Podcasts about EMG

Do you think that patients that complain of shoulder pain should always be evaluated with an EMG?  Perhaps not, but not all shoulder pain is orthopedic in nature. Tune in to this episode to see how EMG can help when the cause of shoulder pain may not be obvious?

Meta Connect had hiccups—but did it just spark the mainstream AR era? XR developer Tom Krikorian (Studio 84) joins Ely Santos to unpack first impressions of Ray-Ban Display, why the EMG wristband is the real breakthrough, and what devs need before this market explodes. We get candid about Horizon Worlds, Meta's SDK (or lack of it), and Apple's tight ecosystem + on-device AI advantage. In this episode:Hands-on with Ray-Ban Display: one-eye HUD, real-world navigation, where it shines—and where it strains. The wristband wow-factor: finger-level intent control that finally feels ready for prime time. Developer reality check: unstable stacks, shifting roadmaps, and why an SDK + clear monetization path are non-negotiable. Meta vs. Apple: open ambitions vs. end-to-end optimization (iPhone, processors, on-device AI). Who's better positioned? Early use cases that actually stick: notifications without the phone, live captions, travel translation, and accessibility. The Horizon Worlds debate: chasing Roblox—or losing the VR plot? Guest: Tom Krikorian — visionOS/XR developer, Studio 84; Host: Ely Santos — Metavertising Podcast. If you're building for XR—or betting your brand on the next wave of consumer wearables—this is your field guide to what's real, what's hype, and what's coming next.

Send us a textWe share a personal update and then dive into how injuries, curiosity, and tough feedback shaped a modular myoelectric hand that is lighter, repairable in minutes, and priced for wider access. Ryan Saavedra of Alt-Bionics walks through additive manufacturing choices, PDAC approval, and a roadmap linking prosthetics with humanoid robotics.• user and clinician pain points shaping design choices • modular finger architecture for fast field repairs • nylon 12 and MJF for strength, weight, and cost • tolerancing to reduce lash and improve reliability • pricing philosophy, PDAC approval, and reimbursement nuance • EMG control foundations, firmware over AI for stability • balancing service bureaus vs in-house manufacturing • funding from bootstrap to mission-aligned investors • product roadmap focused on hands and higher DoF • cross-pollination between prosthetics and roboticsSpecial thanks to Advanced 3D for sponsoring this episode.Support the show

LEITURA BÍBLICA DO DIA: GÊNESIS 2:8-9; 3:16-19 PLANO DE LEITURA ANUAL: ISAÍAS 59–61; 2 TESSALONICENSES 3   Já fez seu devocional hoje? Aproveite e marque um amigo para fazer junto com você! Confira:  Meu pai amava estar ao ar livre com a criação de Deus, acampar, pescar e colecionar pedras. Ele gostava de trabalhar em seu quintal e jardim. Dava muito trabalho! Passava horas podando, capinando, plantando sementes e flores, arrancando ervas daninhas, cortando a grama e regando o jardim. Os resultados valiam a pena, gramado ajardinado, tomates saborosos e lindas rosas da paz. Todos os anos, ele podava as roseiras rentes ao solo, e elas cresciam, preenchendo os sentidos com fragrância e beleza. Em Gênesis, lemos sobre o jardim do Éden, onde Adão e Eva viveram, prosperaram e caminharam com Deus. Ali, “Deus fez brotar do solo árvores de todas as espécies, árvores lindas que produziam frutos deliciosos” (GÊNESIS 2:9). Imagino que aquele jardim perfeito também tinha flores lindas e perfumadas, talvez até mesmo rosas sem os espinhos! Após a rebelião de Adão e Eva contra Deus, eles foram expulsos do jardim e precisaram plantar e cuidar de seus próprios jardins, o que significava capinar terreno duro, lutar com espinhos e outros desafios (3:17-19,23-24). No entanto, Deus continuou a prover para eles (v.21), e o Senhor não deixou a humanidade sem a beleza da criação para nos atrair a Ele (ROMANOS 1:20). As flores no jardim nos lembram do contínuo amor de Deus e da promessa de uma nova criação — símbolos de esperança e conforto!   Por: ALYSON KIEDA 

Chilvana Patel, MD interviews Devon Rubin, MD on "Lessons Learned in the EMG Lab: Elevating EMG Practice Through International Exchange." Dr. Rubin will be sharing his extensive experience in EMG education and collaborative practice across the countries. That effort is the exchange of best practices and the development of collaborations that ultimately improve patient care and promote health equity worldwide.

Looking for the best dumbbell chest exercises to build size at home, without a bench or fancy setup? In this video, I'm ranking the Top 5 Dumbbell Chest Exercises for Hypertrophy, from good to absolutely essential, so you can build a bigger, fuller chest fast. ► 30% Off Afterburn Dumbbell Program Inside My App: https://www.liveleantv.com/programs/a... (code: Afterburn30) ► Find The Perfect Program - Live Lean Body Quiz: https://www.liveleantv.com/quiz ► Free 7 Day Trial To My Workout App: https://www.liveleantv.com ► Free 7 Day Meal Plan And Recipes: https://www.liveleantv.com/free-stuff I'll break down: ✅ Which exercises activate the most chest muscle (EMG-backed) ✅ Proper form and mistakes to avoid ✅ Bench-free modifications you can do on the floor ✅ Upper, mid, and lower chest targeting for complete development ✅ A pro tip at the end that instantly improves chest activation Timestamps 00:00 Intro 00:35 How I Ranked the Exercises 01:14 Decline Dumbbell Press 02:05 Dumbbell Flyes 02:50 Guillotine Press 03:34 Dumbbell Flat Press 04:12 Dumbbell Incline Press 04:53 Best Chest Activation Tip 05:11 Chest Anatomy & Function 06:19 30% Off My Dumbbell Program Subscribe Here! http://bit.ly/SubLiveLeanTV Check Out Our Top Videos! http://bit.ly/LiveLeanTVTopVideos Read the blog here: https://www.liveleantv.com/best-dumbb... Listen to the podcast here: https://www.liveleantv.com/podcast WANT MORE DAILY TIPS ON HOW TO LIVE LEAN?: ► INSTAGRAM: / bradgouthro ► INSTAGRAM: / jessicagouthrofitness ► INSTAGRAM: / liveleantv ► SNAPCHAT: / bradgouthro ► FACEBOOK: / liveleantv ► TWITTER: / bradgouthro ► TWITTER: / liveleantv ► TIK-TOK: / bradgouthro ► TIK-TOK: / liveleantv #chestworkout #chestday #LiveLeanTV About Live Lean TV: Welcome to Live Lean TV. The online fitness and nutrition show, hosted by Brad and Jessica Gouthro, teaching you how to LIVE THE LEAN LIFESTYLE 365 days a year. Watch hundreds of fat blasting & muscle building workouts, easy and delicious recipes, as well as fitness and nutrition tips to get you your dream body (and maintain it 365 days a year). Make sure you click the SUBSCRIBE button for new fitness and nutrition episodes every week! Business Enquiries: info@LiveLeanTV.com Best Exercises For Chest Growth With Dumbbells At Home (RANKED) • Best Exercises For Chest Growth With Dumbb... Live Lean TV / liveleantv

Myasthenia gravis is een auto-immuunziekte van de neuromusculaire overgang, gekenmerkt door zwakte in specifieke spiergroepen die typisch fluctueert met inspanning en rust. Hoewel het klassieke beeld vaak goed te herkennen is, maken atypische presentaties de diagnose soms lastig. In deze aflevering spreken we met Martijn Tannemaat, neuroloog en klinisch neurofysioloog in het LUMC.  Hij neemt ons mee in de wereld van de myasthene syndromen, met de focus op myasthenia gravis, en hun onderliggende pathofysiologie. Hoe ontstaat autoreactiviteit? Wat is het typische klinische beeld, en welke diagnostiek kun je direct in de spreekkamer inzetten? En hoe onderscheid je dubbelzien door een myastheen syndroom van andere oorzaken?We bespreken de rol van aanvullende diagnostiek, waarin we ingaan op de verschillende (zeldzame) antistoffen, en ook de meerwaarde van klinisch neurofysiologisch onderzoek zoals repetitieve zenuwstimulatie en het single fiber EMG.Verder bespreken we de behandeling van myasthenia gravis, en dan met name de chronische behandeling. Wat heb je aan pyridostigmine? Wanneer moet je immuunmodulerende therapie inzetten? De behandeling van een myasthene crisis komt in een aparte aflevering van Hersenspinsels aan bod.Verder vertelt Martijn over zijn voorliefde voor de KNF en artificial intelligence. Ook doet hij een boekje open over zijn vroegere ambities om neurochirurg te worden, en zijn avontuur tijdens deelname aan het televisieprogramma Peking Express. Zie ook de Richtlijn Myasthenia Gravis versie 2.0 voor de meest recente aanbevelingen:https://www.spierziektencentrum.nl/guideline/myasthenia-gravis-autoimmuun-consensus-richtlijn/Disclosures: M.R. Tannemaat is betaald voor advieswerkzaamheden voor ArgenX, UCB Pharma, Johnson and Johnson, Peervoice en Medtalks, en heeft onderzoeksfinanciering ontvangen van ZonMW, NWO, ArgenX, USB Pharma, NMD Pharma en Huma.  Alle vergoedingen zijn betaald aan het Leids Universitair Medisch Centrum. Hij is lid van het European Reference Network for Rare Neuromuscular Diseases [ERN EURO-NMD].

