Podcasts about Prednisone

Originally aired in 2023, Molly Jacobson interviews Dr. Kim Agnew about the groundbreaking trials of Monepantel, a sheep dewormer repurposed as a potential treatment for canine lymphoma. Discover the science behind this innovative therapy, its potential to stabilize lymphoma, and how it compares to traditional treatments like chemotherapy and prednisone.  Key Topics:  What is Monepantel, and how was it discovered?  The mechanism behind Monepantel's ability to target cancer cells.  Early trial results: Quality of life and stability for dogs with B-cell lymphoma.  How Monepantel compares to prednisone and chemotherapy.  Insights into ongoing and future clinical trials.  How dog lovers can participate in studies.  Your Voice Matters!   If you have a question for our team, or if you want to share your own hopeful dog cancer story, we want to hear from you! Go to https://www.dogcancer.com/ask to submit your question or story, or call our Listener Line at +1 808-868-3200 to leave a question.   Related Videos:  https://www.youtube.com/watch?v=_92w6TTXM5c   https://www.youtube.com/watch?v=uAZrPM1X8ww   Related Links:  PharmAust Dog Lymphoma Clinical Trial: https://www.pharmaust.com/petdogtrial/  Chapters:  00:00 Introduction  00:30 Meet Dr. Kim Agnew and Monepantel Overview  01:15 The Origin of Monepantel: From Dewormer to Cancer Fighter  03:30 How Monepantel Targets Cancer Cells  07:00 Insights on Clinical Trials for Canine Lymphoma  09:30 Benefits of Monepantel: A Daily Pill for Dogs  12:00 Comparing Monepantel to Chemotherapy and Prednisone  14:45 Early Trial Results: Stabilizing Lymphoma in Dogs  18:30 Why Lymphoma Was Chosen for Trials  20:00 Potential for Treating Other Cancers in Dogs  23:15 How to Enroll Dogs in Clinical Trials  26:45 Side Effects and Safety of Monepantel  31:00 Future Trials and Research Goals  34:30 Closing Thoughts and Resources  33:55 Closing Remarks and Resources    Get to know Kim Agnew, BVSc: https://www.dogcancer.com/people/kim-agnew-bvsc/     For more details, articles, podcast episodes, and quality education, go to the episode page: https://www.dogcancer.com/podcast/   Learn more about your ad choices. Visit megaphone.fm/adchoices

Description: Co-hosts Ryan Piansky, a graduate student and patient advocate living with eosinophilic esophagitis (EoE) and eosinophilic asthma, and Holly Knotowicz, a speech-language pathologist living with EoE who serves on APFED's Health Sciences Advisory Council, interview Anna Henderson, MD, a pediatric gastroenterologist at Northern Light Health in Maine, about bone mineral density in EoE patients. They discuss a paper she co-authored on the subject. Disclaimer: The information provided in this podcast is designed to support, not replace, the relationship that exists between listeners and their healthcare providers. Opinions, information, and recommendations shared in this podcast are not a substitute for medical advice. Decisions related to medical care should be made with your healthcare provider. Opinions and views of guests and co-hosts are their own.   Key Takeaways: [:50] Co-host Ryan Piansky introduces the episode, brought to you thanks to the support of Education Partners Bristol Myers Squibb, Sanofi, Regeneron, and Takeda. Ryan introduces co-host Holly Knotowicz.   [1:17] Holly introduces today's topic, eosinophilic esophagitis (EoE), and bone density.   [1:22] Holly introduces today's guest, Dr. Anna Henderson, a pediatric gastroenterologist at Northern Light Health in Maine.   [1:29] During her pediatric and pediatric gastroenterology training at Cincinnati Children's Hospital, she took a special interest in eosinophilic esophagitis. In 2019, Dr. Henderson received APFED's NASPGHAN Outstanding EGID Abstract Award.   [1:45] Holly, a feeding therapist in Maine, has referred many patients to Dr. Henderson and is excited to have her on the show.   [2:29] Dr. Henderson is a wife and mother. She loves to swim and loves the outdoors. She practices general pediatric GI in Bangor, Maine, at a community-based academic center.   [2:52] Her patient population is the northern two-thirds of Maine. Dr. Henderson feels it is rewarding to bring her expertise from Cincinnati to a community that may not otherwise have access to specialized care.   [3:13] Dr. Henderson's interest in EoE grew as a GI fellow at Cincinnati Children's. Her research focused on biomarkers for disease response to dietary therapies and EoE's relationship to bone health.   [3:36] As a fellow, Dr. Henderson rotated through different specialized clinics. She saw there were many unanswered questions about the disease process, areas to improve treatment options, and quality of life for the patients suffering from these diseases.   [4:00] Dr. Henderson saw many patients going through endoscopies. She saw the social barriers for patients following strict diets. She saw a huge need in EoE and jumped on it.   [4:20] Ryan grew up with EoE. He remembers the struggles of constant scopes, different treatment options, and dietary therapy. Many people struggled to find what was best for them before there was a good approved treatment.   [4:38] As part of Ryan's journey, he learned he has osteoporosis. He was diagnosed at age 18 or 19. His DEXA scan had such a low Z-score that they thought the machine was broken. He was retested.   [5:12] Dr. Henderson explains that bone mineral density is a key measure of bone health and strength. Denser bones contain more minerals and are stronger. A low bone mineral density means weaker bones. Weaker bones increase the risk of fracture.   [5:36] DEXA scan stands for Dual Energy X-ray Absorptiometry scan. It's a type of X-ray that takes 10 to 30 minutes. A machine scans over their bones. Typically, we're most interested in the lumbar spine and hip bones.   [5:56] The results are standardized to the patient's height and weight, with 0 being the average. A negative number means weaker bones than average for that patient's height and weight. Anything positive means stronger bones for that patient's height and weight.   [6:34] A lot of things can affect a patient's bone mineral density: genetics, dietary history, calcium and Vitamin D intake, and medications, including steroid use. Prednisone is a big risk factor for bone disease.   [7:07] Other risk factors are medical and auto-immune conditions, like celiac disease, and age. Any patient will have their highest bone density in their 20s to 30s. Females typically have lower bone mineral density than males.   [7:26] The last factor is lifestyle. Patients who are more active and do weight-bearing exercises will have higher bone mineral density than patients who have more of a sedentary lifestyle.   [7:56] Ryan was told his bone mineral density issues were probably a side-effect of the long-term steroids he was on for his EoE. Ryan is now on benralizumab for eosinophilic asthma. He is off steroids.   [8:36] Dr. Henderson says the research is needed to find causes of bone mineral density loss besides glucocorticoids.   [8:45] EoE patients are on swallowed steroids, fluticasone, budesonide, etc. Other patients are on steroids for asthma, eczema, and allergic rhinitis. These may be intranasal steroids or topical steroids.   [9:01] Dr. Henderson says we wondered whether or not all of those steroids and those combined risks put the EoE population at risk for low bone mineral density. There's not a lot published in that area.   [9:14] We know that proton pump inhibitors can increase the risk of low bone mineral density. A lot of EoE patients are on proton pump inhibitors.   [9:23] That was where Dr. Henderson's interest started. She didn't have a great way to screen for bone mineral density issues or even know if it was a problem in her patients more than was expected in a typical patient population.   [9:57] Holly wasn't diagnosed with EoE until she was in her late 20s. She was undiagnosed but was given prednisone for her problems. Now she wonders if she should get a DEXA scan.   [10:15] Holly hopes the listeners will learn something and advocate for themselves or for their children.   [10:52] If a patient is concerned about their bone mineral density, talking to your PCP is a perfect place to start. They can discuss the risk factors and order a DEXA scan and interpret it, if needed.   [11:11] If osteoporosis is diagnosed, you should see an endocrinologist, specifically to discuss therapy, including medications called bisphosphonates.   [11:36] From an EoE perspective, patients can talk to their gastroenterologist about what bone mineral density risk factors may be and if multiple risk factors exist. Gastroenterologists are also more than capable of ordering DEXA scans and helping their patients along that journey.   [11:53] A DEXA scan is typically the way to measure bone mineral density. It's low radiation, it's easy, it's fast, and relatively inexpensive.   [12:10] It's also useful in following up over time in response to different interventions, whether or not that's stopping medications or starting medications.   [12:30] Dr. Henderson co-authored a paper in the Journal of Pediatric Gastroenterology and Nutrition, called “Prevalence and Predictors of Compromised Bone Mineral Density in Pediatric Eosinophilic Esophagitis.” The study looked at potential variables.   [12:59] The researchers were looking at chronic systemic steroid use. They thought it was an issue in their patients, especially patients with multiple atopic diseases like asthma, eczema, and allergic rhinitis. That's where the study started.   [13:22] Over the years, proton pump inhibitors have become more ubiquitous, and more research has come out. The study tried to find out if this was an issue or not. There weren't any guidelines for following these patients, as it was a retrospective study.    [13:42] At the time, Dr. Henderson was at a large institution with a huge EoE population. She saw that she could do a study and gather a lot of information on a large population of patients. Studies like this are the start of figuring out the guidelines for the future.   [14:34] Dr. Henderson wanted to determine whether pediatric patients with EoE had a lower-than-expected bone mineral density, compared to their peers. [14:44] Then, if there were deficits, she wanted to determine where they were more pronounced. Were they more pronounced in certain subgroups of patients with EoE?   [14:59] Were they patients with an elemental diet? Patients with an elimination diet? Were they patients on steroids or PPIs? Were they patients with multiple atopic diseases? Is low bone mineral density just a manifestation of their disease processes?   [15:14] Do patients with active EoE have a greater propensity to have low bone mineral density? The study was diving into see what the potential risk factors are for this patient population.   [15:45] The study was a retrospective chart review. They looked at patients aged 3 to 21. You can't do a DEXA scan on a younger patient, and 21 is when people leave pediatrics.   [16:03] These were all patients who had the diagnosis of EoE and were seen at Cincinnati Children's in the period between 2014 and 2017. That period enabled full ability for chart review. Then they looked at the patients who had DEXA scans.   [16:20] They did a manual chart review of all of the patients and tried to tease out what the potential exposures were. They looked at demographics, age, sex, the age of the diagnosis of EoE, medications used, such as PPIs, and all different swallowed steroids.   [16:44] They got as complete a dietary history as they could: whether or not patients were on an elemental diet, whether that was a full elemental diet, whether they were on a five-food, six-food, or cow's milk elimination diet.   [16:58] They teased out as much as they could. One of the limitations of a retrospective chart review is that you can't get some of the details, compared to doing a prospective study. For example, they couldn't tease out the dosing or length of therapy, as they would have liked.   [17:19] They classified those exposures as whether or not the patient was ever exposed to those medications, whether or not they were taking them at the time of the DEXA scan, or if they had been exposed within the year before the DEXA scan.   [17:40] They also looked at whether the patients had other comorbid atopic disorders, to see if those played a role, as well.   [18:03] The study found that there was a slightly lower-than-expected bone mineral density in the patients. The score was -0.55, lower than average but not diagnostic of a low bone mineral density, which would be -2 or below.   [18:27] There were 23 patients with low bone mineral density scores of -2 or below. That was 8.6% of the study patients. Typically, only 2.5% of the population would have that score. It was hard to tease out the specific risk factors in a small population of 23.   [18:57] They looked at what the specific risk factors were that were associated with low bone mineral density, or bone mineral density in general.   [19:12] After moving from Colorado, Holly has transferred to a new care team, and doctors wanted her baseline Vitamin D and Calcium levels. No one had ever tested that on her before. Dr. Henderson says it's hard because there's nothing published on what to do.   [19:58] The biggest surprise in the study was that swallowed steroids, or even combined steroid exposure, didn't have any effect on bone mineral density. That was reassuring, in light of what is known about glucocorticoid use.   [20:16] The impact of PPI use was interesting. The study found that any lifetime use of PPIs did seem to decrease bone mineral density. It was difficult to tease out the dosing and the time that a patient was on PPIs.   [20:34] Dr. Henderson thinks that any lifetime use of PPIs is more of a representation of their cumulative use of PPIs. At the time of the study, from 2014 to 2017, PPIs were still very much first-line therapy for EoE; 97% of the study patients had taken PPIs at some time.   [21:02] There are so many more options now for therapy when a patient has a new diagnosis of EoE, especially with dupilumab now being an option.   [21:11] Dr. Henderson speaks of patients who started on PPIs and have stayed on them for years. This study allows her to question whether we need to continue patients on PPIs. When do we discuss weaning patients off PPIs, if appropriate?   [22:05] Ryan says these podcasts are a great opportunity for the community at large and also for the hosts. He just wrote himself a note to ask his endocrinologist about coming off PPIs.   [22:43] Dr. Henderson says that glucocorticoid use is a known risk factor for low bone mineral density and osteoporosis. In the asthma population, inhaled steroids can slightly decrease someone's growth potential while the patient is taking them.   [23:10] From those two facts, it was thought that swallowed steroids would have a similar effect. But since they're swallowed and not systemic, maybe things are different.   [23:23] It was reassuring to Dr. Henderson that what her study found was that the swallowed steroid didn't affect bone mineral density. There was one other study that found that swallowed steroids for EoE did not affect someone's height.   [23:51] Dr. Henderson clarifies that glucocorticoids include systemic steroids like prednisone and hydrocortisone.     [23:57] Based on Dr. Henderson's retrospective study, fluticasone as a swallowed steroid did not affect bone mineral density. It was hard to tease out the dosing, but the cumulative use did not seem to result in a deficit for bone mineral density.   [24:16] Holly shared that when she tells a family of a child she works with that the child's gastroenterologist will likely recommend steroids, she will now give them the two papers Dr. Henderson mentioned. There are different types of steroids. The average person doesn't know the difference.   [25:15] Dr. Henderson thinks that for patients who have multiple risk factors for low bone mineral density, it is reasonable to have a conversation about bone health with their gastroenterologist to see whether or not a DEXA scan would be worth it.   [25:56] If low bone mineral density is found, that needs to be followed up on.   [26:03] There are no great guidelines, but this study is a good start on what these potential risk factors are. We need some more prospective studies to look at these risk factors in more detail than Dr. Henderson's team teased out in this retrospective study.   [26:23] Dr. Henderson tells how important it is for patients to participate in prospective longitudinal studies for developing future guidelines.   [26:34] Holly points out that a lot of patients are on restrictive diets. It's important to think about the whole picture if you are starting a medication or an elimination, or a restricted diet. You have to think about the impact on your body, overall.   [27:11] People don't think of dietary therapy as medication, but it has risks and benefits involved, like a medication.   [27:50] Dr. Henderson says, in general, lifestyle management is the best strategy for managing bone health. Stay as active as you can with weight-bearing exercises and eating a well-balanced diet. If you are on a restrictive diet, make sure it's well-balanced.   [28:12] Dr. Henderson says a lot of our patients have feeding disorders, so they see feeding specialists like Holly. A balanced diet is hard when kids are very selective in their eating habits.   [29:10] Dr. Henderson says calcium and Vitamin D are the first steps in how we treat patients with low bone mineral density. A patient who is struggling with osteoporosis needs to discuss it with their endocrinologist for medications beyond supplementation.   [29:31] Ryan reminds listeners who are patients always to consult with their medical team. Don't go changing anything up just because of what we're talking about here. Ask your care team some good questions.   [29:47] Dr. Henderson would like families to be aware, first, that some patients with EoE will have bone mineral density loss, especially if they are on PPIs and restrictive diets. They should start having those discussions with their providers.   [30:04] Second, Dr. Henderson would like families to be reassured that swallowed steroids and combined steroid exposure didn't have an impact on bone mineral density. Everyone can take that away from today's chat.   [30:18] Lastly, Dr. Henderson gives another plug for patient participation in prospective studies, if they're presented with the opportunity. It's super important to be able to gather more information and make guidelines better for our patients. [30:35] Holly thanks Dr. Henderson for coming on Real Talk — Eosinophilic Diseases and sharing her insights on bone mineral density, and supporting patients in Maine.   [30:57] Dr. Henderson will continue to focus on the clinical side. She loves doing outreach clinics in rural Maine. It's rewarding, getting to meet all of these patients and taking care of patients who would otherwise have to travel hours to see a provider.   [32:01] Ryan thinks the listeners got a lot out of this. For our listeners who would like to learn more about eosinophilic disorders, please visit APFED.org and check out the links in the show notes.   [32:11] If you're looking to find specialists who treat eosinophilic disorders, we encourage you to use APFED's Specialist Finder at APFED.org/specialist.   [32:19] If you'd like to connect with others impacted by eosinophilic diseases, please join APFED's online community on the Inspire Network at APFED.org/connections.   [32:28] Ryan thanks Dr. Henderson for joining us today for this great conversation. Holly also thanks APFED's Education Partners Bristol Myers Squibb, Sanofi, Regeneron, and Takeda for supporting this episode.   Mentioned in This Episode: Anna Henderson, MD, a pediatric gastroenterologist at Northern Light Health in Maine Cincinnati Children's “Prevalence and Predictors of Compromised Bone Mineral Density in Pediatric Eosinophilic Esophagitis.” Journal of Pediatric Gastroenterology and Nutrition   APFED on YouTube, Twitter, Facebook, Pinterest, Instagram Real Talk: Eosinophilic Diseases Podcast apfed.org/specialist apfed.org/connections   Education Partners: This episode of APFED's podcast is brought to you thanks to the support of Bristol Myers Squibb, Sanofi, Regeneron, and Takeda.   Tweetables:   “DEXA scan stands for dual-energy X-ray absorptiometry scan. It's a type of X-ray where a patient lies down for 10 to 30 minutes. A machine scans over their bones. Typically, we're most interested in the lumbar spine and hip bones.” — Anna Henderson, MD   “We wondered whether or not all of those steroids and those combined risks even put our EoE population at risk for low bone mineral density. There's not a lot published in that area.” — Anna Henderson, MD   “If a patient is worried [about their bone mineral density], their PCP is a perfect place to start for that. They're more than capable of discussing the risk factors specific for that patient, ordering a DEXA scan, and interpreting it if need be.” — Anna Henderson, MD   “I think we need some more prospective studies to look at these risk factors in a little bit more detail than we were able to tease out in our retrospective review.” — Anna Henderson, MD   “Just another plug for the participation in prospective studies, if you're presented with the opportunity. It's super important to be able to gather more information and to be able to make guidelines better for our patients about these risks.” — Anna Henderson, MD

