Podcasts about Pla

Les sardanes d'estiu a Lloret de Mar celebren el centenari aquesta temporada i l'Ajuntament vol celebrar-ho d'una manera especial. Per això aquest dissabte es farà un concert commemoratiu. Anirà a càrrec de la Cobla Orquestra Montgrins, que precisament és la cobla–orquestra més antiga en actiu. L'actuació es farà a l'amfiteatre dels Jardins de Santa Clotilde, un espai idíl·lic per a una actuació d'aquesta rellevància, tal com assenyala la regidora de Cultura i Festes, Vereda López. “Vam pensar que l'amfiteatre dels Jardins de Santa Clotilde era l'espai més idíl·lic i especial que tenim actualment al municipi per fer el concert commemoratiu d'aquest centenari”Vereda López En aquest concert, s'estrenarà la sardana Cent anys de sardanes a Lloret del lloretenc Xavier Albertí. Un concert d’Elena Gadel a l’amfiteatre dels Jardins de Santa Clotilde (Ajuntament de Lloret de Mar) L'actuació començarà a dos quarts de nou del vespre i l'Ajuntament posa a disposició de la ciutadania un autocar gratuït per anar fins als Jardins de Santa Clotilde, que sortirà a dos quarts de vuit des de la Plaça de la Vila. L'entrada és gratuïta, però cal descarregar la invitació a través del portal web de Patrimoni Cultural o bé de manera presencial al Puntet. El centenari de les sardanes d'estiu a Lloret arriba amb una novetat: s'oferiran uns tallers perquè tothom que ho vulgui pugui aprendre a ballar sardanes. Serà abans de començar les audicions dels tres primers dissabtes, és a dir, el 27 de juny i també el 4 i 11 de juliol. A més, com cada any, l'estiu estarà farcit d'audicions cada dissabte, amb les principals cobles del país. La regidora considera que arribar als cent anys de sardanes a Lloret demostra l'aposta del municipi per la cultura popular. “Lloret, a més de tot el que tothom sap que és, també és això, també és cultura, és gaudir, és aquesta cultura popular catalana que segueix viva”Vereda López Les audicions es faran cada dissabte, a les deu de la nit, a la Plaça de la Vila. Els dies que hi hagi taller, es farà mitja hora abans. La primera cobla convidada, dissabte 27 de juny, és la Ciutat de Girona.

Conversem amb l'artista ampostina per endinsar-nos en un disc de dream pop que transita entre la veu i l'experimentaci

#DigitālāsBrokastis galdā svaigākie mākslīgā intelekta jaunumi, kuru priekšgalā ir stāsts par “Apple”, kas beidzot izziņojis savu mākslīgā intelekta Siri risinājumu. Tikmēr “OpenAI” uzlabo “ChatGPT” atmiņu ar jauno "dreaming" pieeju. Mākslīgā intelekta uzņēmumi dodas uz biržu. Igaunija skolēniem piešķir bezmaksas ChatGPT piekļuvi. Un FIFA Pasaules kausā futbolā mākslīgais intelekts ir klātesošs. Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

Kas ir sauszemes droni un kādi ir to pielietojuma veidi? Kā Latvijas uzņēmumi veido moderno aizsardzības infrastruktūru? Pie Digitālo brokastu galda divi uzņēmēji — Kristaps Puķe un Armands Sakne. Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

Sekošana līdzi savai veselībai un patērētajai pārtikai ir kļuvisi teju par modes tendenci, un tam palīgā nāk arī tehnoloģijas. Viena no tām ir lietotne “Cal AI”, kura nesen izpelnījusies lielu uzmanību lietotņu tirgū, un to pat pārpirkuši “MyFitnessPal” veidotāji. Ko ar to var izdarīt un kāda no tā vispār jēga? Noskaidrojam apskatā! * Lietotni testam sagādājām paši. Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

On today's show Andrew and Bill begin with takeaways from Xi's visit to North Korea this week, including the conspicuous silence on North Korea's nuclearization, Kim Jong Un's assistance to Russia's war in Ukraine, Beijing as Kim's top priority, U.S.-Japan dialogue on regional nuclear threats, and an email about the PRC as a communist country. From there: CPPCC Chairman Wang Huning leads an inspection tour of Xinjiang ahead of the July 1st implementation of the national ethnic unity law, plus thoughts on Xinjiang's strategic importance generally and why Beijing sees its recent efforts as successful. At the end: China preps for an AI infrastructure buildout, the Pentagon alleges that Alibaba, Baidu and BYD are linked to the PLA, the Busan truce is being tested by both sides, and two Knicks stars wish students good luck on the GaoKao.

Henrikas III Valois vienas kertinių Geriausių Tautų Respublikos politinės sistemos architektų. Nors eilėje iki gimtosios Prancūzijos sosto jis laukė ne pats pirmas, tačiau motinos širdyje jis nuo pirmų dienų užėmė privilegijuotą vietą. Ši spalvinga asmenybė nepasižymėjo nei kuklumu, nei santūrumu, tačiau už plėvėsiško fasado slėpėsi talentingas, nors kiek netašytas, administravimo deimantas. O talentingų valdovų valstybei reikėjo kaip vandens kupranugariui - sostų žaidime iš visų pusių smūgiavo galios ištroškę didikai, religiniai fanatikai, bei artimiausi šeimos nariai. Kokiu vardu Henrikas buvo žinomas vaikystėje? Kodėl Prancūzijos valdovas keliavo nuo rūmų prie rūmų? Kokios traumos suformavo būsimo GTR karaliaus asmenybę? Ir kuo jis susijęs su pimpačkiukais?   Pasiilgai Geriausių Tautų Respublikos? Nes ji pasiilgo tavęs...   TELE2 5G Namų internetas – greitas ir pigus. Plačiau: https://tele2.lt/namu-internetas Domina karjera Omnisend? Eik į https://www.omnisend.com/katuciaveiki   Iliustracija: @korinavaicikone Muzika: Freesound_community, SoundReality, Wings of Freedom, Alex Grohl, Ashot_Danielyan, Inplusmusic, Petrushkasound, Zec53, Gregor Quendel, nesrality, Iithop, Emand Edroff, Artarea Studio, Denis Pavlov, SamuelFJohanns

L’inquinamento da plastiche passa anche attraverso la produzione di compost di cattiva qualità, inquinato a sua volta da microplastiche se il materiale di partenza è contaminato a monte. A complicare le cose c’è poi il fatto che questi residui sono difficili da distinguere dai residui di polimeri compostabili - bioplastiche come il mater-b o il PLA -, che invece hanno tutto il diritto di trovarsi frammisti al compost. Da qui, il lavoro fatto congiuntamente da Cnr-Isafom e Università degli Studi di Milano: un nuovo protocollo di analisi che permette in modo facile ed economico di quantificare eventuali plastiche “cattive” presenti nel compost, che potrebbe presto diventare un test standard. Ne parliamo con Mirko Cucina, primo ricercatore dell’Istituto per i Sistemi Agrari e Ambientali nel Mediterraneo, del CNR di Perugia.

Užsienio kultūros naujienos: septyni Sakartvelo piliečiai teisiami dėl retų rusiškos literatūros klasikos leidinių vagysčių iš Prancūzijos bibliotekų; į Vašingtono statybos projektus nukreiptas Jungtinių Amerikos Valstijų nacionalinių parkų bilietų pardavimo pajamas; teatro apdovanojimai „Tony“. Plačiau – Ieva Radzevičiūtė.Ar galima važiuojant pro šalį aplankyti „Dingusį štetlą“ Šeduvoje? Ar būtina iš anksto registruotis? Muziejaus lankymo galimybes aiškinamės su muziejaus komunikacijos vadove Marija Dautartaite.Prieš trisdešimt metų Pažaislio vienuolyne nuskambėjo didingas klasikos kūrinys – Ludwigo van Beethoveno oratorija „Kristus Alyvų kalne“. Tokia buvo tradicinio Pažaislio muzikos festivalio pradžia, kurią prisiminsime su vienu iš festivalio įkūrėjų Justinu Krėpšta, o apie šių metų festivalį kalbėsime su kuratore Lina Krėpštaite.Klaipėdoje atidaryta VDA Klaipėdos fakulteto galerija DARŽAS. Naujojoje ekspozicinėje erdvėje – studentų baigiamųjų darbų paroda. Kaip išsiaiškino DARŽE apsilankiusi žurnalistė Agnė Bukartaitė, pirmiausia čia suteikiama galimybė iš arti susipažinti su nauja kūrėjų karta ir jos formuojamu šiuolaikinės kultūros lauku, taip pat galima pamatyti ir profesionalių menininkų kūrybą.Mažiems ir dideliems daug džiaugsmo savo eitynėmis ir koncertais parko rotondoje teikiantis Palangos orkestras pradeda „Orkestro vasaros“ sezoną. Plačiau – pokalbis su Palangos orkestro meno vadovu ir vyriausiasiu dirigentu Vygantu Rekašiumi.Ved. Jolanta Kryževičienė

