Podcasts about Excess

Most HR professionals treat the annual retirement plan review like a status update. The advisor presents, the team nods, and everyone gets back to the work they actually understand. The plan keeps running. Probably. Andrew Farrell, Retirement Plan Advisor at North County Wealth, has spent 20 years working with businesses on plan design, compliance, and the advisor relationship. He's watched the same dynamic play out across organizations of every size: HR teams that know everything about their company but defer entirely on the plan. In this episode, he breaks down: Why HR is already a subject matter expert in the room, and how to use that expertise to get more out of every advisor meeting How to draw a clear line between what HR should own and what should get handed off the moment it leaves the building Why missed payroll contributions are the most common retirement plan compliance failure, and the simple calendar habit that catches them before they become a problem Timestamps [00:00:39] Where HR's ownership starts in the retirement plan decision chain, from matching rates to enrollment [00:02:00] Why getting employees enrolled early is HR's most impactful retirement plan move, and why starting late is the primary headwind in savings [00:03:32] When auto-enrollment creates more administrative burden than it's worth, and the specific scenario where high turnover changes the math [00:05:08] The "excess deference" dynamic: why HR professionals hand over authority in advisor meetings they should be keeping [00:06:17] Why your plan advisor needs your company knowledge as much as you need their 401(k) expertise, and what a conversation of equals actually looks like [00:07:00] Asking your advisor to teach you something, and why using their availability is part of what you're paying for [00:08:18] The front door rule: what HR should own in plan administration and what to hand off the moment it leaves the building [00:11:19] Why missed payroll contributions are the most common compliance failure, and how to build the redundancy and calendar checks that catch them [00:15:38] Why "no" is a valid answer when your advisor pitches new plan features, and the ongoing operational cost of adding any bell or whistle [00:18:40] What "fiduciary" actually means, how to know if you're a named one, and the four primary duties that come with it Brought to You by Paylocity Paylocity is the fastest growing unified platform for HR, Finance, and IT. Paylocity brings your people, processes, and data together in one place so HR leaders can spend less time managing systems and more time doing the work that actually moves their organizations forward. Learn more at paylocity.com Keywords: 401k, 403b, retirement plan, fiduciary responsibility, HR compliance, auto-enrollment, plan advisor, payroll contributions, benefit design, retirement savings, plan document, financial wellness, employee benefits, retirement readiness, plan sponsor, benefits administration, excess deference, contribution timing, investment liability, HR strategy

Unlock the secrets to achieving quality skin and understanding excess skin in our latest episode of the Cutting Edge Podcast! Join host Mel Gottfried, along with Dr. Omar Beidas and Ashlyn Douglass-Barnes, LCSW, as they dive deep into the complexities of skin quality and the impact of weight loss on your body. Key takeaways: Discover the difference between quality skin and excess skin. Learn about innovative techniques like radio frequency tightening and fat grafting. Understand the importance of choosing the right surgeon for your body contouring needs. Explore the psychological aspects of dealing with skin changes after weight loss. What's your biggest challenge with skin quality after weight loss? Drop it in the comments! Subscribe for weekly insights on plastic surgery and mental health! #PlasticSurgery #SkinQuality #BodyContouring Did your surgeon talk to you about skin quality or radiofrequency tightening during your consultation? Are you considering a 360 lower body lift? Drop your questions and experiences in the comments below!

Brian "locked in" by eating more than he ever has before. Chicago’s best morning radio show now has a podcast! Don’t forget to rate, review, and subscribe wherever you listen to podcasts and remember that the conversation always lives on the Q101 Facebook page. Brian & Kenzie are live every morning from 6a-10a on Q101. Subscribe to our channel HERE: https://www.youtube.com/@Q101 Like Q101 on Facebook HERE: https://www.facebook.com/q101chicago Follow Q101 on Twitter HERE: https://twitter.com/Q101Chicago Follow Q101 on Instagram HERE: https://www.instagram.com/q101chicago/?hl=en Follow Q101 on TikTok HERE: https://www.tiktok.com/@q101chicago?lang=enSee omnystudio.com/listener for privacy information.

Brian "locked in" by eating more than he ever has before. Chicago’s best morning radio show now has a podcast! Don’t forget to rate, review, and subscribe wherever you listen to podcasts and remember that the conversation always lives on the Q101 Facebook page. Brian & Kenzie are live every morning from 6a-10a on Q101. Subscribe to our channel HERE: https://www.youtube.com/@Q101 Like Q101 on Facebook HERE: https://www.facebook.com/q101chicago Follow Q101 on Twitter HERE: https://twitter.com/Q101Chicago Follow Q101 on Instagram HERE: https://www.instagram.com/q101chicago/?hl=en Follow Q101 on TikTok HERE: https://www.tiktok.com/@q101chicago?lang=enSee omnystudio.com/listener for privacy information.

Around 700,000 gallbladder removals are performed in the US every year. Discover the gallbladder removal side effects they don't tell you about, what happens years after gallbladder removal, and why a healthy gallbladder is vital for digestive health.0:00 Gallbladder removal 0:27 Gallbladder function 1:24 The importance of bile1:53 SIBO (small intestinal bacterial overgrowth)2:45 Gallbladder stones5:40 Gallbladder removal side effects6:22 Fiber and digestive health6:59 Bile deficiency symptoms8:42 Pancreatic function after gallbladder removal 9:41 Choline and gallbladder health11:07 Gallbladder pain

Disclaimer: This episode and article are for educational purposes only and are not a substitute for individualized medical advice. Always consult your physician before making changes to your diet, exercise routine, or medications.TL;DR* The CDC's top 10 causes of death account for roughly 74% of all deaths in the US each year, and up to 80% of chronic disease is estimated to be driven by lifestyle factors — diet, movement, sleep, stress, and toxin exposure. * This episode goes cause by cause (heart disease, cancer, COVID/respiratory disease, accidents, stroke, Alzheimer's, diabetes, kidney disease, flu/pneumonia, and suicide) and lays out what the peer-reviewed research says about preventing, slowing, or reversing each one.* Ready to build your own plan? Book a free Metabolic Blueprint Session →The List, and the Number That Should Keep You Up at NightAccording to the CDC, ten conditions account for about 74% of all deaths in the United States every year:* Heart disease* Cancer* COVID-19 / chronic lower respiratory diseases* Accidents (unintentional injuries)* Stroke* Alzheimer's disease* Diabetes* Kidney disease* Influenza and pneumonia* SuicideHere's the part that doesn't get said enough: research estimates that up to 80% of chronic disease is driven by lifestyle factors — diet, movement, sleep, stress, and toxin exposure. Those aren't destinies. They're decisions, and decisions can change.A landmark paper in the Journal of the American Medical Association found that poor diet alone accounts for more deaths in the US than tobacco use. The fork may be more dangerous than the cigarette. And yet, most patients who end up on four to six medications were never once asked what they eat, how they sleep, or whether they move their bodies before those prescriptions were written. That's the difference between a sick care system and a healthcare system.Heart Disease and Stroke: The Most Reversible Disease We KnowHeart disease kills one American every 34 seconds and has held the #1 spot for over a century. But it's also one of the most reversible conditions in medicine.In 1990, Dr. Dean Ornish's landmark Lifestyle Heart Trial showed that intensive lifestyle changes — diet, exercise, stress management, and social support — could reverse coronary artery disease without drugs or surgery. Arterial plaques reduced. Blood flow improved. Chest pain decreased. A 2019 study in the Journal of the American College of Cardiology confirmed it: a plant-predominant diet, regular aerobic exercise, not smoking, and a healthy weight reduced cardiovascular risk by over 80%.What that looks like in practice:* A Mediterranean or whole-food, plant-based diet rich in omega-3s and antioxidants (this isn't the only diet that works — it's the broadest evidence-backed baseline)* Zone 2 cardio (low-intensity aerobic work, three to five times a week — a brisk walk counts)* Managing chronic inflammation, the real driver of arterial plaque formation* Reducing refined sugar and seed oils, which damage the endothelial lining of the arteries* Prioritizing sleep, since poor sleep spikes cortisol and directly harms the heartStroke follows the same playbook: control blood pressure through diet, exercise, and stress reduction. The DASH diet in particular has been shown in multiple studies to lower blood pressure as effectively as medication in many patients.Cancer: 30–50% Preventable, and Possibly More Metabolic Than GeneticTo be clear, this isn't a claim about curing cancer — it's about how much prevention research is available and underused. The World Health Organization estimates that 30–50% of cancers are preventable. The American Institute for Cancer Research has identified lifestyle factors that dramatically reduce risk across multiple cancer types:* Diet. A 2022 meta-analysis in The Lancet linked poor dietary patterns — low fiber, low fruit/vegetable intake, high processed meat — to one in five cancer deaths globally.* Obesity. Excess weight is now the second-leading risk factor for cancer after smoking, because fat tissue is metabolically active and produces hormones like estrogen and inflammatory cytokines that fuel cancer growth.* Exercise. A 2019 study of over 1.4 million people found higher physical activity was associated with lower risk across 13 cancer types, including breast, colon, and endometrial cancer.* Fasting and metabolic health. Emerging research on intermittent fasting and time-restricted eating shows it can lower insulin-like growth factor, a key promoter of cancer cell proliferation.* Toxin reduction. Chronic low-grade exposure to pesticides, plastics (BPA, phthalates), and environmental pollutants has been linked to increased cancer risk. Choosing organic where possible, filtering water and air, and reducing plastic in cookware and storage all matter.There's also a deeper shift happening in how researchers understand cancer itself. It's long been treated primarily as a genetic disease, but the evidence is increasingly pointing toward cancer as a metabolic disease. Dr. Thomas Seyfried at Boston College has done groundbreaking work showing that cancer cells overwhelmingly rely on glucose and glutamine fermentation for energy — which is why a ketogenic or low-glycemic approach may help starve cancer cells while protecting healthy ones. The research is still evolving, but it's compelling.Think of your body like a garden. You can water it, enrich the soil, and pull the weeds — or you can dump sugar and chemicals on it and wonder why nothing grows right. Disease doesn't happen to you. It grows in an environment you create, consciously or not. The good news: you can change that environment starting today.Alzheimer's: “Type 3 Diabetes”Alzheimer's is rising, and it's terrifying to watch someone go through it. But emerging science shows it has a strong lifestyle and metabolic component — researchers now sometimes call it “type 3 diabetes” because of the profound link between insulin resistance and neurodegeneration.Dr. Dale Bredesen's ReCODE Protocol, published in the Journal of Aging, showed that a multimodal lifestyle intervention — diet, exercise, sleep optimization, hormone balancing, and stress reduction — reversed early cognitive decline in the majority of patients treated. In his 2014 case series, 9 of 10 patients with early Alzheimer's or mild cognitive impairment showed measurable improvement, with several returning to work.Key strategies from the research:* Sleep is non-negotiable. The brain's glymphatic (waste clearance) system activates primarily during sleep, flushing out amyloid beta plaque. Poor sleep means plaque buildup.* Exercise. A 2020 study showed aerobic exercise increases BDNF (brain-derived neurotrophic factor) — essentially fertilizer for neurons — and reduces dementia risk by up to 35%.* Mediterranean-MIND diet. Shown to slow cognitive aging by an estimated 7.5 years in adherent individuals.* Blood sugar management. Insulin resistance in the brain disrupts its ability to use glucose for energy, effectively starving neurons.* Social connection. Harvard's 80-year longitudinal study found relationship quality was the single strongest predictor of cognitive health and longevity in old age.Type 2 Diabetes and Kidney Disease: One Disease Driving AnotherOver 37 million Americans have type 2 diabetes, and another 96 million are pre-diabetic. Here's the part that surprises people: type 2 diabetes is reversible in the majority of cases, and this isn't controversial — the science is overwhelming.The 2019 DiRECT trial, published in The Lancet, found that nearly half of type 2 diabetics achieved full remission through an intensive dietary intervention alone, with no medication. Other studies using low-carbohydrate and very-low-calorie diets have replicated these results consistently.Diabetic nephropathy — kidney damage from chronically high blood sugar — is the leading cause of kidney failure in the US. Control the upstream issue (blood sugar and metabolic dysfunction) and you protect the downstream organ.The diabetes reversal framework from the research:* Dramatically reduce refined carbohydrates and added sugars to blunt insulin spikes and reduce fat storage in the liver and pancreas* Practice time-restricted eating or intermittent fasting to improve insulin sensitivity* Add resistance training — muscle is your biggest glucose sink, so more muscle means better blood sugar control* Reduce visceral fat (the fat around and inside your organs), a key driver of insulin resistance and inflammationIf this list feels like a lot, that's fair. But it's not about blame — it's about empowerment. You have more control over your health outcomes than the medical system has typically told you.Respiratory Disease, Flu, and PneumoniaChronic lower respiratory diseases like COPD and emphysema are largely driven by smoking and air quality. Quitting smoking remains the single most impactful intervention here, full stop.For flu and pneumonia risk, immune resilience matters: vitamin D optimization, zinc and elderberry, and gut microbiome health (roughly 70% of your immune system lives in your gut). Even mild sleep deprivation has been shown to quadruple susceptibility to viral infection.Suicide and Mental HealthThis is the cause of death nobody wants to talk about, but it's in the top 10 — and it's the leading cause of death for men under 50, and the second-leading cause of death for people ages 10–34.While this is complex and deeply personal, research points to several lifestyle levers:* Exercise functions as an effective antidepressant, largely through endorphin release* The gut-brain axis matters more than most people realize — over 75% of the body's serotonin is produced in the gut, so gut health directly shapes brain chemistry* Social connection and purpose. Viktor Frankl's Man's Search for Meaning, written while he was in a Nazi concentration camp, captured this: meaning and connection to purpose are essential for survival, and perspective shapes both physiology and outcome* Reducing alcohol, a central nervous system depressant that worsens anxiety and depression despite short-term reliefIf you or someone you love is struggling, crisis hotlines are available 24/7. There's often a reason it's happening, and there's often a way through it.The Common ThreadAcross every single one of these conditions — heart disease, stroke, cancer, Alzheimer's, diabetes, kidney disease, respiratory disease, immune function, mental health — the same five levers keep showing up: diet, exercise, sleep, toxin reduction, and blood sugar balance.Your daily habits are either building disease or building resilience. Every meal, every walk, every night of good sleep compounds — just like interest in a bank account.This Week's Action Plan* Do a food audit. For the next three days, write down everything you eat. No judgment, just awareness — you can't change what you can't see.* Walk 30 minutes a day. It lowers blood pressure, improves insulin sensitivity, boosts BDNF, and reduces cortisol. Simple, not easy — do it anyway.* Optimize your sleep. Consistent bedtime, dark room, cool temperature, no screens.* Get your labs done. Know your fasting glucose, A1C, CRP, vitamin D, triglycerides, and LDL — these are your early warning systems.* Address one stress source this week. A conversation you need to have, a boundary you need to set, or ten minutes of breathwork. Chronic stress is silently killing you — take action on it.Ready for a Real Plan?If you're ready to take your health seriously and want a clear roadmap instead of guesswork, book a Metabolic Blueprint Session. In 30–45 minutes, we'll walk through your top three priority areas and how programs like the Metabolic Momentum Accelerator, the Cellular Reboot Accelerator, and the Total Health Restore Protocols can help.Schedule your complimentary consult →Know someone this could help? Share this episode with them — it might change or even save their life.If this episode was valuable, leaving a review on Spotify or Apple Podcasts helps this show reach more people.ReferencesStudies and sources cited in this episode:* CDC — Leading Causes of Death data* Journal of the American Medical Association (JAMA) — poor diet and US mortality* Ornish, D. et al. (1990) — The Lifestyle Heart Trial* Journal of the American College of Cardiology (2019) — plant-predominant diet, exercise, and cardiovascular risk reduction* DASH diet research on blood pressure control* World Health Organization (WHO) — cancer preventability estimates* American Institute for Cancer Research — lifestyle and cancer risk* The Lancet (2022 meta-analysis) — dietary patterns and cancer deaths* Physical activity and cancer risk study (2019, ~1.4 million participants)* Seyfried, T., Boston College — cancer as a metabolic disease* Bredesen, D., Journal of Aging — the ReCODE Protocol; 2014 case series* BDNF and exercise study (2020) — dementia risk reduction* Mediterranean-MIND diet and cognitive aging research* Harvard Study of Adult Development (80-year longitudinal study) — relationships and longevity* DiRECT Trial, The Lancet (2019) — type 2 diabetes remission through dietary intervention* Frankl, V. — Man's Search for MeaningThis article summarizes claims and studies as presented in the podcast episode. Listeners are encouraged to review primary sources directly and consult their own physician before making health decisions. This is a public episode. If you'd like to discuss this with other subscribers or get access to bonus episodes, visit tripleplaydoc.substack.com/subscribe