Our heroes finally come to an end with their long awaited encounter with Prednisone and are in for a surprise when Flargelbuns alters the battlefield. As they look to escape the hope to stumble into better lands than they have been in. Check out our Linktree where we have Twitter, Facebook, Discord and Instagram! Come join in the and chat about your favorite moments with the cast!linktr.ee/thedhcastMusic credits: www.epidemicsound.com

Our heroes encounter much more than the illusion that Nox bargained for as they continue their altercation with Prednisone. Can they gain some needed information from this enounter and will they be able to survive long enough to use it?Check out our Linktree where we have Twitter, Facebook, Discord and Instagram! Come join in the and chat about your favorite moments with the cast!linktr.ee/thedhcastMusic credits: www.epidemicsound.com

Our heroes continue to push in battle against Prednisone while trying their best to protect their friend Flargelbuns. As they push to defeat this enemy they have searched so long for, can they save their friend in the process.Check out our Linktree where we have Twitter, Facebook, Discord and Instagram! Come join in the and chat about your favorite moments with the cast!linktr.ee/thedhcastMusic credits: www.epidemicsound.com

Prednisone

Our heroes finally confront their former companion Prednisone determined to put an end to him. They showed up prepared to fight but are caught off guard by an old friend. Can you put an end to Prednisone once and for all?Check out our Linktree where we have Twitter, Facebook, Discord and Instagram! Come join in the and chat about your favorite moments with the cast!linktr.ee/thedhcastMusic credits: www.epidemicsound.com

Dr. Jack Cush reviews the news, journal and regulatory reports from this week on RheumNow.com.  Interesting trends and results with prednisone in lupus, cannabis in RA and opioid deaths too!

Our heroes finally understand their past, and how their former friend has wronged them. They head back to Mucinex in order to formulate a plot to finally take down Prednisone one and for all.Check out our Linktree where we have Twitter, Facebook, Discord and Instagram! Come join in the and chat about your favorite moments with the cast!linktr.ee/thedhcastMusic credits: www.epidemicsound.com

Take a sneak peak at this month's Fertility & Sterility! Articles discussed this month are:   4:08 Classification system of human ovarian follicle morphology: recommendations of the National Institute of Child Health and Human Development - sponsored ovarian nomenclature workshop  12:32 Impact of Prednisone on Vasectomy Reversal Outcomes (iPRED Study): Results from a Randomized, Controlled Clinical Trial 21:38 Triggering oocyte maturation in IVF treatment in normal responders: a systematic review and network meta-analysis 33:57 Parental Balanced Translocation Carriers do not have Decreased Usable Blastulation Rates or Live Birth Rates Compared to Infertile Controls  45:28 A re-look at the relevance of TSH and thyroid autoimmunity for pregnancy outcomes: Analyses of RCT data from PPCOS II and AMIGOS View Fertility and Sterility May 2025, Volume 123, Issue 5:  https://www.fertstert.org/issue/S0015-0282(25)X0004-2 View Fertility and Sterility at https://www.fertstert.org/

Discussing:Association between risk-reducing surgeries and survival in young BRCA carriers with #BreastCancer DESTINY-Breast11 Update from IndustryPALMIRA in Breast #Cancer (Palbo rechallenge)PEACE V STORM in #Prostate CancerMAGNITUDE :Niraparib and Abiraterone Acetate plus Prednisone in Met CR Prostate CancerOS EGFR-mutant AdvancedNon-Small Cell #LungCancer Treated with 1L Osimertinib Cemiplimab monotherapy as 1L treatment of patients with brain metastases from advanced #NSCLC with  PDL1 ≥50%Beyond fluorodeoxyglucose: Molecular imaging of cancer in precision medicine and more

Today's sponsor is Freed AI! Freed's AI medical scribe listens, transcribes, and writes notes for you. Over 15,000 healthcare professionals use Freed and you should too! Learn more here! Prednisone is a commonly used medication for its antiinflammatory and immunosuppressive action. On this episode I discuss prednisone pharmacology, ADRs, and much more. In the episode, I discuss how prednisone affects the HPA system in the body. Cortisol production can be greatly impacted by long term use of prednisone. GI adverse effects can be problematic with oral prednisone which is why we often give this medication with food or milk. Hyperglycemia is a complication from the use of prednisone. I've commonly seen blood sugar spike by 50-100+ mg/dL due to this medication. Osteoporosis is a long term risk with the use of prednisone. Prednisone can also impact the benefits of vaccination. I discuss this in greater depth in the drug interactions portion of the podcast.

