Podcasts about American Heart Association

What if the biggest improvements to your health aren't hiding inside another supplement, expensive biohack, or complicated protocol, but in the signals you're sending your body every single day? In this special episode of the SuperLife Podcast, Darin walks through his personal daily routine from the moment he wakes up, explaining not only what he does, but why he does it. From getting outside before touching his phone to morning light, hydration, meditation, breathwork, journaling, cacao, movement, food, and eventually shutting off the breaker to his bedroom at night, Darin breaks down the practices he has built into the rhythm of his life. But this isn't a prescription to copy Darin's routine. It's an invitation to understand the biological signals behind it and build your own. Darin's central idea is simple: food is information, light is information, breath is information. Every input you give your body becomes a set of instructions your cells have to respond to. Drawing from research on circadian biology, hydration, meditation, nervous system regulation, expressive writing, cacao flavanols, resistance training, cardiovascular health, and longevity, Darin explains how simple behaviors compound over decades. The healthiest and longest-lived people he has encountered around the world didn't necessarily follow elaborate protocols. Their health was embedded into a rhythm of light, movement, real food, darkness, and sleep. This is a practical look inside the habits Darin actually uses, and a reminder that your health isn't created by one massive intervention. It's built through the seemingly insignificant decisions you repeat every day. What You'll Learn What Darin does from the moment he wakes up each morning Why he goes outside before looking at his phone How morning light helps synchronize your circadian rhythm and nighttime sleep Why hydration comes before coffee or other stimulants How meditation and conscious breathing can change your relationship with stress Why Darin considers breath one of the fastest ways to influence your nervous system How journaling can help process emotions, gratitude, grief, and mental stress Why cacao has become part of Darin's morning ritual How resistance training and aerobic movement support long-term health Why Darin believes health is ultimately about creating a biological rhythm rather than blindly following someone else's protocol Chapters 00:00:03 – Welcome to SuperLife 00:00:34 – Sponsor: Bite 00:02:49 – What Darin does before touching his phone every morning 00:03:11 – Why your biology is waiting for the signal of morning light 00:03:44 – The purpose behind Darin's daily routine 00:04:12 – Why you shouldn't simply copy someone else's protocol 00:04:37 – Food, light, and breath are information 00:05:06 – How the modern day disrupts your natural biology 00:05:51 – The danger of stacking small "fatal conveniences" for decades 00:06:20 – Why Darin built his life around biological rhythm 00:06:47 – Darin's morning hydration routine 00:07:11 – Why Darin wakes up between 4 and 5 A.M. 00:07:38 – Morning sunlight, no sunglasses, and no screens 00:07:48 – Your body's master circadian clock 00:08:40 – What research says about morning sunlight and sleep 00:09:21 – The cheapest thing you can do tonight for better sleep 00:09:44 – Why Darin drinks water before anything else 00:10:22 – Darin's daily meditation practice 00:10:54 – What research shows about mindfulness, anxiety, depression, and pain 00:11:33 – Sponsor: Fatty15 00:15:14 – Training yourself not to be hijacked by the modern world 00:15:43 – Your breath is the steering wheel of your nervous system 00:16:33 – The five-minute breathing practice that changed mood and respiratory rate 00:17:13 – How conscious breathing can shift your nervous system 00:17:42 – Darin's journaling and gratitude practice 00:18:15 – Journaling through grief after losing his mother 00:18:44 – What expressive writing research reveals about mental health 00:19:17 – Why an unprocessed thought can become physiological stress 00:19:30 – The sequence Darin follows before consuming anything stimulating 00:19:39 – Why cacao is part of Darin's morning ritual 00:20:06 – Darin reveals the cacao project he's developing 00:20:32 – The massive COSMOS cacao study 00:21:36 – Cacao and potential cognitive benefits 00:22:13 – Flavanols, nitric oxide, theobromine, magnesium, and polyphenols 00:22:50 – What to avoid when choosing cacao 00:23:19 – Darin's approach to resistance training 00:23:43 – Why movement doesn't need to be complicated 00:24:19 – Resistance training and longevity 00:24:54 – The powerful combination of strength training and aerobic activity 00:25:17 – Why walking may be one of the simplest longevity tools 00:25:46 – Muscle, blood sugar, myokines, and grip strength 00:26:15 – How injury changed Darin's relationship with training 00:26:51 – Training to experience more of life 00:27:14 – The principle the entire episode builds toward: food is information 00:27:26 – What you've learned from Darin's morning routine 00:27:49 – What Darin eats every day and his nighttime routine 00:28:18 – Continue Darin's complete daily protocol on Patreon Thank You to Our Sponsors: Bite Toothpaste: Go to trybite.com/DARIN20 or use code DARIN20 for 20% off your first order. Fatty15: Get an additional 15% off their 90-day subscription Starter Kit by going to fatty15.com/DARIN and using code DARIN at checkout. Join the SuperLife Patreon: This is where Darin now shares the deeper work: - weekly voice notes - ingredient trackers - wellness challenges - extended conversations - community accountability - sovereignty practices Join now for only $7.49/month at https://patreon.com/darinolien Connect with Darin Olien: Website: darinolien.com Instagram: @darinolien Book: Fatal Conveniences Platform & Products: superlife.com New Show: Roadmap to Happiness Key Takeaway "Your health isn't built from one perfect supplement, workout, meal, or morning routine. It's built from the information you repeatedly give your body. Light is information. Breath is information. Food is information. Movement is information. Darkness is information. Every day, your cells are listening to the signals you send them, whether you're consciously choosing those signals or allowing the modern world to choose them for you. You don't need to copy my routine. Understand why these practices matter, listen to your own body, and build a rhythm that works for your life. Because the seemingly insignificant things you do every morning, every afternoon, and every night eventually become your biology." Bibliography/Sources Morning Light Anderson, et al. (2025). Does sunlight exposure predict next-night sleep? A daily diary study among U.S. adults. Journal of Health Psychology . https://pubmed.ncbi.nlm.nih.gov/39077837/ Silva, et al. (2025). The role of sunlight in sleep regulation: Analysis of morning, evening and late exposure. BMC Public Health . https://link.springer.com/article/10.1186/s12889-025-24618-8 Meditation, Breath, Journaling Balban, et al. (2023). Brief structured respiration practices enhance mood and reduce physiological arousal. Cell Reports Medicine, 4(1), 100895 . https://www.cell.com/cell-reports-medicine/fulltext/S2666-3791(22)00474-8 Goyal, et al. (2014). Meditation programs for psychological stress and well-being: A systematic review and meta-analysis. JAMA Internal Medicine, 174(3), 357–368 . https://jamanetwork.com/journals/jamainternalmedicine/fullarticle/1809754 Pennebaker, J. W., & Beall, S. K. (1986). Confronting a traumatic event. Journal of Abnormal Psychology, 95(3), 274–281 . Smyth, et al. (2018). Online positive affect journaling… a preliminary randomized controlled trial. JMIR Mental Health, 5(4), e11290 . Cacao Sesso, et al. (2022). Effect of cocoa flavanol supplementation for the prevention of cardiovascular disease events: COSMOS. The American Journal of Clinical Nutrition, 115(6), 1490–1500 . https://ajcn.nutrition.org/article/S0002-9165(22)00275-1/fulltext Vyas, et al. (2023). Cocoa extract and cognitive function: COSMOS clinic subcohort. The American Journal of Clinical Nutrition, 117(1), 40–52 . https://ajcn.nutrition.org/article/S0002-9165(23)66275-6/fulltext Resistance Training Saeidifard, et al. (2019). The association of resistance training with mortality. European Journal of Preventive Cardiology, 26(15), 1647–1665 . https://pubmed.ncbi.nlm.nih.gov/31104484/ Shailendra, et al. (2022). Resistance training and mortality risk: A systematic review and meta-analysis. American Journal of Preventive Medicine, 63(2), 277–285 . https://pubmed.ncbi.nlm.nih.gov/35599175/ Fruit, Nuts, Plant Diversity Aune, et al. (2016). Nut consumption and risk of CVD, cancer, all-cause mortality. BMC Medicine, 14, 207 . Aune, et al. (2017). Fruit and vegetable intake and the risk of CVD, cancer and all-cause mortality. International Journal of Epidemiology, 46(3), 1029–1056 . McDonald, et al. (2018). American Gut. mSystems, 3(3), e00031-18 . Tempeh Effects of fermented soy on cognition in older adults: RCT. (2025). . https://pmc.ncbi.nlm.nih.gov/articles/PMC12472887/ Stephanie, et al. (2018). Tempeh consumption enhanced beneficial bacteria in the human gut . https://www.researchgate.net/publication/328100975 Baru Nut Baru almond oil supplementation in hemodialysis patients: RCT. (2021). Clinical Nutrition . https://www.sciencedirect.com/science/article/abs/pii/S0965229920303575 de Souza, et al. (2019). Baru almonds increase the activity of glutathione peroxidase in overweight and obese women: RCT. Nutrients, 11(8), 1750 . https://www.mdpi.com/2072-6643/11/8/1750 Phenolic and flavonoid content, antioxidant capacity of baru almond. (2021). . https://pmc.ncbi.nlm.nih.gov/articles/PMC8181485/ Pumpkin Seeds Dotto, J., & Chacha, J. (2020). Pumpkin seeds as a functional food ingredient. Scientific African, 10, e00575 . U.S. Department of Agriculture. (n.d.). FoodData Central: Pumpkin seed kernels, dried . Avocado HAT: Life's Essential 8 ancillary. (2025). Journal of the American Heart Association . https://www.ahajournals.org/doi/10.1161/JAHA.124.039130 Lichtenstein, et al. (2022). HAT: 1 avocado/day vs habitual diet on visceral adiposity. Journal of the American Heart Association . https://pubmed.ncbi.nlm.nih.gov/35861827/ Unlu, et al. (2005). Carotenoid absorption enhanced by avocado. The Journal of Nutrition, 135(3), 431–436 . Evening Light, Sleep Environment Gooley, et al. (2011). Room light before bedtime suppresses melatonin onset. The Journal of Clinical Endocrinology & Metabolism, 96(3), E463–E472 . https://pubmed.ncbi.nlm.nih.gov/21193540/ Greco, et al. (2023). Eye mask during overnight sleep improves episodic learning and alertness. Sleep, 46(3) & Sleep, 46(8) . https://academic.oup.com/sleep/article/46/3/zsac305/6912219 https://academic.oup.com/sleep/article/46/8/zsad105/7143720 Hu, et al. (2010). Earplugs and eye masks on nocturnal sleep, melatonin and cortisol. Critical Care, 14(2), R66 . Mason, et al. (2022). Light exposure during sleep impairs cardiometabolic function. Proceedings of the National Academy of Sciences, 119(12) . https://www.pnas.org/doi/10.1073/pnas.2113290119 World Health Organization. (2018). Environmental noise guidelines for the European region . RF-EMF and Sleep Does radiofrequency radiation impact sleep? Double-blind, randomised, placebo-controlled crossover pilot. (2024). Frontiers in Public Health . https://www.frontiersin.org/journals/public-health/articles/10.3389/fpubh.2024.1481537/full Loughran, et al. (2005). Mobile phone EMF and human sleep. NeuroReport, 16(17), 1973–1976 . Mohler, et al. (2012). RF-EMF exposure and sleep quality: Prospective cohort. PLOS One, 7(5), e37455 .

Welcome to the latest Midlife Minute. This episode unpacks ongoing research on Lp(a), explores how women's lipids change during the perimenopause-to-menopause transition, and clarifies what we can do about it. Stay tuned for more! IN THIS EPISODE, YOU WILL LEARN: How total cholesterol, LDL, and ApoB tend to rise most steeply around the final menstrual period, rather than increasing gradually with chronological aging Why women should have their Lp(a) checked both before and after menopause How oral and transdermal estradiol can affect LDL, HDL, triglycerides, and Lp(a) differently Why I recommend including ApoB when checking your lipids during the perimenopause window How your lifestyle can reduce your cardiovascular risk, even when your Lp(a) is elevated Why omega-3 fatty acids and commonly used lipid medications do not lower Lp(a) Why supplements are not replacements for statins in genuinely high-risk patients  I review the various PCSK9 inhibitors currently available, how they are administered, and their effects  How a CAC, a CT angiogram, and an AI-assisted CT angiogram can help women and their providers determine how aggressive their approach should be when Lp(a) is elevated Connect with Cynthia Thurlow   Follow on X, Instagram & LinkedIn Check out Cynthia's website. Submit your questions to support@cynthiathurlow.com  Join other like-minded women in a supportive, nurturing community: The Midlife Pause/Cynthia Thurlow.  Purchase Cynthia's book, The Menopause Gut. Cynthia's Intermittent Fasting Transformation Book The Midlife Pause Supplement Line References: 1. Menopause Transition and Cardiovascular Disease Risk: Implications for Timing of Early Prevention: A Scientific Statement From the American Heart Association.Circulation. 2020. El Khoudary SR, Aggarwal B, Beckie TM, et al.Guideline 2. Menopause: A Cardiometabolic Transition. The Lancet. Diabetes & Endocrinology. 2022. Nappi RE, Chedraui P, Lambrinoudaki I, Simoncini T.Review 3. Dyslipidemia Across the Menopause Transition: Mechanisms, Trajectories, and Opportunities for Cardiovascular Prevention. Maturitas. 2026. Castaneda R, Tatit CP, Hurtado Andrade MD, Faubion SS, Shufelt CL.RecentReview 4. Lipoprotein(a) and Women's Cardiovascular Health: A Review. JACC. Advances. 2026. Michos ED, Saucier S, Mehran R, Koschinsky ML. 5. Lipid Metabolism in Women: A Review. Atherosclerosis. 2025. van Oortmerssen JAE, Mulder JWCM, Kavousi M, Roeters van Lennep JE.Review 6. Are Changes in Cardiovascular Disease Risk Factors in Midlife Women Due to Chronological Aging or to the Menopausal Transition?. Journal of the American College of Cardiology. 2009. Matthews KA, Crawford SL, Chae CU, et al.Observational 7. Trajectories of Lipids Around the Menopause Transition in Chinese Women: Results of the Kailuan Cohort Study. Fertility and Sterility. 2023. Dai Q, Wu S, Cao Z, et al. 8. Lipid Management in Patients With Endocrine Disorders: An Endocrine Society Clinical Practice Guideline. The Journal of Clinical Endocrinology and Metabolism. 2020. Newman CB, Blaha MJ, Boord JB, et al.Guideline 9. The Effects of Menopause Hormone Therapy on Lipid Profile in Postmenopausal Women: A Systematic Review and Meta-Analysis. Frontiers in Pharmacology. 2022. Nie G, Yang X, Wang Y, et al.SR 10. Perspectives on Dyslipidemia and Coronary Heart Disease in Women. Journal of the American College of Cardiology. 2005. Bittner V.Review 11. Dyslipidemia in Midlife Women: Approach and Considerations During the Menopausal Transition. Maturitas. 2022. Torosyan N, Visrodia P, Torbati T, Minissian MB, Shufelt CL.Review 12. Effects of gender, age and menopausal status on serum apolipoprotein concentrations.13.Lipoprotein(a).The Journal of the American Medical Association. 2025. Mora S, Kronenberg     14. Why, how and in whom should we measure levels of lipoprotein(a): A review of the latest evidence and clinical implications. Diabetes, Obesity & Metabolism. 2025. Razavi AC, Bhatia HS, Blumenthal RS, Shapiro MD, Mehta A.RecentReview 15. Red Yeast Rice for Hypercholesterolemia: JACC Focus Seminar. Journal of the American College of Cardiology. 2021. Cicero AFG, Fogacci F, Zambon A.Review 16. Overall and Sex-Specific Effect of Berberine for the Treatment of Dyslipidemia in Adults: A Systematic Review and Meta-Analysis of Randomized Placebo-Controlled Trials. Drugs. 2023. Blais JE, Huang X, Zhao JV.SR 17. Non-Genetic Influences on Lipoprotein(a) Concentrations. Atherosclerosis. 2022. Enkhmaa B, Berglund L.Review 18. NHLBI Working Group Recommendations to Reduce Lipoprotein(a)-Mediated Risk of Cardiovascular Disease and Aortic Stenosis. Journal of the American College of Cardiology. 2018. Tsimikas S, Fazio S, Ferdinand KC, et al.Review 19. Consumption of a defined, plant‐based diet reduces lipoprotein(a), inflammation, and other atherogenic lipoproteins and particles within 4 weeks. Clinical Cardiology. 2018. Najjar RS, Moore CE, Montgomery BD. 20. Lipoprotein(a): Current Evidence for a Physiologic Role and the Effects of Nutraceutical Strategies. Clinical Therapeutics. 2019. Santos HO, Kones R, Rumana U, et al.Review 21. Effect of Lipid-Lowering Therapies on Lipoprotein(a) Levels: A Comprehensive Meta-Analysis of Randomized Controlled Trials. Atherosclerosis. 2025. Xie S, Galimberti F, Olmastroni E, et al.Recent 22. Effect of Omega-3 Fatty Acid Supplementation on the Postprandial Metabolism of Apolipoprotein(a) in Familial Hypercholesterolemia. Journal of Atherosclerosis and Thrombosis. 2023. Ying Q, Croyal M, Chan DC, et al.RCT 23.Lipoprotein(a) and Cardiovascular Disease. Lancet. 2024. Nordestgaard BG, Langsted A.Review 24. Lipoprotein(a): A Genetically Determined, Causal, and Prevalent Risk Factor for Atherosclerotic Cardiovascular Disease: A Scientific Statement From the American Heart Association. Arteriosclerosis, Thrombosis, and Vascular Biology. 2022. Reyes-Soffer G, Ginsberg HN, Berglund L, et al.Guideline 25.Clinical Trial Design for Lipoprotein(a)-Lowering Therapies: JACC Focus Seminar 2/3.Journal of the American College of Cardiology. 2023. Malick WA, Goonewardena SN, Koenig W, Rosenson RS.Review 26.2026 ACC/AHA/AACVPR/ABC/ACPM/ADA/AGS/APhA/ASPC/NLA/PCNA Guideline on the Management of Dyslipidemia: A Report of the American Collegeof Cardiology/American Heart Association Joint Committee on Clinical Practice Guidelines. Circulation. 2026. Writing Committee Members, Blumenthal RS, Morris PB, et al.Guideline 27. Noninvasive Coronary Atherosclerotic Plaque Imaging. JACC. Cardiovascular Imaging. 2023. Kwiecinski J, Tzolos E, Williams MC, et al.Review 28. Computed Tomography Angiography Versus Agatston Score for Diagnosis of Coronary Artery Disease in Patients With Stable Chest Pain: Individual Patient Data Meta-Analysis of the International COME-CCT Consortium. European Radiology. 2022. Wieske V, Walther M, Dubourg B, et al.SR 29. Association of Coronary Artery Calcium Score With Qualitatively and Quantitatively Assessed Adverse Plaque on Coronary CT Angiography in the SCOT-HEART Trial. European Heart Journal. Cardiovascular Imaging. 2022. Osborne-Grinter M, Kwiecinski J, Doris M, et al.Clinical Trial 30. AI-Guided Quantitative Plaque Staging Predicts Long-Term Cardiovascular Outcomes in Patients at Risk for Atherosclerotic CVD. JACC. Cardiovascular Imaging. 2024. Nurmohamed NS, Bom MJ, Jukema RA, et al.Observational 31. The Lancet Commission on Rethinking Coronary Artery Disease: Moving From Ischemia to Atheroma. Lancet. 2025. Zaman S, Wasfy JH, Kapil V, et al.Review 32. Efficacy and safety of novel lipoprotein(a)‐targeted therapies: A systematic review and meta‐analysis of randomized controlled trials. Diabetes, Obesity & Metabolism. 2026. Santana AUL, Yusufzai MO, Freitas APS, et al. Recent Opinion

