Podcasts about Neuro

Et si certaines migraines, douleurs cervicales ou dorsales, troubles digestifs, articulaires, gynécologiques, une hypersensibilité ou encore une dépression cachaient en réalité une maladie de Lyme ?

Et si l'iA rabotait notre cerveau, et nous enfermait comme dans une camisole numérique ?Ce podcast explore si l'iA modifie la plasticité de notre cerveau au point de nous en faire perdre le contrôle et devenir une « civilisation spectrale » pour citer Eric Saadin.Écoutez l'épisode complet Un neurologue face à l'iA avec Grégoire Hinzelin :

Temas: Prohibiciones, Control Social, MK-Ultra, Monarca, Programación Social, Miedo, Neuro, Necesidades, Realidades, Emisión: 17/04/2025 Temporada 11 Episodio 11 Abstract: En la actualidad, tanto los gobiernos como las creencias históricas han empleado la prohibición como un instrumento de control, con el propósito de prevenir daños en la población y manipular sus necesidades. No obstante, cabe cuestionar la verdadera eficacia de tales prohibiciones. Mónica Maciel y Salvador Gaviño Romero ponen sobre la mesa del misterio las historias, los fenómenos y un acercamiento a la verdad. Conviértete en miembro de este canal para disfrutar de ventajas: https://www.youtube.com/channel/UCovCkTauWfbvVYKbYjAYw1w/join Gracias por Suscribirte: http://bit.do/byjsq Follow en Twitter: http://bit.do/byjqZ Like en FB: http://bit.do/byjri WEB: http://www.elajoproducciones.com Podcast Ivoox: http://bit.do/bKae7 Ajófono: (+52) 56.100.56.1.56 (MX) Ajomail: elajo.producciones@gmail.com #Misterio3 Animación Intro El Ajo Producciones: Cortesía El Último Escriba Animación Intro Misterio 3: Cortesía El Último Escriba Animación Intraterreno: Cortesía El Último Escriba Música Fondo: Kevin Macleod https://incompetech.com/music/royalty-free/ Copyright Disclaimer! Title 17, US Code (Sections 107-118 of the copyright law, Act 1976): All media in this video is used for purpose of review & commentary under terms of fair use. All footage, & images used belong to their respective companies. Fair use is a use permitted by copyright statute that might otherwise be infringing. ***** Enlaces de Interés ***** Anacrónico ¡Ya Disponible! : https://a.co/d/8Z5OABJ PODCAST Dante: https://www.youtube.com/watch?v=rZkE2IKIJVc Dante Vanzetti spotify: https://open.spotify.com/artist/08MlOZSrQ6psjZbZWEVCgH?si=j6fSsfBATw-HwwjInMyOCg Dante Vanzetti YT: https://youtu.be/N8BJxFrRbGQ?si=ACbIH2GEOQoVzbkK Yasfer Cuadrante Mágico: https://yasferlvx.wixsite.com/arcano-obscuro-radio Marcos Urbex: https://youtube.com/@markoz320?si=qH2JyDW1gX2ohDH_ Mónica Canal Misterio: https://youtube.com/@proyectoguionenblancomiste3209?si=xt2T2iYtlIlwag-f Canal Vladimir Chargoy: https://www.youtube.com/@vladimirchargoy1711

Starlight Thursdays Episode 261 featuring Mixtress Pendula. It is springtime. Time for New beginnings: we've passed the vernal equinox. I wanted to give you about an hour, maybe a little more, of some high energy neurofunk drum and bass. This mix was a lot of fun to make. It's got some good energy. And I hope that the humor that runs underneath it is obvious. If nothing else it's a good something to get you going in your moves outdoors enjoying this new season.  Hope to see all of you somewhere outside at a venue this summer. And in the interim I'm frequently on Twitch as well. But I hope this mix puts a little pep in your step. Mixtress Pendula

The Homeland Security Department has canceled a Federal Workforce Initiative for Neuro diversity. The move ends a years long effort to accommodate people with conditions like autism as part of federal recruiting and retention. The initiatives were aimed at filling gaps in crucial fields like cyber security. For the latest, Federal News Network's Justin Doubleday. Learn more about your ad choices. Visit podcastchoices.com/adchoices

The science fiction trope of humans superpowered by computer and bionic implants is fast becoming a reality, and today, a startup hoping for a role in how that plays out is announcing some funding.  Phantom Neuro, which is developing a wristband-like device that gets implanted under the skin to let a person control prosthetic limbs. Learn more about your ad choices. Visit podcastchoices.com/adchoices

So, what the one thing most people don't realize about transformation? this podcast began as an assignment from Daniel's coach, he was to record it on the fly, stream of consciousness... one to discover something about himself AND to share the process with you. The result? Gold. If you're ready, you're going to get it.Be the Cure...ANNOUNCING: THE LIMITLESS EXPERIENCEDaniel D'Neuville is a peak performance coach, spiritual teacher, and master life and business coach. A Master Practitioner/Trainer in Neuro-linguistic Programming and a Consulting Hypnotist he has worked with over 10,000 people over the past 30 plus years. LINKS http://yesdaniel.com Daniel's YouTube CHANNEL FACEBOOK GROUPSPODCAST LISTENER'S FBCOMMUNITY EXTREME GRATITUDE PROJECT BassSlap Intro written and performed by bass player & producer: Miki SantamariaMiki'sYouTube Channel: https://www.youtube.com Hosted on Acast. See acast.com/privacy for more information.

In this episode of 'Just the Guys,' Jeremy and Dan pivot from their original plan due to an unexpected airline mishap, turning the moment into a meaningful exploration of their personal and professional insights on coaching from an autistic perspective. They delve into what makes effective coaching, highlighting the importance of adaptability, openness to change, and focusing on potential rather than limitations. The conversation emphasizes the courage required to question long-held beliefs and behaviors, and the necessity of cultivating self-awareness and emotional growth. Rich with relatable analogies from sports and personal experiences, Jeremy and Dan discuss overcoming resistance to change, the power of vulnerability, and the critical distinction between being open to new skills versus changing one's core identity. Whether you're seeking personal growth, better relationships, or practical strategies for life's complexities, this candid discussion offers valuable wisdom for those navigating neurodiverse relationships and personal development.

The eustachian tube connects the middle ear to the nasopharynx (back of the nose). It helps aerate the middle ear as well as equalizing pressure such as when flying in an airplane. Sometimes the eustachian tube does not function as it is supposed to and sometimes this may be related to concurrent nasal and sinus issues. Learn more about what can go wrong and how to remedy it.Michael Yong, MD, MPH, MBA, is board certified in Otolaryngology – Head & Neck Surgery, and fellowship-trained in Neurorhinology – Advanced Sinus & Skull Base Surgery. At Pacific Neuroscience Institute®, Dr. Yong provides full-service general Otolaryngology care, with an expertise in sinonasal and skull base disorders. He specializes in using minimally invasive endoscopic techniques for the treatment of nasal obstruction, smell disorders, allergic and non-allergic rhinitis, facial pain, acute and chronic sinusitis, nasal polyps, and benign and malignant tumors of the sinonasal cavities, among others. In addition, he works closely with a multidisciplinary team including his Neurosurgery colleagues to perform endoscopic endonasal surgeries that address benign and malignant skull base disorders in a way that maximizes preservation of function and quality of life.

