Podcasts about variants

On Healthy Mind, Healthy Life, host Avik Chakraborty sits down with Julie Alsaker, founder of Lux Bioanalytics and holistic genetic specialist, to unpack how nutrigenomics, methylation pathways, and nervous system regulation shape clarity, energy, and resilience. We move beyond generic wellness advice to explore DNA-informed strategies for burnout, brain fog, and “doing everything right” yet still feeling off. If you've wondered why breathwork, journaling, or clean eating sometimes stall, this episode shows how aligning biology with belief and behavior can create practical, measurable change. About the guest  : Julie Alsaker is a holistic genetic specialist with 25+ years in alternative health. Through Lux Bioanalytics, she integrates DNA insights, functional blood analysis, and nutrition to help clients—from bedridden patients to high performers—identify root causes and build sustainable, personalized protocols. Key takeaways: Genetics is strategy, not destiny: DNA reveals tendencies; choices and environments shape outcomes. Nutrigenomics > one-size-fits-all: Diet, stress tools, and routines work best when calibrated to your genetic blueprint. Methylation matters: Variants across 13+ genes (e.g., MTHFR, MTR/MTRR, CBS, COMT) can limit energy access; nutrient form/amount can be the unlock. Biology sets the ceiling: Ignoring physiology (gut, immune, detox pathways) caps results from mindset and mindfulness work. Parasympathetic is where healing happens: Your body repairs when it feels safe; resilience includes nervous system retraining. Symptoms are data, not destiny: Don't normalize persistent brain fog, poor sleep, or “just getting older.” Track and investigate. Gut–brain loop: >70% of serotonin is produced in the gut; microbiome balance influences mood and motivation. Test, then tailor: DNA plus detailed bloodwork narrows root causes and informs targeted, safe interventions. Language shapes physiology: Identity and words matter—reframing fear can reduce stress chemistry and support healing. Start simple: Practice “grounded awareness”—honest symptom logging—before adding protocols or supplements. Connect with the guest  : Follow Julie on Facebook: https://www.facebook.com/julie.alsaker Website: https://www.luxbioanalytics.com/   Notes: The episode discusses genetics, nutrition, supplements, and mindset in general, educational terms without making dangerous claims or instructing self-harm. Listeners should consult qualified professionals for personal medical decisions. Want to be a guest on Healthy Mind, Healthy Life? DM on PM - Send me a message on PodMatch DM Me Here: https://www.podmatch.com/hostdetailpreview/avik Disclaimer: This video is for educational and informational purposes only. The views expressed are the personal opinions of the guest and do not reflect the views of the host or Healthy Mind By Avik™️. We do not intend to harm, defame, or discredit any person, organization, brand, product, country, or profession mentioned. All third-party media used remain the property of their respective owners and are used under fair use for informational purposes. By watching, you acknowledge and accept this disclaimer. Healthy Mind By Avik™️ is a global platform redefining mental health as a necessity, not a luxury. Born during the pandemic, it's become a sanctuary for healing, growth, and mindful living. Hosted by Avik Chakraborty—storyteller, survivor, wellness advocate—this channel shares powerful podcasts and soul-nurturing conversations on: • Mental Health & Emotional Well-being• Mindfulness & Spiritual Growth• Holistic Healing & Conscious Living• Trauma Recovery & Self-Empowerment With over 4,400+ episodes and 168.4K+ global listeners, join us as we unite voices, break stigma, and build a world where every story matters.

Each of the Variants brings a handful of Letterboxd reviews to the table and has the other 2 try to guess what movie the review is from. Trust Us, You'll Like It.Please leave us a 5 star review on your podcast platform, drop us a follow on the socials, consider joining the Patreon, & share with your friends & family! Thank you for listening & thank you for all the support! Links for all of our stuff can be found here: https://linktr.ee/VariantVendettaYou can find our good friend's Podcasts here:https://www.abingerspodcast.com/https://linktr.ee/anyonescomichttps://linktr.ee/goingmerrypod

The Variants are back with King taking another trip into the Twilight Zone with Season 1 Episodes 4, 5, and 6!Please leave us a 5 star review on your podcast platform, drop us a follow on the socials, consider joining the Patreon, & share with your friends & family! Thank you for listening & thank you for all the support! Links for all of our stuff can be found here: https://linktr.ee/VariantVendettaYou can find our good friend's Podcasts here:https://www.abingerspodcast.com/https://linktr.ee/anyonescomichttps://linktr.ee/goingmerrypod

By contrast, varied doses of the same genes decrease or increase the odds of five other conditions, with distinct biological consequences, two new preprints show.

In this episode I discuss the following: ⭐️ DJ Spin .. I talk about the “too many cards” controversy and why I don't agree .. I also touch on the cheater bots and why I think they need to be toned down especially in certain modes ⭐️ The Request Line has answers from YOU to a question on X⭐️ SNAP talk .. where I finished on ladder .. decks I'm playing to start the new season. Variants / albums and bundles , conquest & more ⭐️ Comics Corner with Viv & Sparky .. origins / appearances and where you can read about them ⭐️ MCU minute with Marvel Zombies (Blade) Tom Holland on his future playing Spider-Man and the Doom / Peggy Carter / Cap rumors for Doomsday ⭐️ Dazzlers Guess the Lyric .. win a NO Prize ! (EZ this week) Thanks for listening!

Episode Overview In this episode of The Design Vault, hosts Albert Shum and Thamer Abanami explore the extraordinary story of the Citroën DS, arguably the most audacious automobile ever created. Born from the devastation of post-WWII France, this revolutionary car emerged from an 18-year development odyssey that challenged every automotive convention. With insights from retired Apple and Motorola design leader Tim Parsey, who owned multiple DS models, this episode reveals how a dream team of engineers and designers created a vehicle so advanced it seemed to come from the future. From its magical hydropneumatic suspension to its aerodynamic sculpture-on-wheels aesthetic, the DS completely reimagined what a car could be. Original Air Date: August 26, 2025 Episode Length: 38:31 Hosts: Albert Shum, Thamer Abanami Guest: Tim Parsey (Former Apple, Motorola, Mattel Design Leader)   Key Segments & Timestamps The Context: Post-War France's Design Challenge (00:20 - 03:58) Post-WWII devastation creating space for radical innovation Rough roads, high fuel taxes, and the culture of efficiency Charles de Gaulle's “grandeur” vision driving technological ambition How constraints became catalysts for breakthrough thinking The Automotive Landscape: A World Ripe for Disruption (03:58 - 06:10) American excess era: 42-inch tail fins and chrome measured by weight Germany's people's car philosophy with the Beetle Britain maintaining pre-war conservatism France's strategy to leapfrog rather than catch up Citroën's Culture of Radical Innovation (06:10 - 08:45) André Citroën's front-wheel-drive gamble with the Traction Avant The critical 1934 bankruptcy and Michelin's revolutionary takeover Pierre Boulanger's radical decision: “Keep engineers, fire accountants” The 2CV's parallel development funding DS ambitions The Dream Team (08:45 - 11:50) André Lefebvre: Aeronautical engineer with a backlog of innovations Paul Magès: Self-taught genius behind hydropneumatic suspension Flaminio Bertoni: Italian sculptor turned automotive stylist Why letting creative minds loose is “highly risky but necessary” The 18-Year Development Odyssey (11:50 - 16:21) Simple question: Why improve roads when you can improve cars? Secret development during WWII The hydropneumatic breakthrough: Gas compresses, liquid transmits Systems integration: One technology powering suspension, brakes, steering 40% of build cost invested in hydraulic complexity The Theatrical Launch: Paris 1955 (17:16 - 20:03) Grand Palais transformed into theater The silk sheet drops, crowds gasp 12,000 pre orders—a record until Tesla Model 3 The strategic 500-customer beta program with dedicated engineers Living with Revolutionary Complexity (20:03 - 23:05) The infamous “mushroom brake” and its quirks Tim's near-death experience  “Marking territory with hydraulic fluid” Why the experience had to be driven to be understood The Meditative Magic: What Made DS Special (23:05 - 27:03) “Like gliding around… a meditative experience” Magic carpet ride over speed bumps Why no other manufacturers copied the formula Engineering complexity as competitive moat Evolution and Variants (27:03 - 28:55) From “frog eyes” to swiveling directional headlights (1967) Power progression: DS 19, DS 21, DS 23 Safari wagons, Pallas luxury, SM with Maserati power “Frogs have personality. Fairings don't.” Design Philosophy: Engineering as Art (28:55 - 32:39) Perfect tension between engineering and sculptural beauty Authentic aerodynamics vs. American “rocket ship” styling Three-dimensional airflow management with under-car panels Flush door handles decades before Tesla Interior as Living Room (32:39 - 35:20) Four interior lights creating ambient atmosphere Bench seats and column-mounted gear shifter maximizing space Single-spoke steering wheel for unobstructed view Dashboard-mounted mirror at natural eye level Personal Connection: Tim's First DS Story (35:20 - 38:27) £30 for two broken cars to make one working DS Brilliant engineering: body panels removable with single bolts Digging holes in frozen ground to replace hydraulic lines The devotion that revolutionary design inspires Legacy and Lessons for Modern Innovators (38:27 - 38:31) Showing possibilities people never imagined The courage to exist “outside of time” Why serving people sometimes means ignoring market research Dream teams without financial constraints    Connect With The Design Vault The Design Vault explores iconic products from the innovation-rich 1970s-early 2000s, extracting strategic insights for today's designers, engineers, and business leaders. Each episode combines nostalgic storytelling with actionable lessons for modern product development. Subscribe: Available on all major podcast platforms including Spotify, Apple, and more Follow us: Instagram: @thedesignvaultpodcast, LinkedIn: Thamer Abanami, Albert Shum We'd love to hear your thoughts, episode ideas and feedback via the links above.   Credits Hosts: Albert Shum and Thamer Abanami Guest: Tim Parsey Editor: Rachel James Intro Music: Red Lips Media LLC Brand Design: Rafael Poloni​​​​​​​​​​​​​​​​

