POPULARITY
8 episodes with hyperplasia
5 episodes with hyperplasia
2 episodes with hyperplasia
2 episodes with hyperplasia
2 episodes with hyperplasia
2 episodes with hyperplasia
2 episodes with hyperplasia
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The FDA issues alert regarding a compounded topical finasteride; plant-based alkaloid shows improvement for smoking cessations; potential new treatment option for GERD; and the CDC's Advisory Committee on Immunization Practices make new recommendations.
BUFFALO, NY - April 15, 2025 – A new #research paper was #published in Oncotarget, Volume 16, on April 4, 2025, titled “Association between two single nucleotide polymorphisms of the Prostaglandin-Endoperoxide Synthase 1 and 2 genes and cell proliferative prostatic diseases in Lebanon." The team of researchers led by first author Brock J. Sheehan and corresponding author Ruhul H. Kuddus, from Utah Valley University, discovered that a specific genetic variation in the PTGS2 gene is associated with a higher risk of benign prostate hyperplasia (BPH), a common condition in aging men. The study, which focused on Lebanese men, suggests that the C allele of the -765 G>C polymorphism in the PTGS2 gene may increase risk to this non-cancerous but problematic prostate condition. This finding could help identify men at greater risk earlier and lead to better treatment choices. Benign prostate hyperplasia and prostate cancer are two common conditions that involve abnormal cell growth in the prostate gland. While prostate cancer is malignant and potentially life-threatening, BPH is a non-cancerous enlargement that can still significantly affect quality of life. Both conditions are widespread in older men, with BPH affecting over 70% of men above 60. Researchers have long suspected that inflammation-related genes may play a role in their development. In this study, the focus was to study PTGS1 and PTGS2, genes that help produce enzymes involved in inflammation. Using DNA samples from 168 Lebanese men, including 61 with prostate cancer, 51 with BPH, and 56 healthy controls, the researchers analyzed two common gene variants. They found no link between the PTGS1 variant and either condition. However, the PTGS2 variant showed a strong association with BPH. Men carrying the C version of this gene were more than twice as likely to have BPH compared to those without it. While a similar trend was observed in men with prostate cancer, the results were less conclusive. "The C allele of SNP-765G>C of the PTGS2 gene was significantly associated with an increased risk of BPH (OR = 2.30, p-value = 0.01)." This is the first study to report a genetic link between the C allele of the -765 G>C polymorphism in the PTGS2 gene and BPH in Lebanese men. It builds on earlier findings that associated this gene variant with various cancers, including prostate, colon, and stomach cancers. Although based on a relatively small and specific population, the study offers new insight that could help improve genetic screening and guide prevention strategies. The research also points to the potential benefits of COX-2 inhibitors—drugs already used to treat prostate conditions—which may be more effective for men with certain PTGS2 gene types. Further studies in larger and more diverse groups are needed to confirm these results and explore how this gene variant influences prostate disease. In the future, simple genetic tests could help identify men at higher risk before symptoms appear, allowing for earlier and more personalized care. Continue reading: DOI: https://doi.org/10.18632/oncotarget.28710 Correspondence to: Ruhul H. Kuddus — ruhul.kuddus@uvu.edu Subscribe for free publication alerts from Oncotarget - https://www.oncotarget.com/subscribe/ To learn more about Oncotarget, please visit https://www.oncotarget.com and connect with us: Facebook - https://www.facebook.com/Oncotarget/ X - https://twitter.com/oncotarget Instagram - https://www.instagram.com/oncotargetjrnl/ YouTube - https://www.youtube.com/@OncotargetJournal LinkedIn - https://www.linkedin.com/company/oncotarget Pinterest - https://www.pinterest.com/oncotarget/ Reddit - https://www.reddit.com/user/Oncotarget/ Spotify - https://open.spotify.com/show/0gRwT6BqYWJzxzmjPJwtVh MEDIA@IMPACTJOURNALS.COM
Ever feel like your workouts just aren't delivering the same results anymore? You're training hard, lifting heavy, and doing everything “right,” but somehow, progress stalls. Strength fades. Recovery slows. And your joints? Yeah, they're not loving it either.Here's the truth: it's not your age—it's your approach.That's why I brought in Nick Nilsson, aka The Mad Scientist of Muscle. This guy has spent decades reinventing the way we build muscle, train smart, and stay strong—especially as we get older. His Return to Prime system isn't about grinding yourself into the ground. It's about training smarter, not harder, by optimizing the four key systems that fuel muscle growth.I put his method to the test, and let me tell you—it humbled me in the best way possible. This workout forced my body to adapt in ways I didn't even think were possible.Timestamps:09:29 Nick's origin story and fitness background21:00 How Nick created his "best exercises you've never done" brand28:48 The inspiration behind the Return to Prime program33:36 Phase 1: Angiogenesis training for rebuilding circulation46:08 Phase 2: Connective tissue training and hydraulic circulation1:00:14 Phase 3: Hyperplasia training for increasing muscle fiber numbers1:10:30 Phase 4: Nervous system efficiency and activation training1:24:42 Nutrition recommendations for each training phase1:32:10 Final thoughts: Have fun whith training!Mentioned Resources:Mad Scientist of Muscle website: https://www.madscientistofmuscle.com/ The Best Exercises You've Never Heard Of: https://www.fitstep.com/2/the-best-exercises-index/index.htm Return to Prime: https://www.fitstep.com/2/return-to-prime/index-story.htm
Discover insights into the challenging landscape of Cushing syndrome, where adrenal variations require careful diagnostic and therapeutic strategies. In this podcast, Sina Jasim, MD, MPH, Editor-in-Chief of the AACE Clinical Reports (ACCR) and Associate Professor of Medicine at Washington University School of Medicine, leads a compelling discussion on two unique cases featured in the ACCR article, Histopathologic Differences Between Adrenocorticotropic Hormone–Dependent and Adrenocorticotropic Hormone–Independent Adrenal Hyperplasia Causing Cushing Syndrome. Authors, Amir H Hamrahian, MD, Endocrinologist, Associate Professor, and Medical Director of the Comprehensive Adrenal Center at Johns Hopkins University School of Medicine, and Ezra Baraban, MD, Assistant Professor of Pathology at Johns Hopkins University School of Medicine, explain the diagnostic challenges for each case, the decision-making process between medical and surgical approaches, and the critical role of pathology in guiding clinical care. Read the full case report in ACCR at https://www.aaceclinicalcasereports.com/article/S2376-0605(22)00066-9/fulltext.
In this episode of the IJGC podcast, Editor-in-Chief Dr. Pedro Ramirez is joined by Andrea Rosati. Mr. Rosati is a consultant at the Department of Gynecologic Oncology at Fondazione Policlinico Universitario Agostino Gemelli in Rome (Italy). He is currently attending a second level master "Gynecologic Oncology International Master" at the Catholic University of the Sacred Heart (Rome, Italy) accredited as a Subspecialty Fellowship by the European Society of Gynaecological Oncology. His main interest areas are gynecological cancer, surgical anatomy, and gynecologic oncology surgery. Highlights: This study evaluated the prevalence of concurrent endometrial cancer in patients with pre-operative diagnoses of atypical endometrial hyperplasia undergoing hysterectomy. Among 460 patients, 47.2% were found to have concurrent endometrial cancer. Sentinel lymph node biopsy provided prognostic and therapeutic information in 60.8% of cases. It also allowed for the adjustment of adjuvant therapy in 12.3% of high to intermediate-risk patients without increasing operative time or complication rates. The study suggests sentinel lymph node biopsy can provide valuable prognostic and therapeutic insights in managing atypical endometrial hyperplasia.
Summary In this episode of the Vet Dental Show, Dr. Brett Beckman, Board Certified Veterinary Dentist, answers listener questions about managing gingival hyperplasia in boxers and other brachycephalic breeds. Dr. Beckman discusses the importance of radiographs before treatment, when to refer complex cases, and the nuances of dealing with epulides. Tune in for expert advice and practical tips to enhance your veterinary dental practice. Guest, Cast, and Crew Information Host: Dr. Brett Beckman, Board Certified Veterinary Dentist Sponsor: Veterinary Dental Practitioner Program Main Talking Points Introduction: Overview of the episode and sponsorship details. Listener Question: Mandy's question on treating gingival hyperplasia in boxers. Radiographs Importance: The necessity of taking radiographs before treatment. Treatment Approach: Steps to handle gingival hyperplasia and epulides. When to Refer: Guidance on referring complex brachycephalic cases. Histopathology: The importance of submitting tissue for histopathology. Maintenance and Follow-Up: Managing recurrent gingival hyperplasia. Interesting Quotes "You do not want to go in and start removing tissue without first taking radiographs." "Brachycephalic breeds often have dense cortical bone, making extractions more challenging." "Gingival hyperplasia will come back and requires maintenance every 6 to 18 months." Timestamps 00:00 - 00:30: Introduction 00:31 - 02:00: Listener Question from Mandy 02:01 - 04:00: Importance of Radiographs 04:01 - 06:00: Treatment Approach for Gingival Hyperplasia 06:01 - 08:00: When to Refer Complex Cases 08:01 - 10:00: Histopathology and Tissue Submission 10:01 - 11:30: Maintenance and Follow-Up 11:31 - 13:00: Summary and Conclusion [Veterinary dentistry, gingival hyperplasia, brachycephalic breeds, radiographs, dental extractions, epulides, histopathology, veterinary dental training, Dr. Brett Beckman] Key Points Summary Radiographs Importance: Always take full mouth radiographs before treating gingival hyperplasia. Treatment Approach: Remove affected teeth and contour tissue for closure. Referral Guidance: Refer complex brachycephalic cases to specialists. Histopathology: Submit all excised tissue for pathology to ensure an accurate diagnosis. Maintenance: Regular follow-up and maintenance are necessary for managing recurrent gingival hyperplasia. Affiliate & Sponsor Links IVDI.org/inv - Submit your request for an invitation to the Veterinary Dental Practitioner Program.
I'm honored to share Episode 2 of the first season of Perform with Dr. Andy Galpin. Dr. Andy Galpin is a tenured full professor at California State University, Fullerton, where he co-directs the Center for Sport Performance and leads the Biochemistry and Molecular Exercise Physiology Laboratory. Andy is both a friend and a colleague, and I'm delighted to have assisted in the creation of this podcast. I'm certain you'll both enjoy and learn from it. Season 1 features 10 episodes, airing every Wednesday for 10 weeks. Dr. Galpin will cover everything from building strength, the importance of strength for long-term health, the science of breathing, the benefits of sleep extension, genetic testing for personalized training, and nutrition for injury recovery. While we have Episode 2 of Perform with Dr. Andy Galpin here, please be sure to subscribe and follow Perform with Dr. Andy Galpin on your preferred platform. Show notes for this episode can be found at performpodcast.com. Timestamps 00:00:00 Introduction from Dr. Andrew Huberman 00:01:06 Skeletal Muscle 00:04:06 Sponsors: Absolute Rest & Momentous 00:07:20 Quantity & Quality; Organ System; Health & Performance 00:12:58 Plasticity, “Look Good, Feel Good, Play Good”; Muscle Types 00:15:46 What is Muscle?, Muscle Fibers, Tendon 00:21:37 Muscle Fiber Number, Hyperplasia, Anabolic Steroids, Age 00:24:03 Myonuclei & Adaptability 00:26:27 Muscle Fiber Types, Variable Muscle Functions 00:32:24 Fiber Type & Lifestyle Factors 00:34:54 Sponsors: David Protein & AG1 00:37:37 Age & Muscle Loss, Slow vs. Fast-Twitch Fibers; Motor Units 00:46:36 Muscle Size vs. Muscle Strength, Quantity vs. Quality 00:50:56 Investigate: Muscle Quantity, Fat-Free Mass Index (FFMI) 00:56:21 FFMI, Elite Athletes, Muscle Mass 01:00:59 Muscle Asymmetry; Too Much Muscle Possible? 01:03:49 Interpret: Muscle Mass, FFMI Calculations & Percentiles 01:09:28 Tool: Intervene - Increase Muscle Mass, 72-Hour Rule 01:15:27 Sponsors: Maui Nui & Renaissance Periodization 01:17:51 Investigate: Muscle Quality & 4 Movement Principles 01:23:34 Muscle Quality & 3 Performance Principles 01:26:42 Interpret: Muscle Speed, Age 01:32:45 Muscle Power, Vertical Jump, Broad Jump 01:36:17 Muscle Strength, Powerlifting Elite, Bench Press, Leg Press, Grip Strength 01:44:05 Increasing Strength, Improve Health & Longevity 01:46:44 Tool: Intervene - Improve Muscle Quality, 4 Training Principles, 3-to-5 Rule 01:53:56 Zero-Cost Support, YouTube, Spotify & Apple Subscribe & Reviews, Sponsors, YouTube Feedback, Social Media 01:56:10 Conclusion from Dr. Andrew Huberman Disclaimer
Are you afraid of the dark? You might be after this spooky story! Sara shares a recent “encounter” Zoe and her had with a shadowy figure in their home. SOS! In less frightening news, we have our favorite board-certified dermatologist and co-founder of Facile, Dr. Nancy Samolitis, back on the podcast to answer some mailbag questions we couldn't about sebacious hyperplasia, melasma, and her thoughts on the bill to ban children from using anti-aging skincare. Kirbie also chats with Dr. Sam about her recent CoolPeel and PRP treatment. Shop this episodeGlossAngelesPod.comhttps://glossangeles.komi.ioCALL US: 424-341-0426Shop products from our episodesJoin our FB Group: https://www.facebook.com/groups/glossangelespodcastInstagram: @glossangelspod, @kirbiejohnson, @saratanTwitter: @glossangelespod, @kirbiejohnson, @saratanEmail: glossangelespodcast@gmail.com Hosted on Acast. See acast.com/privacy for more information.
In this episode of the IJGC podcast, Editor-in-Chief Dr. Pedro Ramirez is joined by Andrea Rosati. Mr. Rosati is a consultant at the Department of Gynecologic Oncology at Fondazione Policlinico Universitario Agostino Gemelli in Rome (Italy). He is currently attending a second level master "Gynecologic Oncology International Master" at the Catholic University of the Sacred Heart (Rome, Italy) accredited as a Subspecialty Fellowship by the European Society of Gynaecological Oncology. His main interest areas are gynecological cancer, surgical anatomy, and gynecologic oncology surgery. Highlights: This study evaluated the prevalence of concurrent endometrial cancer in patients with pre-operative diagnoses of atypical endometrial hyperplasia undergoing hysterectomy. Among 460 patients, 47.2% were found to have concurrent endometrial cancer. According to ESGO-ESTRO-ESP classification, 71.4%, were low-risk, 9.7% intermediate, 11.1% high to intermediate and 7.8% high-risk tumors. Positive Lymph node were found in 12 patients, accounting for the 7.6% of concurrent endometrial cancers who underwent SLN biopsy. SLN biopsy allowed for the adjustment of adjuvant therapy in 12.3% of high to intermediate-risk patients without increasing operative time or complication rates.
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In this installment of the Best of Muscle For Life, you'll hear hand-picked clips from three popular MFL episodes: an interview with Eric Helms on the problems with staying too lean, a monologue on whether you should train for hypertrophy or hyperplasia, and book club episode on Titan by Ron Chernow. Some people—my favorite people—listen to most or even all of my podcasts, but my wizbang analytics tell me that while many listeners tune in on a regular basis, they don't catch every installment of Muscle for Life and thus miss out on insights that could help them do at least a little better inside and outside the gym. That's why I do “best of” episodes that contain a few of the most practical and compelling ideas, tips, and moments from the more popular episodes I've published over the years. This way, you can learn interesting insights that you might have otherwise missed and find new episodes of the show to listen to. So, in this installment of The Best of Muscle for Life, you'll be hearing hand-picked morsels from three episodes: Eric Helms on Trying To Stay Too Lean (What the Science Says) (Originally published 8/18/2021) Should You Train For Hypertrophy or Hyperplasia? (Originally published 10/4/2021) Book Club: Titan by Ron Chernow (Originally published 8/4/2017) And we'll be starting with number one, Eric Helms on Trying To Stay Too Lean (What the Science Says). Timestamps: 0:00 - Please leave a review of the show wherever you listen to podcasts and make sure to subscribe! Eric Helms on Trying To Stay Too Lean (What the Science Says) Should You Train For Hypertrophy or Hyperplasia? Book Club: Titan by Ron Chernow TIME - Ad Mentioned on the Show: Find the Perfect Strength Training Program for You in Just 60 Seconds: http://www.muscleforlife.show/trainingquiz
Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2023.06.22.546113v1?rss=1 Authors: Vieira, P., Kantor, M. R., Jansen, A., Handoo, Z., Eisenback, J. D. Abstract: The beech leaf disease nematode, Litylenchus crenatae subsp. mccannii, is recognized as a newly emergent nematode species that causes beech leaf disease (BLD) in beech trees (Fagus spp.) in North America. Changes of leaf morphology induced by BLD can provoke dramatic effects into the leaf architecture and consequently to tree performance and development. The initial symptoms of BLD appear as dark green interveinal banding patterns of the leaf. Despite the fast progression of this disease, the cellular mechanisms leading to the formation of such type of aberrant leaf phenotype remains totally unknown. To understand the cellular basis of BLD, we employed several microscopy approaches to provide an exhaustive characterization of nematode-infected buds and leaves. Histological sections revealed a dramatic cell change composition of these nematode-infected tissues. Diseased bud scale cells were typically hypertrophied and showed a high variability of size. Moreover, while altered cell division had no influence on leaf organogenesis, induction of cell proliferation on young leaf primordia led to a dramatic change in cell layer architecture. Hyperplasia and hypertrophy of the different leaf cell layers, coupled with an abnormal proliferation of chloroplasts specially in the spongy mesophyll cells, resulted in the typical interveinal leaf banding. These discrepancies in leaf cell structure were depicted by an abnormal rate of cellular division of the leaf interveinal areas infected by the nematode, promoting significant increase of cell size and leaf thickness. The formation of symptomatic BLD leaves is therefore orchestrated by distinct cellular processes, to enhance the value of these feeding sites and to improve their nutrition status to the nematode. These results revealed a high specialized mode of parasitism of L. crenatae subsp. mccannii. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC
On Episode 86 of Limb Lengthening LIVE - We were going to do a LLD Roundtable for patients but no one confirmed. So I just did a LL LIVE Q&A __________________ Timestamps: 0:00 - intro 1:10 - Nail malfunction 5:45 - Age limit for LL 9:00 - good surgeon for a good price 12:20 - talking normally with WB nail 17:20 - my crooked nose septum 19:00 - preparing for LL as a 20 year old 23:00 - does bone density and flexibility matter 25:30 - doing over 8 cm LON 27:50 - Hyperplasia in muscles after LL 30:00 - weight-bearing nail 34:00 - nuVasive precise max 34:50 - soft tissue shoulder injury 36:00 - preop procedure 38:00 - The C4LLECTIVE overview 43:25 - can you use crutches with WB nail 46:00 - 6 cm on Femurs and walking in 6 mo 49:00 - obesity and chances of embolism 56:40 - Quadrilateral lengthening 58:30 - PSA for leg length discrepancy 1:02:00 - Outro __________________
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Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2023.04.11.536468v1?rss=1 Authors: Giri, S., Suo, C., Pardi, R., Fishbein, G. A., Rezvani, K., Chen, Y., Wang, X. Abstract: BACKGROUND: Neointimal hyperplasia (NH) is a common pathological response to vascular injury and mediated primarily by vascular smooth muscle cell (VSMC) migration and proliferation. The COP9 signalosome (CSN) is formed by 8 canonical subunits (CSN1 through CSN8) with its deneddylation activity residing in CSN5. Each or some of CSN subunits may have deneddylation-independent function. Despite strong evidence linking the CSN to cell cycle regulation in cancer cells, the role of the CSN in vascular biology remains obscure. METHODS: Neointimal CSN5 expression in the lung tissue of pulmonary hypertension (PAH) patients was assessed with immunohistochemistry. Adult mice with smooth muscle cell-restricted CSN5 knockout (CSN5-SMKO) or CSN8 hypomorphism (CSN8-hypo) and cultured mouse VSMCs were studied to determine the role and governing mechanisms of the CSN in NH. NH was induced by ligation of the left common carotid artery (LCCA) and PDGF-BB stimulation was used to mimic the vascular injury in cell cultures. RESULTS: Remarkably higher CSN5 levels were detected in the neointimal VSMCs of the pulmonary arteries of human PAH. LCCA ligation induced NH and significantly increased the mRNA and protein levels of CSN subunits in the LCCA wall of adult wild type mice. CSN5-SMKO impaired Cullin deneddylation and the nuclear export of p27 in vessel walls and markedly inhibited VSMC proliferation in mice. On the contrary, CSN8-hypo significantly exacerbated NH and VSMC proliferation in vivo and in cellulo. Cytoplasmic CSN5 mini-complexes and the nuclear export of p27 were significantly increased in CSN8-hypo mouse vessels and cultured CSN8-hypo VSMCs. Nuclear export inhibition with leptomycin attenuated the PDGF-BB-induced increases in VSMC proliferation in both CSN8-hypo and control VSMCs. Further, genetically disabling CSN5 nuclear export but not disabling CSN5 deneddylase activity suppressed the hyperproliferation and restored p27 nuclear localization in CSN8 hypomorphic VSMCs. Interestingly, CSN deneddylase inhibition by CSN5i-3 did not alter the hyperproliferation of cultured CSN8-hypo VSMCs but suppressed wild type VSMC proliferation in cellulo and in vivo and blocked neointimal formation in wild type mice. CONCLUSION: The CSN promotes VSMC proliferation and NH in injured vessels through deneddylation activity and CSN5-mediated nuclear export. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC
In this episode, we review the high-yield topic of Endometrial Hyperplasia from the Reproductive section. Follow Medbullets on social media: Facebook: www.facebook.com/medbullets Instagram: www.instagram.com/medbulletsofficial Twitter: www.twitter.com/medbullets --- Send in a voice message: https://anchor.fm/medbulletsstep1/message
Hosted by: Gregg Nelson, MD, PhD, Social Media Editor of Gynecologic Oncology Featuring: Jennifer Mueller MD, Memorial Sloan Kettering Cancer Center Emad Matanes MD, Rambam Medical Center Emma Rossi MD, Duke University Medical Center Editor's Choice Papers: Sentinel lymph node mapping in patients with endometrial hyperplasia: A practice to preserve or abandon? Is sentinel lymph node assessment useful in patients with a preoperative diagnosis of endometrial intraepithelial neoplasia? Editorial: Routine SLN biopsy for endometrial intraepithelial neoplasia: A pragmatic approach or over-treatment?
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Editor's Choice Papers: Sentinel lymph node mapping in patients with endometrial hyperplasia: A practice to preserve or abandon? Is sentinel lymph node assessment useful in patients with a preoperative diagnosis of endometrial intraepithelial neoplasia? Editorial: Routine SLN biopsy for endometrial intraepithelial neoplasia: A pragmatic approach or over-treatment? Hosted by:Gregg Nelson, MD, PhD, Social Media Editor of Gynecologic Oncology Featuring: Jennifer Mueller MD, Memorial Sloan Kettering Cancer CenterEmad Matanes MD, Rambam Medical CenterEmma Rossi MD, Duke University Medical Center
abdominal cavity · abdomin/o = abdomen · -al = pertaining to o The cavity beneath the thoracic cavity that is separated from the thoracic cavity by the diaphragm; contains the liver, gallbladder, spleen, stomach, pancreas, intestines, and kidneys. abdominopelvic cavity · abdomin/o = abdomen · pelv/i = pelvis · -ic = pertaining to o A term that describes the abdominal and pelvic cavity collectively; refers to the space between the diaphragm and the groin. anaplasia · (an-ah-PLAY-zee-ah) · ana- = not, without · -plasia = formation, growth o A change in the structure and orientation of cells, characterized by a loss of differentiation and reversion to a more primitive form. Hyperplasia · hyper- = high · -plasia = formation, growth anterior · Pertaining to the front of the body or toward the belly of the body. aplasia · (ah-PLAY-zee-ah) · a = without, not · -plasia formation, growth o A developmental failure resulting in the absence of any organ or tissue. cardiac muscle · cardi/o = heart · -ac = pertaining to o The muscle that makes up the muscular wall of the heart. cervical vertebrae · (SER-vic-al VER-teh-bray) · cervic/o = neck · -al = pertaining to o The first seven segments of the spinal column; identified as C1 through C7. chromosomes · (KROH-moh-sohm) · The threadlike structures within the nucleus that control the functions of growth, repair, and reproduction for the body. coccyx · (COCK-siks) · The tailbone. Located at the end of the vertebral column, the coccyx results from the fusion of four individual coccygeal bones in the child. connective tissue · Tissue that supports and binds other body tissue and parts. cranial · (KRAY-nee-al) · crani/o skull · -al pertaining to · Pertaining to the skull or cranium. cytology · (sigh-TALL-oh-jee) · cyt/o cell · -logy the study of · The study of cells. cytoplasm · (SIGH-toh-plazm) · cyt/o cell · -plasm living substance · A gel-like substance that surrounds the nucleus of a cell. The cytoplasm · contains cell organs, called organelles, which carry out the essential · functions of the cell. --- Support this podcast: https://anchor.fm/liam-connerly/support
Link to bioRxiv paper: http://biorxiv.org/cgi/content/short/2022.12.08.519564v1?rss=1 Authors: Royan, M. R., Kayo, D., Weltzien, F.-A., Fontaine, R. Abstract: The two pituitary gonadotropins, Fsh and Lh, regulate the reproductive function in vertebrates. While many studies have investigated the regulation of gonadotropin production and release by the sex steroid feedback, its role on the regulation of gonadotrope cell number remains unclear. Using medaka as a model and an optimized protocol to restore physiological sex steroids levels following gonadectomy, we show that gonadal sex steroids not only decrease fshb transcript levels, but also Fsh cell number in both sexes. We then investigated the origin of the Fsh cell hyperplasia induced by gonadectomy. In both sexes, BrdU incubation shows that this is achieved via Fsh cell mitosis. In situ hybridization reveals that new Fsh cells also originate from transdifferentiating Tsh cells in females, but not in males. Both phenomena are inhibited by sex steroid supplementation via feeding. In males (but not females), gonadectomy (without recovery with sex steroid supplementation) also reduces sox2 transcript levels and Sox2-immunopositive population volume, suggesting that sox2-progenitors may be recruited to produce new Fsh cells. Opposite to Fsh cells, gonadectomy decreases lhb levels in both sexes, and levels are not restored by sex steroid supplementation. In addition, the regulation of Lh cell number also seems to be sex dependent. Removal of gonadal sex steroids stimulates Lh cell mitosis in male (like Fsh cells), but not in females. To conclude, our study provides the first evidence on sexually dimorphic mechanisms used in the fish pituitary to remodel gonadotrope populations in response to sex steroids. Copy rights belong to original authors. Visit the link for more info Podcast created by Paper Player, LLC
Thank you for joining us for our 2nd Cabral HouseCall of the weekend! I'm looking forward to sharing with you some of our community's questions that have come in over the past few weeks… Jade: Hi Dr. Cabral. Thank you for all that you do for all of us. You're a true blessing! I have Gilbert's Syndrome and fibromyalgia. I have horrible stomache issues and all over body pain. I use CBD oil for sleeping and for the body pain. Am I putting more strain on my liver with the CBD oil? Am I causing more damage? I also drink fermented drinks and eat fermented foods. Is this good for Gilbert's? My bilirubin has been 1.8 for the last 2 years.I've gone though a lot of stress and emotional grief. I'm thinking this caused my bilirubin to be higher then before which was around 1.3. Thank you for your time. Blessings! Tiffany: Hello Dr. Cabral I was diagnosed with vertical heterophoria a couple of years ago. Even with corrective lenses it still affects me when I drive on the freeway and the dizziness makes me feel very anxious. It has only gotten worse over the years. What can I do if anything to find the root cause of this or at least relieve my symptoms. It has gotten so bad that sometimes I have to just pull over for a few minutes because of the anxiety. Thanks so much for all the great information. I listen daily! Nicole: Hi Dr. Cabral! I have been listening to your podcasts for 2 years. Thank you for you for sharing your vast amount of knowledge with your community. My question is regarding my 7 year old son. After a long journey we have finally been diagnosed with Non Classic Congenital Adrenal Hyperplasia which has put his body into early puberty. For this he has been put on Hydrocortisone. After 3 months of taking hydrocortisone his hormone levels are finally dropping. This was great news except for the fact after my request to check his A1C verified he is prediabetic. Now I am at a loss. Do you have any tips for me or know of any good resources? My goal will be to focus on his nutrition as much as possible, as that is something we can control. I often think of you and your journey back to health. I know how intimidating this feels to me I can only imagine how you felt. Thank you for your time and knowledge. Jamie: Hi Dr Cabral, I was wondering if there is a difference between stomach bloat and lower belly bloat (under belly button) bloat. I sometimes find that while my upper stomach seems not bloated, I have severe bloat in my lower belly, even first thing in the morning. This bloat doesn't budge with ginger and other natural remedies (or even may still be there after defacation) What are the differences and what may cause lower belly bloat vs upper belly bloat? Holly: Hi Dr. C, hope you're well? I have one daily vice - coffee! Yes it's organic and the second is a swiss water processed decaf :) HOWEVER I'm a latte drinker and do not enjoy the taste of any of the nut milks in coffee. My go to has been Oat milk however it contains veg oils that i know are not good. My question is, would i be better off with cows milk vs. oat milk with oils? Which is the lesser of two evils? Thanks, Holly Thank you for tuning into this weekend's Cabral HouseCalls and be sure to check back tomorrow for our Mindset & Motivation Monday show to get your week started off right! - - - Show Notes and Resources: StephenCabral.com/2480 - - - Get a FREE Copy of Dr. Cabral's Book: The Rain Barrel Effect - - - Join the Community & Get Your Questions Answered: CabralSupportGroup.com - - - Dr. Cabral's Most Popular At-Home Lab Tests: > Complete Minerals & Metals Test (Test for mineral imbalances & heavy metal toxicity) - - - > Complete Candida, Metabolic & Vitamins Test (Test for 75 biomarkers including yeast & bacterial gut overgrowth, as well as vitamin levels) - - - > Complete Stress, Mood & Metabolism Test (Discover your complete thyroid, adrenal, hormone, vitamin D & insulin levels) - - - > Complete Food Sensitivity Test (Find out your hidden food sensitivities) - - - > Complete Omega-3 & Inflammation Test (Discover your levels of inflammation related to your omega-6 to omega-3 levels) - - - Get Your Question Answered On An Upcoming HouseCall: StephenCabral.com/askcabral - - - Would You Take 30 Seconds To Rate & Review The Cabral Concept? The best way to help me spread our mission of true natural health is to pass on the good word, and I read and appreciate every review!
Scott Stevenson talks Bodybuilding Science. TIME STAMPS BELOW- With Scott McNally
In this episode of the IJGC podcast, Editor-in-Chief Dr. Pedro Ramirez is joined by Devon Abt to discuss SLN mapping in endometrial hyperplasia. Devon is a fourth-year Resident in Obstetrics & Gynecology at Beth Israel Deaconess Medical Center/Harvard Medical School currently applying into Gynecologic Oncology fellowship. Highlights: - Fewer than 30% of patients with preoperative diagnosis of endometrial intraepithelial neoplasia had concurrent endometrial cancer on final pathology. Endometrial stripe ≥20mm was associated with 2 times the risk of concurrent endometrial cancer. - Routine SLND in all patients with preoperative diagnosis of endometrial intraepithelial neoplasia is not cost-effective and would result in overtreatment. - Endometrial stripe may be a criterion for selectively using a sentinel lymph node algorithm in patients with preoperative diagnosis of endometrial intraepithelial neoplasia who are poor candidates for lymphadenectomy or at institutions where frozen section is not available or reliable.
Intro Hyperplasia is different from hypertrophy in that the adaptive cell change in hypertrophy is an increase in the size of cells, whereas hyperplasia involves an increase in the number of cells. Body Hypertrophy seems to generally lead to hyperplasia Swimming pool analogy Hypertrophy for beginners, intermediate, advanced Common hypertrophy rep ranges Mechanical tension model Pure volume model Periodization changes what works best Sensitization to intensity, volume, etc Not just for bodybuilders Dietary considerations Lean gains vs “bulking” Blood flow restriction training - should we mention Conclusions Periodization!
In Episode #219, I sit down with Layne Norton, PhD to answer one very topical question: does insulin make us fat? Welcome to the first bite-sized episode of The Proof. In these short conversations, I am joined by a guest to tackle a single topic in a brief, digestible time frame. In this episode, Layne Norton, PhD addresses recent online claims by Ben Bikman, PhD. Dr Bikman claims that if we keep insulin low and control seed oils, fat cells can stay smaller and healthier. But is there any truth to this claim, and would that necessarily be a good thing? In this episode, Dr Norton takes a highly scientific approach to the topic at hand. We discuss adipocyte hypertrophy, the difference between and significance of hypertrophy versus hypoplasia, and the consequences of misinformation. We also examine the uses and drawbacks of mechanistic data. Specifically, we cover: Intro [0:00] Dr. Bikman's Claims [2:24] Hyperplasia and hypertrophy [7:19] Gap of Research [12:17] Why this is problematic [15:56] Motives [20:14] Outro [22:53] To connect with Layne Norton, PhD, you can find him on Instagram and Twitter @biolayne, and on Facebook as Layne Norton. You can also visit his website, https://biolayne.com/, to learn more about his work and other resources. The best way to support the show is to use the products and services offered by our sponsors. To check them out, and enjoy great savings, visit theproof.com/friends. Enjoy, friends. Simon Want to support the show? The best way to support the show is to use the products and services offered by our sponsors. To check them out, and enjoy great savings, visit theproof.com/friends. Simon Hill, Msc, Bsc (Hons) Creator of Theproof.com and host of The Proof with Simon Hill Author of The Proof is in the Plants Watch the episodes on YouTube, or Listen on Apple/Spotify Connect with me on Instagram, Twitter and Facebook Download my complimentary two week meal plan and plant performance Learn more about your ad choices. Visit megaphone.fm/adchoices
Pathology : Robbins & Cotran : Adaptations of cellular growth & differentiation Hypertrophy| Hyperplasia| Atrophy | Metaplasia Hypertrophy Hypertrophy is an increase in the size of cells resulting in an increase in the size of the organ. In contrast, hyper- plasia (discussed next) is an increase in cell number. Stated another way, in pure hypertrophy there are no new cells, just bigger cells containing increased amounts of structural proteins and organelles. Hyperplasia is an adaptive response in cells capable of replication, whereas hypertro- phy occurs when cells have a limited capacity to divide. Hypertrophy and hyperplasia also can occur together, and obviously both result in an enlarged organ. Hypertrophy can be physiologic or pathologic and is caused either by increased functional demand or by growth factor or hormonal stimulation. Hyperplasia Hyperplasia is an increase in the number of cells in an organ that stems from increased proliferation, either of differentiated cells or, in some instances, less differenti- ated progenitor cells. As discussed earlier, hyperplasia takes place if the tissue contains cell populations capable of replication; it may occur concurrently with hypertrophy and often in response to the same stimuli. Hyperplasia can be physiologic or pathologic; in both situations, cellular proliferation is stimulated by growth factors that are produced by a variety of cell types.Metaplasia Metaplasia is a change in which one adult cell type (epi- thelial or mesenchymal) is replaced by another adult cell type. In this type of cellular adaptation, a cell type sensitive to a particular stress is replaced by another cell type better able to withstand the adverse environment. Metaplasia is thought to arise by the reprogramming of stem cells.Atrophy Atrophy is shrinkage in the size of cells by the loss of cell substance. When a sufficient number of cells are involved, the entire tissue or organ is reduced in size, or atrophic Although atrophic cells may have diminished function, they are not dead. Causes of atrophy include a decreased workload (e.g., immobilization of a limb to permit healing of a fracture), loss of innervation, diminished blood supply, inadequate nutrition, loss of endocrine stimulation, and aging (senile atrophy). Although some of these stimuli are physiologic (e.g., the loss of hormone stimulation in menopause) and others are pathologic (e.g., denervation), the fundamental cellular changes are similar. They represent a retreat by the cell to a smaller size at which survival is still possible; a new equilibrium is achieved between cell size and dimin- ished blood supply, nutrition, or trophic stimulation. Cellular atrophy results from a combination of decreased protein synthesis and increased protein degradation. • Protein synthesis decreases because of reduced meta- bolic activity. • The degradation of cellular proteins occurs mainly by the ubiquitin-proteasome pathway. Nutrient deficiency and disuse may activate ubiquitin ligases, which attach multiple copies of the small peptide ubiquitin to cellular proteins and target them for degradation in protea- somes. This pathway is also thought to be responsible for the accelerated proteolysis seen in a variety of cata- bolic conditions, including the cachexia associated with cancer. • In many situations, atrophy also is associated with autophagy, with resulting increases in the number of autophagic vacuoles. As discussed previously, autoph- agy is the process in which the starved cell eats its own organelles in an attempt to survive.
Listen as Dr. London Smith (.com) and his producer Cameron discuss Focal Nodular Hyperplasia with special guest Chak (Cassie Walker). Not so boring! https://www.patreon.com/join/jockdocpodcast Hosts: London Smith, Cameron Clark. Guest: Cassie Walker. Produced by: Dylan Walker Created by: London Smith
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In this episode of the IJGC podcast, Editor-in-Chief Dr. Pedro Ramirez, is joined by Dr. Jason Wright to discuss SLN in endometrial atypical hyperplasia. Dr. Wright is the Sol Goldman Associate Professor and Chief of the Division of Gynecologic Oncology at Columbia University. Highlights: -Performance of sentinel lymph node mapping is increasing in women undergoing hysterectomy for complex atypical endometrial hyperplasia. -Sentinel lymph node mapping in women with atypical endometrial hyperplasia does not increase perioperative morbidity or mortality. -The addition of sentinel lymph node mapping to surgery for endometrial hyperplasia is associated with increased hospital costs.
This podcast is going to cover a technical aspect of muscle building. I'm discussing which is more important for getting bigger and stronger: hypertrophy or hyperplasia? What do those words even mean? Well, hypertrophy is when your muscle cells get bigger, and hyperplasia is when the number of muscle cells increases. Scientists aren't sure whether hyperplasia even happens in humans, but “believers” contend that both hypertrophy and hyperplasia contribute to muscle growth. Thus, according to them, you should train to produce both of these phenomena to gain muscle as quickly as possible. Focusing on hypertrophy or hyperplasia would change how you train, so this topic has practical applications I'm going to break down for you in this podcast. Timestamps: 4:27 - What is hypertrophy? 4:46 - What are the components of muscle? 4:56 - What is myofibrilar hypertrophy? 5:16 - What is muscle hyperplasia? 5:37 - Does hyperplasia happen in humans? 7:16 - Does hyperplasia exist? 8:10 - How do you induce hyperplasia in animals? 9:26 - What does the science say about hyperplasia in humans? 16:56 - How do we cause hypertrophy? 18:28 - Why does training near failure produce hypertrophy? 19:06 - What about hyperplasia and training? 20:12 - Does stretching cause hyperplasia? Mentioned on the Show: Legion VIP One-on-One Coaching: https://buylegion.com/vip
Atypical Breast Hyperplasia may present either as Atypical Ductal Hyperplasia or Atypical Lobular Hyperplasia. Which of these two is considered the more “pre-malignant”? Is surgical excision necessary for all atypical breast hyperplasia? Who qualifies for tamoxifen? And, how does breast tomosynthesis fit into the management scheme? We will dive into these questions and more in this session.
Peak Human - Unbiased Nutrition Info for Optimum Health, Fitness & Living
Welcome back, friends! I'm Brian Sanders and this is the Peak Human podcast. I'm also the owner of NoseToTail.org where you can get all kinds of properly raised meats and the creator of the Food Lies film. We continue to work on the film daily and are just as desperate to finish it and get out to the world as you are. I know it's taking longer than expected, but we are making something truly special and there are a lot of factors we're dealing with. It will be worth it, trust me. This episode today is very special and a long time in the making. I got the top 2 audience favorites on one podcast. This nutrition stuff is so complex, it's never enough to get one side of the story so I thought I could at least get 2. I try to stay out of giving my opinions in this one. Perhaps 3 would be a crowd. We talk about so many interesting topics such as the carbohydrate-insulin model of obesity, the alternatives to this. The personal fat threshold, the insulin model of disease which is separate from how one becomes overweight. Satiety and how it fits into this. The processing of foods and how it plays in. Basal insulin vs. bolus. THe satiety vs. calorie theory. Also the big PUFA debate - are they as bad as they're made out to be? I think they are, but these 2 don't seem to think there's anything uniquely problematic with them other than they supply empty calories and we eat way too much of them. So they do think you should cut them out. I try to not interject much. I'm not trying to insert myself too much into these discussions. I have all kinds of guests on with differing opinions to expose people to all the angles of this complex nutrition world. I think something particularly with these guys and PUFAs, especially omega 6, is that they haven't spent the time to really dig into these details enough. They have busy lives and are focused on other areas of health. Just an idea. Really we're looking at the mystery of what goes on with food we eat and it's role in disease and/or obesity (which are independent of each other). Everyone should be interested in this to some extent. Maybe not to the level of a 90 minute podcast into some of the small details, but these are important questions that need to be answered. Dr. Ted Naiman is a board-certified Family Medicine physician in the department of Primary Care at a leading major medical center in Seattle. His research and medical practice are focused on the practical implementation of diet and exercise for health optimization. He has an undergraduate degree in mechanical engineering and utilizes engineering principles when dealing with complex systems such as human health and nutrition. Dr. Ben Bikman is a researcher and professor specializing in metabolic function, insulin resistance, and the drivers of metabolic disorders. He has a PhD in bioenergetics, he did a postdoctoral fellowship with the Duke National University of Singapore in metabolic disorders, and he's currently exploring the contrasting roles of insulin and ketones as key drivers of metabolic function. GET THE MEAT! http://NosetoTail.org GET THE FREE SAPIEN FOOD GUIDE! http://Sapien.org SHOW NOTES [1:00] The 4 steps of insulin resistance. [3:00] A deep dive into how insulin resistance spreads throughout the body. [8:00] Hypertrophy and Hyperplasia of fat cells. [14:00] Carbohydrate insulin model. [27:00] How refined food plays a role in obesity. [31:45] How processing, cooking, and pulverizing our food can lead to different hormonal reactions. [42:20] Differences between basal and postprandial insulin. [46:00] Obesity is a function of insulin and excessive energy intake [56:00] Are PUFAs truly as bad as they are portrayed to be? Soybean Oil Is More Obesogenic and Diabetogenic than Coconut Oil and Fructose in Mouse: Potential Role for the Liver [1:09:40] How refined fats and carbs affect satiation and brain chemistry. [1:18:20] A recap and discussing Brian's Uganda trip. GET THE MEAT! http://NosetoTail.org GET THE FREE SAPIEN FOOD GUIDE! http://Sapien.org Follow along: http://twitter.com/FoodLiesOrg http://instagram.com/food.lies http://facebook.com/FoodLiesOrg
When it comes to the human body, Size Matters. And this is particularly so for those who are interested in increasing their muscle mass. What exactly are we increasing, however, when our muscles grow? And how much bigger should we try to be? Is it just a matter of personal taste, or is there an optimal degree of hypertrophy for human movement? Some think bigger is better, while others believe that too much can be detrimental to movement and motor skill. Once again we’re faced with a fitness controversy, having no dearth of opinions. To help us sift through the science and uncover some of the secrets behind the size, we sat down with professor of physiology, researcher, and former Mr. United States Natural Bodybuilding Federation champion, Dr. Fred DiMenna. Our previous two episodes, Power Play and It’s About Time, centered on rate of tension development (RTD), fatigue and the neuromotor implications of those constructs, so hypertrophy was a logical next subject. We cover some exercise physiology studies, techniques and perspectives on developing hypertrophy, and merge them with some of the motor control theories and personal perspectives discussed in prior episodes, to create a richer tapestry of What Really Matters when it comes to size. Three reasons why you should listen to this episode: First of all, what, exactly, is hypertrophy? Dr. DiMenna explains what’s occurring at the cellular level when a muscle undergoes hypertrophy, or hyperplasia and how it can be measured both in a lab and in the gym. There’s been some speculation as to what we’re actually experiencing during human muscle growth, and Dr. DiMenna offers his perspective. How important is Growth Hormone (HGH)? Conventional wisdom tells us that post-exercise HGH, circulating in the blood, is an accurate marker of subsequent muscle growth. Dr. DiMenna clues us in to why that might not be the case. Dr. DiMenna also identifies a more accurate proxy of an environment amenable to creating hypertrophy that often gets overlooked by mainstream media, listen in to hear what this is! Intensity is obviously necessary for hypertrophy, right? Perhaps, but what does intensity really mean? How do we quantify it? It seems like intensity and load should be synonymous, but does the load need to be heavy for the exercise to be intense? Can we create enough intensity with light loads to reach a stimulus significant enough to drive hypertrophy? How does that compare to using heavier loads? Think learning about any of this can help you in your workouts? We do! In this episode we discuss: The cellular dynamics of hypertrophy Hypertrophy vs hyperplasia Protein synthesis Fast and slow twitch fiber transitions due to hypertrophy training Is hypertrophy training helpful as we age? Glossary: Concentric Failure - The point during a muscle-shortening contraction at which a repetition can no longer be completed Eccentric Failure - The point during a muscle-lengthening contraction at which a repetition can no longer be completed Hyperplasia - Muscle growth due to an increase in the number of muscle fibers Hypertrophy - Muscle growth due to an increase in the size of muscle fibers Mitochondria - An organelle found in most cells responsible for governing processes of respiration and energy production Protein Synthesis - Two stage process; transcription and translation in which cells make new proteins, can be used as a proxy to measure hypertrophic effect of an exercise condition References: Mitchell CJ, Churchward-Venne TA, West DW, et. al. (2012) Resistance exercise load does not determine training-mediated hypertrophic gains in young men. Journal of Applied Physiology. 113: 71–77. Takarada, Y., Nakamura, Y., Aruga, S., et. al. (2000) Rapid increase in plasma growth hormone after low-intensity resistance exercise with vascular occlusion. Journal of Applied Physiology. 88(1): 61–65. Trappe, S., Williamson, D., Godard, M., et. al. (1985) Effect of resistance training on single muscle fiber contractile function in older men. Journal of Applied Physiology. 89(1): 143-52. West DW, Burd NA, Tang JE, et. al. (2010) Elevations in ostensibly anabolic hormones with resistance exercise enhance neither training-induced muscle hypertrophy not strength of the elbow flexors. Journal of Applied Physiology. 108: 60–67. West DW, Kujbida GW, Moore DR, et. al. (2009) Resistance exercise-induced increases in putative anabolic hormones do not enhance muscle protein synthesis or intracellular signalling in young men. Journal of Physiology 587: 5239–5247. You might also like: Season 1 Episode 9 - Lessons from an Icon Season 2, Episode 7 - Power Play Season 2, Episode 8 - It’s About Time ADVERTISE WITH US: Reach dedicated exercise professionals, future trainers, and exercise enthusiasts all over the world. Send us an email to get the conversation started, hello@thinkfitbefitpodcast.com SUBSCRIBE TO THE NEWSLETTER: Dive deeper with us. Sign up here. We offer a unique view on muscles, portals to new ways to respect the body and health. Learning and ‘enjoy the process’ is a buzzy term. We take learning seriously and want to take our listeners on that journey with us and through us SUBSCRIBE: Subscribe to the podcast to make sure you never miss an episode. episode. You can find us on a variety of podcast apps: Spotify Pandora iHeartRadio Amazon music TuneIn Google podcasts WRITE A REVIEW: Leave us a rating and a written review on iTunes so more listeners can find us. JOIN THE CONVERSATION: If you have a question or a topic you want us to address, send us an email here. You can also connect to us through Twitter, Facebook, and Instagram. Tag #thinkfitbefit for a chance to be featured! NEW TO THE SHOW? Don't be afraid to start with the trailer or our recap episodes. the trailer best of 2020 episode part 1 Recap Season 1 of Fitness for Consumption part 1 Recap Season 1 of Fitness for Consumption part 2 SUPPORT THE SHOW: by checking out LADDER SPORT, a line of high performance nutritional supplements created by Lebron James and his trainer. They are high quality and NSF certified. Use the code BEFIT10 for a special discount! HOST can be found at: Instagram https://www.instagram.com/fitnessforconsumption/
Today, I am blessed to have here with me for a THIRD time Dr. Benjamin Bikman. He is a biomedical scientist, professor, and expert on insulin resistance. Dr. Benjamin Bikman studies diabetes and obesity, and much of what he focuses on is a seemingly obscure condition called insulin resistance. But it's, in fact, not very obscure at all. What is insulin resistance? Why did Dr. Benjamin Bikman devote his career to studying something like this? Well, insulin resistance is essentially two things. First of all, some cells in your body aren't responding very well to the hormone insulin. Second, you have more insulin than you did before, a condition called hyperinsulinemia. Dr. Bikman's research focus is to elucidate the molecular mechanisms that mediate the disruption that causes and accompanies metabolic disorders, such as obesity, type 2 diabetes, and dementia. Driven by his academic training (Ph.D. in Bioenergetics and postdoctoral fellowship with the Duke-National University of Singapore in metabolic disorders), he is currently exploring the different roles of insulin and ketones as critical drivers of metabolic function. He frequently publishes his research in peer-reviewed journals and presents at international science meetings. In this episode, Dr. Bikman starts by telling us about his journey with social media. In this, he explains his thoughts on why people are so addicted to hatred. He then describes his new manuscript about the differences in the hippocampus of Alzheimer's brains vs. control brains. Next, he discusses sphingolipids, different types of fat growth, and the importance of our humble kidneys! Dr. Bikman finishes up by discussing if people perish purely from diabetes and the link between salt and insulin sensitivity. Pre Order Keto Flex: http://www.ketoflexbook.com -------------------------------------------------------- / / E P I S O D E S P ON S O R S PureForm Omega Plant Based Oils (Best Alternative to Fish Oil): http://www.purelifescience.com Use ben4 for $4.00 off. Upgraded Formulas Hair Mineral Deficiency Analysis & Supplements: http://www.upgradedformulas.com Use BEN10 at checkout for 10% off your order. Text me +1 (786) 364-5002
Thank you for joining us for our 2nd Cabral HouseCall of the weekend! I’m looking forward to sharing with you some of our community’s questions that have come in over the past few weeks… Let’s get started! Monique: Hi Dr. Cabral, I discovered you after listening to a podcast where Melissa Ambrosini interviewed you and it’s changed my life so much. So thank you for all that you do! I have a question about a Herb Tea I found in a store called Cat’s Claw (Uncaria Tomentosa). On the back of the packet it states: “Cat’s Claw is an immune stimulant, rich in proanthocyandin, a powerful antioxidant and free radical scavenger. This combined with it’s white blood cell increasing effect, makes it effective in the treatment of cancer. It cleanses the intestinal tract, assisting with Crohn’s disease, arthritis, hemorrhoids, parasites, leaky bowel syndrome, ulcers, gastritis, allergic disorders, diverticulitis and other gastro-in-test I also disorders. Also helpful for chemotherapy recovery.” It states to simmer 1 teaspoon in 1 cup of water for 10 minutes , take 3 times a day. I’m wondering if you have heard of it and if so what your views are? Thanks for your time! Kelly: Hello Dr. Cabral. I was diagnosed with atypia hyperplasia in one of my breast 8 months ago. I had the area surgical removed and no cancer was detected. I am now taking Tamoxifen (20 mg) daily to reduce the risk of breast cancer. I am working with a Naturopath who works specifically with people who have cancer or who are at risk of cancer. He has me on a protocol that includes 20 mg of Melatonin. I had to work up to that amount over a period of a few weeks but now I am fine with it. My question though is I have noticed my morning blood sugar level is higher than what my levels typically are. I used to wake with a blood glucose level between 75 and 85. Now it is typically between 95 and 105. I eat a mostly paleo diet, exercise 6 days week, and keep myself very hydrated. So my question is, can that much melatonin have an impact on my blood sugar levels? Laura: Hi Dr. Cabral! I recently took the 3-day stool test. My results came back with a 1+ level of Candida Parapsilosis. Is this treated the same way that other types of Candida are treated? I'd love to hear from you regarding what causes this and how to treat it. Thank you SO much for your time and expertise - we're all getting a little smarter because of you. Laura Tom: Hi Dr Cabral, first of all, thank you very much for sharing your knowledge with the community! So, everyone knows that they must exercise and that there are a lot of benefits for it. But why SHOULD you exercise? If you're healthy, happy, weight is good, you're fit... why should exercise still be a must? Christina: Hi doctor Cabral! I hope you’re doing great! There’s this weird health question that’s been on my mind, and I figured that if anyone would know the answer, it would be you. So, I’m a 40 yo female and have been celibate for over 4 years. When I was in a relationship, my periods were normal and regular. After my relationship ended, not so much anymore. One month they are 29 days, the other 42, and so on. Can it be that the fact that I’m not having sex anymore is influencing my hormones and making my periods irregular? I know it’s a weird question, but I have a gut feeling that could be it. I’m super curious to hear your thoughts about it. Thanks a million! Christina Dar: Hi love the show big fan. Have a two part question. Do you know anyone that's gone on a mucusless diet based on Arnold Ehret's results and if so how did they feel? The guy ate leafy greens and fruits exclusively and was able to hike up mountains while fasting, walk 56 hours consecutively and bike I believe something like 1000 kilometers at one time which for a non athlete is mind blowing. Also how long do you think it would take the average person to clean their bowels on a juice fast I'm on day 8 and surprisingly had a bowel movement. Ate the sad diet up until the last few years. Now going more plant based. Thanks! Thank you for tuning into this weekend’s Cabral HouseCalls and be sure to check back tomorrow for our Mindset & Motivation Monday show to get your week started off right! - - - Show Notes & Resources: http://StephenCabral.com/1906 - - - Dr. Cabral's New Book, The Rain Barrel Effect https://amzn.to/2H0W7Ge - - - Join the Community & Get Your Questions Answered: http://CabralSupportGroup.com - - - Dr. Cabral’s Most Popular At-Home Lab Tests: > Complete Minerals & Metals Test (Test for mineral imbalances & heavy metal toxicity) - - - > Complete Candida, Metabolic & Vitamins Test (Test for 75 biomarkers including yeast & bacterial gut overgrowth, as well as vitamin levels) - - - > Complete Stress, Mood & Metabolism Test (Discover your complete thyroid, adrenal, hormone, vitamin D & insulin levels) - - - > Complete Stress, Sleep & Hormones Test (Run your adrenal & hormone levels) - - - > Complete Food Sensitivity Test (Find out your hidden food sensitivities) - - - > Complete Omega-3 & Inflammation Test (Discover your levels of inflammation related to your omega-6 to omega-3 levels)
This week we welcome Benjamin Bikman. Ben earned his Ph.D. in Bioenergetics and was a postdoctoral fellow with the Duke-National University of Singapore in metabolic disorders. Currently, his professional focus as a scientist and professor to better understand chronic modern-day diseases, with special emphasis on the origins and consequences of obesity and diabetes. With a particular emphasis on the role of insulin. He frequently publishes his research in peer-reviewed journals and presents at international science meetings. In this episode, We discuss all things Insulin Resistance and Insulin Sensitivity. Such as Linoleic Acid Effects on Insulin, PCOS, Erectile Dysfunction, and Migraines. Ben also gives us his dietary recommendations for managing insulin resistance. Join The Better! Community On Facebook. Get the show notes here.
Welcome to Episode 27 Today I am joined by ChiChi @makingbabyo_ who is Nigerian & British residing in the UK. She joins us to share her Infertility journey, pregnancy loss, natural killer cells, Endometrial hyperplasia and the surgery to treat it. She also shares her reasons for opting for IVF treatment abroad, the cost concerns as well as race concerns that crosses your mind when traveling abroad for treatment. Our conversations included the idea that IVF feels like gambling with zero guarantees. ChiChi has travelled to several countries in Europe for consultations and fertility treatment. . Join us to listen to her incredible journey. . Remember to Subscribe, leave a review & give this podcast a 5* rating to help spread more awareness about In(fertility). . . If you would like to share your story, I would love to hear from you. Please email me at: . fertilityconversations@gmail.com . . Follow me on Instagram @fertilityconversations . . Read my blog: www.fertilityconversations.com
How to "get yoked": An in-depth look on how to program for muscle development for your best off season gains.In this weeks episode Tim and Brandon break down the process of adding lean muscle tissue or "getting yoked" into a more easily understood concept. Follow along with the conversation as they discuss: MindestAre you lean enough for this? have you been in a dieting phase and reached a point where you can go into a good reverse diet now?Scheduling: This is good information for all other aspects of your life.Training: How hard should you are training, if you're even training hard at allNutrition: This is the cornerstone to any phase whether it be weight loss or gainCardio / Daily Steps: How is your daily output are you getting in nearly 10k steps every day?How far is enough and when to transition into the next phase?What is your next phase?It was a great conversation with many great key takeaways. As always we appreciate your support if you feel like this podcast has given you value please share with your community. Thank You!Tim English - @Everycaloriecounts [https://www.instagram.com/everycaloriecounts/]Brandon DaCruz - @brandondacruz_ [https://www.instagram.com/brandondacruz_/]Alex Zaro - @EveryCalorieCounts.Alex [https://www.instagram.com/everycaloriecounts.alex/]
John Jaquish, PhD. has spent years researching and developing improved approaches to health. He is the inventor of the most effective bone density building medical technology which is now partnered with Tony Robbins and OsteoStrong for rapid clinic deployment.Inventor of X3, a technology that is proven to develop muscle much faster than conventional weight lifting, all with the lowest risk of joint injury, Dr. Jaquish methods are used in training the world's most elite athletes and associations such as the entire Miami Heat organization, various NFL and NBA players, as well as Olympians. 1:00- Intro to Dr. Jaquish and his book 4:00- Major problem in the fitness industry 11:15- Explaining the X3 system 14:45- Why can’t we simply use bands in the gym? 17:45- Why can’t we use heavy partials? 19:45- Band tension when it comes to exercises with different force curves e.g. rows 23:45- Dr. Jaquish’s thoughts on Layne Norton 28:00- Is the fitness industry scientific? 34:00- Why isn’t this “a band and a stick”? 37:00- Home workout products 44:00- Dietary approach 47:00- Hyperplasia / extreme stretch researchDr. Jaquish's website: https://www.jaquishbiomedical.com/doc...Website and Coaching: https://drdavemaconi.com/Smile Fund: https://secure.operationsmile.org/sit...Youtube Homepage: https://www.youtube.com/channel/UCW-P...
The Flex Success Podcast episode #39 - Greg Nuckols - Hypertrophy, dieting and the study of hyperplasia Greg Nuckols is one of the industry good guys. He is also an industry professional you can trust as his deep dive into research offers an unbiased dialogue on what we currently know, versus what we think we know. In this episode Greg discusses his recall of information, how he is currently managing his diet, what he wish he knew when he started lifting and how he would research hyperplasia if ethics didnt exist. Something Worth Sharing Nick Lane - Power, Sex, Suicide: Mitochondria and the Meaning of Life Fan of the show? If you find value in the Flex Success Podcast, we'd love if you could show your support by leaving a review on iTunes. This will help expose the show to a bigger audience so we can help more people be less shit. To find out more about Flex Success, head to: www.flexsuccess.com.au Flex Success Instagram To find out more about Greg Nuckols, head to: Greg's Instagram --- Send in a voice message
Endometrial hyperplasia is common in peri-menopause. It's simply an abnormal thickening of the uterine lining or endometrium. It is not cancer but in some cases, it can increase your risk of later developing uterine cancer. Endometrial hyperplasia and Peri-Menopause Discovery Call Facebook group
In her latest radio interview, Dr. Jegasothy shares more of her expert skincare knowledge.Listen to this podcast to find out which skin care ingredients you should avoid & how to spot them if they are not on ingredient labels, what is sebaceous hyperplasia, and what the causes and treatments of excessive sweating are. Thanks again to Heidi Godman for featuring Dr. Jegasothy again on Health Check for WSRQ Sarasota Talk Radio 106.9 FM (1220 AM).
We welcome Dr John Jaquish back to "The Hacked Life" podcast for the third time!!! In our last two episodes we broke down variable resistance, the X3 bar, Osteostrong, and tons, tons, of info. Well, he comes back a third time and we go deep again, but this time delving deep into his bestselling and highly controversial book "Weightlifting Is A Waste of Time & So Is Cardio." We talk about veganism and why it'll be one of the greatest scandals in history, why the fitness industry is an absolute joke, why fasting actually builds muscle, and much, much more. We just scratch the surface of what the book breaks down into detail. Make sure to like the video that's how people can this information and please subscribe or leave me a comment and let me know what you liked. Last but not least, pick up Dr J's book, it's phenomenal and it's an easy read.If you want the show notes and other resources, head over to https://www.joelevancoaching.com/ If you're interested in life/health coaching, email me at info@joelevancoaching.comTimeline7:10 - Why write a book?9:50 - Why most peer reviewed studies are not full proof?14:22 - Is there ever a time to life weights?20:00 - Why the fitness industry is the most failed industry in the world26:50 - Why veganism will go down as the greatest scandal in history32:00 - Why fasting builds muscle36:59 - Why amino acids are important44:18 - Why carbs are not a macronutrient48:00 - What's Dr Jaquish's daily protocol like54:00 - 10 reps increases testosterone, so why not just do 10 reps?56:50 - Should I lift heavy or do more reps?1:10:30 - Myofibrils & sarcoplasm, and why X3 works out both.1:13:00 - Blood flow restriction training (BFR) induces hypoxia, why not just do that?1:16:00 - How hypoxia works1:19:00 - Time under tension and the force curve 1:24:43 - Hyperplasia and how to split muscles cells to increase growth1:34:00 - The difference between professional athletes' training & the average athlete1:44:45 - What is the fitness industry going to be like in 5 years?
In this episode we will be covering Hypertrophy, Metaplasia, Hyperplasia, and Atrophy. --- Support this podcast: https://anchor.fm/ben-haseen/support
Podcast Editor, Tracy Carlson, interviews author Kristen Hobbie to discuss the article, "Evaluation of Cystic Endometrial Hyperplasia and the Normal Estrous Cycle in Longitudinal Sections of Uterus from Female Harlan Sprague-Dawley Rats" which can currently be found in Vol. 48, Issue 5 of Toxicologic Pathology. Click here to read the article
In this episode, Kristin discusses the classification, diagnosis, and management of endometrial hyperplasia. At the end of this episode, listeners will be able to: Define the classification system for endometrial hyperplasia Review important investigations for the patient with abnormal uterine bleeding Discuss the management of hyperplasia with and without atypia For references for this podcast, please visit: http://obginyeg.libsyn.com
1. APGO medical student educational objectives 10th edition Faculty version 2. APGO medical student basic science teaching script on Endometrial Hyperplasia 3. Beckman CRB et al. Obstetrics and Gynecology. 8th ed. Philadelphia: Wolters Kluwer, 2019
In this episode of the IJGC podcast, Editor-in-Chief Dr. Pedro Ramirez, is joined by Dr. Ross Harrison to discuss fertility outcomes in women with endometrial hyperplasia or cancer. Before starting fellowship at MD Anderson Cancer Center in 2018, Dr. Harrison completed residency in obstetrics & gynecology at the University of Wisconsin. His research interests are related to the costs of cancer care, financial toxicity, and cost-effectiveness.
We’re back from our winter break with an excellent episode to kick off Season 2. This episode features an important update from the Game Changers cinematic universe, and some incredible feats of strength involving athletes with unbelievable longevity in a variety of physically demanding sports. Greg discusses a new documentary with some explosive allegations about doping in weightlifting, and then we get into some science-heavy segments with a Q&A and a Research Roundup. Topics include collagen supplementation, muscle hyperplasia, new caffeine research, and more. Finally, we discuss some more practical information about goal setting, programming your training during weight loss, and how to make really, really good caramel.TIME STAMPS0:00:52 Winter break recap, show structure moving forward. 0:06:23 Hugely important developments from the Game Changers cinematic universe. 0:09:02 Feats of Strength. 0:21:05 New documentary about doping in weightlifting. 0:40:06 Q&A. 0:40:21 What is the best way to achieve/plan for lifting goals going into the new year? 0:45:10 Is anyone looking into how we can increase our overall potential for muscle growth by boosting hyperplasia? 0:59:35 Does collagen or glycine supplementation provide any benefit to connective tissue, muscle, or skin, beyond simply increasing protein quantity? 1:13:13 What do you think about Brian Minor’s theory that being able to lift more is a result of hypertrophy, rather than hypertrophy being a result of lifting heavier? 1:26:09 Research Roundup. 1:26:32 Effects of acute caffeine, theanine and tyrosine supplementation on mental and physical performance in athletes. https://jissn.biomedcentral.com/articles/10.1186/s12970-019-0326-3. 1:35:45 High Compared with Moderate Protein Intake Reduces Adaptive Thermogenesis and Induces a Negative Energy Balance during Long-term Weight-Loss Maintenance in Participants with Prediabetes in the Postobese State: A PREVIEW Study. https://academic.oup.com/jn/advance-article/doi/10.1093/jn/nxz281/5637681. 1:43:51 Coach’s Corner: Programming your training during weight loss. 1:53:28 To Play Us Out: Mailbag update on processed meat meta-analysis, and how to make really good caramels at home.
See all the Healthcast at https://www.biobalancehealth.com/healthcast-blog/ This week we are examining a story about a young man who had a hereditary disease. He was one of several in his family who apparently had suffered these same symptoms which are believed to mean that they had the same illness. Doctors had not been able to diagnose what was wrong. This young man became crippled and bed -ridden and medical science was not helping him get better. The best they could do was help him survive in bed, suffering from progressively intense weakness and fearing that he would die. He could not work, get out of bed, go to school or the store, all he could do was lie in bed and read old medical texts. He scoured old books to see if he could find references that described his symptomology. Over a period of eleven years, he read all these old books and educated himself about what he was reading and what he was suffering. He finally found what he thought was wrong with him and he and a friend went to a medical convention to try to convince the doctors at the convention that he had one of only 32 recorded cases of bilateral adrenal medullary hyperplasia. He was so insistent with his presentation that the conferences began to allow him to present on his situation. In order to go to the convention, he would have to buy an entire row of seats on a plane and then get an ambulance or a station wagon to take him to the hotel, and his friend would help him get up and get to the stage to present. What an amazing story of intelligence, deliberation, determination and success! He and some of the doctors at the convention who bought into his research even though he was not a doctor and had not graduated from college, began to work on treatments and experiment with things to do to help him get stronger. The organization where he obtained acceptance and a positive reception was the National Dysautonomia Research Foundation, a group who focuses on adrenal research. This young man was able to rent a van and have a friend drive him over 500 miles to Birmingham to repurpose a drug called Levophed, which is basically an injection of noradrenaline. The process of repurposing means that doctors are free to use a drug off label or to use it to treat things which in their medical judgment would help even though that is not what the FDA allows the drug to be used for. Doctors often do this but there is always risk for the patient and the doctor when they prescribe off label uses for a medication. He eventually determined that his adrenal glands were acting as if they had a tumor even though the tumor did not show on the scans. But he convinced a surgeon that his adrenal glands needed to have the center of them, the medulla popped out. This surgery was not one that was accepted by mainstream medicine. It took over 17 months to find and convince a physician to undertake this surgery as an experimental process. But he did find one at the University of Alabama. The treatment was a success and he has been able to get up and stay on his feet for hours and to go back to school where he is now doing research for others suffering from rare and unusual diseases and trying to find new treatments for them. This is a miracle story about the human spirit and the courage and determination of a single man who changed medicine and the trajectory of his own life. Listen to our discussion and be encouraged!
Joel and Garrett are now on the 10th episode of the Flatline Fitness Podcast. In this episode they discuss hypertrophy and hyperplasia. It's all about building muscle and how to do so. Subjects talked about in this episode go from intense training techniques to just the basics of getting results from weight lifting!
In this episode of the School of Muscle podcast, Dr. Andy Galpin discusses muscle hyperplasia and much more! Time Stamps: 00:12 - Intermittent vs Continuous Dieting 8:15 - The spectrum of fiber types 16:00 - Strength phases for hypertrophy 19:00 - Anabolic window specific to fiber types 24:50 - Hyperplasia 30:30 - Artificial Intelligence in fitness/health 38:00 - Lessons learned from mentors and advice around social media Instagram : @ryanjsolomon Youtube.com/ryansolomon
In this episode of the School of Muscle podcast, Dr. Andy Galpin discusses muscle hyperplasia and much more! Time Stamps: 00:12 - Intermittent vs Continuous Dieting 8:15 - The spectrum of fiber types 16:00 - Strength phases for hypertrophy 19:00 - Anabolic window specific to fiber types 24:50 - Hyperplasia 30:30 - Artificial Intelligence in fitness/health 38:00 - Lessons learned from mentors and advice around social media Instagram : @ryanjsolomon Youtube.com/ryansolomon
Your "genetic" limitations on muscle size are NOT genetic. You've stopped growing because your training hasn't optimized the four key physiological factors that are essential for accessing your TRUE muscle growth potential. In today's episode Coach Chris connects with the Mad Scientist of Muscle Nick Nilsson. Find out how exercise science has proven that these four factors can be TRAINED and IMPROVED so that you can finally build the muscle mass that your body is TRULY capable of building and unlock your hidden potential. Discover How to Unlock 5, 10, even 20 Pounds of Your TRUE "Untapped" Muscle Growth Potential in the Next 90 Days with Metamorphosis. "Connective tissue unlike muscle tissue really does not go away with time, you develop this connective tissue strength, you've got it for a long time.." -Nick Nilsson Subscribe to iTunes Love the podcast? Leave a review on iTunes! What do you want to hear from the Critical Bench Podcast? Tell us here! Time Stamps: 02:40 How Nick developed the Metamorphosis program over 25 years of in-depth hands on experience 04:21 Definition of Angiogenesis training 07:46 Why Connective Tissue takes so long to strengthen 16:47 How Phase 3 Hyperplasia training will help you grow MORE muscle fibers 24:50 Super-human nervous system activation that is the SPARK that makes everything happen 33:04 How often the Mad Scientist works in his gym laboratory 36:14 Putting all 4 phases together of the Metamorphosis program 39:50 How ALL future training programs are impacted by your Metamorphosis training 42:08 Meet Nick's 3 dogs and eating fur for added protein 44:06 You are literally FAST-FORWARDING yourself into muscle and strength building over 90 days Resources Text "TESTO” to 345-345 for the Free 7 Worst Testosterone Killers Report madscientistmuscle.com metabolicmonsters.com Connect with Nick: madscientistofmuscle.com YouTube Instagram Facebook Connect with Critical Bench: Facebook Instagram Youtube www.criticalbench.com
Talk to a Dr. Berg Keto Consultant today and get the help you need on your journey (free consultation). Call 1-540-299-1557 with your questions about Keto, Intermittent Fasting, or the use of Dr. Berg products. Consultants are available Monday through Friday from 8:30 am to 9 pm EST. Saturday & Sunday 9 am to 5 pm EST. USA Only. Take Dr. Berg's Free Keto Mini-Course! Intermittent Fasting Basics: https://www.messenger.com/t/drericberg Today, Dr. Berg talks about two different ways we get overweight. It all boils down to genetics because some people shoot off multiple fat cells on the inside which is called Hyperplasia. They rarely get subcutaneous fat even though the body might look lean. The damage is mainly done deep within the body. The rest of the people get overweight from the fat cell expanding with high insulin 24/7, inflammation, and insulin resistance. Dr. Eric Berg DC Bio: Dr. Berg, 51 years of age is a chiropractor who specializes in weight loss through nutritional & natural methods. His private practice is located in Alexandria, Virginia. His clients include senior officials in the U.S. government & the Justice Department, ambassadors, medical doctors, high-level executives of prominent corporations, scientists, engineers, professors, and other clients from all walks of life. He is the author of The 7 Principles of Fat Burning. ABOUT DR. BERG: https://bit.ly/2FwSQQT DR. BERG'S STORY: https://bit.ly/2RwY5GP DR. BERG'S SHOP: https://bit.ly/2RN11yv DR. BERG'S VIDEO BLOG: https://bit.ly/2AZYyHt DR. BERG'S HEALTH COACHING TRAINING: https://bit.ly/2SZlH3o Follow us on FACEBOOK: https://www.messenger.com/t/drericberg TWITTER: https://twitter.com/DrBergDC YOUTUBE: https://www.youtube.com/user/drericberg123 Send a Message to Dr. Berg and his team: https://www.messenger.com/t/drericberg
In this Episode we hear from senior author Hervé Lefebvre discuss his recent paper 'Mast cell hyperplasia is associated with aldosterone hypersecretion in a subset of aldosterone-producing adenomas.'
In BONUS Episode 142 of Beat Infertility, Dr. Allison Rodgers answers five listener questions and then does a deep dive into endometrial hyperplasia. If you need additional support, consider joining Hope University or our Infertility Warrior Tribe. For details on both, please visit https://beatinfertility.co/hopeu.
In BONUS Episode 142 of Beat Infertility, Dr. Allison Rodgers answers five listener questions and then does a deep dive into endometrial hyperplasia. Infertility coach Heather Huhman helps warriors like you make scientifically-based, well-informed decisions about your next steps. To schedule your free 30-minute call, go to https://beatinfertility.co/hope.
In this episode I sit down to a fun conversation with one of my partners, Dr. Dean Tortorelis. We discussed Dr. Tortorelis' path in to Urology, why he is passionate about Men's Health, and, of course, the Urolift procedure for treatment of BPH. We had a lot of fun making this episode. I hope you enjoy it.
Summary: In this edition of the JDD podcast “Ask and Investigator,” host Dr. Adam Friedman from the GW School of Medicine and Health Sciences interview internationally recognized and NIH funded...
Dr Hieken speaks with ecancertv at EACR 2016 about the significance of a potential biomarker for breast cancer in hyperplasia patients. She describes how the subpopulation of patients, those with atypical hyperplasia of the breast and high levels of ERß, can act on this information with further breast cancer treatment, and the choice faced in risk reduction. Dr Hieken also summarises the interests and technologies being investigated in her lab, and the future of breast cancer prevention.
Journals for 2010 with Nicholas Fogelson and Paul Browne. Topics include NICHD Strip Classification, Oral vs Intrauterine Progestins for Hyperplasia, Fetal Lung Maturity Outcomes Less than 39 Weeks, Congenital Toxo and more! Academic OB/GYN Podcast Episode 28 – Journals for December 2010
Fakultät für Chemie und Pharmazie - Digitale Hochschulschriften der LMU - Teil 01/06
Fri, 14 Mar 2003 12:00:00 +0100 https://edoc.ub.uni-muenchen.de/868/ https://edoc.ub.uni-muenchen.de/868/1/Haider_Ursula.pdf Haider, Ursula G. B. ddc:540, ddc:500, Fakultät für Chemie und Ph
This paper presents the first case of extensive, diffuse, somatostatin- immunoreactive D-cell hyperplasia in the human stomach and duodenum. It occurred in a 37-yr-old woman, who showed clinical signs of dwarfism, obesity, dryness of the mouth, and goiter. The density of the distribution of D cells was increased 39-fold in the stomach fundus, 23- fold in the proximal antrum, 25-fold in the distal antrum, and 31-fold in the upper duodenum in comparison with normal values. At the same time, the gastrin-immunoreactive cells were increased 2.3-fold in the antrum. Although the range in size of the D cells was within normal limits in all regions examined, the G cells showed pronounced hypertrophy of up to 127%. A possible relationship between the immuno- histochemical findings and the clinical picture is discussed.
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