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The CKNYC podcast returns after a hiatus. Same ingredients for the past 17 years. Vocal House + a dash of retro melodies remixed + some Madonna (duh) + a perfect mix every time. Produced and mixed by Kerry John Poynter. Track List (41:00): Sultan & Shepard & Shallou - Twilight Fahlberg - Looking Good DJ Fave! Madonna - One Step Away. A fave track from the new Confessions II album. The Queen. Who Made Who & Kolsch - Heartless (Fidelis Remix) Tinlicker & Hero Baldwin - I Started A Fire  Madonna - La Isla Bonita (Mitch Oliver & Carl Sylvestri Remix) Retro Alert! Martin Strauts feat. Cosmogram - Enjoy The Silence (Original Mix) Depeche Mode Retro Alert! The Police - Every Breath You Take (Waltry Afro House Remix) Retro Alert! Is It Over Now (Original Mix) - Elderbrook http://www.clubkerrynyc.com About CKYNC: Number 1 Indie Dance (Goodpods). Ranked a top 1% most popular global-Reach podcast by Listen Notes. Listed a popular "Podcasts Worth A Listen" on PlayerFM. Chartable.com "Global Reach" is a top 20 music podcast. "Stylistically superior. The best vocal house podcast on the net" (iTunes Review). Celebrating 17 years: 2009-2026. More info at https://www.clubkerrynyc.com/about/. Kerry John Poynter has been podcast producing for over 17 years. He also produces and hosts "Like A Muse: The Madonna Remixes Culture Podcast". He completed an audio engineering degree from SAE Institute in NYC in 2024 with highest honors. Link Tree Audio: https://linktr.ee/kerrypoynter.

In the business news section of this week's episode, Axiom Biosciences recently announced its plans to go public and made headlines for its plans to do so on the Hong Kong Exchange rather than a U.S. market. We dig into the company's reasoning. Then we turn our attention to Latigo's report of positive Phase IIb data for its non-opioid acute pain candidate, and its plans to go through a planned IPO soon. Switching to some science news, we discuss findings from a study that identified genetic risk factors for fibromyalgia syndrome as well as links to other neurodegenerative, autoimmune, and metabolic diseases. And lastly, a potential base editing therapy for Huntington's disease that has shown promise in mice.Join GEN editors Corinna Singleman, PhD, Alex Philippidis, and Uduak Thomas for a discussion of the latest biotech and biopharma news. Listed below are links to the GEN stories referenced in this episode of Touching Base: StockWatch: Axiom CEO Explains Plans for Hong Kong IPO By Alex Philippidis, GEN Edge, July 27, 2026Latigo Reports Positive Phase IIb Data for Non-Opioid Acute Pain Candidate By Alex Philippidis, GEN Edge, July 30, 2026Genetic Study of Fibromyalgia Points to Neurological Basis GEN, July 29, 2026Base Editing Strategy Alleviates Huntington's Disease in Mice GEN, July 29, 2026Touching Base Podcast Hosted by Corinna Singleman, PhD Behind the Breakthroughs Hosted by Jonathan D. Grinstein, PhD The State of Biologics Testing 2026Charles River Laboratories and GEN, June 10, 2026 Hosted on Acast. See acast.com/privacy for more information.

How do you grow an Airbnb from barely breaking even to one of the most wish listed properties in your state?Janelle Gallucci joins Kenny Bedwell to share how she transformed a struggling Lake Tahoe Airbnb into one of the highest performing properties in Nevada. Starting with a property that barely covered its mortgage, she reveals the strategies that helped grow annual revenue from $25,000 to $165,000 while many hosts in competitive markets watched their earnings decline.From identifying the right guest avatar to creating memorable experiences for families and pets, Janelle explains why thoughtful hospitality consistently beats chasing the latest trends. She also shares the operational systems, marketing strategies, and guest touches that helped her property become the most wish listed Airbnb in Nevada.If you've ever wondered whether your current property still has room to grow, this episode offers practical strategies you can apply to create better guest experiences, increase revenue, and stand out in any market.Timestamped Highlights02:45 How Janelle grew one property from $25,000 to $165,000 in annual revenue08:42 Why firing the property manager completely changed the business15:07 The biggest mistake new hosts make when designing their property20:54 How defining one guest avatar transformed bookings26:38 The family friendly and pet friendly upgrades that guests love36:15 Creating premium pet experiences that justify higher fees44:20 The welcome basket strategy that generates five star reviews53:12 Why amenities create experiences instead of filling space59:46 Building a direct booking strategy that actually works1:07:18 Why optimizing one property can outperform buying anotherAbout the GuestJanelle Gallucci is a short term rental investor, educator, and co founder of Seven Summers Host Academy. After leaving a successful career in tech sales, she now helps Airbnb hosts increase revenue through hospitality, branding, and operational excellence. Her Lake Tahoe property became the most wish listed Airbnb in Nevada while consistently increasing revenue in one of the country's most competitive vacation rental markets.Important LinksWant us to find the deals for you? https://strinsights.comInstagram: @kenny_bedwellYouTube: Cash Flow PositiveLinkedIn: Kenneth BedwellGuest Instagram: @sevensummershostacademyProperty Instagram: @thetahoehouseCash Flow Positive is an original podcast hosted by Kenny Bedwell. Production and editing by Podcast Your Brand.

This week, we're with an absolute legend in food writing, Anissa Helou and her culinary celebration of Lebanon.Listed by Arabian Business magazine as one of the "500 Most Influential Arabs" and "100 Most Powerful Arab Women, she's among the greats of modern food writing with the likes of Claudia Roden and Elisabeth Luard. Now, after 32 years of writing about the everyday food of the Middle East and beyond, Lebanon, A Culinary Celebration takes her back to where she began. Pop over to Gilly's Substack for Extra Bites of Lebanon. If you'd like to support CTB which is advertising and sponsorship free, contribute whatever you like via this link, or become a paid subscriber on Substack which gives you access to Second Helpings, monthly Zooms with a CTB guest, and a massive archive of Gilly's articles. Hosted on Acast. See acast.com/privacy for more information.

Send us Fan MailNicky Jackson has been a CPG brand owner before, so she knew the ropes. When she first talked to Target US, she didn't ask for a trial, she asked for 700 doors. What came next was a surprise even to Nicky. Target came back with an offer of over 1,900.Nicky is the founder of Lioness, a daily functional snack range built around what women actually want throughout their day. Before Lioness, Nicky spent years at Kellogg Company  and Pepsi Co, then built RangeMe, the platform that changed how brands and retailers find each other.In episode 167 of the Foodpreneur with Chelsea Ford podcast, Nicky and I get into why she built a product the category didn't have, and what it actually cost her to get there.Don't miss this episode, you'll learn: 

What does it actually take to become one of the highest earning Airbnb properties in your market?In this episode, Kenny Bedwell shares the strategies behind one of Kentucky's top performing short term rentals and explains why success is about much more than adding expensive amenities. From choosing the right market to understanding exactly who your property is designed for, he walks through the framework that has helped his properties consistently outperform the competition.You'll learn why patience matters more than most hosts realize, how to evaluate a market before investing, and why the best properties are built around a specific guest instead of trying to appeal to everyone.If you want to increase revenue, stand out in your market, and create a property guests remember, this episode is packed with practical lessons you can apply.Timestamped Highlights00:00 Why becoming a top performing property starts with the right strategy02:10 The numbers behind one of Kentucky's highest earning short term rentals05:08 Why your second year is often your most important08:44 How investing more than your competition can create an advantage13:15 When spending more stops producing better returns17:02 How to find markets where you can stand out22:10 Why many hosts struggle to keep up with changing guest expectations28:06 How knowing your ideal guest influences every decision33:08 The marketing mistake that causes properties to blend in37:15 Four lessons every host can use to build a stronger businessAbout the HostKenny Bedwell is a short term rental investor, educator, and founder of STR Insights. He owns and operates vacation rentals across the United States, including one of the highest earning properties in Kentucky. Through Cash Flow Positive, he shares practical strategies that help investors increase revenue, improve the guest experience, and build stronger hospitality businesses.Mentioned ResourcesSTR InsightsAirbnbVRBOBooking.comKentucky Bourbon TrailImportant LinksWant us to find the deals for you? https://strinsights.comInstagram: @kenny_bedwellYouTube: Cash Flow PositiveLinkedIn: Kenneth BedwellCash Flow Positive is an original podcast hosted by Kenny Bedwell. Brought to you by STR Insights.