With this episode, it goes over some variants to bridge so that people can engage in alternate means of playing the game.  So with that said, we hope you enjoy. CreditsWriter - Bradley P. ThomasProducer - Bradley P. ThomasVoice Talent – ElevenLabs: TaylorEditor - Bradley P. Thomas https://linktr.ee/Check_Point_Gaming Copyright Disclaimer: Copyright Disclaimer under section 107 of the Copyright Act 1976, allowance is made for “fair use” for purposes such as criticism, comment, news reporting, teaching, scholarship, education, and research.  Fair use is a use permitted by copyright statute that might otherwise be infringing.  Non-profit, educational, or personal use tips the balance in favor of fair use.https://www.copyright.gov/legislation/dmca.pdf

AI and genetic medicine are converging to transform how we diagnose, treat, and prevent disease. Gene Yeo, Ph.D., unites RNA biology with artificial intelligence to speed the path from genome sequencing to personalized RNA therapeutics. Advances in sequencing have reduced costs dramatically, making interpretation and translation into treatments the real challenge. Using deep learning and large datasets of RNA-binding proteins, Yeo predicts disease vulnerabilities and identifies therapeutic targets, including in neurodegeneration and muscular diseases. Alexis Komor, Ph.D., focuses on DNA, explaining human genetic variation—particularly single-nucleotide variants—and how genome editing technologies like CRISPR can target them. She highlights strategies to correct harmful mutations and explores precise, programmable interventions. Together, their research drives discovery and enables more effective, personalized therapies. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40459]

AI and genetic medicine are converging to transform how we diagnose, treat, and prevent disease. Gene Yeo, Ph.D., unites RNA biology with artificial intelligence to speed the path from genome sequencing to personalized RNA therapeutics. Advances in sequencing have reduced costs dramatically, making interpretation and translation into treatments the real challenge. Using deep learning and large datasets of RNA-binding proteins, Yeo predicts disease vulnerabilities and identifies therapeutic targets, including in neurodegeneration and muscular diseases. Alexis Komor, Ph.D., focuses on DNA, explaining human genetic variation—particularly single-nucleotide variants—and how genome editing technologies like CRISPR can target them. She highlights strategies to correct harmful mutations and explores precise, programmable interventions. Together, their research drives discovery and enables more effective, personalized therapies. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40459]

AI and genetic medicine are converging to transform how we diagnose, treat, and prevent disease. Gene Yeo, Ph.D., unites RNA biology with artificial intelligence to speed the path from genome sequencing to personalized RNA therapeutics. Advances in sequencing have reduced costs dramatically, making interpretation and translation into treatments the real challenge. Using deep learning and large datasets of RNA-binding proteins, Yeo predicts disease vulnerabilities and identifies therapeutic targets, including in neurodegeneration and muscular diseases. Alexis Komor, Ph.D., focuses on DNA, explaining human genetic variation—particularly single-nucleotide variants—and how genome editing technologies like CRISPR can target them. She highlights strategies to correct harmful mutations and explores precise, programmable interventions. Together, their research drives discovery and enables more effective, personalized therapies. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40459]

AI and genetic medicine are converging to transform how we diagnose, treat, and prevent disease. Gene Yeo, Ph.D., unites RNA biology with artificial intelligence to speed the path from genome sequencing to personalized RNA therapeutics. Advances in sequencing have reduced costs dramatically, making interpretation and translation into treatments the real challenge. Using deep learning and large datasets of RNA-binding proteins, Yeo predicts disease vulnerabilities and identifies therapeutic targets, including in neurodegeneration and muscular diseases. Alexis Komor, Ph.D., focuses on DNA, explaining human genetic variation—particularly single-nucleotide variants—and how genome editing technologies like CRISPR can target them. She highlights strategies to correct harmful mutations and explores precise, programmable interventions. Together, their research drives discovery and enables more effective, personalized therapies. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40459]

AI and genetic medicine are converging to transform how we diagnose, treat, and prevent disease. Gene Yeo, Ph.D., unites RNA biology with artificial intelligence to speed the path from genome sequencing to personalized RNA therapeutics. Advances in sequencing have reduced costs dramatically, making interpretation and translation into treatments the real challenge. Using deep learning and large datasets of RNA-binding proteins, Yeo predicts disease vulnerabilities and identifies therapeutic targets, including in neurodegeneration and muscular diseases. Alexis Komor, Ph.D., focuses on DNA, explaining human genetic variation—particularly single-nucleotide variants—and how genome editing technologies like CRISPR can target them. She highlights strategies to correct harmful mutations and explores precise, programmable interventions. Together, their research drives discovery and enables more effective, personalized therapies. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40459]

AI and genetic medicine are converging to transform how we diagnose, treat, and prevent disease. Gene Yeo, Ph.D., unites RNA biology with artificial intelligence to speed the path from genome sequencing to personalized RNA therapeutics. Advances in sequencing have reduced costs dramatically, making interpretation and translation into treatments the real challenge. Using deep learning and large datasets of RNA-binding proteins, Yeo predicts disease vulnerabilities and identifies therapeutic targets, including in neurodegeneration and muscular diseases. Alexis Komor, Ph.D., focuses on DNA, explaining human genetic variation—particularly single-nucleotide variants—and how genome editing technologies like CRISPR can target them. She highlights strategies to correct harmful mutations and explores precise, programmable interventions. Together, their research drives discovery and enables more effective, personalized therapies. Series: "Stem Cell Channel" [Health and Medicine] [Science] [Show ID: 40459]

Send comments and feedbackWhat are variants of uncertain significance (VUS) in epilepsy genetic testing? How are they defined, and can those definitions change over time? Sharp Waves talks with Dr. Gemma Carvill as part of our genetic testing series.RESOURCESStandards for interpreting variants (American College of Medical Genetics)Gene ReviewsClinVarGeneMatcherGnomadClinGenILAE genetic literacy seriesEpiPred website for STXBP1 (developed by EpiMVP project - will be final by end of July)Think Genetics paper – genetic testing in South Africa projectLink to Sharp Waves episode on genetic testing in LMICs  Sharp Waves episodes are meant for informational purposes only, and not as clinical or medical advice.Let us know how we're doing: podcast@ilae.org.The International League Against Epilepsy is the world's preeminent association of health professionals and scientists, working toward a world where no person's life is limited by epilepsy. Visit us on Facebook, Instagram, and LinkedIn.

Surfing, skating, & Heath Ledger.. What more could you ask for? Join the Variants this week as they shred through the true beginning of skateboarding history! Please leave us a 5 star review on your podcast platform, drop us a follow on the socials, consider joining the Patreon, & share with your friends & family! Thank you for listening & thank you for all the support! Links for all of our stuff can be found here: https://linktr.ee/VariantVendettaYou can find our good friend's Podcasts here:https://www.abingerspodcast.com/https://linktr.ee/anyonescomichttps://linktr.ee/goingmerrypod

Senator Ron Johnson, The Blaze's Joe Hanneman, WMC's Scott Manley, Hometown Pharmacy in Wisconsin's Dan Strauss, Developer Terrence Wall We are replaying Sen. Ron Johnson

Marlon Hue is a standup comedian & producer based in Chicago!

For review:1.Arab League Condemns 7 October Attack; Calls for Hamas to Disarm. Arab and Muslim countries, including Saudi Arabia, Qatar, Egypt, Jordan and Turkey, signed a declaration (New York Declaration) Tuesday condemning for the first time Hamas's onslaught of October 7, 2023, and calling on the Palestinian terror group to release all the hostages it is holding, disarm and end its rule of Gaza.2. Givati Infantry Brigade combat in Northeast Gaza town of Beit Hanoun. 3. US Secretary of State Marco Rubio revealed in an interview with Fox Radio on Thursday that the White House is nearing a critical decision point regarding the ongoing conflict in Ukraine.4. India Rejects F-35 Purchase.5. The first of Japan's Short Take Off and Vertical Landing (STOVL) F-35B Lightning II fifth-generation fighters will arrive from the United States in early August, according to the country's defense ministry.6. A US Navy pilot is “safe” after their F-35C crashed near Naval Air Station Lemoore on Wednesday evening, according to a Navy spokesperson.7. AM General has received a $126.5-million contract to deliver additional M1165A1B3 Humvees, for the US Army. The Pentagon did not disclose the quantity of the latest order.Work on the program will be carried out in South Bend, Indiana, with completion expected by August 2027.8. Senate Appropriators Pass Committee Defense Spending Bill.Senate appropriators passed their $852.5 billion defense spending bill for fiscal 2026 today, adding $21.7 billion to the Pentagon's topline. 