When families have support, children and youth do better at home, in school and in their communities. Yet many conversations about “student success” focus narrowly on classrooms and test scores, overlooking the pregnancy, early childhood, housing, mental health, special‑needs and basic‑needs supports that shape whether children can show up ready to learn in the first place. This program will explore how family resource centers and other family support services operate in San Francisco, drawing on the experiences of community leaders and parents who know these systems firsthand. Through stories from their work and lives, the panel will highlight how family support helps families navigate complex systems, build stability, and prevent crises before they escalate. The conversation will also consider what we can all do to support cradle‑to‑career supports for families, so that all San Francisco children and youth can thrive.About the Speakers Our moderator will be Kula Koenig, the chief policy officer at Safe & Sound, where she leads policy, advocacy and strategic partnerships to strengthen child abuse prevention through upstream investment, cross-sector collaboration, and systems change that centers families most impacted by inequities. She directly oversees Safe & Sound's role as the backbone organization of the Family Services Alliance, working to make San Francisco a place where all children, youth, and families can thrive. Kula grounds her leadership in the belief that prevention and civic engagement must be inclusive, accessible and healing. She brings more than 15 years of leadership experience at the intersection of public health, early childhood, and racial equity, including senior roles at Public Health Advocates, United Way, and the American Heart Association. She is also the founder and executive director of Social Justice PolitiCorps. Kula holds a B.A. from UCLA and an MBA from The George Washington University. Gloria Tan has been the executive director of the San Francisco nonprofit Gum Moon since 1986. Gum Moon operates a residence hall in Chinatown for survivors of domestic violence and human trafficking. In addition, Gum Moon has 3 family resource centers in the Chinatown, Richmond and Sunset neighborhoods. The FRC's are all funded through the San Francisco Department of Early Childhood's Family Resource Center Initiative. Originally from Singapore, Gloria came to the United States for college and earned a Business Administration degree from the University of San Francisco. In 2020, the Chinese Culture Center of San Francisco honored her with its Lifetime Achievement Award in Community Building for her decades-long community leadership. In May 2026, the San Francisco Human Rights Commission recognized her contributions on behalf of women, children, immigrant families and underserved communities across the city. She is also an active member of the Family Services Alliance. Vicente Milan is a proud San Franciscan, born and raised. A former U.S. Army Specialist who deployed to Iraq, Vicente later worked in landscaping before stepping into his most important role: primary caregiver to three young boys in a blended family. Vicente and his family have turned to Compass Family Services' Family Resource Center for parenting support, help with one of his children's special needs, and housing stability services.  He now serves as a parent leader in Compass's “Repeat and Repair” group and brings to this panel the perspective of a father who has seen how family resource centers can strengthen families and improve children's outcomes. He loves taking his kids to museums and connecting with other parents.  A Social Impact Member-led Forum program. Forums at the Club are organized and run by volunteer programmers who are members of The Commonwealth Club, and they cover a diverse range of topics. Learn more about our Forums. ORGANIZER Virginia Cheung Learn more about your ad choices. Visit megaphone.fm/adchoices

Most retirement calculators assume your money needs to last until age 95. But if you're 55 years old today, average life expectancy is actually 79 for men and 82 for women. This single hidden assumption could mean the difference between retiring five years earlier or leaving millions unspent. Key Topics Discussed 00:00:00 - Introduction: The Hidden Assumption Brad introduces the concept that retirement calculators make an invisible assumption about longevity—typically planning to age 90-95—and explains why this matters for financial independence planning. 00:05:30 - Why Planning to 95 Is the Default Dr Bobby Dubois and Aubrey Williams explain why financial advisors default to age 95, the fear of running out of money, and how this ignores the risk of over-saving and under-living. 00:12:00 - Layers of Financial Conservatism Brad discusses multiple layers of conservative assumptions in retirement planning: lower returns, lower withdrawal rates, zero Social Security, and now longevity assumptions. 00:15:45 - Real Life Expectancy Numbers Dr Bobby provides actuarial data showing average life expectancy from birth (71-76) versus age 55 (79-82), and the probability of reaching age 90. 00:21:00 - The Financial Impact of Longevity Aubrey presents modeling showing how nest egg requirements change dramatically based on longevity assumptions—from $714K at age 79 to 41% higher at age 90. 00:28:30 - Dynamic Planning and Annual Updates Discussion of how mortality-adjusted planning changes over time and why annual plan updates are essential rather than lock-and-load strategies. 00:35:00 - Tools to Estimate Your Longevity Dr Bobby outlines practical methods: actuarial calculators, family history, cardiovascular risk calculators like the AHA Prevent tool, and genetic testing including APOE for dementia risk. 00:45:00 - Biological Age vs Chronological Age Discussion of biological clock testing and why these tests aren't ready for prime time, despite heavy marketing in the longevity space. 00:50:00 - Extending Your Healthspan Dr Bobby covers evidence-based interventions to reduce risk of heart disease and dementia: exercise, sleep, blood pressure control, weight management, and avoiding smoking. 00:58:00 - Wrap-up and Resources Final thoughts on updating assumptions, where to find Dr Bobby and Aubrey, and invitation for community feedback on future analysis scenarios. Notable Quotes Brad Barrett: "Every financial calculator has to make assumptions... but there's one assumption that's almost never discussed, even though it might be the single biggest one in the entire model. How long does your money need to last?" Dr Bobby Dubois: "The nest egg you need depends a lot on how long you're going to live. Imagine you're sixty-five and you're only going to live five years. Well, you don't need a whole lot of money. Imagine you're sixty-five and you're going to live to be one hundred five." Aubrey Williams: "Planning to ninety-five does answer one question, but by far, it's not the only question we should be looking at... it completely ignores the opposite risk that we live a shorter life and either we've saved too much, worked too long or spent too little." Dr Bobby Dubois: "If you are 60, what's the likelihood you'll live to be 90? For men, it's about a third, meaning two thirds won't. Women, it's about half might live to be 90." Aubrey Williams: "If hitting FI at 65, you live to age 79, you need $714,000. But if you live to 90, 11 years longer, then that nest egg needs to be 41% higher." Key Takeaways Calculate your own life expectancy using actuarial tools from the Society of Actuaries rather than accepting calculator defaults of 90-95 Research your family history of longevity—genetics account for roughly 50% of how long you'll live, especially for those reaching 100+ Use the American Heart Association's Prevent calculator to assess your 10-year and 30-year cardiovascular disease risk Consider genetic testing for APOE status to understand dementia ris…

Contributor: Travis Barlock, MD Educational Pearls: What is pulmonary edema?  Pulmonary edema is the accumulation of fluid initially in the interstitium of the lungs, that when severe enough can also accumulate in the alveolar air sacs. It develops when the rate of fluid filtration through the pulmonary vasculature out-paces the lymphatics ability to drain the fluid.  There are 2 theories for what causes pulmonary edema in the setting of naloxone administration: catecholamine surge vs negative pressure/barotrauma Catecholamine Surge : Naloxone administration precipitates an acute opioid withdrawal in which epinephrine and norepinephrine surge causing marked vasoconstriction on both the heart and the lungs   Negative Pressure/Barotrauma: This mechanism is not directly naloxone related but may be confounded by opioid effects. High dose synthetic opioids can induce sustained laryngospasm that is not mu-opioid mediated (thus not easily reversed by naloxone). With rapid awakening, respiratory muscles induce a negative pressure in the thoracic cavity against a closed glottis. This results in an alveolar barotrauma and a transudative pulmonary edema. What is the treatment for pulmonary edema secondary to opioid overdose reversal? Positive pressure ventilation is the mainstay treatment (CPAP/BiPAP) with oxygen supplementation. Importantly, diuretics are not recommended. The patient is not fluid overloaded like in the case of other pulmonary edemas, and diuresing the patient can worsen kidney injury which is already at an increased risk in opioid overdoses (rhabdomyolysis in particular).     References: Saari TI, Strang J, Dale O. Clinical Pharmacokinetics and Pharmacodynamics of Naloxone. Clin Pharmacokinet. 2024;63(4):397-422. doi:10.1007/s40262-024-01355-6 DailyMed - NALOXONE HYDROCHLORIDE injection, solution. https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=201fdedf-1736-4e52-9d7d-de14292547fd Boyer EW. Management of Opioid Analgesic Overdose. New England Journal of Medicine. 2012;367(2):146-155. doi:10.1056/NEJMra1202561 Dezfulian C, Orkin AM, Maron BA, et al. Opioid-Associated Out-of-Hospital Cardiac Arrest: Distinctive Clinical Features and Implications for Health Care and Public Responses: A Scientific Statement From the American Heart Association. Circulation. 2021;143(16):e836-e870. doi:10.1161/CIR.0000000000000958 Kienbaum P, Thurauf N, Michel M, Scherbaum N, Gastpar M, Peters J. Profound Increase in Epinephrine Concentration in Plasma and Cardiovascular Stimulation after [micro sign]-Opioid Receptor Blockade in Opioid-addicted Patients during Barbiturate-induced Anesthesia for Acute Detoxification Anesthesiology. 1998;88(5):1154-1161. doi:10.1097/00000542-199805000-00004 Summarized by Dan Orbidan, OMS3 | Edited by Dan Orbidan & Ahmed Abdel-Hafiz, NREMT-P   Donate: https://emergencymedicalminute.org/donate/   Join our mailing list: http://eepurl.com/c9ouHf

Christopher Gardner, PhD, is a nutrition scientist and Professor of Medicine at Stanford University, where he has led more than 20 large-scale randomized clinical trials comparing diets like keto, vegan, low-fat, and low-carb. He's the past chair of the American Heart Association's Nutrition Committee and the author of the upcoming book Food Sense, which argues there's far more scientific consensus about what to eat than the internet's diet wars would suggest.In this episode, we discuss:(1:51) The Biggest Diet Mistake Everyone Makes(3:44) Oreos Are Vegan: The Low-Fat Diet Myth(8:48) Stanford's 609-Person Diet Study(11:32) Why Some Diets Work Better For You(17:00) $8M Study: Genes and Weight Loss(29:34) Keto vs Low-Fat: Cholesterol Results(36:27) Best and Worst Diets, Ranked by Science(39:01) 6 Foods Every Diet Expert Agrees On(47:17) Is Organic Food Actually Worth It?(53:58) Are Vegetables Losing Their Nutrients?(1:07:52) How Seed Oils Are Really Made(1:13:58) The Omega-3 vs Omega-6 Myth Explained(1:19:23) Why Inflammation Tests Don't Work(1:26:03) The Truth About the Seed Oil Panic(1:29:49) Eat Food, Not Too Much, Mostly PlantsLearn more about Christopher Gardner here:Website: https://nutrition.stanford.eduX (Twitter): https://x.com/GardnerPhDInstagram: https://www.instagram.com/cgardnerphdBook (pre-order, out Oct 13): Food Sense

