Podcasts about pathological

"MK Confidential" fades in on a motel room in Daytona Beach, Florida. On January 22nd, 2009, George Anthony takes about seventy pills, drinks as much beer as he can, and begins writing an eight-page letter he never finishes. Losing his granddaughter has brought him to the edge. Two and a half years later, a prosecutor reads that letter aloud to the twelve people deciding whether his daughter is responsible. Megyn Kelly tracks the defense case through the trial. Lead defense attorney Jose Baez tells the jury that Caylee was not murdered, but instead drowned in the aboveground pool behind the family's house, and her grandfather found her. He accuses the men in the Anthony house of abusing Casey. Both men deny it, and no witness ever supports it. Cindy Anthony testifies that searches for “chloroform” on the family computer were hers, made while she looked into why her Yorkie kept eating bamboo leaves in the backyard; her employer's records put her at the office both afternoons. Then the state gets the last word. A detective searches the hard drive all day and overnight. Inside the courtroom, Judge Perry finds absolutely no evidence behind the abuse allegations and bars them from closing argument, while letting the drowning stand on the strength of photographs: a two-year-old climbing a pool ladder, and opening a sliding glass door by herself. And prosecutor Linda Drane Burdick, the last lawyer to speak, puts two photographs on the screen and leaves the jury with a single question: whose life was better after Caylee's death? This is Episode 4 of the Casey Anthony story. OneSkin: Get 15% off OneSkin with the code MEGYN at https://www.oneskin.co/MEGYN #oneskinpod Lean: Discover why LEAN is becoming the choice for real weight‑loss results—shop now at https://TAKELEAN.com use code KELLY Follow The Megyn Kelly Show on all social platforms:YouTube: https://www.youtube.com/MegynKellyTwitter: https://Twitter.com/MegynKellyShowInstagram: https://Instagram.com/MegynKellyShowFacebook: https://Facebook.com/MegynKellyShowFind out more information at: https://www.devilmaycaremedia.com/megynkellyshow Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

"MK Confidential" follows the case to trial. In her opening statement, prosecutor Linda Drane Burdick doesn't start with chloroform or duct tape. She begins with the story of a little girl, walking the jury through every day of Caylee's short life before landing on the thirty-one days no one reported her missing. Defense attorney Jose Baez fires back with a name the prosecution never said out loud: Roy Kronk, the meter reader who found Caylee's skull.Megyn sifts through the physical evidence piece by piece. A trunk that smelled like nothing to Casey's boyfriend— but smells like “death” to a tow-yard owner three weeks later, a hair traced back to someone from Caylee's maternal line inside that trunk, and an online search about how to make chloroform. Meanwhile, three medical examiners looking at the same skull, are unable to agree on whether that tape held Caylee's jaw in place or was never holding anything at all.By the time the state rests, it has built a case on a mother's lies stacked one on top of another — leaving the defense one afternoon to explain them away. This is Episode 3 of the Casey Anthony story.Lean: Discover why LEAN is becoming the choice for real weight‑loss results—shop now at https://TAKELEAN.com use code KELLYOneSkin: Get 15% off OneSkin with the code MEGYN at https://www.oneskin.co/MEGYN #oneskinpod Follow The Megyn Kelly Show on all social platforms:YouTube: https://www.youtube.com/MegynKellyTwitter: https://Twitter.com/MegynKellyShowInstagram: https://Instagram.com/MegynKellyShowFacebook: https://Facebook.com/MegynKellyShowFind out more information at: https://www.devilmaycaremedia.com/megynkellyshow Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

"MK Confidential" opens on the front door of Zenaida Fernandez-Gonzalez. Sheriff's deputies knock on the door and ask to search her house for a missing toddler. She has never met Casey Anthony, or her daughter Caylee. She's cleared by the police that same day — and then her phone number gets out, the threatening calls start. She loses her job and her reputation. All for sharing a name with someone Casey Anthony invented.Megyn traces that invented nanny back to 2006, two years before Caylee disappears. Casey describes a babysitter her family never meets, attached to a man Casey barely knows and a little boy who does not exist. On June 16, 2008, Casey tells her father that Caylee will be staying overnight with “Zanny.” It the last day George sees his granddaughter.Thirty-one days of excuses follow, as Casey empties a friend's checking account, tattoos “Bella Vita" on her shoulder, and parks a white Pontiac beside a dumpster, with the car seat still in the back. Finding the car is just the beginning, as Casey's lies begin to fall apart. This is Episode 2 of the Casey Anthony story.OneSkin: Get 15% off OneSkin with the code MEGYN at https://www.oneskin.co/MEGYN #oneskinpodHome Title Lock: Go to https://hometitlelock.com/megyn and use promo code MEGYN to get a FREE title history report and a FREE TRIAL of their Triple Lock ProtectionFollow The Megyn Kelly Show on all social platforms:YouTube: https://www.youtube.com/MegynKellyTwitter: https://Twitter.com/MegynKellyShowInstagram: https://Instagram.com/MegynKellyShowFacebook: https://Facebook.com/MegynKellyShowFind out more information at: https://www.devilmaycaremedia.com/megynkellyshow Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

"MK Confidential" examines the story of Casey Anthony. It all starts with a certified-mail notice that sits unnoticed on a pink front door for days— eventually leading to an impounded Pontiac Sunfire with a stench oh-so-familiar to Casey's former deputy sheriff father. George Anthony's granddaughter Caylee hasn't been seen in over a month.Megyn reconstructs the night Casey's mother, Cindy, makes three separate 911 calls in ninety minutes — the first about a stolen car, the last about a smell she can't explain — all while her daughter Casey sits in the next room insisting Caylee is just fine. Casey takes detectives to an empty apartment, a job she hasn't worked at in two years, and lies about having a nanny named Zenaida whom she says has Caylee. Behind it all is a woman fluent in lies, a pregnancy nobody quite believed, and a mother who keeps choosing not to look too closely. This is Episode 1 of the Casey Anthony story.Home Title Lock: Go to https://hometitlelock.com/megyn and use promo code MEGYN to get a FREE title history report and a FREE TRIAL of their Triple Lock ProtectionLean: Discover why LEAN is becoming the choice for real weight‑loss results—shop now at https://TAKELEAN.com use code KELLYFollow The Megyn Kelly Show on all social platforms:YouTube: https://www.youtube.com/MegynKellyTwitter: https://Twitter.com/MegynKellyShowInstagram: https://Instagram.com/MegynKellyShowFacebook: https://Facebook.com/MegynKellyShowFind out more information at: https://www.devilmaycaremedia.com/megynkellyshow Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

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A single drug undid the damage in mice who'd suffered early-life trauma. Also, a contagious form of 'cringe avoidance' is spreading — and listen in to the soft sounds of Jurassic crickets.

hello hotties and welcome back to smoke sesh! we have a lot to chat about today as we talk what I've been up to, dive bar experiences, seduction, flirting, the art of seduction, feeling yourself, fitting in, traveling, cosplaying, becoming a woman, growing pains, body changes, second puberty, moving your body, body neutrality, fear of driving, learning to drive, overeating, emotional eating, eating rationality, learning to cook, getting out of a funk, finding the light, learning to how fun, pathological liars, lying, growing up, existential dread, world worries, doing something about it, lingerie, living alone, friends with exes, what are we conversation, trusting yourself, monitoring spirits, getting flown out, healing and much much more!watch the vlogs on YouTube at hannahmarlene444join the patreon! new episodes (and more) at patreon.com/smokeseshshawtysubmit your questions and call ins every week on instagram.com/hannahmarlene

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In today's episode, Elizabeth sits down with Dr. Judith Joseph, a board certified psychiatrist and researcher who led the first ever peer reviewed study on high functioning depression. They talk about her upbringing as a Caribbean immigrant, the pressure to be constantly high achieving, and how she ended up depressed while giving a talk on how to help others get through crisis. Dr. Judith breaks down the difference between burnout and high functioning depression, explains why so many high achievers become pathologically productive as a way to outrun unprocessed pain, and shares her five Vs framework for reclaiming joy: validation, venting, values, vital signs, and vision. They also get into phone addiction, the link between hormones and depression, and why planning joy is not as unromantic as it sounds.If you want to go DEEPER with me, my Substack is where I share even more behind-the-scenes, personal reflections, and wellness experiments, with new posts dropping every Thursday: https://substack.com/@thewellnessprocessFollow Dr. Judith Joseph:Instagram: https://www.instagram.com/drjudithjosephWebsite: https://www.drjudithjoseph.comBook: https://www.highfunctioningbook.comFollow us: Instagram: https://www.instagram.com/thewellnessprocesspodTikTok: https://www.tiktok.com/@thewellnessprocessYouTube: https://www.youtube.com/@TheWellnessProcessSponsors:Use coupon code TWP to save 15%. The website is boncharge.com.NOBL gives you real travel peace of mind — security, design, and convenience all in one. Head to NOBLTravel.com for 46% off your entire order! #NOBL #adGet 20% off your DeleteMe plan when you go to joindeleteme.com/TWP and use promo code TWP at checkout.Use code WELLNESS at Monarch.com to get your first year of Monarch Core half off at just $50.head to koraorganics.com and use code WELLNESS20 for 20% off sitewide. Start your new morning ritual & get up to 43% off your @MUDWTR with code TWP at mudwtr.com/TWP! #mudwtrpodVisit Carawayhome.com/TWP10 to take an additional 10% off your next purchase.Please note that this episode may contain paid endorsements and advertisements for products and services. Individuals on the show may have a direct or indirect financial interest in products or services referred to in this episode.Produced by Dear Media.See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

We talk with Dr. Paul Jenkins about a clear positivity model that explains why our brains swing between judging what is and building what's next, and how that swing shows up in marriage. We walk away with simple tools that replace helpless frustration with choice, hope, and daily actions that actually change the tone at home. • Dr. Jenkins' shift from traditional therapy to positive psychology after a humbling bankruptcy • Awareness precedes choice as a foundation for personal growth and relationship repair • Two constant mind processes: evaluation of what is and creation of what is to be • How comparison drives emotion and why “it could be worse” can create real relief • Hope versus anxiety as outcomes of what we imagine is coming next • Toxic positivity as denial of reality versus healthy positivity as making peace with reality • Gratitude power-up as a five-day practice to notice what is good right now • BB8 Better By Eight as a small daily creation habit for immediate upgrades • Choosing love as a verb and a daily decision that shapes the marriage Happy Hacks: 101 Science-Backed Ways to Boost Happiness, Reduce Stress, and Build a More Meaningful Life.Available now starting at 99¢ on Amazon and Dave's Book StoreWritten by Dr. Dave Schramm, this practical and uplifting new book offers simple, research-based strategies to help you build greater happiness, strengthen resilience, reduceThe opinions, findings, conclusions, and recommendations expressed in this podcast do not necessarily reflect the views of the Utah Marriage Commission.Visit our site for FREE relationship resources and regular giveaways: Strongermarriage.org  Podcast.stongermarriage.orgYouTube: https://www.youtube.com/@StrongerMarriageLifeTikTok: https://www.tiktok.com/@strongermarriagelifeInstagram: https://www.instagram.com/strongermarriagelife/Facebook: https://www.facebook.com/strongermarriage/Facebook Marriage Group: https://www.facebook.com/groups/770019130329579Dr. Dave Schramm: http://drdaveschramm.comhttp://drdavespeaks.com Dr. Liz Hale: http://www.drlizhale.com/ 