This week on the Toy Power Podcast; we are still down a Scot, as he is still unwell. But that will not slow him down in contributing to this ep! Before we get into the main segments, we chat about Ben & Frank's latest Plastic Addiction, in the form of Transformers Blokees! Prior to chatting about the Superman Film itself, we focus on the Toy likeness compared to what the actor actually looks like! With a little Jurassic World banter in between. Then, without another moment to spare, we get right into the James Gunn driven film itself! We chat towards our Cinema experiences and start off with a non-spoiler discussion; before we head into a chat about the whole Film itself! Giving it our typical DVG rating out of 19 too. Then changing gears, we introduce a New segment - named: Variant Schmariant! We each pick two variants of a pre-existing character (in toy form) that just doesn't need to exist. We unearth some quite silly versions of characters that had a legitimate Toy Release; that clearly logic was not a concern at all!! Plus we tie a nice bow around the proposed Nick-Names for both Frank & Scot. Enjoy!!Support the show: http://patreon.com/toypowerpodcastSee omnystudio.com/listener for privacy information.

2025年2月には、100万ダウンロード突破しています。

Variants in genes tied to both conditions derail the formation of cilia, the tiny hair-like structure found on almost every cell in the body, a new study finds.

For decades, Alejandro Portes (CASBS fellow 1980-81) has been among our most distinguished scholars elucidating the causes and consequences of immigration and assimilation. René D. Flores (CASBS fellow 2023-24) engages Portes in a conversation spanning large swaths of Portes's formidable intellectual biography, including his personal journey from Cuba and its influence on his academic trajectory, as well as his approach to social science inquiry and its delivery of insights leading to some of his most celebrated and consequential works.ALEJANDRO PORTES: Princeton faculty page | CV | Univ. of Miami faculty page | Wikipedia page | on Google Scholar | Biographical sketches of Portes: American Sociological Association | National Academy of Education | Princeton | National Institutes of Health | Proceedings of the National Academy of Sciences | Robert K. Merton's full quote about Alejandro Portes is contained in the September/October 1998 issue of Footnotes, a publication of the American Sociological Association. Access the full text.  Works referenced in this episodeAlejandro Portes, "Rationality in the Slum: An Essay on Interpretive Sociology," Comparative Studies in Society and History, v13 n3, June 1972.Alejandro Portes, "Dilemmas of a Golden Exile: Integration of Cuban Refugee Families in Milwaukee," American Sociological Review, v34 n4, August 1969.Alejandro Portes and Robert Bach, Latin Journey: Cuban and Mexican Immigrants in the United States. Univ. of California Press, 1985.Alejandro Portes and Min Zhou, "The New Second Generation: Segmented Assimilation and its Variants," Annals of the American Academy of Political and Social Science, November 1993.Alejandro Portes and Ruben Rumbaut, Immigrant America: A Portrait, Univ. of California Press, 2024 (fifth ed.)Alejandro Portes and Ruben Rumbaut, Legacies: The Story of the Immigrant Second Generation, Univ. of California Press, 2001.Jennifer Lee and Min Zhou, The Asian American Achievement Paradox, Russell Sage Foundation, 2015. (Notably, Jennifer Lee was a CASBS fellow in 2002-03; Min Zhou was a CASBS fellow in 2006-06.)Bonus: 2019-20 CASBS fellow Catherine Ramírez discusses the influence of Alejandro Portes in "What Does Assimilation Mean?" Public Books, Feb. 27, 2020. The essay was written as part of CASBS's partnership with Public Books. Ramírez writes, "By showing that there are many strata in society into which people assimilate, and many outcomes of assimilation, Portes and his coauthors have enriched our understanding of the processes by which people become American, however precarious that status may be.” René D. Flores: Univ. of Chicago faculty page | CASBS page | on Google Scholar | Personal website |    Center for Advanced Study in the Behavioral Sciences (CASBS) at Stanford UniversityExplore CASBS: website|Bluesky|X|YouTube|LinkedIn|podcast|latest newsletter|signup|outreach​Human CenteredProducer: Mike Gaetani | Audio engineer & co-producer: Joe Monzel |

In the Electrek Podcast, we discuss the most popular news in the world of sustainable transport and energy. In this week's episode, we discuss Tesla announcing new Model 3 and Model Y variants, the robotaxi expansion wars, big Lucid news, and more The show is live every Friday at 4 p.m. ET on Electrek's YouTube channel. As a reminder, we'll have an accompanying post, like this one, on the site with an embedded link to the live stream. Head to the YouTube channel to get your questions and comments in. After the show ends at around 5 p.m. ET, the video will be archived on YouTube and the audio on all your favorite podcast apps: Apple Podcasts Spotify Overcast Pocket Casts Castro RSS We now have a Patreon if you want to help us avoid more ads and invest more in our content. We have some awesome gifts for our Patreons and more coming. Here are a few of the articles that we will discuss during the podcast: Tesla announces Model YL, a larger 6-seater SUV coming this fall Tesla prepares to launch Model 3+ with 800 km (500 mile) range Tesla launches a long list of new discounts and incentives in the US Tesla's long-time head of sales in North America is out Tesla's penis-shaped Robotaxi expansion illustrates how unserious the business is Waymo outlengths Tesla: Elon's phallic Robotaxi map backfires in Austin's expansion battle Uber to deploy 20,000 Lucid Gravity robotaxis equipped with Nuro Driver, beginning next year [Video] Mercedes-Benz unveils the new CLA Shooting Brake EV with impressive range Subaru enters Uncharted EV territory Faraday Future unveils FX Super One, an EV minivan with a funny F.A.C.E. Here's the live stream for today's episode starting at 4:00 p.m. ET (or the video after 5 p.m. ET: https://www.youtube.com/live/6AoMC46FO04

In this episode of The BlueHat Podcast, host Nic Fillingham is joined by George Hughey from Microsoft who returns to discuss his Blue Hat India talk on variant hunting, explaining how MSRC uses submission data from hacking competitions like Pwn2Own and Tianfu Cup to uncover additional security vulnerabilities in Windows. George shares how incentives in competitions differ from bug bounty programs, how tools like CodeQL assist variant hunting, and why collaborating with the security research community is key to improving Windows security.    In This Episode You Will Learn:   How hacking competitions help find real-world Windows vulnerabilities  The role of MSRC in hunting variants beyond submitted vulnerabilities  Why fuzzing is not always effective for modern edge cases    Some Questions We Ask:  How do you decide which cases to pursue for variant hunting?  What advice do you have for researchers submitting variants?  How does the CodeQL team collaborate with your team?      Resources:       View George Hughey on LinkedIn      View Wendy Zenone on LinkedIn    View Nic Fillingham on LinkedIn     Related Microsoft Podcasts:    Microsoft Threat Intelligence Podcast    Afternoon Cyber Tea with Ann Johnson    Uncovering Hidden Risks       Discover and follow other Microsoft podcasts at microsoft.com/podcasts      The BlueHat Podcast is produced by Microsoft and distributed as part of N2K media network.  

Commentary by Dr. Jian'an Wang.

In this episode of Talking Drupal, we dive into the world of Drupal user groups and meetups with guests Lee Walker, Bernardo Martinez, and Bo Shipley. Our guests share their experiences in organizing and participating in Drupal communities and the vital role these meetups play in fostering continuous learning and professional development. We also explore the newest features of Drupal Core 11.2 in the Module of the Week. For show notes visit: https://www.talkingDrupal.com/508 Topics Meet the Guests: Lee, Bo, and Bernardo Module of the Week: Drupal Core 11.2 Diving into Drupal User Groups and Meetups Personal Journeys into Drupal User Groups The Role of Meetup.com in Drupal Communities Organizing and Attending Meetups vs. Conferences Challenges and Strategies for Growing Meetups Virtual and Hybrid Meetups: Impact on Attendance Success Tips for Organizing Meetups Keeping Meetups Simple and Engaging Preventing Organizer Burnout Challenges and Changes in Meetup Cadence Finding and Retaining Meetup Members Communication Tools for Meetup Groups The Importance of In-Person Meetups Advice for Starting or Restarting Meetups Conclusion and Contact Information Resources Drupal.org Events The Drop Times Events Meetup.com Drupal Chattanooga Drupal Users Group Chattanooga Drupal Camp Guests Lee Walker - www.codejourneymen.com mr_scumbag Bo Shipley - simplyshipley Bernardo Martinez - linkedin bernardm28 Hosts Stephen Cross - stephencross John Picozzi - epam.com johnpicozzi JD Leonard - modernbizconsulting.com jdleonard Module of the Week with Martin Anderson-Clutz - mandclu.com mandclu Drupal Core 11.2 Single Directory Components (SDCs) have been a focus of excitement for Drupal's front end developers since they were added to Drupal 10.1 as an experimental module, and merged into 10.3 as a stable feature. With Drupal 11.2, SDCs now have a concept of variants, to allow for different ways of presenting a component's information. Some component frameworks like Storybook have a somewhat different concept of variants, which is really a set of property value presets that are useful for testing. Variants with Drupal SDCs strike me as being analogous to view modes for content types, in that you can have separate template files for each variant, or you can have conditional logic within a single template based on the variant in use. Our own nicxvan, chx, and some others put some significant work into allowing preprocess hooks to be defined as OOP classes, which bring us a significant step closer to not needing .module files anymore. Hooks (and .module files) are Drupalisms, so removing the need for them is a big improvement for Developer Experience, and makes it easier for developers to get started with Drupal In Drupal 11.2 the module installer has been updated to only rebuild the container after several modules have been installed, which significantly speeds up installing multiple modules at once. Drupal 11.2 also brings us a Recipe Unpack composer extension, so when you composer require a recipe, the dependencies get automatically added to your site's composer.json file, so you can apply and then remove the recipe and still have a fully functional site Package Manager is now a hidden module in Drupal core, which is critical for initiative like Automatic Updates and Project Browser, that the community has been working on for years Drupal core now also supports the next-generation AVIF format, with WEBP as a fallback with servers that don't support generating them Of course there are also a variety of dependency updates as well, for CKEDitor, Symfony, composer and more, as well as too many minor improvements and bugfixes to cover in detail here