La recreació del mític Floridita de l'Havana, on es va popularitzar el daiquiri de la mà del lloretenc Constantí Ribalaigua, és un dels atractius principals de la Fira dels Americanos, que aquest cap de setmana celebra la 13a edició a Lloret de Mar. Un altre dels punts forts de la fira és, sens dubte, el mercat indià, organitzat per l'empresa Balconet Recreacions. El seu responsable és Juan Cantero, que remarca que hi haurà una seixantena de parades variades que està convençut que atraparan al públic. “Tindrem varietat, sobretot d'artesania: joieria, bijuteria, sabons, bosses… També alguns espais de garrapinyades, caramels i una mica d'alimentació”Juan Cantero El mercat indià obrirà les portes demà divendres, a les tres de la tarda, i es podrà visitar fins diumenge a les deu de la nit. Durant aquests tres dies, s'han previst una quarantena d'activitats variades entre el passeig Verdaguer i la Plaça de la Vila. Hi haurà cercaviles, espectacles de circ modernista, animacions teatrals i un dels punts forts: tallers de balls caribenys i sessions musicals. Entre les propostes musicals destacades hi haurà les actuacions de Mixael Cabrera i Mariana y La Makinaria. La programació musical de l'esdeveniment va a càrrec de Dynamo Entertainment, amb Armand Galarraga al capdavant. “Intentem sempre que l'ambient faci ebullició, que hi hagi diversió i també el punt cultural musical i que les actuacions siguin d'un rigor i una qualitat que crec que estan a l'altura de la fira”Armand Galarraga A més, també hi haurà exhibicions de Dynamo Dance Team, les representacions teatrals d'Els periodistes i Els banyistes i la tradicional cantada d'havaneres amb el grup Peix Fregit. Tot el programa es pot consultar a través del portal web de l'Ajuntament. Armand Galarraga considera que hi ha un conjunt d'aspectes que fan especial la Fira dels Americanos de Lloret de Mar. “Lloret, l'entorn, el microclima lloretenc, l'estima per aquest tipus de cultura, la música, l'ambient distès i festiu i les 13 edicions avalen la trajectòria de la fira”Armand Galarraga Una de les novetats d'aquest any serà el repartiment de 500 barrets oficials de la Fira dels Americanos.

La Plata Alejandro Dolina, Patricio Barton, Gillespi Introducción • 0:09:57 Presentación en La Plata y agradecimiento al público • 0:12:06 Recuerdo de la primera salida del programa desde Buenos Aires hacia La Plata • 0:12:35 Presentación de Patricio Barton y Gillespi Segmento Inicial • 0:13:21 Qué significa ser romántico hoy • 0:16:06 Regalos románticos recomendados y objeciones • 0:20:37 Desayuno sorpresa a domicilio y sus inconvenientes • 0:27:24 Notas manuscritas como gesto romántico • 0:30:39 Escapadas de fin de semana y paseos románticos • 0:32:23 Regalar cachorros y responsabilidades afectivas • 0:34:44 Cocinar para la pareja como prueba de amor • 0:36:22 Pasacalles, sándwiches en la costanera y otros gestos desaconsejados • 0:40:29 Regalos domésticos, dinero y publicaciones en redes sociales • 0:45:05 Peluches gigantes, mensajes aéreos y límites del romanticismo • 0:52:23 Regalar canciones como gesto amoroso Segmento Dispositivo • 0:54:56 Las plañideras y el llanto profesional en la antigüedad • 0:56:21 Lloronas en hebreos, griegos y romanos • 0:59:28 Rituales funerarios egipcios con plañideras • 1:02:57 Plañideras en Grecia, Roma y la Edad Media • 1:06:06 Función ritual del llanto y reflexión sobre las lágrimas Segmento Humorístico • 1:13:01 Cuidado con la automedicación • 1:14:46 Confusiones entre gotas, dosis excesivas y antibióticos mal usados • 1:20:32 Pastillas por color, medicamentos vencidos y remedios veterinarios • 1:24:15 Interrumpir tratamientos, tragar antisépticos y triturar pastillas • 1:28:14 Intercambio de remedios, curanderos y peligros al tomar cápsulas sin agua • 1:31:15 Mezcla de sedantes con alcohol y cierre sobre el botiquín doméstico Sordo Gancé / Trío Sin Nombre • 1:42:21 Presentación del Sordo Gancé y el Trío Sin Nombre • 1:43:16 "Mañana campestre" ♫ • 1:46:02 "Tu casa ya no está" ♫ • 1:49:04 Ingreso de Gillespi y "Blue Moon" ♫ • 1:51:36 Recuerdo de "Golondrina" • 1:55:15 Homenaje musical a un amigo • 1:56:54 "Todo un palo" ♫ • 1:59:17 "Solo le pido a Dios" ♫ • 2:00:23 Cierre musical y agradecimiento al público (Resumen generado automáticamente con IA, puede contener errores)

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

Vilnių galima pažinti ir per kiną – jau startavo kino turizmo maršrutai po filmavimo vietas. Plačiau apie tai pasakoja Vilniaus kino biuro direktorė Jūratė Pazikaitė.Ved. Agnė Skamarakaitė.

„Popiežius Leonas birželio 6–12 d. su apaštaliniu vizitu lankysis Ispanijoje. Tenykštės Bažnyčios vadovai teigia, kad susitikimas su katalikų bendruomene vyksta lemiamu momentu: nors pastaraisiais dešimtmečiais katalikais save laikančių ispanų skaičius sumažėjo, atkreipiamas dėmesys į atgimstantį jaunimo susidomėjimą katalikybe.“ Plačiau - vedamojo skiltyje.Vilniuje prasideda Pasaulinis Apaštalinis Gailestingumo kongresas. Gailestingumo temai skirtas ir žurnalo „Artuma“ birželio numeris, kurį pristatys vyriausiasis redaktorius Darius Chmieliauskas.Augmino Petronio komentaras: eitynės už lygybę.„Krikščioniškos minties puslapis“: William Willimon „Kaip pamilti gailestingumą?“Kun. Mozė Mitkevičius apie gailestingumo terminus ir turinį Senajame bei Naujajame Testamente.Eleonoros Terleckienės radijo apybraiža „Apie poilsį“.Redaktoriai Rūta Tumėnaitė ir Julius Sasnauskas.

Garrett Chaffin-Quiray and Ed Rosa wonder if bears prefer eating old or young white dudes. Bon appétit, Bart!***Referenced media:“The Silence of the Lambs” (Jonathan Demme, 1991)“First Blood” (Ted Kotcheff, 1982)“Glengarry Glen Ross” (James Foley, 1992)“Once Were Warriors” (Lee Tamahori, 1994)“The Texas Chainsaw Massacre 2” (Tobe Hooper, 1986)“Road House” (Rowdy Herrington, 1989)“The Game” (David Fincher, 1997)“LA Confidential” (Curtis Hanson, 1997)“The Ice Storm” (Ang Lee, 1997)“U Turn” (Oliver Stone, 1997)“Boogie Nights” (Paul Thomas Anderson, 1997)“The Sweet Hereafter” (Atom Egoyan, 1997)“The Devil's Advocate” (Taylor Hackford, 1997)“Prometheus Rising” (Robert Anton Wilson, 1983) Audio quotation:“The Edge” (Lee Tamahori, 1997), including “End Title”, “The Edge”, “Rich Man”, and “Deadfall/Bear Fight” by Jerry Goldsmith, https://www.youtube.com/playlist?list=PLoTgadYyOMmYIku-y2NBhrY1nJE4WdBt_“Campfire by the Lake Ambience with Crickets, Owls, Water, & Night Sounds for Relaxation & Sleep” (2021), posted by Calmed by Nature, https://www.youtube.com/watch?v=9nBFKH3qhGE“First Blood” (Ted Kotcheff, 1982), including “Escape Route” by Jerry Goldsmith, https://www.youtube.com/playlist?list=PLA_6nw_SiTT23YKnzHA24tICcBsYJH-b1“Glengarry Glen Ross” (James Foley, 1992)“bolt action rifle sound effects” (2011), posted by MasterHand125, https://www.youtube.com/watch?v=STbDaAgcvsU“Glasses clinking sound #glass #cheers” (2025), posted by @sound_magic73, https://www.youtube.com/shorts/4VuUSk3xGos“Dersu Uzala” (Akira Kurosawa, 1975), including “Journal” by Isaak Shvarts, https://www.youtube.com/watch?v=6zNtQVYyC4M&list=RD6zNtQVYyC4M&start_radio=1

Vēl pirms dažiem gadiem mākslīgā intelekta radīti dziļviltojumi jeb "deepfakes" šķita drīzāk tehnoloģiju pasaules kuriozs, taču mākslīgā intelekta rīku straujā attīstība ļauj krāpniekiem ļoti reālistiski klonēt cilvēka balsi un seju, un pat imitēt to video zvanā. Mākslīgā intelekta dziļviltojumi no izklaides ir pārtapuši par nopietnu kiberdrošības apdraudējumu, kas ietekmē pat sabiedrības procesus. Pie Digitālo brokastu galda CERT.LV kiberdrošības eksperts Mārtiņš Vecstaudžs un privātās izlūkošanas aģentūras “Rossad” direktors Madars Balodis. Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