We're constantly swamped by messages like rest or idleness leads to laziness or that if you rest, then you'll fall behind — it's been repeated by so many self-proclaimed gurus, business experts, and biohacking influencers that we've started to believe the false messaging. The truth is that you're wired to take purposeful action, to have a healthy store of motivation, and to rest so that you can do those things when needed.  So, how come so many of us feel stuck in the swing between pathological productivity and then guilt when resting? We'll dig into the concept of idleness aversion — the deeply human tendency to detest doing nothing — and explains its evolutionary roots. We are wired to assess whether action is needed and to feel motivated when it is. That's not a flaw. The flaw is when that drive gets hijacked by external messaging that equates busyness with moral virtue and rest with weakness. Henry brings the neuroscience of mind-wandering: when the mind is allowed to wander, there's a kind of syncing up and rest that can happen, almost like rebooting a computer or closing all the apps on your phone. Far from being a waste, it's the precondition for efficient activation when you actually need it.  We'll get into some practical strategies to help you embrace more rest. For example, get a hobby that cannot be turned into a side hustle. Find a third place where people aren't doing anything productive. Let yourself daydream with a timer.  And if you're looking for the episode that followed this one when it played originally, here it is: From FOMO to GOMO: Discovering Freedom and Joy in The Choices We Make [ep. 224]   Note: This is an encore episode, originally aired during Joy Lab's Element of Fun. It's replaying here during the month of Re-Creation because rest and play go hand in hand — and if part of you felt a flicker of guilt pressing play on a podcast instead of doing something "productive," this episode is exactly what you need to hear. Preferably in a hammock. Not because you deserve it, but because you need it.   About: The Joy Lab Podcast is an Ambie-nominated podcast that blends science and soul to help you cope better with stress, anxiety, and depression. It's hosted by integrative psychiatrist Dr. Henry Emmons and holistic mental health researcher Dr. Aimee Prasek. The podcast is best paired with the Joy Lab App.   Bonus: spread some joy and keep this podcast ad-free by donating (Joy Lab is powered by the nonprofit Pathways North and your donations are tax-deductible).   Listen & follow the Joy Lab Podcast on your favorite listening app: Spotify | Apple | YouTube   Sources and Notes: Joy Lab Experiments: Take the next leap in your wellbeing journey with step-by-step practices to help you build and maintain the elements of joy in your life. Joy Lab episodes referenced: Are You Stuck in Joyless Urgency? [ep 222] Where's Your Third Place? [ep. 171] Sometimes I Just Sits... (the power of solitude) [ep. 74] Four Thousand Weeks by Oliver Burkeman Series on authenticity from our Joy Lab podcast: Unmasking Your True Self: Exploring Authenticity and Awe [ep. 216] The Truth Behind Your Thoughts And How to Calm Your Mind (encore) [ep. 141] Embrace Your True Self: Accepted, Connected, & In The Game [ep. 217] The Road Most Travelled: Awakening Through Suffering [ep. 218]  Follow Your Bliss: Awakening to Joy [ep. 219] The Still Small Voice: Awakening with soulfulness [ep. 220] Just think: The challenges of the disengaged mind (this is the study of people shocking themselves out of boredom) Chandola, T., Ling, W., & Rouxel, P. (2025). Are anxious Mondays associated with HPA-axis dysregulation? A longitudinal study of older adults in England. Journal of Affective Disorders, 389. Access here.   Questions this episode was made for: Q: Why do I feel guilty when I'm not being productive, even when I know I need rest? A: This episode names and traces the moral weight most of us carry around rest. There is a culturally reinforced message that busyness is virtuous and idleness is weak. We'll dig into the evolutionary roots of idleness aversion and the neuroscience of why consciously letting your mind wander is genuinely restorative, not a waste.   Q: Is it possible to actually retrain a stressed-out nervous system that's gotten used to constant productivity? A: Yes, but it takes practice. It takes time to create new pathways and patterns. This episode explains how those neural pathways form and what gentle, practical steps (hobbies, third places, daydreaming, solitude) begin to build new ones.    Key moments: [00:00] Aimee's intro to the encore: idleness aversion, the moral weight on rest, and the invitation to listen in a hammock — not because you deserve it, but because you need it [02:00] Pathological productivity defined: on a spectrum from no productivity (feels bad for long, briefly good) to overwork (most of us have been there) to pathological productivity (causing harm, planting seeds of future illness) [03:30] Work addiction as a real condition — as hard to break as any other addiction once it sets in [05:00] Idleness aversion: the very common human tendency to detest being idle [06:00] The evolutionary reason: we're wired to assess whether action is needed and motivated when it is — not pathologically lazy creatures by nature [07:00] The paradox: we don't like idleness AND we want a reason to be busy — this sets us up for anxiety around rest and unconscious pathological productivity [09:30] Getting comfortable with rest is a skill — we work on it here at Joy Lab [10:00] The Anxious Mondays study. Finding: those who felt anxious on Mondays had elevated cortisol lasting at least two months. The twist: this continued even in retirees. [12:00] We're training ourselves to be stressed by productivity, and the neural pathway outlasts the job [13:00] The habit of being stressed by productivity — whatever the driver (money, pleasing others, excellence) — sets us up for long-term problems if we don't build in genuine downtime [15:00] The moral layer on rest: the deeply warped message that idleness leads to evil, laziness, or poverty. Guru-culture messaging: the gist is that rest is morally weak and busyness is morally strong — and it's false [16:00] The opposite of work is not evil — just as the opposite of play is not work but depression; we've gotten the opposites wrong [17:00] Our inner wisdom calls us to play, rest, AND work — a rhythm we're wired to follow rather than fight [19:00] The neuroscience of mind-wandering: Henry's image of the brain as a domed web of nodes and connections, always active [21:00] During mind-wandering: everything settles; hyperactive areas rest; underactive areas come online; whole system syncs up; Like rebooting a computer or closing all apps — fresh starting place, more efficient activation when you need it [24:00] Positive mind-wandering (playful, creative, inspiring) vs. rumination — similar states, very different outcomes [25:00] Practice ideas: guided visualizations, set a timer and let yourself daydream creatively [25:30] Get a hobby — something that cannot be turned into a side hustle, is not quantifiable, and just makes you smile [27:00] Identify a third place with a relationship to rest: meditation class, library, quiet coffee shop — somewhere people aren't checking things off lists [28:00] Oliver Burkeman, 4,000 Weeks: "In an age of instrumentalization, the hobbyist is a subversive" [29:00] Find something you think is a waste of time and try it — birding, train spotting, stamp collecting; whatever your version of that is [30:30] Oliver Burkeman closing: "Productivity is a trap... The day will never arrive when you finally have everything under control. But you know what? That's excellent news."   If you enjoyed this episode, please rate and review us wherever you listen to your favorite podcasts!    Please remember that this content is for informational and educational purposes only. It is not intended to provide medical advice and is not a replacement for advice and treatment from a medical professional. Please consult your doctor or other qualified health professional before beginning any diet change, supplement, or lifestyle program. Please see our terms for more information. If you or someone you know is struggling or in crisis, help is available. Call the NAMI HelpLine: 1-800-950-6264 available Monday through Friday, 10 a.m. – 10 p.m., ET. OR text "HelpLine" to 62640 or email NAMI at helpline@nami.org. Visit NAMI for more. You can also call or text SAMHSA at 988 or chat 988lifeline.org. Find more mental health resources here.  

Welcome to There is Nothing to Fear, written by Callmesalticidae and read by Sam Gabriel! This story is serialized in the form of multiple fics with different names, recorded and presented in order on this feed. All text can be found on ao3. My website can be found at http://samgabrielvo.com. Enjoy!

Pathological optimism meets its cathartic downside. Listen to a 2-minute burst about what sucks in life as I ride my recumbent trike. It's lame & heartfelt. Click here to view the printable newsletter. More readable than a transcript. Click here to view the verbatim transcript Summary Pathological optimism doesn’t mean things don’t suck. Sometimes you just need a 2-minute burst of complaining. I hate leaking misery in dribs and drabs. I let loose about my sax struggles, MS fatigue, too many pills, old-man prostate, and empty nesting. It’s ridiculous, it’s cathartic, and by the end, my annoyances are down a notch. Episode Proem: The Downside of Pathological Optimism Okay. Good morning. I’m riding my trike, and I’m feeling the need for a 2-minute bitch. I don’t know if I’ve talked to you before; I might have, about the 2-minute bitch. See, one downside of being pathologically optimistic is that it doesn’t mean things still don’t suck. So every once in a while, I just must have a 2-minute bitch. I don’t like it to— I don’t like my miserableness, or unhappiness, or whatever it is to, like, leak out in drips and drabs and— you know, I’d rather have a burst. Sourpuss Kick-off: I Suck at Music. I Quit. So, today—last night, I had a rehearsal. Oh my God. A rehearsal with the Latin band, Lechuga Fresca. Oh, man. You know, right now I’m— I was the only horn there, and I’m often the only horn. And I’m a Bari sax player, and I’m never the only horn. So this is a really new experience for me. Well, it’s not so new; it’s been going on for a year, so. But anyway, I’m— oh man, I just— I just don’t hear the music, or I’m not counting the 1, and I’m just, like, I feel like, you know, there’s like 3 percussionists— no, what were there? Today— yesterday— there was just— last night— there was just 2 percussionists, a keyboard player, and that was it. So it was not enough. Clearly not enough. But it’s stuff I’ve been working on. I’ve just been working on it, and I— I just can’t get it. I just don’t hear it. And then I sort of freeze up, and I— ugh, I just hate that. And I feel like I’m remedial, you know? Like, my band mates were— they’re really nice, and they work with me, and— I don’t know. But anyway, I just— I just have this feeling that I’m going to quit. I’m going to quit this band. This is too much. I’m also playing with a Dixieland band and rehearsing once a week. I still play an hour a day. An hour a day. And I’m just not— I don’t know— not getting better fast enough, I guess. Tired of Tired And I’m tired. I just hate being tired all the time. I wake up in the morning, and I’m exhausted. I hate that. I hate being exhausted. And then, yeah, I don’t sleep that well. I usually say, “Oh, good enough.” And it is good enough. I mean, I do what I do. But I want to have more energy. I don’t know. What would I do with more energy? Anyway, I’m just going to be miserable. Let’s see. What else can I be miserable about? I have cramps. I have cramps. Cramps in my calves. Cramps in my back. Cramps in my shoulders. It’s just annoying. It’s like stuff I've got to manage all the time. I don’t want to manage this stuff. I want to be 74 years old and not have pain. Ugh, that sounds ridiculous. Anyway, I’m bitching. Ugh. What else? Well, here I am, riding my trike. I had— oh, my legs really aren’t that strong. I wish they were stronger. And it’s actually my strong right leg that feels weak. I mean, it makes sense. You know? I got MS, but anyway, I’m bitching. And what have I done? Ridden the trike— what, so far? 6 times, maybe, this season. And I don’t feel like I’m getting stronger. I mean, I don’t have to be stronger. I got a lovely ICE trike. It’s a top-of-the-line trike. It’s got great gears. It’s very comfortable to ride. Old White Man with Old White Man Problems Wait a minute. I’m bitching. What else can I bitch about? I wish I wasn’t an old man with prostate issues. How do you like them apples? It’s just annoying. Annoying, annoying, annoying. Oh goodness. What else can I— I’m going to dig deep here. Find something else to bitch about. I don’t have anything else. Ugh. Oh, here. I got another one. I take too many pills. You know? I have to sit there for 20 minutes every Sunday and lay out my pills. What am I doing taking so many pills? Pills for my cholesterol. Pills for my MS. Pills for anemia. Pills for diabetes. Ugh. What have I gotten to? Pills, pills, pills. I might have just run out of steam on that. Let me ride a little longer and see if I can come up with something else to bitch about. Here, I can bitch about having to wait for a red light. How you like that? How you like that, huh? Oh, I didn’t have to wait. Oh well. That was a wasted bitch. Man, it’s a great temperature. Really nice. It’s cloudy, and it’ll probably rain. I could bitch about the rain, even though we need it. I like rain. It’s so much work to be at the top of my game. I want to be at the top of my game all the time. There. There’s another bitch. Man, this is really lame. My lame bitching. All right. I’ll turn it on again if I think of more. Round 2. Losing It All right. I’m a little farther along now, and I have a couple more things to bitch about. One is, I got back on the video because my wife just called to say that some very nice man stopped at the house on his electric bike, bringing my wallet, which I dropped on the bike path here. So, you know, I’m such an idiot. I— I know these pockets suck, and I don’t— I thought I had my wallet in this pocket that has a flap on it, but it’s not really, I guess, a flap that closes. So anyway. There. I’m bitching about being such an idiot, doing stupid things. Empty Nesting I have another thing I thought I had to bitch about. So my—my son, my daughter-in-law, and my grandson moved. They lived upstairs from us for 18 years. We had 18 years with them. And they needed to go. It was enough with the in-laws. But I really miss my son and my grandson. My grandson’s going to college. He’s such a sweetie. And he— he’s gone. And I feel it even more than when my— I guess, when my kids left. I don’t know. Simon, I was glad to see him go. He was just ready to be away from us. He hated me. And then when Ruben left, Mike was still there. When Mike left, that was hard. Really hard. I missed him. I still miss him. He died at 26, God. Anyway. But Leon. I miss Leon. I try to text him almost every day. I’m a pain in the ass, Opa. But he says it’s fine. He likes it. But anyway. There you go. Something else I can feel sorry for myself about. Cycling / Walking Community Pretty nice, isn’t it? Look at this beautiful here. I’m going so slow. It’s a little bit uphill. I think there’s a limit to how strong I can get. There. I got to bitch about that. If I already bitched about that, I don’t like to be repetitive. Anyway. It’s pretty lame. Good morning. (as I pass a cluster of people on the bike path talking). I like seeing people out here. Chatting and exercising. It’s nice being in a community. All right. I’m going to turn it off again. Anyway, I just listened to it. That was it. Reflection: Catharsis. Downgrading Annoyances It was cathartic. It was ridiculous. I hate complaining. Though, when I’m with other people who are gossiping, I gossip. When I’m— when I sour on a relationship or a job, I can be really negative. And when I’m sour or negative for more than a few minutes, I can feel it. All my annoyances escalate a step. It’s my scale. Not 1 to 10, but annoyances. I go from annoying to moderately annoying, to seriously annoying, to catastrophizing, to crippling. So. That was it. A little longer than 2 minutes, but you know, I packed it right in there. Anyway. Thanks for spending this time with me. Later. Please comment and ask questions: at the comment section at the bottom of the show notes on LinkedIn  via email YouTube channel  DM on Instagram, TikTok to @healthhats Substack Patreon Production Team Kayla Nelson: Web and Social Media Coach, Dissemination, Help Desk  Steve Heatherington: Help Desk and podcast production counseling Joey van Leeuwen, Drummer, Composer, and Arranger, provided the music for the intro, outro, proem, and reflection Claude, Perplexity, Auphonic, Descript, Grammarly, DaVinci Resolve, DaVinci AI Art Generator, OpenArt AI Creator Studio Inspired by and Grateful to: Peter Lehman, Jeff Harrington, Shel Schenkler Artificial Intelligence in Podcast Production Health Hats, the Podcast, utilizes AI tools for production tasks such as editing, transcription, and content suggestions. While AI assists with various aspects, including image creation, most AI suggestions are modified. All creative decisions remain my own, with AI sources referenced as usual. Questions are welcome. Creative Commons Licensing CC BY-NC-SA This license enables reusers to distribute, remix, adapt, and build upon the material in any medium or format for noncommercial purposes only, and only so long as attribution is given to the creator. If you remix, adapt, or build upon the material, you must license the modified material under identical terms. CC BY-NC-SA includes the following elements:    BY: credit must be given to the creator.   NC: Only noncommercial uses of the work are permitted.    SA: Adaptations must be shared under the same terms. Please let me know. dannyhealthhats@gmail.com  Material on this site created by others is theirs, and use follows their guidelines. Disclaimer The views and opinions presented in this podcast and publication are solely my responsibility and do not necessarily represent the views of the Patient-Centered Outcomes Research Institute®  (PCORI®), its Board of Governors, or Methodology Committee. Danny van Leeuwen (Health Hats)