See all the Healthcasts at https://www.biobalancehealth.com/healthcast-blog In my Anti-Aging-Longevity practice, one of the complaints my new patients tell me about is the fact that their hair is thinning, falling out or changing to a brittle texture, as well as the fact that their hair is turning grey. Hair DOES get thinner, coarser, and greyer as we age!  That is a fact, and one I can't fully explain to you, except that it happens to everyone!  My philosophy is that each of us should work with what we have, and optimize it, as well as treat any condition that makes hair loss work instead of wishing and wishing we had the hair we did when we were 25! Accepting the fact that your hair gets thinner after 40 and working with the hair you have takes knowledge to determine what is normal and what is not. Some of the things that everyone should know before looking for the best path forward to healthy hair. Aging and Slower Hair Growth Low Growth Hormone Lack of Estradiol and T in women and lack of T in men Aging causes Growth Hormone (GH) to decrease, which slows the growth of hair, fingernails, bone.  Your hair falls out at the same rate as it did when you were young, but the growth slows which results in losing more hair than you replace which thins your hair! If you started your young life with fine, thin hair, then this difference between growth and loss of hair can make your hair very thin.  To get to the root of the problem (I apologize) increasing GH will make your hair grow a bit faster. Testosterone replacement and Estradiol replacement both increase the growth of GH. Both hormone replacements increase hair thickness. Testosterone has a second benefit.  Testosterone makes your scalp oiler, which in turn increases the longevity of your hair. At menopause the lack of these two hormones causes a big change in hair thickness. Loss of hair in specific areas—Balding Genetics Scarring alopecia Androgenic alopecia Extensions Dread-locks Balding and alopecia both cause a person to lose hair follicles, not just slow the rate of hair growth.  Balding usually is genetically determined, so look at the older members of your family to determine what is in your future.  This type of hair loss is very difficult to treat. 50% of men have some balding by age 50, and 1/3 of women experience it sometime in their lives.  Until recently there was nothing to stop this process or grow more hair in those areas, however both men and women can resurrect their hair follicles (if they haven't been gone too long) and make them grow with the TED hair restoration painless ultrasound treatment by Alma. Other options are Hans Weiman hair transplants or weaves, both of which are extremely expensive, don't necessarily look natural (President Biden and his son) and must be redone every couple of years. Women can just cover over the problem with a wig, but even that answer has drawbacks—they are hot and itchy until you get used to it. Women can also get extensions to make their hair look thicker, but it ruins the quality of the hair you have which in the long run causes even more hair loss from scarring of the scalp. Women of color have traditionally used tight braids, cornrowing or dreadlocks to control their hair.  This cultural process causes them to break their hair off at the scalp and damage the hair follicle from tension, which results in hair no longer growing in multiple areas of the scalp. Change in Hair Texture and Dryness At menopause for women and when men's testosterone gets very low, we notice a change in texture of our hair. The cuticle area that covers the hair shaft becomes fragile and stops protecting the hair shaft, so the texture becomes frizzy, and hair breaks causing a dull look similar to what my mother used to call a “birds nest”. Our scalps become dry, and the oil glands dry up with age and loss of sex hormones, so hair is dry and frizzy, making us look like we just stuck our fingers in a light switch.  You can read about many “natural remedies” but beyond taking collagen, Biotin and B vitamins most supplements don't work in a dramatic way that would be noticeable. The remedies for hair loss include all the following and you will have to do most of them to improve your aging hair! Hormone replacement of Estradiol and Testosterone Conditioners (which only work a little) Hair color which covers the shaft with pigment and strengthens hair Brazilian treatment that drives straightener into the hair shaft and seals it with heat. Take supplements of Collagen every morning Take methyl B12 and Biotin daily Stop bleaching your hair Eat a diet with healthy fats and protein Wash your hair every 2-3 days Take the fat soluble vitamins A.E.K,D If you are anemic take iron supplements Avoid statins if possible   Other medical causes of frizzy, broken hair can be found in low thyroid hormones which slows hair growth, decreases oil production, and results in brittle hair all over the head. Replacing your thyroid hormones and supplementing your iodine can overcome this obstacle. All medicine changes should be managed by your doctor. Many drugs cause hair loss, and you can't change some of them: Metoprolol or any Beta blocker Blood pressure medication Prednisone and all steroids Cancer treatments Anything that inhibits your B vitamins like some autoimmune diseases What can you do to fix what you can fix! Nutrition: Hair is protein; however hair requires oil (fat) to grow and be beautiful—a diet rich in protein, and healthy fats give you the building blocks to make healthy hair and skin. Add Vitamins of A/E/K/D, vitamin C, Multi Methyl B vitamins with methyl folate and Biotin because our diets aren't perfect!   Hair care: Get hair products without sulfides.  They break hair and make it weak!  Wash your hair as little as possible. Decrease the use of hot hair tools like flat irons.  Color your hair and or Brazilian it to make it stronger. Medications not to take are listed above. But the medications to take to help your hair are your sex hormones, Estradiol, Testosterone, and make sure your DHT doesn't get too high which can cause hair loss in the male pattern. You may need Finasteride or Minoxidil if you have male pattern hair loss. Sun damage is important to maintain your hair in sunny areas.  Wear a hat or scarf when outside and comb some conditioner through your hair at the pool to “cover your hair from sun damage”.   Summary: Now that you know the possible causes and treatments for hair loss, you can do everything possible up to seeking medical care, and then medical care may be necessary. Hair thinning is often familial and also due to our estradiol and testosterone hormones decreasing after 35-40 years of age. You may need a scalp biopsy from a Dermatologist if your hair loss is in patches or severe over a short period of time, which implies an autoimmune disease. When you see a doctor be prepared with a list of your hair products, your diet, a list of medicines and a timeline for your hair loss.  This preparation will get you the best treatment per doctor visit, and your doctor will appreciate not having to ask you all those questions!

Prednisone and cortisone shots can wreak havoc on blood sugar, but sometimes they're unavoidable. In this episode, I break down exactly what happens, why it happens, and how to handle it without losing your mind (or your blood sugar control). I'll share my own experience, tips for managing insulin resistance, and how to stay on top of your diabetes game!   Want the best blood sugars you've ever had while enjoying great food? Peep this: https://www.yourdiabetesinsider.com/coaching   RESOURCES: Download these FREE guides that will help you on your diabetes, nutrition, and exercise journey! https://www.yourdiabetesinsider.com/free-stuff Join our EXCLUSIVE Facebook group! https://www.facebook.com/groups/266766620895432 Watch my food breakdowns here → https://www.youtube.com/@yourdiabetesinsider   LET'S TALK! Instagram: @manoftzeel Tiktok: @manoftzeel  

All it takes is a small amount of oxidative stress to the eye's lens to develop a cataract. A healthy lens is composed of an organized protein called crystallin. When this tissue becomes damaged, it is not replaced. Glycation can damage the lens of the eye. It results from sugar connecting to a protein and then becoming stuck and unusable to the body. Steroids like Prednisone can also increase the risk of cataracts. Not all surgeries can eliminate cataracts, and they can come with complications. According to “The Cataract Cure” by Dr. Marios Kyriazis, there is a safe, effective remedy for cataracts. Carnosine is a unique compound that can penetrate the lens of the eye and help with cataracts. Carnosine is a potent antioxidant that can dissolve damaged proteins. It can also activate your own antioxidants. NAC (n-acetyl carnosine) eye drops are one of the best remedies for cataracts.In an experiment conducted at the Moscow Helmholtz Research Institute of Eye Diseases, scientists used NAC eyedrops at 1% concentration on rabbits. They found that NAC could reach the inner parts of the eye within 15 to 30 minutes. No side effects were reported during this experiment. The author concluded that NAC is a proposed eye treatment for eye disorders, including cataracts, glaucoma, corneal disorders, inflammation, and complications from diabetes.The same group of scientists who performed the rabbit studies also tested NAC on humans. They selected 49 volunteers with an average age of 65 who had established age-related cataracts. After 6 months, researchers found that:•41% of people presented a significant reduction of clouding in the lens•90% showed a gradual improvement in vision•89% showed up to a 100% improvement in sensitivity to glare NAC for cataracts must be used for at least 3 to 5 months. Antioxidants such as those found in onions, egg yolks, leafy greens, and grass-fed red meat can protect you against cataracts.Vitamin A is vital for eye health and can be found in cod liver oil and grass-fed liver. B vitamins, especially B2, B6, and folate, are also essential. BOOK LINK: https://www.amazon.com/Cataract-Cure-... DATA:https://pmc.ncbi.nlm.nih.gov/articles...https://pubmed.ncbi.nlm.nih.gov/12001...https://pubmed.ncbi.nlm.nih.gov/26084...

Mike is an IT Infrastructure Manager in Las Vegas, and he's coming up on 1 year of Carnivore in March. He was diagnosed with Mixed Connective Tissue disorder in 2016, and has in the past been on Methotrexate, Prednisone, Enbrel, and Rinvoq. MCTD consists of elements of Sjogens, Rheumatoid Arthritis, and Lupus. Since going Carnivore, the only non-Carnivore thing he still uses that helps is Qunol Tumeric and Ginger gummies to help with inflammation. He has lost 50 pounds, his symptoms are gone until he eats something he shouldn't, and he has more energy and his hands have stopped hurting. Timestamps: 00:00 Trailer 01:18 Introduction 07:49 Sedentary job, unhealthy diet 08:58 Loss of basic functionality 13:34 Keto diet success and weight loss 16:06 Beef-centric meal prep routine 18:35 Rib eye diet cures symptoms 22:30 Couple's weight loss success 24:40 Prioritizing quality meats over extras 29:38 Diet's role in rheumatism neglected 31:43 Air fried rib eyes & snacks 35:06 Curbing cravings with ribeye 38:15 Revitalizing lives through dietary change 40:45 Easing into a carnivore diet 43:10 Mindful eating on vacation 46:34 Where to find Mike Join Revero now to regain your health: https://revero.com/YT Revero.com is an online medical clinic for treating chronic diseases with this root-cause approach of nutrition therapy. You can get access to medical providers, personalized nutrition therapy, biomarker tracking, lab testing, ongoing clinical care, and daily coaching. You will also learn everything you need with educational videos, hundreds of recipes, and articles to make this easy for you. Join the Revero team (medical providers, etc): https://revero.com/jobs ‪#Revero #ReveroHealth #shawnbaker  #Carnivorediet #MeatHeals #AnimalBased #ZeroCarb #DietCoach  #FatAdapted #Carnivore #sugarfree Disclaimer: The content on this channel is not medical advice. Please consult your healthcare provider.

