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What are you keeping and why? Let's discuss what's in your house, and why letting go feels so hard. It certainly helped us to understand ourselves and our family members. And maybe it will help you sort through your things and make some hard decisions.The things we keep representmemoriesidentityhopes for the futureguiltobligationcomfortand even fearListen to our episode on the art of Swedish Death cleaning HEREWe participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.Check out Kelly's app HERE !Anita's crush is the podcast Dish HEREKelly's crush is the follow up book to The Gentle Art of Swedish Death Cleaning - The Swedish Art of Aging Exuberantly! HERENeed help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 15:33 / 22:53See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

What if your burnout isn't just a scheduling problem — what if it's an energy problem? In this powerful episode, we explore why emotional healing is the missing piece in most nurse leader recovery plans. Drawing from Scripture, quantum physics, and the bioresonance technology behind WholeBio Insights, Shan reveals how unresolved emotions are stored in the body as disrupted frequencies — and why you can't think your way out of what your nervous system is holding.   Joined by Aligned leadership coach, Kassandra Hamilton, this episode takes you inside a real burnout story and the emotional work that made recovery possible. If you've tried all the strategies and still feel depleted, this episode will help you understand why — and what to do about it.   What You'll Learn in This Episode This episode is for you if you've been asking why rest isn't enough, why strategies aren't sticking, or why you still feel empty even when things look okay on the outside. Here's what we cover:   Why burnout is fundamentally an energy problem — and what the Bible has always said about it The Hebrew meaning of Shalom and why it's actually a description of energetic wholeness What Proverbs 17:22 reveals about the body-emotion-health connection (that science is just now catching up to) How Mark Virkler's "Prayers that Heal the Heart" framework reframes emotional healing as a clinical necessity, not just spiritual work What quantum physics' Observer Effect has to do with your burnout recovery Why emotion is literally "energy in motion" — and what happens when you suppress it How bioresonance technology (WholeBio Insights™) reads the body's frequencies to reveal emotional disruption before symptoms become diagnosable Kassandra Hamilton's honest account of what burnout looked and felt like as a leader — and what it actually took to heal   About Our Guest: Kassandra Hamilton Leadership & Alignment Coach, Best-Selling Author, Facilitator, Bioenergy & Sound Healer, and quality improvement specialist in the healthcare field.

What I do: I align high achieving women with a life that feels as good on the inside as it does on the outside. 

Think More energy, time freedom, less stress.

Kassandra's links: 
LinkedIn: https://www.linkedin.com/in/kassandra-hamilton-15b8b175/

Facebook: https://www.facebook.com/VancouverIslandHealing

Instagram: https://www.instagram.com/kassandra_hamilton/
Tik tok: https://www.tiktok.com/@coachingwithkassandra?_r=1&_t=ZS-92Iynv6RAoe

Website: www.readysetrealign.ca

Link to BestSeller Book: https://a.co/d/2yWISSu
   Key Takeaways "Burnout is not a scheduling problem. It is an energy problem. And you cannot fix an energy problem with a schedule change."   "Management and healing are not the same thing. You can manage your emotions your whole life and still be running on frequencies of unresolved fear and grief that are quietly depleting you."   "The word emotion is energy in motion. When we suppress emotion, we stop that energy in its tracks — and stored emotional energy creates disruption in the body's field."   Scriptures Referenced "He gives strength to the weary and increases the power of the weak."  — Isaiah 40:29, NIV "A cheerful heart is good medicine, but a crushed spirit dries up the bones."  — Proverbs 17:22, NIV "For as he thinks in his heart, so is he."  — Proverbs 23:7, NKJV "The Spirit of God has made me; the breath of the Almighty gives me life."  — Job 33:4, NIV "We all, who with unveiled faces contemplate the Lord's glory, are being transformed into his image with ever-increasing glory."  — 2 Corinthians 3:18, NIV   Resources & People Mentioned Books & Teachers cwgministries.org Prayers that Heal the Heart by Mark & Patti Virkler — glorywaves.org Charity Virkler Kayembe — quantum physics and Christian spirituality — "Quantum Glory" (framework referenced) — the science of heaven invading earth   Work With Shan: The Wholly Well Intensive To learn more: DM "360" to Shan on Instagram @theshanwright or email: hello@theshanwright.com Connect With Shan Instagram: @theshanwright Website: theshanwright.com Stress Less Community (Storehouse Wellness)    Shalom Shalom,  Xx, Shan  ……CONNECT…… The STAT Protocol: 5 min Emergency Reset Take the Free QUIZ-  Are you in burnout or just stressed??