******Support the channel******Patreon: https://www.patreon.com/thedissenterPayPal: paypal.me/thedissenterPayPal Subscription 1 Dollar: https://tinyurl.com/yb3acuuyPayPal Subscription 3 Dollars: https://tinyurl.com/ybn6bg9lPayPal Subscription 5 Dollars: https://tinyurl.com/ycmr9gpzPayPal Subscription 10 Dollars: https://tinyurl.com/y9r3fc9mPayPal Subscription 20 Dollars: https://tinyurl.com/y95uvkao ******Follow me on******Website: https://www.thedissenter.net/The Dissenter Goodreads list: https://shorturl.at/7BMoBFacebook: https://www.facebook.com/thedissenteryt/Twitter: https://x.com/TheDissenterYT This show is sponsored by Enlites, Learning & Development done differently. Check the website here: http://enlites.com/ Dr. Nima Bassiri is a social theorist, historian & philosopher of the human sciences, and associate professor at Duke University, where he teaches in the Program in Literature, Duke's interdisciplinary humanities and cultural studies program. He is also the co-director of Duke's Institute for Critical Theory, where he convenes the Critical Theory Workshop. He is the author of Madness and Enterprise: Psychiatry, Economic Reason, and the Emergence of Pathological Value. In this episode, we focus on Madness and Enterprise. We discuss how psychiatry developed under capitalism, the link between economic value and mental illness, the sociocultural context of psychiatry, and the madness of business enterprise. We discuss an economic style of psychiatric reasoning as a hermeneutic strategy for self-intelligibility, psychiatric diagnoses as interpretations, and when immoral behaviors are labelled as illnesses. Finally, we discuss a critique of capitalism, and a realistic perspective on psychiatry.--A HUGE THANK YOU TO MY PATRONS/SUPPORTERS: PER HELGE LARSEN, BERNARDO SEIXAS, ADAM KESSEL, MATTHEW WHITINGBIRD, ARNAUD WOLFF, TIM HOLLOSY, HENRIK AHLENIUS, ROBERT WINDHAGER, RUI INACIO, ZOOP, MARCO NEVES, COLIN HOLBROOK, PHIL KAVANAGH, SAMUEL ANDREEFF, FRANCIS FORDE, TIAGO NUNES, FERGAL CUSSEN, HAL HERZOG, NUNO MACHADO, JONATHAN LEIBRANT, JOÃO LINHARES, STANTON T, SAMUEL CORREA, ERIK HAINES, MARK SMITH, JOÃO EIRA, TOM HUMMEL, SARDUS FRANCE, DAVID SLOAN WILSON, YACILA DEZA-ARAUJO, ROMAIN ROCH, YANICK PUNTER, CHARLOTTE BLEASE, NICOLE BARBARO, PAWEL OSTASZEWSKI, NELLEKE BAK, GUY MADISON, GARY G HELLMANN, SAIMA AFZAL, ADRIAN JAEGGI, JOÃO BARBOSA, JULIAN PRICE, HEDIN BRØNNER, FRANCA BORTOLOTTI, GABRIEL PONS CORTÈS, URSULA LITZCKE, SCOTT, ZACHARY FISH, TIM DUFFY, SUNNY SMITH, JON WISMAN, WILLIAM BUCKNER, LUKE GLOWACKI, GEORGIOS THEOPHANOUS, CHRIS WILLIAMSON, PETER WOLOSZYN, DAVID WILLIAMS, DIOGO COSTA, ALEX CHAU, CORALIE CHEVALLIER, BANGALORE ATHEISTS, LARRY D. LEE JR., OLD HERRINGBONE, DAN SPERBER, ROBERT GRESSIS, JEFF MCMAHAN, JAKE ZUEHL, MARK CAMPBELL, TOMAS DAUBNER, LUKE NISSEN, KIMBERLY JOHNSON, JESSICA NOWICKI, LINDA BRANDIN, VALENTIN STEINMANN, ALEXANDER HUBBARD, BR, JONAS HERTNER, URSULA GOODENOUGH, DAVID PINSOF, SEAN NELSON, MIKE LAVIGNE, JOS KNECHT, LUCY, MANVIR SINGH, PETRA WEIMANN, CAROLA FEEST, MAURO JÚNIOR, TONY BARRETT, NIKOLAI VISHNEVSKY, STEVEN GANGESTAD, TED FARRIS, HUGO B., JORDAN MANSFIELD, CHARLOTTE ALLEN, PETER STOYKO, DAVID TONNER, LEE BECK, PATRICK DALTON-HOLMES, NICK KRASNEY, RACHEL ZAK, DENNIS XAVIER, CHINMAYA BHAT, RHYS, ALEX MACLEOD, HAIDAR, JULIEN PORCHER, ROBERT SUNDSTRÖM, JON STEWART, AND JENNY M!A SPECIAL THANKS TO MY PRODUCERS, YZAR WEHBE, JIM FRANK, ŁUKASZ STAFINIAK, TOM VANEGDOM, BERNARD HUGUENEY, CURTIS DIXON, THOMAS TRUMBLE, KATHRINE AND PATRICK TOBIN, JONCARLO MONTENEGRO, NICK GOLDEN, CHRISTINE GLASS, IGOR NIKIFOROVSKI, PER KRAULIS, ADAM HUNT, ANTHONY DI LORENZO, AND JOÃO BARBOSA!AND TO MY EXECUTIVE PRODUCERS, MATTHEW LAVENDER,SERGIU CODREANU, AND GREGORY HASTINGS!

Read the full article at: https://oncdata.com/ai-driven-pathological-subtyping-pancreatic-cancer-ashish-manne Artificial intelligence (AI) continues to generate excitement across oncology, but its real-world clinical impact remains inconsistent. In this episode of Exploring AI in Oncology, Dr. Ashish Manne, a Gastrointestinal Medical Oncologist at The Ohio State University Comprehensive Cancer Center, Arthur G. James Cancer Hospital and Richard J. Solove Research Institute, shares his American Association for Cancer Research (AACR) presentation on AI-driven pathological subtyping in pancreatic cancer and highlights the critical shift that is moving AI from theoretical promise to practical application.

The Meatballs thought they were just recapping Maternal Instincts but turns out it was a free therapy session also. Join us as we relive some old memories, watch us get triggered over questionable friendships and discover why we are the way we are. Come laugh and spiral with us. 

Most treatments for Borderline Personality Disorder focus on emotional dysregulation. The goal is to reduce distress, self-harm, anxiety, and crises. But that misses the mark. In this episode, I argue that modern psychiatry has adopted a model of Borderline Personality Disorder that is incomplete. By focusing almost exclusively on suffering, it neglects the maladaptive strategies harm others. I also examine the striking contrast between how psychiatry approaches Borderline Personality Disorder—predominantly diagnosed in women—and Antisocial Personality Disorder, diagnosed predominantly in men. Why do we define one disorder primarily by the patient's suffering and the other by the harm inflicted on others? And what has this double standard done to treatment? In the second half of the episode I outline what I believe a more realistic treatment model would look like. If you've ever wondered why Borderline Personality Disorder remains so difficult to treat, this episode offers a very different way of thinking about the problem.

Send us Fan MailIn this Journal Club episode, Daphna reviews a retrospective cohort study from Istanbul examining clinical, laboratory, and ultrasound factors associated with UTI in neonates hospitalized for unexplained hyperbilirubinemia. Among 96 term and near-term infants, 31% had culture-proven UTIs, a striking prevalence. Pathological renal ultrasound findings were independently associated with UTI, with affected neonates 4.6 times more likely to have a concurrent infection. Notably, standard laboratory markers including CRP and white blood cell count failed to distinguish UTI-positive from UTI-negative infants. The findings prompt a practical question: should urine culture be part of the routine workup for neonatal hyperbilirubinemia?----Renal ultrasonography findings are associated with urinary tract infection in neonates with asymptomatic hyperbilirubinemia. Sarı EE, Salihoğlu Ö.J Perinatol. 2026 Apr 13. doi: 10.1038/s41372-026-02686-x. Online ahead of print.PMID: 41975209Support the showAs always, feel free to send us questions, comments, or suggestions to our email: nicupodcast@gmail.com. You can also contact the show through Instagram or Twitter, @nicupodcast. Or contact Ben and Daphna directly via their Twitter profiles: @drnicu and @doctordaphnamd. The papers discussed in today's episode are listed and timestamped on the webpage linked below.Enjoy!

In today's episode, Tanner and Anne provide ideas on how to get to the root cause of your chronic pain or symptoms.Neuroplastic pain/symptoms occur when there's a sense of danger and dysregulation in the brain and nervous system - not due to physical disease or damage. So it makes sense to ask - what's creating this sense of danger?You'll learn about pain/symptoms can be fueled by:• Prolonged high stress, including perfectionism and people pleasing• Childhood adversity and trauma as sensitizing factros• Pathological views of the body fueled by medical ideas, Googling, ChatGPT, fear, and avoidance • Emotional resistance• Dysregulated responses to feeling pain/symptoms throughout the day.Tanner Murtagh and Anne Hampson are therapists who treat neuroplastic pain and mind-body symptoms. They are also married!In his 20s, Tanner overcame chronic pain and a fibromyalgia diagnosis by learning his symptoms were neuroplastic, not structural. Post-healing, Tanner and Anne have dedicated their lives to developing effective treatment and education for neuroplastic pain and symptoms.Listen and learn how to assess your own chronic pain and symptoms, gain tools to retrain the brain and nervous system, and make changes in your life and health!The Mind-Body Couple podcast is owned by Pain Psychotherapy Canada Inc. This podcast is produced by Alex Klassen, one of the wonderful therapists at our agency in Calgary, Alberta. https://www.painpsychotherapy.ca/Tanner, Anne, and Alex also run the MBody Community, an in-depth online course that provides a step-by-step process for assessing, treating, and resolving mind-body pain and symptoms. https://www.mbodycommunity.comCheck out Tanner's YouTube channel for more free education and practices: https://www.youtube.com/channel/UC-Fl6WaFHnh4ponuexaMbFQAnd follow us for daily education posts on Instagram: @painpsychotherapyDisclaimer: The information provided on this podcast is for general in...

"Corresponding member of the Swedish Pathological Society" [HOUN]    When James Mortimer, M.R.C.S. left behind his walking stick in The Hound of the Baskervilles, it was filled with clues — enough to lead Watson to his medical directory, which listed Mortimer as a member of the Swedish Pathological Society.   In his 2008 Morley-Montgomery Award-winning article in Vol. 58, No. 4 of The Baker Street Journal, Mattias Boström, BSI ("The Swedish Pathological Society") determined that Mortimer's membership was an impossibility. He explains why and it's just a Trifle.     If you have a question for us, please email us at trifles@ihearofsherlock.com. If you use your inquiry on the show, we'll send you a thank you gift.   There's a new "Trifling Trifles" episode out — short-form content that doesn't warrant a full episode. We release these at the beginning of every month. The latest episode is "for those with ears attuned to catch the distant view-halloo!" This is a benefit exclusively for our paying subscribers. Check it out (Patreon | Substack).   Our Merch Store is now open: Trifles mugs, notepads, and oval stickers can be yours (or someone else's, if you'd like to make it a gift). Start shopping today.      Leave Trifles a five-star rating on Apple Podcasts and Spotify; listen to this episode here or wherever you get podcasts   Download | 21.9 MB 23:04   Links The Morley-Montgomery Award episode series (Patreon | Substack) All of our social links: https://linktr.ee/ihearofsherlock Email us at trifles @ ihearofsherlock.com    Music credits Performers: Uncredited violinist, US Marine Chamber Orchestra Publisher Info.: Washington, DC: United States Marine Band. Copyright: Creative Commons Attribution 3.0      

In 1991, the quiet Cotswold town of Tetbury became the centre of a devastating double murder involving family, violence, mental illness, and a confession that stunned police…*** LISTENER CAUTION IS ADVISED *** This episode was researched and written by Eileen Macfarlane.Script editing, additional writing, illustrations and production direction by Rosanna Fitton.Audio editing by Joel Porter at Dot Dot Dot Productions.Narration, additional audio editing and mixing, and script editing by Benjamin Fitton.To get early ad-free access, including Season 1, sign up for They Walk Among PLUS, available from Patreon or Apple Podcasts.More information and episode references can be found on our website https://theywalkamonguspodcast.comSOCIAL MEDIA: https://linktr.ee/TheyWalkAmongUsSupport this show http://supporter.acast.com/theywalkamongus. Hosted on Acast. See acast.com/privacy for more information.

