Podcasts about psychomotor

Her son Hudson was born with a rare genetic syndrome called Koolen-de Vries. Kaci shares their journey which led to founding Stone Wall Meadows, TN which provides belonging, growth and job to individuals with special needs and disabilities and their families. Find her @stonewallmeadows   KdVS is characterized by congenital malformations, developmental delay / intellectual disability, neonatal/childhood hypotonia, epilepsy, dysmorphisms, and behavioral features. Psychomotor developmental delay is noted in all individuals from an early age. NIH.gov   Check out our website: greenergrasspodcast.com Get the Greener Grass Newsletter HERE Part of the Digitent Podcast Network   Find us at: IG: @grandrevecreative Twitter: @grass_podcast Email:  kerry@grandrevecreative.com The Expecting Aerialist Podcast  See omnystudio.com/listener for privacy information.

Learn More (Video Study Course): https://www.prepareforems.comGet my EKG/12-Lead EKG Masterclass: https://www.ekgforems.comI had to report on yet another important EMS education update from NREMT. The NREMT psychomotor exams for ALS providers such as advanced EMT and Paramedic level exams are being discontinued starting July 1st, 2024. I believe this is a good step in the right direction for our profession as one of the biggest complaints of NREMT candidates would be an inconsistent testing experience due to different types of proctors or testing centers. This will be especially helpful for those students who if failed had to previously travel to other locations (some time hours and hours away) for another attempt to pass. Last year NREMT had removed the EMR and EMT psychomotor exams and now they have followed that up with removing the AEMT and Paramedic psychomotor exams. Now students at all levels will have their skills signed off by their class instructors and program directors. Now every level of certification will have one cognitive examination as reported by NREMT, I never thought I would see the day! Be great, Evan, The Paramedic Coach #emt #nremt #ems

5.10 Mood Disorders (Depression and Bipolar) Psychiatry Review for the USMLE Step 1 exam Mood disorders are persistent disruptions in emotion, categorized into bipolar disorders and depressive disorders. Bipolar disorders are characterized by manic or hypomanic episodes, while depressive disorders feature periods of depression. Major Depressive Disorder (MDD) is characterized by feelings of sadness, guilt, worthlessness, and anhedonia lasting for at least two weeks. SIG E CAPS is an acronym used to remember the symptoms of depression: Sleep changes, Interest loss, Guilt, Energy loss, Concentration difficulties, Appetite changes, Psychomotor retardation, and Suicidal ideation. In MDD, at least 5 of the SIG E CAPS symptoms must persist for more than 2 weeks. Persistent Depressive Disorder (dysthymia) is a chronic, low-grade form of MDD that lasts for at least two years. Mania is a symptom common to all bipolar disorders, characterized by elevated or irritable mood and increased activity or energy. Mania lasting for at least 1 week is called a manic episode, while hypomania is a less severe form lasting longer than 4 days. The symptoms of mania can be remembered using the mnemonic DIG FAST: Distractibility, Irritable mood/insomnia, Grandiosity, Flight of ideas, Agitation/increased activity, Speedy thoughts/speech, and Thoughtlessness. Bipolar I involves manic episodes, and bipolar II involves hypomanic episodes and major depressive episodes. Bipolar I requires at least one manic episode, while bipolar II requires depressive episodes. Mood stabilizers such as lithium, valproate, carbamazepine, and lamotrigine are used to treat bipolar disorders. Litium is the best (notable exeptions however). Antidepressants are contraindicated for bipolar patients due to the risk of flipping into mania. The risk of suicide is high in bipolar patients, with 25-50% attempting suicide and 10-15% dying by suicide. Cyclothymia is a less common form of bipolar disorder characterized by cycling between hypomania and mild depression over many years.

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https://psychiatry.dev/wp-content/uploads/speaker/post-10910.mp3?cb=1669144551.mp3 Playback speed: 0.8x 1x 1.3x 1.6x 2x Download: The Behavioral Mapping of Psychomotor Slowing in Psychosis Demonstrates Heterogeneity Among Patients Suggesting Distinct Pathobiology – Niluja Nadesalingam et al. Schizophrenia Bulletin.Full EntryThe Behavioral Mapping of Psychomotor Slowing in Psychosis Demonstrates Heterogeneity Among Patients Suggesting Distinct Pathobiology –

https://psychiatry.dev/wp-content/uploads/speaker/post-10017.mp3?cb=1664244358.mp3 Playback speed: 0.8x 1x 1.3x 1.6x 2x Download: Paratonia, Gegenhalten and psychomotor hypertonia Back to the roots – PubMed Jack R Foucher et al. Schizophrenia Research. 2022. In the firstFull EntryParatonia, Gegenhalten and psychomotor hypertonia Back to the roots – PubMed

On today's episode, Rachel and Alex sit down with Dr. Corey Keeton to discuss how to best take care of patients who are depressed and/or have suicidal ideations.  Dr. Corey Keeton is a a board-certified Family Medicine and Psychiatry physician. Dr. Keeton is currently the medical director of the inpatient consultation and liaison psychiatry service at the University of Cincinnati Medical Center and a learning community preceptor. He earned his medical degree at the Joan C. Edwards School of Medicine at Marshall University and completed his residency in both Family Medicine and Psychiatry at the University of Cincinnati College of Medicine. It is our hope that after listening to this episode you'll feel more comfortable addressing depression and suicidal ideation not only in your patients, but also family and friends. Together we can help erase mental health stigmatization and help identify those in need of help.General resources:SIGECAPS (Sleep, Interest, Guilt, Energy, Concentration, Appetite, Psychomotor activity, Suicidal ideations)PHQ-9 questionnaire for depression: https://www.mdcalc.com/phq-9-patient-health-questionnaire-9Note from Unscripted Medicine Team: If you are experiencing depression, anxiety, suicidal ideations know that you do not have to go at it alone.  Please reach out to those close to you or use the numbers listed below. Local (Cincinnati) resources:Psychiatry Emergency Services: 513-584-8577Holmes Clinic (on call nights & weekends): 513-584-4457CAPS same day emergent services: 513-556-0648 M-F 8-5pCAPS 24 hour helpline: 513-556-0648 and press 1National resources: National Suicide Prevention Lifeline: 1-800-273-TALK (8255)Crisis text line: text “connect” to 741741

Chris McCarthy is the EMS Program Manager for Northeast Emergency Training Solutions, LLC. With her 20 years of experience as an EMS educator, Instructor Coordinator and National Registry Representative, she will discuss the new portfolio competency program that will be replacing the psychomotor testing for EMT testing. Interested to hear more? Tune in to see how […]

