Podcasts about Salp

durée : 00:58:42 - Mauvais genres - par : François Angelier, Céline du Chéné - Avec "On m'a trouvé grandie", la chorégraphe et magicienne Valentine Losseau évoque, au travers de la figure de "Madeleine", le destin et la parole des internées de la Salpêtrière. - réalisation : Laurent Paulré - invités : Valentine Losseau Magicienne, dramaturge, metteuse en scène et anthropologue, fondatrice du mouvement artistique de la magie nouvelle

durée : 00:58:42 - Mauvais genres - par : François Angelier, Céline du Chéné - Avec "On m'a trouvé grandie", la chorégraphe et magicienne Valentine Losseau évoque, au travers de la figure de "Madeleine", le destin et la parole des internées de la Salpêtrière. - réalisation : Laurent Paulré - invités : Valentine Losseau Magicienne, dramaturge, metteuse en scène et anthropologue, fondatrice du mouvement artistique de la magie nouvelle

"Antes eu acreditava em uma só espécie: a minha. Mas me decepcionei ao descobrir que somos atrozes, violentos, horríveis, que estamos nos destruindo e a nosso planeta. Descobri também que faço parte de um universo enorme de espécies, e que se a minha desaparecer, não há problema". A frase é do fotógrafo brasileiro Sebastião Salgado, homenageado desde o dia 1° de março com uma grande retrospectiva com 166 de suas obras no centro cultural Les Franciscaines, em Deauville, na França.  "Descobri que sou parte de tudo isso", insistia Sebastião Salgado, 81 anos, na abertura da grande exposição Sebastião Salgado: Obras da Coleção da MEP, que homenageia não apenas suas séries icônicas de fotografias em preto e branco, mas também sua vida e seu olhar às vezes terno, às vezes dramático, e muitas vezes crítico sobre o mundo. O evento destaca um trabalho de mais de 40 anos percorrendo os quatro cantos do planeta, que resultou em trabalhos como "Exôdos" (1993-2000), sobre as grandes migrações humanas, "Gênesis" (2004-2011), sobre a natureza intocada do planeta, ou "A Mão do Homem" (1986-1992), sobre os trabalhadores, a precarização do trabalho artesanal e as grandes transformações do mundo industrial.A retrospectiva, que faz parte do calendário de comemorações oficiais do ano do Brasil na França, conta com a parceria da Maison Européenne de la Photographie (MEP), entidade que apoiou o fotógrafo brasileiro desde o início de sua carreira, quando trocou a formação de economista em Londres pelas lentes que imortalizariam grandes momentos da humanidade, dos animais e da natureza."Não sou um ser que domina o que está em volta, eu sou parte de tudo isso, dos minerais, dos vegetais", diz, Salgado, quando perguntado sobre o que aprendeu ao realizar a série "Gênesis", que ele afirma ter "reacendido sua esperança na vida e no planeta"."Todos os minerais têm uma inteligência inacreditável, assim como os vegetais. Uma vez fotografei uma árvore na Serra Nevada, nos Estados Unidos. Ela tinha sido parcialmente queimada, e cientistas que me acompanhavam me disseram que ela havia sido tocada pelo fogo de um determinado lado há mais de 1.500 anos. Incrível. Para ficar frente a um ser como esse é necessário muito respeito, e tempo para compreendê-lo, e para que ele possa compreender você também. Para que possamos fotografar a dignidade presente nesta árvore", argumentou um Sebastião Salgado emocionado, mas incansável durante a sequência de perguntas da imprensa internacional. "Não estou 100%"Integrante da Academia de Belas Artes da França desde 2017 e premiado pela Organização Mundial de Fotografia, em Londres, o fotógrafo surpreendeu o público presente no centro cultural Les Franciscaines, em Deauville, na região da Normandia, no norte da França, com um tom emocionado, pedindo "antes de tudo desculpas" e dizendo que não estava "100%". "Fui internado na semana passada num hospital em São Paulo para continuar um tratamento e volto na semana que vem para lá. É uma doença bem forte que eu adquiri há cerca de 15 anos trabalhando no projeto 'Gênesis', na Nova Guiné. Eu peguei malária e os médicos em Paris, no [tradicional hospital] Salpêtrière, me disseram que eu deveria descansar durante seis meses, porque a doença ataca o corpo inteiro, mas eu tive que desligar porque já estava no platô do Colorado", contou Salgado em francês, fazendo a audiência rir."Eu não pude interromper a excursão fotográfica, mas quando voltei a Paris, minha defesa imunológica despencou e eu desenvolvi uma infecção generalizada, tomando doses cavalares de antibiótico. Minha máquina de produzir glóbulos brancos e vermelhos se danificou para sempre. É uma espécie de câncer que adquiri, sou tratado por oncologistas", revelou. "Tomo medicamentos há 15 anos e isso ajuda um pouco, fiz toda a série na Amazônia assim, mas há duas semanas meu corpo começou a rejeitar o remédio e eu tive uma hemorragia no baço. Me desculpem, eu não estou nem com 50% da minha energia", disse Salgado, em meio a aplausos da plateia. O privilégio "enorme" de "estar vivo"Sebastião Salgado chegou a chorar ao lembrar de seus périplos pelo planeta, na abertura do evento, e ao celebrar colegas mortos durante sua trajetória. "O dia mais feliz da minha vida foi quando completei 80 anos... Simplesmente porque eu estava aqui. Eu não estava morto. Quantos amigos perdi, éramos todos amigos durante quatro anos em Goma [,na República Democrática do Congo], quatro fotógrafos foram assassinados, eu estava lá. Então para mim, estar vivo com 80 anos, é um privilégio enorme", confessou.Durante a coletiva, Salgado falou durante 25 minutos sobre suas experiências nos quatro cantos do globo. "Eu tive o privilégio de ir a esses lugares. Algumas vezes as pessoas me dizem que sou um artista, eu digo que não, que sou um fotógrafo. Porque vamos sozinhos a todas essas regiões do mundo, face a todos os problemas que vocês possam imaginar, todos os desafios, e temos dúvidas, questões éticas, de legitimidade, de segurança, e somos nós, fotógrafos, que devemos encontrar respostas para essas perguntas", testemunhou. O fotógrafo lembrou de quando perdeu parte da audição no Kuwait. "Foi quando houve a explosão em quase seiscentos poços de petróleo. Eu estava num barco, fotografando uma história para The New York Times. Eu me lembro que as tropas norte-americanas estavam lá, era a guerra contra Saddam Hussein. Foi um momento terrível e sublime da minha vida", destacou. "Terrível porque foi a maior poluição já vista, tinha dias que não víamos o sol. Era tão surpreendente, porque num determinado momento batia um vento, conseguíamos ver entre as nuvens e podíamos finalmente ver um raio de sol", contou. "Eram homens para mim heróicos aqueles que entravam no fogo do petróleo que tentavam tapar os poços. Havia um medo enorme de ser queimado vivo e o barulho que esses poços produziam era como trabalhar atrás da turbina de um avião", lembrou. "Quando fui embora do Kuwait, havia perdido mais da metade da minha audição", relatou Salgado.A "mão do homem" e as belezas intocadas do planetaA retrospectiva dos trabalhos em preto e branco do fotógrafo brasileiro na França resgata desde suas primeiras reportagens sobre os danos causados pela seca e pela fome na África (1984-1985), até seu trabalho sobre a condição dos trabalhadores imigrantes na Europa, passando na série "Outras Américas" (1977-1984), onde ele revisita a América Latina, com uma visão humanista e universal.Na sequência, a exposição mostra registros captados no final da década de 1980, quando Sebastião Salgado começa a trabalhar seus grandes murais fotográficos. "A Mão do Homem" (1986-1992) é uma homenagem ao trabalho manual e à condição humana. "Eu estudei geopolítica, antropologia, vi que nós estávamos chegando ao fim da primeira grande revolução industrial e que as máquinas inteligentes estavam substituindo o proletariado em toda linha de produção, que os robôs estavam substituindo o homem", declarou em entrevista à RFI em Deauville. "Eu resolvi fazer um retrato da classe trabalhadora antes que ela desaparecesse, e fiz. Minha formação [em economia com ênfase social] me permitiu ver o momento histórico que eu estava vivendo e fotografando", sublinhou."Mas eu pude ver que uma outra maior revolução estava acontecendo. Que o fato da gente estar terminando um tipo de indústria nessa parte sofisticada do planeta, não significava que ela estava acabando, mas se transferindo para a China, para o Brasil, a Indonésia, o México, esses grandes países em território e população. Trabalho barato, mão de obra barata", atestou Salgado.Com "Êxodos" (1993-2000), Salgado documenta os grandes movimentos populacionais ao redor do mundo, relacionados aos conflitos e à pobreza resultante das transformações econômicas que abalam a nossa época. "Eu fui atrás dessa reorganização da família humana que estava acontecendo no mundo. Durante seis anos eu fotografei o que se transformou no livro "Êxodos". "Então essa minha herança visual, histórica, essa minha formação que permitiu me situar. Não que eu seja um militante, não que eu quis fazer coisas diferentes dos outros, mas o que eu fiz, eu fiz com uma certa coerência política", definiu Salgado à RFI.Por fim, "Gênesis" (2004-2012), fruto de oito anos de expedições épicas pelos quatro cantos do globo, mostra a beleza de nosso planeta e permite descobrir paisagens, animais e seres humanos que até então haviam escapado à pressão do mundo contemporâneo. "Cerca de 47% do planeta Terra continua intacto", sublinhou o fotógrafo, feroz opositor ao acordo bilateral entre a União Europeia e o Mercosul. "A Europa quer nos vender produtos industrializados a baixo custo em troca de insumos agrícolas baratos, mas sabemos que as terras cultiváveis que restam no Brasil são indígenas, e deveriam ser preservadas", martelou.O inferno do genocídio em Ruanda"Foi a coisa mais dura que eu vivi na minha vida", disse Sebastião Salgado sobre o genocídio em Ruanda, entre abril e julho de 1994, quando aproximadamente quase 1 milhão de pessoas, a maioria da etnia tutsi, foram brutalmente assassinadas por extremistas hutus. "Não era fácil chegar num campo de refugiados e ver morrer por dia cerca de 20 mil pessoas, com um grande trator que escavava buracos enormes no chão para depositar os cadáveres. Era tão insuportável que cheguei a ficar doente", relatou.Consultado pelo médico indicado por Cartier-Bresson, amigo da família, Salgado conta que o especialista disse que seu corpo "estava perfeito" e que "ele estava morrendo em Ruanda". "Foi quando fomos como Lélia [Wanick Salgado, esposa e parceira do fotógrafo] e os meninos para Trancoso, na Bahia onde alugamos uma casa. Nesse momento, meus pais decidiram nos doar a fazenda onde nasci. Foi quando eu tomei a decisão de abandonar a fotografia. Eu tinha vergonha de ser fotógrafo, porque até então eu havia fotografado apenas uma espécie, a nossa. Então tomei a decisão com a Lélia de me tornar fazendeiro. Começamos a plantar, mas uma enorme chuva destruiu um morro que meu pai havia feito no terreno, matando um riacho onde eu nadava na infância. Lélia me disse então: 'vamos abandonar tudo, vamos pegar essa terra e replantar a floresta", lembra, rememorando as origens do Instituto Terra, que já restaurou cerca de 709 hectares de Mata Atlântica e plantou mais de 2,7 milhões de árvores nativas no Vale do Rio Doce, em Minas Gerais."Céus dramáticos de Minas Gerais""Se você colocar 300 fotógrafos em um evento, você vai ter 300 fotografias diferentes, porque você só fotografa com a sua herança, com tudo que está dentro de você. As minhas fotografias têm séries, céus dramáticos, carregados. Isso vem de onde eu nasci, vem da chegada da época de chuva naquelas montanhas de Minas Gerais que meu pai me levava para ver... no pico mais alto da nossa fazenda, pra se chegar àquelas nuvens incríveis, para ver o raio de sol passar através dessas nuvens, ver a chuva. Então aquelas imagens ficaram em mim", diz o fotógrafo."Memória da sociedade""Cada vez que você aperta no botãozinho da câmera e faz uma imagem, você faz um corte representativo do planeta naquele momento, e você só o faz naquele momento. Precisa ter a realidade em frente para essa imagem existir, para ela ser vista como fotografia, senão ela vai ser vista como um objeto criado como um artistismo, mas não como fotografia. Fotografia é a memória da sociedade" rebate o fotógrafo."Fotografia é a memória, e a memória tem que existir. E a memória só pode ser feita através da realidade. Uma ficção não pode criar memória, então eu acho que a fotografia jamais perderá sua função", insiste. "Eu não estou querendo tirar o lugar da inteligência artificial. Eu acho que ela vai fazer coisas fantásticas, talvez até melhor do que a gente. Com a nossa inteligência normal o que nós fizemos foi destruir o planeta, fizemos guerra, fizemos violência. Talvez uma inteligência artificial seja realmente inteligente para levar a gente em outra direção", diz."Eu não sou contra a inteligência artificial, mas fotografia mesmo, só é fotografia. Quando você pega a fotografia que você faz no telefone celular, isso não é fotografia. Isso é uma linguagem de comunicação por imagem, mas que não tem nada a ver com a memória", ressalta. "Eu acho que o inteligência artificial não vai mudar absolutamente nada na fotografia porque a inteligência artificial só pode criar a partir do que já existe. Pode imaginar, transformar, mas a fotografia é outra coisa", conclui Salgado.A retrospectiva Sebastião Salgado: Obras da Coleção da MEP fica em cartaz no centro cultural Les Franciscaines, em Deauville, até o dia 1° de junho de 2025.

Odcinek #190, w którym w krakowskiej księgarni Compare Bookstore rozmawiam z Bartoszem Sadulskim o jego drugiej powieści „Szesnaście na Bourbon”. Wyruszamy w P jak podróż na wyspę, na której urodził się Michel Houellebecq. Cofamy się do 1673 roku i dołączamy do kobiet uciekających ze szpitala Salpêtrière.Jaki będzie ich los?Wszystkiego nie zdradzamy, ale mówimy o Z jak zabawie literaturą i G jak globusem, o historycznej nie-stylizacji i o zawłaszczaniu historii. Jest K jak  kulturowa rozpusta i K jak klasowość, D jak dwór króla, M jak mikrohistorie, P jak polowanie na czarownice oraz walka z D jak demonami. Pojawia się K jak kot Lucjan – talizman i obrońca w jednym. Autor sięga po opis ataku O jak ośmiornicy z powieści Verne'a.Na liście I jak inspiracji znajdujemy też Josepha Conrada. I zachwyca nas też pani Bourdon.W końcu musimy rozprawić się musimy z polskimi wątkami w książce, z tajemniczą postacią historyczną i z kobietą, która wygląda jak słynna Marysieńka. W tym odcinie słychać, co Bartosza cieszy w literaturze. Z jak zderzamy więc z sobą skojarzenia, tak jak on zderza frazy, stylizacje. Rozmowę kończymy na dywagacjach o przekładzie, o R jak recepcji książki na rynku francuskim.I liczymy na S jak skandal.