Optic neuropathies encompass all congenital or acquired conditions affecting the optic nerve and are often a harbinger of systemic and central nervous system disorders. A systematic approach to identifying the clinical manifestations of specific optic neuropathies is imperative for directing diagnostic assessments, formulating tailored treatment regimens, and identifying broader central nervous system and systemic disorders. In this episode, Gordon Smith, MD, FAAN speaks with Lindsey De Lott, MD, MS, author of the article “Optic Neuropathies” in the Continuum® April 2025 Neuro-ophthalmology issue. Dr. Smith is a Continuum® Audio interviewer and a professor and chair of neurology at Kenneth and Dianne Wright Distinguished Chair in Clinical and Translational Research at Virginia Commonwealth University in Richmond, Virginia. Dr. De Lott is an assistant professor of neurology and ophthalmology at the University of Michigan in Ann Arbor, Michigan. Additional Resources Read the article: Optic Neuropathies Subscribe to Continuum®: shop.lww.com/Continuum Earn CME (available only to AAN members): continpub.com/AudioCME Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com Social Media facebook.com/continuumcme @ContinuumAAN Host: @gordonsmithMD Guest: @lindseydelott Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME, subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Smith: Hello, this is Dr Gordon Smith. Today I'm interviewing Dr Lindsey De Lott about her article on optic neuropathies, which appears in the April 2025 Continuum issue on neuro-ophthalmology. Lindsey, welcome to the podcast, and perhaps you can introduce yourself to our audience. Dr De Lott: Thank you, Dr Smith. My name is Lindsey De Lott and I am a neurologist and a neuro-ophthalmologist at the University of Michigan. I also serve as the section lead for the Division of Neuro-Ophthalmology, which is actually part of the ophthalmology department rather than the neurology department. And I spend a good portion of my time as a researcher in health services research, and that's now about 60% of my practice or so. Dr Smith: I'm super excited to spend some time talking with you. One, I'm a Michigan person. As we were chatting before this, I trained with Wayne Cornblath and John Trobe, and it's great to have you. I wonder if we maybe can begin- and by the way, your article is outstanding. It is such a huge topic and it was actually really fun to read, so I encourage our listeners to check it out. But you begin by talking about misdiagnosis as being a common problem in this patient population. I wonder if you can talk through why that is and if you have any pearls or pitfalls in avoiding it? Dr De Lott: Yeah, I think there's been a lot of great research looking at misdiagnosis in specific types of optic neuropathies; in particular, compressive optic neuropathies and optic neuritis. A lot of that work has come out of the group at Emory and the group at Washington University. But a lot of neuro-ophthalmologists across the country really contributed to those data. And one of the statistics that always strikes me is that, you know, for example, in patients with optic nerve sheath meningiomas, something like 70% of them are actually misdiagnosed. And a lot of those errors in diagnosis, whether it's for compressive optic neuropathy or some other type of optic neuropathy, really comes down to the way that physicians are really incorporating elements of the history in the physical. For example, in optic neuritis, we know that physicians tend to anchor pretty heavily on pain in general. And that often tends to lead them astray when optic neuritis was never the diagnosis to begin with. So, it's really overindexing on certain things and not paying attention to other features of the physical exam; for example, say presence of an afferent pupillary defect. So, I think it just really highlights the need to have a really relatively structured approach to patients that you think have an optic neuropathy when you're trying to sort of plan your diagnostic testing and your treatment. Dr Smith: I do maybe five or six weeks on our hospital service each year, and I don't know if it's just a Richmond thing, but there's always at least two people in my week who come in with an optic neuropathy or acute vision loss. How common is this in medical practice? Or neurologic practice, I should say? Dr De Lott: Optic neuropathies themselves… if you look across, unfortunately we don't have any great data that puts together all optic neuropathies and gives us an actual sort of prevalence estimate or an incidence estimate from year to year. We do have some of those data for specific types of optic neuropathies like optic neuritis and NAION, and you're probably looking around five-ish per one hundred thousand. So, these aren't that common, but at the same time they do get funneled to- often to emergency rooms and to neurologists from our ophthalmology colleagues and optometry colleagues in particular. Dr Smith: So, one other question I had before kind of diving into the topic at hand is how facile neurologists need to be in recognizing other causes of acute visual loss. I mean, we see acute visual loss as neurologists, we think optic neuropathy, right? Optic neuritis is sort of the go-to in a younger patient, and NAION in someone older. But what do neurologists need to know about other ophthalmologic causes? So, glaucoma or acute retinal disorders, for instance? Dr De Lott: Yeah, I think it's really important that neurologists are able to distinguish optic neuropathies from other causes of vision loss. And so, I would really encourage the listeners to take a look at the excellent article by Nancy Newman about vision loss in this issue where she really kind of breaks it down into vision loss that is acute and chronic and how you really think through distinguishing optic neuropathies from other causes of vision loss. But it is really important. For example, a patient with a central retinal artery occlusion may potentially be eligible for treatments. And that's very different from a patient with optic neuritis and acute vision loss. So, we want to be able to distinguish these things.  Dr Smith: So maybe we can pivot to that a little bit. Just for our listeners, our focus today is going to be on- not so much on optic neuritis, although obviously we need to talk a little bit about how we differentiate optic neuritis from non-neuritis optic neuropathies. It seems like the two most common situations we encounter are ischemic optic neuropathies and optic neuritis. Maybe you can talk a little bit about how you distinguish these two? I mean, some of it's age, some of it's risk factors, some of it's exam. What's the framework, of let's say, a fifty-year-old person comes into the emergency room with acute vision loss and you're worried about an optic neuropathy? Dr De Lott: The first step whenever you are considering an optic neuropathy is just making sure that the features are present. I think, really going back to your earlier question, making sure that the patient has the features of an optic neuropathy that we expect. So, it's not only vision loss, but it's also the presence of an apparent pupillary defect in a patient with a unilateral optic neuropathy. In a person who has a bilateral optic neuropathy, that apparent pupillary defect may not be present because it is relative. So, you really would have to have asymmetric vision loss between the two eyes. They should also have impairment of their color vision, and they're probably going to have some kind of visual field defect, whether that's central scotoma or an arcuate scotoma or an altitudinal defect that really respects the horizontal meridian. So, you want to make sure that, first and foremost, you've got a patient that really meets most of those- most of those features. And then from there, we're looking at the other features on their history. How acute is the onset of the vision loss? What is the progression over time? Is there pain associated or not associated with the vision loss? What other medical issues does the patient have? And you know, one of the things you already brought up, for example, is, what's the age of the patient? So, I'm going to be much more hesitant to make a diagnosis of optic neuritis in a much older patient or a diagnosis on the other side, of ischemic optic neuropathy, in a much younger patient, unless they have really clear features that push me in that direction. Dr Smith: I wonder if maybe you could talk a little bit about features that would push you away from optic neuritis, because, I mean, people who are over fifty do get optic neuritis- Dr De Lott: They do. Dr Smith: -and people who get ischemic optic neuropathies who are younger. So, what features would push you away from optic neuritis and towards… let's be broad, just a different type of optic neuropathy? Dr De Lott: Sure. We know that most patients with optic neuritis do have pain, but that pain is accompanied---within a few days, typically---with vision loss. So, pain alone going on for a number of days without any visual symptoms or any of those other things I listed, like the afferent papillary defect, the visual field defect, would push me away from optic neuritis. But in general, yes, most optic neuritis is indeed painful. So, the presence of optic disc edema is unfortunately one of those things that an optic neuritis may be present, may not be present, but in somebody with ischemia that is anterior---and that's the most common type of ischemic optic neuropathy, would be anterior ischemic optic neuropathy---they have to have optic disc edema for us to be able to make that diagnosis, and that is a diagnosis of NAION, or nonarteritic ischemic optic neuropathy. An APD in this case, again, that's just a feature of an optic neuropathy. It doesn't really help you to distinguish, individual field defects are going to be relatively similar between them. So then in patients, I'm also looking, like I said, at their history. So, in a patient where I'm entertaining a diagnosis of ischemic optic neuropathy, I want to make sure that they have vascular risk factors or that I'm actually doing things like measuring their blood pressure in the office if they haven't seen a physician recently or checking a lipid panel, hemoglobin A1c, those kinds of things, to look for vascular risk factors. One of the other features on exam that might push me more- again, in a patient with ischemic optic neuropathy, where it might suggest ischemia over optic neuritis, would be some other features on exam like a crowded optic disc that we sometimes will see in patients with ischemic optic neuropathy. I feel like that was a bit of a convoluted answer. Dr Smith: I thought that was a great answer. And when you say crowded optic disc, that's the- is that the “disc at risk”? Dr De Lott: That is the “disk at risk,” yes. So, crowded optic disk is really a disk that is smaller than what we see in the average population, and the average cup to disk ratio is 0.3. So, I think that's where 30% of the disk should be. So, this extra wiggle room, as I sometimes will explain to my patients. Dr Smith: And then, I wonder if you could talk a little bit about more- just more about exam, right? You raised the importance of recognizing optic disc edema. Are there aspects of that disc edema that really steer you away from optic neuritis and towards ischemia-like hemorrhages or whatnot? And then a similar question about the importance of careful visual field testing? Dr De Lott: So, on the whole, optic disc edema is optic disc edema. And you can have very severe optic neuritis with hemorrhages, cotton wool spots, which is essentially just an infarction of the retinal nerve fiber layer either overlying the disc or other parts of the retina. And ischemia, you can have some of the same features. In patients who have giant cell arteritis, which is just one form of anterior ischemic optic neuropathy, patients can have a pallid optic disc edema where the optic disc is swollen and white-looking. But on the whole, swelling is swelling. So, I would caution anyone against using the features of the optic nerve swelling to make any type of, sort of, definitive kind of diagnosis. It's worth keeping in mind, but I just- I would caution against using specific features, optic nerve swelling. And then for visual field testing, there are certain patterns that sometimes can be helpful. I think as I mentioned earlier, in patients with ischemic optic neuropathy, we'll often see an altitudinal defect where either the top half or, more commonly, the bottom half of the vision is lost. And that vision loss in the field corresponds to the area of swelling on the disk, which is really rewarding when you're actually able to see sectoral swelling of the disk. So, say the top half of the disk is swollen and you see a really dense inferior defect. And other types of optic neuropathy such as hereditary optic neuropathies, toxic and nutritional optic neuropathies, they often cause more central field loss. And in patients who have optic neuropathies from elevated intracranial pressure, so papilladema, those folks often have more subtle visual field loss in an arcuate pattern. And it's only once the optic nerves have sustained a pretty significant injury that you start to see other patterns of field loss and actual decline in visual acuity in those patients. I do think a detailed visual field assessment can often be pretty helpful as an adjunct to the rest of the exam. Dr Smith: So, we haven't talked a lot about neuroimaging, and obviously, neuroimaging is really important in patients who have optic neuritis. But how about an older patient in whom you suspect ischemic optic neuropathy? Do those patients all need a MRI scan? And if so, is it orbits and brain? How do you- how do you protocol it? Dr De Lott: You're asking such a good question, totally controversial in in some ways. And so, in patients with ischemic optic neuropathy, if you are confident in your diagnosis: the patient is over the age of fifty, they have all the vascular, you know, they have vascular risk factors. And those vascular risk factors are things like diabetes, hypertension, high blood pressure, hyperlipidemia, obstructive sleep apnea. They have a “disc at risk” in the fellow eye. They don't have pain, they don't have a cancer history. Then doing an MRI of the orbits is probably not necessary to rule out another cause. But if you aren't confident that you have all of those features, then you should absolutely do an MRI of the orbit. The MRI of the brain probably doesn't provide you with much additional information. However, if you are trying to distinguish between an ischemic optic neuropathy and, say, maybe an optic neuritis, in those patients we do recommend MRI orbits and brain imaging because the brain does provide additional information about other CNS demyelinating disorders that might be actually the cause of a patient's optic neuritis. Dr Smith: I wonder if you could talk a little bit about posterior ischemic optic neuropathy. That's much less common, and you mentioned earlier that those patients don't have optic disk edema. So, if there's a patient who has vision loss that- in a similar sort of clinical scenario that you talked about, how do you approach that and under what circumstances do we see patients who have posterior ischemic optic neuropathy? Dr De Lott: So, you're going to most often see patients with posterior ischemic optic neuropathy who, for example, have undergone a recent surgery. These are often associated with things like spinal surgeries, cardiac surgeries. And there are a number of risk factors that are associated with it. Things like blood pressure, drain surgery, the amount of blood loss, positioning of patient. And this is something that the surgeons and anesthesiologists are very sensitive to at this point in time, and many patients are often- this can be part of the normal informed consent process at this point in time since this is something that is well-recognized for specific surgeries. In those patients, though… again, unless you're really certain, for example, maybe the inpatient neurology attending and you've been asked to consult on a patient and it's very clear that they went into surgery normal, they came out of surgery with vision loss, and all the rest of the features really seem to be present. I would recommend that in those cases you think about orbital imaging, making sure you're not missing anything else. Again, unless all of the features really are present- and I think that's one of the themes, definitely, throughout this article, is really the importance of neuroimaging in helping us to distinguish between different types of optic neuropathy. Dr Smith: Yeah, I think one of the things that Eric Eggenberger talks about in his article is the need to use precise nomenclature too, which I plan on talking to him about. But I think having this very structured approach- and your article does it very well, I'll tell our listeners who haven't seen it there's a series of really great tables in the article that outline a lot of these. I wonder, Lindsey, if we can switch to talk about arteritic optic neuropathy. Is that okay? Dr De Lott: Sure. Yeah, absolutely. Dr Smith: How do you sort that out in an older patient who comes in with an ischemic optic neuropathy? Dr De Lott: Yeah. In patients who are over the age of fifty with an ischemic optic neuropathy, we always need to be thinking about giant cell arteritis. It is really a diagnosis we cannot afford to miss. If we do miss it, unfortunately, patients are likely to lose vision in their fellow eye about 1/3 to 1/2 the time. So, it is really one of those emergencies in neuro-ophthalmology and neurology. And so you want to do a thorough review systems for giant cell arteritis symptoms, things like headache, jaw claudication, myalgias, unintentional weight loss, fevers, things of that nature. You also want to check their inflammatory markers to look for evidence of an elevated ESR, elevated C-reactive protein. And then on exam, what you're going to find is that it can cause an anterior ischemic optic neuropathy, as I mentioned earlier. It can cause palette optic disc swelling. But giant cell arteritis can also cause posterior ischemic optic neuropathy. And so, it can be present without any swelling of the optic disc. And in fact, you know, you mentioned one of my mentors, John Trobe, who used to say that in a patient where you're entertaining the idea of posterior ischemic optic neuropathy, who is over the age of fifty with no optic disc swelling, you should be thinking about number one, giant cell arteritis; number two, giant cell arteritis; number three, giant cell arteritis. And so, I think that is a real take-home point is making sure that you're thinking of this diagnosis often in our patients who are over the age of fifty, have to rule it out. Dr Smith: I'll ask maybe a simple question. And presumably just about everyone who you see with a presumed ischemic optic neuropathy, even if they don't have clinical features, you at least check a sed rate. Is that true? Dr De Lott: I do. So, I do routinely check sedimentation rate and C-reactive protein. So, you need to check both. And the reason is that there are some patients who have a positive C-reactive protein but a normal sedimentation rate, so. And vice versa, although that is less common. And so both need to be checked. One other lab that sometimes can be helpful is looking at their CBC. You'll often find these patients with giant cell arteritis have elevated platelet counts. And if you can trend them over time, if you happen to have a patient that's had multiple, you'll see it sort of increasing over time. Dr Smith: I'm just thinking about how you sort things out in the middle, right? I mean, so that not all patients with GCF, sky-high sed rate and CRP…. And I'm just thinking of Dr Trobe's wisdom. So, when you're in an uncertain situation, presumably you go ahead and treat with steroids and move to biopsy. Maybe you can talk a bit about that pathway? Dr De Lott: Yeah, sure. Dr Smith: What's the definitive diagnostic process? Do you- for instance, the sed rate is sky-high, do you still get a biopsy? Dr De Lott: Yes. So, biopsy is still our gold-standard diagnosis here in the United States. I will say that is not the case in all parts of the world. In fact, many parts of Europe are moving toward using other ancillary tests in combination with labs and exam, the history, to make a definitive diagnosis of giant cell arteritis. And those tests are things like temporal artery ultrasound. We also, even though we call it temporal artery ultrasound, we actually need to image not only the temporal arteries but also the axillary arteries. The sensitivity and specificity is actually greater in those cases. And then there's high-resolution imaging of the vessels and the- both the intracranial and extracranial distributions. And both of those have shown some promise in their predictive values of patients actually having giant cell arteritis. One caution I would give to our listeners, though, is that, you know, currently in the US, temporal artery biopsy is still the gold standard. And reading the ultrasounds and the MRIs takes a really experienced radiologist. So, unless you really know the diagnostic accuracy at your institution, again, temporal artery biopsy remains the gold standard here. So, when you are considering giant cell arteritis, start the patient on steroids and- that's high dose, high dose steroids. In patients with vision loss, we use high dose intravenous methylprednisolone and then go ahead and get the biopsy. Dr Smith: Super helpful. And are there other treatments, other than steroids? Maybe how long do you keep people on steroids? And let's say you've got a patient who's, you know, diabetic or has other factors that make you want to avoid the course of steroids. Are there other options available? Dr De Lott: So, in the acute phase steroids are the only option. There is no other option. However, long term, yes, we do pretty quickly put patients on tocilizumab, which is really our first-line treatment. And I do that in conjunction with our rheumatology colleagues, who are incredibly helpful in managing and monitoring the tocilizumab for our patients. But when you're seeing the patients, you know, whether it's in the emergency room or in the hospital, those patients need steroids immediately. There are other steroid-sparing agents that have been tried, but the efficacy is not as good as tocilizumab. So, the American College of Rheumatology is really recommending tocilizumab as our first line steroid-sparing agent at this point. Dr Smith: Outstanding. So again, I will refer our listeners to your article. It's just chock-full of great stuff. This has been a great conversation. Thank you so much for joining me today. Dr De Lott: Thank you, Dr Smith. I really appreciate it.  Dr Smith: The pleasure has been all mine, and I know our listeners will be enjoying this as well. Again, today I've been interviewing Dr Lindsey De Lott about her article on optic neuropathies, which appears in the most recent issue of Continuum on neuro-ophthalmology. Be sure to check out Continuum Audio episodes from this and other issues. I already mentioned Dr Eggenberger and I will be talking about optic neuritis, which will be a great companion to this discussion. Listeners, thank you for joining us today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use the link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

This week, Kirsty speaks with Dr. Mandi Freger, author of "From Exhausted to Energized" and a licensed behavioral specialist. Together, they speak about children with mental illnesses with a focus on ASD, energy psychology, and ways to help your child through their difficult behaviors.   Show Notes Get Mandi's Book! https://link.edgepilot.com/s/f499c0c8/9PoHSmJjNkqyZ0jKK_W5Lg?u=http://www.mandifreger.com/ https://link.edgepilot.com/s/4cf16363/zNuXqkVI90WXXiVjEcjOjg?u=https://www.facebook.com/mandifreger https://link.edgepilot.com/s/ce69036a/Ye2teQ3J-ESexIZTRvrhxA?u=http://www.linkedin.com/in/mandifreger https://www.attachmenttheoryinaction.com/  https://www.tkcchaddock.org/events/atiawebinar/  https://shop.tkcchaddock.org/  https://www.facebook.com/share/g/19Xm5Nhk2K/  https://www.facebook.com/TKCChaddock https://www.linkedin.com/company/tkcchaddock/posts/?feedView=all https://www.instagram.com/tkcchaddock https://www.facebook.com/krugglesatchaddock https://www.linkedin.com/in/kirstynolan84/

Coach Ted talks about enabling the formation of the pathways that allow for transformation and growth.

