Podcasts about tests

In episode 2 of the Tim G miniseries, Tim dives into a raw and intense firsthand account of life inside one of Texas' most violent prison units. He breaks down what it was like arriving young, white, and unprotected in a heavily segregated environment where respect had to be earned fast. He talks about the politics between prison gangs, corrupt officers, contraband hustles, riots, extortion, violence, and the daily survival mindset required to make it through. From guards being flipped for drops, to inmates running serious money operations behind the walls, this conversation exposes how chaotic and dangerous Texas prison life could really get. Tim also reflects on how older inmates taught him discipline, street wisdom, and the importance of carrying himself with respect — lessons that shaped the way he moved both inside and after prison. A brutal, unfiltered look at prison politics, survival, and the hidden economy behind the walls. Topics covered: Texas prison violence, prison gangs, Bloods and Crips, Mexican Mafia, Texas Syndicate, contraband, corrupt guards, prison riots, extortion, survival, redemption, and life after incarceration. Go Support Tim! IG: https://www.instagram.com/tattoos_bykriminalkustoms/ Join The Patreon For Bonus Content! https://www.patreon.com/theconnectshow 00:00 Intro & Life in the Most Violent Prisons 01:14 Terrell Unit: The Worst Prison in Texas 02:57 Death Row, Security Changes & Contraband 07:06 Prison Hustles & Black Market Economy 12:17 Adapting, Survival & Earning Respect 18:29 Race, Gangs & Violence Inside 21:15 This Episode Is Sponsored By Shopify 23:13 Respect, Snitching, and Survival Rules 32:38 Corrupt Guards, Prison Parties, and Lessons from Older Inmates 42:47 Riot Stories & Texas Prison Politics 44:30 This Episode Is Sponsored By Superpower 46:13 Racial Conflict and Survival Fights 54:42 Initiations, Tests & Early Survival 01:10:24 Prison Hierarchy, Hits & Corrupt Guards 01:20:00 Violence on the Inside: Stabbings & Fights 01:34:08 Race, Respect, and Code of Silence 01:46:03 Bloods, Politics, and Earning Your Place 02:00:04 Endurance, Anxiety & Becoming Hardened 02:14:43 Self-Reflection & Emotional Toll 02:16:49 Wrap-Up & Outro Learn more about your ad choices. Visit podcastchoices.com/adchoices

Unser Showpartner ist JOIN, die KI-basierte Trainings-App für Radfahrer:innen. Trainiere die ersten 30 Tage kostenlos, ohne dabei schon Zahlungsdaten hinterlegen zu müssen. https://join.cc/campaigns/faszinationrennrad. Du willst ROADBIKE zwei Monate gratis lesen? Dann sichere dir dein Probe-Abo unter: https://www.roadbike.de/podcast-abo ROADBIKE ist Faszination Rennrad! Tests, News, Tipps, Interviews, Reise- und Szene-Reportagen rund um deine Leidenschaft Rennradfahren findest du bei uns auf allen Kanälen. Monatlich erscheint eine neue gedruckte ROADBIKE-Ausgabe - erhältlich am Kiosk, digital als ePaper oder ganz praktisch als Abonnement. https://shop.motorpresse.de/zeitschriften/sport-freizeit/roadbike/abo-print.html Auch mit unserer Website https://www.bike-x.de/rennrad/ oder über unsere Social Media-Kanäle Instagram und Facebook sowie YouTube bleibst du stets auf dem Laufenden. Die wichtigsten Nachrichten aus der bunten Rennradwelt fasst einmal wöchentlich unser Newsletter zusammen - hier abonnieren: https://www.bike-x.de/newsletter/?mtm_campaign=newsletter-anmeldung-podcast&mtm_medium=podcast Du interessierst Dich auch für andere Facetten des Radfahrens und Outdoor-Lebens? Dann hör doch mal in unsere anderen Podcasts rein: - Alles ist fahrbar – der MOUNTAINBIKE-Podcast https://www.bike-x.de/mtb/ratgeber/mtb-podcast-alles-ist-fahrbar-sprechstunde-mit-der-mountainbike-redaktion/?mtm_campaign=newsletter-anmeldung-podcast&mtm_medium=podcast - Hauptsache raus – der OUTDOOR-Podcast https://www.outdoor-magazin.com/wandertipps/hauptsache-raus-jetzt-den-neuen-podcast-anhoeren/?mtm_campaign=newsletter-anmeldung-podcast&mtm_medium=podcast

