Podcasts about stays

In Hour 2 of the show, Ben Maller talks about how Austin Reaves got such a lucrative deal to stay in Los Angeles and rips the Lakers for their decision. Plus, Ben also discusses why the Spurs have faith in DeAaron Fox still, we have Maller to the Third Degree, and more!!See omnystudio.com/listener for privacy information.

And like that, 13 years in these podcast streets comes to an end. On our terms, as we see fit. All of Team SKiM is inthe building to say goodbye, celebrate what we've accomplished and of course, taste test one last Oreo. If this is your first listen, boy howdy did you pick a time to arrive. If you are a long-time listener, thank you so much for your support. Cleveland's longest running podcast, is no more, and that's okay.Team SKiM“We'll be reunited and it feels so hood Have the whole world saying "How you still so good?" Well, I do this in my slumber, Summer I ain't none of these half-assed newcomers You know how I do, Summer”Tatum | TAYREL713 | Lunchbox | LISTEN | RSS | Apple Podcast | Spotify | TuneIn | Bluesky | Amazon Music | YouTube | Email | Amazon Wish List | Merch | Patreon  PHONE l 216-264-6311

The Never Weres Tom and Tim Brown meet up to talk about the huge Giannis Antetokounmpo trade to the Heat. Then they discuss the fallout between Jaylen Brown and the Celtics before moving on to the NBA draft. Finally, they tell their dream ballpark meals.

Ben Garrett and Zach Berry of the Ole Miss Spirit/On3 are LIVE following 4-star in-state offensive lineman Antonio Berry's commitment to the Rebels.Our Sponsors:* Check out BetterHelp and use my code betterhelp.com for a great deal: https://www.betterhelp.com* Check out BetterHelp and use my code betterhelp.com for a great deal: https://www.betterhelp.com* Check out Outdoorsy and use my code TOC for a great deal: https://www.outdoorsy.com* Check out Quince and use my code quince.com/toc for a great deal: https://www.quince.com* Check out Underdog Fantasy and use my code CHAMPIONS for a great deal: https://underdogfantasy.comAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

Hour 2 of Taco Tuesday's 3 Man Front featured more good news from the recruiting trail for the Auburn Tigers, the Braves' tough night against the Padres & tons of your texts on the SF Giants Pride Night controversy. See omnystudio.com/listener for privacy information.

Kaôh F***ing Rong Rewatch Ep 7 It's merge time on Survivor: Kaôh R?ng as Rob Cesternino and co-host Chappell break down the nonstop drama and brutal twists of episode seven in this recap. The Brains vs. Beauty vs. Brawn season hits a turning point when medical emergencies wreck carefully laid plans, sending shockwaves through alliances and putting player safety front and center. When Neil is suddenly pulled from the game by the medics, the expected fireworks at Tribal Council fizzle—but the fallout is anything but predictable. Rob and Chappell dive deep into the chaos of camp as infections run wild and players trade war stories about their wounds, with Dr. Rupert stepping in and changing the course of the game. The merge feast brings celebration and strategic maneuvering, as the Beauties find themselves in the swing position and both Brains and Brawn vie for their loyalty. Aubry appears doomed after her social missteps, but Neil's medevac and his idol complicate everything, leading to a series of what-if scenarios and hard feelings. The hosts dig into the ripple effects—how Neil's exit becomes a sliding doors moment for Aubry, the questionable strategies from both sides, and the relentless pace of health scares that push production's limits. – The medevac sequence and Survivor's cavalier attitude toward infections, capped by Neil's shocking evacuation – The Beauties as a crucial swing vote, with Nick and Michele weighing their options between Brains and Brawn – Insight on Aubry's vulnerable position and why her alliance choices almost send her home – The merge feast antics, hidden idol talk, and why Debbie's recruitment strategy rubs her tribemates the wrong way – Reflections on Survivor history: past idol handoffs, scratched-out votes, and what happens when production bends medical rules Will Aubry's luck finally run out, or does Neil's sacrifice set her on an unexpected path to Survivor glory? Tune in for all the tense decisions, shifting alliances, and “what could have been” twists as Survivor: Kaôh R?ng hits its wildest turning point yet. Chapters: 0:00 Merge Episode Begins, No Merge Vote 1:00 Neil Medically Evacuated, Aubry in Trouble 3:00 Contestants Joke About Infections 5:00 Medical Triage at Immunity Challenge 7:00 Aubry Gets Antibiotics, Stays in Game 11:00 Neil's Infection, Med Evac, and Jury 13:15 Neil's Bitter Exit and Idol Legacy 16:00 Sliding Doors: Aubrey Spared, Not Voted Out 20:15 Neil Weighs Giving Idol to Aubry 22:00 The Julia Crossout Fallout 28:15 Aubry's Iconic Oregon Trail Speech 33:40 Scott, Jason, Tai Share Idol Info 36:51 Mark the Chicken and Coconut Moment 39:00 Brains Tribe Desperate, Debbie Overplays 44:59 Balls on a Disc Immunity Challenge 47:19 Aubry's POV: Neil's Emotional Exit To order Rob’s book, The Tribe and I Have Spoken, visit www.robhasabook.com Never miss a minute of RHAP's extensive Survivor coverage! LISTEN: Subscribe to the Survivor podcast feed WATCH:  Watch and subscribe to the podcast on YouTube SUPPORT:  Become a RHAP Patron for bonus content, access to Facebook and Discord groups plus more great perks!