BUFFALO, NY — June 12, 2025 — A new #research paper was #published in Aging (Aging-US) Volume 17, Issue 5, on May 3, 2025, titled “APOE genotype and biological age impact inter-omic associations related to bioenergetics.” In this study, led by first author Dylan Ellis and corresponding author Noa Rappaport from the Institute for Systems Biology, researchers discovered that different versions of the APOE gene—particularly ε2 and ε4—are linked to metabolic patterns associated with aging and Alzheimer's disease risk. Both variants were linked to increased levels of diacylglycerols, a type of fat molecule connected to insulin resistance and inflammation, suggesting shared disruptions in how the body regulates energy. The research team analyzed data from over 2,200 adults without an Alzheimer's diagnosis, exploring how APOE genotypes influence biological age, a measure of health that reflects how quickly or slowly someone is aging at a cellular level. They found that the same metabolic disturbances seen in ε2 carriers were also present in people considered biologically older, revealing unexpected overlap between genetic risk and aging-related metabolic changes. To examine these connections in more detail, the researchers used a multi-omics approach, combining blood-based metabolism and protein data, gut bacteria analysis from stool samples, and clinical chemistry data. This method allowed them to map how genetic differences and biological aging affect the body's energy systems. They observed altered connections between glucose metabolism, inflammatory markers, and key molecules that play roles in energy production, indicating early disruptions that could contribute to age-related diseases. One of the study's surprising findings was that the ε2 variant, usually associated with longer life and reduced Alzheimer's risk, showed metabolic traits similar to those found in insulin-resistant individuals. This suggests that ε2 may carry metabolic disadvantages earlier in life, with its protective effects becoming more pronounced later. Conversely, ε4—linked to greater Alzheimer's risk—may exert its influence based on interactions with lifestyle factors like diet, sex, and overall health status. “‘Omics association patterns of ε2-carriers and increased biological age were also counter-intuitively similar, displaying significantly increased associations between insulin resistance markers and energy-generating pathway metabolites.” By identifying these shared biological signatures, this study offers a new framework for understanding how genes and metabolism work together to influence aging. These findings could support more personalized health strategies aimed at delaying biological aging and reducing the risk of chronic diseases. As aging populations grow worldwide, understanding these pathways is essential to improving healthspan. DOI - https://doi.org/10.18632/aging.206243 Corresponding author - Noa Rappaport - noa.rappaport@isbscience.org Video short - https://www.youtube.com/watch?v=75hZQoO5U0U Sign up for free Altmetric alerts about this article - https://aging.altmetric.com/details/email_updates?id=10.18632%2Faging.206243 Subscribe for free publication alerts from Aging - https://www.aging-us.com/subscribe-to-toc-alerts Keywords - aging, apolipoprotein E (APOE), biological age, metabolism, Alzheimer's disease (AD), insulin resistance To learn more about the journal, please visit our website at https://www.Aging-US.com​​ and connect with us on social media at: Facebook - https://www.facebook.com/AgingUS/ X - https://twitter.com/AgingJrnl Instagram - https://www.instagram.com/agingjrnl/ YouTube - https://www.youtube.com/@AgingJournal LinkedIn - https://www.linkedin.com/company/aging/ Bluesky - https://bsky.app/profile/aging-us.bsky.social Pinterest - https://www.pinterest.com/AgingUS/ Spotify - https://open.spotify.com/show/1X4HQQgegjReaf6Mozn6Mc MEDIA@IMPACTJOURNALS.COM

The Ars Magica roleplaying game is currently complete, but the community is still mining great gaming material out of medieval history as part of a Share Alike License. The Games From Folktales podcast gives you new, odd, useful material each week, for roleplaying games set in European-styled fantasy settings.   Mythic Europe Magazine is now available from Drivethru, Patreon and itch.io. https://www.patreon.com/c/timothyferguson/shop https://timothy-ferguson.itch.io/mythic-europe-magazine-issue-one https://www.drivethrurpg.com/en/product/520248/Mythic-Europe-Magazine-Issue-One?affiliate_id=1776916

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Explains language models (LLMs) advancements. Scaling laws - the relationships among model size, data size, and compute - and how emergent abilities such as in-context learning, multi-step reasoning, and instruction following arise once certain scaling thresholds are crossed. The evolution of the transformer architecture with Mixture of Experts (MoE), describes the three-phase training process culminating in Reinforcement Learning from Human Feedback (RLHF) for model alignment, and explores advanced reasoning techniques such as chain-of-thought prompting which significantly improve complex task performance. Links Notes and resources at ocdevel.com/mlg/mlg34 Build the future of multi-agent software with AGNTCY Try a walking desk stay healthy & sharp while you learn & code Transformer Foundations and Scaling Laws Transformers: Introduced by the 2017 "Attention is All You Need" paper, transformers allow for parallel training and inference of sequences using self-attention, in contrast to the sequential nature of RNNs. Scaling Laws: Empirical research revealed that LLM performance improves predictably as model size (parameters), data size (training tokens), and compute are increased together, with diminishing returns if only one variable is scaled disproportionately. The "Chinchilla scaling law" (DeepMind, 2022) established the optimal model/data/compute ratio for efficient model performance: earlier large models like GPT-3 were undertrained relative to their size, whereas right-sized models with more training data (e.g., Chinchilla, LLaMA series) proved more compute and inference efficient. Emergent Abilities in LLMs Emergence: When trained beyond a certain scale, LLMs display abilities not present in smaller models, including: In-Context Learning (ICL): Performing new tasks based solely on prompt examples at inference time. Instruction Following: Executing natural language tasks not seen during training. Multi-Step Reasoning & Chain of Thought (CoT): Solving arithmetic, logic, or symbolic reasoning by generating intermediate reasoning steps. Discontinuity & Debate: These abilities appear abruptly in larger models, though recent research suggests that this could result from non-linearities in evaluation metrics rather than innate model properties. Architectural Evolutions: Mixture of Experts (MoE) MoE Layers: Modern LLMs often replace standard feed-forward layers with MoE structures. Composed of many independent "expert" networks specializing in different subdomains or latent structures. A gating network routes tokens to the most relevant experts per input, activating only a subset of parameters—this is called "sparse activation." Enables much larger overall models without proportional increases in compute per inference, but requires the entire model in memory and introduces new challenges like load balancing and communication overhead. Specialization & Efficiency: Experts learn different data/knowledge types, boosting model specialization and throughput, though care is needed to avoid overfitting and underutilization of specialists. The Three-Phase Training Process 1. Unsupervised Pre-Training: Next-token prediction on massive datasets—builds a foundation model capturing general language patterns. 2. Supervised Fine Tuning (SFT): Training on labeled prompt-response pairs to teach the model how to perform specific tasks (e.g., question answering, summarization, code generation). Overfitting and "catastrophic forgetting" are risks if not carefully managed. 3. Reinforcement Learning from Human Feedback (RLHF): Collects human preference data by generating multiple responses to prompts and then having annotators rank them. Builds a reward model (often PPO) based on these rankings, then updates the LLM to maximize alignment with human preferences (helpfulness, harmlessness, truthfulness). Introduces complexity and risk of reward hacking (specification gaming), where the model may exploit the reward system in unanticipated ways. Advanced Reasoning Techniques Prompt Engineering: The art/science of crafting prompts that elicit better model responses, shown to dramatically affect model output quality. Chain of Thought (CoT) Prompting: Guides models to elaborate step-by-step reasoning before arriving at final answers—demonstrably improves results on complex tasks. Variants include zero-shot CoT ("let's think step by step"), few-shot CoT with worked examples, self-consistency (voting among multiple reasoning chains), and Tree of Thought (explores multiple reasoning branches in parallel). Automated Reasoning Optimization: Frontier models selectively apply these advanced reasoning techniques, balancing compute costs with gains in accuracy and transparency. Optimization for Training and Inference Tradeoffs: The optimal balance between model size, data, and compute is determined not only for pretraining but also for inference efficiency, as lifetime inference costs may exceed initial training costs. Current Trends: Efficient scaling, model specialization (MoE), careful fine-tuning, RLHF alignment, and automated reasoning techniques define state-of-the-art LLM development.

