Podcasts about Sami

It's time for another real-time match commentary! This week, host Tim Kail provides commentary to the modern classic, Sami Zayn vs Johnny Knoxville at WrestleMania 38. Tim discusses Knoxville's sincerity, Sami's focus, and the fascinating way this match exists as an advertisement for a movie and great art simultaneously. Then, in the outro, Tim examines why he belives "no one is over today", and why the common criticisms by veteran wrestlers and journalists fail to capture the reality of WWE's failing creative apparatus. Get a tee-shirt at www.prowrestlingtees.com/workofwrestling. Subscribe to WOW's Substack at www.workofwrestling.substack.com. Thank you for listening!

PWTorch editor Wade Keller is joined by PWTorch's Javier Machado to review WWE Smackdown with live chat and caller interactions throughout starting with thoughts on Jey Uso advancing to the King of the Ring semi-final and how KOTR could play out from here. A lot of talk about the dynamic with the Bloodline and Bloodline-adjacent wrestlers interacting in ambiguous, intriguing ways and whether it will pay off or end up softening fan passion for a certain outcome. Also, a lot of praise for the Sami Zayn-Cody Rhodes dynamic, the messy and convoluted women's match (and the ridiculous "This is awesome!" chant from the crowd), LA Knight's hot streak on the mic (with a couple misspeaks tonight), Jade Cargill's preoccupation with her wig coming loose, Royce Keys-Solo Sikoa intrigue, and more.Become a supporter of this podcast: https://www.spreaker.com/podcast/wade-keller-pro-wrestling-post-shows--3275545/support.

Alice Nellis studovala na konzervatoři hru na flétnu a zároveň anglistiku a amerikanistiku. Pak se obojím chvíli živila. Když potom ale nastoupila ještě na FAMU, pochopila, že se jejím světem stane film a divadlo. Začala psát scénáře, točit filmy, režírovat v divadle. A to nejenom činohru, ale také operní představení. V Černošicích, kde žije, má k tomu všemu navíc hotovou zvířecí farmu.

Představíme vám novou obyvatelku Safari Parku Dvůr Králové. Je to dvouletá samička pralesní žirafy okapi. Vzácné zvíře pocházející z Demokratické republiky Kongo sdílí společnou expozici s domácí samičkou Quirou a dvorští chovatelé pro ně hledají partnera - jejich cílem je odchov dalších mláďat.

On this week's @betches, Sami, Aleen, and Jordana are back together to break down Ariana Grande and Ethan Slater's reported breakup and debate whether anyone is actually surprised. They also dive into the wild story of Jennifer Lawrence allegedly helping Bravo track down the Summer House reunion leak, and discuss the kind of investigative skills it takes to uncover the truth. Plus, they unpack the viral Dot Cakes phenomenon, NYC's obsession with waiting in line for food, and what they would (and definitely would not) wait hours for. Go to the Betches Podcast YouTube page to watch full length episodes every Friday: Youtube.com/@betchespod   Learn more about your ad choices. Visit megaphone.fm/adchoices

There is a place at the top of the world where the land runs out, a sheer thousand-foot cliff rising straight from the Arctic Ocean, and nothing beyond it but open sea all the way to the North Pole. This is the North Cape - the northernmost point of continental Europe and getting there means sailing one of the most spectacular coastlines on Earth. From Oslo, we travel north through towering fjords and fishing villages so remote the ship is their only connection to the outside world. We pass the Lofoten Islands rising like a mountain range planted in the middle of the ocean, Alta - nicknamed the Northern Lights City - and on into the deep Arctic silence of Finnmark. Norway's coast is not just beautiful. It is staggering.The Northern Lights wait for us there too. The Sami called them Govsov - the light you can hear, the light that speaks. Science calls them solar particles colliding with the earth's atmosphere. But standing beneath them, none of that matters. What you are seeing is the invisible touch of our sun, raining down in colour all around you - and it is unlike anything you have ever experienced before.Join host Aaron Millar, and two expert guests, as we set sail on the North Cape Line with the legendary adventure ships of Hurtigruten. This is more than an expedition cruise, it is a journey to the edge of the world along one of the most awe-inspiring coastlines in the world in search of the Aurora at its most breathtaking.Produced in Armchair Explorer's signature documentary style, this immersive audio adventure is designed not just to let you hear what it's like to chase the Northern Lights along the Norwegian coast - but to feel it.Highlights include:Sailing the Trollfjord - a fjord so narrow it feels like you could reach out and touch the mountains on either side, sheer walls of rock and snow rising straight from the water around you.Standing at the North Cape - the northernmost point in Europe where beyond the cliff edge there nothing but the Arctic Ocean, all the way to the North Pole.Sitting inside a Sami Lavvu - warm with firelight and wood smoke - hearing stories, sharing food and listening to yoik, the ancient song form where every melody carries the spirit of a specific person, animal or place.Husky sledding into the Arctic wilderness - the moment the dogs stop barking, set off, and everything goes utterly silent. Just you, the animals, and the snow.The food of Norway's coast - fresh Arctic cod from the world's largest cod fishery in Lofoten, king crab hauled from the icy waters of Finnmark and cooked within the hour, cheese from a family farm whose recipes are 300 years old, and wild herbs foraged from cliff edges above the fjords.The Aurora itself - the crowning corona, where the lights rain down in every direction around you - “like taking a dream out of your head and placing it into the sky”FIND OUT MOREProduced in collaboration with Lonely Planet: LonelyPlanet.comThis journey is run by expedition cruise company Hurtigruten, whose ships have sailed this coastline for generations. This is what they promise: If you're on one of their 11-day or longer voyage during the Auroral season and the Lights don't appear, they'll give you another chance on a future sailing at no extra cost.Find out more at hurtigruten.comConnect with expert Aurora chaser Tom Kerss at TomKerss.com or follow him on Instagram at @tomkerssThank you also to Andre Pettersen, the man behind Hurtigruten's extraordinary food and community connections along the Norwegian coast.Know Someone Who Needs This?If this episode lit something up in you, do me a favour - send it to just one person. One friend, one family member, one person who needs a little wonder in their life right now. Or go old school and tell someone about it over a cheeky pint. Every single share genuinely moves the needle.Hit the share button in your podcast app - it takes about ten seconds, and helps us continue to bring these stories to you.FOLLOW US: Instagram: @armchairexplorerpodcast Facebook: @armchairexplorerpodcastCONNECT WITH US: If you enjoy the show, please subscribe on whatever podcast player you're reading this on right now. Go on, do it! It helps us grow the show, and means you won't miss an episode (we've got some good ones coming up!).Armchair Explorer is written and presented by Aaron Millar. Audio editing and sound design by Charles Tyrie. Theme music by Sweet Chap. Produced by Armchair Productions. Find our other shows at Armchair-Productions.com Mentioned in this episode:Check out all of our other travel podcasts from around the worldThis podcast is part of the Voyascape Network, a collection of some of the world's best travel podcasts. Explore more at Voyascape.com. For advertising or sponsorship opportunities across the network, see the link below.Voyascape Podcast NetworkCheck out the Smart Travel PodcastThis week's show is supported by the new Smart Travel Podcast. Travel smarter — and spend less — with help from NerdWallet. Check out Smart Travel at the Link below:Smart Travel Podcast

The wpov crew discuss Sami zayne next champ?, oba/brock, danhausen, mjf,bloodline rules match, nick kahn/ hhh, creative writing, this week in wrestling 5 second shuffle championship and more!

