Podcasts about pnes

Paroxysmal movement disorders refer to a group of highly heterogeneous disorders that present with attacks of involuntary movements without loss of consciousness. These disorders demonstrate considerable and ever-expanding genetic and clinical heterogeneity, so an accurate clinical diagnosis has key therapeutic implications. In this episode, Kait Nevel, MD, speaks with Abhimanyu Mahajan, MD, MHS, FAAN, author of the article “Paroxysmal Movement Disorders” in the Continuum® August 2025 Movement Disorders issue. Dr. Nevel is a Continuum® Audio interviewer and a neurologist and neuro-oncologist at Indiana University School of Medicine in Indianapolis, Indiana. Dr. Mahajan is an assistant professor of neurology and rehabilitation medicine at the James J. and Joan A. Gardner Family Center for Parkinson's Disease and Movement Disorders at the University of Cincinnati in Cincinnati, Ohio. Additional Resources Read the article: Paroxysmal Movement Disorders Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @IUneurodocmom Guest: @MahajanMD Full episode transcript available here Dr Jones: This is Doctor Lyell Jones, editor in chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Nevel: Hello, this is Dr Kait Nevel. Today I'm interviewing doctor Abhi Mahajan about his article on diagnosis and management of paroxysmal movement disorders, which appears in the August 2025 Continuum issue on movement disorders. Abhi, welcome to the podcast and please introduce yourself to the audience. Dr Mahajan: Thank you, Kait. Thank you for inviting me. My name is Abhi Mahajan. I'm an assistant professor of neurology and rehabilitation medicine at the University of Cincinnati in Cincinnati, Ohio. I'm happy to be here. Dr Nevel: Wonderful. Well, I'm really excited to talk to you about your article today on this very interesting and unique set of movement disorders. So, before we get into your article a little bit more, I think just kind of the set the stage for the discussion so that we're all on the same page. Could you start us off with some definitions? What are paroxysmal movement disorders? And generally, how do we start to kind of categorize these in our minds? Dr Mahajan: So, the term paroxysmal movement disorders refers to a group of highly heterogeneous disorders. These may present with attacks of involuntary movements, commonly a combination of dystonia and chorea, or ataxia, or both. These movements are typically without loss of consciousness and may follow, may follow, so with or without known triggers. In terms of the classification, these have been classified in a number of ways. Classically, these have been classified based on the trigger. So, if the paroxysmal movement disorder follows activity, these are called kinesigenic, paroxysmal, kinesigenic dyskinesia. If they are not followed by activity, they're called non kinesigenic dyskinesia and then if they've followed prolonged activity or exercise they're called paroxysmal exercise induced dyskinesia. There's a separate but related group of protogynous movement disorders called episodic attacks here that can have their own triggers. Initially this was the classification that was said. Subsequent classifications have placed their focus on the ideology of these attacks that could be familiar or acquired and of course understanding of familiar or genetic causes of paroxysmal movement disorders keeps on expanding and so on and so forth. And more recently, response to pharmacotherapy and specific clinical features have also been introduced into the classification. Dr Nevel: Great, thank you for that. Can you share with us what you think is the most important takeaway from your article for the practicing neurologist? Dr Mahajan: Absolutely. I think it's important to recognize that everything that looks and sounds bizarre should not be dismissed as malingering. Such hyperkinetic and again in quotations, “bizarre movements”. They may appear functional to the untrained eye or the lazy eye. These movements can be diagnosed. Paroxysmal movement disorders can be diagnosed with a good clinical history and exam and may be treated with a lot of success with medications that are readily available and cheap. So, you can actually make a huge amount of difference to your patients' lives by practicing old-school neurology. Dr Nevel: That's great, thank you so much for that. I can imagine that scenario does come up where somebody is thought to have a functional neurological disorder but really has a proximal movement disorder. You mentioned that in your article, how it's important to distinguish between these two, how there can be similarities at times. Do you mind giving us a little bit more in terms of how do we differentiate between functional neurologic disorder and paroxysmal movement disorder? Dr Mahajan: So clinical differentiation of functional neurological disorder from paroxysmal movement disorders, of course it's really important as a management is completely different, but it can be quite challenging. There's certainly an overlap. So, there can be an overlap with presentation, with phenomenology. Paroxysmal nature is common to both of them. In addition, FND and PMD's may commonly share triggers, whether they are movement, physical exercise. Other triggers include emotional stimuli, even touch or auditory stimuli. What makes it even more challenging is that FND's may coexist with other neurological disorders, including paroxysmal movement disorders. However, there are certain specific phenom phenotypic differences that have been reported. So specific presentations, for example the paroxysms may look different. Each paroxysm may look different in functional neurological disorders, specific phenotypes like paroxysmal akinesia. So, these are long duration episodes with eyes closed. Certain kinds of paroxysmal hyperkinesia with ataxia and dystonia have been reported. Of course. More commonly we see PNES of paroxysmal nonepileptic spells or seizures that may be considered paroxysmal movement disorders but represent completely different etiology which is FND. Within the world of movement disorders, functional jerks may resemble propiospinal myoclonus which is a completely different entity. Overall, there are certain things that help separate functional movement disorders from paroxysmal movement disorders, such as an acute onset variable and inconsistent phenomenology. They can be suggestibility, distractibility, entrainment, the use of an EMG may show a B-potential (Bereitschaftspotential) preceding the movement in patients with FND. So, all of these cues are really helpful. Dr Nevel: Great, thanks. When you're seeing a patient who's reporting to these paroxysmal uncontrollable movements, what kind of features of their story really tips you off that this might be a proximal movement disorder? Dr Mahajan: Often these patients have been diagnosed with functional neurological disorders and they come to us. But for me, whenever the patient and or the family talk about episodic movements, I think about these. Honestly, we must be aware that there is a possibility that the movements that the patients are reporting that you may not see in clinic. Maybe there are obvious movement disorders. Specifically, there's certain clues that you should always ask for in the history, for example, ask for the age of onset, a description of movements. Patients typically have videos or families have videos. You may not be able to see them in clinic. The regularity of frequency of these movements, how long the attacks are, is there any family history of or not? On the basis of triggers, whether, as I mentioned before, do these follow exercise? Prolonged exercise? Or neither of the above? What is the presentation in between attacks, which I think is a very important clinical clue. Your examination may be limited to videos, but it's important not just to examine the video which represents the patient during an attack, but in between attacks. That is important. And of course, I suspect we'll get to the treatment, but the treatment can follow just this part, the history and physical exam. It may be refined with further testing, including genetic testing. Dr Nevel: Great. On the note of genetic testing, when you do suspect a diagnosis of paroxysmal movement disorder, what are some key points for the provider to be aware of about genetic testing? How do we go about that? I know that there are lots of different options for genetic testing and it gets complicated. What do you suggest? Dr Mahajan: Traditionally, things were a little bit easier, right, because we had a couple of genes that have been associated with the robust movement disorders. So, genetic testing included single gene testing, testing for PRRT2 followed by SLC2A. And if these were negative, you said, well, this is not a genetic ideology for paroxysmal movement disorders. Of course, with time that has changed. There's an increase in known genes and variants. There is increased genetic entropy. So, the same genetic mutation may present with many phenotypes and different genetic mutations may present with the similar phenotype. Single gene testing is not a high yield approach. Overall genetic investigations for paroxysmal movement disorders use next generation sequencing or whole exome sequence panels which allow for sequencing of multiple genes simultaneously. The reported diagnostic yield with let's say next generation sequencing is around 35 to 50 percent. Specific labs at centers have developed their own panels which may improve the yield of course. In children, microarray may be considered, especially the presentation includes epilepsy or intellectual disability because copy number variations may not be detected by a whole exome sequencing or next generation sequencing. Overall, I will tell you that I'm certainly not an expert in genetics, so whenever you're considering genetic testing, if possible, please utilize the expertise of a genetic counsellor. Families want to know, especially as an understanding of the molecular underpinnings and knowledge about associated mutations or variations keeps on expanding. We need to incorporate their expertise. A variant of unknown significance, which is quite a common result with genetic testing, may not be a variant of unknown significance next year may be reclassified as pathogenic. So, this is extremely important. Dr Nevel: Yeah. That's such a good point. Thank you. And you just mentioned that there are some genetic mutations that can lead to multiple different phenotypes. Seemingly similar phenotypes can be associated with various genetic mutations. What's our understanding of that? Do we have an understanding of that? Why there is this seeming disconnect at times between the specific genetic mutation and the phenotype? Dr Mahajan: That is a tough question to answer for all paroxysmal movement disorders because the answer may be specific to a specific mutation. I think a great example is the CACNA1A