Kaôh F***ing Rong Rewatch Ep 7 It's merge time on Survivor: Kaôh R?ng as Rob Cesternino and co-host Chappell break down the nonstop drama and brutal twists of episode seven in this recap. The Brains vs. Beauty vs. Brawn season hits a turning point when medical emergencies wreck carefully laid plans, sending shockwaves through alliances and putting player safety front and center. When Neil is suddenly pulled from the game by the medics, the expected fireworks at Tribal Council fizzle—but the fallout is anything but predictable. Rob and Chappell dive deep into the chaos of camp as infections run wild and players trade war stories about their wounds, with Dr. Rupert stepping in and changing the course of the game. The merge feast brings celebration and strategic maneuvering, as the Beauties find themselves in the swing position and both Brains and Brawn vie for their loyalty. Aubry appears doomed after her social missteps, but Neil's medevac and his idol complicate everything, leading to a series of what-if scenarios and hard feelings. The hosts dig into the ripple effects—how Neil's exit becomes a sliding doors moment for Aubry, the questionable strategies from both sides, and the relentless pace of health scares that push production's limits. – The medevac sequence and Survivor's cavalier attitude toward infections, capped by Neil's shocking evacuation – The Beauties as a crucial swing vote, with Nick and Michele weighing their options between Brains and Brawn – Insight on Aubry's vulnerable position and why her alliance choices almost send her home – The merge feast antics, hidden idol talk, and why Debbie's recruitment strategy rubs her tribemates the wrong way – Reflections on Survivor history: past idol handoffs, scratched-out votes, and what happens when production bends medical rules Will Aubry's luck finally run out, or does Neil's sacrifice set her on an unexpected path to Survivor glory? Tune in for all the tense decisions, shifting alliances, and “what could have been” twists as Survivor: Kaôh R?ng hits its wildest turning point yet. Chapters: 0:00 Merge Episode Begins, No Merge Vote 1:00 Neil Medically Evacuated, Aubry in Trouble 3:00 Contestants Joke About Infections 5:00 Medical Triage at Immunity Challenge 7:00 Aubry Gets Antibiotics, Stays in Game 11:00 Neil's Infection, Med Evac, and Jury 13:15 Neil's Bitter Exit and Idol Legacy 16:00 Sliding Doors: Aubrey Spared, Not Voted Out 20:15 Neil Weighs Giving Idol to Aubry 22:00 The Julia Crossout Fallout 28:15 Aubry's Iconic Oregon Trail Speech 33:40 Scott, Jason, Tai Share Idol Info 36:51 Mark the Chicken and Coconut Moment 39:00 Brains Tribe Desperate, Debbie Overplays 44:59 Balls on a Disc Immunity Challenge 47:19 Aubry's POV: Neil's Emotional Exit To order Rob’s book, The Tribe and I Have Spoken, visit www.robhasabook.com Never miss a minute of RHAP's extensive Survivor coverage! LISTEN: Subscribe to the Survivor podcast feed WATCH:  Watch and subscribe to the podcast on YouTube SUPPORT:  Become a RHAP Patron for bonus content, access to Facebook and Discord groups plus more great perks!

Elizabeth Ziman, who performs as Elizabeth and the Catapult, is a singer-songwriter from Brooklyn. Over the past twenty years, she and a slowly rotating cast of musical friends have released six LPs, full of songs that offer a neat, often unexpected blend of the witty and the vulnerable. Her latest release is called Responsible Friend, "about slowing down in a world that keeps accelerating. It's a commitment to friends, family, and self, at a time when everyone seems to be carrying more than they can reasonably hold" (Bandcamp). Elizabeth and the Catapult play in our studio.  Set list: 1. 50/50 2. When the Doctor Needs A Doctor 3. I Love You Still Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Wil Ravelo spent years as a Green Beret developing the kind of environmental awareness, threat assessment discipline, and rapid decision-making under pressure that most people will never need and most operators carry with them for the rest of their lives, and when he transitioned into law enforcement and eventually SWAT, he discovered that the tools were the same even when the rules of engagement were entirely different. This episode is a deep dive into how elite military training rewires the way you perceive and process a threat environment, what translates from a Special Forces deployment to a domestic hostage situation, and how Wil learned to calibrate the speed and intensity of his threat response to fit a civilian law enforcement context without dulling the instincts that kept him alive overseas. If you want to understand how the highest-performing operators in the world think about space, movement, pattern recognition, and the decision cycle, this conversation is the blueprint.

Bridging the Gap - What You See Stays WIth You by Radio Islam

This week, we break down the Dani Pereira transfer rumors to CF Montréal, Austin FC's decision to buy out Robert Taylor's contract, and the big question everyone is asking... who is actually making roster decisions right now with no Sporting Director and Jim Curtin not officially in charge yet?We also discuss Austin FC's three upcoming friendlies, the return of Terry Boss, Nico Bosch's departure, Austin FC II's incredible run under Jason Shackell, Academy prospect Oliver Hamadi, and the latest Austin Rise news regarding their new home and next match.We finished up by talking about the World Cup and how it's changing the country in big 2026.THANKS FOR THE SUPPORT! HAPPY FATHER'S DAY!

Friday, June 19, 2026 The Dominant Duo – Total Dominance Hour -Jim gets knocked out of the senior WSOP event, Gideon sits in with Al, Berry stays long today, Deion Sanders' future, NBA draft is Tuesday and more. Have a great weekend! Follow the Sports Animal on Facebook, Instagram and X PLUS Jim Traber on Instagram, Berry Tramel on X and Dean Blevins on X Follow Tony Z on Instagram and Facebook Listen to past episodes HERE! Follow Total Dominance Podcasts on Apple, Google and SpotifySee omnystudio.com/listener for privacy information.

Ron Hughley, Brandon Kiley, and Stephen Serda are back to discuss who the Chiefs should take a look at with Cam Jordan returning to the Saints. Rashee Rice is out of jail, and none of us are confident it's the last of his worries. Subscribe: https://youtube.com/live/cQy6exgKdWQ Learn more about your ad choices. Visit megaphone.fm/adchoices

We all have a Father we can look up to. Even if the man we called dad was a disappointment, or abusive, or just less than loving, we have a Father in heaven who loves us. And today on A NEW BEGINNING, Pastor Greg Laurie points out, even if we never knew our earthly father, God is there for us. How does Pastor Greg know? From first-hand experience. Today he brings us a powerful message called, How to Be a Father When You are Fatherless. Harvest Crusade tickets are fully claimed—but it’s not too late to participate and witness what God does on July 11. Invite your loved ones to watch online with you and make sure you join the waitlist in case more tickets become available. — Become a Harvest Partner today and join us in knowing God and making Him known through media and large-scale evangelism, our mission of over 30 years. Explore more resources from Pastor Greg Laurie, including daily devotionals and blogs, designed to answer your spiritual questions and equip you to walk closely with Christ.Support the show: https://bit.ly/anbsupportSee omnystudio.com/listener for privacy information.