Jeff Walter, DPT, NCS, returns to Neuro Navigators to share even more clinical practice tips for clinicians who are both new and experienced with vestibular rehab. Host JJ Mowder-Tinney and Jeff dive into the vestibular management hidden gems that could be the strategies to transform your clinical practice. From alternative benign paroxysmal positional vertigo (BPPV) assessment techniques to under-recognized treatments for Meniere's disease to emerging treatments for vestibular-related imbalance and falls, you'll walk away with practical tools you can apply immediately. Whether you are a physical or occupational therapy practitioner, this episode is for you. Don't miss this engaging discussion filled with actionable takeaways to enhance your confidence in treating dizziness and balance disorders.Learning ObjectivesAnalyze the evidence regarding optimal management of vestibular disordersApply evidence-based, practical strategies to actionably address the efficient evaluation and treatment of vestibular-related dizzinessSolve patient case scenarios involving frequent falls and disabling vertigo to support participation in instrumental activities of daily living (IADLs), such as community mobility and home managementTimestamps(00:00:00) Welcome(00:00:15) Welcome back, guest Jeff Walter, DPT, NCS(00:00:55) Jeff's background and work at Geisinger Medical Center(00:02:11) Evolution of Jeff's vestibular specialization(00:03:12) Overview: tips for clinicians with foundational vestibular knowledge(00:04:42) Sidelying test: what it is and why to use it(00:06:54) How to perform the sidelying test with exact head positioning cues(00:08:21) Embedding the sidelying test into functional mobility assessments(00:11:55) Splinting the patient's head: comfort and compliance tips(00:13:21) Half Dix-Hallpike: identifying short-arm posterior canal BPPV(00:16:40) Flashlight fixation-blocking: a goggle-free nystagmus test(00:18:11) When to use it and how to prep the patient(00:25:10) Mastoid vibration test: screening for vestibular hypofunction(00:26:00) Interpretation: direction-fixed nystagmus and its implications(00:26:40) When and why to use mastoid vibration (TBI, falls, etc.)(00:34:04) Gentamicin injections: managing Meniere's-related vertigo(00:40:00) Vestibular drop attacks (Tumarkin events): signs and screening tips(00:42:30) Real-life example and how to follow up when falls are unexplained(00:46:00) Vibrotactile belt: a future-forward sensory substitution device(00:47:00) Who it's for, how it works, and early user feedback(00:52:50) Wrapping up: Jeff's top takeaways for novice and experienced physical and occupational therapy practitioners(00:56:25) Superpower time: Jeff's vestibular-themed wishes(00:58:00) Closing remarks and where to listen to Episode 1Resources Mentioned in EpisodeAlonso, S. M., & Caletrío, Á. B. (2024). Clinical Advancements in Skull Vibration-Induced Nystagmus (SVIN) over the Last Two Years: A Literature Review. Journal of Clinical Medicine, 13(23), 7236.Neuro Naviagators is brought to you by Medbridge. If you'd like to earn continuing education credit for listening to this episode and access bonus takeaway handouts, log in to your Medbridge account and navigate to the course where you'll find accreditation details. If applicable, complete the post-course assessment and survey to be eligible for credit. The takeaway handout on Medbridge gives you the key points mentioned in this episode, along with additional resources you can implement into your practice right away.To hear more episodes of Neuro Naviagators, visit https://www.medbridge.com/neuro-navigatorsIf you'd like to subscribe to Medbridge, visit https://www.medbridge.com/pricing/

It is Autism Acceptance and Action Month, and we have Dave Glick as our guest neurodivergent coach and clinician.Today, Dr. Stephanie and Barbara ask David a few questions about behaviors.We ask:Can you differentiate freeze and fawning?What are your thoughts on pervasive demand avoidance?What behaviors can actually change?Can those on the spectrum learn regulation and relational skills? About our Guest & Colleague:Dave writes on his website: https://triadpsych.org/therapists/david-glick/My specialties are quite extensive and diverse, as I have two degrees, one in Education and the other in Social Work. I was originally trained in behaviorism and usually merge behavior therapies with personality trait theories. This approach, while demanding, usually produces very positive results for my clients. My practice is a safe place where people can share and be open. Once trust is established, it becomes a virtual Swiss army knife, in that the therapy is adaptable and useful regardless of the situation. Essentially, I want my clients to be empowered with a new sense of self and confident in their abilities.

Staying up on the latest evidence in neuro rehab? This interview is for you!  Erin Gallardo, PT, DPT, NCS interviewed Dr. Timothy Faw, a neuroscientist and physical therapist about his research path - where he's been and where it's going. Tim discusses the work his labs have done in a few areas including the plasticity of gray matter and myelin and the implications on motor learning and movement in spinal cord injury. He also discusses tactics on eccentric gait training - that are basically the opposite of what we think of as high intensity - and how they are impacting outcomes. More recently his work has led him to study genetic factors in neurologic recovery including the markers for better or worse outcomes and drug trials. And to ensure listeners leave the conversation with action items, he'll provide his takeaways and clinical pearls that you can start incorporating immediately. Tim emphasizes that rehabilitation is about more than just intensity—it's about creating meaningful, challenging, and novel movement experiences that engage the brain's adaptive capabilities. Clinicians and students interested in cutting-edge neuro rehab approaches can contact Dr. Faw at tfaw@som.umaryland.edu for more information. X - @timothy_faw Faculty Website - https://www.medschool.umaryland.edu/profiles/faw-timothy/

Kentaro Yoshimura and his business partner Ryan Chen built Neuro from zero to nearly $100 million in just ten short years. This is just part of the story...Check out Neuro here:https://neurogum.com/Support the show

Send us a textDr. Ashley Aaroe joins us today to talk all about Neuro-oncology, from brain tumor classification, to complications of chemotherapy and immunotherapy, to the importance of patient advocacy.Find her on X/Twitter.  Check out our website at www.theneurotransmitters.com to sign up for emails, classes, and quizzes! Would you like to be a guest or suggest a topic? Email us at contact@theneurotransmitters.com Follow our podcast channel on

While FreshEd is away, we are going to replay some of our favourite episodes about education in a digital society. -- Today we unpack the neuro-affective turn in education. With me are Kirsi Yliniva and Audrey Bryan. Kirsi Yliniva is a PhD researcher and university teacher in the Faculty of Education and Psychology at the University of Oulu. Audrey Bryan is an associate professor of sociology in the School of Human Development at Dublin City University's Institute of Education. Together with Kristiina Brunila, they have recently published the article “‘The future we want'? – The ideal twenty-first century learner and education's neuro-affective turn.” https://freshedpodcast.com/yliniva-bryan/ -- Get in touch! Twitter: @FreshEdpodcast Facebook: FreshEd Email: info@freshedpodcast.com Support FreshEd: www.freshedpodcast.com/support/

On this episode, we welcome Gavin Stone, author of the bestselling book How to Tell if Someone is Lying. Gavin served as a security and intelligence covert specialist. He has over 20 years of applied experience, globally deployed by government organizations such as the British Ministry of Defence, corporations, and ultra-high-net-worth VIPs. The post 391: The Secret Methods Used by Government Intelligence Agencies to Detect Lying first appeared on Persuasion by the Pint.

In this episode of The Med-Tech Talent Lab podcast, host Mitch Robbins interviews Mary McNamara-Cullinane, Vice President of Regulatory Affairs at ClearPoint Neuro, about her 25+ year journey in the medical device regulatory space.Key Highlights:Mary shares her background as the youngest of eight children and how her early work ethic shaped her career pathLearn how Mary transitioned from research to regulatory affairs through roles at Brigham and Women's, CRBARD, and a 17-year consulting careerDiscover what makes an outstanding regulatory professional in today's environmentUnderstand the challenges regulatory executives face when balancing company goals with regulatory requirementsPractical Takeaways:Why proactive research and understanding the "why" behind regulations separates exceptional regulatory professionalsThe importance of managing leadership expectations around FDA timelinesWhy submitting complete FDA submissions is more efficient than rushing deficient onesThoughts on remote work effectiveness in regulatory rolesMary also discusses ClearPoint Neuro's exciting work with gene therapy companies and their recent partnership with PTC Therapeutics to address rare diseases in children.Whether you're a regulatory professional looking to advance your career or an executive trying to better understand the regulatory landscape, this conversation offers valuable insights from a seasoned industry leader.Mary McNamara-Cullinane on LinkedIn: https://www.linkedin.com/in/marymcnamaracullinane/ClearPoint Neuro on the web:https://www.clearpointneuro.com/

Hey there, Shiny Minds!