Il fait chaud, vous vous préparez une grande et belle salade pleine de couleurs, et vous avez le sentiment d'avoir fait les choses parfaitement. Sauf qu'à 16 h, vous tournez déjà autour du placard. Cette salade « healthy » qui ne rassasie pas, c'est ce que j'appelle la salade-piège : généreuse en légumes, souvent chargée en glucides… et presque toujours pauvre en protéines. Dans cet épisode, je vous donne une méthode simple pour transformer n'importe quelle salade en vrai repas, un repas qui tient jusqu'au suivant et qui soutient vos entraînements.Composez vos salades en quelques secondes avec l'assistant Salade Express : https://go.soulier.xyz/saladesnLiens complémentaires :Gratuit : Le kit Reboot pour retrouver la forme et l'énergie avec la méthode SAMi et des outils : https://sn.soulier.xyz/kitLe Protocole Perte de Gras : https://go.soulier.xyz/protocolesnLa Stratégie FlowFit pour bouger et plus et prendre du muscle (tarif de lancement spécial) : https://go.soulier.xyz/flowfitsnTous les liens complémentaires et anciens épisodes : https://sn.soulier.xyz/187Rejoindre le Hamsters Running Club : https://sn.soulier.xyz/hrcL'été, on se tourne naturellement vers les salades : fraîcheur, légumes de saison, format pique-nique. Le problème, c'est qu'à force d'empiler les légumes et d'ajouter des pâtes ou du riz, on obtient des assiettes magnifiques mais déséquilibrées, où le grand absent est toujours le même : la protéine. Résultat, une salade peu rassasiante, des fringales l'après-midi, et une récupération sportive qui ne suit pas.Je vous partage ce que je teste au quotidien et ce que disent les études. On verra pourquoi la protéine est le nutriment qui construit ET qui rassasie. Et quel repère viser réellement sur un repas-salade en dépassant l'idée reçue du « 20 grammes maximum », qui ne s'applique pas à un repas mixte. Bonne surprise au passage : vos pâtes et votre riz refroidis ne sont pas vos ennemis. En refroidissant, ils forment de l'amidon résistant, qui nourrit votre microbiote. Le piège glucidique peut donc devenir un atout, à condition d'équilibrer le reste.Vous repartirez avec une approche concrète et souple : cibler votre besoin du jour, sécuriser vos protéines, et composer en mode « mix and match » sans rien peser. Je vous montre aussi mes astuces perso pour monter les protéines sans y penser, et l'outil que j'utilise pour ne plus jamais sécher devant le frigo.Dans cet épisode :Pourquoi votre salade « healthy » ne vous rassasie pas jusqu'au repas suivant ?Comment repérer le déséquilibre caché des salades de pâtes et de riz ?Pourquoi la protéine est le nutriment le plus rassasiant — et quel repère viser par repas ?Faut-il vraiment se limiter à 20 grammes de protéines par repas ?Pourquoi vos féculents refroidis nourrissent mieux votre microbiote ?Comment construire une salade-repas complète en mode « mix and match » ?Quelles sont mes astuces pour augmenter les protéines sans y penser ?Je vous donne même la recette de la salade signature de Tibo InShapeNouveau : Le protocole Perte de Gras 2025 ❤️ Me suivre Tous les liens sont ici

6/11/26 (Host - Bill Newman) Amherst-Pelham School Superintendent Xiomara Herman: the accomplishments this year, the challenges and finances of the next. Live in the studio -- Django in June in Northampton: Musicians Sam Farthing, Giacomo Smith & Sami Arefin -- their music is amazing -- and Andrew Lawrence, founder and director. Rabbi Riqi Kosovske: war & peace, scripture & poetry. Ruth Griggs, Pres, Northampton Jazz Festival, on Eugene Uman's Convergence Project @ the Vermont Jazz Center.

PWTorch editor Wade Keller presents the Tuesday Flagship edition of the Wade Keller Pro Wrestling Podcast with guest co-host Rich Fann, a PWTorch columnist. They discuss these topics:An in-depth look at the realistic and preferred paths left in the King of the Ring and Queen of the Ring tournaments, including pros and cons for favorite(s) and dark horses.An email on whether Paul Levesque is leaning too much on multi-man matches beyond regular singles and tag team matchesThe CM Punk heat he took on social media for his bulked up physique and Ryback coming to his defenseA look at the Owen Hart Memorial Cup Tournament men's and women's bracketsA preview of DynamiteThe latest developments with Roman Reigns, Jacob Fatu, and the UsosReaction to the latest with Cody Rhodes, Sami Zayn, and Gunther, and some discussion on who Cody's Summerslam opponent should beAn email on Beck Lynch's role in WWE recently and whether she's being underutilizedBecome a supporter of this podcast: https://www.spreaker.com/podcast/wade-keller-pro-wrestling-podcast--3076978/support.

Host Marina Franklin chats with the hilarious Ophira Eisenberg and rising star Sami Beason about Ophira's new comedy special, toxic positivity, AI hallucinations, parenting and more! Tune in now for laughs and insights!  Ophira Eisenberg is a Standup Comedian, Writer, and Podcast Host (NPR, CBS, CNN, The Moth, HBO, Comedy Central, iHeart) who headlines theaters and comedy venues across the world, tours regularly with the Moth Mainstage, and hosts the weekly parenting-comedy podcast Parenting is a Joke (Webby Finalist for Best Host). Her new comedy special, I Used to Be Nicer produced by Lewis Black/Stark Raving Black Productions, is streaming now. She also hosted NPR's trivia comedy show Ask Me Another for 9 years. Her breakout memoir Screw Everyone: Sleeping My Way to Monogamy(Seal Press) has been optioned for a television series, and her solo show Leaving A Mark: A Comedy About Scarswon Women in the Arts & Media Award for Solo Show Scripts and just finished a successful European tour. Sami Beason is a rising comedy talent from Denver, celebrated for their quick wit, authentic storytelling, and constant giggle. In 2024, they took home the Best of Fest award at the Colorado Springs Festival and now has a special on YouTube called "First Gen" through open bar. Always hosted by Marina Franklin - One Hour Comedy Special: Single Black Female ( Amazon Prime, CW Network), TBS's The Last O.G, Last Week Tonight with John Oliver, Hysterical on FX, The Movie Trainwreck, Louie Season V, The Jim Gaffigan Show, Conan O'Brien, Stephen Colbert, HBO's Crashing, and The Breaks with Michelle Wolf. Writer for HBO's 'Divorce' and the new Tracy Morgan show on Paramount Plus: 'Crutch