Welcome to the latest Midlife Minute. Today, I'm answering listeners' questions about cardiovascular risk, focusing on Lp(a), LDL, statins, and the changes women experience after menopause. Stay tuned for more! IN THIS EPISODE, YOU WILL LEARN: Why women should have their Lp(a) checked both before and after menopause How Lp(a) works as a risk enhancer, and how cardiovascular risk becomes more significant when elevated Lp(a) occurs alongside high LDL Why family history is particularly helpful for women with a 10-year calculated cardiovascular risk that looks reassuringly low I review the current guidelines for primary prevention Why, even though research has shown reductions in major cardiac events with GLP-1s, GLP-1s do not replace statins for lowering LDL  What a CAC score can reveal about coronary artery calcification, and what it cannot detect How AI-assisted CT angiography (such as Clearly) can map, quantify, and characterize plaque throughout the coronary branches Connect with Cynthia Thurlow   Follow on X, Instagram & LinkedIn Check out Cynthia's website. Submit your questions to support@cynthiathurlow.com  Join other like-minded women in a supportive, nurturing community: The Midlife Pause/Cynthia Thurlow.  Purchase Cynthia's book, The Menopause Gut. Cynthia's Intermittent Fasting Transformation Book The Midlife Pause Supplement Line References: 1.  ​Lipoprotein(a) and Women's Cardiovascular Health: A Review. JACC. Advances. 2026. Michos ED, Saucier S, Mehran R, Koschinsky ML.Recent 2.  ​Sex Differences of Lipoprotein(a) Levels and Associated Risk of Morbidity and Mortality by Age: The Copenhagen General Population Study. Atherosclerosis. 2022. Simony SB, Mortensen MB, Langsted A, et al. 3. Managing Atherosclerotic Cardiovascular Risk in Young Adults: JACC State-of-the-Art Review. Journal of the American College of Cardiology. 2022. Stone NJ, Smith SC, Orringer CE, et al.Review 4. Thirty-Year Risk of Cardiovascular Disease Among Healthy Women According to Clinical Thresholds of Lipoprotein(a). JAMA Cardiology. 2026. Nordestgaard AT, Chasman DI, Moorthy V, et al.RecentObservational 5.  ​Lipoprotein(a).The Journal of the American Medical Association. 2025. Mora S, Kronenberg  6. Clinical Practice Guideline on Lipid Management for Cardiovascular Disease Risk Reduction. Department of Veterans Affairs (2026). 2026. Paul Heidenreich, Lance Spacek, Neil Gregor, et al. Guideline 7. Lipoprotein(a) and Family History Predict Cardiovascular Disease Risk. Journal of the American College of Cardiology. 2020. Mehta A, Virani SS, Ayers CR, et al.Observational 8. 2010 ACCF/AHA Guideline for Assessment of Cardiovascular Risk in Asymptomatic Adults: A Report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines. Journal of the American College of Cardiology. 2010. Greenland P, Alpert JS, Beller GA, et al.Guideline 9. Prevalence and Prognostic Implications of Coronary Artery Calcification in Low-Risk Women. The Journal of the American Medical Association. 2016. Kavousi M, Desai CS, Ayers C, et al.SR 10. ​Coronary Artery Calcium Scores and Risk for Cardiovascular Events in Women Classified as “Low Risk” Based on Framingham Risk Score: The Multi-Ethnic Study of Atherosclerosis (MESA). Archives of Internal Medicine. 2007. Lakoski SG, Greenland P, Wong ND, et al.Observational 11. 2019 ACC/AHA Guideline on the Primary Prevention of Cardiovascular Disease: A Report of the American College of Cardiology/American Heart Association Task Force on Clinical Practice Guidelines. Journal of the American College of Cardiology. 2019. Arnett DK, Blumenthal RS, Albert MA, et al.Guideline 12. HOPE for Rational Statin Allocation for Primary Prevention: A Coronary Artery Calcium Picture Is Worth 1000 Words. Mayo Clinic Proceedings. 2020. Orringer CE, Maki KC.Review 13. Summary of Updated Recommendations for Primary Prevention of Cardiovascular Disease in Women: JACC State-of-the-Art Review. Journal of the American College of Cardiology. 2020. Cho L, Davis M, Elgendy I, et al.Review 14. Health Maintenance in Postmenopausal Women. American Family Physician. 2025. Plesa M, Wong A, Katsaggelos E.Guideline 15.  ​Semaglutide and Cardiovascular Outcomes by Baseline HbA1c and Change in HbA1c in People With Overweight or Obesity but Without Diabetes in SELECT. Diabetes Care. 2024. Lingvay I, Deanfield J, Kahn SE, et al.RCT 16.  ​Semaglutide and Cardiovascular Outcomes in Obesity without Diabetes. The New England Journal of Medicine. 2023. Lincoff AM, Brown-Frandsen K, Colhoun HM, et al.RCT 17. Long-Term Weight Loss Effects of Semaglutide in Obesity Without Diabetes in the SELECT Trial. Nature Medicine. 2024. Ryan DH, Lingvay I, Deanfield J, et al.RCT 18. Interventions for the Prevention and Management of Cardiometabolic Multiple Long-Term Conditions. Lancet. 2026. Valabhji J, Hope D, Sayed NE, et al.RecentReview 19. Glucagon‐Like Peptide‐1 Receptor Agonists and Major Adverse Cardiovascular Events in Patients With and Without Diabetes: A Meta‐Analysis of Randomized‐Controlled Trials. Clinical Cardiology. 2024. Hosseinpour A, Sood A, Kamalpour J, et al.SR 20. ​FDA Orange Book. FDA Orange Book. 2026. 21. 10. Cardiovascular Disease and Risk Management: Standards of Care in Diabetes-2026. Diabetes Care. 2026. American Diabetes Association Professional Practice Committee for Diabetes*.RecentGuideline 22. Premature Coronary Artery Disease in Women: Sex-Specific Risk Factors, Pathogenetic Mechanisms and Clinical Implications. Annals of Medicine. 2026. Li F, Hong D, Yang M, et al.RecentReview 23. Cardiovascular Disease Risk Factors in Women: The Impact of Race and Ethnicity: A Scientific Statement From the American Heart Association. Circulation. 2023. Mehta LS, Velarde GP, Lewey J, et al.Guideline 24. Preventing CVD in Women: Common Questions and Answers. American Family Physician. 2023. Westfall E, Viere AB, Genewick JE.Review 25. Menopause Transition and Cardiovascular Disease Risk: Implications for Timing of Early Prevention: A Scientific Statement From the American Heart Association. Circulation. 2020. El Khoudary SR, Aggarwal B, Beckie TM, et al.Guideline

On the Evolving Wellness podcast, host Sarah interviews returning guest Dr. Anthony Jay (PhD biochemistry; former Mayo Clinic researcher) about his book Sugar Beat: How Sugar, Not Cholesterol, Destroys Arteries and his broader critique of mainstream cardiology. Jay cites a 28,000-person study where insulin resistance had the highest heart-attack hazard ratio (6.4) versus hypertension (2.2), triglycerides (2.1), and LDL (1.38), arguing doctors overemphasize cholesterol while neglecting insulin and HOMA-IR. He claims statins increase arterial calcification and diabetes risk, criticizes organizations like the American Heart Association, and disputes fears around LDL, ApoB, and Lp(a), describing them as fat-transport/oxidation-related. Jay recommends focusing on metabolic health, diet and fasting to promote fat burning and plaque reversal, cautions about processed foods and mislabeled oils, discusses CAC vs CCTA imaging variability, and notes genetic nuances such as iron-related risk and sitosterolemia.— Connect with Dr. Anthony Jaywebsite - https://www.ajconsultingcompany.com/YT - https://www.youtube.com/@dranthonyjay Books:Sugar Beat - https://amzlink.to/az0hIdy8AMV03Estrogeneration:  https://amzlink.to/az0U0rLUAUr8f _________Sponsored By:→ Bon Charge | Go to https://boncharge.com/products/demi-red-light-device?rfsn=8108115.26608d & use code SARAHKLEINER for 15% off storewide._________Timestamp:00:00 Insulin Resistance vs LDL01:05 Meet Dr Anthony Jay04:03 Censorship and Free Speech10:14 Seed Oils and Food Fraud14:04 Statins and Calcified Plaque21:53 Sugar Beat Heart Disease Mechanism30:59 Saturated Fat Genes and Lp a40:52 Sleep Stress Insulin Link42:14 Metabolic Flexibility Basics42:55 Genetic High Cholesterol Cases43:40 Oreo Study LDL Trap45:45 Statins Guidelines Corruption51:02 CAC vs CCTA Scans57:04 Labs Genes Wrap Up——— This video is not medical advice & as a supporter to you and your health journey - I encourage you to monitor your labs and work with a professional!________________________________________Get all my free guides and product recommendations to get started on your journey!https://www.sarahkleinerwellness.com/all-free-resourcesCheck out all my courses to understand how to improve your mitochondrial health & experience long lasting health! (Use code PODCAST to save 10%) -  https://www.sarahkleinerwellness.com/coursesMy free product guide with all product recommendations and discount codes:https://www.canva.com/design/DAF7mlgZpJI/xVyE4tiQFEWJmh_Xwx8Kbw/view?utm_content=DAF7mlgZpJIFree Webinar on Light & Health (includes free light bulb guide) - https://www.sarahkleinerwellness.com/mycircadianapp-free-webinarGet Early Access to Podcast Episodes & my Seasonal Food Course + UVB+Red Light Therapy course for free -  https://open.substack.com/pub/sarahkleinerwellness/p/uvbred-light-protocol?r=5eztl9&utm_campaign=post&utm_medium=web&showWelcomeOnShare=true

Send Zorba a message!On our 50th episode: Zorba looks at new research showing 1 in 4 NFL players have CTE, a progressive, degenerative brain disease linked to repetitive head trauma. He also talks about how this plays into youth sports. The American Heart Association calls out Zorba for something he said on the podcast. We hear a Mom Joke and Zorba talks about his recent time in Nepal, and how folks can help with the relief efforts following the recent disasters in the area.Link to support Nepal disaster reliefSupport the showProduction, edit, and music by Karl ChristensonSend your question to Dr. Zorba (he loves to help!):Phone: 608-492-9292 (call anytime)Email: askdoctorzorba@gmail.comWeb: www.doctorzorba.orgStay well!

The apprenticeship model of academia requires more than just passive learning from students. How can applicants seeking entry to selective programs demonstrate that they have the ability and ambition to contribute to their desired fields? Amy and Mike invited professor Robert Malkin to consider what student research matters the most to colleges. What are five things you will learn in this episode? What are the different forms of research high school students have access to? Why are in-person lab experiences becoming increasingly valuable for high school students? What are the biggest mistakes students make when trying to find research or lab opportunities? How can students approach professors, universities, or labs even if they don't have prior experience? What makes a research experience truly meaningful from a college admissions perspective? MEET OUR GUEST Dr. Robert Malkin is a Professor of the Practice of Biomedical Engineering and Global Health, Emeritus at Duke University in Durham, North Carolina. He is also the Founder and Executive Director of the International Research Institute of North Carolina.  Previously, he was the Herbert Herff Professor of Biomedical Engineering at The University of Memphis in Memphis, Tennessee, and The University of Tennessee. Before moving to Tennessee, Dr. Malkin was a professor of Electrical Engineering at The City College of New York and a member of the graduate faculty at The City University of New York and a research associate at Columbia University. Dr. Malkin was the founder and Director of Engineering World Health and The Global Public Service Academies, organizations dedicated to improving healthcare in the developing world. Dr. Malkin received his MS and PhD in Electrical Engineering from Duke University in 1993. Prior to attending graduate school, Dr. Malkin taught English in Thailand, worked at EM Microelectronics in Switzerland designing integrated circuits, worked for Cordis Corporation designing pacemakers, and worked for Sarns Incorporated designing heart-lung machines. Dr. Malkin served as an expert advisor to the World Health Organization's Advisory Group on Healthcare Technology, Advisory Group on Innovative Technologies, and on the World Health Organization's subcommittee on medical equipment donations. Dr Malkin received the BS degree in Electrical Engineering from The University of Michigan in 1984. Dr. Malkin has received numerous awards, including service awards from The Republic of Nicaragua; EM Microelectronics and Cordis Corporation. He was named an Engineering Hero Award by IEEE and received an Outstanding Faculty Research Award from The College of Engineering, an Established Investigator Award from the American Heart Association, and an award for Innovation and Excellence in Undergraduate Education from The President of The City College of New York. Currently, he serves as the Founder and Executive Director of IRI, wherein they provide STEM remote research opportunities to students around the world with the goal of empowering young minds that can potentially advance human knowledge, especially through their peer-reviewed journal publications. Bob can be reached at robert.malkin@duke.edu. LINKS International Research Institute of North Carolina RELATED EPISODES HOW STUDENT RESEARCH IMPACTS COLLEGE ADMISSIONS CREATING A PASSION PROJECT CULTIVATING AND RECOGNIZING ACADEMIC CURIOSITY FINDING YOUR AUTHENTIC SELF IN THE PATH TO COLLEGE ABOUT THIS PODCAST Tests and the Rest is THE college admissions industry podcast. Explore all of our episodes on the show page. ABOUT YOUR HOSTS Mike Bergin is the president of Chariot Learning and founder of TestBright, Roots2Words, and College Eagle. Amy Seeley is the president of Seeley Test Pros and LEAP. If you're interested in working with Mike and/or Amy for test preparation, training, or consulting, get in touch through our contact page.  

In this episode of *PICU Doc on Call*, Drs. Pradip Kamat and Rahul Damania dive into the topic of beta-blocker toxicity. They walk through a case of a 15-year-old who comes in with bradycardia, hypotension, hypoglycemia, and altered mental status after an intentional ingestion. Using a board-style question, they break down how to tell the difference between beta-blocker and calcium channel blocker poisoning and review key points about cardiac physiology and receptor signaling.Drs. Kamat and Damania discuss management strategies, including glucagon, high-dose insulin therapy, vasopressors, calcium, and ECMO as a last resort. They also spend some time focusing on specific agents like propranolol and sotalol, highlighting what makes their toxicologic profiles unique. This is a high-yield episode packed with pearls for pediatric intensivists!Show Highlights:Clinical case of a 15-year-old patient with bradycardia and hypotension due to intentional drug ingestionBeta-blocker toxicity and its clinical featuresDifferentiation between beta-blocker and calcium channel blocker toxicityKey physiological concepts related to cardiac action potentials and beta receptor functionClinical presentation and diagnostic workup for beta-blocker toxicityManagement strategies for beta blocker overdose, including airway control and fluid resuscitationSpecific toxicologic profiles of common beta-blockers (e.g., propranolol, sotalol, metoprolol)Importance of recognizing hypoglycemia and CNS effects in beta-blocker toxicitySummary of critical management steps and take-home points for pediatric intensivistsEncouragement for further learning and engagement on related topicsReferences:Fuhrman & Zimmerman - Textbook of Pediatric Critical Care Chapter 126. Toxidromes and their treatment. Joshi P. Pages 1504-1505Reference 1: Rogers textbook of Pediatric Intensive Care: Chapter 36: Poisoning. Nares M, Jeyapalan A, Weisman R: pages 525-543Reference 2: Lavonas EJ, Akpunonu PD, Arens AM, Babu KM, Cao D, Hoffman RS, Hoyte CO, Mazer-Amirshahi ME, Stolbach A, St-Onge M, Thompson TM, Wang GS, Hoover AV, Drennan IR; American Heart Association. 2023 American Heart Association Focused Update on the Management of Patients With Cardiac Arrest or Life-Threatening Toxicity Due to Poisoning: An Update to the American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Circulation. 2023 Oct 17;148(16):e149-e184. doi: 10.1161/CIR.0000000000001161. Epub 2023 Sep 18. PMID: 37721023.Suarez F, Koyfman A, Long B. Pearls and Pitfalls for the Emergency Clinician: Beta Blocker and Calcium Channel Blocker Toxicity. J Emerg Med. 2026 May;84:1-11. doi: 10.1016/j.jemermed.2026.01.021. Epub 2026 Jan 27. PMID: 41833262.

*JOIN THE PHYSIONIC INSIDERS [PREMIUM CONTENT]*Join the Physionic Insiders: https://bit.ly/PhysionicInsiders2 *HEALTH AUTONOMY [COURSE]*Learn to Analyze & Apply Studies for Yourself: https://bit.ly/healthautonomy*JOIN THE COMMUNITY*Join my Community [It's Free!]: https://bit.ly/PhysionicCommunity2*EMAIL LIST*1-2 Weekly Email of Value [It's Free!]: http://bit.ly/2AXIzK6*HIRE ME FOR CONSULTING:* Consulting: https://bit.ly/3dmUl2H Original Podcast: https://youtu.be/q57CNySsYxs?si=PIwVEVGj_-cYkDCU0:00 - Introduction1:54 - Genetics that cause High Cholesterol5:29 - The American Heart Association is lying to you8:05 - Extremely High Cholesterol doesn't Kill17:48 - High Cholesterol at 60? Protection from Death25:20 - Other ways of testing this?27:12 - Main PointsReferences[1] Mundal L, Sarancic M, Ose L, et al. Mortality among patients with familial hypercholesterolemia: a registry-based study in Norway, 1992–2010. *J Am Heart Assoc*. 2014;3(6):e001236. doi:10.1161/JAHA.114.001236[2] Brown MS, Goldstein JL. Familial hypercholesterolemia: defective binding of lipoproteins to cultured fibroblasts associated with impaired regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity. *Proc Natl Acad Sci U S A*. 1974;71(3):788-792. doi:10.1073/pnas.71.3.788[3] Shishikura D, Harada-Shiba M, Michikura M, et al. Comparison of clinical characterization and therapeutic strategies between homozygous familial hypercholesterolemia and heterozygous familial hypercholesterolemia. *J Atheroscler Thromb*. 2026;33. Advance publication online March 27, 2026. doi:10.5551/jat.65959[4] Wiegman A, de Groot E, Hutten BA, et al. Arterial intima-media thickness in children heterozygous for familial hypercholesterolaemia. *Lancet*. 2004;363(9406):369-370. doi:10.1016/S0140-6736(04)15467-6[5] EAS Familial Hypercholesterolaemia Studies Collaboration (FHSC). Global perspective of familial hypercholesterolaemia: a cross-sectional study from the EAS Familial Hypercholesterolaemia Studies Collaboration (FHSC). *Lancet*. 2021;398(10312):1713-1725. doi:10.1016/S0140-6736(21)01122-3[6] Clarke R, Emberson JR, Parish S, et al. Cholesterol fractions and apolipoproteins as risk factors for heart disease mortality in older men. *Arch Intern Med.* 2007;167(13):1373-1378.[7] Ding M, Wennberg A, Ek S, et al. The association of apolipoproteins with later-life all-cause and cardiovascular mortality: a population-based study stratified by age. *Sci Rep.* 2021;11(1):24440. doi:10.1038/s41598-021-03959-5.[8] Mortensen MB, Nordestgaard BG. Elevated LDL cholesterol and increased risk of myocardial infarction and atherosclerotic cardiovascular disease in individuals aged 70-100 years: a contemporary primary prevention cohort. *Lancet*. 2020;396(10263):1644-1652. doi:10.1016/S0140-6736(20)32233-9Please use the following link to submit your critique: https://bit.ly/PhysionicCritiqueDisclaimer: None of the information provided by this brand is a replacement for your physician's advice. This brand is information for the sake of knowledge and the options of choice it provides, not in any way a personalized prescription. Please consult your physician before making any health related changes.

Matt DiNardo joins Jeff to chat about the heat and an opportunity to help raise money for The Leaders of Impact with The American Heart Association.

Relive the magic of outdoor cinema with the White Plains Hospital Screenings Under the Stars series at Kensico Dam Plaza in Valhalla. Nothing quite compared to the simple joy of watching a favorite film under the stars on a warm summer night, blending the enchantment of movies with the peaceful serenity of nature. Families and friends gathered with picnic blankets and plenty of snacks to enjoy these fun, free community evenings. Set against the stunning backdrop of Kensico Dam Plaza, the beloved series provided local residents with the perfect setting to relax, bond with loved ones, and make unforgettable summer memories together.Westchester Talk Radio host Andrew Castellano spoke with Kristen Alouisa, the Executive Director of the American Heart Association. During the interview, they highlighted the strong partnership between White Plains Hospital and the Heart Association, particularly through senior vice president Dawn French, who serves on the organization's board. She explained how easy it is to learn hands-only CPR in as little as 90 seconds, noting the organization's goal to have at least one person trained in CPR in every household by 2030. Additionally, they discussed the importance of managing blood pressure, mitigating stress, getting quality sleep, and adopting healthy lifestyle habits to combat heart disease.

In this episode, Aleah Johnson, Senior Program Lead, American Heart Association, and Jilyana Miklos, Community Health Director, YMCA Buffalo Niagara, discuss how the YMCA is supporting hypertension awareness through blood pressure kiosks, self-monitoring programs, education and community partnerships. They also explore how community-based organizations can help connect patients with clinical care and promote heart health.This episode is sponsored by American Heart Association

What if one of the most powerful tools for improving your cardiovascular resilience was already built into your body—and completely free? In this science-forward solo episode, Darin dives into the fascinating physiology of breath holding and the mammalian dive response, an ancient survival mechanism humans share with seals, whales, and dolphins. Simply holding your breath triggers an extraordinary cascade inside the body: your heart rate slows, blood vessels constrict to preserve oxygen for vital organs, and your spleen contracts to release oxygen-rich red blood cells into circulation. Darin explores research on elite freedivers, Southeast Asian sea nomads, cardiac hypoxic resistance, and the remarkable ways the human body can adapt to repeated breath-hold training. But he also separates the evidence from the hype, explaining why breath holding alone isn't a miracle intervention and why some of the most promising benefits appear when hypoxic stress is combined with movement and resistance exercise. This episode is a reminder that some of the most extraordinary tools for improving human performance aren't expensive supplements or complicated technologies—they're capacities your body has possessed all along. What You'll Learn What happens inside your body the moment you hold your breath How the mammalian dive response slows your heart rate and redirects blood flow Why your spleen acts like a biological reservoir for oxygen-rich red blood cells How humans share this ancient survival mechanism with marine mammals Why Southeast Asian sea nomads developed remarkable adaptations for freediving How breath-hold training can amplify the body's natural dive response What researchers discovered when elite divers reached extraordinarily low blood oxygen levels How repeated training may create greater hypoxic resilience in the heart Why the long-term benefits of breath-hold training in everyday people are still being studied Why combining hypoxic stress with movement may be more powerful than breath holding alone Chapters 00:00:03 – Welcome to SuperLife 00:00:34 – Sponsor: Alkemis 00:03:26 – The incredible thing happening when you hold your breath 00:03:57 – The ancient survival program you share with dolphins and whales 00:04:31 – The hidden cardiovascular tool almost nobody trains 00:05:03 – A free physiological capacity already built into your body 00:05:38 – What the science actually says about breath-hold training 00:06:09 – Understanding the mammalian dive response 00:06:37 – Your heart, blood vessels, and spleen react within seconds 00:07:11 – Your spleen is a biological oxygen reservoir 00:08:05 – Sponsor: Manna Vitality 00:09:59 – Why cold water amplifies the dive response 00:10:07 – The extraordinary physiology of Southeast Asian sea nomads 00:10:39 – Humans performing underwater like marine mammals 00:11:14 – The mammalian dive response is trainable 00:11:54 – What happened when elite divers reached extremely low blood oxygen 00:12:30 – How the heart adapts during extreme breath holds 00:13:08 – Cardiac hypoxic resistance and a stronger trained heart 00:13:55 – The important limitations of the current research 00:14:41 – Why breath holding alone isn't a miracle 00:15:09 – Combining hypoxic stress with resistance exercise 00:15:42 – Breath holding as a multiplier, not a replacement 00:16:33 – Practical takeaways for training the dive response 00:17:48 – Final thoughts and closing Thank You to Our Sponsors Alkemis: Go to https://alkemispaint.com/ and use code DARIN10 for 10% off your order. Manna Vitality: Go to mannavitality.com/ and use code DARIN12 for 12% off your order. Join the SuperLife Patreon: This is where Darin now shares the deeper work: - weekly voice notes - ingredient trackers - wellness challenges - extended conversations - community accountability - sovereignty practices Join now for only $7.49/month at https://patreon.com/darinolien Find More from Darin Olien: Website: darinolien.com Instagram: @darinolien Book: Fatal Conveniences Platform & Products: superlife.com New Show: Roadmap to Happiness Key Takeaway "You already have an ancient cardiovascular survival mechanism built into your body. Breath holding can trigger measurable changes in heart rate, blood flow, oxygen availability, and the body's response to hypoxic stress—and with training, that response appears capable of adapting. The goal isn't to treat breath holding like a miracle hack. It's to recognize it as another powerful physiological tool that may become even more effective when intelligently combined with movement and training. Bibliography/Sources Hypoxic Training & Cardiovascular Adaptation Bosco, G., et al. (2021). Effect of apnea-induced hypoxia on cardiovascular adaptation and circulating biomarkers of oxidative stress in elite breath-hold divers. Frontiers in Physiology . https://pmc.ncbi.nlm.nih.gov/articles/PMC8458773/ Combined training in hypoxic environments improves cardiometabolic health in older adults: A systematic review and meta-analysis of randomized controlled trials. (2025). Frontiers in Medicine . https://www.frontiersin.org/journals/medicine/articles/10.3389/fmed.2025.1728637/full Costalat, G., et al. (2021). Physiology, pathophysiology and (mal)adaptations to chronic apnoeic training: A state-of-the-art review. European Journal of Applied Physiology, 121, 2669–2685 . https://link.springer.com/article/10.1007/s00421-021-04664-x Palada, I., Bakovic, D., Valic, Z., et al. (2007). Cardiovascular regulation during apnea in elite divers. Hypertension, 50(2) . https://www.ahajournals.org/doi/10.1161/hypertensionaha.108.127530 Patrizio, I., et al. (2021). Cardiac hypoxic resistance and decreasing lactate during maximum apnea in elite breath-hold divers. Scientific Reports, 11, 3676 . https://www.nature.com/articles/s41598-021-81797-1 The Mammalian Dive Reflex & Genetic Adaptations Ilardo, M. A., Moltke, I., Korneliussen, T. S., et al. (2018). Physiological and genetic adaptations to diving in sea nomads. Cell, 173(3), 569–580 . https://news.berkeley.edu/2018/04/19/enlarged-spleen-key-to-diving-endurance-of-sea-nomads/ Schagatay, E. (2000). Studies on trained apnea divers' amplification of the diving response. Mid Sweden University . https://freedivecafe.com/2019/06/04/42-transcript-erika-schagatay-the-science-of-freediving/ Speck, D. F., & Bruce, D. S. (1978). Effects of varying thermal and apneic conditions on the human diving reflex. Undersea Biomedical Research . https://www.scientificamerican.com/article/breath-holding-dive-reflex-extends/ Inspiratory Muscle Strength Training (IMST) Craighead, D. H., Heinbockel, T. C., Freeberg, K. A., et al. (2021). Time-efficient inspiratory muscle strength training lowers blood pressure and improves endothelial function, NO bioavailability, and oxidative stress in midlife/older adults with above-normal blood pressure. Journal of the American Heart Association, 10(13) . https://www.ahajournals.org/doi/10.1161/JAHA.121.022203

Dr. Daniel W. Jones, M.D., FAHA — Professor Emeritus and former Dean of the University of Mississippi School of Medicine — joins us to break down the American Heart Association's new high blood pressure guidelines. As chair of the committee that wrote them, he explains why 130/80 is the new target, the surprising link between blood pressure control and lower dementia risk, why an arm cuff beats your smartwatch, and the simple lifestyle changes that make a difference. Informational only — always consult your clinician.

Current State and Future: Role of Physical Activity in Public Health - Prof Russell Pate (Pt2) Dr. Pate is Professor in the Department of Exercise Science in the Arnold School of Public Health at the University of South Carolina. He has held several administrative positions including Chair, Department of Exercise Science; Associate Dean for Research, Arnold School of Public Health; and Vice Provost for Health Sciences. Dr. Pate is an exercise physiologist with interests in physical activity and physical fitness in children and the health implications of physical activity. His research has been supported by NIH, CDC, the American Heart Association, and several private foundations and corporations. He coordinated the effort that led to the development of the recommendation on Physical Activity and Public Health of the CDC and the American College of Sports Medicine (ACSM) (1995). He served on the 2005 Dietary Guidelines Advisory Committee, the 2008 Physical Activity Guidelines Advisory Committee, and an Institute of Medicine panel that developed guidelines on prevention of childhood obesity. He currently serves as Chair of the National Physical Activity Plan Alliance. In 2012, he received the Honor Award from the ACSM. --- This podcast episode is sponsored by Fibion Inc. | The New Gold Standard for Sedentary Behaviour and Physical Activity Monitoring Learn more about Fibion: fibion.com/research --- Collect, store and manage SB and PA data easily and remotely - Discover new Fibion SENS Motion: https://sens.fibion.com/

Our hearts are restless, says Saint Augustine, until they find their rest in God. The biological science and the spiritual metaphor are in agreement here. Over the course of billions of beats, our hearts never stop to rest.  Your magnificent heart exchanges or pumps your entire blood volume, just over a gallon, every single minute. (Athlete hearts can pump up to 10 gallons per minute!) In this episode, Mark Labberton discusses matters of the heart with his close friend Dr. David Anderson, a cardiologist of four decades who trained at Johns Hopkins and helped launch one of the first interventional cardiology programs in San Francisco. Anderson reflects on the medical science of cardiology, the profound life-saving medical advances over the past forty years, heart disease versus coronary artery disease, how the brain and the heart communicate, and what happens during a heart attack. In the second half of their conversation, they move from physiology to spirituality of the heart: Pharaoh's hardened heart, "out of the heart the mouth speaks," and the human need to find somewhere to locate the self. Anderson closes with what a cardiologist would tell anyone worried about their own heart, and with a case for medicine as a noble profession. Episode Highlights "When I first began in cardiology, if your heart was damaged, imagining that the heart could actually improve its function was like imagining someone could grow back a new arm." "One of the distinctive things about the heart is unlike other muscles, it doesn't have a time to rest. When it gets insufficient blood supply, it can't just say, 'Whoa, I'm gonna stop here.' It asks you to slow down because it starts hurting." "I think that's part of the intrigue of the heart, that it is inside us … but in some ways it's apart from us. It's responding to environmental things that we're not really even aware of. … I think it's this sense of detachment of the heart from us—but yet its centrality—that has led people to posit the soul or the will or the passions or almost anything that you would think was the real you. "Atherosclerosis … I would describe as it's like having a pimple inside your artery." "First of all, know yourself. That is, it's not a time to be fatalistic because your father had a heart attack in his fifties or your brother just had a heart attack. It's a time to be proactive." About David Anderson David Anderson is a cardiologist who practiced for more than four decades before retiring. He studied at Stanford as an undergraduate and earned his medical degree at Johns Hopkins University School of Medicine. He stayed at Hopkins for residency, then trained in San Francisco at the dawn of interventional cardiology, helping to build an angioplasty program in the early 1980s. Helpful Links and Resources Alta Bates Summit Medical Center, Sutter Health, the Oakland hospital Anderson thanks by name: https://www.sutterhealth.org/about-us/our-hospitals/alta-bates-summit-medical-center DeWood et al., "Prevalence of Total Coronary Occlusion during the Early Hours of Transmural Myocardial Infarction," the 1980 study that identified clot as the cause of heart attack: https://www.nejm.org/doi/full/10.1056/NEJM198010163031601 Waagstein et al., "Effect of chronic beta-adrenergic receptor blockade in congestive cardiomyopathy," the Swedish beta-blocker work behind the recovery he describes: https://pubmed.ncbi.nlm.nih.gov/1191416/ What Is Atherosclerosis, National Heart, Lung, and Blood Institute: https://www.nhlbi.nih.gov/health/atherosclerosis Warning Signs of a Heart Attack, American Heart Association: https://www.heart.org/en/health-topics/heart-attack/warning-signs-of-a-heart-attack Angina (Chest Pain), American Heart Association, on the exertional pain he describes at the close: https://www.heart.org/en/health-topics/heart-attack/angina-chest-pain Show Notes The impact of family gene pool, lifelong heart awareness How David Anderson got into medicine, and cardiology in particular A Hopkins autopsy elective, a teenager dead in a parking lot, a mentor who followed From internists interested in the heart to interventionists inside it Catheters, pressures, and the arrival of echocardiography Seeing a beating heart inside a living body for the first time, as a senior resident A colleague's heart attack in 1979 The 1980 angiography study that identified clot as the trigger Spring 1983: an artery occludes, the cath lab opens it, pain gone in fifteen minutes Bypass surgery before that: elective, scheduled, never mid-attack Prevention running under the technology: blood pressure, smoking, the lipid theory Fixing one artery and never having to fix another Coronary artery disease versus disease of the muscle itself Plasticity, not regeneration Adrenaline as an emergency system built for days, not decades Moving from technological advances to molecular-based understanding of heart problems Understanding of the pathophysiology of atherosclerosis and its prevention Beta blockers, ejection fraction, and hearts that came back The heart as a muscle with no rest The heart's own electrical center, and the backup pacemakers below it Your heart doesn't rely on you or your consciousness to beat Why the ancients may have located the self in the heart organ Jeremiah's pounding heart, Pharaoh's hardened one Pascal, the soul, and needing somewhere to put "the real you" Maintaining an appropriate degree of separation from the patient and their family in order to care properly and effectively The difficulty of delivering bad news to patients and their familes Digging for a shared history to call on in a crisis "Medicine is a noble profession." Heart care advice from a cardiologist: First, know yourself. Heart health checklist: cholesterol, blood pressure, weight, knowing your own history The pain that warrants a phone call #Conversing #MarkLabberton #Cardiology #Heart #MedicalScience #HeartHealth #Biology #ChristianHumanism Production Credits Conversing is produced and distributed in partnership with Comment magazine and Fuller Seminary.

Dr. Hoffman continues his conversation with cardiologist and electrophysiologist Dr. Gregory Marcus, chair of the American Heart Association scientific statement writing group.

Cardiologist and electrophysiologist Dr. Gregory Marcus, chair of the American Heart Association scientific statement writing group, reveals evidence on coffee/caffeine and cardiovascular health. He discusses how earlier assumptions that caffeine triggers arrhythmias are complicated by genetics, individual risk factors, and mostly observational research. Marcus reviews randomized and observational findings: the CRAVE trial showed no increase in premature atrial contractions but more premature ventricular contractions on coffee days; a trial in post-cardioversion patients found less atrial fibrillation with daily coffee. Observational data suggest lower risks of type 2 diabetes, heart attack, stroke, and AFib among coffee drinkers, while heart failure risk may be lower at low intake but higher around 4–5 drinks/day. He cautions against extrapolating benefits to energy drinks, discusses additives like sugar, touches on alcohol, cannabis, Mediterranean/DASH diets, pollution, and evolving AFib treatments, wearables, and pacing/ablation technologies.

Como a mudança de paradigma na neuromonitorização do trauma, a padronização do choque em pediatria e o mapeamento genômico do câncer impactam sua conduta?Neste episódio do Afya News, analisamos o consenso latino-americano publicado na Neurosurgical Review sobre a detecção precoce da deterioração neurológica em pacientes com traumatismo cranioencefálico, integrando pupilometria, Doppler transcraniano e tomografia de controle.Discutimos também a nova declaração científica da American Heart Association sobre choque cardiogênico pediátrico, focando na redução da mortalidade por meio de reconhecimento rápido e suporte circulatório. Por fim, apresentamos no Radar o estudo da Nature Medicine que utilizou biópsia líquida para desvendar as mutações de escape tumoral em pacientes com câncer de pâncreas.O Afya News apresenta notícias da medicina com informação confiável e atualizada no seu tempo. Criado pela Afya, o maior hub de educação e soluções para a prática médica do Brasil, nosso propósito é transformar a saúde junto com quem tem a medicina como vocação.Fontes do episódio aqui:⁠https://portal.afya.com.br/podcasts/afya-news/18-08-2026Conteúdo 100% validado por equipe médica.Médico Responsável: Dr. Guilherme Rodrigues — CRM-RJ 1049461.

Send us Fan MailJoin the conversation as Dr. Tori Hudson and host Dr. Jaclyn Smeaton discuss the evolution of menopause hormone therapy through the eras: pre-2002, post-Women's Health Initiative, and the current state of menopause hormone therapy, with a particular focus on testosterone therapy. This episode also covers: Testosterone usage and dosing and the correlation with libido, mood, energy, and muscle mass Dr. Hudson's dosing protocols and concerns about supraphysiological dosing Methods of delivery for estrogen therapy Cardiovascular disease as an underappreciated priority in menopause care The importance of individualized care over following hormone therapy trends Show Notes Check out The American Heart Association's PREVENTä Online Calculator mentioned in this episode.Read Dr. Hudson's DUTCH Article on testosterone prescribing in postmenopausal women, and check out the testosterone guidelines discussed in this episode. Learn more about Dr. Tori Hudson and her practice, A Woman's Time! Explore easy at-home collection for patients with DUTCH Direct. Become a DUTCH Provider to get access to peer-reviewed and validated research, comprehensive patient reports, and expert clinical support. 

Biscuit welcomes in Jacob Dockery to talk about his work with the American Heart Association and upcoming charity events. They also talk about the upcoming football season.See omnystudio.com/listener for privacy information.

Dr. Pate is Professor in the Department of Exercise Science in the Arnold School of Public Health at the University of South Carolina. He has held several administrative positions including Chair, Department of Exercise Science; Associate Dean for Research, Arnold School of Public Health; and Vice Provost for Health Sciences. Dr. Pate is an exercise physiologist with interests in physical activity and physical fitness in children and the health implications of physical activity. His research has been supported by NIH, CDC, the American Heart Association, and several private foundations and corporations. He coordinated the effort that led to the development of the recommendation on Physical Activity and Public Health of the CDC and the American College of Sports Medicine (ACSM) (1995). He served on the 2005 Dietary Guidelines Advisory Committee, the 2008 Physical Activity Guidelines Advisory Committee, and an Institute of Medicine panel that developed guidelines on prevention of childhood obesity. He currently serves as Chair of the National Physical Activity Plan Alliance. In 2012, he received the Honor Award from the ACSM. --- This podcast episode is sponsored by Fibion Inc. | The New Gold Standard for Sedentary Behaviour and Physical Activity Monitoring Learn more about Fibion: fibion.com/research --- Collect, store and manage SB and PA data easily and remotely - Discover new Fibion SENS Motion: https://sens.fibion.com/

CardioNerds (Dr. Apoorva Gangavelli, Dr. Cory Sejo, and Dr. Joseph Kassab), discuss tricuspid regurgitation evaluation and management with Dr. Sunil Mankad. This episode was produced as part of the CardioNerds Academy curriculum by House Einthoven under the guidance of House Chief, Dr. Apoorva Gangavelli and Academy Program Director, Dr. Gurleen Kaur. A matching review article will be published in US Cardiology Review, the official journal of CardioNerds. This discussion was planned in collaboration with the Mayo Clinic Cardiovascular Board Review Course.  Audio editing by CardioNerds intern Emma Winakur. Enjoy this Circulation 2022 Paths to Discovery article to learn about the CardioNerds story, mission, and values. CardioNerds Episode PageCardioNerds AcademyCardionerds Healy Honor Roll CardioNerds Journal ClubSubscribe to The Heartbeat Newsletter!Check out CardioNerds SWAG!Become a CardioNerds Patron! Key Points: Tricuspid regurgitation is common and associated with increased mortality at every stage, regardless of etiology. Outcomes are worse with worsening severity, so accurate grading is critical. Etiology is critical to guide treatment decisions. Etiology includes primary vs secondary (atrial or ventricular) vs CIED-related TR. 3D echocardiography can be very helpful in determining TR etiology, especially in CIED-related TR. Diuresis with the goal of euvolemia is step one. Additionally, underlying contributory conditions (eg. pulmonary HTN, HFrEF, atrial fibrillation) should be addressed, if appropriate, and then TR severity reassessed. The choice between T-TEER and TTVR hinges on anatomy, RV function, pulmonary hypertension, and the ability to tolerate anticoagulation. T-TEER is generally first line in atrial functional TR with appropriate anatomy, in patients with poor RV function who cannot tolerate a sudden increase in RV afterload, or in patients who cannot tolerate the necessary anticoagulation with TTVR. TTVR is preferred with wide coaptation gaps and CIED-related TR. This is a team sport. Multidisciplinary discussions utilizing imaging (TTE/TEE, CT), risk scores (TRI-SCORE or TRIO), patient preference, and prior institutional experience are essential for the effective treatment of severe TR. Notes: What is the clinical importance of tricuspid regurgitation? TR is very common with approximately 4% of people over 75 having moderate or greater severity. TR (even mild) is associated with increased mortality. Those outcomes worsen as the TR severity worsens, and this phenomenon is independent of the mechanism of regurgitation. What is unique about the tricuspid valve compared to the other cardiac valves? It is at an anterior location which allows it to be imaged well with transthoracic echocardiography It is the largest valve and composed generally of 3 leaflets (but very often can have 4+ leaflets). Importantly, the RV is compliant and changes size and shape readily based on loading conditions. The TV annulus similarly changes size and shape based on hemodynamic conditions such as preload. What is a good framework for approaching the causes of tricuspid regurgitation? Determine the presence and define the severity of TR. Using TTE, we want to measure the right atrial size, the RV size, and any other concomitant valvular lesions.  Use TTE (2D and 3D) to characterize leaflet anatomy and characteristics. Subtypes of TR mechanisms (many times etiology is mixed). Primary: primary leaflet abnormality, occurs in ~10% of cases. Look for prolapse, flail, endocarditis, etc. Secondary/functional: leaflets normal but surrounding structures are abnormal. Atrial: RA and tricuspid annular dilation but normal RV size/shape, and can be related to arrhythmias like atrial fibrillation. Ventricular: RV dilated and/or dysfunctional with leaflet tethering. Can be related to pulmonary hypertension or primary RV disease. Cardiac implantable electronic device (CIED): Related to device (usually pacemakers or ICD) interaction with TV leaflets. Includes perforation, entanglement in subvalvular apparatus, impingement, etc. 3D TTE particularly helpful to evaluate How do we grade TR severity? It is very important to grade the severity of TR, and this is generally done with echocardiography. There are both quantitative and qualitative methods which use Doppler and various equations to estimate TR severity. Current recommendations have expanded TR severity beyond mild/moderate/severe to include “massive” and “torrential” categories. The most important parameters measured/calculated are vena contracta width, regurgitant volume, regurgitant fraction, and effective regurgitant orifice area. Helpful qualitative metrics include hepatic venous flow reversal. When should additional studies beyond transthoracic echocardiography, such as transesophageal echocardiography (TEE), cardiac computed tomography (CT), and cardiac magnetic resonance imaging (MRI) be pursued? TEE is particularly helpful if TTE views are poor. Since TEE is used during transcatheter intervention, a pre-procedure TEE to define anatomy, determine procedure candidacy, and plan for the procedure is critical.  CT is also helpful for procedure planning and has particular strengths in defining annulus size and geometry. A CT is required prior to transcatheter tricuspid valve replacement (TTVR). MRI is helpful for measuring RV volumes and function, but is not generally used to assess TR severity.  What is the approach to the treatment for severe tricuspid regurgitation? The first step is to try to determine the etiology. For secondary TR, treating the underlying condition is indicated. For example, pulmonary vasodilators for pulmonary HTN or guideline therapy for heart failure with reduced ejection fraction. Diuretics are the mainstay for treatment, with the goal to obtain euvolemia. This may require inpatient admission to optimize volume status and medication regimen. Once reversible etiologies are addressed, if the patient is still symptomatic from TR, additional therapies can be considered. What is the role of right heart catheterizations (RHC) in patients with severe TR? RHC is very helpful for many reasons. We use it in TR to help determine volume status, cardiac output, and RV function. Additionally, identifying and characterizing pulmonary hypertension (with pulmonary artery pressures and calculating pulmonary vascular resistance) is an important factor when choosing future therapies.  With severe tricuspid regurgitation, when should we refer for intervention (either with surgery or transcatheter repair or replacement)? Once reversible etiologies are addressed and euvolemia has been achieved, if the patient is still symptomatic from TR despite aggressive medical optimization, additional therapies can be considered. Once euvolemic, a repeat TTE should be ordered to reassess the severity of the TR. Use calculators (for example, either the TRI-SCORE or TRIO score) to predict operative mortality for isolated TR surgery. What are our transcatheter treatment options in severe tricuspid regurgitation, and how do we choose between them? The primary approved transcatheter treatment options for severe TR include transcatheter tricuspid edge-to-edge repair (T-TEER) and transcatheter tricuspid valve replacement (TTVR), of which the Edwards EVOQUE valve is the only one currently approved by the FDA. There are other TTVR device under investigation. These decisions should be made with a multi-disciplinary team including representation from cardiac imaging, interventional cardiology, and cardiothoracic surgery. Factors that go into the decision between T-TEER and TTVR include anatomy (annulus width, coaptation gap, leaflet length), RV reserve, pulmonary hypertension presence, ability to tolerate anticoagulation, patient preference, and institutional experience.  T-TEER is generally the first line with atrial functional and suitable anatomy. It is successful at reducing TR but does not generally eliminate it.  TTVR with EVOQUE is preferred in certain anatomic considerations like a large coaptation gap or when there is CIED-related TR (as this was excluded in T-TEER trials). Patients must be suitable for anticoagulation to receive TTVR as there is risk of leaflet thrombosis without it. If moderate/severe pulmonary hypertension is present, or there is poor RV function, TTVR may be avoided as the sudden elimination of TR causes a sudden increase in RV afterload which may not be tolerated. What is the role in advanced metrics for evaluating RV function? Advanced metrics like RV/PA coupling are under investigation but have not made it into the guidelines. The clinical utility is not yet known.  Assessing the RV function is important as stated above. Dr. Mankad prefers using 3D TTE to calculate an RVEF, or tracking RV longitudinal free wall strain. If you do encounter CIED-related TR, how do you treat it? Evaluate with TTE or TEE. 3D is very helpful to identify relative anatomy and leaflet-device interactions. There is no clear consensus about treatment if CIED-related TR is the primary mechanism of severe TR. If recently implanted, repositioning may be a valid option, but requires discussions with multiple teams including electrophysiology, advanced cardiac imaging, CT surgery, and interventional cardiology. References O’Gara PT, Lindenfeld J, Hahn RT, et al. 10 Issues for the Clinician in Tricuspid Regurgitation Evaluation and Management: 2025 ACC Expert Consensus Decision Pathway. J Am Coll Cardiol. 2025;S0735-1097(25)07047-0. O’Gara PT, Little SH, Badhwar V, et al. Operator and Institutional Recommendations and Requirements for Tricuspid Interventions: 2026 ACC/AHA/ASE/HRS/STS Expert Consensus Systems of Care Document. J Am Coll Cardiol. 2026;S0735-1097(26)05481-1. Hahn RT. Tricuspid Regurgitation. N Engl J Med. 2023;388(20):1876-1891. Davidson LJ, Tang GHL, Ho EC, et al. The Tricuspid Valve: A Review of Pathology, Imaging, and Current Treatment Options: A Scientific Statement From the American Heart Association. Circulation. 2024;149(22):e1223-e1238.

In this segment, we discussed the recent American Heart Association guidance on LDL cholesterol and the sobering estimate that only about one in three adults has their cholesterol adequately managed. For patients with risk factors for cardiovascular disease, the LDL target is now 55 mg/dL or lower — a number that is all too often overlooked when lab results are reviewed. The result is that many people are walking around with elevated LDL cholesterol, unaware that they are at meaningful risk for serious cardiovascular events including heart attack, stroke, and heart failure.The call to action for listeners is straightforward: log into your hospital or clinic patient portal, pull up your most recent lipid panel, and confirm that your LDL cholesterol is in range. Diet and exercise absolutely play a role in improving LDL, but lifestyle change alone is frequently not enough — particularly for patients with diabetes, hypertension, obesity, or fatty liver disease. In these cases, statin therapy is often the necessary next step, and it should not be viewed as a failure of lifestyle effort but as appropriate risk reduction.We also covered two additional recommendations. First, the AHA now advises a one-time measurement of lipoprotein(a). An elevated Lp(a) signals a substantially higher cardiovascular risk, and because the level is genetically determined, it typically will not improve with diet or exercise — making it essential information for guiding how aggressively other risk factors are treated. Second, a coronary calcium scan can bring your individual risk into sharp focus. A positive calcium score is a clear indication that aggressive intervention is warranted. Taken together, these three steps — knowing your LDL, checking your Lp(a) once, and considering a calcium score — give you and your clinician a far more complete picture of your true cardiovascular risk.How To Reach Dr. Joe Galati and his Team:For an on-line consultation or press inquiries, contact Teresa Reyes at 713-794-0700Dr. Galati's Newsletter Sign-UpLiver Specialists of TexasGet a Copy of Dr. Galati's BookDr. Galati on FacebookMessage Dr. Galati and his team Hosted on Acast. See acast.com/privacy for more information.

OPENER: Stephen A Smith DESTROYS WNBA After HIDING BRITTNEY GRINER'S DIRTY SECRET! Defend Sophie Cunningham! - 5:11 Brandon Gill Gets Witness To Admit Who DEI Punishes - 5:13 “I went to court to argue what a woman is” — Sall Grover at ARC 2026 -1:00 Jason Arday Whistleblower: ‘People Knew The Truth And Covered It Up' - 4:00 Quick, easy, worthless: Students let AI do the thinking | Principia Scientific, Intl. - Text It's Time to Save America - 10:00   HEALTH NEWS Arginine Supplement May Strengthen Immune Response to Cancer and Viral Infections Yeast food supplement helps immune cells fight cancer more effectively Physical activity linked to lower stroke and death risk in adults with atrial fibrillation Phthalates may contribute to high blood pressure in pregnant women Daily Collagen Supplement Alters Bone-Related Blood Markers in Female Runners, Study Says Arginine Supplement May Strengthen Immune Response to Cancer and Viral Infections Rockefeller University, August 10, 2026 (Natural News) A study published in the journal Cell reports that arginine, an amino acid found in protein-rich foods and produced by the body, may help the immune system recognize and respond to cancer and viral infections. The research found that low arginine levels were linked to reduced production of MHC-1, a protein that displays abnormal cellular signals to T cells, according to the report. In mouse models, arginine-rich diets were associated with fewer colon tumors and milder influenza and SARS-CoV-2 infections, the researchers reported. The findings add to prior work linking declining immune function to cancer progression. When arginine is scarce, cells struggle to produce MHC-1 proteins, which appear on the surfaces of cells throughout the body and display foreign or abnormal proteins to T cells. Researchers repeated the dietary studies using mouse models of influenza and SARS-CoV-2, conditions previously associated with unusual levels of different amino acids. One of the most dramatic patterns to emerge was that arginine was the most depleted amino acid in all of these diseases. Not only did mice with an arginine-rich diet have milder symptoms from viral infections, giving the mice arginine after influenza infection improved their outcomes too. Yeast food supplement helps immune cells fight cancer more effectively Trinity College Dublin & University College Dublin, July 9 2026 Researchers from Trinity College Dublin (TCD) and University College Dublin (UCD) have shown for the first time that a food supplement made from yeast helps the body to make stronger immune cells that can fight cancer. The research teams found that adding a yeast-based supplement to the food of obese laboratory mice changes how their immune cells grow and help to make better cancer‑fighting cells. Mice were fed a standard or high fat diet supplemented with yeast beta-glucan for 4-12 weeks and then their immune system was challenged by different types of cancer cells (colorectal, skin and breast cancer). The study also tested whether yeast supplementation could overcome immune dysfunction caused by obesity and whether protective effects persist after weight loss. Crucially, this dietary intervention restores anti-tumor innate immunity in obese mice and reverses long-term immune memory defects that persist even after weight loss, a major unmet clinical challenge. Physical activity linked to lower stroke and death risk in adults with atrial fibrillation Arctic University of Norway, August 5 2026 (News-Medical) Being physically active was linked to a lower risk of stroke and death for adults with atrial fibrillation, or AFib, according to new, independent research published in the Journal of the American Heart Association. AFib is a quivering or irregular heartbeat that occurs in the heart's upper chamber known as the atria. According to the American Heart Association, it can lead to blood clots, stroke and other heart-related conditions. The prevalence of AFib in the U.S. was estimated at 4.48% of the adult U.S. population. The study tracked more than 87,000 adults in Norway for about 15 years, capturing information about AFib, stroke and death from national health registries. What did the analysis find? Compared to adults who were inactive, adults with a low, moderate or high level of physical activity had a 9%, 19% and 18% lower risk of stroke, respectively. Having a low, moderate or high level of physical activity was associated with an 11%, 18% and 22% lower risk of death from any cause, respectively. For people with AFib, staying active was tied to living an average of 0.5 to 1.2 years longer compared to those who were inactive Phthalates may contribute to high blood pressure in pregnant women Harvard School of Public Health, August 10 2026 (News-Medical) Phthalates, which are chemicals found in plastics, personal care products and hundreds of other consumer goods, may contribute to high blood pressure in pregnant women, according to a study published in the Journal of the Endocrine Society. The researchers measured phthalate exposure and blood pressure in 338 pregnant women at three points during pregnancy. They analyzed whether higher levels of these chemicals-individually and in combination-were linked to higher blood pressure or an increased risk of pregnancy-related high blood pressure disorders. The authors found that pregnant women with higher urine concentrations of fragrance-associated phthalates and those in personal care products had higher systolic and diastolic blood pressure markers of an increased risk of hypertensive disorders of pregnancy. About 13% of participants in the study developed a pregnancy-related high blood pressure disorder. Women with higher levels of certain phthalates, especially those found in personal care products, tended to have higher blood pressure later in pregnancy. Daily Collagen Supplement Alters Bone-Related Blood Markers in Female Runners, Study Says Fitchburg State University, University of Connecticut & Mayo Clinic, August 4 2026 (Natural News) A four-week randomized, double-blind, placebo-controlled trial found that female distance runners who took 20 grams of collagen peptides per day showed an approximately 5.1 percent rise in P1NP over four weeks, a blood marker of bone formation, while the placebo group showed no such change, according to a study published in Frontiers in Nutrition. The collagen group also showed a drop in IL-6, an inflammatory protein linked to bone breakdown, while placebo levels held steady or rose, the study reported. The trial included 22 female runners ages 18 to 35 who ran at least 35 miles per week, and no adverse effects were reported. Collagen is the most common and abundant protein in the body, accounting for about 30 percent of total protein content and providing structural support to bones, tendons, ligaments, and other connective tissues, according to published research.       Discussion about this episode

CardioNerds (Dr. Apoorva Gangavelli, Dr. Rebecca Garber, and Dr. Tina Reddy), discuss pre-pregnancy risk stratification and counseling with Dr. Katy Young across a range of risks.  This episode was produced as part of the CardioNerds Academy curriculum by House Einthoven under the guidance of House Chief, Dr. Apoorva Gangavelli and Academy Program Director, Dr. Gurleen Kaur. A matching review article will be published in US Cardiology Review, the official journal of CardioNerds. This discussion was planned in collaboration with the Mayo Clinic Cardiovascular Board Review Course. Audio editing by CardioNerds intern, Dr. Patrick Pekyi-Boateng. Enjoy this Circulation 2022 Paths to Discovery article to learn about the CardioNerds story, mission, and values. CardioNerds Episode PageCardioNerds AcademyCardionerds Healy Honor Roll CardioNerds Journal ClubSubscribe to The Heartbeat Newsletter!Check out CardioNerds SWAG!Become a CardioNerds Patron! Notes:  Why is pregnancy considered a “physiologic stress test,” and why does risk extend beyond delivery? Blood volume, heart rate, and cardiac output rise while systemic vascular resistance falls, peaking in the late second/early third trimester; underlying (even undiagnosed) heart disease can be unmasked or worsened. Postpartum (“fourth trimester”) is a high-risk period, not a safe zone – fluid shifts, rising SVR, and bleeding risk can precipitate decompensation in patients with heart failure, pulmonary hypertension, valvular disease, or aortopathy. Adverse pregnancy outcomes (hypertensive disorders, gestational diabetes, preterm birth, fetal growth restriction, peripartum cardiomyopathy) are markers of future cardiovascular risk and warrant long-term preventive follow-up. What is the practical framework for approaching pre-pregnancy cardiovascular risk? Four broad categories: (1) patients who may need cardiac screening before pregnancy, (2) patients needing risk-factor/medication optimization, (3) known cardiovascular disease where pregnancy is reasonable with structured risk stratification, and (4) high-risk disease where pregnancy may need to be delayed, modified by intervention, or discouraged. Testing should be targeted, not blanket – reserved for symptoms, abnormal exam, concerning family history, or reduced functional capacity. How is risk stratified in patients with known cardiovascular disease? Use a combination of tools per 2025 ESC guidelines: mWHO 2.0 (broad maternal risk category), CARPREG II (additional predictors of maternal cardiac events), and ZAHARA (useful in congenital heart disease). Key lesion-specific factors: aortic size/growth, valve severity, ventricular function, symptoms, blood pressure, and family history of dissection. Translate risk into practical terms for patients rather than leading with a numerical score. Which cardiovascular medications require review before conception? ACE inhibitors, ARBs, and ARNIs should be transitioned off before pregnancy; statins, MRAs, and SGLT2 inhibitors also need review. DOACs are contraindicated in pregnancy and lactation; mechanical valve anticoagulation requires individualized shared decision-making, as no strategy is risk-free for mother and fetus. Medication changes are best made proactively, before conception, rather than reactively. This is not an exhaustive list! The medication list needs to be reviewed carefully. Which conditions carry high or prohibitive risk in pregnancy? Pulmonary arterial hypertension, Eisenmenger syndrome, severe ventricular dysfunction, prior peripartum cardiomyopathy with residual LV dysfunction, severe left-sided obstructive valve disease (e.g., severe mitral stenosis), mechanical valves, significant aortopathy, cyanotic congenital heart disease, and Fontan physiology. Common theme: limited cardiovascular reserve and high risk of decompensation, thrombosis, arrhythmia, heart failure, aortic dissection, or death. These patients need expert multidisciplinary evaluation before pregnancy. Severe mitral stenosis is poorly tolerated because tachycardia shortens diastolic filling time and raises left atrial pressure, risking pulmonary edema and decompensation. When should genetic testing or counseling be offered? Consider when a diagnosis may be inherited or affect the patient, pregnancy, or family members: inherited cardiomyopathies, aortopathies, channelopathies, select congenital heart disease, and some pulmonary hypertension syndromes. Recurrence risk of congenital heart disease in offspring is roughly 6-10% when the mother has CHD; fetal echocardiography should be offered. How should contraception be approached in high-risk cardiac patients? Frame contraception as part of the cardiac care and reproductive safety plan to prevent unplanned high-risk pregnancy. Long-acting reversible contraception is often preferred; progestin-only methods are generally safer than estrogen-containing options with thrombosis risk, pulmonary hypertension, or mechanical valves. What are key delivery-planning considerations for cardiac patients? Vaginal delivery is preferred unless there is an obstetric indication for cesarean or a specific cardiac reason (e.g., unstable maternal status, therapeutic INR) to avoid labor. Planning should address delivery location, anesthesia involvement, telemetry needs, fluid management, and postpartum monitoring, clearly communicated across the multidisciplinary team in advance. How should clinicians counsel patients when pregnancy is discouraged but strongly desired? Acknowledge the patient’s goals and the emotional weight of the conversation; separate the goal (family building) from the timeline (safety now vs. after optimization). If pregnancy remains prohibitively risky, discuss alternatives for family building and ensure adequate patient support. What are the key gaps and future directions in cardio-obstetric risk stratification? Current risk tools (mWHO, CARPREG II, ZAHARA) provide common language but do not fully capture functional status, prior pregnancy history, or how risk evolves over time. Future direction: individualized, dynamic risk prediction incorporating imaging, biomarkers, exercise capacity, and social drivers of health, with better long-term links between pregnancy complications and cardiovascular prevention. References 1. European Society of Cardiology. 2025 ESC Guidelines for the management of cardiovascular disease and pregnancy. 2. Mehta LS, et al. Cardiovascular Considerations in Caring for Pregnant Patients: A Scientific Statement From the American Heart Association. Circulation. 2020;141:e884-e903. PMID: 32362133. doi:https://doi.org/10.1161/CIR.0000000000000772 3. ACOG Practice Bulletin No. 212. Pregnancy and Heart Disease. Obstet Gynecol. 2019;133(5):e320-e356. PMID: 31022123. doi:https://doi.org/10.1097/AOG.0000000000003243

This week, I reviewed the cyclosporiasis outbreak a little more; not much has changed. I’m waiting to hear a friend’s experience with treatment. Then, I got into how much caffeine is enough, or too much? According to the American Heart Association, 400 mg is fine for most people! It’s NOT fine for me. I’d be […]

In the fourth installment of our church security series, Lloyd discusses the importance of training and the different kinds of training your church security team should partake of and how to role-play emergency situations inside your church worship space. Support the Show  Armed Lutheran Radio is a listener-supported podcast. If you value the information and entertainment we provide, consider supporting the show by joining our membership site, The Reformation Gun Club! http://www.ReformationGunClub.com  Links of Interest American Red Cross - https://www.redcross.org/take-a-class/cpr/cpr-training American Heart Association- https://cpr.heart.org/ SABRE Personal Safety Training - https://www.sabrered.com/personal-safety-training AVADE Pepper Spray Training - https://avadetraining.com/product/avade-pepper-spray-defense-e-learning-course/ USCCA Free Guide - https://www.usconcealedcarry.com/uscca-info/guide/protecting-house-of-worship-guide/ Sheepdog Church Security - https://sheepdogchurchsecurity.net/ Gatekeepers - https://gatekeeperstx.com/ NOCSSM 2026 Conference - https://gatekeeperstx.com/nocssm-conference/ Buy Duty to Defend, Volume 2 on Amazon – https://amzn.to/3D3frE5 Prayer of the Week Grant, O Lord, we implore You, that the course of this world may be so peaceably ordered by Your governance, that Your Church may joyfully serve You in all godly quietness; through Jesus Christ, Your Son, our Lord. Amen. Get in Touch Visit our Feedback Page - https://armedlutheranradio.libsyn.com/contact Please tell your friends about us, leave an iTunes review, and like us on Facebook Join our Facebook group - https://www.facebook.com/groups/fansofarmedlutheranradio Subscribe to us and follow us on Youtube - https://www.youtube.com/armedlutheran Check Out More at our Website- http://www.armedlutheran.us Original Music by Reformer  https://www.youtube.com/ReformerBand   

Send us Fan Mail"It is probably nothing."The swelling is probably the third trimester. The breathlessness is probably the stairs. The ache in your calf is probably that run you should not have done. And most of the time, all of that is exactly right.Most of the time is doing a lot of work in that sentence.Dr. Rachel Rosovsky is a hematologist at Massachusetts General Hospital, where she has spent twenty years on blood clots, and she co-authored the first ever AHA/ACC guideline on acute pulmonary embolism. The American Heart Association reached out about this one, and I said yes immediately, because this is the conversation I do not think most of us have ever actually had.

Join Dr. Martin in today's episode of The Doctor Is In Podcast.  

Family, friends and world leaders gathered for ceremonies in the nation's capital to celebrate the life of late Sen. Lindsey Graham — the farewells continue in his home state of South Carolina. TODAY shines a light on new high blood pressure guidelines from the American Heart Association, including a cardiovascular disease risk calculator tool and easy lifestyle changes to improve health outcomes. Tom Holland confirms his marriage to Zendaya after getting grilled by elementary school students on “Celebrity Substitute.” Plus, Macaulay Culkin eyes a return to the holiday silver screen after pitching a “Home Alone” reboot to Disney. Hosted by Simplecast, an AdsWizz company. See pcm.adswizz.com for information about our collection and use of personal data for advertising.

How many grams of protein do you need to be healthy? What about fiber? Supplements? It feels like we're changing our diets daily to adhere to constantly shifting recommendations and none of it is really working to optimize our health. Christopher Gardner, Professor of Medicine in Nutrition Science at Stanford University, and featured advisor on the Netflix show, “You Are What You Eat: A Twin Experiment”, joins us today to talk about how the industry is stacked against us and why the solution to our diet woes is as simple as beans, peas, and lentils. For more than 30 years Christopher Gardner, PhD, has studied what to consume and to avoid for optimal health, and how best to motivate individuals to achieve healthy dietary behaviors. He recently completed a two-year term serving on the US Dietary Guidelines Advisory Committee and is the past chair of the American Heart Association's Nutrition Committee. He has conducted and published dozens of human nutrition intervention trials, including trials of Mediterranean, Ketogenic, Vegan, Low-Fat and Low-Carb diets and their effects on cardiometabolic health. Some of his current interests include Stealth Nutrition, Unapologetic Deliciousness and Institutional Food Settings. He is currently working on personalized nutrition explorations with several colleagues, with particular focus on the gut microbiome.

Play 16:28 30:46 Mute Settings On this episode of The Community Cast, Carissa sits down with Ashley Deeb, Marketing Manager at Mainely Tubs, to discuss the growth of Maine's employee-owned hot tub, swim spa, and sauna company. Ashley shares how Mainely Tubs has expanded across New England, the benefits of employee ownership, and how her team helps customers create spaces for relaxation, wellness, and connection at home. Beyond her professional role, Ashley opens up about her personal journey as a heart disease survivor and how that experience inspired her commitment to community service through the American Heart Association and Make-A-Wish Maine. From leadership and volunteerism to resilience and purpose, this conversation highlights the power of giving back and the impact one person can have on the lives of others.

Investigators are trying to piece together what happened in a Michigan home that led to 47-year-old Kris Karolkiewicz shooting and killing his six children, his wife and then himself after setting the family home on fire. We know Kris recently lost his job as a Vice President with the American Heart Association, but it’s still unclear if that played a role in the unthinkable tragedy. Investigators say they found his licensed firearm near his body, and the rest of the family members shot to death in their bedrooms. While police confirmed he did not leave behind a note, investigators have seized what they call “a lot of documentation” from the home.See omnystudio.com/listener for privacy information.

Investigators are trying to piece together what happened in a Michigan home that led to 47-year-old Kris Karolkiewicz shooting and killing his six children, his wife and then himself after setting the family home on fire. We know Kris recently lost his job as a Vice President with the American Heart Association, but it’s still unclear if that played a role in the unthinkable tragedy. Investigators say they found his licensed firearm near his body, and the rest of the family members shot to death in their bedrooms. While police confirmed he did not leave behind a note, investigators have seized what they call “a lot of documentation” from the home.See omnystudio.com/listener for privacy information.

Investigators are trying to piece together what happened in a Michigan home that led to 47-year-old Kris Karolkiewicz shooting and killing his six children, his wife and then himself after setting the family home on fire. We know Kris recently lost his job as a Vice President with the American Heart Association, but it’s still unclear if that played a role in the unthinkable tragedy. Investigators say they found his licensed firearm near his body, and the rest of the family members shot to death in their bedrooms. While police confirmed he did not leave behind a note, investigators have seized what they call “a lot of documentation” from the home.See omnystudio.com/listener for privacy information.

Investigators are trying to piece together what happened in a Michigan home that led to 47-year-old Kris Karolkiewicz shooting and killing his six children, his wife and then himself after setting the family home on fire. We know Kris recently lost his job as a Vice President with the American Heart Association, but it’s still unclear if that played a role in the unthinkable tragedy. Investigators say they found his licensed firearm near his body, and the rest of the family members shot to death in their bedrooms. While police confirmed he did not leave behind a note, investigators have seized what they call “a lot of documentation” from the home.See omnystudio.com/listener for privacy information.

This week's supplement spotlight—spermidine; American Heart Association does a 180° on coffee; How accurate are health tracker wearables for sleep, blood pressure, heart rate? A skin disorder practically no one has heard of is often confused for skin cancer; Vitamin D—what forms and what dosages are optimal? Popular sugar substitutes may be worse for the brain than natural sugar.

In this episode of *Pediatric Critical Care Insights*, Dr. Monica Gray and Dr. Pradip Kamat chat about how capnography, specifically end-tidal CO2 monitoring, is used in the pediatric ICU. They walk through a real-life case of a 9-year-old with respiratory failure from influenza A, showing how ETCO2 monitoring helps confirm endotracheal tube placement, guides ventilation, spots cardiac arrest, and even helps assess the quality of CPR. Along the way, they break down how to interpret capnography waveforms, discuss different types of devices, and explain the key physiological concepts. The episode is packed with practical, bedside tips for intensivists caring for critically ill kids.Show Highlights:Importance of capnography (end-tidal CO2 monitoring) in the pediatric intensive care unit (PICU)Clinical case study of a 9-year-old boy with respiratory failure due to influenza AUse of capnography for confirming endotracheal tube placement and assessing ventilation statusDetection of cardiac arrest and guidance for CPR quality through ETCO2 monitoringOverview of capnography physics and physiology, including terminology distinctionsTypes of capnography: mainstream vs. sidestream, and their applications in pediatric patientsAssumptions for accurate ETCO2 approximation of arterial CO2 and conditions affecting this relationshipAnalysis of capnography waveform phases and their clinical significancePrognostic value of ETCO2 during cardiac arrest and its correlation with patient outcomesPractical applications of ETCO2 monitoring in critical care, focusing on airway, breathing, and circulation managementReferences:Noninvasive respiratory monitoring and assessment of gas exchange. David F. Butler; Kenneth A. Schenkman. Fuhrman and Zimmerman's Pediatric Critical Care, 43, 483-491.e3Humphreys S, Schibler A, von Ungern-Sternberg BS. Carbon dioxide monitoring in children—A narrative review of physiology, value, and pitfalls in clinical practice. Pediatr Anaesth. 2021;31:839–845. https://doi.org/10.1111/pan.14208Lasa JJ, Dhillon GS, Duff JP, et al. Part 8: Pediatric Advanced Life Support: 2025 American Heart Association and American Academy of Pediatrics Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Pediatrics. 2026;157(1):e2025074351O'Flaherty. Capnography: principles and practice. London: BMJ Publishing Group; 1994.Aminiahidashti H, Shafiee S, Zamani Kiasari A, Sazgar M. Applications of End-Tidal Carbon Dioxide (ETCO2) Monitoring in Emergency Department; a Narrative Review. Emerg (Tehran). 2018;6(1):e5. Epub 2018 Jan 15. PMID: 29503830; PMCID: PMC5827051.

Public Health is about more than hospitals and healthcare; it's also about the policies that help people live healthier lives every day.This week on the Conduit Street Podcast, Michael Sanderson and Karrington Anderson are joined by Laura Hale, State Government Relations Director for the American Heart Association, to discuss how counties can help improve community health through smart public policy.Laura shares practical examples of how counties are working with the AHA to make a difference, from expanding access to nutritious school meals and reducing youth exposure to tobacco to helping families make more informed dining choices through informed dining initiatives. She also explains how local governments can use partnerships, policy, and local authority to create healthier communities across Maryland. Plus, you'll hear one surprising statistic that may change the way you think about sugary drinks: the average child consumes the equivalent of a bathtub full each year.Tune in to hear this timely conversation on how thoughtful local policy can improve public health, one school meal, one community partnership, and one informed decision at a time.Follow us on Socials!MACo on TwitterMACo on FacebookLearn More: Why Give | American Heart Association

In this episode, Scott Becker discusses new findings from the American Heart Association suggesting that drinking up to five cups of coffee a day may support heart health and reduce stroke risk.

CardioNerds (Drs. Apoorva Gangavelli, Jenna Skowronski, and Hannah Every) discuss the continuum of prevention and heart failure with Drs. Anu Lala and Martha Gulati. Grounded in a clinical case of a 55-year-old woman with uncontrolled hypertension, type 2 diabetes, and obesity who is on the trajectory toward heart failure, this episode unpacks a paradigm-shifting framework from a joint HFSA/ASPC Scientific Statement. The discussion explores how prevention should not be siloed from heart failure management but rather integrated across a patient’s lifespan—from primary prevention in at-risk individuals, to secondary prevention in those with established heart failure, to tertiary prevention in patients with advanced therapies such as LVADs and heart transplantation. The experts highlight the importance of aggressive risk factor management, biomarker-guided screening, the AHA’s Life’s Essential 8, and the need for multidisciplinary collaboration and systems-level change to shift heart failure care from reactive to proactive. Audio editing for this episode was performed by CardioNerds Intern, Dr. Julia Marques Fernandes. Enjoy this Circulation 2022 Paths to Discovery article to learn about the CardioNerds story, mission, and values. US Cardiology Review is now the official journal of CardioNerds! Submit your manuscript here. CardioNerds Prevention PageCardioNerds Episode PageCardioNerds AcademyCardionerds Healy Honor Roll CardioNerds Journal ClubSubscribe to The Heartbeat Newsletter!Check out CardioNerds SWAG!Become a CardioNerds Patron! Pearls Systemic inflammatory diseases are associated with an elevated CVD risk that has significant implications for early detection, risk Heart failure prevention is a continuum, not a checkpoint. Prevention applies at every stage—from at-risk (Stage A) through advanced/post-transplant care—and every clinical encounter is an opportunity to intervene. The AHA’s Life’s Essential 8 (diet, physical activity, nicotine exposure, sleep, BMI, blood lipids, blood glucose, blood pressure) forms the foundation at every stage. Hypertension carries the highest population-attributable risk for heart failure of any modifiable risk factor. In the Framingham Heart Study, 91% of patients with newly diagnosed HF had pre-existing hypertension. The SPRINT trial demonstrated a 38% reduction in HF incidence with intensive blood pressure targets (30 ng/L or NT-proBNP >125 ng/L) identify individuals at heightened risk for progression to symptomatic HF. The ACC/AHA/HFSA guidelines give a Class IIa recommendation for natriuretic peptide screening in at-risk patients. Urine albumin-to-creatinine ratio (UACR) is an underutilized screening tool that provides additional insight into CKM risk. The heart failure label does not close the prevention window—it accentuates it. Secondary prevention through GDMT optimization (quadruple therapy in HFrEF) and continued risk factor management remains critical. Tertiary prevention extends to post-LVAD and post-transplant patients, where hypertension, diabetes, obesity, and CKD management remain essential to long-term outcomes. Show notes For a comprehensive review, please review the full HFSA/ASPC Joint Scientific Statement: Lala A, Beavers C, Blumer V, et al. The Continuum of Prevention and Heart Failure in Cardiovascular Medicine. J Card Fail. 2026;32:75-105. doi:10.1016/j.cardfail.2025.06.013 1. What is the “continuum of prevention” framework, and how does it differ from traditional approaches to heart failure prevention? Historically, prevention and heart failure management have been treated as separate disciplines—primary prevention handled by preventive cardiologists and treatment managed by heart failure specialists. This joint HFSA/ASPC Scientific Statement reframes prevention as a dynamic, continuous process that spans a patient’s entire lifespan, regardless of HF stage or ejection fraction. The framework maps onto the ACC/AHA HF staging system: Primary prevention targets Stage A (“at risk”) and Stage B (“pre-HF”) patients to reduce the burden of incident HF. Secondary prevention targets Stage C (symptomatic) and Stage D (advanced) patients to reduce the impact of established HF through GDMT optimization and ongoing risk factor management. Tertiary prevention encompasses risk factor management in patients with LVADs or heart transplants—populations where hypertension, diabetes, and obesity still drive outcomes. The Central Figure of the statement illustrates that Life’s Essential 8 (blood pressure and lipid control, diabetes management, exercise, sleep, smoking cessation, weight management, and diet/nutrition counseling) forms the foundation at every stage, with pharmacologic and device-based therapies layered on top as disease progresses (Figure) 2. How do traditional risk factors drive heart failure, and what should clinicians prioritize? Hypertension carries the greatest population-attributable risk for HF. In the Framingham Heart Study (N=5,143), HTN was associated with a 2- to 3-fold increased risk of HF, with a population-attributable risk of 39% in men and 59% in women. The SPRINT trial showed a 38% reduction in HF incidence and 25% reduction in the primary composite outcome with intensive BP targets (30 ng/L or NT-proBNP >125 ng/L) are associated with heightened risk for progression to symptomatic HF. In the ARIC study, incorporating NT-proBNP reclassified 20% of older adults without HF into Stage B. Factors that affect interpretation include age, sex, obesity (lower values), and CKD (higher values). High-sensitivity cardiac troponin (hs-cTn): Concentrations above the 99th percentile are now included in the definition of Stage B HF. Troponin testing may complement natriuretic peptides, particularly when BNP/NT-proBNP values are ambiguous. Risk scores: The PCP-HF equation predicts 10-year HF risk using traditional risk factors plus QRS duration. The AHA PREVENT score incorporates HF risk calculation and includes markers of kidney function (albuminuria, eGFR), though it may underestimate risk in men and Black adults. The CKM syndrome staging framework (Stages 0–4) provides a holistic approach to assessing systemic cardiovascular-kidney-metabolic risk. 4. What are the key nontraditional risk factors and cross-cutting themes in heart failure prevention? Genetics: Pathogenic cardiomyopathy variants exist in ~1 in 200 individuals in the general population. The HFSA and ACMG recommend cascade testing to identify at-risk family members. Polygenic risk scores for dilated cardiomyopathy show a 3.8-fold risk for DCM in the top 10th percentile compared with the median. Sex-specific considerations: Women have 2.8 times the odds of developing HFpEF, while men have similarly increased odds of HFrEF. A complete obstetric/gynecologic history is essential—preeclampsia is associated with a 4-fold increased risk of HF. Peripartum cardiomyopathy requires intentional screening in high-risk populations. Cardiotoxic exposures: Clinicians should be aware of medications that cause direct myocardial toxicity (e.g., anthracyclines, trastuzumab, tyrosine kinase inhibitors). A team-based approach with pharmacists can help optimize medication selection and risk factor modification. Social determinants of health: Environmental exposures (air pollution, arsenic, lead, cadmium), food insecurity, financial instability, and limited healthcare access contribute to HF risk and progression. Equity-focused, risk-based prevention strategies are needed. Psychological health: Depression is common in HF and independently associated with worse outcomes. Screening with brief questionnaires (e.g., PHQ-2) is recommended. Meditation, spirituality, and holistic wellness approaches remain underutilized. 5. What systems-level and policy changes are needed to move the needle on heart failure prevention? Multidisciplinary HF prevention clinics that bring together preventive cardiologists, HF specialists, endocrinologists, nephrologists, dietitians, pharmacists, exercise physiologists, and genetic counselors are advocated by the statement. EHR-embedded risk stratification could proactively flag patients on a trajectory toward HF—analogous to sepsis alerts or fall risk flags—enabling earlier intervention, particularly for patients who may not reach a cardiologist. Cardiac rehabilitation remains underutilized, particularly in HFrEF (Class 2b recommendation) and HFpEF (not yet covered by Medicare). The HF-ACTION trial showed quality-of-life benefits, and the REHAB-HF trial showed particular benefit in older patients with HFpEF. Policy priorities include expanding insurance coverage for preventive screening and novel therapies (SGLT2i, GLP-1 RAs, nsMRAs), reducing clinical inertia through team-based care models with closer follow-up intervals, and ensuring equitable access to evidence-based therapies across diverse populations. Digital health and AI hold promise for personalized risk prediction, remote monitoring (e.g., wearable devices, implantable PA pressure monitors), and virtual cardiac rehabilitation to overcome access barriers. Figure  Lala A, Beavers C, Blumer V, et al. The continuum of prevention and heart failure in cardiovascular medicine: a joint scientific statement from the Heart Failure Society of America and the American Society for Preventive Cardiology. J Card Fail. 2026;32(1):75-105. doi:10.1016/j.cardfail.2025.06.013) References Key references are bolded. Lala A, Beavers C, Blumer V, et al. The continuum of prevention and heart failure in cardiovascular medicine: a joint scientific statement from the Heart Failure Society of America and the American Society for Preventive Cardiology. J Card Fail. 2026;32(1):75-105. doi:10.1016/j.cardfail.2025.06.013 Heidenreich PA, Bozkurt B, Aguilar D, et al. 2022 AHA/ACC/HFSA guideline for the management of heart failure: a report of the American College of Cardiology/American Heart Association Joint Committee on Clinical Practice Guidelines. Circulation. 2022;145(18):e895-e1032. doi:10.1161/CIR.0000000000001063 Lloyd-Jones DM, Allen NB, Anderson CAM, et al. Life’s Essential 8: updating and enhancing the American Heart Association’s construct of cardiovascular health: a presidential advisory from the American Heart Association. Circulation. 2022;146(5):e18-e43. doi:10.1161/CIR.0000000000001078 SPRINT Research Group, Wright JT Jr, Williamson JD, et al. A randomized trial of intensive versus standard blood-pressure control. N Engl J Med. 2015;373(22):2103-2116. doi:10.1056/NEJMoa1511939 Levy D, Larson MG, Vasan RS, Kannel WB, Ho KK. The progression from hypertension to congestive heart failure. JAMA. 1996;275(20):1557-1562. doi:10.1001/jama.1996.03530440037034 Major outcomes in high-risk hypertensive patients randomized to angiotensin-converting enzyme inhibitor or calcium channel blocker vs diuretic: the Antihypertensive and Lipid-Lowering Treatment to Prevent Heart Attack Trial (ALLHAT). JAMA. 2002;288(23):2981-2997. doi:10.1001/jama.288.23.2981 Yusuf S, Sleight P, Pogue J, et al. Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients. N Engl J Med. 2000;342(3):145-153. doi:10.1056/NEJM200001203420301 Zinman B, Wanner C, Lachin JM, et al. Empagliflozin, cardiovascular outcomes, and mortality in type 2 diabetes. N Engl J Med. 2015;373(22):2117-2128. doi:10.1056/NEJMoa1504720 Anker SD, Butler J, Filippatos G, et al. Empagliflozin in heart failure with a preserved ejection fraction. N Engl J Med. 2021;385(16):1451-1461. doi:10.1056/NEJMoa2107038 Solomon SD, McMurray JJV, Claggett B, et al. Dapagliflozin in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2022;387(12):1089-1098. doi:10.1056/NEJMoa2206286 Filippatos G, Anker SD, Agarwal R, et al. Finerenone reduces risk of incident heart failure in patients with chronic kidney disease and type 2 diabetes: analyses from the FIGARO-DKD trial. Circulation. 2022;145(6):437-447. doi:10.1161/CIRCULATIONAHA.121.057983 Solomon SD, McMurray JJV, Vaduganathan M, et al. Finerenone in heart failure with mildly reduced or preserved ejection fraction. N Engl J Med. 2024;391(16):1475-1485. doi:10.1056/NEJMoa2407107 Lincoff AM, Brown-Frandsen K, Colhoun HM, et al. Semaglutide and cardiovascular outcomes in obesity without diabetes. N Engl J Med. 2023;389(24):2221-2232. doi:10.1056/NEJMoa2307563 Deanfield J, Verma S, Scirica BM, et al. Semaglutide and cardiovascular outcomes in patients with obesity and prevalent heart failure: a prespecified analysis of the SELECT trial. Lancet. 2024;404(10454):773-786. doi:10.1016/S0140-6736(24)01498-3  Kosiborod MN, Abildstrøm SZ, Borlaug BA, et al. Semaglutide in patients with heart failure with preserved ejection fraction and obesity. N Engl J Med. 2023;389(12):1069-1084. doi:10.1056/NEJMoa2306963 Ndumele CE, Neeland IJ, Tuttle KR, et al. A synopsis of the evidence for the science and clinical management of cardiovascular-kidney-metabolic (CKM) syndrome: a scientific statement from the American Heart Association. Circulation. 2023;148(20):1636-1664. doi:10.1161/CIR.0000000000001175 Khan SS, Matsushita K, Sang Y, et al. Development and validation of the American Heart Association’s PREVENT equations. Circulation. 2024;149(6):430-449. doi:10.1161/CIRCULATIONAHA.123.067626 Khan SS, Ning H, Shah SJ, et al. 10-year risk equations for incident heart failure in the general population. J Am Coll Cardiol. 2019;73(19):2388-2397. doi:10.1016/j.jacc.2019.02.057 Bozkurt B, Fonarow GC, Goldberg LR, et al. Cardiac rehabilitation for patients with heart failure: JACC expert panel. J Am Coll Cardiol. 2021;77(11):1454-1469. doi:10.1016/j.jacc.2021.01.030 Packer M. Leptin-aldosterone-neprilysin axis: identification of its distinctive role in the pathogenesis of the three phenotypes of heart failure in people with obesity. Circulation. 2018;137(15):1614-1631. doi:10.1161/CIRCULATIONAHA.117.032474 Lala A, Tayal U, Hamo CE, et al. Sex differences in heart failure. J Card Fail. 2022;28(3):477-498. doi:10.1016/j.cardfail.2021.10.006 Bozkurt B, Coats AJS, Tsutsui H, et al. Universal definition and classification of heart failure. Eur J Heart Fail. 2021;23(3):352-380. doi:10.1002/ejhf.2115 Hershberger RE, Givertz MM, Ho CY, et al. Genetic evaluation of cardiomyopathy—a Heart Failure Society of America practice guideline. J Card Fail. 2018;24(5):281-302. doi:10.1016/j.cardfail.2018.03.004 Levine GN, Cohen BE, Commodore-Mensah Y, et al. Psychological health, well-being, and the mind-heart-body connection: a scientific statement from the American Heart Association. Circulation. 2021;143(10):e763-e783. doi:10.1161/CIR.0000000000000947 Ezekowitz JA, Colin-Ramirez E, Ross H, et al. Reduction of dietary sodium to less than 100 mmol in heart failure (SODIUM-HF): an international, open-label, randomised, controlled trial. Lancet. 2022;399(10333):1391-1400. doi:10.1016/S0140-6736(22)00369-5

July 22, 2026: Your daily rundown of health and wellness news, in under 5 minutes. Today's top stories: Garmin launches CIRQA Smart Band, a screenless, subscription-free wearable, as hardware becomes table stakes and data interpretation becomes the real battleground Kalshi partners with AppliedXL to launch prediction markets forecasting clinical trial outcomes and FDA decisions, making drug development probabilities tradable American Heart Association guidance finds up to 400mg of caffeine daily is safe for most adults and may reduce heart disease risk, while warning on energy shots More from Fitt: Fitt Insider breaks down the convergence of fitness, wellness, and healthcare — and what it means for business, culture, and capital. Subscribe to our newsletter → insider.fitt.co/subscribe  Work with our recruiting firm → https://talent.fitt.co/ Follow us on Instagram → https://www.instagram.com/fittinsider/ Follow us on LinkedIn → linkedin.com/company/fittinsider  Reach out → insider@fitt.co 

Dave Rubin of "The Rubin Report" gives a first look at the stories you need to know to start your day, including President Donald Trump rejecting New York City Mayor Zohran Mamdani's threat to arrest Israeli Prime Minister Benjamin Netanyahu over the International Criminal Court's warrant, declaring America will protect its closest ally; a suspect in custody after an incendiary device attack outside a federal immigration building in Manhattan, where authorities say anti-ICE materials, a manifesto, and pellet guns were recovered; and a new American Heart Association study finding that drinking up to five cups of black coffee a day is safe for most adults and may reduce the risk of heart disease, stroke, type 2 diabetes, and other cardiovascular conditions; and much more.

P.M. Edition for July 20. The Magnificent Seven have dominated the stock market for years. But now, as markets reporter Hannah Erin Lang discusses, there are signs that everyday investors are buying fewer shares of the megacap tech companies as they look to find the next big AI stock. Plus, a judge puts a temporary restraining order on the $81 billion merger of Paramount and Warner Bros. Discovery. And how much coffee should you drink for a healthy heart? A new scientific statement from the American Heart Association has the answer… and it may be more than you think. Alex Ossola hosts. Sign up for the WSJ's free What's News newsletter. Learn more about your ad choices. Visit megaphone.fm/adchoices

CardioNerds co-chairs Dr. Dinu Balanescu and Dr. Billy Joe Mullinax, along with FIT lead Dr. Shiavax Rao, discuss the evolving landscape of randomized controlled trials in pulmonary embolism with Dr. Jay Giri, interventional cardiologist, Associate Professor of Medicine, and Director of the Cardiovascular Catheterization Laboratories at the Hospital of the University of Pennsylvania. This episode examines the historical evidence behind systemic thrombolysis, the emergence of catheter-directed therapies and mechanical thrombectomy, and the landmark RCTs – STORM-PE, PEERLESS, HI-PEITHO, and PEERLESS II – that are reshaping intermediate-risk PE management. The discussion highlights challenges in PE trial design, the critical importance of clinical deterioration as an endpoint, and why this era represents an unprecedented wave of evidence generation in PE. Audio editing for this episode was performed by CardioNerds Intern, Dr. Julia Marques Fernandes. Dr. Dinu Balanescu and Dr. Billy-Joe Mullinax are Co-chairs for the CardioNerds PE Series, developed in collaboration with the PERT Consortium.   Enjoy this Circulation 2022 Paths to Discovery article to learn about the CardioNerds story, mission, and values. CardioNerds Pulmonary Embolism PageCardioNerds Episode PageCardioNerds AcademyCardionerds Healy Honor Roll CardioNerds Journal ClubSubscribe to The Heartbeat Newsletter!Check out CardioNerds SWAG!Become a CardioNerds Patron! Pearls: Systemic thrombolysis in intermediate-risk PE reduces hemodynamic decompensation but at the cost of ~1.5–2% intracranial hemorrhage risk – a near-zero net benefit that has driven the search for safer catheter-based alternatives. “Focus on clinical deterioration, not mortality” – Due to crossover design in contemporary PE RCTs, control-arm patients who decompensate are rescued with advanced therapies, biasing mortality toward the null. Clinical deterioration is the most informative endpoint to watch in HI-PEITHO, PRAGUE-26, and PEERLESS II. HI-PEITHO is the first large RCT to demonstrate that catheter-directed fibrinolysis plus anticoagulation significantly reduces the composite of PE-related death, cardiorespiratory decompensation, or PE recurrence versus anticoagulation alone (RR 0.39; 95% CI 0.20–0.77; P=0.005), with no intracranial hemorrhage in either arm. The four major upcoming/recently reported PE RCTs (HI-PEITHO, PRAGUE-26, PEERLESS II, PE-TRACT) enroll progressively different risk populations – from the most enriched (HI-PEITHO) to the most permissive (PE-TRACT, which includes intermediate-low risk patients) – enabling a nuanced understanding of which patients benefit most from intervention. PE device clearance follows a fundamentally different FDA pathway than structural heart devices (single-arm safety/efficacy studies vs. mandated RCTs), yet market forces and clinical need have ultimately driven industry and government to sponsor large-scale RCTs – a lesson in how evidence development can evolve organically alongside regulatory frameworks. Notes: Notes drafted by Dr. Shiavax Rao. Question #1: What is the current evidence behind advanced PE therapies? Systemic thrombolysis: Sixteen RCTs over 40 years (1972–2014) enrolling nearly 2,000 patients have studied systemic thrombolysis in intermediate-risk PE. The landmark PEITHO trial (n=1,006) showed that tenecteplase reduced the composite of death or hemodynamic collapse (2.6% vs. 5.6%; P=0.015), driven primarily by reduced hemodynamic decompensation (1.6% vs. 5.0%; P=0.002). However, this came at the cost of increased major bleeding (6.3% vs. 1.5%; P

Episode 2821 - Vinnie Tortorich and Chris Shaffer discuss marketing ploys by Big Food, poorly done food studies, and that exercise is king. https://vinnietortorich.com/2026/07/exercise-is-king-episode-2821 PLEASE SUPPORT OUR SPONSORS Pure Vitamin Club Pure Coffee Club NSNG® Foods VILLA CAPPELLI EAT HAPPY KITCHEN YOU CAN WATCH THIS EPISODE ON YOUTUBE - @FitnessConfidential Podcast Vinnie's workout videos are available to purchase! Choose from a 2-day, 4-day, or 6-day workout–or buy all three at a discount! TO PURCHASE VINNIE'S WORKOUT VIDEOS, CLICK THIS LINK: https://vinnietortorich.com/workout Exercise is King Vinnie tells a story of "who is influencing whom?" (3:00) We can all do better, encourage each other, and be an example. The American Journal of Public Health recently published an article that highlights the connections between the marketing approaches of Big Food and Big Tobacco. (13:00) Lunchables are marketed as having a good amount of protein; however, the breakdown shows otherwise. (19:00) Big Food's entire goal is to get you addicted to their products. (28:00) You can find a book focusing on this topic in Vinnie's Book Club on his Amazon page. The book is "Salt, Sugar, Fat" by Michael Moss: https://www.amazon.com/dp/B07DF5QS3H?linkCode=ssc&tag=vinnitorto-20&creativeASIN=B07DF5QS3H&asc_item-id=amzn1.ideas.DLRU885XQ4BV&ref_=cm_sw_r_cp_ud_aipsfshop_aipsfvinnietortorich_109ME21EYG8380MBNX34_asin It appears that the American Heart Association finally agrees with what Vinnie has been saying for years: exercise is a poor way to lose weight. (43:00) Exercise is king for health and longevity! However, you need to eat real food, too. Another epidemiological study on meat consumption; however, it compares processed meats (like salami, pepperoni) to white meat, such as chicken (not processed). The comparison is off, so the outcome is off. (50:00) For a recent study on drinking tea, the article included all kinds of caveats, but the article about meat did not. Update on the PVC Magnesium product. (1:05:30) Anna's products are now linked to PureVitamin Club's website. Look under the "Food and Snacks" section to purchase them there, too. https://purevitaminclub.com/collections/food-and-snacks Vinnie hopes to add other products as well, all of which will be health-related. The NSNG® VIP GROUP IS NOW CLOSED AGAIN AS OF SUNDAY, MARCH 15TH Anna's next cookbook, Eat Happy Cocktail Hour, is filled with cocktails, mocktails, and appetizers and is available for pre-order right now. If you pre-order, you'll get bonus goodies! You can preorder from a wide variety of booksellers at https://eathappycocktailhour.com/ Save your receipt from wherever you preorder, you'll need it for your bonuses! Physical Release Date is October 2026 You can book a consultation with Vinnie to get guidance on your goals. https://vinnietortorich.com/phone-consultation-2/ More News Serena has added some of her clothing suggestions and beauty product suggestions to Vinnie's Amazon Recommended Products link. Self Care, Beauty, and Grooming Products that Actually Work! https://www.amazon.com/shop/vinnietortorich/list/3GPVU29UHHPMY?ref_=aipsflist Don't forget to check out Serena Scott Thomas on Days of Our Lives on the Peacock channel. "Dirty Keto" is available on Amazon! You can purchase or rent it here.https://amzn.to/4d9agj1 Please make sure to watch, rate, and review it! Eat Happy Italian, Anna's second cookbook, is available! You can go to https://eathappyitalian.com You can order it from Vinnie's Book Club. https://amzn.to/3ucIXm Anna's recipes are in her cookbooks, on her website, and on Substack —they will spice up your day! https://annavocino.substack.com/ PURCHASE DIRTY KETO (2024) The documentary launched in August 2024! Order it TODAY! This is Vinnie's fourth documentary in just over five years. Visit my new Documentaries HQ to find my films everywhere: https://vinnietortorich.com/documentaries Then, please share my fact-based, health-focused documentary series with your friends and family. Additionally, the more views it receives, the better it ranks, so please watch it again with a new friend! REVIEWS: Please submit your REVIEW after you watch my films. Your positive REVIEW does matter! PURCHASE BEYOND IMPOSSIBLE (2022) Visit my new Documentaries HQ to find my films everywhere: https://vinnietortorich.com/documentaries FAT: A DOCUMENTARY 2 (2021) Visit my new Documentaries HQ to find my films everywhere: https://vinnietortorich.com/documentaries FAT: A DOCUMENTARY (2019) Visit my new Documentaries HQ to find my films everywhere: https://vinnietortorich.com/documentaries