Max Riese spricht über seinen Sieg bei dem Bikepacking-Race durch Griechenland Unser Showpartner ist JOIN, die KI-basierte Trainings-App für Radfahrer:innen. Trainiere die ersten 30 Tage kostenlos, ohne dabei schon Zahlungsdaten hinterlegen zu müssen. https://join.cc/campaigns/faszinationrennrad. Du willst ROADBIKE zwei Monate gratis lesen? Dann sichere dir dein Probe-Abo unter: https://www.roadbike.de/podcast-abo ROADBIKE ist Faszination Rennrad! Tests, News, Tipps, Interviews, Reise- und Szene-Reportagen rund um deine Leidenschaft Rennradfahren findest du bei uns auf allen Kanälen. Monatlich erscheint eine neue gedruckte ROADBIKE-Ausgabe - erhältlich am Kiosk, digital als ePaper oder ganz praktisch als Abonnement. https://shop.motorpresse.de/zeitschriften/sport-freizeit/roadbike/abo-print.html Auch mit unserer Website https://www.bike-x.de/rennrad/ oder über ... Dieser Podcast wird vermarktet von der Podcastbude.www.podcastbu.de - Full-Service-Podcast-Agentur - Konzeption, Produktion, Vermarktung, Distribution und Hosting.Du möchtest deinen Podcast auch kostenlos hosten und damit Geld verdienen?Dann schaue auf www.kostenlos-hosten.de und informiere dich.Dort erhältst du alle Informationen zu unseren kostenlosen Podcast-Hosting-Angeboten. kostenlos-hosten.de ist ein Produkt der Podcastbude.

Bob Zimmerman discusses the crew selection for NASA's Artemis 3 mission, which has been simplified to focus on Earth-orbit docking tests. He also examines private sector developments, including German startup Isar's funding, Stoke Space's reusable rocket design, and an orbital servicing mission by Catalyst intended to rescue a decaying NASAtelescope. (7)1904

Mike Matthews investigates the fascinating news from the week and Mike answers what is happening in the odd world of summer spending. Join Mike as he podcasts live from Café Anyway in podCastro Valley with Chely Shoehart, Floyd the Floorman, and John Deer the Engineer. Next show Mike Talks to Benita, the Disgruntled Fiddle Player, and the Brewmaster.

Japanese startup Heartseed Inc. has said it conducted a clinical trial to inject via catheter small masses of heart muscle cells produced from induced pluripotent stem, or iPS, cells into patients with severe heart failure.

Purchase My Zine Here: https://www.blurb.com/b/12865606-caug... You can purchase my photographic compositions here: https://www.piratechristianphoto.com Support Fighting for the Faith Join Our Crew: http://www.piratechristian.com/join-o... Patreon:   / piratechristian   Merchandise: https://www.moteefe.com/store/pirate-... Fighting for the Faith Radio Program: http://fightingforthefaith.com Social Media Facebook:   / piratechristian   Twitter:   / piratechristian   Instagram:   / piratechristian   Video Sermons    / @kongsvingerlutheran670   Sermons http://www.kongsvingerchurch.org/sermons Sunday Schools http://www.kongsvingerchurch.org/bibl... Bible Software Used in this Video: https://www.accordancebible.com Video Editing Software: https://adobe.ly/2W9lyNa Video Recording Software: https://www.ecamm.com Scripture quotations are from The ESV® Bible (The Holy Bible, English Standard Version®), copyright © 2001 by Crossway, a publishing ministry of Good News Publishers. Used by permission. All rights reserved. Scripture quoted by permission. Quotations designated (NET) are from the NET Bible® copyright ©1996-2016 by Biblical Studies Press, L.L.C. http://netbible.com http://netbible.com/ All rights reserved.

It's time for NZ Sport History and today Jesse is joined by one of the country's greatest cricketers Stephen Fleming Stephen played 111 Tests for New Zealand and 279 ODIs, he captained the Black Caps when they won the 2000 ICC KnockOut Trophy which was New Zealand's first ICC trophy Widely respected for his tactical brilliance and leadership, Australian legend Shane Warne hailed Fleming as "the best captain I played against in my time, he knew how to get the best of his teammates"

Send us Fan MailWelcome to season 5 of The Cricket Slouch, a podcast that was not expected to last this long but has trundled along nicely at a pace that would have made Haseeb Hamid proud. In this episode Shounak and SP talk about the recently concluded test matches between India-Afghanistan and England-New Zealand, focusing on some notable performances, off-field shenanigans and an unnecessary detour into T20 franchise territory before rounding up with news from the ongoing ACC Women's Premier Cup in Malaysia.We also tease some exciting things to happen for the podcast in the near future and might make an announcement about that at some stage.Stay tuned and enjoy this episode !

Youtubers The Godfrey Twins joins Oliver to talk about amassing millions of followers thanks to their twin telepathy tests, comedy sketches and pranks.

