sarcoidosis generally diagnosed? What exactly is the Delphi methodology your team used to build consensus?  The suggested algorithm for imaging in suspected cardiac sarcoidosis uses pretest probability, how was that defined? Was cardiac MRI superior to PET in your study? Does one have to be first? How do experts decide how often to do follow up PET imaging in established cardiac sarcoidosis? What were areas of priority research that were identified? The research study cited by Dr. Kafil was published on June 2, 2025. Click the following link to view the paper: https://www.jacc.org/doi/full/10.1016/j.jcmg.2025.02.010   Connect with Mayo Clinic's Cardiovascular Continuing Medical Education online at https://cveducation.mayo.edu or on Twitter @MayoClinicCV and @MayoCVservices. LinkedIn: Mayo Clinic Cardiovascular Services Cardiovascular Education App: The Mayo Clinic Cardiovascular CME App is an innovative educational platform that features cardiology-focused continuing medical education wherever and whenever you need it. Use this app to access other free content and browse upcoming courses. Download it for free in Apple or Google stores today! No CME credit offered for this episode.   Podcast episode transcript found here.

Scientists are expanding our understanding of MS at an unprecedented pace. This week, Dr. Leorah Freeman discusses why, as new discoveries and medications enter clinical practice, neurologists and MS specialists should ask themselves 3 important questions when considering a patient's treatment plan. Dr. Freeman is the Director of the Multiple Sclerosis and Neuroimmunology Center at Dell Medical School at the University of Texas at Austin, where she also leads the MS and Neuroimmunology fellowship program and the MS Imaging and Outcomes Research Laboratory.  We'll also tell you about study results that reveal two distinct biologically-informed MS subtypes. We're explaining some of the confusing background to the FDA's decision not to approve a disease-modifying therapy that achieved positive results in its Phase 3 clinical trial. And did we really need a study to tell us that people living with MS fear experiencing a relapse or disease progression? Well, yes. We'll explain why. We're also reminding you to mail your insurance premium payments and other important documents earlier than you have in the past.  And we're sharing details about the two clinical trials that received $4.1 million in funding as part of the International Progressive MS Alliance Experimental Medicine Trial Awards. We have a lot to talk about! Are you ready for RealTalk MS??! This Week: 3 questions your neurologist should be asking themselves  :22 Public Service Announcement: How the new rule about postmarks could affect your healthcare  1:16 FDA says it's not ready to approve Tolebrutinib  3:16 Study identifies two biologically-informed MS subtypes  6:29 Study results remind us that people with MS fear relapse and progression  10:09 The International Progressive MS Alliance invests $4.1 million in two clinical trials  14:04 Dr. Leorah Freeman discusses why neurologists need to ask themselves 3 important questions when considering a patient's treatment plan  17:18 Share this episode  33:16 SHARE THIS EPISODE OF REALTALK MS Just copy this link & paste it into your text or email: https://realtalkms.com/436 ADD YOUR VOICE TO THE CONVERSATION I've always thought about the RealTalk MS podcast as a conversation. And this is your opportunity to join the conversation by sharing your feedback, questions, and suggestions for topics that we can discuss in future podcast episodes. Please shoot me an email or call the RealTalk MS Listener Hotline and share your thoughts! Email: jon@realtalkms.com Phone: (310) 526-2283 And don't forget to join us in the RealTalk MS Facebook group! LINKS If your podcast app doesn't allow you to click on these links, you'll find them in the show notes in the RealTalk MS app or at www.RealTalkMS.com The Multiple Sclerosis Insider https://themultiplesclerosisinsider.substack.com STUDY: Combined Magnetic Resonance Imaging and Serum Analysis Reveals Distinct Multiple Sclerosis Types https://academic.oup.com/brain/article/148/12/4578/8321558 STUDY: Fear of Disease Progression and Relapse in Multiple Sclerosis: A Systematic Scoping Review https://frontiersin.org/journals/psychiatry/articles/10.3389/fpsyt.2025.1680781/full JOIN: The RealTalk MS Facebook Group https://facebook.com/groups/realtalkms REVIEW: Give RealTalk MS a rating and review http://www.realtalkms.com/review Follow RealTalk MS on Twitter, @RealTalkMS_jon, and subscribe to our newsletter at our website, RealTalkMS.com. RealTalk MS Episode 436 Guests: Dr. Leorah Freeman Privacy Policy

In this episode, Brian sits down with Darren from Oregon, a seasoned paranormal investigator turned dedicated Bigfoot researcher. Darren recounts the life-changing encounter that occurred during a bachelor party in the wilderness—an experience that shifted his focus from ghost hunting to tracking cryptids.What began as one startling sighting of Bigfoot evolved into years of ongoing research, multiple encounters, and the collection of compelling evidence.Darren shares remarkable details, including thermal camera footage, massive footprints, and hidden nesting sites deep in the forest. But Bigfoot isn't the only cryptid on his radar.Darren also dives into terrifying Dogman encounters—describing chilling moments that hint at a dangerous, possibly territorial conflict between these two legendary creatures.His stories provide a rare glimpse into the shadowy edge of the unknown, where cryptids clash and researchers tread carefully.  Darren also shores some of the tools that he uses in the field, including game cameras, thermal imaging, and the unexpected role of medicinal plant research in uncovering patterns in cryptid behavior.DBK Investigations YouTube Get Our FREE NewsletterGet Brian's Books Leave Us A VoicemailVisit Our WebsiteBecome a supporter of this podcast: https://www.spreaker.com/podcast/sasquatch-odyssey--4839697/support.