¡Difícil! Clint Eastwood se retira, Pedro Sánchez no. El Guapo, hasta el infinito y más allá. Sí, el seguro de vida de Perro Sanxe, ‘el One', es Abascal. La casita, el confesionario, la trama, la cloaca, el pisito, el empujón, la nariz rota, la Plaça de la Mare de Déu. Paquita la de Mónaco, Leire y Haaland. Carisma y tirria. Dos Españas, dos relatos. Florentino, Riquelme. Y nosotros, ¡la periferia! Juanfran, ¿eres capaz de ver el aguilucho y Lo Rat Penat en menos de 30 segundos? Semana de toreros y espontáneos: el salto de la rana. Feijóo —“ofrezco decencia y elecciones”— decepciona. Again. Trump, ante la amenaza de un nuevo Vietnam y las elecciones de noviembre, le chilla a Netanyahu: “Estás jodidamente loco”. Què li diu el mort al degollat? Leonor se tira en paracaídas. Juanfran, ¡al ataque! ¡Es que los sindicalistas van provocando! Rovira —la ilustración negra— y Mazón —el impugnador del Ventorro— me bloquean al president. Juanfran es un falso llaurador, un tiburón atrapado dentro de Paco Martínez Soria. Juanfran, mucho bla, bla, bla…, ¡si yo nunca he roto un plato! ¡Sí, mante! Pero Juanfran —en verdad, en verdad os digo— está curtido en fajarse con mafiosos, dulces e inofensivas abuelitas y traficantes de tierras raras, drones y voluntades. El president del Partit Popular del País Valencià no baja al cara a cara de la negociación con los huelguistas —poca broma, vergonya, dignitat!— porque se juega su permanencia y futuro. Aznar prefiere a María José Catalá. ¡El Mediterráneo hierve! ¿Cómo elegir un buen melón? Bueno, suena Unai Sordo. Sin Mónica Oltra no hay tu tía. Barcala, alcalde por la gracia de Vox, dos años sin fumar, rompió su voto de silencio para soltar: “No contesto”. ¡Ja estem en Fogueres! El dominio del fuego, deshielo glacial. Valencians, alacantins, levantinos, ¡Arcaya dimissió! Letizia de blanco, Charlene de azul, por el jardín. Estampida de caballos en Roma. ¡Milagro! Los obispos españoles —siempre más a la derecha— vuelven a creer en Dios: Leo Messi, por supuesto. “Dios nos coja confesados”, León XIV. ¡Que me quede como estoy! ¡Difícil!

#DigitālāsBrokastis servē svaigāko tehnoloģiju ziņu topu! Hakeri ar "Meta" MI palīdzību uzlauzuši "Instagram" kontus. Pētījums: cena un laiks motivē latviešus iepirkties internetā. NASA šogad plāno vēl pāris Mēness misijas. Ķīnā policija sāk izmantot MI viedbrilles seju un numurzīmju atpazīšanai. "Oura" prezentē līdz šim mazāko viedu gredzenu "Ring 5". Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

La Corporació de Salut del Maresme i la Selva ha obtingut uns resultats superiors a la mitjana catalana en satisfacció i fidelitat de les gestants durant l'embaràs, el part i el puerperi. Així es desprèn de les dades del Pla d'Enquestes de Percepció, Experiència i Satisfacció d'Usuaris del Servei Català de la Salut (PLAENSA), que avalen el model assistencial desplegat els darrers anys. Des de la Corporació atribueixen aquests resultats a diverses accions impulsades per millorar l'experiència de les dones. La cap del Servei de Ginecologia i Obstetrícia, Marta González, assenyala tres elements clau: la renovació dels espais destinats al part natural, la formació conjunta dels professionals i l'establiment de protocols compartits que garanteixen una mateixa línia d'actuació. En aquest sentit, González destaca que l’èxit de les enquestes es relaciona amb «les noves sales de part natural, on la introducció de la banyera, tècniques com l’aromateràpia o l’aparellatge de part natural donen una gran satisfacció a les nostres gestants». La responsable també posa en valor la formació específica de llevadores i ginecòlegs, així com l’existència de protocols unificats que permeten oferir una atenció coherent i transmetre més confiança a les pacients. «Intentem no només cuidar el cos sinó veure la dona com un tot més global donant moltíssima importància també a la part emocional» Marta González Les valoracions de les usuàries també reflecteixen la importància que té l’acompanyament emocional durant aquesta etapa. Segons explica la cap del servei, el model assistencial va més enllà de l’atenció estrictament mèdica i busca donar resposta a les necessitats globals de cada dona. La doctora González assegura que el nou model d’atenció «dona moltíssima importància el tracte respectuós, més humà i més proper que donem a les dones». En aquesta línia, destaca el paper que tenen iniciatives com la matronatació o les sessions de ioga en l’acompanyament de les gestants al llarg de tot el procés. Les dades recollides pel Servei Català de la Salut mostren que la Corporació supera la mitjana de Catalunya en diferents indicadors vinculats a l’experiència de les usuàries i a la seva predisposició a tornar a escollir el mateix centre o recomanar-lo a altres dones. Uns resultats que consoliden l’aposta de l’organisme per un model d’atenció centrat tant en la qualitat assistencial com en el benestar emocional de les gestants.

Sankt Peterburge prasidedant Tarptautiniam ekonomikos forumui, buvo atakuotas vienas svarbiausių Rusijos naftos terminalų esantis Suomijos įlankoje. Pasak Ukrainos žiniasklaidos, objekte nugriaudėjo keli stiprūs sprogimai, kilo gaisrai. Tuo metu Sankt Peterburge prasidėję Rusijos investicijų forumas surengtas penktą kartą nuo Rusijos įsiveržimo į Ukrainą pradžios. Plačiau – LRT radijo bendradarbis, politologas Maksimas Milta.

Latvijas Transportlīdzekļu apdrošinātāju biroja veiktajā aptaujā atklājies, ka gandrīz puse autovadītāju nezina, kā pareizi rīkoties pēc ceļu satiksmes negadījuma. Kāda būtu pareiza rīcība pēc avārijas, skaidrojam raidījumā Kā labāk dzīvot. Plašāk stāsta Latvijas Transportlīdzekļu apdrošinātāju biroja IT daļas vadītājs Agris Daukste un satiksmes eksperts Oskars Irbītis. Oskars Irbītis norāda, ka nokļūšana negadījumā, par laimi, nav ikdienišķs pasākums cilvēkiem, ko papildina apjums, stress. Tāpēc saprast, kā pareizi noformēt saskaņoto paziņojumu par negadījumu bieži vien ir diezgan lielas problēmas.  "Vislielākās grūtības cilvēkiem sagādā, kad ir jāaizpilda saskaņotais paziņojums. Mani novērojumi, izskatot arī sūdzības, ka cilvēki jauc elementārām lietas un jēdzienus, piemēram, kas īsti ir apdzīšana un kas ir iebraukšana blakus joslā. Liela nesaprašana ir par dažādu manevru atšifrēšanu. Ja saskaņotajā paziņojumā kāds ir izdomājis apzīmēt vairāk par trīs ķeksīšiem, tas nozīmē, ka kaut kas nav saprasts," norāda Oskars Irbītis. "99% gadījumu būs jāatzīmē tikai viens ķeksītis, viena situācija, ko atlasām no attiecīgā saraksta. Ļoti daudzos gadījumos nevajadzēs atķeksēt nevienu ķeksīti cilvēkam, kurš vienkārši brauc taisni. Dažas elementāras lietas, kas sagādā milzīgas grūtības. Tāpēc mans uzskats - par to ir jārunā, jārunā, jārunā un vēlreiz jārunā," atzīst Oskars Irbītis. Abi viesi mudina nepieciešamības gadījumā lejplādēt Latvijas Transportlīdzekļu apdrošinātāju biroja lietotni, kur pieejams saskaņotais paziņojums, bet tajā pašā laikā nebūtu slikti, ja mašīnā būtu tas papīra formātā, jo negadījumi mēdz notikt arī vietās, kur nav sevišķi labs interneta pārklājums. Ja vien negadījumā nav cietušie, viesi noteikti mudina nelaimes gadījumā iekļuvušajiem vienoties, jo gaidīt jau tā noslogoto policiju būs ilgi un tas raisīs papildus daudz nepatīkamu emociju.

Naujausiame „Basketnews.lt podkasto“ epizode Jonas Miklovas, Karolis Tiškevičius ir Tomas Purlys aiškinosi, kuris pralaimėjo Eurolygos prognozių lažybas ir kokia bausmė vieno iš jų lauks sąskrydyje. Trijulė taip pat aptarė lūžtančias tribūnas Utenoje, „Žalgirio“ skolininkus rūbinėje, NBA finalą pasitikančio Wembanyamos fenomeną bei Šarūno Jasikevičiaus sūnų 14-mečių rinktinėje. Tinklalaidės partneriai: Jon-Chedar – čederio gabaliukai, priversiantys skonio receptorius šokti. Svajonių užkandis ieškantiems kažko gardaus kelionėms, vakarėliams ar vakarui su krepšiniu.  Telia Play. Krepšinis, filmai, serialai – viskas vienoje vietoje. O dabar Samsung televizorius tik 69€ su Telia Play planu MIX. Plačiau. Nealkoholinis alus „Gubernija“.  Tėčiams, mylintiems krepšinį – BN+ narystė dovanų! Įsigyk čia.Temos: Vyrai, paskaičiuokime Eurolygos finalo ketverto pavasario viščiukus (0:00); LOGIN konferencijoje – įstrigę Pauliaus Motiejūno žodžiai (09:17); Milžiniškas anšlagas Utenoje ir istorinis šansas finalo link (16:41); Nekuklios Eglinsko frazės ir filmas apie „Juventus“ (24:30); Europos taurės renesanse – skambūs vardai ir naujas formatas (26:42); Augantys savivaldybių pinigai klubams ir gėda Panevėžiui (31:30); Keisti Čanako priekaištai teisėjams (41:45); „Žalgirio“ skolininkai ir nestebinančios kauniečių pergalių atkarpos (46:10); Kitų Europos grandų atkrintamųjų situacija vietiniuose čempionatuose (48:50); Išspręstas Atamano ateities galvosūkis ir grįžtantis Željko? (51:29); LKL sezono finišo datos, padėsiančios rinktinei (54:06); Pasiteisinęs į ACB atkrintamąsias įšokusio Margo sprendimas? (55:04); Intriguojantis NBA finalas, bilietų kainos Niujorke ir antžmogiško Wembanyamos branda (58:11); Lukui Jasikevičiui – kvietimas į 14-mečių rinktinę ir tėčio aikštės matymas (1:10:56); Aplodismentai „Auksinio proto“ finalą užtikrintai laimėjusiam BN pliusui Benjaminui (1:14:46).