Diagnosing and differentiating among the many possible localizations and causes of vision loss is an essential skill for neurologists. The approach to vision loss should include a history and examination geared toward localization, followed by a differential diagnosis based on the likely location of the pathophysiologic process.  In this episode, Aaron Berkowitz, MD, PhD, FAAN speaks with Nancy J. Newman, MD, FAAN, author of the article “Approach to Vision Loss” in the Continuum® April 2025 Neuro-ophthalmology issue.  Dr. Berkowitz is a Continuum® Audio interviewer and a professor of neurology at the University of California San Francisco in the Department of Neurology and a neurohospitalist, general neurologist, and clinician educator at the San Francisco VA Medical Center at the San Francisco General Hospital in San Francisco, California.  Dr. Newman is a professor of ophthalmology and neurology at the Emory University School of Medicine in Atlanta, Georgia.  Additional Resources Read the article: Approach to Vision Loss Subscribe to Continuum®: shop.lww.com/Continuum  Earn CME (available only to AAN members): continpub.com/AudioCME  Continuum® Aloud (verbatim audio-book style recordings of articles available only to Continuum® subscribers): continpub.com/Aloud More about the American Academy of Neurology: aan.com  Social Media facebook.com/continuumcme  @ContinuumAAN  Host: @AaronLBerkowitz  Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about earning CME subscribing to the journal, and exclusive access to interviews not featured on the podcast. Dr Berkowitz: This is Dr Aaron Berkowitz, and today I'm interviewing Dr Nancy Newman about her article on the approach to visual loss, which she wrote with Dr Valerie Biousse. This article appears in the April 2025 Continuum issue on neuro-ophthalmology. Welcome to the podcast, Dr Newman. I know you need no introduction, but if you wouldn't mind introducing yourself to our listeners. Dr Newman: Sure. My name's Nancy Newman. I am a neurologist and neuro-ophthalmologist, professor of ophthalmology and neurology at the Emory University School of Medicine in Atlanta, Georgia. Dr Berkowitz: You and your colleague Dr Biousse have written a comprehensive and practical article on the approach to visual loss here. It's fantastic to have this article by two of the world's leading experts and best-known teachers in neuro-ophthalmology. And so, readers of this article will find extremely helpful flow charts, tables and very nuanced clinical discussion about how to make a bedside diagnosis of the cause of visual loss based on the history exam and ancillary testing. We'll talk today about that important topic, and excited to learn from you and for our listeners to learn from you. To begin, let's start broad. Let's say you have a patient presenting with visual loss. What's your framework for the approach to this common chief concern that has such a broad differential diagnosis of localizations and of causes? Where do you start when you hear of visual loss? How do you think about this chief concern? Dr Newman: Well, it's very fun because this is the heart of being a neurologist, isn't it? Nowhere in the nervous system is localization as important as the complaint of vision loss. And so, the key, as any neurologist knows, is to first of all figure out where the problem is. And then you can figure out what it is based on the where, because that will limit the number of possibilities. So, the visual system is quite beautiful in that regard because you really can exquisitely localize based on figuring out where things are. And that starts with the history and then goes to the exam, in particular the first localization. So, you can whittle it down to the more power-for-your-buck question is, is the vision lost in one eye or in two eyes? Because if the vision loss clearly, whether it's transient or persistent, is in only one eye, then you only have to think about the eyeball and the optic nerve on that side. So, think about that. Why would you ever get a brain MRI? I know I'm jumping ahead here, but this is the importance of localization. Because what you really want to know, once you know for sure it's in one eye, is, is it an eyeball problem---which could be anything from the cornea, the lens, the vitreous, the retina---or is it an optic nerve problem? The only caveat is that every once in a while, although we trust our patients, a patient may insist that a homonymous hemianopia, especially when it's transient, is only in the eye with the temporal defect. So that's the only caveat. But if it's in only one eye, it has to be in that side eyeball or optic nerve. And if it's in two eyes, it's either in both eyeballs or optic nerves, or it's chiasmal or retrochiasmal. So that's the initial approach and everything about the history should first be guided by that. Then you can move on to the more nuanced questions that help you with the whats. Once you have your where, you can then figure out what the whats are that fit that particular where. Dr Berkowitz: Fantastic. And your article with Dr Biousse has this very helpful framework, which you alluded to there, that first we figure out, is it monocular or binocular? And we figure out if it's a transient or fixed or permanent deficit. So, you have transient monocular, transient binocular, fixed monocular, fixed binocular. And I encourage our listeners to seek out this article where you have a table for each of those, a flow chart for each of those, that are definitely things people want to have printed out and at their desk or on their phone to use at the bedside. Very helpful. So, we won't be able to go through all of those different clinical presentations in this interview, but let's focus on monocular visual loss. As you just mentioned, this can be an eye problem or an optic nerve problem. So, this could be an ophthalmologic problem or a neurologic problem, right? And sometimes this can be hard to distinguish. So, you mentioned the importance of the history. When you hear a monocular visual loss- and with the caveat, I said you're convinced that this is a monocular visual problem and not a visual field defect that may appear. So, the patient has a monocular deficit, how do you approach the history at trying to get at whether this is an eye problem or an optic nerve problem and what the cause may be? Dr Newman: Absolutely. So, the history at that point tends not to be as helpful as the examination. My mentor used to say if you haven't figured out the answer to the problem after your history, you're in trouble, because that 90% of it is history and 10% is the exam. In the visual system, the exam actually may have even more importance than anywhere else in the neurologic examination. And we need as neurologists to not have too much hubris in this. Because there's a whole specialty on the eyeball. And the ophthalmologists, although a lot of their training is surgical training that that we don't need to have, they also have a lot of expertise in recognizing when it's not a neurologic problem, when it's not an optic neuropathy. And they have all sorts of toys and equipment that can very much help them with that. And as neurologists, we tend not to be as versed in what those toys are and how to use them. So, we have to do what we can do. Your directive thalmoscope, I wouldn't throw it in the garbage, because it's actually helpful to look at the eyeball itself, not just the back of the eye, the optic nerve and retina. And we'll come back to that, but we have in our armamentarium things we can do as neurologists without having an eye doctor's office. These include things like visual acuity and color vision, confrontation, visual fields. Although again, you have to be very humble. Sometimes you're lucky; 30% of the time it's going to show you a defect. It has to be pretty big to pick it up on confrontation fields. And then as we say, looking at the fundus. And you probably know that myself and Dr Biousse have been on somewhat of a crusade to allow the emperor's new clothes to be recognized, which is- most neurologists aren't very comfortable using the direct ophthalmoscope and aren't so comfortable, even if they can use it, seeing what they need to see. It's hard. It's really, really hard. And it's particularly hard without pupillary dilation. And technology has allowed us now with non-mydriatic cameras, cameras that are incredible, even through a small pupil can take magnificent pictures of the back of the eye. And who wouldn't rather have that? And as their cost and availability- the cost goes down and their availability goes up. These cameras should be part of every neurology office and every emergency department. And this isn't futuristic. This is happening already and will continue to happen. But over the next five years or so… well, we're transitioning into that. I think knowing what you can do with the direct ophthalmoscope is important. First of all, if you dial in plus lenses, you can't be an ophthalmologist, but you can see media opacities. If you can't see into the back of the eye, that may be the reason the patient can't see out. And then just seeing if someone has central vision loss in one eye, it's got to be localized either to the media in the axis of vision; or it's in the macula, the very center of the retina; or it's in the optic nerve. So, if you get good at looking at the optic nerve and then try to curb your excitement when you saw it and actually move a little temporally and take a look at the macula, you're looking at the two areas. Again, a lot of ophthalmologists these days don't do much looking with the naked eye. They actually do photography, and they do what's called OCT, optical coherence tomography, which especially for maculopathies, problems in the macula are showing us the pathology so beautifully, things that used to be considered subtle like central serous retinopathy and other macula. So, I think having a real healthy respect for what an eye care provider can do for you to help screen away the ophthalmic causes, it's very, very important to have a patient complaining of central vision loss, even if they have a diagnosis like multiple sclerosis, you expect that they might have an optic neuritis… they can have retinal detachments and other things also. And so, I think every one of these patients should be seen by an eye care provider as well. Dr Berkowitz: Thank you for that overview. And I feel certainly as guilty as charged here as one of many neurologists, I imagine, who wish we were much better and more comfortable with fundoscopy and being confident on what we see. But as you said, it's hard with the direct ophthalmoscope and a non-dilated exam. And it's great that, as you said, these fundus photography techniques and tools are becoming more widely available so that we can get a good look at the fundus. And then we're going to have to learn a lot more about how to interpret those images, right? If we haven't been so confident in our ability to see the fundus and analyze some of the subtle abnormalities that you and your colleagues and our ophthalmology colleagues are more familiar with. So, I appreciate you acknowledging that. And I'm glad to hear that coming down the pipeline, there are going to be some tools to help us there. So, you mentioned some of the things you do at the bedside to try to distinguish between eye and optic nerve. Could you go into those in a little bit more detail here? How do you check the visual fields? For example, some people count fingers, some people wiggle fingers, see when the patient can see. How should we be checking visual fields? And what are some of the other bedside tasks you use to decide this is probably going to end up being in the optic nerve or this seems more like an eye? Dr Newman: Of course. Again, central visual acuity is very important. If somebody is older than fifty, they clearly will need some form of reading glasses. So, keeping a set of plus three glasses from cheapo drugstore in your pocket is very helpful. Have them put on their glasses and have them read an ear card. It's one of the few things you can actually measure and examine. And so that's important. The strongest reflex in the body and I can have it duke it out with the peripheral neurologists if they want to, it's not the knee jerk, it's looking for a relative afferent pupillary defect. Extremely important for neurologists to feel comfortable with that. Remember, you cut an optic nerve, you're not going to have anisocoria. It's not going to cause a big pupil. The pupils are always equal because this is not an efferent problem, it's an afferent problem, an input problem. So basically, if the eye has been injured in the optic nerve and it can't get that information about light back into the brain, well, the endoresfol nuclei, both of them are going to reset at a bigger size. And then when you swing over and shine that light in the good optic nerve, the good eye, then the brain gets all this light and both endoresfol nuclei equally set those pupils back at a smaller size. So that's the test for the relative afferent pupillary defect. When you swing back and forth. Of course, when the light falls on the eye, that's not transmitting light as well to the brain, you're going to see the pupil dilate up. But it's not that that pupil is dilating alone. They both are getting bigger. It's an extremely powerful reflex for a unilateral or asymmetric bilateral optic neuropathy. But what you have to remember, extremely important, is, where does our optic nerve come from? Well, it comes from the retinal ganglion cells. It's the axons of the retinal ganglion cells, which is in the inner retina. And therefore inner retinal disorders such as central retinal artery occlusion, ophthalmic artery occlusion, branch retinal artery occlusion, they will also give a relative afferent pupillary defect because you're affecting the source. And this is extremely important. A retinal detachment will give a relative afferent pupillary defect. So, you can't just assume that it's optic nerve. Luckily for us, those things that also give a relative afferent pupillary defect from a retinal problem cause really bad-looking retinal disease. And you should be able to see it with your direct ophthalmoscope. And if you can't, you definitely will be able to see it with a picture, a photograph, or having an ophthalmologist or optometrist take a look for you. That's really the bedside. You mentioned confrontation visual fields. I still do them, but I am very, very aware that they are not very sensitive. And I have an extremely low threshold to- again, I have something in my office. But if I were a general neurologist, to partner with an eye care specialist who has an automated visual field perimeter in their office because it is much more likely to pick up a deficit. Confrontation fields. Just remember, one eye at a time. Never two eyes at the same time. They overlap with each other. You're going to miss something if you do two eyes open, so one eye at a time. You check their field against your field, so you better be sure your field in that eye is normal. You probably ought to have an automated perimetry test yourself at some point during your career if you're doing that. And remember that the central thirty degrees is subserved by 90% of our fibers neurologically, so really just testing in the four quadrants around fixation within the central 30% is sufficient. You can present fingers, you don't have to wiggle in the periphery unless you want to pick up a retinal detachment. Dr Berkowitz: You mentioned perimetry. You've also mentioned ocular coherence tomography, OCT, other tests. Sometimes we think about it in these cases, is MRI one of the orbits? When do you decide to pursue one or more of those tests based on your history and exam? Dr Newman: So again, it sort of depends on what's available to you, right? Most neurologists don't have a perimeter and don't have an OCT machine. I think if you're worried that you have an optic neuropathy, since we're just speaking about monocular vision loss at this point, again, these are tests that you should get at an office of an eye care specialist if you can. OCT is very helpful specifically in investigating for a macular cause of central vision loss as opposed to an optic nerve cause. It's very, very good at picking up macular problems that would be bad enough to cause a vision problem. In addition, it can give you a look at the thickness of the axons that are about to become the optic nerve. We call it the peripapillary retinal nerve fiber layer. And it actually can look at the thickness of the layer of the retinal ganglion cells without any axons on them in that central area because the axons, the nerve fiber layer, bends away from central vision. So, we can see the best we can see. And remember these are anatomical measurements. So, they will lag, for the ganglion cell layer, three to four weeks behind an injury, and for the retinal nerve fiber, layer usually about six weeks behind an entry. Whereas the functional measurements, such as visual acuity, color vision, visual fields, will be immediate on an injury. So, it's that combination of function and anatomy examination that makes you all-powerful. You're very much helped by the two together and understanding where one will be more helpful than the other. Dr Berkowitz: Let's say we've gotten to the optic nerve as our localization. Many people jump to the assumption it's the optic nerve, it's optic neuritis, because maybe that's the most common diagnosis we learn in medical school. And of course, we have to sometimes, when we're teaching our students or trainees,  say, well, actually, not all optic nerve disease, optic neuritis, we have to remember there's a broader bucket of optic neuropathy. And I remember, probably I didn't hear that term until residency and thought, oh, that's right. I learned optic neuritis. Didn't really learn any of the other causes of optic nerve pathology in medical school. And so, you sort of assume that's the only one. And so you realize, no, optic neuropathy has a differential diagnosis beyond optic neuritis. Neuritis is a common cause. So how do you think about the “what” once you've localized to the optic nerve, how do you think about that? Figure out what the cause of the optic neuropathy is? Dr Newman: Absolutely. And we've been trying to convince neuro-radiologists when they see evidence of optic nerve T2 hyperintensity, that just means damage to the optic nerve from any cause. It's just old damage, and they should not put in their read consistent with optic neuritis. But that's a pet peeve. Anyway, yes, the piece of tissue called the optic nerve can be affected by any category of pathophysiology of disease. And I always suggest that you run your categories in your head so you don't leave one out. Some are going to be more common to be bilateral involvement like toxic or metabolic causes. Others will be more likely unilateral. And so, you just run those guys. So, in my mind, my categories always are compressive-slash-infiltrative, which can be neoplastic or non-neoplastic. For example, an ophthalmic artery aneurysm pressing on an optic nerve, or a thyroid, an enlarged thyroid eye muscle pressing on the optic nerve. So, I have compressive infiltrative, which could be neoplastic or not neoplastic. I have inflammatory, which can be infectious. Some of the ones that can involve the optic nerve are syphilis, cat scratch disease. Or noninfectious, and these are usually your autoimmune such as idiopathic optic neuritis associated with multiple sclerosis, or MOG, or NMO, or even sarcoidosis and inflammation. Next category for me would be vascular, and you can have arterial versus venous in the optic nerve, probably all arterial if we're talking about causes of optic neuropathy. Or you could have arteritic versus nonarteritic with the vascular, the arteritic usually being giant cell arteritis. And the way the optic nerve circulation is, you can have an anterior ischemic optic neuropathy or a posterior ischemic optic neuropathy defined by the presence of disc edema suggesting it's anterior, the front of the optic nerve, or not, suggesting that it's retrobulbar or posterior optic nerve. So what category am I- we mentioned toxic, metabolic nutritional. And there are many causes in those categories of optic neuropathy, usually bilateral. You can have degenerative or inherited. And there are causes of inherited optic neuropathies such as Leber hereditary optic neuropathy and dominant optic atrophy. And then there's a group I call the mechanical optic neuropathies. The obvious one is traumatic, and that can happen in any piece of tissue. And then the other two relate to the particular anatomy of the eyeball and the optic nerve, and the fact that the optic nerve is a card-carrying member of the central nervous system. So, it's not really a nerve by the way, it's a tract. Think about it. Anyway, white matter tract. It is covered by the same fluid and meninges that the rest of the brain. So, what mechanically can happen? Well, you could have an elevated intraocular pressure where that nerve inserts. That's called glaucoma, and that would affect the front of the optic nerve. Or you can have elevated intracranial pressure. And if that's transmitted along the optic nerve, it can make the front of the optic nerve swell. And we call that specifically papilledema, optic disk edema due specifically to raised intracranial pressure. We actually even can have low intraocular pressure cause something called hypotony, and that can actually even give an optic neuropathy the swelling of the optic nerve. So, these are the mechanical. And if you were to just take that list and use it for any piece of tissue anywhere, like the heart or the kidney, you can come up with your own mechanical categories for those, like pericarditis or something like that. And then all those other categories would fit. But of course, the specific causes within that pathophysiology are going to be different based on the piece of tissue that you have. In this case, the optic nerve. Dr Berkowitz: In our final moments here, we've talked a lot about the approach to monocular visual loss. I think most neurologists, once we find a visual field defect, we breathe a sigh of relief that we know we're in our home territory here, somewhere in the visual task base that we've studied very well. I'm not trying to distinguish ocular causes amongst themselves or ocular from optic nerve, which can be very challenging at the bedside. But one topic you cover in your article, which I realized I don't really have a great approach to, is transient binocular visual loss. Briefly here, since we're running out of time, what's your approach to transient binocular visual loss?  Dr Newman: We assume with transient binocular vision loss that we are not dealing with a different experience in each eye, because if you have a different experience in each eye, then you're dealing with bilateral eyeball or optic nerve. But if you're having the same experience in the two eyes, it's equal in the two eyes, then you're located. You're located, usually, retro chiasmally, or even chiasm if you have pituitary apoplexy or something. So, all of these things require imaging, and I want to take one minute to talk about that. If you are sure that you have monocular vision loss, please don't get a brain MRI without contrast. It's really useless. Get a orbital MRI with contrast and fat suppression techniques if you really want to look at the optic nerve. Now, let's say you you're convinced that this is chiasmal or retrochiasmal. Well then, we all know we want to get a brain MRI---again, with and without contrast---to look specifically where we could see something. And so, if it's persistent and you have a homonymous hemianopia, it's easy, you know where to look. Be careful though, optic track can fool you. It's such a small little piece, you may miss it on the MRI, especially in someone with MS. So really look hard. There's very few things that are homonymous hemianopias MRI negative. It may just be that you didn't look carefully enough. And as far as the transient binocular vision loss, again, remember, even if it's persistent, it has to be equal vision in the two eyes. If there's inequality, then you have a superimposed anterior visual pathway problem, meaning in front of the chiasm on the side that's worse. The most common cause of transient binocular vision loss would be a form of migraine. The visual aura of migraine usually is a positive phenomenon, but sometimes you can have a homonymous hemianopic persistent defect that then ebbs and flows and goes away. Usually there's buildup, lasts maybe fifteen minutes and then it goes away, not always followed by a headache. Other things to think of would be transient ischemic attack in the vertebra Basler system, either a homonymous hemianopia or cerebral blindness, what we call cortical blindness. It can be any degree of vision loss, complete or any degree, as long as the two eyes are equal. That should last only minutes. It should be maximum at onset. There should be no buildup the way migraine has it. And it should be gone within less than ten minutes, typically. After fifteen, that's really pushing it. And then you could have seizures. Seizures can actually be the aura of a seizure, the actual ictal phenomenon of a seizure, or a postictal, almost like a todd's paralysis after a seizure. These events are typically bright colors and flashing, and they last usually seconds or just a couple of minutes at most. So, you can probably differentiate them. And then there are the more- less common but more interesting things like hyperglycemia, non-ketonic hyperglycemia can give you transient vision loss from cerebral origin, and other less common things like that. Dr Berkowitz: Fantastic. Although we've talked about many pearls of clinical wisdom here with you today, Dr Newman, this is only a fraction of what we can find in your article with Dr Biousse. We focused here on monocular visual loss and a little bit at the end here on binocular visual loss, transient binocular visual loss. But thank you very much for your article, and thank you very much for taking the time to speak with us today. Again, today I've been interviewing Dr Nancy Newman about her article with Dr Valerie Biousse on the approach to visual loss, which appears in the most recent issue of Continuum on neuro-ophthalmology. Be sure to check out Continuum audio episodes from this and other issues. Thank you so much to our listeners for joining us today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in-depth and clinically relevant information important for neurology practitioners. Use this link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

This week Kate, Gary, Mark and Henry discuss discontinuation of benzodiazepines and treatment of insomnia, the value of baseline cognitive testing of college athletes, vonoprazan vs PPI for preventing and treating ulcers, and whether herpes zoster vaccine reduces dementia risk.Show links:Essential Evidence Plus: www.essentialevidenceplus.comTapering benzos: https://pubmed.ncbi.nlm.nih.gov/39374004/ Baseline neuro eval for athletes: pubmed.ncbi.nlm.nih.gov/39741470/ David Kaufman, “We Need You in the Locker Room” https://thesagergroup.net/books/in-the-locker-room Vonoprazan vs PPIs for ulcers: https://pubmed.ncbi.nlm.nih.gov/39294424/ Zoster and dementia: https://pubmed.ncbi.nlm.nih.gov/40175543/ 

Join Dr. Stephanie and Tiffany in an interactive discussion that will help you delve deeper into understanding your rights as a parent with a school-age child with disabilities. You will have a solid understanding of the variety of support plans, how to navigate the school teams, and how to ensure your child gets everything they need to succeed in school!About our Guest:Tiffany Yandle is a non-attorney Special Education Advocate and President of In Bloom Advocacy. She has a 15+ year history of teaching Special Education and school administration in North Carolina public schools. She is passionate about reaching an equitable, personalized education for all students. She specializes in Special Education Laws and regulations, Multi-Tiered Systems of Support, Community Engagement in schools, and Autism Spectrum Disorders.