This week Nick talks to Gabriel Sacks Gabriel is lead manager of Aberdeen Asia Focus plc and a member of the investment team managing Aberdeen's Global Emerging Markets Income and Global Emerging Markets Smaller Companies strategies. During his time in Singapore, he also managed the Asian Smaller Companies Fund until returning to London in late 2022, while contributing to a range of Asian and global emerging markets portfolios. Gabriel holds an MA (Hons) in Land Economy from Selwyn College, Cambridge, and is a CFA charterholder. Nick and Gabriel discuss the investment case for Asian smaller companies and the evolving outlook for emerging markets. Drawing on more than 17 years of experience at Aberdeen, Gabriel explains why he believes the region offers attractive long-term growth opportunities. The conversation explores the drivers behind renewed investor interest in emerging markets, including diversification away from the US, the recovery in sentiment towards China, and the structural growth stories in India and Vietnam. Gabriel outlines Aberdeen Asia Focus plc's investment philosophy, emphasising high-quality businesses, strong governance, disciplined portfolio construction, and the importance of identifying under-researched companies with sustainable competitive advantages. Book Choices Range: Why Generalists Triumph in a Specialized World by David EpsteinGodfather of the Kremlin by Paul KlebnikovMusic ChoicesJamiroquai - Virtual InsanityAlmir Guineto - ConselhoThis content is issued by Zeus Capital Limited (“Zeus”) (Incorporated in England & Wales No. 4417845), which is authorised and regulated in the United Kingdom by the Financial Conduct Authority (“FCA”) for designated investment business, (Reg No. 224621) and is a member firm of the London Stock Exchange. This content is for information purposes only and neither the information contained, nor the opinions expressed within, constitute or are to be construed as an offer or a solicitation of an offer to buy or sell the securities or other instruments mentioned in it. Zeus shall not be liable for any direct or indirect damages, including lost profits arising in any way from the information contained in this material. This material is for the use of intended recipients only.

In this episode of the Grow A Small Business Podcast host Troy Trewin interviews Cheryl Contee, founder of BrightWorks AI, shares how she built a purpose-driven SaaS company serving nonprofits, scaled it to a 12-person team with multi-million-dollar revenue, and achieved a historic exit as the first Black female founder to sell a tech startup to a Nasdaq-listed company. Cheryl discusses the realities of fundraising, hiring, leadership, and planning for a successful exit while emphasizing the power of relationships and reputation. She also explains why she is now helping nonprofits adopt AI ethically through her latest venture. This conversation is packed with practical lessons on resilience, innovation, and building a business that creates lasting impact. Also check Cheryl Contee's book, AI for Nonprofits: Putting Artificial Intelligence to Work for Your Cause, and her appearance on the Built to Sell Radio podcast, where she shares how she built and successfully sold Attentive.ly, an AI-powered social listening platform acquired by Blackbaud. Why would you wait any longer to start living the lifestyle you signed up for? Balance your health, wealth, relationships and business growth. And focus your time and energy and make the most of this year. Let's get into it by clicking here. Troy delves into our guest's startup journey, their perception of success, industry reconsideration, and the pivotal stress point during business expansion. They discuss the joys of small business growth, vital entrepreneurial habits, and strategies for team building, encompassing wins, blunders, and invaluable advice. And a snapshot of the final five Grow A Small Business Questions:  What do you think is the hardest thing in growing a small business? Cheryl Contee shares that the hardest part of growing a small business is having the confidence to believe in yourself, especially when no one else believes in your vision. Cheryl Contee shares that entrepreneurs must be willing to put in 110% effort and embrace hard work every day. Cheryl Contee shares that self-belief is what keeps founders moving forward through uncertainty and setbacks. Cheryl Contee shares that lasting success comes from combining confidence with consistent dedication and perseverance. What's your favorite business book that has helped you the most? Cheryl Contee shares that her favorite business book is Who Moved My Cheese? because its lessons about adapting to change are even more relevant in today's fast-changing business world. Cheryl Contee shares that entrepreneurs must be willing to follow new opportunities instead of clinging to old ways of working. Cheryl Contee shares that embracing change with the right mindset helps business owners stay resilient and competitive. Cheryl Contee shares that continuous adaptation is essential for long-term business success. Are there any great podcasts or online learning resources you'd recommend to help grow a small business? Cheryl Contee shares that she highly recommends Apple News+ for access to trusted publications like The Wall Street Journal, Bloomberg, The Atlantic, and The Guardian, offering broad and in-depth business insights. Cheryl Contee shares that YouTube has also become one of her favorite learning platforms, especially for independent experts and international news sources. Cheryl Contee shares that exploring multiple perspectives helps business owners make better-informed decisions. Cheryl Contee shares that continuous learning from diverse, credible sources is essential for staying ahead in a rapidly changing world. What tool or resource would you recommend to grow a small business? Cheryl Contee shares that one of the best tools for growing a small business is a well-crafted pitch deck rather than a lengthy business plan. Cheryl Contee shares that a 10–15 slide pitch deck helps entrepreneurs clearly define their market, customers, competitors, business model, and growth strategy. Cheryl Contee shares that answering these core questions creates clarity and prepares business owners for investors, partners, and opportunities. Cheryl Contee shares that a strong pitch deck is a practical resource for building and scaling a successful business. What advice would you give yourself on day one of starting out in business? Cheryl Contee shares that if she could go back to day one, she would remind herself, "You've got this," and trust her ability to succeed. Cheryl Contee shares that embracing what makes you different allows you to see opportunities others may overlook. Cheryl Contee shares that being authentic and courageous opens the door to creating things that have never been done before. Cheryl Contee shares that believing in yourself from the very beginning is one of the greatest advantages an entrepreneur can have.  Book a 20-minute Growth Chat with Troy Trewin to see if you qualify for our upcoming course. Don't miss out on this opportunity to take your small business to new heights! Enjoyed the podcast? Please leave a review on iTunes or your preferred platform. Your feedback helps more small business owners discover our podcast and embark on their business growth journey.     Quotable quotes from our special Grow A Small Business podcast guest: It's okay to be different because different people see opportunities others miss - Cheryl Contee Your reputation is the most powerful marketing tool your business will ever have - Cheryl Contee Plan your exit from day one because successful businesses are built with the end in mind - Cheryl Contee    

BOGO is back and we are so excited to talk about this. Yes, we are talking all about this BOGO is such a fun way to save money, stock up and refill your stash, or even gift it to your oil loving friends.  Listed below is a few of the episodes from the past you can tune into and dive deeper into them at your leisure. Lavender  https://mcdn.podbean.com/mf/web/g4vhap/TheEssentialOilScoopEp7.mp3 Peppermint https://mcdn.podbean.com/mf/web/eu7f2cnaq967wvbf/Ep288peppermint.mp3 Helicrysum  Eucalyptus https://mcdn.podbean.com/mf/web/gnzxkbee2cdqdvzv/Ep188.mp3 Spearmint Coming out end of month Adaptiv  https://mcdn.podbean.com/mf/web/fzbvvxtt7pim8dxp/Ep175.mp3 Balance  Coming out this summer  Cheer  https://mcdn.podbean.com/mf/web/t4cb2qg7ehxvbstp/Ep102.mp3 Grapefruit   Frankincense Copaiba #doTERRA #doTERRABOGO #BOGOWeek #EssentialOils #Frankincense #Helichrysum #Copaiba #PeppermintOil #LavenderOil #Adaptiv #BalanceBlend #CheerBlend #NaturalWellness #HolisticHealth #EssentialOilEducation Welcome into our little essential oil world where we talk about the physical and emotional support of our essential oils.   Hi friends, don't forget to leave us a review, your feedback is always welcome, and helps this podcast reach more ears. Join us in our New Facebook Community! Connect on Instagram  Follow us on YouTube: https://www.youtube.com/@TheEssentialOilScoopPodcast   We upload a brand new episode every Tuesday and Thursday!   Want to learn more about us? theessentialoilscoop.com   Remember to like, share, and subscribe to our podcast so you will be notified every time we upload a brand new episode.  Leave us a review as well, your feedback is always welcome. Also opt-in to our newsletter at https://theessentialoilscoop.com/newsletter   If you have any questions or have subject ideas you would like us to cover please email us at theessentialoilscoop@gmail.com   Tag us on socials using #theessentialoilscoop #essentialoilpodcast #oilpodcast   Disclaimer:  Welcome to The Essential Oil Scoop Podcast. We want to remind our listeners that the information provided in this podcast is for educational purposes only and should not be considered as a substitute for professional medical advice, diagnosis, or treatment. The use of essential oils is a personal choice and should be done at your own risk. We are not medical professionals and cannot diagnose, treat, or prescribe any medical condition. Please consult with a qualified healthcare provider before using any essential oils or making changes to your healthcare routine. Any information or opinions expressed in this podcast are those of the hosts and guests and do not necessarily reflect the views of any particular organization. Thank you for listening.  