We all have a Father we can look up to. Even if the man we called dad was a disappointment, or abusive, or just less than loving, we have a Father in heaven who loves us. And today on A NEW BEGINNING, Pastor Greg Laurie points out, even if we never knew our earthly father, God is there for us. How does Pastor Greg know? From first-hand experience. Today he brings us a powerful message called, How to Be a Father When You are Fatherless. Harvest Crusade tickets are fully claimed—but it’s not too late to participate and witness what God does on July 11. Invite your loved ones to watch online with you and make sure you join the waitlist in case more tickets become available. — Become a Harvest Partner today and join us in knowing God and making Him known through media and large-scale evangelism, our mission of over 30 years. Explore more resources from Pastor Greg Laurie, including daily devotionals and blogs, designed to answer your spiritual questions and equip you to walk closely with Christ.Support the show: https://bit.ly/anbsupportSee omnystudio.com/listener for privacy information.

Denny Hamlin is on an absolute heater! The NASCAR Cup Series rolled into Pocono Raceway for The Tricky Triangle weekend, and Hamlin delivered his third thrilling win in a row, shaking up the playoff conversation and raising a huge question: are the top two drivers in the NASCAR Cup Series points already a lock? Kerry Murphey and Toby Christie break down all the action from Pocono, including Hamlin's dominant run, the biggest storylines from the race weekend, and what it means moving forward in the NASCAR playoff picture. Plus, NASCAR news of the week from TobyChristie.com features Chris Buescher's contract extension, The Wood Brothers announcing their new driver for the iconic No. 21 car in 2027, and Denny Hamlin opening up about his retirement plans. Then it's time to look ahead to one of the most unique races on the NASCAR schedule: the all-new street course at Naval Base Coronado on Coronado Island in San Diego. The guys preview what to expect from NASCAR's newest street course challenge, and Rowdy Dragon stops by with his Fantasy NASCAR Picks for Naval Base Coronado. It's Pocono drama, Hamlin hype, NASCAR news, San Diego street course chaos, fantasy picks, and more on The Final Lap Weekly with Kerry Murphey and Toby Christie! NASCAR News Provided By: http://tobychristie.com -Listen on Spotify -Listen on Apple Podcasts -Click To Play Fantasy NASCAR With Us!  -Giving Fun Is Here: Patreon.com/thefinallap   NASCAR, NASCAR podcast, The Final Lap Weekly, Kerry Murphey, Toby Christie, Denny Hamlin, Denny Hamlin Pocono win, Pocono Raceway, Tricky Triangle, NASCAR Cup Series, NASCAR race recap, NASCAR Pocono recap, Hamlin three wins in a row, NASCAR playoffs, NASCAR points standings, Chris Buescher contract extension, Wood Brothers Racing, Wood Brothers 21 car, NASCAR 2027 driver news, Denny Hamlin retirement, Naval Base Coronado, San Diego street course, NASCAR San Diego, Coronado Island, NASCAR street course, Rowdy Dragon, Fantasy NASCAR Picks, NASCAR fantasy picks, NASCAR news, TobyChristie.com Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Follow The ThunderCast on social media so you never miss an episode or a ticket giveaway!! ThunderCast.Online Instagram Tik Tok Threads Twitter Facebook YouTube   The ThunderCast is brought to you each week by Leasure Oliver PLLC. Please remember, if you are ever the victim of a car wreck, contact Leasure Oliver PLLC at 304carwreck.com Jason and Matt are local attorneys proudly serving West Virginia, Kentucky, & Ohio. Like them on Facebook as well.   5 Things Every Herd Fan Needs To Know This Week is sponsored by Ignite Link, The Tri-State's Premier IT Management Team.  Contact Ignite Link for all of your business' IT and media consulting needs at (304)908-9424 or online at: Website Facebook Twitter   Learn how you or your business can be a part of The Thunder Trust Follow The Thunder Trust on all Social Media Outlets Instagram Twitter Facebook   Join the Big Green for as little as $8.33/Month, so you can take advantage of all of the money saving Herd Perks that come along with membership, in addition to from providing critical scholarship funding for our Herd Athletes.     An all new way to support Herd Athletics is here... at no cost to you!! The next time you book a hotel online, go to HerdHotels.com.  Its free, easy, and 50% of the booking commission goes right back to Marshall Athletics.   ALWAYS buy your tickets to ALL Marshall Home Games, Away Games, Tournaments, & Bowl Games at HerdZone.com or by calling 800-The-Herd   Sign your kids up for The Thundering Herd Kids Club and let's build a new era of passionate Herd Fans!!   We'll see you around The Joan...

You get 101 nights to try Ghost Bed at home. If you don't feel the difference, you can send it back, risk-free. Right now, GhostBed is offering my audience their lowest prices of the season, plus an extra 10% off; just got to https://www.GHOSTBED.COM/POSO and use promo code POSO.Support the show