Kanto: “Ni kunvenas”,de Kaj Tiel plu el la kompaktdisko PLaĉas al mi Legado: Heather el Gazetara komuniko “ La 113a Universala kongreso en Kaŭnas en 2028″. Parolado : Brendan pri la trajnsistemo en Melburno kaj Viktorio. Kanto: “La blondulineto” de ĴoMo el la kompaktdisko   Liberecanoj . Legado: Heather 1) el la revuo Esperanto “ Kvaronjarcento […]

China and the United States should implement the important consensus reached by the two countries' leaders and work toward a stable and sustainable military-to-military relationship, Chinese scholars said at a high-level security forum in Singapore, as global security faces rising risks from hegemonism, disorder of global governance and emerging technologies.当前，霸权主义、全球治理失序以及新兴技术等因素令全球安全风险不断攀升。中国学者在新加坡一场高级别安全论坛上表示，中美两国应落实两国元首达成的重要共识，推动两军关系朝着稳定、可持续的方向发展。Major General Meng Xiangqing, a professor at China's National Defense University, made the remarks on Saturday during a parallel session of the three-day Shangri-La Dialogue, which concluded on Sunday.为期三天的香格里拉对话会于周日落幕。中国国防大学教授孟祥青少将在周六的专题分论坛上发表了上述观点。His remarks came after US Defense Secretary Pete Hegseth referred to the recent China-US top leaders' meeting in Beijing and their consensus, during a plenary speech earlier on Saturday.周六早些时候，美国国防部长皮特・赫格塞思在全体大会发言中提及近期中美两国元首在北京举行的会晤及双方达成的共识，随后孟祥青作出相关表态。Meng said the level of attention the two leaders' meeting received at the forum showed that stability in China-US relations serves not only the interests of the two peoples, but also regional stability and world peace.孟祥青表示，此次元首会晤在论坛上受到高度关注，这说明中美关系稳定不仅符合两国人民的利益，也有益于地区稳定与世界和平。The most important political consensus reached by the two sides is to build a constructive relationship of strategic stability between China and the US, Meng said.他指出，双方达成的最重要政治共识，是构建中美建设性战略稳定关系。"We expect China and the US to meet each other halfway, translate the consensus into concrete actions, and push military-to-military relations toward healthy, stable and sustainable development," he said.他说：“我们期待中美双方相向而行，将共识转化为实际行动，推动两军关系实现健康、稳定、可持续发展。”Responding to a question from a member of the Chinese delegation after his speech, Hegseth said the new vision of building a constructive US-China relationship of strategic stability is "real, substantive and meaningful for the history of peace in the region and the world".赫格塞思发言结束后，回答了中方代表团成员的提问。他表示，构建中美建设性战略稳定关系这一新愿景是切实、务实的，对地区乃至世界和平发展历程都具有重要意义。Hegseth said he was present when the leaders discussed constructive strategic stability. "I think that was a great framing from both leaders about what they want from that relationship," he said, adding that there is "a mutual respect, a recognition of capabilities and power and how that could be most usefully leveraged in the world today".赫格塞思称，两国元首探讨建设性战略稳定相关内容时他在场。他表示：“两国元首为双边关系发展指明了良好方向。” 他还提到，双方相互尊重，正视彼此的实力与影响力，并思考如何在当今世界合理发挥这些力量的作用。Wang Dong, a professor at Peking University's School of International Studies, said that Hegseth's speech this year contained far fewer negative references to China compared with speeches by US defense chiefs in previous years, and did not mention Taiwan or the South China Sea, two hot topics that had often been cited in the past.北京大学国际关系学院教授王栋表示，相较于往年美国国防部长的发言，赫格塞思今年的讲话针对中国的负面表述大幅减少，也没有提及以往频频出现的台湾、南海两大热点议题。Wang, who participated in the security summit, said the change in Hegseth's tone reflected a more cautious approach by the US in handling relations with China after the two countries agreed to build a constructive relationship of strategic stability.出席本次安全峰会的王栋认为，美方态度出现转变，反映出在双方就构建建设性战略稳定关系达成共识后，美国处理对华关系时变得更为谨慎。"Over the past year or so, China, through engagement and struggle, has made the US realize that it cannot gain an advantage in a trade war with China, and may even face countermeasures from China," he said. "The US is working with China to build a new paradigm, which is very important for the two countries to find the right way to get along."他说：“过去一年多来，中国通过沟通与斗争，让美方认识到在对华贸易博弈中无法占到便宜，甚至会遭到反制。如今美方愿意同中国探索构建新型相处模式，这对两国找到正确相处之道至关重要。”In his speech, Meng, the PLA professor, also warned that global strategic stability faces unprecedented challenges, including the impact of hegemonism on regional security, rising risks of global nuclear conflict, serious erosion of international arms control, disarmament and non-proliferation systems, and disorder in global governance.孟祥青在发言中还警示，全球战略稳定正面临前所未有的挑战：霸权主义冲击地区安全，全球核冲突风险上升，国际军控、裁军与防扩散体系遭到严重破坏，全球治理陷入失序状态。Some countries are engaging in power politics, seeking absolute strategic superiority and provoking bloc confrontation, he said, adding that such moves have intensified arms races and regional conflicts.他指出，部分国家大搞强权政治，谋求绝对战略优势，煽动阵营对抗，此类行径加剧了军备竞赛与地区冲突。"These risks are intertwined and mutually reinforcing, making current strategic stability highly fragile," Meng said, noting that countries, especially major powers, should shoulder their due responsibilities in safeguarding strategic stability.孟祥青表示，各类风险交织叠加、相互激化，令当下的全球战略稳定变得十分脆弱。各国尤其是大国，应当承担起维护战略稳定的应有责任。He called for firmly defending the postwar international order, saying that it is essential to building the political foundation for strategic stability.他呼吁坚决维护战后国际秩序，这是筑牢战略稳定政治根基的关键。"As the world again stands at a crossroads, countries must stay alert to any revival of militarist thinking and firmly safeguard the outcomes of World War II and the postwar international order," Meng said, criticizing recent actions by the Japanese side in the security and military fields.孟祥青批评了日方近期在安全和军事领域的相关举动，并表示，当今世界再次来到历史十字路口，各国必须警惕军国主义思想回潮，坚定捍卫二战胜利成果和战后国际秩序。On emerging technologies, the scholar warned against a "rules vacuum" in their military use.谈及新兴技术，这位学者提醒，要警惕其在军事应用领域出现 “规则真空”。"Allowing algorithms to control matters of life and death could very likely lead to technological loss of control," he said. "At all times, control over war and related weapon systems must be firmly kept in human hands."他表示：“若任由算法掌控生杀大权，极易引发技术失控。战争以及各类武器系统的控制权，必须始终牢牢掌握在人类手中。”consensus /kənˈsensəs/n. 共识，一致意见hegemonism /hɪˈɡemənɪzəm/n. 霸权主义strategic /strəˈtiːdʒɪk/adj. 战略的；战略性的fragile /ˈfrædʒaɪl/adj. 脆弱的；易受损的

Warum scheitern viele 3D‑Druck‑Anwendungen nicht an der Maschine, sondern am Material? In dieser Interviewfolge spricht Johannes Lutz mit Polymer‑Experte Uwe Stenglin von DIPRO®Mat darüber, warum Hochleistungspolymere der entscheidende Hebel für echten industriellen 3D‑Druck sind. Es geht um minimale Schwindung statt Verzug, um Temperatur‑, UV‑ und Chemikalienbeständigkeit – und um Anwendungen, die wirklich funktionieren, von Großbauteilen bis zu gedruckten Booten. Eine Folge für alle, die weg von PLA‑Kompromissen wollen und verstehen möchten, wie Materialkompetenz Innovation im 3D‑Druck erst möglich macht.