Promise hits Parkinson's disease therapy, as a Phase I/II clinical study has demonstrated the feasibility of transplanting stem-cell-derived dopamine progenitor cells into the brain. In preclinical research, a new platform uses the brain's fluid transport pathways to effectively deliver AAVs to therapeutic targets in mice. We'll also cover the accelerating infrastructure moment for AI-driven drug discovery, with billion-dollar investments flowing into end-to-end platforms driven by models and compute, rather than single drug assets. In business, Insilico's revenue leaps as the company's AI-developed lead candidate moves to Phase III, while Vertex acquires Crinetics for $10 billion. Join GEN editors Corinna Singleman, PhD, Alex Philippidis, Fay Lin, PhD, and Uduak Thomas for a discussion of the latest biotech and biopharma news.  Listed below are links to the GEN stories referenced in this episode of Touching Base:  Engineered AAVs Harness Glymphatic System to Reach Brain Targets in Mice GEN, July 9, 2026 Stem Cell Therapy Shows Promise in First Human Parkinson's Disease Trial GEN, July 9, 2026 Pharma Races to Scale AI as Billions Flow into Drug Discovery By Fay Lin, PhD, GEN Edge, July 6, 2026 StockWatch: Insilico Projects Profit, Revenue Leaps as AI-Developed Lead Candidate Moves to Phase III By Alex Philippidis, GEN Edge, July 12, 2026 Vertex Eyes Expansion Beyond Cystic Fibrosis with Planned $10B Crinetics Buyout By Alex Philippidis, GEN Edge, July 7, 2026 Top 20 Drugs Heading for the Patent Cliff, 2026-2029 By Alex Philippidis, GEN Magazine, Nov 1, 2025 Touching Base Podcast  Hosted by Corinna Singleman, PhD  Behind the Breakthroughs  Hosted by Jonathan D. Grinstein, PhD  The State of Biologics Testing 2026 Charles River Laboratories and GEN, June 10, 2026 Hosted on Acast. See acast.com/privacy for more information.

Baltimore and KC continue to have beef, Linebacker list has dropped and we react, and is Lamar Jackson listed too low on NFLs top 100? full 2735 Wed, 15 Jul 2026 01:46:43 +0000 WtNgL98Uv3NFAa3W42tEvNVL3uDcISlQ sports After Hours on 96.5 The Fan sports Baltimore and KC continue to have beef, Linebacker list has dropped and we react, and is Lamar Jackson listed too low on NFLs top 100? After Hours on 96.5 The Fan 2024 © 2022 Audacy, Inc.

Recent reports indicate the listed retirement sector in New Zealand has continued to struggle in 2026. Share market expectations were high in the new year, but the predicted growth has since turned flat and negative. Milford Asset Management expert Jeremy Hutton explained further. LISTEN ABOVESee omnystudio.com/listener for privacy information.

Recent reports indicate the listed retirement sector in New Zealand has continued to struggle in 2026. Share market expectations were high in the new year, but the predicted growth has since turned flat and negative. Milford Asset Management expert Jeremy Hutton explained further. LISTEN ABOVESee omnystudio.com/listener for privacy information.

-Here's your list: Vince Young (QB); Adrian Peterson (RB); Larry Fitzgerald, Michael Crabtree, Julio Jones (WR); Brock Bowers (TE);Bryant McKinnie, Joe Thomas (OT); Quentin Nelson, David DeCastro (OG); and Barrett Jones (C)-Defense: Terrell Suggs, Will Anderson Jr. (Edge); Ndamukong Suh, Aaron Donald (DT); Luke Kuechly, Micah Parsons (LB); PatrickPeterson, Patrick Surtain II (CB); Tyrann Mathieu (NB); Troy Polamalu, Ed Reed (S). What a damn list of studs.Advertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

This week Nick talks to Pedro Gonzalez de CosioPedro Gonzalez de Cosio is the Chief Executive Officer and co-founder of Pharmakon. Before co-founding the firm, he spent 14 years in structured finance, holding senior roles at Deutsche Bank and JPMorgan in New York. There, he specialized in structuring complex collateralized financing and derivative transactions for U.S. and international clients, including several years focused on the biotechnology and pharmaceutical sectors. Pedro is also the Investment Manager at BioPharma Credit PLC. Nick and Pedro discuss Pharmakon's approach to providing non-dilutive debt financing to life sciences companies, how the firm supports biotech businesses through key growth stages, and what differentiates its investment strategy. They also explore successful investments, the role of non-dilutive capital in funding innovation and acquisitions, the competitive landscape for biotech financing, differences between U.S. and European capital markets, and emerging healthcare trends, including orphan diseases, oncology, GLP-1 therapies, AI in drug discovery, and the future of cancer treatment. Pedro's Book ChoicesThe Godfather by Mario Puzo The Emperor of All Maladies by Siddhartha Mukherjee Pedro's Music ChoicesPiano Man by Billy JoelPagliacci OperaThis content is issued by Zeus Capital Limited (“Zeus”) (Incorporated in England & Wales No. 4417845), which is authorised and regulated in the United Kingdom by the Financial Conduct Authority (“FCA”) for designated investment business, (Reg No. 224621) and is a member firm of the London Stock Exchange. This content is for information purposes only and neither the information contained, nor the opinions expressed within, constitute or are to be construed as an offer or a solicitation of an offer to buy or sell the securities or other instruments mentioned in it. Zeus shall not be liable for any direct or indirect damages, including lost profits arising in any way from the information contained in this material. This material is for the use of intended recipients only.

One of the founding directors at Sesfikile Capital weighs in on the opportunities in advanced real estate markets like the US, UK and Europe, Asian hub Singapore and old South African investor favourite Australia, noting that global property is up around 12% year-to-date in US dollar terms. Podcast series on Moneyweb

➢ Check out our website www.colossusfitness.comIn this episode we go over 7 different ways to naturally increase your energy levels. There's no worse feeling than always feeling sluggish, tired, or just not in the right head space.These strategies and tips should help you feel energized.Having low energy is the absolute worst and there's no worse feeling.Listed points:1- Aim to do more of what inspires you“You often feel tired, not because you've done too much, but because you've done too little of what sparks a light in you.”2- Spend 15-60 minutes getting inspired each dayAudio booksPodcastsBooksMotivational videos3- Eat a balanced dietA lot of people don't realize, but a poor diet can substantially decrease energy levels.Ex- heavy fast food meal will weigh you down.Lighter, healthy meal will give you energy and leave you feeling greatProtein helps to stabilize blood sugar levels and provides a longer-lasting energy source. Include sources like lean meats, beans, nuts, and tofu.4- Get rid of the energy draining habitsExamples:Waking up and scrollingLaying in bed after the alarm goes offAlcoholSaying yes to everything and overcommitting5- Get Adequate Sleep/Manage StressAim for 7-9 hours of quality sleep each night.Stick to a consistent sleep schedule by going to bed and waking up at the same time every day.10 hours before bed: No more caffeine. 3 hours before bed: No more food or alcohol. 2 hours before bed: No more work. 1 hour before bed: No more screen time (shut off all phones, TVs and computers). 0: The number of times you hit snooze in the morning.6- Morning sunlightWakes up your brain, Improves sleep at night, Boosts daytime energy7- Get more movementThanks for listening! We genuinely appreciate every single one of you listening.➢Follow us on instagram @colossusfit➢Apply to get your Polished Physique: https://colossusfitness.com/

Featuring: Judah Thomas, David LaManna, Lenny Salgado, Johnny V., Ben Cossette, Mike McHugh, and James Gowell.Edited by: Tim NicholsonIn this episode of Thriving in the Word, we continue The Hall of Faith series and land on one of the most unlikely heroes in all of Scripture — a woman the world had already written off, living in a city God was about to bring down.We open in Hebrews 11:31, where Judah reads the actual text — just one verse. But behind that one verse is a story that rewrites everything you think you know about who God uses and why.James opens things up by painting a scene. Think Quentin Tarantino's Inglourious Basterds — that opening farmhouse interrogation. Hans Landa, the Jew Hunter, sitting across from a terrified man who knows exactly what's hidden beneath his floor. The pressure is unbearable. The fear is suffocating. And the man folds.Rahab didn't.Soldiers showed up at her door. The king's men. The most powerful city in the region behind them, and two foreign spies hiding on her roof. She had every reason to hand them over, every incentive to protect herself — and she looked the authorities dead in the eye and lied through her teeth for the God she was just beginning to believe in.That's not recklessness. That's raw, unpolished, courageous faith.We dig into Jericho — one of the most fortified cities in the ancient world, surrounded by a wall so massive that homes were literally built into it. Rahab's house was in that wall. Possibly an inn. A woman on the margins, living on the edge of everything, in more ways than one. And yet God sent His spies directly to her door. That wasn't coincidence. That was a divine appointment.From there, we explore one of the most staggering threads in all of Scripture — Rahab's place in the lineage of Jesus. A Gentile. A former prostitute. Listed by name in Matthew 1, honored in Hebrews 11, and woven into the bloodline of the Messiah. We talk through several angles on what that means, why it matters, and what it says about a God who doesn't just tolerate the imperfect — He chooses them.This episode asks the hard questions: What walls are you afraid of? What would it cost you to protect what God has placed in your hands? And are you willing to let your past be the very thing God uses to build something eternal?Topics include: Hall of Faith, Hebrews 11:31, Rahab, Joshua 2, faith under pressure, Jericho, walls of Jericho, Rahab and the spies, divine appointment, God uses the imperfect, Rahab in the lineage of Jesus, Matthew 1, genealogy of Jesus, Gentiles in the Bible, faith and fear, courage in Scripture, hiding the spies, women of faith in the Bible, scarlet cord, redemption, imperfect people God used, Inglourious Basterds, Hall of Faith series, Thriving in the Word, Thrive Church, Christian podcast, Bible teaching, Bible study on Rahab, Old Testament faith, faith in action, what is faith, spiritual formation, faith-based living, New Testament connections to the Old Testament.More Info: https://www.thrive.church Give: https://www.thrive.church/give/ Need prayer? prayer@thrive.churchThis is a presentation of Thrive.Church. © All Rights Reserved.

Seth and Luke discuss the Athletic listing the Texans as one of the teams with the biggest concerns, look at the other teams named, and why the Texans are on a higher level than those teams.

Sixtysomething Season 3, Episode 3 - Mark Died & I Don't Know What to DoIn this episode of Sixtysomething, your host, Grace Taylor Segal, shares this deeply personal story of recently losing her younger brother, Mark, at age 66—and the unexpected emotions that followed. Instead of overwhelming sadness, she found herself living with unanswered questions, complicated memories, unresolved feelings, and a surprising emotional numbness.This isn't an episode about having the perfect ending or finding easy closure. It's about the reality of loving someone with whom you've shared both joy and pain. It's about family relationships that never fit neatly into a box, the difficult conversations that never happened, and learning that grief isn't always what we expect.If you've ever lost someone with whom your relationship was complicated—or if you've wondered whether you're grieving "the right way"—this conversation is for you.Sometimes simply knowing you're not alone is enough.* * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * *In This EpisodeWhy grief doesn't always look the way we expectLosing a sibling after a lifetime of shared historyThe challenge of saying goodbye when relationships are complicatedWhy death doesn't magically erase old hurtsThe "loss inside the loss" when closure never comesFeeling numb instead of devastatedHow family members can mourn the same person in very different waysLearning to remember someone as their whole, imperfect selfFinding peace without getting all the answers* * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * *Hey Friends! It's me, Grace! I just want to thank you for listening. I hope you'll let me know what you think about the podcast and if any particular episodes resonate with you.Listed just below here is my contact information and all of the social channels where you can find me, as well as the link to our Facebook Group.Contact InfoGrace Taylor SegalEmail: grace@gracetaylorsegal.comFacebook: 60something Page(https://www.facebook.com/profile.php?id=61553062496332)Instagram: @60somethingpodFacebook Group: 60Something Podhttps://www.facebook.com/groups/1665326354000332RESOURCESThe poem "Death Is Nothing at All" by Henry Scott HollandCreditsSixtysomething Theme SongMusic & lyrics by Lizzy SanfordVocals by Lizzy SanfordGuitar: Lizzy & Coco SanfordTo Leave a Review: On Apple Podcasts (no link is available--here are the steps)Open the Apple Podcasts app.Search for the podcast you want to review.Select the podcast show page (not an individual episode).Scroll to the bottom of the page.Click "Write a Review".On the Sixtysomething Podcast Websitehttps://www.sixtysomething.net/reviews/new/Thank you so much for taking the time to review the show!Timestamps:00:00 Introduction: Why this episode was so hard to record01:00 My brother Mark's death and why grief surprised me03:00 Mark as a child: The brother I grew up with05:00 Our adult relationship and his health struggles09:00 The devastating diagnosis: Six months to two years to live11:00 The emergency call and rushing to Tennessee13:00 A miraculous improvement—and one last conversation16:00 "I want to tell you goodbye."19:00 Our complicated relationship23:00 The loss inside the loss: When there isn't closure25:00 Mark's final hours26:00 Waiting for grief that never came29:00 When grief doesn't look like grief30:00 Mourning someone different than everyone else knew33:00 How do you make peace with someone who's gone?34:00 Remembering the whole person—not just the good36:00 Finding peace after goodbye37:00 What I've learned about complicated grief39:00 "Death Is Nothing at All"41:00 Final thoughts and invitation to connect

Kristen, Asher, and Arden board the Culture Train Time Machine and travel to two extraordinary moments in history. First, they land on the Persian Royal Road in 400 BCE, where merchants and travelers are exchanging stories from distant lands - including eyewitness accounts of mysterious one-horned creatures from beyond the rivers of India. Then they leap forward to a Medieval European castle, where knights and nobles are marveling at what they believe is a real unicorn horn - not knowing it is actually the spiral tusk of a narwhal, carried thousands of miles by clever traders. Along the way, kids learn how stories travel, how imagination fills in the gaps, and how a legend is born when one curious mind sparks an idea and another passes it on. Fun Facts from This Episode The earliest known written description of a unicorn-like creature was recorded by the Greek historian Ctesias around 400 BCE Historians believe early unicorn stories may have been inspired by travelers describing a rhinoceros to people who had never seen one In Medieval Europe, narwhal tusks were sold as unicorn horns and were sometimes worth more than gold Traders carried narwhal tusks thousands of miles south from the Arctic, and most buyers had no idea what they really were Unicorn legends appear independently in ancient India, China, Persia, and Europe- showing how powerful a traveling story can be SUPPORT CULTURE KIDS If you enjoyed today's adventure, please subscribe, leave a review, and share Culture Kids with a family you love. Every share helps more children discover new places, meet new people, and grow curious about the world around them. Culture Kids Productions is a 3x Webby Award-winning nonprofit podcast dedicated to sparking curiosity, connection, and wonder in children around the world. Rated five stars by Common Sense Media. Listed as an Apple Podcasts Essentials pick for Kids and Family. Join our newsletter:https://culturekidsproductions.org Support our mission:https://culturekidsproductions.org Follow Culture Kids: Instagram:https://www.instagram.com/culturekidsproductions Credits: Host & Producer: Kristen Kim Co Host & Co Producer: Asher Kim and Arden Kim Script Review/Academic Consultant: Elisha Li Audio Engineering: Robin Lai Music & Sound FX: Envato Elements Merchant: Kristen Kim This episode is a work of fiction created for children's entertainment and educational enrichment purposes only. The characters, dialogue, and specific story events depicted are fictionalized and should not be relied upon as an accurate, complete, or authoritative account of any culture's traditions, beliefs, or history. Culture Kids Productions has made a good faith effort to represent the cultural and mythological source material respectfully, but listeners seeking factual or academic information about these traditions should consult qualified cultural experts, historians, or primary sources.

Link Up w/The Morning Sickness Digitally All Over:Instagram: @hms_98_official, @bosskupd, @bretvesely, @dickToledoX/Twitter: @HMSon98, @DickToledo, @bretveselyFacebook: @HMSKUPDYouTube: @hmspodcast9320, @98kupdRequest/Call in/Wakeup Song line:(IN AZ) 602.585.9800More HMS: holmbergpodcast.com, 98kupd.comEmail: dtoledo@98kupd.com, bvesely@98kupd.com, bbogen@98kupd.comSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Emmet Kennedy, Andy Newton, Jaime Wrenn and Jake Wilkes return with the Final Furlong Podcast Weekend Betting Guide, previewing the biggest betting opportunities from Sandown, Newmarket and Saratoga. The feature race is the Coral Eclipse, where Constitution River is a red-hot favourite to give Aidan O'Brien a 10th Eclipse victory and his 12th Group 1 success of the season.

Link Up w/The Morning Sickness Digitally All Over:Instagram: @hms_98_official, @bosskupd, @bretvesely, @dickToledoX/Twitter: @HMSon98, @DickToledo, @bretveselyFacebook: @HMSKUPDYouTube: @hmspodcast9320, @98kupdRequest/Call in/Wakeup Song line:(IN AZ) 602.585.9800More HMS: holmbergpodcast.com, 98kupd.comEmail: dtoledo@98kupd.com, bvesely@98kupd.com, bbogen@98kupd.comSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

This week Nick talks to Chris McVey. Chris McVey is Deputy Head of Octopus Quoted Companies at Octopus Investments, where he has worked since 2016 and is lead manager of the Octopus UK Multi Cap Income Fund. He has more than 20 years of experience in the UK quoted smaller companies market. Prior to joining Octopus, Chris was a UK small- and mid-cap equity research analyst at Citigroup, and before that spent seven years at Gartmore as an investment manager and analyst, building extensive expertise in UK smaller company investing. Nick and Chris discuss the significant opportunity in the UK small and mid-cap market, where valuations remain near historic lows despite strong underlying company fundamentals. Chris argues that quality growth businesses, particularly on AIM, are trading at substantial discounts to their intrinsic value due to a lack of investor flows rather than operational weakness. He believes the sector is poised for a re-rating as sentiment improves, creating an attractive entry point for long-term investors. Chris's Book Choice: Financial Times Guide to the Financial Markets by Glen ArnoldChris's Music Choice:The Best of Ludovico Einaudi playlistThis content is issued by Zeus Capital Limited (“Zeus”) (Incorporated in England & Wales No. 4417845), which is authorised and regulated in the United Kingdom by the Financial Conduct Authority (“FCA”) for designated investment business, (Reg No. 224621) and is a member firm of the London Stock Exchange. This content is for information purposes only and neither the information contained, nor the opinions expressed within, constitute or are to be construed as an offer or a solicitation of an offer to buy or sell the securities or other instruments mentioned in it. Zeus shall not be liable for any direct or indirect damages, including lost profits arising in any way from the information contained in this material. This material is for the use of intended recipients only.

-Here are the guarantees per CBS Sports' Brad Crawford: 1) Notre Dame embraces the villain role; 2) ACC flexes with second CFP entry; 3)Several SEC teams complain after exclusion; 4) Group of Six produces first-time CFP entrant; 5) LSU enjoys highest year-over-year increase inviewership-6) Heisman Winner comes from a top-four seed; 7) Texas Tech/BYU offers high December drama; 8) B1G wins a 4 th straight nationalchampionship; 9) Expect more calendar complaints; and 10) Only one unbeaten entering the playoffsAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

Daniel Lam addresses the underperformance of Hong Kong-listed China equitiesvis-à-vis the high-end technology growth markets such as the US, Japan, South Korea and Taiwan, highlighting the key catalysts that could trigger a turnaround.Speaker: - Daniel Lam, Head, Cross-Asset Derivative Strategy, Standard Chartered Bank For the latest market insights, visit our on-the-go Market Views or subscribe to Standard Chartered Wealth Insights on YouTube.

Fifty episodes in and we are still pinching ourselves that we get to do this. For the Season 7 finale we sat down with Katerina Giannouka, a passionate hotelier and one of the most well travelled people we know and she took us on a journey that left us completely speechless. Japan, Mongolia, Norway, northern Greece and so much more.   Episode Highlights & Destination Gems: 1. Kumano Kodo Trail, Japan - A Pilgrimage That Will Change You • A trail walked by Emperors, Samurai and villagers for over a thousand years. Katerina says it is unlike anything else on earth. • One of only two UNESCO World Heritage trails in the world, the other being Santiago de Compostela in Spain • Begins in Mount Koya, a mountain village with over 100 stone temples where you stay as a guest of the monks and join morning prayers • Ryokans along the way owned by local families who cook kaiseki meals from their own gardens and rivers • Accessible for all levels with routes ranging from three to seven days • No plastics, no crowds and a deep connection to nature   https://www.tb-kumano.jp/en/kumano-kodo/ 2. Cape Town, South Africa - The One Destination Everyone Must Visit Katerina's go-to destination at every phase of life and a place she says has something for everyone. • A food scene that is vibrant, diverse and constantly evolving • The Oranjezicht market bringing together flavours from across the world in one extraordinary space • A city with cross cultural roots, youthfulness and a community of people actively giving back 3. Genghis Khan Retreat, Mongolia - Off the Map and Utterly Unforgettable • One of the most remote and extraordinary travel experiences on the planet. • Located in the Orkhon Valley • A tented retreat built by a Mongolian family that packs up completely each winter and leaves no trace • No phone signal, no roads and no civilization within reach • Horseback riding, polo, kayaking, mountain picnics and nights that go on under an endless sky https://genghiskhanretreat.com/ 4. Hong Kong - The City That Shapes You • A city that makes you live with presence because you know from the moment you arrive that you will not be there forever • Seventy five percent of the territory is protected natural land with pristine beaches that take hours to reach on foot • Trail running, hiking and coastlines that feel like Thailand and yet are entirely Hong Kong • A city where everyone is up to something, standing for something and constantly in motion 5. Al Khalili Market, Cairo - Solo Travel That Surprises You Katerina went against all advice as a solo woman traveller and found one of her warmest travel memories. • A market that has been part of Cairo's ecosystem for thousands of years • Welcoming, gracious and full of warmth for anyone willing to arrive with an open mind • A reminder that the places we fear most often have the most to give 6. Vikos Gorge, Northern Greece - The Deepest Canyon in the World Katerina's pick for the most underrated destination on earth and a place that left her completely speechless. • Listed in the Guinness Book of Records as the deepest canyon in the world • A five to six hour hike along the canyon floor starting from a stone village at the top of a mountain • At the end of the trail, the source of a river born from freezing water bubbling straight out of rock forming crystal blue pools at four degrees • Well marked, accessible and world class in every sense • Northern Greece deserves far more recognition than it currently receives 7. Sydney, Australia - The Best Food City in the World • Produce so exceptional and so fresh that very little needs to be done to it in the kitchen • From Michelin starred restaurants to a breakfast bowl at Bondi Beach, the quality never drops • Soil that has not been overused and land mass that allows ingredients to be what they are meant to be 8. Haukasen Lodge, Norway - The Hidden Gem • A hunting lodge in the Norwegian fjords that Katerina was almost reluctant to share. • Reachable via Oslo and a one and a half hour flight northwest to Sogndal • Open only four or five months a year and booked many years in advance • A sauna overlooking the fjord, glacier hikes, kayaking, ice bathing and evenings by the fireplace • Slow luxury with no agenda, no performance and nothing between you and nature https://www.haukasenlodge.no/ 9. Socotra, Yemen - Bucket List • Blood dragon trees and Jurassic Park landscapes found nowhere else on earth • Thirty seven percent of species are endemic meaning they exist only here • Pristine beaches, extraordinary nature and visitor numbers still tightly controlled • A destination that remains genuinely untouched 10. Oman - Bucket List • Some of the best diving in the world • Close enough from Dubai to make it completely accessible and yet still deeply under visited Connect with Katerina Giannouka: https://www.linkedin.com/in/katerinaqod/ Thank you for tuning in to Travel Stories with Moush! If you loved this episode, please hit subscribe on your favorite podcast platform and leave us a rating or review - it truly helps us reach more travelers like you.   Drop a comment and tell us which destination from today's episode is going straight to your bucket list? Stay connected with me on https://www.instagram.com/moushtravels/ to find out who's joining me next week.   Explore all past episodes and destinations here: https://podcasts.apple.com/ae/podcast/travel-stories-with-moush/id1691525895 https://open.spotify.com/show/1pAUXiXuRLv1E9WFznWm7T?si=qA_E3Cf8RqKT97pUJcINxQ https://www.youtube.com/@travelstorieswithmoush Until next time…safe travels and keep adventuring. Connect with me on the following: Instagram @moushtravels Facebook @travelstorieswithmoush LinkedIn @Moushumi Bhuyan You Tube @travelstorieswithmoush "Want a spotlight on our show? Visit https://admanager.fm/client/podcasts/moushtravels and align your brand with our audience."Connect with me on the following:Instagram @moushtravelsFacebook @travelstorieswithmoushLinkedIn @Moushumi BhuyanYou Tube @travelstorieswithmoush Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Last week, while attending a social media conference in Lima, Ohio, I heard a speaker recommend something I had never seriously considered before: Submit your podcast to IMDb. The idea was that IMDb has become a highly trusted source that AI tools and search engines often reference when gathering information about people. If that's true, having an accurate IMDb profile and podcast listing could influence what people learn about you when they search your name. That immediately made me curious. So instead of speculating, I decided to run an experiment. In this episode, I share what it was actually like to submit one of my podcasts to IMDb, the surprisingly convoluted process I encountered, how AI helped me navigate it, and whether I believe the investment of time is actually worthwhile. More importantly, I explore the bigger questions: Does an IMDb listing actually increase your authority? Will it help people discover your podcast? Who is most likely to benefit from being listed? Is this a strategic move, or simply another shiny object? By the end of this conversation, I have a much clearer perspective on where an IMDb listing might fit into a podcaster's overall strategy and where it probably doesn't. If you've ever wondered whether your podcast belongs on IMDb, or you're curious how AI is changing the way people discover information about us online, I think you'll enjoy this episode. Momentum Mastermind Also, if you are on the entrepreneurial journey and you are tired of trying to figure everything out on your own, I invite you to check out Next Level Mastermind: Momentum. This is a weekly mastermind experience for entrepreneurs who want a room where they can tell the truth about where they are, get clear on the next aligned step, and build real momentum with the support of other business owners who understand the decisions, opportunities, and challenges they are facing. You can learn more at: https://cliffravenscraft.com/momentum

Emmet Kennedy, Andy Newton, George Gorman and Jaime Wrenn return with the Final Furlong Podcast Weekend Betting Guide, previewing the biggest betting opportunities from a superb Saturday featuring two Group 1s at the Curragh, competitive handicaps at York and Newcastle, and the prestigious Northumberland Plate. After a profitable Royal Ascot, the team return with confident NAPs, major handicap angles and several double-figure selections, including a 14/1 NAP, 12/1, 8/1 and 5/1 betting plays.

On this episode of Animal Spirits: Talk Your Book, ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Michael Batnick⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ and ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Ben Carlson⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ are joined by Dr. Ankur Crawford from Alger to discuss: what everyone is getting wrong about the AI trade, why this technology is different, letting the market tell you if you're wrong or right, and how to manage a concentrated portfolio of stocks.  Find complete show notes on our blogs... Ben Carlson's ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠A Wealth of Common Sense⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Michael Batnick's ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠The Irrelevant Investor⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Feel free to shoot us an email at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠animalspirits@thecompoundnews.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ with any feedback, questions, recommendations, or ideas for future topics of conversation. Check out the latest in financial blogger fashion at The Compound shop: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://idontshop.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Investing involves the risk of loss. This podcast is for informational purposes only and should not be or regarded as personalized investment advice or relied upon for investment decisions. Michael Batnick and Ben Carlson are employees of Ritholtz Wealth Management and may maintain positions in the securities discussed in this video. All opinions expressed by them are solely their own opinion and do not reflect the opinion of Ritholtz Wealth Management. See our disclosures here: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://ritholtzwealth.com/podcast-youtube-disclosures/⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ The Compound Media, Incorporated, an affiliate of ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Ritholtz Wealth Management⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠, receives payment from various entities for advertisements in affiliated podcasts, blogs and emails. Inclusion of such advertisements does not constitute or imply endorsement, sponsorship or recommendation thereof, or any affiliation therewith, by the Content Creator or by Ritholtz Wealth Management or any of its employees. For additional advertisement disclaimers see here ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://ritholtzwealth.com/advertising-disclaimers⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠. Alger Disclaimer: The views expressed are the views of Fred Alger Management, LLC (FAM) and its affiliates as of June 2026. This material is not meant to provide investment advice and should not be considered a recommendation to purchase or sell securities. Holdings are subject to change. Risk Disclosures: Investing in the stock market involves risks, including the potential loss of principal. Growth stocks may be more volatile than other stocks as their prices tend to be higher in relation to their companies' earnings and may be more sensitive to market, political, and economic developments. The following represented the noted percentage of CNEQ assets as of 3/31/26: Micron 0%; Astera Labs 2.0%; Nebius 5.2%; Anthropic 4.4%; TSMC 5.8%; Lam Research 0%; GE Vernova 3%; Nvidia 14.9%; QXO 4%; Heico 2.2%. Before investing, carefully consider the Fund's investment objective, risks, charges, and expenses. For a prospectus and summary prospectus containing this and other information or for the Fund's most recent month-end performance data, visit www.alger.com, call (800) 223-3810 or consult your financial advisor. Read the prospectus and summary prospectus carefully before investing. Distributor: Fred Alger & Company, LLC. Listed on NYSE Arca, Inc. NOT FDIC INSURED. NOT BANK GUARANTEED. MAY LOSE VALUE. Learn more about your ad choices. Visit megaphone.fm/adchoices

Listed on the National Register of Historic places, the Wyoming Territorial Prison is an imposing stone structure built in 1872  and restored in 1990. Visitors usually take a self-guided tour of the prison grounds, but during the summer season a limited number of drop-in, guided tours are available. Visit the prison Broom Factory to learn about the history of the buliding and how prisoners made brooms.After the prison closed, the Site spent most of its life as an experimental stock farm station for the University of Wyoming. Visit the historic horse barn to see the exhibit, "Science on the Range" and learn more. If you are curious about the spider silk producing goats that Loren mentioned follow these links to learn more: https://digitalcommons.usu.edu/student_showcase/11/ https://www.discoveryscientificsolutions.com/item/31

Listed as a British “wrong-com,” the first episode of Alice and Steve covers the inciting incident that sets up the premise of this six-episode series. You probably already know Jemaine Clement and Nicola Walker, just like we knew Seth Rogen and Rose Byrne going into Platonic. The difference is that this one leans confidently British, with a plot that veers more toward sharp awkwardness than anything particularly heartwarming.Created by Sex Education writer Sophie Goodhart, this new Hulu/Disney+ series arrives right around midyear, just as we're starting to get a good impression of what 2026 TV is all about. On the podcast, we run down the episode, ask the big questions, make comparisons, discuss the reception, and, of course, give our rating.Tune in and enjoy. Welcome to Today's Episode.