Interview with Leonoor F. J. M. Wijdeveld, BSc and Sean J. Jurgens, MD, MSc, PhD, authors of Cardiomyopathy-Associated Gene Variants in Atrial Fibrillation. Hosted by Sharlene Day, MD. Related Content: Cardiomyopathy-Associated Gene Variants in Atrial Fibrillation Genetic Testing in Early-Onset Atrial Fibrillation

Interview with Leonoor F. J. M. Wijdeveld, BSc and Sean J. Jurgens, MD, MSc, PhD, authors of Cardiomyopathy-Associated Gene Variants in Atrial Fibrillation. Hosted by Sharlene Day, MD. Related Content: Cardiomyopathy-Associated Gene Variants in Atrial Fibrillation Genetic Testing in Early-Onset Atrial Fibrillation

The scientific method involves observation, questioning, forming hypotheses, testing predictions and altering theories to align with results; it is not the altering of results to align with a hypothesis.  There are three acceptable narratives about COVID-19: a wet mart (official), 5G (conspiracy), and laboratory leak (alternative). Whereas the https://www.science.org/content/article/cia-bribed-its-own-covid-19-origin-team-reject-lab-leak-theory-anonymous-whistleblower to reject the lab theory, https://apnews.com/article/covid-cia-trump-china-pandemic-lab-leak-9ab7e84c626fed68ca13c8d2e453dde1 explanation. This announcement was made just days after the former President regained the White House. As of April 2025, the https://www.whitehouse.gov/lab-leak-true-origins-of-covid-19/?fbclid=IwY2xjawJv5wdleHRuA2FlbQIxMAABHrhaCm1LYQx2UbG8uGLw5gkhCvB3N4a2gNrAgdarT7Z6C-XKZijSXHb3PctU_aem_hUJWc6XJ_Gfj14AvDa1VSA as “the true origins of COVID-19.”  This new official designation means that at one time or another two totally different explanations were given, ultimately with the consequence of censorship and ridicule if a person thought or said anything different. Both explanations still result in justufciaotn for past, and future, measures such as: social distancing, masking, https://www.theatlantic.com/ideas/archive/2021/04/end-hygiene-theater/618576/ as a form of a theater, vaccines, etc.  Prior to the recent shift in narratives, undercover video reportedly proved that Pfizer was indeed conducing gain of function research in a laboratory. But that lab was not a viral facility or a Wuhan institute; instead, it was a computer lab. In fact, https://www.pfizer.com/news/announcements/pfizer-responds-research-claims stating: “With a naturally evolving virus, it is important to routinely assess the activity of an antiviral. Most of this work is conducted using computer simulations…” Such computer simulations were used to predict mass casualties from COVID, too, and are the same ones being employed for Climate Change narratives. But what is COVID-19 or the virus designated SARS-COV-2. It is a list or complex of symptoms that are classified into categories of disease. Examination of COVID's symptoms prove they are nearly identical to the common cold and flu, among others. In fact, the https://hsph.harvard.edu/news/a-sharp-drop-in-flu-cases-during-covid-19-pandemic/. According to Harvard, this was the result of “wearing masks and distancing,” though they did not explain how such measures stopped the flu but not SARS-COV-2. Consider these three sets of symptoms from the CDC website: https://www.cdc.gov/common-cold/about/index.html: runny nose or nasal congestion, cough, sneezing, sore throat, headache, mild body aches, fever.https://www.cdc.gov/flu/signs-symptoms/index.html: runny or stuffy nose, cough, sore throat, headaches, muscle or body aches, fatigue, fever, and vomiting or diarrhea.  https://www.cdc.gov/covid/signs-symptoms/index.html: congestion or runny nose, cough, sore throat, headache, muscle or body aches, fatigue, fever or chills, nausea or vomiting, and diarrhea.  The only distinct symptoms of COVID were “shortness of breath or difficulty breathing” and “new loss of taste or smell.” The first symptom is already https://www.cdc.gov/nchs/fastats/leading-causes-of-death.htm in the United States - chronic lower respiratory disease. Since COVID was first tested CLRD has been bumped to sixth, though many of these respiratory deaths have been listed as COVID. In other words, what would have been diagnosed as CLRD was categorized instead as COVID-19. This is the same reason flu nearly disappeared as reported cases. These breathing problems were, within the COVID diagnosis, themselves sub-categorized as COVID-Pneumonia, and https://my.clevelandclinic.org/health/diseases/24002-covid-pneumonia.   The second symptom of losing a senes of taste or smell varied between total loss and partial loss, something that also occurs with the common cold and flu. This distinct and often promoted https://www.healthline.com/health-news/who-is-most-likely-to-lose-their-sense-of-smell-and-taste-from-covid-19, and even so only involved some often minor or unspecified form of loss.  Thus we can determine that between 15-37% of COVID cases had “distinct” symptoms arguably different than the overall symptoms that classify cold or flu, which means at liberal estimates over two thirds of COVID cases were nothing more than a case of the cold or flu. When defining what caused these other symptoms, we know recategorized pneumonia was one. But what about other causes that resulted in loss of senses?  Other than injuries or inflammation, often caused by what we call allergies, https://www.livestrong.com/article/13731552-food-suddenly-tastes-different/ and https://pubmed.ncbi.nlm.nih.gov/27178656/, as can neurological disorders. Other than the obvious and physical, there is also the psychological. Anxiety and stress are well known to alter sense perceptions, including https://www.calmclinic.com/anxiety/signs/smell and https://pmc.ncbi.nlm.nih.gov/articles/PMC10668578/. Consider how much anxiety and stress were cultivated by 24-hour coverage of cases, deaths, symptoms, videos from China, etc., and how wiling the public was to adopt any perceptually legal or even illogical dictate for the purposes of keeping themselves and others “safe.” There is a long history of such mass psychogenic pathogen.Much of this fear was generated by variant names like “KRAKEN,” a mythical monster, as was https://www.unmc.edu/healthsecurity/transmission/2023/09/19/meet-the-man-who-named-covids-new-variants/ who likewise believed this naming heightened the public's perception of a terror they should be feeling. Such fear became so intense that one analysis suggested that COVID activated “archetypes of evil” and thus “added psychological suffering.” The study suggested: “Fear and grief caused by the pandemic have produced a powerful unconscious narrative in the collective psyche that the coronavirus is driven by an https://pmc.ncbi.nlm.nih.gov/articles/PMC8441919/. The resulting archetypal dimension of fear causes an extra layer of psychological suffering in individuals.”  Such mythical, theological, and even magical terms were not lost in the New England Journal of Medicine which openly declared in 2020 that masks were little more than talismans: “Masks are not only tools, https://www.nejm.org/doi/full/10.1056/NEJMp2006372...” A spirit or demon possessing a body is an impure form that makes one sick. Items such as crosses or holy water are employed in its exorcism - to exercise/exorcise the demon and make healthy again - along with the name of the unclean. The same is done today in modern vaccine administration. The holy water is replaced by a vaccine vial, the cross is replaced by a syringe and plunger, the demon's name is replaced by the variant or virus name, and the ritual robes are replaced by white lab coats.  The pandemic was not about a virus and a distinct set of symptoms. Instead it was about inducing archetypical fear and https://www.weforum.org/stories/2020/10/the-rich-got-richer-during-the-pandemic-and-that-s-a-daunting-sign-for-our-recovery/. It was at best https://www.history.com/articles/mysterious-illnesses-mass-hysteria, in the middle a conspiracy of fraud and psychological terror, and at worst a dark magical ritual to induce trauma.  Further evidence of the fraud can be found in reports like this one from the New York Times that discuss the ultra amplification of PCR testing cycles from the low 30s to the mid 40s -“https://absa.org/wp-content/uploads/2020/09/NYT-200829-Your-Coronavirus-Test.pdf” - “In three sets of testing data that include cycle thresholds, compiled by officials in Massachusetts, New York and Nevada, up to 90 percent of people testing positive carried barely any virus…In Massachusetts, from 85 to 90 percent of people who tested positive in July with a cycle threshold of 40 would have been deemed negative if the threshold were 30 cycles.”  Even ‘true' positive tests do not indicate symptoms or disease, or the future development of such, which brings us back to the White House website and the statement about the lab leak: “The virus possesses a biological characteristic that is not found in nature.” This may be true, as per whatever is being assumed to exist, or observed under a microscope, or played with in a computer model, yet it does not prove any disease, especially in https://abcnews.go.com/Health/covid-transmission-asymptomatic/story?id=84599810. *The is the FREE archive.Become a supporter of this podcast: https://www.spreaker.com/podcast/the-secret-teachings--5328407/support.

In this week's episode we'll learn more about how phosphoseryl-tRNA kinase inhibition promotes cell death in acute myeloid leukemia, or AML; APOE gene variants and their association with post-hematopoietic stem cell transplant outcomes in AML; and pathways by which chronic inflammation and oxidative stress may lead to cardiomyopathy in patients with sickle cell disease.Featured Articles:PSTK inhibition activates cGAS-STING, precipitating ferroptotic cell death in leukemic stem cells Common Hereditary Variants of the APOE Gene and Posttransplant Outcome in Acute Myeloid Leukemia 17R-Resolvin D1 Protects Against Sickle Cell Related Inflammatory Cardiomyopathy in Humanized Mice 

Philosophers have for over two millennia debated the irresistible force paradox, usually formulated as ‘What happens when an unstoppable force meets an immovable object?' Variants of the problem date back as far as China in the third century BCE and ancient Greece. The world got one answer to the question on March 26, 2024, when a Singapore-flagged boxship hit the Francis Scott Key Bridge that spanned Baltimore harbour in the US. The vessel was inevitably damaged, the bridge collapsed, and six people from a maintenance crew working on the bridge at the time were killed. Within days, the owners and managers of Dali filed for limitation of liability. If their suit is successful, Grace Ocean, Synergy Marine and their insurers will pay out no more than $44m. In this edition of the podcast, Lloyd's List insurance and law editor David Osler unpacks what happens next in what could turn out to be one of the costliest marine casualties of all time.

In this episode I discuss the following ⭐️ DJ Spin .. I discuss high voltage and what makes it kind of boring.. also i go over this months cards so far (high expectations can lead to disappointment). ⭐️ Listener questions (thank you to both who sent them this week! ) please send me YOURS via my X/ Blusky or my discord !! ⭐️ my week in snap & what's in the shop with where I am for the month.. how has the climb been? Decks I've played , variants , bundles and more! ⭐️ Strange Supreme in comics corner (no comic presence just the origin from what if ) Also a look at the new books in the marvel unlimited app⭐️ MCU minute with a daredevil finale review and my thoughts on the entire season/ early (non spoiler) reviews from people who pre screened Thunderbolts, Fantastic Four promo art and the rest of the MCU projects coming out this year!