„Melancholie“, „Wahnsinn“, „Hysterie“. In der Geschichte hatten psychische Krankheiten viele Namen. Schon in der Antike waren seelische Leiden bekannt. Nur, woher sie kamen und wie man sie behandeln konnte, wusste man nicht. Oft wurden psychisch Kranke für „wahnsinnig“ gehalten und spätestens ab dem ausgehenden Mittelalter häufig in Irrenanstalten oder Tollhäusern weggesperrt. Das sollte sich Anfang des 19. Jahrhunderts ändern. Damals entstanden die ersten psychiatrischen Kliniken in Deutschland. Was als Reformbewegung begann, endete spätestens mit der Herrschaft der Nationalsozialisten 1933. Auch psychisch kranke Menschen galten jetzt als „unwertes Leben.“ Psychiatrische Kliniken gerieten wegen der Euthanasie-Programme der Nationalsozialisten in Verruf. Ab den 1970er Jahren gab es viele Reformen, aber viele Vorurteile blieben bis heute bestehen. Filme wie „Shutter Island“ oder „Einer flog über das Kuckucksnest“ verstärkten das noch. Ein Podcast unter anderem über wahnsinnige Könige, hysterische Frauen und die Frage: Wieso gab es nach dem Zweiten Weltkrieg noch Medikamentenversuche in deutschen Kinder- und Jugendpsychiatrien? Und wie sieht es heute aus bei der Behandlung psychischer Erkrankungen? **Gesprächspartner*innen:** - Burkhart Brückner - Boris Böhm - Heiner Fangerau - Astrid Freisen - Linus Günther **Literatur:** - Alvarado, Carlos S. (2009): „Nineteenth-Century Hysteria and Hypnosis: A Historical Note on Blanche Wittmann. Australian Journal of Clinical and Experimental Hypnosis. Volume 37, 1, S: 21-36. - Böhm, Boris (2011): Die Geschichte der Heil– und Pflegeanstalt Sonnenstein, 1811-1939. - Brückner, Burkhart (2023): Kurze Geschichte der Psychiatrie. - Didi-Hubermann, Georges (2004): Invention of Hysteria. Charcot and the Photographic Iconography of the Salpêtrière. - Fahmüller, Eva-Maria (2018): Geniale Psychopathen – labile Kommissare: Filmfiguren mit psychischen Störungen im aktuellen deutschen Krimi. - Freisen, Astrid (2023): Wir fliegen hoch, wir fallen tief. Eine Psychiaterin spricht offen über ihre bipolare Störung und zeigt, wie wir mit der Krankheit umgehen können. - Fleming, Gillian B. (2011): Juana I and the struggle for power in an age of transition (1504-1521). PhD thesis, London School of Economics and Political Science. - Freud, Sigmund; Breuer; Joseph (1895): Studien über Hysterie. - Guenther, Linus Paul Frederic (2023): The concept of hysteria as mirror of the relation between clinical and cultural psychology. In: Culture & Psychology, Vol. 30 (3), S. 538 – 550. - Hartig, Christiane (2020): Medikamentenversuche an Kindern und Jugendlichen im Rahmen der Heimerziehung in Niedersachsen zwischen 1945 und 1978. Robert Bosch Stiftung. - Kamen, Henry (2001): Philip V of Spain: The King Who Reigned Twice. - Nasser, Mervat (1987): Psychiatry in Ancient Egypt, Bulletin of the Royal College of Psychiatrists, Vol. 11. - Paulon, Moreno (2022): Hysteria. Rise and fall of a baffling disease. A review on history of ideas in medicine. In: Journal of Psychopathology. 2022;28: 152-161. - Ronel, Joram/Holl-Hussong, Michael/Claas Lahmann (2018): Von der Hysterie zur F45.0 - Geschichte, Konzepte, Epidemiologie und Diagnostik. In: Psychotherapie im Dialog 3 (9. Jg.), S. 207 – 216. - Vitecek, Daniel (2023): Die Geschichte der niederösterreichischen Psychiatrie von 1784 bis 1870. **Internetquellen:** - https://pubmed.ncbi.nlm.nih.gov/17691194/ - https://www.linkedin.com/in/linus-paul-frederic-guenther-478007281/ - https://www.stsg.de/cms/pirna/startseite - https://www.dimdi.de/static/de/klassifikationen/icd/icd-10-who/kode-suche/htmlamtl2019/block-f30-f39.htm - https://www.neurologen-und-psychiater-im-netz.org/psychiatrie-psychosomatik-psychotherapie/diagnostik/ - https://www.destatis.de/DE/Themen/Gesellschaft-Umwelt/Gesundheit/Krankenhaeuser/Tabellen/krankenhaeuser-fa.html - https://www.destatis.de/DE/Themen/Gesellschaft-Umwelt/Gesundheit/Krankenhaeuser/krankenhaeuser.html - http://www.gedenkstaette-grafeneck.

Starting off on a sad note, LD, Thrill, and Admin try to bring a little fun to you today. We talk about conspiracy theories, movies people hate that you love, and the best monologues in movies. AUDITION FOR IDOL - Lynly.ehrlich@americanidol.com Our social stuff: Patreon.com/rockandrollheaven Twitter: @rockandrolllt Instagram: Rockandrollheavenlt Facebook: Rock and Roll Heaven Pod Our website: https://rockandrollheavenl.wixsite.com/mysite Tick Tok: rockandrollheavenpod Email us! rockandrollheavenlt@gmail.com Check out the other awesome Pantheon Podcast at www.pantheonpodcasts.com Learn more about your ad choices. Visit megaphone.fm/adchoices

Willkommen in Kookhausen! Kasper und Till sind zurück wie Marty McFly in die Zukunft! Wir sprechen über den besten Surf-Sommer seit 10 Jahren, und welche Lehren du daraus ziehen kannst, damit auch du den besten Sommer deines Lebens erleben kannst. Es geht um Camping-Equipment, notgeile Delfine, Salp (?) und um die Körperflüssigkeiten deines Autos. Dazu natürlich Welle oder Schelle und mehr! Meldet euch gerne zurück via till@kookhausen.de, kasper@kookhausen.de, über Insta bei @kookhausen.podcast oder bei Soul-Surfers.de. Um den Kookhausen-Podcast zu unterstützen, werdet Kookhausen-Local, empfehlt uns weiter und besucht gerne die Seiten unserer Partner: www.rusty.eu (15 % Rabatt mit Code kookhausen15) www.its-on-zinc.com (10 % Rabatt mit Code "kookhausen") www.mokenvision.com (10 % Rabatt mit Code kookhausen10) mightyottersurfboards.com (10 % Rabatt mit Code kookhausen) srface.com (10 % Rabatt mit Code KOOKHAUSEN10) soul-surfers.de summersurf.de surfandfashion.net (10 % Rabatt mit Kook10) Danke fürs Zuhören!

En France, la prévalence de l'obésité a doublé depuis 1997. Selon les derniers chiffres de la Haute autorité de santé, 17% des adultes et 4% des enfants et adolescents de 6 à 17 ans en souffrent aujourd'hui.Une étape importante a été franchie dans sa prise en charge. Deux médicaments, le liraglutide -nom commercial : Saxenda et le sémaglutide ou Wegovy, initialement connus pour soigner le diabète de type 2, se sont révélés capables de réduire le poids des personnes atteintes d'obésité. Un résultat sans précédent. Sans équivalent, ils font les gros titres des journaux dans le monde entier et affolent les réseaux sociaux.Mais d'abord, comment agissent ces nouveaux médicaments amaigrissants ? Sont-ils aussi efficaces qu'on le dit ? Changent-ils à ce point la donne ? Et révolutionnent-ils vraiment le traitement de l'obésité ?Pour répondre à toutes ces questions, Aline Perraudin, directrice de la rédaction de Santé magazine, échange avec la Pr Judith Aron-Wisnewsky, professeure de nutrition, chercheuse et médecin à l'hôpital de la pitié Salpêtrière à Paris. Spécialiste de l'obésité complexe et des maladies cardiovasculaires qui lui sont associées, la Pr Aron-Wisnewsky est présidente du groupement de coordination et concertation des centres spécialisés obésité.CRÉDITSHypercondriaque est un podcast de Santé magazine animé par Aline PerraudinRédaction et réalisation : Nathalie Courret, Nicolas Jean et Aline PerraudinExtraits : FR3 -18.30 - 14/02/23 ; Franceinfo - 01/07/21 ; TF1 - 10/05/23 ; BFMTV -07/03/24 ; Europe1 - 22/12/14Musique : François ClosIdentité graphique : Upian Communication : Suzanne Méthé, Marianne MeynielMise en ligne : 7 juin 2024Enregistrements : 27 mai et 6 juin 2024 Hébergé par Acast. Visitez acast.com/privacy pour plus d'informations.

Entre la fin du XVIe siècle et le traité de Paris, en 1763, qui voit la rétrocession de ces terres au Royaume-Uni, la France administre un vaste territoire canadien, connu sous le nom de Nouvelle-France.Les autorités peinent à peupler ces immenses contrées, qui ont Québec pour capitale. Et les femmes manquent beaucoup plus que les hommes. Au XVIIe siècle, en effet, elles représentaient à peine plus de 6 % de la population.Ce grand déséquilibre entre les sexes menace, à terme, le peuplement du territoire. Il faut donc trouver une solution pour y remédier.Puisqu'il y a très peu de femmes sur place, le seul moyen est d'en faire venir de la métropole. Louis XIV charge donc son ministre Colbert de trouver des femmes prêtes à s'embarquer pour le Nouveau Monde.L'offre pouvait paraître alléchante à des femmes pauvres. En effet, le Roi s'engageait à leur verser une dot et à payer aux candidates un trousseau neuf ainsi que la traversée pour la Nouvelle-France.C'est bien pourquoi, d'ailleurs, on prit l'habitude d'appeler ces femmes les "filles du Roi". Contrairement aux idées reçues, Colbert ne recrute pas des contingents de filles de joie.Cette rumeur a commencé à naître quand le ministre, pour parer au plus pressé, s'adresse à l'hôpital de la Salpêtrière pour trouver des candidates à l'émigration. Si quelques prostituées étaient bien recluses dans cet hospice réservé aux femmes, beaucoup de ses résidentes étaient des femmes sans ressources.C'est notamment parmi ces déshéritées que Colbert recrute les quelque 700 femmes qui, entre 1663 et 1673, rejoindront le Canada. On les choisit jeunes et célibataires. Elles doivent avoir une santé robuste et une vertu éprouvée.Pour s'en assurer, elles devaient présenter un "certificat de bonne conduite", signé par le curé de la paroisse. On note aussi la présence, parmi ces "filles du Roi", de nombreuses orphelines qui, n'ayant plus d'attaches en France, se sentaient plus libres de partir à l'aventure.Arrivées au Canada, elles se marient rapidement. En 10 ans, on enregistre plus de 4 500 naissances. Hébergé par Acast. Visitez acast.com/privacy pour plus d'informations.

Aujourd'hui, le livre qu'on croque, c'est La Louisiane, un roman de Julia Malye.C'est l'histoire d'un évènement historique qui m'était totalement inconnu : le fait que des femmes aient été envoyées en Louisiane en 1720 pour y devenir des épouses dévouées, fertiles et discrètes. Dans ce roman, on suit trois héroïnes, toutes différentes, toutes envoyées depuis l'hôpital de la Salpêtrière à Paris vers les rivages inconnus de la Louisiane, en passant par une traversée de l'Atlantique périlleuse. C'est donc les orphelines, les laissées pour compte par leur famille, celles qui dérangent et qui sont considérées comme folles — et qui sont en âge de procréer (donc à partir de 12-13 ans) — qui vont partir pour aller donner une descendance aux colons français arrivés en Louisiane au début du 18e siècle. C'est un roman choral où nous suivons Geneviève, qui a fait un court séjour en prison pour une raison que je vous tairais aujourd'hui (suspense !), Pétronille, une aristocrate enfermée par ses parents, et Charlotte orpheline élevée à la Salpêtrière. On suit ces trois femmes entre les décès, les naissances, les mariages, les guerres contre les tribus indiennes, et durant leur voyage et leur adaptation difficile à ce Nouveau monde.J'ai aimé ce roman, déjà parce que je ne connaissais pas cette période historique, et que j'ai appris plein de choses ! C'est une histoire si inconnue que même à la Salpêtrière, il n'y a aucune plaque pour commémorer leur départ ou leur existence… Cette histoire nous amène sur les traces des femmes envoyées en Louisiane pour peupler la récente colonie française, et le moins qu'on puisse dire, c'est que leur vie n'est pas de tout repos. Tout est très bien documenté, et c'est normal, car l'autrice a passé 8 ans à se renseigner et faire de longues recherches. Je suis également assez fascinée par le fait que l'autrice a écrit son livre deux fois : une fois en anglais… et une fois en français !En plus, c'est un roman choral, et vous connaissez mon amour pour ce type de roman : ça permet de s'attarder un peu plus longuement sur plusieurs personnages, de voir plusieurs situations différentes, de comprendre un peu mieux chacune des personnages principal. On y voit des femmes se créer une nouvelle vie, envers et contre tout, loin de leur pays natal, et sans avoir beaucoup de marge de manoeuvre. J'ai aimé la lecture ! Ce roman va être adapté en série prochainement, et je suis intriguée et impatiente de voir Geneviève, Charlotte et Pétronille représentées à l'écran.Je vous recommande La Louisiane de Julia Malye si vous cherchez un roman historique avec des héroïnes fémininesJ'ai lu ce livre dans le cadre d'un partenariat non rémunéré avec le Printemps du livre de Grenoble 2024 : ce festival a lieu du 3 au 7 avril 2024 dans les différentes bibliothèques de Grenoble & dans les lieux partenaires. Merci à Julia Malye de nous avoir accordé du temps avec le podcast La Page Sensible. Vous pouvez retrouver son interview passionnante dans l'épisode bonus ou en allant écouter La Page Sensible. Pour vous plonger dans l'ambiance du Printemps du livre, sachez que les hirondelles du Printemps se posent dans diverses structures, bibliothèques, commerces à partir du 19 mars et jusqu'au 3 avril. Le programme est en description. Si vous voulez rencontrer Julia Malye l'autrice du roman dont je vous ai parlé, c'est possible pour une lecture le samedi 6/04 à 17h30 à la Bobine et le dimanche 7/04 à 10h30 pour une table ronde avec Cécile Coulon (j'y serais !) ou à 15h pour une lecture au musée de Grenoble. Et si vous voulez faire dédicacer vos livres et rencontrer les auteurs et autrices en tête à tête, notez dans vos agendas la soirée librairie-dédicace organisée le samedi 6/04 à 19h à la bibliothèque d'étude et du patrimoine de Grenoble.Toutes les informations/le programme et les réseaux sociaux pour le Printemps du livre 2024 à Grenoble sont ici : BlogDécouvre l'épisode "Rencontre-entretien avec Julia Malye", enregistré avec Marion de La Page Sensible (qui a aussi un joli compte Instagram) dans l'épisode 70Bis du podcast ou en allant écouter sur la page de Marion Livre chroniqué : La Louisiane, de Julia Malye, Stock 2024Musique du générique :Credits: Not The King - Ice Tea - Royalty Free Vlog Music — Music By Not The KingMusique d'ambiance pour la lecture :"Waves and Tears - Sad Piano Music with Calm Ocean Waves", Music by Julius H. from PixabayContacts :Pour retrouver tous les liens pour me retrouver/me contacter/me suivre : https://linktr.ee/lacroqueusedelivrespodcastPour info, je suis sur Instagram, mais aussi sur Facebook et tu peux retrouver mon Blog : www.lacroqueusedelivrespodcast.frMail : lacroqueusedelivres@gresille.orgSi vous voulez être invité-e dans le podcast ou me faire un retour, n'hésitez pas !N'hésitez pas à le partager autour de vous pour le faire découvrir ou à le noter sur votre application de podcast préférée.Merci pour votre écoute.A très vite, pour découvrir un nouveau livre à croquer... ou à dévorer !

On se souvient tous de notre 13 novembre 2015. De l'instant précis où nous avons appris que des attaques étaient en cours à Paris et à Saint-Denis. Que reste-t-il de nos souvenirs ? Comment se relever ? Comment juger l'horreur ? Dans ce huitième épisode, vous allez écouter Romain Rousseau. En 2015, il était chirurgien à l'hôpital de la Salpêtrière. Toute la nuit il a réparé les corps des blessés fauchés par les balles des kalachnikovs.

ایدۀ این قسمت از داستان یک گل شکل گرفت. در مستند سیارۀ سبز نشون میده که بعد از 15 سال در نقطه‌ای از یک جنگل آتش‌سوزی اتفاق میوفته و تقریباً تمام گیاهان از بین میرن ولی چهار روز بعد، از زیر خاکستر گلی زیبا سر بر میاره (سوسن نارنجی). بذری که 15 سال زیر خاک خواب بوده و حالا با دود و آتیش بیدار شده و گل میده و تبدیل به تنها منبع شهد برای پرنده‌های اون ناحیه میشه و حیاتی دوباره رو از دل یک نیستی و نابودی ایجاد می‌کنه. بعد از چند ماه طبیعت دوباره خودش رو احیا می‌کنه. سوسن نارنجی به زیر خاک برمی‌گرده تا آتش‌سوزی بعد اتفاق بیوفته. از دلِ ویرانی، درد و رنج چیز تازه‌ای خلق میشه. تو این قسمت سراغ همین موضوع رفتیم. حامی این قسمت: ایرانیکارت راه‌های حمایت از پادکست اورسی:حامی باشPayPalکانال تلگرام اشکان شریعت معرفی کتاب: جهان مردگان آرژانتین، دنیل لودل، ترجمۀ: شهرزاد بیات موحد.محتوا و موسیقی‌های استفاده‌شده: Patrick Watson - Perfect Day (Piano Komorebi Version)Prem Joshua – Dance of KaliAtrium Carceri – GodessAigiri Nandini - Mahishasura Mardini StotramNick Cave & The Bad Seeds - Wild GodAsaf Avidan - Ride to SalpêtrièreDance With Light Pooriya Faraji –Sarband Ensemble - Gymnopedie No. 1Komitas & Sergey KhachatryanSingers of the Art of Living - Shiva Manas PujaArmand Amar - Poêm of the Atoms I Hosted on Acast. See acast.com/privacy for more information.

L'émission 28 Minutes du 29/02/2024 « La Louisiane » : ces femmes que l'Histoire de France a oubliées  « Adolescente je lisais beaucoup Zola, Stendhal, Balzac. Ils ont été des sources précieuses pour écrire mon premier roman. » C'est à 14 ans que Julia Malye écrit son premier ouvrage — « La fiancée de Tocqueville » — un récit d'Histoire romancée à la Jean Teulé. Publié un an plus tard, le livre connaît un certain succès et gagne le Prix des lycéens au Salon du livre du Touquet. À vingt ans, elle part pour l'Oregon et suit un master de création littéraire pendant deux ans. C'est une visite en Louisiane qui lui inspire son futur roman : « Je suis tombée sur l'histoire de ces femmes envoyées [dans cet État] au début du 18e siècle. […] Je n'avais jamais entendu parler de ces dizaines de femmes qui s'étaient portées volontaires pour venir épouser un inconnu à des milliers de kilomètres de chez elles. » Après huit ans de recherches sur ces Françaises de la Salpêtrière envoyées pour peupler la colonie de la Nouvelle-Orléans, Julia Malye publie « La Louisiane » aux éditions Stock. Un roman qu'elle a d'abord écrit en anglais. « Ce qui m'intéressait beaucoup, c'était vraiment de voir dans quelle mesure l'on raconte des choses différentes d'une langue à une autre ». Julia Malye est notre invitée. Comment l'agriculture française est-elle devenue un champ de bataille politique ? Longtemps présentée comme « une et indivisible », les crises successives font aujourd'hui apparaître une agriculture française divisée entre plusieurs modèles. Trois principaux syndicats s'opposent : la FNSEA, qui défend une agriculture productiviste tournée vers l'exportation, la Coordination rurale — très à droite — qui revendique un positionnement « anti-écolo » et la Confédération paysanne qui dénonce le modèle intensif et défend l'environnement. Le dialogue semble d'autant plus compliqué que chacun porte un agenda politique de plus en plus revendiqué. Si la FNSEA reste l'interlocuteur privilégié du gouvernement, la Coordination rurale est aujourd'hui accusée de rouler pour le RN face à une Confédération paysanne qui n'a jamais caché ses sympathies avec les partis de gauche. Pour ou contre l'écologie, le libre-échange ou les méga-bassines… Dans la perspective des élections européennes, comment rassembler des agriculteurs que tout semble diviser ? Est-il encore possible de trouver un consensus ? Nos invités en débattent. Enfin, retrouvez également les chroniques de Xavier Mauduit et Marie Bonnisseau ! 28 Minutes est le magazine d'actualité d'ARTE, présenté par Elisabeth Quin du lundi au jeudi à 20h05. Renaud Dély est aux commandes de l'émission le vendredi et le samedi. Ce podcast est coproduit par KM et ARTE Radio.  Enregistrement : 29 février 2024 - Présentation : Élisabeth Quin - Production : KM, ARTE Radio

Julia Malye "La Louisiane" (Stock)" Pour la première fois depuis trois mois, elles discernent enfin le sable que leur cachait l'eau lors de la traversée de l'Atlantique, ce fond de l'océan qu'elles ont brièvement aperçu ce matin en débarquant de La Baleine. Personne ne leur a expliqué où elles seraient logées ce soir, dans combien de temps elles seraient fiancées. On ne dit pas tout aux femmes. "Paris, 1720. Marguerite Pancatelin, la Supérieure de la Salpêtrière, est mandatée pour sélectionner une centaine de femmes « volontaires » qui seront envoyées en Louisiane afin d'y épouser les colons français. Parmi elles, trois amies improbables : une orpheline de douze ans à la langue bien pendue, une jeune aristocrate désargentée et rejetée par sa famille ainsi qu'une femme condamnée pour avortement. Comme leurs compagnes à bord de La Baleine, Charlotte, Pétronille et Geneviève ignorent tout de ce qui les attend au-delà des mers. Et n'ont pas leur mot à dire sur leur avenir. Ces étrangères réunies par le destin devront braver l'adversité – maladie, guerre, patriarcat –, traverser une vie faite de chagrins d'amour, de naissances et de deuils, de cruauté et de plaisirs inattendus. Et d'une amitié forgée dans le feu.Un roman d'une profondeur et d'une émotion saisissantes, qui nous transporte au cœur d'une terre impitoyable, aux côtés d'héroïnes animées d'une extraordinaire soif d'amour et de vie.Musique: Queen “Cool Cat” et Beach House “Silver Soul”

Paris, 1894. Qui est Fanni qui prétend s'être laissée enfermer volontairement à l'Hôpital de la Salpêtrière ? Cherchant sa mère parmi la multitude des femmes accusées de folie, Fanni découvre une réalité de l'asile toute autre que ce qu'elle imaginait, ainsi que l'amitié inattendue de compagnes d'infortune. Le dernier grand […] The post Le bal des Captives tourne en rond first appeared on Radio Vostok.

Paris, 1894. Qui est Fanni qui prétend s'être laissée enfermer volontairement à l'Hôpital de la Salpêtrière ? Cherchant sa mère parmi la multitude des femmes accusées de folie, Fanni découvre une réalité de l'asile toute autre que ce qu'elle imaginait, ainsi que l'amitié inattendue de compagnes d'infortune. Le dernier grand […] The post Le bal des Captives tourne en rond first appeared on Radio Vostok.

« Captives » est un long-métrage d'Arnaud des Pallières, qui bénéficie du talent d'interprétation de Mélanie Thierry dans le rôle principal, celui de Fanni. Nous sommes à Paris en 1894. Fanni a un comportement mystérieux. Elle se laisse volontairement enfermer à l'Hôpital de la Salpêtrière. En fait, on apprend que l'héroïne est à la recherche de sa mère, parmi une multitude de femmes considérées comme folles. Elle va aussi nouer des liens inattendus avec ses compagnes d'infortune. Mais sa démarche va s'avérer périlleuse. Seul l'événement mondain du Bal des Folles pourrait la sauver. Copyright Cécile Burban / Prélude

On se souvient tous de notre 13 novembre 2015. De l'instant précis où nous avons appris que des attaques étaient en cours à Paris et à Saint-Denis. Que reste-t-il de nos souvenirs ? Comment se relever ? Comment juger l'horreur ? Dans ce huitième épisode, vous allez écouter Romain Rousseau. En 2015, il était chirurgien à l'hôpital de la Salpêtrière. Toute la nuit il a réparé les corps des blessés fauchés par les balles des kalachnikovs. "Les Voix du crime" vous propose d'écouter "Mémoires du 13 novembre", un podcast diffusé pour la première fois en septembre 2021, à l'occasion de l'ouverture du procès des attentats du 13 novembre 2015.

Le 10 mai 2021, dans l'après-midi, Michel Fourniret est conduit en urgence à l'hôpital de la Salpêtrière à Paris, où il meurt quelques heures plus tard. A 79 ans, « l'ogre des Ardennes », condamné à la perpétuité incompressible pour les meurtres et les viols de sept jeunes femmes entre 1987 et 2001, emporte avec lui de nombreux secrets, suspecté par la justice dans plusieurs autres affaires. Depuis sa mort, tous les regards se portent désormais sur son ex-épouse et complice, Monique Olivier. Aujourd'hui âgée de 74 ans, celle que l'on désigne comme « la gardienne des secrets » a été condamnée à la réclusion à perpétuité pour sa complicité dans cinq meurtres et un viol en réunion commis par son ex-mari Michel Fourniret. Son procès devant la cour d'assises des Hauts-de-Seine à Nanterre pour sa complicité dans l'enlèvement d'Estelle Mouzin ainsi que dans le meurtre de Marie-Angèle Domèce et de Joanna Parrish, s'ouvrira le 28 novembre prochain. L'occasion pour elle, peut-être, de livrer les derniers secrets de Michel Fourniret.Pour Code source, Vincent Gautronneau, journaliste au service police-justice du Parisien, retrace le parcours criminel de Monique Olivier. Ecoutez Code source sur toutes les plateformes audio : Apple Podcast (iPhone, iPad), Google Podcast (Android), Amazon Music, Podcast Addict ou Castbox, Deezer, Spotify.Crédits. Direction de la rédaction : Pierre Chausse - Rédacteur en chef : Jules Lavie - Reporter : Ambre Rosala - Production : Raphaël Pueyo, Barbara Gouy et Thibault Lambert - Réalisation et mixage : Julien Montcouquiol - Musiques : François Clos, Audio Network - Archives : INA, France Info. Hébergé par Acast. Visitez acast.com/privacy pour plus d'informations.

Friends! This week we are on deadline and/or under a terrifying pile of ungraded papers, so we're giving folks a chance to enter the wild world of The Standard Edition with this freshly unlocked episode that tackles Freud's earliest work, his personal and professional anxieties, and the complicated disorder(s) he and his contemporaries called hysteria. (Please join us on Patreon if you like it!) And for our Patreon supporters, a lot more Fliess is coming soon in the next installments of the SE, plus Wild Analysis on settler colonialism and Thanksgiving…Unlocked Patreon episode. Support Ordinary Unhappiness on Patreon to get access to all the exclusive episodes. patreon.com/OrdinaryUnhappinessIn this second episode of The Standard Edition, we finally start doing the reading, tackling the first three texts in Volume I of the Standard Edition: “Report on My Studies in Paris and Berlin,” (1886), “Preface to the Translation of Charcot's Lectures on the Diseases of the Nervous System” (1886) and “Observation of a Severe Case of Hemi-Anesthesia in A Hysterical Male” (1886). We do some ground-clearing about the history of medicine and the various disciplines in which the young Freud sought recognition and met with frustration; sketch out Freud's biography and the world into which he was born and came of age; and discuss the figure of Jean-Martin Charcot and Freud's time studying hysteria with him at the Salpêtrière in Paris. We pay close attention to an early case study of a male hysteric and how it prefigures some of Freud's later case studies. Along the way, we also talk about Freud's history with eels, anti-Semitism, cocaine, hypnosis, and his lifelong habit of making best friends and breaking up with them. We offer a handful of sources we're consulting, whether for general edification or for reference for anyone who might be embarking on this project alongside us. Have you noticed that Freud is back? Got questions about psychoanalysis? Or maybe you've traversed the fantasy and lived to tell the tale? Leave us a voicemail! 484 775-0107 A podcast about psychoanalysis, politics, pop culture, and the ways we suffer now. New episodes on Saturdays. Follow us on social media: Linktree: https://linktr.ee/OrdinaryUnhappiness Twitter: @UnhappinessPod Instagram: @OrdinaryUnhappiness Patreon: patreon.com/OrdinaryUnhappiness Theme song: Formal Chicken - Gnossienne No. 1 https://open.spotify.com/album/2MIIYnbyLqriV3vrpUTxxO Provided by Fruits Music

Emma and Christy use Eugène Grasset's lithograph Morphinomaniac (1897) as a starting point to talk about artistic depictions of morphine and historical opioid addiction, as well as decadence and degeneration in fin-de-siècle Parisian society. In this episode, we cover vampires, hypodermic syringes, Orientalism and Japonisme, 'dangerous' women, masturbation, pleasure, and sex work, true crime waxworks, and gendered consumption — of women, goods, and drugs. CLICK HERE TO VIEW THE IMAGES WE DISCUSS, as well as complete show notes, references, and suggestions for further reading. MEDIA DISCUSSED Eugène Grasset, Morphinomaniac (1897) Photographs of a ‘hysterical' woman yawning at the Salpetrière from Nouvelle Iconographie de la Salpêtrière (c. 1888-1918) Eugène Grasset, Inquiétude (1897) Aubrey Beardsley, cover illustrations for The Yellow Book, An Illustrated Quarterly (1894) Henri de Toulouse-Lautrec, Divan Japonais poster (1892-93) Bernini, detail from Rape of Proserpina (1621-22) Edvard Munch, Vampire II (Vampyr II) (1895) Walter Sickert, Reclining Nude (Le lit de cuivre) (c. 1906) Examples of Parisian wax work: Death of Marat at the Musée Grévin (photograph taken 1959) Albert Joseph Pénot, La Femme Chauve-Souris ('The Bat-Woman') (c. 1890) Luis Ricardo Falero, Vision of Faust (1878) Eugène Grasset, Vitrioleuse (The Acid Thrower) (1894) Katsushika Hokusai, The Waterfall Where Yoshitsune Washed His Horse at Yoshino in Yamato Province(c. 1832) Jules Cheret, Vin Mariani (c. 1896-1900) Jean Bernard Restout, Morpheus (Sleep) (c. 1771) Pablo Picasso, Waiting (Margot) (1901) Pablo Picasso, Morphinomanes (1900) Paul-Albert Besnard, Morphine Addicts (Morphinomanes) (1887) CREDITS This season of ‘Drawing Blood' was funded in part by the Association for Art History. Follow our Twitter @drawingblood_ ‘Drawing Blood' cover art © Emma Merkling All audio and content © Emma Merkling and Christy Slobogin Intro music: ‘There Will Be Blood' by Kim Petras, © BunHead Records 2019. We're still trying to get hold of permissions for this song - Kim Petras text us back!!

Cet été, nous avons sélectionné les meilleurs épisodes des "Fabuleux Destins" . (Re)découvrez les épisodes qui ont fait battre le cœur des auditeurs et auditrices ! Un lieu surprenant Dans certains couloirs de l'hôpital de la Salpêtrière, résonne encore l'ombre d'une époque où la folie était considérée comme le fardeau exclusif des femmes. Nous sommes au 19eme siècle, les femmes peuvent être enfermées pour des raisons absurdes. Comme par exemple, refuser le devoir conjugal, avoir recourt à l'avortement ou avoir de mauvaises fréquentations. À la Salpêtrière, la majorité des femmes malades sont issues de classes sociales précaires. Pour la plupart, elles ont vécu des violences physiques et psychologiques. C'est dans ce contexte, qu'un homme va y trouver sa renommée... Pour découvrir d'autres récits passionnants, cliquez ci-dessous : Harry Styles, le phénomène planétaire de la pop culture Les Reptiliens, la théorie du complot la plus répandue dans le monde Le crash du vol Malaysia Airlines 370, le drame aérien le plus mystérieux de notre époque Un podcast Bababam Originals Ecriture : Clémence Setti Voix : Andréa Brusque Production : Bababam (montage Célia Brondeau, Antoine Berry Roger) Date de première diffusion : 16 mars 2023 Learn more about your ad choices. Visit megaphone.fm/adchoices

Hàng thế kỷ qua, dòng sông Seine uốn khúc chảy qua trung tâm thủ đô Pháp, như một chứng nhân lịch sử trước bao thăng trầm của kinh đô ánh sáng. Dòng sông biểu tượng của Paris vốn bị ô nhiễm từ nhiều năm qua, đã bị cấm bơi lội từ năm 1923, thì nay với đòn bẩy từ Thế vận hội mùa hè 2024, có thể được làm sạch, cho phép tổ chức thi bơi, hứa hẹn là một địa điểm làm mát cho cư dân thủ đô những ngày hè nóng bức.  Sông Seine được tạp chí Time ví như dòng sông lãng mạn nhất thế giới, đã truyền cảm hứng cho bao văn hào Pháp. Victor Hugo từng gắn sông Seine với sự lãng quên hay cái chết trong tác phẩm Những Người Khốn Khổ. Đối với thi hào Pháp Jacques Prévert, (Chanson de la Seine) thì sông Seine như là một cô gái, “thật may mắn, không có muộn phiền, êm đềm chảy ngày cũng như đêm, …ở giữa những điều huyền bí cũng như những điều khốn khổ của Paris”. Vào thế kỷ XIX, không khó để bắt gặp người dân người mặc đồ bơi, bơi lội, phơi nắng ở ven bờ sông của thủ đô. Nhưng tình trạng ô nhiễm sông Seine lên đến mức báo động do ô nhiễm từ nước thải sinh hoạt, nước thải nhà máy hoặc nước mưa… cũng như xác chết của các động vật. Vào năm 1889, báo Figaro đã đưa ra một danh sách các chất thải tìm thấy tại sông Seine chỉ trong vài tháng : hơn 2000 xác chó, 977 xác mèo, 10 xác cừu, 8 xác dê… , nhưng điều này cũng không ngăn cản sự thành lập của các câu lạc bộ bơi lội trên sông Seine ở thủ đô. Chính vì vậy mà từ năm 1923, thành phố Paris đã cấm tắm, bơi ở sông Seine, nếu ai cố ý vi phạm sẽ phải nộp phạt 15 euro.  Nhưng lệnh cấm này sẽ được dỡ bỏ nhờ Thế vận hội mùa hè diễn ra tại Paris, từ ngày 26/07-11/08/2024. Bắt nguồn từ vùng Bourgogne rồi chảy ra biển (qua thành phố Le Havre), sông Seine là con sông dài thứ hai nước Pháp (774km). Dự án làm sạch đoạn sông chảy qua thủ đô đã được thị trưởng thành phố Paris Anne Hildago khởi xướng từ năm 2016, cho phép người dân cũng như khách du lịch tại paris có thêm lựa chọn để làm mát vào những ngày hè nóng bức, khi nhiệt độ được dự báo có thể lên đến 50 độ C vào những thập kỷ tới. Hồi tháng Ba, cách lễ khai mạc Thế vận hội 500 ngày, trên mạng xã hội X (Twitter), tổng thống Pháp Emmanuel Macron nhấn mạnh đến mục tiêu của Pháp : khiến sông Seine và sông Marne có thể bơi được (một cách an toàn), nhờ vào khoản đầu tư lên đến 1,4 tỷ euro, một nửa là từ ngân sách Nhà nước. Đính kèm bài đăng là ảnh bìa của tạp chí Time, với tựa “Cứu lấy sông Seine” (Save the Seine), nguyên thủ Pháp khẳng định rằng đây sẽ là một trong những “di sản đẹp nhất mà Thế vận hội 2024 để lại cho nước Pháp”.    Sông Seine cũng được lựa chọn làm nơi tổ chức lễ khai mạc Thế vận hội, trên đoạn sông dài 6 km, từ cầu Austerlitz đến cầu Iéna. Nếu lời hứa làm sạch nước sông Seine được thực hiện, những người đầu tiên có thể bơi trên sông là các vận động viên tham gia Thế vận hội trong các cuộc thi như bơi tự do 10 km, thi bơi trong ba bộ môn phối hợp (triahlon) và para triathlon. Lộ trình bơi bắt đầu từ cầu Alexandre III đên cầu Pont de l'Alma.  Thế vận hội Paris 2024 là đòn bẩy về tài chínhTrên đài truyền hình France 24, phó thị trưởng thành phố Paris phụ trách thể thao, Thế vận hội Olympic và Paraolympic, ông Pierre Rabadan cho rằng kế hoạch làm sạch nước sông kéo dài 8 -10 năm, không phải là một ý tưởng điên rồ, mà là một cao vọng : “Tất cả đều được thực hiện trong một khuôn khổ rất chặt chẽ. Chúng tôi phải đáp ứng các yêu cầu của châu Âu về chất lượng nước. Vào mùa hè năm 2022, chất lượng nước sông đã cải thiện đáng kể và có thể cho phép tổ chức các cuộc thi bơi. Yêu cầu của Liên đoàn thể thao quốc tế sẽ được đáp ứng. Nếu không có Thế vận hội, chúng tôi cũng có thể hoàn thành mục tiêu làm sạch nước sông Seine, nhưng có lẽ là phải trong 30 hay 40 năm nữa, nhưng nhờ Thế vận hội mà chúng tôi có thể thực hiện điều này trong 8 đến 10 năm. Chúng tôi có hẹn với người dân Paris vào năm 2025, mọi người có thể mặc đồ bơi và khám phá nước sông Seine”.   Hôm 10/07 vừa qua, Thành phố Paris đã công bố các địa điểm có thể tắm được ở sông Seine, dự trù mở cửa vào năm 2025, một năm sau kỳ Thế vận hội Paris 2024. Cư dân thủ đô có thể đến tắm ở sông Seine tại Bras Marie (quận 4) đối diện Île Saint-Louis ở hữu ngạn, tại Bras de Grenelle (quận 15) và tại Bercy (quận 12) đối diện thư viện quốc gia của Pháp. Nước tại các khu vực này sẽ được kiểm tra hàng ngày và được phân cách với làn dành cho tàu thuyền qua lại.  Đây không phải là lần đầu tiên giới chính khách Pháp hứa hẹn làm sạch sông Seine để có thể bơi mà không gặp rủi ro về sức khoẻ. Hồi tháng 05/1990, cựu tổng thống Pháp Jacques Chirac, lúc đó là thị trưởng Thành phố Paris, đã tuyên bố sẽ làm sạch sông Seine, và chính ông sẽ tự mình bơi ở sông để kiểm chứng. Thế nhưng lời hứa này đã không được thực hiện. Rủi ro đối với sức khỏeLàm sao để khử trùng, làm sạch sông Seine là một thách thức lớn. Dòng sông từng là nơi ném xác người (trong chiến tranh tôn giáo) ném xác động vật vào thế kỷ XVI, thì nay là nơi “ném” xe đạp, ti vi, chai nhựa…hay các loại rác thải khác. Khoảng 360 tấn chất thải được thu gom từ sông Seine mỗi năm. Nguồn gây ô nhiễm nhất cho dòng sông ngày nay là hàng tấn nước thải sinh hoạt hay công nghiệp. Theo tạp chí Time, năm 2022 1,9 triệu mét khối nước thải chưa qua xử lý đã đổ ra sông Seine. Quan chức Pháp cho rằng điều này là cần thiết để tránh làm “quá tải mạng lưới nước thải của Paris và làm ngập thành phố khi mưa lớn ập đến.”  Ngoài ra, theo Le Figaro, nhiều đường ống nước thải từ khoảng 23.000 hộ gia đình ở thượng nguồn Paris, không được kết nối với các nhà máy xử lý mà lại đổ trực tiếp vào sông Marne hoặc sông Seine.   Điều đáng quan ngại nhất khi bơi ở sông Seine, là rủi ro với sức khoẻ khi nước sông có chứa các loại vi khuẩn như Escherichia coli và enterococci đường ruột, có nguồn gốc từ phân người hoặc động vật. Theo France 24, Hồi đầu tháng 05/2023, kết quả xét nghiệm cho thấy trong 100 ml nước sông, có khoảng 100 đến 1000 khuẩn Escherichia coli. Để có thể bơi được trên sông Seine thì tỷ lệ này phải là dưới 900 Escherichia coli trên 100 ml khi tiến hành xét nghiệm liên tục, trong 90% thời gian của năm. Nhà thuỷ văn học, Jean-Marie Mouchel, nhà nghiên cứu tại đại học Sorbonne nhận định trên France 24 : “Các loại vi khuẩn có nguồn gốc từ phân, lúc nào cũng hiện diện ở sông Seine. Nhưng không phải vì có các loại vi khuẩn này mà chúng ta không thể bơi được ở sông Seine. Vấn đề là ở tiêu chuẩn và mật độ hiện diện của chúng, và mật độ đó cần phải thấp, khiến cho rủi ro nhiễm các vi khuẩn này cũng thấp theo”Xử lý ô nhiễm từ thượng nguồnTheo Le Monde, ngoài xử lý các đường ống nước thải đổ trực tiếp ra sông Seine, dự án làm sạch dòng sông của thủ đô cũng tính đến việc cải thiện hiệu quả của các nhà máy xử lý nước thải ở thượng nguồn Paris. Mặc dù các nhà máy này đã có thể xử lý, loại bỏ 99 % các loại vi khuẩn có nguồn gốc từ phân, nhưng vẫn xả hàng triệu mét khối nước chứa nhiều vi khuẩn ra sông. Với khoản đầu tư trong khuôn khổ Thế vận hội, trạm xử lý nước thải Noisy-le-Grand và Marne Aval, đã áp dụng công nghệ khử trùng bằng tia cực tím vào năm 2022. Giám đốc phụ trách về đổi mới của Dịch vụ vệ sinh công cộng Ile-de-France (SIAAP - Service public d'assainissement francilien),trên France 24, ông Vincent Rocher giải thích : “Hai nhà máy xử lý nước thải này có vị trí chiến lược nằm ở cửa ngõ vào Paris. Chúng tôi đã nỗ lực rất nhiều để hai nhà máy này có thể đạt được hiệu suất cao. Chúng tôi sẽ thấy chất lượng của nước sông chảy vào thành phố Paris được cải thiện.” Thành phố Paris cũng trang bị cho các cảng dọc trên sông Seine ở thủ đô, một mạng lưới vệ sinh, để ngăn 260 tàu (péniche) neo đậu tại đây, xả nước thải ra sông. Hiện một nửa số tàu này đã được kết nối vào mạng lưới này, số còn lại, dự trù, sẽ phải hoàn thành trước Thế vận hội.  Ngoài ra, một trong những rủi ro lớn khiến nước sông bị ô nhiễm là từ nước mưa. Trong trường hợp bão lớn, nước mưa có thể chảy vào đường ống nước thải, làm quá tải và gây tràn cống. Hiện nay, một công trình khổng lồ, xây dựng bể chứa nước mưa ở sau nhà ga Austerlitz đang được gấp rút hoàn thành. Quản lý công trình này, Samuel Colin-Canivez, cho biết “để tránh nước mưa làm ngập đường phố, sông Seine được được sử dụng như một lối thoát an toàn để thoát nước. Trong tương lai, với công trình này, nước mưa sẽ bị chặn lại nhờ một đường ống dài 600 mét, với đường kính 2 mét rưỡi. Nước chảy qua đường ống sẽ đổ vào bể chứa rộng 50 mét và sâu 30 mét. Thể tích tương đương với 20 bể bơi Olympic, ẩn sâu dưới lòng đất. Sau khi trời mưa trong vòng dưới 24 giờ, chúng tôi sẽ bơm nước từ bể chứa, đổ ra cống ở đại lộ l'Hôpital. Nước sẽ chảy về nhà máy xử lý nước thải trước khi thải ra môi trường. Công trình này sẽ giảm các sự cố khiến lượng nước thải đổ ra sông Seime, cải thiện hệ sinh thái và chất lượng nước về mặt vệ sinh”. Ngoài công trình này, một dự án xây dựng bể chứa nước mưa khổng lồ khác cũng đang được tiến hành gần lối vào bệnh viện Salpêtrière. Mặc dù có nhiều dự án, công trình được thực hiện để làm sạch nước sông Seine, song, theo Le Monde, trong trường hợp mưa lớn, phải mất ít nhất hai ngày thì chất lượng nước mới quay trở lại mức “đạt tiêu chuẩn”, có thể bơi được. Do đó, các cuộc thi bơi của Olympic có nguy cơ bị hoãn nếu xảy ra mưa lớn. Các cơn bão lớn, khiến nước sông dâng cao, có thể cho thấy rõ mức độ ô nhiễm qua hàng tấn rác thải nhựa, trôi dại trên sông. Hiện tại, 20 đập nổi “barrage flottant”, đã được lắp đặt trên sông Seine và sông Marne, vào mùa hè này, các tàu thu gom rác cũng sẽ hoạt động mạnh hơn. Tuy nhiên, vẫn còn rào cản tâm lý có thể khiến nhiều người quan ngại có nên đi tắm sông hay không. Nếu như vào thế kỷ 20, có những người e ngại đi tắm sông Seine vì không muốn lộ cơ thể, thì nay rào cản này đến từ rủi ro đối với sức khoẻ.   

My guest this week is best-selling, award-winning author Jennifer Cody Epstein. Jennifer's latest novel "The Madwomen of Paris" is a work of historical fiction set in Paris's 19th-century Salpêtrière, a notorious women's asylum. Here, women are treated for hysteria by world famous Doctor Jean-Martine Charcot, often publicly, on stage and under hypnosis. When Josephine is admitted to the hospital in a fit of rage and amnesia, she is placed under the care of Laure, a former-resident-turned-attendant. They quickly form a strong connection that forces them to separate truth from fiction in all facets of their lives… which may include murder.In our conversation, Jennifer shares what drew her to this particular place, period and story. She discusses the power of hysteria, hypnosis, and of beauty, how she played with perception, and why there are no heroes in her book. Then, she treats us to a reading from "The Madwomen of Paris.”https://jennifercodyepstein.comhttps://twitter.com/JennCodyEpsteinhttps://www.instagram.com/jennepstein/ Join our Book Club: patreon.com/parisundergroundradioFind Us OnlineWebsite: https://www.parisundergroundradio.com/storytimeinparisFacebook: https://www.facebook.com/parisundergroundradioInstagram: https://www.instagram.com/parisundergroundradio/ CreditsHost and Producer: Jennifer Geraghty. Facebook, Instagram, and Twitter: @jennyphoria; Website: http://jennyphoria.comMusic CreditsHip Hop Rap Instrumental (Crying Over You) by christophermorrow https://soundcloud.com/chris-morrow-3​ Creative Commons — Attribution 3.0 Unported— CC BY 3.0 Free Download / Stream: http://bit.ly/2AHA5G9​ Music promoted by Audio Library https://youtu.be/hiYs5z4xdBU​ About UsSince well before Victor Hugo looked up at Notre Dame and thought, "Huh... what if a hunchback lived in there?" authors have been inspired by Paris. The Storytime in Paris podcast will help keep this tradition alive with short interviews and readings from your favorite contemporary authors with a French connection. Every episode will feature five questions, asked by you, our authors' biggest fans, and answered live on air. Then, our authors will treat us to a reading of an excerpt from their book. Who knows? Maybe you'll even be inspired to write your own Great French Novel. Happy listening!

Subscribe to get access to the full episode, the episode reading list, and all premium episodes! www.patreon.com/OrdinaryUnhappinessIn this second episode of The Standard Edition, we finally start doing the reading, tackling the first three texts in Volume I of the Standard Edition: “Report on My Studies in Paris and Berlin,” (1886), “Preface to the Translation of Charcot's Lectures on the Diseases of the Nervous System” (1886) and “Observation of a Severe Case of Hemi-Anesthesia in A Hysterical Male” (1886). We do some ground-clearing about the history of medicine and the various disciplines in which the young Freud sought recognition and met with frustration; sketch out Freud's biography and the world into which he was born and came of age; and discuss the figure of Jean-Martin Charcot and Freud's time studying hysteria with him at the Salpêtrière in Paris. We pay close attention to an early case study of a male hysteric and how it prefigures some of Freud's later case studies. Along the way, we also talk about Freud's history with eels, anti-Semitism, cocaine, hypnosis, and his lifelong habit of making best friends and breaking up with them. We offer a handful of sources we're consulting, whether for general edification or for reference for anyone who might be embarking on this project alongside us.Have you noticed that Freud is back? Got questions about psychoanalysis? Or maybe you've traversed the fantasy and lived to tell the tale? Leave us a voicemail! 484 775-0107 A podcast about psychoanalysis, politics, pop culture, and the ways we suffer now. New episodes on Saturdays. Follow us on social media: Linktree: https://linktr.ee/OrdinaryUnhappiness Twitter: @UnhappinessPod Instagram: @OrdinaryUnhappiness Patreon: patreon.com/OrdinaryUnhappiness Theme song: Formal Chicken - Gnossienne No. 1 https://open.spotify.com/album/2MIIYnbyLqriV3vrpUTxxO Provided by Fruits Music

durée : 01:59:13 - Les Matins du samedi - par : Quentin Lafay - . - invités : Louise Auvitu journaliste; Jacqueline Wendland professeure dans le domaine de la petite enfance à l'Université Paris-Cité et psychologue clinicienne à la Salpêtrière ; Véronique Escames Auxiliaire de puériculture, co-secrétaire du Syndicat national des professionnel.le.s de la petite enfance

Although on the outside we may all seem very similar, we experience the world in wildly different ways. A fascinating illustration of this is 'Tickertape Synesthesia' in which people experience live subtitles of every sound around them appearing in front of their very eyes. Joining Jonathan to discuss the condition is Professor Laurent Cohen, Professor of Neurology at the Salpêtrière Hospital in Paris. Also joining Jonathan is Professor Séamus Davis, Professor of Quantum Physics at University College Cork. Together they discuss quantum computing and its future. Dr. Susan Kelleher, Assistant Professor of Polymer Chemistry at DCU & Dr. Fergus McAuliffe, Communications and Public Engagement Manager at iCRAG, the Science Foundation Ireland Research Centre for Applied Geosciences also join us to go through the top science stories from throughout the week for Newsround.

Dans cet épisode, Andréa Brusque vous parle d'un événement mondain insolite du 19eme siècle. À cette époque, le Tout-Paris aimait faire la fête, la capitale regorge de bals, d'opérettes et autres festivités. L'évènement dont je vais vous parler se déroule à la mi-carême, dans un lieu particulier : un hôpital. Cette soirée a inspiré l'écrivaine Victoria Mas et plus récemment la réalisatrice Mélanie Laurent. Son nom : le Bal des folles. Un bal insolite qui a ameuté tout Paris, découvrez sa True Story. Un lieu surprenant Dans certains couloirs de l'hôpital de la Salpêtrière, résonne encore l'ombre d'une époque où la folie était considérée comme le fardeau exclusif des femmes. Nous sommes au 19eme siècle, les femmes peuvent être enfermées pour des raisons absurdes. Comme par exemple, refuser le devoir conjugal, avoir recourt à l'avortement ou avoir de mauvaises fréquentations. À la Salpêtrière, la majorité des femmes malades sont issues de classes sociales précaires. Pour la plupart, elles ont vécu des violences physiques et psychologiques. C'est dans ce contexte, qu'un homme va y trouver sa renommée... Pour découvrir d'autres récits passionnants, cliquez ci-dessous : Le “manifeste des 343”, l'appel fondateur des femmes pour le droit à l'avortement Les Suffragettes, celles qui se sont sacrifiées pour le droit de vote L'affaire « personne », le jour où les femmes ont obtenu l'un des droits les plus fondamentaux Un podcast Bababam Originals Ecriture : Clémence Setti Voix : Andréa Brusque Production : Bababam (montage Célia Brondeau, Antoine Berry Roger) Learn more about your ad choices. Visit megaphone.fm/adchoices

Demain, découvrez le dernier épisode spécial de notre série autour de la Journée des droits des femmes.  Jeudi, retrouvez une nouvelle True Story, cette fois-ci sur une sombre expérience de médecine qui a eu lieu à la Salpêtrière… Et tout au long de la semaine comme toujours, (ré)écoutez nos meilleures True Story et nos meilleures Love Story, sur toutes les plateformes audio !  Learn more about your ad choices. Visit megaphone.fm/adchoices

On Episode 24 of the Stroke Alert Podcast, host Dr. Negar Asdaghi highlights two articles from the January 2023 issue of Stroke: “Covert Brain Infarction as a Risk Factor for Stroke Recurrence in Patients With Atrial Fibrillation” and “Subarachnoid Hemorrhage During Pregnancy and Puerperium.” She also interviews Dr. Georgios Tsivgoulis about his article “Clinical, Neuroimaging, and Genetic Markers in Cerebral Amyloid Angiopathy-Related Inflammation: A Systematic Review and Meta-Analysis.” Dr. Negar Asdaghi:         Let's start with some questions. 1) When during pregnancy is an intracranial aneurysm at the highest risk of rupture? 2) What does the presence of covert brain infarcts mean in the setting of atrial fibrillation? 3) And, finally, how is the inflammatory form of cerebral amyloid angiopathy different from the classic CAA form, and why is it important to differentiate between the two? We'll be answering these questions and much more in today's podcast. We're covering the latest in cerebrovascular disorders, and this is the best in Stroke. Stay with us. Welcome back to another issue of the Stroke Alert Podcast. My name is Negar Asdaghi. I'm an Associate Professor of Neurology at the University of Miami Miller School of Medicine and your host for the monthly Stroke Alert Podcast. Together with my co-editors, Drs. Nastajjia Krementz and Eric Goldstein, here's our article selection for the month of January. Symptomatic intracerebral hemorrhage is a feared complication of reperfusion therapies in acute stroke, so there's a lot of interest in looking for predictors of development of this complication, especially when you're making decisions for pursuing endovascular therapy. For many years now, we've known about some of these predictors, such as presence of a large infarct core and high blood glucose levels. But in the recent years, other radiographic markers of tissue viability, such as a poor collateral status and unfavorable venous outflow profile, have been shown to be predictors of post-reperfusion hemorrhagic transformation. In this issue of the journal, we learn about another imaging marker that can potentially predict parenchymal hemorrhage occurrence post-endovascular therapy, which is high hypoperfusion intensity ratio, or HIR, as measured by perfusion imaging. What is HIR? It's a long name for a simple ratio that can easily be measured by dividing the volume of tissue with Tmax delay of over 10 seconds to the volume of tissue with Tmax delays of over 6 seconds. Simply put, Tmax 10 divided by Tmax 6. These volumes, as you know, are typically provided to us by almost all post-processing perfusion softwares, and so this ratio can be easily calculated in the acute setting. So, in this paper led by Dr. Tobias Faizy from University Medical Center in Hamburg and colleagues, we learned that higher hypoperfusion intensity ratios are strongly associated with parenchymal hemorrhage occurrence after endovascular therapy. So, in summary, HIR, that is a quantitative ratio, can be used as a marker to risk stratify patients that are undergoing endovascular therapy in terms of helping us predicting the risk of development of intracerebral hemorrhage after reperfusion therapies. In a separate study in this issue of the journal, we read a very interesting paper titled "Anti-Epileptic Drug Target Perturbation and Intracranial Aneurysm Risk." How are intracranial aneurysms even related to anti-epileptic drugs? Well, first of all, it's been known for a long time based on genome-wide association studies that there are multiple common genes that are associated with increased risk of intracranial aneurysm development. Now, some of the largest genetic studies to date have shown pleiotropy between genetic causes of development of intracranial aneurysms and genes encoding targets for anti-epileptic drugs. Now that's a fascinating finding because finding commonalities between these genes may help find new treatment targets for intracranial aneurysms. So, in this paper in this issue of the journal, the investigators from the University Medical Center in Utrecht found an association in the expression of anti-epileptic drug target gene CNNM2 and intracranial aneurysm risk. They found that certain anti-epileptic drugs, such as phenytoin, valproic acid, and carbamazepine, that are expected to lower CNNM2 levels in the blood may subsequently lead to a lower risk of development of intracranial aneurysms. And, of course, a reasonable follow-up study to this would be to investigate whether persons exposed to these anti-epileptic drugs have indeed a lower risk of unruptured intracranial aneurysms and subarachnoid hemorrhage, and how variation in CNNM2 expression can lead to development of aneurysms. Bottom line, CNNM2 may be a relevant drug target for treatment of cerebral aneurysms. As always, I encourage you to review these papers in detail in addition to listening to our podcast today. My guest on the podcast today is the Chairman of Neurology at the University of Athens, Dr. Georgios Tsivgoulis. He joins me all the way from Greece to talk about cerebral amyloid angiopathy-related inflammation, or CAA-ri. He's a remarkable researcher, and I can say with absolute confidence that we cannot find a better summary of this very tough topic elsewhere. He ends the interview with an intriguing account of the early description of dementia in Greek mythology. But first, with these two articles. What are covert brain infarcts, or CBIs? Are these the John Wick or the James Bond of the stroke world? After all, they operate undercover. They're ominous and attack without warning. That's probably why they're also called silent infarcts. Now, whatever we call them, we need to know how prevalent they are and what does their presence actually mean. Let's dive into this topic. For at least two centuries, if not longer, we've known about covert brain infarcts. Early description of these lesions is credited to Amédée Dechambre, a medical intern at Salpêtrière Hospital in Paris who noted that there are strokes that can cause symptoms like hemiplegia, but also strokes that are asymptomatic, or so he thought at the time. In the modern times, while we agree with our pathology forefathers that CBIs are different from symptomatic strokes, we also know that they are not entirely asymptomatic. The symptoms can be subtle and tend to sneak up on the patient, but what is clear is that amassing of covert brain infarcts results in an overall decline in cerebrovascular reserve of the brain. With the advent of neuroimaging, we now know that CBIs are age-dependent and prevalent, seen in almost 10 to 30% of even healthy adults, but much more prevalent in those with vascular risk factors, and they can be caused by nearly the entire spectrum of neurovascular disease, including large vessel, small vessel disorders, cardioembolism, and others. Now, how do these covert infarcts catch up in those with atrial fibrillation? Neuroimaging studies have shown that patients with A-fib, especially those untreated, have a higher percentage of embolic-appearing CBIs, and conversely, those with embolic formed pattern of CBIs are more likely to have undiagnosed A-fib. So the question is, what's the significance of CBI in those with confirmed A-fib? In this issue of the journal, Dr. Do Yeon Kim from Seoul National University and colleagues help us answer this question using the EAST-AF, which stands for East Asian Ischemic Stroke Patients With Atrial Fibrillation Study. So, the paper included over 1300 patients with A-fib and first-ever stroke without a prior history of TIA or stroke. And then they categorized these patients into those who had evidence of CBI on neuroimaging and those who didn't. So, what did they find? Forty-two percent of patients with A-fib and first-ever stroke had evidence of covert brain infarcts on neuroimaging. Let's think about it for a moment. These patients presented with what was thought to be their first-ever stroke, not knowing they already had some in their brain. Now, what makes things really worse is that over a quarter of these subjects had more than just one covert infarct. Not surprisingly, those with CBI tended to be older, had higher blood pressure, and had worse white matter hyperintensity burden. This is kind of expected and also not expected was the fact that most of these covert infarcts were actually embolic in pattern. Over 60% of them were embolic. Another 14% of cases had combined embolic and non-embolic-appearing CBIs. Now, overall, the one-year incidence of ischemic stroke and all-cause mortality was higher in those that had CBIs at baseline. When they started looking at the specific patterns of CBIs, those embolic-appearing CBIs had a threefold higher risk of recurrent ischemic stroke, whereas those with non-embolic-appearing covert infarcts had oddly a higher all-cause mortality rate but not recurrent ischemic stroke. And finally, just briefly, the authors noted that the addition of CBIs to the classic CHA2DS2-VASc score didn't meaningfully otherwise statistically improve the scoring metrics, so they left it at that. So, the take-home message is that 42% of A-fib patients presenting with first-ever stroke actually had prior strokes without even knowing based on this study. And most of these strokes were embolic-appearing, and these covert brain infarcts can be used as predictors of future clinical strokes in this population. Strokes should be the last thing to worry about when we think of pregnancy. In the United States, around 30 in 100,000 women, unfortunately, experienced a stroke during pregnancy, and between 6 to 8 in 100,000 deliveries are complicated by subarachnoid hemorrhage. What's the most common cause of pregnancy-associated subarachnoid hemorrhage? In the general population, close to 80% of subarachnoid hemorrhage cases are aneurysmal. Is this true for the pregnant population as well? And importantly, what's the contemporary incidence trend, risk factors, and outcomes of pregnancy-related subarachnoid hemorrhage? In this issue of the journal, Dr. Korhonen and Petra [Ijäs] and their colleagues from the Departments of Neurology and Obstetrics and Gynecology at Helsinki University Hospital will give us the answers to some of these questions through a nationwide population-based study in Finland. So, they looked at over one and a half million pregnant women who gave birth during a 30-year time period between 1987 to 2016. Subarachnoid hemorrhage was identified through appropriate ICD codes and then further adjudicated based on confirmatory information, including neuroimaging and data from lumbar puncture. A total of 57 cases of pregnancy-related subarachnoid hemorrhage was identified in this paper. The mean age of women was 33, ranging from 23 to 45, and the clinical presentation was typical for subarachnoid hemorrhage, including thunderclap headache and mild neurological symptoms. So, what did they find? So, first off, in terms of general observations, the overall incidence rate of pregnancy-related subarachnoid hemorrhage in this study was 3 over 100,000 deliveries. This is almost half the incidence rate reported from the nationwide registries in the United States. Seventy-seven percent of pregnancy-related subarachnoid hemorrhage cases were aneurysmal, so very similar to the general population. The other 23% were non-aneurysmal cases, but it's important to note that 40% of those non-aneurysmal cases also had vascular etiologies, so etiologies such as moyamoya syndrome, postpartum angiopathy, AVM, to name a few. Like non-pregnant patients with subarachnoid hemorrhage, the aneurysmal cases were sicker patients in general. They had a lower GCS at presentation, higher Hunt and Hess scores, and required more ICU admissions. The next finding is very important because it actually shows that development of subarachnoid hemorrhage during pregnancy significantly impacted obstetrical care. A total of 66% of women with subarachnoid hemorrhage during pregnancy ended up having a C-section and a high percentage of these cesarean sections were actually elective. This is in contrast with subarachnoid hemorrhages in the postpartum period where 67% of women had spontaneous vaginal deliveries. The other important finding of the paper was really highlighting the differences between pregnancy-related aneurysmal versus non-aneurysmal subarachnoid hemorrhages. We already talked about how, in general, aneurysmal cases had more severe neurological presentations, so, not surprisingly, they also had worse outcomes with a mortality rate of 16% for the aneurysmal subarachnoid hemorrhage cases, and only 68% of women with pregnancy-related aneurysmal subarachnoid hemorrhage reached a favorable outcome, which was defined in this study as modified Rankin Scale of 0 to 2. Other important differences included the fact that the incidence of aneurysmal subarachnoid hemorrhage increased towards the end of pregnancy and was highest in the third trimester. This ties in with the findings from prior studies all indicating that rupture of an aneurysm is most common in the third trimester. By contrast, the incidence of non-aneurysmal subarachnoid hemorrhage peaked in the second trimester in this study. And finally, in terms of risk factors, first let's talk about age. The incidence rate of pregnancy-associated subarachnoid hemorrhage increased with age of the mother. So, in this study, there were no cases noted amongst women aged below 20 years of age to an incident rate of 12 per 100,000 deliveries among women aged 40 years or over. So that's a fourfold increase from the overall incidence rate of pregnancy-related subarachnoid hemorrhage, and very important point that we learned from this paper. Apart from age, smoking beyond 12 weeks of gestation and hypertension were also independent factors associated with pregnancy-related subarachnoid hemorrhage. So, overall, hypertension, smoking are bad and are significant risk factors for pregnancy-related subarachnoid hemorrhage. And if we have to remember just one thing from this paper, let it be this one: The rupture of an aneurysm is most common in the third trimester of pregnancy. Cerebral amyloid angiopathy, or CAA, is an important cause of intracranial hemorrhage and refers to deposition of β-amyloid fibrils in the wall of the small- and medium-sized cerebral blood vessels, mostly involving cortical and leptomeningeal arteries. It is believed that the deposition of β-amyloid results in architectural disruption of the blood vessels, which then leads to perivascular leakage. That's the pathophysiological mechanism behind the development of cerebral microbleeds. And this process, of course, can cause frank vascular rupture resulting in cortical intracerebral hemorrhage or development of high-convexity subarachnoid hemorrhages. It is important to note that varying amounts of perivascular inflammation, that is inflammation surrounding β-amyloid-laden blood vessels, may be present in some CAA cases, rendering them the designation of inflammation-related CAA. However, frank vasculitic destruction of the vessel wall, such as what is found in amyloid-β-related angiitis, or ABRA, and primary angiitis of the central nervous system, is usually absent in most CAA-related inflammation cases. How these entities are best defined, diagnosed, and treated is subject of intense research. In this issue of the journal, in the study titled "Clinical, Neuroimaging, and Genetic Markers in CAA-Related Inflammation," Dr. Georgios Tsivgoulis and colleagues take us through a systematic review and meta-analysis of published studies of patients with CAA-related inflammation. I am joined today by Dr. Tsivgoulis himself to discuss this paper. He's a Professor of Neurology and Chairman of the Second Department of Neurology at the University of Athens School of Medicine. Dr. Tsivgoulis is the residency program director and the director of cerebrovascular fellowship program with extensive research and expertise in the field of stroke. Good morning, Georgios, and welcome to our podcast. Dr. Georgios Tsivgoulis: Good morning, Negar. I'm delighted to be here and delighted to present our findings, on behalf of all our co-authors. Dr. Negar Asdaghi:         Thank you very much for being here and congrats again on the paper. So, Georgios, let's start with this interest that's going on with using clinical and radiographic features to make the diagnosis of CAA-related inflammation in contrast to moving ahead and performing brain biopsy. Can you please start us off with a brief review of the newly proposed clinico-radiographic criteria for this condition, please? Dr. Georgios Tsivgoulis: Yes. As you mentioned, Negar, CAA-ri is a distinct, however, rare subset of cerebral amyloid angiopathy. Firstly, Greenberg and the Boston group published in Neurology in 2007 a paper highlighting that a diagnosis of a probable CAA-ri patient could be made on the basis of characteristic clinical and neuroimaging findings without requiring a biopsy. Following this observation, Chung and colleagues in 2010, in a seminal paper in JNNP, proposed the first diagnostic criteria for probable and definite CAA-ri. For the definite diagnosis, besides the typical clinical presentation with headache, encephalopathy, focal neurological signs and seizures, and the characteristic neuroimaging findings with T2 or FLAIR hyperintense asymmetric white matter lesions complicated with microbleeds and leptomeningeal or parenchymal gadolinium enhancement, and histopathological confirmation with amyloid deposition within cortical leptomeningeal vessels associated with perivascular, transmural or intramural inflammation was also required. The latest criteria developed in 2015 by Auriel and colleagues that were published in JAMA Neurology using a validation study modified the current criteria for the diagnosis of CAA-ri. In this paper, the author supported the use of empirical immunosuppressive therapy, avoiding brain biopsy, for patients meeting the criteria proposed for probable CAA-ri. They suggested that a brain biopsy should be considered in empirically treated patients who failed to respond to corticosteroid therapy within three weeks. The criteria by Auriel and colleagues are widely applicable in everyday clinical practice, and we also use this criteria for the inclusion of studies in our current meta-analysis. I would like to highlight for our audience that the latest criteria for CAA-ri were published in 2015 by Auriel and colleagues. However, these are different for the criteria for cerebral amyloid angiopathy than the latest criteria were published in 2022 in Lancet Neurology, OK? Dr. Negar Asdaghi:         Georgios, that was a great start for this interview. You had mentioned a lot of information here. I just want to highlight what you just said. So, we are using for this meta-analysis, the latest criteria in CAA-related inflammation published in JAMA by Auriel and colleagues. That's slightly different than, we're not referring to the 2022 criteria of cerebral amyloid angiopathy. It's an important distinction. We're going to talk about this a little more as we go through the interview, but I want to come back to your current paper and start from there. Can you please tell us about the importance of this paper, why doing a meta-analysis was important in your view, and tell us a little bit about the studies that were included in your paper? Dr. Georgios Tsivgoulis: Yes, thank you for that question. CAA-ri is an increasingly recognized entity since the recent diagnostic criteria by Auriel and colleagues published in 2015. In collaboration with the greater availability of the high-resolution MR, we can have now a reliable non-invasive diagnosis of possible or probable CAA-ri, avoiding the risk of brain biopsy. However, I need to highlight that the early diagnosis remains a great challenge for the clinicians and neurologists. Searching the literature, we observe that there is scarce data regarding the prevalence of the distinct clinical, neuroimaging, and genetic markers among patients diagnosed with CAA-ri. We believe that pooling all this information in the current meta-analysis would be very helpful for every clinician, increasing a comprehensive understanding of this rare cerebrovascular disorder. Consequently, we conducted this meta-analysis including 21 studies that recruited a total of 378 patients with CAA-ri. Our study involved only 4 prospective and 17 retrospective hospital-based cohorts of patients diagnosed with CAA-ri based on autopsy or biopsy or on the recent Auriel diagnostic criteria that do not require autopsy or biopsy. Due to limited data in the literature regarding this entity, we had to include only small cohort studies with at least five patients in our meta-analysis. We excluded case reports and case series with less than five patients. This is, by far, the largest available sample of CAA-ri patients in the literature. Dr. Negar Asdaghi:         OK, great. So, let me just recap this, more so for myself. So, we have 21 studies, and you excluded studies that included less than 5 patients. So, practically speaking, case reports. Dr. Georgios Tsivgoulis: Yes, and single-case reports. Dr. Negar Asdaghi:         Yes. And practically speaking, of the total number of patients that are included in this meta-analysis, you have 378 cases, and basically the diagnosis of CAA-related inflammation was either based on the newly proposed criteria or based on biopsy-confirmed or autopsy cases. Dr. Georgios Tsivgoulis: Which is the standard criteria. Dr. Negar Asdaghi:         So, now, I'm dying to ask you about these clinical and radiographic characteristics of patients with CAA-related inflammation in this meta-analysis. Dr. Georgios Tsivgoulis: The mean age of patients in the included studies was approximately 72 years old, and there was no obvious gender predominance. Fifty-two percent of the patients were of female sex. In our study, 70% of the included patients presented with cognitive decline, which was the most common neurological manifestation, while 50% of the total sample had focal neurological signs and 54% encephalopathy presentation. Symptoms such as headache and seizures were less common, 37 and 31% respectively. With regard to the radiological findings, hyperintense T2 FLAIR white matter lesions were very, very common in 98% of our patients, and they were also complicated with lobar cerebral microbleeds, with a prevalence of 96%, and these two were, by far, the most prevalent neuroimaging findings, that white matter hyperintensities coupled with a cerebral microbleed. The pooled prevalence rates of gadolinium-enhanced lesions was 54%, and also the prevalence of cortical superficial siderosis was 51%, which is also very high in this cohort of patients with CAA-ri. Dr. Negar Asdaghi:         OK. So many of the features Georgios said, you mentioned, from presence of white matter hyperintense lesions on T2 FLAIR to presence of cortical microbleeds or superficial siderosis, these features are also seen in patients with cerebral amyloid angiopathy. What are some of the important differentiating features between the two conditions? Dr. Georgios Tsivgoulis: Yes, this is an excellent clinical question. First of all, the lower age threshold for CAA-ri is 40 years old, whereas in cerebral amyloid angiopathy, the lower age threshold is 50 years. So, patients who are younger than 50 years can be diagnosed with CAA-ri, but they cannot be diagnosed with CAA. Another issue is that comparing the result of this meta-analysis with another recent meta-analysis focusing on CAA, on cerebral amyloid angiopathy, that our international multi-collaborative group published in Stroke in 2002, we also evaluated the presence of clinical phenotypes and radiological markers among patients with cerebral amyloid angiopathy. We have documented that transient focal neurological episodes are much more common in patients with cerebral amyloid angiopathy in contrast to patients with CAA-ri. These episodes, which are called TFNEs, transient focal neurological episodes, are attributed to cortical subarachnoid hemorrhage or cortical superficial siderosis. So, I think this is another important clinical distinction. The most important, however, differentiating features between the two entities are neuroimaging markers, in specific, in particular, T2 FLAIR hyperintense unifocal or multifocal lesions with mass effect. These are the most prevalent neuroimaging features among patients with CAA-ri, but they're very seldomly described in patients with cerebral amyloid angiopathy, in patients with CAA. Another characteristic neuroimaging finding very indicative of the inflammation is the leptomeningeal or parenchymal gadolinium enhancement. This finding has been very rarely described in patients with non-inflammatory cerebral amyloid angiopathy. So, the clinical distinction is not so solid. However, the neuroimaging distinction would provide us with very strong information that can help us differentiate these two conditions. Dr. Negar Asdaghi:         Excellent points, I have to say, golden points, not just excellent points. I'm going to try to recap this and see if I understood it correctly. So, for our listeners, we have two conditions that potentially have many common points. One is the cerebral amyloid angiopathy, and the second one, which is obviously the subject of this interview, is cerebral amyloid angiopathy-related inflammation. The most important differentiating factors between the two are actually the neuroimaging features, as Georgios mentioned. So, the first feature that was mentioned is presence of T2 FLAIR hyperintense lesions. Some of them are large and have actually mass effects. This feature is rarely seen in patients with CAA, and it's an important radiographic factor that is seen in patients with CAA-related inflammation. The second distinguishing feature was leptomeningeal enhancement, again, rarely seen in non-inflammatory CAA, but was seen in a significant proportion of patients with CAA-related inflammation. These were the neuroimaging features. You also mentioned two other factors. The median age of CAA-related inflammation was lower than CAA. That can be helpful. And also the entity of transient focal neurological episodes, or TFNE, is rarely seen in inflammatory cases of CAA, whereas it is described in cases with cerebral amyloid angiopathy and mostly related to development of either cortical subarachnoid hemorrhage or cortical superficial siderosis. I think I got this all, correct? Dr. Georgios Tsivgoulis: Excellent. Dr. Negar Asdaghi:         All right, so let's come now to the genetics of CAA. The apolipoprotein E gene is associated with the presence of amyloid angiopathy and development of lobar intracerebral hemorrhage, and we've learned about this in cases with cerebral amyloid angiopathy. Is there an association with ApoE, and did you find anything in this meta-analysis? Dr. Georgios Tsivgoulis: Another very exciting question. In 2007, there was a first report that the apolipoprotein ε4 homozygosity may be considered a risk factor for CAA-ri, and there was a strong correlation reporting a high prevalence of 77% of this apolipoprotein ε4 alleles among patients with CAA-ri. To justify this correlation, the hypothesis was that an underlying pathogenic mechanism, which increases the amyloid-β deposition and has a pro-inflammatory effect, may be suspected as the cause of this disorder. The largest, however, prospective cohort of CAA-ri patients conducted by Antolini and colleagues and was published in 2021 in Neurology, reported a much lower prevalence of apolipoprotein ε4 carriers accounting for 37%, 23% heterozygotes and 14% homozygotes. So, we also documented a pool prevalence of apolipoprotein ε4 homozygosity of 34%. So, we did not confirm the initial finding of 77%. However, in our meta-analysis, the homozygosity was 34%, and we need to have a cautious interpretation of these results because data is limited, and we need larger future population-based studies and in larger cohorts to evaluate the prevalence rate of these specific genetic markers. So, we can confirm an association between apolipoprotein ε4 homozygosity, however not as strong as originally reported in 2007. Dr. Negar Asdaghi:         OK. So, Georgios, thank you. And again, very important factor to keep in mind for our clinicians listening in. Unfortunately, based on what you mentioned, we don't have yet a genetic marker to, for sure, tell us if we're dealing with CAA-related inflammation, yes or no, as you mentioned. Just to recap, earlier on, there was studies to suggest a very strong association between apolipoprotein ε4 homozygosity and CAA-related inflammation. But later on, this was not confirmed by subsequent studies, and in your meta-analysis, you found 34% ApoE ε4 homozygosity amongst patients with CAA-related inflammation and could not confirm that original high association. OK, so with all of that, it's a lot of information. I have to go to the next question regarding controversies involving the levels of Aβ40, Aβ42, and P-tau proteins in CSF in the setting of CAA-related inflammation. Can you please tell us more about these biomarkers? Dr. Georgios Tsivgoulis: Yes. The overlap of Alzheimer's disease and CAA can be attributed to the coexistence of some degree of cerebrovascular amyloid deposition and amyloid plaque pathology, which is very common. And, of course, the evaluation of amyloid and tau proteins in CSF is of high significance for the prognosis and the evolution of CAA patients. In our previous review, we have summarized the literature and noticed that CSF concentrations of Aβ40 and, secondarily, Aβ42 were much lower in patients with cerebral amyloid angiopathy compared with Alzheimer's disease. Total tau and phospho-tau CSF levels were comparable to healthy controls in CAA and lower than patients with Alzheimer's disease. Moving now to CAA-ri, there were scarce data about these biomarkers amongst CAA-ri patients. The majority of the relevant studies have found relatively low levels of Aβ42 and Aβ40 in the CSF and high levels of P-tau. In the present meta-analysis, the pooled means of biomarker levels were based on the findings of only two studies with heterogeneity, and these limit substantially the validity of our observations. However, they confirm the previous reports indicating, as I said before, but I would like to repeat, low levels of Aβ42 and Aβ40 in the CSF and high levels of P-tau. Dr. Negar Asdaghi:         Perfect. So, thank you, Georgios. I'm going to recap what you said. So, we're talking about CSF biomarkers, and first what you mentioned is going back to the original studies concentrated on using these biomarkers as ways of differentiating between cerebral amyloid angiopathy and Alzheimer's disease. And very briefly, to recap what you said, in general, the levels of Aβ40 and, secondarily, Aβ42 was found to be much lower than the Alzheimer's levels in patients with CAA. Now coming to the inflammatory form of CAA, what you mentioned and what you found in this meta-analysis, practically speaking, confirmed that the levels of Aβ40 and Aβ42 in CSF are low and the levels of P-tau are high in this condition as well. So, one thing I want to ask as a secondary question to that is, that it sounds like these biomarkers are more or less similar in CAA and CAA -related inflammation, not that different. Is that correct? Dr. Georgios Tsivgoulis: It's absolutely correct. And I would also like to highlight a major limitation of the meta-analysis that we had available data from only two studies to pool the mean of these CSF biomarker levels. So, these results need to be acknowledged with caution, and we would love to repeat our meta-analysis after the publication of more studies and prospective cohorts measuring the CSF biomarkers in patients with CAA-ri. Dr. Negar Asdaghi:         OK. So, again, important to note, as you mentioned, that there's heterogeneity in data because of just paucity of information on this, but as we stand today, the biomarkers won't really help us in terms of differentiating between the two conditions that are CAA or CAA-related inflammation. And so, I think I've learned a lot from this interview myself, but I think we have to just talk briefly about the available therapies for CAA-related inflammation. Dr. Georgios Tsivgoulis: Yes. In our meta-analysis, we sought to summarize the available information regarding different therapeutic strategies and outcomes among CAA-ri patients. Our results supported our clinical experience indicating that corticosteroids represent the first-line treatment in these patients' outlook. Steroids have been associated with clinical and radiological improvement of the primary disease episode and decreased risk of subsequent relapses in patients with CAA-ri. Additional immunosuppressive therapies, including cyclophosphamide, mycophenolate mofetil, azathioprine, IVIG, or rituximab, have been also reported as adjunct therapies in selected cases with a more severe course of the disease. However, this is another limitation that needs to be acknowledged. That data regarding the treatment and the outcomes are limited and heterogeneous, which prevented us from drawing robust conclusions using a meta-analytical approach. And we believe that we need future cohort studies with prospective data validation in order to generate a proposal for a therapeutic algorithm management in these cases. Dr. Negar Asdaghi:         Thank you, Georgios. So, we have a condition that is now being more and more recognized. We now have criteria based on clinical and radiographic presentation features of patients that might help us with this diagnosis to differentiate it from cerebral amyloid angiopathy. And in terms of therapies, the idea is that the most studied drug is really just first-line therapy, that's corticosteroids. And then there's positive data regarding use of all other forms of immunosuppression, including, as you mentioned, cyclophosphamide, rituximab, and oral agents such as mycophenolate mofetil or azathioprine. We have limited information about those, but I want to highlight something you actually mentioned earlier on in the interview, which is the field is moving towards making these diagnoses based on clinical features and radiographic features that you had highlighted and actually giving patients immunosuppression early on and only move on to a biopsy if the patient had failed these therapies for a period of time, which you mentioned three weeks. So, I think it's important for us as clinicians to keep this evolving criterion and recommendations in mind. And before we end, I want to ask you a hypothetical question, Georgios. In your opinion, what's an ideal randomized trial for CAA-related inflammation in the future? Dr. Georgios Tsivgoulis: I think before going to the randomized, the ideal randomized trial for CAA-ri, and designing this trial, we need much more information regarding the underlying pathophysiological mechanisms. There are many unanswered questions. What is the diagnostic value of CSF biomarkers such as amyloid, we discussed earlier, and tau protein? And, of course, what is the value of CSF and the amyloid-β autoantibodies, if there is any? What is the value of genetic markers such as apolipoprotein E genotype and a correlation with the co-existing inflammation in CAA-ri? However, I don't want to defer this question. So, a typical answer would be that with regard to the ideal patients, we would want a young patient without comorbidities after the first manifestation of CAA-ri who has shown a good clinical and radiological response to corticosteroids in order to define the best second-line therapy. However, before answering all these questions in a clinical trial, if we can, I think that we need to understand the CSF and genetic biomarkers in order to uncover mechanisms regarding pathophysiology that can help us to design more targeted clinical trials studying novel disease-modifying treatments. Dr. Negar Asdaghi:         Thank you. Dr. Georgios, it's been a pleasure having you on the podcast, and I can say we've learned a lot. We look forward to having you back here and talk about that hypothetical randomized trial, and I'm sure one day hopefully will happen in our lifetime. Thank you for being here. Dr. Georgios Tsivgoulis: Thank you. Thank you for having me. It was a pleasure. Dr. Negar Asdaghi:         Thank you. Homer, the legendary Greek poet, described a case of dementia in his seminal work, The Odyssey, in the late eighth century before Christ. He described the cognitive decline of Odysseus's father, King Laertes. The detailed account of the king's mental decline, loss of short-term memory with retention of long-term memory combined with his depression and despair over the loss of his son, is dramatically accurate for a nearly 3,000-year-old description of dementia. Before I ended the interview, I had to use this opportunity to ask Georgios about lessons learned from ancient Greeks and this seemingly timeless disease. Dr. Georgios Tsivgoulis: Thank you for this question. King Laertes was indeed Odysseus's father, and it's a great paradigm describing dementia. However, the ancient history of dementia may be separated according to the Greek philosopher Posidonius in two periods. The first period is called dementia appearing due to old age, which is called in Greek, eros. And the second one is dementia appearing in other ages and mainly due to other reasons, called morosis. Posidonius of Rhodes was a Greek stoic philosopher of the second first century BC who strongly believed and suggested that morosis, which is that dementia appearing in younger ages due to other disorders, should be treated immediately after its onset. So, if I would like to end this podcast, I would just suggest that CAA-ri could be classified as morosis according to Posidonius. And what we could learn is that the early diagnosis is essential since the prompt initiation of corticosteroids should not be unreasonably delayed. Dr. Negar Asdaghi:         And this concludes our podcast for the January 2023 issue of Stroke. Please be sure to check this month's table of contents for the full list of publications, including a series of Focused Updates on post-stroke neurological recovery, from management of post-stroke attention deficit, neglect and apraxia to post-stroke memory decline. And with this, we end the start of our 2023 podcast series. Like all new things, a new beginning can come with new directions, and sometimes a new direction is all that we need. After all, as the legend has it, it was a direction of that falling apple back in the year 1666 that gave Isaac Newton the idea of the universal law of gravitation. Now, Isaac Newton has, without a doubt, given science some of its biggest discoveries in mathematics, physics, and astronomy. But most may not know that Newton had a pretty rough start in life. A January-born premature baby, he was thought not to survive the first few days of life. Newton had a difficult childhood, and at the age of 16, he was pulled out of school by his family and forced to become a farmer, a job he didn't like and he was miserably bad at. So, as we start a new year, let's remember that even the smartest people are not good at everything, and it does take time to find one's passion in life. Now, while things may not always be clear, what is clear is that a great way to find that center of gravity is, as always, staying alert with Stroke Alert. This podcast is produced by Wolters Kluwer and supported by the editorial team of Stroke. Our Stroke Alert podcast and production staff includes Danielle Cross, Eric Goldstein, Nastajjia Krementz, Ishara Ratnayaka, Erinn Cain, Rebecca Seastrong, and Negar Asdaghi. This program is copyright of the American Heart Association, 2023. The opinions expressed by speakers in this podcast are their own and not necessarily those of the editors or of the American Heart Association. For more, visit AHAjournals.org.

Florence Millot, psychopédagogue et psychologue. Spécialisée dans le suivi des enfants et adolescents, et auteur du livre « Se libérer de son enfant intérieur » (Ed. De Vinci). Florence Millot, Psychologue pour enfants et adolescents PARIS Nina Bataille, Coach professionnelle certifiée, et spécialisée en neuroscience. Nina Bataille – Coach professionnelle, Auteur, Conférencière (ninabataille-coaching.com) Son dernier livre « Les 50 règles d'or de l'autorité tranquille » (Ed. Larousse). Et paraîtra le 16 février prochain :  « J'arrête de courir après le temps » (Ed. ESF Sciences Humaines) dont un chapitre est consacré à la gestion des écrans.   Christian Flavigny, pédopsychiatre, psychanalyste, ancien directeur du Département de Psychanalyse de l'Enfant à l'hôpital de la Salpêtrière. Dernier livre paru « Aider les enfants transgenres » (Ed. Tequi).Christian Flavigny – Psychanalyse – Site officiel – Accueil

Au programme :  - Invité : Renaud Piarroux, pédiatre et biologiste, spécialiste des Maladies infectieuses, chef du service de Parasitologie à l'Hôpital de la Salpêtrière à Paris.   Renaud Piarroux a inauguré une collection de podcasts sur France Culture pour mieux comprendre l'histoire des virus, des épidémies et des pandémies d'hier et d'aujourd'hui.  - La chronique Les dessous de l'infox de Gregory Genevrier  L'OMS cible privilégiée des infox autour de la Covid-19.   - La chronique d'AFP-factuel par Marin Lefevre :  Non, le Qatar n'a pas refusé l'entrée sur son territoire à l'avion de l'équipe allemande de football à cause d'un «logo gay». 

Por Miguel Ángel Alcarria.Suscríbete a CesarVidal.TV y escucha este audio antes que nadie y sin publicidad: https://www.cesarvidal.tv

Por Miguel Ángel Alcarria.Suscríbete a CesarVidal.TV y escucha este audio antes que nadie y sin publicidad: https://www.cesarvidal.tv

Welcome to PsychEd, the psychiatry podcast for medical learners, by medical learners. This episode delves into the history of psychiatry with Dr David Castle, the inaugural Scientific Director of the Centre for Complex Interventions at the Centre for Addictions and Mental Health and a Professor in the Department of Psychiatry at the University of Toronto. Prior to migrating to Canada in 2021, he spent 15 years as a Professor of Psychiatry at St Vincent's Hospital and the University of Melbourne in Australia.   The learning objectives for this episode are as follows:   By the end of this episode, you should be able to… Gain an appreciation for the historical context of the field of psychiatry Understand how illness categories and treatments have been shaped by this history  Compare and contrast how various past societies viewed and conceptualized mental illness Apply lessons learned from historical practices to appraise current approaches   Guest: Dr David Castle   Hosts: Dr Alex Raben (Staff Psychiatrist), Gaurav Sharma (PGY4), Nikhita Singhal (PGY4), Andreea Chiorean (CC4)   Audio editing by: Dr Alex Raben   Show notes by: Dr Nikhita Singhal   Interview Content:   1:45 - Learning Objectives   3:25 - Ancient Times   14:42 - Middles Ages    23:56 - Renaissance to Enlightenment   34:55 - 19th-20th Centuries   47:55 - 20th-21st Centuries   1:00:48 - Final Thoughts   Resources: Shrinks: The Untold Story of Psychiatry (Jeffrey A Lieberman)   References: The Emotional Foundations of Personality: A Neurobiological and Evolutionary Approach (Kenneth L Davis, Jaak Panksepp) Illustration of Bedlam (William Hogarth) Pinel, médecin en chef de la Salpêtrière en 1795 (Tony Robert-Fleury) Castle, D., Bassett, D., King, J., & Gleason, A. (2013). A primer of clinical psychiatry. Elsevier Health Sciences. de Leon J. DSM-5 and the research domain criteria: 100 years after Jaspers' General psychopathology. Am J Psychiatry. 2014 May;171(5):492-4. https://doi.org/10.1176/appi.ajp.2013.13091218 Eisenberg L. Mindlessness and brainlessness in psychiatry. Br J Psychiatry. 1986 May;148:497-508. https://doi.org/10.1192/bjp.148.5.497 Engel GL. The need for a new medical model: a challenge for biomedicine. Science. 1977 Apr 8;196(4286):129-36. https://doi.org/10.1126/science.847460 Kendell R, Jablensky A. Distinguishing between the validity and utility of psychiatric diagnoses. Am J Psychiatry. 2003 Jan;160(1):4-12. https://doi.org/10.1176/appi.ajp.160.1.4 Robins E, Guze SB. Establishment of diagnostic validity in psychiatric illness: its application to schizophrenia. Am J Psychiatry. 1970 Jan;126(7):983-7. https://doi.org/10.1176/ajp.126.7.983 Rosenhan DL. On being sane in insane places. Science. 1973 Jan 19;179(4070):250-8. https://doi.org/10.1126/science.179.4070.250 Scheff TJ. The labelling theory of mental illness. Am Sociol Rev. 1974 Jun;39(3):444-52. https://doi.org/10.2307/2094300 Szasz T. The myth of mental illness: 50 years later. The Psychiatrist. Cambridge University Press; 2011;35(5):179–182. https://doi.org/10.1192/pb.bp.110.031310   CPA Note: The views expressed in this podcast do not necessarily reflect those of the Canadian Psychiatric Association.   For more PsychEd, follow us on Twitter (@psychedpodcast), Facebook (PsychEd Podcast), and Instagram (@psyched.podcast). You can provide feedback by email at psychedpodcast@gmail.com. For more information, visit our website at psychedpodcast.org.

In the 115th episode of The Strength Connection Podcast, Mike and our special guests, Creators of Let's Eliminate Your Limits Program, Brain Grasso, speaker and author of Mindset Matters Most and Audacity Of Success and Carrie Campbell, a BFF Pro Athlete, Personal Development Coach, and a Clinical Counselor will talk about Join us in this insightful and captivating talk! In this chapter, you will discover:(1:05) Introducing our special guests Creators of Let's Eliminate Your Limits Program, Brain Grasso, speaker and author of Mindset Matters Most and Audacity Of Success and Carrie Campbell, a BFF Pro Athlete, Personal Development Coach, and a Clinical Counselor @eliminateyourlimits(2:35) The intention behind creating ELI(3:45) Carrie's experience with “troubled teens”(4:20) Creating a mindset system(4:50) Interruption vs. intervention model(5:25) “You can interrupt a human beings pain and their experiences younger in age and avoid intervention at the age of 40, 30, 50.” - Carrie Campbell @eliminateyourlimits(5:50) Brian's reason behind creating ELI(6:10) Brian & Carrie's proactive intentions(7:30) The hate for categorization(7:40) Definition of categorization by Brian(8:15) The ranges of normal human experiences and emotions(8:55) The Unstoppable Girls Tribe(9:25) “There is no child or adolescent on the planet, who is going to label themselves without having the information to be labeled.” - Carrie Campbell @eliminateyourlimits(9:45) The reality of opposition in this world(10:15) Reason behind intervention in the school system(10:55) More about ELI(11:15) “You don't change the world with a quick grab.” - Carrie Campbell @eliminateyourlimits(12:50) Hopelessness in teachers today(13:35) Who is responsible?(14:00) Brian's suggestion for bettering the school system(15:20) ELI's backstory(15:50) “The backstory matters.” - Carrie Campbell @eliminateyourlimits(16:00) What's ELI?(16:50) Buying Pitbull tickets for their daughter @pitbull(17:45) Meeting Pitbull @pitbull(18:20) Connecting with Pitbull @pitbull(19:20) The first business order(20:00) Moving to Miami(20:15) The pilot project(20:25) The COVID-pivot(21:25) The initial intention behind ELI(22:10) SALP model(22:50) Reason behind the importance of the backstory(24:30) Invitation exclusive event, February 2023(24:50) Shout out to Alice Cooper @alicecooper(25:20) The launch party, September 2023(27:10) Pitbull's old boombox @pitbull(28:25) The acceptance of failure(29:30) Why didn't Carrie quit?(31:35) The struggles with building this new business(33:50) Step out of it!(35:30) Real-world reward system(37:00) About ELI's gamification(37:45) About ELI's built-in AI(39:05) More about ELI (43:20) “The simplicity is not a lack of consideration.” - Carrie Campbell @eliminateyourlimits(44:20) The real crises (46:35) A gamified journal(48:50) “You don't need a solution right off the bat, you need momentum.” - Michael Kurkowski @mike_strength_connectionWhere to find Brian and Carrie? @eliminateyourlimits

Historiquement Vôtre réunit 3 folles pas si folles : celle qu'on surnommait “Jane la folle” à la Salpétrière où elle a fait un passage remarqué : Jane Avril, passée du Bal des folles de l'hôpital au célèbre Moulin Rouge où la danse lui a permis de continuer à vivre. Puis Jeanne Devidal, “la Folle de Saint-Lunaire” comme on l'appelait dans son village, à cause de son étrange maison faite de bric et de broc, qu'elle a bâtie elle-même, devant les yeux ébahis des gens du pays. Et une folle autoproclamée qui en a fait le titre d'un de ses albums, mais peut-être pas si folle en réalité : la chanteuse Brigitte Fontaine.

Historiquement Vôtre réunit 3 folles pas si folles : celle qu'on surnommait “Jane la folle” à la Salpétrière où elle a fait un passage remarqué : Jane Avril, passée du Bal des folles de l'hôpital au célèbre Moulin Rouge où la danse lui a permis de continuer à vivre. Puis Jeanne Devidal, “la Folle de Saint-Lunaire” comme on l'appelait dans son village, à cause de son étrange maison faite de bric et de broc, qu'elle a bâtie elle-même, devant les yeux ébahis des gens du pays. Et une folle autoproclamée qui en a fait le titre d'un de ses albums, mais peut-être pas si folle en réalité : la chanteuse Brigitte Fontaine.

Full Text of ReadingsFriday of the Twenty-second Week in Ordinary Time Lectionary: 435All podcast readings are produced by the USCCB and are from the Catholic Lectionary, based on the New American Bible and approved for use in the United States _______________________________________The Saint of the day is Blessed John Francis Burt and Companionsclass="content"> Sep 2, 2020 Franciscan Media Image: Massacre à la Salpêtrière, 3 septembre 1792 | anonymous (Unrelated, but similar incident during the French Revolution.) Saints of the Day for September 2 Blessed John Francis Burté and Companions (d. September 2, 1792 and January 21, 1794) Audio file Blessed John Francis Burté's and Companions' Stories These priests were victims of the French Revolution. Though their martyrdom spans a period of several years, they stand together in the Church's memory because they all gave their lives for the same principle. In 1791, the Civil Constitution of the Clergy required all priests to take an oath which amounted to a denial of the faith. Each of these men refused and was executed. John Francis Burté became a Franciscan at 16 and after ordination taught theology to the young friars. Later he was guardian of the large Conventual friary in Paris until he was arrested and held in the convent of the Carmelites. Appolinaris of Posat was born in 1739 in Switzerland. He joined the Capuchins and acquired a reputation as an excellent preacher, confessor, and instructor of clerics. Preparing for his assignment to the East as a missionary, he was in Paris studying Oriental languages when the French Revolution began. Refusing the oath, he was swiftly arrested and detained in the Carmelite convent. Severin Girault, a member of the Third Order Regular, was a chaplain for a group of sisters in Paris. Imprisoned with the others, he was the first to die in the slaughter at the convent. These three plus 182 others—including several bishops and many religious and diocesan priests—were massacred at the Carmelite house in Paris on September 2, 1792. They were beatified in 1926. Born in 1737, John Baptist Triquerie became a Conventual Franciscan. He was the chaplain and confessor of Poor Clare monasteries in three cities before he was arrested for refusing to take the oath. He and 13 diocesan priests were martyred in Laval on January 21, 1794. He was beatified in 1955. Reflection “Liberty, Equality, Fraternity” was the motto of the French Revolution. If individuals have “inalienable rights,” as the Declaration of Independence states, these must come not from the agreement of society—which can be very fragile—but directly from God. Do we believe that? Do we act on it? Saint of the Day, Copyright Franciscan Media

Aujourd'hui à 15 heure, Elisabeth Borne réunira les préfets et les directeurs de l'ARS. Le but de cette réunion : avoir des recommandations pour les semaines à venir à la suite de la hausse de la covid 19. Le point sur ce qu'il pourrait être mis en place. Depuis 3 semaines le nombre de personnes touché par la covid commence à augmenter. En cause, le sous variant BA.5 qui porte le nombre de cas à 60 000 par jour, c'est donc la 7 ième vague en France. Le gouvernement envisage de recourir aux passes sanitaires à l'arrivée dans l'Hexagone, en Corse et en Outre-mer. La ministre de la Culture, Rima Abdul Malak, a expliqué qu'il faut être très vigilant, se laver les mains, reprendre les gestes barrière, se tester à chaque fois qu'on a un doute. La ministre de la Santé, Brigitte Bourguignon, a demandé hier aux Français de remettre le masque dans les transports par civisme. Un « comité de veille et d'anticipation des risques sanitaires » est envisagé. Il s'agira d'une instance devant produire des recommandations en fonction de l'évolution de la situation sanitaire. Et puis une nouvelle dose de rappel de vaccin pourrait être nécessaire. Les personnes concernées recevront une lettre du gouvernement. Il s'agit des personnes de 60 ans et plus ainsi que des personnes immunodéprimées. L'état d'urgence sanitaire et les mesures de sortie de crise prendront fin le 31 juillet prochain. Mais le gouvernement incite à rester vigilants.  Le vaccin produit-il des effets indésirables très rares ? Louis Daufresne : Un peu partout, on entend encore les campagnes en faveur des doses de rappel. Les autorités sont discrètes sur les effets indésirables des injections. Je vous propose d'écouter un témoignage qui illustre la difficulté à pouvoir faire un lien de cause à effet, bien que l'on soit amené à penser qu'il y en a un. Marion, 25 ans, est artiste-plasticienne. Après s'être fait vacciner, sa vue en a pâti lourdement, au point qu'elle a dû se faire hospitaliser à la Salpêtrière. Difficile dans son cas, comme dans beaucoup d'autres, de savoir si le vaccin est la cause directe de son mal. Néanmoins, son cas appartient aux milliers de cas de personnes qui, favorables à la vaccination, ont été conduites à nuancer leur vision des choses, et la formule est bien trouvée, comme elle nous l'explique pour Un Jour une Info :

It's that time again- the time when we all go a little slapnuts!! This week take a trip with the troubled trio where we discuss Our favorite video games, Novelty and Parady song, and True crime!! Our social stuff: Patreon.com/rockandrollheaven Twitter: @rockandrolllt Instagram: Rockandrollheavenlt Facebook: Rock and Roll Heaven Pod Our website: https://rockandrollheavenl.wixsite.com/mysite Tick Tok: rockandrollheavenpod Please email us! rockandrollheavenlt@gmail.com Check out the other awesome Pantheon Podcast at www.pantheonpodcasts.com Learn more about your ad choices. Visit megaphone.fm/adchoices

It's that time again- the time when we all go a little slapnuts!! This week take a trip with the troubled trio where we discuss Our favorite video games, Novelty and Parady song, and True crime!!Our social stuff:Patreon.com/rockandrollheavenTwitter: @rockandrollltInstagram: RockandrollheavenltFacebook: Rock and Roll Heaven PodOur website: https://rockandrollheavenl.wixsite.com/mysiteTick Tok: rockandrollheavenpodPlease email us! rockandrollheavenlt@gmail.comCheck out the other awesome Pantheon Podcast at www.pantheonpodcasts.com

The Washington State Department of Agriculture has begun trapping season on invasive pests, and WSDA's Karla Salp talks about what's being done.

The Washington State Department of Agriculture has begun trapping season on invasive pests, and WSDA's Karla Salp talks about what's being done.

KGMI's Joe Teehan talks to Karla Salp from the Washington State Department of Agriculture about their efforts to trap invasive species.

Enzo Fileno Carabba"Il digiunatore"Ponte alle Graziehttps://www.ponteallegrazie.it/Nato a metà Ottocento a Cesenatico Ponente, terra di mangiatori, Giovanni Succi si impone sulla scena del mondo come il più grande digiunatore di tutti i tempi. C'è qualcosa in lui di invulnerabile, che non si arrende neanche all'evidenza. Qualcosa che ha imparato ancora bambino dalle carovane dei circhi, quando scendevano dal Paradiso Terrestre verso la pianura romagnola. Alla saggezza errante dei saltimbanchi, Giovanni deve la sua gioia e la sua salvezza, l'urgenza di diventare quello che è: uno spirito sensibile, un leone indomabile, un profeta immortale. Guidato dall'utopia del socialismo e dal battito del suo cuore, veleggia libero come un elisir attraverso deserti e savane, cespugli e radure, nuvole e gabbie, e mette il suo digiuno al servizio dell'umanità. Coltivando in sé la sorgente di una speranza illimitata – riflessa in donne dai nomi armoniosi quali Ginevra, Gigliola, Guerranda –, segue il suo respiro per il mondo, dal Canale di Suez al manicomio della Lungara, dalle strade del Cairo e di Milano alle corsie della Salpêtrière. Incontra donne-belve e grandi esploratori, Sigmund Freud e Buffalo Bill, mentre l'Occidente sfocia nella modernità e perde per sempre l'innocenza. In questa biografia sentimentale, Carabba parte da una storia vera per trasfigurarla in un grande romanzo, che ci svela il valore del dubbio, le acrobazie dell'entusiasmo, la fierezza della semplicità. Perché è proprio lì, sul confine tra il pieno e il vuoto, dove la nebbia personale si dissolve nell'incontro con gli altri, che si nasconde la promessa dell'eternità.Enzo Fileno Carabba è nato a Firenze nel 1966. È autore di romanzi pubblicati in Italia e all'estero, di racconti, sceneggiature radiofoniche, libri per bambini, libretti d'opera e poesie. Nel 1990 ha vinto il Premio Calvino con il romanzo Jakob Pesciolini e il suo ultimo libro è Vite sognate del Vasari (Bompiani, 2021). Vive a Impruneta.https://enzofilenocarabba.it/IL POSTO DELLE PAROLEascoltare fa pensarehttps://ilpostodelleparole.it/

El Trío Avileño, el Dúo Cabrisas Farach, Laíto Sureda con el Conjunto Casino, y el Conjunto Estrellas de Chocolate.