In this episode of The Hormone Genius Podcast, we sit down with Olivia Chasteen, Director of SymptoPro Fertility Education, to have an honest and empowering conversation about women's health, fertility awareness, and the gap between conventional care and true root-cause solutions. So many women are handed quick fixes, band-aid solutions that often mask symptoms rather than address what's really going on. Hormonal contraception is frequently prescribed as a way to “regulate” cycles, yet it doesn't actually restore or regulate the body's natural rhythms, and it comes with real side effects that can impact nearly every system in a woman's body. Olivia shares how the SymptoPro method, a sympto-thermal approach to Natural Family Planning (NFP), offers a science-based, effective alternative that works with a woman's body instead of against it. We explore the history and effectiveness of SymptoPro, including how it has helped couples confidently avoid pregnancy when used correctly, while also serving as a powerful tool for those trying to conceive. But this conversation goes far beyond family planning. Cycle tracking is a window into a woman's overall health. Whether married, single, trying to conceive, or simply wanting to understand her body better, charting can reveal valuable insights from hormonal imbalances to thyroid concerns, luteal phase defects, and more. Olivia explains how even women with no immediate fertility goals can benefit deeply from learning to read the signs their bodies are giving them each day. We also dive into the reality that charting isn't always straightforward—and that's okay. If your charts feel confusing or inconsistent, it doesn't mean your body is broken. It means there's more to uncover. Olivia walks through practical troubleshooting strategies and emphasizes the importance of individualized support and education. In a culture that often paints the female body as inconvenient or flawed, true healing begins when we learn to understand and respect what our bodies are communicating. There is real support. There are real answers. And there is real beauty in understanding your fertility. What the SymptoPro method is and how long it's been helping women and couples How effective SymptoPro is for avoiding pregnancy when used correctly How it supports couples trying to conceive Why cycle tracking is a powerful health tool for all women, not just those planning families What your fertility chart can reveal about your overall health Practical tips for when charting feels confusing or overwhelming Why one method may not fit everyone, and how to find the right approach for you The deeper issue with “quick fix” solutions in women's healthcare Olivia also shares her work as a Certified Lactation Consultant, wellness professional, and instructor serving families in rural Wisconsin where access to fertility education and medical support can be limited, yet deeply needed. Sponsors

Sami Clarke is back with her self-love and growth series for a Q+A straight from your questions — and she's going deep on the ones you've been sitting with. From navigating the single era in your 30s, to what it actually means to trust your intuition, to how to feel like yourself again when the sparkle feels far away.Tune in as Sami shares what this season of life has really looked like for her: the hard nights, the big realizations, the cry-on-the-couch-watching-Sex-and-the-City moments, and why she's genuinely excited about where she is. She gets into why feeling "behind" in life is a timeline someone else handed you, how your desires are never there by accident, and what it looks like to move your body in a way that actually responds to the season you're in. If you're doing the work on yourself right now and some days feel harder than others, this one is for you.Transform Instagram - click here!Sami Spalter Instagram - click here!Sami Clarke Instagram - click here!FORM Shop - click here!FORM Website - click here!Code TRANSFORM for 20% off an annual membership.This episode may contain paid endorsements and advertisements for products and services. Individuals on the show may have a direct, or indirect financial interest in products, or services referred to in this episode.This episode is brought to you by:Jolie: Try Jolie risk-free for 60 days by going to jolieskinco.com/TRANSFORMRitual: Save 25% on your first month at Ritual.com/TRANSFORMTheRealReal: Go to TheRealReal.com/transform and get $25 off your first purchase plus an extra $100 to shop when you sell for the first timeOllie: Ollie. Feed the obsession. Go to ollie.com/transform and use code transform to get 70% off your first box!BiOptimizers: Head to bioptimizers.com/samispalter and use my exclusive code SAMI to get 15% off any order. Osea: Get 10% off your first order sitewide with code TRANSFORM at oseamalibu.com Produced by Dear MediaSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

Het WK 2026 komt eraan — en wij kunnen niet meer wachten.In deel 2 zetten we onze tanden in het grootste toernooi van het jaar. Welke teams gaan verrassen? Wie zijn de échte favorieten? En de hamvraag: hoe ziet ónze ideale opstelling van Oranje eruit? We bouwen 'm van keeper tot spits — en niet iedereen aan tafel is het eens.

Willkommen am Stammtisch Rückblick auf das Spiel SV Werder Bremen vs. BVB Das Saisonfazit Unsere Prognose und das WM-Tippspiel Das Werder Stürmer Ranking Das Fundstück der Woche Die Stammtisch Playlist 2025/26 Abgesang Das Tippspiel Herzlichen Glückwunsch an “Ph” unseren Gewinner des Bundesliga-Tippspiels 2025/26 Wir haben ein WM-Tippspiel gestartet Die WM-Kicktipp Runde der Werder Raute Das Fundstück der Woche ***Werbung*** Carsten Dexter - Wiedererwachen (ParamountPlus) Die Toten Hosen - Trink aus, wir müssen gehen uns Alles muss raus (Amazon/Affiliate) Sami Michael (Kino) Kalle Masters of the Universe (Kino) Derpanski WM 1994 – Elf Helden, ein Albtraum (ARD-Mediathek) Spendet uns (Auphonic) Zeit Hier könnt ihr uns Auphonic Zeit spenden, für einen guten Sound Die Werder Raute Playlist Werder Stammtisch 2025/26 - Hotte macht die Bremer Flotte (Spotify)

Money is a topic that affects nearly every relationship, yet it can be one of the most difficult conversations for couples to have. In this episode of The Lebanese Physicians Podcast, Dr. Khalil Diab speaks with Sami Al Natour about the communication patterns that often emerge when couples discuss spending, saving, debt, investing, and major life decisions. The conversation explores how personal experiences, family influences, and differing perspectives can shape financial discussions and relationship dynamics. Watch the full episode on YouTube and listen on all major podcast platforms. #Relationships #PersonalFinance #marriage #Communication #MoneyMindset #Podcast #TheLebanesePhysiciansPodcast #subscribe

In this episode of the Modern Direct Seller Podcast, we chat with digital marketing strategist and agency founder Sami Bedell-Mulhern about why your email list is the one platform you actually own. Sami has spent years helping businesses turn email into their most reliable engine for connection and sales, and she isn't shy about ranking it above social. Together, we get into why email still converts better than social, the mindset shift that trips up most direct sellers, and where to begin if email marketing has never been on your radar. We also dig into lead magnets, sending frequency, and the simple content habits that keep people engaged without wearing you out.You can connect with Sami on Facebook or through her digital marketing business The First Click. She's also part of the team behind Oh My Hi, the platform built for direct sellers, where you can claim a free Capture and Convert homepage to get started.Time-Based Notes:1:38 - Rapid Fire Questions3:43 - Getting to Know Sami4:46 - The Biggest Email Marketing Mistakes6:17 - Why Email Beats Social for Sales12:43 - Where to Start: Your Customer List14:13 - Why Letting People Opt Out Matters15:45 - Building Your List & Lead Magnets That Convert20:26 - Email Frequency & What to Send22:11 - Repurposing Content You Already Have25:53 - Email Mistakes to Avoid28:46 - One Action to Take This WeekShow sponsored by CinchShare: The number one most trusted social media scheduling tool for direct sellers. Start your 60 day trial today with coupon code KEYBOARD60 and spend less time posting and more time socializing!Get the full show notes at https://moderndirectseller.com/episode292