Episode 210: Heat Stroke BasicsWritten by Jacob Dunn, MS4, American University of the Caribbean. Edits and comments by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice. Definition:Heat stroke represents the most severe form of heat-related illness, characterized by a core body temperature exceeding 40°C (104°F) accompanied by central nervous system (CNS) dysfunction. Arreaza: Key element is the body temperature and altered mental status. Jacob: This life-threatening condition arises from the body's failure to dissipate heat effectively, often in the context of excessive environmental heat load or strenuous physical activity. Arreaza: You mentioned, it is a spectrum. What is the difference between heat exhaustion and heat stroke? Jacob: Unlike milder heat illnesses such as heat exhaustion, heat stroke involves multisystem organ dysfunction driven by direct thermal injury, systemic inflammation, and cytokine release. You can think of it as the body's thermostat breaking under extreme stress — leading to rapid, cascading failures if not addressed immediately. Arreaza: Tell us what you found out about the pathophysiology of heat stroke?Jacob: Pathophysiology: Under normal conditions, the body keeps its core temperature tightly controlled through sweating, vasodilation of skin blood vessels, and behavioral responses like seeking shade or drinking water. But in extreme heat or prolonged exertion, those mechanisms get overwhelmed.Once core temperature rises above about 40°C (104°F), the hypothalamus—the brain's thermostat—can't keep up. The body shifts from controlled thermoregulation to uncontrolled, passive heating. Heat stroke isn't just someone getting too hot—it's a full-blown failure of the body's heat-regulating system. Arreaza: So, it's interesting. the cell functions get affected at this point, several dangerous processes start happening at the same time.Jacob: Yes: Cellular Heat InjuryHigh temperatures disrupt proteins, enzymes, and cell membranes. Mitochondria start to fail, ATP production drops, and cells become leaky. This leads to direct tissue injury in vital organs like the brain, liver, kidneys, and heart.Arreaza: Yikes. Cytokines play a big role in the pathophysiology of heat stroke too. Jacob: Systemic Inflammatory ResponseHeat damages the gut barrier, allowing endotoxins to enter the bloodstream. This triggers a massive cytokine release—similar to sepsis. The result is widespread inflammation, endothelial injury, and microvascular collapse.Arreaza: What other systems are affected?Coagulation AbnormalitiesEndothelial damage activates the clotting cascade. Patients may develop a DIC-like picture: microthrombi forming in some areas while clotting factors get consumed in others. This contributes to organ dysfunction and bleeding.Circulatory CollapseAs the body shunts blood to the skin for cooling, perfusion to vital organs drops. Combine that with dehydration from sweating and fluid loss, and you get hypotension, decreased cardiac output, and worsening ischemia.Arreaza: And one of the key features is neurologic dysfunction.Jacob: Neurologic DysfunctionThe brain is extremely sensitive to heat. Encephalopathy, confusion, seizures, and coma occur because neurons malfunction at high temperatures. This is why altered mental status is the hallmark of true heat stroke.Arreaza: Cell injury, inflammation, coagulopathy, circulatory collapse and neurologic dysfunction. Jacob: Ultimately, heat stroke is a multisystem catastrophic event—a combination of thermal injury, inflammatory storm, coagulopathy, and circulatory collapse. Without rapid cooling and aggressive supportive care, these processes spiral into irreversible organ failure.Background and Types:Arreaza: Heat stroke is part of a spectrum of heat-related disorders—it is a true medical emergency. Mortality rate reaches 30%, even with optimal treatment. This mortality correlates directly with the duration of core hyperthermia. I'm reminded of the first time I heard about heat stroke in a baby who was left inside a car in the summer 2005. Jacob: There are two primary types: -nonexertional (classic) heat stroke, which develops insidiously over days and predominantly affects vulnerable populations like children, the elderly, and those with chronic illnesses during heat waves; -exertional heat stroke, which strikes rapidly in young, otherwise