CHECK OUT OUR WEBSITE FAT LOSS GUIDE- www.colossusfitness.com1- Eat MindfullyPay attention to what and how much you're eating. Avoid distractions like watching TV or using your phone while eating. Chew your food slowly and savor each bite.2- EAT BETTER FOOD!!!Prioritize protein, eat enough fibre & drink lots of water3- Understand the difference between tolerable and intolerable hungerTolerable Hunger: You're hungry, but still in control. You can focus, function normally, and wait until your next meal.Intolerable Hunger: Hunger is consuming your thoughts. You're irritable, distracted, and likely to overeat when food becomes available.4- Get rid of the liquid/empty/garbage caloriesFill up with more low calorie5- Have regular consistent meals & be in a reasonable deficitAs Huberman says, “eat like an adult.”Eating regular, balanced meals and snacks can help stabilize blood sugar levels and prevent extreme hunger, which can lead to overeating.6- Manage Stress & Get Enough SleepLack of sleep can disrupt hunger hormones, leading to increased appetite. Aim for 7-9 hours of quality sleep per night.Listed points:Thanks for listening! We genuinely appreciate every single one of you listening.➢Follow us on instagram @colossusfit➢Apply to get your Polished Physique: https://colossusfitness.com/

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Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

Sixtysomething_S3_Ep2 – Meet CanvaHave you heard people talking about Canva but aren't quite sure what it is—or why so many people love it?In this episode of Sixtysometing, your host, Grace Taylor Segal, shares introduces listeners to Canva, the free online design platform that has transformed the way millions of people create everything from greeting cards and photo books to business materials, social media graphics, family cookbooks, presentations, and legacy projects.But this episode isn't really about software. It's about creativity. It's about having access to tools that simply didn't exist for ordinary people for most of our lives.Grace shares her own journey from teaching herself Photoshop with a bootleg copy and learning design from books, to creating magazines, online courses, podcast graphics, legacy projects, and more. She explains why she believes Canva has “democratized design” and why that's such an exciting development for people in our stage of life.You'll learn:• What Canva is and how it works• The inspiring story behind Canva founder Melanie Perkins• Why Canva changed the design world forever• What you can create with Canva• What's included in the free version• Whether Canva Pro is worth considering• Favorite Canva features like Background Remover, Magic Resize, Magic Write, and AI Image Generation• How Canva can help with family history, memory books, photo collections, cookbooks, travel journals, and other legacy projects• Why Canva is especially valuable for people over 60• How creativity and learning don't have an expiration dateMost importantly, you'll be encouraged to think about what you've always wanted to create—and what might finally be possible now.☀️ ☀️ ☀️Canva ResourcesWant to learn more about Canva?I've included links to several beginner-friendly Canva tutorials as well as Canva Design School, which offers free courses, tutorials, and certifications.Whether you're a complete beginner or just curious about what's possible, these resources are a great place to start.Canvahttps://www.canva.comCanva Tutorial by Canvahttps://youtu.be/V9LtRF6EbyY?si=FDRCnNUYxeC8-vEfIn this video we'll show you the basics of Canva and how easy it makes design. This Canva for Beginners video series is here to show you how to get started with Canva and bring your ideas to life.With limitless potential for customization and templates, it's simple to make designs your own. Once you learn how to navigate around the editor, you'll be designing like a pro in no time.

Leftism has reached the point where it cannot define itself by accomplishment, prosperity, security, freedom, patriotism, family, or even happiness. The only way to understand modern Leftism is to chronicle what it promotes. And once you make that list, you begin wondering whether you're reading a political platform or the side effects section of a pharmaceutical product.The deeper problem isn't any individual policy. It's that modern Leftism increasingly defines virtue as the inversion of common sense. If a border works, open them. If standards work, lower them. If merit works, reward mediocrity. If a tradition survives, dismantle it. If people succeed, punish them, then take credit for their achievements. Eventually the movement stops building things and becomes a demolition crew searching for its next wall to demolish.So what are our examples? Let's look at the SPLCWe have the SPLC PAYING FOR RACISM. A blind man can see the irony and hypocrisy of this on Jupiter and without a telescope.Nothing says ‘we defeated the racism' like requesting a W-9” from the white supremacist you just hired.What a story. Allegations reported in a lawsuit and summarized in reporting by Just the News show that when they SPLC was approached by a former white nationalist who wanted out of the movement, they hired him. The dude declared that the SPLC had won.Dude went to the KKK and declared, “I'm done. I would like to return this ideology. Please process my refund.”See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Become a Spotify Subscriber and gain access to a 30 minute bonus episode every single week!Thanks to Dragon Shield for being an official sponsor of the Uncommon Energy Podcast! They make the best sleeves, accessories, and gaming products in the entire industry. Get 5% off your order by using code "UEPOD" online at: https://dragonshield.com/?ref=uncommonenergy

Internet sleuths just pulled permits for a brand new building in the Dark Universe portal at Epic Universe. Building 1570. Area 680. Listed as a special project. And the building numbers line up with exactly the kind of show building you'd need for a new attraction. Mike and EB have a theory about what it is — and it involves a glass bottom boat, the Creature from the Black Lagoon, and an idea that Coaster Radio listeners actually designed in a Real Design Contest years ago. If Universal puts a Coaster Radio t-shirt on one of the bodies in that ride, that's all the confirmation the show needs. Before the speculation though — Hagrid's Magical Creatures Motorbike Adventure has been removed from Universal Express. The longest line at Epic Universe is now a standby-only experience, and the people paying for Express Pass are not happy about it. Nate Bargatze wants to build a theme park based on his own life in Nashville and EB has thoughts. And RMC's Wild Moose prototype track has appeared at their Idaho headquarters, with the most interesting theory being that it could go indoors at Kentucky Kingdom in the space that used to hold a Schwarzkopf coaster. Then in the second half — the interactive question that started with Verbolten's new overlay at Busch Gardens Williamsburg produces one of the best listener response segments of the season. Rides that could use a refresh. Listeners suggest Journey to Atlantis, Men in Black Alien Attack, Dinosaur at Animal Kingdom, Flight of Fear, Big Thunder Mountain, the Haunted Mansion, Test Track, and the entire frontier area at Cedar Point. Mike and EB debate each one — and somewhere in the Men in Black discussion, EB invents what might be the greatest unused attraction concept in Universal history. Also — EB is going to Kentucky Kingdom on June 10th, there are collectible pins being considered, and the show closes with a detailed discussion of the 1997 horror film Wishmaster that you absolutely did not see coming. Stay through the Patreon section. You'll understand why.

Sixtysomething_S3_Episode 1 – There's No Place Like Home…Except Maybe Las VegasIn this episode of Sixtysometing, I'm sharing what's ahead for Season 3 of Sixtysomething—and why I believe this may be our most meaningful season yet.This year, we're diving into heartfelt conversations about friendship, family, creativity, reinvention, adult children, grandparenting, loneliness, marriage, purpose, and the emotional realities of this stage of life. We'll also be talking about practical and inspiring topics like starting businesses after 60, creating meaningful legacy projects, preserving family stories and photos, and finding new ways to express ourselves creatively through writing, collage, journaling, podcasting, and more.We'll explore everything from AI artwork and storytelling to the bigger emotional and ethical questions many of us are asking about technology and the future.I'll also be introducing a brand-new Sixtysomething Book Club, revisiting some favorite topics from Seasons 1 and 2, and sharing more personal reflections and listener-centered conversations along the way.Most of all, this season is about staying curious, connected, creative, hopeful, and engaged with life—because there is still so much ahead of us.I'm so glad you're here for Season 3.You'll Hear in This EpisodeWhat inspired the direction of Season 3New themes and topics coming this yearWhy creativity matters after 60Conversations about adult children, friendship, and connectionHow AI can help with creativity and everyday lifeThe new Sixtysomething Book ClubRevisiting some favorite topics from earlier seasonsWhy reinvention doesn't end at a certain age* * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * * *Hey Friends! It's me, Grace! I just want to thank you for listening. I hope you'll let me know what you think about the podcast and if any particular episodes resonate with you.Listed just below here is my contact information and all of the social channels where you can find me, as well as the link to our Facebook Group. Contact InfoGrace Taylor SegalEmail: grace@gracetaylorsegal.comFacebook: 60something Page(https://www.facebook.com/profile.php?id=61553062496332)Instagram: @60somethingpodFacebook Group: 60Something Podhttps://www.facebook.com/groups/1665326354000332RESOURCES6LINKSSixtysomething Podcast - Episode - - NOTEShttps://drive.google.com/file/d/1kfJIQ6uvPYy2zkjoQE7yT9g6eDZa7FEO/view?usp=sharingCreditsSixtysomething Theme SongMusic & lyrics by Lizzy SanfordVocals by Lizzy SanfordGuitar: Lizzy & Coco SanfordTo Leave a Review: On Apple Podcasts (no link is available--here are the steps)Open the Apple Podcasts app.Search for the podcast you want to review.Select the podcast show page (not an individual episode).Scroll to the bottom of the page.Click "Write a Review".On the Sixtysomething Podcast Websitehttps://www.sixtysomething.net/reviews/new/Thank you so much for taking the time to review the show!Timestamps:00:00 Welcome and Introduction03:15 What's Ahead in Season 308:40 Friendship, Family & Connection14:10 Creativity, Legacy & Reinvention20:05 AI, Technology & Staying Curious26:40 Introducing the Sixtysomething Book Club29:15 Final Thoughts & Looking Ahead

Welcome to the Wholesale Hotline Podcast Weekend Edition (Flipping Mastery Edition), where Jerry teaches how to master the art of house flipping, wholesaling, and new construction development.Show notes -- in this episode we'll cover:Straightforward, step-by-step training on making six and seven figures from real estate deals.Insider tactics for finding motivated sellers, analyzing deals, and raising private money.Learn how to flip houses virtually from anywhere—even with zero experience.Whether you're a beginner or scaling up, Jerry gives you the blueprint to build real wealth through real estate.  Please give us a rating and let us know how we are doing!➖➖➖➖➖➖➖➖➖➖➖➖➖➖➖ ☎️ Welcome to Wholesale Hotline & Flipping Mastery Breakout! ☎️Jerry Norton went from digging holes for minimum wage in his mid 20's to becoming a millionaire by the age of 30. Today he's the nation's leading expert on flipping houses and has taught thousands of people how to live their dream lifestyle through real estate. **NOTE: To Download any of Jerry's FREE training, tools, or resources…Click on the link provided and enter your email. The download is automatically emailed to you. If you don't see it, check your junk/spam folder, in case your email provider put it there. If you still don't see it, contact our support at: support@flippingmastery.com or 888) 958-3028. ➖➖➖➖➖➖➖➖➖➖➖➖➖➖➖ 

Link Up w/The Morning Sickness Digitally All Over:Instagram: @hms_98_official, @bosskupd, @bretvesely, @dickToledoX/Twitter: @HMSon98, @DickToledo, @bretveselyFacebook: @HMSKUPDYouTube: @hmspodcast9320, @98kupdRequest/Call in/Wakeup Song line:(IN AZ) 602.585.9800More HMS: holmbergpodcast.com, 98kupd.comEmail: dtoledo@98kupd.com, bvesely@98kupd.com, bbogen@98kupd.comSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Matt O'Leary discusses the New York Jets head coach being listed as a big winner after the 2026 schedule dropped Sign up for PrizePicks with code: HMA and get $50 in lineups instantly when you play your first $5+ lineup! https://link.prizepicks.com/LME0/OLEARY Find LUCY near you at lucy.co/stores, or save 20% on your first online order at lucy.co/HMA with promo code HMA. Play in my Free to Play Win $100 on PeopleGuess: https://www.peopleguess.com/ All videos now available in Podcast Form: Apple : http://bit.ly/3V7KcNQ Spotify : https://bit.ly/4mc8bX7 le Chrome Extension: https://www.swvle.com/r/G7cAw5VyQ1tv Subscribe to the Talkin Jets Channel: https://www.youtube.com/@TalkinJets/ For Serious Inquiries: mattoleary95@gmail.com MattOLearyNY Merch Shop: https://mattolearyny.com Social: Twitter: https://twitter.com/mattolearyny TikTok: https://www.tiktok.com/@mattolearyny?is_from_webapp=1&sender_device=pc Instagram: https://instagram.com/mattolearyny ​ Call in and leave a voicemail: ‪(631) 517-0756‬ JUST JETS PODCAST: https://linktr.ee/justjets MUSIC - Saint Saxon Sound Lia Saint Saxon - Lofi Hip Hop Beats to Relax/Study to https://www.youtube.com/watch?v=9uuBuZy0pxE&t=2314s Saint Saxon Sound Youtube Channel: https://bit.ly/3CWknIA Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Link Up w/The Morning Sickness Digitally All Over:Instagram: @hms_98_official, @bosskupd, @bretvesely, @dickToledoX/Twitter: @HMSon98, @DickToledo, @bretveselyFacebook: @HMSKUPDYouTube: @hmspodcast9320, @98kupdRequest/Call in/Wakeup Song line:(IN AZ) 602.585.9800More HMS: holmbergpodcast.com, 98kupd.comEmail: dtoledo@98kupd.com, bvesely@98kupd.com, bbogen@98kupd.comSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

-Staples wrote this weekend these 10 storylines, in order, that are most meaningful: 1) The Legend of Arch Manning; 2) Lane Kiffin'sFirst Year at LSU; 3) Is Mike Norvell in his last year at FSU?; 4) Lincoln Riley's “window;” 5) 5-star QBs taking the SEC by storm-6) Young coach takeover in the SEC; 7) Change of the guard in the B1G?; 8) What happened to Clemson?; 9) Texas Tech's BrendanSorsby situation; and 10) Deion Sanders and Colorado's next moveOur Sponsors:* Check out Hims: https://hims.com/EARLYBREAKAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

-Staples wrote this weekend these 10 storylines, in order, that are most meaningful: 1) The Legend of Arch Manning; 2) Lane Kiffin'sFirst Year at LSU; 3) Is Mike Norvell in his last year at FSU?; 4) Lincoln Riley's “window;” 5) 5-star QBs taking the SEC by storm-6) Young coach takeover in the SEC; 7) Change of the guard in the B1G?; 8) What happened to Clemson?; 9) Texas Tech's BrendanSorsby situation; and 10) Deion Sanders and Colorado's next moveOur Sponsors:* Check out Hims: https://hims.com/EARLYBREAKAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

-Nebraska will end up probably between 8 th to 11 th on almost every list you see for the B1G in power rankings, so 8 th is nice to see fromESPN, who basically said Nebraska has had good starts under Matt Rhule—but November has been a problem and needs to be fixed-The real story is who is at No. 4---OHIO STATE!!—behind Indiana, Oregon, and USC. Now THAT would be a story if they fell thatfar…does The Stool see that happening? Between Ohio State and Nebraska is Penn State, Michigan, and Iowa…Our Sponsors:* Check out Hims: https://hims.com/EARLYBREAKAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

NBA duos tier list! #nba   Check out the TD3 merch: https://the-deep-3-shop.fourthwall.com/   Listen on Spotify!: https://open.spotify.com/show/3elbbqVumwqz8wlIdknsLW   Listen on Apple Podcasts!: https://podcasts.apple.com/us/podcast/the-deep-3-podcast/id1657940794   Follow us on TikTok!: https://www.tiktok.com/@thedeepthree   Follow us on Instagram!: https://www.instagram.com/thedeep3podcast/   Isaac's twitter: https://twitter.com/byisaacg   Mo's twitter: https://twitter.com/Mojo99_   Donnavan's twitter: https://twitter.com/Dsmoot3D Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.