Molly Sims is a model, actress, producer, founder of YiSE Beauty, and host of the Lipstick on the Rim podcast. After decades in the beauty and fashion industry, she's amassed an incredible wealth of knowledge on what actually works when it comes to looking and feeling your best.In this episode, Molly shares the beauty lessons she's learned from modeling around the world, why she's more confident in her 50s than she was in her 20s, and how she shifted from seeking external validation to finding confidence from within.We discuss the European approach to aging, why restraint is often the key to looking great as you get older, and the importance of staying true to yourself rather than chasing every beauty trend. Molly also opens up about dealing with acne, hyperpigmentation, and insecurities throughout her career, despite being one of the industry's most recognizable faces.Plus, we get into her favorite lasers and beauty treatments, her peptide obsession, exact Sports Illustrated prep, five-minute makeup routine, fragrance favorites, how to find great providers, and the best and worst beauty advice she's ever received.You can find Molly @mollybsims on Instagram, and don't forget to check out her beauty brand @yisebeauty.This episode is brought to you by:Visit hungryroot.com/blonde to get 40% off your first box and a free item of your choice.Visit ProlonLife.com/BLONDE to get 15% off their 5-day nutrition program.Head to armra.com/WELL or enter WELL to get 30% off your first subscription order.Go to fatty15.com/WELL and use code WELL at checkout for an additional 15% off their Starter Kit.Go to Ritual.com/WELL for 25% off your first month.Head over to thisisneeded.com and use code WELL for 20% off your first orderHead to ladder.fit/WELL for a 7-day free trial and $10 off your first month.This episode may contain paid endorsements and advertisements for products and services. Individuals on the show may have a direct, or indirect financial interest in products, or services referred to in this episode.Produced by Dear Media See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Most podcasters don't realise how much leverage their show actually gives them. Erin Treloar figured it out, and she's been cashing in ever since. Erin is the host of Well Within, a podcast for women ready to feel their best in body and life, covering health, wealth, relationships, spirituality, and more. She's been podcasting for six years, releasing episodes almost every single week. An award-winning podcast host and health coach, Erin's work has been featured in The Huffington Post, the LA Times, and Marie Claire. Based in Vancouver, she recently rebranded her show from Raw Beauty Talks to Well Within and successfully maintained her audience through the transition. She shares/breaks down: ◼️ How she negotiated hotel partnerships that now cover her LA recording trips in full ◼️ Why she started charging guests $500 to appear on her show, and how most said yes immediately ◼️ The three-month social media and podcast package she sells to founder guests ◼️ How her podcast directly converted a cold lead into a paying coaching client ◼️ What the O-1 extraordinary ability visa process taught her about her own accomplishments ◼️ Why she's launching a branded podcast tour, and how she's pitching sponsors to fund it ◼️ The metric mistake she admits cost her years of insight into what was actually working   Follow Erin Troulor: ◼️ Instagram: https://www.instagram.com/erintreloar/ ◼️ Website: https://erintreloar.com/  ◼️ Well Within Podcast: https://podcasts.apple.com/gb/podcast/well-within/id1454866000    Want to grow your visibility through podcast guesting? Explore how PodWritten can help: https://podwritten.com/services/   Bonus tips and resources: ◼️ Blog: https://podwritten.com/blog/ ◼️ Instagram: https://www.instagram.com/podwritten/ ◼️ LinkedIn: https://www.linkedin.com/company/podwritten   Questions or want to say hey? Email us at sam@podwritten.com   Please leave us a review on Apple Podcasts or Spotify.

Brothers, this episode is for you.James sits down and delivers a Father's Day message that every Catholic father needs to hear. He starts with the hard truth: America is in a fatherlessness catastrophe. One in four children grows up without their biological father. The research on what that produces is not subtle. And some of the most dangerous absences are not the men who walked out the front door — but the men who are still sitting at the dinner table and have already checked out.But this episode is not about guilt. It is about weight and dignity and the sacred office that most men are carrying without ever being told what it actually is.James unpacks why Jesus calls God Father. He walks through Saint Joseph: a man who never speaks a single word in Scripture, who just acts, stays, protects, and obeys. He talks about the thanklessness of faithful fatherhood and how staying anyway is one of the most countercultural things a man can do in 2026.And then he gets personal. The exhaustion. The moments of checking out. The weight of running a home, working full time, homeschooling, podcasting, and still trying to show up. This one is honest.

Drew and Steak analyze the impact of global stars like Lionel Messi and Kylian Mbappé playing in the United States. They dive into the controversy surrounding Brendan Sorsby's gambling ruling and his potential future in the NFL. Finally, the discussion turns to Georgia baseball's record-breaking season ahead of a matchup against Oklahoma. 01:53 - World Cup Star Power 04:01 - Brendan Sorsby Gambling Ruling 10:31 - Georgia Baseball's Historic Season

Laureano v. Att'y Gen. U.S., No. 24-2433 (3d Cir. May 29, 2026)petition for review withholding of removal; reinstatement; Riley; Matter of Y-L- presumption Matter of Herrera-Nunez, 29 I&N Dec. 691 (BIA 2026)stay pending motion to reopen; requirement to file stay with DHS first; procedural rules; administrative efficiency; power to create discretionary policies Matter of Herrera-Nunez, 29 I&N Dec. 695 (BIA 2026)sua sponte motion to reopen; equities acquired post-removal order Dodaj v. Blanche, No. 25-3409 (6th Cir. June 8, 2026)LPR cancellation of removal; BIA reweighing evidence; waiver of removability argument; HYTA youth conviction in Michigan as a minor Senatus v. U.S. Att'y Gen., No. 24-10514 (11th Cir. June 8, 2026)Borden; crime of violence; recklessness; Fla. Stat. § 784.021(a); Somers; Florida Supreme Court change in law tells what statute always meant; favorable exhaustion standard Tiah v. Blanche, No. 24-2010 (8th Cir. June 8, 2026)INA § 237(a)(2)(E)(ii); violation of a protective order; circumstance specific approach; no requirement to submit violation; N.D.C.C. § 12.1-31.2-02Kurzban Kurzban Tetzeli and Pratt P.A.Immigration, serious injury, and business lawyers serving clients in Florida, California, and all over the world for over 40 years.eimmigration"Immigration law software you'll love to use."get.eimmigration.com/IRP Gonzales & Gonzales Immigration BondsP: (833) 409-9200immigrationbond.com Stafi"Remote staffing solutions for businesses of all sizes"Click me!Want to become a patron?Click here to check out our Patreon Page!CONTACT INFORMATION:Email: kgregg@kktplaw.comFacebook: @immigrationreviewInstagram: @immigrationreviewTwitter: @immreviewAbout your hostCase notesRecent criminal-immigration article (p.18)Featured in San Diego VoyagerSupport the show