While genetic testing has replaced muscle biopsy in the diagnosis of many genetic myopathies, clinical assessment and the integration of clinical and laboratory findings remain key elements for the diagnosis and treatment of muscle diseases. In this episode, Casey Albin, MD, speaks with Margherita Milone, MD, PhD, FAAN, FANA, author of the article “A Pattern Recognition Approach to Myopathy” in the Continuum® October 2025 Muscle and Neuromuscular Junction Disorders issue. Dr. Albin is a Continuum® Audio interviewer, associate editor of media engagement, and an assistant professor of neurology and neurosurgery at Emory University School of Medicine in Atlanta, Georgia. Dr. Milone is a professor of neurology and the director of the Muscle Pathology Laboratory at Mayo Clinic College of Medicine and Science in Rochester, Minnesota. Additional Resources Read the article: A Pattern Recognition Approach to Myopathy Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @caseyalbin Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Albin: Hello, this is Dr Casey Albin. Today I'm interviewing Dr Margherita Milone on her article on a pattern recognition approach to myopathy, which appears in the October 2025 Continuum issue on muscle and neuromuscular junction disorders. Welcome to the podcast, Dr Milone. Thank you so much for joining us. I'll start off by having you introduce yourself to our listeners. Dr Milone: Hello Casey, thank you so much for this interview and for bringing the attention to the article on muscle diseases. So, I'm Margherita Milone. I'm one of the neuromuscular neurologists at Mayo Clinic in Rochester. I have been interested in muscle disorders since I was a neurology resident many years ago. Muscle diseases are the focus of my clinical practice and research interest. Dr Albin: Wonderful. Thank you so much. When I think about myopathies, I generally tend to think of three large buckets: the genetic myopathy, the inflammatory myopathies, and then the necrotizing myopathies. Is that a reasonable approach to conceptualizing these myopathies? Dr Milone: Yeah, the ideology of the myopathies can be quite broad. And yes, we have a large group of genetic muscle diseases, which are the most common. And then we have immune-mediated muscle diseases, which include inflammatory myopathies as well as some form of necrotizing myopathies. Then we have some metabolic myopathies, which could be acquired or could be genetic. And then there are muscle diseases that are due to toxins as well as to infection. Dr Albin: Wow. So, lots of different etiologies. And that really struck me about your article, is that these can present in really heterogeneous ways, and some of them don't really read the rule book. So, we have to have a really high level of suspicion, for someone who's coming in with weakness, to remember to think about a myopathy. One of the things that I like to do is try to take us through a little bit of a case to sort of walk us through how you would approach if someone comes in. So, let's say you get, you know, a forty-year-old woman, and she's presenting with several months of progressive weakness. And she says that even recently she's noted just a little bit of difficulty swallowing. It feels to her like things are getting stuck. What are some of the things when you are approaching the history that would help you tease this to a myopathy instead of so many other things that can cause a patient to be weak? Dr Milone: Yes. So, as you mentioned, people who have a muscle disease have the muscle weakness often, but the muscle weakness is not just specific for a muscle disease. Because you can have a mass weakness in somebody who has a neurogenic paralysis. The problem with diagnosis of muscle diseases is that patients with these disorders have a limited number of symptom and sign that does not match the large heterogeneity of the etiology. So, in someone who has weakness, that weakness could represent a muscle disease, could represent an anterior horn cell disease, could represent a defect of neuromuscular junction. The clinical history of weakness is not sufficient by itself to make you think about a muscle disease. You have to keep that in the differential diagnosis. But your examination will help in corroborating your suspicion of a muscle disease. Let's say if you have a patient, the patient that you described, with six months' history of progressive weakness, dysphagia, and that patient has normal reflexes, and the patient has no clinical evidence for muscle fatigability and no sensory loss, then the probability that that patient has a myopathy increases. Dr Albin: Ah, that's really helpful. I'm hearing a lot of it is actually the lack of other findings. In some ways it's asking, you know, have you experienced numbness and tingling? And if not, that's sort of eliminating that this might not be a neuropathy problem. And then again, that fatigability- obviously fatigability is not specific to a neuromuscular junction, but knowing that is a hallmark of myasthenia, the most common of neuromuscular disorders. Getting that off the table helps you say, okay, well, it's not a neuromuscular junction problem, perhaps. Now we have to think more about, is this a muscle problem itself? Are there any patterns that the patients describe? I have difficulty getting up from a chair, or I have difficulty brushing my hair. When I think of myopathies, I historically have thought of, sort of, more proximal weakness. Is that always true, or not so much? Dr Milone: Yeah. So, there are muscle diseases that involve predominantly proximal weakness. For example, the patient you mentioned earlier could have, for example, an autoimmune muscle disease, a necrotizing autoimmune myopathy; could have, perhaps, dermatomyositis if there are skin changes. But a patient with muscle disease can also present with a different pattern of weakness. So, myopathies can lead to this weakness, and foot drop myopathies can cause- can manifest with the weakness of the calf muscles. So, you may have a patient presenting to the clinic who has no the inability to stand on tiptoes, or you may have a patient who has just facial weakness, who has noted the difficulty sealing their lips on the glasses when they drink and experiencing some drooling in that setting, plus some hand weakness. So, the muscle involved in muscle diseases can vary depending on the underlying cause of the muscle disease. Dr Albin: That's really helpful. So, it really is really keeping an open mind and looking for some supporting features, whether it's bulbar involvement, extraocular eye muscle involvement; looking, you know, is it proximal, is it distal? And then remembering that any of those patterns can also be a muscle problem, even if sometimes we think of distal being more neuropathy and proximal myopathy. Really, there's a host of ranges for this. I really took that away from your article. This is, unfortunately, not just a neat way to box these. We really have to have that broad differential. Let me ask another question about your history. How often do you find that patients complain of, sort of, muscular cramping or muscle pain? And does that help you in terms of deciding what type of myopathy they may have? Dr Milone: Many patients with muscle disease have muscle pain. The muscle pain could signal a presence of inflammation in skeletal muscle, could be the result of overuse from a muscle that is not functioning normally. People who have myotonia experience muscle stiffness and muscle pain. Patients who have a metabolic myopathy usually have exercise-induced muscle pain. But, as we know, muscle pain is also very nonspecific, so we have to try to find out from the patient in what setting the pain specifically occurs. Dr Albin: That's really helpful. So, it's asking a little bit more details about the type of cramping that they have, the type of pain they may be experiencing, to help you refine that differential. Similarly, one of the things that I historically have always associated with myopathies is an elevation in the CK, or the creatinine kinase. How sensitive and specific is that, and how do you as the expert sort of take into account, you know, what their CK may be? Dr Milone: So, this is a very good point. And the elevation of creatine kinase can provide a clue that the patient has a muscle disease, but it is nonspecific for muscle disease because we know that elevation of creatine kinase can occur in the setting of a neurogenic process. For example, we can see elevation of the creatine kinase in patients who have ALS or in patients who have spinal muscular atrophy. And in these patients---for example, those with spinal muscular atrophy---the CK elevation can be also of significantly elevated up to a couple of thousand. Conversely, we can have muscle diseases where the CK elevation does not occur. Examples of these are some genetic muscle disease, but also some acquired muscle diseases. If we think of, for example, cases where inflammation in the muscle occurs in between muscle fibers, more in the interstitium of the muscle, that disease may not lead to significant elevation of the CK. Dr Albin: That's super helpful. So, I'm hearing you say CK may be helpful, but it's neither completely sensitive nor completely specific when we're thinking about myopathic disorders. Dr Milone: You are correct. Dr Albin: Great. So, coming back to our patients, you know, she says that she has this dysphasia. How do bulbar involvement or extraocular eye movement involvement, how do those help narrow your differential? And what sort of disorders are you thinking of for patients who may have that bulbar or extraocular muscle involvement? Dr Milone: Regarding dysphagia, that can occur in the setting of acquired myopathies relatively frequent; for example, in inclusion body myositis or in other forms of inflammatory myopathy. Your patient, I believe, was in their forties, so it's a little bit too young for inclusion body myositis. Involvement of the extraocular muscles is usually much more common in genetic muscle diseases and much less frequent in hereditary muscle disease. So, if there is involvement of the extraocular muscles, and if there is a dysphagia, and if there is a proximal weakness, you may think about oculopharyngeal muscular dystrophy, for example. But obviously, in a patient who has only six months of history, we have to pay attention of the degree of weakness the patient has developed since the symptom onset. Because if the degree of weakness is mild, yes, it could still be a genetic or could be an acquired disease. But if we have a patient who, in six months, from being normal became unable to climb stairs, then we worry much more about an acquired muscle disease. Dr Albin: That's really helpful. So, the time force of this is really important. And when you're trying to think about, do I put this in sort of a hereditary form of muscle disease, thinking more of an indolent core, something that's going to be slowly progressive versus one of those inflammatory or necrotizing pathologies, that's going to be a much more quick onset, rapidly progressive, Do I have that right? Dr Milone: In general, the statement is correct. They tend, acquired muscle disease, to have a faster course compared to a muscular dystrophy. But there are exceptions. There have been patients with immune mediated necrotizing myopathy who have been misdiagnosed as having limb-girdle muscular dystrophy just because the disease has been very slowly progressive, and vice versa. There may be some genetic muscle diseases that can present in a relatively fast way. And one of these is a lipid storage myopathy, where some patients may develop subacutely weakness, dysphagia, and even respiratory difficulties. Dr Albin: Again, I'm hearing you say that we really have to have an open mind that myopathies can present in a whole bunch of different ways with a bunch of different phenotypes. And so, keeping that in mind, once you suspect someone has a myopathy, looking at the testing from the EMG perspective and then maybe laboratory testing, how do you use that information to guide your work up? Dr Milone: The EMG has a crucial role in the diagnosis of muscle diseases. Because, as we said earlier, weakness could be the result of muscle disease or other form of neuromuscular disease. If the EMG study will show evidence of muscle disease supporting your diagnostic hypothesis, now you have to decide, is this an acquired muscle disease or is this a genetic muscle disease? If you think that, based on clinical history of, perhaps, subacute pores, it is more likely that the patient has an acquired muscle disease, then I would request a muscle biopsy. The muscle biopsy will look for structural abnormalities that could help in narrowing down the type of muscle disease that the patient has. Dr Albin: That's really helpful. When we're sending people to get muscle biopsies, are there any tips that you would give the listeners in terms of what site to biopsy or what site, maybe, not to biopsy? Dr Milone: This is a very important point. A muscle biopsy has the highest diagnostic yield if it's done in a muscle that is weak. And because muscle diseases can result in proximal or distal weakness, if your patient has distal weakness, you should really biopsy a distal muscle. However, we do not wish to biopsy a muscle that is too weak, because otherwise the biopsy sample will result just in fibrous and fatty connected tissue. So, we want to biopsy a muscle that has mild to moderate weakness. Dr Albin: Great. So, a little Goldilocks phenomenon: has to be some weak, but not too weak. You got to get just the right feature there. I love that. That's a really good pearl for our listeners to take. What about on the flip side? Let's say you don't think it's an acquired a muscular disease. How are you handling testing in that situation? Dr Milone: If you think the patient has a genetic muscle disease, you pay a lot of attention to the distribution of the weakness. Ask yourself, what is the best pattern that represent the patient's weakness? So, if I have a patient who has facial weakness, dysphagia, muscle cramping, and then on examination represent myotonia, then at that point we can go straight to a genetic test for myotonic dystrophy type one. Dr Albin: That's super helpful. Dr Milone: So, you request directly that generic test and wait for the result. If positive, you will have proof that your diagnostic hypothesis was correct. Dr Albin: You're using the genetic testing to confirm your hypothesis, not just sending a whole panel of them. You're really informing that testing based on the patient's pattern of weakness and the exam findings, and sometimes even the EMG findings as well. Is that correct? Dr Milone: You are correct, and ideally, yes. And this is true for certain muscle diseases. In addition to myotonic dystrophy type one, for example, if you have a patient who has fascial scapulohumeral muscular weakness, you can directly request a test for FSHD. So, the characterization of the clinical phenotype is crucial before selecting the genetic test for diagnosis. Dr Albin: Wonderful. Dr Milone: However, this is not always possible, because you may have a patient who has just a limb-girdle weakness, and the limb-girdle weakness can be limb-girdle muscular dystrophy. But we know that there are many, many types of limb-girdle muscular dystrophies. Therefore, the phenotype is not sufficient to request specific genetic tests for one specific form of a limb-girdle muscular dystrophy. And in those cases, more complex next-generation sequencing panels have a higher chance of providing the answer. Dr Albin: Got it, that makes sense. So, sometimes we're using a specific genetic test; sometimes, it is unfortunate that we just cannot narrow down to one disease that we might be looking for, and we may need a panel in that situation. Dr Milone: You are correct. Dr Albin: Fantastic. Well, as we wrap up, is there anything on the horizon for muscular disorders that you're really excited about? Dr Milone: Yes, there are a lot of exciting studies ongoing for gene therapy, gene editing. So, these studies are very promising for the treatment of genetic muscle disease, and I'm sure there will be therapists that will improve the patient's quality of life and the disease outcome. Dr Albin: It's really exciting. Well, thank you again. Today I've been interviewing Dr Margarita Malone on her article on a pattern recognition approach to myopathy, which appears in the October 2025 Continuum issue on muscle and neuromuscular junction disorders. Be sure to check out Continuum Audio episodes from this and other issues, and thank you to our listeners for joining us today. And thank you, Dr Milone. Dr Milone: Thank you, Casey. Very nice chatting with you about this. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

In this episode of the Programming Electronics Academy Podcast, we talk with the Red Snapper team—three high school students from Italy who designed and built a prosthetic arm using 3D printing, servos, EMG sensors, and Arduino technology. They share how the project began, the challenges they faced with design and control systems, and how their innovation won them first place at the national RoboCup robotics competition. Follow the Red Snapper team on Instagram: https://www.instagram.com/pac_tech_ Or check out their website here: https://pactech.mystrikingly.com/ Learn more about Programming Electronics Academy: https://programmingelectronics.com