Love as Neural Colonialism fMRI scans reveal entitled partners exhibit 34% less anterior cingulate cortex activity—the brain's empathy hub (Durvasula). This neural detachment mirrors colonial resource extraction: love becomes a transaction where emotional labor is siphoned into psychic ledgers.

Love as Neural Colonialism fMRI scans reveal entitled partners exhibit 34% less anterior cingulate cortex activity—the brain's empathy hub (Durvasula). This neural detachment mirrors colonial resource extraction: love becomes a transaction where emotional labor is siphoned into psychic ledgers.

This conversation hit me hard as my own grandmother suffered from Alzheimer's for 14 years before passing. If you're worried about cognitive decline for yourself or someone you love, this clip delivers game-changing insights. The neurologists break down why women are more susceptible to Alzheimer's, and reveal the NEURO plan - a simple framework anyone can follow to protect their brain health. They debunk the oversimplified "Type 3 Diabetes" theory and explain how up to 80% of dementia cases might be preventable through lifestyle changes. This isn't just about adding years to your life, but life to your years by protecting your most precious asset - your mind.***Thank you to my wonderful sponsors! VivoBarefoot | 'If you can't be barefoot, be Vivobarefoot'For 20% off, use code LWBW20www.vivobarefoot.comOneSkin | Topical Products for Healthy Aginghttp://oneskin.co/LWBWUse code LWBW for 15% off your first order ***Sign up to Sarah's Compassionate Cure newsletter: Science Simplified, Health Humanised. Join thousands in exploring actionable insights that prioritise compassion, clarity, and real-life impact. https://sarahmacklin.substack.com/***Let's be friends!

Video: ⁠https://mehlmanmedical.com/hy-usmle-q-1292-neuro⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠IG: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.instagram.com/mehlman_medical/⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠Telegram: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://mehlmanmedical.com/subscribe/⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠FB: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.facebook.com/mehlmanmedical⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠

Article: Why INSiGHT Scans Improve Before Symptoms DoArticle: What are the INSiGHT Scans?Episode on Neuro 'Soft Signs'Episode on NeurodetoxDr. Tony Ebel addresses a common concern among parents whose children are receiving chiropractic care for neurological issues: why do the nervous system scans show improvement while symptoms persist? Dr. Tony explains the science of inside-out healing, detailing how neurological improvement happens in stages, with symptom relief often being the last step in the healing process. He contrasts this natural healing approach with conventional medicine's symptom-focused model and provides parents with a new framework for understanding their child's healing journey.[00:00:00] IntroductionIntroduction to Dr. Tony's approach to drug-free nervous system focused healthcare[00:06:00] Understanding Neurological INSiGHT ScansExplanation of how nervous system scans work as objective functional testingIntroduction to the main question: Why scans improve before symptoms change[00:10:00] The Inside-Out Healing ProcessThe four-step sequence of dysfunction: triggers → nervous system dysfunction → system dysfunction → symptomsWhy healing must work in reverse order, with nervous system improvement happening first[00:15:00] Analogies That Explain the ProcessThe messy room analogy: medications hide symptoms like shoving mess in a closetWeather radar analogy: knowing change is coming before you can see it[00:20:00] Three Things to Track in Healing ProgressINSiGHT scans: measuring functional nervous system improvementNeurological soft signs: sleep, digestion, immune function, breathing, movementSymptoms: the conditions parents most want resolved[00:27:00] Why Scans Matter in Natural HealingHow the scanning technology allows practitioners to practice differently than conventional medicineThe sequence of healing: scans improve first, soft signs second, symptoms third[00:31:00] Encouragement for ParentsReminder that healing isn't always fast, smooth, or linearThe importance of staying the course even when progress seems slowReassurance that "your kiddo is not broken and healing is entirely possible"-- Follow us on Socials: Instagram: @pxdocs Facebook: Dr. Tony Ebel & The PX Docs Network Youtube: The PX Docs For more information, visit PXDocs.com to read informative articles about the power of Neurologically-Focused Chiropractic Care. Find a PX Doc Office near me: PX DOCS DirectoryTo watch Dr. Tony's 30 min Perfect Storm Webinar: Click HereSubscribe, share, and stay tuned for more incredible episodes unpacking the power of Nervous System focused care for children!

Guest Whitney Fallon, NP shares how your body works on an Individual basis with your own genetic processes understanding Neuro divergence with ADHD and Autism. How or why, you can or can't lose weight, insulin resistance, the best detox or nutrient absorption. Whitney Fallon Links: https://linktr.ee/woodedlakewellness https://Wellnessrenovation.com Feel the reassurance of a Psychiatric Service Dog—Train your own dog if they recognize and reduce any psychological symptoms, to fly, be in 'no-pet' housing, with no pet fees and access public places under ADA law. Training from Joanne S. Williams, LCSW. A 30-second free guide to see if you qualify at ServiceDogPro.com!   Free 30 minute focus call with Joanne to talk about what would work best for you for your emotional health. Free Cultivate patience worksheet to make the world a more patient place.  

¡ALERTA NEUROLÓGICA! El reconocido Dr. Alejandro Andersson, neurólogo experto en neuroinflamación, revela los HÁBITOS QUE SALVAN TU CEREBRO del deterioro y enfermedades como Alzheimer y Parkinson.⚠️ Descubre en este episodio exclusivo:✅ Cómo DESINFLAMAR tu cerebro de forma natural✅ El proceso de NEUROGÉNESIS: regenera neuronas a cualquier edad✅ Los 5 HÁBITOS CLAVE para prevenir Alzheimer, Parkinson y migrañas✅ La VERDAD sobre el TDAH que nadie te cuenta✅ Cómo el TRAUMA afecta tu cerebro y cómo repararlo✅ La conexión entre neuroinflamación y enfermedades crónicas

I'm excited to share my conversation with Dr. Scott Sherr, where we dive deep into methylene blue - a compound you've probably seen all over social media lately (those blue tongues!). As someone whose primary research focuses on Alzheimer's disease, I was fascinated to explore the science behind this molecule that dates back to the 1870s and was actually the first drug registered with the FDA in 1897.  The most fascinating insight for me was learning how methylene blue can serve as an electron acceptor in our mitochondria - essentially functioning as a substitute for oxygen when our cells are under stress. This explains why it shows promise for everything from traumatic brain injury to cognitive enhancement and even as a potential alternative to stimulant medications.   If you're interested in optimizing brain performance, understanding mitochondrial health, or simply curious about this blue compound that's suddenly everywhere, this episode provides both the scientific foundation and practical applications you need. Subscribe to The Neuroscience Experience for more conversations at the intersection of brain science and performance. I'm committed to bringing you evidence-based insights that you can apply to your own health journey.  SponsorsA huge thank you to my sponsors for supporting this episode. Check them out and enjoy exclusive discounts:Hone Health – Take control of your hormones with at-home testing designed specifically for men. Get personalized insights and expert guidance to optimize your health and performance. Learn more at: https://honehealth.com/Momentous – Science-backed supplements trusted by elite athletes and experts. Whether you're looking to improve recovery, performance, or overall wellness, Momentous has you covered. Use code NEURO for 20% off your order: https://www.livemomentous.com/neuroTroscriptions – Unlock your cognitive potential with precision-dosed nootropics developed by medical experts. Whether you need more focus, clarity, or energy, Troscriptions offers innovative solutions.- Get 10% off: https://troscriptions.com/NEURO- https://troscriptions.com/discount/NEUROMAIL?utm_source=affiliate&utm_medium=email&utm_campaign=NeuroMailTimestamps 00:00 Introduction 02:30 Methylene Blue's Role in Neurotransmitter Regulation 07:54 Mitochondrial Health and Methylene Blue 17:02 Methylene Blue and Modern Health Challenges 25:27 Methylene Blue in Hypobaric and Hyperbaric Environments 29:47 Understanding Hyperbaric Oxygen Therapy 32:27 Methylene Blue: A Mitochondrial Rescue 36:52 Bioavailability of Methylene Blue 42:17 Dosing and Applications of Methylene Blue 47:46 Combining Methylene Blue with Other Treatments 54:02 Conclusion and Further ResourcesThe Neuro Athletics Newsletter Instagram: @louisanicola_Twitter : @louisanicola_YouTube: @Louisa NicolaThe Neuro Experience Podcast is proud to have hosted: Dr Andrew Huberman, Dr Gabrielle Lyon, Dr Layne Norton, Thomas DeLauer, Shawn Stevenson, Dr. Rocio Salas-Whalen, Saad Alam, Uma Naidoo, Dr. Lanna Cheuck, Angela Lee Pucci, Jillian Turecki, Dr. Jordan Feigenbaum, Dr. Darren Candow, Dr. Sue Varma, Evy Poumpouras, Dr Casey Means, Renee Deehan, Dr Chris Palmer, Dr Charles Brenner, Dr Joe Zundell, Dr Ray Dorsy, Dr Dale Bredeson, Dr. Ben Bikman

In this episode of Coffey and Code, host Ashley Coffey engages with Dr. Rolando Masís-Obando, a computational neuroscience postdoc at Johns Hopkins University. They discuss the intersection of neuroscience, technology, and ethics, particularly focusing on neuro rights and the implications of brain-computer interfaces. Rolando shares insights from his research, the importance of ethical considerations in technology, and the evolving landscape of neural data privacy. The conversation highlights the need for a human-centered approach in shaping the future of technology and neuroscience, emphasizing the importance of curiosity and interdisciplinary connections.Want to hear even more? Listen to Dr. Rolando Masis-Obando's Talk on "Should We Fear AI?" at Princeton Research Day, 2023Chapters03:15 The Intersection of Neuroscience and Ethics09:07 Curiosity and the Journey into Neuroscience15:28 Neuro Rights and Technology21:39 Protecting Neural Rights in the Digital Age24:48 The State of Consumer Privacy26:31 Neuroscience and Technology: Surprising Insights32:34 Brain-Computer Interfaces: Opportunities and Ethical Concerns39:30 The Future of Neuroscience and Neuroethics45:33 The Journey of Innovation and AdaptabilityConnect with Dr. Rolando Masis-Obando on Linkedin and follow his research on neuro rights and the future of innovation. EPISODE CREDITS:Produced and edited by Ashley Coffey. Cover art designed by Ashley Coffey.Headshot by Brandlink MediaIntroduction music composed and produced by Ashley Coffey LINKSFollow Coffey & Code on Instagram, Facebook, Linkedin, and YouTube for the latest emerging tech updates! Subscribe to the Coffey & Code Podcast wherever you get your podcasts to be notified when new episodes go live. © 2025 Coffey & Code Podcast. All rights reserved. The content of this podcast, including but not limited to text, graphics, audio, and images, is the property of Ashley Coffey and may not be reproduced, redistributed, or used in any manner without the express written consent of the owner.

Hearing loss affects millions of people worldwide, particularly older adults who can suffer age-related hearing loss (ARHL). Beyond the obvious impact on communication and quality of life, recent research studies show that hearing loss leads to a higher risk of developing the diseases of cognitive decline such as dementia and Alzheimer's. Hearing loss demands comprehensive understanding and professional care. It is more than a mere audiological condition; hearing loss is profoundly connected with cognition, communication, and emotional well-being. If you find yourself confronting hearing loss, do not hesitate to seek assistance at Pacific Eye, Ear, and Skull Base Center.