Šiandien didžiąją laidos dalį skirsime gegužės 25 d. paskelbtai pirmajai popiežiaus Leono XIV enciklikai „Magnifica humanitas“. „Ką reiškia būti žmogumi, kai pačią mąstymo, darbo ir tikėjimo struktūrą perrašo protingos mašinos? Tai pagrindinis klausimas, kurį kelia „Magnifica humanitas“ – pirmoji popiežiaus Leono XIV enciklika. Šis dokumentas yra traktatas, bet ne apie dirbtinį intelektą, o apie žmogaus asmenį, parašytas iš technologinės revoliucijos, jau pakeitusios patirties koordinates, vidinės perspektyvos" Plačiau - vedamojo skiltyje ir pokalbyje su filosofu bei kultūros istoriku Vytautu Ališausku.Artėjant Pasauliniam gailestingumo kongresui - VU TSPMI dėstytojos ir tyrėjos Rositos Garškaitės-Antonowicz komentaras apie sociopolitines tikėjimo gailestingu Dievu pasekmes.„Ex cathedra“. Leono XIV enciklika „Magnifica humanitas“: meilės civilizacija skaitmeniniame amžiuje.Kun. Mozė Mitkevičius: „Apaštalas Paulius ir Švč. Trejybė“.Filosofo Povilo Aleksandravičiaus radijo apybraiža „Seksualumas ir teisingumas Bažnyčioje“.Redaktoriai Rūta Tumėnaitė ir Julius Sasnauskas.

Kas notiek tajās pāris sekundēs, kad pie maksājumu termināļa pieliekam telefonu vai maksājumu karti? Kā iespējams, ka maksājums notiek gandrīz acumirklī, vienlaikus iesaistot bankas, šifrēšanu un virkni drošības mehānismu? Un vai nākotnē par ierastu norēķinu līdzekli kļūs ne tikai telefoni, bet arī gredzeni, brilles vai pat mūsu biometriskie dati? Pie Digitālo brokastu galda Eleonora Bušmane-Zvaigzne, kā arī Kirils Solovjovs un Elviss Strazdiņš. Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

Mākslīgā intelekta kafijas pauzē kopā ar Anetu Gūtmani: Savienotāji jeb "connectors" mākslīgā intelekta platformās. Iespējams, Tu izmanto mākslīgā intelekta platformas, kā arī pārslēdzies starp dažādām programmām un servisiem. Rezultātā darba diena bieži ir nepārtraukta pārslēgšanās, un tieši šo problēmu risina savienotāji – tie savieno mākslīgā intelekta platformas ar citām ārējām lietotnēm.   Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

Šonedēļ #DigitālāsBrokastis ziņu topā augšgalā stāsts par "latvija.gov.lv" portālu, kas piedzīvojis lielāko modernizāciju kopš izveides. Kritis dronu ātruma rekords: 730 km/h. "Starlink Mini" interneta uztvērējs bez ārējā strāvas avota? "Meta" klusi izlaiž jaunu lietotni "Forum". Un "Starbucks" pēc deviņiem mēnešiem atsakās no MI produktu uzskaites rīka. Plašāk par tehnoloģiju jaunumiem lasi arī LSM portālā.

Stāsta dzejniece un tulkotāja Ingmāra Balode; pārraides producente – Signe Lagzdiņa Par 20. gadsimta populārāko, pasaulē vislasītāko poļu rakstnieku bez lielām šaubām var nosaukt Staņislavu Lemu – esejistu, filozofu, futurologu, zinātniskās fantastikas romānu autoru un lielā mērā – fantastikas kanona veidotāju. Lems dzimis 1921. gada 12. septembrī Ļvivā, miris 2006. gada 27. martā Krakovā. Šo enciklopēdijas šķirkļa pamata informāciju gan ironiskā kārtā tūdaļ var apstrīdēt vai papildināt: vairākos avotos minēts, ka Lems piedzima 13. septembrī, taču tīrās māņticības dēļ [1] vecāki nolēma dokumentos rakstīt labvēlīgāku datumu, savukārt brīnišķo  pilsētu, kurā dzīvoja Lemu ģimene, tolaik sauca par Ļvovu (poliski: Lwów) un tā bija Austrumpolijas metropole, trešā lielākā pilsēta Polijas Republikā starpkaru periodā. Uz Ļvovā pavadīto bērnību Lems atskatās autobiogrāfiskajā romānā “Augstā pils” (Wysoki Zamek, 1966). Tā ir arī viena no pirmajām poļu grāmatām, kurā plaši un atklāti reflektēts par šīs pilsētas nozīmi, var teikt arī – mītu poļu kultūrā, ko turpinājuši visdažādāko žanru autori un mākslinieki. Lems nenoliedzami ir viens no šī mīta veidotājiem. Ļviva, tobrīd Ļvova, iemiesoja domas brīvību laikā, kad Polija pastāvēja, sadalīta starp trim lielvarām. Ļvivas Universitāte, kā arī Matemātikas skola līdz ar citām domas un zinātnes institūcijām padarīja Lema pilsētu ne tikai par garīga patvēruma vietu spožiem prātiem, bet arī par zinātnes un laikmetīgās kultūras centru. Runādams par Vīni, Lems reiz to nosauca par “stipri palielinātu Ļvovu”; abu pilsētu līdzība ir ievērojama, un arī Vīne var ar šo to lepoties, tomēr Lema sirds piederēja viņa dzimtajai pilsētai. Lems bija vienīgais dēls laringologa Samuēla Lema un viņa sievas Sabīnes ģimenē; vecāki, līdzīgi kā daudzi tā laika Polijas ebreji, bija asimilēti un uzskatīja sevi par poļiem. Ģimene bija visai pārtikusi, tiem piederēja īres nams pilsētas centrā. Lems mācījās ģimnāzijā ar paplašinātu vācu valodas programmu un jau skolas laikā vēlējās studēt politehnikumā un kļūt par zinātnieku. “Man ir tāda vājība, ka dažus cilvēkus uzskatu par prātīgākiem nekā citi – un tie ir zinātnieki,” rakstnieks un filozofs Lems saka intervijā [2] ap nesenāko gadsimtu miju. Līdz mūža galam Lems lasīja visdažādākos zinātnes izdevumus angļu, franču, vācu, poļu, ukraiņu valodā, bet par literatūras ceļiem izteicās ironiski un nelabprāt. Pārsātinātību gan viņš uzskatīja par kaitīgu ne tikai literatūrā. “Šausmīga biežņa valda ne tikai sabiedriskajā transportā, bet arī zinātnē,” Lems secina un turpina: “arī tur katrs indivīds vēlas pastiept gaisā roku un iesaukties: mīļais Dievs, es esmu šeit! Katrs grib atklāt vai izgudrot kaut ko pilnīgi unikālu. Tādi nu cilvēki ir. Es tāds neesmu.” Lems daudzkārt uzsvēris, ka pievēršanās fantastikai bija veids, kā patverties no sociālistiskā reālisma plakanības un cenzūras spaidiem. Atgriežoties pie Lema Ļvovas (poļu literatūrzinātnē reizumis apspēlē arī pilsētas vācisko nosaukumu, LEMbergu), jāpiemin, ka ģimene dzīvoja Brajerovskas ielā – tagadējā Bohdana Lepkija ielā 4. Nams, kura trešajā stāvā uz balkona stāvēja zēns, kam patika izjaukt visas savas rotaļlietas un kura vārdā nosaukts asteroīds 3836 [3], vēl arvien, par laimi, ir savā vietā. No Lema tēva kabineta balkona pavērās skats uz augošo, grezno Ļvovas centru, bet uz otru pusi no nama sākās košuma pilnais ebreju kvartāls. Lems bija īpaši iemīļojis pilsētas parkus – plašo Strijas parku, pie tā izvērstos tirgus paviljonus, kas reprezentēja gadsimtu mijas Galīcijas saimniecisko uzplaukumu, kā arī arhitektūras, kultūras un mākslas sasniegumus. “Tur varēja just Eiropas elpu, bet mēs ar klasesbiedriem turp skrējām galvenokārt tādēļ, lai par velti iedzertu buljonu “Maggi” un sagrābtos pilns rokas reklāmlapiņu,” rakstnieks atceras [4]. Šajā parkā Lems pirmoreiz apmeklējis cirku, pieredzot, ka arī pieauguši cilvēki, tostarp viņa tēvs, visu cienītais ārsts, var brīnīties, sastapdamies ar ērmībām, un tikt pārsteigti ar cilvēka izdomu. Īpašs bija arī tagadējais Ivana Franko parks jeb Jezuītu dārzs, kurp Lemu ģimene bieži gāja kājām, nevis brauca ar drošku kā uz Strijas parku. “Un žēl, ka tā,” Lems raksta “Augstajā pilī” – “jo brauktuve Universitātes priekšā bija izlikta ar īpašu koka bruģi, un zirgu pakavi, sitot pa to, radīja burvīgu skaņu, tādu, it kā zem kājām atrastos plaša neredzama telpa.” Šo parku, kura priekšā gandrīz katru dienu esot stāvējis vīrs ar laimes ratu, Lems iedzīvinājis arī vienā no saviem agrīnajiem stāstiem “Tumsas dārzs” (Ogród ciemności, 1947). Ļvovas elementi, iespaidi, tiešas un netiešas atsauces uz to atrodamas daudzos Lema darbos; iemīļoto varoni Pirksu (Pirx) arī saista ar autora dzimto pilsētu. [5] No Ļvovas laika nāk arī Lema mīlestība uz saldumiem, īpaši halvu [6], ar ko Lems turpināja mieloties arī tad, kad veselības dēļ to vairs nedrīkstēja darīt. (Kad rakstnieks nomira, viņa dēls aiz grāmatplaukta atrada tūkstošiem saldumu papīrīšu.) Vācu okupācijas laikā Staņislavs Lems ar viltotiem dokumentiem strādāja par automehāniķi un metinātāju vācu uzņēmuma garāžās, turklāt pamanījās palīdzēt arī poļu pretošanās kustībai. Lemam un viņa vecākiem izdevās izdzīvot Holokaustā. Uz šiem gadiem rakstnieks atsaucies reti. Nevēlēdamies kļūt par PSRS pilsoņiem, viņi 1945. gadā no Ļvovas aizbrauca un apmetās uz dzīvi Krakovā. “Pirms kara es ne reizes nebiju redzējis Varšavu, nebiju bijis Krakovā. Tāpēc varu sacīt, ka Ļvova ir daļa no manis, un es – daļa no Ļvovas. Esmu ieaudzis Ļvovā kā koks,” saka Lems. Stipro saikni ar Ļvivu varu apliecināt arī es, lai arī nemēdzu lielīties ar lietām, kas nav mans nopelns. Mēs, grupiņa poļu vasaras skolas studentu, kā lielu balvu saņēmām iespēju paviesoties pie Lema viņa Krakovas savrupnamā. Bija svelmaina diena, pie visiem namiem melni karogi, jo nupat bija nomiris Česlavs Milošs. Tikām brīdināti, ka Lems slikti jūtas – drauga zaudējums un slimība rakstnieku novārdzinājusi, tāpēc drīkstējām uzdot tikai dažus jautājumus. Sasēdāmies ēnainā viesistabā, uzmanīgi turējām rokās porcelāna tasītes, un pie varena kamīna sēdēja neliela auguma vīrs, kurš tiešām izskatījās, it kā būtu no citas pasaules. Kāds sadūšojās uzdot pirmo jautājumu par rakstnieka bērnību Ļvivā. Brūnajām acīm zibot, Lems četrdesmit minūtes no vietas stāstīja par saviem parkiem, skolu, par pirmajiem izgudrojumiem. Otrais jautājums bija par romāna “Solāris” [7] (Solaris, 1961) jaunāko, Stīvena Soderberga ekranizāciju; tai autors veltīja tikai divdesmit minūtes, beigu beigās nosakot: “Ja es būtu vēlējies, lai mani varoņi bučojas saulrietā, man nevajadzētu viņus sūtīt kosmosā; to taču var darīt arī tepat uz zemes!” [1] Triskaidekafobija – slimīgas bailes no skaitļa trīspadsmit. [2] Ar Staņislavu Lemu sarunājas Gžegožs Brauns: https://www.youtube.com/watch?v=37K-P77tZXo [3] Īsumā par asteroīdu: https://english.lem.pl/gallery/59-home/facebook-post/213-asteroid-3836-lem [4] Patriks Zakševskis, LEMberg. Lema vietas Ļvovā. https://culture.pl/pl/artykul/lemberg-lwowskie-miejsca-lema [5] Turpat. [6] Lems to sākotnēji pircis Kavurasa kunga kioskā Sv. Gara ielas stūrī; dzīvodams Krakovā, rakstieks reizēm pasūtinājis, lai draugi atgādā saldumus no Ļvivas. Plašāk: https://culture.pl/pl/dzielo/stanislaw-lem-wysoki-zamek [7] “Solāris” latviešu valodā pirmoreiz izdots 1970. gadā, to tulkojis Zigfrīds Trenko. Romāna fragmenti publicēti žurnālā "Zinātne un Tehnika" jau 1962. gadā. Atkārtoti latviski izdots 2006. gadā.