सुनिए स्नेहा भाटिया के जीवन की प्रेरक कहानी। स्नेहा भाटिया ने पेशेवर जीवन में बेहतर करने के साथ साथ अपने व्यक्तिगत जीवन को भी बहुत सुन्दरता से संवारा है।जी हाँ जहां ओर पेशे से शिक्षिका होने के नाते इन्होंने अपने छात्रों का जीवन संवारने में अपना अहम योगदान दिया, तो वही एक माँ होने के नाते अपनी दोनों बच्चियों की भी अच्छी तरह से परवरिश की और घर के हर सदस्य के प्रति अपनी सभी ज़िम्मेदारीयों को समझते हुए उन्हें बखूबी निभाया । आपको बतादें स्नेहा भाटिया का जन्म और पालन पोषण अजमेर शहर में हुआ। शहर के ही एक प्रतिष्ठित सेंट मेरिज कान्वेंट स्कूल से स्कूली शिक्षा प्राप्त करने के बाद सोफ़िया गर्ल्स कॉलेज से अपनी स्नातक की पढ़ाई पूरी की और फिर शादी के बाद इन्होंने मुंबई शहर में प्रवेश किया। मुंबई जैसे महानगर में भी इन्होंने अपने स्तर पर खुद को स्थापित करने का प्रयास किया। जी हाँ आपको बतादें स्नेहा भाटिया को हमेशा से ही खुद के साथ-साथ लोगों को संवारना, उन्हें तैयार करना बेहद पसंद था और इसके लिए इन्होंने ब्यूटिशियन का कोर्स भी किया था। ऐसे में इन्होंने इसी में इन्होने इसी क्षेत्र में प्रवेश करते हुए एक छोटे से ब्यूटी क्लीनिक से अपने करियर की शुरुआत की जहां इन्हें अपनी क्रिएटिविटी दिखाने का मौका मिला। कुछ वक़्त इसी क्षेत्र में बेहतर करने के बाद जब हस्बैंड का दुबई में ट्रांसफर हुआ। तो ये भी दुबई ही शिफ्ट हो गयी और फिर इन्होंने आगे बढ़ने के लिए नए क्षेत्र का चयन किया और एक शिक्षिका के रूप में अपने करियर को आगे बढ़ाया। आपको बतादें स्नेहा भाटिया ने 'अवर ओन इंग्लिश हाई स्कूल', शारजाह में एक दशक से भी अधिक समय तक काम किया और जब परिवार संग दुबई से उदयपुर शिफ्ट हुई तो उदयपुर में भी बतौर शिक्षिका अपने सफर को जारी रखा और उदयपुर शहर के प्रतिष्ठित स्कूल में कुछ वर्षों तक अपनी सेवाएं दी। आज ये युवाओं को भी अपनी इस कहानी के माध्यम से शिक्षण पेशे को अपनाते हुए आगे बढ़ने के लिए प्रेरित कर रही है। पूरी कहानी पढ़ें: https://stories.workmob.com/sneha-bhatia-education-academiaवर्कमोब द्वारा #मेरीकहानी कार्यक्रम के माध्यम से एक नयी पहल शुरू की गयी है जिसके ज़रिये हर कोई छोटे बड़े बिज़नेस ओनर्स अपनी प्रेरक कहानियों को यहाँ सभी के साथ साझा कर सकते है। क्योंकि हर शख्स की कहानी में है वो बात जो जीवन को बदलकर एक नयी दिशा दिखाएगी, और ज़िन्दगी में ले आएगी आशा की एक नयी चमकती किरण। #प्रेरककहानियाँ #स्नेहा भाटिया #शिक्षिका #सेंट मेरिज कान्वेंट स्कूल #सोफ़िया गर्ल्स कॉलेज #ब्यूटिशियन #ब्यूटी क्लीनिक #क्रिएटिविटी #शिक्षण जानिए वर्कमोब के बारे में: जुड़िये वर्कमोब पर अपनी कहानी साझा करने और प्रेरणादायक कहानियाँ देखने के लिए। ये एक ऐसा मंच है जहां आप पेशेवरों, लघु व्यापारियों, उद्यमियों और सामाजिक कार्यकर्ताओं की वीडियो कहानियां देख सकते हैं और दूसरों को प्रेरित करने के लिए अपनी व्यक्तिगत और व्यावसायिक कहानी सभी के साथ साझा कर सकते हैं। आपकी कहानी में लोगों को आशा देने, प्रेरणा देने और दूसरों का जीवन बदलने में मदद करने की एक अद्भुत क्षमता है। यह 100% मुफ़्त है। इस लिंक पर क्लिक करें और देखें प्रेरक कहानियां https://stories.workmob.com/हमारे ऐप्प को डाउनलोड करें: Android: https://play.google.com/store/apps/details?id=com.workmob iOS: https://apps.apple.com/in/app/workmob/id901802570  

In this episode of Inside EMS, co-hosts Chris Cebollero and Kelly Grayson discuss the launch of the EMS Trend Survey 2022, which will provide the data for the 7th annual EMS Trend Report. During the conversation, the hosts emphasize the importance of field providers participating in the survey.  "This is their opportunity to have their voices heard," Cebollero said. Grayson echoed his sentiment. "This is a chance to take stewardship of our profession," he said.  The pair also discuss Grayson's recent article, "Whatever will we do without the skill sheets?" regarding NREMT's plan to sunset skill sheets for paramedic certification, and what other changes are needed in EMS edcuation. Cebollero and Grayson also discuss a recent news item from Lexington, Kentucky, that found a nearly 7% decrease in call volume due to the area's robust community paramedicine program. 

The International Gifted Consortium (IGC), Research Center for The Highly-Profoundly Gifted in collaboration with The University of Antwerp invite parents to participate in The 1st Study of Highly-Profoundly Gifted Children and Emotional, Intellectual, Imaginational, Psychomotor, and Sensual Overexcitabilities. --- Send in a voice message: https://anchor.fm/intl-gifted-consortium/message

Almost everyone recognizes Bloom's taxonomy and the use of the Cognitive Domain. But there are two other domains, the Affective and Psychomotor, which are extremely important in implementing in our lessons, courses, and assessments. Together, all three domains make up the head, the heart, and the hands of education. Listen in to review these two domains and their importance in learning development as a curriculum developer and teacher.

In today's episode, we continue our overexcitabilities series for parents of gifted children. Today, the focus is psychomotor excitabilities and our gifted children. Colleen discusses what these overexcitabilities are and how to help your child. 