Americans are more stressed than ever before due to the pandemic, financial situations, medications, overconsumption of alcohol,imbalanced diet, poor gut health, and environmental toxins. As a result, many people are tired, depressed, have poor memory and focus, and have weakened immune systems.According to research, Boosting B vitamin levels quickly improves energy. A deficiency of just one B vitamin will negatively affect the complex production of energy (ATP) in your cells. While people usually think of Supplemental B12 for energy, one will typically get better results with a full spectrum B complex since many of the B's are involved in ATP production.Recently Dr. Mark Stengler, NMD. put out a helpful article that can be found here.Many medications deplete the body of B vitamins. Examples include acid reflux medications such as the Proton Pump Inhibitors (PPIs), Prednisone, Metformin, Antibiotics, Birth Control Pills, Anti-seizure medications, Antidepressants, Chemotherapy drugs such as Methotrexate, and several others. Research has shown that the elderly who take three or more medications are prone to B vitamin deficiencies such as B2, B6, Folate, and B12.Helpful ArticleLearn more about Emerald Labs Listeners of the show can save 20% with the code: Forever 

Welp, it's time once again for the Monday morning podcast you can rely on - Bubbles' Mushrooms! This week we have our friends the Puzzle Broads back at it again! That's right, Ann and Sharon are back hanging with the gang to talk about puzzles. Edward decides to start his own gang called the Puzzle Dudes to rival the Puzzle Broads at puzzles and have puzzle showdowns at libraries across the nation. Katie has an injury just ahead of the great puzzle contest that the Broads are excited to enter, Ann brought a special friend with her who ended up punching Luke in the balls, we have a medical corner update from Katie (who is on some brand new medicine), Edward calls Katie some bad names and Sharon tells us about her FroUp Bowl which is not a green Tupperware from her mom's house. Turns out Katie's new medicine has turned her a bit surly and she also mixes in a cup of BMAD style coffee to help rile her up even further, but thankfully she isn't chewing any gum. Ann decides it's time to whip her family into shape and have a come to Jesus with her children, Luke unfortunately doesn't have any cyber bully updates and we have a few viewer emails to read from Brian and Nathan Skarunchy. Next up is game time, of course! This week we have one of the games of all time to play called Family Feud… or Family Feudish. Our host Edward challenges the Ann & Luke family who are taking on the Katie and Sharon family. What kinds of things would ruin a blind date with a blind person? Are the number one answers more from the Richard Dawson or the Steve Harby era? Is Jesus of Nazareth a white-haired douche? Find out this week only on Bubbles' Mushrooms! Email the show at bubbmush@gmail.com and follow the show on Instagram (and maybe TikTok?) @bubbmush - thanks for checking out the show!

Welcome back to the Karma Stories podcast! In today's episode, host Rob shares five gripping tales from the 'Traumatize Them Back' subreddit. From serving icy cold karma to a nosy coworker to schooling a judgmental stranger at the grocery store, these stories of instant retribution are sure to captivate you. Plus, Rob discusses the strain and stigma associated with adoption through a personal lens. Don't miss out on this rollercoaster of emotions and epic clapbacks. Tune in and enjoy these astonishing tales of karma in action!Submit your own stories to KarmaStoriesPod@gmail.com.Check out our NEW PODCAST! Karma Crime!https://kccyt.com/KarmaCrimeApplePodcastshttps://kccyt.com/KarmaCrimeSpotifyhttps://kccyt.com/KarmaCrimeAmazonMusichttps://kccyt.com/KarmaCrimeDeezerKarma Stories is available on all major Podcasting Platforms and on YouTube under the @KarmaStoriesPodcast handle. We cover stories from popular Reddit Subreddits like Entitled Parents, Tales From Tech Support, Pro Revenge and Malicious Compliance. You can find new uploads here every single day of the week!Rob's 3D Printing Site: https://Dangly3D.comUSE CODE "NEWYEAR2025" FOR 25% OFF YOUR ENTIRE ORDER!Become a supporter of this podcast: https://www.spreaker.com/podcast/karma-stories--5098578/support.

Recommended Apple Cider Vinegar: https://www.fairchildsvinegar.com/ Most people are familiar with the more popular benefits of apple cider vinegar, including the following: •Supports digestion •Decreases bloating •Improves indigestion and acid reflux •Supports healthy blood sugar levels •Helps make insulin more sensitive •Decreases a fatty liver •Decreases inflammation Both good bacteria and bad bacteria can go into a dormant state. Microbes in a dormant state are protected from many environmental stresses and chemicals, especially antibiotics. Dormant microbes can survive for decades or even thousands of years! A change in environment, like a change in pH, can wake good bacteria or cause them to go dormant. Most good bacteria thrive in an acidic environment. Apple cider vinegar acidifies the environment for good microbes. Things like kombucha, pickle juice, sauerkraut, and kimchi have similar effects. Certain microbes create butyrate, which helps with insulin resistance. These microbes thrive in an acidic environment. We have a synergistic relationship with the microbes in our gut. We provide a home, and they provide immune protection, help with digestion, vitamin production, and more. By making their environment more acidic, you help activate them. H. pylori releases ammonia, which alkalizes the stomach acid. This allows it to do its damage, causing ulcers in the stomach and small intestine. C. diff, E. coli, and salmonella go into a state of dormancy when the pH drops below 6. When your immune system is suppressed, bad bacteria can come out of dormancy. Antibiotics, stress, Prednisone, and vitamin D deficiency can all suppress the immune system and activate pathogenic microbes. Adequate vitamin D in amounts of at least 10,000 IU daily may help keep pathogenic microbes in remission. Try drinking 1 tablespoon of apple cider vinegar in a glass of water every evening with a straw to support your beneficial microbes. Fairchild's Apple Cider Vinegar is an excellent option.

Guest: Corinna Bellizzi, MBA is a natural products industry executive and omega-3 expert who successfully creates new nutrition categories, pioneers brands, and develops educational strategies that disrupt markets.As an avid podcaster, she started a new podcast, Nutrition Without Compromise, to lean into the simple truth that great nutrition and bountiful health are a human right. She is also the host and creator of another great podcast, Care more be better.Last time Corinna joined me we shared a groundbreaking study conducted by an international team of researchers that unveiled a pioneering approach to boosting the production of active vitamin B12 in Spirulina. For those of you who missed that episode it is Ep 553 if you want to listen to it after this show.This study presents Spirulina as a viable, sustainable alternative to traditional animal sources, reducing environmental impacts from livestock farming. Also, a great source for people that don't consume animal-based proteins.Vitamin B12 deficiency affects over a billion people worldwide, leading to serious health issues. This study presents Spirulina as a viable, sustainable alternative to traditional animal sources, reducing environmental impacts from livestock farming.Many medications deplete the body of B vitamins. Examples include acid reflux medications such as the Proton Pump Inhibitors (PPIs), Prednisone, Metformin, Antibiotics, Birth Control Pills, Anti-seizure medications, Antidepressants, Chemotherapy drugs such as Methotrexate, and several others. Research has shown that the elderly who take three or more medications are prone to B vitamin deficiencies such as B2, B6, Folate, and B12.Boosting B vitamin levels quickly helps improve energy. A deficiency of just one B vitamin will negatively affect the complex production of energy (ATP) in your cells.Örlö is the future of nutrition – omega-3s and immune boosting micronutrients that provide all the benefits - without damaging earth's ecosystems - so people and the planet can thrive. Visit orlonutrition.com today.  Get an exclusive 20% discount at Orlonutrition.com when using the code FOREVER at checkout. (Does not work on Subscriptions).

Today, we're going to take a look at the underlying cause of autoimmune disease. Your immune system has two parts: the innate immune system that you're born with and the acquired immune system that's created through a series of infections. Over time, the acquired immune system becomes stronger and offers protection against infection. The innate immune system is not the problem when it comes to autoimmune diseases. Autoimmune disorders typically involve problems with the T-regulatory cells or T-cells. Also called suppressor T-cells, these cells are the peacemakers of the immune system and stop the immune reaction when the job is done. Prednisone, the synthetic version of cortisol, is commonly used as a treatment for autoimmune diseases. Both prednisone and vitamin D have reduce inflammation, but Vitamin D empowers and enhances the immune system. Prednisone suppresses the innate and the acquired immune systems. Vitamin D shifts the immune system to increase the T-cells. Inflammatory TH1 and TH17 cells are usually too high when someone has an autoimmune disease. Vitamin D suppresses both of these cells, reducing inflammation. Prednisone raises your blood glucose levels while vitamin D does not. Vitamin D enhances the cells that makes insulin and helps to regulate your blood sugars. Prednisone can break down your bone, leading to osteoporosis. Vitamin D helps you absorb calcium and supports bone remineralization. The thymus gland makes T-cells. As we age, the thymus gland deteriorates and our immune systems decline. Vitamin D slows down this process, but you need larger, therapeutic doses. Vitamin D does not work without the cofactors magnesium, vitamin K2, and zinc. Dr. Coimbra of Brazil created a protocol that involves increasing vitamin D to penetrate any resistance. This lowers the parathyroid hormone which means that vitamin D levels have significantly increased. DATA: https://www.coimbraprotocol.com/gener... https://ajcn.nutrition.org/article/S0... http://www.vitamindprotocol.com/vitam...

Let's talk about the health benefits of turmeric. One study found that curcumin was just as effective as ibuprofen at reducing pain from arthritis without side effects! It's also shown to be as effective as aspirin. Another study showed that curcumin had comparable results to Prednisone in reducing inflammation for rheumatoid arthritis, asthma, and IBS. It has also been shown to produce antidepressant effects similar to Prozac and Zoloft. Curcumin has anti-diabetic properties and effects similar to Metformin. It also has anticoagulant properties similar to aspirin and Warfarin. Research has shown that curcumin has benefits similar to statins and can help reduce LDL cholesterol and triglycerides. One study compared curcumin to 5-fluorouracil, a chemotherapy drug. Turmeric has been shown to be as effective as anti-inflammatory drugs, especially for irritable bowel disease and digestive problems. It may also help reduce blood pressure and inhibit pathogens, especially fungi. To prepare turmeric water, combine ½ teaspoon of turmeric powder, a pinch of black pepper, and half of a lemon in a glass of warm water. To prepare golden milk, combine ½ teaspoon of turmeric, ¼ teaspoon of cinnamon, and a pinch of black pepper in a cup of milk or coconut milk. Heat the mixture and remove from heat just before it comes to a boil. You can also add turmeric to a smoothie with berries and kefir. If you have a cough, try drinking a cup of hot water with a teaspoon of turmeric and a tablespoon of raw honey. DATA: https://www.ncbi.nlm.nih.gov/pmc/arti... https://pubmed.ncbi.nlm.nih.gov/10404... https://pubmed.ncbi.nlm.nih.gov/23832... https://www.ncbi.nlm.nih.gov/pmc/arti... https://www.sciencedirect.com/science... https://www.ncbi.nlm.nih.gov/pmc/arti... https://www.sciencedirect.com/science... https://pubmed.ncbi.nlm.nih.gov/17101... https://pubmed.ncbi.nlm.nih.gov/23142... https://www.ncbi.nlm.nih.gov/pmc/arti...