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

We hear Ana's honest recovery story, from a gradual slide into Long Covid to the moment her nervous system finally stopped treating everyday life as a threat. We talk through neurodivergence, trauma, hypermobility and the practical tools that helped her rebuild a calm, creative life. • The slow build of stressors that leads to a major crash • Hypermobility and why intense workouts can backfire • ADHD and autism discovery and how it reframes burnout • Tests, uncertainty and how fear can keep symptoms loud • Restrictive diets, supplement spirals and removing fear around food • Nervous system regulation using polyvagal ideas and body listening • Meditation with music, creative visualisation and breathing• Trauma as unmet safety needs plus small experiences that stack up • The “spreading” practice and learning to handle activation • Returning to work slowly and dropping all-or-nothing thinking • Creativity as a recovery pillar and building a life that fits Links:Ana's Substack: https://becomingcreative.substack.com/Somia Heal: https://somiainternational.com/heal/ Jackie's Insight Timer (as mentioned by Ana at the start) https://insig.ht/sT6iUelAR3b Ed the Osteopath who Ana found super helpful: https://movementandwellbeingclinic.co.uk/about-us/#ed-madeleyMessage me! (I can't reply to these messages) For more information about Long Covid Breathing courses & workshops, please check out LongCovidBreathing.com (music credit - Brock Hewitt, Rule of Life) Support the show~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~The Long Covid Podcast is self-produced & self funded. If you enjoy what you hear and are able to, please Buy me a coffee or purchase a mug to help cover costsTranscripts available on individual episodes herewww.LongCovidPodcast.comFacebook Instagram  Twitter Facebook Creativity GroupSubscribe to mailing listI love to hear from you, via socials or LongCovidPodcast@gmail.com**Disclaimer - you should not rely on any medical information contained in this Podcast and related materials in making medical, health-related or other decisions. Please consult a doctor or other health professional**

Themen: Teure Hörnchen? Eis-Preise in NRW [00:29 Min.] | Manchmal lebensgefährlich: Wechselwirkungen bei Medikamenten [08:57 Min.] | Abzocke mit schlechten Pflege-Angeboten [18:03 Min.] | Schnäppchenjagd Sonnenbrillen [26:48 Min.] | Im Labortest: Bodylotion [35:16 Min.]

In Episode 517 testen wir den neuen Helm Kyros von Fizik und wir zeigen euch, wie sich der leichte Backcountry Hydration Backpack von Apidura auf dem MTB und Gravelrad bewährt. Wie immer gibt es etwas zu gewinnen.

Drei Amerikaner und ein Italiener sollen die Artemis-3-Tests in Erdnähe durchführen, 2028 soll dann Artemis 4 zum Mond fliegen und landen. Wie realistisch ist der straffe Zeitplan und was bedeutet das für die europäische Raumfahrt? Jochen Steiner im Gespräch mit Raumfahrtexperte David Beck aus der ARD-Wissenschaftsredaktion

Today on Ascend: The Great Books Podcast, Deacon Harrison Garlick is joined by Dr. Frank Grabowski and Dr. Alex Petkas (host of the Cost of Glory) for a rich discussion of Odyssey Books 13 and 14. Check out all our resources on the great books!Check out the Cost of Glory!Check our the Porch and Altar, Dr. Grabowski's Substack.Odysseus finally reaches Ithaca—sleeping peacefully through the voyage as the Phaeacians carry him ashore—only to awaken uncertain and disguised by Athena as a beggar. The conversation explores his strategic plotting with the goddess, the symbolic Cave of the Nymphs (including a fascinating Neoplatonic reading from Porphyry), the poignant encounter with the loyal swineherd Eumaeus, and Odysseus's layered “lies” that reveal deep truths about his character. The guests delve into themes of homecoming, the restoration of patriarchal order, xenia (guest-friendship), internal moral growth, and the slowing narrative pace as Homer shifts focus from external adventures to the subtle work of reclaiming one's house.This episode offers fresh, layered insights perfect for both first-time readers and Odyssey veterans, blending literary analysis, philosophy, and practical wisdom on loyalty, cunning, and self-mastery. Whether you're drawn to the theological depth of the cave, the psychological realism of Odysseus' deceptions, or the timeless lessons on rebuilding order amid chaos, Dcn. Garlick, Dr. Grabowski, and Dr. Petkas make these often-overlooked books come alive with warmth, humor, and profound appreciation. Tune in for an engaging, accessible conversation that will deepen your love for Homer and leave you eager for the next leg of the journey.Chapters00:00 Introduction to the Odyssey and Guests08:38 The Slow Pace of the Odyssey13:15 Themes of Homecoming and Restoration18:07 The Significance of Sleep and Rebirth23:22 Divine Intervention and the Phaeacians28:24 Odysseus' Identity and the Role of Athena36:26 The Art of Deception: Odysseus and Athena's Intrigue37:19 The Mist of Disguise: Odysseus' Identity Crisis39:46 Inner Odyssey: The Journey of Self-Discovery42:34 The Goddess of Trickery: Athena's Role in Odysseus' Return44:39 The Cave of the Nymphs: Symbolism and Allegory51:03 Depositing Treasures: Spiritual Growth and Cooperation with the Divine59:13 The Loyal Swineherd: Eumaeus and the Theme of Hospitality01:02:39 The Encounter with the Dogs01:03:44 Loyalty and the Commoner's Perspective01:05:11 The Swineherd's Knowledge and Loyalty01:06:16 Odysseus's Tests and Coalition Building01:07:59 Reflections on War and Leadership01:11:35 Odysseus's Crafty Storytelling01:14:58 The Nature of Lies and Truth01:18:12 Piety and Character in the Odyssey01:22:06 The Art of Storytelling and Requests01:25:20 Final Thoughts and Future EncountersBe sure to check out our website for more resources on the great books!