Paroxysmal movement disorders refer to a group of highly heterogeneous disorders that present with attacks of involuntary movements without loss of consciousness. These disorders demonstrate considerable and ever-expanding genetic and clinical heterogeneity, so an accurate clinical diagnosis has key therapeutic implications. In this episode, Kait Nevel, MD, speaks with Abhimanyu Mahajan, MD, MHS, FAAN, author of the article “Paroxysmal Movement Disorders” in the Continuum® August 2025 Movement Disorders issue. Dr. Nevel is a Continuum® Audio interviewer and a neurologist and neuro-oncologist at Indiana University School of Medicine in Indianapolis, Indiana. Dr. Mahajan is an assistant professor of neurology and rehabilitation medicine at the James J. and Joan A. Gardner Family Center for Parkinson's Disease and Movement Disorders at the University of Cincinnati in Cincinnati, Ohio. Additional Resources Read the article: Paroxysmal Movement Disorders Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @IUneurodocmom Guest: @MahajanMD Full episode transcript available here Dr Jones: This is Doctor Lyell Jones, editor in chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Nevel: Hello, this is Dr Kait Nevel. Today I'm interviewing doctor Abhi Mahajan about his article on diagnosis and management of paroxysmal movement disorders, which appears in the August 2025 Continuum issue on movement disorders. Abhi, welcome to the podcast and please introduce yourself to the audience. Dr Mahajan: Thank you, Kait. Thank you for inviting me. My name is Abhi Mahajan. I'm an assistant professor of neurology and rehabilitation medicine at the University of Cincinnati in Cincinnati, Ohio. I'm happy to be here. Dr Nevel: Wonderful. Well, I'm really excited to talk to you about your article today on this very interesting and unique set of movement disorders. So, before we get into your article a little bit more, I think just kind of the set the stage for the discussion so that we're all on the same page. Could you start us off with some definitions? What are paroxysmal movement disorders? And generally, how do we start to kind of categorize these in our minds? Dr Mahajan: So, the term paroxysmal movement disorders refers to a group of highly heterogeneous disorders. These may present with attacks of involuntary movements, commonly a combination of dystonia and chorea, or ataxia, or both. These movements are typically without loss of consciousness and may follow, may follow, so with or without known triggers. In terms of the classification, these have been classified in a number of ways. Classically, these have been classified based on the trigger. So, if the paroxysmal movement disorder follows activity, these are called kinesigenic, paroxysmal, kinesigenic dyskinesia. If they are not followed by activity, they're called non kinesigenic dyskinesia and then if they've followed prolonged activity or exercise they're called paroxysmal exercise induced dyskinesia. There's a separate but related group of protogynous movement disorders called episodic attacks here that can have their own triggers. Initially this was the classification that was said. Subsequent classifications have placed their focus on the ideology of these attacks that could be familiar or acquired and of course understanding of familiar or genetic causes of paroxysmal movement disorders keeps on expanding and so on and so forth. And more recently, response to pharmacotherapy and specific clinical features have also been introduced into the classification. Dr Nevel: Great, thank you for that. Can you share with us what you think is the most important takeaway from your article for the practicing neurologist? Dr Mahajan: Absolutely. I think it's important to recognize that everything that looks and sounds bizarre should not be dismissed as malingering. Such hyperkinetic and again in quotations, “bizarre movements”. They may appear functional to the untrained eye or the lazy eye. These movements can be diagnosed. Paroxysmal movement disorders can be diagnosed with a good clinical history and exam and may be treated with a lot of success with medications that are readily available and cheap. So, you can actually make a huge amount of difference to your patients' lives by practicing old-school neurology. Dr Nevel: That's great, thank you so much for that. I can imagine that scenario does come up where somebody is thought to have a functional neurological disorder but really has a proximal movement disorder. You mentioned that in your article, how it's important to distinguish between these two, how there can be similarities at times. Do you mind giving us a little bit more in terms of how do we differentiate between functional neurologic disorder and paroxysmal movement disorder? Dr Mahajan: So clinical differentiation of functional neurological disorder from paroxysmal movement disorders, of course it's really important as a management is completely different, but it can be quite challenging. There's certainly an overlap. So, there can be an overlap with presentation, with phenomenology. Paroxysmal nature is common to both of them. In addition, FND and PMD's may commonly share triggers, whether they are movement, physical exercise. Other triggers include emotional stimuli, even touch or auditory stimuli. What makes it even more challenging is that FND's may coexist with other neurological disorders, including paroxysmal movement disorders. However, there are certain specific phenom phenotypic differences that have been reported. So specific presentations, for example the paroxysms may look different. Each paroxysm may look different in functional neurological disorders, specific phenotypes like paroxysmal akinesia. So, these are long duration episodes with eyes closed. Certain kinds of paroxysmal hyperkinesia with ataxia and dystonia have been reported. Of course. More commonly we see PNES of paroxysmal nonepileptic spells or seizures that may be considered paroxysmal movement disorders but represent completely different etiology which is FND. Within the world of movement disorders, functional jerks may resemble propiospinal myoclonus which is a completely different entity. Overall, there are certain things that help separate functional movement disorders from paroxysmal movement disorders, such as an acute onset variable and inconsistent phenomenology. They can be suggestibility, distractibility, entrainment, the use of an EMG may show a B-potential (Bereitschaftspotential) preceding the movement in patients with FND. So, all of these cues are really helpful. Dr Nevel: Great, thanks. When you're seeing a patient who's reporting to these paroxysmal uncontrollable movements, what kind of features of their story really tips you off that this might be a proximal movement disorder? Dr Mahajan: Often these patients have been diagnosed with functional neurological disorders and they come to us. But for me, whenever the patient and or the family talk about episodic movements, I think about these. Honestly, we must be aware that there is a possibility that the movements that the patients are reporting that you may not see in clinic. Maybe there are obvious movement disorders. Specifically, there's certain clues that you should always ask for in the history, for example, ask for the age of onset, a description of movements. Patients typically have videos or families have videos. You may not be able to see them in clinic. The regularity of frequency of these movements, how long the attacks are, is there any family history of or not? On the basis of triggers, whether, as I mentioned before, do these follow exercise? Prolonged exercise? Or neither of the above? What is the presentation in between attacks, which I think is a very important clinical clue. Your examination may be limited to videos, but it's important not just to examine the video which represents the patient during an attack, but in between attacks. That is important. And of course, I suspect we'll get to the treatment, but the treatment can follow just this part, the history and physical exam. It may be refined with further testing, including genetic testing. Dr Nevel: Great. On the note of genetic testing, when you do suspect a diagnosis of paroxysmal movement disorder, what are some key points for the provider to be aware of about genetic testing? How do we go about that? I know that there are lots of different options for genetic testing and it gets complicated. What do you suggest? Dr Mahajan: Traditionally, things were a little bit easier, right, because we had a couple of genes that have been associated with the robust movement disorders. So, genetic testing included single gene testing, testing for PRRT2 followed by SLC2A. And if these were negative, you said, well, this is not a genetic ideology for paroxysmal movement disorders. Of course, with time that has changed. There's an increase in known genes and variants. There is increased genetic entropy. So, the same genetic mutation may present with many phenotypes and different genetic mutations may present with the similar phenotype. Single gene testing is not a high yield approach. Overall genetic investigations for paroxysmal movement disorders use next generation sequencing or whole exome sequence panels which allow for sequencing of multiple genes simultaneously. The reported diagnostic yield with let's say next generation sequencing is around 35 to 50 percent. Specific labs at centers have developed their own panels which may improve the yield of course. In children, microarray may be considered, especially the presentation includes epilepsy or intellectual disability because copy number variations may not be detected by a whole exome sequencing or next generation sequencing. Overall, I will tell you that I'm certainly not an expert in genetics, so whenever you're considering genetic testing, if possible, please utilize the expertise of a genetic counsellor. Families want to know, especially as an understanding of the molecular underpinnings and knowledge about associated mutations or variations keeps on expanding. We need to incorporate their expertise. A variant of unknown significance, which is quite a common result with genetic testing, may not be a variant of unknown significance next year may be reclassified as pathogenic. So, this is extremely important. Dr Nevel: Yeah. That's such a good point. Thank you. And you just mentioned that there are some genetic mutations that can lead to multiple different phenotypes. Seemingly similar phenotypes can be associated with various genetic mutations. What's our understanding of that? Do we have an understanding of that? Why there is this seeming disconnect at times between the specific genetic mutation and the phenotype? Dr Mahajan: That is a tough question to answer for all paroxysmal movement disorders because the answer may be specific to a specific mutation. I think a great example is the CACNA1A mutation. It is a common cause of episodic ataxia type 2. Depending on when the patient presents, you can have a whole gamut of clinical presentations. So, if the patient is 1 year old, the patient can present with epileptic encephalopathy. Two to 5 years, it can be benign paroxysmal torticollis of infancy. Five to 10 years, can present with learning difficulties with absence epilepsy and then of course later, greater than 10 years, with episodic ataxia (type) 2 hemiplegic migraine and then a presentation with progressive ataxia and hemiplegic migraines has also been reported. So not just episodic progressive form of ataxia has also been reported. I think overall these disorders are very rare. They are even more infrequently diagnosed than their prevalence. As such, the point that different genetic mutations present with different phenotypes, or the same genetic mutation I may present with different phenotypes could also represent this part. Understanding of the clinical presentation is really incomplete and forever growing. There's a new case report or case series every other month, which makes this a little bit challenging, but that's all the more reason for learning about them and for constant vigilance for patients who show up to our clinic. Dr Nevel: Yeah, absolutely. What is our current understanding of the associated pathophysiology of these conditions and the pathophysiology relating to the genetics? And then how does that relate to the treatment of these conditions? Dr Mahajan: So, a number of different disease mechanisms have been proposed. Traditionally, these were all thought to be ion channelopathies, but a number of different processes have been proposed now. So, depending on the genetic mutation that you talk about. So certain mutations can involve ion channels such as CACMA1A, ATP1A3. It can involve solute carriers, synaptic vesicle fusion, energy metabolism such as ECHS1, synthesis of neurotransmitters such as GCH1. So, there are multiple processes that may be involved. I think overall for the practicing clinician such as me, I think there is a greater need for us to understand the underlying genetics and associated phenotypes and the molecular mechanisms specifically because these can actually influence treatment decisions, right? So, you mentioned that specific genetic testing understanding of the underlying molecular mechanism can influence specific treatments. As an example, a patient presenting with proximal nocturnal dyskinesia with mutation in the ADCY5 gene may respond beautifully to caffeine. Other examples if you have SLC2A1, so gluc-1 (glucose transporter type 1) mutation, a ketogenic diet may work really well. If you have PDHA1 mutation that may respond to thiamine and so on and so forth. There are certain patients where paroxysmal movement disorders are highly disabling and you may consider deep brain stimulation. That's another reason why it may be important to understand genetic mutations because there is literature on response to DBS with certain mutations versus others. Helps like counselling for patients and families, and of course introduces time, effort, and money spent in additional testing. Dr Nevel: Other than genetic testing, what other diagnostic work up do you consider when you're evaluating patients with a suspected paroxysmal movement disorder? Are there specific things in the history or on exam that would prompt you to do certain testing to look for perhaps other things in your differential when you're first evaluating a patient? Dr Mahajan: In this article, I provide a flow chart that helps me assess these patients as well. I think overall the history taking and neurological exam outside of these paroxysms is really important. So, the clinical exam in between these episodic events, for example, for history, specific examples include, well, when do these paroxysms happen? Do they happen or are they precipitated with meals that might indicate that there's something to do with glucose metabolism? Do they follow exercise? So, a specific example is in Moyamoya disease, they can be limb shaking that follows exercise. So, which gives you a clue to what the etiology could be. Of course, family history is important, but again, talking about the exam in between episodes, you know, this is actually a great point because out– we've talked about genetics, we've talked about idiopathic paroxysmal movement disorders, –but a number of these disorders are because of acquired causes. Well, of course it's important because acquired causes such as autoimmune causes, so multiple sclerosis, ADEM, lupus, LGI1, all of these NMDAR, I mentioned Moyamoya disease and metabolic causes. Of course, you can consider FND as under-acquired as well. But all of these causes have very different treatments and they have very different prognosis. So, I think it's extremely important for us to look into the history with a fine comb and then examine these patients in between these episodes and keep our mind open about acquired causes as well. Dr Nevel: When you evaluate these patients, are you routinely ordering vascular imaging and autoimmune kind of serologies and things like that to evaluate for these other acquired causes or it does it really just depend on the clinical presentation of the patient? Dr Mahajan: It mostly depends on the clinical presentation. I mean, if the exam is let's say completely normal, there are no other risk factors in a thirty year old, then you know, with a normal exam, normal history, no other risk factors. I may not order an MRI of the brain. But if the patient is 55 or 60 (years) with vascular risk factors, then you have to be mindful that this could be a TIA. If the patient has let's say in the 30s and in between these episodes too has basically has a sequel of these paroxysms, then you may want to consider autoimmune. I think the understanding of paraneoplastic, even autoimmune disorders, is expanding as well. So, you know the pattern matters. So, if all of this is subacute started a few months ago, then I have a low threshold for ordering testing for autoimmune and paraneoplastic ideology is simply because it makes such a huge difference in terms of how you approach the treatment and the long-term prognosis. Dr Nevel: Yeah, absolutely. What do you find most challenging about the management of patients with paroxysmal movement disorders? And then also what is most rewarding? Dr Mahajan: I think the answer to both those questions is, is the same. The first thing is there's so much advancement in what we know and how we understand these disorders so regularly that it's really hard to keep on track. Even for this article, it took me a few months to write this article, and between the time and I started and when I ended, there were new papers to include new case reports, case series, right? So, these are rare disorders. So most of our understanding for these disorders comes from case reports and case series, and it's in a constant state of advancement. I think that is the most challenging part, but it's also the most interesting part as well. I think the challenging and interesting part is the heterogeneity of presentation as well. These can involve just one part of your body, your entire body can present with paroxysmal events, with multiple different phenomenologies and they might change over time. So overall, it's highly rewarding to diagnose such patients in clinic. As I said before, you can make a sizeable difference with the medication which is usually inexpensive, which is obviously a great point to mention these days in our health system. But with anti-seizure drugs, you can put the right diagnosis, you can make a huge difference. I just wanted to make a point that this is not minimizing in any way the validity or the importance of diagnosing patients with functional neurological disorders correctly. Both of them are as organic. The importance is the treatment is completely different. So, if you're diagnosing somebody with FND and they do have FND and they get cognitive behavioral therapy and they get better, that's fantastic. But if somebody has paroxysmal movement disorders and they undergo cognitive behavioral therapy and they're not doing well, that doesn't help anybody. Dr Nevel: One hundred percent. As providers, obviously we all want to help our patients and having the correct diagnosis, you know, is the first step. What is most interesting to you about paroxysmal movement disorders? Dr Mahajan: So outside of the above, there are some unanswered questions that I find very interesting. Specifically, the overlap with epilepsy is very interesting, including shared genes, the episodic nature, presence of triggers, therapeutic response to anti-seizure drugs. All of this I think deserves further study. In the clinic, you may find that epilepsy and prognosis for movement disorders may occur in the same individual or in a family. Episodic ataxia has been associated with seizures. Traditionally this dichotomy of an ictal focus. If it's cortical then it's epilepsy, if it's subcortical then it's prognosis for movement disorders. This is thought to be overly simplistic. There can be co-occurrence of seizures and paroxysmal movement disorders in the same patient and that has led to this continuum between these two that has been proposed. This is something that needs to be looked into in more detail. Our colleagues in Epilepsy may scoff this, but there's concept of basal ganglia epilepsy manifesting as paroxysmal movement disorders was proposed in the past. And there was this case report that was published out of Italy where there was ictal discharge from the supplementary sensory motor cortex with a concomitant discharge from the ipsilateral coordinate nucleus in a patient with paroxysmal kinesigenic cardioarthidosis. So again, you know, basal ganglia epilepsy, no matter what you call it, the idea is that there is a clear overlap between these two conditions. And I think that is fascinating. Dr Nevel: Really interesting stuff. Well, thank you so much for chatting with me today. Dr Mahajan: Thank you, Kait. And thank you to the Continuum for inviting me to write this article and for this chance to speak about it. I'm excited about how it turned out, and I hope readers enjoy it as well. Dr Nevel: Today again, I've been interviewing doctor Abhi Mahajan about his article on diagnosis and management of paroxysmal movement disorders, which appears in the August 2025 Continuum issue on movement disorders. I encourage all of our listeners to be sure to check out the Continuum Audio episodes from this and other issues. As always, please read the Continuum articles where you can find a lot more information than what we were able to cover in our discussion today. And thank you for our listeners for joining today. And thank you, Abhi, so much for sharing your knowledge with us today. Dr Monteith: This is Dr Teshamae Monteith, associate editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