Raspberries, ellagic acid reveal benefits in two studies Oregon State University, October 1, 2021.    Articles that appeared recently in the Journal of Berry Research report that raspberries and compounds present in the fruit could help support healthy body mass and motor function, including balance, coordination and strength.   In one study, Neil Shay and colleagues at Oregon State University fed mice a high fat, high sugar diet plus one of the following: raspberry juice concentrate, raspberry puree concentrate, raspberry fruit powder, raspberry seed extract, ellagic acid (a polyphenol that occurs in a relatively high amount in raspberries), raspberry ketone, or a combination of raspberry ketone and ellagic acid. Additional groups of animals received a high fat, high sugar diet alone or a low fat diet.   While mice that received the high fat and sugar diet alone experienced a significant increase in body mass, the addition of raspberry juice concentrate, raspberry puree concentrate or ellagic acid plus raspberry ketone helped prevent this effect. Of note, mice that received raspberry juice concentrate experienced gains similar to those of animals given a low fat diet. "We hope that the findings from this study can help guide the design of future clinical trials," Dr Shay stated.   In another study, Barbara Shukitt-Hale, PhD, and her associates at Tufts University's Human Nutrition Research Center on Aging gave 19 month old rats a control diet or a diet enhanced with raspberry extract for 11 weeks. Psychomotor behavior was assessed during week 7 and cognitive testing was conducted during weeks 9-10.   Animals that received raspberry performed better on psychomotor coordination and balance, and had better muscle tone, strength and stamina than those that received a control diet. "These results may have important implications for healthy aging," stated Dr Shukitt-Hale. "While further research in humans is necessary, animal model studies are helpful in identifying deficits associated with normal aging."       Massage doesn't just make muscles feel better, it makes them heal faster and stronger Harvard University, October 6, 2021 Massage has been used to treat sore, injured muscles for more than 3,000 years, and today many athletes swear by massage guns to rehabilitate their bodies. But other than making people feel good, do these "mechanotherapies" actually improve healing after severe injury? According to a new study from researchers at Harvard's Wyss Institute for Biologically Inspired Engineering and John A. Paulson School of Engineering and Applied Sciences (SEAS), the answer is "yes." Using a custom-designed robotic system to deliver consistent and tunable compressive forces to mice's leg muscles, the team found that this mechanical loading (ML) rapidly clears immune cells called neutrophils out of severely injured muscle tissue. This process also removed inflammatory cytokinesreleased by neutrophils from the muscles, enhancing the process of muscle fiber regeneration. The research is published in Science Translational Medicine. "Lots of people have been trying to study the beneficial effects of massage and other mechanotherapies on the body, but up to this point it hadn't been done in a systematic, reproducible way. Our work shows a very clear connection between mechanical stimulation and immune function. This has promise for regenerating a wide variety of tissues including bone, tendon, hair, and skin, and can also be used in patients with diseases that prevent the use of drug-based interventions," said first author Bo Ri Seo, Ph.D., who is a Postdoctoral Fellow in the lab of Core Faculty member Dave Mooney, Ph.D. at the Wyss Institute and SEAS. Seo and her coauthors started exploring the effects of mechanotherapy on injured tissues in mice several years ago, and found that it doubled the rate of muscle regeneration and reduced tissue scarring over the course of two weeks. Excited by the idea that mechanical stimulation alone can foster regeneration and enhance muscle function, the team decided to probe more deeply into exactly how that process worked in the body, and to figure out what parameters would maximize healing. They teamed up with soft robotics experts in the Harvard Biodesign Lab, led by Wyss Associate Faculty member Conor Walsh, Ph.D., to create a small device that used sensors and actuators to monitor and control the force applied to the limb of a mouse. " The device we created allows us to precisely control parameters like the amount and frequency of force applied, enabling a much more systematic approach to understanding tissue healing than would be possible with a manual approach," said co-second author Christopher Payne, Ph.D., a former Postdoctoral Fellow at the Wyss Institute and the Harvard Biodesign Lab who is now a Robotics Engineer at Viam, Inc.  Once the device was ready, the team experimented with applying force to mice's leg muscles via a soft silicone tip and used ultrasound to get a look at what happened to the tissue in response. They observed that the muscles experienced a strain of between 10-40%, confirming that the tissues were experiencing mechanical force. They also used those ultrasound imaging data to develop and validate a computational model that could predict the amount of tissue strain under different loading forces. They then applied consistent, repeated force to injured muscles for 14 days. While both treated and untreated muscles displayed a reduction in the amount of damaged muscle fibers, the reduction was more pronounced and the cross-sectional area of the fibers was larger in the treated muscle, indicating that treatment had led to greater repair and strength recovery. The greater the force applied during treatment, the stronger the injured muscles became, confirming that mechanotherapy improves muscle recovery after injury. But how? Evicting neutrophils to enhance regeneration To answer that question, the scientists performed a detailed biological assessment, analyzing a wide range of inflammation-related factors called cytokines and chemokines in untreated vs. treated muscles. A subset of cytokines was dramatically lower in treated muscles after three days of mechanotherapy, and these cytokines are associated with the movement of immune cells called neutrophils, which play many roles in the inflammation process. Treated muscles also had fewer neutrophils in their tissue than untreated muscles, suggesting that the reduction in cytokines that attract them had caused the decrease in neutrophil infiltration. The team had a hunch that the force applied to the muscle by the mechanotherapy effectively squeezed the neutrophils and cytokines out of the injured tissue. They confirmed this theory by injecting fluorescent molecules into the muscles and observing that the movement of the molecules was more significant with force application, supporting the idea that it helped to flush out the muscle tissue. To pick apart what effect the neutrophils and their associated cytokines have on regenerating muscle fibers, the scientists performed in vitro studies in which they grew muscle progenitor cells (MPCs) in a medium in which neutrophils had previously been grown. They found that the number of MPCs increased, but the rate at which they differentiated (developed into other cell types) decreased, suggesting that neutrophil-secreted factors stimulate the growth of muscle cells, but the prolonged presence of those factors impairs the production of new muscle fibers. "Neutrophils are known to kill and clear out pathogens and damaged tissue, but in this study we identified their direct impacts on muscle progenitor cell behaviors," said co-second author Stephanie McNamara, a former Post-Graduate Fellow at the Wyss Institute who is now an M.D.-Ph.D. student at Harvard Medical School (HMS). "While the inflammatory response is important for regeneration in the initial stages of healing, it is equally important that inflammation is quickly resolved to enable the regenerative processes to run its full course." Seo and her colleagues then turned back to their in vivo model and analyzed the types of muscle fibers in the treated vs. untreated mice 14 days after injury. They found that type IIX fibers were prevalent in healthy muscle and treated muscle, but untreated injured muscle contained smaller numbers of type IIX fibers and increased numbers of type IIA fibers. This difference explained the enlarged fiber size and greater force production of treated muscles, as IIX fibers produce more force than IIA fibers. Finally, the team homed in on the optimal amount of time for neutrophil presence in injured muscle by depleting neutrophils in the mice on the third day after injury. The treated mice's muscles showed larger fiber size and greater strength recovery than those in untreated mice, confirming that while neutrophils are necessary in the earliest stages of injury recovery, getting them out of the injury site early leads to improved muscle regeneration. "These findings are remarkable because they indicate that we can influence the function of the body's immune system in a drug-free, non-invasive way," said Walsh, who is also the Paul A. Maeder Professor of Engineering and Applied Science at SEAS and whose group is experienced in developing wearable technology for diagnosing and treating disease. "This provides great motivation for the development of external, mechanical interventions to help accelerate and improve muscle and tissue healing that have the potential to be rapidly translated to the clinic." The team is continuing to investigate this line of research with multiple projects in the lab. They plan to validate this mechanotherpeutic approach in larger animals, with the goal of being able to test its efficacy on humans. They also hope to test it on different types of injuries, age-related muscle loss, and muscle performance enhancement. "The fields of mechanotherapy and immunotherapy rarely interact with each other, but this work is a testament to how crucial it is to consider both physical and biological elements when studying and working to improve human health," said Mooney, who is the corresponding author of the paper and the Robert P. Pinkas Family Professor of Bioengineering at SEAS. "The idea that mechanics influence cell and tissue function was ridiculed until the last few decades, and while scientists have made great strides in establishing acceptance of this fact, we still know very little about how that process actually works at the organ level. This research has revealed a previously unknown type of interplay between mechanobiology and immunology that is critical for muscle tissue healing, in addition to describing a new form of mechanotherapy that potentially could be as potent as chemical or gene therapies, but much simpler and less invasive," said Wyss Founding Director Don Ingber, M.D., Ph.D., who is also the Judah Folkman Professor of Vascular Biology at (HMS) and the Vascular Biology Program at Boston Children's Hospital, as well as Professor of Bioengineering at SEAS.   