Send us Fan MailThis episode is technically about grandparenting. But it is really about something most of us deal with every day: how to correct someone you love without making them feel like something is wrong with them. That shows up in how you give feedback at work, how you argue with a partner, how you talk to yourself when you mess something up. And yes, how you talk to a kid when they are driving you absolutely crazy.Sami and Angela use grandparenting as the lens because it is where the stakes feel especially clear: you love these kids completely, you only get so many reps, and the patterns you absorbed from your own upbringing have a way of showing up without permission. Their conversation centers on the difference between guilt (I did something wrong) and shame (there is something wrong with me), a distinction borrowed from Brene Brown that is one of the most practically useful frameworks in the episode. Once you have it, you will start noticing it everywhere. In this episode, they dig into:Why shame shows up in grandparenting even when no one intends itHow telling a child to "be careful" all the time might be quietly building their anxietyThe difference between correcting a behavior and attacking an identityAngela's ABCs (and Sami's three Rs) for interacting with grandkids without shameWhy repair matters just as much as getting it right in the first placeSami and Angela get personal here. Angela talks about the very real capacity limits of grandparenting (and why "I love my grandkids but send them home" is not a character flaw). Sami talks about what it is like to watch a grandparent say something she also says, and realize the two are not that different. They walk through the backpack metaphor, the sleeping-grandchild test, and why knowing better is not the same as saying you did it wrong. If you grew up hearing "be careful" constantly and have spent your adult life with an anxiety you cannot fully explain, this one might give you a word for it.You do not need a grandchild, or even a child, to walk away from this one with something real.Press play. The kid who grew up being told to be careful might need to hear this one.MENTIONED IN THIS EPISODEBrene Brown's work on shame vs. guilt (brenebrown.com)The motivational triad (avoid pain, seek pleasure, be efficient) -- referenced in discussionSupport the showSign up at bfreakingawesome.com to get the latest news, insights, and episodes straight to your inbox.Follow Be Freaking Awesome on Facebook, LinkedIn, Youtube, and Instagram.Let us know what questions you want to be answered and discussed by emailing us at podcast@bfreakingawesome.com.

Faptele apostolilor 5 1. Dar un om, numit Anania, a vândut o moşioară, cu nevasta sa Safira,2. şi a oprit o parte din preţ, cu ştirea nevestei lui; apoi a adus partea cealaltă şi a pus-o la picioarele apostolilor.3. Petru i-a zis: „Anania, pentru ce ţi-a umplut Satana inima ca să minţi pe Duhul Sfânt […]

Hoy en SmackDown pasó de todo y vamos a hablar claro de cada momento importante de la noche.Gunther exigió una revancha contra Cody Rhodes después del controversial final de su lucha en WWE Clash in Italy, pero el segmento se calentó cuando Sami Zayn interrumpió para dejar claro que él también puede ser una amenaza seria para Cody como campeón. Todo terminó en una tremenda refriega entre Gunther, Cody y Sami.También hablaremos del avance de Raquel Rodriguez en el torneo Queen of the Ring, luego de vencer a Bayley, Jacy Jayne y Kiana James. Además, Giulia puso fin a su alianza con Kiana James, dejando dudas sobre qué viene ahora para ella.En la división femenina, Rhea Ripley fue interrumpida por Charlotte Flair, Tiffany Stratton, Jade Cargill, B-Fab y Michin, lo que terminó provocando una lucha en parejas donde Jade Cargill y Michin vencieron a Charlotte y Tiffany.También analizamos la victoria de Ricky Saints sobre Carmelo Hayes, el triunfo de Lash Legend sobre Chelsea Green, y el gran resultado de la noche: Dominik Mysterio venciendo a Bron Breakker, Damian Priest y Trick Williams para avanzar a las semifinales del torneo King of the Ring.Esta noche repasamos todo lo que sucedió en SmackDown, lo bueno, lo malo y lo que puede cambiar el camino hacia los próximos eventos de WWE.#SmackDown #WWE #CodyRhodes #Gunther #DominikMysterio #KingOfTheRing #QueenOfTheRing #100WrestlingPodcast

PWTorch editor Wade Keller is joined by PWTorch's Greg Parks to review WWE Smackdown with live chat and caller interactions throughout starting with thoughts on Dominik Mysterio's King of the Ring opening round win and prospects for him against Oba Femi, plus a look at the other side of the bracket and its possibilities. Also, Queen of the Ring possibilities with Iyo Sky vs. Raquel Rodriguez now set in the semis. Also, Gunther gets to pick a stip for a rematch against Cody Rhodes, and how might Sami Zayn get involved. Plus, Chad Gable, Blake Monroe, and more.Become a supporter of this podcast: https://www.spreaker.com/podcast/wade-keller-pro-wrestling-post-shows--3275545/support.

In today's episode the gang talks about the Bloodline 2.0 and if we are okay with recycled stories??? Seems like WWE is digging into the books but not digging very far. We get into you just made the list and do our best to help out LA Knight!!! All this and so much more......CHEERS!!!JERKING THE CURTAINROUND TABLE OF TOPICSNEWSAsuka introduces her new puppy Big Body BossMike Santana WWE bound???Finn traded to SmackdownSpeaking of Smackdown……they are moving back to 2 hours Curtis Axel facing multiple charges for DWI“You Just Made the List” Give me two storylines for LA Knight  SMACKDOWNJade kicks off RAW and gets Dirty Dom treatment Jade strikes again, real heeling shit going down hahaSami continues his need for attention and Cody isn't having it When will Miz believe in the DanHausen curse???Ugh I'll say it again, Chelsea putting over Tiffany is bad for business Poor Candice, Johnny and Sami need to be a tag team……looks like the locker room is getting tired of Sami as well Chelsea gets the win with an assist from Tiffany…..blahhhhEasy to find hard to beat……strong words from our champ!!! CLASH IN ITALYItaly is alive and they love them some Cody Rhodes Was it me or did the camera feel different but in a good way Cody and Gunther put on a solid match…..right decision for match Gotta say Jade looked better in this match, Charlotte to the rescue You have got to be fucking kidding me……match was going great and Oba loses now!?! Trilogy activated for SummerSlam Santino Marella in Italy is grrrrreat for business Wow, wish I could say I'm happy for Sol buuut I'm not Ethan Page with the finger is hilarious Bloodline is officially back, Jacobs addition will be interesting…..great match between Fatu and Roman put on a solid match RAWOTC kicks off RAW, it's Acknowledgement day!!! Bloodline 2.0 is here, are we okay with a recycled story this soon???First round of KOR gets ruled Does Cody and Romans silent stare move the needle???Rey and Dragon Lee put on a great matchLA Knight giving my guy the business, I hate to say it but I'm seeing the same These match ups are fantastic and hard to predict a winner. QOR was damn good, Iyo moves on I still don't care for Maxine's story but can WWE just pull bandaid already Lyra vs Sol is great for business, does Bayley retaliate???Liv is a POS and phenomenal at her job The Architect is back baby and 3 steps ahead of the Vision, great ending to RAWTNAWe are officially taking a break from NXT until it gets better We will break news for TNA next week 10 count with Always Ready Arianna Check out the Smackdown Siblings on TikTok @ariannaandthomasEpisodes dropping weekly!!!Follow us on TikTok @the.funkaholiks.pod THEE POD THAT TALKS WHAT THEY LOVE 

This week on News Weakly, Sami Shah dives into the latest Australian culture war, where an autism podcast somehow becomes a referendum on Israel, Gaza, free speech, antisemitism, cancellation, and who should be allowed to work.The ABC finds itself under fire after commissioning a podcast hosted by Grace Tame, despite controversy surrounding her comments about October 7, Hamas sexual violence, and the phrase "globalise the intifada". Meanwhile, Charlie Pickering discovers the dangers of talking to Avi Yemini, and Australia once again debates whether disagreement should be answered with argument or unemployment.Along the way, Sami examines the Jayson Gillham controversy, the limits of institutional neutrality, why cancellation campaigns rarely achieve their stated goals, and the difference between offensive speech, political speech, artistic speech, and work that is itself the speech.All that, and more.Sami Shah is a multi-award-winning comedian, writer, journalist, and broadcaster.For more: http://thesamishah.comTheme music 'Historic Anticipation' by Paul MottramThis podcast is written, hosted, and produced by Sami Shah. Hosted on Acast. See acast.com/privacy for more information.