Everyone I know pulls their hair out because it's one thing to organize the house...it's another for it to STAY organized. That's what today's episode is for! I break down the difference between macro and micro organizing, when to use each type, and how to make sure your house doesn't explode 30 seconds after you finally have it looking good. MENTIONED THIS EPISODE How to Declutter Your Kitchen (YouTube video) Ep 77: What If I End Up Needing What I've Decluttered? Ep 5: Decluttering Sentimental Items Ep 50: The Guilt & Fear of Decluttering Gifts Shoe Organizing Bin PARING DOWN (SHANNON LEYKO): Sign up for my newsletter! ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠The L.E.S.S. Express⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Website: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠www.shannonleyko.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Instagram: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠@shannonleyko⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ TikTok: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠@shannon_leyko⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Youtube: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.youtube.com/@shannonleyko⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Facebook: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠www.facebook.com/shannonleyko.paringdown⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Substack: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Blog & Additional Support (free trial!)⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠TAKE THE QUIZ!!⁠⁠⁠⁠ "What's Your Decluttering Type?" & receive a customized playlist with 10 episodes of Paring Down for your exact needs. ⁠⁠⁠⁠⁠⁠⁠⁠⁠PARING DOWN RESOURCES⁠⁠⁠⁠⁠⁠⁠⁠⁠: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠CLICK HERE for free checklist, hacks, worksheet, & more!⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ SPONSORS: Get 10% off your first order of OSEA skincare (sitewide) with code PARING at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠OSEAMalibu.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ $250 off Air Doctor Pro air purifier:⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ https://airdoctorpro.com/ -⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Use code PARING 20% OFF any AquaTru water purifier when you go to ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠AquaTru.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ and use promo code PARING Ethical, luxury women's clothing at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Quince.com/paring⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ for 365-day returns, plus free shipping on your order! Go to ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠BornShoes.com ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠today for a 15% discount plus free ground shipping on all full-price shoes when you use my promo code, PARING 10 Free Meals from Hello Fresh + Free Breakfast For Life:⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠www.hellofresh.com/paring10fm⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Only $2.99 per meal from Every Plate + 10% off for a month: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠www.everyplate.com/podcast⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ - CODE: paring299 Find furniture, decor, and essentials that fit your unique style and budget. ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.wayfair.com/⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ 15% off ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠oneskin.co/PARING⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ with code PARING Learn more about your ad choices. Visit megaphone.fm/adchoices

Jace and Rocky break down all 13 DC releases for the week of June 17, 2026. They discuss Batwoman #4, where Kate Kane and Renee Montoya begin searching for Alice as the mystery involving Beth, the Darkseid cult and Gotham's elite finally starts coming into clearer focus. Absolute Flash #16 sends Wally and Linda deeper into their search for Ray Palmer as young Grodd leads them toward Gorilla City and the Rogues close in. Batman/Superman: World's Finest #52 continues the heroes' adventure in Skartaris as Robin joins Travis Morgan and his allies while Tyrant Rex uses magic to turn Batman and Superman against them. Deathstroke the Terminator #4 finds Rose Wilson saving Deathstroke before father and daughter struggle to connect during a tense conversation that forces Slade to confront what kind of parent he has become. Lobo #4 follows the Main Man's attempt to break into Superman's off-world "Fortress of Storitude," bringing him into conflict with Supergirl while Krypto and Dawg create their own chaos. Absolute Green Arrow #2 sees Dinah Lance and Merlyn questioning figures from Oliver Queen's past while the mysterious archer targeting billionaires remains one step ahead of their investigation. Catwoman #88 reveals that Katarina Belova survived the fire and has been manipulating Black Mask's campaign to destroy Selina, bringing the European conspiracy and Gotham storylines together. New Titans #36 finds the team settling into Titans Mountain as Starfire leaves, Cyborg secretly works to activate a massive Mother Box and questions continue to surround Eva's nanite-based body. Justice League: Dream Girls #3 sends Galaxy back into Dreamer's mind to rescue her from the Key while the Justice League questions Galaxy's actions, and the backup follows Harley Quinn and Poison Ivy through a quiet day together. Nightwing #139 sees Dick Grayson use his scientific training to examine bodies recovered from the water while facing pressure to surrender for a crime he did not commit, leading to a confrontation with a priest whose actions create a difficult moral dilemma. Superman Unlimited #14 intensifies the questions surrounding the young Jon Kent as kryptonite, old memories and Batman's investigation begin exposing cracks in his story. Absolute Batman #21 pits Batman against the Robins before Jack Grimm confronts Bruce Wayne and claims responsibility for creating the man he became. End of Life #5 shows Eddie at his most effective as a killer before his father forces him to confront the fact that he may no longer have a place in Pluto. They also give a rundown of this week's collected editions, reprints and facsimile releases. As always, all books are ranked from top to bottom and each host gives a Book of the Week pick.

There was a time when everything on the internet seemed like it would be there forever, whether a Facebook status or a government website. But today the prospect of maintaining massive amounts of digital history is in doubt. This poses a particular issue for the open science movement, which has advocated for wide access to scientific data and papers over the last 25 years. The movement's successes have led to the creation of digital research infrastructure such as data libraries and preservation systems that serve large and small research communities. Now, as the system is maturing, it's becoming clear that open science isn't free: it requires investment and significant effort to stay alive. Without it, open research cannot be verified or advanced. On this episode, host Megan Nicholson is joined by Jen Gibson and Kaitlin Thaney, authors of “Who Will Keep Research Data Infrastructure Open and Running?” in our Spring 2026 issue. Gibson is the executive director of Dryad, an open-access international research data repository and curation service. Thaney is the executive director of Invest in Open Infrastructure, a nonprofit that advances open-source solutions and systems for research communities. Resources:Read “Who Will Keep Research Data Infrastructure Open and Running?” for more on the importance of maintaining digital infrastructure.Visit Dryad and Invest in Open Infrastructure's websites to learn more about open data, open infrastructure, and how you can support their work. 