Vitamin E could help protect older men from pneumonia University of Helsinki (Finland), October 7 2021.    An article that appeared in Clinical Interventions in Aging reported a protective role for vitamin E against pneumonia in older men.   For the current investigation, Dr Harri Hemilä of the University of Helsinki, Finland analyzed data from the Alpha-Tocopherol Beta-Carotene (ATBC) Cancer Prevention Study conducted in Finland. The trial included 29,133 men between the ages of 50 to 69 years who smoked at least five cigarettes daily upon enrollment. Participants received alpha tocopherol (vitamin E), beta carotene, both supplements, or a placebo for five to eight years.   The current study was limited to 7,469 ATBC participants who started smoking at age 21 or older. Among this group, supplementation with vitamin E was associated with a 35% lower risk of developing pneumonia in comparison with those who did not receive the vitamin.  Light smokers who engaged in leisure time exercise had a 69% lower risk compared with unsupplemented members of this subgroup. The risk in this subgroup of developing pneumonia by age 74 was 12.9%.   Among the one-third of the current study's population who quit smoking for a median period of two years, there was a 72% lower risk of pneumonia in association with vitamin E supplementation. In this group, exercisers who received vitamin E experienced an 81% lower pneumonia risk.   Dr Hemilä observed that the benefit for vitamin E in this study was strongest for older subjects—a group at higher risk of pneumonia.   "The current analysis of individual-level data suggests that trials on vitamin E and pneumonia on nonsmoking elderly males are warranted," he concluded.       Toxic fatty acids to blame for brain cell death after injury New York University, October 7, 2021 Cells that normally nourish healthy brain cells called neurons release toxic fatty acids after neurons are damaged, a new study in rodents shows. This phenomenon is likely the driving factor behind most, if not all, diseases that affect brain function, as well as the natural breakdown of brain cells seen in aging, researchers say. Previous research has pointed to astrocytes—a star-shaped glial cell of the central nervous system—as the culprits behind cell death seen in Parkinson's disease and dementia, among other neurodegenerative diseases. While many experts believed that these cells released a neuron-killing molecule to "clear away" damaged brain cells, the identity of this toxin has until now remained a mystery. Led by researchers at NYU Grossman School of Medicine, the new investigation provides what they say is the first evidence that tissue damage prompts astrocytes to produce two kinds of fats, long-chain saturated free fatty acids and phosphatidylcholines. These fats then trigger cell death in damaged neurons, the electrically active cells that send messages throughout nerve tissue. Publishing Oct. 6 in the journal Nature, the study also showed that when researchers blocked fatty acid formation in mice, 75 percent of neurons survived compared with 10 percent when the fatty acids were allowed to form. The researchers' earlier work showed that brain cells continued to function when shielded from astrocyte attacks.  "Our findings show that the toxic fatty acids produced by astrocytes play a critical role in brain cell death and provide a promising new target for treating, and perhaps even preventing, many neurodegenerative diseases," says study co-senior author Shane Liddelow, Ph.D. Liddelow, an assistant professor in the Department of Neuroscience and Physiology at NYU Langone Health, adds that targeting these fats instead of the cells that produce them may be a safer approach to treating neurodegenerative diseasesbecause astrocytes feed nerve cells and clear away their waste. Stopping them from working altogether could interfere with healthy brain function. Although it remains unclear why astrocytes produce these toxins, it is possible they evolved to destroy damaged cells before they can harm their neighbors, says Liddelow. He notes that while healthy cells are not harmed by the toxins, neurons become susceptible to the damaging effects when they are injured, mutated, or infected by prions, the contagious, misfolded proteins that play a major role in mad cow disease and similar illnesses. Perhaps in chronic diseases like dementia, this otherwise helpful process goes off track and becomes a problem, the study authors say. For the investigation, researchers analyzed the molecules released by astrocytes collected from rodents. They also genetically engineered some groups of mice to prevent the normal production of the toxic fats and looked to see whether neuron death occurred after an acute injury. "Our results provide what is likely the most detailed molecular map to date of how tissue damage leads to brain cell death, enabling researchers to better understand why neurons die in all kinds of diseases," says Liddelow, also an assistant professor in the Department of Ophthalmology at NYU Langone. Liddelow cautions that while the findings are promising, the genetic techniques used to block the enzyme that produces toxic fatty acids in mice are not ready for use in humans. As a result, the researchers next plan is to explore safe and effective ways to interfere with the release of the toxins in human patients. Liddelow and his colleagues had previously shown these neurotoxic astrocytes in the brains of patients with Parkinson's, Huntington's disease, and multiple sclerosis, among other diseases.   Clinical trial for nicotinamide riboside: Vitamin safely boosts levels of important cell metabolite linked to multiple health benefits University of Iowa Health Care, October 3, 2021   In the first controlled clinical trial of nicotinamide riboside (NR), a newly discovered form of Vitamin B3, researchers have shown that the compound is safe for humans and increases levels of a cell metabolite that is critical for cellular energy production and protection against stress and DNA damage.   Studies in mice have shown that boosting the levels of this cell metabolite -- known as NAD+ -- can produce multiple health benefits, including resistance to weight gain, improved control of blood sugar and cholesterol, reduced nerve damage, and longer lifespan. Levels of NAD+ diminish with age, and it has been suggested that loss of this metabolite may play a role in age-related health decline.   These findings in animal studies have spurred people to take commercially available NR supplements designed to boost NAD+. However, these over-the-counter supplements have not undergone clinical trials to see if they work in people.   The new research, reported in the journal Nature Communications, was led by Charles Brenner, PhD, professor and Roy J. Carver Chair of Biochemistry at the University of Iowa Carver College of Medicine in collaboration with colleagues at Queens University Belfast and ChromaDex Corp. (NASDAQ: CDXC), which supplied the NR used in the trial. Brenner is a consultant for ChromaDex. He also is co-founder and Chief Scientific Adviser of ProHealthspan, which sells NR supplements under the trade name Tru NIAGEN®.   The human trial involved six men and six women, all healthy. Each participant received single oral doses of 100 mg, 300 mg, or 1,000 mg of NR in a different sequence with a seven-day gap between doses. After each dose, blood and urine samples were collected and analyzed by Brenner's lab to measure various NAD+ metabolites in a process called metabolomics. The trial showed that the NR vitamin increased NAD+ metabolism by amounts directly related to the dose, and there were no serious side effects with any of the doses.   "This trial shows that oral NR safely boosts human NAD+ metabolism," Brenner says. "We are excited because everything we are learning from animal systems indicates that the effectiveness of NR depends on preserving and/or boosting NAD+ and related compounds in the face of metabolic stresses. Because the levels of supplementation in mice that produce beneficial effects are achievable in people, it appears than health benefits of NR will be translatable to humans safely."   The next step will be to study the effect of longer duration NR supplementation on NAD+ metabolism in healthy adults, but Brenner also has plans to test the effects of NR in people with diseases and health conditions, including elevated cholesterol, obesity and diabetes, and people at risk for chemotherapeutic peripheral neuropathy.   Prior to the formal clinical trial, Brenner conducted a pilot human study -- on himself. In 2004, he had discovered that NR is a natural product found in milk and that there is pathway to convert NR to NAD+ in people. More than a decade of research on NR metabolic pathways and health effects in mice and rats had convinced him that NR supplementation had real promise to improve human health and wellness. After consulting with UI's institutional review board, he conducted an experiment in which he took 1 gram of NR once a day for seven days, and his team analyzed blood and urine samples using mass spectrometry. The experiment showed that Brenner's blood NAD+ increased by about 2.7 times. In addition, though he reported immediate sensitivity to flushing with the related compound niacin, he did not experience any side effects taking NR.   The biggest surprise from his metabolomic analysis was an increase in a metabolite called NAAD, which was multiplied by 45 times, from trace levels to amounts in the micromolar range that were easily detectable.   "While this was unexpected, I thought it might be useful," Brenner says. "NAD+ is an abundant metabolite and it is sometimes hard to see the needle move on levels of abundant metabolites. But when you can look at a low-abundance metabolite that goes from undetectable to easily detectable, there is a great signal to noise ratio, meaning that NAAD levels could be a useful biomarker for tracking increases in NAD+ in human trials."   Brenner notes this was a case of bidirectional translational science; having learned something from the initial human experiment, his team was able to return to laboratory mice to explore the unexpected NAAD finding in more detail.   Brenner's mouse study showed that NAAD is formed from NR and confirmed that NAAD levels are a strong biomarker for increased NAD+ metabolism. The experiments also revealed more detail about NAD+ metabolic pathways.   