In this episode of In The LOOP Podcast, Jordan Jo sits down with Sami Jo Smith for a powerful conversation about leadership, legacy, women's rodeo, faith, and the behind the scenes work it takes to build something that truly moves the industry forward. From growing up as a first generation rodeo kid in Cheyenne, Wyoming, to serving as Miss Frontier and building a career across RFD TV, The American, ERA, WCRA, and now Premier Women's Rodeo, Sami Jo shares the people, moments, and opportunities that shaped her path in the Western industry. The two also dive into the growth of PWR, the evolution of professional women's rodeo, the addition of rough stock, national television opportunities, athlete stories, family legacies, and why creating a true home for women in rodeo matters now more than ever. As the Director of Operations and Rodeo Administration for PWR, Sami Jo reflects on what it means to serve behind the scenes, build opportunities for cowgirls, and help carry a movement that is opening doors for the next generation. At the heart of the conversation is a reminder that legacy is not just about titles or organizations. It is about the impression you leave, the people you serve, and the impact that lasts long after the event is over.

The FIFA World Cup begins in just one week, and once again the debate is underway. Will politics overshadow the football? Or will the football eventually overshadow the politics? On this episode of Roqe, Jian Ghomeshi welcomes Atbin Arian and Shayan Sami'i for a conversation about the upcoming World Cup, FIFA's contradictions, the controversy surrounding the Lion and Sun flag, Team Melli, nationalism, identity, free expression, and the difficult questions many Iranians continue to wrestle with whenever international sport and Iranian politics collide. Will Iranian fans support Team Melli? Has FIFA's handling of the Lion and Sun flag created more controversy rather than less? And when the first whistle blows, will football once again reclaim center stage? But first, Jian opens the show with an essay entitled “They Always Tell Us To Keep Politics Out Of It”, exploring why so many people are told to separate politics from culture and sport, and why that separation is often easier said than done. Guests: Atbin Arian Shayan Sami'i Sponsors: Stellar Law Quasar Homes Recorded June 4, 2026.

Sami Godlove is the is the Central Oregon Field Coordinator at Oregon Wild, which works to protect and restore Oregon's wild lands, wildlife, and waters as an enduring legacy for future generations.Sami spoke about his early influential moments in nature, the importance of the Roadless Rule, potential large environmental rollbacks, and the effects these policies have on large ecosystems, old growth forests and other natural places that they protect. Oregon Wild@oregonwild@thewolfconnectionpod

In today's episode of Sami's Nesting Series, Sami opens up about how pregnancy has completely shifted her relationship with movement and nutrition, and how the expectations she once had for herself were very different from the reality of what her body truly needed. From letting go of discipline and control to learning how to move and nourish herself from a place of love, Sami shares the mindset shifts that have helped her embrace this new season with more grace and trust.Tune in as Sami shares how she's currently moving through this season, including the workouts she's loving, what she's had to let go of when it comes to fitness, and how nourishing her body has helped her feel even more connected to herself. To close the episode, she shares the affirmations helping her reframe her mindset around movement and food. If you're in a season of pregnancy, learning to trust your body, or redefining what feeling good looks like for you, this episode is for you.Transform Instagram - click here!Sami Spalter Instagram - click here!Sami Clarke Instagram - click here!FORM Shop - click here!FORM Website - click here!Code TRANSFORM for 20% off an annual membership.This episode may contain paid endorsements and advertisements for products and services. Individuals on the show may have a direct, or indirect financial interest in products, or services referred to in this episode.This episode is brought to you by:IQ Bar: Text TRANSFORM to 64000 for 20% off all IQ Bar products plus FREE shippingOllie: Go to ollie.com/transform and use code transform to get 60% off your first box!Spindrift: Visit drinkspindrift.com and use code transform for 15% off your next Spindrift orderQuince: Head to Quince.com/transform for free shipping on your order and 365-day returnsCozy Earth: Head to cozyearth.com and use code TRANSFORM for an exclusive 20% offProduced by Dear Media See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Send us Fan MailYou know that feeling where you did something wrong and instead of fixing it, you just... keep feeling bad about it? Like feeling bad is the apology, the penance, and the plan all rolled into one? Yeah. That's not guilt doing its job. That's guilt overstaying its welcome, and there's a difference.In this episode, Sami and Angela get real about guilt: what it's actually for, what it looks like when it goes sideways, and how to tell the difference between guilt that's moving you toward repair and guilt that's just becoming your whole personality. They dig into mom guilt, survivor guilt, the guilt you pick up on behalf of other people for things you had nothing to do with, and the sneaky habit of dumping your guilt on the very person you wronged and asking them to make you feel better about it. In this episode, we dig into:Why guilt is only useful if it's pushing you to do somethingThe difference between guilt (I did something wrong) and shame (I am something wrong)How to offer a real repair without making the other person comfort you in the processWhat to do with guilt when the relationship can't be repairedHow to feel your feelings fully without turning them into everyone else's burdenSami shares why she rarely experiences mom guilt and what Angela modeled that made that possible. Angela gets honest about her own anxious attachment history, the time she raged at her husband over a text message misunderstanding, and the friend with a hard medical diagnosis who taught her something important about allowing someone to offer a sincere apology. These are real stories, not tidy examples.The takeaway from this one is simple but not easy: guilt is an emotion, which means it's designed to move you. When it motivates repair, it's working. When it just sits there and collects weight, it's a drain on you and everyone around you. You don't have to be perfect. You do have to be willing to do something about it.Press play. This one is going to resonate.Mentioned in this episode:Atlas of the Heart by Brene Brown: brenebrown.com/book/atlas-of-the-heart/Next week's episode: Guilt vs. Shame (part two of this arc)Support the showSign up at bfreakingawesome.com to get the latest news, insights, and episodes straight to your inbox.Follow Be Freaking Awesome on Facebook, LinkedIn, Youtube, and Instagram.Let us know what questions you want to be answered and discussed by emailing us at podcast@bfreakingawesome.com.