Carrie Ford, CNCT interviews Clark Moser, MD, ABEM, ABPN on "Electromyography Referrals: Principles and Practices for Optimizing EMG Orders ". This podcast is for neurologists, PMRs, orthopedists, PCPs, NPs, PAs, and residents who refer patients for EMG and NCS. It also helps NCS technologists understand the clinical intent behind each referral—guided by the EMGer's diagnostic focus. This episode offers practical strategies for improving the quality of the EMG referrals by treating each order as a focused clinical study. We review what the EMG evaluates—nerve and muscle function—and how to understand results in terms of localization and severity. Listeners will learn to move beyond vague referral terms like “weakness,” “pain,” or “paresthesia” and instead indicate in the EMG order what specific neuromuscular or electrodiagnostic question they want answered.

Mándanos un mensajePuntos Clave de DiscusiónIntroducción al Debate: Discusión sobre las posturas de barra alta y barra baja en sentadillas y por qué son tan debatidas en los gimnasios.Análisis del Estudio Científico: Un estudio de van den Tillaar (2020) que investiga las diferencias biomecánicas y de activación muscular entre ambas técnicas usando EMG.Resultados del Estudio: No se encontraron diferencias significativas en la cinemática del movimiento, pero hubo variaciones en la activación muscular, especialmente en cuádriceps con barra alta.Aplicaciones en el Gimnasio: Cómo los diferentes posicionamientos de la barra pueden afectar el crecimiento muscular y la fuerza, y cuándo podría ser mejor utilizar cada técnica.Consideraciones Prácticas: Factores como la comodidad, movilidad, lesiones previas, y objetivos personales pueden influir en la elección de la técnica.Support the showInstagram: andieillanesPágina web: andieillanes.com.mx

Episode 198: In this episode of the Inform Performance Podcast, Andy McDonald is joined by Jeff Riepe, a distinguished expert at the intersection of sports science, rehabilitation, and human performance. Riepe earned his Doctorate in Physical Therapy from the University of Southern California, followed by a fellowship in Clinical & Sports Biomechanics at the Movement Performance Institute under the mentorship of Chris Powers. His career spans elite sports medicine roles with the NFL's Minnesota Vikings and Los Angeles Rams, numerous collegiate athletic programs, and the EXOS NFL Combine Training Program. Specializing in advanced movement analysis, Riepe integrates cutting-edge technologies such as 2D and 3D high-speed motion capture, force plate analysis, and wireless electromyography (EMG) to optimize rehabilitation and performance outcomes. Topics Discussed: MoveLab's Advanced Biomechanics Lab Data-Driven Rehabilitation Strategies Integration of Clinical Research and Normative Data Challenges in Late-Stage Rehabilitation The Role of VO₂ Max - Where you can find Jeff Riepe: LinkedIn Move Lab Instagram X -  Sponsors VALD Performance, makers of the Nordbord, Forceframe, ForeDecks and HumanTrak. VALD Performance systems are built with the high-performance practitioner in mind, translating traditionally lab-based technologies into engaging, quick, easy-to-use tools for daily testing, monitoring and training Hytro: The world's leading Blood Flow Restriction (BFR) wearable, designed to accelerate recovery and maximise athletic potential using Hytro BFR for Professional Sport.  -  Where to Find Us Keep up to date with everything that is going on with the podcast by following Inform Performance on: Instagram Twitter Our Website - Our Team Andy McDonald Ben Ashworth Alistair McKenzie Dylan Carmody Steve Barrett  Pete McKnight

CNET's Scott Stein goes hands-on with Meta's newest lineup of smart glasses, including the Meta Ray-Ban Display Glasses and the next-gen Ray-Ban Meta. He also gets to try out the new neural wristband, which uses electromyography (EMG) to detect subtle muscle movements, offering a glimpse into the future of how we'll interact with augmented reality.

CNET's Scott Stein goes hands-on with Meta's newest lineup of smart glasses, including the Meta Ray-Ban Display Glasses and the next-gen Ray-Ban Meta. He also gets to try out the new neural wristband, which uses electromyography (EMG) to detect subtle muscle movements, offering a glimpse into the future of how we'll interact with augmented reality.

¡Nueva temporada! En este episodio de #MemoriasDelMetaverso nos ponemos en modo “antesala” de la Meta Connect para destripar filtraciones y expectativas… y alguna bomba gamer.Resumen del episodio (rápido y al grano)Meta presentará sus nuevas smart glasses con display monocular: no es “realidad aumentada” como tal, sino realidad asistida (información en una ventanita sobre el mundo, sin anclaje 3D al entorno). Clave: una pulsera de interfaz neuronal/EMG para escribir y controlar sin cámaras de manos. También repasamos por qué esto no compite con AR auténtica (ejemplo: navegación AR en coches tipo BMW con flechas integradas en la calzada vs. un simple HUD). La otra gran protagonista podría ser Valve: todo apunta a un visor “Steam Frame” centrado en llevar tu biblioteca de Steam a una pantalla gigante virtual, con mandos que recuperan la cruceta para juegos planos y la opción de soñar con 3D estereoscópico. Ideal como “caballo de Troya” para que más jugadores prueben la VR sin renunciar a sus AAA. Cerramos con Apple Vision Pro: nuevo curso con mejoras (avatares más naturales, compatibilidad de mandos, demos de producto a escala real) y debate sobre potencia local para experiencias pro (modelos 3D gordos… y cómo se le pueden “buscar las cosquillas” al chip actual). Además, guiños a traducción en llamadas y usos reales de colaboración remota. SEO / palabras clave: Meta Connect, smart glasses con display, realidad asistida vs realidad aumentada, pulsera EMG, Valve Steam Frame, Steam Deck, pantalla gigante virtual, controladores con cruceta, Apple Vision Pro, visionOS, BMW AR HUD, Xreal, juegos AAA en VR.Por qué escucharlo:Descubres la diferencia práctica entre asistencia visual y AR real, qué cambia con una pulsera neuronal en tu muñeca, y cómo Valve puede redefinir el juego en XR sin pelear por precio. Además, te llevas ideas claras para trabajo remoto y demos de producto con Vision Pro.

How on earth can a neck injection eliminate teeth sensitivity? Can a patient's tooth sensitivity really be linked to their occlusion? Is occlusal adjustment ever indicated for sensitivity? And what's the actual mechanism behind those cases where everything looks fine — no cracks, no significant wear, no exposed dentine — yet the patient still complains their teeth are sensitive? In this episode, Dr. Nick Yiannios shares the concept of Sympathetic Dentin Hypersensitivity (SDH), a groundbreaking way of understanding sensitivity that goes beyond the usual suspects like caries, erosion, or leakage. We dive into how the sympathetic nervous system in the pulp can drive unexplained pain, why traditional approaches often fail, and how objective tools like T-Scan and EMG can reveal what articulating paper misses. This could completely change the way you diagnose and manage those “mystery” sensitivity cases that just don't add up. https://youtu.be/a2Mg72Y_zkw Watch PDP240 on Youtube Protrusive Dental Pearl: When fitting a resin-bonded bridge (RBB), if you're unsure about the fit and cement gap, use light-bodied PVS on the intaglio surface of the wing. After setting and peeling it away, the thickness of the PVS shows you the expected cement layer. Ideally, it should be thin and even; a thicker area highlights where your gap is excessive. Key Takeaways: The T-scan technology revolutionizes occlusal analysis. Sensitive teeth can be linked to occlusion and bite adjustments. Frictional dental hypersensitivity (FDH) is a key concept in understanding sensitivity. Sympathetic responses may contribute to dental hypersensitivity. Innovative treatments include laser therapy and ozone application. Addressing root causes is essential for long-term solutions. Dentists should explore literature for new insights and techniques. Critical thinking is vital in dental practice. Advanced technology can enhance patient care and outcomes. Objective data is essential for effective occlusal adjustments. Understanding joint function is crucial for dental health. Differentiating between types of dental hypersensitivity is important. The sympathetic nervous system plays a significant role in dental pain. Educating patients about their conditions fosters better outcomes. The beaker of pain concept helps in understanding patient symptoms. Continuous learning is vital for dental professionals. Objective metrics are necessary for accurate diagnosis and treatment. Highlights of this episode: 00:00 Teaser 00:39 Intro 03:51 Protrusive Dental Pearl 05:42: Dr. Nick Yiannios' Journey and Innovations 07:46 T-Scan and Digital Occlusal Analysis 08:29 FIRST INTERJECTION 13:46 T-Scan and Digital Occlusal Analysis 14:07 Discovery of Occlusion–Sensitivity Link 20:44 Second interjection 24:25 Student Case – Sensitivity from a Bridge 26:04  Dentine Hypersensitivity 28:39 Cervical Dentine Hypersensitivity 30:44 The Role of Lasers and Ozone in Dental Treatment 35:24 Alternatives for Dentists Without Lasers 43:12 Alternatives for Dentists Without Lasers 44:00 Frictional Dental Hypersensitivity Explained 47:15 The Importance of T-Scan in Dentistry 50:57 Neck Blocks and Sympathetic Responses. 58:24 Third interjection 01:00:01 Neck Block Mechanism 01:12:34 The Beaker of Pain Concept 01:14:38 Fourth interjection 01:16:23 The Beaker of Pain Concept 01:16:59 Community and Collaboration 1:20:57 Outro Curious to dive deeper?You can explore more of Dr. Nick's work and insights through these resources: Upcoming course: CNO6 – Sympathetics in Dentistry: The Missing Link in General & Specialty Practice AES (American Equilibration Society) – check out their upcoming conference for world-class learning in occlusion and TMD. CNO – Center for Neural Occlusion Facebook community: Neural Occlusion YouTube channel: Dr.