This episode continues the Heal NPD Seminar Series with Dr. Mark Ettensohn, joined by his associates Deanna Young, Psy.D., and Danté Spencer, Ph.D. In this session, the group discusses the paper “Living with Pathological Narcissism: A Qualitative Study” (Day et al., 2020), which examines narcissistic personality pathology from the perspective of partners and family members. Unlike most research on narcissism, this study does not rely on self-report or clinician ratings. Instead, it draws on qualitative descriptions from over 400 individuals in close relationships with someone exhibiting high levels of pathological narcissistic traits. These accounts provide a window into how narcissism is experienced interpersonally, particularly in intimate and long-term relationships. The discussion focuses on the study's central finding: that pathological narcissism is best understood as a system characterized by the co-occurrence of grandiosity and vulnerability. Loved ones described patterns of entitlement, arrogance, and need for admiration alongside insecurity, hypersensitivity, emotional instability, and chronic feelings of emptiness. In the majority of cases, these features were not separate “types,” but fluctuating states within the same individual. The group explores how these findings challenge common assumptions about narcissism, including the tendency to equate it with overt grandiosity or interpersonal abusiveness. Particular attention is given to the limitations of DSM-based models, which emphasize observable traits while underrepresenting the internal dysregulation and vulnerability that define the disorder. The conversation also examines broader relational and developmental themes, including: The oscillation between grandiose and vulnerable self-states The role of dissociation and splitting in personality organization The impact of early attachment trauma and “empathic failures” How narcissistic dynamics are expressed and amplified within close relationships The tendency for polarized, dehumanizing narratives to emerge in response to relational injury Finally, the group discusses the concept of “narcissistic abuse,” noting that while experiences of harm in these relationships are real and often significant, the term itself is not a well-defined clinical construct. The discussion emphasizes the importance of distinguishing between lived experience and explanatory frameworks, and of maintaining a nuanced, non-reductive understanding of personality pathology. Key themes include: Pathological narcissism as a dysregulated self-state system The interdependence of grandiosity and vulnerability Limitations of categorical and trait-based models of narcissism The relational expression of personality pathology The role of trauma, attachment, and development in narcissistic adaptation Clinical implications for assessment, formulation, and treatment This series is intended for clinicians, trainees, and viewers seeking a nuanced, clinically grounded understanding of narcissism beyond popular discourse. To learn more about our work, visit: www.HealNPD.org Additional Resources: Newsletter: https://healnpd.substack.com Assessment and therapy inquiries: https://healnpd.org/contact Purchase Unmasking Narcissism: A Guide to Understanding the Narcissist in Your Life: https://amzn.to/3nG9FgH LISTEN ON APPLE PODCASTS: https://rb.gy/cklpum LISTEN ON GOOGLE PODCASTS: https://rb.gy/fotpca LISTEN ON AMAZON MUSIC: https://rb.gy/g4yzh8 Citation: Day, N. J. S., Townsend, M. L., & Grenyer, B. F. S. (2020). Living with pathological narcissism: A qualitative study. Borderline Personality Disorder and Emotion Dysregulation, 7(19).  Full Text Link: https://pmc.ncbi.nlm.nih.gov/articles... About Heal NPD Heal NPD is a clinical practice specializing in the assessment and treatment of pathological narcissism, narcissistic personality disorder, and related personality difficulties. We offer comprehensive diagnostic assessments, individual psychotherapy, and consultations for partners and family members. Learn more or inquire about services: https://healnpd.org

Open Discussion!

CME credits: 1.00 Valid until: 15-05-2027 Claim your CME credit at https://reachmd.com/programs/cme/aldosterone-targeted-therapies-uncontrolled-hypertension-chronic-kidney-disease/56405/ Chronic kidney disease (CKD) and high blood pressure (BP) are closely linked and, when not managed appropriately, can lead to serious outcomes such as myocardial infarction, stroke, heart failure, and progression to kidney failure. Pathological excess of aldosterone induces hypertension by forcing the kidneys to excessively retain sodium and water, increasing blood volume, and lowering potassium. Mineralocorticoid receptor antagonists (MRAs), which target the renin-angiotensin-aldosterone system (RAAS), do not fully inhibit the deleterious effects of aldosterone and may increase serum aldosterone concentrations. Highly selective aldosterone synthase inhibitors (ASIs) have emerged as potential alternatives to MRAs; ASIs decrease aldosterone synthesis, providing another strategy to inhibit aldosterone effects without affecting cortisol production.Want to dive deeper into the discussion? Check out Audience Q&A From the Live Symposium in the Related Content section for additional expert insights and practical perspectives on CKD, hypertension, and emerging aldosterone-targeted therapies. =

In this episode of Giant Ideas, Jimmy Wales, founder of Wikipedia, discusses how trust, purpose, and community design shaped one of the world's most used websites. He reflects on three photographs that define his life, Wikipedia's core purpose and why transparency, clear purpose, and walking the walk are central to trust.He also shares the trust framework of building trust, and why execution matters more than idea secrecy, and the real‑world costs of living in mistrust.----------------------------------------------------------------------------------------------------------------Building a purpose driven company? Read more about Giant Ventures at www.Giant.vcMusic credits: Bubble King written and produced by Cameron McLain and Stevan Cablayan aka Vector_XING.Please note: The content of this podcast is for informational and entertainment purposes only. It should not be considered financial, legal, or investment advice. Always consult a licensed professional before making any investment decisions.Building a purpose driven company? Read more about Giant Ventures at www.Giant.vc.Music credits: Bubble King written and produced by Cameron McLain and Stevan Cablayan aka Vector_XING.Please note: The content of this podcast is for informational and entertainment purposes only. It should not be considered financial, legal, or investment advice. Always consult a licensed professional before making any investment decisions.

Who Knew It with Matt Stewart is a comedy game show podcast hosted by Australian comedian Matt Stewart. Episode 191 features comedians JNaomi Higgins, Jett Archer and Josephine Bowser!Buy tickets for the 200th episode: https://tickets.oztix.com.au/outlet/event/7bb3026b-b8a8-40b8-8693-2cadee9f423cCheck out Pathological episode 1 feat. Big Wet: https://youtu.be/sdV-rKRBkTk?si=IDU8MEllQ69kBXSdSupport the show via http://patreon.com/dogoonpod and you can submit questions for the show!Check out Matt's stand up special 'Best Man': https://youtu.be/ZgukEPerWZc?si=SW8PttGAB-ly_GF8And his stand up special 'Live at Stupid Old Studios': https://youtu.be/cWStRpI-BhESee the podcast/Matt live: https://www.mattstewartcomedy.com/Check out Matt's podcast network: https://dogoonpod.com/Theme song by Evan Munro-Smith, Logo by Murray Summerville and edited by Connor Schmidt! Hosted on Acast. See acast.com/privacy for more information.

BPD Joint Therapy Fantasy BPD Right-Fighting & Pathological Lying vs Your Eggshell-WalkingBPD joint therapy doesn't work and is a fantasy bonded nightmare of Borderline victim/martyr cs your Codependent walking on eggshells and trauma bond addiction to intermittent reinforcement.Borderlines - unless clinically proven to actually be recovered need their own therapy and you need yours. BPD relationships without full BPD recovery are toxic and you're not being “loved” while being abused.BPD right-fighting and pathological lying block anything meaningful in joint therapy as they control the process initiating BPD Induced Conversation and projecting on to the therapist as they do you in life. Stop trying to recycle toxic relationships after the first breakup!https://ajmahari.ca/sessions - Sessions with A.J.https://ajmahari.ca/podcastshttps://ajmahari.com - Online Store100 Best Relationship Podcasts You Must Follow in 2025Million Podcasts has ranked this podcast in the top 60 Codependency Podcasts,the topThis podcast is ranked in the Top 100 Relationships Podcasts on feedspot.com at: 100 Narcissistic Abuse Podcasts and the top 100 in their Toxic RelationshipMillion Podcasats:https://www.millionpodcasts.com/codependency-podcasts/https://www.millionpodcasts.com/narcissistic-abuse-podcasts/https://www.millionpodcasts.com/toxic-relationship-podcasts/

What does it mean to say the world is fundamentally open for play - and why does it take something to even have to say it at all? In this episode - the third and final in a live-recorded three-part series with Ethan Hsieh, Taylor Barratt, and John Vervaeke - the conversation centers on Ethan as he unpacks the distinction between teaching and facilitation, the purpose of TIAMAT, and the deep personal why that drives his work. John maps the teacher/facilitator divide onto Aristotle's sophia and phronesis, while the group works through how theory and practice function as mutual correctives - each able to expose the other's blind spots. They examine phenomenological adequacy (how a theory can be causally sound yet fail to account for what's actually showing up in lived practice), the necessity of an ecology of practices over any single panacea, and why no closed overarching theory can substitute for genuine interdisciplinary dialogue. Ethan unpacks TIAMAT's purpose as psycho-education toward a good life - affording self-knowledge and heightened religiosity (bindedness to self, other, and world) without becoming a religion - and walks through the SPIRE framework (Service, Pilgrimage, Inquiry, Ritual, Enlightenment). The conversation deepens into the primordial nature of relationality, the actor training roots of TIAMAT, and Ethan's core conviction: that serious play - wrestling fully with what matters, using every faculty of one's being - is the most human way to stay genuinely coupled to a reality that always exceeds our grasp. The episode closes on joy: not pleasure, not comfort, but contact. Ethan Hsieh is the Director of Community Development and Partnerships at the Vervaeke Foundation. He comes from an acting background focused on character development. LinkedIn Taylor Barratt is the Director of Practice and Education at the Vervaeke Foundation. He has over a decade of experience in relational leadership through Authentic Relating Toronto. LinkedIn X 00:00 Welcome to the Lectern 01:30 Introducing Ethan - the third and final session 03:00 Teaching vs. facilitation - the core distinction 04:20 The knowing-doing and being-becoming questions 06:30 What truly distinguishes a teacher from a facilitator? 08:00 Responsibility, longitudinal tracking, and development 09:00 Training containers vs. drop-in practice 11:10 Sophia and phronesis - Aristotle on wisdom 12:30 Self-correction and attachment to theory or practice 14:10 Adaptive fit vs. adaptive transfer 17:30 When to bring theory in as a leader 20:00 Theory as legitimation of practice 22:00 Does practice challenge theory? Practice as research 24:00 Phenomenological adequacy - what theory can miss 26:00 Being too precious about theory or practice 27:00 Voice work and the emotional dimension as data 28:30 Deficit, excess, and the normativity of practice 30:30 Ecology of practices as pedagogical design 32:20 Why there's no closed theoretical system 33:00 Why there's no panacea discipline 35:00 TIAMAT as a living, evolving system 35:50 Predictive processing, CBT, and Jungian thought 36:30 Propositional knowledge must afford participation 38:10 What's ours to do? Defining scope of practice 41:20 What is TIAMAT actually for? 43:00 Pathological vs. positive psychology 46:10 TIAMAT: psycho-education for a good life 47:00 Religiosity without religion 48:30 SPIRE - Service, Pilgrimage, Inquiry, Ritual, Enlightenment 49:30 Enriching religio and relationship 50:20 Relationality is primordial - all of it is real 52:00 Depersonalization and the world-as-instrument trap 54:00 Why Taylor does this work 56:40 "The world is open for play" 58:00 Joy as good 59:00 Serious play as anamnesis - recovering what was forgotten 01:00:00 Joy vs. pleasure - genuine coupling to reality 01:01:00 Daoism, Zen, and the blurry line with philosophy 01:02:00 Actor training as the origin of TIAMAT 01:03:30 Anger and sadness at unnecessary suffering 01:08:30 "Why do I have to tell you that you matter?" 01:10:00 Holding the suchness of where someone is 01:11:10 Joy as developing relationship - closing thoughts The Vervaeke Foundation is committed to advancing the scientific pursuit of wisdom and creating a significant impact on the world. Become a part of our mission. Join Awaken to Meaning to explore practices that enhance your virtues and foster deeper connections with reality and relationships. Follow John Vervaeke: Website | Twitter | YouTube | Patreon Thank you for listening!

Read the full article at: https://oncdata.com/ai-driven-pathological-subtyping-pancreatic-cancer-ashish-manne Artificial intelligence (AI) continues to generate excitement across oncology, but its real-world clinical impact remains uneven. In this episode of Exploring AI in Oncology, Dr. Ashish Manne, a Gastrointestinal Medical Oncologist at The Ohio State University Comprehensive Cancer Center, Arthur G. James Cancer Hospital and Richard J. Solove Research Institute, shares his American Association for Cancer Research (AACR) presentation on AI-driven pathological subtyping in pancreatic cancer and highlights a critical shift: moving AI from theoretical promise to practical application.

Zammit is missing, presumed dead. We can only assume that all his bad decisions have finally caught up with him. But never fear as the beautiful and normal hosts are joined by the beautiful and normal Naomi Higgins. Reality TV is pop culture, we dare you to say otherwise and today the gang try and figure out the best way to win Big Brother. They also get into the nitty gritty of Big Brother Up Late, decide what counts as an act of terrorism, and explore how Duscher requires ego death if he's to even have a chance of meeting Gretel Killeen. So listen on, enjoy and do your best to avoid intruders Frank and Oz who wish to do you some serious harm.Go listen to Naomi's podcast 3 Lemons Carcass Out wherever you listen to podcasts. It really is the perfect pop culture podcast. And check out her new show Pathological over at Humdinger Studios on YouTube starting May 7th.Links to everything at https://linktr.ee/plumbingthedeathstar including our terrible merch, social media garbage and where to become a subscriber to Bad Brain Boys+ Hosted on Acast. See acast.com/privacy for more information.

Dan speaks to Aydin Paladin about why Mass democracy is dangerous and stupid, much like the voters. 

Welcome to Light ‘Em Up!At the conclusion of the editing and postproduction process for this episode in preparation for its upload and debut - DHS Secretary Kristi Noem was fired from her role as the Secretary of the Department of Homeland Security.Tonight, on this brand-new, intense episode we'll investigate and examine the more than 32 deaths that have happened at the hands of Immigration & Customs Enforcement (ICE).It may come as a surprise, but the killing of Renee Good (a mother of three) and Alex Pretti (an ICU nurse at the Minneapolis Veterans Affairs Health Care System Hospital in Minnesota) were not the first and they won't be the last.We'll drill down on the Department of Homeland Security — an organization led by individuals who lack both qualifications and integrity.This witch's brew often forces an organization — whether desired or not -- to eventually face severe consequences, ranging from operational inefficiencies to catastrophic, fatal incidents.Research indicates that when leadership prioritizes self-interest over competence and honesty, it creates a toxic culture that undermines safety and performance. ICE has made our streets unsafe with its tyrannical tactics, terrorizing our own people in the name of Trump.Since returning to office in 2025, President Trump has implemented a "hardline" and "maximalist" immigration policy focused on executing the largest domestic deportation operation in U.S. history.The administration doesn't care how the policy is carried out — ONLY that it is — so that Stephen Miller can satisfy his own sick, sadistic wishes.Miller is credited with shaping the racist and draconian immigration policies of President Trump, and Noem oversaw carrying those decisions out, which include:—   zero-tolerance policy, also known as family separation,—   the Muslim ban and—   ending the Deferred Action for Childhood Arrivals (DACA) program.As we go to air with this episode, a federal judge accuses the White House of ‘terror' against immigrants in the U.S.The administration has pursued a whole-of-government approach to identify, detain, and remove undocumented immigrants, aiming for a "1 million per year" deportation goal.But, in the process, there've been:—  tremendous oversights,—  poor procedural safeguards and protocols put in place,—  very little to no training for new hires,—  negligence and outright lies which have led to more than 32 people's death across the nation.We'll introduce you to those who have lost their lives at the hands of the governmental “political theater” that is unfolding right in front of our own eyes, daily.We will report that the (former) DHS Secretary, Kristi Noem is an outright liar — as is her boss.  That isn't opinion — that is fact and we have the receipts to prove it.Be sure to tune in for our reporting regarding a completely fabricated story — that Noem stated in one of her press briefings to the nation — about a man in deportation proceedings “cannibalizing” others, then, himself, like the fictitious character Hannibal Lecter, who Trump thinks is a real person.As we speak truth to power, we never cower — as we expose the fact that Secretary Noem has deported U.S. military veterans AND U.S. citizens and lied and covered up about that as well.As a painter paints and a singer sings, a liar lies.The streets of the U.S. are safer with Noem ousted at the top of DHS, but the man tapped to be her replacement isn't much of an improvement.Tune in for this episode and our sponsors Newsly & Feedspot!We want to hear from you!Support the show

Send a textIn this episode, we sit down with Marley to hear her two very different birth stories and how they shaped her journey in the motherhood space. Marley is a doula, birth educator, nanny, and babysitter who continues to support families through pregnancy, birth, and early parenting.Her first pregnancy followed the cascade of interventions, ultimately leading to an instrumental birth. During her second pregnancy, Marley planned a home birth. However, after discovering her baby was breech and having open discussions with her midwife and hospital team, weighing up the information available to her at the time, she chose to have a caesarean birth. She proactively wrote to the hospital outlining her wishes and the context surrounding her decision, advocating for a respectful and supported experience.Her second baby was admitted to NICU with pathological jaundice, and Marley also navigated recovery from a caesarean infection.Support the show@homebirthstoriesaustralia Support the show by buying us a coffee! Please be advised that this podcast may contain explicit language. Listener discretion is advised.The information, statistics, and research presented in this podcast are for informational purposes only and are not intended to constitute or replace medical or midwifery advice. All information discussed can be found online and is provided in the links in the show notes. It is always recommended to conduct your own research and make informed decisions. We advise you to discuss any topics or concerns with your healthcare provider. While we strive to incorporate the most up-to-date research in our episodes, we do not warrant or guarantee the accuracy of the information discussed on the show.

Will Tom Brady be the modern-day Bugsy Siegel in Las Vegas? Being a great player is no guarantee of future success as a coach or executive. Michael Jordan, a majority owner of the Charlotte Bobcats/Hornets, and Wayne Gretzky, behind the bench for the Phoenix Coyotes, were classic examples. Jason questions whether Brady, a minority owner of the Las Vegas Raiders, will follow suit as the sometimes not-so-stealthy leader of the team. The Raiders imploded last season with a new head coach and quarterback and now are on the verge of losing the face of the franchise, Maxx Crosby, while beginning anew with a different head coach and quarterback. Steve Kim and Jay Skapinac join the conversation, responding to Stephen A. Smith explaining how he moved on up from the mean streets; Kendrick Perkins claiming that a title will elevate Jalen Brunson to the greatest player in New York Knicks history; and Fight Night at a women's college basketball game. Shaun King shares his takes on Logan Paul offering $1 million to any NFL player who can beat him in combat; the A's telling Kyler Murray he can come home; and the Los Angeles Rams sending a bevy of draft picks to Kansas City for premium cornerback Trent McDuffie. ​​Today's Sponsors: PreBorn PreBorn has helped rescue more than 400,000 babies, and every single day, they continue that work by offering mothers something powerful and life-changing: an ultrasound. Will you help us? Just dial #250 and say the keyword “BABY” or donate securely at https://Preborn.com/FEARLESS  ➢ Subscribe Jason's other channel https://www.youtube.com/@JasonWhitlockHarmony  https://www.youtube.com/@JasonWhitlockBYOG  ➢ Connect with Jason on Social Media:  https://x.com/WhitlockJason https://www.instagram.com/realjasonwhitlock/ https://www.facebook.com/jasonwhitlock ➢ Send Jason an Email FearlessBlazeShow@gmail.com ➢ Support The Blaze Visit https://TheBlaze.com. Explore the all-new ad-free experience and see for yourself how we're standing up against suppression and prioritizing independent journalism. Support Conservative Voices! Subscribe to BlazeTV at https://www.fearlessmission.com and get $20 off your yearly subscription. Learn more about your ad choices. Visit megaphone.fm/adchoices

Episode 212: Managing HFpEFHyo Mun and Jordan Redden (medical students) explain how to manage HFpEF with medications and touch some basics about nonpharmacologic treatments. Dr. Arreaza asks insightful questions to guide the discussion. Written by Hyo Mun, MSIV, American University of the Caribbean; and Jordan Redden, MSIV, Ross University School of Medicine. Comments by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice.Treatment of HFpEFArreaza: Mike, if you had to name the one therapy everyone with HFpEF should be on, what is it?Mike: That's easy! SGLT-2 inhibitors. This is the one slam-dunk we have in HFpEF. Empagliflozin (Jardiance) or dapagliflozin (Farxiga) should be started in essentially every patient with HFpEF, and it doesn't matter if they have diabetes or not.Jordan: And that's worth repeating, because people still think of these as “diabetes drugs.” They're not anymore. In HFpEF, SGLT-2 inhibitors reduce heart-failure hospitalizations, improve symptoms, improve quality of life, and even reduce cardiovascular death.Dr. Arreaza: They're also simple. Empagliflozin 10 mg daily or dapagliflozin 10 mg daily. No titration, no drama. The effectiveness of these meds was established around 2019 with DAPA-HF and later with DELIVER. These were trials thatdemonstrated that dapagliflozin reduces worsening heart failure and cardiovascular events across the full spectrum of heart failure, from reduced to preserved ejection fraction, independent of diabetes status.Mike: And the number needed to treat is about 28 to prevent one heart-failure hospitalization. That's excellent for a disease where we historically had almost nothing that worked.Jordan: They're also safe in chronic kidney disease down to an eGFR of about 25, which makes them even more useful in this population.Dr. Arreaza: Alright. We got SGLT-2 inhibitor, what's next?Mike: Volume management. Loop diuretics are still the backbone of symptom control in HFpEF. If the patient is volume overloaded, you diurese, and you diurese aggressively.Jordan: The goal is euvolemia. Dry weight, no edema, no orthopnea, no waking up gasping for air. A lot of these patients end up needing chronic oral loop diuretics to stay there.Dr. Arreaza: Something to remember: HFpEF patients don't tolerate congestion well, and being “a little wet” is not benign. Let's move into RAAS inhibition. Where do ARBs and ACE inhibitors fit in?Mike: Between ARBs and ACE inhibitors, ARBs are the winners in HFpEF. They actually reduce heart failure hospitalizations—drugs like candesartan, losartan, valsartan. ACE inhibitors? Not so much. They showed minimal benefit in older HFpEF patients, which is why we go with ARBs instead.Jordan: But a lot of clinicians get nervous about ACE inhibitors and ARBs because of kidney function, so it's worth talking through how these drugs actually work in the kidney.Dr. Arreaza: Yes, misunderstanding may lead to unnecessary drug discontinuation.Jordan: Under normal conditions, the afferent arteriole brings blood into the glomerulus, and the efferent arteriole is constricted by angiotensin II. That constriction keeps pressure high in the glomerulus and maintains filtration.Mike: Here's what happens with an ACE inhibitor: you block angiotensin II, the efferent arteriole relaxes, glomerular pressure drops, and GFR dips slightly. Creatinine bumps up a little, and that scares people, but that's actually the whole point—that's how you get kidney protection long-term.Jordan: High intraglomerular pressure causes hyperfiltration injury and scarring over time. Lowering that pressure protects the kidney long-term. The short-term GFR drop is the price you pay for long-term benefits.Dr. Arreaza: So let's talk about CKD, because this is where people panic.Mike: Right. ACE inhibitors and ARBs are not contraindicated in chronic kidney disease. In fact, they're recommended even in advanced stages. They reduce progression to kidney failure by about a third.Jordan: The key is how you use them. Start low. Check creatinine and potassium one to two weeks after starting, then periodically. A creatinine rise up to 30% from baseline is acceptable. That's not kidney injury, that's physiology.Dr. Arreaza: And what about potassium creeping up?Mike: You adjust the dose or add a potassium binder. You don't just automatically stop the drug.Dr. Arreaza: Now there is one absolute contraindication everyone needs to know about! (board exam test)Jordan: Bilateral renal artery stenosis. This is the big one. In these patients, the kidneys are completely dependent on angiotensin II–mediated efferent constriction to maintain GFR. Take that away, and GFR collapses.Mike: Creatinine can jump dramatically within days. If you see a creatinine rise of 20% or more shortly after starting an ACE inhibitor, you should be thinking about bilateral renal artery stenosis and stopping the drug immediately.Dr. Arreaza: After revascularization, though, many patients can tolerate ACE inhibitors again, so this isn't always permanent. What about cardiorenal syndrome? That's where things get uncomfortable.Mike: It is uncomfortable, but cardiorenal syndrome isn't a contraindication. These patients have severe heart failure and kidney disease, and their mortality is actually higher than patients with heart failure alone.Jordan: ACE inhibitors still reduce mortality and slow kidney disease progression in this group. Studies show that stopping ACE inhibitors during acute heart-failure admissions increases in-hospital mortality three- to four-fold.Dr. Arreaza: So we are cautious, but we don't avoid it.Mike: Exactly. Start low, titrate slowly, monitor labs closely, accept up to a 30% creatinine rise. You only stop if kidney function keeps worsening, or potassium gets dangerously high.Dr. Arreaza: Alright. Let's move on. What about mineralocorticoid receptor antagonists… MRA?Jordan: Spironolactone or eplerenone might reduce hospitalizations in HFpEF, but the data is mixed. This is more of a “select patients” situation.Mike: And you have to watch potassium and kidney function carefully, especially if they're already on an ACE inhibitor or ARB.Dr. Arreaza: What about sacubitril-valsartan, also known as Entresto®?Mike: Entresto may help patients with mildly reduced EF roughly in the 45 to 57% range. It's not first-line for HFpEF, but in select patients, it's reasonable.Dr. Arreaza: Now let's clarify one of the biggest sources of confusion: beta blockers.Jordan: Beta blockers are not a treatment for HFpEF itself. They're only indicated if the patient has another reason to be on them, like coronary disease or atrial fibrillation.Mike: And timing really matters here. You absolutely do not start beta blockers during acute decompensated heart failure. Their negative inotropic effects can make things worse when patients are volume overloaded.Jordan: But, and this is critical, you also don't stop them if the patient is already taking one. Abrupt withdrawal causes a sympathetic surge and dramatically increases mortality.Dr. Arreaza: If a patient is admitted on a beta blocker, what do we do?Mike: Continue it at the same dose or reduce it slightly if they're really unstable. Once they're euvolemic and stable, you can carefully titrate up.Jordan: And watch for chronotropic incompetence. HFpEF patients often rely on heart-rate response to exercise, and beta blockers can worsen exercise intolerance.Dr. Arreaza: Beyond medications, HFpEF is really about treating comorbidities. Aerobic activity can be an initial strategy to improve exercise intolerance and has evidence of improving aerobic function and quality of life. Sodium restriction: improves symptoms, does not decrease risk of death or hospitalizations.Mike: Hypertension control is huge. For diabetes, the SGLT-2 inhibitors will perform double duty. For obesity, weight loss improves symptoms, and GLP-1 agonists like semaglutide are absolute gamechangers.Jordan: Don't forget sleep apnea, atrial fibrillation, and lifestyle. Exercise improves the quality of life, even if it doesn't change hard outcomes. Lifestyle is the main treatment. Dr. Arreaza: And when should you refer to cardiology?Mike: You should refer when the diagnosis isn't clear; symptoms are not responding to treatment, difficult volume management, end-organ dysfunction, or if you are concerned about advanced heart failure.Dr. Arreaza: So, it has been a great discussion. What is the takeaway?Mike: HFpEF treatment isn't about one magic drug -- it's about volume control, SGLT2 inhibitors, smart use of RAAS blockade, and aggressive management of comorbidities.Jordan: And it's understanding the physiology, so you don't withhold life-saving therapies out of fear.Dr. Arreaza: Well said. If you found this helpful, share it with a friend or colleague and rate us wherever you listen. This is Dr. Arreaza, signing off.Jordan/Mike: Thanks! Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! _____________________References:Barzin A, Barnhouse KK, Kane SF. Heart Failure With Preserved Ejection Fraction. Am Fam Physician. 2025;112(4):435-440.Heidenreich PA, Bozkurt B, Aguilar D, et al. 2022 AHA/ACC/HFSA guideline for the management of heart failure. Circulation. 2022;145(18):e895-e1032.Kittleson MM, Panjrath GS, Amancherla K, et al. 2023 ACC expert consensus decision pathway on management of heart failure with preserved ejection fraction. J Am Coll Cardiol. 2023;81(18):1835-1878.Anker SD, Butler J, Filippatos G, et al. Empagliflozin in heart failure with a preserved ejection fraction. N Engl J Med. 2021;385(16):1451-1461.Solomon SD, McMurray JJV, Claggett B, et al. Dapagliflozin in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2022;387(12):1089-1098.Pitt B, Pfeffer MA, Assmann SF, et al. Spironolactone for heart failure with preserved ejection fraction. N Engl J Med. 2014;370(15):1383-1392.Yusuf S, Pfeffer MA, Swedberg K, et al. Effects of candesartan in patients with chronic heart failure and preserved left-ventricular ejection fraction. Lancet. 2003;362(9386):777-781.Solomon SD, McMurray JJV, Anand IS, et al. Angiotensin-neprilysin inhibition in heart failure with preserved ejection fraction. N Engl J Med. 2019;381(17):1609-1620.Kosiborod MN, Abildstrøm SZ, Borlaug BA, et al. Semaglutide in patients with heart failure with preserved ejection fraction and obesity. N Engl J Med. 2023;389(12):1069-1084.Xie Y, Xu E, Bowe B, Al-Aly Z. Long-term cardiovascular outcomes of COVID-19. Nat Med. 2022;28(3):583-590.Puntmann VO, Carerj ML, Wieters I, et al. Outcomes of cardiovascular magnetic resonance imaging in patients recently recovered from COVID-19. JAMA Cardiol. 2020;5(11):1265-1273.Basso C, Leone O, Rizzo S, et al. Pathological features of COVID-19-associated myocardial injury. Eur Heart J. 2020;41(39):3827-3835.Nalbandian A, Sehgal K, Gupta A, et al. Post-acute COVID-19 syndrome. Nat Med. 2021;27(4):601-615.Badve SV, Roberts MA, Hawley CM, et al. Effects of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers in adults with estimated GFR less than 60 mL/min per 1.73 m². Ann Intern Med. 2024;177(8):953-963.Navis G, Faber HJ, de Zeeuw D, de Jong PE. ACE inhibitors and the kidney: a risk-benefit assessment. Drug Saf. 1996;15(3):200-211.Textor SC, Novick AC, Tarazi RC, et al. Critical perfusion pressure for renal function in patients with bilateral atherosclerotic renal vascular disease. Ann Intern Med. 1985;102(3):308-314.Hackam DG, Spence JD, Garg AX, Textor SC. Role of renin-angiotensin system blockade in atherosclerotic renal artery stenosis and renovascular hypertension. Hypertension. 2007;50(6):998-1003.Ronco C, Haapio M, House AA, et al. Cardiorenal syndrome. J Am Coll Cardiol. 2008;52(19):1527-1539.Prins KW, Neill JM, Tyler JO, et al. Effects of beta-blocker withdrawal in acute decompensated heart failure. JACC Heart Fail. 2015;3(8):647-653.Jondeau G, Neuder Y, Eicher JC, et al. B-CONVINCED: Beta-blocker CONtinuation Vs. INterruption in patients with Congestive heart failure hospitalizED for a decompensation episode. Eur Heart J. 2009;30(18):2186-2192.Theme song, Works All The Time by Dominik Schwarzer, YouTube ID: CUBDNERZU8HXUHBS, purchased from https://www.premiumbeat.com/.

Episode 211: Understanding HFpEF.  Hyo Mun and Jordan Redden (medical students) explain the pathophysiology of heart failure with preserved ejection fraction (HFpEF) and how it differentiates from HFrEF. Dr. Arreaza asks insightful questions and summarizes some key elements of HFpEF. Written by Hyo Mun, MS4, American University of the Caribbean; and Jordan Redden, MS4, Ross University School of Medicine. Comments and edits by Hector Arreaza, MD.You are listening to Rio Bravo qWeek Podcast, your weekly dose of knowledge brought to you by the Rio Bravo Family Medicine Residency Program from Bakersfield, California, a UCLA-affiliated program sponsored by Clinica Sierra Vista, Let Us Be Your Healthcare Home. This podcast was created for educational purposes only. Visit your primary care provider for additional medical advice.What is EF? Just imagine, the heart is a pump, blood gets into the heart through the veins, the ventricles fill up and then squeeze the blood out. So, the percent of blood that is pumped out is the EF. Let's start at the beginning. What is HFpEF?Mike: HFpEF stands for heart failure with preserved ejection fraction. Basically, these patients squeeze normally—their ejection fraction is 50% or higher—but here's the thing: the heart can't relax and fill the way it should. The muscle gets stiff, almost like a thick leather boot that just won't stretch. And because the ventricle can't fill properly, pressure starts backing up into the lungs and the rest of the body. That's when patients start experiencing shortness of breath, leg swelling, fatigue—all those classic symptoms.Dr. Arreaza: And this is where people get fooled by the ejection fraction.Mike: Exactly. The ejectionfraction tells you total left ventricular emptying, not just forward flow.Jordan: The classic example is severe mitral regurgitation. You can eject 60% of your blood volume and still be in cardiogenic shock because most of that blood is leaking backward into the left atrium instead of going into the aorta. So, you get pulmonary edema, hypotension, fatigue, all with a “normal” EF. Which is honestly terrifying if you're over-relying on echo reports without thinking clinically.Dr. Arreaza: And in HFpEF, functional mitral regurgitation often shows up later in the disease. It's not usually the primary cause; it's more of a marker of advanced disease. Moderate to severe MR in HFpEF independently predicts worse outcomes, including a higher risk of mortality or heart failure hospitalization. So, let's contrast this with HFrEF. How are these two different?Mike: HFrEF—heart failure with reduced ejection fraction—is a pumping problem. The heart muscle is weak and can't contracteffectively. Ejection fraction drops below 40%, and this is your classic systolic dysfunction.Jordan: HFpEF, on the other hand, is diastolic dysfunction. The heart muscle is thick, fibrotic, and noncompliant. It squeezes fine, but it just doesn't relax, even though the EF looks reassuring on paper.Mike: I like to explain it this way: HFrEF is a weak heart that can't squeeze. HFpEF is a stiff heart that can't relax. Totally different problems.Dr. Arreaza: And then there's the gray zone: heart failure with mildly reduced EF, or HFmrEF. That's an EF between 41 and 49% with evidence of elevated filling pressures. It really shares the features of both worlds. So, what actually causes HFpEF versus HFrEF?Jordan: HFpEF is basically what happens when all the problems of modern living catch up with you. You've got chronic hypertension, obesity, diabetes, metabolic syndrome, aging, systemic inflammation—all of these things slowly remodel the heart over years. The muscle gets thick and stiff, and eventually the ventricle just loses its ability to relax. So, HFpEF is really a disease of metabolic dysfunction and chronic stress in the heart. Mike: HFrEF is more about direct injury. Think about myocardial infarctions, ischemic cardiomyopathy, viral myocarditis, alcohol toxicity, chemotherapy like doxorubicin, genetic cardiomyopathies, or chronic uncontrolled tachycardia. These insults actually damage or kill heart muscle cells, leading to a dilated, weak ventricle that can't pump effectively.Dr. Arreaza: So the short version: HFpEF is caused by chronic metabolic and hypertensive stress, while HFrEF is caused mainly by myocardial damage. A question we get a lot: does HFpEF eventually turn into HFrEF? What do you guys think?Mike: In most cases, no. HFpEF patients usually stay HFpEF throughout their disease course. They don't just “burn out” and turn into HFrEF.Jordan: They're generally separate disease entities with different pathophysiology. A patient with HFpEF can develop HFrEF if they have a big myocardial infarction or ongoing ischemia that damages the muscle, but that's not the natural progression.Mike: Interestingly though, the opposite can happen. Some HFrEF patients actually improve their ejection fraction with good medical therapy—that's called HF with improved EF—and it's a great sign that treatment is working.Dr. Arreaza: Another question. How do HFpEF and HFrEF compare to restrictive cardiomyopathy and constrictive pericarditis?Jordan: Clinically, they can all look very similar: dyspnea, edema, fatigue, but the underlying mechanisms are completely different.Mike: In HFpEF, the myocardium itself is stiff from hypertrophy and fibrosis. The problem is intrinsic to the heart muscle, and EF stays preserved. Echoshows diastolic dysfunction with elevated filling pressures.Jordan: In HFrEF, the myocardium is weak. The ventricle is often dilated and contracts poorly, with a reduced EF.Mike: Restrictive cardiomyopathy is different. Here, the myocardium gets infiltrated by abnormal stuff—amyloid, iron, sarcoid—and that makes it extremely stiff. It can look like HFpEF on the surface, but it's usually more severe. On Echo You'll see biatrial enlargement, small ventricles, and preserved EF. And importantly, it's a pathologic diagnosis, so you need advanced imaging or biopsy to confirm it.Jordan: Constrictive pericarditis is another mimic, but here the myocardium is usually normal. The problem is that the pericardium is thickened, calcified, and rigid. This will physically prevent the heart from being filled. Imaging shows pericardial thickening, septal bounce, and respiratory variation in flow, and cath shows equalization of diastolic pressures, which is the hallmark of constrictive pericarditis.Dr. Arreaza: So the takeaway is: HFpEF is a clinical syndrome driven by common metabolic and hypertensive causes, while restrictive and constrictive diseases are specific pathologic entities. If “HFpEF” is unusually severe or not responding to treatment, you need to think beyond HFpEF. Which type of heart failure is more common right now?Mike: Good question, the answer is: HFpEF. It now accounts for up to 60% of all heart failure cases, and it's still rising.Dr. Arreaza: Why is that?Jordan: Because people are living longer, gaining weight, and developing more metabolic syndrome. HFpEF thrives in older, or people with obesity, hypertension, or diabetes: basically, the modern American population. At the same time, better treatment of acute MIs means fewer people are developing HFrEF from massive heart attacks.Mike: HFpEF is the heart failure epidemic of the 21st century. It's honestly the cardiology equivalent of type 2 diabetes.Dr. Arreaza: Let's talk aboutCOVID-19. (2025 and still talking about it) Does it actually increase heart failure risk?Mike: Yes, absolutely. COVID increases both acute and long-term heart failure risk.Jordan: During acute infection, COVID can cause myocarditis, trigger massive inflammation, and precipitate acute decompensated heart failure, especially in patients with pre-existing disease. It also causes microthrombi, which can injure the myocardium.Mike: And after infection, even mild cases are linked to a significantly higher risk of developing new heart failure within the following year. Both HFpEF and HFrEF rates go up.Dr. Arreaza: I remember seeing this in 2021, we had a patient with acute COVID and HFrEF, her EF was about 10%, I lost contact with the patient and at the end I don't know what happened to her. What's the pathophysiology of COVID and heart failure?Mike: COVID causes direct viral injury through ACE2 receptors, triggers massive inflammation that damages the endothelium and heart muscle, leads to microvascular clotting and fibrosis—all mechanisms that promote HFpEF.Jordan: Add autonomic dysfunction, persistent low-grade inflammation, and worsening metabolic syndrome, and you've got a perfect storm for heart failure.Dr. Arreaza: Bottom line: COVID is a cardiovascular disease as much as a respiratory one. If someone had COVID and now has unexplained dyspnea or fatigue, think about heart failure. Get an echo, get a BNP, start treatment. Last big question: why did we have so many therapies for HFrEF but essentially none for HFpEF for years?Mike: HFrEF is mechanistically straightforward. You've got a weak heart with excessive neurohormonal activation going on — so you block RAAS, block the sympathetic system, drop the afterload. The drugs make sense.Jordan: HFpEF is messy. It's not one disease. It's stiffness, fibrosis, inflammation, microvascular dysfunction, metabolic disease, atrial fibrillation, all overlapping. One drug can't fix all of that.Mike: And some drugs that worked beautifully in HFrEF actually made HFpEF worse. Take Beta blockers, for example.  They slow heart rate, which is a problem because HFpEF patients rely on heart rate to maintain their cardiac output.Jordan: The breakthrough came with SGLT-2 inhibitors: diabetes drugs that unexpectedly addressed multiple HFpEF mechanisms at once: volume, metabolism, inflammation, and myocardial energetics.Dr. Arreaza: The miracle drug for HFpEF! Alright, let's wrap up.Mike: Bottom line: HFpEF is common, complex, and dangerous: even if the EF looks “normal.”Jordan: And if you're relying on ejection fraction alone, HFpEF will humble you every time.Dr. Arreaza: If you liked this episode, share it with a friend or a colleague and rate us wherever you listen. This is Dr. Arreaza, signing off.Even without trying, every night you go to bed a little wiser. Thanks for listening to Rio Bravo qWeek Podcast. We want to hear from you, send us an email at RioBravoqWeek@clinicasierravista.org, or visit our website riobravofmrp.org/qweek. See you next week! _____________________References:Barzin A, Barnhouse KK, Kane SF. Heart Failure With Preserved Ejection Fraction. Am Fam Physician. 2025;112(4):435-440.Heidenreich PA, Bozkurt B, Aguilar D, et al. 2022 AHA/ACC/HFSA guideline for the management of heart failure. Circulation. 2022;145(18):e895-e1032.Kittleson MM, Panjrath GS, Amancherla K, et al. 2023 ACC expert consensus decision pathway on management of heart failure with preserved ejection fraction. J Am Coll Cardiol. 2023;81(18):1835-1878.Anker SD, Butler J, Filippatos G, et al. Empagliflozin in heart failure with a preserved ejection fraction. N Engl J Med. 2021;385(16):1451-1461.Solomon SD, McMurray JJV, Claggett B, et al. Dapagliflozin in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2022;387(12):1089-1098.Pitt B, Pfeffer MA, Assmann SF, et al. Spironolactone for heart failure with preserved ejection fraction. N Engl J Med. 2014;370(15):1383-1392.Yusuf S, Pfeffer MA, Swedberg K, et al. Effects of candesartan in patients with chronic heart failure and preserved left-ventricular ejection fraction. Lancet. 2003;362(9386):777-781.Solomon SD, McMurray JJV, Anand IS, et al. Angiotensin-neprilysin inhibition in heart failure with preserved ejection fraction. N Engl J Med. 2019;381(17):1609-1620.Kosiborod MN, Abildstrøm SZ, Borlaug BA, et al. Semaglutide in patients with heart failure with preserved ejection fraction and obesity. N Engl J Med. 2023;389(12):1069-1084.Xie Y, Xu E, Bowe B, Al-Aly Z. Long-term cardiovascular outcomes of COVID-19. Nat Med. 2022;28(3):583-590.Puntmann VO, Carerj ML, Wieters I, et al. Outcomes of cardiovascular magnetic resonance imaging in patients recently recovered from COVID-19. JAMA Cardiol. 2020;5(11):1265-1273.Basso C, Leone O, Rizzo S, et al. Pathological features of COVID-19-associated myocardial injury. Eur Heart J. 2020;41(39):3827-3835.Nalbandian A, Sehgal K, Gupta A, et al. Post-acute COVID-19 syndrome. Nat Med. 2021;27(4):601-615.Badve SV, Roberts MA, Hawley CM, et al. Effects of angiotensin-converting enzyme inhibitors and angiotensin receptor blockers in adults with estimated GFR less than 60 mL/min per 1.73 m². Ann Intern Med. 2024;177(8):953-963.Navis G, Faber HJ, de Zeeuw D, de Jong PE. ACE inhibitors and the kidney: a risk-benefit assessment. Drug Saf. 1996;15(3):200-211.Textor SC, Novick AC, Tarazi RC, et al. Critical perfusion pressure for renal function in patients with bilateral atherosclerotic renal vascular disease. Ann Intern Med. 1985;102(3):308-314.Hackam DG, Spence JD, Garg AX, Textor SC. Role of renin-angiotensin system blockade in atherosclerotic renal artery stenosis and renovascular hypertension. Hypertension. 2007;50(6):998-1003.Ronco C, Haapio M, House AA, et al. Cardiorenal syndrome. J Am Coll Cardiol. 2008;52(19):1527-1539.Prins KW, Neill JM, Tyler JO, et al. Effects of beta-blocker withdrawal in acute decompensated heart failure. JACC Heart Fail. 2015;3(8):647-653.Jondeau G, Neuder Y, Eicher JC, et al. B-CONVINCED: Beta-blocker CONtinuation Vs. INterruption in patients with Congestive heart failure hospitalizED for a decompensation episode. Eur Heart J. 2009;30(18):2186-2192.Theme song, Works All The Time by Dominik Schwarzer, YouTube ID: CUBDNERZU8HXUHBS, purchased from https://www.premiumbeat.com/. 

Welcome to The Mental Breakdown and Psychreg Podcast! Today, Dr. Berney and Dr. Marshall discuss the signs of typical cognitive decline that happens as we age, and symptoms that suggest the need for an evaluation to rule out a potential dementia. Read the articles from The National Library of Medicine here, from Presbyterian Living here, and from the Alzheimer's Association here. You can now follow Dr. Marshall on twitter, as well! Dr. Berney and Dr. Marshall are happy to announce the release of their new parenting e-book, Handbook for Raising an Emotionally Healthy Child Part 2: Attention. You can get your copy from Amazon here. We hope that you will join us each morning so that we can help you make your day the best it can be! See you tomorrow. Become a patron and support our work at http://www.Patreon.com/thementalbreakdown. Visit Psychreg for blog posts covering a variety of topics within the fields of mental health and psychology. The Parenting Your ADHD Child course is now on YouTube! Check it out at the Paedeia YouTube Channel. The Handbook for Raising an Emotionally Health Child Part 1: Behavior Management is now available on kindle! Get your copy today! The Elimination Diet Manual is now available on kindle and nook! Get your copy today! Follow us on Twitter and Facebook and subscribe to our YouTube Channels, Paedeia and The Mental Breakdown. Please leave us a review on iTunes so that others might find our podcast and join in on the conversation!

Josh Slocum is back after almost 4 years of pushing back against this culture war. We talk about the madness becoming mainstream, cluster B psychology, the external locus of control, happiness in Canada, Alberta separation, media influence, reversal of reality, the climate thing, Trump and Carney and civil war.   In the second half we chat about American exceptionalism, the AWFUL's, these bitches, solutions, sex typical hard wire, sports, Tate on PBD, man hating, what they will say in the future, election fraud, hard realities, EU collapse, and cultural fatigue on this rise.   Mommy has been running the show, Dad hasn't come home for 60 years. Illuminating the abnormal psychology that's become the new normal.   We are in an abusive relationship with our government, our media, and increasingly, ourselves. Today's activists use the same tools as abusive families. They keep us in fear while we wonder if we've lost our minds. It's not a passing fashion and its not well-meaning misguided activism. It's abuse dynamics. Pathological narcissism, indiscriminate rage, and tactical emotional break-downs by bullies who claim that they are the victims. Domestic abuse has gone public, and feral. Join us to talk about it. https://www.youtube.com/@DisaffectedPodcast https://substack.com/@disaffectedpod?utm_source=account-card https://disaffected.com/   To gain access to the second half of show and our Plus feed for audio and podcast please clink the link http://www.grimericaoutlawed.ca/support.   For second half of video (when applicable and audio) go to our Substack and Subscribe. https://grimericaoutlawed.substack.com/ or to our Locals  https://grimericaoutlawed.locals.com/ or Rokfin www.Rokfin.com/Grimerica Patreon https://www.patreon.com/grimericaoutlawed   Support the show directly: https://grimericacbd.com/ CBD / THC Tinctures and Gummies https://grimerica.ca/support-2/ Our Adultbrain Audiobook Podcast and Website: www.adultbrain.ca Our Audiobook Youtube Channel:  https://www.youtube.com/@adultbrainaudiobookpublishing/videos Check out our next trip/conference/meetup - Contact at the Cabin www.contactatthecabin.com Other affiliated shows: www.grimerica.ca The OG Grimerica Show Join the chat / hangout with a bunch of fellow Grimericans  Https://t.me.grimerica grimerica.ca/chats   Discord Chats Darren's book www.acanadianshame.ca Eh-List Podcast and site: https://eh-list.ca/ Eh-List YouTube: https://www.youtube.com/@TheEh-List www.Rokfin.com/Grimerica Our channel on free speech Rokfin Leave a review on iTunes and/or Stitcher: https://itunes.apple.com/ca/podcast/grimerica-outlawed http://www.stitcher.com/podcast/grimerica-outlawed Sign up for our newsletter http://www.grimerica.ca/news SPAM Graham = and send him your synchronicities, feedback, strange experiences and psychedelic trip reports!! graham@grimerica.com InstaGRAM https://www.instagram.com/the_grimerica_show_podcast/  Purchase swag, with partial proceeds donated to the show www.grimerica.ca/swag Send us a postcard or letter http://www.grimerica.ca/contact/ ART - Napolean Duheme's site http://www.lostbreadcomic.com/  MUSIC Tru Northperception, Felix's Site sirfelix.bandcamp.com 

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Dr. Margarita Fedorova outlines how genetic, environmental, and pathological factors interact in Parkinson's disease and what this means for patient counseling.  Show citation:  Blauwendraat C, Morris HR, Van Keuren-Jensen K, Noyce AJ, Singleton AB. The temporal order of genetic, environmental, and pathological risk factors in Parkinson's disease: paving the way to prevention. Lancet Neurol. 2025;24(11):969-975. doi:10.1016/S1474-4422(25)00271-6  Show transcript:  Dr. Margarita Federova: Welcome to Neurology Minute. My name is Margarita Fedorova, and I'm a neurology resident at the Cleveland Clinic. Today we're exploring a framework for understanding how genetic, environmental, and pathological factors interact in Parkinson's disease and what this means for how we counsel our patients. A personal view paper by Blauwendraat and colleagues, published in The Lancet Neurology in September 2025, addresses a critical question. We've identified over 100 genetic loci for Parkinson's, but how do they act? The common saying is genetics loads the gun and environment pulls the trigger, but this paper suggests the relationship may be more complex. The key tool here is alpha-synuclein seeding amplification assays or SAAs. These detect misfolded alpha-synuclein protein in cerebrospinal fluid. Over 90% of Parkinson's patients test positive for misfolded alpha-synuclein using this assay. But here's what's notable. 2% to 16% of neurologically healthy older adults also test positive with prevalence increasing with age. This means there are more asymptomatic people with detectable alpha-synuclein pathology than people with actual Parkinson's disease. Most of these asymptomatic individuals will never develop symptoms. This raises an important question. What determines who converts to a disease and who doesn't? By integrating SAA results with genetic data, researchers can examine whether genetic factors drive initial protein misfolding or whether they modulate the response to pathology triggered by environmental or random events. Preliminary data suggests polygenic risk scores don't strongly associate with SAA positivity in healthy older adults. In other words, people with high genetic risk for Parkinson's aren't necessarily more likely to have misfolded alpha-synuclein if they're healthy. This suggests most Parkinson's genetic risk factors may not be causing initial misfolding. Instead, they may be determining what happens afterward, such as whether the pathology progresses to clinical disease. LRRK2 mutations support this model. About 33% of LRRK2 related Parkinson's patients are SAA-negative compared to only 7% in sporadic disease. This means many people with LRRK2 mutations develop Parkinson's without the typical alpha-synuclein pathology. LRRK2 mutations also show varied pathology. Sometimes alpha-synuclein, sometimes tau, sometimes neither. This suggests LRRK2 may modulate responses to different initiating events rather than directly causing protein misfolding. What does this mean for us as clinicians? Asymptomatic SAA-positive individuals could represent a window for intervention. If we can understand what protects them from converting to disease or what triggers that conversion, we could enable earlier identification of at risk individuals and potentially intervene before symptoms develop. The authors call for large scale studies using SAAs in older populations, combined with genetic analysis and longitudinal follow-up. By integrating pathological biomarkers with genetic and environmental data, we can better understand the temporal sequence of events in development of Parkinson's. This approach could fundamentally change how we think about disease prevention and early intervention, potentially allowing us to identify at risk individuals before symptoms appear and develop targeted prevention strategies. That's your neurology minute for today. Keep exploring, and we'll see you next time. If you want to read more, please find the paper by Cornelis Blauwendraat et al titled The Temporal Order of Genetic, Environmental and Pathological Risk Factors in Parkinson's Disease: Paving the Way to Prevention, published online in September 2025 in Lancet Neurology.

Chelsey Cole is a psychotherapist focusing on narcissistic abuse and complex trauma. She is the best-selling author of If Only I'd Known, and has joined Dr. Rob to explore what narcissism really is and what it isn't. She offers clear signs and strategies of a grandiose narcissist, and offers hope for any partner who has been frustrated by assuming their narcissistic partner views relationships through the same lens they do. Unless you understand the views of a narcissist, you will never succeed in true communication and connection with them.    TAKEAWAYS: [2:10] Is he just selfish or is he a true narcissist?  [5:26] Characteristics of the grandiose narcissist.  [9:38] The danger of transactional views of relationships.  [10:31] Stages of the narcissist spectrum.  [14:45] Remorse motivation in a narcissist's mind.  [17:07] Steps in the cycle of narcissistic abuse.  [20:03] Characteristics of the moderate to severe narcissists.  [23:01] You're not crazy! Narcissists actually want the conflict you are avoiding.  [25:49] Pathological insecurity versus healthy insecurity.    RESOURCES: Sex and Relationship Healing @RobWeissMSW Sex Addiction 101  Seeking Integrity Free Sexual Addiction Screening Assessment Partner Sexuality Survey Chelsey Cole   Seeking Integrity Podcasts are produced in partnership with Podfly Productions.    QUOTES: “True narcissists don't have the stuff that it takes to create deep, committed relationships.”  “Narcissists believe they are always the exception to every rule.”  “For a true narcissist, their default is to not care about you, and to put their needs first.”  “Narcissists actually seek the conflict you are avoiding.” 

Welcome to episode 78 of Rapaport's Reality! Starring Kebe & Michael Rapaport. This is the reality television podcast that the whole reality world has been waiting for. The Rapaport's are here to discuss: The Golden Age of Reality TV Gia Guiadice Huge Shoutout New Years Resolution Jen Shaw coming out of jail Real Housewives Redemption Pathological In Potomac? Wendy & Eddie's jail situation 9th Year of Marriage Being in Israel Leak Gate & Grading Heather Gay Taking That Reality Ride This episode is not to be missed! An iHeartPodcasts Show Stand Up Comedy Tickets on sale at: MichaelRapaportComedy.com Produced by DBPodcasts.comFollow @dbpodcasts, @rapaportsreality, @michaelrapaport on Instagram & X Subscribe to Rapaport's Reality Feeds: iHeartRadio: https://www.iheart.com/podcast/867-rapaports-reality-with-keb-171162927/ Apple Podcasts: https://podcasts.apple.com/us/podcast/rapaports-reality-with-kebe-michael-rapaport/id1744160673 Spotify: https://open.spotify.com/show/3a9ArixCtWRhfpfo1Tz7MR Pandora: https://www.pandora.com/podcast/rapaports-reality-with-kebe-michael-rapaport/PC:1001087456 Amazon Music: https://music.amazon.com/podcasts/a776919e-ad8c-4b4b-90c6-f28e41fe1d40/rapaports-reality-with-kebe-michael-rapaportSee omnystudio.com/listener for privacy information.

This episode marks the beginning of a new educational series from Heal NPD, featuring Dr. Mark Ettensohn and his associates: Deanna Young, Psy.D. and Danté Spencer, MA. This series offers a rare window into clinical reasoning and supervision, bringing viewers inside real discussions about theory, diagnosis, and treatment of personality pathology. In this first seminar, the group examines an influential paper by Pincus & Lukowitsky (2010) and explores one of the central challenges in the field: how to define pathological narcissism. The conversation addresses the criterion problem surrounding narcissism. That is, the lack of a unified construct definition. It traces how this has led to conflicting models and measures of narcissism. Topics include the distinction between pathological narcissism and NPD, the interplay of grandiosity and vulnerability, the overlap with depression and trauma, and emerging dimensional approaches to understanding personality. This series is designed for clinicians, students, and anyone interested in a deeper and more integrative understanding of narcissism, personality, and self-regulation. To learn more about our work, visit www.HealNPD.org Citation for the article discussed: Pincus, A. L., & Lukowitsky, M. R. (2010). Pathological narcissism and narcissistic personality disorder. Annual Review of Clinical Psychology, 6, 421–446. https://doi.org/10.1146/annurev.clinpsy.121208.131215

Distinguishing Humane Nationalism from Pathological Ideologies Guest Name: Daniel Mahoney Summary:Nationalism must be distinguished from pathological forms like "blood and soil" ideology, which champions ethnic rooting and the subordination of others. Moderate, humane national loyalty is tied to self-government and common humanity, rejecting the path that leads to "zoological wars." Critics often unfairly conflate nationalism with isolationism or imperialism. 1916 SWITZERLAND

Distinguishing Humane Nationalism from Pathological Ideologies Guest Name: Daniel Mahoney Summary:Nationalism must be distinguished from pathological forms like "blood and soil" ideology, which champions ethnic rooting and the subordination of others. Moderate, humane national loyalty is tied to self-government and common humanity, rejecting the path that leads to "zoological wars." Critics often unfairly conflate nationalism with isolationism or imperialism. 1865 MONTENEGRO

We asked Dr. Im about the role of sleep in injury recovery, what is considered optimal sleep, and when someone should be referred for pathological sleep.Timestamps(8:50) What signs & symptoms may indicate less than optimal sleep(11:09) Sleep and recovery(17:16) Stages of sleep(18:23) Adapting stages of sleep(21:32) Recommendations to improve sleep quality(25:40) Special considerations for maintaining optimal sleep(27:21) Role of naps in improving sleep quality(30:39) Optimal timing of naps(31:49) Relationship of mental health and sleep(34:40) Pathological sleep(41:05) Changing the circadian rhythm (43:59) When to refer an athlete with pathological sleep(46:30) Monitoring sleepAction Item: What are some talking points you use with athletes to educate about their sleep and importance of sleep?--AT CORNER FACEBOOK GROUP: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.facebook.com/groups/atcornerpodcast⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Instagram, Website, YouTube, and other links: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠atcornerds.wixsite.com/home/links⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠EMAIL US: atcornerds@gmail.comSAVE on ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Medbridge⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠: Use code ATCORNER to get $101 off your subscriptionWant to host a podcast like ours? Use our link to sign up for Zencastr, the service we use to record our interviews: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://zencastr.com/?via=atcorner⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Music: Jahzzar (betterwithmusic.com) CC BY-SA---Sandy & Randy

Your favorite psychologists, John Gartner and Harry Segal, review Trump's disastrous Alaska summit and marvel at how he has reverted to Putin's absurd Ukraine talking points. They also speak with Richard Wood, eminent clinical psychologist and expert on malignant narcissism, who shares his insights on Trump's pathology. Be sure to subscribe wherever you get your podcasts: Our site Subscribe on iTunes Subscribe on Spotify Subscribe on Amazon Music Subscribe on iHeartRadio We open with a “therapy session” framework—our way of inviting you into a collective examination of how Trump's behavior has infiltrated our daily lives. Too often, political coverage tip-toes around psychological insights. Not here. We argue that to understand Trump's volatility, his tantrums, and his rule-by-instinct style, you have to start with his inner life. Dr. Woods steps in to define malignant narcissism: a toxic cocktail of grandiosity, ruthlessness, and paranoia. He walks us through examples—obsessive self-aggrandizement, zero empathy for victims, and a ferocious need to dominate. When you hear Trump lob insults or bully subordinates, that's not business as usual. It's the playbook of someone for whom status and control override every other impulse. Too many pundits tiptoe around Trump's slurred speeches, jumbled tweets, and off-script rants. We don't. We break down the warning signs of cognitive fragility—memory lapses, word-salad tangents, even motor-skill stumbles. These aren't just gaffes; they're red flags that a leader who can't stay on message is a risk to national security. Why do millions still cheer him on? In a frank discussion, we and Dr. Woods explore how Trump transforms fear into solidarity. His promises of vengeance and identity-based appeals speak to grievances deeper than policy. When democracy feels abstract, demagoguery feels personal—and that's how authoritarian figures like Trump cement loyalty. We draw parallels to his moves in Washington itself: weaponizing local police, twisting museum exhibits to sanitize his image, and rewriting history to suit his narrative. It's not accidental. It's an orchestrated strategy to freeze dissent and manufacture consent. This episode doesn't leave you helpless. We outline concrete steps—peer education, fact-based conversations, and refusing to normalize destructive behavior cloaked in charismatic populism. Psychological insight isn't therapy; it's ammunition. It helps us name the tactics, defang the threats, and reclaim our public life. If you want to see past the tweets and the rallies and understand the man behind the façade, tune in to Shrinking Trump wherever you get your podcasts. Let's sharpen our collective lens—because democracy demands more than headline-chasing. It demands that we understand, confront, and ultimately shrink the power of rulers unfit to lead. Learn more about your ad choices. Visit megaphone.fm/adchoices

Across centuries and continents, documented cases reveal supernatural forces that escalated beyond typical paranormal activity — resulting in physical harm, unexplained deaths, and encounters that defy conventional explanations.Join the DARKNESS SYNDICATE: https://weirddarkness.com/syndicateIN THIS EPISODE: Many ghost stories are frightening but leave the person encountering them shaken, but no more worse for wear. Perhaps a light touch on the shoulder, the moving of an object, a whisper in the ear, or a cold chill for the more intense cases. But then there are those hauntings that leave the person hysterical… or physically hurt… or even dead. We'll look at some of the most violent ghosts and hauntings of all time. (History's Most Violent Ghosts) *** Dixon, Illinois needed a bridge – Mr. L.E. Truesdell was an expert bridge builder. But people had doubts as to whether what he was being asked to do could be accomplished. The naysayers were ignored – and tragedy arrived, (The Truesdell Bridge Disaster) *** The witch trials in Salem, Massachusetts were horrid… the trials in Europe even more so. Now, imagine being a soldier tried as a witch because you returned with supernatural knowledge imparted to you after you were abducted by extraterrestrials. It sounds like the makings of a terrible novel and an even worse movie… but it's the true story of Major Thomas Weir. (The True Story of the Alien Abduction Witch Trial) *** He's believed to have killed over 50 women inside his soundproof torture trailer – which he called his “toy box.” We'll look at the case of serial killer David Parker Ray. (Evils of the Toybox Killer) *** Our criminal justice system tries to be the most fair in the world – considering the accused “innocent until proven guilty.” And while our system is not perfect, for the most part it does its job well. But because it is not perfect, once in a while a murderer walks… or even worse, an innocent defendant goes to prison. And in some cases, that innocent person is convicted and imprisoned based on one thing – they confessed to the crime that they didn't commit. Why would someone do that? (Why Do Innocent People Confess?) *** Port St. Lucie, Florida is full of disturbing urban legends – some true, many false – as urban legends go. But somehow, most all of these terrifying stories tie back to a single oak tree in a neighborhood park, and Florida's first serial killer. (The Devil Tree Hauntings)ABOUT WEIRD DARKNESS: Weird Darkness is a true crime and paranormal podcast narrated by professional award-winning voice actor, Darren Marlar. Seven days per week, Weird Darkness focuses on all thing strange and macabre such as haunted locations, unsolved mysteries, true ghost stories, supernatural manifestations, urban legends, unsolved or cold case murders, conspiracy theories, and more. On Thursdays, this scary stories podcast features horror fiction along with the occasional creepypasta. Weird Darkness has been named one of the “Best 20 Storytellers in Podcasting” by Podcast Business Journal. Listeners have described the show as a cross between “Coast to Coast” with Art Bell, “The Twilight Zone” with Rod Serling, “Unsolved Mysteries” with Robert Stack, and “In Search Of” with Leonard Nimoy.DISCLAIMER: Ads heard during the podcast that are not in my voice are placed by third party agencies outside of my control and should not imply an endorsement by Weird Darkness or myself. *** Stories and content in Weird Darkness can be disturbing for some listeners and intended for mature audiences only. Parental discretion is strongly advised.CHAPTERS & TIME STAMPS (All Times Approximate)…00:00:00.000 = Lead-In00:00:55.371 = Show Open00:04:12.885 = The Devil Tree Hauntings00:15:28.295 = History's Most Violent Ghosts00:36:32.777 = The True Story of the Alien Abduction Witch Trial00:45:55.817 = Evils of the Toybox Killer00:57:04.271 = The Truesdell Bridge Disaster01:07:45.917 = Why Do Innocent People Confess?01:15:48.277 = Show CloseSOURCES AND RESOURCES FROM THE EPISODE…“History's Most Violent Ghosts” by Robert F. Mason for Ranker: https://weirddarkness.tiny.us/3x42y575“The True Story of the Alien Abduction Witch Trial” by Marcus Lowth for UFO Insight: https://weirddarkness.tiny.us/bdhrv3hu“The Devil Tree Hauntings” posted at MiamiHaunts.com: https://weirddarkness.tiny.us/2sp2wakz“Evils of the Toybox Killer” by Jaclyn Anglis for All That's Interesting: https://weirddarkness.tiny.us/jep7tnxf“The Truesdell Bridge Disaster” by Kathi Kresol for HauntedRockford.com: https://weirddarkness.tiny.us/4c94hvjy“Why Do Innocent People Confess?” by Lea Rose Emery for Ranker: https://weirddarkness.tiny.us/bjtvenfv=====(Over time links may become invalid, disappear, or have different content. I always make sure to give authors credit for the material I use whenever possible. If I somehow overlooked doing so for a story, or if a credit is incorrect, please let me know and I will rectify it in these show notes immediately. Some links included above may benefit me financially through qualifying purchases.)= = = = ="I have come into the world as a light, so that no one who believes in me should stay in darkness." — John 12:46= = = = =WeirdDarkness® is a registered trademark. Copyright ©2025, Weird Darkness.=====Originally aired: July 24, 2023EPISODE PAGE at WeirdDarkness.com (includes list of sources): https://weirddarkness.com/ViolentHauntings

Megyn Kelly is joined by Jordan Peterson, co-founder of Peterson Academy and author of "We Who Wrestle with God," to discuss the insane leftist policies that drove men and boys away from the Democratic party, how they're still missing the point even after Trump's landslide win, how it is "too little too late" for schools and universities to salvage their reputation, the decline of the Ivy League status, how young people are the more politically divided by gender than ever before, how Dems don't understand why there's this split and still think everything is about abortion, Andrew Tate's “pathological masculinity" and the wrong messenger to men and boys, why Dems are misguided about what makes Trump popular, what Olivia Wilde got wrong about him in the character based on him in “Don't Worry Darling,” what Peterson learned about Chris Pine after the movie came out, how Disney dropped the ball on Snow White, challenges young women face in the dating world, the issue with the mentality of “settling,” and more.More from Peterson: https://petersonacademy.com/Herald Group: Learn more at https://GuardYourCard.com  Done with Debt: https://www.DoneWithDebt.com  & tell them Megyn sent you!FYSI: https://FYSI.com/Megyn  or call 800-877-4000 Follow The Megyn Kelly Show on all social platforms:YouTube: https://www.youtube.com/MegynKellyTwitter: http://Twitter.com/MegynKellyShowInstagram: http://Instagram.com/MegynKellyShowFacebook: http://Facebook.com/MegynKellyShow Find out more information at: https://www.devilmaycaremedia.com/megynkellyshow

Sarah roped Susie into watching Love is Blind and now they have to talk about it (even though Susie is mad about it). We discuss the particular hellscape that is a reality tv reunion and the trauma it provides. We discuss why bangs are never the answer and you should never get a tattoo when you're in the throes of life's worst moments. Sarah talks about Belle Gibson who became a successful influencer, cookbook author, and lifestyle blogger based on a lie that she had terminal cancer she was treating homeopathically. We find out why young people aren't getting romantically involved and debate whether that's a bad or good thing. And we hear why reality tv causes insecurity in men and women in different ways. Plus, Susie explains what a "bed party" is and why people are spending lots of money for an instagram picture celebrating their kid going to college.Listen to more podcasts like this: https://wavepodcastnetwork.comJoin our Candy Club, shop our merch, sign-up for our free newsletter, & more by visiting The Brain Candy Podcast website: https://www.thebraincandypodcast.comConnect with us on social media:BCP Instagram: https://www.instagram.com/braincandypodcastSusie's Instagram: https://www.instagram.com/susiemeisterSarah's Instagram: https://www.instagram.com/imsarahriceBCP on X: https://www.x.com/braincandypodSponsors:Get 20% OFF by going to https://www.honeylove.com/braincandy! #honeylovepodGet up to 35% off PLUS 2 free gifts using code BRAINCANDY at https://shopbeam.com/BRAINCANDYSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.