DOWNLOAD THE WALLET CUTOUT CARD HERE: https://bit.ly/3TsJJ8s In this podcast, I'm going to tell you how to reshape your face naturally and get rid of a moon face. One of the most common symptoms of Cushing's syndrome is a “moon face.” Cushing's syndrome is characterized by high levels of the stress hormone cortisol. Chronic high cortisol over a period of time can cause fat face syndrome. It can also be caused by the drug Prednisone. Cortisol can redistribute fat to different parts of the body, causing you to store fat in the face and the midsection. Over time, it can cause a hump on the back, red cheeks, thinner legs, and weak muscles in the legs and buttocks. Your cholesterol and lipids may rise, and you'll start losing potassium and retaining sodium. This sends your blood pressure straight up, vitamin D levels decrease, and your immune system suffers. Cortisol indirectly causes a puffy face by breaking down protein and turning it into glucose. This spikes insulin, a fat-storing hormone. In the presence of too much insulin, you won't be able to lose fat. Carbohydrates, frequent eating, seed oils, nutrient deficiencies, stress, overexercising, and certain medications also trigger insulin. Try these 5 tips to reshape your face. 1. Eat less frequently Cut out the snacks between the meals and do intermittent fasting. This forces your body to use body fat as fuel between meals. 2. Change your diet Cut down your carb intake, specifically avoiding sugar and starch. Print my card cutout that shows you the ingredients to avoid! 3. Increase protein Look for high-quality grass-fed meat such as beef, lamb, or goat. 4. Lower stress levels Go for long walks, exercise, and do physical work. Avoid excessive phone use. 5. Try to get extra sleep Make sure you're getting enough sleep. Even if you wake up early, try to go back to sleep if possible!

In this CCO Nephrology podcast episode, hear from nephrologists Pietro Canetta, MD, MS, and Andy Bomback, MD, PhD, experts in clinical management and research on glomerular diseases as they discuss key updates in managing IgAN. Faculty highlight the importance of a comprehensive supportive care regimen to protect patients' kidneys and prevent progression of disease. In addition, they review the merits and place in therapy of novel and emerging therapies.  Topics include:Supportive care as the foundation of IgAN managementPlace in therapy for new and emerging agentsTargeted-release formulation of budesonideEndothelin receptor antagonists (eg, sparsentan)Factor B inhibitors (eg, iptacopan)The role of clinical trial involvementLearn more about IgA nephropathy with educational activities and resources here:  CME-certified text module with animated pathophysiology video and patient voice audio clipClinicalThought commentariesResources on IgAN from the American Kidney Fund

Today, I'm going to share a few natural sciatica pain remedies! The sciatic nerve is the longest and largest nerve in the body. It extends from the lower back, down the back of the leg, to the bottom of the foot. Surgery for sciatica can cause side effects like failed back surgery syndrome. Because 90% of sciatica pain will often resolve by itself, invasive procedures are best saved as a last resort. Sciatic pain often gets worse in the winter, which would explain why vitamin D deficiency is a major contributor. If you're dealing with inflammation, you need high amounts of vitamin D daily. Vitamin D is a potent anti-inflammatory and works similarly to Prednisone without the side effects. It helps to repair the nerves, disc, connective tissue, and muscles involved with sciatica. Magnesium deficiency can also contribute to sciatica because it's essential for keeping your muscles relaxed. You also need adequate magnesium for vitamin D to work in the body. Vitamin B12 is involved with several aspects of the nervous system and can cause numbness, tingling, and burning pain if you're deficient. It's highly anti-inflammatory, and a deficiency can also contribute to sciatica. Most people don't get enough sun, so unless you're taking 10,000 IU of vitamin D each day, you could end up deficient. If you have sciatica, try upping your dose of vitamin D3 to 20,000 to 30,000 IU daily. Around 800 mg of magnesium each day can be beneficial for sciatica. Sunflower seeds, pumpkin seeds, chocolate, almonds, leafy greens, bone broth, and spinach are good food sources of magnesium. Red meat and other animal products provide plenty of vitamin B12. Try these simple sciatica exercises focused on stretching the fascia to say goodbye to your sciatic nerve pain!

At age two, Jon Brent was diagnosed with a type of blood cancer, acute lymphoblastic leukemia.  He underwent an aggressive chemotherapy regimen, including vincristine, dexamethasone, methotrexate and prednisone, but after that still needed a bone marrow transplant.  He has achieved survivorship but is still in pain and expects to some extent he will be for the rest of his life.  Jon can no longer compete in contact sports but is an active participant in ultimate Frisbee.

On this episode of the Real Life Pharmacology podcast, I continue my education on the top 200 drugs. Raloxifene, prednisone, phenytoin, fish oil, and ezetimibe are covered in this podcast episode. Prednisone is a corticosteroid that may cause hyperglycemia, insomnia, GI upset, osteoporosis, HPA suppression, and hypertension as primary adverse effects. Raloxifene is classified as a SERM and can be used for osteoporosis and breast cancer. DVT and hot flashes are significant adverse effect concerns. Fish oil (Lovaza) is used to reduce triglycerides. Elevated triglycerides can increase the risk of pancreatitis. Ezetimibe inhibits the absorption of cholesterol through the gut. It lowers LDL but not to the extent of statins. Phenytoin is a narrow therapeutic index medication (NTI) that is used as an anticonvulsant. Ataxia, confusion, GI upset, and vertical nystagmus are potential signs of toxicity.

In this CCO Nephrology podcast episode, hear from Kelly Chen, a nephrology nurse practitioner and patient living with IgA nephropathy, as she discusses her experiences with Dr Pietro Canetta, Associate Professor of Medicine at Columbia University and expert in glomerular diseases. Episode outline: Seeking and obtaining an IgAN diagnosis Patient self-advocacy Individualizing IgAN treatment plansClinical trial involvement Burdens of IgAN: physical, emotional, social, and beyondTo learn more about IgA nephropathy, find more educational activities and resources with the links below: CME-certified text module with animated pathophysiology video and patient voice audio clipsClinicalThought commentaries Resources on IgAN from the American Kidney Fund 

Episode 2496 - On this Monday's show Vinnie Tortorich and Anna Vocino chat about Vinnie's cancer protocol, dealing with insurance companies, and more. https://vinnietortorich.com/2024/06/dealing-with-insurance-episode-2496 PLEASE SUPPORT OUR SPONSORS YOU CAN WATCH ALL THE PODCAST EPISODES ON YOUTUBE - Dealing With Insurance Vinnie is grateful for all the well-wishes from his followers/supporters. (2:00) He gives an update about his cancer treatment, and he also wants to discuss advocating for yourself. (3:00) He advocated for himself regarding his medications. He is not against doctors, he but emphasizes the importance of communicating with them. (15:00) You don't have to get adversarial, just be sure to have the conversations and advocate for yourself. Anna was on Prednisone recently and it helped her, but she was miserable on it. (17:00) They discuss insurance; you pay for insurance to ensure you get the care you need when needed. (21:30) Vinnie had an injection that cost $16,000! He explains where there was a discrepancy between the hospital and the pharmacy Anna shares her experience with insurance regarding her recent hysterectomy. (30:00) There are insurance companies that fight patients' insurance until the patients have to file bankruptcy. Anna tells a story about a wine tasting, a prank, and a celebrity or two. (47:00) The “Dirty Keto” documentary landing page should be up by May 30th. You can go to Vinnie's website and enter your email to get notified. When it comes out, make sure you watch and rate it. There's a new NSNG® Foods promo code you can use! (53:00) The promo code ONLY works on the NSNG® Foods website, NOT on Amazon. Anna's upcoming Eat Happy Italian book is on the way. You can go to: —upload your receipt and get bonus content.  You can preorder it from . [the_ad id="20253"] PURCHASE BEYOND IMPOSSIBLE (2022) The documentary launched on January 11! Order it TODAY! This is Vinnie's third documentary in just over three years. Get it now on Apple TV (iTunes) and/or Amazon Video! Link to the film on Apple TV (iTunes):  Then, Share this link with friends, too! It's also now available on Amazon (the USA only for now)!  Visit my new Documentaries HQ to find my films everywhere: REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter! FAT: A DOCUMENTARY 2 (2021) Visit my new Documentaries HQ to find my films everywhere: Then, please share my fact-based, health-focused documentary series with your friends and family. The more views, the better it ranks, so please watch it again with a new friend! REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter! FAT: A DOCUMENTARY (2019) Visit my new Documentaries HQ to find my films everywhere: Then, please share my fact-based, health-focused documentary series with your friends and family. The more views, the better it ranks, so please watch it again with a new friend! REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter!  