Beijing's spin cycle - washing away the "so-called" excess capacity problem. Listen for more o info on Two Minutes in Trade.

→ Puori 7/12 | Go to https://puori.com/DRG and use the code DRG at checkout to get 32% off your first Puori Creatine+ subscription order. → Timeline | Timeline's clinically proven formula is now available at a new, lower price. Mitopure now starts at $79, when you subscribe at https://timeline.com/DRG → Ora Oganic | Real ingredients, planet-friendly packaging, easy to love. Head to https://ora.organic/pages/drg and use my code HEALTHYSELF30 to enjoy 30% off your first Ora order today. Episode Description: Every person comes into this world with a spiritual container. If your container can only hold 60 watts of energy, that is all you are getting. No strategy, no opportunity, no amount of hustle changes that. What changes it is expanding the vessel itself. David Ghiyam is a Kabbalah teacher and spiritual coach who has spent 25 years working with people at the intersection of consciousness, abundance, and transformation. This is one of the most expansive conversations Heal Thy Self has ever had. It moves from money ceilings and competing belief systems to anxiety as a message from your highest destiny, to darkness as excess light, to the energetic pipes of mother and father that govern healing and abundance. It goes places most podcasts never go and it earns every minute of it. In this episode, you'll discover:  •  Why the ceiling you keep hitting with money, love, or opportunity has nothing to do with the outside world, and why fixing your circumstances without expanding your spiritual vessel is fighting a shadow instead of the seed  •   What competing belief systems are and how a hidden belief you are completely unaware of can quietly sabotage every goal your conscious mind is working toward  •   Why anxiety is not a problem to solve but a signal that your higher destiny is trying to come in and cannot, and what it actually takes to open the channel This one will stay with you. Find David Ghiyam: Website: https://davidghiyam.com/yis Instagram: https://www.instagram.com/davidghiyam/?hl=en Timestamps: 0:00 - Intro 1:00 - Your Spiritual Container: The Real Reason Wealth, Love and Opportunity Stop at a Certain Point 2:30 - The Two Belief Systems That Are Quietly Blocking Everything You Want 4:00 - The Hidden Desire That Was Costing David Millions Without Him Knowing It 6:00 - Why Moving Cities, Changing Jobs or Finding New People Won't Fix the Problem 8:30 - 100% Aligned Desires = Instant Manifestation — Here's What That Means 11:55 - Anxiety Is Not a Disorder — It's Your Higher Destiny Trying to Come In 13:00 - Darkness Is Not the Absence of Light — It's the Excess of It 13:35 - The Single Biggest Block for High-Achieving, High-Anxiety People 14:00 - Why Every Best Idea You've Ever Had Came When You Were Alone 15:00 - The Real Reason People Fear Public Speaking (It Has Nothing to Do With Confidence) 17:50 - Why Staying in Something That's Not Working Is Hurting Everyone Around You 19:00 - Giving From the Left Column vs. the Right Column (And Why Most Leaders Only Do One) 23:00 - The Energy of One Disengaged Employee Is Quietly Killing Your Company 25:00 - The Amazon $5,000 Quit Check — And Why David Uses the Same Principle 26:00 - Why You Can't Attract a Partner Who Has What You're Not Willing to Build in Yourself 29:00 - The Real Estate Broker, the $50M Listing, and the Lesson About Her Mom 31:50 - What Blaming Your Parents Is Actually Blocking in Every Area of Your Life 32:00 - Father Energy and Mother Energy: The Two Pipes That Govern Abundance and Love 34:30 - Prayer Is Not Woo Woo — It's a Technology That Expands the Vessel 35:30 - How to Call In the Healing (Even If You Don't Want to Want It Yet) 37:00 - Where to Find David and How to Go Deeper Learn more about your ad choices. Visit megaphone.fm/adchoices

In this special rerun episode of The Dairy Nutrition Blackbelt Podcast, Dr. Pedro Melendez, Clinical Full Professor at City University of Hong Kong, explains how abdominal adiposity and hypocalcemia affect transition cow health. He discusses genetic links to internal fat, risks of metabolic disease, urine pH monitoring, anionic diets, and the use of calcium boluses for older cows. Listen now on all major platforms!"Excess abdominal adiposity can trigger lipolysis, inflammation, mastitis, ketosis, and fatty liver while reflecting an important genetic component."Meet the guest: Dr. Pedro Melendez is a Clinical Full Professor in Bovine Production Medicine at City University of Hong Kong. He earned his DVM from the University of Chile and his MS and PhD from the University of Florida, with work focused on dairy production medicine, nutritional management, and metabolic disease. Learn more from Dr. Pedro Melendez on The Dairy Nutrition Blackbelt Podcast, available on all major platforms.Liked this one? Don't stop now — Here's what we think you'll love!What will you learn: (00:00) Highlight(01:44) Introduction(02:14) Guest background(03:30) Abdominal adiposity risks(06:41) Genetic factors role(08:14) Preventing hypocalcemia(10:49) Calcium bolus use(12:08) Closing thoughtsThe Dairy Nutrition Blackbelt Podcast is trusted and supported by the innovative companies:* Fortiva* Adisseo* Vetagro* Barentz* Priority IAC* Kemin- Esmilco Inc.- Virtus Nutrition- DietForge

Luxury yachting is often seen from the outside as glamour, freedom, celebrities, billionaires, royals, and floating palaces. But behind that image is a very different world of service, sacrifice, pressure, extraordinary human moments, and stories most people never hear.In this episode of Captain's Chat, Captain Liam Devlin speaks with Timmo Elliott and Gemma Masferrer, authors of Sailing Through the Excess, a book that pulls back the curtain on life inside the luxury yachting world.Timmo shares how years in hospitality, travel, and yachting led to a memoir filled with remarkable encounters, unforgettable guests, and the realities behind elite service. Gemma explains how she helped bring those stories together, shaping Timmo's experiences into a book with wider context around climate change, gender equality at sea, the MeToo movement, language, identity, and the sacrifices behind maritime life.This is a conversation about luxury yachting, but also about storytelling, human connection, nature, resilience, and the courage to step outside your comfort zone.Guests: Timmo Elliott & Gemma Masferrer Book: Sailing Through the Excess

Importers are watching to see whether upcoming Section 301 tariffs on 16 economies will add to forced-labor duties and conflict with existing trade agreements. For more information, listen to today's Two Minutes in Trade. 

In this episode of Slowmade, I'm talking with Los Angeles jewelry artist Tzipora Hoynik.Tzipora has been making and selling jewelry for around forty years, but her creative life began long before that. As a little girl, she told her father she wanted to be an artist, even though she didn't yet know exactly what that meant. Over the years, that pull toward making has taken many forms, from drawing and painting to glass blowing, ceramics, weaving, beads, metalsmithing, and eventually the bold, colorful, deeply textured jewelry she creates today.Tzipora's work begins with stones. Color is her starting point, and her pieces often feel layered with pattern, texture, movement, and embellishment. In our conversation, she shares how gemstones guide her designs, why she doesn't like to leave a surface blank, and how her love of color continues to keep the work exciting after so many years.We also talk about the evolution of a long creative career, including her early years making beaded jewelry, her time doing wholesale shows, and the moment production work started to feel more like work than play. Tzipora now focuses mostly on one-of-a-kind pieces, allowing the stones, the process, and her own instincts to lead.Along the way, we talk about her Los Angeles studio, her very practical relationship with tools, her longtime connection to Burning Man, the special jewelry pieces she has made for that community, and her Warrior Women series, a design she returned to after many years and reimagined with more detail, strength, and meaning.This conversation is also about what it means to keep creating over the long arc of a life. Tzipora talks about the need to make things as something deeply innate, something that feeds her and feels woven into who she is. Even after decades of working as an artist, she still finds joy in coming into the studio, playing with stones, and seeing what wants to become.We talk about creative devotion, staying true to your own voice, selling work through Instagram and shows, working with collectors, the reality of pricing handmade jewelry, and why the value of a piece is never only about the materials. It also carries the years of experience, the choices, the eye, the hands, and the voice of the artist who made it.If you've ever wondered what it looks like to build a life around making, not for a season, but for decades, I think you'll love this conversation with Tzipora.Follow Tzipora...Tzipora on Instagram: @opalgirlTzipora's Website: spiraljewelry.etsy.comLove the Podcast?Support an episode: ⁠Click here to learn more!⁠Without your support I couldn't continue the Slowmade Podcast. Thank you so much!Follow Christine and Slowmade...Instagram: ⁠@christinemighion⁠Jewelry Website: ⁠christinemighion.com⁠Slowmade: ⁠slowmadepodcast.com

Recorded live at the House of Suze during Tales of the Cocktail in New Orleans 2026.Why This Episode MattersDiscover how the American beefsteak became a tool for political organizing in New York and New Jersey.Learn the feast's essential rules: endless meat, bread as a plate and no concern for remaining clean.Hear how Prohibition and women's suffrage changed public drinking—and the beefsteak itself.Find out how New Jersey preserved the tradition after it faded elsewhere.The ConversationMark Pascal, Francis Schott and Sother Teague take the stage together to explore one of America's messiest, and most political, dining traditions.The American beefsteak began as a nineteenth-century feast where politicians served voters and ward leaders all the steak and beer they could handle. Matching aprons erased the appearance of social rank, and the room became a place to reward loyalty, recruit supporters and build political alliances.The panel lays out the beefsteak's non-negotiables: slices of beef served continuously on pieces of bread, no forks, no knives and traditionally no napkins. Diners eat the meat, stack the bread and use their aprons to wipe their hands. As Sother puts it, the guiding principle was “opulence over elegance.”They trace the tradition through Tammany Hall, organized labor and the East Side–West Side beefsteak rivalry. They also examine how Prohibition and women's suffrage helped transform the formerly all-male event into a more social and mixed-gender gathering.When beefsteaks began disappearing after World War II, New Jersey butcher Garrett “Hap” Nightingale revived them as fundraisers for fire departments, churches, and community organizations. The conversation also compares the beefsteak with Southern pig pickin's, crab boils, clambakes and pancake breakfasts.Along the way, Sother recounts the six-plane journey that brought him to Tales, while Mark and Francis explain how they transported steaks, produce and Himalayan salt bricks from New Jersey to New Orleans.The evening's menu includes brisket, roast beef, Wagyu, house-made mozzarella and steaks finished on 500-degree salt bricks. Steak sauce is permitted, although putting it on the Wagyu may cause Francis emotional distress.Guest BioSother Teague is a bartender, hospitality professional and drinks historian best known for Amor y Amargo and Overthrow Hospitality. He is also a co-host of The Speakeasy and Sauced podcasts.Timestamps00:00 Live from the House of Suze at Tales of the Cocktail02:15 What is an American beefsteak?05:00 Tammany Hall, political organizing and “opulence over elegance”10:00 Six planes, ten cooler bags and salt bricks on an airplane13:00 Excess, Prohibition and the end of the all-male beefsteak21:00 How New Jersey revived the tradition23:00 Pig pickings and other regional communal feasts25:00 Wagyu, salt bricks and the steak-sauce debateInfoSother Teague https://www.overthrowhospitality.com/teamPodcasts: The Speakeasy, SaucedRecorded AtHouse of SuzeTales of the CocktailNew Orleans, LouisianaFeatured PartnersSuze and BCIOld ForesterSerene Craft Beer DistributorsSubscribe: Restaurant Guys' Regularhttps://restaurantguysregulars.buzzsprout.com/Magyar Bankhttps://www.magbank.com/Stage Left Wine Shophttps://www.stageleftwineshop.com/Our PlacesStage Left Steakhttps://www.stageleft.com/Catherine Lombardi Restauranthttps://www.catherinelombardi.com/Stage Left Wineshophttps://www.stageleftwineshop.com/Reach Out to The Guys!TheGuys@restaurantguyspodcast.com

The episode begins with an introduction to the panel and research objectives. This sets the stage for a deeper discussion on how elevated liver copper levels may influence immune resilience in crossbred calves. The panel examines why the impact of excess concentrations of liver copper is significant for animal health. (00:00 – 02:55) Dr. Hansen then provides an overview of the featured Journal of Animal Science study, emphasizing its interdisciplinary approach and the motivation behind evaluating copper status in emerging beef-on-dairy production systems. She also references how excess copper concentrations in the liver can alter expected outcomes. (02:55 – 05:47) As the discussion progresses, the panel highlights how copper accumulates throughout the production cycle, from late gestation through milk replacer feeding. This results in many calves entering the feedlot with elevated liver copper levels. The excess may influence health outcomes. Importantly, the impact of liver copper concentrations in excess is considered in feedlot transition and calf growth. (05:47 – 10:33) A key theme throughout the episode is the concept of subclinical copper toxicity, where animals may experience negative physiological effects without obvious clinical signs. The panel explains how traditional definitions of copper toxicity may overlook subtle yet economically significant impacts on immune function and disease susceptibility, this is particularly important in relation to BRD. In the context of excess liver copper concentrations, even mild impact on metabolic processes is discussed. (10:33 – 12:28) Dr. Hansen then walks through the experimental design, outlining how calves were assigned to high and adequate-copper groups. They were subjected to a controlled BRD challenge that incorporated stress, viral exposure, and bacterial infection. These steps better simulate real-world conditions. The impact of concentrations exceeding normal liver copper thresholds is addressed in their study design. (12:28 – 17:46) The findings reveal that calves with elevated liver copper levels experienced more severe disease outcomes. This includes higher clinical scores, increased lung consolidation, and slower recovery. Therefore, excess copper in the liver clearly demonstrates a measurable impact on calf recovery and health status. (19:18 – 23:50) The panel explores potential mechanisms behind these results, including increased oxidative stress, reduced antioxidant capacity, and disruptions to normal immune signaling pathways. Next, the discussion also examines how excess copper may interfere with nutritional immunity. This happens by altering the balance and movement of other essential trace minerals, such as zinc and iron, during infection. Ultimately, this compromises the animal's ability to respond effectively to disease challenges. (27:17 – 29:10) From a practical standpoint, the episode provides valuable guidance on managing copper supplementation in cattle diets. The panel emphasizes the importance of evaluating total mineral intake across life stages. They recommend carefully formulating diets to avoid over-supplementation. They discuss the challenges associated with reducing liver copper once it has accumulated and the time required to bring levels back into an optimal range. Most notably, managing the impact of excess concentrations in liver copper requires proactive nutrition strategies. (34:26 – 36:07) The episode concludes with a discussion on diagnostic tools and future research needs, including the role of liver biopsies in accurately assessing copper status and the limitations of blood biomarkers. There is also a need for a deeper understanding of mineral dynamics during disease. The panel highlights the importance of industry education and awareness as this issue continues to gain attention. To summarize, the impact of excess liver copper concentrations is a subject demanding ongoing research and producer awareness. (43:12 – 47:15) Please subscribe and share with your industry friends to invite more people to join us at the Real Science Exchange virtual pub table.  If you want one of our Real Science Exchange t-shirts, screenshot your rating, review, or subscription, and email a picture to anh.marketing@balchem.com. Include your size and mailing address, and we'll mail you a shirt.

Watch the full discussion here: https://youtu.be/MwUJGWWFOSUJoin us for 'Fanon & Anders: On Revolution': https://www.patreon.com/acidhorizonpodcast/posts/final-summer-on-164624401?utm_medium=clipboard_copy&utm_source=copyLink&utm_campaign=postshare_fan&utm_content=web_shareWhat does it mean when even compliance with the demands of the neoliberal university no longer guarantees institutional legitimacy or security? As universities become increasingly governed through financial imperatives, visa regimes, managerial restructurings, and strategic repositioning, what forms of university life become impossible? What survives beyond them? If the university increasingly appears unable to sustain the conditions for collective intellectual life, where does study go? What forms of inquiry exceed the university, and what kinds of institutions – or anti-institutions – might emerge in its wake? To discuss the capitalisation of the university and the future of study, Adam C. Jones of Acid Horizon Research Commons hosts a panel alongside theorists Stevphen Shukaitis, John Preston, Angela McRobbie, and Giovanni Mormont.Link to Minor Compositions: https://www.minorcompositions.info/Support the showSupport the podcast:AHRCCurrent classes at Acid Horizon Research Commons (AHRC): acidhorizonresearchcommons.comAHRC Course Archive: https://www.acidhorizonpodcast.com/ahrc-course-archivesSubmit your course proposal: acidhorizonresearchcommons@gmail.comMore LinksWebsite: https://www.acidhorizonpodcast.com/Linktree: https://linktr.ee/acidhorizonAcid Horizon on Patreon: https://www.patreon.com/acidhorizonpodcastBoycott Watkins Media: https://xenogothic.com/2025/03/17/boycott-watkins-statement/Subscribe to us on your favorite podcast: https://pod.link/1512615438Merch: http://www.crit-drip.comSubscribe to us on your favorite podcast platform: https://pod.link/1512615438LEPHT HAND: https://www.patreon.com/LEPHTHANDHappy Hour at Hippel's (Adam's blog): https://happyhourathippels.wordpress.com​Split Infinities (Craig's Substack): https://splitinfinities.substack.com/​Music: https://sereptie.bandcamp.com/ and https://thecominginsurrection.bandcamp.com/

As part of the federal government's AI strategy, hyperscale data centres are being built across the country with many more projects being proposed. But, communities are pushing back, and it's capturing the attention of local governments.Projects in Ontario, British Columbia, and Saskatchewan have been the subject of protests and petitions for moratoriums. A recent Angus Reid poll reported that 68 per cent of Canadians object to a data centre being built near them.As Prime Minister Mark Carney pushes an agenda of digital sovereignty and AI expansion, we look at how Canadians are responding to the rapid construction of data centres, and the risks AI poses to the environment and local communities.Paris Marx joins us. He is a tech journalist and the host of the podcast, Tech Won't Save Us. He's got a book coming out this October called 'Hyperscale: The Ambition and Excess of Big Tech's Data Empires.'For transcripts of Front Burner, please visit: https://www.cbc.ca/radio/frontburner/transcripts

Mathew Pritchard is best known as one of the stars of MTV's Dirty Sanchez but, this chat goes far beyond the stories everyone has already heard... In this episode, Mathew joins Davi and Olly to talk about who he was before the fame, how skateboarding shaped his life, how Pritchard vs Dainton became Dirty Sanchez, what MTV saw in the group and what life was really like when the show exploded around the world. Mathew opens up about the difference between 'Mathew' and 'Pritchard', the pressure to perform in public, the money, the partying, the loss of routine and what happened when the fame slowly dwindled away. He also speaks honestly about addiction, anxiety, going cold turkey on 6 October 2022, the support system that helped him through and why routine, training, food and purpose are now central to his life. The conversation also covers The Road of Excess, Mathew's new documentary about rowing across the Atlantic, fame, excess, addiction, mental health and life after Dirty Sanchez. Plus veganism, Dirty Vegan, Dirty Health, Ironman Wales, Everest plans, why mountain bikers are 'nuts' and whether a Dirty Sanchez reunion could ever happen. Go see The Road Of Excess: https://mbentertainment.co.uk/the-road-of-excess/ Don't forget to book your ticket to TRC Live on 31st of July, details here:  https://www.haslemerehall.co.uk/sales/genres/events/the-ride-companion---bike-nigh Get early access & ad-free episodes → https://www.patreon.com/theridecompanion Episode Sponsors:- - RX Sport: RX Sport are your one stop eyewear shop. From prescription sunglasses, to riding glasses and everything in-between. Use code TRC at https://rxsport.avln.me/c/kdvMdXvBBUjH for a deal on Pit Viper products! - invisiFrame: 15% off with code REFRESHANDRIDE at https://www.invisiframe.co.uk You can also support our long term partners: Marin Bikes → marinbikes.com/gb Focus Bikes → focus-bikes.com SRAM: sram.com/en/sram adidas FiveTen: adidas.co.uk/five_ten invisiFrame: 15% off with code REFRESHANDRIDE at invisiframe.co.uk Manta Sleep → 10% off with code theridecompanion tinyurl.com/theridecompanion HUEL → 15% off with code RIDE: huel.com/ Mudhugger → Get 10% off with code ridecompanion10 at themudhugger.co.uk Compex → 20% off with code THERIDECOMPANION: compex.com/uk/ Igloo → igloocoolers.com/ Kecks → https://kecks.co.uk use code THERIDECOMPANION for 10% off Feedback Sports: feedbacksports.com WORX → 15% off with code THERIDECOMPANION at https://uk.worx.com/aff/theridecompanion/ HKT Products → 10% off with code PODCAST at hktproducts.co.uk Follow The Ride Companion Instagram @theridecompanion YouTube @TheRideCompanion Olly Wilkins Instagram @odub_23 YouTube @owilkins23 YouTube clips and BTS channel @moreridecompanion Get official Ride Companion merch, find old episodes and more theridecompanion.co.uk

Welcome to Insurance Covered, the podcast that covers everything insurance.In this episode, Wendy Hauser, President of US Wholesale and Specialty at Markel, shares insights into the world of excess and surplus insurance, its regulation, and its significance in the evolving insurance landscape. Discover how E&S fills gaps in coverage, the differences between admitted and non-admitted insurers, and the future of this dynamic market.We hope you enjoyed this episode, if you did please subscribe to be notified when new episodes release. Hosted on Acast. See acast.com/privacy for more information.

07 16 26 Excess Lime in Soil by Ag PhD

Weekly sermon from our evening service.

When cash flow gets tight, most practice owners look at revenue first. But one of the biggest drains on your cash may already be sitting on your shelves. Excess inventory doesn't just take up space—it ties up working capital, increases expiration risk, and quietly chips away at your margins.  In this episode, I break down why inventory management deserves more attention in your financial strategy and share practical ways to manage injectables, retail skincare, and consumables more effectively. Small changes in how you purchase, track, and replenish inventory can have a meaningful impact on your cash reserves and overall profitability.  Buying More Inventory Doesn't Always Save You Money  Bulk discounts can be tempting, but they're not always the best financial decision. Purchasing more product than you can realistically use may lower your cost per unit, but it also locks up cash that could be used for payroll, marketing, or other growth opportunities.  Inventory should support your practice—not compete with it for cash. Before placing a large order, consider your usage rate, seasonality, payment terms, and how quickly that inventory will actually generate revenue.  Every Product Category Needs a Different Strategy  Not all inventory should be managed the same way. Injectables, retail skincare, and everyday consumables each serve a different purpose in your practice and carry different levels of financial risk. Creating category-specific inventory strategies helps improve cash flow, reduce waste, and ensure you're investing in products that support both patient demand and long-term profitability.  Establish inventory par levels for injectables, retail skincare, and consumables  Track inventory turnover and usage by provider  Monitor expiration dates, shrinkage, spoilage, and waste  Assign one team member ownership of inventory reconciliation  Adjust purchasing based on seasonal demand instead of automatic reordering  Negotiate flexible payment terms with vendors whenever possible  Make sure every product on your shelves has a purpose. When you consistently monitor inventory levels and adjust purchasing decisions based on real usage, you free up cash, protect your margins, and create a more efficient operation.  04:00 Managing inventory responsibilities  07:31 Managing Inventory and Cash Flow  11:19 Tracking inventory and usage metrics  15:47 Upselling and Packaging Services  16:52 Managing inventory and pricing strategy Good Inventory Data Leads to Better Financial Decisions  Inventory counts should do more than confirm what's on the shelf. They should tell you how inventory is moving through the practice and whether it's contributing to healthy cash flow.  When you regularly review inventory tracking alongside your financial reports, it's much easier to identify underutilized inventory, margin loss, or products that aren't generating the return you expected. That visibility allows you to make adjustments before small issues become expensive ones.  Cash Should Keep Working for Your Business  As your med spa grows, inventory becomes more than an operational task—it becomes a strategic financial decision. Expanding locations, hiring providers, investing in new technology, or building cash reserves all require liquidity. Every dollar tied up in excess inventory is a dollar that can't be invested elsewhere.  Practices that scale successfully understand how to balance inventory levels with demand. They know what products generate revenue, what products move slowly, and when it's worth negotiating better payment terms instead of purchasing more inventory. Managing inventory with intention doesn't just protect margins—it creates the financial flexibility needed to grow with confidence.  Follow Shannon & Keep What You Earn:   Shannon Weinstein is the founder of a fractional CFO firm specializing in helping 7-figure aesthetics and wellness practices scale with clarity, cash flow, and confidence.  Shannon is committed to helping med spa owners understand, fix, and maximize their business's enterprise value, offering actionable advice and resources, including a popular free video series specifically for aesthetics practice owners.  Connect with Shannon:  Fractional CFO Services and Executive Financial Review: https://www.keepwhatyouearn.com/  Connect with Shannon: https://www.linkedin.com/in/shannonweinstein  Watch full episodes: https://www.youtube.com/@KeepWhatYouEarn  Listen on your favorite podcast app: https://pod.link/1580071347  Instagram: https://www.instagram.com/shannonkweinstein/  The information shared is for educational purposes only and is not individualized financial advice. Aesthetics practice owners should consult a qualified professional before implementing financial strategies discussed here. 

Enroll now at AHRC: https://www.acidhorizonpodcast.com/ahrc-coursesAttend 'University of Excess' free: https://www.patreon.com/acidhorizonpodcast/posts/new-panel-of-and-163158129?utm_medium=clipboard_copy&utm_source=copyLink&utm_campaign=postshare_creator&utm_content=join_linkBuy the Book: https://www.versobooks.com/products/3232-alexandre-kojeve?srsltid=AfmBOorrKB0ugBN0vBNZjBdT9aeqMWBFGCK5bOl9l8sOeWm7nmKwWQtpIn this intellectual biography, critic and philosopher Boris Groys turns to the Arthur Rimbaud of modern bureaucracy, Alexandre Kojève, a philosopher of little-known writings and profound influence. Kojève was fascinated with Hegel's dialectics and with communism and envisioned a universal empire as the end of history. Kojève drew on Buddhism and also proclaimed himself a Stalinist. At the same time, he was one of the creators of a nascent European Union. His concept of the human as something defined by negation and unique among animals in being separated from nature is highly political. It explains why humans can never be fully satisfied by a political system based on their allegedly ‘natural' rights.Groys reveals a Kojève with a unique perspective on our political capacities and human condition.Support the showSupport the podcast:AHRCCurrent classes at Acid Horizon Research Commons (AHRC): acidhorizonresearchcommons.comAHRC Course Archive: https://www.acidhorizonpodcast.com/ahrc-course-archivesSubmit your course proposal: acidhorizonresearchcommons@gmail.comMore LinksWebsite: https://www.acidhorizonpodcast.com/Linktree: https://linktr.ee/acidhorizonAcid Horizon on Patreon: https://www.patreon.com/acidhorizonpodcastBoycott Watkins Media: https://xenogothic.com/2025/03/17/boycott-watkins-statement/Subscribe to us on your favorite podcast: https://pod.link/1512615438Merch: http://www.crit-drip.comSubscribe to us on your favorite podcast platform: https://pod.link/1512615438LEPHT HAND: https://www.patreon.com/LEPHTHANDHappy Hour at Hippel's (Adam's blog): https://happyhourathippels.wordpress.com​Split Infinities (Craig's Substack): https://splitinfinities.substack.com/​Music: https://sereptie.bandcamp.com/ and https://thecominginsurrection.bandcamp.com/

If you've ever lost weight, gained it back, and convinced yourself it must be a willpower problem, this episode is for you. Kamy's whole message is the opposite. Excess weight in a teen is almost never about willpower, the food itself, or how much you move. It's a symptom of something deeper that nobody has helped you name yet. Today's guest is Kamy Moussavi, founder of Step Together, an organization that helps families address the true roots of children's excess weight (which Kamy sees as emotional stress, undiagnosed ADHD patterns, family dynamics, and life overwhelm, not just food). Kami grew up overweight and spent more than two decades cycling through Jenny Craig, Weight Watchers, restrictive diets, fasting, binge eating, and bulimia before he finally realized none of it was working because none of it was addressing what was actually happening underneath.   In this episode you'll hear why weight is a symptom and not the root problem, why diet and exercise alone don't break the cycle, the simple pattern recognition tool you can start using tonight, why your developing brain literally cannot do this work on its own (and what to do about that), how to have the kind of conversation with your parents that actually helps, the dopamine rerouting tools that can stand in for food when emotions hit, and why parents have to lead with role modeling instead of lecturing if anything is going to change.   Why excess weight in teens is almost never about willpower or food itself The yo yo cycle and what actually drives it underneath Why parents focusing on the food alone keeps the loop going The pattern recognition tool (catalog what happened before the ice cream, not just the ice cream) Why your frontal lobe is still developing into your mid 20s and what that means for impulse control How to ask a parent for support without making it worse Why "hold the space, don't jump to solutions" is the most powerful thing a parent or friend can do The dopamine rerouting list (touch, play, creativity, movement, social connection) Why connecting how food makes you FEEL is more powerful than counting calories Why a strong "why" matters more than any meal plan How a parent role modeling new habits rewires what their teen absorbs at home   Parents: This is one of the most important episodes for any parent of a teen who is struggling with their weight, with restrictive eating, with binge eating, or with the sense that food has quietly become bigger than it should be. Kamy's central message is one that almost every weight focused program misses: the focus should not be on your child at all, it should be on what the home environment and the emotional ecosystem are teaching them. He shares specific ways to role model the behavior you want to see, hold space when your teen is struggling, and become the parent your teen comes to instead of the parent your teen hides from. Listen with your teen if you can.   Heads up before you dive in: This episode talks openly about excess weight, binge eating, bulimia, restrictive dieting, weight shame, and the emotional roots underneath all of it. If any of this feels heavy for you right now, please take care of yourself and come back when you're ready, or listen with a parent or trusted adult nearby. If you've been struggling with these patterns for months or longer, please reach out to a professional for support.   Step Together website:  steptogether.com   Connect with Leslie: Ready to talk through what your teen is navigating and what kind of support would actually help? Book a free clarity call with Leslie at leslierosecoaching.com/chat/   Grab Leslie's free guide: 5 Ways to Help Your Teen Eat Better Without Making Things Worse Website: leslierosecoaching.com Instagram: instagram.com/leslierosecoaching Facebook: facebook.com/leslierosecoaching Disclaimer    

On the surface, the USA is thriving and a "light unto the world", as Trump recited in his speeches marking the 250th anniversary of the US Declaration of Independence on July 4th 1776. But is this 'national mythos' still valid? Was it ever? Does the US live up to its claims of uniqueness with respect to 'freedom' and 'democracy'? If not, what is this exceptional nation's 'chief feature'? Excess? By extreme contrast, the country the US arguably just lost a war against, Iran, is undergoing...

Reports that Meta is selling excess AI compute sparked an important question across the investment community:Is this the first sign that AI demand is slowing?Or is the market reading too much into a normal part of operating hyperscale infrastructure?In this episode, we separate headlines from fundamentals and examine what Meta's move could actually mean for the broader AI investment thesis.⭐ Sponsored by Podcast10x - Podcasting agency for VCs - https://podcast10x.comKey topics we explore:– Why Meta may be selling excess compute capacity– Does this signal weakening AI demand or simply better capital allocation?– How hyperscalers think about infrastructure utilization– What this means for Nvidia, Micron, and the broader AI supply chain– Why compute utilization is becoming just as important as compute capacity– The key indicators investors should monitor to assess the health of the AI buildoutThe bigger question:Is Meta's decision an early warning sign for the AI infrastructure boom, or evidence that the industry is entering a more mature phase where optimizing compute matters as much as building it?For investors, understanding the difference between excess capacity and weakening demand is critical to evaluating the next phase of the AI race.LINKSPrashant Choubey - ⁠https://www.linkedin.com/in/choubeysahab⁠Subscribe to VC10X newsletter - ⁠https://vc10x.beehiiv.com⁠Subscribe on YouTube - ⁠https://youtube.com/@VC10X⁠Subscribe on Apple Podcasts - ⁠https://podcasts.apple.com/us/podcast/vc10x-investing-venture-capital-asset-management-private/id1632806986⁠Subscribe on Spotify - ⁠https://open.spotify.com/show/7F7KEhXNhTx1bKTBFgzv3k?si=WgQ4ozMiQJ-6nowj6wBgqQ⁠VC10X website - ⁠https://vc10x.com⁠For sponsorship queries reach out to prashantchoubey3@gmail.comThis channel is for asset managers, allocators, and investors who want analysis that holds up — not headlines dressed as insight.Subscribe for weekly data-driven breakdowns of the forces reshaping capital markets.#Meta #AI #ArtificialIntelligence #Datacenter #Nvidia #Micron #Semiconductors #CloudComputing #Investing #Stocks #TechStocks #VC10X #Infrastructure #CapitalMarkets #Finance #VentureCapital #WallStreet #BigTech

In this episode, Alexis Holifield shares her journey into real estate, focusing on excess proceeds and overages, and how her faith and family background influence her business approach. Discover how delegation, market research, and a kingdom mindset drive her success in helping families recover wealth during challenging times.   Professional Real Estate Investors - How we can help you: Investor Fuel Mastermind:  Learn more about the Investor Fuel Mastermind, including 100% deal financing, massive discounts from vendors and sponsors you're already using, our world class community of over 150 members, and SO much more here: http://www.investorfuel.com/apply   Investor Machine Marketing Partnership:  Are you looking for consistent, high quality lead generation? Investor Machine is America's #1 lead generation service professional investors. Investor Machine provides true 'white glove' support to help you build the perfect marketing plan, then we'll execute it for you…talking and working together on an ongoing basis to help you hit YOUR goals! Learn more here: http://www.investormachine.com   Coaching with Mike Hambright:  Interested in 1 on 1 coaching with Mike Hambright? Mike coaches entrepreneurs looking to level up, build coaching or service based businesses (Mike runs multiple 7 and 8 figure a year businesses), building a coaching program and more. Learn more here: https://investorfuel.com/coachingwithmike   Attend a Vacation/Mastermind Retreat with Mike Hambright: Interested in joining a "mini-mastermind" with Mike and his private clients on an upcoming "Retreat", either at locations like Cabo San Lucas, Napa, Park City ski trip, Yellowstone, or even at Mike's East Texas "Big H Ranch"? Learn more here: http://www.investorfuel.com/retreat   Property Insurance: Join the largest and most investor friendly property insurance provider in 2 minutes. Free to join, and insure all your flips and rentals within minutes! There is NO easier insurance provider on the planet (turn insurance on or off in 1 minute without talking to anyone!), and there's no 15-30% agent mark up through this platform!  Register here: https://myinvestorinsurance.com/   New Real Estate Investors - How we can work together: Investor Fuel Club (Coaching and Deal Partner Community): Looking to kickstart your real estate investing career? Join our one of a kind Coaching Community, Investor Fuel Club, where you'll get trained by some of the best real estate investors in America, and partner with them on deals! You don't need $ for deals…we'll partner with you and hold your hand along the way! Learn More here: http://www.investorfuel.com/club   —--------------------

Late notice to an excess carrier is one of those “small” steps that can explode into a coverage dispute at the worst possible time, like right before mediation or on the eve of trial. We talk through why excess claims notification is getting tougher in today's market and how retail agents can protect their clients and themselves when a claim starts creeping toward the tower.We're joined by CRC Group's claims pros who live this every day: Dave Gilfillian, Chief Claims Officer and leader of Claims Advocacy, Ari Shapiro, Claims Advocacy Director, and Cammie Powell, Senior Claims Coordinator. Together, we unpack what's driving bigger claims, how plaintiff expectations and litigation funding can change the exposure curve, and why excess carriers are more likely to take a hard line on late notice. We also explain the concept of “prejudice,” how state law affects the analysis, and what a reservation of rights letter can signal when notice comes in too late.We also challenge a dangerous assumption: “follow form” does not automatically mean “same rules.” Excess policies often include pages of exceptions, unique notice provisions, and surprises around defense costs and endorsements, especially in complex towers with multiple carriers. Our practical takeaway is straightforward: when the thought pops into your head that excess might be involved, send notice, document it clearly, and keep communication moving so no layer is blindsided. Visit REDYIndex.com for critical pricing analysis and a snapshot of the marketplace.Do you want to take your career to the next level? Join #TeamCRC to get access to best-in-class tools, data, exclusive programs, and more! Send your resume to resumes@crcgroup.com today!

What does it mean to trust that life will work out—not as a belief, but as a lived, embodied experience?In this solo episode, Dr. Amy Wheeler explores the intersection of the Yamas—the ethical foundation of yoga—and the neuroscience of safety, regulation, and predictive processing. Rather than approaching ethics as a set of rules to follow, this conversation reframes them as natural expressions of a regulated, supported nervous system.At the center of this inquiry is a simple but powerful question: Can we live in alignment with our values when we feel unsafe?Drawing from yoga philosophy, polyvagal theory, and lived experience, Amy offers a grounded perspective on why ethical behavior becomes more accessible when the body feels steady—and why it often breaks down in states of survival.Key Themes ExploredEthics as a State, Not a Rulebook The Yamas are not imposed from the outside. They emerge when the system is regulated, resourced, and supported.Ahaṅkāra and the Survival Response The drive to secure, control, and protect—while necessary—can narrow perception and disrupt our ability to act with clarity and integrity. The Nervous System and Trust A flexible, regulated nervous system supports connection, openness, and discernment. When dysregulated, the system shifts toward threat detection, reactivity, and contraction. Predictive Processing and Learned Safety The brain continuously predicts what will happen next based on past experience. Trust is not naïve—it is built through repeated experiences of support, repair, and resilience. From Fear-Based Reactivity to Possibility Through intentional practice, the mind can shift from worst-case predictions toward openness, receptivity, and grounded optimism.The Yamas Through a Nervous System LensAhimsa (Non-harming): Begins internally. A regulated system reduces reactivity, blame, and harsh self-judgment.Satya (Truthfulness): Requires humility and discernment. When fear and ego soften, truth becomes less about being right and more about being aligned.Asteya (Non-stealing): Trust reduces grasping. When we feel supported, we are less likely to take what is not ours—ideas, credit, or energy.Brahmacharya (Moderation): Regulation is sustained through balance. Excess and depletion disrupt clarity and ethical consistency.Aparigraha (Non-grasping): Letting go of fear-based holding creates space for discernment, freedom, and right timing.Clinical InsightEthical challenges in yoga therapy—and in healthcare more broadly—are often not failures of character. They are reflections of nervous system states.When practitioners are exhausted, unsupported, or operating in survival mode, their window of tolerance narrows, and ethical clarity becomes harder to access. This reframing invites a shift from judgment to understanding, and from rules to regulation.A Subtle ReorientationAs safety becomes more embodied:The breath becomes more adaptableThe heart rate more flexibleSocial engagement comes onlineAmbiguity is interpreted with more generosityThe future is no longer predicted as threat, but as possibility From this place, trust is not forced—it emerges.Reflection for ListenersConsider where your system feels:Steady and openContracted and protectiveHow might your ethical responses shift depending on that state?And what practices help you return to a place where clarity, connection, and trust are more available?ClosingThis episode offers a reframe that may feel both grounding and challenging: That living the Yamas is not about trying harder—but about creating the internal conditions where they can naturally arise.

As tech companies race to build hyperscale data centers, communities are coming together to push back. Saul Levin joins Paris Marx to discuss how rising opposition to data center construction is uniting people across party lines and prompting broader conversations around what infrastructure people want instead. Saul Levin is community organizer and host of The Hum.Tech Won't Save Us offers a critical perspective on tech, its worldview, and wider society with the goal of inspiring people to demand better tech and a better world. Support the show on Patreon.The podcast is made in partnership with The Nation. Production is by Kyla Hewson.Also mentioned in this episode:You can now pre-order Paris's new book, Hyperscale: The Ambition and Excess of Big Tech's Data Empires.Saul wrote a piece with Astra Taylor about the bigger picture of data center organizing.The Seminole Nation in Oklahoma passed a ban on AI data centers on their lands.Sam Altman and Gov. Gretchen Whitmer posing together at the site of a new data center drew wide criticism.Support the show

SHOW NOTES: World Cup attendance and revenues are disappointing. That's hardly surprising. The hype and publicity and exaggerated claims create a level of expectation that's hard to support, and the inflated prices of everything from hot dogs to hotel rooms is absurd. This kind of insistence on constantly "topping" what's come before exists with Super Bowl halftime shows, entertainment award shows (they are proliferating in number as well as production extremes), parades for any variety of causes, political rallies, and even fundraising. The more the pressure to "better" and "outdo" the past attempts as a metric of current success, the more you're prone to fall short. No one has ever sung the Star Spangled Banner better than Whitney Houston, so stop trying to create a canonical version, and just sing the song. Tom Brady wasn't trying to be better than Joe Montana or Johnny Unitas, just the best quarterback he could be.Picasso was just trying to be Picasso. They're wondering why attendance is down when a train ticket was $15 is now $150 to get to the Meadowlands in New Jersey from Penn Station in Manhattan, and it can cost as much as $175 to park at Gillette Stadium on Foxboro, MA for the World Cup (where it's $50 for Patriot game). The road to excess had a lot of potholes.

Discover all of the podcasts in our network, search for specific episodes, get the Optimal Living Daily workbook, and learn more at: OLDPodcast.com. Episode 3606: Dori Cameron challenges five common mental shortcuts that make unnecessary purchases feel justified, from assuming you can return an item to believing it can always be stored, repaired, donated, or resold later. By exposing the hidden costs in time, money, and effort behind these rationalizations, she offers a practical framework for making more intentional buying decisions and reducing clutter before it starts. Read along with the original article(s) here: https://www.becomingminimalist.com/myths-that-cause-overspending/ Quotes to ponder: "If you are thinking “I can always return it,” the item may not be something worth purchasing in the first place." "A rule of thumb when making a purchase is to assume it has no resale value." "When you are in buying mode, ask yourself where in your house the object will likely be a year from now." Episode references: eBay: https://www.ebay.com Craigslist: https://www.craigslist.org Poshmark: https://poshmark.com Wealthfront's high-yield Cash Account: ⁠⁠https://wealthfront.com/OFD⁠⁠ This experience may not be representative of other Wealthfront clients, and there is no guarantee of future performance or success. Experiences will vary. The Optimal Finance Daily Podcast, Diana Merriam (collectively "Media Partner") are not clients of Wealthfront. The Media Partner receives cash compensation from Wealthfront Brokerage for this paid endorsement placed in their video, creating a conflict of interest. More details available via the referral link. The Direct Deposit Plus Investing Program from Wealthfront Advisers LLC and Wealthfront Brokerage LLC provides eligible clients a 0.25% APY increase above the base APY on eligible Cash Account balances (up to an overall boosted rate of 4.30% for a limited time when including the 0.75% APY boost for new clients) when you direct deposit $1,000 a month, plus open, fund, and maintain an investing account. Wealthfront may change or end the program at any time and determine eligibility at its discretion. Terms apply. Full details at ⁠⁠wealthfront.com/promo-terms⁠⁠.  The Cash Account, which is not a deposit account, is offered by Wealthfront Brokerage LLC ("Wealthfront Brokerage"), Member FINRA/SIPC. Wealthfront Brokerage is not a bank. The Annual Percentage Yield ("APY") on cash deposits as of January 30, 2026, is representative, requires no minimum, and may change at any time. References to the APY for the Wealthfront Cash Account, including any APY increase, are to the APY paid by insured depository institutions that participate in our cash sweep program (the "Program Banks”).. Wealthfront Brokerage sweeps cash balances to Program Banks, where they earn the variable APY. Investing involves risk, including the possible loss of principal. Securities investments are not bank deposits, bank-guaranteed or FDIC-insured, and may lose value. Investment advisory services are provided by Wealthfront Advisers LLC, an SEC-registered investment adviser. Learn more about your ad choices. Visit megaphone.fm/adchoices

[00:00] - Intro [01:01] - How acid lowers pH [03:14] - Hypochlorite chlorines [04:20] - How hypochlorites are made [06:20] - Chlorines leave behind muriatic acid in water [07:28] - Excess hydroxides [09:50] - What can be done to limit the alkalinity rise? [12:01] - Closing ______________________________Connect with us! Realize your full potential.Watershape University®Water chemistry questions?Orenda®Questions? Comments? Or apply to sponsor the show:ruleyourpool@gmail.com Facebook: @ruleyourpoolYouTube: @rule-your-pool

Discover all of the podcasts in our network, search for specific episodes, get the Optimal Living Daily workbook, and learn more at: OLDPodcast.com. Episode 3606: Dori Cameron challenges five common mental shortcuts that make unnecessary purchases feel justified, from assuming you can return an item to believing it can always be stored, repaired, donated, or resold later. By exposing the hidden costs in time, money, and effort behind these rationalizations, she offers a practical framework for making more intentional buying decisions and reducing clutter before it starts. Read along with the original article(s) here: https://www.becomingminimalist.com/myths-that-cause-overspending/ Quotes to ponder: "If you are thinking “I can always return it,” the item may not be something worth purchasing in the first place." "A rule of thumb when making a purchase is to assume it has no resale value." "When you are in buying mode, ask yourself where in your house the object will likely be a year from now." Episode references: eBay: https://www.ebay.com Craigslist: https://www.craigslist.org Poshmark: https://poshmark.com Wealthfront's high-yield Cash Account: ⁠⁠https://wealthfront.com/OFD⁠⁠ This experience may not be representative of other Wealthfront clients, and there is no guarantee of future performance or success. Experiences will vary. The Optimal Finance Daily Podcast, Diana Merriam (collectively "Media Partner") are not clients of Wealthfront. The Media Partner receives cash compensation from Wealthfront Brokerage for this paid endorsement placed in their video, creating a conflict of interest. More details available via the referral link. The Direct Deposit Plus Investing Program from Wealthfront Advisers LLC and Wealthfront Brokerage LLC provides eligible clients a 0.25% APY increase above the base APY on eligible Cash Account balances (up to an overall boosted rate of 4.30% for a limited time when including the 0.75% APY boost for new clients) when you direct deposit $1,000 a month, plus open, fund, and maintain an investing account. Wealthfront may change or end the program at any time and determine eligibility at its discretion. Terms apply. Full details at ⁠⁠wealthfront.com/promo-terms⁠⁠.  The Cash Account, which is not a deposit account, is offered by Wealthfront Brokerage LLC ("Wealthfront Brokerage"), Member FINRA/SIPC. Wealthfront Brokerage is not a bank. The Annual Percentage Yield ("APY") on cash deposits as of January 30, 2026, is representative, requires no minimum, and may change at any time. References to the APY for the Wealthfront Cash Account, including any APY increase, are to the APY paid by insured depository institutions that participate in our cash sweep program (the "Program Banks”).. Wealthfront Brokerage sweeps cash balances to Program Banks, where they earn the variable APY. Investing involves risk, including the possible loss of principal. Securities investments are not bank deposits, bank-guaranteed or FDIC-insured, and may lose value. Investment advisory services are provided by Wealthfront Advisers LLC, an SEC-registered investment adviser. Learn more about your ad choices. Visit megaphone.fm/adchoices

California cherry growers are facing one of the most difficult seasons in recent memory after multiple spring rain events devastated what had initially appeared to be a promising crop. According to longtime industry veteran Michael Jameson of Morada Produce, the 2026 California cherry season may be the worst he has seen in nearly four decades in the business. Jameson, who has worked in the cherry industry for 38 years, said growers entered the season with optimism. Strong winter chill accumulation, favorable bloom conditions, and excellent fruit set had many expecting an ideal crop of approximately 8.5 to 9 million cartons. That production level is considered optimal for California's harvesting, packing, and marketing infrastructure while also providing profitable returns to growers. However, Mother Nature had other plans. A series of rainstorms struck California cherry-producing regions during critical stages of fruit development. The first major rain event arrived in April, followed by several additional storms that damaged both early- and late-season varieties. According to Jameson, cherries become highly vulnerable once they begin transitioning from green to red. Excess moisture absorbed through the roots and fruit stems can cause cherries to crack when the fruit skin cannot stretch enough to accommodate the additional water. The result was widespread crop damage across multiple growing regions. Jameson noted that San Joaquin County alone experienced estimated losses exceeding 63 percent of its cherry crop, with economic losses reaching approximately $174 million. When factoring in damage across all California cherry-producing districts, total losses likely climbed into the hundreds of millions of dollars. Even fruit that survived the storms presented challenges. Modern optical sorting technology allows packinghouses to identify and remove defective cherries more effectively than in previous decades. While that technology helped salvage portions of the crop, Jameson explained that many cherries still suffered from reduced shelf life due to repeated weather events. Fruit that appeared marketable when packed often deteriorated during transportation to distant markets. That created significant problems throughout the supply chain. Shipments arriving at retailers sometimes failed to meet quality standards, forcing receivers to reject loads. Those rejected loads then had to be diverted to wholesale markets, often resulting in substantial financial losses. Jameson said rejected cherry shipments can cost growers and shippers between $100,000 and $150,000 per load. During this season, some operations experienced multiple rejected loads in a single day. Beyond the financial impact, Jameson expressed concern about the effect on consumers. When shoppers purchase cherries that lack flavor, firmness, or shelf life, they are less likely to return and buy additional fruit. Maintaining consumer confidence is critical for specialty crops like cherries, where repeat purchases drive seasonal demand. Despite the challenges, growers remain committed to producing high-quality fruit and navigating difficult conditions. Jameson emphasized that farming is inherently unpredictable, with weather often determining whether a season becomes highly profitable or financially devastating. For California's cherry industry, 2026 will likely be remembered as a season that tested the resilience of growers, packers, shippers, and marketers alike.

Upcoming Events Snowfighters Institute Webinars: Join us live for monthly webinars built to help snow pros run stronger, more profitable operations. All sessions run 10:00 to 11:00 AM. Pricing & Estimating Review | Tuesday, July 7, 2026 Are you pricing for profit or just hoping to break even? Finding & Managing Subcontractors | Tuesday, August 11, 2026 How do you find subcontractors who actually show up when it snows? Capacity Planning | Tuesday, September 8, 2026 How do you determine your true operational capacity? Recruiting | Tuesday, October 13, 2026 Why can't you find good people to hire, and what can you do about it? Incentive Compensation & Rewards | Tuesday, November 10, 2026 Are your bonuses and rewards actually driving the results you want? Client & Employee Appreciation | Tuesday, December 8, 2026 Are you truly appreciating your clients and employees, or just going through the motions? See the full webinar list → In-Person Event GROW! Snow | September 22 to 23, 2026 An in-person event built for snow leaders and their teams. Two days of snow-specific breakout sessions, a facility tour, and content designed to drive real change at your business. Details coming soon. Episode #57Matt Delborrello, Commercial Insurance Consultant with Alera Group and an Accredited Professional in Risk and Insurance, joins Phil to demystify the world of business insurance for snow and ice contractors. From understanding how umbrella liability provides broader protection than raising general liability limits, to why being rated on payroll instead of sales can dramatically lower your premiums, to navigating the excess and surplus marketplace, Matt shares why responsiveness and relationships drive his work, how site selection affects insurability, and what every contractor needs to know before the next slip and fall claim hits. Key Learnings Umbrella Liability Beats Raising General Liability Limits - Umbrella coverage isn't auditable like general liability, so it gives you broader protection over auto and employer's liability without driving up your audit exposure. Sales vs Payroll Rating Changes Everything - Being rated on snow payroll instead of total snow sales gives carriers a more accurate picture of your true exposure and can dramatically lower your premiums. Guaranteed Cost Contracts Distort Sales Numbers - When clients pay $500,000 whether it snows 2 inches or 200, sales doesn't reflect actual risk exposure, which is why the payroll rating shift matters so much. Choose Your Clients Wisely - You can run a flawless snow operation, but if your client list is heavy on gas stations and big box retail, insurance carriers may decline to write the account because of slip and fall frequency. Site Type Matters More Than Industry Label - A local bank and a Walmart are both retail, but they carry completely different risk profiles, so generic application categories without conversation create real problems. Camera Footage Defends Against Bogus Claims - Forward-facing, rear-facing, and driver-facing cameras give carriers the evidence they need to fight inflated or fraudulent claims rather than just settling them. You Can Be Involved... Chapters (00:00:20) - Welcome and Introductions(00:01:55) - Inside Alera Group(00:05:49) - Insurance 101 for Contractors(00:07:47) - Why Umbrella Beats Higher GL Limits(00:10:24) - A Day in the Life of an Agent(00:14:54) - Why Carriers Don't Get Snow(00:20:18) - The Sales vs Payroll Shift(00:24:24) - The Clients Carriers Hate(00:28:15) - The Bogus Slip and Fall Story(00:31:29) - Who Really Decides Your Claim(00:38:32) - The Excess and Surplus Trap(00:43:16) - One Entity or Two(00:45:01) - How to Reach Matt

Welcome to the land podcast, a platform for people looking to educate themselves in the world of land ownership, land investing, staying up to date with current land trends in the Midwest, and hearing from industry experts and professionals.  On today's episode, we are back in the studio with Alec Bean. We discuss: Section 180 has existed since the late 1950s. Excess soil fertility can create significant tax deductions. Most farmland owners have never utilized the deduction. Recreational farms with tillable acres can qualify. Timberland generally does not qualify. Active participation creates larger tax advantages. Some buyers use soil tests before making offers. Midwest farms commonly show $500-$2,500/acre in value. One client generated over $2.5 million in deductions. Data center money is creating major farmland demand. And so much more! Connect with Alec: https://asmlabs.net/irs-180/  Thanks again for all of the support from our partners—none of this would've been possible without them! - Buck Land Funding: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.firstbankers.com/bucklandfunding⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ -Hawke Optics | Use Code WHTL for 15% off:⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ https://bit.ly/hawkeoptics_⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ -OnX:⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ https://bit.ly/onX_Hunt⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ -Painted Arrow: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠bit.ly/PaintedArrow⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ - Latitude Outdoors: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.latitudeoutdoors.com/⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ - Whitetail Master Academy ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.whitetailmasteracademy.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ - Use code '⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠HOFER' to save 10% off at ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠www.theprairiefarm.com⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ - Massive potential tax savings: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ASMLABS.Net⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠

“You're Fat, Stop Eating”: The Power of Fasting And A New Way to Think About Body Fat by Oman Morales https://www.amazon.com/dp/B0DSB22DKM Fatistimenotweight.com “You're Fat, Stop Eating”: The Power of Fasting And A New Way to Think About Body Fat: By Oman Morales has achieved a remarkable feat by being as Top #50 best seller in One of Amazon’s categories. #38 Best Seller ★ in Teen & Young Adult Diet & Nutrition eBooks (Feb 18, 2025) Are you ready to transform your health and your life? You've tried countless diets, workout programs, and quick fixes, but nothing seems to deliver the lasting results you're after. You're not alone. The truth is, most approaches fail because they don't address the root of the problem. Excess body fat, low energy, and a rocky relationship with food aren't just physical issues—they're the result of a broken system that's been feeding us lies about health for decades. This book is your wake-up call. In You’re Fat Stop Eating, I strip away the noise and give you a clear, proven path to reclaim control over your body and mind. Through the science and simplicity of fasting, you'll learn how to unlock your body's natural power to heal, burn fat, and thrive. This isn't another diet gimmick or fitness fad—it's the key to real transformation. Here's what you'll discover: Why fasting is the ultimate reset for your body and brain. How to start fasting today, no matter your lifestyle. The surprising truth about body fat—and why it's not your enemy. How fasting supercharges longevity, immunity, and overall health. Practical strategies to break free from food cravings and emotional eating. This isn't just a book; it's a movement to empower you to live longer, feel stronger, and take control of your future. Whether you want to lose weight, improve your fitness, or prevent chronic illness, You’re Fat Stop Eating is your guide to unlocking the healthiest, happiest version of yourself. You've been lied to about what your body needs. It's time to rewrite the story. Your transformation starts now. About the author With over 30 years of expertise in the fitness industry, Oman Morales has dedicated his life to transforming how people approach health and wellness. Combining deep knowledge of fitness, nutrition, and human behavior, Oman delivers straightforward, actionable advice that empowers individuals to achieve lasting physical, mental, and emotional well-being. Oman's unique ability to simplify complex concepts has made him a trusted mentor to countless individuals seeking practical solutions to their health challenges. His no-nonsense approach is rooted in real-world experience and a passion for helping others break through barriers to unlock their full potential. Originally from Northern California, Oman now lives in Knoxville, Tennessee, with his wife and daughter. When he's not coaching or writing, Oman enjoys exploring new fitness methodologies, connecting with the community, and continuing his mission to inspire change one person at a time. Oman's work is a must-read for anyone ready to take control of their health and embrace a better future.

SpaceTime Series 29 Episode 74 Why Venus spins backwards A new study suggests that the strange retrograde spin of the planet Venus is the result of a massive impact event. Could Dark Matter explain what's happening at the centre of our galaxy A new study has failed to rule out Dark Matter as the source of the so called Galactic Center Excess at the heart of the Milky Way galaxy. Trying to solve a meteor cold case Last month astronomers detected a small near Earth meteoroid on a collision course with our planet. The Science Report The deadly H5N1 strain of bird flu detected on the Australian mainland for the first time. Australia's Bureau of Meteorology has officially declared an El Niño. The risk of suicide among males can persist for years following a relationship break up. Research continues on nuclear diamond batteries that could last thousands of years. A new species of shark discovered in the tropical Pacific, north of Australia. Skeptics guide to five lessons on misinformation from the ancients. Our Guests This Week: Dr Hadrien Devillepoix from Curtin University NASA Swift scientists Brad Cenko and Regina Caputo Katalyst CEO Ghonhee Lee Katalyst LINK lead Kieran Wilson   And our regular guests: Alex Zaharov-Reutt from techadvice.life Tim Mendham from Australian Skeptics  

In this episode, I discuss pitfalls of unhealthy masculinity, including a lack of self control, lack of restraint, and a lack of ability to constrain themselves. A lot of men have confused healthy masculinity with excess, gluttony, and overconsumption, being overly concerned with porn, drinking and eating in excess, following tons of titty accounts on Instagram, and other fast but empty pleasures.-Secrets of a Witch is a podcast by writer, artist, and spiritual teacher Sabrina Scott. She's been a practicing witch and medium for more than 25 years, and in this casual, mellow show she shares her secrets and musings about how to overcome pain and live a happy, magical life. She is the author of five books: Witchbody; Curse and Cure: Magic for Real Life; Rapeseed: Poetry and Writing About Life After Rape; Bodymagic: A Graphic Novel About Witchcraft, Trauma, and Healing; and A Witch at Home: 18 Rituals for Life, Love, and Healing. You can learn more about Sabrina, book a tarot reading, and learn about her courses at sabrinamscott.com, and say hi on Instagram @sabrinamscott. Email her at ceo@sabrinamscott.com

Lion’s Mane Mushroom and Brain Health: What Four Clinical Trials Actually Found Many stroke survivors and people managing cognitive decline more broadly eventually ask the same question: Is there anything beyond physiotherapy and medication that can actively support brain healing? Not symptom management. Actual repair. Lion’s Mane mushroom (Hericium erinaceus) is one compound that has gathered genuine clinical attention. It is not a cure, the human trial evidence is still limited in scale, and it is not a replacement for the fundamentals of brain health. But the mechanism is unusual, the safety profile is consistently good, and for anyone serious about their brain, the research warrants an honest look. Why Lion’s Mane Is Neurologically Unusual Most supplements that claim to support brain health cannot cross the blood-brain barrier, the tightly regulated membrane that controls what enters the brain. Without crossing it, any direct effect on brain tissue is limited. Lion’s Mane contains two families of bioactive compounds found almost nowhere else in nature. Hericenones come from the fruiting body, the visible mushroom. Erinacines come from the mycelium, the root-like underground network. Both stimulate the production of Nerve Growth Factor (NGF) and Brain-Derived Neurotrophic Factor (BDNF). These are proteins the brain uses to grow new neurons, maintain existing ones, and strengthen the connections between them. Crucially, erinacine A, one of the key mycelium compounds, has been confirmed in preclinical studies to cross the blood-brain barrier. That is not a trivial distinction. It is one of the reasons researchers have taken this mushroom seriously. “These are proteins your brain uses to grow new neurons, maintain existing ones, and build and strengthen the connections between them. They are, in a very real sense, your brain’s repair and maintenance crew.” — Bill Gasiamis What the Human Clinical Trials Found Four published human clinical trials have examined Lion’s Mane. Here is what each found: Mori et al. (2009): In a randomised, double-blind, placebo-controlled trial, 30 older adults with mild cognitive impairment (MCI) took Lion’s Mane supplement or placebo for 16 weeks. The Lion’s Mane group showed significantly better cognitive function scores at weeks 8, 12, and 16. When supplementation stopped, scores declined again within four weeks, suggesting the effect was tied to ongoing intake, not a placebo response. Saitsu et al. (2019): A multicenter RCT tested 12 weeks of oral Lion’s Mane in older adults. Participants in the treatment group showed significant improvement on the Mini-Mental State Examination (MMSE) compared to placebo. No adverse effects were observed. Nagano et al. (2010): A 4-week RCT using Lion’s Mane-enriched cookies found significant reductions in self-reported depression and anxiety in women compared to placebo, suggesting effects extend beyond cognition to mood and emotional regulation, possibly via the gut-brain axis. Docherty et al. (2023): A double-blind pilot study from Northumbria University tested 41 healthy young adults aged 18–45. After a single dose, participants performed significantly faster on the Stroop task, a measure of cognitive processing speed and flexibility. After 28 days, there was a trend toward reduced subjective stress. This was a small study, and results should be interpreted cautiously, but it suggests Lion’s Mane effects are not limited to populations already experiencing cognitive decline. The Stroke-Specific Preclinical Data For stroke survivors, the preclinical research adds another dimension. In a 2014 animal study, erinacine A reduced brain infarct volume by 22–44% in ischemic stroke models (depending on dose), and significantly lowered pro-inflammatory cytokines, including IL-1β, IL-6, and TNF-α markers of the neuroinflammatory cascade that follows stroke. A 2022 study found that erinacine A helps preserve glutamate clearance in the brain after ischemic injury. Excess glutamate is one of the key mechanisms of neuronal death after stroke, so anything that helps regulate it post-injury is clinically relevant. These are animal studies. They do not translate directly to human outcomes. But they provide a biological rationale that supports why clinical researchers are now investigating Lion’s Mane in neurological recovery contexts. What the Research Does Not Yet Tell Us The limitations matter, and any honest assessment must include them. All four human trials are relatively small, none exceeds 100 participants. We do not yet have large-scale, long-term RCTs in stroke survivor populations specifically. The optimal dose, duration, and form (fruiting body vs mycelium vs dual extract) have not been established in human trials. Direct confirmation that erinacines cross the blood-brain barrier in humans rather than in animal models does not yet exist. Bill says it directly in the video: “The human trial data is still relatively limited in scale. We need larger, longer trials.” Practical Questions to Raise with Your Doctor If you are considering Lion’s Mane supplementation, the following questions are worth raising with your neurologist or GP: Is it safe alongside my current medications? Theoretical interactions exist with anticoagulants (warfarin, aspirin, clopidogrel) and antidepressants, not confirmed in human trials, but worth disclosing. Anyone on blood thinners following a stroke should have this conversation before starting. What form should I look for? Products should specify standardised hericenone content (fruiting body extract) or erinacine A content (mycelium extract). Products listed only as “mycelial biomass on grain” typically contain very low levels of active compounds and high levels of starch from the growth substrate. If the label does not specify active compound content, treat that as a quality flag. Are there any trials I could join? ClinicalTrials.gov lists current recruiting studies for Hericium erinaceus and cognitive function worth checking if you are interested in contributing to the evidence base. More information: https://recoveryafterstroke.com/book | Support the podcast: https://patreon.com/recoveryafterstroke *This blog is for informational purposes only and does not constitute medical advice. Please consult your doctor before making any changes to your health or recovery plan. The post Can a Mushroom Help Your Brain Heal? The Science Says Maybe appeared first on Recovery After Stroke.

Excess moisture, escalating weed pressure, and delayed spray operations are emerging as the biggest challenges facing Prairie canola growers this season, according to feedback gathered through the Canola Council of Canada's Canola Watch network. Speaking with RealAgriculture at RDAR's Round-Up near Cremona, Alta., Jay Whetter, manager of knowledge and technology transfer with the Canola Council... Read More

If you've ever struggled with heavy periods, hormonal mood changes, painful boobs, feeling swollen before your period, or have been told you have “high oestrogen” without much explanation… this episode is for you.Because contrary to Instagram wellness culture, it's usually not as simple as “your body is making too much oestrogen.” Hormones are a LOT more nuanced than that — and in this episode, we're breaking down what's really going on behind symptoms like PMS, bloating, breast tenderness, headaches, mood swings, fluid retention, and feeling like your jeans suddenly shrunk overnight.We also dive into the science of hormone detoxification in a way that's practical, understandable, and actually useful — without turning this into a biochemistry lecture from hell.GET THE OESTROGEN SUPPORT RECIPE COLLECTION HEREUse the code THHP for 25% off.GET THE PROGESTERONE BLUEPRINT RECIPE COLLECTION HEREUse the code THHP for 25% off.FIND NAT BELOW:Website - https://nataliekdouglas.com/Instagram - https://www.instagram.com/natalie.k.douglasBook a Free Assessment Call - https://NatalieKDouglas.as.me/?appointmentType=50255874EndoNourish - Endometriosis and Adenomyosis Guide - https://nataliekdouglas.com/endonourish-holistic-endometriosis-adenomyoisis-care-guide/SacredSeeds - Preconception Care Guidehttps://nataliekdouglas.com/preconception-care-guide/PCOS Wellness Guidehttps://nataliekdouglas.com/pcos-holistic-guide/Thyroid Rescue - Self guided programhttps://nataliekdouglas.com/thyroid-rescue/Coming Off The Pill/IUD Holistic Guidehttps://nataliekdouglas.com/coming-off-the-pill-mini-course/PMS/PMDD Natural Solutons Masterclass https://nataliekdouglas.com/pms-pmdd-natural-solutions-masterclass/Restore and Nourish Gut Reset - https://nataliekdouglas.com/restore-nourish-gut-reset/Perimenopause Masterclass -https://nataliekdouglas.com/perimenopause-masterclass-holistic-toolkit/Become a one-to-one clienthttps://nataliekdouglas.com/1-1-naturopathic-nutrition-consultations/FIND AMIE BELOW:Book a Free Assessment Call: https://p.bttr.to/3yBdmu3   Book Yourself In: https://l.bttr.to/ZDxWOWebsite - https://whatthenaturopathsaid.com   Instagram - https://www.instagram.com/thatnaturopathJoin the mailing list - https://elysium-clinic-of-natural-medicine.ck.page/69663ce14a  PS: yes we are practitioners, but we're not YOUR practitioners - it should go without saying but our episodes are not medical advice.

Set the context for a joyful, exuberant day with a short, powerful message from Sadhguru. Explore a range of subjects with Sadhguru, discover how every aspect of life can be a stepping stone, and learn to make the most of the potential that a human being embodies.  Conscious Planet: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://www.consciousplanet.org⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Sadhguru App (Download): ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://onelink.to/sadhguru__app⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Official Sadhguru Website: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://isha.sadhguru.org⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Sadhguru Exclusive: ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠https://isha.sadhguru.org/in/en/sadhguru-exclusive⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠ Inner Engineering Link: isha.co/ieo-podcast Yogi, mystic and visionary, Sadhguru is a spiritual master with a difference. An arresting blend of profundity and pragmatism, his life and work serves as a reminder that yoga is a contemporary science, vitally relevant to our times. Learn more about your ad choices. Visit megaphone.fm/adchoices

One long trusted economic indicator may no longer reliably indicate a noninflationary environment. Confluence Chief Market Strategist Patrick Fearon-Hernandez joins Phil Adler to discuss how governments might be misled and what investors should watch for.

Burn Fat While You Sleep, Boost Metabolism & Fast the Smart Way with guest expert Dr. William Li #64What if everything you've been told about metabolism, fat, and weight loss is wrong? In this episode, Samantha sits down with Dr. William Li for a second conversation in season 2 (Check out Episode #62, if you missed it - and Season 1 Episode #14 as well!).Dr. Li dismantles everything you thought you knew about metabolism, visceral fat, and intermittent fasting — revealing the science-backed strategies that let your body burn dangerous fat while you sleep, eat, and live your life.Dr. Li is an internationally renowned physician-scientist, president of the Angiogenesis Foundation, and New York Times bestselling author of Eat to Beat Your Diet — to expose the myths that have kept so many women stuck in a cycle of dieting, frustration, and self-blame.Spoiler: your metabolism is not broken. It never was.From the newly discovered science proving that human metabolism stays rock-stable from age 20 to 60, to the hidden danger of "skinny fat" and its shocking link to breast cancer, to the brilliantly simple 12-hour fasting protocol Dr. Li himself follows every day — this episode is a masterclass in working with your body instead of against it. Plus, the true origin story of the famous 16:8 intermittent fasting method that no one is talking about.WHAT YOU'LL LEARN:Your metabolism is NOT slowing down: Learn the findings from a landmark study of 6,000 people that completely rewrites what we thought we knew about metabolism — and where the real culprit actually lives.Fat is a hormone-producing organ: Discover why healthy fat is essential to your metabolic function, and what happens to your hormones when it starts to grow out of control.Excess fat behaves like a tumor: Find out why Dr. Li says too much body fat follows the same dangerous playbook as cancer — and why that makes chronic inflammation so much more urgent to address.Skinny fat is a real and serious risk: Learn what a 13-year Cornell study found when it scanned nearly 3,500 normal-weight women — and why the results are a wake-up call for women who think they're in the clear.Foods that burn fat at the cellular level: Discover which everyday grocery store foods contain natural bioactives that work against fat growth — and how they do it without you changing everything on your plate.The tape measure test: Find out why your waistline tells a more accurate story about your health than your BMI, your scale, or your body size — and how to use it at home for free.Why 12 hours of fasting is enough: Learn the surprisingly achievable protocol Dr. Li follows himself every single day — and why the research says you don't need to go longer to see real results.The truth about 16:8: Discover the origin story of the most popular intermittent fasting method — and why its famous time window has almost nothing to do with human science.Insulin is the metabolic switch: Understand the simple biological reason your body burns fat while you sleep — and how the timing of your last meal determines how hard it works overnight.Ultra-lean is not ultra-safe: Find out why the medical literature puts extreme leanness in the same dangerous category as extreme obesity — and what that means for how we define a healthy goal.About Our GuestDr. William Li is an internationally renowned physician, scientist, researcher, and author whose work has impacted over 70 diseases, including cancer, diabetes, and cardiovascular disease. He is the founder and president of the Angiogenesis Foundation and is best known for his groundbreaking TED Talk, Can We Eat to Starve Cancer?, which has been viewed by millions worldwide. He is the New York Times bestselling author of Eat to Beat Disease and Eat to Beat Your Diet: Burn Fat, Heal Your Metabolism, and Live Longer.

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

This is a preview episode. To listen to the full episode, subscribe for as little as $5 a month at www.patreon.com/10kpostspodcast . ----more---- This week, Hussein talks to writer, author and tech critic Paris Marx about how the global shortage of hardware has led to a surge in prices of most electronic devices, and have also made them worse in quality. They also talk about how this has led to the worsening of existing internet infrastructure, especially search, where the pursuit of AI at all costs has made search engines near impossible to use, while also making tech companies more reliant on owning and storing your data.    Read and subscribe to Disconnect: https://disconnect.blog/ Listen to Tech Won't Save Us: https://techwontsave.us/ Pre Order Paris' new book, Hyperscale : The Ambition and Excess of Big Tech's Data Empires : https://bookshop.org/p/books/hyperscale-the-ambition-and-excess-of-big-tech-s-data-empires-paris-marx/bef8c026f7d016e2?ean=9798217048526&next=t&next=t&affiliate=18331   PALESTINE AID LINKS -You can donate to Medical Aid for Palestinians and other charities using the links below. https://www.map.org.uk/donate/donate https://www.savethechildren.org.uk/how-you-can-help/emergencies/gaza-israel-conflict -Palestinian Communist Youth Union, which is doing a food and water effort, and is part of the official communist party of Palestine https://www.gofundme.com/f/to-preserve-whats-left-of-humanity-global-solidarity -Water is Life, a water distribution project in North Gaza affiliated with an Indigenous American organization and the Freedom Flotilla https://www.waterislifegaza.org/ -Vegetable Distribution Fund, which secured and delivers fresh veg, affiliated with Freedom Flotilla also https://www.instagram.com/linking/fundraiser?fundraiser_id=1102739514947848 -Thamra, which distributes herb and veg seedlings, repairs and maintains water infrastructure, and distributes food made with replanted veg patches https://www.gofundme.com/f/support-thamra-cultivating-resilience-in-gaza -------- PHOEBE ALERT Okay, now that we have your attention; check out her Substack Here! Check out Masters of our Domain with Milo and Patrick, here! -------- Ten Thousand Posts is a show about how everything is posting. It's hosted by Hussein (@HKesvani), Phoebe (@PRHRoy) and produced by Devon (@Devon_onEarth).