Lietuvos nacionaliniame dramos teatre – paskutinė šio sezono premjera – „Geismas“.Žemaitės apsakymas „Marti“ adaptuotas lengvai suprantama kalba.Vienas po kito aiškėja skandalingi atvejai, kai šiuolaikinės literatūros kūriniai generuojami dirbtinio intelekto. Ar prasidėjo naujas kovos už literatūros žmogiškumą etapas? Plačiau apie tai – Europos rašytojų tarybos vadovas, Katalonijos rašytojas ir vertėjas Sebastià Portell ir Lietuvos rašytojas, vertėjas, literatūros kritikas Marius Burokas.Užsienio kultūros naujienų apžvalgoje apie Jungtinių Valstijų dailės komisijos pritarimą Triumfo arkos projektui, planuojamą parodą apie Agathą Christie ir kuriamą metalo legendos Ozzy'io Osbourne'o avatarą. Plačiau Ieva Radzevičiūtė.„Kurį laiką man nedavė ramybės grobuoniškumo tema, pasaulinių megastruktūrų kompleksiškumas. Deja, vyksta penktieji karo metai“, – sako parodos „(Ne)saugumo ribos“ kuratorė, menotyrininkė Skaidra Trilupaitytė.Ved. Jolanta Kryževičienė

Today I do some number swapping, some late night metal detecting, and I talk to an entire family of psycho bitches. This show is all thanks to Billio Dillio and the billions of others who support my shows on Patreon and Spotify and Tim Apple. The opening song is by BarberShopJon on Fiverr. The background music is Black Mages by Emergency Pizza Party. The end song is Wheels by Ghost Rappy. You know, I just watched a trailer for La Brea and it looks kind of cool. I doubt I'll ever watch it, but I would probably like it if I did.LEARN MORE ABOUT THE SHOW: To find out more about the music you hear on this show, visit our page of artists. For a bit of history and a studio tour, this is a good starting page. Request a prank call and find links related to shows at snowplowshow.com. If you're searching for a specific thing said in a show, shitbradsays.com is a great resource for that.SUPPORT THE PLA: For extra weekly shows, subscribe on Patreon, Spotify, or Apple. Get our t-shirts and stickers at our TeePublic store. Get coins, vinyl records, CDs, and more on our Bandcamp page. We also have thing in our Zazzle store in our Spreadshirt store. Buy important things for us at PLA's wishlist or Brad's wishlist, but most importantly, please tell your friends, family, and strangers about all about us.JOIN THE COMMUNITY: Add PLA on Prankcast for live shows, add PLA on Facebook, add Snow Plow Show on Facebook, add PLA on X, add PLA on Tik Tok, add PLA on Bluesky, add PLA on Tumblr, add PLA on YouTube, add Snow Plow Show on YouTube, PLA on Twitch, PLA on Instagram, join a Prank Call Discord, the PLA Reddit, add RBCP on Instagram, add RBCP on X, add RBCP on YouTube, and leave a voice message for the show at 814-422-5309.PLA FRIENDS AND STAFF: Nik Caesar from scary-art.com is the unofficial PLA artist.  Buy PLA designs from Nik's Spreadshirt store.  You might also like the cactus and PLA designs in Laugh Track Matt's store.  Clownsec does a lot of cool things for PLA so go honk the PLAnet.  Watch JIAD's Calls of Mass Confusion and listen to a few of the prank call shows over at World of Prank Calls. Joe DiVita and Vista Blue do lots of music for us, but so do lots of other people, so visit the SPS music page for a very incomplete list.GAME WITH PLA Join us Animal Crossing addicts by becoming our friends on Switch. If you're into GTA5, join the PLA Radio crew.

The Approach Angle Nate Schwartz (@_nateschwartz) and Kyle Bland (@blandalytics) about ERA estimators and how to apply them. The discussion starts by talking about why ERA needs to be estimated. There are so many factors that go into run prevention that make it hard to value a pitcher just on ERA. They first dive into FIP, breaking down why it makes sense to remove balls in play from the equation entirely. This starts the focus on strikeouts and walks as the main identifer across the board. The chat moves to xFIP, which adjusts for league-average home run rate. Next, the two talk about SIERA, and how it's complex compared to the others and is the most predictive. After covering the main ERA estimators, they shift to how xERA is powered by Statcast data, but that doesn't make it more predictive. Finally, they wrap up by mentioning PLA, Pitcher List's scaled stat for pitch level value (PLV) and the players that stand out across these estimators. Articles discussed: The Relative Value of FIP, xFIP, SIERA, and xERA Pt. II, Dan Richards; pFIP: Pitch Height, Launch Angle, and the FIP Framework, Alex Chamberlain; Predictive Classified Run Average, Connor Kurcon; What is PLV?, Nick Pollack Join Our Discord & Support The Show: PL+ | PL Pro - Get 15% off Yearly with code PODCASTProud member of the Pitcher List Fantasy Baseball Podcast Network Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

Vilniaus „Ryto“ čempioniška šventė sostinėje prasidėjo nuo BN LIVE. Augustas Marčiulionis, stebint komandos draugams ir keliems šimtams fanų gyvai, dalinosi ČL finalo ketverto emocijomis ir užkulisiais. Tinklalaidės partneriai: – BRITE. Pasinaudok -25% nuolaida internetinėje parduotuvėje su kodu BN: https://get.britedrinks.com/basketnews – Telia Play. Krepšinis, filmai, serialai – viskas vienoje vietoje. O dabar Samsung televizorius tik 69€ su Telia Play planu MIX. Plačiau: https://www.telia.lt/privatiems/televizija/planai?su-isipareigojimais – Nannow. Greitas ir patikimas auklių pasirinkimas – kada tik prireikia. Atsisiųskite programėlę naudodami App Store ar Google Play: https://www.nannow.app/lt/

Dissabte la Plaça Catalunya serà escenari de la 23a Festa per una societat inclusiva de les persones amb discapacitat 2026. Des de les 17h fins les 22h l'associació Societat Inclusiva Garraf ha organitzat un munt d'activitats i actuacions musicals per celebrar allò assolit en matèria d'inclusió i alhora reivindicar tot el que queda per fer fins arribar a construir una societat inclusiva real. D'inclusió, del que fem bé i malament, del que falta i sobretot de com veuen el futur n'hem parlat amb tres joves activistes per la inclusió, Matato Campos, Marta Domínguez i Iván Moreno. L'entrada Joves activistes per la inclusió ha aparegut primer a Radio Maricel.