Listen in as Real Science Radio host Fred Williams and co-host Doug McBurney review and update some of Bob Enyart's legendary list of not so old things! From Darwin's Finches to opals forming in months to man's genetic diversity in 200 generations, to carbon 14 everywhere it's not supposed to be (including in diamonds and dinosaur bones!), scientific observations simply defy the claim that the earth is billions of years old. Real science demands the dismissal of the alleged million and billion year ages asserted by the ungodly and the foolish.     * Finches Adapt in 17 Years, Not 2.3 Million: Charles Darwin's finches are claimed to have taken 2,300,000 years to diversify from an initial species blown onto the Galapagos Islands. Yet individuals from a single finch species on a U.S. Bird Reservation in the Pacific were introduced to a group of small islands 300 miles away and in at most 17 years, like Darwin's finches, they had diversified their beaks, related muscles, and behavior to fill various ecological niches. Hear about this also at rsr.org/spetner.  * Finches Speciate in Two Generations vs Two Million Years for Darwin's Birds?  Darwin's finches on the Galapagos Islands are said to have diversified into 14 species over a period of two million years. But in 2017 the journal Science reported a newcomer to the Island which within two generations spawned a reproductively isolated new species. In another instance as documented by Lee Spetner, a hundred birds of the same finch species introduced to an island cluster a 1,000 kilometers from Galapagos diversified into species with the typical variations in beak sizes, etc. "If this diversification occurred in less than seventeen years," Dr. Spetner asks, "why did Darwin's Galapagos finches [as claimed by evolutionists] have to take two million years?" * Opals Can Form in "A Few Months" And Don't Need 100,000 Years: A leading authority on opals, Allan W. Eckert, observed that, "scientific papers and textbooks have told that the process of opal formation requires tens of thousands of years, perhaps hundreds of thousands... Not true." A 2011 peer-reviewed paper in a geology journal from Australia, where almost all the world's opal is found, reported on the: "new timetable for opal formation involving weeks to a few months and not the hundreds of thousands of years envisaged by the conventional weathering model." (And apparently, per a 2019 report from Entomology Today, opals can even form around insects!) More knowledgeable scientists resist the uncritical, group-think insistence on false super-slow formation rates (as also for manganese nodules, gold veins, stone, petroleum, canyons and gullies, and even guts, all below). Regarding opals, Darwinian bias led geologists to long ignore possible quick action, as from microbes, as a possible explanation for these mineraloids. For both in nature and in the lab, opals form rapidly, not even in 10,000 years, but in weeks. See this also from creationists by a geologist, a paleobiochemist, and a nuclear chemist. * Blue Eyes Originated Not So Long Ago: Not a million years ago, nor a hundred thousand years ago, but based on a peer-reviewed paper in Human Genetics, a press release at Science Daily reports that, "research shows that people with blue eyes have a single, common ancestor. A team at the University of Copenhagen have tracked down a genetic mutation which took place 6-10,000 years ago and is the cause of the eye color of all blue-eyed humans alive on the planet today." * Adding the Entire Universe to our List of Not So Old Things? Based on March 2019 findings from Hubble, Nobel laureate Adam Riess of the Space Telescope Science Institute and his co-authors in the Astrophysical Journal estimate that the universe is about a billion years younger than previously thought! Then in September 2019 in the journal Science, the age dropped precipitously to as low as 11.4 billion years! Of course, these measurements also further squeeze the canonical story of the big bang chronology with its many already existing problems including the insufficient time to "evolve" distant mature galaxies, galaxy clusters, superclusters, enormous black holes, filaments, bubbles, walls, and other superstructures. So, even though the latest estimates are still absurdly too old (Google: big bang predictions, and click on the #1 ranked article, or just go on over there to rsr.org/bb), regardless, we thought we'd plop the whole universe down on our List of Not So Old Things!   * After the Soft Tissue Discoveries, NOW Dino DNA: When a North Carolina State University paleontologist took the Tyrannosaurus Rex photos to the right of original biological material, that led to the 2016 discovery of dinosaur DNA, So far researchers have also recovered dinosaur blood vessels, collagen, osteocytes, hemoglobin, red blood cells, and various proteins. As of May 2018, twenty-six scientific journals, including Nature, Science, PNAS, PLoS One, Bone, and Journal of Vertebrate Paleontology, have confirmed the discovery of biomaterial fossils from many dinosaurs! Organisms including T. Rex, hadrosaur, titanosaur, triceratops, Lufengosaur, mosasaur, and Archaeopteryx, and many others dated, allegedly, even hundreds of millions of years old, have yielded their endogenous, still-soft biological material. See the web's most complete listing of 100+ journal papers (screenshot, left) announcing these discoveries at bflist.rsr.org and see it in layman's terms at rsr.org/soft. * Rapid Stalactites, Stalagmites, Etc.: A construction worker in 1954 left a lemonade bottle in one of Australia's famous Jenolan Caves. By 2011 it had been naturally transformed into a stalagmite (below, right). Increasing scientific knowledge is arguing for rapid cave formation (see below, Nat'l Park Service shrinks Carlsbad Caverns formation estimates from 260M years, to 10M, to 2M, to it "depends"). Likewise, examples are growing of rapid formations with typical chemical make-up (see bottle, left) of classic stalactites and stalagmites including: - in Nat'l Geo the Carlsbad Caverns stalagmite that rapidly covered a bat - the tunnel stalagmites at Tennessee's Raccoon Mountain - hundreds of stalactites beneath the Lincoln Memorial - those near Gladfelter Hall at Philadelphia's Temple University (send photos to Bob@rsr.org) - hundreds of stalactites at Australia's zinc mine at Mt. Isa.   - and those beneath Melbourne's Shrine of Remembrance. * Most Human Mutations Arose in 200 Generations: From Adam until Real Science Radio, in only 200 generations! The journal Nature reports The Recent Origin of Most Human Protein-coding Variants. As summarized by geneticist co-author Joshua Akey, "Most of the mutations that we found arose in the last 200 generations or so" (the same number previously published by biblical creationists). Another 2012 paper, in the American Journal of Physical Anthropology (Eugenie Scott's own field) on High mitochondrial mutation rates, shows that one mitochondrial DNA mutation occurs every other generation, which, as creationists point out, indicates that mtEve would have lived about 200 generations ago. That's not so old! * National Geographic's Not-So-Old Hard-Rock Canyon at Mount St. Helens: As our List of Not So Old Things (this web page) reveals, by a kneejerk reaction evolutionary scientists assign ages of tens or hundreds of thousands of years (or at least just long enough to contradict Moses' chronology in Genesis.) However, with closer study, routinely, more and more old ages get revised downward to fit the world's growing scientific knowledge. So the trend is not that more information lengthens ages, but rather, as data replaces guesswork, ages tend to shrink until they are consistent with the young-earth biblical timeframe. Consistent with this observation, the May 2000 issue of National Geographic quotes the U.S. Forest Service's scientist at Mount St. Helens, Peter Frenzen, describing the canyon on the north side of the volcano. "You'd expect a hard-rock canyon to be thousands, even hundreds of thousands of years old. But this was cut in less than a decade." And as for the volcano itself, while again, the kneejerk reaction of old-earthers would be to claim that most geologic features are hundreds of thousands or millions of years old, the atheistic National Geographic magazine acknowledges from the evidence that Mount St. Helens, the volcanic mount, is only about 4,000 years old! See below and more at rsr.org/mount-st-helens. * Mount St. Helens Dome Ten Years Old not 1.7 Million: Geochron Laboratories of Cambridge, Mass., using potassium-argon and other radiometric techniques claims the rock sample they dated, from the volcano's dome, solidified somewhere between 340,000 and 2.8 million years ago. However photographic evidence and historical reports document the dome's formation during the 1980s, just ten years prior to the samples being collected. With the age of this rock known, radiometric dating therefore gets the age 99.99999% wrong. * Devils Hole Pupfish Isolated Not for 13,000 Years But for 100: Secular scientists default to knee-jerk, older-than-Bible-age dates. However, a tiny Mojave desert fish is having none of it. Rather than having been genetically isolated from other fish for 13,000 years (which would make this small school of fish older than the Earth itself), according to a paper in the journal Nature, actual measurements of mutation rates indicate that the genetic diversity of these Pupfish could have been generated in about 100 years, give or take a few. * Polystrates like Spines and Rare Schools of Fossilized Jellyfish: Previously, seven sedimentary layers in Wisconsin had been described as taking a million years to form. And because jellyfish have no skeleton, as Charles Darwin pointed out, it is rare to find them among fossils. But now, reported in the journal Geology, a school of jellyfish fossils have been found throughout those same seven layers. So, polystrate fossils that condense the time of strata deposition from eons to hours or months, include: - Jellyfish in central Wisconsin were not deposited and fossilized over a million years but during a single event quick enough to trap a whole school. (This fossil school, therefore, taken as a unit forms a polystrate fossil.) Examples are everywhere that falsify the claims of strata deposition over millions of years. - Countless trilobites buried in astounding three dimensionality around the world are meticulously recovered from limestone, much of which is claimed to have been deposited very slowly. Contrariwise, because these specimens were buried rapidly in quickly laid down sediments, they show no evidence of greater erosion on their upper parts as compared to their lower parts. - The delicacy of radiating spine polystrates, like tadpole and jellyfish fossils, especially clearly demonstrate the rapidity of such strata deposition. - A second school of jellyfish, even though they rarely fossilized, exists in another locale with jellyfish fossils in multiple layers, in Australia's Brockman Iron Formation, constraining there too the rate of strata deposition. By the way, jellyfish are an example of evolution's big squeeze. Like galaxies evolving too quickly, 