healthy individuals, often during intense exercise in hot, humid conditions. Arreaza: In our community, farm workers are especially at risk of heat stroke, but any person living in the Central Valley is basically at risk.Jacob: Risk factors amplify vulnerability across both types, including dehydration, cardiovascular disease, medications that impair sweating (e.g., anticholinergics), and acclimatization deficits. Notably, anhidrosis (lack of sweating) is common but not required for diagnosis. Hot, dry skin can signal the shift from heat exhaustion to stroke. Arreaza: What other conditions look like heat stroke?Differential Diagnosis:Jacob: Presenting with altered mental status and hyperthermia, heat stroke demands a broad differential to avoid missing mimics. -Environmental: heat exhaustion, syncope, or cramps. -Infectious etiologies like sepsis or meningitis must be ruled out. -Endocrine emergencies such as thyroid storm, pheochromocytoma, or diabetic ketoacidosis (DKA) can overlap. -Neurologic insults include cerebrovascular accident (CVA), hypothalamic lesions (bleeding or infarct), or status epilepticus. -Toxicologic culprits are plentiful—sympathomimetic or anticholinergic toxidromes, salicylate poisoning, serotonin syndrome, malignant hyperthermia, neuroleptic malignant syndrome (NMS), or even alcohol/benzodiazepine withdrawal. When it comes to differentials, it is always best to cast a wide net and think about what we could be missing if this is not heat stroke. Arreaza: Let's say we have a patient with hyperthermia and we have to assess him in the ER. What should we do to diagnose it?Jacob: Workup:Diagnosis is primarily clinical, hinging on documented hyperthermia (>40°C) plus CNS changes (e.g., confusion, delirium, seizures, coma) in a hot environment. Arreaza: No single lab confirms it, but targeted testing allows us to detect complications and rule out alternative diagnosis. Jacob: -Start with ECG to assess for dysrhythmias or ischemic changes (sinus tachycardia is classic; ST depressions or T-wave inversions may hint at myocardial strain). -Labs include complete blood count (CBC), comprehensive metabolic panel (electrolytes, renal function, liver enzymes), glucose, arterial blood gas, lactate (elevated in shock), coagulation studies (for disseminated intravascular coagulation, or DIC), creatine kinase (CK) and myoglobin (for rhabdomyolysis), and urinalysis. Toxicology screen if history suggests. Arreaza: I can imagine doing all this while trying to cool down the patient. What about imaging?-Imaging: chest X-ray for pulmonary issues, non-contrast head CT if neurologic concerns suggest edema or bleed (consider lumbar puncture if infection suspected). It is important to note that continuous core temperature monitoring—via rectal, esophageal, or bladder probe—is essential, not just peripheral skin checks. Arreaza: TreatmentManagement:Time is tissue here—initiate cooling en route, if possible, as delays skyrocket morbidity. ABCs first: secure airway (intubate if needed, favoring rocuronium over succinylcholine to avoid hyperkalemia risk), support breathing, and stabilize circulation. -Remove the patient from the heat source, strip clothing, and launch aggressive cooling to target 38-39°C (102-102°F) before halting to prevent rebound hypothermia. -For exertional cases, ice-water immersion reigns supreme—it's the fastest method, with immersion in cold water resulting in near-100% survival if started within 30 minutes. -Nonexertional benefits from evaporative cooling: mist with tepid water (15-25°C) plus fans for convective airflow. -Adjuncts include ice packs to neck, axillae, and groin; -room-temperature IV fluids (avoid cold initially to prevent shivering); -refractory cases, invasive options like peritoneal lavage, endovascular cooling catheters, or even ECMO. -Fluid resuscitation with lactated Ringer's or normal saline (250-500 mL boluses) protects kidneys and counters rhabdomyolysis—aim for urine output of 2-3 mL/kg/hour. Arreaza: What about medications?Jacob: Benzodiazepines (e.g., lorazepam) control agitation, seizures, or shivering; propofol or fentanyl if intubated. Avoid antipyretics like acetaminophen. For intubation, etomidate or ketamine as induction agents. Hypotension often resolves with cooling and fluids; if not, use dopamine or dobutamine over norepinephrine to avoid