In particular, the researchers compared the ability of all three NAD+ precursor vitamins -- NR, niacin, and nicotinamide -- to boost NAD+ metabolism and stimulate the activity of certain enzymes, which have been linked to longevity and healthbenefits. The study showed for the first time that oral NR is superior to nicotinamide, which is better than niacin in terms of the total amount of NAD+ produced at an equivalent dose. NR was also the best of the three in stimulating the activity of sirtuin enzymes. However, in this case, NR was the best at stimulating sirtuin-like activities, followed by niacin, followed by nicotinamide.   The information from the mouse study subsequently helped Brenner's team design the formal clinical trial. In addition to showing that NR boosts NAD+ in humans without adverse effects, the trial confirmed that NAAD is a highly sensitive biomarker of NAD+ supplementation in people.   "Now that we have demonstrated safety in this small clinical trial, we are in a position to find out if the health benefits that we have seen in animals can be reproduced in people," says Brenner, who also is co-director of the Obesity Research and Education Initiative, professor of internal medicine, and a member of the Fraternal Order of Eagles Diabetes Research Center at the UI.   Protecting the ozone layer is delivering vast health benefits Montreal Protocol will spare Americans from 443 million skin cancer cases National Center for Atmospheric Research, October 7, 2021 An international agreement to protect the ozone layer is expected to prevent 443 million cases of skin cancer and 63 million cataract cases for people born in the United States through the end of this century, according to new research. The research team, by scientists at the National Center for Atmospheric Research (NCAR), ICF Consulting, and U.S. Environmental Protection Agency (EPA), focused on the far-reaching impacts of a landmark 1987 treaty known as the Montreal Protocol and later amendments that substantially strengthened it. The agreement phased out the use of chemicals such as chlorofluorocarbons (CFCs) that destroy ozone in the stratosphere. Stratospheric ozone shields the planet from harmful levels of the Sun's ultraviolet (UV) radiation, protecting life on Earth. To measure the long-term effects of the Montreal Protocol, the scientists developed a computer modeling approach that enabled them to look to both the past and the future by simulating the treaty's impact on Americans born between 1890 and 2100. The modeling revealed the treaty's effect on stratospheric ozone, the associated reductions in ultraviolet radiation, and the resulting health benefits.  In addition to the number of skin cancer and cataract cases that were avoided, the study also showed that the treaty, as most recently amended, will prevent approximately 2.3 million skin cancer deaths in the U.S. “It's very encouraging,” said NCAR scientist Julia Lee-Taylor, a co-author of the study. “It shows that, given the will, the nations of the world can come together to solve global environmental problems.” The study, funded by the EPA, was published in ACS Earth and Space Chemistry. NCAR is sponsored by the National Science Foundation. Mounting concerns over the ozone layer Scientists in the 1970s began highlighting the threat to the ozone layer when they found that CFCs, used as refrigerants and in other applications, release chlorine atoms in the stratosphere that set off chemical reactions that destroy ozone. Concerns mounted the following decade with the discovery of an Antarctic ozone hole. The loss of stratospheric ozone would be catastrophic, as high levels of UV radiation have been linked to certain types of skin cancer, cataracts, and immunological disorders. The ozone layer also protects terrestrial and aquatic ecosystems, as well as agriculture. Policy makers responded to the threat with the 1987 Montreal Protocol on Substances that Deplete the Ozone Layer, in which nations agreed to curtail the use of certain ozone-destroying substances. Subsequent amendments strengthened the treaty by expanding the list of ozone-destroying substances (such as halons and hydrochlorofluorocarbons, or HCFCs) and accelerating the timeline for phasing out their use. The amendments were based on Input from the scientific community, including a number of NCAR scientists, that were summarized in quadrennial Ozone Assessment reports. To quantify the impacts of the treaty, the research team built a model known as the Atmospheric and Health Effects Framework. This model, which draws on various data sources about ozone, public health, and population demographics, consists of five computational steps. These simulate past and future emissions of ozone-destroying substances, the impacts of those substances on stratospheric ozone, the resulting changes in ground-level UV radiation, the U.S. population's exposure to UV radiation, and the incidence and mortality of health effects resulting from the exposure. The results showed UV radiation levels returning to 1980 levels by the mid-2040s under the amended treaty. In contrast, UV levels would have continued to increase throughout this century if the treaty had not been amended, and they would have soared far higher without any treaty at all.  Even with the amendments, the simulations show excess cases of cataracts and various types of skin cancer beginning to occur with the onset of ozone depletion and peaking decades later as the population exposed to the highest UV levels ages. Those born between 1900 and 2040 experience heightened cases of skin cancer and cataracts, with the worst health outcomes affecting those born between about 1950 and 2000. However, the health impacts would have been far more severe without the treaty, with cases of skin cancer and cataracts rising at an increasingly rapid rate through the century.  “We peeled away from disaster,” Lee-Taylor said. “What is eye popping is what would have happened by the end of this century if not for the Montreal Protocol. By 2080, the amount of UV has tripled. After that, our calculations for the health impacts start to break down because we're getting so far into conditions that have never been seen before.” The research team also found that more than half the treaty's health benefits could be traced to the later amendments rather than the original 1987 Montreal Protocol. Overall, the treaty prevented more than 99% of potential health impacts that would have otherwise occurred from ozone destruction. This showed the importance of the treaty's flexibility in adjusting to evolving scientific knowledge, the authors said. The researchers focused on the U.S. because of ready access to health data and population projections. Lee-Taylor said that the specific health outcomes in other countries may vary, but the overall trends would be similar. “The treaty had broad global benefits,” she said.     What is Boron? The trace mineral boron provides profound anti-cancer effects, in addition to maintaining stronger bones. Life Extension, September 2021 Boron is a trace mineral found in the earth's crust and in water. Its importance in human health has been underestimated. Boron has been shown to have actions against specific types of malignancies, such as: Cervical cancer: The country Turkey has an extremely low incidence of cervical cancer, and scientists partially attribute this to its boron-rich soil.1 When comparing women who live in boron-rich regions versus boron-poor regions of Turkey, not a single woman living in the boron-rich regions had any indication of cervical cancer.2(The mean dietary intake of boron for women in this group was 8.41 mg/day.)  Boron interferes with the life cycle of the human papillomavirus (HPV), which is a contributing factor in approximately 95% of all cervical cancers.1  Considering that HPV viruses are increasingly implicated in head and neck cancers,3,4 supplementation with this ultra-low-cost mineral could have significant benefits in protecting against this malignancy that is increasing in prevalence. Lung cancer: A study conducted at the University of Texas MD Anderson Cancer Center between 1995 and 2005 found that increased boron intake was associated with a lower risk of lung cancer in postmenopausal women who were taking hormone replacement therapy. Prostate cancer: Studies point to boron's ability to inhibit the growth and spread of prostate cancer cells.  In one study, when mice were exposed to boric acid, their tumors shrank by as much as 38%.6 One analysis found that increased dietary boron intake was associated with a decreased risk of prostate cancer.7 Several human and animal studies have confirmed the important connection between boron and bone health. Boron prevents calcium loss,8 while also alleviating the bone problems associated with magnesium and vitamin D deficiency.9 All of these nutrients help maintain bone density. A study in female rats revealed the harmful effects a deficiency in boron has on bones, including:10 Decreased bone volume fraction, a measure of bone strength, Decreased thickness of the bone's spongy inner layer, and Decreased maximum force needed to break the femur. And in a study of post-menopausal women, supplementation with3 mg of boron per day prevented calcium loss and bone demineralization by reducing urinary excretion of both calcium and magnesium.8 In addition to its bone and anti-cancer benefits, there are nine additional reasons boron is an important trace mineral vital for health and longevity. It has been shown to:1 Greatly improve wound healing, Beneficially impact the body's use of estrogen, testosterone, and vitamin D, Boost magnesium absorption, Reduce levels of inflammatory biomarkers, such as high-sensitivity C-reactive protein (hs-CRP) and tumor necrosis factor α (TNF-α), Raise levels of antioxidant enzymes, such as superoxide dismutase (SOD), catalase, and glutathione peroxidase, Protect against pesticide-induced oxidative stress and heavy-metal toxicity, Improve the brain's electrical activity, which may explain its benefits for cognitive performance, and short-term memory in the elderly, Influence the formation and activity of key biomolecules, such as S-adenosyl methionine (SAM-e) and nicotinamide adenine dinucleotide (NAD+), and Potentially help ameliorate the adverse effects of traditional chemotherapeutic agents. Because the amount of boron varies in the soil, based on geographical location, obtaining enough boron through diet alone can be difficult. Supplementing with low-cost boron is an effective way to maintain adequate levels of this overlooked micronutrient.