Iedzīvotāji bieži jauc zvērinātus tiesu izpildītājus ar parāda atgūšanas pakalpojumu sniedzējiem. Kurš ir tiesīgs piedzīt parādu, vai likums ļauj bloķēt kontus un vai cilvēku var atstāt bez iztikas līdzekļiem, ja viņš ir parādnieks? Par to raidījumā Kā labāk dzīvot. Plašāk skaidro Zvērinātu tiesu izpildītāju padomes vecākā juriste Evelīna Šteinberga, Latvijas Zvērinātu tiesu izpildītāju padomes locekle Inta Kešāne, Latvijas Ārpustiesas parādu atguvēju asociācijas vadītājs Jānis Lukaševskis un Patērētāju tiesību aizsardzības centra Patērētāju tiesību uzraudzības departamenta Otrās finanšu pakalpojumu uzraudzības daļas vadītāja Solveiga Kārkliņa.

La cita serà el proper dissabte dia 23 de maig a la Plaça Catalunya i comptarà amb tot un seguit d'activitats des de quarts de cinc de la tarda fins a les deu la nit. Aquesta tarda es presenta l'acte oficialment al Miramar amb la presència de Margarida Saiz , presidenta de l'Associació Societat Inclusiva Garraf amb qui hem pogut conversar sobre el concepte de societat inclusiva per al qual treballen i lluiten i per al que s'han aconseguit millores significatives, algunes de molt recents, però per al que encara manquen la implantació de drets fonamentals com el de treballar. I per damunt de tot parlem de la importància de canviar la mirada per aconseguir una societat inclusiva de debò. L'entrada Sitges acollirà la 23a Festa per una Societat Inclusiva que mostra avenços assolits i reclama millores pendents en inclusió social ha aparegut primer a Radio Maricel.

Trump heads to Beijing this week for one of the most consequential U.S.-China meetings in years — with trade wars, Taiwan, AI, rare earths, and the fallout from the Iran conflict all hanging over the talks. Alice Han and James Kynge break down what Trump and Xi really want from the summit, why China may have more leverage than many in Washington realize, and how Beijing quietly used globalization to accelerate its technological rise. They also unpack a striking new study showing Chinese investors heavily targeted research-intensive firms across Europe and North America — raising a bigger question: did the West help build the competitor it's now trying to contain? Plus, Xi Jinping's military purge is intensifying. China has handed suspended death sentences to two former defense ministers as Xi continues reshaping the PLA ahead of a more dangerous geopolitical era. They discuss why Xi is trying to build a world-class fighting force while simultaneously hollowing out large parts of its leadership. Also: China Decode will be LIVE this Friday at 10AM ET on Prof G Plus with Kevin Xu to break down the first day of the Trump-Xi talks and what comes next. Subscribe to China Decode on Substack for weekly analysis, livestreams, and deep dives into the biggest story shaping the global economy: chinadecode.profgmedia.com Learn more about your ad choices. Visit podcastchoices.com/adchoices

Vào ngày 8/4, Quân ủy Trung ương Trung Quốc (CMC) triệu tập một khóa đào tạo "chỉnh đốn" dành cho các sĩ quan cao cấp còn lại trong Quân đội Trung Quốc (PLA). Chủ tịch Tập Cận Bình đọc diễn văn khai mạc. Trên bục danh dự bên cạnh ông tại Đại học Quốc phòng ở Bắc Kinh chỉ có duy nhất một đồng nghiệp: Trương Thăng Dân.Xem thêm.

I studion: Martin Soneby, Johan Wicklén, Tobias Öjerfalk, Adeel Faqih För 90SEK/mån får du 5 avsnitt i veckan:4 Vanliga AMK MORGON + AMK FREDAG med Isak Wahlberg Se till att bli Patron via webben och inte direkt i iPhones Patreon-app för att undvika Apples extraavgifter:Öppna istället din browser och gå till www.patreon.com/amkmorgon Önska Karakou till Gröna Lund!https://faq.gronalund.com/support/tickets/new Köp Johan Wickléns bok "Du Gröna Nya Värld"https://volanteshop.com/bok/du-grona-nya-varld/ Relevanta länkar: ...Kanal Gratis med Tejbzhttps://www.youtube.com/@kanalgratisdotse/search?query=tejbz ...tonfiskehttps://www.capecodsportsmen.com/wp-content/uploads/2022/10/IMG_0095-768x1024.jpg https://www.coastalfishing.com/cdn/shop/articles/BF2_500x.jpg?v=1686741637 ...Thermacellhttps://www.bygghemma.se/tradgard-och-utemiljo/odling-och-tradgardsskotsel/skadedjursbekampning/myggfangare/myggskydd-thermacell-portabel-mr300-c24/p-1760176-1760177?utm_source=google&utm_medium=cpc&utm_campaign=PLA%20-%20All%20Products%20-%20Medium%20Prio%20-%20Shopping&utm_id=19814304156&gad_source=1&gad_campaignid=19814304156&gbraid=0AAAAAD8-GQlsnx4UNqSTAngJWBoBL_3mq ...mygglasernhttps://www.facebook.com/watch/?v=1693845437984114 ...myggdrönarenhttps://dronelife.com/2025/07/09/china-unveils-mosquito-sized-drone-for-stealth-surveillance/ ...Hunter Seekerhttps://static0.srcdn.com/wordpress/wp-content/uploads/2021/11/Why-didnt-the-hunter-seeker-kill-paul-immediately-dune.jpg https://3dreplicas.net/cdn/shop/products/il_fullxfull.2649382844_gys0.jpg?v=1654520731&width=1946 ...Stargate Universehttps://www.imdb.com/title/tt1286039/ ...Kristerssons memehttps://www.aftonbladet.se/nyheter/a/vrk2GX/ilskan-efter-moderaternas-glassbild-motherf https://www.svt.se/nyheter/inrikes/senaste-nytt-om-val-2026?inlagg=ab9d6df11b048e5c934d39eba98fd134 ...Rålambshovsparken på Valborghttps://www.svt.se/nyheter/lokalt/stockholm/hog-berusningsgrad-i-ralambshovsparken-tva-tonaringar-gripna https://omni.se/nattvandrare-arets-valborg-var-varre-an-nagonsin/a/aJGE5E https://www.tv4.se/artikel/5NEBGvB25D2ARqU4iqGHIJ/nattvandrare-vittnar-om-valborgsnatten-fruktansvaert https://static.bonniernews.se/images/30/b1/30b19f5213604b42b5df8ea503d44611/16x9/original.jpg ...Kungsan-kravallerna 1910https://sklivedata.blob.core.windows.net/contentfiles/SSA/Biblioteket/119_1A_25_AT_100905.pdf ...JPMorgan-chefenhttps://media.licdn.com/dms/image/sync/v2/D5627AQEvcOMG75cSHA/articleshare-shrink_800/B56Z3jgWVAIYAI-/0/1777638401767?e=2147483647&v=beta&t=MflPkFhNZgpUNnTHDmPhwrnr1ls3JqnumdDq1y-CimI https://www.eviemagazine.com/post/jpmorgans-viral-harassment-lawsuit-may-have-been-fabricated https://images.lids-us.com/2026/05/i-bet-your-little-asian-fish-head-wife-doesnt-have-these-cannons-anime-shirt.webp Alla låtar finns i AMK Morgons spellista här:https://open.spotify.com/user/amk.morgon/playlist/6V9bgWnHJMh9c4iVHncF9j?si=so0WKn7sSpyufjg3olHYmg

An island nation only one-third the size of Virginia, Taiwan produces more than 90 percent of the world's most advanced chips and more than 90 percent of the servers powering the AI revolution. And last year, Taiwan became the United States' fourth-largest trading partner—after Mexico, Canada, and China.More than one-fifth of global maritime trade goes through the Taiwan Strait, according to a Center for Strategic and International Studies analysis, and any conflict over Taiwan would be devastating for the global economy—and likely far worse than the economic disruptions caused by the Iran War.Chinese leader Xi Jinping has told the People's Liberation Army (PLA) to be ready for a successful Taiwan invasion by 2027, the PLA's 100th anniversary.In this episode, I sit down with Taiwan's representative to the United States, Ambassador Alexander Yui, to understand why Taiwan matters and what's at stake as the Chinese Communist Party has ramped up its campaign to isolate, intimidate, and encircle Taiwan in recent years.Taiwanese President Lai Ching-te's recent visit to Eswatini—Taiwan's only African ally—had to be abruptly postponed when Seychelles, Madagascar, and Mauritius revoked overflight permissions—presumably due to pressure from Beijing.“They are constantly harassing our naval and air surroundings, trying to create panic and uneasiness,” Yui says.Since 2013, Beijing has built more than two dozen militarized outposts in disputed waters in the South China Sea and has recently been militarizing yet another artificial island known as Antelope Reef.Views expressed in this video are opinions of the host and the guest, and do not necessarily reflect the views of The Epoch Times.