In this JCO Article Insights episode, Alexandra Rojek provides a summary on "Romidepsin Plus Cyclophosphamide, Doxorubicin, Vincristine, and Prednisone Versus Cyclophosphamide, Doxorubicin, Vincristine, and Prednisone in Patients With Previously Untreated Peripheral T-Cell Lymphoma: Final Analysis of the Ro-CHOP Trial" by Camus et al published February 16th, 2024 and the associated editorial written by Dr. Mehta-Shah and Dr. Horwitz. TRANSCRIPT The guest on this podcast episode has no disclosures to declare. Alexandra Rojek: Hello and welcome to JCO Article Insights. I'm your host, Alexandra Rojek, and today we will be discussing a clinical trial update published in the May 10th issue of JCO addressing the long term follow up of the addition of romidepsin to CHOP chemotherapy for previously untreated peripheral T-cell lymphoma, or PTCL. This report by Camus et al discusses a five-year follow up of the original Ro-CHOP trial, which did not meet its primary endpoint of progression free survival. The original Ro-CHOP study, conducted by the LYSA group, was published in 2021 in JCO and was conducted as a one-to-one randomized phase III study of Ro-CHOP versus CHOP for patients aged 18 to 80 years with PTCL. 98 international centers were included, and the study enrolled patients between 2013 and ‘17. Nodal follicular helper T-cell, or TFH lymphoma was defined in this study follow-up as a PTCL expressing at least two of five TFH markers according to the latest classifications. The study's primary endpoint was PFS with secondary endpoints of OS and duration of response, or DOR. Five year follow up was reached in December 2022. In the original study report, the addition of romidepsin to CHOP did result in a reduction of dose intensity of chemotherapy. However, in this updated follow up, there were no new safety signals reported. A total of 421 patients were enrolled in the trial with a median age of 65 years. Notably, those who were randomized to the Ro-CHOP arm had a higher proportion of IPI 4-5 scores at 33%, versus 24% of those who were assigned to CHOP alone despite randomization. Nearly half of patients carried a histologic diagnosis of angioimmunoblastic T-cell lymphoma. 30% were characterized as PTCL NOS, not otherwise specified, 10% ALK negative anaplastic large cell lymphoma, leaving 13% reported as other. Over 60% of patients had stage four disease at enrollment, with nearly half having more than two sites of extranodal involvement. Median follow up was six years with a median PFS of 12 months for Ro-CHOP and 10.2 months for CHOP, which did not reach statistical significance as reported in the original study publication. Estimated five year OS available as a part of this clinical trial update was 50% for Ro-CHOP and 43% for CHOP alone, and also did not reach significance. There was, however, a longer duration of response observed in the Ro-CHOP arm at 52 months versus 24 months for CHOP, which is a new finding in this extended follow up of the study. In an effort to better understand whether there are subgroups which may benefit from romidepsin despite the overall negative outcome of this study, the authors are able to provide new insights from this Ro-CHOP study in line with our current and updated understanding of PTCL. When the authors evaluated the study population for subgroups which may benefit from addition of romidepsin, TFH lymphomas, which included angioimmunoblastic, follicular, and NOS subtypes, were noted to have an improved response to the addition of this HDAC inhibitor. This subgroup, numbering 201 patients, experienced a mean PFS of over 19 months with Ro-CHOP versus over 10 months for those who received CHOP, and this difference achieved statistical significance. Similarly, there was a higher complete response rate and longer duration of response for those in the TFH subgroup who received romidepsin. The authors also make note that those patients in this subgroup with high IPI appeared to particularly benefit. However, those in the PTCL NOS group who did not fit the TFH subtype experienced poor outcomes with shorter PFS and OS as compared to other histologic subtypes, which is in line with other reported data in the field. The authors in this update also report on treatments and outcomes after first progression or relapse of disease, which includes a total of 274 patients, of whom 251 received second line therapy. The median PFS in OS after progression or relapse was only 3.3 months and 11.5 months, respectively, and again, patients with the TFH subtype experienced a longer median OS than other included histologies. Only 8% of patients proceeded to undergo autologous stem cell transplantation overall, and 5% underwent allogeneic stem cell transplantation. While the authors note that they did not observe any notable outcome differences between various included second line therapies, they do note of a possible benefit of the antibody drug conjugate brentuximab vedotin or BV in combination with chemotherapy backbones. Those who received a BV containing regimen in the second line experienced a better CR rate and a longer PFS, too. However, the OS too was not significantly different. Again, the authors note that most of the benefit of the addition of BV to chemotherapy in the second line appeared focused on this TFH lymphoma subgroup. Of note, CD30 status was not recorded as a part of the study for included patients and could not be further reported on. The findings of the authors present in this subgroup analysis of the negative Ro-CHOP study seek to find benefit in future lessons for patients with these rare lymphomas who do not have a multitude of effective treatments available to them. The subgroup benefit in this clinical trial update based on the TFH phenotype fits our evolving understanding of PTCL in the modern era. Mutations characteristic of the TFH subgroup, in this case often involved in chromatin modification and the tumor microenvironment, have been linked to improved responses when treating with epigenetic targeting drugs such as romidepsin, thus making the findings of TFH lymphoma in the Ro-CHOP trial confirmatory of what we would expect from correlative studies. Despite the benefit of Ro-CHOP for those with TFH lymphoma, this updated analysis of a major frontline study of PTCL highlights the critical need in the T-cell lymphoma field to, first of all, better understand and separate what we have come to recognize as the heterogeneous diseases historically lumped together as PTCL. This historic classification, based on a lack of deeper molecular understanding of disease biology is unlikely to represent the true biologic diversity of its component subtypes, including the focus on TFH lymphoma presented here. The second critical unmet need, which this subgroup analysis highlights, is the need to investigate the benefit of new therapeutic agents in these distinct subgroups of PTCL, where the need for improved survival outcomes is sorely needed when examining historical outcomes with currently available therapies, both in the frontline setting as well as those in the relapsed refractory group. The associated editorial, written by Dr. Mehta-Shah and Dr. Horwitz, elegantly discusses the challenges of designing studies in rare diseases such as PTCL, which are both biologically informed as well as appropriately powered. As the authors discuss, it is rare to get a second chance for a drug, as is the case for romidepsin, where these negative results led to its voluntary withdrawal in PTCL, affecting availability of the drug for patients in the relapsed setting, as well as future clinical trials, which could have had potential for success. Additionally, better understanding of the biological underpinnings of PTCL needs to lead to better designed and appropriately powered clinical trials, as these subsets of patients are in great need of therapeutic advances. While none of these tasks is easy nor straightforward, this clinical trial update of the RoCHOP study suggests a path forward from learning from prior, promising yet failed attempts at moving the standard of care for frontline therapy of T-cell lymphoma forward. This is Alexandra Rojek thank you for listening to JCO Article Insights. Don't forget to give us a rating or review and be sure to subscribe so you never miss an episode. You can find all ASCO shows at ascp.org/podcasts. The purpose of this podcast is to educate and to inform. This is not a substitute for professional medical care and is not intended for use in the diagnosis or treatment of individual conditions. Guests on this podcast express their own opinions, experience, and conclusions. Guest statements on the podcast do not express the opinions of ASCO. The mention of any product, service, organization, activity, or therapy should not be construed as an ASCO endorsement.  

*Check out EZ's morning radio show "The InZane Asylum Q100 Michigan Mornings with Eric Zane" Click here*Support the show and enjoy great products! Check out EZ on Vouch!*Get a FREE 7 day trial to Patreon to "try it out."*Watch the show live, daily at 8AM EST on Twitch! Please click here to follow the page.Email the show on the Shoreliners Striping inbox: eric@ericzaneshow.comTopics:*EZ on cloud nine this morning as the Prednisone kicks in. Despite the highly regarded opinions of show dopes, Kenney, Joe Martinez and David in Hudsonville, EZ is listening to his doctor.*Discount Tire in Grandville, Michigan is the freaking best. *A police chase started in "The City-a-Warn." That led EZ to revisiting his time on "89.1 WPHS."*Griffs beat Milwaukee*Boxing announcer botches winning fighter call; gets butthurt because internet is mad at him.*An EZ Show audience member second guesses his doctor.*Girl wears dude's suit to prom, gets sent home. Told to dress like a girl.*Former Grand Rapids Christian Services worker behaved in a "not-so-Christian" way....because he stuck his ding dong in a child.*Asshole of the Day BTYB TC PaintballSponsors:Berlin Raceway, Impact Powersports, Frank Fuss / My Policy Shop Insurance, Kings Room Barbershop, A&E Heating and Cooling, The Mario Flores Lakeshore Team of VanDyk Mortgage, Shoreliners Striping, Ervines Auto Repair Grand Rapids Hybrid & EV, TC PaintballInterested in advertising? Email eric@ericzaneshow.com and let me design a marketing plan for you.Contact: Shoreliners Striping inbox eric@ericzaneshow.comDiscord LinkEZSP TikTokSubscribe to my YouTube channelHire me on Cameo!Tshirts available herePlease subscribe, rate & write a review on Apple Podcastspatreon.com/ericzaneInstagram: ericzaneshowTwitterOur Sponsors:* Check out Express VPN: www.expressvpn.com* Check out eufy: us.eufy.comSupport this podcast at — https://redcircle.com/the-eric-zane-show-podcast/donationsAdvertising Inquiries: https://redcircle.com/brandsPrivacy & Opt-Out: https://redcircle.com/privacy

Do you have a prescription for Prednisone? Save up to 85 percent off your medication on your next trip to the pharmacy by using CareCard! Visit https://carecard.com/drugs/prednisone for more. CareCard City: New York Address: 6 St Johns Ln. Website: https://www.carecard.com

Live IVIRMA Journal Club from February 15th. Dr. Pavan Gill presents an article from JAMA on the role of Prednisone versus Placebo for Recurrent Implantation Failure. Drs. Elena Labarta and Filippo Ubaldi comment on the effect of prednisone and its potential harms, followed by Q&A from the audience including expert commentary by Dr. Diana Alecsandru. Podcast website: https://www.ivi-rmainnovation.com/fertilipod/

Dylan and Connor are joined by Solea Pfeiffer (Hadestown, Almost Famous). The twins welcome their literal queen Solea Pfeiffer to the DRAMA fam in this wonderful, free-flowing, insightful chat that's been years in the making. After pulling double and triple duty over the last year, Solea takes us through her various projects, including starring as Eurydice in Hadestown and Ophelia in Hamlet last summer. From making her Broadway debut as Penny Lane in Almost Famous to being NYCC's Evita and Songs for a New World Songstress, Solea is taking the theatre world by storm. She gives great insight into her mindset as a performer, shares her love story with her boyfriend Kevin Csolak, gushes over Betty Who and even spills the tea on her favorite Drag Race season 16 queens. We dive into country music, Vanessa Williams in The Devil Wears Prada, and Studio Ghibli standom. You must… love… ME.Follow Solea on InstagramFollow DRAMA. on Twitter & Instagram & TiktokFollow Connor MacDowell on Twitter & InstagramFollow Dylan MacDowell on Twitter & InstagramEdited by DylanSupport the podcast by subscribing to DRAMA+, which also includes bonus episodes, Instagram Close Friends content, and more!

Scott ended up in the Hospital this week for 3 full day Plusl.  He did not see this coming and it has rocked his world.  We are going to dive into what happend and look for different ways we can do this nutritionally

In this podcast, we're going to look at the root causes of inflammation and the best natural remedies for inflammation. Here are 10 common causes of inflammation. 1. Gluten consumption and food allergies Gluten can lead to a leaky gut, a condition where food particles leak through the small intestine. Leaky gut is one of the most common causes of inflammation. Pinpointing a food allergy can also help clear up gut inflammation. 2. Ultra-processed food ingredients Ultra-processed foods like synthetic sugars, modified food starches, and seed oils go through several industrial processes to the point that they no longer resemble their original form. These foods cause free-radical damage and inflammation in the body. 3. Viruses Viruses can go in and out of remission, causing inflammatory conditions that come and go. This is often triggered by stress which increases your cortisol levels, suppressing your immune system. Viruses can also block vitamin D. 4. Insulin resistance Frequent eating and carb consumption leads to insulin resistance. Most of the population is insulin-resistant. 5. Lack of cortisol Chronic stress over a long period and medications like Prednisone can lead to cortisol resistance. 6. Old injuries Lack of movement and a sedentary lifestyle can lead to inflammation. 7. Sludge in bile ducts Without adequate bile salts, bile sludge can back up into the liver causing right shoulder pain and rhomboid pain. This can be corrected by a healthy diet. 8. Too much iron This is typically caused by a genetic issue and is more common in men. 9. Hypoxia Conditions like COPD, cirrhosis of the liver, clogged arteries, and sleep apnea can cause hypoxia—a lack of oxygen. 10. Uric acid Uric acid is one of the most common causes of high blood pressure and comes from high fructose consumption. Here are the best remedies for inflammation: •Kefir, sauerkraut, kimchi, or a probiotic •Vitamin D3 •Intermittent fasting and periodic prolonged fasting •Cruciferous vegetables and salads •Keto diet •Carnivore diet •TUDCA •Tocotrienols •Cold therapy •Stretching and exercise •Omega-3 fatty acids (cod liver oil, sardines, salmon)

In this podcast, we're going to talk about candida overgrowth. Candida is a fungal infection that typically affects the mouth, private areas, or toenails. Candida often develops after antibiotics destroy the bacteria that keep yeast and fungus at bay. The majority of your gut microbiome is bacteria. However, you have a small percentage of yeast, fungus, and friendly candida. When antibiotics wipe out your friendly bacteria, this leaves room for candida overgrowth. Some of the symptoms of candida overgrowth include itchiness, burning, fatigue, cracks at the corners of the mouth, and ammonia odor. Candida changes your pH, making it more alkaline. Sugar is the primary fuel source of candida. Other conditions can lower your immune system and cause candida overgrowth. HIV/AIDS, organ transplants, medications like Prednisone, pregnancy, and excessive sugar consumption can all cause candida. Oftentimes, candida overgrowth is paired with a vitamin D deficiency. Vitamin D has antifungal properties and is also a broad-spectrum antimicrobial. If you have candida, there are three things that you need to do! 1. Take 20,000 to 30,000 IU of vitamin D 2. Remove sugar from the diet 3. Consume probiotic foods (sauerkraut, kimchi, kefir, fermented vegetables) DATA: https://www.sciencedirect.com/science... https://vitamindwiki.com/tiki-index.p... SUBSCRIBE TO MY NEWSLETTER HERE: httphttps://bit.ly/49rtbTB

We are on a mission to get as many lucrative sponsorship deals as possible!!!! Air Up, Prednisone, Blue Chew, Celsius, Impossible Meats, Dude Wipes - please reach out post haste. Our listeners care deeply about we have to say. And what's even better is that we are joined by renowned NYC funny lady Lauren Servideo on our quest.