Ever wondered what lab tests are essential when you hit perimenopause? Then don't miss this episode of the Mind Movement Health Podcast with Cindy Dupuie, a seasoned functional medicine nutritionist, who shares her expertise on how women can better understand and manage changes during perimenopause and menopause. We explore the importance of comprehensive lab testing, the role of hormones and gut health, gallbladder health and thyroid must-knows and how to advocate for yourself in medical settings.

Ali Mac tests Mike Johnson's, Dylan Mathews', and Beau Morgan's movie, sports, and general history knowledge as she takes them back in the day for the halftime portion of the show!

Haben Sie auch schon Ihr Mikrobiom analysieren lassen, wiegen Sie täglich die Ballaststoffe in Ihrem Essen oder sind Sie Team „Pflanzen-Challenge“? Dann liegen Sie voll im Trend. Was wir essen und was unser Körper daraus macht, beschäftigt viele Menschen, denn wir wissen mittlerweile mehr über den Darm, in dem Immunsystem, Psyche und Hormone mit der Ernährung zusammenspielen. Und andere machen Geld daraus, dass sich Menschen für ihren Darm mehr denn je interessieren, bieten Tests sowie Beratung mit zweifelhaften Versprechen an. Warum ist der Darm für unsere Gesundheit so bedeutend? Wie eng arbeitet er mit unserem Hirn zusammen? Und was können wir wissenschaftlich erwiesenermaßen dem Verdauungsorgan und damit dem ganzen Körper Gutes tun? Darüber sprechen wir mit der Ernährungsmedizinerin Luisa Werner, dem Gastroenterologen Ulrich Pape und dem Künstler Mike Bouchet. Podcast-Tipp: Die blaue Couch Dr. Ulrich Selz, Darm Doc Als Kind hatten er und sein Bruder selbst massive Darmprobleme, heute beschäftigt er sich intensiv mit unserem "Bauch-Gehirn". Warum wir einen Regenbogen essen sollten, wie der Magen knurrt, was Fasten bewirkt und warum es bei ihm daheim keine Süßigkeiten-Schublade gibt, darüber spricht Ulrich Selz mit Dominique Knoll. https://www.ardsounds.de/episode/urn:ard:episode:e00791e0b4a3f378/

Stundenlang gefangen im Scrollen von Kurzvideos, zugeschnitten auf die eigenen Interessen. Wie gefährlich ist Tiktok für Jugendliche? Länder diskutieren Nutzungsverbote für unter 16-Jährige, auch die Schweiz. Experimente zeigen: Tiktok bedient selbst Minderjährige mit problematischen Inhalten. Hirnforscherin: Zuviel Tiktok schädigt unser Hirn Barbara Studer ist Neurowissenschaftlerin und warnt: Tiktok wurde von Hirnforschern beraten. Der Algorithmus bediene uns konstant mit intensiver Stimulation, mit Dopaminausschüttung, und wecke das Gefühl, ständig mehr zu wollen. Bis hin zum emotionalen Burnout. Die Folge: Immer mehr Stimulation wird nötig, um sich gut zu fühlen. Dabei nimmt die Leistung ab, Aufmerksamkeit und Gedächtnis lassen nach, es gibt weniger soziale Interaktionen. Testfall Australien: Wirkt ein Verbot? Australien kennt seit Dezember 2025 bereits ein Tiktok-Verbot für unter 16-Jährige. Wie sind die Erfahrungen? Das Verbot lasse sich leicht umgehen, berichten Jugendliche. Etwa, indem ein falsches Geburtsdatum angegeben werde. Der Besuch in einer Schule in Lismore in der Nähe von Brisbane zeigt: Lehrpersonen unterstützen das Verbot. Schweizer Politikerin: Tiktok braucht Alterslimite Die SVP-Politikerin Nina Fehr Düsel will ein Social-Media-Verbot für unter 14-Jährige in der Schweiz und hat eine Motion ins Parlament eingebracht. Diese wird von einzelnen Vertretern aus verschiedenen Parteien unterstützt. Wirksame technische Alterskontrollen seien möglich, findet sie. Medienkompetenz in der Schule aneignen sei gut, aber nicht ausreichend. Das Experiment: Selbst Minderjährige sehen heikle Inhalte Der Tessiner Journalist Nicola Agostinetti macht ein Experiment: Er kreiert ein neues Profil eines Minderjährigen auf Tiktok. Nach ein paar Minuten erscheinen Videos mit Drogen und Waffen. Trotz scheinbarer Alterskontrolle. Dann erscheinen Videos mit dünnen Menschen, gerade für gefährdete Jugendliche ein heikles Thema. Die Französin Stéphanie Mistre hat Tiktok nach dem Suizid ihrer Tochter in Frankreich vor Gericht gebracht. Sie will, dass der Techgigant mehr Verantwortung für die Inhalte übernehmen muss.