„Pokud by Česká republika poslala oficiálně vojáky na Ukrajinu, byť třeba jako instruktory, tak se automaticky stávají normálním cílem pro vojenské operace Ruska. Bez ohledu na to, že jsme členskou zemí NATO, by se naši vojáci automaticky stali cílem, a s vysokou pravděpodobností by jich poměrně značná část zemřela, protože by byli napadeni,“ říká bezpečnostní analytik Jaroslav Štefec v rozhovoru pro pořad Kupředu do minulosti. 3. díl, 02.06.2026, www.RadioUniversum.cz

Ioan 10 11. Eu sunt Păstorul cel bun. Păstorul cel bun Îşi dă viaţa pentru oi.12. Dar cel plătit, care nu este păstor şi ale cărui oi nu sunt ale lui, când vede lupul venind, lasă oile şi fuge; şi lupul le răpeşte şi le împrăştie.13. Cel plătit fuge, pentru că este plătit şi nu-i […]

What if assessment made thinking visible and turned AI into a learning partner instead of a shortcut? Jackie sat down with Hamza Sami to close our series by unpacking practical ways to design for real understanding—where reasoning, judgment, and context take center stage.We start by reframing purpose: assessment is for learning, not just measurement. That lens leads to formative moves that build trust and invite responsible AI use. Hamza breaks down how to set clear AI norms, teach limits and risks, and require transparent acknowledgment with screenshots, links, and prompt logs. From there, we get tactical: in-class AI critiques, compare-and-verify exercises, and concise reflections that reveal what the model did well, where it failed, and how students adapted outputs to their goals.For summative checks, we spotlight formats that hold up with AI in play: presentations with Q&A, reflective blogs, portfolios of evidence, capstones grounded in authentic problems, and open-ended scenarios that demand justification. Hamza also shares three quick wins any instructor can deploy next term—explain-your-thinking checkpoints, brief peer feedback moments, and explicit AI process citations—plus one culture shift: stop letting assessment end at submission and bring learning to life through dialogue.Subscribe, share this episode with a colleague who's redesigning a course, and leave a review to help more educators find the show. What's the first assessment move you'll try next?

On this week's episode of @Betches, Sami and Jordana break down the explosive Summer House reunion and why Amanda and West's explanations still are not adding up. They discuss Ciara's emotional confrontation, Amanda's response to the backlash, Kyle's surprising role in the reunion, and what the drama reveals about friendships, accountability, and reality TV. Plus, they share their thoughts on Taylor Swift and Travis Kelce's viral Knicks date night, debate whether the couple actually enjoys the spotlight, and play a Taylor wedding prediction game featuring everything from the venue and dress to the bridesmaids, honeymoon destination, and first dance song. Go to the Betches Podcast YouTube page to watch full length episodes every Friday: Youtube.com/@betchespod   Learn more about your ad choices. Visit megaphone.fm/adchoices

Sami from Saskatoon, SK is a naturopathic doctor and joins us on this episode of Moony Birth Stories. Sami was followed by the midwives throughout her pregnancy, but when her water broke at 34 weeks + 3 days she was transferred to the care of OB's for delivery. Sami went on to have an unmedicated delivery that was augmented with pitocin. Her daughter spent 22 days in the NICU as well. At around 6 weeks postpartum, Sami experienced bleeding and retained products of conception which led to a D&C. Find us on Instagram: @moonybirthstoriespodcast @alivitrihSupport the show

Chad shares a recent pivot in ReadySetGo's development, where they see the app entering a founder's development and what they're doing to ensure a wide set of features for all users. Our hosts also pick apart the impact of AI on developers, how valuable is speed when working with AI, is it still a good measure of success when coding, and Sami takes a moment to ask the all knowing ChadGPT about his excessive running and exercise regime. — If you'd like to meet some of our hosts in person, be sure to check the links below to secure your place at thoughtbot's UK meet ups over the month of June. London Tech Leader Meetup - Tuesday June 23rd Brighton Tech Leader Meetup - Wednesday June 24th Brighton Ruby - Thursday June 25th Evolve - Friday June 26th You can find Chad all over social media as @cpytel and Sami through his website. You can also connect with the trio via their LinkedIn pages - Chad - Will - Sami. If you would like to support the show, head over to our GitHub page, or check out our website. Got a question or comment about the show? Why not write to our hosts: hosts@giantrobots.fm This has been a thoughtbot podcast. Stay up to date by following us on social media - LinkedIn - Mastodon - YouTube - Bluesky © 2026 thoughtbot, inc.

Sami is joined by one of her dear friends who has been such a pivotal part of her life lately, Hannah Eve — and this conversation is exactly the kind of girl chat we all need. From how their friendship formed to the big, messy, beautiful moments that brought them both to where they are now, this one goes deep in the best way.Tune in as Hannah opens up about one of the hardest seasons of her life: the heartbreak of a relationship ending, the lessons that came with it, and how she found faith and herself in the middle of it all. They talk about what it really means to look at life as happening for you rather than to you, the power of your circle, and how Hannah believes she genuinely manifested her dream relationship.They close it out with a rapid fire round you won't want to skip, from what Hannah would tell herself in her darkest moment, the one habit that changed everything, and the best advice she's received recently. Transform Instagram - click here!Sami Spalter Instagram - click here!Sami Clarke Instagram - click here!FORM Shop - click here!FORM Website - click here!Code TRANSFORM for 20% off an annual membership.This episode may contain paid endorsements and advertisements for products and services. Individuals on the show may have a direct, or indirect financial interest in products, or services referred to in this episode.This episode is brought to you by:Quince: Head to Quince.com/transform for free shipping on your order and 365-day returnsHungryroot: For a limited time get 40% off your first order PLUS get a free item in every box for life when you go to Hungryroot.com/transform and use code transformJolie: Try Jolie risk-free for 60 days by going to jolieskinco.com/transform Wayfair: Head to Wayfair.com right now to shop all things home.Ollie: Go to ollie.com/transform and use code transform to get 60% off your first box!Salt & Stone: Go to SaltandStone.com/TRANSFORM and use code TRANSFORM at checkout for 15% off your first order Produced by Dear MediaSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

In this episode of F-Stop Collaborate and Listen, host Matt Payne interviews Belgian photographer Hanneke Van Camp, whose work is rooted in her deep love for nature and her background in biology and science communication. Hanneke Van Camp shares the motivations behind her award-winning “Sápmi Living Landscapes” project, which explores the land and culture of Northern Scandinavia and the indigenous Sami people, as well as her experiences living seasonally in the Arctic for most of the year. The conversation delves into topics like the importance of presence over production in photography, the unique connection between environment and culture in the far north, practical ways photographers can infuse storytelling and meaning into their work, and how a values-driven approach—such as sustainability and giving back—can shape your creative life. They also discuss the process and challenges of turning a long-term project into a book, the impact of print vs. digital, and the influence of other photographers and storytellers on her journey. Links and Resources: Hanneke Van Camp Natural Landscape Photography Awards (NLPA) Order the Sápmi Book by Hanneke Van Camp Muench Workshops Vital Impacts (Amy Vitale's organization) Mother Magazine (Melissa Schäfer) Kevin Morgans – Prince of Puffins Feli Hansen (referenced project "Guilty Trashures") Marcus Westberg Freedom to Roam (Allemansrätten) Rewilding Europe Rewilding Sweden Matt's Arborglyphs Project Bonus Episode here on Patreon

Ioan 16 7. Totuşi vă spun adevărul: vă este de folos să Mă duc; căci, dacă nu Mă duc Eu, Mângâietorul nu va veni la voi; dar, dacă Mă duc, vi-L voi trimite.8. Şi când va veni El, va dovedi lumea vinovată în ce priveşte păcatul, neprihănirea şi judecata.9. În ce priveşte păcatul: fiindcă ei […]

Has the Unnamed Automotive Podcast really never reviewed a Volkswagen Jetta? That's situation has been resolved, and Benjamin has a full and thorough test of the German compact car, as Sami asks him tons of questions. What makes the Jetta a worthy pick over other small sedans? And how does it compare to hybrid options out there? Benjamin has plenty answers as we review this sedan for the first time ever. Then Sami checks out the 2026 Ford Explorer Tremor, an off road variant of the three-row crossover that replaces the old Timberline trim. Packed with a 3.0L turbocharged engine, the Ford Explorer has plenty of cool features and equipment, but Sami was really caught off guard by the hands-free driving tech. Thanks for listening!