Every year, a few times a year, I have a bad reaction to poison ivy. This time, it has affected my eye. Each time I was exposed and had a bad reaction, I was given prednisone. It got to the point where, after I was exposed, the effects of the poison ivy wouldn't go away after using prednisone. People typically take prednisone for its anti-inflammatory or immune suppressant effects. But prednisone has side effects. Prednisone is a synthetic version of cortisol. Cortisol has benefits. It's a hormone that helps the immune system and prepares the body for stress. But high cortisol levels can lead to a lot of health concerns. You don't want to be on prednisone forever. The more you take it, the more it weakens the system. But if you stop taking it, your symptoms may come back. So, why don't we get to the root problem? Keep in mind that once you start taking prednisone, it's important not to come off of it too fast. Prednisone and cortisol also deplete the body of numerous vitamins and minerals. The best ways to lower cortisol: 1. Lower your stress (do physical work, exercise, spend more time on a hobby, and take certain nutrients) 2. Take ashwagandha 3. Try breathing exercises 4. Use an HRV device to better understand your stress Download ALL 25 Natural Ways to Lower Cortisol: https://drbrg.co/3Tk2eN5 DATA: https://karger.com/nen/article/92/Sup...

Omega-3s help isotretinoin - Covid and autoimmune disease - Transient abdominal telangiectasia of the newborn - Systemic tacrolimus in derm - MTX + prednisone for alopecia totalis/universalis - Tofacitinib for BP - Check out our video content on YouTube: https://www.youtube.com/@dermaspherepodcast and VuMedi!: https://www.vumedi.com/channel/dermasphere/ The University of Utah's Dermatology ECHO: ⁠⁠https://physicians.utah.edu/echo/dermatology-primarycare - ⁠⁠Connect with us! - Web: ⁠https://dermaspherepodcast.com/⁠ - Twitter: @DermaspherePC - Instagram: dermaspherepodcast - Facebook: https://www.facebook.com/DermaspherePodcast/ - Check out Luke and Michelle's other podcast, SkinCast! ⁠https://healthcare.utah.edu/dermatology/skincast/⁠ Luke and Michelle report no significant conflicts of interest… BUT check out our friends at: - ⁠Kikoxp.com ⁠(a social platform for doctors to share knowledge) - ⁠https://www.levelex.com/games/top-derm⁠ (A free dermatology game to learn more dermatology!)

Sable can smell again (thank you prednisone) so this is a big catch-up chat that includes new scents launched this Fall, the Glossier You formula controversy, Tynan's second (Chanel) Chance, The Kayali Oudgasm universe, soft boy smells, Halloween scents,  and MORE.Find more info, episodes, and merch at Smellyalater.liveLeave us a message on the SYL Hotline at Speakpipe.com/smellyalaterFollow us on Instagram @smellyalater.mp3Leave a (nice) comment & (5-star) review wherever you stream

Detailed Shownotes for This Episode of The 6-8 Weeks Podcast:-- What is Knee Arthritis? https://www.hopkinsmedicine.org/health/conditions-and-diseases/knee-arthritis#:~:text=What%20is%20knee%20arthritis%3F,to%20smoothly%20bend%20and%20straighten.-- What is Bursitis? https://www.mayoclinic.org/diseases-conditions/bursitis/symptoms-causes/syc-20353242-- What is Return to Play? https://www.nm.org/healthbeat/healthy-tips/what-does-return-to-play-really-mean-- How Often Does Arthritis Impact Construction Workers? https://www.safetyandhealthmagazine.com/articles/20166-construction-agricultural-workers-at-higher-risk-of-knee-osteoarthritis-study-- What is an Anti-Inflammatory? https://www.healthline.com/health/pain-relief/otc-anti-inflammatories-- What is Advil? https://www.advil.com/faqs-en/-- What is Aleve? https://www.aleve.com/frequently-asked-questions-safety-and-usage?gclid=CjwKCAjwp8OpBhAFEiwAG7NaElXGOEjUEaFYifyMEVBvwGwZaj2pvDOTihkIH0ASBdL0rycLV3ewkRoCtXAQAvD_BwE-- What is Motrin? https://www.motrin.com/what-is-motrin-- What is Meloxicam? https://www.mayoclinic.org/drugs-supplements/meloxicam-oral-route/description/drg-20066928-- How Does the Human Bloodstream Carry Medications? https://www.nigms.nih.gov/education/Inside-Life-Science/Pages/A-Medicines-Life-Inside-the-Body.aspx-- What is Prednisone? https://my.clevelandclinic.org/health/drugs/20469-prednisone-tablets-- Learn All About the Knee Joint: https://www.youtube.com/watch?v=_q-Jxj5sT0g-- What is Synovium? https://www.physio-pedia.com/Synovium_%26_Synovial_Fluid-- What is a Narcotic? https://www.dea.gov/sites/default/files/2020-06/Narcotics-2020.pdf-- What is Vicodin? https://www.deadiversion.usdoj.gov/drug_chem_info/hydrocodone.pdf-- What is Norco? https://www.accessdata.fda.gov/drugsatfda_docs/label/2019/040148s073lbl.pdf-- What is Hip Arthritis? https://www.hopkinsmedicine.org/health/conditions-and-diseases/hip-arthritis-- What are Meniscus Root Tears? https://tcomn.com/meniscus-root-tears-on-the-rise/-- Be Sure to Listen to Our Episode on Meniscus Tears! https://music.amazon.com/podcasts/74f824ce-3c64-4e14-8c64-da985a8ea19a/episodes/d4b9f236-ce61-4abb-9eb5-dba27edd8a7d/6-8-weeks-perspectives-on-sports-medicine-meniscus-tears---when-should-you-worry-- What is Knee Replacement? https://www.mayoclinic.org/tests-procedures/knee-replacement/about/pac-20385276-- Learn All About the British Medical Journal: https://www.bmj.com/-- What are the Tell-Tale Symptoms of Arthritis? https://www.painandtherapy.com/blog/5-telltale-signs-of-arthritis-- What is a Knee Injection? https://www.mayoclinic.org/drugs-supplements/hyaluronic-acid-injection-route/description/drg-20074557-- What is a Spine Injection? https://orthoinfo.aaos.org/en/treatment/spinal-injections/-- Learn All About the Journal of The American Medical Association (JAMA): https://jamanetwork.com/journals/jama -- What is a Placebo? https://www.youtube.com/watch?v=5RhG_ySxhDA-- What is the Value of Utilizing a Placebo? https://www.health.harvard.edu/mental-health/the-power-of-the-placebo-effect-- What is an ACL Tear? https://orthoinfo.aaos.org/en/diseases--conditions/anterior-cruciate-ligament-acl-injuries/-- What is Bicep Tendinitis? https://orthoinfo.aaos.org/en/diseases--conditions/biceps-tendinitis/-- What is Arthroscopic Surgery? https://losrobleshospital.com/your-health/video/what-is-arthroscopy-or-arthroscopic-surgery-- What is a Hamstring?— A Little Something About Steroid Injections to Read Over on Reddit:https://www.reddit.com/r/science/comments/j32d4q/cortisone_injections_increased_risk_for_knee/— Will There Be Trouble When Getting Injections Prior to Surgeries?https://pubmed.ncbi.nlm.nih.gov/37058158/https://pubmed.ncbi.nlm.nih.gov/36737032/https://pubmed.ncbi.nlm.nih.gov/35829737/— Should You Get a Steroid Injection Before Rotator Cuff Surgery?https://pubmed.ncbi.nlm.nih.gov/36734466/ — Learn About the Differences Between Cuff tendinopathy Vs PRP: https://pubmed.ncbi.nlm.nih.gov/34020672/=== Connect with Dr. Brian Feeley:On the Web: https://twitter.com/drbrianfeeley On X: https://twitter.com/drbrianfeeley=== Connect with Dr. Nirav Pandya:On the Web: https://www.ucsfhealth.org/providers/dr-nirav-pandyaOn X: https://twitter.com/drniravpandya=== Connect with Dr. Drew Lansdown:On the Web: https://www.ucsfhealth.org/providers/dr-drew-lansdown

We all have preconceived notions when it comes to "injected steroids". Now - thanks to our three orthopedic surgeons, it's time to learn all about HOW AND WHY, in many cases, injecting steroids makes definitive sense. Whether it be to treat a soon-to-be-vacationing construction worker or a troubled athlete, all of the answers to resolve your questiosn are here inside this episode of The 6-8 Weeks Podcast. Connect with The 6-8 Weeks Podcast: There's a LOT of detail included in this program. Do you want to share YOUR perspective about it? Connect with The 6-8 Weeks Podcast Now! Subscribe to, Like and Share The 6-8 Weeks Podcast Everywhere:     The Detailed Shownotes for This Episode of The 6-8 Weeks Podcast: -- What is Knee Arthritis? https://www.hopkinsmedicine.org/health/conditions-and-diseases/knee-arthritis#:~:text=What%20is%20knee%20arthritis%3F,to%20smoothly%20bend%20and%20straighten. -- What is Bursitis? https://www.mayoclinic.org/diseases-conditions/bursitis/symptoms-causes/syc-20353242 -- What is Return to Play? https://www.nm.org/healthbeat/healthy-tips/what-does-return-to-play-really-mean -- How Often Does Arthritis Impact Construction Workers? https://www.safetyandhealthmagazine.com/articles/20166-construction-agricultural-workers-at-higher-risk-of-knee-osteoarthritis-study -- What is an Anti-Inflammatory? https://www.healthline.com/health/pain-relief/otc-anti-inflammatories -- What is Advil? https://www.advil.com/faqs-en/ -- What is Aleve? https://www.aleve.com/frequently-asked-questions-safety-and-usage?gclid=CjwKCAjwp8OpBhAFEiwAG7NaElXGOEjUEaFYifyMEVBvwGwZaj2pvDOTihkIH0ASBdL0rycLV3ewkRoCtXAQAvD_BwE -- What is Motrin? https://www.motrin.com/what-is-motrin -- What is Meloxicam? https://www.mayoclinic.org/drugs-supplements/meloxicam-oral-route/description/drg-20066928 -- How Does the Human Bloodstream Carry Medications? https://www.nigms.nih.gov/education/Inside-Life-Science/Pages/A-Medicines-Life-Inside-the-Body.aspx -- What is Prednisone? https://my.clevelandclinic.org/health/drugs/20469-prednisone-tablets -- Learn All About the Knee Joint: https://www.youtube.com/watch?v=_q-Jxj5sT0g -- What is Synovium? https://www.physio-pedia.com/Synovium_%26_Synovial_Fluid -- What is a Narcotic? https://www.dea.gov/sites/default/files/2020-06/Narcotics-2020.pdf -- What is Vicodin? https://www.deadiversion.usdoj.gov/drug_chem_info/hydrocodone.pdf -- What is Norco? https://www.accessdata.fda.gov/drugsatfda_docs/label/2019/040148s073lbl.pdf -- What is Hip Arthritis? https://www.hopkinsmedicine.org/health/conditions-and-diseases/hip-arthritis -- What are Meniscus Root Tears? https://tcomn.com/meniscus-root-tears-on-the-rise/ -- Be Sure to Listen to Our Episode on Meniscus Tears! https://music.amazon.com/podcasts/74f824ce-3c64-4e14-8c64-da985a8ea19a/episodes/d4b9f236-ce61-4abb-9eb5-dba27edd8a7d/6-8-weeks-perspectives-on-sports-medicine-meniscus-tears---when-should-you-worry -- What is Knee Replacement? https://www.mayoclinic.org/tests-procedures/knee-replacement/about/pac-20385276 -- Learn All About the British Medical Journal: https://www.bmj.com/ -- What are the Tell-Tale Symptoms of Arthritis? https://www.painandtherapy.com/blog/5-telltale-signs-of-arthritis -- What is a Knee Injection? https://www.mayoclinic.org/drugs-supplements/hyaluronic-acid-injection-route/description/drg-20074557 -- What is a Spine Injection? https://orthoinfo.aaos.org/en/treatment/spinal-injections/ -- Learn All About the Journal of The American Medical Association (JAMA): https://jamanetwork.com/journals/jama -- What is a Placebo? https://www.youtube.com/watch?v=5RhG_ySxhDA -- What is the Value of Utilizing a Placebo? https://www.health.harvard.edu/mental-health/the-power-of-the-placebo-effect -- What is an ACL Tear? https://orthoinfo.aaos.org/en/diseases--conditions/anterior-cruciate-ligament-acl-injuries/