PJ talks to Ger Hyland from The IRHA who says it's ridiculous the list is moving in the wrong direction Hosted on Acast. See acast.com/privacy for more information.

Description: We discuss why this week's macro setup favors caution, as last week's strong jobs report makes the upcoming CPI print critical to the Fed's reaction function. We cover why Bitcoin's institutional bid is being tested, with ETF outflows acting as a double-edged sword and $60K standing out as a key level to hold. We also discuss how AI-assisted discovery of a critical Zcash vulnerability is reshaping how investors price protocol security risk, and what US-regulated perps and options means for Hyperliquid. Main sections: (01:20 - 08:01) Labor, inflation, and the CPI setup (08:01 - 14:02) Bitcoin's institutional bid (14:02 - 19:35) Zcash, AI, and protocol security risk (19:35 - 29:12) Hyperliquid's competition amidst the arrival of US perps & optionsSpeakers: Colin Basco - Quantitative Strategist, Coinbase (X: https://x.com/colin_basco) Greg Sutton - Head of Prime Trading, Coinbase Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

EPISODE 447: Before you hire your next superintendent, project manager, or estimator, you need to know whether you have a capacity problem or a process problem. Todd Dawalt walks construction business owners through a five-step hiring framework to make smarter, more strategic staffing decisions. ⚡Schedule a Business Evaluation Call with The Construction Leading Edge Team:  HERE 

Most rifle data is hidden behind biased reviews or confusing specs—until now. Jim Harmer, aka Backfire, shares groundbreaking findings from his rifle accuracy database that shatter the industry status quo. Discover how a simple yet rigorous testing system reveals the truth about factory vs. custom rifles, and why seven out of ten top performers are custom builds that cost less than you think.Check out the new website:https://www.redbeardoutdoors.net/DISCOUNTS and Support The Show 1st Phorm Favorites:https://1stphorm.com/products/post-workout-stack/?a_aid=RedBeardOutdoorsSheepFeet Custom Orthotics:https://sheepfeetoutdoors.com/?ref=REDBEARDCode - REDBEARDLAST LIGHT:https://lastlightllc.com/?ref=REDBEARDCode - REDBEARDBarbell Apparel:Https://www.barbellapparel.com/redbeardCode - RedBeardOllin Digiscoping:https://ollin.co/?ref=REDBEARDCode: RedBeardGoRuck:https://alnk.to/gKTTRMeSumet:https://www.sumet.com/?ref=RedBeardCode - RedbeardTuffJug:https://tuffjug.com?sca_ref=10529106.pS2vdXlnFlA0z5p3Code - REDBEARDStar-Batt:https://star-batt.com/ref/redbeardoutdoors/Code: RedBeardoutdoorsGrayboe:https://www.grayboe.com/?aff=15Code - REDBEARDCRUZR Saddles:https://cruzr.com/id/20/Code - RedBeardPrimary Arms:https://alnk.to/fwuB01v Initial Ascent:https://initialascent.comCode: Redbeard*Disclosure: I may earn commissions on some of these links/codes used*

"Test, don't guess and stress."  It's a phrase we say often, and today's episode, we are putting the phrase to the test.  Join us in taking a deep dive into the powerful testing capabilities of RGCC International and how they are reshaping personalized cancer care. From identifying circulating tumor cells to mapping out which therapies (both conventional and natural) may be most effective, their tests can provide actionable, individualized insight so that you and your BCC Coach can make a solid plan of action.  Our guest, Dr. Terri Beim, has spent over a decade working directly with RGCC's tests and technologies and is dedicated to educating medical professionals on how to use these tools to support real patients in real time.  Now, you get to hear directly from her on how their liquid biopsy tests and other innovative technologies can help guide you and your BCC Coach into turning your results into a clear, data-driven healing plan that can be monitored every step of the way.  Because the more you know, the more effectively you can heal.

Eng v NZ Daily 2026, 1st Test, Lord's Review: It's the day after one of the shortest 40-wicket Tests on record and Adam has Daniel Norcross with him to ponder what it all means. The Lord's pitch is where attention continues to sit, albeit with MCC presenting a path to a better future for the contentious 22 yards. Casting to The Oval, where the next Test begins next Wednesday, is it simply the case that Jofra Archer vaults straight into the squad and, indeed, the XI? How might Shoaib Bashir's use, or otherwise, inform this? As for the Black Caps, they simply cannot lose their bundle with their WTC implications. Support the show with a Nerd Pledge at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠patreon.com/thefinalword⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ and win a signed copy of Wisden, or a case of Stomping Ground: browse their range at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠stompingground.beer⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Experience England's cricket tour of South Africa 2026/27 LIVE with Gullivers Sports Travel. Find out more and book at ⁠⁠⁠⁠⁠⁠⁠⁠gulliverstravel.co.uk⁠⁠⁠⁠⁠⁠⁠  Check out the Lord's Performance Centre for School Holiday activities and courses: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠lords.org/lords/performancecentre⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠  Stop snoring with 10% off a Zeus device: use code TFW2026 at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠zeussleeps.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Get your This is W̶o̶m̶e̶n̶'̶s̶ Cricket t-shirt here, and learn about Lacuna Sports bespoke cricket wear, created by women for women:⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠lacunasports.co.uk/en/shop/limited-edition/world-cup-t-shirt/⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Get your big NordVPN discount: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠nordvpn.com/tfw⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ or 10% off ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Duncan Fearnley ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠bats and kit with code TFW10 or 10% off Glenn Maxwell's sunnies: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠t20vision.com/FINALWORD⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ or 15% off Step One clothes at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠uk.stepone.life/discount/TFW148⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ or 10% off BIG Boots UK boots and socks at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠bigboots.co.uk/?ref=thefinalword⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Find previous episodes at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠finalwordcricket.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Title track by ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Urthboy⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Learn more about your ad choices. Visit podcastchoices.com/adchoices