In this episode, Lyell K. Jones Jr, MD, FAAN, speaks with Valérie Biousse, MD, who served as the guest editor of the Continuum® April 2025 Epilepsy issue. They provide a preview of the issue, which publishes on April 3, 2025.   Dr. Jones is the editor-in-chief of Continuum: Lifelong Learning in Neurology® and is a professor of neurology at Mayo Clinic in Rochester, Minnesota.  Dr. Biousse is a professor in the departments of neurology and ophthalmology, as well as the Reunette Harris Chair of Ophthalmic Research, at Emory University in Atlanta, Georgia.  Additional Resources Read the issue: Neuro-ophthalmology Subscribe to Continuum®: shop.lww.com/Continuum More about the American Academy of Neurology: aan.com  Social Media  facebook.com/continuumcme  @ContinuumAAN  Host: @LyellJ  Guest: @vbiouss  Full episode transcript available here Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum. Thank you for listening to Continuum Audio. Be sure to visit the links in the episode notes for information about subscribing to the journal, listening to verbatim recordings of the articles, and exclusive access to interviews not featured on the podcast. Dr Jones: This is Dr Lyell Jones, Editor-in-Chief of Continuum: Lifelong Learning in Neurology. Today, I'm interviewing Dr Valerie Biousse, who recently served as Continuum's guest editor for our latest issue on neuro-ophthalmology. Dr Biousse is a professor in the departments of neurology and ophthalmology at Emory University in Atlanta, Georgia where she's also the Renette Harris Chair of Ophthalmic Research. Dr Biousse, welcome and thank you for joining us today. Why don't you introduce yourself to our listeners? Dr Biousse: In addition to what you just mentioned, I would like to highlight that I have a French accent because I was born and raised and went to medical school in France in Saint Pete Pierre, where I trained as a neurologist. And I even practiced as a stroke neurologist and a headache specialist in the big university in Paris before I decided to move to the US to pursue my passion, which was really neuro-ophthalmology. And at the time, it was impossible to get a license in the US, so I had to repeat a residency and became an ophthalmologist. And this is what led me where I am today. Dr Jones: We're fortunate that you did that. I'm glad you did all that extra work because your contributions to the field have obviously been magnificent, especially this issue, which I think is an incredibly important topic for neurologists. This is why we include it in the rotation of Continuum topics. We all know the saying that the eyes are the windows to the soul, but for neurologists they are also the windows to the brain. The only part of the CNS that's visible to us is the optic disc. I think in spite of that, I think neurologists, our readers and our listeners would acknowledge the importance of the ophthalmic exam and respect the importance of that aspect of the neurologic exam. It's an area that feels challenging to us, and many of us, even with lots of years of experience, don't always feel very comfortable with this. So, it's a really important topic and I'm glad you have edited this. And let's start off with, you know, as you've reviewed all these articles from, really, the pinnacle experts in their specific topics in neuro-ophthalmology, as you were editing this issue, Dr Biousse, what would you say is the one biggest, most important practice-changing message about neuro-ophthalmology you would want to convey to our listeners? Dr Biousse: I think its technology, advances in technology. Without any doubt. The ophthalmology world cannot evaluate a patient anymore without access to fundus photography, optical coherence tomography (OCT) of the back of the eye, not just the optic nerve, but the retina. These advantages in technology have completely changed the way we practice ophthalmology. The same applies to neuro-ophthalmology. And these techniques can really help neurologists do a basic eye exam. Dr Jones: So, let's get right into that. And I'm glad you started with that because I still feel, even though I've done it thousands of times, I still feel a little fumbly and awkward when I'm trying to examine and fundus through an undilated pupil, right? And so, this is I think where technology has helped us quantitate with, as you mentioned, OCT, but I think from an accessibility perspective, I think nonmydriatic fundus photography is a very interesting tool for neurologists and non-neurologists.  Tell us how, how does that work and how could neurologists implement that in their practice? Dr Biousse: It's a very important tool that of course neurology should be able to use every day. You can take fundus photographs of the back of the eye without dilating the pupil. The quality of the photographs is usually very good. You only have access to what we call the posterior pole of the eye, so the optic nerves and the macula and the vascular arcade. You don't see the periphery of the retina, but in neuro-ophthalmology or neurology you don't need access to the periphery of the retina, so it doesn't matter. What is remarkable nowadays is that we have access to very highly performing fundus cameras which can take pictures through very, very small pupils or in patients of all ages. You can use it on a two-year-old in a pediatric clinic. You can use it on a much older person who may have a cataract or other eye problems. And what's really new and what this issue highlights is that it's not just that we can take pictures of the back of the eye, we can also perform OCT at the same time using the same camera. So, that's really a complete game changer for neurologists. Dr Jones: And that's extremely helpful. If I'm in a neurology clinic and I would like to use this technology, how would I access that? Do I need special equipment? Can I use my smartphone and an app? How would that work in terms of getting the image but also getting an interpretation of it? Dr Biousse: It all depends on what your ultimate goal is. The fundus cameras, they are like regular cameras or like any technology that would allow you to get brain imaging. The more sophisticated, the better the quality of the image, the more expensive they are. You know, that's the difference between a three-tesla MRI and a head CT. You buy a camera that's more expensive, you're going to have access to much easier cameras and to much higher resolution of images, and therefore you're going to be much happier with the results. So, I always tell people be very careful not to get a tool that is not going to give you the quality of images you need or you may make mistakes. You basically have two big sorts of cameras. You have what we call the tabletop cameras, which is a little more bulky camera, a little more expensive camera that's sitting on the table. The table can be on wheels, so you can move the table to the patient or you can move the patient to the table. That's very convenient in a neurology clinic where most patients are outpatient. It works in the emergency department. It's more difficult at bedside in the hospital. Or you can have a handheld camera, which can be sophisticated, a device that just uses a handheld camera or, as you mentioned, a small camera that you place on your smartphone, or even better, a camera that you can attach to some of the marketed direct ophthalmoscopes. In all situations, you need to be able to transfer those images to your electronic medical records so that you can use them. You can do that with all tabletop cameras, most handheld cameras; you cannot do it with your smartphone. So that gives you an idea of what you can use. So yes, you can have a direct ophthalmoscope with a little camera mounted. This is very inexpensive. It is very useful at bedside for the neurologists who do- who see patients every day, or the resident on call. But if you really want to have a reliable tool in clinic, I always recommend that people buy a tabletop camera that's connected to the electronic medical record. Dr Jones: You know, the photos always make it so much more approachable and accessible than the keyhole view that I get with my direct ophthalmoscope in clinic. And obviously the technology and the tools are part of the story, but also, it's access to the expertise. Right? There are not many neuro-ophthalmologists in the world, and getting access to the experts is a challenge, I think, everywhere, everywhere in the world really. When you think about how technology can expand that---and here I'm getting at AI, which I hesitate to bring up because it feels like we talk about AI a lot---are there tools that you think are here now or will be coming soon that will help clinicians, including neurologists, interpret fundus photography or other neuro-ophthalmologic findings, maybe eye movements, to make that interpretation piece a little more accessible? Dr Biousse: Absolutely. It's going to happen. It's not there yet. OK? I always tell people, AI is very important and it's a big part of our future without any doubt. But to use AI you need pictures. To get pictures, you need a camera. And so I tell people, first you start with the camera, you implement the camera, you incorporate the camera in your electronic medical record. Because if you do that, then the pictures become accessible to everyone, including the ophthalmologist who's maybe offsite and can review the pictures and provide an official interpretation of the pictures to help you. You can also transfer those pictures using secure mode of transfers and not your smartphone text application, which you really don't want to use to transfer medical information. And that's why I insist on the fact that those pictures should definitely appear in the patient's medical record. Otherwise you're going to break HIPAA laws, and that's an issue that comes up quite often. Once you have the pictures in the electronic medical record and once you have the pictures in the camera, you can do three things. You can look at them yourself. And many of my neurology colleagues are very competent at declaring that an optic nerve is normal or an optic nerve is swollen or an optic nerve is pale. And very often that's all we need. You can say, oh, I don't know about that one, and page the ophthalmologist on call, give the patient 's medical record number, have them look at the pictures, provide an interpretation, and that's where you have your answer. And this can be done in real time, live, when you're at bedside, no problem. Or you can use AI as what I call “Diagnostic A.” I always compare it as, imagine if you had a little robot neuro-ophthalmologist in your pocket that you could use at any time by just taking a picture, clicking submit on the AI app. The app will tell you never, it's normal or it's papilledema or it's pale. The app will tell you, the probability of this optic disk of being normal is 99% or the probability that this is papilledema. And when I say papilledema, I mean papilledema from rest intracranial pressure that's incredible as opposed to optic disc edema from an optic neuritis or from an ischemic optic neuropathy. And the app will tell you, the probability that this is papilledema is eighty six percent. The probability that it's normal is zero. The probability that it's another cause of disc edema is whatever. And so, depending on your probability and your brain and your own eyes, because you know how to interpret most fundus photographs, you really can make an immediate diagnosis. So that is not available for clinical use yet because the difficulty with the eye, as you know, is to have it have a deep learning algorithm cleared by the FDA. And that's a real challenge. But many research projects have shown that it can be done. It is very reliable, it works. And we know that such tools can either be either incorporated inside the camera that you use---in which case it's the camera that gives you the answer, which I don't think is the ideal situation because you have one algorithm per camera---or you have the algorithm on the Cloud and your camera immediately transfers in a secure fashion the images to the Cloud and you get your answer that way directly in your electronic medical record. We know it can be done because it happens every day for diabetic retinopathy. Dr Jones: Got it. And so, it'll expand, and obviously there has to be a period of developing trust in it, right? Once it's been validated and it becomes something that people use. And I get the sense that this isn't going to replace the expertise of the people that use these tools or people in neuro-ophthalmology clinics. It really will just augment. Is that a fair statement? Dr Biousse: Absolutely. Similar to what you get when you do an EKG. The EKG machine gives you a tentative interpretation, correct? And when the report is “it's normal,” you really can trust it, it's normal. But when it says it's not normal, this is when you look at it and you ask for a cardiology consultation. That's usually what happens. And so, I really envision such AI tools as, “it's normal,” in which case you don't need a consultation. You don't need to get an ophthalmology consultation to be sure that there is no papilledema in a patient with headache, in a patient with possible cerebrospinal fluid shunt malfunction. You don't need it because if the AI tool tells you it's normal, it's normal. When it's not normal, you still need the expertise of the ophthalmologist or the neuro-ophthalmology. The same applies to the diagnosis of eye movement. So that's a little more difficult to implement because, as you know, to have an AI algorithm, you need to have trained the algorithm with many examples. We have many examples of pathology of the back of the eyes, because that's what we do. We take pictures every day and there are databases of pictures, there are banks of pictures. But how many examples do we have of abnormal line movement in myasthenia, of videos or downbeat nystagmus? You know, even if we pulled all our collections together, we would come up with what, two hundred examples of downbeat nystagmus around the world? That's not enough to train an AI system, and that's why most of the research on eye movement right now is devoted to creating algorithm that mimic abnormal eye movements so that we can make them and then train algorithm which job will be to diagnose the abnormal eye movement. There's an extra difficult step, it's actually quite interesting. But it's going to happen. You would be able to have the patient look at the camera on the computer and get a report about “it's normal” or “the saccades, whatever, are not normal. It's most likely an internucleosomal neuralgia” or “it is downbeat nystagmus.” And that's not, again, science fiction. There are very good groups right now working on this. Dr Jones: That's really fascinating, and that- you anticipated my next question, which is, I think neurologists understand the importance of the ocular motor exam from a localizing perspective, but it's also complex and challenging. And I think that's certainly an area of potential growth. And you make a good point that we need some data to train the models. And until we have these tools, Dr Biousse, that will sort of democratize and provide access through technology to diagnosis and, you know, ultimately management of neuro-ophthalmology disorders, we know that there are gaps in the care of these patients right now in the modern day. In your own practice, in your own work at Emory, what do you see as the biggest gap in practice in caring for these patients?  Dr Biousse: I think there is a lack of confidence amongst many neurologists regarding their ability to perform a basic eye exam and provide a reliable report of their finding. And the same applies to most ophthalmologists. And that's very interesting because we have, often, a large cohort of patients who are in between the two specialties and are getting a little bit lost. The ophthalmologist doesn't know what to do. The neurologist usually knows what to do, but he's not completely sure that it's the right thing to do. And that's where the neuro-ophthalmologist comes in. And when you have a neuro-ophthalmologist right there, it's fantastic, okay? We bridge the two specialties, and we often just translate what the ophthalmologist said to the neurologist or what the neurologist said to the ophthalmologist and suddenly everything becomes clear. But unfortunately, there are not enough neuro-ophthalmologists. There is a definite patient access issue even when there is a neuro-ophthalmologist because not only is there a coverage heterogeneity in the country and in the world, but then everybody is too busy to be able to see a patient right away. And so, this gap impairs the quality of patient care. And this is why despite all this technology, despite the future, despite AI, we teach ophthalmologists and neurologists how to do a neuro-op examination, how to use it for localization, how to use it to increase the value and the power of a good neurologic examination so that nothing is missed. And I'm taking a very simple example. Neurologists see patients with headaches all the time. The vast majority of those headaches are benign headaches. 90% of headache patients are either migraine or tension headache or analgesic abuse headaches, but they are not secondary headache that are life threatening or neurologically threatening. If the patient has papilledema, it's a huge retina that really should prompt immediate workup, immediate prevention of vision loss with the help of the ophthalmologist. And unfortunately, that's often delayed because the patients with headaches do not see eye doctors. They see their primary care providers who does not examine the back of the eye, and then they reach neurology sometimes too late. And when the neurologist is comfortable with the ophthalmoscope, then the papilledema is identified. But when the neurologist is not comfortable with the ophthalmoscope, then the patient is either misdiagnosed or sent to an eye care provider who makes the diagnosis. But there is always a delay in care. You know, most patients end up with a correct diagnosis because people know what to do. But the problem is the delay in appropriate care in those patients. And that's where technology is a complete life-changing experience. And, you know, I want to highlight that I am not blaming neurologists for not looking at the back of the eye with a direct ophthalmoscope without pharmacologic dilation of the pupil. It is not possible to do that reliably. The first thing I learned when I transitioned from a neurologist to an ophthalmologist is that no eye care provider ever attempts to look at the back of the eyes without dilating the pupils because it's too hard. Why do we ask neurologists to do it? It's really unfair, correct? And then the ophthalmoscope is such an archaic tool that gives only a very small portion of the back of the eye and is extraordinarily difficult to use. It's really not fair. And so, until we give the appropriate tools to neurologists, I don't think we should complain about neurologists not being reliable when they look at the back of the eye. It's a major issue.  Dr Jones: I appreciate you giving us some absolution there. I don't think we would ask neurologists to check reflexes but then not give them a reflex hammer, right? So maybe that's the analogy to not dilating the pupil. So, for you and your practice, in our closing minutes here, Dr Biousse, what's the most rewarding thing for you in neuro-ophthalmology? What do you find most rewarding in the care of these patients?  Dr Biousse: Well, I think the most rewarding is the specialty itself. I'm a neurologist at heart. This is where my heart belongs. What's great about those neuro-ophthalmology patients is that it is completely unpredictable. They are unpredictable. They can have anything. I am super specialized because I'm a neuro-ophthalmologist, but I am a general neurologist and I see everything in neurology. So my clinic days are fascinating. I never know what's going to happen. So that's, I think, the most rewarding part of my job as an neuro-ophthalmologist. I'm having fun every day because it's never the same, I never know what's going to happen. But at the same time, we are so useful to those patients. When you use the neuro-ophthalmologic examination, you really can provide exquisite localization of the disease. You're better than the best of the MRIs. And when you know the localization, your differential diagnosis is always right, always correct, and you can really help patients. And then I want to highlight one point that we made sure was covered in this issue of Continuum, which is the symptomatic treatment of patients who have visual disturbances from neurologic disorders. You know, a patient with chronic diplopia is really disabled. A patient with decreased vision cannot function. And being able to treat the diplopia and provide the low vision resources to those patients who do not see well is extremely important for the quality of life of our patients with neurologic disorders. When you don't walk well, if you don't see well, you fall. When you're cognitively impaired, if you don't see well, you are very cognitively impaired. It makes everything worse. When you see double, you cannot function. When you have a homonymous anopia, you should not drive. And so, there is a lot of work in the field of rehabilitation that can greatly enhance the quality of life of those patients. And that really covers the entire field of neurology and is very, very important. Dr Jones: Clearly important work, and very exciting. And your enthusiasm is contagious, Dr Biousse. I can see how much you enjoy this work. And it comes through, I think, in this interview, but I think it also comes through in the articles and the experts that you have. And I'd like to thank you again for joining us today for a great discussion of neuro-ophthalmology. I learned a lot, and hopefully our listeners did too.  Dr Biousse: Thank you very much. I really hope you enjoyed this issue. Dr Jones: Again, we've been speaking with Dr Valerie Biousse, guest editor of Continuum's most recent issue on neuro-ophthalmology. Please check it out, and thank you to our listeners for joining today. Dr Monteith: This is Dr Teshamae Monteith, Associate Editor of Continuum Audio. If you've enjoyed this episode, you'll love the journal, which is full of in depth and clinically relevant information important for neurology practitioners. Use this link in the episode notes to learn more and subscribe. AAN members, you can get CME for listening to this interview by completing the evaluation at continpub.com/audioCME. Thank you for listening to Continuum Audio.

À l'occasion de la Journée mondiale de sensibilisation à l'autisme, nous parlons de ces troubles du neurodéveloppement. Les troubles du spectre de l'autisme apparaissent dans l'enfance et persistent à l'âge adulte. Difficultés à établir des interactions sociales et à communiquer, anomalies comportementales… les manifestations de ces troubles sont multiples.   Quelles sont les causes de ces troubles ? Comment les prendre en charge ? Comment changer de regard sur les personnes neuro-atypiques et favoriser l'inclusion ?  Dr Aurélie Clavel, pédiatre au sein du Département TND-TSA (Troubles de Neuro développement - Troubles de Spectre de l'Autisme) du Service Universitaire de Pédopsychiatrie du Pr Baleyte, du Centre Hospitalier Intercommunal de Créteil en région parisienne   Scila Toscana, cadre coordonnatrice au sein du Département TND-TSA (Troubles de Neurodéveloppement - Troubles de Spectre de l'Autisme) du Service Universitaire de Pédopsychiatrie du Pr Baleyte, du Centre Hospitalier Intercommunal de Créteil en région parisienne   Alida Inès Oket, psychologue à Brazzaville au Congo.   Un reportage de Raphaëlle Constant. À lire aussiSensibilisation à l'autisme: quand on peut «être handicapé et travailler dans la publicité» Programmation musicale :► Gorillaz – On melancholy hill ► Burna Boy – Update.

À l'occasion de la Journée mondiale de sensibilisation à l'autisme, nous parlons de ces troubles du neurodéveloppement. Les troubles du spectre de l'autisme apparaissent dans l'enfance et persistent à l'âge adulte. Difficultés à établir des interactions sociales et à communiquer, anomalies comportementales… les manifestations de ces troubles sont multiples.   Quelles sont les causes de ces troubles ? Comment les prendre en charge ? Comment changer de regard sur les personnes neuro-atypiques et favoriser l'inclusion ?  Dr Aurélie Clavel, pédiatre au sein du Département TND-TSA (Troubles de Neuro développement - Troubles de Spectre de l'Autisme) du Service Universitaire de Pédopsychiatrie du Pr Baleyte, du Centre Hospitalier Intercommunal de Créteil en région parisienne   Scila Toscana, cadre coordonnatrice au sein du Département TND-TSA (Troubles de Neurodéveloppement - Troubles de Spectre de l'Autisme) du Service Universitaire de Pédopsychiatrie du Pr Baleyte, du Centre Hospitalier Intercommunal de Créteil en région parisienne   Alida Inès Oket, psychologue à Brazzaville au Congo.   Un reportage de Raphaëlle Constant. À lire aussiSensibilisation à l'autisme: quand on peut «être handicapé et travailler dans la publicité» Programmation musicale :► Gorillaz – On melancholy hill ► Burna Boy – Update.

This is not just an episode—it's a doorway. In this guided workshop, I share the story of John Goddard and his awe-inspiring Master Life List, then invite you to begin crafting your own.Grab a pen. Grab some paper. This is your moment to unleash the dreams that have been whispering from within. Together, we'll take your raw desires and begin shaping them into clarity, direction, and real-world goals... to activate the law of attraction and turn your desires into manifestations.This is where longing meets intention—where vision takes form. And if you're ready to go deeper into the full alignment and execution journey, we invite you into the Master Life Intention System. To get started, reach out to Daniel at yesdaniel.com. Your future is waiting.ANNOUNCING: THE LIMITLESS EXPERIENCEDaniel D'Neuville is a peak performance coach, spiritual teacher, and master life and business coach. A Master Practitioner/Trainer in Neuro-linguistic Programming and a Consulting Hypnotist he has worked with over 10,000 people over the past 30 plus years. LINKS http://yesdaniel.com Daniel's YouTube CHANNEL FACEBOOK GROUPSPODCAST LISTENER'S FBCOMMUNITY EXTREME GRATITUDE PROJECT BassSlap Intro written and performed by bass player & producer: Miki SantamariaMiki'sYouTube Channel: https://www.youtube.com Hosted on Acast. See acast.com/privacy for more information.

We all want a marriage that feels like home. We long for a partner who is there for us through all of life's challenges and griefs, who is in on all our inside jokes, who delights in the family we've built together, who looks with hope toward the future with a shared faith and an arm firmly around our shoulders. But how do we get there? What actually works in the real world and honors the picture of marriage found in Scripture?Based on the findings of their meticulous research, Sheila Wray Gregoire and Dr. Keith Gregoire dispel the pervasive myths about what makes a happy marriage. Rather than relying on gender stereotypes, they look at what actually creates intimacy, emotional health, and connection, asking deeper questions that lead to real healing and growth in your relationship with your spouse.With enlightening stories, survey results, and practical tools, this book will help readers create a marriage they love. And with the evidence-based, Scripture-honoring advice in this book, that just may be closer than you imagine. Today, Dr. Stephanie talks with Sheila & Keith about the new book The Marriage You Want, and Dr. Stephanie adds in some of the neurodiverse lens.Dr. Stephanie asks Sheila & Dr. Keith:What did you find about the marriage triangle (God at the top and husband and wife on the bottom) and how that triangle can get skewed?You open a chapter with the controversial statement, "You should not prioritize sex in marriage." Talk a bit more about that as authors of books on healthy sexual marital relationships.What is the unfairness threshold?How does a couple grow intimacy?Do Christian books empower men or make them seem fragile and needy?Dr. Stephanie will do a course based on the new book this summer! Join now!https://www.christianneurodiversemarriage.com/contact-usAbout our Guests:Sheila Wray Gregoire is the face behind BareMarriage.com as well as a sought-after speaker and an award-winning author of 9 books, including the Great Sex Rescue and She Deserves Better. Sheila is passionate about changing the evangelical conversation about sex and marriage to make it healthy, evidence-based, and biblical.Dr. Keith Gregoire is a physician and coauthor with Sheila on The Good Guy's Guide to Great Sex, a companion to Sheila's award-winning The Good Girl's Guide to Great Sex.The couple lives in Ontario, Canada.Find out more at: https://baremarriage.com/

JASON BRETT SERLE is a British writer, filmmaker, musician, Neuro-linguistic Programming (NLP) Master and licensed hypnotherapist with a particular focus on themes involving psychology, spirituality, wellness, and human potential.   He has written articles for Jain Spirit and Watkins magazines as well as interviewing people such as Eckhart Tolle, Robert Anton Wilson, Andrew Cohen, Jan Kersschot, and Amado Crowley. He is cited in Crowley's 2002 book Liber Alba: The Questions Most Often Asked of an Occult Master as being the only other person to have seen The Book of Desolation; a book purported to have been brought back from Cairo by his father, Aleister Crowley, in 1904.   In 2012 he wrote and produced his first documentary film, 'Mind Your Mind: A Primer for Psychological Independence' which looks at the psychological methods used to manipulate people and what they can do to protect themselves. He also composed and performed most of the soundtrack. The film is distributed by Journeyman Films in the UK and Film Media Group in the US, and it was an official selection for the London International Documentary Festival (LIDF) in 2012. We ltalk about:   1. What exactly is crazy wisdom, and what makes it a path worth exploring?   2. How does crazy wisdom differ from what you call in the book divine madness?   3. How does The Monkey in the Bodhi Tree challenge our conventional understanding of sanity?   4. Why is trans-rational thought—going beyond logic and reason—so often misunderstood?   5. What are some of the most striking historical examples of crazy-wisdom?   6. How can embracing crazy-wisdom lead to greater clarity and self-realization?   7. How has crazy-wisdom influenced art, literature, and culture throughout history?   8. Why do spiritual movements sometimes attract charlatans, and how can seekers distinguish authenticity from deception?   9. What inspired you to explore this topic, and what impact has it had on your own perspective?   10. If someone wants to begin exploring crazy-wisdom, what is the first step they should take?   11. Where can people read The Monkey in the Bodhi Tree? O books Presents The Monkey in the Bodhi Tree Crazy-Wisdom & the Way of the Wise-Fool by Jason Brett Serle Release date: March 1st 2025 Categories: Eastern, Mindfulness & meditation, Rituals & Practice CLICK HERE TO VIEW THE BOOK COVER   Unleash the radical, transformative power at the heart of the world's great wisdom traditions.   Of the many paths up the mountain, that of crazy-wisdom, although one of the lesser travelled, presents a dramatic and formidable climb to those that are so inclined. Now for the first time, the true spiritual landscape of the wise-fool has been laid bare and its features and principal landmarks revealed.   Written in two parts, loosely based on the theory and practice of crazy-wisdom, The Monkey in the Bodhi Tree is the first comprehensive look at this universal phenomenon, from its origins and development to the lives of its greatest adepts and luminaries.   In addition to the theoretical foundations laid down in Part I, Part II deals with its practice and aims to demonstrate crazy-wisdom in action. To this end, 151 teaching tales from around the world have been meticulously gathered and retold to illustrate the methods of the great masters and adepts - stories that not only give practical insight but also, like Zen koans, can be used as contemplative tools to illuminate and provoke epiphany.   From the enigmatic Mahasiddhas of ancient India to the eccentric Taoist poet-monks of China, from the uncompromising insights of the Buddhist Tantrikas to the unconventional wisdom of Sufi heretics and the utter surrender to God displayed by the Fools for Christ, this book will take you to a place where the boundaries of logic and reason dissolve and enlightenment awaits those daring enough to venture forth.   BOOK LINK: https://www.collectiveinkbooks.com/o-books/our-books/monkey-bodhi-tree-crazy-wisdom   JASON'S WEBSITE: www.fasonbrettserle.com  

We’ll discuss these hidden risks with Brady Dahmer, author of the new book “Blindspots”. The post 390: What Are the Hidden Risks of Assumptions in Marketing? first appeared on Persuasion by the Pint.

Warning: This piece discusses my menstrual cycle in depth. If that doesn't interest you, please do something else with your time. I won't be offended. Thanks!“Man is an intelligence, not served by, but in servitude to his organs.” — Aldous Huxley, Brave New World.Dear Wonderful Readers,I am not a scientific expert when it comes to a complex topic like the menstrual cycle. But as a writer who is curious about the world, since I got my first period 19 years ago, I've gathered a lot of data on one menstrual cycle in particular: my own. Today, I'm going to share with you one aspect of my menstrual cycle that I hope you will find reassuring, cool, and downright witchy. It gives me the kind of all-knowing power that I definitely would have been burned at the stake for 400 years ago.The Phenomena of Phenomenal ThinkingEvery month, after I finish my period, I get a few days of incredible clarity on basically every aspect of my life. I also feel incredibly energized. I'm currently in that phase. Just yesterday, I found myself biking extra fast on my way to my co-working space and improvising on the piano like an absolute boss.I first noticed this experience a couple of years ago. I've since started tracking it in my period-tracking app, Clue, which is an awesome company that I also happen to work for (I don't make money from telling you this because Clue doesn't have an affiliate program). In Clue, I've made a special tag for this experience. I call it my “Post-Period Strategic Clarity.”How does my “Post-Period Strategy Clarity” manifest in my life? Well, this week, I pitched five new clients before midday on Wednesday. I'd also been feeling a bit lost in my career, but when I went to a concert on Tuesday, I could picture my future self, sitting on that same stage, selling out an audience of 3,500 people as I talked about my writing tools and techniques and sharing them with the world. In the last couple of days, as I go to bed, I find myself reaching for a piece of paper and a pen to jot down strings of incredible ideas on who I should be reaching out to, how I should be asking for help, and where I should be focusing my energy because I know where I'm going. It's like unrolling a blueprint created by my brain and knowing all my next moves. I even redownloaded Hinge and started messaging people.There's evidence that I'm not making this up. Here's what hormonal health expert Alissa Vetti says about the menstrual cycle and the follicular phase:“So first, you have the follicular phase. Fascinating phase. And neuro-chemically these hormonal ratios change your brain chemistry. So, you are a different person week-over-week within a month…In that follicular phase where the eggs are coming up to maturity, one is going to make it to the fallopian tube, very exciting time. Neuro-chemically speaking, you have the most access to creative energy than that you will have the entire month. Effortlessly. This is a perfect time to begin new projects: mastermind plan, dream big, all of that.”My next question is: why does my body do this? Well, here is my totally non-scientific answer. I'm 30 years old, and these days I'm feeling super intense baby fever and what can only be described as extreme horniness every time I ovulate, which occurs in the days right after the follicular phase. So, right after my period ends, once my body has rid itself of the last cycle, it's gearing up again for ovulation. And so, I get a surge of energy. And this energy makes sense because, for survival, it would probably be a good idea for me to gather a lot of resources (i.e., pitch a s**t ton of clients to make sure I have plenty of enough income) and find a mate (i.e., download a dating app and start going out and flirting with whoever is hanging around). Maybe I need to nest a bit (i.e., decide which relationships and things in my house are working for me and discard some that aren't). So, yes, I'm living my modern life equivalent. But from a biological perspective, I can see how this corresponds to what's going on in my cycle. My brain might be speaking in emails, client pitches, and party invitations. But my body is making sure that I create the most awesome, successful, and habitable environment for myself, attract a great mate, and get some optimal offspring going. Pretty cool! And yes, I literally am ready to run a half-marathon this weekend before a party with some of the coolest musicians and hot, bright young things in Mexico City.Before you start rolling your eyes and thinking, “Well, isn't this girl's life just pretty and perfect!” let me remind you what happened just before my Post-Period Strategy Clarity: I had my period. All the greatness that follows my period during this phase happens despite my recent mood swings, extreme irritability, propensity to cry for no reason, and depression during my PMS for at least another week before. Plus, the depressing point is that this clarity doesn't last forever. As I've written about before, after a couple of days, I know all the insanity of ovulation will start happening again, and I'll be back to not having a clue what I am doing with my life. See how Mother Nature's a b***h? And how what goes up must come down?Three years ago, I was on this same cycle, just as I will be until I hit menopause, I expect. In May 2022, I wrote,“Right before I get my period or even the first or second day, I feel incredibly alone, timid, and shy. I feel self-conscious and hyper-acutely aware of all my mistakes in conversations. But the strangest thing happens at the end of my period, like now. I feel incredibly energized, and my thoughts become completely clear. It usually comes to me like a flash of inspiration on one night of the month. I can see crystal clear who I am trying to become, what I need to do to push myself that month, what I'm truly feeling about things, and what needs to change. I can see that now.”For those of you who want the extra details, you should know that this all happens despite the fact that I've had a Hormonal IUD for the last 8 years. I recently got mine replaced after it expired because my periods have been historically insanely painful, and I don't plan on having a period again if I can avoid it. These 8 years, I haven't had cramping and bleeding on my period, but I still get those mood swings! So, I still get my Post-Period Strategic Clarity. At least in my experience with Hormonal IUDs, everybody wins.In summary, if you have a menstrual cycle and you're not currently using an app to track any of your symptoms, maybe try it out for a while and see if you find it helpful. There are plenty of apps on the market besides Clue. Obviously, I'm not a pacha mama earthy spiritual lady telling you to live off the grid and never put another medication in your body ever again. But looking at the science, it makes sense to try to master matching our lives with the energetic flow of our menstrual cycles, especially when it serves us as baddies living our best lives. Alissa Vetti called this “[leveraging] your body as a power tool.” And that's what I plan to continue to do.Here's to harnessing the cyclical nature of being a female for my own benefit! Moahaha!