YESS BIG SHOW The boys are back together! Chom's back from Perth, Booker back from paternity leave & Butts from Thailand. Geez they're excited to be back, and they talk: - Soceroos making us proud - Earliest sporting memories - Booker is a father - Chom's time in Perth - Butts Thailand stories (not to be missed) & Much more! 200 Plus live show: https://clubbysports.com/collections/events/products/live-and-uncut Enjoy plums and remember to GET THE KNEES UP! Send us your voice messages here: https://memo.fm/200pluspodcast/ Produced by Ben Raimondo 200 PLUS 2026 Membership: https://www.patreon.com/c/200PLUS 200 PLUS Instagram: https://www.instagram.com/200pluspod/ Nick Butler: https://www.instagram.com/nick_butler10/ Charlie Comben: https://www.instagram.com/charliecomben/ Darcy Cameron: https://www.instagram.com/darcy_cameron/ Sam Draper: https://www.instagram.com/draper/ Clubby Sports: https://www.instagram.com/ClubbySports To get in contact with Clubby Sports email Hello@ClubbySports.com

title says it all. quick episode to touch about football recruiting, all of HCCA's media appearances last week and the Sorsby of it all. Learn more about your ad choices. Visit podcastchoices.com/adchoices

Send us a message!Welcome to the second episode of Pour Decisions: When Stays Go Sideways, presented by Safely.In this episode, Alex and Annie are joined again by Amanda Martins, VP of Business Development at Safely, for another round of real guest stories, summer stay surprises, and practical takeaways for short-term rental operators.This conversation is all about “vacation brain” and the strange, messy, and sometimes costly decisions guests make when they are out of their normal routine.From poolside surprises to hot tub chaos and grill-related mishaps, this episode brings the stories, the laughs, and the reminders operators can use before the next stay goes sideways.We discuss:05:38 - Why summer season can bring more guest-related damage08:38 - How vacation brain shows up in real short-term rental claims18:53 - What operators can learn from pool, hot tub, and grill-related incidents25:29 - Why clear amenity instructions can help reduce preventable issues10:24 - How protection programs can support owner trust08:20 - Why guest involvement is not always required in many damage claims11:49 - How Safely helps property managers explain coverage to homeowners38:30 - Why operators should think about preparation before the busy season gets messyConnect with Amanda:LinkedIn: https://www.linkedin.com/in/amandakatmartins/Learn more about Safely:Website: https://safely.com/alex-and-annieLinkedIn: https://www.linkedin.com/company/safely-stay/Instagram: http://instagram.com/safely/Facebook: https://www.facebook.com/safelyprotectionThis episode's cocktail: Hugo Spritz with a twistA refreshing summer spritz inspired by Annie's favorite vacation drink, with Amanda's optional vodka twist.Ingredients:ProseccoElderflower liqueurFresh limeFresh mintClub sodaVodka, optionalHow to make it:Build over ice in a wine glass. Add elderflower liqueur, lime, mint, and vodka if using. Top with Prosecco and club soda, then stir gently.Best enjoyed responsibly and preferably nowhere near a hot tub full of seafood.#vacationrentals #shorttermrentals #vacationbrain

James, Darragh and Rocco are back for a summer catch-up - and despite "nothing happening," there's plenty to chew over. We kick off with the new Adidas 26/27 away kit (Rocco's not sold on the navy shorts), the historic trefoil logo and Leeds moving up Adidas' premium tier, before getting stuck into Paraag Marathe's annual address and what it tells us about how the 49ers are running the club. Then the big one: Ethan Ampadu's new four-year deal, no relegation clause, and why locking down the captain so early is the biggest statement Leeds could make this summer. The boys debate Farke's future, the transfer targets doing the rounds (Diomande, Harry Wilson and the never-ending goalkeeper search), who's likely to leave, and whether Gnonto could be another one that gets away. Plus: Rocco's off to Mexico for Uruguay v Spain at the World Cup, a genius (or daft?) idea for fixing extra time, why Karl Darlow might just be the answer in goal, and a lovely Rafinha tribute to Liam Cooper to finish. Sponsored by Bass & Bligh - https://bassandbligh.com - the finest purveyors of binoculars, spotting scopes and camera gear. Pop into 6 Beulah Street in Harrogate and make a day of it.

title says it all. quick episode to touch about football recruiting, all of HCCA's media appearances last week and the Sorsby of it all. Learn more about your ad choices. Visit podcastchoices.com/adchoices

Nebraska had another productive official visit weekend, landing two commitments, trending for more coming out of a smaller, but still notable visit weekend. Inside Nebraska's Tim Verghese talks Nebraska''s latest additions: wide receiver Justyn Lindsay and linebacker Caleb Green, who the Huskers flipped from Missouri. Verghese also talks the latest with top targets in town, including two of Geep Wade's top remaining offensive line targets and two of Addison Williams' top remaining defensive back targets. The Huskers have another big week ahead, set to host one last major official visit weekend, plus the Battle of the Boneyard 7-on-7 tournament on Saturday. Subscribe for Nebraska football and recruiting: nebraska.rivals.com Follow us on Twitter: https://twitter.com/NebraskaRivals Follow us on Instagram: @NebraskaRivals #Nebraska #NebraskaFootball #NebraskaCornhuskers #GBR #HuskerFootball

#Sooners remain SCORCHING HOT at #MCWS in #Omaha after beat #Alabama 9-0 in round one. Now they are set to face off against fellow #SEC foe #Georgia in the #winners #bracket on Monday at 6 PM CST. OUInsider's Travis Davidson and SEC Unfiltered's Chris Phillips are breaking down it all as #BoomerSooner and #GoDawgs try and remain in the winner's bracket. What does #OU need to do? Can #Oklahoma's pitching and fielding withstand the aggressive nature of #UGA bats and base running? Can #Oklahoma and Skip Johnson's squad keep the power behind their bats vs #UGA pitching? They give you the full rundown, so join us and be as prepared as possible for the next big MCWS matchup between two top SEC powerhouses! Want the best Sooners' intel and info in the industry? Want to be a part of a community of THOUSANDS of Sooner fans that you can talk with on a daily basis? Become a member of OU Insider and stay updated daily with insights from the OUI staff!!

Inflation reached its highest level in three years while the labor market remained surprisingly strong. Kathy Fettke breaks down the latest CPI and jobs data, what it could mean for Federal Reserve rate cuts, and why mortgage rates may stay elevated for real estate investors.   Source: https://finance.yahoo.com/economy/article/annual-cpi-inflation-surges-to-42-in-may-the-highest-level-since-2023-as-energy-prices-rise-182143107.html 

Why can some people lose & maintain on so much higher calories than others? Over time will you be able to increase your maintenance intake so that it matches or is even higher than theirs?You have questions, today's episode has answers.Chapters00:00 Understanding Caloric Disparities02:53 The Role of Body Size and Muscle Mass06:01 Non-Exercise Activity and Its Impact08:46 Health Considerations in Caloric Needs12:09 The Importance of Accurate Tracking14:45 Social Media and Its Influence on Perception17:52 Strategies for Improving Caloric Intake20:58 Conclusion and Key TakeawaysLinksApply for Coaching: https://form.typeform.com/to/ubUfJiEu?utm_source=podcastLiving Lean Podcast: https://www.buzzsprout.com/712032Follow Jeremiah on Instagram: https://www.instagram.com/jeremiahbair/Follow Andrea on Instagram: https://www.instagram.com/andirogersfit/Follow Natalie on Instagram: https://www.instagram.com/natalieatswell/Keywordscalorie needs, maintenance calories, body size, muscle mass, metabolism, fat loss, health, tracking accuracy, social media influenceTo Apply For Coaching With Our Team: CLICK HERE

Claudia Sahm says inflation remains relatively narrow despite recent energy shocks but warns the breadth of price pressures bears watching. She highlights a growing disconnect between consumer sentiment and market performance, driven by prolonged inflation and a gradually slowing labor market.======== Schwab Network ========Empowering every investor and trader, every market day.Subscribe to the Market Minute newsletter - https://schwabnetwork.com/subscribeDownload the iOS app - https://apps.apple.com/us/app/schwab-network/id1460719185Download the Amazon Fire Tv App - https://www.amazon.com/TD-Ameritrade-Network/dp/B07KRD76C7Watch on Sling - https://watch.sling.com/1/asset/191928615bd8d47686f94682aefaa007/watchWatch on Vizio - https://www.vizio.com/en/watchfreeplus-exploreWatch on DistroTV - https://www.distro.tv/live/schwab-network/Follow us on X – https://twitter.com/schwabnetworkFollow us on Facebook – https://www.facebook.com/schwabnetworkFollow us on LinkedIn - https://www.linkedin.com/company/schwab-network/About Schwab Network - https://schwabnetwork.com/about

Does Mitch Keller belong in the bullpen? The Pirates have decisions to make on a multitude of players. Marcell Ozuna, Henry Davis, Dennis Santana, and Jared Triolo have all struggled. We discuss if we would keep those players on the roster or who we would move on from. 

Hour 2 with Joe Starkey: Steelers reporter Ray Fittipaldo of the Pittsburgh Post-Gazette joined the show. The Steelers are closing OTAs today and the young quarterbacks are getting all the reps. Ray thinks the Steelers are giving Will Howard every opportunity to win the backup job. Ray thinks Will Howard will end up on the team. Drew Allar has made "good progress" since starting his Steelers career. The Pirates have decisions to make on a multitude of players - Marcell Ozuna, Henry Davis, Dennis Santana, and Jared Triolo.

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

Bestselling Regency and Victorian romance author Kathleen Ayers joins Samantha Tennant to talk about leaving a 30-year career in media sales to follow a dare from her best friend — and ending up with a romance career she never saw coming. Kathleen shares how classic gateway novels like SWEET SAVAGE LOVE and THIS LOVING TORMENT shaped her love of historical fiction, why morally gray men are still the most irresistible heroes in any era, and what it was really like to write alongside authors like Cathy Maxwell and Jenna MacGregor for the Busty Bodice Club anthology. She also opens up about her process as a self-described pantser, the one fictional character who refused to stop talking to her, and why she will never apologize for being an unapologetic Ruby Dixon super-fan. Plus: Buc-ee's Beaver Nuggets, Nine Inch Nails concert tees, and the mysterious ancestor named Barbara who just disappeared from the family records.

Welcome to BARBERSHOP CONFIDENTIAL the Podcast   produced by @d0peclick Episode 262, back with another episode, this week we have Katelyn aka Kata, we had a dope conversation about life and her journey in the content creating space, we talked about being outside and toxic behavior. We keep it 100 on this podcast and we hope you enjoy it!. We had a dope fun convo and I hope you find entertaining and meaningful as we did, please follow Drew and support.  As always we never intend to offend anyone with our opinions.    Any questions or comments please email at bscpdcst@gmail.com   https://bscpdcst.wixsite.com/bscpodcast SUBSCRIBE to YOUTUBE: https://www.youtube.com/channel/UCMk3TS3qnykSLkO6wk2IrdQ   PLEASE follow us on Instagram @BarbershopConfidential Host: @fkinjona Guest: @katelynocamp0   Dale Gas merch @dale.gas.sd   #podcast #chisme #sugarbabby #sugardaddy #gossip #storytime #cheating #sidepiece #musician  #toxicrelationships #chisme #delulu #womeninmensfeild #toxicadvise #god #relationshipwithgod #church #faith #religion #crashout 

What makes a great leader? Not talent.Not charisma.Not position. According to Mr. Black, great leaders are built on a triangle—and triangles are the strongest structures in creation. Remove one point and the structure weakens. Keep all three and it can withstand tremendous pressure. On this episode of Like It Matters Radio, Mr. Black builds on last week’s discussion of the power of triangles and introduces what he calls the Leadership Triangle: OMNI — Living with purpose on purpose SEMPER — Always flexible, always growing HINENI — All in, fully present Together, these three words create a framework for transformational leadership. You’ll discover why words matter more than most people realize, how identity shapes behavior, and why great leaders are intentional about what they think, say, and believe. Drawing from Scripture, neuroscience, epigenetics, and leadership psychology, Mr. Black explores the connection between thoughts, beliefs, and outcomes. This episode also unpacks: Why purpose gives meaning to both success and suffering The leadership principle of Requisite Variety—the most adaptable person has the most influence Why presence is becoming one of the rarest and most valuable leadership skills in today’s distracted world How words can reinforce identity, shape culture, and change lives At its core, this episode is a challenge to become the kind of leader who: Sees purpose in everything Stays flexible in anything Shows up with everything Because leadership isn’t just about what you do. It’s about who you’re becoming. You were created by an OMNITELIC God.Designed to live a SEMPER life.And called to walk with a HINENI heart. This is the Triangle of a Great Leader. Inspiration. Education. Application.When you live your life like it matters… it does.See omnystudio.com/listener for privacy information.

Connor and Ace talk about the massive week in recruiting, which includes 7 total football commits, including the #1 player in the State of Kansas!Twitter: https://x.com/AggievilleACatsMerch: https://aggievillealleycats.myspreadshop.com/

On this week's Mean Age Daydream, Brian has no pity for the fired Scott Pelley of 60 Minutes fame, one of the most overrated shows ever in its modern incarnation. Also: Spencer Pratt advances into a finale vs. Karen Bass in Los Angeles, and the IPCC is exposed as charlatans. Buy my new card game: WOKE WORD WARS - https://www.amazon.com/Woke-Word-Wars-Ridiculously-Players/dp/B0FLF8Y16S

Brighton put up a good fight but were ultimately blown away by Manchester City in the FA Cup Final. What's more, Andrée Jeglertz' team has been given a massive boost by Bunny Shaw announcing she'll be staying! Could this be the start of a sky blue dynasty?While City fans celebrate, Arsenal fans have been left somewhere between shocked and fuming by Katie McCabe's transfer to rivals Chelsea. It is hard to resist the champions of the World Sevens, in fairness. Rachel was on the ground at that wacky tournament and is here to tell us all about it.Finally, it's that time again. England vs Spain for what feels like the millionth time.Follow us on X, Instagram, TikTok and YouTube! Email us show@upfrontpod.com.For ad-free episodes and much more from across our football shows, head over to the Football Ramble Patreon and subscribe: patreon.com/footballramble.**Please rate and review us on Apple, Spotify or wherever you get your pods. It means a lot and makes it easy for other people to find us. Thank you!**Upfront is your definitive women's football podcast, covering the WSL, the Lionesses, and beyond. Subscribe for new podcasts every Tuesday throughout the season. Hosted on Acast. See acast.com/privacy for more information.

   The Kansas Jayhawks get exactly the kind of break it takes to have a magical season as Oklahoma wins at Georgia Tech.  Tech was the top team left in the tourney and would have hosted KU with a win.  Instead, Tech is toast and OU is coming to Lawrence.  This thing just got real!    We got a sneak preview of the new Morton Amphitheater Monday night in Riverside and you're going to love this place.  KC needed it, we deserve it and our quality of life just got a little bit better.  You almost feel like it's too nice for an outdoor venue!    Multiple reports indicate President Trump had a very heated phone call with Israel's Bibi Netanyahu and it was pretty ugly.  We discuss what it could all mean.    The Kansas Governor's race got wild Monday, just before the official filing deadline.     A dude with too much time on his hands checked out all 32 NFL team websites and provides a list of teams that don't celebrate, mention or recognize pride month.  We have a crazy new poll for GOP presidential nominee in 2028, you have to hear how Vance v Rubio is shaping up.    The Royals are undefeated in June!  Yes, the team got a nice win but we have some player rankings for you that explains exactly why the Royals are so bad.     The Rams make a big trade, a Super Bowl quarterback is retiring and become an announcer and we've got the first set of confirmed celebrities for the Travis and Taylor wedding.

Gary Parrish and Matt Norlander break down the most impactful NBA Draft withdrawal deadline decisions. Tounde Yessoufou is coming back and heading to St. John's. Milan Momcilovic is back. Koa Peat is gone. All-Americans are returning to the sport and plenty more from the May 27 deadline. (0:00) Intro + we got deadline decisions to discuss! (1:00) Younde Yessoufou is coming back … and headed to St. John's (8:00) Where will Milan Momcilovic play next season (16:30) Two players that are officially gone: Koa Peat & Christian Anderson (24:00) Important players returning to college (32:00) GP played golf at PGA Frisco … let's talk (35:40) News & notes: College sports legislation and a conference name change Theme song: “Timothy Leary,” written, performed and courtesy of Guster Eye on College Basketball is available for free on the Audacy app as well as Apple Podcasts, Spotify and wherever else you listen to podcasts. Follow our team: @EyeonCBBPodcast @GaryParrishCBS @MattNorlander @Boone @DavidWCobb @TheJMULL_ Visit the ⁠betting arena on CBSSports.com⁠ for all the latest in ⁠sportsbook reviews⁠ and ⁠sportsbook promos⁠ for ⁠betting on college basketball⁠. You can listen to us on your smart speakers! Simply say, “Alexa, play the latest episode of the Eye on College Basketball podcast,” or “Hey, Google, play the latest episode of the Eye on College Basketball podcast.” Email the show for any reason whatsoever: ShoutstoCBS@gmail.com Visit Eye on College Basketball's YouTube channel: ⁠https://www.youtube.com/channel/UCeFb_xyBgOekQPZYC7Ijilw⁠ For more college hoops coverage, visit ⁠https://www.cbssports.com/college-basketball/⁠ To hear more from the CBS Sports Podcast Network, visit ⁠https://www.cbssports.com/podcasts/

On Friday's Mark Levin Show, President Trump has canceled plans to attend his son Donald Trump Jr's wedding and instead remains at the White House, which might be a signal that the clock is ticking and decisive action against Iran may be imminent. It looks like the Iranian regime is digging in its heels and the President has had enough. It is critical that this ends the right way by defeating the enemy. If there was a deal, does anyone seriously believe the Iranian Nazi terror regime will adhere to it over time? And if you do, based on what? They exist to conquer, devour, and destroy. And believe this is commanded by Allah. By any means. Also, the Muslim Brotherhood is a terrorist organization with members operating openly in the US and Europe seeking to conquer and destroy Western societies rather than assimilate, having already taken control of many cities in Britain, France, and the rest of Europe. The Democrat Party is becoming a wholly owned subsidiary of the Muslim Brotherhood by nominating, electing, and collaborating with these figures without denunciation. The Woke Reich serves this agenda from within while the MAGA movement remains unified in strong support of the president. Later, Rep Hakeem Jeffries has a brother, Hassan Kwame Jeffries, who's an extremist. He invoked abolitionist John Brown's philosophy of violence, quoting Brown's statement that the crimes of the land would never be purged without blood and declaring that White supremacists must be eliminated “by any means necessary.” This is a larger pattern in which university employees normalize political violence. Afterward, Welles Crowther, the "man in the red bandana," saved more than 18 lives on 9/11 and today Trump posthumously awarded him the Presidential medal of freedom. Almost 25 years after 9/11, when the city suffered such hell at the hands of Islamists, they elected Zohran Mamdani as Mayor. Learn more about your ad choices. Visit podcastchoices.com/adchoices