If you don't already have children before the onset of your disease, it can be an overwhelming prospect. Will you still be able to properly care for a child? Is it fair to bring them into a home where you may be incredibly limited? What about pregnancy? Should you get pregnant? Will your disease or your medications negatively affect your child?   I go into all of this and more in this episode.   Please don't forget to click that subscibe button whereever you listen to the podcast and do me a HUGE favor and leave a review! These two things help others find the podcast, find our community, and feel a little less alone in their journey through life with chronic illness!   Join us on Instagram: https://www.instagram.com/teamvasculitis Join the Email List: https://teamvasculitis.com/team-vasculitis-email

During this episode, Heathar Shepard, classical homeopath, author of The Sunlight Rx and homesteader, discusses her concerns around steroid drugs, namely, prednisone. She discusses the side effects of prednisone, how long it takes to recover from its use, and why she never recommends this drug to anyone. Heathar also shares her concerns around surgery and how to use homeopathy to help recover post surgery.

During this episode Heathar Shepard, classical homeopath, author of The Sunlight Rx, homesteader and ancestral foods chef, takes you on a rheumatoid arthritis (RA) deep dive. She discusses western treatment and approach - how and why this is ineffective and immunosuppressive. She talks about root causes of RA, reveals the myth of anti-inflammatory foods, discusses the role of homeopathy vs the role of western medicine when treating RA and much more.

Thank you for joining us for our 2nd Cabral HouseCall of the weekend! I'm looking forward to sharing with you some of our community's questions that have come in over the past few weeks…   Kate: What to do for very large 9cm & 8cm fibroids?   Sidney: Hi doctor Cabral, I have a thick, deformed toenail and the skin around it often gets inflamed, and hurts a lot. I read that the only thing that helps is removing the nail altogether, but I would rather keep it. Is there any way to make it healthy again, or to stop it hurting as much? Thanks so much for you answer! Best, Sidney   Kristin: I've been suffering from hives for a year. They started with what I thought was a spider bite on my scalp. A few weeks later I started getting itchy red hives all over my body. I have seen my general practitioner, dermatologist and allergist. I did a ton of bloodwork and they couldn't find any underlying issues or allergies. Prednisone cleared it up, but came right back when stopped. Zyrtec (at first 4 a day, now just 1 a day) cleared up the body hives, but heat and tight clothing bring them back. The itchy bumps on my scalp have never gone away. I have tried apple cider vinegar, avocado oil, coconut oil. I changed bedding, shampoos, etc. I don't know what else to do. Do you have any advice?   Eric: Hi Dr. Cabral. I'm a 64-year-old male that has struggled for many years with urinary tract problems (urinary tract stricture as a child that was surgically treated, recurring bouts of urinary tract irritation from various substances and stress, but never any indication of a UTI). Over the past several months I've been doing infrared sauna sessions 2X per week, which seems to make my urinary tract problems worse. I use a binder before each sauna session. Two questions: (1) Can a sauna session trigger a large jump in toxins being processed through the kidneys/urinary tract, including oxalate dumping (I'm high in oxalates on the OAT test)? (2) How would you suggest one treat irritation symptoms (raw feeling) in the urethra/bladder area? Thanks!   Jessi: I've searched the podcasts and can't find that you have ever spoken about the SAAT procedure specifically used for Alpha gal reactions. I've read several testimonials that it has greatly benefited people dealing with this frustrating affliction. Thank you for all your free resources and I'm looking forward to hearing your opinion on this subject.    Thank you for tuning into this weekend's Cabral HouseCalls and be sure to check back tomorrow for our Mindset & Motivation Monday show to get your week started off right!   - - - Show Notes and Resources: StephenCabral.com/2704 - - - Get a FREE Copy of Dr. Cabral's Book: The Rain Barrel Effect - - - Join the Community & Get Your Questions Answered: CabralSupportGroup.com - - - Dr. Cabral's Most Popular At-Home Lab Tests: > Complete Minerals & Metals Test (Test for mineral imbalances & heavy metal toxicity) - - - > Complete Candida, Metabolic & Vitamins Test (Test for 75 biomarkers including yeast & bacterial gut overgrowth, as well as vitamin levels) - - - > Complete Stress, Mood & Metabolism Test (Discover your complete thyroid, adrenal, hormone, vitamin D & insulin levels) - - - > Complete Food Sensitivity Test (Find out your hidden food sensitivities) - - - > Complete Omega-3 & Inflammation Test (Discover your levels of inflammation related to your omega-6 to omega-3 levels) - - - Get Your Question Answered On An Upcoming HouseCall: StephenCabral.com/askcabral - - - Would You Take 30 Seconds To Rate & Review The Cabral Concept? The best way to help me spread our mission of true natural health is to pass on the good word, and I read and appreciate every review!  

Here's an interesting natural prednisone or steroid protocol. Check this out. Dr. Berg's Keto and IF Lab: https://www.facebook.com/groups/drbergslab/ How to Bulletproof your Immune System FREE Course: https://bit.ly/39Ry3s2 FREE MINI-COURSE ➜ ➜ Take Dr. Berg's Free Keto Mini-Course! ADD YOUR SUCCESS STORY HERE: https://bit.ly/3z9TviS Find Your Body Type: https://www.drberg.com/body-type-quiz Talk to a Product Advisor to find the best product for you! Call 1-540-299-1557 with your questions about Dr. Berg's products. Product Advisors are available Monday through Friday 8 am - 6 pm and Saturday 9 am - 5 pm EST. At this time, we no longer offer Keto Consulting and our Product Advisors will only be advising on which product is best for you and advise on how to take them. Dr. Eric Berg DC Bio: Dr. Berg, 51 years of age is a chiropractor who specializes in weight loss through nutritional & natural methods. His private practice is located in Alexandria, Virginia. His clients include senior officials in the U.S. government & the Justice Department, ambassadors, medical doctors, high-level executives of prominent corporations, scientists, engineers, professors, and other clients from all walks of life. He is the author of The 7 Principles of Fat Burning. Dr. Berg's Website: http://bit.ly/37AV0fk Dr. Berg's Recipe Ideas: http://bit.ly/37FF6QR Dr. Berg's Reviews: http://bit.ly/3hkIvbb Dr. Berg's Shop: http://bit.ly/3mJcLxg Dr. Berg's Bio: http://bit.ly/3as2cfE Dr. Berg's Health Coach Training: http://bit.ly/3as2p2q Facebook: https://www.facebook.com/drericberg Messenger: https://www.messenger.com/t/drericberg Instagram: https://www.instagram.com/drericberg/ YouTube: http://bit.ly/37DXt8C Pinterest: https://www.pinterest.com/drericberg/

Episode 2227 - On this Saturday's show, we replay the first time Vinnie Tortorich speaks to Dr. Jason Fung and they discuss his book , insulin and weight loss, metabolic syndrome, intermittent fasting, and more. https://vinnietortorich.com/best-of-insulin-weight-loss-dr-jason-fung-episode-2227 PLEASE SUPPORT OUR SPONSORS INSULIN & WEIGHT LOSS Vinnie welcome Dr. Jason Fung, who is a kidney specialist. (2:05) He works often with type 2 diabetics -- the biggest cause of kidney nephropathy. Type 2 is linked closely with obesity. Insulin has negative effects.   Vinnie tells Dr. Fung about his visit to his hometown of Donaldsonville. (7:05) Everyone he met was on multiple medications. They all had some kind of sugary drink in their hands. They discuss how obesity is misunderstood; it is linked with several diseases and is often a part of metabolic syndrome. (8:00) Dr. Fung recommends . (9:00) Sugar is the enemy, not calories! Calories In, Calories Out (CICO) doesn't work or even make sense. (9:10) When you eat fewer calories, your energy expenditure (and thus fat/fuel usage) goes down. You simply get hungrier. It takes time to lose weight. Lots of things raise insulin, not just sugars and grains. (19:40) Insulin resistance leads to higher insulin levels. INTERMITTENT FASTING A key way to break insulin resistance is intermittent fasting. (23:20) Intermittent fasting should happen every day--Dr. Fung explains. (27:50) Having snacks doesn't allow your body to process all the hormones properly. (29:00) Shorter fasts aren't really enough. Dr. Fung explains the various fasting methods he recommends, even longer ones under medical supervision. Body switches to fat burning. (33:15) Obesity is a hormonal imbalance, not a caloric one. (42:00) Cortisol is a stress hormone -- encourages getting fat. Prednisone is a synthetic form of cortisol. Leptin is produced by fat cells and regulates how much fat is in the body If you have lots of fat, leptin is released and tells your brain to stop gaining more weight. When you are quite fat, your body stops responding to leptin. Leptin is a secondary hormone, so it doesn't pass the causal test. Ghrelin is another one of these secondary hormones; as a result, no lasting effect so doesn't pass the causal test either. Vinnie and Dr. Fung discuss a bit more about why CICO doesn't work. (45:00) Type 2 diabetes can be reversed in most cases; however, you can't treat a disease of having too much insulin by giving more insulin! (51:45) They give great comparisons as examples. There needs to be a grassroots movement for changes in the system; there are too many conflicts of interest with people who want to keep the meds going and how they fund things like universities. (53:40) NOTE: At the end of the show, Vinnie references Anna Vocino's old website, but she can now be found at . [the_ad id="20253"] PURCHASE BEYOND IMPOSSIBLE The documentary launched on January 11! Order it TODAY! This is Vinnie's third documentary in just over three years. Get it now on Apple TV (iTunes) and/or Amazon Video! Link to the film on Apple TV (iTunes):  Then, Share this link with friends, too! It's also now available on Amazon (the USA only for now)!    Visit my new Documentaries HQ to find my films everywhere: REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter! FAT: A DOCUMENTARY 2 (2021) Visit my new Documentaries HQ to find my films everywhere: Then, please share my fact-based, health-focused documentary series with your friends and family. The more views, the better it ranks, so please watch it again with a new friend! REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter! FAT: A DOCUMENTARY (2019) Visit my new Documentaries HQ to find my films everywhere: Then, please share my fact-based, health-focused documentary series with your friends and family. The more views, the better it ranks, so please watch it again with a new friend! REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter!