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Twitter/X Facebook A message from Dr. Thomas Patterson of Harvard Univerity: “Tomorrow, Sept. 17, is National Constitution Day— marking the 239th anniversary of the signing of the U.S. Constitution. As in other years, it will pass largely unnoticed despite its significance and relevance at this critical moment in our politics. I'm trying to elevate its visibility by encouraging people to take my free, nonpartisan HarvardX course on the U.S. Constitution (6 brief sessions). I'd be grateful if you would share the link with your social media followers and others: https://bit.ly/FreeCourseHarvard (pick ‘audit' for free version) Thank you.” I encourage you to take up Dr. Patterson’s opportunity! As your host on this podcast, I explain in this episode the background of the Constitution and why the American Revolution had such contrasting impacts on American and on France in the 18th century. You will learn how the French Revolution, while catastrophically different than the American, was brought to you by the American Revolution! –Rick Reiman Twitter/X Facebook
In this episode, I have the pleasure of speaking with Dr. Neil Nathan about the intersection between brain symptoms and complex illness. Dr. Nathan has practiced medicine for over 50 years. He is one of the leading experts in complex chronic illness and mold toxicity — author of the bestselling Toxic: Heal Your Body from Mold Toxicity, Lyme Disease, Multiple Chemical Sensitivity, and Chronic Environmental Illness and The Sensitive Patient's Healing Guide. I am grateful to call Dr. Nathan a mentor and colleague. His pioneering work has been responsible for my healing from chronic illness, as well as that of many patients I've treated. Together, we co-lead a small peer group (within the larger mentoring group he provides with Marie Matheson, ND), supporting practitioners who treat those with complex chronic illness.In this episode we discuss:03:43 – Dr. Nathan's path to understanding complex illness11:20 – Why complex illness is rising and underrecognized19:30 – Why new medical ideas take decades to reach patients23:15 – Mold, Lyme, and long-COVID: the “big three” root causes25:58 – The limbic-vagal-mast cell “trifecta”30:31 – Are some people born more vulnerable?44:39 – The risk in “just do limbic retraining” advice47:35 - Biochemical contributors49:26 – Structural root: jaw and cervical instability53:58 – Differentiating Between Mold, Lyme & Long-COVID58:49 – A message of hope, and where to learn moreFull Transcript belowYou can find Dr. Nathan's books, consultations and mentoring program through his website at neilnathanmd.com. If you'd like to help get this information out into the world, please consider liking, sharing, or commenting.Until next time,CourtneyCourtney Snyder MDDr. Courtney Snyder MD is a holistic and functional child and adult psychiatrist and host of the Holistic Psychiatry Podcast and can be found at courtneysnydermd.comMedical DisclaimerThis podcast is for educational purposes only and is not intended or implied to be a substitute for professional medical advice, diagnosis, or treatment. Consult your own physician for any medical issues you may be having.Full TranscriptDr. Nathan's Path to Understanding Complex Illness03:43Courtney: I'm so glad we're doing this, and I feel like it's long overdue. As you know, in the mental health space in psychiatry, there's a lot of recognition of a dysregulated nervous system, threat response in the body, but oftentimes that's attributed to external forces or trauma, and not necessarily internal threats that we see, obviously, in complex chronic illness, which we'll be talking about. There's also not the recognition of the relationship between physical symptoms and brain-related symptoms. So I know you're going to help us connect a lot of these dots. As we sort of just lay the groundwork for what complex chronic illness is — when in your path did you start to recognize the distinction, when was the beginning of your work and appreciation of complex health issues?Neil: Okay, there are two answers to that question. First, when I went to medical school, my hope was to become a healer. And I was very disappointed that when I got there, I realized they weren't going to teach me to be a healer. And back then I used to question my professors at medical school — why do you do it this way, and how is that? And I kept feeling in all of our interactions that we were missing something. And my professors would go, “Okay, Mr. Wise Guy, smart guy, what are we missing?” And I'd go, “I don't know, I just know we're missing something.” Later I would come to recognize that we were missing the emotional and spiritual component to whatever was affecting people in their illnesses. And I have language for it now — I didn't then. The person in medical school who influenced me the most was Elisabeth Kübler-Ross, who I view as my main mentor in medical school. She, unlike anyone else, had a one-and-a-half-hour seminar once a week. Back then there weren't video cameras — you were in a small confined room with a two-way mirror, and you're sitting there watching someone do this. And she would simply interview one of her patients. She was a psychiatrist, but she was the only one in medical school who demonstrated how to really listen to people, how to really communicate with them, and not have this standard review of systems — here's my questions, here's your answers, and that's what I do, and this is what I write up. She was really listening. She was trying to really take in, okay, what happened to you, and what's going on with you? So I would want to give her credit as a role model to change how I learn to listen. Okay, that was step one.I'm going to fast forward to the early 1980s. And I am, at that point, the medical director of a regional pain clinic. And we are starting to see a whole bunch of patients who had this complicated process of fatigue, unrefreshed sleep, irritable bowel syndrome, and pain migrating all over their body. And back then we called it fibrositis. Now we call this fibromyalgia. And so whenever medicine encounters a thing that they don't know what they're looking at, it's psychological until proven otherwise. And people would get therapy, and they would take antidepressants and anti-anxiety agents. Didn't help much. And so it became clear to me that we didn't understand what this was at all. It was not psychological. Something was going on in their bodies — now I recognize it as an inflammatory process. Back then there was no concept of that at all. And then, slowly, in the early nineties, we began to find pieces of the puzzle to understand what was triggering fibromyalgia. I was working back then with Jacob Teitelbaum, who did a lot of work with chronic fatigue and fibromyalgia, and we both independently came up with an understanding that a deficiency of magnesium, DHEA, adrenal issues, thyroid issues, sex hormone deficiencies, GI dysbiosis, and food allergy — if we looked at those and treated those, the majority of people with fibromyalgia got cured. It was different for different patients. It was not like there was one thing that caused it. And as time evolved, we began to realize that there was lots of other biochemical imbalances in the body that would contribute to this kind of a process that represented chronic fatigue and fibromyalgia.We discovered that Lyme disease and coinfections was a huge piece of that. And later still we discovered that mold toxicity was a huge piece of that. And so my understanding of the complexity — and it's not really confined to chronic fatigue and fibromyalgia at this point, we're really talking about almost all chronic illness — with a huge psychological component to that. So we've begun to understand this as essentially some version of inflammation. And if we start to really dig into what are the potential causes of that inflammation, we can make a huge impact for them. So this is a multi-year evolving understanding of how a person's genetics, biochemistry, physiology impacts their whole being in every way. And that inflammation extends to the nervous system, contributing greatly, if not causing directly, a large number of what are now called psychiatric diagnoses.Courtney: Right. So even among those patients that you were seeing — you were seeing, I'm assuming, depression, anxiety, brain fog?Neil: Yeah, the vast majority of our patients with mold toxicity have brain fog and anxiety, depression, OCD behaviors, mood swings, depersonalization, derealization. I mean, all of that is super common in our patients.Complex Illness Is Rising — and Underrecognized11:20Courtney: Do you feel like this is more recognized now — or do you feel like the number of people being hit with these complex health issues is increasing — or both?Neil: Both. I would love to say that the medical profession is increasingly recognizing all of this as important, but that recognition is coming very, very slowly. The majority of folks out there, if they were to list the symptoms that they had, their doctors would basically look at them like, “I have no idea what's causing it, so this must be in your head.” And that is the message they're getting from their physicians at this point. So many patients are being gaslighted, so many patients are being told it's in your head. And the repercussions of that are the family is hearing that same message, and the families become less supportive of those people, because they're saying, “This is not real, so why would I support you? This is still in your head, go get your head fixed.” And they're wrong, and it's really a travesty. But so yes, very slowly, this awareness is growing. There are now more medical meetings that I'm aware of where psychiatrists can learn more about this than there were ten, fifteen years ago, for sure. But not enough — it's still not enough.The other part of that is some of the main triggers for this inflammatory process are increasing. So we're seeing more and more mold toxicity, we're seeing more and more Lyme and coinfections, partly because the environment that we are all exposed to has gotten extremely increasingly toxic. So it's now known, for example, that there are 350,000 new chemicals in our environment than existed 70 years ago. The vast majority of those have never been tested for safety in human beings. So human beings have a tremendous amount of chemical exposure that they never had before. Human beings have a tremendous amount of EMF, electromagnetic exposures that they never had before in the history of the human race. So these exposures are cumulative. Our major organs, like our liver and our kidney, that have to deal with toxicity, are overloaded. And so, yes, we are seeing an epidemic of a whole bunch of these conditions. In children, we are aware that autism has skyrocketed — it went from one in several thousand kids to one in thirty kids in many states now have autism. And forgive me, but the pediatric profession is not taking this seriously. It's like, “Yeah, that's sad, it's unfortunate.” It's like — really, you don't want to look at this? This means one in every three kids born are getting autism — does that not concern you? But, forgive me, the medical profession is very slow to embrace new information, and that is really to the detriment.We're seeing an epidemic of Alzheimer's, Parkinson's, ALS, all of which involve neuroinflammation. We've seen already an epidemic of chronic fatigue and fibromyalgia. And all of these things are interrelated, because — yes, I'm supposed to be an expert on mold toxicity, and I probably am — but mold toxicity doesn't exist in a vacuum. You have mold toxicity as one of multiple environmental toxins we're being exposed to. It is simply one of the most common, the easiest one to diagnose, because we have tests for it. We don't have tests for the hundreds of thousands of chemicals out there. We're just beginning to be able to have some tests that will tell us what's in someone's body that might be making them sick.Courtney: Right, right. And I would imagine that this typical medical model of looking for one cause to autism, one cause to dementia — it's a completely different way of thinking. When you get away from that and you look at complexity of the human body, that way of thinking holds back the research, it holds back any progress.Neil: What I think is helpful for people to look at is: we are all uniquely, biochemically, genetically different. And so the same inflammation in one person could trigger cardiovascular disease, and another person could trigger Alzheimer's, and another person could trigger chronic fatigue. So we need to take a step back and go, okay — and this is not a brand new idea. For the last ten years, there are many papers in the medical journals that are saying chronic illness is almost certainly an inflammatory process. Now, it hasn't really integrated into what people are being taught in medical school or in their residency program. But this is not a brand new idea, and there's tons of medical research documenting that.What behooves us as physicians is to look at every human being and go, “Okay, this is your named chronic condition — what are the triggers for you?” We have to kind of be like a medical detective and dig in and go, okay, what's triggering your inflammation, so I can treat the cause — not put a band-aid on it, which is, forgive me, in the psychiatric field, taking antidepressants and benzodiazepines. That's a band-aid. It's a helpful band-aid, it could be a very important band-aid, help someone to function. But you really need to be looking at, okay, I have this great band-aid, but wouldn't it be nice to actually cure this? And I think that would be, for me, to your audience, Courtney, my take-home message — which is always look for the root cause, because it is possible that we will find that root cause, and you won't have whatever name you've been given, depression, anxiety, OCD, for the rest of your earthly life. Maybe.And I've treated thousands of people successfully here. So this is not — I've had one or two successes here — we're talking thousands of people that have responded to this way of practicing medicine, with fabulous results. People who've been literally bedridden, incapacitated by their illness, are now out there working again and being with their family and living full lives. So that's the take-home message.Courtney: Right. Which makes it so satisfying to treat. And I mean, I can speak to that, having experienced it personally. But at the same time, you do get pushback from other areas of medicine that don't give it necessarily credibility, even though people, on the other side, are thriving and healing, and it's really amazing.New Medical Ideas & Research Take Decades to Reach Patients19:30Neil: You know, the history of all science, including medicine, is: new technology, new drugs are fast-tracked to get into our lives. Ideas, not so much. Typically it takes a generation — twenty, twenty-five years — for a new idea to come into acceptance. And the first reaction of any science, medicine included, is to get into denial, which is, “There isn't enough data to support this concept, and I can't be perceived as practicing bad medicine by embracing a concept that isn't proven yet.” But that means that all of their patients are going to have to wait twenty years — until suffering — until that person goes, “Now there's enough evidence, now I can learn how to do this, and now I'll begin to help you.” I'm not wired that way. I'm wired that if I have a suffering person, and I have something that I can offer them that might help, I'm going to try it. I'm going to do it in the safest way I know how, but I can't leave someone dangling for twenty years and say, “I'm sorry, I have to wait until my profession figures out what to do.” That's not me.I worked at the medical school at the University of Minnesota Duluth for eleven years. And I was not only on the faculty, but I was on the committee that determined what new information was put into the curriculum. For example, AIDS was just coming into vogue at that particular point, and it was obvious that we needed to have courses on AIDS for the medical students, because this was big. What a lot of people wouldn't understand is that each department in a medical school fiercely guards its time allotment. Everyone gets a certain number of hours, and they're not open to adding new things or giving up any of their time slots for anything new. And so what I would watch is, as new information came into the medical school, each department fiercely refused to add any new lectures in their department, or give up any of the time that they had to anybody else to add that information. So we're talking about university politics now — this is not even medicine, this is people guarding their turf. And I don't even think that would occur to any consumers, that that would happen like that — well, certainly, why would that happen in a medical school? Well, it's because human beings are responsible for each department, and they guard their own turf. So it's a kind of an insidious process about how new information isn't embraced by the profession, to go into the medical school curriculum.Mold, Lyme, and Long-COVID: The “Big Three”23:15Courtney: I want to come back to what you were saying about inflammation being at the core of chronic illness and complex chronic illness. And when it comes to complex chronic illness, whether it's SIRS or mast cell activation from mold or Lyme, we talk about multiple systems being affected — the central nervous system, gastrointestinal tract, endocrine, skin, just you name it. If the inflammation then is from a source of oxidative stress or a major insult — would you say that mold toxicity is the most common, or would you put Lyme or now even COVID on a par with that, as far as the deepest roots causing the inflammation? Not that the chemicals, metals, and everything else we're being exposed to aren't at play.Neil: In my experience, those three are the biggies — mold toxicity, Lyme with its co-infections, and long-haul COVID. Those are the three most common. Of those, in my experience, mold is the most common, and the one that has to be treated first if you have some or all of them. So, for example, in people who have long-haul COVID or Lyme, often their body will not respond to that treatment optimally until you first get the mold out. So there's a little hierarchy of what we do, in what order. And then within that hierarchy, there's also an order in which we do things for people to respond optimally. So, for example, mold toxicity will trigger — and Lyme and Bartonella will do the same thing — limbic dysfunction, vagal nerve dysfunction, and mast cell activation in most of our patients. Not all, but in most. And the longer they have it, the more likely those things are going to get triggered. Once somebody does not feel safe — and the systems that we have in our body to monitor that safety and to keep us safe are the limbic system, the vagal system, and mast cell activation — we have to get the body safer, or it will not be able to take what you need to take to treat mold or Lyme or long-haul COVID. People will literally shut down — their limbic system will essentially go, “Mm, I see what you're trying to do there, and I can't let you do it, because I'm not sure that's safe.” And it will literally not allow people to do it.The Limbic-Vagal-Mast Cell “Trifecta”25:58Courtney: So with one of those, or a combination of those, on board, these threat mechanisms would be kicked up — mast cell activation, limbic system dysfunction, autonomic nervous system dysfunction. If they look similar, or a timeline isn't easily helping parse out what might be primary, how able do you feel to make the distinction — versus if mold is present, starting there?Neil: If we look at our patient's symptoms, they will point us clearly to whether there's a limbic or vagal or mast cell piece, to be specific. The limbic system's job involves primarily monitoring and regulating and keeping emotion and sensitivity in check. So any symptoms that involve those two things are limbic almost by definition. So on the sensitivity side, if someone describes an increased sensitivity to light, sound, touch, smell, chemicals, EMFs, food — they're already telling me my limbic system is dysfunctional. In some ways it doesn't matter what the cause is; if someone has this increased sensitivity, regardless of the cause, I know we have to treat their limbic system first.On the vagal side, the symptoms would primarily run into symptoms that involve the autonomic nervous system, which would involve things like temperature dysregulation, palpitations, insomnia, POTS, blood pressure dysregulation, or symptoms that involve the gastrointestinal tract, because the vagus nerve controls almost the entire intestinal tract. So any GI symptoms — gas, bloating, distension, reflux, diarrhea, constipation, abdominal pain — again, that would lead me to go, the vagus is involved here. So by listening to my patient's symptoms, I can immediately know what's dysregulated here, and what my first treatment arm will be — to get the limbic and vagal systems back up and running again at a safer level, because otherwise our patients aren't going anywhere. They can do binders and antifungals and antibiotics for Lyme, and they will get worse. Their body will basically go, “I know I might need that, but why are you doing that? I can't even get to a place of safety to respond. I'm on self-preservation mode here.”And in the mast cell category — again, all of the same symptoms that could be mold or Lyme or long-haul COVID could also be mast cell. Again, it's a multi-systemic process. A tip-off, which I find very helpful, is if someone describes a symptom that comes on immediately after eating, sometimes while eating. Nothing causes that kind of reaction that fast except mast cell activation. Allergy — and most people, if they do have some symptom that comes after eating, they're going to be thinking, “It must be what I ate, what did I just eat?” But it's not food allergy, it's mast cell activation. In that context, many of our patients have already observed that they can eat a particular food one day and have a very strong reaction, and the next day, same food, nothing. And they'll go, “That doesn't make sense — if this was allergy, I ought to react the same way every time.” And so that is classical mast cell activation, which fluctuates. So — for the first part of your question — that's what I'm looking for. When I take a history, when I talk with patients, I'm going to really dig into: are they already manifesting symptoms that clearly tell me what we need to do first, to get them safer, so that now they can respond properly to the treatment that will really get at their root cause.Are Some People Born More Vulnerable?30:31Courtney: I know you take deep timelines when you meet with someone, so you're really accounting for a lot. But one of the things that I think about a lot is: who is most vulnerable? Do some of us come into the world already with a vulnerability similar to Elaine Aron's highly sensitive person model? And then maybe there's trauma, early attachment disruption — and already vulnerabilities — but then the toxic load that we're all experiencing. But then something like mold or Lyme then hits. And then, all the years and all the patients you've seen — I'm curious how many you feel already had sort of a vulnerability. Not that they ever had to really become sick necessarily, but already had a vulnerability.Neil: Honestly, I would say most. Our limbic system evolves from the time we were in our mother's uterus, so that almost no one has had a perfect childhood. And so whatever someone has had to deal with in childhood — it could be recurrent ear infections or throat infections, or it could be a surgical procedure, or it could be parents, both of whom worked and they didn't get enough time, or they could be abusive parents, sexually, physically, verbally abusive — whatever someone has been exposed to, that forces their limbic system to have to deal with it. Because that's the job of the limbic system, is to keep you safe. So your limbic system is basically going, “Okay, I'm just a little thing here, what do I need to do to cope with this?” And so their limbic system slowly, inexorably, becomes more and more hypervigilant to protect them. Let's say someone had an alcoholic parent, and they would come home, and you were never sure what their status would be — and so you'd kind of hold yourself back from that, which is, “I've got to see what this person is showing me, is it safe for me to come up and give them a hug, or do I want to hang out in my room for a bit until whatever this is settles down?” That's the beginning of this limbic hypervigilance process. And then it just goes on through life. So that if you've had difficult relationships, or surgeries, or severe infections, or anything in the physical or emotional or spiritual plane, a betrayal of some type — the limbic system becomes slowly more and more and more hypervigilant, when the straw hits the camel's back. And that straw could be COVID, or Lyme, or mold toxicity, or another infection of some type — that's the straw that breaks the camel's back. At that point the limbic system goes into shutdown, which — we know the vagal system goes into shutdown, literally, that's something the vagal system is trained to do. It's “okay, we are so unsafe that I'm shutting down my systems here.” And so that predisposes to illness. But I want to tie that back to inflammation, which — a lot of people don't understand that the vagus nerve, for example, controls inflammation in the body also. There are branches of the vagus that go to the spleen, to the gut-associated lymphoid tissue, and to the thymus, all of which are major immune organs. And if the vagus is dysfunctional, inflammation can rage out of control. So again, this comes back to understanding this complexity of inflammation, and how we need to both understand it and know where it is we want to look to get at that inflammation. So there is a profound interconnected limbic-vagal-mast cell piece. Each totally connects to the other. There's an interaction between the thousand biochemical mediators that mast cells make and the nervous system itself. So I call this interconnection the trifecta, which is: you can't just treat the limbic system in a vacuum, because if you're not also looking at the vagal and mast cell system, your treatment may be a little bit helpful, or it might not even work at all, until you combine all three — so that you really understand this is a package deal here, in terms of how we work with it.There was one more piece I wanted to add, Courtney, to understanding the inflammation piece: whether or not someone gets mold toxicity or Lyme or COVID depends on how robust their immune system is. So you could have a family living in a moldy environment — one person is really sick, several are a little bit sick, and one is not sick at all. And the one who's not sick at all can easily go, “I'm not sure what's wrong with you all, but I'm fine in this environment, it can't be my environment because I'm fine.” And what they're not understanding is: yes, they are fine, as long as their immune system stays robust. For the others, they were at risk for all of these conditions because their immune system took a hit. And for many people who were living in a moldy environment, during COVID, for example, when we isolated and people were spending twenty-four hours in their home rather than the eight hours out, they were many now being exposed to more mold at home than they were before. I don't think people began to think about that or look at that. So in that environment, whether or not they were affected depends on the hit they took. Now, the things that will hit an immune system to weaken it, and allow whatever's there to manifest, are: COVID was a biggie, any severe infection, a surgical procedure, childbirth, menopause, or any emotional upheaval will weaken that immune system, and then the immune system loses containment, and now we are off to the races. So what I try to convince people who are living in a moldy environment who are well is that they're living with a ticking time bomb. Should they take an immune hit, they will be just as sick as their family member who they're kind of lording it over, which is, “I'm not sick, what's wrong with you?” You'll be right there yourself if you take that hit — to try to help them understand, yeah, you're okay for now, but once your immune system weakens. So I'm trying to tie together the immune system, its ability to regulate inflammation, and this whole process here.Courtney: Right, right. Which I guess is why we call it complex, complex illness.Neil: It is the way human beings are. We're not making up a new word of complex illness — illness was always complex. We're just beginning to understand how complex it always has been.The Limits of Treating Symptoms Alone44:39Courtney: Right, so there's communities out there addressing mast cell activation. There's communities out there addressing POTS and dysautonomia. Communities out there addressing OCD and more obvious limbic-type issues. And then there's the whole mental health field. And the dots aren't getting connected. I mean, I've yet to see someone with OCD — I think it's like eighty to ninety-five percent have mold toxicity as part of the picture.Neil: You know, an example — and I won't name names — is there's an eminent pediatric physician who is very well known for his work in autism, who thought he was ADD his whole life until he recognized that he had mold toxicity and started taking antifungals, and all of a sudden his brain went online and he doesn't have it anymore. So that's just a very clear example of what you're talking about. And what we are talking about is: it's absolutely essential that all people who work in the medical field, all healthcare providers, embrace this complexity, because if you're only working on a piece of it, you will help some people to a certain extent, but you will not help people optimally. I mean, that is the absolute bottom line.The understanding that mast cell activation was huge came in 2016, when Larry Afrin wrote his book, Never Bet Against Occam. It was a huge game changer. When I read his book, it was like, “My goodness, I've been missing this in my patients.” And the whole world embraced this information, so that every medical center in the country now has a mast cell activation clinic. However, they're looking at it as a single thing with a label to it, and they're treating it just that way. They have not recognized that if they don't add limbic and vagal work to it, their treatment is not going to be adequate. And so, yes, they're helping people, but absolutely not optimally. And to make it worse, very few of those clinics have recognized that what's causing mast cell activation, which absolutely needs to be treated, is mold toxicity and Lyme. So people go to these clinics and get a certain amount of help, but they're not being treated comprehensively, which is a pet peeve of mine — which is, come on, if you're going to have a clinic, understand the whole interactions of whatever name it is you're working with. If you're going to be an ENT specialist, understand everything that impacts that area. If you're going to be a cardiologist, understand everything that impacts it. And we just don't see that. We see people kind of in their little box — “No, no, I've learned this, this is what I do, this is how I work” — and it's just unfortunate that they're not looking at the bigger picture.Courtney: Right, I think add to that PANS and PANDAS, because in psychiatry, people feel like they're deep diving because they're getting to PANS and PANDAS. It's like, okay, you've got to go deeper than that, because even those conferences, they talk about the need for treating relapse, and it's just going to keep reoccurring.Neil: I mean, they're using IVIG to help. That's nice, but they're not getting to cause. And we know when PANS and PANDAS first emerged on the scene, it was all about strep, and that was the focus for a long time. But as time emerged, it was very clear that this was a neurological inflammation triggered — here we go, broken record — Lyme, mold, other infectious agents had to be looked for. And if we treated those, you could cure PANS. It wasn't something that needed IVIG for the rest of your earthly life.Courtney: Even among those, would you say very often mold is underlying the mycoplasma, the candida?Neil: I think mold is one of the most missed components to illness that I can think of. If I have a goal in life, it would be to raise the consciousness of our profession as to the importance of mold toxicity, how common it is. We're talking millions and millions of people who are not diagnosed, because their doctors don't know about it, and therefore are not making the diagnosis. I'd love to see the consciousness get to the point that someone would present with these complicated stories and a doctor would say, “Why don't we think about mold toxicity?” That would be the most important thing I would have done in my career.Courtney: Yeah, well, you're certainly raising awareness.Neil: I'm working — it is slow. As we said, I'm working on a twenty-year generational issue here.Courtney: You've mentioned that the underlying source needs to be addressed, but the nervous system and immune systems need to be addressed. So one of the things that comes up with limbic system retraining is: I've had people hear the message that if they just address their limbic and autonomic nervous system, then they wouldn't need to address what I see as an ongoing threat, such as a mold toxin. So I'm hoping you can speak to that, and the importance of these programs, despite that.Neil: Sure. A couple of the better-known limbic retraining programs, the coaches get a little bit zealous about — “limbic retraining is so important, this is all you need to do alone, don't even do anything else, just do limbic retraining.” And I suspect that there are a handful, very few, but a handful of people who've gotten well just doing limbic retraining. If, for example, if you had mold toxicity, if it was relatively new, if you had not colonized, and you moved out of your mold exposure soon, it is possible that the limbic retraining would help reboot it to the point that your immune system rebooted itself and you'd be fine. I believe there are a few people like that. The vast majority of people who have limbic dysfunction — you need to do limbic retraining and vagal retraining. You may also need to do mast cell retraining, and please look for the cause of that. Those are downstream effects, those are not named diagnoses that you treat alone and it fixes it. Please look for at least mold and Lyme, which are the two most common things that are triggering this at this point. And people are getting the wrong message if they think they need to do it in a vacuum. To make it worse, they're being told, “No, no, just do limbic retraining, that's all you need.” And the truth is, no, you need to combine limbic retraining and vagal rebooting and mast cell activation most of the time. And of course you've got to get back to cause.Courtney: Right, I mean I try to liken it to — if they were in an abusive relationship or toxic relationship, they wouldn't just limbic-system their way out of that, if there was an external threat.Biochemical Contributors47:35Neil: No, you've got to fix what's primary here. I want to add to this discussion — we're focusing on inflammation from toxins and infections, but there are also other biochemical issues, some of them downstream, that need to be looked for and addressed in order for people to recover completely. So, for example, Lyme and mold will trigger kryptopyrrole issues, or imbalances in the ratio between zinc and copper, or they'll trigger methylation issues super commonly. And I just wanted to expand this discussion — I know it's complicated enough, but I don't want to oversimplify it either — that there are other biochemical things that we want to be looking for, because they're treatable, and they will help people to heal faster, if we're also addressing methylation, the need for zinc, the need for magnesium or B6 — those are common components of this whole process here. And so I just want to kind of tie that all together for a moment.Courtney: No, I'm glad you did, because those are topics that I talk about here, so it's important to connect those. The other thing I see, because I integrate the Walsh approaches with treating people that have complex health issues, is zinc seems so amazing when it comes to mast cell activation — making sure that's optimized. As well as connective tissue — and that was something else I wanted to ask you about — is structural issues that can be impacting the autonomic nervous system. I just see a lot of people — I inquire about it because I had it myself — but who have upper cervical instability as part of this whole picture.Structural Contributors49:26Neil: Again, all of these things are interrelated, and the physical structure can prevent people from moving forward in several different ways. One is the structure of the jaw and the face — the body puts a priority on teeth occluding properly. And if the teeth don't meet just quite right, and the jaw is a little bit out of structure, people often talk about TMJ — the body basically freaks out, it goes into sympathetic overdrive. And for some patients — it's not common, but I've seen several dozen — where the jaw had to be realigned by a combination of dentists specially trained in this work, working with osteopathic physicians, to realign the jaw. Often in those patients, they couldn't respond to the limbic and vagal treatment until their jaw got realigned.Same thing is true with what you're talking about, as cervical cranial instability, which is basically a weakening of the ligaments at the base of the skull, where it attaches to the first cervical vertebra. Now, these are all again intertwined — for example, it begs the question of what weakens those ligaments. And the answer is: if you have mast cell activation, some of the materials that the mast cell makes weakens the ligaments. So the ligaments get weak, and where the skull attaches to the first cervical vertebra, instead of being pulled apart, like ligaments do, it collapses. And that influences the blood flow to the brain, the nerve flow to the brain. And again, the body recognizes that as a priority, and often you can't get that better — you can't get limbic and vagal response until that gets fixed.So there are a bunch of structural issues that also need to be looked at, when people become overly sensitive and are not responding properly. And there's more in that area. So, for example, there's a chapter by Tasha Turzo, who's an expert on the jaw structure. There's a chapter in there by Andy Maxwell on cervical cranial instability. So that book would give people a really good overview of how complicated things are, and they might, chapter by chapter, get a clue of, “I haven't looked at this piece yet.” I mean, we haven't talked about oxalate or salicylate imbalances, which can add to that. So I just think that is a resource for folks that could really help them understand how they've gotten where they are, and understand the path out.Courtney: Right, right, because not everyone has all of these areas, but certainly could be held back or have a unique something that they could be getting relief from early on in the process. So I think it's both, to the degree that people are able to learn, and your book Toxic, too — it's like an encyclopedia, even if someone doesn't read straight through it, they can go to the mast cell section, or they can go to the Bartonella section.Neil: It was intended to be a readable way to understand this complexity, because it basically will talk about almost everything we've talked about today, and help people to get better understanding. I mean, we've talked about the bigger picture today. We didn't talk about, okay, how do you diagnose Lyme, how do you diagnose mold toxicity, how do you diagnose long-haul COVID, and most important, because this is very complicated, how do you decide which of those is the biggest player that you want to address first?Distinguishing Between Mold, Lyme & Long-COVID53:58Courtney: Right, I have shared resources on diagnosing mold toxicity. Would you comment on your last point — how do you recognize, are you using specific testing that you want to reference, the RealTime?Neil: I do. But I would emphasize, we're taught in medical school that if you really listen to patients, ninety-five percent of the time you will make the diagnosis by history alone. And I don't want to get too focused on testing, because listening to the patient and having them describe in detail what they're going through will really give you the little pieces you need to tease it apart to a certain degree. That's only possible to a certain degree, because there's a huge overlap between the symptoms of Lyme and Bartonella and mold and long-haul COVID and mast cell activation. Huge overlap. So there's little teeny pieces that will tend to lean more in one way than another. And honestly, that's what's required from all of us, literally years of study, to really get a handle on — someone will say something, okay, that's much more of a mold symptom than it is a Lyme symptom, something else. So I do encourage people, if they think they might have mold, to get a urine mycotoxin test. That has revolutionized our ability to make these diagnoses.Courtney: Would you still set RealTime as the preferred? That's what I've communicated to this audience.Neil: I would. Any test can help make the diagnosis, but for follow-up testing, the only one that's been really consistent is RealTime. And you want a follow-up test, so you want to know what your baseline starting is, and then you want to know, okay, am I getting these toxins out of my body? From my perspective, you know that you're done treating it when that RealTime comes back reading not present in every category. We have better tests now for Lyme than we've had in the past. Years ago, our tests were inadequate to say the least. And the CDC recognized years ago that the tests were so inadequate that it was basically a diagnosis made by a physician by examining all of the history and information that you had. That was enough — you didn't need a lab report to make the diagnosis of Lyme disease. A new test that I'm particularly fond of is Bruce Patterson's fourteen-panel cytokine test. This is a test that measures cytokines, which are the molecules of inflammation — cytokines are made by your immune system to deal with, or fight, different inflammational causes. And so what Dr. Patterson has discovered is there's a pattern of cytokines that we see with long-haul COVID, which is different than the pattern we see for Lyme. And I'm working with him now on the pattern for mold. So we can literally look at that test and look at, okay, what is inflaming this person primarily now, because I'm looking at that inflammational pattern, and I can literally read that. Some of my patients will have all of those on a particular test, or only one, but it really helps tease apart what is — I call it public enemy number one for my patient, what do I need to address as root cause right now. So our science is improving as we go here.A Message of Hope — and Where to Learn More58:49Courtney: And that's huge. Before we go — I think all of your books, but I'm thinking specifically about Toxic and The Sensitive Patient — all the information you generously share gives people a lot of hope who've been struggling out there. And I think your mentoring and teaching so many of us helps extend some of that reach. Is there anything you'd want to convey to people out there in the throes of chronic illness who are feeling hopeless and not getting the feedback from their doctors that this is something they can get beyond?Neil: Sure. My take-home message is always: everything we talked about today is treatable. Is there hope in there? You bet there is. But if you are not getting the help you need from whoever you are seeing, find someone who understands these things. For example, I do have on my website a list of physicians that I've trained over the years that I'm comfortable that they really know what they're doing — it's a growing list. There's someone like Courtney, who's been trained for years to do this, she's good at it. Find someone who knows what they're talking about, so that you can get someone who does embrace this complexity, and will look at it in its entirety, and then get you moving in the right direction, doing the right things in the right order. To me, this can be done. I mean, I have personally helped to cure four or five thousand people with mold toxicity, and an equal number of people with Lyme disease. So we can help the vast majority of people that we're seeing. So, regardless of how long you've been sick, if you haven't been properly diagnosed and you haven't gotten the right treatment, you can be helped.Courtney: Right, right, that's great. And for physicians out there who are interested in learning more about complex chronic illness — are there resources you would point to? I'll be sharing your books, because I think those are great references — but also how people can find what you're doing currently.Neil: Sure, my books — particularly with this discussion, Toxic and The Sensitive Patient's Healing Guide would be the two most appropriate. I would add that I have just finished writing a book on this whole subject of inflammation. It's currently being edited, I hope it'll be out by early next year. So if you stay tuned to my website, I'll let people know when that's going to be available. And I think, Courtney, what you're referring to is: I do have a mentorship program for healthcare providers. We now have three hundred and fifty-plus people in the mentorship program, it keeps growing. You were in one of the first groups — we started with eight, and then people kept wanting to get in on it. So we welcome people to join. If you go to my website, which is simply neilnathanmd.com, there's a whole thing which talks about the mentorship and how to sign up for it. So I certainly welcome people who want to learn more.Courtney: And then you offer consultations and professional consultations also?Neil: I do. My consultations — I am, at this point, since I'm semi-retired, not seeing people directly. But I do offer consultations with a patient and their primary treating physician — so we all get on a Zoom together, and we will go over the details of that person's history and their lab work, and I will help outline a treatment program that looks to me like it will get people well. And actually, Courtney, I actually do a lot of that — I'm not really retired, my wife will tell you. I stay pretty busy. I love helping people. I love being able to take this complicated information and tease it apart, so that I can help people to understand what they need to do, in what order, in order to get better.Courtney: And we're all grateful — grateful that you've followed your path, because so many people have benefited personally and professionally. So thank you for bringing your wisdom and expertise, really appreciate it.Neil: You're very welcome, happy to do that.You can find Dr. Nathan's information, consultations, and mentoring program through his website at neilnathanmd.com. If you'd like to help get this information out into the world, please consider liking, sharing, or commenting.Until next time,CourtneyCourtney Snyder MDDr. Courtney Snyder MD is a holistic, functional, and environmental psychiatrist for children and adults. Find her at courtneysnydermd.com and on the Holistic Psychiatry Podcast on all major platforms.Medical DisclaimerThis podcast is for educational purposes only and is not intended or implied to be a substitute for professional medical advice, diagnosis, or treatment. Consult your own physician for any medical issues you may be having. This is a public episode. If you would like to discuss this with other subscribers or get access to bonus episodes, visit courtneysnydermd.substack.com
Beat writer for the Detroit Pistons Hunter Patterson breaks down his view on the Jalen Duren situation and more on Detroit's offseason. Hosted by Simplecast, an AdsWizz company. See https://pcm.adswizz.com for information about our collection and use of personal data for advertising.
This week, J.John speaks with Kate Patterson, author of Ashes to Crowns, which looks at her journey through grief and the hope she discovered in the midst of loss. Drawing on the promise of Isaiah 61:3, Kate reflects on God's faithfulness through suffering and how she learnt to grieve with hope.
This week, Tee is joined by Dr. Jaquel Patterson, an internationally recognized naturopathic physician, 3x Amazon bestselling author, and Forbes contributor. As CEO of Fairfield Family Health, Dr. Jaquel brings over 16 years of clinical expertise treating Lyme disease, autoimmune conditions, ADD/ADHD, PANS/PANDAS, and environmental illness. She's been named the top naturopathic physician in Fairfield County for six consecutive years, has been featured in USA Today, Forbes, and on CBS, Fox, and NBC, and serves as Treasurer of the American Academy of Environmental Medicine. In this episode, Dr. Jaquel unpacks lifestyle medicine and proactive prevention as the true foundation of long-term wellness, rather than chasing symptoms after they appear. She explains how simple, intentional daily habits can help prevent chronic disease and bring the body back into balance. Tee and Dr. Jaquel discuss how these principles apply not just to adults, but to protecting the whole family's health. The conversation also dives into one of Tee's favorite topics: how modern environmental stressors quietly undermine our health over time. Dr. Jaquel draws on her background in environmental medicine to share how families can build resilience against these everyday exposures through proactive, root-cause care. Whether you're managing a chronic condition or simply want to future-proof your family's health, this episode offers a practical foundation to build from. Connect with Dr. Jaquel: Website LinkedIn Facebook Instagram Phone: (908) 581-8181 Follow Therese "Tee" Forton-Barnes and The Green Living Gurus: The Green Living Gurus Website Instagram YouTube Facebook Healthy Living Group on Facebook Austin Air Purifiers: For podcast listeners, take 15% off any Austin Air product; please email Tee@thegreenlivinggurus.com and mention that you want to buy a product and would like the discount. See all products here: Austin Air Tip the podcaster! Support Tee and the endless information that she provides: Patreon Venmo: @Therese-Forton-Barnes last four digits of her cell are 8868 For further info, contact Tee: Email: Tee@thegreenlivinggurus.com Cell: 716-868-8868 DISCLAIMER: ALL INFORMATION PROVIDED HERE IS GENERAL GUIDANCE AND NOT MEANT TO BE USED FOR INDIVIDUAL TREATMENT. PLEASE CONTACT YOUR PROVIDER OR DOCTOR FOR MEDICAL ADVICE. Produced By: Social Chameleon
Today, we are sitting back down with Janci Patterson to discuss the new Cytoverse novel, Blightfall! We have Eric (Chaos), Ian (Weiry), Jessie (Lady Lameness), Will (#1 Taln Fan), and of course, Janci herself! 0:00:00 Intros 0:01:38 Non-spoiler section 0:09:15 Spoiler section If you like our content, support us on Patreon: https://www.patreon.com/17thshard Purchase merch here! https://store.17thshard.com/ For discussion, theories, games, and news, come to https://www.17thshard.com Come talk with us and the community on the 17th Shard Discord: https://discord.gg/17thshard Want to learn more about the cosmere and more? The Coppermind Wiki is where it's at: https://coppermind.net Read all Words of Brandon on Arcanum: https://wob.coppermind.net Subscribe to Shardcast: http://feeds.soundcloud.com/users/soundcloud:users:102123174/sounds.rss Send your Who's That Cosmere Characters to wtcc@17thshard.com
Cory Brada, Jovon Johnson, Kyle Spence, and Coach Don Patterson react to the Hawkeyes' nail-biting win over ISU.Watch 'FARMS OF AMERICA' series (Seasons 1 & 2) FREE: https://youtube.com/playlist?list=PLPWrfk1hFOt9JY8IwMOzixSweNKgzsh3n&si=YzOShNXFijPR2Ep5IOWA SMOKEHOUSE - "TASTING IS BELIEVING": https://www.iowasmokehouse.com/Big Grove Brewery is the Official Craft Brewery of the Hawkeyes - Locations in Iowa City, Des Moines, Cedar Rapids, and Solon! - https://biggrove.comBrad Van Meter (State Farm Insurance) - GET YOUR FREE QUOTE TODAY! - https://bradvanmeter.com/Iowa Floor Covering - https://www.IowaFloorCovering.com/ - DO IT YOURSELF FLOORING by IFC in Bondurant, IAJOIN OUR DISCORD SERVER! (use any of the links below) - Cash App: https://cash.app/$HawkeyeoftheStormVenmo: https://venmo.com/HawkeyeoftheStormPayPal or credit card: https://www.paypal.com/donate/?hosted_button_id=499R9YWL5YGEG
Most families don't know what skilled nursing actually is until the moment they desperately need it — and by then, the system feels impossible to navigate. Joy Patterson has spent 25 years inside that system. She's here to change what you think you know about it.In this episode, Jamie sits down with Joy Patterson, VP of Skilled Nursing Facilities at Your Health, for a straight-talking conversation about what skilled care really looks like, why it's constantly misunderstood, and what separates facilities that deliver exceptional outcomes from the ones that fall short.Key topics covered:Why "we have great relationships" no longer wins hospital referrals — and what C-suite administrators are actually asking forHow avoidable hospitalizations happen, and the clinical interventions that prevent them before a cough becomes pneumoniaThe staffing crisis hitting skilled nursing hardest, and how embedded partnerships change the equationWhat value-based care actually means at the bedside — and why it has nothing to do with cutting cornersThe questions every skilled nursing administrator should be asking a physician partner (but mostly aren't)Why physical presence inside a facility — not remote support — is the single biggest factor in better outcomes for residents and familiesWhether you're a healthcare professional, a family member navigating a loved one's transition, or a Your Health team member building fluency in the landscape — this episode gives you a clearer picture of what excellent skilled care looks like, and why it matters more right now than it ever has before. www.YourHealth.Org
Cory Brada, Jacob Gill, Kyle Spence, and Coach Don Patterson recap Iowa Football's season-opening win over the NIU Huskies.Watch 'FARMS OF AMERICA' series (Seasons 1 & 2) FREE: https://youtube.com/playlist?list=PLPWrfk1hFOt9JY8IwMOzixSweNKgzsh3n&si=YzOShNXFijPR2Ep5IOWA SMOKEHOUSE - "TASTING IS BELIEVING": https://www.iowasmokehouse.com/Big Grove Brewery is the Official Craft Brewery of the Hawkeyes - Locations in Iowa City, Des Moines, Cedar Rapids, and Solon! - https://biggrove.comBrad Van Meter (State Farm Insurance) - GET YOUR FREE QUOTE TODAY! - https://bradvanmeter.com/Iowa Floor Covering - https://www.IowaFloorCovering.com/ - DO IT YOURSELF FLOORING by IFC in Bondurant, IAJOIN OUR DISCORD SERVER! (use any of the links below) - Cash App: https://cash.app/$HawkeyeoftheStormVenmo: https://venmo.com/HawkeyeoftheStormPayPal or credit card: https://www.paypal.com/donate/?hosted_button_id=499R9YWL5YGEG
My interview with Richard North Patterson, author of "Tripwires: 15 Twenty-First-Century Events That Undermined American Democracy―and How to Reclaim It"
Acclaimed Morgan State University professor Ray Winbush joins our classroom. Dr. Winbush will deliver powerful insights into the Democratic Party’s complex relationship with its progressive wing and the critical importance of appealing to Black voters. He’ll also break down the high-stakes meeting between Hakeem Jeffries and Jared Kushner and offer an essential update on reparations progress since the historic Durban Convention 25 years ago. You’ll also hear from WDC activist and holistic health practitioner Dr. Kokayi Patterson, who will preview a pivotal meeting designed to empower Black men facing emotional challenges. Plus, Ghana’s Director of the World Conference of Mayors, Francis Opai Tetteh, will launch the program with actionable guidance on fresh opportunities for African Americans in Ghana.See omnystudio.com/listener for privacy information.
What happens when the practice you built is forced to stand without you?In this powerful reunion episode, Michael reconnects with Dr. Ashley Joves nearly 10 years after her startup journey began with Smile & Co still in the dream stage. Ashley reflects on how her definition of success has changed from building a standout dental brand, hosting events, creating unforgettable patient experiences, and growing a loyal community, to recognizing the person she has become through the hard seasons of business, marriage, motherhood, and leadership.Ashley also opens up about the health crisis that reshaped everything: discovering an oral cancer lesion on her husband, Brian, after what first seemed like a simple tongue irritation. She shares how self-advocacy, a second biopsy, and trusting her instincts made all the difference, then explains how her team, systems, and culture kept the practice running when she had to step away. This conversation is a deeply honest look at ambition, resilience, leadership, and building a dental practice that supports your life instead of consuming it.What You'll Learn in This Episode:Why Ashley's definition of success has shifted after nearly a decade in practice ownership.How a missed oral cancer diagnosis reinforced the importance of screenings and patient advocacy.What practice owners can learn from building systems before a crisis hits.How strong team leads, associates, and clear roles can protect the patient experience.Why culture is built through hiring, boundaries, and difficult conversations.The “low-cost” loyalty driver that matters more than decor or amenities.How to rethink growth when bigger no longer feels better.Why dentistry should fund your life, not become your entire life.How to create a backup plan that gives you more freedom and flexibility.Why finding a supportive dental community can make ownership feel less isolating.Listen now for an honest, grounding conversation on building a practice, protecting your people, and redefining success on your own terms.Sponsors:Net32: Founded by a dentist, for dentists. Net32 is the leading online marketplace for dental supplies, helping dental and medical professionals save on high-quality products for over 25 years. Start saving today at: https://www.net32.com/dentalmarketerClick here for a special offer!Guest: Dr. Ashley JovesCheck out Ashley's Media:Ashley's Instagram: https://www.instagram.com/ashleyjovesdds/Smile & Co. Instagram: https://www.instagram.com/smileandcompany/The Making Of Instagram: https://www.instagram.com/themaking.of/Facebook Community: https://www.facebook.com/groups/themakingofPodcast "The Making of a Dental Startup": https://pod.link/1474094399Practice "Smile & Co. (locations in Folsom, CA and Roseville, CA)": https://www.smileandcompany.com/Mentions & Links:People:Alex Hormozi (entrepreneur/podcaster referenced for insights on success and reps)Organizations, Products & ToolsNet32 (dental supply ordering platform)Patterson (dental supply company)Henry Schein (dental supply company)Studio 8E8DisneyHost: Michael AriasJoin my newsletter: https://thedentalmarketer.lpages.co/newsletter/Join this podcast's Facebook Group: The Dental Marketer SocietyLove the Podcast? Follow on Your Favorite App! https://lnkfi.re/TDMPod
In this episode, Kelly invites Connor Patterson back to continue our series on Church History. This time, Connor looks at the Council of Constantinople. In doing so, Connor leads us in a study that answers the most important question of all: “Who is Jesus?”www.instagram.com/thehishillpodcast/www.hishill.orgkelly@hishill.org
Hunter Patterson of The Athletic joined D-Lo to talk Jalen Duren and Pistons.
Mark is joined by Duane Patterson, producer of The Hugh Hewitt Show, host of the Duane's World Podcast, and contributor to Hot Air, for his signature fast-paced analysis. The two discuss recent polling trends in major political races, high-profile legal updates involving Tiger Woods, national media coverage, and the political dynamics surrounding key Senate and House races across the country.
In terms of criminal jury trials, what unfolded in the infamous death cap mushroom case was unique.Because when the jury was locked away from the outside world to consider a verdict, they were staying in the same hotel, in the same country town, as the police who investigated the case, the lawyers who prosecuted, and the media reporting on it. This strange arrangement has opened the case to suggestions that the jury was tainted – a central tenet of Erin Patterson’s appeal against her conviction. Patterson was sentenced to life imprisonment last year, with a non-parole period of 33 years, for serving a poisoned beef Wellington lunch to her in-laws Don and Gail Patterson, Gail’s sister Heather Wilkinson and her Baptist pastor husband Ian Wilkinson. Today, crime and justice reporter Erin Pearson and senior reporter Chris Vedelago on the curious case of the mushroom murder jury. Background reading: Erin Patterson, the jury hotel and the curious case of the missing CCTV The Morning Edition brings you the story behind the story with the best journalists in Australia. Join host Samantha Selinger-Morris, from the newsrooms of The Sydney Morning Herald and The Age, weekdays from 5am.If you enjoyed this episode and want to hear more, rate, review and subscribe on Apple, Spotify, YouTube or whichever platform you find your pods.Subscribe to The Age & SMH: https://subscribe.smh.com.au/See omnystudio.com/listener for privacy information.
Send us Fan MailFive years after Ohio began implementing the Fair School Funding Plan, former Ohio House Speaker Bob Cupp, a Republican, and former state Rep. John Patterson, a Democrat, reunite to discuss how they worked across party lines to tackle one of Ohio's most complicated public policy issues.Cupp and Patterson share how they built trust, brought school leaders and other experts into the process and found common ground despite their political differences.They also discuss the plan's future, Ohio's property tax debate and how school board members can build stronger relationships with their legislators.For more resources, training and advocacy updates from the Ohio School Boards Association, visit ohioschoolboards.org.
Brooke and Tyler hop in their starfighters and take flight with author Janci Patterson to discuss the release of her and Brandon's latest novel, Blightfall. We discuss the unique writing partnership between Janci and Brandon, the development of Blightfall and upcoming Riftwake novels, and the true stars of the series- Taynix. This episode is divided into two sections and there are spoilers for Blightfall beginning at 52m36s. #SomeSpoilers Read Blightfall before listening past 52m36s. Available now wherever you get your books. Follow Janci on Instagram Janci's website can be found here Support this podcast by becoming a Patron on Patreon Original music by David Gruwier. "Radiant" by David Gruwier.
Last time we spoke about the beginning of the Battle of Hong kong. Japan's rise as a threat to British interests accelerated through the 1920s and intensified after invading China. Britain's response remained contradictory—diplomatically conciliatory yet militarily apprehensive. While Singapore and Malaya received substantial reinforcements, Hong Kong was acknowledged indefensible, yet the 1936 Defence Scheme attempted to fortify it as an island fortress. Governor Northcote urgently advocated evacuation, recognizing the futility of defense. London rejected this, fearing it would demoralize China and forfeit sovereignty claims. Reinforcement requests from commanders like Grasett fell on deaf ears until Grasett independently lobbied Canadian leaders. This persistence finally convinced Churchill to dispatch two additional battalions. On December 8, 1941, Japanese forces attacked across three fronts with overwhelming force. Within hours, the RAF was destroyed on the ground, demolitions proved ineffective, and British units rapidly retreated toward the Gin Drinkers Line, facing an enemy that outnumbered and outmaneuvered them from the opening moments. #217 The Battle of Hong Kong Part 2: the Island Aflame Welcome to the Fall and Rise of China Podcast, I am your dutiful host Craig Watson. But, before we start I want to also remind you this podcast is only made possible through the efforts of Kings and Generals over at Youtube. Perhaps you want to learn more about the history of Asia? Kings and Generals have an assortment of episodes on history of asia and much more so go give them a look over on Youtube. So please subscribe to Kings and Generals over at Youtube and to continue helping us produce this content please check out www.patreon.com/kingsandgenerals. If you are still hungry for some more history related content, over on my channel, the Pacific War Channel where I cover the history of China and Japan from the 19th century until the end of the Pacific War. Around 1500 hours on 9 December, Colonel Doi of the 228th Regiment arrived at Needle Hill, just north of the Gin Drinkers Line, ahead of his main force. For two hours he studied the British defensive positions through binoculars, noting trenches and bunkers—and to his surprise, washing lines fluttering with white clothes. The sight spoke volumes about the 2 Royal Scots' lax discipline. Then fog descended, reducing visibility to barely twenty meters, and heavy rain obscured the landscape. Doi sensed a fleeting advantage but lost radio contact with his battalions. Worse still, the Shing Mun Redoubt lay outside his regimental boundary, making an unauthorized attack a serious breach of protocol. When his units finally arrived, supporting artillery remained unavailable, held up by demolished roads. Doi chose to attack anyway. Doi ordered the 2nd Battalion to scout the eastern approach while the 3rd Battalion spearheaded the main assault. Major Nishiyana commanded the 3rd, sending Lieutenant Wakabayashi and Kasugai with 150 volunteers to lead. The Shing Mun Redoubt itself comprised five concrete pillboxes and an artillery observation post, connected by underground tunnels named after London landmarks—Piccadilly, Haymarket, and others. The whole complex sat on small rises above Jubilee Dam, then the British Empire's tallest. Defending the redoubt was A Company, 2 Royal Scots, under Captain Jones—just three officers and 39 men. One platoon of 25 soldiers under 2nd Lieutenant Thompson held the forward positions. Against this stood 150 Japanese attackers, with another 1,500 troops in reserve. Battalion orders forbade using the concrete works as fighting positions; they were meant for the Vickers machine-gun team and artillery protection. No mines could spare the redoubt's perimeter. On the night of the 9th, roughly half the defenders sheltered in the observation post while the remainder manned pillboxes 401b and 402. At 2000 hours, Thompson led a nine-man patrol northward to check Needle Hill and Shing Mun Valley. Despite two and a half hours in the field, the patrol returned at 2220 hours without detecting the Japanese force massing just 500 meters away. By that time, Lieutenant Kasugai's assault group had crossed Jubilee Dam undetected, bypassed unmanned Post X, and assembled below Pillbox 401b. At 2200 hours, Japanese sappers cut through two lines of wire. At 2300—H-Hour—Lance Corporal Laird heard movement in the bushes and opened fire. The Japanese answered with grenades and rushed the pillbox lines. One team entered the tunnel system; another hurled grenades through the air vents. Corporal Campbell swung his Vickers toward Pillbox 401, catching Japanese headquarters personnel in the open. Sergeant Robb mustered a counter-attack force of thirteen men. Five fell as casualties—Laird, Basnett, Coyle, Casey, and Jardine. Casey was killed; only Jardine escaped. The alert triggered gunfire from the observation post's defenders, and Forward Observer Wilcox called down 4.5-inch and 6-inch artillery fire. Without strong reinforcement and leadership, four Royal Scots and an Indian sentry abandoned the northern positions and withdrew to the observation post. Captain Jones received orders from Brigade to counter-attack, but delegated the task to Thompson. As Thompson attempted to leave the observation post, he found the gate locked from outside. This calamity had a backstory: just before the attack, Thompson had admitted a civilian at the request of Force 163 (SOE's sabotage unit in Singapore). Private Wylie had taken the observation post key from Lance Naik Kishan Singh and, upon exit, locked the gate behind him. The three officers were now literally locked out of the battle. Second Lieutenant Mochizuki and his platoon infiltrated through open trenches and began attacking the observation post from above, attempting to breach the hatches. The defenders held for three hours before Japanese sappers forced explosives down the air vents. At 0230 hours, the blast killed two Indian signallers inside. Thompson sustained a severe eye wound from grenade shrapnel; Jones and Warrant Officer Mead were both wounded. The remaining fifteen defenders surrendered; eleven became casualties. Meanwhile, a Rajput patrol engaging Japanese forces in the river valley managed to drive them back toward the redoubt. One final pillbox endured for another eleven hours before suffering the same sapper treatment. By 0400 hours on 10 December, the entire redoubt was lost. For just two killed, the Japanese had achieved a decisive victory—the breakthrough that would shift the entire campaign's trajectory. By dawn, a Japanese flag flew triumphantly from Point 255. Major-General Maltby's assessment was stark: "This was calamitous, for Shing Mun was the key to the whole of the left position." Yet the Japanese perspective differed sharply. Having achieved a breakthrough, Colonel Doi received astonishing orders to withdraw—his success had transgressed into the 230th Regiment's sector and violated the meticulously prepared war plan. Doi refused. After heated confrontation with Major-General Ito and Lieutenant-General Sakai, his insubordination was later censured, though a divisional staff officer, Oyadomani, bore the scapegoat's burden of rebuke for failing to restrain Doi's initiative. With Shing Mun secured, Doi methodically fed reinforcements into the position. Throughout December 10th, the Japanese remained uncertain of their advantage and adopted a cautious strategy, probing British lines to exploit the evident disorder. Brigadier Wallis ordered the Royal Scots to mount an immediate dawn counterattack supported by the Rajputs and artillery. Lieutenant-Colonel White demurred; his battalion lacked any reasonable prospect of success. The resulting counterattack proved half-hearted at best. At first light on December 11th, the Japanese pressed Golden Hill forcefully, only to be repulsed by Captain Newton's Rajputs and gunfire from HMS Cicala. White positioned D Company in shallow shell scrapes and rusted wire—inadequate defenses against Japanese assault. Captain Pinkerton led a desperate bayonet charge that morning, temporarily clearing the Japanese and enabling the evacuation of wounded. Yet B, C, and D Companies absorbed devastating losses; Captains Rose and Richardson fell commanding their positions. With the Royal Scots collapsing, X Company of the Winnipeg Grenadiers and armored support rushed westward to seal the widening gap along Castle Peak Road. Meanwhile, Maltby grappled with alarming reports: Japanese landings on Lantau Island southwest of Hong Kong. Heavy artillery drove them back. A second probe at Aberdeen, where Special Naval Landing Forces approached within 300 yards of the naval base, was repelled by Canadian machine-gun fire. In hindsight, these were mere feints—never intended as the primary thrust. The stratagem succeeded. Maltby retained his island reserves—the Middlesex and two Canadian battalions—precisely where they would be needed. Had Wallis executed a forceful counterattack on December 10th with the Royal Scots and brigade reserves, the Rajputs might have retaken Shing Mun while the Japanese remained disorganized. Instead, Colonel Doi faced a furious Sakai, who had flown down from 38th Division headquarters at Taipo for what amounted to a pre-court-martial examination. The moment passed. After only 48 hours of combat, Maltby concluded the mainland was indefensible. The three battalions would withdraw intact to Hong Kong Island to assume their fortress defense roles. The naval commodore objected—demolitions, supply transfers, and troop ferrying required more time. The evacuation order was deferred 24 hours until noon on December 11th. At that hour, the decision was finalized: the mainland would be abandoned under cover of darkness. Further demolitions followed—the China Light and Power Station, the cement works on Tsing Yi Island, the docks. Yet Brigadier Wallis believed this merely a precautionary measure; he anticipated holding the mainland for at least another week. The withdrawal avoided Kowloon's congested streets, where street fighting and fifth columnists might have entrapped the British—a calculation that proved prescient, as an IJA advance column of 350 men from the 3/230th infiltrated Kowloon on the morning of December 12th to cut off the retreating garrison. The Royal Scots and Canadians retired south to Shamshuipo Barracks and Jordan Road Pier in western Kowloon; the Rajputs and supporting gunners withdrew eastward to Ma Lau Tong, which anchored the fortified Devil's Peak Peninsula. Maltby intended to hold this commanding position permanently—it was easily supplied across the narrow Lyemun Strait. The evacuation proceeded largely as planned. The Canadians and Royal Scots embarked on the afternoon and evening of December 11th under Punjabi covering fire. The Punjabis faced a harder journey—a moonlit scramble along steep Kowloon hills laden with supplies but critically short of mules. One group lost its way at a crucial junction, splitting into two columns. One proceeded toward Devil's Peak as planned; Battalion Headquarters descended into Kai Tak Airport, threaded through Kowloon City, and was evacuated by Star Ferry at Tsim Sha Tsui, fighting until the final moment as the last man boarded. The Rajputs' commander, unable to reach his Punjabi counterpart, abandoned Ma Lau Tong for the closer Hai Wan line near Devil's Peak. By 0400 hours on December 12th, the Punjabis and one Rajput company, accompanied by the gunners of the 25th Medium Battery, crossed Lyemun Strait to Hong Kong Island. Morning evacuations continued with minimal Japanese interference—a surprising restraint. Naval support proved crucial. The withdrawal completed by the morning of December 13th, though 170 mules were lost to the desertion of Chinese laborers—a deprivation that would haunt the subsequent campaign. During this passage, the IJN attempted shore bombardment. The Japanese cruiser and supporting vessels found themselves outranged by the defenders' 9.2-inch guns. The damage inflicted proved sufficient to discourage future naval adventures; for the remainder of the battle, the IJN maintained a cautious distance and played only a peripheral role. The speed and execution of the withdrawal apparently caught the Japanese unprepared. Their most probable expectation had been a full-scale assault on Devil's Peak accompanied by heavy bombardment—a day earlier, an assault without artillery preparation had been driven back with severe losses. The British appeared stronger than anticipated. The defense of the mainland had lasted only five days. With the mainland secured, the Japanese pivoted toward Hong Kong Island. The first move was diplomatic rather than military. At 0900 hours on December 13th, a small boat bearing a white flag crossed the harbor. It carried Colonel Toda, Lieutenant Mizuno, Mr. Othsu Dak, and two European women—the prominent and heavily pregnant Mrs. Macdonald and Mrs. C. R. Lee, wife of the Governor's secretary, accompanied by her two dachshunds. The surrender terms were flatly rejected. Major Boxer delivered the written refusal personally. Mrs. Macdonald remained in Hong Kong to give birth; Mrs. Lee and her dogs returned to Kowloon. The Japanese commenced systematic shelling of selected targets. On December 13th, a 9.2-inch gun on Mt. Davis was silenced; Belcher's Fort sustained damage. The following day, a 3-inch mount on Mt. Davis was hit, killing Chinese gunners and triggering morale collapse; some deserted. On December 15th, the barrage shifted to the northern shoreline, deliberately targeting pillboxes. At 2100 hours that night, approximately three Japanese companies attempted crossing the harbor in rubber rafts toward Pakshawan but were driven off by machine-gun fire. Fifth columnists intensified their operations—signaling with mirrors, sniping from concealed positions, spreading propaganda to encourage Chinese desertion. Their impact might have been catastrophic had not Rear-Admiral Chan and Colonel Yee, armed with tommy guns and grenades, systematically eliminated many of these saboteurs. By December 16th, half the pillboxes between the racecourse and Lyemun Strait lay destroyed. The intended crossing site was unmistakable. On December 17th, the Japanese dispatched a second peace mission—Colonel Toda's team now accompanied by Mrs. Lee with her dachshunds and a pregnant Russian woman. Again, the overtures were rebuffed. On December 18th, the bombardment reached new intensity. Oil storage tanks ignited, sending dense black smoke cascading across the northeast corner of the island—perfect concealment for what was to come. All signs pointed to imminent assault. I want to note something here, you sometimes hear about how race played a role in the Pacific War and indeed it did. Racist attitudes were found on both the western and Japanese side of the war. There is a fantastic book dedicated to this by the way called, War without Mercy by John Dower, highly recommend it. What is interesting is how these racial attitudes actually affected decision making during the war. Take General Sakai's night amphibious assault for example. Western soldiers such as the Canadians in Hong Kong reported they were told by their commanders that quote "Don't worry about the Japanese, they can't see at night, they all wear glasses" another Canadian soldier wrote in a diary "They don't see well, especially at night we knew this as a matter of fact." Indeed the week before the outbreak of the battle of Hong Kong, Canadian officers attending a briefing by a British officer were informed that "Japan's aircraft were mostly obsolete, its air force had little practice in night flying, and its pilots were myopic and thus unable to carry out dive-bombing attacks". You might be asking yourself, why did they think the Japanese were myopic? The scientific explanation of the day was this "That the Japanese suffered widespread inner-ear damage. What caused this? Japanese motherhood, the practice of strapping babies to their mothers backs, it was explained caused their heads to bounce about, like when they harvested rice and permanently impaired their sense of balance". I particularly love this quote, one of my professors dedicated an entire class to the bizarre racial attitudes before and after the pacific war. There was a belief the Japanese could not see well at night, many western air force commanders actually believed there were times of day like at dawn or dusk where Japanese pilots would not operate because of this hindrance. American military commentator Fletcher Pratt in 1939 analyzed Japan's military strengths and explained it had four major weaknesses. The first was scarcity of resources such as iron, and steel. The second was that the Japanese "can neither make good airplanes nor fly them well" as a result of " the Japanese as a race have defects of the tubes of the inner ear, just as they are generally myopic. This gives them a defective sense of balance, the one physical sense in which an aviator is not permitted to be deficient.". The third was that the Japanese were short-tempered and were inclined to waste men unnecessarily when operations did not go well. The fourth was that the Japanese would simply never date to provoke a war with the US. Again Mr Pratt was an American military commentator whose information came from multiple accounts given by high ranking military officials! The British military maintained that the Japanese avoided night operations on land because they were simply incapable of caring them out well, while they avoided nighttime aerial attacks, as well as dive-bombing for the same reasons Pratt said quote "their pilots were poor and their aircraft inferior.". So take this all to mind when I talk about how certain ranking officers go about making their decisions. By the evening of 14 December, evacuated mainland units had regrouped on Hong Kong Island. The defensive structure was restructured into two brigades: Brigadier Wallis commanded the East Brigade from Taitam Gap, while Brigadier Lawson headed the West Brigade from Wongneichong Gap. This reorganization placed the 5/7 Rajputs across the north-east perimeter, defending over 3,500 metres of coastline with B Company positioned on the hills behind Taikoo Docks. The Royal Rifles of Canada held the north-east corner from Stanley to points south, with reserve positions at Lyemun. Two HKVDC companies supported from Taitam Valley and Pottinger Gap, while the veteran Hughsiliers manned the North Point power station. British and Canadian artillery provided fire support across the northern approaches. The West Brigade held the north-western and south-western shores. The 2/14 Punjab stretched from Causeway Bay to Belcher's Point, protecting Government House and General Maltby's headquarters, with battalion headquarters at Mid-Level. The Winnipeg Grenadiers covered the south-west from Wanchai Gap, with one company detached to the brigade headquarters at Wongneichong Gap. The 2 Royal Scots, severely mauled at Shing Mun, were held in reserve with fresh replacements from the HKVDC. The Middlesex Regiment's companies were dispersed along 72 pillboxes encircling the entire shoreline, while an improvised Z Company of clerks, musicians and cooks defended battalion headquarters at Leighton Hill. Supporting artillery included 9.2-inch and 6-inch guns on Mt Davis and 4.7-inch guns at Belcher's Point. As darkness fell on 17 December, Japanese reconnaissance swimmers reached Taikoo, confirming the crossing site despite detection. Reinforcements arrived: 18 additional heavy bombers from Guangzhou and 26 fighters from Taiwan joined the aerial assault. The crescendo of artillery fire on the 18th, combined with masses of small craft gathering across the harbour, signalled the imminent invasion. Japanese divisional headquarters shifted to Ma Tau Wei near Kai Tak Airport; the 23rd Army moved to Taipo. The invasion force disposed itself with the right flank embarking from east and west of Kai Tak, the left from Devil's Hill area. At 1800 hours, the invasion order was issued; by 1930 hours Sakai himself arrived in Kowloon to oversee the crossing. At precisely 2000 hours, the 2/228th Regiment silently paddled their collapsible craft toward Taikoo Docks and the nearby sugar factory as artillery support commenced. Colonel Doi embarked with 80 headquarters personnel aboard a large barge, leading the second wave. The conditions favoured the invaders—a moonless night, sporadic showers, and thick black smoke from burning oil tanks obscured their movement. They reached the midpoint undetected before searchlights and machine-gun fire scattered the small boats, separating commanders from their units. Despite this chaos, the assault pressed forward. At 2140 hours the artillery shifted to deeper targets; at 2145 hours the first wave of 3/230th landed at North Point, followed minutes later by 2/228th and then 2/229th. Three red flares signalled a successful landing. All six Japanese battalions were ashore by midnight, though wire fencing delayed their advance and Bren carriers added to the confusion on the beach. General Sano arrived at Taikoo one hour later. The 5/7 Rajputs bore the initial brunt of the assault. Despite days of preliminary shelling, the Rajputs mounted a stubborn defence; Japanese casualties mounted rapidly. The anti-tank company suffered so grievously that only a single gun remained operational. The Japanese plan was tactical rather than frontal—bypass isolated positions for later elimination and push rapidly toward high ground. Overwhelmed by numerical superiority and losing most of their officers, Rajput resistance gradually crumbled. Contradicting all evidence, Maltby remained convinced the main Japanese assault would strike directly at Victoria from Kowloon. He therefore withheld the counter-attack, instead dispatching a platoon of Middlesex troops, Royal Marines, naval personnel, and the Rajputs' reserve company—supported by artillery—to establish a defensive line blocking the Japanese advance toward Central District. Three HKVDC armoured cars were sent to protect East Brigade headquarters; two more reinforced the Middlesex battalion headquarters. It was manifestly inadequate against six Japanese battalions. In post-war dispatches, Maltby conceded his grave miscalculation: he believed he faced at most two battalions, not a full division. As the 3/230th broke through D Company of the 5/7 Rajputs, the 2/230th came ashore nearby and wheeled westward toward Victoria. Their advance halted abruptly at the North Point power station, held by an extraordinary force: four officers and 36 men of the HKVDC Hughsiliers Platoon—alongside 39 civilian volunteers from China Light & Power and Hong Kong Electric, including the station manager Vincent Sorby—plus eight Free French soldiers commanded by Captain Jacques Egal, a Shanghai businessman turned soldier. Without exception, these men were World War I veterans, many aged 55 or older. Some had fought in the Boer War. Major J. J. Patterson, the Hughsiliers' commander, had served with Allenby's Camel Corps and been mentioned in dispatches six times; he was chairman of Jardine Matheson and a legislative councillor. Private Sir Edward Des Voeux, 8th Baronet, a millionaire bullion trader and secretary of the Hong Kong Club, had enlisted as a private when deemed too old for mobilization. Private T. A. Pearce, 67 years old, was chairman of J. D. Hutchison & Company and secretary of the Hong Kong Jockey Club. Captain Bruch, the second-in-command, aged 60, chaired a major trading house. The list extended through Hong Kong's business elite, each choosing to fight rather than flee. These old men held an entire regiment at bay. When Patterson called for aid, Maltby dispatched an HKVDC armoured car with a platoon from Z Company, 1 Middlesex, but the reinforcement was ambushed before reaching the station; only nine soldiers, including 2nd Lieutenant Caruthers, broke through. Unable to dislodge the defenders by assault, the Japanese resorted to bombardment. By 0145 hours on 19 December, surrounded and ammunition exhausted, the defenders withdrew to King's Road behind a disabled bus. The Japanese deployed three machine guns, killing or mortally wounding all but Private Geoghan. A Japanese officer, believing all dead, cautiously approached—only to have Geoghan, despite his wounds, surge from cover and charge. He killed the officer and four soldiers before collapsing from wounds. Somehow Geoghan survived. Those old men—civilians and reservists, businessmen and veterans—held the entire western Japanese advance for 18 hours, buying Maltby precious time to withdraw forces and establish a new defensive line. Their stand at North Point became one of the campaign's most improbable acts of defiance. As the 2/229th Regiment advanced southward toward Mt Parker, the 3/229th landed at Aldrich Bay and turned eastward toward Lyemun, where they seized an HKVDC 6-inch gun. Major Bishop of the Royal Rifles of Canada, unaware of the landings, dispatched 15 Platoon under Lieutenant Scott to check on A Company of the Rajputs. Unexpectedly, they encountered armed men in civilian clothes at Lyemun's gate—the first indication of the Japanese presence on the island. Yet senior officers dismissed initial reports of landings and lost positions as exaggerated, a critical miscalculation that proved catastrophic. At Lyemun Fort, the 5th Anti-aircraft Battery of the HKVDC was caught completely unaware and surrendered after minimal resistance. The Japanese then committed one of many atrocities that would characterize the battle, bayoneting 29 survivors. Alerted by 15 Platoon's report, Bishop attempted a counter-attack with two platoons to retake the fort, but the defenders could not scale the six-meter wall and withdrew with nine men killed. As the 2/229th pressed southward toward Mt Parker, they overran the Salesian Mission, which had been converted into an Advance Dressing Station. No. 8 Company of the 2/229th systematized a massacre of medical personnel and the wounded—an atrocity that would later become pivotal evidence at Colonel Tanaka's war crimes trial. Yet amid the chaos, several medical staff and wounded survived to testify: Gunners Y. K. Chan and Martin H. C. Tso of the 5th Battery, Captain Osler Thomas of the HKVDC, Captain Martin Banfill of the Royal Canadian Medical Corps, and Corporal Norman Leath of the Royal Army Medical Corps. C Company of the Royal Rifles, reinforced with captured Japanese machine guns abandoned by fleeing Rajputs, inflicted heavy casualties on the attackers, with one Japanese company suffering over 65 percent losses. Yet overwhelmed by superior numbers and suffering nearly two platoons worth of casualties themselves, the Canadians were forced to withdraw, leaving only a reinforcement platoon under Lieutenant Blaver on Mt Parker. When Captain Clerke arrived at dawn with 16 Platoon to reinforce the position, he found over 100 Japanese already entrenched. With no artillery support and only two platoons available, Clerke had no choice but to withdraw, abandoning Mt Parker to the enemy. By dawn on the 19th, the East Brigade faced catastrophe. Infantry strength had been halved, and losses of artillery were equally devastating. A series of misunderstandings and misinterpreted orders led coastal gun crews at Capes D'Aguilar and Collinson to destroy their own guns in confusion or lose them to the advancing Japanese. Of the entire artillery complement, only one 18-pounder and two 3.7-inch field guns remained operational. Wongneichong—literally "yellow muddy creek" in Chinese—occupies the center of Hong Kong Island, a valley separating the eastern hills (Mt Parker, Mt Butler, Jardine's Lookout) from the western massif (Mt Nicholson, Mt Cameron, Victoria Peak). This narrow gap formed the critical artery linking northern and southern Hong Kong, making Brigadier Lawson's decision to place West Brigade Headquarters there strategically sound yet tactically vulnerable. Recognizing the exposed position after the landings, Major Lyndon had located an alternative site south of Mt Nicholson, scheduled to open the following day. In response to the Japanese landings, Lawson dispatched three platoons of Winnipeg Grenadiers to block the advance. Lieutenant C. D. French took 18 Platoon to Mt Butler, Lieutenant G. A. Birkett's 17 Platoon moved to Jardine's Lookout, and Lieutenant L. B. Corrigan's platoon took the north-west approach to the Gap. As on Mt Parker, both French and Birkett were killed almost immediately, their small forces overwhelmed by battalion-strength enemy forces with artillery support. By morning on the 19th, two battalions each of the 230th and 228th Regiments were pushing for the high ground. The 2/230th rapidly occupied Jardine's Lookout while the 3/230th moved to Mt Nicholson and the 2/228th advanced toward the Gap itself. No. 3 Company of the HKVDC under Major E. Stewart held pillboxes around Jardine's Lookout, while No. 1 Company under Captain Penn defended Taitam Valley with forward positions at Quarry Gap. The pillboxes, despite heavy bombardment, held longer than anticipated—some for as long as 24 hours. Stewart maintained his company headquarters until the 22nd, four days of continuous fighting without adequate ammunition, food, or water. No. 3 Company suffered 80 percent casualties and ceased to exist as a cohesive unit. Realizing that the enemy had penetrated to Mt Butler, Brigadier Lawson decided counter-attack was essential. Major A. B. Gresham of A Company, Winnipeg Grenadiers, was ordered to retake Mt Butler and clear Jardine's Lookout. The Canadians initially succeeded, with Warrant Officer 2 J. R. Osborne leading a bayonet charge, but a strong counter-attack by three Japanese companies drove them back by 2200 hours. The Canadians held firm until 1500 hours, when Gresham attempted surrender despite waving a white flag—the Japanese shot him regardless. In the chaos that followed, a Canadian killed a Japanese officer in response, triggering a grenade barrage. Warrant Officer Osborne caught the grenades and hurled them back as fast as they came, but one landed in an impossible position. Without hesitation, Osborne threw himself on the grenade to save his comrades. This act of supreme self-sacrifice earned him a Victoria Cross—the first Canadian VC of World War II. At dawn on the 19th, Maltby dispatched A Company of the 2 Royal Scots under Captain K. J. Campbell to reinforce the Gap, but they lost all their officers with only 15 men reaching Lawson's headquarters. A group of sailors under Commander A. L. Pears came from the south, but were ambushed en route; only a few reached the Postbridge—a house just south of the Gap—which became an isolated strongpoint. The 3/230th captured the police station perched on a mound at the southern point of the Gap, then swept eastward to seize the anti-aircraft guns of 7th Battery (barely 250 meters from brigade headquarters) and continued uphill toward Taitam Hill to capture additional 6-inch and 3.7-inch guns. Maltby's response was to call down final protective fire from the HKSRA in Happy Valley, inflicting massive casualties on the 3/230th. At 1000 hours on the 19th, Lawson reported that his headquarters was surrounded and he was about to "go outside and shoot it out." Captain H. A. Bush provided covering fire as Lawson and his entire staff, including Middlesex signallers, were cut down by machine-gun fire across the Gap. Lawson's body was discovered two days later halfway up the hill behind his headquarters, dead from a gunshot wound to the thigh. Lieutenant Kerfoot of the 2/14 Punjab with his three Bren carriers arrived minutes too late to save him. IJA who saw the fight made a note stating "he died heroically". At 1330 hours on the 19th, Maltby issued "Operation Order No. 6" calling for a massive counter-attack to commence 90 minutes later. The plan was ambitious: A and D Companies of the 2/14 Punjab were to attack east from Victoria to North Point to relieve the Hughsiliers, while HQ Company of the Winnipeg Grenadiers and the 2 Royal Scots were to attack the Gap and Jardine's Lookout. Eight field guns were promised but never materialized. There was no reconnaissance, no proper orders, and minimal coordination. The Canadians were to rendezvous with the 2 Royal Scots at Middle Gap at 1530 hours, but the Scots were late; the Canadians advanced without them. Captain Pinkerton of D Company, 2 Royal Scots, was ordered at the last minute to attack up Wongneichong Gap Road—the same route A Company had taken only seven hours earlier and been decimated—because it was reported as "lightly held." Captain A. M. S. Slater-Brown led the advance with three Bren carriers and attached engineers, followed by D Company of the Winnipeg Grenadiers. Just as the carriers reached the burnt-out trucks of A Company, they too were ambushed. Mortars and machine-gun fire rained down from Jardine's Lookout, where the Japanese occupied captured HKVDC pillboxes. Slater-Brown and 2nd Lieutenant Bell, the battalion intelligence officer, were killed instantly. The Royal Scots waited in the roadside ditches until 0200 hours before launching an attack on the police station, reaching the steps before Pinkerton was severely wounded. Captain Ford's composite company attack at 0300 hours also failed. Simultaneously, C Company under Lieutenant F. L. Stanier attacked Jardine's Lookout—also unsuccessfully. This series of piecemeal assaults cost the Royal Scots eight officers and 68 soldiers. Growing increasingly desperate, Maltby ordered Major Hodkinson, Officer Commanding HQ Company of the Winnipeg Grenadiers, to attack the Gap and proceed to Mt Parker. Requesting reinforcements from A Company and the Royal Scots, Hodkinson launched his assault with covering fire from Lieutenant Corrigan on Mt Nicholson. The Grenadiers managed to reach Mt Nicholson with only five unwounded men, though Corrigan's group advanced to within 300 meters of the Gap road. Then came an unexpected stroke of fortune: while skirting Mt Nicholson, Hodkinson's group stumbled upon approximately 500 Japanese—probably from the 3/230th—eating lunch without posting sentries. The Winnipeg Grenadiers unleashed devastating fire, inflicting heavy casualties. By 1745 hours, Hodkinson's force was only 100 meters from Repulse Bay Road. With four men and a 2-inch mortar, Hodkinson worked his way to the abandoned brigade headquarters, bringing 20 walking wounded with him. He established a foothold at the Gap, receiving orders to attack the police station at 2200 hours. Two HKVDC armoured cars with Vickers machine guns came in support, but accurate enemy fire disabled both. Like Pinkerton before him, Hodkinson reached near his objective before being severely wounded. For this action of determination and courage, Hodkinson was awarded the Distinguished Service Order. By the morning of the 20th, the Japanese had consolidated control of the Gap, but elements of D Company, Winnipeg Grenadiers, still held various pillboxes north of the position. Initially, 17 and 18 Platoons occupied forward positions with Company Headquarters and 16 Platoon in reserve, positioned across Wongneichong Road almost directly opposite brigade headquarters. Forward positions were rapidly overrun and cut off from the company. They fought without officers until withdrawal to battalion headquarters became necessary. With Captain A. S. Bowman killed and Captain R. W. Philips wounded, command fell to Lieutenant T. A. Blackwood. An attempt by B Company to relieve D Company on the evening of the 20th failed catastrophically, killing all officers and 29 men. With fewer than 50 men remaining—all wounded—Lieutenant Blackwood continued inflicting heavy casualties on the Japanese until 22 December, when exhaustion and the complete absence of ammunition, food, and water forced surrender. D Company killed over 200 Japanese soldiers. Post-war Japanese reports refused to believe that a single company had held the Gap for so long. For their stubborn defense, Philips and Blackwood were awarded the Military Cross, and three enlisted men received the Military Medal. The battle of Wongneichong Gap was characterized by fragmented planning and poor coordination. On the night of the 19th-20th, no fewer than three separate companies attacked the police station, with another company assaulting Jardine's Lookout—all within five hours, all independently organized by local commanders. Had these disparate actions been coordinated into a single simultaneous assault, fortress headquarters' objectives might well have been achieved. By the end of the 20th, the British had lost the vital ground of Wongneichong, though Postbridge House remained as an isolated holdout. For the Japanese, Wongneichong became the single costliest engagement since the invasion began, with over 800 casualties and the regimental commander of the 3/230th seriously wounded. I would like to take this time to remind you all that this podcast is only made possible through the efforts of Kings and Generals over at Youtube. Please go subscribe to Kings and Generals over at Youtube and to continue helping us produce this content please check out www.patreon.com/kingsandgenerals. If you are still hungry after that, give my personal channel a look over at The Pacific War Channel at Youtube, it would mean a lot to me. Japanese landings on 18-19 December overwhelmed the East Brigade. Brigadier Lawson died defending the vital Gap. Desperate, uncoordinated counter-attacks by British and Canadian troops failed to dislodge the enemy. D Company Winnipeg Grenadiers held the position for four days against overwhelming odds before surrendering. The Japanese suffered 800 casualties but secured this critical strategic ground.
Karen Patterson talks with J about learning spirituality and the teachings of Heinz Grill. They discuss the religious impulse, finding truth from within, ashram living, Father Bede Griffiths, Heinz Grill, qualities of soul, informing asana with imagination, essence and meaning, inspired wisdom, awakening self, radiance and potential, enlivening yoga, heart to heart communication, ultimate source, spiritual laws, maintaining integrity, subtle energy, and recognizing when authenticity and truth is embodied and expressed. To subscribe and support the show… GET PREMIUM. Say thank you - buy J a coffee. Check out J's other podcast… J. BROWN YOGA THOUGHTS.
In this podcast, Greg Voisen sits down with Johnny Patterson, Chief of Staff for the Alliance for Responsible Citizens (ARC) and former founding director of Hong Kong Watch, to explore his landmark book, The Age of Reconstruction: Building a Civilisation Worth Inheriting. Starting from his front-row seat watching freedoms stripped away in Hong Kong to analyzing the "meaning crisis" consuming modern Western youth, Patterson delivers a stark wake-up call and a compelling message of hope. Is Western civilization in an irreversible, downward spiral of cynicism and cultural amnesia—or are we on the brink of a massive cultural rebuilding movement? Dive in to discover why dismantling the past was easy, but constructing a future worth inheriting demands an entirely new kind of courage.
durée : 00:05:08 - Le masque et la plume - par : Rebecca Manzoni - Entre drame familial, thriller et portrait du Sud des États-Unis, "Oklahoma Honey" d'Andrew Patterson a suscité des réactions contrastées au sein du Masque, que ce soit à propos de la performance de Matthew McConaughey, du rôle de la musique bluegrass ou de la représentation de l'Amérique rurale. - équipe : Stéphane Le Guennec, Ilinca Negulesco - invités : Marie Sauvion Journaliste à Télérama, Christophe Bourseiller Journaliste France Inter et Transfuge , Jean-Marc Lalanne Critique de cinéma et rédacteur en chef du magazine Les Inrocks, Murielle Joudet Critique de cinéma au Monde Vous aimez ce podcast ? Pour écouter tous les épisodes sans limite, rendez-vous sur Radio France
What does the commercial cow that actually pays her way look like? Trey Patterson (Padlock Ranch) and John Maddox (Maddux Cattle Company) join us to discuss building a cow for the long haul—balancing fertility, genetics, management and production without losing sight of profitability. We'll look at why maximizing performance isn't always the answer, the value of heterosis, and how to build a cow that fits your environment and your operation. #WorkingRanchRadioShow #CommercialCattle #CowCalf #BeefCattle #Ranching #CattleGenetics #Heterosis #CowHerd #BeefProduction #RanchManagement #CattleProducers #CommercialCow #RanchProfitability #BeefIndustry
Send us Fan MailIn this episode of the Hodge Pack Sports Life & Everything in Between, Hodge, Josh and Misti welcome back Friday Night Lights and more.The Texas Rangers are still holding on hope for the playoffs. The Rangers head into a big three game series with the Brewers.Remembering Dolly Parton. Dolly made such a huge impact on so many generations.College football again is having another controversial storyline that has nothing to do with action on the football field. Instead they are talking legal action. What are the most interesting on the field storylines heading into the college football season?Abilene Christian University head football coach Ketih Patterson joins the show to preview the upcoming Wildcat season. Misti's Mulligans question of the week is "If whistles were outlawed what would you relace them with?Who will win the NFL Divisions up North? The returning Hodge Pack Football Pick Em Challenge Champion Mike Dean is in studio to begin his title defense. Support the show
Album 8 Track 19: How Authentic Positioning Drives Success w/Krista PattersonIn this episode of Brands, Beats & Bytes, hosts Darryl "DC" Cobbin and Larry "LT" Taman welcome positioning expert Krista Patterson, Co-Founder and CEO of Positioning Collective.Drawing from her executive tenure at tech giants like Cisco, ServiceNow, and VMware, Krista reveals why systemic business problems almost always trace back to murky positioning.Krista breaks down the distinction between targeting demographics versus targeting mindset, shares deeply personal stories about overcoming career missteps, and discusses managing high-profile partnerships like F1 McLaren. The conversation tackles modern industry shifts, from why communications leaders must question the ecological footprint of AI infrastructure to how Burger King reclaimed its market share through core positioning. Tune in for a masterclass on brand strategy, authentic leadership, and career growth.Don't forget to subscribe, rate, and share with a fellow Brand Nerd!Instagram | LinkedIn
Duane Patterson, with HotAir.com and Host of Duane's World Podcast, joins the show to discuss the Lindsay Clancy murder trial, as well as all things political.
We talk about being tire guy for David Gravel Racing and his own racing career.
On today's show, host Ali Muldrow speaks with the new Co-CEO and Chief People Officer for the Overture Center, Victor Patterson. They discuss the role of the arts in shaping Madison's culture, how Patterson developed his philosophy of leadership, and the jazz music and gospel plays that shaped him. Patterson describes how his upbringing in Chicagoland set him on a path to community building. Supported by his loving extended family and inspired by music and sports, Patterson says that he now makes decisions in order to build a strong legacy. Moving to Madison after decades working in corporate America, Patterson says that he stopped looking for bigger and bigger roles and instead chose Madison for the community. He says he's ready to make a splash in this smaller pond and focus on creating seats at the table in Madison's arts scene. Inclusivity and diversity are authentic realities for Patterson who wants the Overture Center to be the “social heartbeat” for communities of color in Madison and see the venue become a regional player in the arts. They also discuss how leaders build strong teams, remain open to feedback, and refuse to see harm in failure. Victor Patterson previously held senior human resources leadership roles with Lowe's, H.B. Fuller and The Home Depot, where he led workforce planning, succession strategy, leadership development and organizational effectiveness efforts across regional and global teams. At Overture, Patterson oversees the People & Culture function, influencing business strategy and driving inclusion, access and belonging initiatives as well as overall organizational culture. Featured image of Ali Muldrow and Victor Patterson courtesy of Sara Gabler/WORT. Did you enjoy this story? Your funding makes great, local journalism like this possible. Donate hereThe post New Co-CEO hopes Overture becomes Madison's “Social Heartbeat” appeared first on WORT-FM 89.9.
This episode of The C.L. Brown Show features CBS Sports reporter Chip Patterson, who is the host of the Cover 3 podcast on college football, to discuss the upcoming season for Louisville and Kentucky. Patterson believes the Cardinals' Isaac Brown is one of the best running backs in the nation — when he's healthy. Brown's talent is such that he can make UofL's offense proficient without needing quarterback Lincoln Kienholz to be great, Patterson says. But hear why he feels more comfortable pitching Brown as an All-America candidate instead of a Heisman Trophy hopeful. Patterson also explains why he's reserving judgment on UK coach Will Stein until he has a full year at the helm to recruit. Hear about that and more on this week's episode of The C.L. Brown Show.
Webster University Music Professor Trent Patterson joins Megan Lynch following the death of singer Dolly Parton at the age of 80 this week. He comments on her philanthropy, songwriting, acting and her long-lasting career. You just cant help but love her,' says Patterson. (Photo by Frazer Harrison/Getty Images)
Australia wrapped up the Bangladesh series with a dominant innings-and-51-run victory in Mackay, but the two-day Test leaves plenty to discuss. Menners questions the quality of Australian pitches after another Test finished inside six sessions, while also assessing Australia's unresolved batting problems heading into South Africa. Mitchell Starc was the star with 10 wickets, Cameron Green continued to strengthen his case at No. 5, and Nathan Lyon bounced back with three wickets. Menners also looks at Shoriful Islam's outstanding 7-48, Bangladesh's historic series win in Darwin, and what Australia should do with its batting order for the South Africa tour. (0:58) Australia crush Bangladesh — but another two-day Test raises serious pitch questions (5:39) Australia's batting problems remain unresolved despite levelling the series (9:59) Starc destroys Bangladesh as Australia wraps up a huge innings victory (14:19) The South Africa batting lineup — Renshaw, Head, Patterson and the No. 6 dilemma (16:00) Should Labuschagne be dropped? And why Head should remain an opener (18:14) Starc's incredible 10-51 and Australia's road to South Africa We've launched our official Cricket Unfiltered merch store thanks to a brilliant partnership with Exactamundo, a longtime supporter of the show.
Larry Patterson went in for a routine hernia repair. He came out, eventually, with several inches of his bowel removed. Timothy O'Brien, board-certified trial attorney and shareholder at Levin Papantonio, just won $88 million for Patterson and his wife Tammy against Covidien, a Medtronic company. It is the largest compensatory award in fifteen years of mesh litigation, and the first of roughly 2,500 cases to reach a jury. The mesh, Symbotex, sits right against your intestines behind a collagen barrier meant to last 30 days. Covidien's own internal testing showed it was gone in under seven. They knew in 2003. O'Brien walks Gregg through the evidence that turned the trial, where sales staff swore to doctors the barrier lasted a month while R&D said otherwise in writing. He also maps the hernia mesh lawsuit landscape, from the Bard settlement covering 33,000 claimants to why Medtronic Covidien is the last major manufacturer still fighting, and what a bellwether verdict actually means for the thousands of people still waiting on their own cases Join Gregg and Timothy O'Brien on Climate Change Environment Science & the Law as they explore: What You'll Learn Why a generation of surgeons stopped doing tissue-to-tissue repair, and what that traded away How Symbotex's protective barrier was supposed to work, and how fast it actually disappeared The 2003 document that locked in the depositions and became the case's spine Why sales and R&D were telling two completely different stories about the same product What a bellwether trial is, and why the first one carries the most weight Where the Bard, Johnson & Johnson, and Medtronic Covidien litigations stand right now Why about 100 new Covidien cases are still being filed every month What Covidien's planned appeal means for people still waiting How to find out if you or someone you love has a case Time Stamps 0:00 – "They are lying": the sales pitch vs. the truth 0:48 – Meet attorney Timothy O'Brien 1:02 – The hernia mesh "safety feature" that failed 2:06 – Why hernia mesh exists in the first place 4:13 – The Patterson case: what happened to the plaintiff 5:41 – The depositions that exposed Covidien 7:46 – 2003: when they allegedly knew the truth 9:33 – The 3 major mesh manufacturers, explained 11:59 – What a "bellwether trial" means for your case 14:07 – Will Covidien settle or appeal? 17:40 – How to contact Timothy O'Brien about your case Timothy O'Brien is a board-certified trial attorney and shareholder at Levin Papantonio, where he has served as lead or co-lead counsel in mass tort litigation against major hernia mesh manufacturers, including Bard, Johnson & Johnson, and Medtronic Covidien. He recently helped secure an $88 million verdict in the first bellwether trial of the Covidien hernia mesh multidistrict litigation. Contact / Follow Timothy O'Brien: Firm: Levin Papantonio Website: https://levinlaw.com Want more conversations that cut through the noise on science, climate, and the issues shaping our future? Subscribe to Climate Change Environment Science & the Law with Gregg Goldfarb for new episodes every week.
Director Andrew Patterson's big screen journey has been unconventional to say the least. With his debut feature film, The Vast of Night, Patterson patiently watched as his film was shopped around the festival circuit, landed a spot at the 2019 Slamdance Film Festival, going on to win the Audience Award for Best Narrative Feature. It was then acquired by Amazon Studios, where they were going to give it a proper release in 2020, but then the pandemic happened, and most audiences had to discover this bright new talent from the comfort of his own home. In this time, where we were all locked away in our homes, Patterson went back to an old script he was developing, one about an eccentric, beloved, blue grass-playing beekeeper and his estranged former foster daughter, their reconnection, and the passing of a touch between a business and the bloodlines you make along the way; The Rivals of Amziah King. In writing a draft that was close to 360 pages long, Patterson, a former editor before he turned writer-director, slowly stripped his epic down to the version that he wanted to put on the big screen; a sprawling tale that features people, a town, a culture, an environment he grew up around in a small town in rural Oklahoma. This baked in familiarity and personality resulted in his script impressing many, including the actor who plays the titular Amziah King, Academy Award winner Matthew McConaughey, who is a revelation in the film, as well as his co-star Angelina LookingGlass, one of the best discoveries of the year. Together, this acting duo brought to life the unique, brilliant vision of Patterson's expansive second feature and in result, was a rousing success at the 2025 SXSW Film Festival, with buzz building from the moment it screened. After being acquired by Black Bear Pictures, the world finally gets to see an Andrew Patterson film on the big screen, which, per my review from SXSW, is still "one of the best films of the year so far," and announced the director as "a serious name to be reckoned with in the world of cinematic storytelling," as well as high praise for McConaughey, who delivers "one of the best performances of his career and his best work in over a decade." In a recent conversation, Patterson and I spoke about the process of getting the film to the big screen, the patience it took to trust the studio, and his work as an editor shapes new role as a writer-director. We also spoke about recreating the world of Oklahoma in Alabama (where the film was mostly shot), his collaborative process with his crew and two lead actors, as well as his thoughts on the state of independent cinema, and the stories he wants to tell going forward, no matter how big or small they might be. Insightful doesn't begin to describe Patterson, a growing voice in modern cinema that's open to unknown possibilities that lie ahead, willing to still look optimistically at this business after an atypical start to one's cinematic career; humming along to his next project much like bees in his latest film, waiting for his chance to deliver something special yet singular like his first two features. The Rivals of Amziah King is currently in theaters from Black Bear Pictures. You can listen to this interview wherever you stream podcasts, from iTunes, iHeartRadio, Soundcloud, Stitcher, Spotify, Audible, Amazon Music and more. You can also listen on the AW YouTube page. Music: "Quill" from Matthew McConaughey and Ben Hardesty (intro) and "B-3" from BoxCat Games Nameless: The Hackers RPG Soundtrack (outro).
First up on the podcast, the News team's Diverse Voices in Science Journalism interns Mona Patterson and Laura Martín Agudelo join host Sarah Crespi to talk over some of their reporting from the summer. They chat about several stories, including why it's so difficult to track the food-borne illness cyclosporiasis and a new map for a major highway of the human nervous system. Check out all of Patterson's and Martín Agudelo's stories. Next on the show, Leslie Chan, assistant professor in the Wallace H. Coulter Department of Biomedical Engineering at the Georgia Institute of Technology and the Emory School of Medicine, describes an ingestible metabolic probe for the gut microbiome. Her team tested the pill in mice and showed they could detect an enzyme made by the microbiome that causes toxicity during some chemotherapy treatments. The signal is read from volatiles in the breath, making the test a noninvasive way to check on the activity of gut microbes. This week's episode was produced with help from Podigy. Image credit: Melanie Moser/Centers for Disease Control and Prevention Learn more about your ad choices. Visit megaphone.fm/adchoices
Minnesota DNR Research Scientist Kristen Patterson checks in with details on the large muskie study on Leech Lake, now in its second year. She discusses what's being done, what they're hoping to learn and what they know so far. And we even get in to some eelpout talk. See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.
Mark is joined by Duane Patterson, political pundit, Hot Air contributor, host of the Duane's World Podcast, and producer of The Hugh Hewitt Show. They discuss recent primary election developments in Florida and Michigan, analyze key Senate contests, and examine Sen. Jon Ossoff's controversial remarks regarding White House aide Natalie Harp. They also break down media coverage of law enforcement, local news headlines, and national security messaging.
The news of Texas covered today includes:Our Lone Star story of the day: The ACLU and other anti-religion groups have asked the Supreme Court to overturn the Fifth Circuit Court of Appeals and declare that Texas cannot post the Ten Commandments in school classrooms. Maybe we should accept their argument that exposure is equal to advocacy, indoctrination, and establishment. That way we could apply the same to all of their Marxist, secular, Leftist crap and force it all out of schools! Frankly, this whole of issue of separation of church and state is moronically misunderstood. There is no such separation in the Constitution, only the prohibition of government creating an Established Church – which had a very specific meaning when written not a generic anti-religious meaning.Our Lone Star story of the day is sponsored by Allied Compliance Services providing the best service in DOT, business and personal drug and alcohol testing since 1995.Rep. Patterson brings up, again, the idea of starting school in September. We've had this argument for decades as start dates moved earlier. What is missed by reporters and others who are ignorant what has gone before, is that it mostly has to do with football. Going lefty with near “participation trophies” for playoffs added weeks to the schedule.Texas CEO pay: Companies with the widest worker pay gaps. Why is this anyone's business? “Workers” (effectively a Marxist term) are free to buy stock and be owners in publicly traded companies but management pay is only their business as an owner, not an employee. And, note the silly union comment about workers being the ones “who make their companies profitable.” Firstly, it's not “their” company if they are employees. Secondly, if this is true then when companies fail it is the employees who “make their companies” unprofitable.Listen on the radio, or station stream, at 5pm Central. Click for our radio and streaming affiliates. www.PrattonTexas.com
In the third and final episode of this series with Janci Patterson, Jason Conforto, Chad Morris, and Shelly Brown discuss Janci's experience with ADHD and how it has shaped her creative life. Janci and Shelly both share their experiences with ADHD, exploring the challenges and strengths that come with thinking differently. Together, they discuss strategies for managing the writing process, the ways ADHD can influence storytelling, and how neurodiversity can be a creative advantage. It's an honest and encouraging conversation about creativity, writing, and embracing the way your mind works.
IN LATE AUGUST 1943, the crew of the destroyer U.S.S. Patterson scrambled to battle stations off the coast of what is now Vanatu, near Australia. The sonar operator had picked up the signature of a big Japanese submarine submerging close by. The Patterson steamed into battle, depth charges rolling off the deck and lighting up the sea below. Then, on the final depth-charge barrage, a deep undersea explosion could be heard, bigger than any depth charge. Oil and air bubbles boiled to the surface. The Patterson had found its mark. After that day, the most important submarine in the history of the state of Oregon — the Imperial Japanese Navy's I-25 — was never heard from again. (For text and pictures, see https://offbeatoregon.com/2401d-1101e.i25-bombards-fort-stevens-108.632.html)
It was one of the most watched criminal court cases in history, and now it's back.Mushroom killer Erin Patterson's appeal against her conviction for murdering three lunch guests and attempting to murder a fourth begins this week.Today, RMIT criminologist and lawyer Brianna Chesser, on why Patterson's lawyers argue she's been the victim of a miscarriage of justice.Featured:Brianna Chesser, RMIT criminologist and lawyer
In this week's Interview Classic podcasts, we jump back to 15 years ago this week (7-22-2011) for PWTorch editor Wade Keller interview with former WWE Creative Team member (2009-2010) John Piermarini. They discussed the C.M. Punk-John Cena match at Money in the Bank, Daniel Bryan's surprise Money in the Bank win including who in particular in power likely pushed for his win, the Triple H-Vince McMahon angle on Raw and where it might lead, and more including live calls on a variety of subjects including what changes Triple H make say, R-Truth's push, and much more. In the VIP Aftershow, must-listen insight into life after TV tapings and the how the writers, producers, and wrestlers do and don't intermingle, and whether the mood is different after good or bad PPVs and TVs. And finally, fascinating insight into the relationship former WWE booker Pat Patterson has with Raw's lead writer Brian Gewirtz and other WWE creative team members.Become a supporter of this podcast: https://www.spreaker.com/podcast/wade-keller-pro-wrestling-podcast--3076978/support.
"The Rivals Of Amziah King" is an American neo-Western crime thriller film written and directed by Andrew Patterson ("The Vast Of Night") and produced by Black Bear Pictures and Heyday Films. It stars Angelina LookingGlass as Kateri, whose life takes a tragic turn as she loses her mother and leaves foster care. Reuniting with her former foster parent, Amziah (Matthew McConaughey), changes everything: he becomes her mentor and reveals hidden skills when a devastating situation arises. Kateri relies on her newfound strength to seek justice. The film also stars Scott Shepherd, Rob Morgan, Tony Revolori, and Kurt Russell. It had its world premiere at the 2025 South by Southwest Film & TV Festival and received rave reviews for its story, music, performances, direction, and editing. Patterson was kind enough to spend some time talking with us about his work and experience making the film, which you can watch or listen to below. Please be sure to check out the film, which is now playing in theaters from Black Bear Pictures. Thank you, and enjoy! https://youtu.be/e-7xArbZRc4 Check out more on NextBestPicture.com Please subscribe on... Apple Podcasts - https://itunes.apple.com/us/podcast/negs-best-film-podcast/id1087678387?mt=2 Spotify - https://open.spotify.com/show/7IMIzpYehTqeUa1d9EC4jT YouTube - https://www.youtube.com/channel/UCWA7KiotcWmHiYYy6wJqwOw And be sure to help support us on Patreon for as little as $1 a month at https://www.patreon.com/NextBestPicture and listen to this podcast ad-free Learn more about your ad choices. Visit megaphone.fm/adchoices
Lyme disease is called "the great imitator" because it can look like growing pains, fatigue, or nothing at all and this episode shows you what to watch for. Steph sits down with naturopathic physician Dr. Jacquell Patterson, a nationally recognized expert in Lyme disease, tick-borne co-infections, and PANS/PANDAS to cover everything parents need to know about ticks and Lyme disease. You'll learn which areas to check on your kid's body, why the smallest ticks cause the most infections, and how to tell the difference between growing pains and a tick-borne illness that's being missed. Dr. Patterson also shares her own delayed Lyme disease diagnosis and what she wishes every parent knew before tick season hits. Topics Covered In This Episode: Why Tick Season Feels Scarier Than Ever Why Lyme Disease Is Called "The Great Imitator" Different Tick Species, Co-Infections, and Testing Gaps The Bullseye Rash Myth (Only Half of Cases Get One) Tick Prevention: Clothing, Sprays, and Checks That Work How Often to Check Kids and Pets for Ticks What to Do the Moment You Find a Tick Symptoms to Watch For After a Tick Bite What Every Parent Gets Wrong About Lyme Disease Show Notes: Click here to learn more about Dr. Elana Roumell's Doctor Mom Membership, a membership designed for moms who want to be their child's number one health advocate! Click here to explore Steph Greunke, RD's Mindset and Metabolism Substack, nuanced discussions on fat loss and behavior change for women. Watch this episode on our Youtube channel @medschoolformoms Listen to today's episode on our website I am a nationally and internationally recognized naturopathic physician. I am an expert in Lyme disease and other tick borne-illnesses, autoimmune conditions, PANS/PANDAS, women's health and anxiety and depression. I run a large and growing multi-disciplinary integrative medical practice composed of naturopathic physicians, nutritionist, physical therapist and acupuncture. Due to my record of clinical success, I also work as a success coach to other integrative practitioners and help them to grow and scale their businesses so that they may spread and have influence in the world of integrative medicine. I'm a sought-after speaker appearing frequently on the lecture stage, television, print, podcasts and radio. I've served in many leadership capacities, most recently as the President of the American Association of Naturopathic Physicians. This Episode's Sponsors Discover for yourself why Needed is trusted by 15,000+ women's health practitioners, including Dr. Elana and Steph. Needed supports optimal health and nourishment throughout the Motherspan--from preconception through perimenopause. Enjoy their range of practitioner-formulated, third-party tested supplements and get 20% off with code DOCTORMOM. Visit thisisneeded.com Active Skin Repair is a must-have for everyone to keep themselves and their families healthy and clean. Keep a bottle in the car to spray your face after removing your mask, a bottle in your medicine cabinet to replace your toxic first aid products, and one in your outdoor pack for whatever life throws at you. Use code DOCTORMOM to receive 20% off your order + free shipping (with $50 minimum purchase). Visit BLDGActive.com to order. INTRODUCE YOURSELF to Steph and Dr. Elana on Instagram. They can't wait to meet you! @stephgreunke @drelanaroumell Please remember that the views and ideas presented on this podcast are for informational purposes only. All information presented on this podcast is for informational purposes and not intended to serve as a substitute for the consultation, diagnosis, and/or medical treatment of a healthcare provider. Consult with your healthcare provider before starting any diet, supplement regimen, or to determine the appropriateness of the information shared on this podcast, or if you have any questions regarding your treatment plan.
In 1997, scientists travelled to Tsavo, Kenya, in search of a cave believed to have been the den of the man-eating lions that terrorised railway workers in 1898.The two lions stalked Indian and African men building the Uganda Railway, dragging victims from their tents at night. It was nine months before the officer in charge of the railway project, British Lieutenant-Colonel John Henry Patterson, could kill them.Nearly a century later, a team including scientists from Chicago's Field Museum, where the now taxidermied lions are kept, returned to Tsavo to search for the cave Patterson believed had been the lions' den.Thomas Gnoske, one of the scientists on the expedition, and Alan Patterson, John Henry Patterson's grandson, speaks to Stefania Gozzer about the hunt for the cave and the enduring legend of the Tsavo lions.Eye-witness accounts brought to life by archive. Witness History is for those fascinated by and curious about the past. We take you to the events that have shaped our world through the eyes of the people who were there. For nine minutes every day, we take you back in time and all over the world, to examine wars, coups, scientific discoveries, cultural moments and much more. Recent episodes explore everything from how the Excel spreadsheet was developed, the creation of cartoon rabbit Miffy and how the sound barrier was broken.We look at the lives of some of the most famous leaders, artists, scientists and personalities in history, including: the moment Reagan and Gorbachev met in Geneva, Haitian singer Emerante de Pradines' life and Omar Sharif's legendary movie entrance in Lawrence of Arabia.You can learn all about fascinating and surprising stories, like the invention of a stent which has saved lives around the world; the birth of the G7; and the meeting of Maldives' ministers underwater. We cover everything from World War Two and Cold War stories to Black History Month and our journeys into space.(Photo: Tsavo lions in Chicago. Credit: Field Museum of Natural History)
Jim Norton and Matt Serra look ahead to Saturday's UFC Belgrade card while catching up with two fighters coming off statement finishes in Abu Dhabi.First, 10th-ranked heavyweight Valter Walker joins the show after tying Demian Maia's UFC record with his fifth straight submission victory. Fresh off a first-round calf slicer win over Thomas Petersen, Walker discusses his historic leg-lock streak, the heavyweight names hovering around the title picture, and what could be next as “The Clean Monster” continues climbing the rankings.Then, Sam Patterson makes his UFC Unfiltered debut fresh off his knockout win over Santiago Ponzinibbio. Successful in his mission to deliver a fan-friendly fights on the heels of his frustrating setback against Michael “Venom” Page, Patterson reflects on his MMA beginnings and how it's set him on a path for success in the UFC's welterweight division.Finally, Jim and Matt wrap up the show by previewing and predicting a few fights from Saturday's UFC Belgrade card in Serbia.See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.