Most educators can recognize Dr. Benjamin Bloom's Taxonomy as an important part of their field. But most may not know that there are varieties of this Taxonomy that include an old, a revised, a Cognitive, an Affective, and a Psychomotor version, only one of which was actually created by Bloom.

Psycho-Motor Skills Discussion. Interview with Karl Shreeves. Karl is the Director of Training Development at PADI. We discuss psycho-motor skills and how that relates to teaching and performing scuba diving. Psychomotor skills refers to a wide range of actions involving physical movement related to conscious cognitive processing.   https://en.wikipedia.org/wiki/Psychomotor_learning

  Click here for a macine generated transcript   Surviving a stroke is not the end of a medical issue. It's the start of a new journey, with new challenges. Major depression is often one of those challenges. It interferes with recovery, rehab, adjusting to a new life, and maintaining relationships. In short, it's big problem. And it's pretty common. According to new research by Dr. Laura Stein from the Icahn School of medicine, depression after stroke is twice as likely to occur as depression after heart attack. There's something unique about stroke that leads to depression. On top of that, if a person lived with Generalized Anxiety Disorder before stroke, they are 1.7 x as likely to experience Major Depression after stroke. In this conversation, Dr. Laura Stein talk about her research and what survivors, caregivers, and medical practitioners need to know. Bio Laura K. Stein, MD, MPH is an Assistant Professor of Neurology at the Icahn School of Medicine at Mount Sinai and attending physician at the Mount Sinai and Mount Sinai Queens Stroke Centers. She is board certified in Neurology and Vascular Neurology by the American Board of Psychiatry and Neurology. Dr. Stein received a BA from Amherst College and her MD and MPH from the Icahn School of Medicine at Mount Sinai. She completed her internship, neurology residency, and vascular neurology fellowship at Mount Sinai.  Dr. Stein's interests are in stroke clinical care, stroke outcomes research, and medical education. She is the Neurology Residency Associate Program Director and precepts first year medical students in the Art and Science of Medicine preclinical skills course. Dr. Stein received the Department of Neurology Resident Class of 2020 Award of Appreciation for Dedication & Commitment Towards Excellence in Mentorship and 2020 Institute for Medical Education Excellence in Teaching Award.  The Research Dr. Stein's research was part of the American Stroke Association's virtual International Stroke Conference. You can read the paper here. You can read the article where I first encountered her work here. The approach of looking at depression after heart attack vs looking at depression after stroke is an interesting one. It's a nice way of controlling for other potential causes. The research looked at Medicare recipients, Medicare is a US government health insurance program, generally for folks 65 and older. It lets researchers draw from a rich collection of data. There are two significant tradeoffs, of course. First, it's restricted to the US so there's no international representation in the study. Obviously, depending on the research question in particular, that may or may not be an issue. Second, it's restricted to folks 65 and older. Can we say that the results of this study apply to younger stroke survivors? Maybe? Probably? We don't know. And this study was not designed to answer that question. I'd say it does get us closer, though, and it opens up an opportunity to do further research that asks different but related questions. That's what good studies do. The answer specific question with solid evidence and clear analysis. And the result can introduce new questions for researchers to explore in different studies. In the meantime, it's likely safe to say, that regardless of age, stroke survivors are probably more likely to experience depression, and the community needs to be vigilant for the signs of depression and seek treatment accordingly. Symptoms of Major Depression Diagnosing depression involves identifying symptoms from a couple lists. To "earn" a depression diagnosis, a patient needs 5 symptoms from list 1 and all 4 from list 2. They are: List 1 (Pick 5 or more) Depressed Mood (most days) Loss of Interest or pleasure Weight loss or gain Insomnia or hypersomnia (nearly every day) Psychomotor agitation or limitation Fatigue (nearly every day) Feelings worthless or experiencing inappropriate guilt Decreased concentration (nearly everyday) Thoughts of death or suicide List 2 (All 4) Symptoms cause significant distress or social/professional impairment Symptoms not attributable to a substance or medical condition Symptoms not explained by another disorder No history of manic episodes You can read more details about the symptoms here. You can probably see one of the issues. Many of those symptoms may be directly attributed to the disabilities and brain damage of the stroke without it being Major Depression. That's why it's important to work with a medical professional to tease them apart. Symptoms of Generalized Anxiety Disorder Generalized Anxiety Disorder is about more than feeling nervous. It's more complicated and severe. The National Institute of Mental Health describes it this way: People with generalized anxiety disorder (GAD) display excessive anxiety or worry, most days for at least 6 months, about a number of things such as personal health, work, social interactions, and everyday routine life circumstances. The fear and anxiety can cause significant problems in areas of their life, such as social interactions, school, and work. Generalized anxiety disorder symptoms include: Feeling restless, wound-up, or on-edge Being easily fatigued Having difficulty concentrating; mind going blank Being irritable Having muscle tension Difficulty controlling feelings of worry Having sleep problems, such as difficulty falling or staying asleep, restlessness, or unsatisfying sleep You can read more about the condition here. Neuropsychology While most psychologists and psychiatrists can help stroke survivors and others with depression and other conditions, there is a specialty that may be even more helpful -- neuropsychology. A while back, I talked with Dr. Karen Sullivan from I Care For Your Brain. She wrote the "Interactive Stroke Recovery Guide."*  You can listen to that conversation or learn more at http://Strokecast.com/Karen A neuropsychologist has additional, specialized training to work specifically with folks who have brain injuries. They dive deep into the details of the injury to provide the best customized treatment plan, leveraging expertise that generalists don't have. They work with folks with a wide array of functionality and challenges. FLAME vs FOCUS As long as we are talking about depression, we should also talk about antidepressants a little -- specifically SSRIs and how the relate to stroke. In 2011, the FLAME study was published in the Lancet. In short, it demonstrated that stroke survivors who took Prozac (Fluoxetine) experienced stronger motor recovery. Many hospitals saw those results and began putting more stroke survivors on Prozac because, well, why not? If the patient tolerates it well, and it can help folks work better, it's probably a good idea. And if it reduces or prevent post stroke depression (or other depression) that's a solid win. I talked with Dr. Nirav Shah about this back in November of 2018. You can listen to that episode here. That's how I started on an SSRI in my stay. They originally tried Prozac with me, but I had a not great reaction to it. It apparently gave me an anxiety attack. So we quickly stopped that (thank you, Xanax, I think). We talked about it some more and tried again with a different SSRI -- Lexapro (Escitalopram) because I had tolerated it well during a tough time some years earlier. Cheap, no negative side effects, and potentially helpful is win. A month after my interview with Nirav, and a year and a half after my stroke, the FOCUS study came out, attempting to duplicate the results of the FLAME study on a bigger scale. It failed. The scientific consensus now is that SSRIs do not help with motor recovery. They do still help with depression. The hospital no longer recommends SSRIs as part of the motor recovery protocol. So what does that mean for folks like me? I asked my doctor last year if that meant I should stop taking them. We talked about it and she explained I could certainly stop if I wanted to. "But, dude -- it's 2020." And she had a valid point. (Okay, maybe I paraphrased that) And that's why Escitalopram still has a place in my pill organizer. As always, everyone's stroke and circumstances will vary, so talk to your doctor before making any changes to your medication. Hack of the Week Get a dog. A dog can be helpful after stroke, whether it's a service dog or simply a companion animal. When you have a dog, you have a responsibility to take care of it. You have to feed it, walk it, groom it, and give it cuddles. Sometimes that may be reason enough to get out of bed and get moving for the day. Links Where do we go from here? If you think there's a chance you might be experiencing depression, talk to your medical team Share this episode with someone you know with the link http://Strokecast.com/depression Subscribe to the free, monthly Strokecast email newsletter at http://Strokecast.com/news Don't get best…get better.

On this episode, Allen Oelschlaeger is joined by Randy Revling, a retired Sheriff's captain with over 30 years of experience as a law enforcement instructor. Randy developed the original instructor development course (IDC) used by the State of Wisconsin and has personally taught over 600 instructor schools for a wide range of disciplines. Unlike most trainers, Randy has documented positive results of his training methods. The discussion focuses on how to optimize “learning” — defined as creating relatively permanent changes in student behavior that results in the benefits expected from the training. Some of the core principles discussed include: • To optimize learning, the importance of soliciting emotion and making the training practical and relevant to the student • People learn by doing, not by listening to a lecture • To drive learning, the need for a stimulus that is created by practicing inside a real-life and relevant scenario • The four steps that must be completed before delivering any training • The reason why teaching should occur inside scenario practice, rather than before this practice • The application of Kirkpatrick's Four-Level Training Evaluation Model to psychomotor skill training • How to effectively engage with students who start class with a bad attitude • The potentially conflicting goals of a training class generating “learning” versus generating good evaluations

On this episode, Allen Oelschlaeger is joined by Randy Revling, a retired Sheriff’s captain with over 30 years of experience as a law enforcement instructor. Randy developed the original instructor development course (IDC) used by the State of Wisconsin and has personally taught over 600 instructor schools for a wide range of disciplines. Unlike most trainers, Randy has documented positive results of his training methods. The discussion focuses on how to optimize “learning” — defined as creating relatively permanent changes in student behavior that results in the benefits expected from the training. Some of the core principles discussed include: • To optimize learning, the importance of soliciting emotion and making the training practical and relevant to the student • People learn by doing, not by listening to a lecture • To drive learning, the need for a stimulus that is created by practicing inside a real-life and relevant scenario • The four steps that must be completed before delivering any training • The reason why teaching should occur inside scenario practice, rather than before this practice • The application of Kirkpatrick’s Four-Level Training Evaluation Model to psychomotor skill training • How to effectively engage with students who start class with a bad attitude • The potentially conflicting goals of a training class generating “learning” versus generating good evaluations

On this episode, Allen Oelschlaeger is joined by Gerard O’Dea from Dynamis (https://www.dynamis.training), a UK conflict management training company and Professor Chris Cushion from Loughborough University (https://www.lboro.ac.uk/departments/ssehs/staff/chris-cushion), a renown expert in how to teach psychomotor skills to optimize learning (conflict management skills are psychomotor skills). Gerard and Professor Cushion have been working together for the last six years to develop a training system that creates a relatively permanent change in their student’s behavior that results in the benefits Dynamis promises its customers (healthcare, education, law enforcement, residential care). The discussion focuses on the core training principles validated by empirical research over the last 40 years that produce these outcomes. Also discussed are how these principles are so rarely applied in the training of psychomotor skills. Some of the core principles discussed include: - How the goal of a training program is learning (a relatively permanent change in student’s behavior that supports the results expected from the training) rather than getting positive reviews - Training must use whole task scenarios in order to optimize learning (instead of block training of small elements of the whole task that only create the illusion of learning) - The whole task scenarios must reflect real-world situations faced by the student (instead of the skill practice being decontextualized). - Training should be structured to take advantage of the background and experience that the students bring to class

On this episode, Allen Oelschlaeger is joined by Gerard O'Dea from Dynamis (https://www.dynamis.training), a UK conflict management training company and Professor Chris Cushion from Loughborough University (https://www.lboro.ac.uk/departments/ssehs/staff/chris-cushion), a renown expert in how to teach psychomotor skills to optimize learning (conflict management skills are psychomotor skills). Gerard and Professor Cushion have been working together for the last six years to develop a training system that creates a relatively permanent change in their student's behavior that results in the benefits Dynamis promises its customers (healthcare, education, law enforcement, residential care). The discussion focuses on the core training principles validated by empirical research over the last 40 years that produce these outcomes. Also discussed are how these principles are so rarely applied in the training of psychomotor skills. Some of the core principles discussed include: - How the goal of a training program is learning (a relatively permanent change in student's behavior that supports the results expected from the training) rather than getting positive reviews - Training must use whole task scenarios in order to optimize learning (instead of block training of small elements of the whole task that only create the illusion of learning) - The whole task scenarios must reflect real-world situations faced by the student (instead of the skill practice being decontextualized). - Training should be structured to take advantage of the background and experience that the students bring to class