Listen in as Real Science Radio host Fred Williams and co-host Doug McBurney review and update some of Bob Enyart's legendary list of not so old things! From Darwin's Finches to opals forming in months to man's genetic diversity in 200 generations, to carbon 14 everywhere it's not supposed to be (including in diamonds and dinosaur bones!), scientific observations simply defy the claim that the earth is billions of years old. Real science demands the dismissal of the alleged million and billion year ages asserted by the ungodly and the foolish.   * Finches Adapt in 17 Years, Not 2.3 Million: Charles Darwin's finches are claimed to have taken 2,300,000 years to diversify from an initial species blown onto the Galapagos Islands. Yet individuals from a single finch species on a U.S. Bird Reservation in the Pacific were introduced to a group of small islands 300 miles away and in at most 17 years, like Darwin's finches, they had diversified their beaks, related muscles, and behavior to fill various ecological niches. Hear about this also at rsr.org/spetner.  * Finches Speciate in Two Generations vs Two Million Years for Darwin's Birds?  Darwin's finches on the Galapagos Islands are said to have diversified into 14 species over a period of two million years. But in 2017 the journal Science reported a newcomer to the Island which within two generations spawned a reproductively isolated new species. In another instance as documented by Lee Spetner, a hundred birds of the same finch species introduced to an island cluster a 1,000 kilometers from Galapagos diversified into species with the typical variations in beak sizes, etc. "If this diversification occurred in less than seventeen years," Dr. Spetner asks, "why did Darwin's Galapagos finches [as claimed by evolutionists] have to take two million years?" * Opals Can Form in "A Few Months" And Don't Need 100,000 Years: A leading authority on opals, Allan W. Eckert, observed that, "scientific papers and textbooks have told that the process of opal formation requires tens of thousands of years, perhaps hundreds of thousands... Not true." A 2011 peer-reviewed paper in a geology journal from Australia, where almost all the world's opal is found, reported on the: "new timetable for opal formation involving weeks to a few months and not the hundreds of thousands of years envisaged by the conventional weathering model." (And apparently, per a 2019 report from Entomology Today, opals can even form around insects!) More knowledgeable scientists resist the uncritical, group-think insistence on false super-slow formation rates (as also for manganese nodules, gold veins, stone, petroleum, canyons and gullies, and even guts, all below). Regarding opals, Darwinian bias led geologists to long ignore possible quick action, as from microbes, as a possible explanation for these mineraloids. For both in nature and in the lab, opals form rapidly, not even in 10,000 years, but in weeks. See this also from creationists by a geologist, a paleobiochemist, and a nuclear chemist. * Blue Eyes Originated Not So Long Ago: Not a million years ago, nor a hundred thousand years ago, but based on a peer-reviewed paper in Human Genetics, a press release at Science Daily reports that, "research shows that people with blue eyes have a single, common ancestor. A team at the University of Copenhagen have tracked down a genetic mutation which took place 6-10,000 years ago and is the cause of the eye color of all blue-eyed humans alive on the planet today." * Adding the Entire Universe to our List of Not So Old Things? Based on March 2019 findings from Hubble, Nobel laureate Adam Riess of the Space Telescope Science Institute and his co-authors in the Astrophysical Journal estimate that the universe is about a billion years younger than previously thought! Then in September 2019 in the journal Science, the age dropped precipitously to as low as 11.4 billion years! Of course, these measurements also further squeeze the canonical story of the big bang chronology with its many already existing problems including the insufficient time to "evolve" distant mature galaxies, galaxy clusters, superclusters, enormous black holes, filaments, bubbles, walls, and other superstructures. So, even though the latest estimates are still absurdly too old (Google: big bang predictions, and click on the #1 ranked article, or just go on over there to rsr.org/bb), regardless, we thought we'd plop the whole universe down on our List of Not So Old Things!   * After the Soft Tissue Discoveries, NOW Dino DNA: When a North Carolina State University paleontologist took the Tyrannosaurus Rex photos to the right of original biological material, that led to the 2016 discovery of dinosaur DNA, So far researchers have also recovered dinosaur blood vessels, collagen, osteocytes, hemoglobin, red blood cells, and various proteins. As of May 2018, twenty-six scientific journals, including Nature, Science, PNAS, PLoS One, Bone, and Journal of Vertebrate Paleontology, have confirmed the discovery of biomaterial fossils from many dinosaurs! Organisms including T. Rex, hadrosaur, titanosaur, triceratops, Lufengosaur, mosasaur, and Archaeopteryx, and many others dated, allegedly, even hundreds of millions of years old, have yielded their endogenous, still-soft biological material. See the web's most complete listing of 100+ journal papers (screenshot, left) announcing these discoveries at bflist.rsr.org and see it in layman's terms at rsr.org/soft. * Rapid Stalactites, Stalagmites, Etc.: A construction worker in 1954 left a lemonade bottle in one of Australia's famous Jenolan Caves. By 2011 it had been naturally transformed into a stalagmite (below, right). Increasing scientific knowledge is arguing for rapid cave formation (see below, Nat'l Park Service shrinks Carlsbad Caverns formation estimates from 260M years, to 10M, to 2M, to it "depends"). Likewise, examples are growing of rapid formations with typical chemical make-up (see bottle, left) of classic stalactites and stalagmites including: - in Nat'l Geo the Carlsbad Caverns stalagmite that rapidly covered a bat - the tunnel stalagmites at Tennessee's Raccoon Mountain - hundreds of stalactites beneath the Lincoln Memorial - those near Gladfelter Hall at Philadelphia's Temple University (send photos to Bob@rsr.org) - hundreds of stalactites at Australia's zinc mine at Mt. Isa.   - and those beneath Melbourne's Shrine of Remembrance. * Most Human Mutations Arose in 200 Generations: From Adam until Real Science Radio, in only 200 generations! The journal Nature reports The Recent Origin of Most Human Protein-coding Variants. As summarized by geneticist co-author Joshua Akey, "Most of the mutations that we found arose in the last 200 generations or so" (the same number previously published by biblical creationists). Another 2012 paper, in the American Journal of Physical Anthropology (Eugenie Scott's own field) on High mitochondrial mutation rates, shows that one mitochondrial DNA mutation occurs every other generation, which, as creationists point out, indicates that mtEve would have lived about 200 generations ago. That's not so old! * National Geographic's Not-So-Old Hard-Rock Canyon at Mount St. Helens: As our List of Not So Old Things (this web page) reveals, by a kneejerk reaction evolutionary scientists assign ages of tens or hundreds of thousands of years (or at least just long enough to contradict Moses' chronology in Genesis.) However, with closer study, routinely, more and more old ages get revised downward to fit the world's growing scientific knowledge. So the trend is not that more information lengthens ages, but rather, as data replaces guesswork, ages tend to shrink until they are consistent with the young-earth biblical timeframe. Consistent with this observation, the May 2000 issue of National Geographic quotes the U.S. Forest Service's scientist at Mount St. Helens, Peter Frenzen, describing the canyon on the north side of the volcano. "You'd expect a hard-rock canyon to be thousands, even hundreds of thousands of years old. But this was cut in less than a decade." And as for the volcano itself, while again, the kneejerk reaction of old-earthers would be to claim that most geologic features are hundreds of thousands or millions of years old, the atheistic National Geographic magazine acknowledges from the evidence that Mount St. Helens, the volcanic mount, is only about 4,000 years old! See below and more at rsr.org/mount-st-helens. * Mount St. Helens Dome Ten Years Old not 1.7 Million: Geochron Laboratories of Cambridge, Mass., using potassium-argon and other radiometric techniques claims the rock sample they dated, from the volcano's dome, solidified somewhere between 340,000 and 2.8 million years ago. However photographic evidence and historical reports document the dome's formation during the 1980s, just ten years prior to the samples being collected. With the age of this rock known, radiometric dating therefore gets the age 99.99999% wrong. * Devils Hole Pupfish Isolated Not for 13,000 Years But for 100: Secular scientists default to knee-jerk, older-than-Bible-age dates. However, a tiny Mojave desert fish is having none of it. Rather than having been genetically isolated from other fish for 13,000 years (which would make this small school of fish older than the Earth itself), according to a paper in the journal Nature, actual measurements of mutation rates indicate that the genetic diversity of these Pupfish could have been generated in about 100 years, give or take a few. * Polystrates like Spines and Rare Schools of Fossilized Jellyfish: Previously, seven sedimentary layers in Wisconsin had been described as taking a million years to form. And because jellyfish have no skeleton, as Charles Darwin pointed out, it is rare to find them among fossils. But now, reported in the journal Geology, a school of jellyfish fossils have been found throughout those same seven layers. So, polystrate fossils that condense the time of strata deposition from eons to hours or months, include: - Jellyfish in central Wisconsin were not deposited and fossilized over a million years but during a single event quick enough to trap a whole school. (This fossil school, therefore, taken as a unit forms a polystrate fossil.) Examples are everywhere that falsify the claims of strata deposition over millions of years. - Countless trilobites buried in astounding three dimensionality around the world are meticulously recovered from limestone, much of which is claimed to have been deposited very slowly. Contrariwise, because these specimens were buried rapidly in quickly laid down sediments, they show no evidence of greater erosion on their upper parts as compared to their lower parts. - The delicacy of radiating spine polystrates, like tadpole and jellyfish fossils, especially clearly demonstrate the rapidity of such strata deposition. - A second school of jellyfish, even though they rarely fossilized, exists in another locale with jellyfish fossils in multiple layers, in Australia's Brockman Iron Formation, constraining there too the rate of strata deposition. By the way, jellyfish are an example of evolution's big squeeze. Like galaxies e