The newborn with a new onset (some kind of) metabolic disease is a rare event in the Pediatric ER. Add that to the fact that there are so many different types of diseases with so much basic science to remember and you got a pretty bad situation in your hands. That is why I invited Dr. Austin Larson an Associate Professor of Pediatrics-Clinical Genetics and Metabolism on the show to discuss how we can do a better job identifying and treating those children.    

The Today in Manufacturing Podcast is brought to you by the editors of Manufacturing.net and Industrial Equipment News (IEN).This week's episode is brought to you by Spot.AI. Transform your plant floor with AI that understands the physical world.Imagine if your existing security cameras could do more than just record—what if they could actively protect your team and optimize your entire production line?In a new video, Anna Wells dives into how Spot.AI is revolutionizing manufacturing with intelligent video AI agents. You will discover how to leverage the camera infrastructure you already own to uncover hidden risks and streamline your daily operations.Watch the video to learn how to:Enhance safety, like automatically detecting missing PPE and hazardous conditions before accidents happen.Crush operational bottlenecks by identifying idle workstations.And maximize your tech ROI by seamlessly integrating advanced AI with your current camera setup.From basic security deterrence to full-scale manufacturing optimization, this informative session offers a practical blueprint for implementing video AI on your plant floor.Watch the video, right now. Every week, we cover the three biggest stories in manufacturing, and the implications they have on the industry moving forward. This week:- Wisconsin Castings Supplier Suffers 'Significant Fire Event'- U.S. Army Testing First New Hand Grenade in Nearly 60 Years- Boiler-Related Explosion at International Paper's NC Plant Injures Multiple WorkersIn Case You Missed It- How a Stamp Factory Reddit-Sourced Its Rubber Waste Problem - Real-Life AI Part I: 6 Use Cases That Reclaimed Hours of Wasted Time- Which Shocks Pose the Biggest Risks to Global Food Systems Please make sure to like, subscribe and share the podcast. And to email the podcast, you can reach any of us at Jeff, Anna or David@ien.com, with “Email the Podcast” in the subject line. Subscribe to our daily and weekly newsletters.

(Long Voice Only) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Long Music) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Overnight Voice Only) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Short Music) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Overnight Music) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Short Voice Only) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Long Voice Only) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Short Voice Only) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Overnight Voice Only) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Overnight Music) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Short Music) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

(Long Music) Autism Tests Monday's Sleepy Boring Objects #1628 LMBYTS by Jason Newland