Denise Salcedo and Mark Henry break down Sami Zayn abandoning Cody Rhodes in the ring to get attacked by GUNTHER on Smackdown. Also, they preview Saturday Night's Main Event! To visit our partners at Chewy, click here. The Master's Class is now available on its own podcast feed! SUBSCRIBE NOW to hear over 50 episodes of Dave, Bully, Mark, and Tommy taking you behind the scenes like only they can, plus BRAND NEW episodes every week. Subscribe to SiriusXM Podcasts+ to listen to new episodes of Busted Open ad-free and get exclusive access to bonus episodes. Start a free trial now on Apple Podcasts or by visiting siriusxm.com/podcastsplus. Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Wai Ting and Neal Flanagan review WWE SmackDown with Cody Rhodes vs. Sami Zayn, Trick Williams vs. Carmelo Hayes, Charlotte & Rhea vs. Fatal Influence, and the debut of Blake Monroe.XL: Wai & Neal discuss the latest involving Marcel Barthel (Ludwig Kaiser)'s arrest, AEW premiering a new show on TikTok, Scott Coker's new MMA venture, and MyAEW adding its first indy promotion.The XL Edition continues at POSTwrestlingCafe.com with News of the Day and Feedback, ad-free.Latest on Ludwig Kaiser arrestAEW Dynamite pre-show hits TikTokScott Coker's new MMA venture1FW (One Fall Wrestling) added to MyAEWPOST Wrestling Café Schedule:Friday: Rewind-A-SmackDown XLFREE Shows:Friday: Rewind-A-SmackDownSaturday: WWE Saturday Night's Main Event Sunday: AEW Double or NothingOur thanks to Quince: quince.com/postwrestlingAd Inquiries: info@truenativemedia.comPhoto Courtesy: WWERASD Theme by THE IDENTiTY CRiSiS: theidentitycrisis.com / youtube.com/theidcBluesky: https://bsky.app/profile/postwrestling.comX: http://www.twitter.com/POSTwrestlingInstagram: http://www.instagram.com/POSTwrestlingFacebook: http://www.facebook.com/POSTwrestlingYouTube: http://www.youtube.com/POSTwrestlingSubscribe: https://postwrestling.com/subscribePatreon: http://postwrestlingcafe.comForum: https://forum.postwrestling.comDiscord: https://postwrestling.com/discordSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

PWTorch editor Wade Keller is joined by PWTorch's Joshua White to review WWE Smackdown with live chat and caller interactions throughout including the great job Sami Zayn is doing with his current story and how Cody Rhodes is finding the right way to play off of him. Also, the Damien Priest-Royce Keys dynamic, Charlotte Flair-Rhea Ripley tension, Wade Barrett's comment about Brock Lesnar's retirement, and more.Become a supporter of this podcast: https://www.spreaker.com/podcast/wade-keller-pro-wrestling-post-shows--3275545/support.

In this week's episode of the Wade Keller Pro Wrestling Post-show from five years ago (5-21-2021), PWTorch.com editor Wade Keller was joined by PWTorch's Nick Barbati from PWTorch.com to discuss WWE Friday Night Smackdown with live callers and emails including Apollo Crews defending the Intercontinental Title against Big E, Kevin Owens, and Sami Zayn, Shinsuke Nakamura vs. Bianca Belair, a “Parade of Champions,” and Aleister Black attacks Big E, and a lot of excitement over WWE announcing tour dates again.Become a supporter of this podcast: https://www.spreaker.com/podcast/wade-keller-pro-wrestling-post-shows--3275545/support.