Tube Screamers—whether you love them, hate them, or can't decide, their impact on the guitar world is undeniable. In this episode of Four On The Floor, we explore four unique Tube Screamer-style variants, each offering a fresh take on the classic sound with added versatility and tonal options. Your guitar will thank you. Hosted by Todd Novak with Tony Dudzik, Rob Chafe, and Bobby Withers   #guitarpodcast #electricguitar #pedaleffects #pedalfx #theguitarknobs #guitarknobs #guitarinterview #guitaramplifier #guitarpickups #guitarsetup #fuzz #overdrive #reverb #distortion #guitartips    Visit us at theguitarknobs.com Support our show on Patreon.com/theguitarknobs

It's a heated debate in the Star Wars toy collecting world: Variants or Fariants? Tonight, the Five Idiots Talking Toys crew goes LIVE to hash it out! We'll be exploring which action figure variations hold true collectible value and which are potentially being overhyped. From mold differences and plastic degradation to paint breakdown, factory errors, running changes, and COO/factory distinctions, we're leaving no stone unturned. Get ready for an honest and opinionated discussion! #ToyCommunity #VariantDiscussion #FakeOrReal #CollectingTips #PodcastShow

New ransomware group claims attack on US Telecom firm WideOpenWest NSA warned of vulnerabilities in Signal app a month before Houthi strike chat New ReaderUpdate malware variants target macOS users Huge thanks to our episode sponsor, ThreatLocker ThreatLocker® is a global leader in Zero Trust endpoint security, offering cybersecurity controls to protect businesses from zero-day attacks and ransomware. ThreatLocker operates with a default deny approach to reduce the attack surface and mitigate potential cyber vulnerabilities. To learn more and start your free trial, visit ThreatLocker.com.

The biggest stories on the internet from March 20th, 2025.Timestamps:1:23 Emma Chamberlain film debut4:02 Billboard cracks down on album variants5:58 Shay Mitchell's Beis pop up scandalFind our podcast YouTube channel here: https://www.youtube.com/channel/UC18HclY7Tt5-1e3Z-MEP7Jg Subscribe to our weekly Substack: https://centennialworld.substack.com/ Follow us on Instagram: https://www.instagram.com/infinitescrollpodcast/ Follow Lauren on Instagram: https://www.instagram.com/laurenmeisner_/ 

durée : 00:05:08 - Avec sciences - par : Alexandre Morales - Une nouvelle étude publiée dans Science dévoile un nouveau type d'anticorps efficace sur tous les variants de SARS-CoV-2 étudiés par les auteurs.

In this episode, DuDs and Carl discuss all the warm and cold additions to Minecraft. Plus, is the Creaking just the exact opposite of an Enderman? Also, Mending Minecraft is back! Make sure you vote in this episode's poll on Spotify, YouTube, and Discord.Thank you to our Milk level Patrons: Aerington, aubni, FragileRock, Jronman, LOKIOLR, and The Meme Bug.(AD) Do you need a Minecraft server? BisectHosting is recommended by the Podcast Team for easy set-up, 24/7 support with fast response times, and a 3-day money-back guarantee. Interested? Get 25% off your first month of any gaming server at ⁠⁠⁠⁠https://bisecthosting.com/TWE⁠⁠ and use code TWE at checkout. New clients only.Discord: ⁠⁠https://discord.gg/gqnKyeZ⁠⁠Patreon: ⁠⁠⁠⁠https://patreon.com/thewitheringeffect⁠⁠Website: ⁠⁠⁠⁠http://thewitheringeffect.com/⁠⁠E-Mail: ⁠⁠podcast@thewitheringeffect.com⁠⁠X/Twitter: ⁠⁠⁠⁠https://twitter.com/WitheringEffect⁠⁠YouTube: ⁠⁠⁠⁠https://youtube.com/thewitheringeffect⁠⁠Instagram: ⁠⁠⁠⁠https://instagram.com/witheringeffect⁠⁠TikTok: ⁠https://tiktok.com/@witheringeffect⁠Show HostDuDs YouTube: ⁠⁠⁠⁠https://youtube.com/DuDs_vs⁠⁠DuDs X/Twitter: ⁠⁠⁠⁠https://twitter.com/DuDs_vs⁠⁠DuDs Twitch: ⁠⁠https://twitch.tv/DuDs_vs⁠⁠Show Host / Digital ProducerCarlRyds YouTube: ⁠⁠⁠⁠https://youtube.com/CarlRydsGaming⁠⁠CarlRyds X/Twitter: ⁠⁠⁠⁠https://twitter.com/CarlRyds⁠⁠CarlRyds Twitch: ⁠⁠https://twitch.tv/CarlRydsGaming⁠⁠Music MasterDiiKoj YouTube: ⁠⁠⁠⁠https://youtube.com/DiiKoj⁠⁠DiiKoj X/Twitter: ⁠⁠⁠⁠https://twitter.com/DiiKoj

In episode 2 of 'What's in a name' we look how choosing names can help, or hinder, attempts to communicate important messages.Categorizing things is central to science. And there are dozens of systems scientists have created to name everything from the trenches on the sea bed to the stars in the sky.But names have consequences. In our series What's in a name we explore naming in science and how names impact the world — whether the system of naming species remains in step with society, how the names of diseases can create stigma, or even how the names of scientific concepts can drive the direction of research itself.In episode two, we're looking at how the names chosen by scientists help, or hinder, communication with the public.Well chosen names can quickly convey scientific concepts or health messages — in emergency situations they can even save lives. We'll hear how the systems of naming tropical storms and Covid-19 variants came to be, and how they took different approaches to achieve the same outcome.We'll also consider the language used to talk about climate change, and how the ways of describing it have been used to deliberately introduce uncertainty and confusion.Listen to the first episode Should offensive species names be changed? The organisms that honour dictators, racists and criminalsFor a list of sources please visithttps://www.nature.com/articles/d41586-024-04200-9Music creditsPremiumaudio/Pond5Richard Smithson/Triple Scoop Music/Getty Images Hosted on Acast. See acast.com/privacy for more information.

In this episode, I sit down with Natalie Richheimer, a genetic counselor at JScreen, to dig into the world of preconception and prenatal genetic testing. We explore the basics of carrier screening, how to interpret results, and the importance of genetic counseling for both new and experienced parents—especially those who already have a child with a rare disease. Natalie also sheds light on Variants of Uncertain Significance (VUS) and offers insight into talking with family members about genetic risks and results. Throughout our chat, Natalie emphasizes that knowledge truly is power when it comes to family planning and navigating the complexities of genetic information. We discuss strategies for deciding between natural pregnancy, IVF with genetic screening, and other alternatives, helping you feel more confident and informed about your options. Whether you're just beginning your journey or seeking clarity in the midst of it, this episode offers valuable perspectives and heartfelt advice. Join us as we learn how to take charge of our genetic health and family-planning decisions with compassion and confidence. Finally, don't forget about the Once Upon a Gene Revival—an uplifting event designed to support, educate, and connect rare disease caregivers. Registration is open now on my website. Links: JScreen Genetic Testing Instagram Once Upon A Gene Revival

Jump in with Carlos Juico and Gavin Ruta on episode 217 of Jumpers Jump. This episode we discuss: LA Fire theories, Dark Hollywood theories, Alien Ocean theory, Project Blue Beam, Universe theories, Multiverse Jumping theory, The Upside Down is Real, Dark Nemo theory, The Losses before the Wins, Rarest Blood Type theory, Annunaki theories, Sabrina Priest Scandal, Supply & Demand theory, Community Subconscious theory, Square reality theory, Dog poop mandela effect, Burnt toast theory, The Chinese Farmer story, Fate theories, Carlos 22 Variants, Artist theories, Social Media theory, The Meaning of Life, Sentimental Value and much more! Download the DraftKings Sportsbook app and use code JUMPERSJUMP. That's code JUMPERSJUMP for new customers to get $200 in bonus bets instantly, when you bet just five bucks. Only on DraftKings Sportsbook - The Crown Is Yours. Gambling problem? Call one eight hundred Gambler. In New York, call 8778 HOPENY or text HOPENY (467369). In Connecticut, Help is available for problem gambling. Call 8887897777 or visit ccpg.org. Please play responsibly. On behalf of Boot Hill Casino & Resort (Kansas). Twenty-one plus age and eligibility varies by jurisdiction. Void in Ontario. Bonus bets expire one hundred sixty eight hours after issuance. For additional terms and responsible gaming resources, see DKNG.CO/AUDIO. Follow the podcast: @JumpersPodcast Follow Carlos: @CarlosJuico Follow Gavin: @GavinRutaa Check out the podcast on YouTube: https://bit.ly/JumpersJumpYT Learn more about your ad choices. Visit podcastchoices.com/adchoices