Hit-making guitarist Mike Scott—who's worked with everyone from Justin Timberlake to Janet Jackson—showed up in Minneapolis from his hometown of D.C. and started snatching everyone's gigs. Soon, he ended up at Paisley Park, jamming with the man himself and joining his band, New Power Generation.This masterful storyteller joins 100 Guitarists to talk all things Prince, cracking us up along the way with firsthand recollections of playing onstage and in the studio, the only time he played the “Purple Rain” solo live, and some insight on Prince and his gear. Sponsored by EMG: emgpickups.comFollow Nick: https://www.instagram.com/nickmillevoiFollow Jason: https://www.instagram.com/jasonshadrickGet at us: 100guitarists@premierguitar.comCall/Text: 319-423-9734Podcast powered by Sweetwater. Get your podcast set up here! - https://sweetwater.sjv.io/75rE0dSubscribe to the podcast:Spotify: https://open.spotify.com/show/0aXdYIDOmS8KtZaZGNazVb?si=c63d98737a6146afApple: https://podcasts.apple.com/us/podcast/100-guitarists/id1746527331

AJ Bruno is the founder and CEO of QuotaPath, a leading sales compensation management platform that has secured significant investments, including a $41 million Series B. Serving roughly 1,000 customers, QuotaPath helps organizations align company goals with incentive plans to drive winning sales behaviors.Before QuotaPath, AJ co-founded TrendKite, which was acquired by Cision for $225 million in 2019. He also co-hosts Topline, a podcast on revenue growth and sales leadership.In this episode, AJ shares the stories behind launching TrendKite and QuotaPath, his passion for flying, and his perspective on integrating AI into workflows as a founder. He reflects on his days as a Penn State student, studying abroad in Italy, his first job at Meltwater, and how a terrible boss inspired him to start his first company.Later in the episode, recent Penn State grad Paul Allen joins the conversation. A participant in the Invent Penn State Summer Founders Program, Paul asks AJ for advice on raising venture capital, making first hires, and navigating the challenges of early-stage entrepreneurship.Episode Chapters02:13 - 07:06 Growing up in Pittsburgh, acting and flying07:06 - 12:17 Choosing Penn State, squash club, studying abroad12:17 - 15:38 First entrepreneurial venture at Penn State15:38 - 22:48 First job in sales, advice to students and parents22:48 - 29:15 Terrible boss as a catalyst for TrendKite29:15 - 33:02 The problem that inspired the launch of QuotaPath33:02 - 40:33 Navigating AI and future of QuotaPath40:33 - 43:51 The crazy story that led to AJ launching a podcast43:51 - 45:24 Rapid Fire Insights45:24 - 55:20 Recent student Paul Allen interviews AJAbout AJ BrunoAJ Bruno is founder and CEO of QuotaPath. He graduated from the Penn State Schreyer Honors College with a bachelor's degree in economics from the Smeal College of Business in 2007.About Paul AllenPaul is a spring 2025 graduate of the Penn State College of Engineering. He completed the 2025 Invent Penn State Summer Founders Program with his startup, Reflex Technologies. Reflex is building a wearable electromyography (EMG) sensor that provides real-time muscle activation and fatigue insights to athletes and trainers. The Dare to Disrupt podcast is made possible by the generous support of the Penn State Smeal College of Business.

In this Ask Dr. Tony episode, Dr. Tony Ebel breaks down the comprehensive approach neurologically focused pediatric chiropractors use to help children with neurodevelopmental disorders. He explains the three-step process: detailed case history taking, advanced nervous system measurement technology (INSiGHT scans including thermal scanning, EMG, and HRV), and gentle, individualized chiropractic adjustments. Dr. Tony emphasizes how their approach differs from traditional medicine by focusing on the nervous system rather than blaming genetics or gut issues. He shares touching stories about children who initially resist touch but eventually crave the healing adjustments, and explains how their sensory-friendly offices are designed specifically to help kids "get out of the storm" of dysregulation. The episode highlights the patience, persistence, and personalized care required for true neurological healing.----Links & ResourcesThe PX Docs Clinical Process Breakdown----Key Topics & Timestamps([00:01:00]) - The Three-Step Neurologically Focused Approach - Detailed Case History, Advanced Technology, and Physical Examination([00:03:00]) - INSiGHT Scans and HRV Testing - Measuring Nervous System Dysfunction and Vagal Tone([00:05:00]) - Light Force Tonal Adjustments - The Gentle "Avocado Ripeness" Technique([00:07:00]) - The Cal Story - How Touch-Sensitive Kids Learn to Crave Healing Adjustments([00:08:00]) - Creating Sensory-Friendly Care - Environment Design and Long-Term Treatment Plans-- Follow us on Socials: Instagram: @pxdocs Facebook: Dr. Tony Ebel & The PX Docs Network Youtube: The PX Docs For more information, visit PXDocs.com to read informative articles about the power of Neurologically-Focused Chiropractic Care. Find a PX Doc Office near me: PX DOCS DirectoryTo watch Dr. Tony's 30 min Perfect Storm Webinar: Click HereSubscribe, share, and stay tuned for more incredible episodes unpacking the power of Nervous System focused care for children!

Dr. Ronald (James) Cotton who is an electrical engineer, neuroscientist, and physiatrist working as a physician scientist at Shirley Ryan Ability Lab, and assistant professor in the Northwestern University Department of Physical Medicine and Rehabilitation. We have this one paper where we can use diffusion models and generate a bunch of probabilistic samples of movements and they constrain them by what we see in the cameras and have shown that we can actually estimate the confidence and the uncertainty in a reliable way. He indicated that we can use something called the myoskeleton that allows us to track all joints in the body down to the individual fingers. Our Portable Biomechanics Laboratory is kind of a combination of a smartphone app that records the rotation and movement of the phone itself. He discussed how he would like to establish validity as a predictive biomarker. He asked what do we actually want and what do we mean by precision rehabilitation? In his mind, the best formalism of it is something called the optimal dynamic treatment regime, which essentially is some kind of function, probably a learned function that looks at all the health information, all the biomarkersof an individual at any point in rehabilitation, kind of condenses that into a phenotype, and then predicts what is the next intervention that should be given. He spoke briefly about a case studythat applies to another line of research in his lab, which is EMG-based biofeedback and also about Next Generation Brain Machine Interface Chips.

For years, anesthesia providers have relied on subjective assessments to evaluate neuromuscular recovery. These methods, while familiar, often fall short in ensuring patient safety. As evidence mounts regarding the risks associated with residual paralysis, the need for more precise and objective monitoring has never been clearer. That's why we're taking a closer look at Quantatative train-of-four (TOF) monitoring with Richie Flowers, CHSE, CRNA, DNP, FAANA and Lisandro Hernandez, DNP, CRNA to learn more about why it's gaining traction in operating rooms nationwide. Here's some of what you'll hear in this episode:

"How does physical activity differ between children with different body weights?" In this third part of the episode, Dr. Olli Tikkanen and Dr. Ying Gao discuss her findings on physical activity patterns in children, focusing on differences between overweight and normal-weight groups. Dr. Gao explains how accelerometer and EMG data revealed that overweight children show higher muscle activity even when standing, which may contribute to their tendency for more sedentary behavior, as physical tasks can be more exhausting for them. Her research highlights the potential role of physical demands in shaping activity levels among children. Dr. Gao also shares insights from her 7-day measurement study of over 400 children, examining how physical activity, sedentary behavior, and sleep patterns impact body weight and overall health. The episode touches on future research directions and how these findings could inform interventions to promote healthy behaviors in school-aged children. ____________________________________ This podcast episode is sponsored by Fibion Inc. | Better Sleep, Sedentary Behaviour and Physical Activity Research with Less Hassle Collect, store and manage SB and PA data easily and remotely - Discover ground-breaking Fibion SENS --- Learn more about Fibion Flash - a versatile customizable tool with HRV and accelerometry capability.  --- SB and PA measurements, analysis, and feedback made easy.  Learn more about Fibion Research. --- Learn more about Fibion Sleep and Fibion Circadian Rhythm Solutions. --- Fibion Kids - Activity tracking designed for children. --- Explore our Wearables,  Experience sampling method (ESM), Sleep,  Heart rate variability (HRV), Sedentary Behavior and Physical Activity article collections for insights on related articles. --- Refer to our article "Physical Activity and Sedentary Behavior Measurements" for an exploration of active and sedentary lifestyle assessment methods. --- Learn about actigraphy in our guide: Exploring Actigraphy in Scientific Research: A Comprehensive Guide. --- Gain foundational ESM insights with "Introduction to Experience Sampling Method (ESM)" for a comprehensive overview. --- Explore accelerometer use in health research with our article "Measuring Physical Activity and Sedentary Behavior with Accelerometers ". --- For an introduction to the fundamental aspects of HRV, consider revisiting our Ultimate Guide to Heart Rate Variability. --- Follow the podcast on Twitter https://twitter.com/PA_Researcher  Follow host Dr Olli Tikkanen on Twitter https://twitter.com/ollitikkanen  Follow Fibion on Twitter https://twitter.com/fibion  Check our YouTube channel: https://www.youtube.com/@PA_Researcher       

"Can we accurately measure children's physical activity with just one device?" In this insightful episode, Dr. Olli Tikkanen speaks with Dr. Ying Gao about her research on sedentary behavior and physical activity in children, combining accelerometer and EMG data to improve accuracy. Dr. Gao explains the challenges of using only accelerometers, as they capture movement but not muscle contractions, which are essential for understanding true physical activity. She describes her study on setting a reliable threshold to differentiate between sitting, light movement, and active play in children, aiming to create a standardized approach for assessing activity levels. Dr. Gao also discusses her findings on how combining EMG with accelerometer data provides a clearer picture of muscle activity, especially for distinguishing between sedentary and active behaviors in school-aged children. This episode sheds light on new methods in physical activity measurement that could improve research on children's health and activity habits. __________________ This podcast episode is sponsored by Fibion Inc. | Better Sleep, Sedentary Behavior and Physical Activity Research with Less Hassle --- Collect, store and manage SB and PA data easily and remotely - Discover ground-breaking Fibion SENS --- Learn more about Fibion Flash - a versatile customizable tool with HRV and accelerometry capability.  --- SB and PA measurements, analysis, and feedback made easy.  Learn more about Fibion Research. --- Learn more about Fibion Sleep and Fibion Circadian Rhythm Solutions. --- Fibion Kids - Activity tracking designed for children. --- Explore our Wearables,  Experience sampling method (ESM), Sleep,  Heart rate variability (HRV), Sedentary Behavior and Physical Activity article collections for insights on related articles. --- Refer to our article "Physical Activity and Sedentary Behavior Measurements" for an exploration of active and sedentary lifestyle assessment methods. --- Learn about actigraphy in our guide: Exploring Actigraphy in Scientific Research: A Comprehensive Guide. --- Gain foundational ESM insights with "Introduction to Experience Sampling Method (ESM)" for a comprehensive overview. --- Explore accelerometer use in health research with our article "Measuring Physical Activity and Sedentary Behavior with Accelerometers ". --- For an introduction to the fundamental aspects of HRV, consider revisiting our Ultimate Guide to Heart Rate Variability. --- Follow the podcast on Twitter https://twitter.com/PA_Researcher  Follow host Dr Olli Tikkanen on Twitter https://twitter.com/ollitikkanen  Follow Fibion on Twitter https://twitter.com/fibion  Check our YouTube channel: https://www.youtube.com/@PA_Researcher     

Join Pascal as he explores the groundbreaking world of generic neuromotor interfaces with Jesse, Lauren, and Sean. Discover how these technologies enable control of devices with just a flick of the wrist or even a simple intention to move. We'll discuss the role of AI in eliminating the need for personalised training, the differences between non-invasive interfaces and their predecessors, and the exciting implications for accessibility. Don't miss this deep dive into the future of human-computer interaction. Got feedback? Send it to us on Threads (https://threads.net/@metatechpod), Instagram (https://instagram.com/metatechpod) and don't forget to follow our host Pascal (https://mastodon.social/@passy, https://threads.net/@passy_). Fancy working with us? Check out https://www.metacareers.com/. Timestamps Intro 0:06 Jesse introduction 1:29 Lauren introduction 2:42 Sean introduction 3:29 Team's mission statement 3:49 What's a neuromotor interface? 4:24 Paper overview 5:29 Non-invasive interfaces 7:50 How to make it generic 9:42 Design tradeoffs 11:29 Real-world model performance 14:21 Feedback cycle 16:22 LLMs and EMG 17:22 Handwriting vision 18:39 Working with product 20:55 EMG for accessibility 22:25 How Meta helps 25:53 Open-source repos 28:02 What's next? 28:45 Outro 30:51 Links A generic non-invasive neuromotor interface for human-computer interaction - Nature - https://www.nature.com/articles/s41586-025-09255-w  How the low-vision community embraced AI smart glasses - The Verge - https://www.theverge.com/the-vergecast/701018/ray-ban-meta-smart-glasses-be-my-eyes-ceo-accessibility-tech MKBHD on Orion - https://www.youtube.com/watch?v=G0eKzU_fV00 

Join Jay Gunkelman, QEEGD (the man who has analyzed over 500,000 brain scans), and host Pete Jansons for another engaging NeuroNoodle Neurofeedback Podcast episode discussing neuroscience, psychology, mental health, and brain training. Special guests Joshua Moore and Anthony Ramos join in for a deep-dive Q&A.✅ Topic 1 Explained: Jay breaks down the critical links between insomnia and ADHD, highlighting how delayed circadian rhythms and underarousal phenotypes impact life satisfaction and school performance.✅ Topic 2 Deep Dive: Restless Leg Syndrome as an ADHD mimic—Jay explains its dopamine and beta spindle connections, EMG detection methods, and neurofeedback treatment options.✅ Topic 3 Insights: How psychiatric meds, especially antipsychotics and benzos, can impact EEGs, neuroplasticity, and long-term cognitive outcomes—plus safer treatment alternatives.✅ Additional Topics:

Leitura Bíblica Do Dia: GÁLATAS 5:1-7,13-15 Plano De Leitura Anual: NEEMIAS 12–13; ATOS 4:23-37  Já fez seu devocional hoje? Aproveite e marque um amigo para fazer junto com você! Confira:  No Texas, onde eu cresci, havia desfiles e piqueniques nas comunidades negras, todo dia 19 de junho, celebrando o Juneteenth (uma mistura das palavras junho e 19 em inglês). Só quando eu já era adolescente que aprendi o significado chocante dessa data. Nesse dia, em 1865, a população escravizada do Texas descobriu que o presidente Lincoln havia assinado a Declaração de Emancipação, libertando-os, 2 anos e 5 meses antes. Eles tinham permanecido na escravidão porque não sabiam que tinham sido libertos. É possível ser livre e continuar vivendo como escravo. Em Gálatas, Paulo escreve sobre outro tipo de escravidão: viver sob as exigências esmagadoras das regras religiosas. Neste versículo- -chave, Paulo afirma a seus leitores: “Para a liberdade foi que Cristo nos libertou. Por isso, permaneçam firmes e não se submetam, de novo, a jugo de escravidão” (GÁLATAS 5:1, NAA). Os seguidores de Jesus foram libertos das regras exteriores, incluindo o que comer ou de quem ser amigo. Muitos, porém, ainda viviam como escravos. Infelizmente, podemos fazer o mesmo hoje. Entretanto, quando cremos em Jesus, Ele nos liberta de uma vida de medo dos padrões religiosos humanos. A liberdade foi declarada. Sejamos livres em Seu poder!  Por: LISA SAMRA 

Listen, Watch, & Support DTP:  www.thereadinesslab.com/dtp-linksBoost the signal with a $5 monthly donation! Become a TRL Insider Member with a ton of extra content!--------------"We solve complex problems, and we help people."This mantra has embodied the work of the US Army Corps of Engineers (USACE) for the past 250 years.On this, the 250th Birthday of the Corps, the Disaster Tough Podcast hears the perspective of Major General Jason Kelly, its Deputy Commanding General of Civil & Emergency Operations.MG Kelly has an extensive military background, including 30+ years in USACE.  In this episode, he and DTP host, John Scardena, discuss a wide range of topics including:USACE's history dates back to the birth of America shortly after the RevolutionThe corps' efforts to make sure disasters don't do MORE damage than is necessaryHow USACE thinks about infrastructure from a security, risk management, response, and financial point of viewCounseling and advising leaders and stakeholders from a technical standpoint, regardless of politicsUSACE's work with FEMA and other entities is to be ready for disasters such as the upcoming hurricane season on the East Coast, or the recent fire season and cleanup efforts on the West Coast.As Major General Kelly says, "We deal with concrete and steel, but we don't PLACE concrete, and we don't PLACE any steel.  We do that with partners."On this Monday, June 16, DTP celebrates and honors the US Army Corps of Engineers for the way they have fulfilled this mission, and their partnership and contribution to America's prosperity and success.--------------Impulse: Bleeding Control Kits by Professionals for Professionals: https://www.dobermanemg.com/impulseDoberman Emergency Management Group provides subject matter experts in planning and training: www.dobermanemg.comFor sponsorship requests, check out our Sponsorship Portfolio here or email us at contact@thereadinesslab.com

Listen, Watch, & Support DTP:  www.thereadinesslab.com/dtp-linksBoost the signal with a $5 monthly donation! Become a TRL Insider Member with a ton of extra content!--------------This week on the Disaster Tough Podcast, we're joined by Andre Mackey, Chair of NATO's Specific Environments Panel (SPED)—and all-around powerhouse in the world of crisis leadership.Andre is an American based in Turkey, working directly with NATO to address the toughest environments on Earth—think urban warfare, cross-border coordination, and multi-agency response in unstable regions.In this episode, we dive into:NATO's response to current global flashpoints, including Ukraine and GazaThe role of SPED in preparing for and operating in extreme and urban environmentsReal-world insights on leadership during international emergenciesThe reality of coordinating across cultures, agencies, and bordersWe also want to give a big shoutout to our friends on the Specific Environments Panel (SEP). You know who you are—and we're grateful for the work you do.Listen now—this episode brings global perspective from someone who's living it.#DisasterTough #NATO #SPED #SEP #UrbanWarfare #EmergencyManagement #CrisisResponse #GlobalSecurity #Ukraine #Gaza #CrisisLeadership #PodcastEpisode--------------Impulse: Bleeding Control Kits by Professionals for Professionals:https://www.dobermanemg.com/impulse Doberman Emergency Management Group provides subject matter experts in planning and training: www.dobermanemg.com

In this AANEM podcast, Dr. Chilvana Patel interviews Dr. Devin Rubin and Dr. Nortina Shahrizaila about global disparities in EMG and nerve conduction study training. They discuss challenges such as inconsistent educational program standards, limited resources, and lack of mentorship, especially in developing countries. The experts are discussing the current efforts to improve access to hands-on training and standardized education.

Listen, Watch, & Support DTP:  www.thereadinesslab.com/dtp-linksBoost the signal with a $5 monthly donation! Become a TRL Insider Member with a ton of extra content!--------------The more you worry about the future, the less you enjoy the present.” Many working for federal agencies, such as FEMA, are worried about the future because of things happening in the present.For that reason, the Disaster Tough Podcast returns with a timely conversation with former FEMA Administrator Pete Gaynor. Pete served at the highest levels of FEMA and the Department of Homeland Security during President Trump's first term.Since then, he has been providing his expertise in the private sector, including his current role as Vice President of Resiliency and Disaster Recovery at Hill International, Inc.In this episode, Pete shares his thoughts on the current situation at FEMA amid recent reductions in workforce within the agency. He also discusses what employees and emergency management professionals can do to remain viable and ready in the face of uncertainty.He and host, John Scardena, point out the difficulties that EM professionals run into when it comes to response because of bureaucracy.He says the expectations often differ from the process and final response.As such, he calls for systems to be put in place so that state and local agencies have more ability to respond first, and only request federal assistance when necessary.--------------Impulse: Bleeding Control Kits by Professionals for Professionals:https://www.dobermanemg.com/impulse Doberman Emergency Management Group provides subject matter experts in planning and training: www.dobermanemg.com

Replay of our live Q&A today where I also interviewed EMG parents!We covered:-Why these parents joined EMG in the first place (everything from experiencing nerves before games, new sports teams, and lacking confidence)-Biggest wins they've noticed since joining-Favorite parts of the program (ease of use, coach on call texting, live calls, & simple tools!)-How to bring up the "mental game" without causing her to shut downThe What to Say Challenge Discount on EMG expires today at 11pm ET!Grab 50% off the program + bonuses here

Episode 187: In this episode of the Athletic Shoulder Podcast, we're joined by Annelies Maenhout, a visiting professor at Ghent University's Department of Rehabilitation Sciences and a leading researcher in shoulder rehabilitation. As a core member of the university's Upper Limb Research Team, Annelies has contributed significantly to the field through her work on EMG, motor learning, and kinetic chain mechanics. Her research bridges cutting-edge science with clinical practice, offering insights into how shoulder rehab can be refined through eccentric loading, implicit motor learning, and neuromechanics. With a forward-thinking approach, she explores how VR, brain activity, and reaction time play a role in optimizing rehab for overhead athletes. Topics Discussed: EMG research on kinetic chain and plyometric exercises How her view on GIRD (glenohumeral internal rotation deficit) evolved over time Key findings from eccentric training research Transitioning from internal to external attentional focus in rehab Applying implicit motor learning in shoulder rehabilitation The current gap in research on reaction time in overhead athletes Exploring the potential of VR and neuroscience in shoulder rehab Future directions in shoulder research: muscle synergies and brain activity Whether you're a clinician, coach, or researcher, this episode offers valuable insights into the future of evidence-based shoulder rehabilitation. - Where you can find Annelies: LinkedIn ResearchGate -  Sponsors VALD Performance, makers of the Nordbord, Forceframe, ForeDecks and HumanTrak. VALD Performance systems are built with the high-performance practitioner in mind, translating traditionally lab-based technologies into engaging, quick, easy-to-use tools for daily testing, monitoring and training Hytro: The world's leading Blood Flow Restriction (BFR) wearable, designed to accelerate recovery and maximise athletic potential using Hytro BFR for Professional Sport. -  Where to Find Us Keep up to date with everything that is going on with the podcast by following Inform Performance on: Instagram Twitter Our Website - Our Team Andy McDonald Ben Ashworth Alistair McKenzie Dylan Carmody Steve Barrett  Pete McKnight

​ @igrejakyrios  | Igreja Evangélica KyriosFeliz Dia das Mães!Culto do dia 11.05.2025 no período da Manhã - 9h Gênesis 21:9-21 (NVI) Você já se sentiu rejeitado, como se estivesse num deserto, sozinho?Em Gênesis 21:9-21, aprendemos algo poderoso: o que o homem rejeita, Deus assume.Agar foi lançada no deserto com seu filho. Parecia o fim. Mas Deus a viu. Deus ouviu o clamor.Talvez você esteja no seu deserto agora, mas Deus já preparou um poço, um escape, uma nova chance.Às vezes, a dor cega. Mas Deus quer abrir seus olhos para enxergar o que já está aí: sustento, direção, esperança.A rejeição não define você. O que você faz com a vida que Deus te deu, sim.Levante-se! Segure na mão do seu filho, da sua história, dos seus sonhos — e siga em frente.Compartilhe essa palavra com alguém... ✨Ouça nossa música autoral!https://youtu.be/htZ9wZZryaM?si=uGKU5E0CfqprJfqCSe conecte conosco!https://portal.igrejakyrios.com.br/fale-conosco/Inscreva-se no nosso canal: www.youtube.com/@igrejakyrios Nosso Site: http://www.igrejakyrios.com.brInstagram: https://www.instagram.com/igrejakyrios/

How did you fare on your March Madness brackets? Did you win your EMG lab pool? Did you pick any upsets? Tune in and you will be sure to win this Lessons from the Lab March Madness bracket. It's full of surprises and successes, and you won't be “upset".

What if the secret to your daughter's confidence isn't more practice… but a mental game shift?Two sports moms (one with a 9-year-old gymnast, another with a 12-year-old softball catcher) spill exactly how they transformed meltdowns into comebacks and rebuilt their own parenting playbooks along the way.

Send us a textParsonage-Turner Syndrome can mimic several other shoulder conditions, leading to misdiagnosis and ineffective treatment if not properly identified and differentiated.• PTS typically presents with sudden onset of severe shoulder pain lasting 2-3 weeks, often triggered by viruses or vaccinations• Common symptoms include limited active and passive range of motion, weakness, atrophy (especially in deltoids), and sometimes altered sensation• Rotator cuff tears differ by having better passive than active motion and usually having a clear mechanism of injury• Cervical nerve root compression can be distinguished by performing Spurling's test and gentle cervical traction• Adhesive capsulitis has a slower onset than PTS and typically doesn't cause the significant atrophy seen in PTS cases• Diagnostic imaging should be used after thorough clinical examination to confirm suspected diagnosis• EMG/nerve conduction studies are most helpful for confirming PTS after 3-4 weeks of symptoms• Always check for skin changes like pustules or rashes that might indicate shingles, which can cause brachial neuritisJoin us for our upcoming live course on May 31st, 2025 where we'll cover more differential diagnoses like these. Visit the website link in the show notes for more information and to reserve your spot.

This is a replay of the recent training we hosted for Sports Moms "The Do's and Don'ts of Strengthening Your Athlete's Mental Game"! Spring enrollment special (50% off the program + bonuses) expires Friday, April 4th! Grab the program with the discount hereShe's talented, she's dedicated, but something's holding her back… and it's not her skills. If you've ever watched your daughter struggle with confidence, perfectionism, or bouncing back from mistakes on the field, this episode is for you. As a parent, it's heartbreaking to see your girl spiral after a setback, or hear her talk herself down after a game. You've told her she's good enough, she's strong enough, but how can you actually help her believe it?This episode will give you the tools to help her break free from self-doubt and start playing with the confidence she deserves!Here's what we cover in this episode:How to help your athlete bounce back from mistakes using the “Bounce Back in a Snap” method.Why perfectionism is holding her back, and how to help her release the pressure.The power of flipping negative self-talk into productive, confidence-building thoughts.Real-life Q&A with sports moms like you, plus expert advice.A special, limited-time offer for the Elite Mental Game program that's designed to quickly strengthen your daughter's mental game and confidence.Don't wait – this episode is only up for 48 hours! Listen now to learn how you can support your athlete's mental game, and unlock a 50% discount on the Elite Mental Game program before the offer ends this Friday, January 31st!Listen to the full episode now and empower your girl to play with confidence – both in sports and in life.Episode Highlights: [00:00] Raising Confident and Mentally Strong Girl Athletes. This is a 48-hour special, featuring a live training session on strengthening an athlete's mental game.[03:20] Common Challenges Faced by Athletes. Discussing the common concerns of parents about their daughters' handling of sports and life challenges.[05:14] Misconceptions About Building Confidence. Debunking common misconceptions about building confidence in athletes, such as relying on coaches or expecting confidence to develop naturally.[07:56] The Unstoppable Athlete Method. Introducing the Unstoppable Athlete Method, a solution used to coach high school volleyball teams.[10:38] Bounce Back in a Snap. The Bounce Back in a Snap method, which helps athletes recover from mistakes quickly.[23:05] Releasing the Pressure Athletes Feel. Discussing the pressure athletes feel, including perfectionism, comparison, and pregame anxiety and understanding the concept of noticing and shifting thoughts to release the pressure.[34:13] Flipping Negative Mindset. The challenge of athletes turning positive feedback into negative self-talk. Understanding negativity bias and the importance of self-trust in building confidence.[42:40] The Role of Parents in Mental Training. The importance of parents in shaping their children's environment and providing opportunities for mental training. And tips for parents to improve their own self-talk and support their children's mental training.[42:50] The Elite Mental Game Program. Introducing the Elite Mental Game program, a self-paced mental training system for girl athletes.[01:08:27] Live Support and Bonuses. Introducing the live support and bonuses included in the Elite Mental Game program.[01:08:07] Enrollment and Special Offers. Details on how to enroll in the Elite Mental Game program, including the enrollment checkout process. Grab the Spring Enrollment Discount + Bonuses on EMG until April 4th here!

Em Gênesis 22:2, vemos uma história de fé, obediência e entrega total a Deus. O que podemos aprender com a vida de Abraão, que saiu da desobediência para se tornar um exemplo de fé?

PX Docs articles to learn more about POTS: click hereTommie's story of HOPE: click hereEpisode on Vagus Nerve Dysfunction referenced: click hereIn this episode, Dr. Tony Ebel breaks down Postural Orthostatic Tachycardia Syndrome (POTS), a condition that affects many teenagers and young adults, particularly females. Dr. Ebel explains that POTS is not a mystery condition but rather a neurological issue stemming from vagus nerve dysfunction, dysautonomia, nervous system dysregulation, and subluxation. He criticizes conventional medical approaches that merely treat symptoms while missing the root cause and offers a chiropractic perspective on how to effectively address POTS through nervous system-focused care. Dr. Ebel provides hope for those suffering from POTS, emphasizing that healing is possible through natural, drug-free methods.[00:00:00] Introduction & Overview of POTSPOTS is not a mystery condition but a straightforward neurological issuePrimarily affects teenagers and young adults, especially femalesMedical approach fails by merely labeling symptoms without addressing root causes[07:00:00] POTS Symptoms & Root CausesSymptoms: dizziness, rapid heartbeat, chronic fatigue, easily triggered anxietyRoot causes: nervous system dysregulation, vagus nerve dysfunction, dysautonomia, subluxationHow vagus nerve connects brain and gut, affecting everything from balance to anxiety[14:00:00] Conventional vs. Functional ApproachesConventional: salt intake, beta blockers, and "slowing down" recommendationsFunctional medicine: diet, physical activity, gut health, stress managementWhy both approaches still fall short without addressing neurological root causes[18:00:00] Patient Patterns & Physical SignsCommon history: birth interventions, childhood illnesses like colic and ear infectionsPhysical signs: forward head posture ("tech neck"), tension at base of skullThe bowling ball analogy explaining how posture affects nervous system function[23:00:00] Assessment & Treatment ApproachAssessment tools: INSiGHT scans (thermal, EMG), heart rate variability (HRV)Care plan: specific chiropractic adjustments, postural correction techniquesWhy complete healing takes time and follows a non-linear recovery path[28:00:00] Call to ActionFinding a PX doctor through PXdocs.com-- Follow us on Socials: Instagram: @pxdocs Facebook: Dr. Tony Ebel & The PX Docs Network Youtube: The PX Docs For more information, visit PXDocs.com to read informative articles about the power of Neurologically-Focused Chiropractic Care. Find a PX Doc Office near me: PX DOCS DirectoryTo watch Dr. Tony's 30 min Perfect Storm Webinar: Click HereSubscribe, share, and stay tuned for more incredible episodes unpacking the power of Nervous System focused care for children!

Got a strong-willed, fiercely independent kid who's equal parts exhausting and amazing? You're not alone – and this episode is for you. Learn how to parent your ‘spicy one' without losing your mind, and help them thrive in sports and life!What's Covered in This EpisodeWhat is a “spicy one”? Strong-willed, independent, and fiercely competitive kids who march to their own beat.Why spicy ones make great athletes (and challenging kids). Their intensity and drive set them apart.How to handle resistance to feedback. Why stepping back and cheering them on works better than pushing.The power of the pause. Why taking a breath before reacting can change everything.Staying calm as a parent. How your calm demeanor helps regulate your child's emotions.Balancing independence and respect. Teaching your spicy one to advocate for themselves while respecting authority.Letting the world teach lessons. Why natural consequences are sometimes the best teachers.Mary's best advice for parents: “Your main job is to delight in them.”Ready to learn more? Check out Mary Van Geffen's resources at maryvangeffen.com and explore her programs like Moms of Spicy Ones and Kind Class. And if your athlete needs mental training, head to EliteCompetitor.com/EMG to learn more about The Elite Mental Game.Parenting a spicy one isn't easy, but it's worth it. Let's do this together!Episode Highlights: [00:01:15] What Is a “Spicy One”? A “spicy one” is a strong-willed, independent, and fiercely competitive kid who marches to the beat of their own drum. These traits make them incredible athletes but can also be challenging to parent.[00:02:09] Meet Mary Van Geffen. Mary is an international parenting coach who helps moms of spicy ones cultivate warmth, tenderness, and self-compassion. Her mission is to help parents thrive while raising these fiery future leaders.[00:05:26] Traits of a Spicy One. Spicy ones are intense, fiercely independent, and often resist feedback or authority. They're not afraid to take up space, which makes them great athletes but also challenging to parent.[00:11:09] Understanding Adolescent Brains. Teens experience adult-sized emotions without the rational thinking to match, which can lead to big ups and downs. Their brains are still under construction, so patience is key.[00:18:47] The Power of the Pause. Mary's top tip: pause before reacting to your child's behavior. Taking a moment to breathe helps you respond thoughtfully instead of reacting impulsively.[00:24:37] Balancing Independence and Respect. Encourage your spicy athlete's independence while teaching them to respect authority figures like coaches and referees. It's a delicate but important balance.[00:30:50] Mary's Best Advice for Parents. Mary's top advice: “Other people can teach them life skills, but your main job is to delight in them.” Your unconditional love is what they need most.[00:34:58] Closing Thoughts. Parenting a spicy one is challenging but incredibly rewarding – let's keep supporting each other and our amazing kids.Next Steps:Join our FREE Training for Sports Moms - How to Strengthen Your Athlete Daughter's Mental Game so She Believes in Herself as Much as You DoVisit our podcast website for more great episodesThank you in advance for joining us on our mission and leaving a rating and review on Grab your exclusive Elite Mental Game discount for being a podcast listener here!

In this special episode, we're pulling back the curtain on The Elite Mental Game (EMG) with a candid conversation featuring two incredible moms—Samarra, mom to a 12 (soon to be 13)-year-old softball player, and Allegra, mom to a 9-year-old gymnast. They share their firsthand experiences of watching their daughters grow in confidence, handle pressure, and navigate the mental side of their sports with the help of EMG.Even though their athletes are on the younger end of the spectrum, we dive into why ages 11-18 is the ideal time to develop strong mental game skills—and how the foundation they're building now is setting them up for long-term success.Inside This Episode:✅ Why they joined EMG – What struggles led them to seek mental training for their daughters?✅ The biggest mindset shifts – How their daughters have changed in confidence, resilience, and handling mistakes.✅ Handling pressure & expectations – How EMG has helped their athletes navigate tough moments in competition.✅ What surprised them most – Insights they didn't expect from the program that made the biggest difference.✅ The mom's role in mental training – How they've supported their daughters and what they've learned along the way.✅ Live Q&A! – We answer common questions from moms considering EMG, including:How do I know if my daughter is ready for mental training?Will she actually want to do the program?What if I've tried to help her before and nothing has worked?How do we balance this with an already busy sports schedule?This episode is a must-listen for any mom wondering if EMG is the right fit for their daughter. Whether she's struggling with confidence, perfectionism, or performance pressure, these real stories will give you insight into how mental training can be a game-changer. EMG Winter Enrollment is happening NOW! -50% off the program-Ditch the Drama training bundle for FREE-FREE ticket to our Peak Performance Clinic "How to Stay Motivated Through the Grind"Https://elitecompetitor.com/emgspecialDiscount + Bonuses Expire January 31st!

www.patreon.com/banjopodcast Today we have a follow-up episode to #152, which featured the engineers behind the innovative ToneDexter product, by Audio Sprockets. In this one, host Keith Billik demonstrates what ToneDexter can do, by programming wavemaps using every combination of 3 different banjo pickups (by K&K, Schatten, and EMG) and 5 different microphones (by Rode, Behringer, Shure, Audio Technica, and Mesanovic). Sponsored by Elderly Instruments, Peghead Nation, Sullivan Banjos, and Bluegrass Country Radio Episode Time Codes: Intro & explanation of WaveMap Shootout - 03:40   Microphone Base Samples (Not using ToneDexter): Rode NT5 (Small diaphragm condenser) - 12:27 Behringer ECM8000 (Omnidirectional measurement microphone) - 17:27 Shure SM57 (Dynamic cardioid) - 22:29 Audio Technica AT4047(Large diaphragm Condenser) - 27:05 Mesanovic Model 2 (Figure 8 ribbon) - 31:46   Pickup Base Samples (Not using ToneDexter): K&K Banjo Twin - 40:38 Schatten BJ-02 - 47:13 EMG ACB Barrel - 53:47   Wavemap Samples   K&K Pickup wavemaps: Rode NT5 - 42:18 Behringer ECM8000 - 43:18 Shure SM57 - 44:16 Audio Technica AT4047 - 45:14 Mesanovic Model 2 - 46:14   Schatten Pickup WaveMaps: Rode NT5 - 48:26 Behringer ECM8000 - 49:29 Shure SM57 - 50:32 Audio Technica AT4047 - 51:38 Mesanovic Model 2 - 52:42   EMG ACB Barrel Pickup WaveMaps: Rode NT5 - 55:02 Behringer ECM8000 - 56:08 Shure SM57 - 57:16 Audio Technica AT4047 - 58:22 Mesanovic Model 2 - 59:30   Conclusion + Keith's Reaction - 1:00:36   Contact the show: pickyfingersbanjopodcast@gmail.com  

Feeling stuck between wanting the best for your athlete and navigating their eye rolls and closed-off responses? You're not alone. This episode is for every mom who's been in the trenches, wondering if what they're doing is even working. Get ready to hear from three real moms who've walked that road and are sharing their unfiltered experiences with The Elite Mental Game. From the early struggles of getting their daughters to buy in to the game-changing moments that made it all worth it – you're about to get the real talk you've been craving. What You'll Learn in This Episode:Early Wins: The first shifts moms noticed in their daughters' confidence and resilience.Snapback Success: How one simple routine helped athletes bounce back from mistakes in seconds.Parent Perspectives: The biggest mindset shifts moms made to support their athletes.Team Dynamics: Strategies for helping athletes navigate new teams and relationships.Navigating Feedback: How EMG tools help athletes handle constructive criticism and grow from it.Live Coaching Benefits: Why athletes love connecting with coaches during live calls and text support.Flexible Training: How EMG fits seamlessly into even the busiest schedules.Why It's Worth It: Hear firsthand why these moms believe the program has been life-changing – not just for their athletes but for their entire families.Ready to hear the raw, unfiltered truth about what it's like inside The Elite Mental Game? Grab your headphones and listen now. Your perspective as a sports mom might just shift forever.Episode Highlights: [00:00:00] Introducing today's focus: hearing directly from three moms with daughters in the Elite Mental Game (EMG). These moms are at different stages of the program, giving you an inside look at how EMG supports athletes and their families.[00:02:00] Shoutout to Jamie: One of our moms shares how her daughter, an equestrian athlete, used EMG tools to maintain her poise during a challenging horse show, earning praise from her coach for staying calm under pressure.[00:05:00] When to Start: I share why starting mental training between ages 11 and 12 is ideal but emphasize that it's never too late for athletes to build confidence and mental strength.[00:09:30] Answering Common Questions: I address questions I frequently get, like how to help athletes stay consistent with mental training and ways to encourage buy-in.[00:25:00] Why They Joined EMG: Each mom shares what brought them to EMG – whether it was to help their daughters overcome self-doubt, navigate team dynamics, or strengthen their own communication as sports parents.[00:38:00] Long-Term Growth: Kim shares how her daughter, now a senior and team captain, revisits EMG tools to manage feedback, lead her team, and maintain confidence as she transitions to college basketball.[00:45:00] Favorite Parts of EMG: The moms highlight their favorite features, including the flexibility to work at their own pace, the supportive community, and practical tools that empower both athletes and parents.[00:55:00] Watching Game Film: I share how to review post-game recordings constructively, focusing on identifying strengths and actionable goals to guide practice without overwhelming athletes.[01:07:00] Final Reflections: Each mom reflects on the program's impact, from helping their daughters grow as athletes and individuals to strengthening their relationships as families.Next Steps:Join our FREE Training for Sports Moms - How to Strengthen Your Athlete Daughter's Mental Game so She Believes in Herself as Much as You DoVisit our podcast website

In this engaging conversation on the Crazy Wisdom podcast, Stewart Alsop talks with neurologist Brian Ahuja about his work in intraoperative neurophysiological monitoring, the intricate science of brainwave patterns, and the philosophical implications of advancing technology. From the practical applications of neuromonitoring in surgery to broader topics like transhumanism, informed consent, and the integration of technology in medicine, the discussion offers a thoughtful exploration of the intersections between science, ethics, and human progress. Brian shares his views on AI, the medical field's challenges, and the trade-offs inherent in technological advancement. To follow Brian's insights and updates, you can find him on Twitter at @BrianAhuja.Check out this GPT we trained on the conversation!Timestamps00:00 Introduction to the Crazy Wisdom Podcast00:21 Understanding Intraoperative Neurophysiological Monitoring00:59 Exploring Brainwaves: Alpha, Beta, Theta, and Gamma03:25 The Impact of Alcohol and Benzodiazepines on Sleep07:17 The Evolution of Remote Neurophysiological Monitoring09:19 Transhumanism and the Future of Human-Machine Integration16:34 Informed Consent in Medical Procedures18:46 The Intersection of Technology and Medicine24:37 Remote Medical Oversight25:59 Real-Time Monitoring Challenges28:00 The Business of Medicine29:41 Medical Legal Concerns32:10 Alternative Medical Practices36:22 Philosophy of Mind and AI43:47 Advancements in Medical Technology48:55 Conclusion and Contact InformationKey InsightsIntraoperative Neurological Monitoring: Brian Ahuja introduced the specialized field of intraoperative neurophysiological monitoring, which uses techniques like EEG and EMG to protect patients during surgeries by continuously tracking brain and nerve activity. This proactive measure reduces the risk of severe complications like paralysis, showcasing the critical intersection of technology and patient safety.Brainwave Categories and Their Significance: The conversation provided an overview of brainwave patterns—alpha, beta, theta, delta, and gamma—and their connections to various mental and physical states. For instance, alpha waves correspond to conscious relaxation, while theta waves are linked to deeper relaxation or meditative states. These insights help demystify the complex language of neurophysiology.Transhumanism and the Cyborg Argument: Ahuja argued that humans are already "cyborgs" in a functional sense, given our reliance on smartphones as extensions of our minds. This segued into a discussion about the philosophical and practical implications of transhumanism, such as brain-computer interfaces like Neuralink and their potential to reshape human capabilities and interactions.Challenges of Medical Technology Integration: The hype surrounding medical technology advancements, particularly AI and machine learning, was critically examined. Ahuja highlighted concerns over inflated claims, such as AI outperforming human doctors, and stressed the need for grounded, evidence-based integration of these tools into healthcare.Philosophy of Mind and Consciousness: A recurring theme was the nature of consciousness and its central role in both neurology and AI research. The unresolved "hard problem of consciousness" raises ethical and philosophical questions about the implications of mimicking or enhancing human cognition through technology.Trade-offs in Technological Progress: Ahuja emphasized that no technological advancement is without trade-offs. While tools like CRISPR and mRNA therapies hold transformative potential, they come with risks like unintended consequences, such as horizontal gene transfer, and the ethical dilemmas of their application.Human Element in Medicine: The conversation underscored the importance of human connection in medical practice, particularly in neurology, where patients often face chronic and emotionally taxing conditions. Ahuja's reflections on the pitfalls of bureaucracy, private equity in healthcare, and the overemphasis on defensive medicine highlighted the critical need to prioritize patient-centered care in an increasingly technological and administrative landscape.

Listen, Watch, & Support DTP: www.thereadinesslab.com/dtp-links Boost the signal with a $5 monthly donation! Become a TRL Insider Member with a ton of extra content! #emergencymanagement #disastertough #leadership #emergencyservices --------------Creating resilient communities is the goal of today's guest in the Disaster Tough Podcast.  JT White and Susanna Pho joined forces in 2019 to form Forerunner, a software platform designed to empower communities to prepare better, respond, and plan for future disasters and major incidents.The company specializes in its floodplain management platform which is designed to automate workflows for professionals to manage their resources better.  However, the company is continuing to work toward becoming an "All Hazards Resilience Platform."In this episode of the Disaster Tough Podcast, Susanna and JT go back to their days as colleagues at MIT, and discuss how their idea for a company came about. They also discuss, among other things, how they work to lead a growing company from opposite sides of the country.  To learn more about Forerunner and its platform, visit https://www.withforerunner.com/--------------*Major Endorsements: L3Harris's BeOn PPT App.Learn more about this amazing product here: https://www.l3harris.com/ Impulse: Bleeding Control Kits by Professionals for Professionals: https://www.dobermanemg.com/impulseEmergency Management for Dynamic Populations (DyPop): Hot Mess Express: An emergency management leadership course focusing on response tactics during terrorist attacks.Hot Mess Express includes an immersive exercise during an intentional train derailment scenario. Register for DyPop here: https://www.thereadinesslab.com/shop/p/dynamicDoberman Emergency Management Group provides subject matter experts in planning and training: www.dobermanemg.com

Dr. Christina Prevett // #ICEPelvic // www.ptonice.com  In today's episode of the PT on ICE Daily Show, ICE Pelvic division leader Christina Prevett discusses the rising trend of the anti-Kegel movement within pelvic health. She explores the origins of the anti-Kegel sentiment, referencing a study that compared EMG activation from Kegel exercises to that of other core exercises such as planks and leg lifts. This study led some to believe that whole body exercises could replace Kegels, a notion Christina critically examines. She presents both sides of the argument, emphasizing the need for a balanced approach to pelvic health moving forward into 2025. Take a listen to learn how to better serve this population of patients & athletes or check out the full show notes on our blog at www.ptonice.com/blog. If you're looking to learn more about our live pregnancy and postpartum physical therapy courses or our online physical therapy courses, check our entire list of continuing education courses for physical therapy including our physical therapy certifications by checking out our website. Don't forget about all of our FREE eBooks, prebuilt workshops, free CEUs, and other physical therapy continuing education on our Resources tab. Are you looking for more information on how to keep lifting weights while pregnant? Check out the ICE Pelvic bi-weekly newsletter!

Listen, Watch, & Support DTP: www.thereadinesslab.com/dtp-links Boost the signal with a $5 monthly donation! Become a TRL Insider Member with a ton of extra content! #emergencymanagement #disastertough #leadership #emergencyservices --------------Learning from experience and dealing with physically and emotionally draining situations is necessary for anyone working in or adjacent to the Emergency management field.Krista Haugen, National Director of Patient Safety for Global Medical Response does her best to do this daily.As a longtime medical professional with extensive experience as a Registered Nurse, Master of Nursing, and Certified Medical Transport Executive, Director Haugen brings over 25 years of experience in emergency, critical care, and flight nursing.  While often caring for others in crisis, Krista is no stranger to trauma herself, as she is also an EMS helicopter crash survivor.   In this episode of the Disaster Tough Podcast, Director Haugen discusses the many lessons she has learned over multiple decades of emergency response and critical care, along with important attributes such as empathy, adaptability, and responsibility. --------------*Major Endorsements: L3Harris's BeOn PPT App.Learn more about this amazing product here: https://www.l3harris.com/ Impulse: Bleeding Control Kits by Professionals for Professionals: https://www.dobermanemg.com/impulseEmergency Management for Dynamic Populations (DyPop): Hot Mess Express: An emergency management leadership course focusing on response tactics during terrorist attacks.Hot Mess Express includes an immersive exercise during an intentional train derailment scenario. Register for DyPop here: https://www.thereadinesslab.com/shop/p/dynamicDoberman Emergency Management Group provides subject matter experts in planning and training: www.dobermanemg.com