vasoconstriction. Jacob: What IV fluid is recommended/best for patients with heat stroke?Both lactated Ringer's solution and normal saline are recommended as initial IV fluids for rehydration, but balanced crystalloids such as LR are increasingly favored due to their lower risk of hyperchloremic metabolic acidosis and AKI. However, direct evidence comparing the two specifically in the setting of heat stroke is limited. Arreaza: Are cold IV fluids better/preferred over room temperature fluids?Cold IV fluids are recommended as an adjunctive therapy to help lower core temperature in heat stroke, but they should not delay or replace primary cooling methods such as cold-water immersion. Cold IV fluids can decrease core temperature more rapidly than room temperature fluids. For example, 30mL/kg bolus of chilled isotonic fluids at 4 degrees Celsius over 30 minutes can decrease core temperature by about 1 degree Celsius, compared to 0.5 degree Celsius with room temperature fluids. Arreaza: Getting cold IV sounds uncomfortable but necessary for those patients. Our favorite topic.Screening and Prevention:-Heat stroke prevention focuses on public health and individual awareness rather than routine testing. -High-risk groups—elderly, children, athletes, laborers, or those on impairing meds—should acclimatize gradually (7-14 days), hydrate preemptively (electrolyte solutions over plain water), and monitor temperature in exertional settings. -Communities during heat waves need cooling centers and alerts. -For clinicians, educate patients with CVD or obesity about early signs like dizziness or nausea. -No formal "screening" exists, but vigilance in EDs during summer surges saves lives. -Arreaza: I think awareness is a key element in prevention, so education of the public through traditional media like TV, and even social media can contribute to the prevention of this catastrophic condition.Jacob: Ya so heat stroke is something that should be on every physician's radar in the central valley especially in the summer time given the hot temperatures. Rapid recognition is key. Arreaza: Thanks, Jacob for this topic, and until next time, this is Dr. Arreaza, signing off.Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! References:Gaudio FG, Grissom CK. Cooling Methods in Heat Stroke. J Emerg Med. 2016 Apr;50(4):607-16. doi: 10.1016/j.jemermed.2015.09.014. Epub 2015 Oct 31. PMID: 26525947. https://pubmed.ncbi.nlm.nih.gov/26525947/.Platt, M. A., & LoVecchio, F. (n.d.). Nonexertional classic heat stroke in adults. In UpToDate. Retrieved September 7, 2025, from https://www.uptodate.com/contents/nonexertional-classic-heat-stroke-in-adults. (Key addition: Emphasizes insidious onset in at-risk populations and the role of urban heat islands in exacerbating classic cases.) Heat Stroke. WikEM. Retrieved December 3, 2025, from https://wikem.org/wiki/Heat_stroke. (Key additions: Details on cooling rates for immersion therapy, confirmation that anhidrosis is not diagnostic, and fluid titration to urine output for rhabdomyolysis prevention.)Theme song, Works All The Time by Dominik Schwarzer, YouTube ID: CUBDNERZU8HXUHBS, purchased from https://www.premiumbeat.com/. 

The microhematuria guideline just changed. Here's what clinicians need to know. In this episode of BackTable Urology, Dr. Daniel Barocas (Vanderbilt University) joins host Dr. Ruchika Talwar to break down the 2025 updates to the American Urological Association (AUA) Microhematuria Guideline and why these changes matter in everyday practice. --- SYNPOSIS They walk through the evidence driving the updates, including revised risk stratification and the expanding role of urinary biomarkers. The conversation highlights how these recommendations affect patient evaluation, imaging decisions, and shared decision-making, with an emphasis on balancing cancer detection, patient burden, and responsible use of healthcare resources. --- TIMESTAMPS 00:00 - Introduction01:33 - What Prompted the Guidelines Update?02:59 - Challenges of a Negative Microhematuria Evaluation06:21 - Initial Evaluation Guidelines07:58 - Risk Stratification18:11 - Imaging in Hematuria Workups21:16 - Use of Urinary Biomarkers33:25 - Potential Future Guideline Updates37:17 - Takeaways for Urologists --- RESOURCES AUA/SUFU Microhematuria Guidelinehttps://www.auanet.org/guidelines-and-quality/guidelines/microhematuria