What if your AI coding agent is quietly cheating on your tests — and how do you stop it? Julien Verlaguet, who built the type system Meta used to migrate tens of millions of PHP lines, is now building Skipper: a closed-loop coding agent designed to make AI-generated code verifiably correct, without human intervention.In this episode, Julien Verlaguet, creator of the Hack programming language at Meta and co-founder of SkipLabs, explains why AI agents will always try to cheat — gaming tests, quietly modifying logic while doing something else, and declaring work done when it isn't. He draws on his experience migrating Meta's PHP codebase to a statically typed system, drawing sharp parallels between convincing engineers to trust a new type checker and building systems that can trust an LLM. Julien makes the case for spec-driven development with validation layers at every step, where separate AI instances verify correctness and the code-writing agent is locked out of touching tests.He shares the story of an LLM that silently swapped a union for an intersection while splitting a file — a subtle bug that passed all tests — and why no human would ever have made that mistake. He then walks through how Skipper works: you write a spec, hand over control, and a compiler-like agent produces correct, runnable TypeScript without back-and-forth, backed by a sound incremental type system, reachability analysis, and a reactive runtime that applies diffs in milliseconds.He closes with a grounded take on how the developer role is shifting — not disappearing — toward the kind of design, integration, and oversight work that always mattered most.Key topics discussed:Why AI agents will always try to cheat on your testsThe union-vs-intersection bug an LLM introduced silentlySpec-driven development to keep LLMs on trackHow to separate the AI that verifies from the one that fixesSkipper: a compiler-like closed-loop coding agentSound, incremental TypeScript built for AI-speed iterationHot-reloading state without restarting — in millisecondsWhy developers are all becoming tech leadsTimestamps:(00:00:00) Trailer & Intro(00:02:34) How Did Julien Create the Hack Programming Language at Facebook?(00:05:53) Does Static Typing Make Your Code More Secure?(00:09:54) How Did You Convince Facebook Engineers to Adopt Hack at Scale?(00:17:15) How Can Engineers Overcome Skepticism Toward AI Coding Tools?(00:22:44) Should Junior Engineers Trust AI-Generated Code?(00:29:44) How Do You Build Reliable Guardrails for LLM-Generated Code?(00:42:15) What Validation Strategies Prevent AI Agents From Cheating on Tests?(00:45:54) What Is Skipper and How Does a Closed-Loop Coding Agent Work?(00:54:59) How Does Skipper Compare to Claude Code in Terms of Correctness?(00:58:27) How Do You Get Started With Skipper and What Does the Output Look Like?(01:04:50) How Will the Software Developer Role Change in an AI-First World?(01:09:06) 3 Tech Lead Wisdom_____Julien Verlaguet's BioJulien Verlaguet is a programming language designer and the Founder and CEO of SkipLabs. He is best known as the creator of Hack—the gradually typed language he built at Facebook that currently powers over 100 million lines of the company's production code. After creating the open-source reactive framework Skip, Julien founded SkipLabs in 2022. His company recently launched Skipper, a closed-loop coding agent that takes a single prompt from a developer and returns a running, validated service.Follow Julien:LinkedIn – linkedin.com/in/julien-verlaguet-b5710a20X – x.com/JulienVerlaguetSkipLabs - skiplabs.ioSkipper - skipperai.devSkipper's Discord – discord.gg/bsnXyw2F9PLike this episode?Show notes & transcript: techleadjournal.dev/episodes/260.Follow @techleadjournal on LinkedIn, Twitter, and Instagram.Buy me a coffee or become a patron.

durée : 00:58:34 - Grand Reportage - par : Aurélie Kieffer - Selon un sondage, les trois quarts des Français s'intéressent à leurs origines. Plongeons dans les archives de ces généalogistes amateurs, à la recherche d'histoires de famille. Des histoires qui se dévoilent désormais par d'autres moyens : les tests génétiques. - réalisation : La Rédaction de France Culture, Annie Brault, Caroline Bennetot, Éric Chaverou - invités : Catherine Pajares y Sanchez Membre du CESE et secrétaire Fédérale à la CFDT Vous aimez ce podcast ? Pour écouter tous les épisodes sans limite, rendez-vous sur Radio France

HAPPY PRIDE! Venus meets up with Jupiter (and it's not magic), and gets challenged by Chiron (it's not all bad)! And in the middle of those two transits, Mercury and Saturn clash. Tune in for the details. Watch the video version of Ghost of a Podcast on Jessica's Patreon or YouTube: https://www.youtube.com/jessicalanyadoo/videos

In This Hour:-- Federal's announcement of the 6.5 Creedmoor +Peak high velocity ammo excites hunters and long range shooters, but the U.S. military has been experimenting with much higher pressures and velocities.  Mike Milhalski of Sons Of Liberty Gun Works shares some of what they have learned, and how this will change rifles, optics, and more.--  Is there any reason to own a .38 Super handgun?--  Physics is real, and higher velocity ammunition certainly increases felt recoilGun Talk 06.08.26 Hour 2Become a supporter of this podcast: https://www.spreaker.com/podcast/gun-talk--6185159/support.

[2 Peter 2:1] There are clear biblical tests for true and false prophets given in Deuteronomy 18:18-22 and reinforced in 2 Peter 2:1. God protects His people by exposing those who speak presumptuously in His name, blend His truth with other gods through syncretism, or issue predictions that fail to come to pass. These ancient standards can be applied to the modern claims of new apostolic and prophetic offices, particularly within the New Apostolic Reformation; the church of Jesus Christ must reject extra-biblical revelation and hold fast to the sufficiency of Scripture.

From birthright citizenship to the independence of federal agencies, the Supreme Court is poised to decide a series of cases that could redefine the balance of power in Washington. Yale legal scholar and New York Times Magazine staff writer Emily Bazelon joins Ian Bremmer to assess what's at stake and whether the judiciary remains an effective check on presidential authority. Bazelon argues that Trump's effort to end birthright citizenship is unlikely to succeed, but says other pending cases involving the Federal Reserve and the Federal Trade Commission could significantly expand presidential control over agencies that Congress intentionally designed to operate independently. "I think it's very likely the court will rule in the president's favor," she says of the FTC case. The conversation also examines the Court's recent decisions on tariffs and voting rights, including a ruling that further weakened protections against partisan gerrymandering. Bazelon argues that the consequences extend beyond individual cases, contributing to a broader perception that the Court is becoming increasingly political. Yet despite declining public trust, Bazelon sees reasons for cautious optimism. While Congress has largely failed to constrain executive power, she argues that the judiciary, particularly the lower courts, has repeatedly pushed back against actions that exceed legal authority. The bigger question is whether those guardrails will continue to hold as the Court confronts some of the most consequential constitutional disputes still ahead. Subscribe to the GZERO World with Ian Bremmer Podcast on Apple Podcasts, Spotify, or your preferred podcast platform, to receive new episodes as soon as they're published. Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