In 2019, Samantha Josephson, a college senior, was kidnapped and murdered after mistakenly entering a vehicle she believed was her Uber after a night out in Columbia, South Carolina.Join Mike and Gibby as they discuss Samantha Josephson. The case became nationally known because it highlighted rideshare safety concerns and eventually inspired state and federal legislation known as “Sami's Law.”You can help support the show at patreon.com/truecrimeallthetimeVisit the show's website at truecrimeallthetime.com for contact, merchandise, and donation informationAn Emash Digital productionSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Sami Inkinen (@samiinkinen) is a Finnish-born, Stanford-trained entrepreneur and the founder and CEO/president of Trulia and Virta Health. Virta is on a mission to reverse metabolic disease in one billion people using technology, AI, and nutrition. A world-class endurance athlete, Sami is a triathlon age-group world champion and an 8-hour, 24-minute Ironman finisher, having completed the Hawaii Ironman World Championship seven times.This episode is brought to you by:Eight Sleep Pod Cover 5 sleeping solution for dynamic cooling and heating: EightSleep.com/TimAG1 all-in-one nutritional supplement: DrinkAG1.com/TimWealthfront high-yield cash account: Wealthfront.com/Tim Wealthfront disclaimer: New clients get 3.30% base APY from program banks + additional 0.75% boost for 3 months on your uninvested cash (max $150k balance). Terms and conditions apply. The Cash Account offered by Wealthfront Brokerage LLC (“WFB”) member FINRA/SIPC, not a bank. The base APY as of 1/30/26 is representative, can change, and requires no minimum. Tim Ferriss, a non-client, receives compensation from WFB for advertising and holds a non-controlling equity interest in the corporate parent of WFB, which creates a conflict of interest. Individual experiences and outcomes will differ. Instant withdrawals may be limited by your receiving firm and other factors. Investment advisory services provided by Wealthfront Advisers LLC, an SEC-registered investment adviser. Securities investments: not bank deposits, not bank-guaranteed or FDIC-insured, and may lose value.DISCLAIMER:The content of this episode is for informational purposes only. Neither Sami Inkinen nor Tim Ferriss is a medical professional, and nothing discussed here should be taken as medical advice or a substitute for consultation with a qualified healthcare provider.Timestamps:[00:00] Start.[01:45] How Sami uses 15 minutes every Sunday to outrun the universe.[03:37] Virta: at a thousand employees and counting.[04:15] The 5 a.m. boot-up: cold lake, core work, and emptying the dishwasher.[06:45] Why mood follows movement before the brain even boots up.[11:54] Saying no to 99% of what “normal people” do.[19:29] The weekly architecture.[20:29] Two direct reports: the case for radical subtraction.[21:09] 553 CEO letters and the case for one scalable habit.[32:36] The text-file life plan.[33:32] The 15-year personal plan Sami stumbled into by accident.[34:30] The four-pillar formula for not cracking in 26 years of founder life.[38:20] What “white Japanese people” and beer steins in saunas have in common.[45:55] Smoke saunas, löyly, and the one Finnish word worth knowing.[48:37] The lean, ten-percent-body-fat triathlete who was quietly going prediabetic.[53:07] Why 93% of American adults are metabolically unhealthy.[56:05] Reversing type 2 diabetes the way Virta actually does it.[1:00:17] Most surprising interventions.[1:03:32] The pancreatic cancer trial that bought patients 35% more time.[1:07:02] The McDonald's protocol: how to reverse diabetes from the drive-thru.[1:16:00] Why GLP-1 adherence collapses and Virta's doesn't.[1:21:10] Vegans, tofu, and the hardest macronutrient to get right.[1:25:27] The dose-response curve that lets perfect stop being the enemy of progress.[1:29:32] VO2 max blocks: how Sami trains an 80+ engine without burning out.[1:41:56] Hacking 10% off your running speed in four weeks.[1:46:09] Progressive overload, specificity, and the case against the long ride.[1:50:07] 45 days, three hours, and a contract to keep a marriage afloat.[1:55:27] The lightning strike in the middle of the Pacific that started a family.[2:01:15] The 36-year-old who bought his first car only because his wife made him.[2:05:40] The book recommendation no one saw coming: Trejo.[2:07:51] The PSA: chronic, progressive, and irreversible — three words Sami refuses.[2:11:40] Parting thoughts.*For show notes and past guests on The Tim Ferriss Show, please visit tim.blog/podcast.For deals from sponsors of The Tim Ferriss Show, please visit tim.blog/podcast-sponsorsSign up for Tim's email newsletter (5-Bullet Friday) at tim.blog/friday.For transcripts of episodes, go to tim.blog/transcripts.Discover Tim's books: tim.blog/books.Follow Tim:Twitter: twitter.com/tferriss Instagram: instagram.com/timferrissYouTube: youtube.com/timferrissFacebook: facebook.com/timferriss LinkedIn: linkedin.com/in/timferrissSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Welcome back to Sami Spalter's Nesting Series on Transform. Today's episode is a personal one. Sami opens up about what it's been like to navigate pregnancy while also holding grief, stepping into motherhood while missing her own mom. It's an honest conversation about how joy and sadness can exist at the same time, and a reminder that you don't have to choose between feeling both.Throughout, Sami shares how this season has deepened her connection to her mom, along with stories from the FORM community that reflect just how many of us are carrying something similar. Plus, Sami walks through six simple, meaningful ways to honor the people we've lost while still being present in our lives today. If you've ever lost someone and are learning how to carry their love forward, this episode is for you.Transform Instagram - click here!Sami Spalter Instagram - click here!Sami Clarke Instagram - click here!FORM Shop - click here!FORM Website - click here!Code TRANSFORM for 20% off an annual membership.This episode may contain paid endorsements and advertisements for products and services. Individuals on the show may have a direct, or indirect financial interest in products, or services referred to in this episode.This episode is brought to you by:Cozy Earth: Head to CozyEarth.com and use code TRANSFORM for up to 30% off - but only for a limited time. This exclusive offer runs from May 18th through June 1st only, so don't wait.The Real Real: Get $25 off your first purchase when you go to TheRealReal.com/transformWayfair: Head to Wayfair.com right now to shop all things home.Ollie: Go to ollie.com/transform and use code transform to get 60% off your first box!BiOptimizers: Go to BiOptimizers.com/samispalter and use code SAMI for 15% off your entire order + a free bottle of Masszymes added to your order automatically when you use our exclusive code.Spindrift: Visit drinkspindrift.com and use code transform for 15% off your next Spindrift orderProduced by Dear MediaSee Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Every Saturday, we revisit a story from the archives. This originally aired on June 1, 2020. None of the dates, titles, or other references from that time have been changed. When coronavirus hit the cruise industry, many passengers were allowed to disembark and were repatriated to their home countries. But as of mid-May, about 100,000 crew members were still at sea, trying to get home. We speak with Sterling Howell, musician and cruise line entertainer crew member who was able to go home, Krista Thomas, former guest services director for Norwegian Cruises, and with many who are still stuck on board, including Ghoseawon “Albert” Luxmeeparsad, and other Mauritian crew members. For more: If you are struggling with mental health issues, there are suicide crisis lines available in many countries around the world. Here are some of those resources: Open Counseling International Association for Suicide Prevention Global Suicide Hotline Resources – WhatsApp In this episode: Sterling Howell, Cruise Ship Crewmember Krista Thomas, Former Guest Services Director for Norwegian Cruises Dr. Sami, Physician Gan, Cruise Ship Crewmember Episode credits: This episode was updated by Chloe K. Li. The original production team was Amy Walters with Dina Kesbeh, Priyanka Tilve, Ney Alvarez, Alexandra Locke and our host, Malika Bilal. Our sound designer is Alex Roldan. Our engagement producers are Adam Abou-Gad and Vienna Maglio. Andrew Greiner is lead of audience engagement. Alexandra Locke is The Take’s executive producer. Connect with us: @AJEPodcasts on X, Instagram, Facebook, and YouTube

Jordana is officially back from maternity leave and this week she joins Sami to talk a surprisingly deep conversation about love languages, birthday gifts, and Mother's Day texting etiquette. Then, they cover Ciara Miller's post-Summer House scandal success and how Bravo betrayals somehow turn women into superstars. Plus, they debate whether Anne Hathaway is secretly annoying, review why The Devil Wears Prada 2 is the best sequel in years, and unpack NBC turning Wordle into a game show. Go to the Betches Podcast YouTube page to watch full length episodes every Friday: Youtube.com/@betchespod   Learn more about your ad choices. Visit megaphone.fm/adchoices

In 1980, Norway's entry to the Eurovision Song Contest included a traditional Sami joik song from the country's indigenous community. It was the first time joiking was heard by an international audience. The song was originally created by Mattis Haetta as part of a protest against the building of a dam in Finnmark, in northern Norway – which would have flooded traditional Sami reindeer herding routes and villages. Surya Elango speaks to Inga Haetta, the sister of Mattis.Eye-witness accounts brought to life by archive. Witness History is for those fascinated by and curious about the past. We take you to the events that have shaped our world through the eyes of the people who were there. For nine minutes every day, we take you back in time and all over the world, to examine wars, coups, scientific discoveries, cultural moments and much more. Recent episodes explore everything from how the Excel spreadsheet was developed, the creation of cartoon rabbit Miffy and how the sound barrier was broken.We look at the lives of some of the most famous leaders, artists, scientists and personalities in history, including: the moment Reagan and Gorbachev met in Geneva, Haitian singer Emerante de Pradines' life and Omar Sharif's legendary movie entrance in Lawrence of Arabia.You can learn all about fascinating and surprising stories, like the invention of a stent which has saved lives around the world; the birth of the G7; and the meeting of Maldives' ministers underwater. We cover everything from World War Two and Cold War stories to Black History Month and our journeys into space.(Photo: Mattis Hætta on Eurovision 1980. Credit: Stein Dag Jensen/National Library of Norway)