mutation. It is a common cause of episodic ataxia type 2. Depending on when the patient presents, you can have a whole gamut of clinical presentations. So, if the patient is 1 year old, the patient can present with epileptic encephalopathy. Two to 5 years, it can be benign paroxysmal torticollis of infancy. Five to 10 years, can present with learning difficulties with absence epilepsy and then of course later, greater than 10 years, with episodic ataxia (type) 2 hemiplegic migraine and then a presentation with progressive ataxia and hemiplegic migraines has also been reported. So not just episodic progressive form of ataxia has also been reported. I think overall these disorders are very rare. They are even more infrequently diagnosed than their prevalence. As such, the point that different genetic mutations present with different phenotypes, or the same genetic mutation I may present with different phenotypes could also represent this part. Understanding of the clinical presentation is really incomplete and forever growing. There's a new case report or case series every other month, which makes this a little bit challenging, but that's all the more reason for learning about them and for constant vigilance for patients who show up to our clinic. Dr Nevel: Yeah, absolutely. What is our current understanding of the associated pathophysiology of these conditions and the pathophysiology relating to the genetics? And then how does that relate to the treatment of these conditions? Dr Mahajan: So, a number of different disease mechanisms have been proposed. Traditionally, these were all thought to be ion channelopathies, but a number of different processes have been proposed now. So, depending on the genetic mutation that you talk about. So certain mutations can involve ion channels such as CACMA1A, ATP1A3. It can involve solute carriers, synaptic vesicle fusion, energy metabolism such as ECHS1, synthesis of neurotransmitters such as GCH1. So, there are multiple processes that may be involved. I think overall for the practicing clinician such as me, I think there is a greater need for us to understand the underlying genetics and associated phenotypes and the molecular mechanisms specifically because these can actually influence treatment decisions, right? So, you mentioned that specific genetic testing understanding of the underlying molecular mechanism can influence specific treatments. As an example, a patient presenting with proximal nocturnal dyskinesia with mutation in the ADCY5 gene may respond beautifully to caffeine. Other examples if you have SLC2A1, so gluc-1 (glucose transporter type 1) mutation, a ketogenic diet may work really well. If you have PDHA1 mutation that may respond to thiamine and so on and so forth. There are certain patients where paroxysmal movement disorders are highly disabling and you may consider deep brain stimulation. That's another reason why it may be important to understand genetic mutations because there is literature on response to DBS with certain mutations versus others. Helps like counselling for patients and families, and of course introduces time, effort, and money spent in additional testing. Dr Nevel: Other than genetic testing, what other diagnostic work up do you consider when you're evaluating patients with a suspected paroxysmal movement disorder? Are there specific things in the history or on exam that would prompt you to do certain testing to look for perhaps other things in your differential when you're first evaluating a patient? Dr Mahajan: In this article, I provide a flow chart that helps me assess these patients as well. I think overall the history taking and neurological exam outside of these paroxysms is really important. So, the clinical exam in between these episodic events, for example, for history, specific examples include, well, when do these paroxysms happen? Do they happen or are they precipitated with meals that might indicate that there's something to do with glucose metabolism? Do they follow exercise? So, a specific example is in Moyamoya disease, they can be limb shaking that follows exercise. So, which gives you a clue to what the etiology could be. Of course, family history is important, but again, talking about the exam in between episodes, you know, this is actually a great point because out– we've talked about genetics, we've talked about idiopathic paroxysmal movement disorders, –but a number of these disorders are because of acquired causes. Well, of course it's important because acquired causes such as autoimmune causes, so multiple sclerosis, ADEM, lupus, LGI1, all of these NMDAR, I mentioned Moyamoya disease and metabolic causes. Of course, you can consider FND as under-acquired as well. But all of these causes have very different treatments and they have very different prognosis. So, I think it's extremely important for us to look into the history with a fine comb and then examine these patients in between these episodes and keep our mind open about acquired causes as well. Dr Nevel: When you evaluate these patients, are you routinely ordering vascular imaging and autoimmune kind of serologies and things like that to evaluate for these other acquired causes or it does it really just depend on the clinical presentation of the patient? Dr Mahajan: It mostly depends on the clinical presentation. I mean, if the exam is let's say completely normal, there are no other risk factors in a thirty year old, then you know, with a normal exam, normal history, no other risk factors. I may not order an MRI of the brain. But if the patient is 55 or 60 (years) with vascular risk factors, then you have to be mindful that this could be a TIA. If the patient has let's say in the 30s and in between these episodes too has basically has a sequel of these paroxysms, then you may want to consider autoimmune. I think the understanding of paraneoplastic, even autoimmune disorders, is expanding as well. So, you know the pattern matters. So, if all of this is subacute started a few months ago, then I have a low threshold for ordering testing for autoimmune and paraneoplastic ideology is simply because it makes such a huge difference in terms of how you approach the treatment and the long-term prognosis. Dr Nevel: Yeah, absolutely. What do you find most challenging about the management of patients with paroxysmal movement disorders? And then also what is most rewarding? Dr Mahajan: I think the answer to both those questions is, is the same. The first thing is there's so much advancement in what we know and how we understand these disorders so regularly that it's really hard to keep on track. Even for this article, it took me a few months to write this article, and between the time and I started and when I ended, there were new papers to include new case reports, case series, right? So, these are rare disorders. So most of our understanding for these disorders comes from case reports and case series, and it's in a constant state of advancement. I think that is the most challenging part, but it's also the most interesting part as well. I think the challenging and interesting part is the heterogeneity of presentation as well. These can involve just one part of your body, your entire body can present with paroxysmal events, with multiple different phenomenologies and they might change over time. So overall, it's highly rewarding to diagnose such patients in clinic. As I said before, you can make a sizeable difference with the medication which is usually inexpensive, which is obviously a great point to mention these days in our health system. But with anti-seizure drugs, you can put the right diagnosis, you can make a huge difference. I just wanted to make a point that this is not minimizing in any way the validity or the importance of diagnosing patients with functional neurological disorders correctly. Both of them are as organic. The importance is the treatment is completely different. So, if you're diagnosing somebody with FND and they do have FND and they get cognitive behavioral therapy and they get better, that's fantastic. But if somebody has paroxysmal movement disorders and they undergo cognitive behavioral therapy and they're not doing well, that doesn't help anybody. Dr Nevel: One hundred percent. As providers, obviously we all want to help our patients and having the correct diagnosis, you know, is the first step. What is most interesting to you about paroxysmal movement disorders? Dr Mahajan: So outside of the above, there are some unanswered questions that I find very interesting. Specifically, the overlap with epilepsy is very interesting, including shared genes, the episodic nature, presence of triggers, therapeutic response to anti-seizure drugs. All of this I think deserves further study. In the clinic, you may find that epilepsy and prognosis for movement disorders may occur in the same individual or in a family. Episodic ataxia has been associated with seizures. Traditionally this dichotomy of an ictal focus. If it's cortical then it's epilepsy, if it's subcortical then it's prognosis for movement disorders. This is thought to be overly simplistic. There can be co-occurrence of seizures and paroxysmal movement disorders in the same patient and that has led to this continuum between these two that has been proposed. This is something that needs to be looked into in more detail. Our colleagues in Epilepsy may scoff this, but there's concept of basal ganglia epilepsy manifesting as paroxysmal movement disorders was proposed in the past. And there was this case report that was published out of Italy where there was ictal discharge from the supplementary sensory motor cortex with a concomitant discharge from the ipsilateral coordinate nucleus in a patient with paroxysmal kinesigenic cardioarthidosis. So again, you know, basal ganglia epilepsy, no matter what you call it, the idea is that there is a clear overlap between these two conditions. And I think that is fascinating. Dr Nevel: Really interesting stuff. Well, thank you so much for chatting with me today. Dr Mahajan: Thank you, Kait. And thank you to the Continuum for inviting me to write this article and for this chance to speak about it. I'm excited about how it turned out, and I hope readers enjoy it as well. Dr Nevel: Today again, I've been interviewing doctor Abhi Mahajan about his article on diagnosis and management of paroxysmal movement disorders, which appears in the August 2025 Continuum issue on movement disorders. I encourage all of our listeners to be sure to check out the Continuum Audio episodes from this and other issues. As always, please read the Continuum articles where you can find a lot more information than what we were able to cover in our discussion today. And thank you for our listeners for joining today. And thank you, Abhi, so much for sharing your knowledge with us today. Dr Monteith: This is Dr Teshamae Monteith, associate editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

PNES!

Neurologist Dr Parthvi Ravat shares the relationship between epilepsy and Non-Epileptic Events* (also known as dissociative Seizures and PNES), symptoms of the latter, treatment options, and the importance of research and awareness in improving patient outcomes. Parthvi highlights the complexities of diagnosing and managing FND and Dissociative Seizures/PNES/Non-Epileptic Events, the need for a multidisciplinary approach, and the shared experiences of patients experiencing both an epilepsy and NEEs! *A type of Functional Neurological Disorder (FND) ------------------------------------------

Dr. Halley Alexander and Dr. Serena Yin discuss the effectiveness of an electronic medical record best practice alert in preventing iatrogenic interventions for patients with a diagnosis of PNES. Show references:  https://www.neurology.org/doi/10.1212/CPJ.0000000000200457 

Dr. Halley Alexander talks with Dr. Serena Yin about the effectiveness of an electronic medical record best practice alert in preventing iatrogenic interventions for patients with a diagnosis of psychogenic nonepileptic seizures. Read the related article in Neurology® Clinical Practice.  Disclosures can be found at Neurology.org. 

I denne episoden snakker vi om:Søvn (Egenreklame for Susannes meditasjon)Portalen som åpnes 21.marsHva betyr dette for det store bevissthetsskiftetHvordan påvirkes vi som personer?Hvordan påvirkes samfunnet?Skjer det plutselig over natten?Lysspråket kommer tilbake Hosted on Acast. See acast.com/privacy for more information.

I denne episoden snakker vi om:En hektisk hverdagHva vil det si å være skeptiskMål for ny sesongSusannes nyhetsbrev Hosted on Acast. See acast.com/privacy for more information.

Ønsker alle en God Jul og et Godt Nyttår.Support the showTrykk linken over "Support the show".Dette er link til patreon for deg som ønsker mere av Synseligaen på øret.Der får du tilgang på "en episode ekstra hver uke".Medlemskap på Patreon kan benyttes mnd til mnd eller et år av gangen ved ønske.Ved at du er Patreon medlem får du vanlig & en ekstra episode ukentlig avspilt der du hører på podcast, altså på samme platform (eks Spotify, Apple) om ønsklig.(Lydklipp er godkjent av utvalgte spillere & mediahus).Takk til Tv2 sporten & Europort for bruk av lydklipp i denne episoden.

Once called pseudoseizures, psychogenic non-epileptic seizures can confuse even seasoned clinicians — until you know the signs. These seizures mimic epilepsy but have entirely different causes, requiring a unique approach to care. In this episode, we explore how to distinguish PNES from epilepsy, the psychological factors that trigger attacks, and steps in the diagnostic process.Learn how you can identify these episodes, provide compassionate care, and educate patients and their families about this misunderstood condition!Topics discussed in this episode:Three PNES patient storiesWhat are psychogenic non-epileptic seizures?PNES versus epilepsy: key differences and signsDiagnosing PNESTreatment and the role of nursesLearn more about psychogenic non-epileptic seizures here:https://www.epilepsy.com/stories/truth-about-psychogenic-nonepileptic-seizuresMentioned in this episode:CONNECT

Apg. 10: "Vinden blåser dit den vil – Nye dører åpnes" - Andreas Bjørntvedt - Gudstjeneste 24.11.24

PALERMO (ITALPRESS) - “L'obiettivo è quello di rafforzare la cultura progettuale, definire un cantiere nel quale sviluppare al meglio le progettualità. La presenza anche dell'istituto nazionale per le povertà rappresenta una testimonianza concreta di un rapporto di collaborazione che vi è fra la Regionee il Ministero della Salute”. Così Salvatore Iacolino, Dirigente Generale Pianificazione strategica dell'Assessorato Salute della Regione Siciliana, a margine del convegno “L'internazionalizzazione del SSR: nuove sfide per una sanità dinamica”, a Palazzo dei Normanni, a Palermo. “Internazionalizzazione significa anche ricerca, significa cooperazione internazionale - ha aggiunto Iacolino -. La Sicilia è al centro del mediterraneo e deve sviluppare al meglio le potenzialità di cui dispone. Sono presenti direttori generali, rappresentanti di istituzioni varie, dimostrazione che soltanto una rete concreta di rapporti consente di sviluppare progettualità serie. Noi abbiamo 105 milioni di euro sul PNES, Piano Nazionale di Equità nella Salute, che utilizzeremo al meglio mettendo al centro della nostra attività il genere femminile, i consultori, la salute mentale e gli screening oncologici. Il tutto per ridurre il gap nella prevenzione che ha caratterizzato distintamente le attività del passato e per dare un segnale concreto di seria e rigorosa programmazione da parte delle attività che l'assessorato regionale intende concretamente promuovere e sviluppare”. xd6/vbo/gsl

Denne søndagen er det Torgeir Lauvås som taler over Apg. 5:17-5:42 ut fra tema "Når låste dører åpnes".

Dagens gjest er Åse Marie Faldalen, spesialsykepleier innen kriser og traumer, fotograf og forfatter, bla av "Syv veier til lykke" med Johan Galtung. Vi snakker om: * Viktigheten av å stole på det du kjenner i deg * Hva gir vi vår autoritet til? * Erfaringsmuligheten er hellig * Gir vi bort det viktigsrte vi har? * Kvalme og glede * Medfølelse, tilgivelse, forsoning og forståelse * I vår kultur ligger kanskje dette litt dypere begravd * Medfølese vs selv - medfølelse * Autoriteten er deg! Punktum.

In part three of this three-part series on non-epileptic events. Dr. Dina Bolden discusses how to compassionately and effectively communicate a PNES diagnosis and how to develop a treatment plan. Show reference: https://pubmed.ncbi.nlm.nih.gov/26633963/ This podcast is sponsored by argenx. Visit www.vyvgarthcp.com for more information.

In part two of this three-part series on non-epileptic events. Dr. Olga Alexeeva discusses disclosing a diagnosis of PNES to your patients in this episode. Show reference:  https://pubmed.ncbi.nlm.nih.gov/26633963/ This podcast is sponsored by argenx. Visit www.vyvgarthcp.com for more information.

In part one of this three-part series on non-epileptic events. In this episode, Dr. Andy Ho Wing Chan provides an overview of psychogenic non-epileptic seizures (PNES). Show reference: https://onlinelibrary.wiley.com/doi/10.1002/epi4.12060 

Drs. Jeff Ratliff, Dara V. F. Albert, and Afsaneh Talai discuss understanding the gaps in resident education on psychogenic nonepileptic seizures. Show reference: https://www.neurology.org/doi/10.1212/NE9.0000000000200111  This podcast is sponsored by argenx. Visit www.vyvgarthcp.com for more information.

Dr. Jeff Ratliff talks with Drs. Dara V. F. Albert and Afsaneh Talai about understanding the gaps in resident education on psychogenic nonepileptic seizures. Read the related article in Neurology: Education.  Disclosures can be found at Neurology.org.

Sex has always had its share of misinformation, even as it relates to some medical conditions- like epilepsy. "Coitus brevis epilepsia est" ("Sex is a brief seizure") is an ancient proverb attributed to Galen, the influential Greek physician and philosopher in the Roman Empire. Then, in the 18th and 19th centuries, some physicians, including Samuel-Auguste Tissot and Edward Sieveking, argued that excessive masturbation could cause epilepsy! At the time, castration and clitoridectomy (removal of the clitoris) were reportedly performed on people with severe epilepsy. Terrible! Thankfully we now are all SURE that sex does not CAUSE epilepsy, but sexual release (orgasm) and seizure like activity are indeed similar. Can orgasm trigger epilepsy? And is the reverse possible: can seizures give orgasm like effect? How is PNES related to this? We will review this fascinating clinical conundrum in this episode.

Ny lokasjon når Marius inviterer Jan Erik og Christian inn i peisestua på Ørejordet med Hjælmen på telefon fra den andre byen. Det blir selvsagt prat forarbeidet til Bo Hegland og selvsagt scoringen til Alexander Håpnes. Marius øyner muligheter i Sogndal og det blir selvsagt prat om billetthåndteringen inn mot kampen mot Glomme.

Siste nytt fra VG.

You've probably heard of seizures, maybe even seen or experienced them in real life. But have you heard of PNES? Psychogenic Nonepileptic Seizures (PNES) are seizure-like events that occur due to psychological triggers. They are very distressing, especially for the folks experiencing them, and are sometimes elusive for medical providers to uncover. Join us this episode as we unpack the mystery of these seizure lookalikes and how they impact the lives of women, in particular! Join the From Skirts To Scrubs community and meet us at the intersection of feminism, medicine, and history!Follow us on socials:Instagram: @fromskirtstoscrubs Facebook: @fromskirtstoscrubs TikTok: @fromskirtstoscrubsTwitter: @FSTS_Podcast 

Functional seizures, also known as psychogenic nonepileptic seizures (PNES), resemble epileptic seizures. Unlike epilepsy, they are not prompted by any electrical activity in the brain. People with functional seizures live with stigma, stress, and emotional and financial burden — and so do their care partners.Joy Mazur spoke with Shannon Guinard, who cares for her husband, about the challenges and stigmas that face care partners of people with functional seizures. Resources and relevant articles:  Information about psychogenic non epileptic seizuresDepression and anxiety in caregivers of patients with functional seizures Tsamakis K, et al., 2023 Social aspects of life in patients with functional seizures: Closing the gap in the biopsychosocial formulation Asadi-Pooya AA, et al., 2021Welfare consequences for people diagnosed with nonepileptic seizures: A matched nationwide study in Denmark Jennum, et al., 2019 Support the showSharp Waves episodes are meant for informational purposes only, and not as clinical or medical advice.The International League Against Epilepsy is the world's preeminent association of health professionals and scientists, working toward a world where no person's life is limited by epilepsy. Visit us on Facebook, Twitter, and Instagram.

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2023.04.23.537976v1?rss=1 Authors: Vranic-Peters, M. J., O'brien, P., Seneviratne, U., Reynolds, A., Lai, A., Grayden, D. B., Cook, M., Peterson, A. Abstract: Studying states and state transitions in the brain is challenging due to the nonlinear, complex dynamics present. In this research, we analyse the brain's response to non-invasive perturbations. Perturbation techniques offer a powerful method for studying complex dynamics, though their translation to human brain data is under-explored. This method involves applying small inputs, in this case via photic stimulation, to a system and measuring its response. Sensitivity to perturbations can forewarn a state transition, therefore biomarkers of the brain's perturbation response or 'cortical excitability' could be used to indicate seizure transitions. However, perturbing the brain often involves invasive intracranial surgeries or expensive equipment as in transcranial magnetic stimulation (TMS) which is only accessible to a minority of patient groups, or animal model studies. Photic stimulation is a widely used diagnostic technique in epilepsy that can be used as a non-invasive perturbation paradigm to probe brain dynamics during routine electroencephalography (EEG) studies in humans. This involves changing the frequency of strobing light, sometimes triggering a photo-paroxysmal response (PPR), which is an electrographic event that can be studied as a state transition to a seizure state. We investigate alterations in the response to these perturbations in patients with genetic generalised epilepsy (GGE), with (n=10) and without (n=10) PPR, and patients with psychogenic non-epileptic seizures (PNES; n=10), compared to resting controls (n=10). Metrics of EEG time-series data were evaluated as biomarkers of the perturbation response including variance, autocorrelation, and phase-based synchrony measures. We observed considerable differences in all group biomarker distributions during stimulation compared to controls. In particular, variance and autocorrelation demonstrated greater changes in epochs close to PPR transitions compared to earlier stimulation epochs. Comparison of PPR and spontaneous seizure morphology found them indistinguishable, suggesting PPR is a valid proxy for seizure dynamics. Also, as expected, posterior channels demonstrated the greatest change in synchrony measures, possibly reflecting underlying PPR pathophysiology mechanisms. We clearly demonstrate observable changes at a group level in cortical excitability in epilepsy patients as a response to perturbation in EEG data. Our work re-frames photic stimulation as a non-invasive perturbation paradigm capable of inducing measurable changes to brain dynamics. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC

Guro og Hallvard åpner garasjeporten for første gang i 2023. Vi har pratet med ChatGPT, Hallvards mamma anbefaler kefir til bruk i sjokoladekake og vi tar deg fra A til Å i teknologiverden!

The June Bug System is one of the most fascinating people I've ever talked with. They have over 30 personalities or alters, including a cat, men and women, left and right handed people, and even a non potty trained toddler. They also suffer from PNES (psychogenic non epileptic seizures) and PTSD from growing up with extreme trauma. Life gets complicated for The June Bug System, and maybe more so for their partner Cody, as they are parents of a new baby girl. Their main passion is spreading mental health awareness and ending stigmas around DID. The June Bug System on TikTok @junebugsystem and on Instagram @thejunebugssystemlittles Follow Bunny Hugs and Mental Health on Facebook https://www.facebook.com/Bunny-Hugs-and-Mental-Health-103055408571486 on Instagram https://www.instagram.com/bunnyhugspodcast/?hl=en Twitter https://twitter.com/HugsMental TikTok https://tiktok.com/@bunnyhugspodcast/ Check out my children book at https://www.amazon.ca/Sometimes-Daddy-Cries-Todd-Rennebohm/dp/0228834678 Bunny Hugs and Mental Health is currently on the Top Ten Best Canadian Mental Health podcasts list! https://blog.feedspot.com/canadian_mental_health_podcasts/ And the Top 100 Best Mental Health Podcasts on the internet! https://blog.feedspot.com/mental_health_podcasts/

"Tyven kommer bare for å stjele, drepe og ødelegge. Jeg er kommet for at dere skal ha liv og overflod." Joh. 10, 10 Det nærmer seg jul, og vi skal igjen få feire at Jesus kom i menneskeskikkelse og for å frelse oss. Han som er selve livet, er den som kan gi oss liv, både før og etter døden, sier Bibelen (Joh. 11, 25-26; 17, 2-3). Å kjenne Gud og Jesus er et evige liv, og det begynner allerede her! Han gir oss ikke bare liv, men også overflod. Først og fremst overflod av håp! Som det står i Romerene 15: "Måtte håpets Gud fylle dere med all glede og fred i troen, så dere kan bli rike på håp ved Den Hellige Ånds kraft." (v. 13) – det vil si bugne av håp, boble over av håp. Det er overflod! Håp er mangelvare i verden i dag, men vi har fått i gave en utømmelig Kilde. Det er ikke en gave vi må vente med å åpne, slik som det noen ganger kan stå på ei pakke: "åpnes ikke før julaften!". Jeg ER kommet, sa Jesus. Han er her allerede! Måtte vi gi rom for Ham i hjerte og sinn denne adventstida, så liv og håp kan bli født på ny i oss. Da har vi virkelig grunn til å feire når jula kommer! Les om Håpets Kvinner, som står bak denne serien på www.haapetskvinner.no

27.10.2022 | Forkynnerseminar 11. samling

I denne episoden får du møte ergoterapeut Helene M Leten som jobber på spesialsykehuset for epilepsi i Bærum. Hjernen er et spennende fagfelt og vi får et lite innblikk i hvilken betydning denne diagnosen kan ha på hverdagslivet. Hvilke ergoterapeutiske verktøy blir brukt i møte med pasienter med epilepsi? Og hva er PNES? Og hva gjør Helene til en rågod ergoterapeut? Det finner du ut av i denne episoden. Hosted on Acast. See acast.com/privacy for more information.

Spiller nummer 15: Alexander Håpnes er på besøk hos Jan Erik og Christian. Marius er med litt via telefon til overraskende god lyd. Hjælmen var opptatt med turn. Vi prater om kampen mot Odd 2, kommende kamp borte mot Strømmen, rekruttlagets kamp og landslaget. Samtidig vipper vi innom supportere og at Moss Fotballklubb har et stort bortefølge land og strand rundt.

First, the diagnosis was epilepsy. Then, a host of other symptoms besides seizures were identified and treated. By the time Mary Held  finally found a doctor who landed on the right intervention, she'd lost mobility and nearly all the ability to work and live as she'd been previously accustomed.You will learn:*Trauma can create lasting debilitating physical and emotional harm.*Some people are falsely diagnosed with epilepsy actually have PNES  (Psychogenic Non Epileptic Seizures).*EMDR, or Eye Movement Desensitization and Reprocessing, is a highly effective way to alleviate stress regarding traumatic memories.For show notes, click HERE.

Hear about what Psychogenic Non-Epileptic Seizures are, and how children experiencing them can be effectively treated. - with paediatric clinical psychologist Tyson Sawchuk - from the University of Calgary (Canada).---More about Tyson: https://www.torierobinson.com/epilepsy-sparks-insights/tyson-sawchuk---Glossary:There are many terms used in this recording which you can find in the Epilepsy Sparks Glossary: https://www.epilepsysparks.com/glossary---Follow Torie on:- Twitter: https://twitter.com/torierobinson10- LinkedIn: https://www.linkedin.com/torierobinson- Instagram: https://www.instagram.com/torierobinson10- Facebook: https://www.facebook.com/TorieRobinsonSpeaker- Check out the website: https://www.torierobinson.com

Dr. Michael Kentris discusses psychogenic non-epileptic spells [PNES].

2021 has been quite the strange year. Dave Paddon brings us this year's recitation, Christmas Volunturr, to try and sum it all up. All week we will be bringing you archival tape from the 1983 series I Well Minds The Time that Here and Now created to showcase stories from Labrador. Today we hear an interview host Mike Aucoin did with Henry John Williams. Putting on the kettle during the Christmas season is customary in Labrador, but why not spice it up a little this year. We find out how to make delicious chai tea from Kamaljeet Kaur and Harmanpreet Kaur, two students from India living in Happy Valley-Goose Bay. For day 10 of our 12 Days of Christmas book giveaway we hear from Debbie Samson about her book A Recipe for Complex PTSD and PNES. The Sheshatshiu Innu School held a friendly competition before school was out for the holidays to decorate 70 gingerbread houses that were donated for students. We hear from them to get some gingerbread house building advice. A social media campaign is encouraging conversations around alcohol culture and promoting alcohol-free activities to help people avoid fetal alcohol spectrum disorder. We speak about the initiative with Katharine Dunbar-Windsor, executive director of FASD NL. Finally, according to a recent report from the province's Attorney General, the provincial government did not adequately communicate its options regarding the wetland capping of Muskrat Falls. Rodd Laing, Director of Environment with the Nunatsiavut Government, joins us to give his reaction to the report.

Today we are talking to Nathan Pevy, a Ph.D. student from Sheffield Uni looking into the differences between types of losses of consciousness - covering epilepsy, PNES (Psychogenic Non-Epileptic Seizures)/NEAD (Non-Epileptic Attack Disorder), and Syncope (fainting); to figure out how to more effectively diagnose each. After all, to many, each can look pretty similar and we aren't keen on misdiagnoses… Nathan has both a degree in psychology and a master's in computational neuroscience, and is now mixing the two in his Ph.D. to benefit people affected by the aforementioned!**GET INVOLVED IN THE PROJECT**“Using a 'digital doctor' for diagnosis”: https://theipep.shef.ac.uk/projectSummary Patient Information Sheet: https://theipep.shef.ac.uk/patientInformationExternal Witness Information Sheet: https://theipep.shef.ac.uk/witnessInformationExternal Consent to Contact Form: https://theipep.shef.ac.uk/interest **ANY QUESTIONS FOR NATHAN?**ndpevy1@sheffield.ac.uk **CHECK OUT NATHAN**https://twitter.com/nathanpevySheffield Uni: https://www.sheffield.ac.uk/engineering/about/equality-diversity-inclusion/nathans-story **CHECK OUT THE YOUTUBE VIDEO WITH NATHAN**YouTube: https://www.youtube.com/c/TorieRobinson/videos **CONNECT WITH TORIE**Website: https://www.torierobinson.comTwitter: https://twitter.com/torierobinson10LinkedIn: https://www.linkedin.com/torierobinsonFacebook: https://www.facebook.com/TorieRobinsonSpeaker **CHECK OUT TORIE'S YOUTUBE & BLOG**YouTube: https://www.youtube.com/c/TorieRobinsonBlog: https://www.torierobinson.com/blog**HIRE TORIE AS A SPEAKER ON EPILEPSY, MENTAL HEALTH, DISABILITY, & DIVERSITY**https://www.torierobinson.com/contact

Siste nytt fra VG.

Covid-19 kan nå suge hard og lenge på ett egg, for nå har vi åpnet Norge igjen! FH har fått seg ny jobb, og Steffen har stjålet en idé fra Who's Line og Friminutt som han vil teste på de andre. Support the show (https://www.facebook.com/Sannerdet/)

Siste nytt fra VG 24. september 2021.

Norge skal endelig gjenåpnes, eller nei forresten, vi skal vente litt til. Regjeringsingssamtalene er i gang på beste Oslo vest. Journalistikkens Obi-Wan Kenobi, Bob Woodward, er ute med ny bok om tidligere president Donald Trump. Med Anders Giæver, HAnne Skartveit, Astrid Meland og Per Olav Ødegård. Vaktsjef Kristina Kinne. Produsent Magne Antonsen.

Join me as I speak with Author Debbie A Samson. Her autobiography, " A Recipe for C-PTSD and PNES: A True Story of Determination and Hope." From her website: Debbie Samson allows the reader to see into her lifetime of abuse. She shows how she slowly let go of anger, sadness, grief, and trauma and replaced it with love, happiness, forgiveness, tolerance, determination, and gratitude. A Recipe for C-PTSD and PNES will open the public's eyes and show how the effects of childhood trauma and abuse can be a recipe for mental illness. Her book is available now. You can from her book on Amazon and where books are sold. You can find out more about Debbie and her book at: https://debbiesamson.com/ https://www.facebook.com/debbieasamson https://www.instagram.com/debbie_a_samson/ Find out more atThe Trauma Room Podcast: https://linktr.ee/thetraumaroompodcast

Stratus' Chief Medical Officer, Dr. Jeremy Slater, discusses psychogenic non-epileptic seizures, also known as PNES.

Kan venner kjøpes for den rette summen? Norge skal tydeligvis åpnes opp igjen, og vi er ikke enig i dette, og vi snakker om nostalgiske ting fra barndommen vår. Dette og mye mer får du i dagens episode! Support the show (https://www.facebook.com/Sannerdet/)

Contributor:  Katie Sprinkel, MD Educational Pearls: Psychogenic Non-Epileptic Seizures (PNES) are due to a psychogenic rather than an epileptic cause Despite common assumption, PNES are not always volitional  20-40% of those with PNES can also have true epileptic seizures 20-40% True diagnosis requires a video EEG  Characteristics of PNES include: Waxing and waning of the seizure intensity Eyes clenched shut Pelvic thrusting, rolling from side-to-side Ability to respond to verbal stimuli during the seizure  Ability to recall information during the seizure Weeping or stuttering Guarding the face on passive hand drop Characteristics of epileptic seizures: Tongue biting Prolonged postictal state Incontinence Haldol or Zyprexa may be better for PNES and benzodiazepines tend to be better for epileptic seizures References Huff JS, Murr N. Psychogenic Nonepileptic Seizures. 2021 Jan 28. In: StatPearls [Internet]. Treasure Island (FL): StatPearls Publishing; 2021 Jan–. PMID: 28722901. Asadi-Pooya AA. Psychogenic nonepileptic seizures: a concise review. Neurol Sci. 2017 Jun;38(6):935-940. doi: 10.1007/s10072-017-2887-8. Epub 2017 Mar 8. PMID: 28275874.   Summarized by John Spartz, MS3 | Edited by Erik Verzemnieks, MD

Unstoppable tics, convulsions, and sore days, yup! I know what that's like.Today we are talking about non-epileptic seizures also known as psychogenic seizures or PNES. I have been dealing with my TS since I was a child but lately I have struggled with seizures that are becoming more intense and more frequent. We are also discussing the importance of advocating for mental health so that we can make mental health affordable for everyone and anyone. Episode mentions:Tourette Syndrome Association of America The Warrior Room (Online wellness training)Music by Ghostshaft

Dette er siste nytt fra VG.

It’s the JournalFeed Podcast for the week of Sept. 7-11, 2020. We cover NSAIDs for low back pain, the HEART pathway one year out, facial palsy as a manifestation of pediatric leukemia, increased mortality in PNES patients, and COVID-19 secondary attack rates.

This podcast presents an approach to the assessment, diagnosis, and multidisciplinary management of psychogenic non-epileptic seizures (PNES) in the pediatric population. It was developed by Vivienne Beard, a second-year medical student at UBC, in collaboration with Dr. Mary Connolly (Department of Pediatrics, UBC) and Dr. Andrea Chapman (Department of Psychiatry, UBC).

Massegravene fylles i Brasil, mens president Bolsonaro trekker på skuldrene. Nå åpnes landet, hvordan skal landet komme seg ut av helsekrisen i god behold? Med overlege Morten Rostrup og professor ved UIO Benedicte Bull. See acast.com/privacy for privacy and opt-out information.

Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2020.06.12.147777v1?rss=1 Authors: van Rosmalen, L., van Dalum, J., Hazlerigg, D. G., Hut, R. A. Abstract: To optimally time reproduction, seasonal mammals use a photoperiodic neuroendocrine system (PNES) that measures photoperiod and subsequently drives reproduction. To adapt to late spring arrival at northern latitudes, a lower photoperiodic sensitivity and therefore a higher critical photoperiod for reproductive onset is necessary in northern species to arrest reproductive development until spring onset. Temperature-photoperiod relationships, and hence food availability-photoperiod relationships, are highly latitude dependent. Therefore, we predict PNES sensitivity characteristics to be latitude-dependent. Here, we investigated photoperiodic responses at different times during development in northern- (tundra/root vole, Microtus oeconomus) and southern vole species (common vole, Microtus arvalis) exposed to constant short (SP) or long photoperiod (LP). M. oeconomus grows faster under LP, whereas no photoperiodic effect on somatic growth is observed in M. arvalis. Contrastingly, gonadal growth is more sensitive to photoperiod in M. arvalis, suggesting that photoperiodic responses in somatic and gonadal growth can be plastic, and might be regulated through different mechanisms. In both species, thyroid-stimulating-hormone-{beta} subunit (Tsh{beta}) and iodothyronine-deiodines 2 (Dio2) expression is highly increased under LP, whereas Tshr and Dio3 decreases under LP. High Tshr levels in voles raised under SP may lead to increased sensitivity to increasing photoperiods later in life. The higher photoperiodic induced Tshr response in M. oeconomus suggests that the northern vole species might be more sensitive to TSH when raised under SP. Species differences in developmental programming of the PNES, which is dependent on photoperiod early in development, may form part divergent breeding strategies evolving as part of latitudinal adaptation. Copy rights belong to original authors. Visit the link for more info

Episode Notes In this episode of Quarantine & Chill, I sit down with Maria Morrero as we talk about her experiences with invisible illnesses like PTSD and PNES, as well as her experiences as a black woman trying to access medical care. We also have loads of laughs. It was really fun to sit with her and talk about her experiences during COVID19. Enjoy! You can reach out to Maria by e-mail at: morrero@umich.edu If you want to be a part of a Quarantine & Chill Episode, be sure to e-mail us at disabilityafterdark@gmail.com using the subject line "Quarantine and Chill". Thank you for listening! This podcast is powered by Pinecast.

Før korona kom til Norge intervjuet vi utvekslingsstudenten Maren om koronasituasjonen i Kina. Vi tenkte det var gøy å høre det igjen. Vi snakker med førsteplassen fra Studentvalget 2020 ved Universitetet i Oslo - Venstrealliansen! Studentnyhetene presenterer: 10 ting vi ikke gjorde før korona Oslos universiteter og høyskoler er åpne igjen. Vi tok en tur innom noen av bruktbutikkene som finnes i Oslo før Korona. Vi gir deg helgens værmelding som alltid. I studio: Line Rørvik og Nora Torgersen Tekniker: Magnus Thune

Denne uken begynner gjenåpningen av New York. Men det er kun enkelte steder langt unna millionbyen New York City som får pusterom. Korrespondentene må fortsatt regne med flere uker med lockdown.  I mellomtiden diskuterer vi president Donald Trumps pressekonferanse, der han stormet ut etter å ha fått kritiske spørsmål fra to kvinnelige journalister, og Trumps forsøk på å beskylde sin forgjenger Barack Obama for å ha drevet en heksejakt på Michael Flynn. 

Nå åpnes Norge igjen etter åtte uker, regjeringen presenterer planene for hvordan og når, direkte i Dagsnytt 18 Men klarer skolene å holde åpne, og samtidig overholde smittevernreglene? Eller trenger de ikke? Og hvis de ikke trenger, hvorfor skulle de holde stengt da? Også idretten skal få starte opp igjen, men ikke alle, og ikke på vanlig måte. For første gang i historien senket Norges Bank renten til...null, men hvem kommer det til å hjelpe, egentlig? Velkommen til Dagsnytt 18 med Espen Aas.

Dette er siste nytt fra VG, klokken 06:30.

Book a FREE PTSD recovery consultation with our team here: https://www.overcomingptsd.info/call Today Brad and I talk about another big myth that blocks a lot of people from recovery... The belief that other things in their life block them from healing. We lovingly call it "alphabet soup". PTSD, CPTSD, PNES, BPD, GAD, etc. are all labels that can seriously hut someones chances at full recovery... This tricky because PTSD is on of the most misdiagnosed things in the field of mental health... If your misdiagnosed, how are you going to know what help you really need!? You'll likely end up confused and feeling stuck with a lack of direction and not knowing where to turn or what to do. In this episode Brad and I bust this harmful myth for you and give you some incredible steps to move forward without even having a "diagnosis"! Hope you enjoy the episode! Kayleen PS Here's the link to the free PTSD recovery consultation we promised - https://www.overcomingptsd.info/call On the call we'll talk specifically about YOUR situation. We'll help you get clarity on where you are right now and where you'd like to be on the other side of PTSD. Then we'll help you bridge the gap between the two and give you resources to get you *exactly* where you need to go in the shortest amount of time possible.

Utenriksreporter Arild Inge Olsson stiller spørsmålet om svenskene egentlig gjør det riktige i møtet med koronaen. Kommentator Harald Birkevold snakker om den vanskelige åpningen av det norske samfunnet. Jan Zahl lurer på hva som skjer i politikken når dette er over, og Leif Tore Lindø mottar hets fra absolutt alle kanter, inkludert fra sin egen familie. See acast.com/privacy for privacy and opt-out information.

Statsminister Erna Solberg har i dag annonsert at Norge skal åpnes opp igjen gradvis fra og med 20. april. Hvordan kan vi forvente at dette vil slå ut for norsk økonomi fremover? I denne episoden deler DNBs sjeføkonom Kjersti Haugland sitt førsteinntrykk av endringene. Programleder: Marius Brun Haugen, DNB Markets Produsent: Kim A. Farago, DNB Markets

Det er ingen aprilspøk, det åpnes for spilling av golf utendørs, MEN husk å følg nytt regelverk.

Det er sesongavslutning, og vi lukker Eurovision-kapittelet for denne gang med en siste prat om avgjørelsen til NRK, og vi har kanskje funnet den beste løsningen på hele problemet.

MINUTAGEM [02:01] Salves! [03:34] Apresentação Inicial do Caso Clínico [04:35] Diferenciais de perda do nível de consciência [16:50] Anamnese (revisão de prontuário) [19:14] Exame físico [21:30] Diferenciação da sincope por mecanismos [34:28] Representação do problema [38:13] Quais exames pedir? [40:33] Resultados de exames [43:00] Discussão dos exames e hipóteses iniciais para o caso [49:13] Resultado de novos exames [56:30] Fechamento do caso [1:00:00] Desafio da semana Um caso clínico desafiador de síncope! Fred, Pedro e Rapha discutem esse caso apresentado pelo João. Como diferenciar síncope de convulsão? Quais características sugerem uma síncope de causa cardíaca? Tentamos responder essas e outras perguntas nesse episódio. Ficou com alguma dúvida, quer apontar algum erro ou enviar sugestões? Entra em contato com a gente no Instagram @tadeclinicagem ou no e-mail tadeclinicagem@gmail.com! REFERÊNCIAS: JURASCHEK, Stephen P. et al. Association of history of dizziness and long-term adverse outcomes with early vs later orthostatic hypotension assessment times in middle-aged adults. JAMA internal medicine, v. 177, n. 9, p. 1316-1323, 2017. BRIGNOLE, Michele et al. 2018 ESC Guidelines for the diagnosis and management of syncope. European heart journal, v. 39, n. 21, p. 1883-1948, 2018 Shmuely, Sharon, et al. "Differentiating motor phenomena in tilt-induced syncope and convulsive seizures." Neurology 90.15 (2018): e1339-e1346. Fisher, Robert S. "Serum prolactin in seizure diagnosis: Glass half-full or half-empty?." (2016): 100-101. Abubakr, Abuhuziefa, and Ilse Wambacq. "Diagnostic value of serum prolactin levels in PNES in the epilepsy monitoring unit." Neurology: Clinical Practice 6.2 (2016): 116-119. Albassam, Omar T., et al. "Did this patient have cardiac syncope?: the rational clinical examination systematic review." Jama 321.24 (2019): 2448-2457.

Not Myself Anymore CRPS has stolen my identity. I never imagined that CRPS, POTS, PNES, DID, et al. could change me so much—and not in a good way. I have become paralyzed in the pain. I hardly leave the house because I’m afraid I’ll have a seizure and/or fall down. I’m afraid of the pain […]

http://www.transferoutpnes.com So very grateful for the brave souls who choose to stand up to share their message of hope. First and foremost - you are not alone. Now, here is a hefty dose of hope!

http://www.transferoutpnes.com. It is such an honor and privilege to be able to share this conversation with you between Tonee and myself. Tonee is a Mental Health Counselor who happens to have PNES. She intimately invites us into this journey from diagnosis through recovery! Thanks Tonee!!

I dag åpnes gigantfeltet Johan Sverdrup i Nordsjøen. Oljå skal opp i femti år til. Det er grunn til å feire, mener Sylvi Listhaug som er på vei ut til feltet Og Jonas Gahr Støre, som sitter klar her i studio. MDG er ikke enig. Sverdrup er en gigantisk feilinvestering, sier Une Bastholm.

If you have PNES or PSTD and are tired of living in the conditions you're in - you have stopped at the right place! Success begins with attitude, so throw your hands up and surrender yourself to the process of dealing and healing. Interested in more? Email me at Christine.Mauriello32@gmail.com or visit https://www.TransferOutPNES.com

"Kāpnes ir tās, kas cilvēku ratiņkrēslā padara nespējīgu, nevis ratiņkrēsls," 1997. gadā atzīmēja politikas plānotāji Dienvidāfrikā. Šonedēļ raidījumā runāsim par pieejamu un iekļaujošu dizainu. Par iespēju ikkatram sabiedrības loceklim lietot produktus vai saņemt pakalpojumus neatkarīgi no viņu individuālajām atšķirībām. Raidījumā piedalās arhitekte un jaunuzņēmuma Vividly dibinātāja un vadītāja Gunita Kuļikovska un uzņēmuma Turn Digital vadītājs, digitālās vides piekļūstamības eksperts un platformas www.pieklustamiba.lv iniciators Pēteris Jurčenko. Īsumā * Pieejams dizains produktu vai pakalpojumu ļauj lietot jebkuram cilvēkam neatkarīgi no apstākļiem vai fizioloģiskām vajadzībām. * Dizaineri un arhitekti ir līdzatbildīgi apstākļu radīšanā, kas definē invaliditāti. * Nevis nevēlēšanās, bet neaizdomāšanās rada nepieejamu dizainu. * Pēteris Jurčenko: Neiekļaujošs dizains ir diskriminācija. * Gunita Kuļikovska: Dizaineru uzdevums – apzināties, kuras grupas tiek izslēgtas, veidojot dizainu. * Pēteris Jurčenko: Dizaina izglītībā kopumā vēl šī domāšana netiek audzināta. Vairāk un plašāk šeit!

If you love someone with PNES or NEAD you may doubt yourself - you may blame yourself at times or feel helpless. Go and put the oxygen mask on yourself, only then can you truly help someone else! With love and gratitude for all you do! Need more? Visit https://www.TransferOutPNES.com

In this podcast, Barbara Ann Dworetzky, MD, outlines the challenges associated with diagnosing psychogenic nonepileptic seizures (PNES), how neurologists and other providers can overcome these challenges, and more. More at: www.consultant360.com/neurology.

Becky Tilahun, PhD, and Jocelyn Bautista, MD, discuss the challenges of psychogenic non-epileptic seizure (PNES) diagnosis and their multi-disciplinary approach to treating and supporting patients with PNES.

If you love someone with PNES, you may feel at times that you don't know what to do. Sometimes, the simplest actions of support is all that is necessary and appreciated. If you have found this helpful, please share it! If you want more help, visit https://www.TransferOutPNES.com

We review the entity formally known as pseudoseizures and psychogenic non-epileptic seizures (PNES), now referred to as psychogenic non-epileptic attacks (PNEA).  We discuss the history of PNEA as well as relevant literature on the diagnosis and treatment of this entity. Thanks for listening! Jeremy Faust and Lauren Westafer

Keri is baaackkk! She follows up on her last episode talking about her health and diagnosis. Talks about coping with it and how to move forward.Support the show (https://www.patreon.com/kerionthepodcast)

Author: Gretchen Hinson, M.D. Educational Pearls:   PNES vs. epilepsy: postictal state is diagnostic of an epileptic seizure (sonorous respirations and/or confusion, lasting typically 20-30 minutes); Epileptiform seizures show decrease in convulsion frequency, but increase in convulsion amplitude while PNES convulsions demonstrate episodic convulsion amplitudes; and epileptiform seizures usually do not pause. PNES is a form of conversion disorder and can be associated with underlying personality disorder; however there are patients with epilepsy that also can have PNES which complicates the diagnosis and treatment.  Patients that are malingering may have flailing movements and might talk during the episodes - both not typical of epileptic seizures or PNES. Treatment for PNES is with psychotropic medications and psychotherapy as opposed to antiepileptic medications   References: Avbersek, A; Sisodiya, S. (2010). Does the primary literature provide support for clinical signs used to distinguish psychogenic nonepileptic seizures from epileptic seizures?.  Journal of neurology, neurosurgery, and psychiatry. 81(7):719-25. Devinsky, O; Gazzola, D; LaFrance, W. Curt (2011). Differentiating between nonepileptic and epileptic seizures. Nature Reviews. Neurology. 7 (4): 210–220. Lesser, RP. (2003). Treatment and Outcome of Psychogenic Nonepileptic Seizures. Epilepsy Currents. 3(6):198-200. doi:10.1046/j.1535-7597.2003.03601.x. Pillaia, JA; Hautab SR. (2012). Patients with epilepsy and psychogenic non-epileptic seizures: An inpatient video-EEG monitoring study. Seizure. 21(1): 24-27.

Walker Foland is an emergency physician practicing in Michigan and in this episode breaks down why pseudoseizures, now termed PNES (Psychogenic Nonepileptic Seizures), are a real disease.   Sign up for the ERcast mailing list Are patients with PNES ‘faking it’? PNES is a conversion disorder: an unconscious manifestation of psychological trauma. Walker treats PNES patients with haloperidol or olanzapine with the thinking that this is psychological, not true epilepsy PNES is not ‘faking it’ or lying   Challenges Patients with PNES may also have true epileptic seizures Diagnosing PNES, or separating it from epilepsy, may take video EEG monitoring, a neurologist, and sometimes prolonged periods of time to figure things out   How to tell the difference between an grand mal epileptic seizure vs PNES vs faking it? PNES Seizures related to a specific stimulus (sound foods, body movement) Frequency and amplitude of concussions: same frequency through the seizure with varying amplitude. Maintenance of consciousness and may have some of the below may guard the face with passive hand drop resist eyelid opening visual fixation on a mirror Whit Fisher, Dr Procedurettes, squirts water in the face of patients where there is thought of PNES.  If they grimace, probably not an epileptic seizure. Faking Seizures Talking Purposeful movement Avoids injury May use convulsions as a way of harming staff Intermittently awake and vocal during the episode Epileptic seizure Convulsive frequency decreases, amplitude increases as seizure progresses No response to pain Allow passive eye opening   A 2010 article from the Journal of Neurology Neurosurgery and Psychiatry broke down the evidence of what other elements can help distinguish PNES from epileptic seizures. Duration over 2 minutes suggests PNES, but we’ve all seen epileptic seizures last for a long time, status, and some PNES can be super short Happens in sleep. Evidence suggests that if the event happens in sleep, that is probably episode. PNES episodes happen when awake Fluctuating course such as a pause in the rhytmic movement, epileptic seizures usually don’t pause and then restart, a pause favors PNES Flailing. You’d think the flailing patient has PNES for sure because epilepsy doesn’t flail, but it does! Flailing is much more common in PNES, but not so much so that it’s a clear distinguishing factor Urinary incontinence, more common in epilepsy, but does happen in PNES. Post-ictal recovery period. Surely, this is the sine qua non of epilepsy.  It is way way more common following generalized epileptic seizures but happens in around 15% of PNES. The sterterous breathing (noisy, labored) that we see after generalized tonic clonic epileptic seizures suggests epilepsy and is not a characteristic of PNES   Walker’s take home points PNES patients aren’t ‘faking it’ This is a real disorder, it's just not epilepsy References Chen, David K., and W. Curt LaFrance Jr. "Diagnosis and treatment of nonepileptic seizures." CONTINUUM: Lifelong Learning in Neurology 22.1, Epilepsy (2016): 116-131. PMID:26844733 Avbersek, Andreja, and Sanjay Sisodiya. "Does the primary literature provide support for clinical signs used to distinguish psychogenic nonepileptic seizures from epileptic seizures?." Journal of Neurology, Neurosurgery & Psychiatry 81.7 (2010): 719-725.Full Text PMID:20581136  Shen, Wayne, Elizabeth S. Bowman, and Omkar N. Markand. "Presenting the diagnosis of pseudoseizure." Neurology 40.5 (1990): 756-756. Full Text PMID:2330101

London ble igjen rammet av terror tidligere denne uken da en terrorist meide uskyldige fotgjengere ned med bilen sin før han knivstakk og drepte en politimann. Han kunne drepe politimannen, trolig fordi han var ubevæpnet - akkurat slik vårt politi er. Er dette et argument for generell bevæpning i Norge? Ja, fordi sekunder kan utgjøre forskjellen på liv eller død, sier Sigve Bolstad i Politiets Fellesforbund. Nei, det gir lite terrorbeskyttelse for pengene, svarer terrorforsker Thomas Hegghammer. De møter KrF's Kjell Ingolf Ropstad og Underhusets faste makker, Jon Hustad. Yama Wolasmal er som vanlig programleder.

Grønlandshvalene flørter på titalls kilometers avstand. Stemmene er dype og frekvensene lave. Men skipstrafikken lager støy på de samme frekvensene. Og det blir stadig flere båter i Arktis. Så hva gjør de store hannhvalene da for å få kontakt med hunnhvalene? Reporter: Vibeke Røiri

In this episode of Epilepsy.com's Hallway Conversations, Dr. Joseph Sirven, Professor of Neurology at Mayo Clinic Arizona and Editor-in-Chief of Epilepsy.com, interviews Selim R. Benbadis, MD, Director of the Comprehensive Epilepsy Program at Tampa General Hospital about treatment of psychogenic non-epileptic seizures.

v Hans Johan Sagrusten, bibelbrukskonsulent i Bibelselskapet Om å lese bibelen og hvordan bibelen ble til... - Hva er Bibelen egentlig? - Hvorfor gikk en fra skriftrull til bok? - Hva gjør vi når skriften åpnes?

Innslag fra NRK Kulturnytt 23.08.2012