06 04 26 Pulling Your Own Soil Tests by Ag PhD

#281: Chris shares the full playbook he used to take control of his health after digging into his family historical and lab results. He breaks down which tests are actually worth your money and when each one matters. Plus, how he built a personal health AI from his own medical records to surface what doctors missed. Link to Full Show Notes: https://chrishutchins.com/personal-health-ai-diagnostics/. Partner Deals DeleteMe: ⁠20% off removing your personal info from the web Fora: Become a Fora Advisor today Copilot Money: ⁠Free 2 months access to my favorite personal finance app with code HACKS2 Gelt: ⁠Skip the waitlist on personalized tax guidance to maximize your wealth Bilt Rewards: ⁠Earn the most valuable points when you pay rent For all the deals, discounts and promo codes from our partners, go to: ⁠chrishutchins.com/deals Resources Mentioned Doctors & Medical Practices PrimaryMD One Medical Blueberry Zocdoc Biomarker & Blood Testing Function Health SuperPower InsideTracker InstaLab Ulta Lab Tests Quest Diagnostics Labcorp Specialized Diagnostics Nucleus 23andMe Levels Prenuvo Health Data Aggregators & AI Tools Apple Health Google Health Epic Claude ChatGPT Gemini PubMed Tech & Wearables Oura Ring Whoop ATH Podcast #097: Optimizing Your Health: Diagnostic Tests, Choosing a Doctor, Sleep, Longevity, Inflammation and More with Dr. Jordan Shlain Newsletter Leave a review: Apple Podcasts | Spotify Email for questions, hacks, deals, and feedback: podcast@chrishutchins.com Full Show Notes (00:00) Introduction (00:46) Chris' Lifelong Battle with High Cholesterol (07:20) Switching to a More Proactive Doctor (09:39) Family Medical History: The Cheapest Diagnostic Tool (10:59) Why ApoB and Lp(a) Belong on Every Lipid Panel (17:04) Detecting Soft Plaque with a CT Angiogram (20:02) Moving Beyond Statins for Better Cholesterol Control (21:46) How Chris Got a $500/Month Drug Covered by Insurance (25:11) Branching Out to Other Health Diagnostics (26:15) Comparing the Major Biomarker Blood Work Services (32:31) What Whole Genome Sequencing Actually Tells You (34:36) The Real Value of Continuous Glucose Monitoring (36:08) Getting a Full-Body Skin Scan from a Dermatologist (36:32) Using a DEXA Scan to Baseline Body Composition (37:17) Benchmarking Fitness with a VO2 Max Test (37:51) The Case For (and Against) a Prenuvo Whole-Body MRI (39:51) Why You Still Need a Human Doctor in the Loop (44:02) Finding the Right Primary Care Setup for Your Budget (46:21) The Health Basics Worth More Than Any Fancy Test (48:40) Building a Personal Health AI From Your Medical Records (56:56) Smarter Ways to Track Your Diet with AI (01:01:43) Why Chris Records Every Doctor's Appointment (01:02:25) Final Takeaways on Modern Health Diagnostics Connect with Chris ⁠⁠Newsletter⁠⁠ | ⁠Membership⁠⁠ | ⁠X⁠⁠ | ⁠Instagram⁠⁠ | ⁠LinkedIn⁠⁠ Editor's Note: The content on this page is accurate as of the posting date; however, some of our partner offers may have expired. Opinions expressed here are the author's alone, not those of any bank, credit card issuer, hotel, airline, or other entity. This content has not been reviewed, approved or otherwise endorsed by any of the entities included within the post. Learn more about your ad choices. Visit megaphone.fm/adchoices

This is a recording of a Viewer Questions livestream from Youtube. With Jade, Chunky & Corey in this live stream. In this stream we primality discuss viewers dating and relationship questions. But we also talk about how to improve your life, financial help and world events. If you enjoy lively conversation and want your questions answered in real time, click on this link to watch upcoming live streams and be part of the conversation: https://www.youtube.com/@CoachCoreyWayne/streams 

(15) Peter Huessy discusses the confirmation by the U.S. government that China conducted recent underground nuclear tests. Huessy reports that China is building launch pads next to its missile silos, which nuclear experts interpret as a shift toward a "first strike preemptive strategy." This strategy is designed to use a nuclear umbrella to coerce the U.S. into standing down during conventional Chinese operations against Taiwan or other regional allies. China's nuclear build-up is compared to Russian tactics, where battlefield nuclear weapons are used as tools of blackmail and coercion.MAY 1953 ATOMIC CANNON LAS VEGAS