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Have you ever thought about what is actually in your shower routine, not just your products, but the tools, materials, and where it all ends up? This episode features Phoebe Yu, founder of Floofah, a plastic-free, fully compostable shower loofah brand that is reimagining one of the most overlooked tools in our daily lives.We explore her journey from Silicon Valley and the recycling industry to launching Floofah after realizing that her own plastic shower poufs were non-recyclable, shedding microplastics, and adding to long-term landfill waste. We also break down what greenwashing actually looks like in real life, from plant-based and recyclable products where facilities exist, to products that are still mostly plastic, and why consumer trust in sustainable branding is so fragile.We also get into what composting actually means in practice, including the difference between industrial and backyard composting, why some compostable plastics like PLA are considered junk food for compost systems, and how to read labels without feeling like you need to throw everything out and start overIf trying to live more sustainably has ever left you feeling overwhelmed, this conversation will help you spot greenwashing, zoom out, and make more mindful choices that fit into real life, not a perfect oneYou will hearThe moment Phoebe realized her plastic loofah was part of the microplastics problemWhat most people misunderstand about recyclable and compostable labelsWhy oral care and kitchen tools might matter even more than what is on your bathroom shelfHow looking to nature and time-tested materials can upgrade your routine without a full lifestyle overhaulMentionedFloofah: www.floofah.comFloofah on Instagram and TikTok @tryfloofah

Plači, Pinki! Jesi video Tita, ali to se bilo tako namestilo, Maršal ti ni-kad nije telefonirao! Plači i ti, Jeremija iz Biblije, jer je plač tvoj ustuknuo pred plakanijem Mirkovim: dedica su se rasplakali kad se u njihovom skromnom penzionerskom domu ukazao Vučević, gorostas nauka pravnih i prepravnih! The post RETROVIZOR Neizreciva milost appeared first on Istinomer Podcast.

Plači, Pinki! Jesi video Tita, ali to se bilo tako namestilo, Maršal ti ni-kad nije telefonirao! Plači i ti, Jeremija iz Biblije, jer je plač tvoj ustuknuo pred plakanijem Mirkovim: dedica su se rasplakali kad se u njihovom skromnom penzionerskom domu ukazao Vučević, gorostas nauka pravnih i prepravnih! The post RETROVIZOR Neizreciva milost appeared first on Istinomer Podcast.

In this week's episode of China Insider, Miles Yu discusses Xi Jinping's campaign for the sinicization of religion by the CCP and the increased repression of religious freedom in China, ten years later. Next, Miles reviews the latest updates from the U.S. naval blockade of the Strait of Hormuz and how this maritime operation impacts PLA strategic calculus and potential future engagements involving Taiwan. Finally, Miles covers recent comments from Chinese real estate tycoon, Pan Shiyi, calling China's real estate market a Ponzi scheme following Evergrande's founder, Xu Jiayin's, entered guilty plea in the ongoing legal proceedings involving allegations of fraud and embezzlement. China Insider is a weekly podcast project from Hudson Institute's China Center, hosted by China Center Director and Senior Fellow, Dr. Miles Yu, who provides weekly news that mainstream American outlets often miss, as well as in-depth commentary and analysis on the China challenge and the free world's future. 

Fluent Fiction - Catalan: Sant Jordi's Miracle: From Anxious Heart to Hopeful Spirit Find the full episode transcript, vocabulary words, and more:fluentfiction.com/ca/episode/2026-04-19-07-38-19-ca Story Transcript:Ca: La primavera bufa suaument, escampant l'aroma de la rosa fresca a través de la Plaça de Catalunya.En: Spring blows gently, spreading the aroma of fresh rosa through Plaça de Catalunya.Ca: Avui és el dia de Sant Jordi, una festa plena de llibres i flors.En: Today is the day of Sant Jordi, a festival full of books and flowers.Ca: Júlia camina, sentint cada pas com un eco en el seu pensament.En: Júlia walks, feeling each step like an echo in her mind.Ca: Té ganes de gaudir del dia, però el pensament de les proves mèdiques no la deixa.En: She wants to enjoy the day, but the thought of her medical tests won't leave her.Ca: Pau, el seu millor amic, l'espera amb un gran somriure i una rosa vermella a la mà.En: Pau, her best friend, waits for her with a big smile and a red rosa in his hand.Ca: "Hola, Júlia!En: "Hello, Júlia!Ca: Avui ho passarem bé.En: We're going to have a great time today.Ca: Mira tot això!En: Look at all this!"Ca: ", crida, assenyalant els llocs plens de llibres i roses.En: he shouts, pointing to the places filled with books and roses.Ca: Pau sap com la Júlia pateix per les seves proves, per això vol distreure-la.En: Pau knows how Júlia suffers over her tests, so he wants to distract her.Ca: És una amiga valuosa en moments com aquests.En: She is a valuable friend in times like these.Ca: "Avui, oblidaré les meves preocupacions", decideix Júlia.En: "Today, I'll forget my worries," Júlia decides.Ca: Junts recorren la plaça, comprant llibres i parlant de les històries que els han captivat.En: Together they walk through the square, buying books and talking about the stories that have captivated them.Ca: La multitud és alegre, les veus són com un rumb constant de rialles i converses.En: The crowd is joyful, the voices are like a constant hum of laughter and conversations.Ca: De sobte, mentre Júlia està mirant un llibre, el telèfon sona.En: Suddenly, while Júlia is looking at a book, the phone rings.Ca: El cor li fa un salt.En: Her heart skips a beat.Ca: És una trucada de la clínica.En: It's a call from the clinic.Ca: Respira profundament i mira a Pau, qui li somriu i li estreny la mà.En: She takes a deep breath and looks at Pau, who smiles at her and squeezes her hand.Ca: "Habla", li diu amb mirades encoratjadores.En: "Answer," he tells her with encouraging looks.Ca: Júlia contesta.En: Júlia answers.Ca: La seva veu és tremolosa, però ferma.En: Her voice is trembling, but firm.Ca: "Hola, sí?En: "Hello, yes?"Ca: ", comença.En: she begins.Ca: La veu a l'altre costat li explica els resultats.En: The voice on the other side explains the results to her.Ca: Els minuts s'estiren com hores.En: The minutes stretch like hours.Ca: Però finalment, Júlia gira el cap cap a Pau i comença a somriure.En: But finally, Júlia turns her head towards Pau and starts to smile.Ca: Els resultats no són tan dolents com temia.En: The results aren't as bad as she feared.Ca: El seu cor es relaxa, i ara pot respirar alleujada.En: Her heart relaxes, and she can now breathe a sigh of relief.Ca: "És moment de celebrar!En: "It's time to celebrate!"Ca: ", exclama, sentint com si un pes enorme li hagués caigut.En: she exclaims, feeling as if a huge weight has been lifted.Ca: L'alegria de Sant Jordi, l'olor de roses i llibres la cobreixen.En: The joy of Sant Jordi, the smell of roses and books envelops her.Ca: Pau li entrega la rosa.En: Pau hands her the rosa.Ca: "Aquesta és per escritora saludable i feliç que seràs", li diu.En: "This is for the healthy and happy writer you'll be," he says.Ca: Júlia sent una nova força dins seu.En: Júlia feels a new strength within her.Ca: És el moment d'apreciar cada instant, d'escriure les històries que sempre ha volgut.En: It's time to appreciate every moment, to write the stories she's always wanted to.Ca: Avui, a la Plaça de Catalunya, Júlia decideix viure plenament i deixar que el futur sigui escrit amb el seu nou entusiasme.En: Today, at Plaça de Catalunya, Júlia decides to live fully and let the future be written with her new enthusiasm.Ca: La festa de Sant Jordi ha fet el seu miracle: el cor de Júlia ara està lliure i ple d'esperança.En: The festival of Sant Jordi has worked its miracle: Júlia's heart is now free and full of hope. Vocabulary Words:spring: la primaverablows: bufagently: suaumentspreading: escampantaroma: l'aromastep: el pasecho: l'ecomind: el pensamenttests: les provessuffer: pateixdistract: distrèurecrowd: la multitudjoyful: alegrehum: el rumblaughter: les riallesconversations: les conversessuddenly: de sobterings: sonadeep breath: respira profundamentsqueeze: estrenytrembling: tremolosafirm: fermastretched: s'estirenhead: el caprelaxes: es relaxasigh of relief: alleujadaweight: el pesenvelops: cobreixenstrength: la forçaappreciate: apreciar

Hello, hello!In this episode of the Good Garbage Podcast, host Ved Krishna sits down with Sandeep Kulkarni, Founder of Kool Earth Solutions and a global expert in sustainable packaging.With experience spanning PepsiCo's Advanced Research team, packaging innovation, and circular economy systems, Sandeep brings a rare insider perspective into how materials, business realities, and sustainability intersect.

Preview for Later TodayJack Burnham discusses the security risks of Chinese tech companies, specifically Hikvision. He highlights its role as a top-tier PLA supplier and its use in surveillance for the mass detention of Uyghurs.1954

                                                           Tom Waits is celebrated.Here's the Pla...