Terri Cheney was a successful entertainment lawyer who represented some of the biggest names in the business, like Michael Jackson and Quincy Jones. All the while, she hid her mental illness and the slash marks on her wrist from a suicide attempt. She bravely started writing about her experience living with bipolar disorder after several horrific suicide attempts, encounters with the law, broken relationships and, eventually, a stint in a mental hospital and several rounds of electroshock therapy. The writing led to her first memoir, Manic, which went on to become a bestseller and the basis of an episode of Netflix's Modern Love where Anne Hathaway did a phenomenal job capturing Terri’s mania and depression.   In Terri’s new book, Modern Madness- An Owner’s Manual, she combines clinical research with her own lived experience- stories like her manic highs accompanied by extreme drinking, driving, and dating. Her new book brilliantly bridges the gap between the one-size-fits-all approach of modern medicine and makes it relatable and valuable for everyone. Those dealing with mental health issues and their family and friends who are often left to pick up the pieces will undoubtedly appreciate her humor and her insight.   Terri lives in Los Angeles, teaches personal essay writing workshops, and keeps up her blog, The Bipolar Lens for Psychology Today.    SHOW NOTE LINKS:   Terri Cheney’s “Modern Madness”   Terri Cheney’s “Manic”   Terri Cheney’s “Dark Side of Innocence”   Terri Cheney’s Blog, “The Bipolar Lens” on Psychology Today   Fourteen Insightful Memoirs about Mental Illness and Addiction Rachel interviews her mom, Laurie Kallen Schwent    CONNECT WITH US! *Dear Family, Podcast Page *Write Now Rachel Website *Rachel's Blog @Medium *Rachel’s Twitter *Facebook *Instagram   PLEASE JOIN: *Dear Family Members, the Private Facebook Group     WAYS TO HELP THE PODCAST: *PLEASE Leave a 5-Star Review and ***Listen and Subscribe via iTunes!!!  ***Listen and Subscribe via Stitcher!!! ***Listen and Subscribe via Spotify!!!   Thank you! Your support means the world to me. Wishing you love, happiness, and good mental health always.

Vanessa Wood dialogs with Jared about the similarities of psychomotor overexcitability and ADHD through his life experiences. For the best understanding, identification and support, Giftedness and overexcitabilities must be considered first or in addition to ADHD or other co-existing behaviors and/or development. Jared, the inspiration for this podcast, is a Philosophy major at Connecticut College. Thank you to Rick Reineke and RRaud.com --- Send in a voice message: https://anchor.fm/intl-gifted-consortium/message

#58.  We squeeze a lifetime's worth of motor skill development into a single episode.  The stages of learning a movement, some wisdom from Mestre Acordeon, and how to sidestep an existential crisis. Easy viewing for the psychomotor pyramid here. Original paper here.

The psychomotor domain of Bloom’s taxonomy doesn’t get as much love as the cognitive domain. Which is interesting to me since the psychomotor domain is the area where we teach skills involving movement or manipulation of stuff which in many cases is a significant amount of the teaching domain that we are responsible for. So what’s in the psychomotor domain? And how can we use this to help our students?

In our blended DPT curriculum, we interweave psychomotor presence along with cognitive presence in the Community of Inquiry framework. In this episode, Mary Blackinton interviews Tim Miller about his experiences and recommendations for teaching classes with significant psychomotor skills in the blended environment. We explore instructional design concepts related to facilitating psychomotor skill development, as well as best practices on how to encourage practice of skills in a digital environment. Tim is in the dissertation phase for his EdD in Distance Education from University of Florida and is currently conducting research about how students perceive and learn from blended teaching strategies.

Tim Fearon, Lead Faculty of Phoenix Manual Therapy & Owner of Fearon Physical Therapy, comes onto HET Podcast to talk about development and fostering psychomotor skill and clinical reasoning in healthcare professionals and much more! Tim is celebrating 40 years as a clinician.  Tim's Message to the PT Profession Physical therapy students enter their educational pursuits seeking the skill required to treat patients. The successful attainment of the degree leads the student to a level of unconscious incompetence which patients will rapidly awaken the intellectually honest amongst them to. The rising clinician then begins to seek the path to conscious competence.  This is where the current system lets the aspiring clinician down. The self motivated  and driven student finds a way to create a unique mentoring path that works for them. This is our system for creating the future of the profession. The primary education is delivered by skillful academicians who are in turn fed by skillful researchers.  This can only supply the student with knowledge, not the skill required to apply it, question it, build from it and master the interface between that which is known and that which we are learning through the skill & artistic application of our intellect.  Until our educational process balances the scales between research, academics, and skill we will continue to produce a preponderance of mediocre clinicians and as such a mediocre professional body. The father of EBM, David Sackett warned a long time ago  “Without clinical expertise, practice risks becoming tyrannized by evidence, for even excellent external evidence may be inapplicable to, or inappropriate for an individual patient.” No one chooses to attend the performance of a violinist who has mastered the reading and writing of sheet music yet never devoted time to picking up the instrument in the mindful practice of summoning the sound of it's music from the skills required to bring all three components to life.  Tim's Email Address: tofpt@mac.com   Phoenix Manual Therapy Website: http://www.phoenixmanualtherapy.com/  Evidence in Motion Website: https://www.evidenceinmotion.com/  "Atlas Shrugged" by Ayn Rand: https://www.amazon.com/Atlas-Shrugged-Ayn-Rand/dp/0451191145  The PT Hustle Website: https://www.thepthustle.com/  Schedule an Appointment with Kyle Rice: www.passtheptboards.com    HET LITE Tool: www.pteducator.com/het      Anywhere Healthcare: https://anywhere.healthcare/ (code: HET)     Biography: Timothy Fearon received his PT degree from The Ohio State University.  He completed the graduate program in the Musculoskeletal Sequence at Northwestern University.  He completed his Doctorate in Physical Therapy at Northern Arizona University. He founded Phoenix Manual Therapy (PMT), which initially offered courses, based on the Nordic System of orthopedic manual therapy.  Phoenix Manual Therapy progressed to offering a long-term course frame for study of the Australian clinical reasoning approach to orthopedic manual therapy with integration of Norwegian techniques and therapeutic exercise.  The course has been running over the last 25 years.  In 2011 PMT began to sponsor Dr. Peter Gibbons & Dr. Phillip Tehan's spinal manipulation course work and Fearon is lead faculty for course one.  He earned his Full Fellowship status in the American Academy of Orthopedic Manual Therapy by the challenge process in 1999; he renewed that status in 2010.  Dr. Fearon is currently adjunct faculty at A. T. Still University where he teaches manual therapy of the spine and extremities at the entry level and in the residency track.  He has been a guest instructor for NAU, Arizona School of Health Sciences, Regis University, Langston University, and North Georgia College.  He is lead faculty for Evidence in Motion manual therapy courses, the Clinical Decision Making Class and Fellowship Virtual Rounds. He has taught manual therapy courses over the last 20 years, spoken at numerous meetings for the Arizona Physical Therapy Association, and for the APTA. Dr. Fearon currently practices in an outpatient private practice specializing in orthopedic manual therapy and spinal rehabilitation where he has been for the last 28 years and has achieved 40 years in clinical practice.

According to a March 30, 2017 press release by the WHO, depression is the leading cause of ill health and disability worldwide with more than 300 million people living with depression. This is an increase of more than 18% between 2005 and 2015. Trauma, grief, financial troubles, unemployment, SAD, smoking, thyroid disease, poor sleep habits, Facebook/Instagram overload, the end of a TV series or movie, urban environments, too many choices, lack of omega-3 fatty acids, BCP, anxiety and insomnia drugs, including Valium and Xanax; Lopressor, for high blood pressure; cholesterol-lowering drugs including Lipitor; and Premarin for menopausal symptoms are all cited as possible causes of depression. Wow. Fortunately, there is such a simple, elegant solution for many who suffer from depression! A breakthrough nutritional study conducted at the Larner College of Medicine at the University of Vermont and published in PLoS ONE has found that just 248mg of magnesium per day leads to an astounding reversal of depression symptoms in study subjects. “New clinical research results show magnesium is effective at addressing symptoms and is safer and easier on the wallet than prescription therapies,” reports Science Daily, echoing information that Dr. Carolyn Dean has published in her landmark thesis on magnesium deficiency, The Magnesium Miracle, published in 2004 and recently updated [2017 Edition]. Today, even mainstream science is now confirming that magnesium is a safe, affordable and effective treatment for depression. As Dr. Dean has written in her many books, blogs and seminars, stress, hypothyroidism, insomnia, anxiety, high blood pressure, high cholesterol, and menopausal symptoms can all be related to magnesium deficiency. What if magnesium deficiency is the underlying cause of what eventually develops into depression? Let’s look at the symptoms of depression and how they relate to magnesium deficiency: * Seep disturbance – definitely a symptom of magnesium deficiency * Fatigue or loss of energy – definitely symptoms of magnesium deficiency * Psychomotor agitation or retardation – definitely symptoms of magnesium deficiency * Diminished ability to think or concentrate – definitely symptoms of magnesium deficiency When you fail to treat the above symptoms with magnesium, then the following symptoms can develop: * Diminished interest or loss of pleasure in most activities * Depressed mood * Appetite disturbance * Feelings of worthlessness * Recurrent thoughts of death or suicide Around 2008 several studies reported that antidepressant medication might only work 40% of the time, which is little better than placebo. Even so, allopathic medicine continues to tell us that antidepressant medication, with or without psychotherapy, is the best treatment for depression. But from our vantage point, for or mild to moderate depression Dr. Dean recommends ReMag Magnesium Solution. Just 1 teaspoon will give you 248 mg highly absorbed picometer elemental magnesium recommended within the parameters of the clinical study. Dr. Dean also urges you to use ReMag in appropriate doses for stress, insomnia, fatigue, agitation, and poor concentration and not succumb to the temptation of quick-fix drugs for these magnesium deficiency problems.

What characterizes the brain activity of an elite performer? How does brain activity change during the process of skill acquisition? Do cultural environments influence motor skills? Are there “motor styles” common to members of a given cultural group?   Articles: Brain Dynamics and Motor Behavior: A Case for Efficiency and Refinement for Superior Performance Action, Movement, and Culture: Does Culture Shape Movement?   More information: http://perceptionaction.com/ My Research Gate Page (pdfs of my articles) My ASU Web page Podcast Facebook page (videos, pics, etc)   Subscribe in iOS/Apple Subscribe in Anroid/Google   Support the podcast and receive bonus content   Credits: The Flamin' Groovies - Shake Some Action Mark Lanegan - Saint Louis Elegy via freemusicarchive.org and jamendo.com

According to a March 30, 2017 press release by the WHO, depression is the leading cause of ill health and disability worldwide with more than 300 million people living with depression. This is an increase of more than 18% between 2005 and 2015. Trauma, grief, financial troubles, unemployment, SAD, smoking, thyroid disease, poor sleep habits, Facebook/Instagram overload, the end of a TV series or movie, urban environments, too many choices, lack of omega-3 fatty acids, BCP, anxiety and insomnia drugs, including Valium and Xanax; Lopressor, for high blood pressure; cholesterol-lowering drugs including Lipitor; and Premarin for menopausal symptoms are all cited as possible causes of depression. Wow. Fortunately, there is such a simple, elegant solution for many who suffer from depression! A breakthrough nutritional study conducted at the Larner College of Medicine at the University of Vermont and published in PLoS ONE has found that just 248mg of magnesium per day leads to an astounding reversal of depression symptoms in study subjects. “New clinical research results show magnesium is effective at addressing symptoms and is safer and easier on the wallet than prescription therapies,” reports Science Daily, echoing information that Dr. Carolyn Dean has published in her landmark thesis on magnesium deficiency, The Magnesium Miracle, published in 2004 and recently updated [2017 Edition]. Today, even mainstream science is now confirming that magnesium is a safe, affordable and effective treatment for depression. As Dr. Dean has written in her many books, blogs and seminars, stress, hypothyroidism, insomnia, anxiety, high blood pressure, high cholesterol, and menopausal symptoms can all be related to magnesium deficiency. What if magnesium deficiency is the underlying cause of what eventually develops into depression? Let’s look at the symptoms of depression and how they relate to magnesium deficiency: * Seep disturbance – definitely a symptom of magnesium deficiency * Fatigue or loss of energy – definitely symptoms of magnesium deficiency * Psychomotor agitation or retardation – definitely symptoms of magnesium deficiency * Diminished ability to think or concentrate – definitely symptoms of magnesium deficiency When you fail to treat the above symptoms with magnesium, then the following symptoms can develop: * Diminished interest or loss of pleasure in most activities * Depressed mood * Appetite disturbance * Feelings of worthlessness * Recurrent thoughts of death or suicide Around 2008 several studies reported that antidepressant medication might only work 40% of the time, which is little better than placebo. Even so, allopathic medicine continues to tell us that antidepressant medication, with or without psychotherapy, is the best treatment for depression. But from our vantage point, for or mild to moderate depression Dr. Dean recommends ReMag Magnesium Solution. Just 1 teaspoon will give you 248 mg highly absorbed picometer elemental magnesium recommended within the parameters of the clinical study. Dr. Dean also urges you to use ReMag in appropriate doses for stress, insomnia, fatigue, agitation, and poor concentration and not succumb to the temptation of quick-fix drugs for these magnesium deficiency problems.

Devoid of or antagonistic to sociable instincts or practices – Alouette is a rock duo from Chicago, formed in 2015 when endless posted a “bedroom punk” demo on the diy forums, and dB, Austin, TX transplant, replied. The two formed a band, one-half raspy vocals and guitar, one-half pulsing drums, discarded matter and refuse, the buzz on a telephone, over mutual love of Fugazi, PJ Harvey and linguistics. They began to play shows around Chicago in 2016 and their début EP, “Psychomotor,” is out now on tape and Bandcamp.

In this episode we discuss depression. We look at the symptoms, possible causes, and ways to address others who may be suffering from it.  First we look at the connection between anxiety and depression and moderate to severe picky eating in children. Check out the article at:  The symptoms of depression are: http://www.cnn.com/2015/08/03/living/young-picky-eaters-more-depression-anxiety-feat/index.html 1. Depressed mood most of the day, almost every day, indicated by your own subjective report or by the report of others. This mood might be characterized by sadness, emptiness, or hopelessness. 2. Markedly diminished interest or pleasure in all or almost all activities most of the day nearly every day. 3. Significant weight loss when not dieting or weight gain. 4. Inability to sleep or oversleeping nearly every day. 5. Psychomotor agitation or retardation nearly every day. 6. Fatigue or loss of energy nearly every day. 7. Feelings of worthlessness or excessive or inappropriate guilt (which may be delusional) nearly every day. 8. Diminished ability to think or concentrate, or indecisiveness, nearly every day. 9. Recurrent thoughts of death (not just fear of dying), recurrent suicidal ideation without a specific plan, or a suicide attempt or a   specific plan for committing suicide.   Find out more about the symptoms at: http://evolutioncounseling.com/major-depressive-disorder-dsm-5-criteria/   Treatment is necessary given that over 8,000,000 people in the US go untreated each year. Find more statistics at http://www.huffingtonpost.com/2015/01/20/depression-statistics_n_6480412.html.   I plan on starting a new discussion group regarding my podcast topics on Facebook. Look for the Everything is Psychological discussion group. If you'd like to participate message me, tweet me, or send me an email.    I encourage you to connect with me, so that we can dialogue about these topics further at: info@drneilstafford.com Facebook: Dr Neil Stafford Twitter: @drseab

Rob Ford's go to drugs..... Music "Stay Gold" by Annakin Slayd

Albert Pesso, co-founder with his wife, Diane Boyden-Pesso, of PBSP, Pesso Boyden System Psychomotor and President of the Psychomotor Institute, Inc. was formerly Associate Professor and Director of the Dance Division at Emerson College, Supervisor of Psychomotor Therapy at McLean Hospital in Massachusetts and Consultant in Psychiatric Research at the Boston VA Hospital.  He has […]

The influence of antipsychotic treatment on the neuropsychological and psychomotor performance of schizophrenic patients is still a subject of investigation. The present study was designed to evaluate the effects of atypical neuroleptics in comparison with a conventional dopamine antagonist neuroleptic (haloperidol) on several dimensions of psychomotor performance (visual perception, attention, reaction time, and sensorimotor performance) considered to be of relevance in evaluating driving fitness. Psychomotor performance was assessed by means of the ART 90, a computerized Act and React Test which is generally used in diagnosis of psychomotor performance. The 49 participating patients were examined at discharge following psychopathological stabilisation; 20 received haloperidol, 29 received an atypical neuroleptic. Our findings demonstrate a remarkably reduced psychomotor performance in the haloperidol-treated group of schizophrenic patients compared with patients treated with atypical neuroleptics. Only 1 (5%) subject passed all subtests without major failures and could be regarded as competent to drive. Among patients with atypical neuroleptics, 7 patients (24%) passed all test parameters without major failures. Copyright (C) 2003 S. Karger AG, Basel.