Have you as a leader, had families wanting to join your co-op that have students who have special needs or learning difficulties?  Maybe these families have suffered in a traditional school setting and are homeschooling because their student did not “fit the mold”.  Are you equipped to welcome them into your homeschool community?  You don't want to just say “No, you can't join our community,” but you are also nervous about what it will look like on a practical basis.  We want to show you a path forward so that you can welcome students of all learning styles and even neuro-divergent learners into your community.  Listen in today for some principles that we have discovered after having some of these special students in our communities!  Next Steps:  Schedule a coaching call:  https://homeschoolcommunitybuilders.com/   Join our Facebook group- Lead Your Homeschool Co-op https://www.facebook.com/groups/72507320516066 Become a Lead Your Homeschool Co-op Insider and get first dibs on valuable resources to help you lead, organize, and connect your community.  https://homeschoolcommunitybuilders.com/contact  

Alzheimer's doesn't just “happen” later in life—it's a slow, silent process that can begin decades before symptoms appear. In this solo episode, we challenge the myth that Alzheimer's is purely genetic, and break down what the science really says about your risk. I explore the role of the APOE4 gene, how it interacts with lifestyle and metabolic health, and why women—especially during and after menopause—may face a unique vulnerability. Most importantly, I introduce three cutting-edge blood tests that can detect the earliest signs of Alzheimer's, long before memory loss sets in. We'll also talk prevention: from blood sugar and insulin resistance to inflammation, hormone balance, and brain energy metabolism—everything you can start doing right now to safeguard your cognitive future. This is more than a conversation about Alzheimer's—it's a roadmap for taking control of your brain health. SponsorsA huge thank you to my sponsors for supporting this episode. Check them out and enjoy exclusive discounts:ZocDoc – Finding a great doctor has never been easier. With ZocDoc, you can browse top-rated physicians, read real patient reviews, and book appointments online - all in one place. Get started here: https://www.zocdoc.com/neuroHone Health – Take control of your hormones with at-home testing designed specifically for men. Get personalized insights and expert guidance to optimize your health and performance. Learn more at: https://honehealth.com/Momentous – Science-backed supplements trusted by elite athletes and experts. Whether you're looking to improve recovery, performance, or overall wellness, Momentous has you covered. Use code NEURO for 20% off your order: https://www.livemomentous.com/neuroTroscriptions – Unlock your cognitive potential with precision-dosed nootropics developed by medical experts. Whether you need more focus, clarity, or energy, Troscriptions offers innovative solutions.- Get 10% off: https://troscriptions.com/NEURO- https://troscriptions.com/discount/NEUROMAIL?utm_source=affiliate&utm_medium=email&utm_campaign=NeuroMailTimestamps: 00:00 Top Alzheimer's Myth: Genetics Don't Seal Your Fate01:43 APOE4 Gene Explained: Risk Factors vs. Reality 04:00 Alzheimer's Risk Isn't Just in Your DNA10:50 Top 3 Alzheimer's Biomarkers You Can Test for Today14:47 APOE4 Prevention Plan: Lifestyle Changes That Protect Your Brain The Neuro Athletics Newsletter Instagram: @louisanicola_Twitter : @louisanicola_YouTube: @Louisa NicolaThe Neuro Experience Podcast is proud to have hosted: Dr Andrew Huberman, Dr Gabrielle Lyon, Dr Layne Norton, Thomas DeLauer, Shawn Stevenson, Dr. Rocio Salas-Whalen, Saad Alam, Uma Naidoo, Dr. Lanna Cheuck, Angela Lee Pucci, Jillian Turecki, Dr. Jordan Feigenbaum, Dr. Darren Candow, Dr. Sue Varma, Evy Poumpouras, Dr Casey Means, Renee Deehan, Dr Chris Palmer, Dr Charles Brenner, Dr Joe Zundell, Dr Ray Dorsy, Dr Dale Bredeson, Dr. Ben Bikman

Depression, Anxiety, Parenting, NLP & Hypnotherapy Practice with Craig Meriwether - S7 E58Starting when I was a teenager I struggled for many years with depression and anxiety. It was with the birth of my son that I found the resolve to heal. My mother struggled with depression as did I and whether it was nurture or nature I want to make sure I would be able to help my son if he fell into the dark hole of depression.  I found healing by learning to take back control of my subconscious mind and nervous system through hypnotherapy and NLP techniques. After learning so much I trained to become a clinical hypnotherapist myself. I am a Certified Clinical Hypnotherapist, Medical Hypnosis Specialist, Neuro-linguistic programming (NLP) specialist, and Founder of Arizona Integrative Hypnotherapy helping people eliminate the negative emotions and limiting beliefs that may be keeping them from reaching their full potential. For over 12 years I've been helping people heal from early childhood trauma, helping cancer patients with pain control, veterans with post-traumatic stress disorder, students with test anxiety, children with nightmares, entrepreneurs with confidence, athletes with peak performance, and anyone who may be dealing with overwhelm, fear and anxiety. I have also created Ace Any Test, the most comprehensive course for test anxiety relief so people can eliminate anxiety around exams, auditions, job interviews and public speaking, as well as increase confidence and self-esteem. Possible Topics to Discuss: •  Overcoming fear and anxiety •  Program yourself for confidence •  What is hypnosis and how it can help you heal (talk about the research behind it)•  Eliminate test anxiety (as well as anxiety around auditions, job interviews, public speaking) •  Getting "in state" at the start of your day •  Using hypnotherapy for confidence, growth and success FIND HIM HERE:Arizona Integrative Hypnotherapyhttps://arizonaintegrativehypnotherapy.com/Ace Any Testhttps://aceanytest.com/Program Yourself For Confidence – Download 5 Free Recordings Today!https://aceanytest.com/boost-your-confidence/Social media - mostly linked to Ace Any Testhttps://www.youtube.com/@aceanytesthttps://www.facebook.com/AceAnyTesthttps://www.instagram.com/ace_any_test/https://www.linkedin.com/in/craig-meriwether-44286718b/If this interests you, I wanted to let you know that I have an affiliate program for my Ace Any Test course. When you sign up you get a custom affiliate link which you can put in the show notes, use in social media and use in an email newsletter. You'll earn a 25% commission when someone using your affiliate link buys the course. That's $36.75 every time someone with your affiliate link purchases Ace Any Test.You can find out more about the course at information more about the affiliate program and the course at https://aceanytest.com/affiliate-program/

Essential tremor is the most common movement disorder in humans, and its causes are among the most mysterious. It's a neurological condition that causes involuntary shaking. It can begin in one's 20s or 30s, or much later in life. Dr. Natalie Diaz treats essential tremors. Its origins may be unknown, but there are treatments. The first step is to talk to your doctor to rule out other forms of tremor because there are many.

In this two part episode Daniel D'Neuville shares insights, techniques, and frameworks from 36 years of coaching and teaching to generate clarity in your thinking and focus, overcoming ambiguous and ill defined goals and intentions. Getting clear about your desires, accelerates your progress, building momentum and maintaining enthusiasm so that you engage the law of attraction.We are always manifesting, the question is are you manifesting what you want, or what you don't want?ANNOUNCING: THE LIMITLESS EXPERIENCEDaniel D'Neuville is a peak performance coach, spiritual teacher, and master life and business coach. A Master Practitioner/Trainer in Neuro-linguistic Programming and a Consulting Hypnotist he has worked with over 10,000 people over the past 30 plus years. LINKS http://yesdaniel.com Daniel's YouTube CHANNEL FACEBOOK GROUPSPODCAST LISTENER'S FBCOMMUNITY EXTREME GRATITUDE PROJECT BassSlap Intro written and performed by bass player & producer: Miki SantamariaMiki'sYouTube Channel: https://www.youtube.com Hosted on Acast. See acast.com/privacy for more information.

Part 2 of a 2 Part Episode... Listen to Part 1 First for continuityIn this two part episode Daniel D'Neuville shares insights, techniques, and frameworks from 36 years of coaching and teaching to generate clarity in your thinking and focus, overcoming ambiguous and ill defined goals and intentions. Getting clear about your desires, accelerates your progress, building momentum and maintaining enthusiasm so that you engage the law of attraction.We are always manifesting, the question is are you manifesting what you want, or what you don't want?ANNOUNCING: THE LIMITLESS EXPERIENCEDaniel D'Neuville is a peak performance coach, spiritual teacher, and master life and business coach. A Master Practitioner/Trainer in Neuro-linguistic Programming and a Consulting Hypnotist he has worked with over 10,000 people over the past 30 plus years. LINKS http://yesdaniel.com Daniel's YouTube CHANNEL FACEBOOK GROUPSPODCAST LISTENER'S FBCOMMUNITY EXTREME GRATITUDE PROJECT BassSlap Intro written and performed by bass player & producer: Miki SantamariaMiki'sYouTube Channel: https://www.youtube.com Hosted on Acast. See acast.com/privacy for more information.

Today, Dr. Stephanie is joined by Dan to talk with Dr. Naseef about the impact of passive or engaged fathers on children, especially their autistic children. Dr. Naseef discusses the psyche of a man and how to engage fathers for a positive impact on their marriage, family system, and the autistic child.About our Guest:Robert Naseef, Ph.D., has a distinct voice as a psychologist and father of an autistic adult son. He has spoken nationwide and trained professionals internationally in treating autism and other developmental disorders and supporting families. He has a special interest in the psychology of men and fatherhood.Along with Stephen Shore, Ed.D., Dr. Naseef served as a lead consultant to the Arc of Philadelphia and SAP's “Autism at Work” program, which involved collaboration with the Pennsylvania Department of Education and the Bureau of Vocational Rehabilitation to develop the curriculum, “Preparing Neurodiverse Youth for the Workplace.”Dr. Naseef's 2013 book, Autism in the Family: Caring and Coping Together (Brookes Publishing), integrates advances in research and treatment with clinical experience to help families navigate the emotional landscape and the practical roadmap through the lifespan. Special Children, Challenged Parents: The Struggles and Rewards of Parenting a Child with a Disability (1996), his first book, received international recognition. He has appeared on radio and television. He is the co-editor with Cindy N. Ariel of Voices from the Spectrum: Parents, Grandparents, Siblings, People with Autism, and Professionals Share Their Wisdom (2006).In 2008, Robert Naseef was honored by Variety, the Children's Charity, for his outstanding contributions to the autism community. On World Autism Awareness Day, April 2, 2017, Dr. Naseef gave a TEDx talk entitled “How autism teaches us about being human,” which you can see on YouTube. Dr. Naseef is also a member of the Panel of Professional Advisors of the Autism Society of America. He also serves on the Leadership Council of the AJ Drexel Autism Institute.

"The saddest words of tongue and pen, are the words, what might have been" John Greenleaf Whittier. Bronnie Ware, a hospice nurse in Australia wrote a small book called The 5 Regrets of the Dying. In this episode Daniel D'Neuville goes over each regret and expands on them so you can implement new procedures and actions in your life to avoid the inevitable quicksand. Speaking of quicksand, Daniel actually tells you his story of when he encountered quicksand. Be Unreasonably Happy. Make it a condition of being alive.Stay in touch with those you love. Don't keep score. Go the extra mile assume responsibility for staying connected.Work less and be less concerned about money. In the end, nobody wishes they spent more time at the office.Express yourself and make your life the story of your self expression.Embrace the courage to live your life as an epic adventure of your design, not the agenda of other people.ANNOUNCING: THE LIMITLESS EXPERIENCEDaniel D'Neuville is a peak performance coach, spiritual teacher, and master life and business coach. A Master Practitioner/Trainer in Neuro-linguistic Programming and a Consulting Hypnotist he has worked with over 10,000 people over the past 30 plus years. LINKS http://yesdaniel.com Daniel's YouTube CHANNEL FACEBOOK GROUPSPODCAST LISTENER'S FBCOMMUNITY EXTREME GRATITUDE PROJECT BassSlap Intro written and performed by bass player & producer: Miki SantamariaMiki'sYouTube Channel: https://www.youtube.com Hosted on Acast. See acast.com/privacy for more information.