What questions should you be asking the people you love while you still can?That's the thread running through this conversation with Dorothy Roberts, whose memoir The Mixed Marriage Projectstarted with a stack of boxes. After her parents passed, Dorothy opened them and found nearly 500 interviews her white father had conducted with interracial couples in Chicago, beginning in 1937, almost two decades before he married her Black mother. Inside were wild parties, a nudist camp, a turn-of-the-century club for mixed couples, and a file labeled number 224 that turned out to be about her.We talk about the interviews her father never published and the book contracts he kept walking away from, why she hid her father's whiteness from her Black classmates at Yale, and the painful stereotypes that surfaced even inside a club devoted to interracial marriage. We get into the big question at the heart of the memoir too: whether love, the everyday intimate kind, can actually dismantle racism. Spoiler, it's complicated.Dorothy Roberts is a scholar, professor, and author of five books on race, gender, and the systems that devalue Black women and mothers. The Mixed Marriage Project is her first memoir, built from the nearly 500 interviews her father left behind. If this conversation moved you, share it with a friend and leave a review and rating.Purchase The Mixed Marriage ProjectSupport the show:On PatreonBuy us a bookBuy cute merchSubscribe to the Babes in Bookland SubstackConnect with us and suggest a great memoir!Follow us on instagram! @babesinbooklandpod Thank you for listening!Xx, Alex Hosted on Acast. See acast.com/privacy for more information.

Ian de la Rosa debuta en el largometraje con Iván & Hadoum, una historia de amor ambientada en los invernaderos de Níjar (Almería) que llega a los cines tras conquistar el Teddy Award del Festival de Berlín y dos Biznagas de Plata en el Festival de Málaga. La película sigue la relación entre Iván, un joven trans, y Hadoum, una chica de origen marroquí, y aborda cuestiones como la identidad, el amor y la búsqueda de un futuro en un entorno marcado por la precariedad laboral y las diferencias culturales.La actualidad cultural pasa también por F. Javier Cárdenas García, ganador del XVIII Premio de Poesía Joven RNE-Fundación Montemadrid por Latitud cero, un poemario que ha destacado por su madurez poética y su capacidad para evocar Ecuador a través de imágenes sugerentes. Además, el Centro KBR de Barcelona dedica una gran retrospectiva a Minor White, una de las figuras fundamentales de la fotografía del siglo XX, con imágenes que se muestran por primera vez en Europa y que recorren algunos de los temas centrales de su obra, como el paisaje, el retrato y el desnudo masculino.El programa se acerca también a la historia del teatro y del cine español con la adaptación de La escopeta nacional, la célebre comedia de Luis García Berlanga y Rafael Azcona, dirigida por Juan Echanove en el Teatro Español de Madrid. Nuestra colaboradora Xaviera Torres recuerda el descubrimiento de los infrarrojos por parte del astrónomo William Herschel, mientras que Enrique Mejías nos lleva hasta el Festival de Bayreuth, el certamen creado por Richard Wagner que celebra este verano su 150 aniversario y sigue siendo una de las grandes citas de la música clásica internacional.Escuchar audio

Fue una de las figuras más singulares de la cultura española del siglo XX. Hija y nieta de maestras, la vida de Josefina Aldecoa (1926-2011) está marcada por la herencia pedagógica de la Segunda República. Imbuida del espíritu de la Institución Libre de Enseñanza, entendió la enseñanza como una forma de transformación social y la escritura como aliada de la memoria. Conoció de primera mano los sistemas educativos británico y estadounidense. En 1959, en plena dictadura, funda el Colegio Estilo en Madrid, un espacio de libertad, creatividad y pensamiento crítico por el que pasaron hijos de destacados escritores, cineastas, artistas e intelectuales de la época como Antonio Buero Vallejo, Alfonso Sastre, Fernando Vizcaíno Casas, Juan Antonio Bardem, Luis García Berlanga o Antonio López.Este documental sonoro, con guion de Minerva Oso y realización de Samuel Alarcón, recorre la biografía de la autora, desde su infancia en las montañas de León hasta la vinculación con la generación del 50. Fue crucial su relación con el escritor Ignacio Aldecoa, del que tomó el apellido tras su prematura muerte. En obras como 'Historia de una maestra', 'La enredadera' o 'Desde la distancia' plasmó su mirada sobre el contexto social y educativo de la España contemporánea. El programa cuenta con los testimonios de su hija, Susana Aldecoa; los periodistas Juan Cruz y Amelia Castilla; la doctora en literatura Estefanía Cabello; y la historiadora de la educación María del Mar del Pozo. Participan también los antiguos alumnos Teo Planell, Manuela Velasco, Marcos Giralt Torrente, Pablo Mingote y Carlos Saura, y profesoras del Colegio Estilo como Carmen Carbajosa. Además, suena la voz de la propia Josefina, conservada en el Archivo RTVE.Escuchar audio

En 1991, un equipo de geólogos de la Universidad de Boston realizó estudios sísmicos alrededor de la Esfinge y detectó una cámara rectangular de varios metros de profundidad exactamente debajo de sus patas delanteras. Nunca se excavó. En 1987, un equipo japonés de la Universidad de Waseda ya había reportado un túnel orientado de norte a sur corriendo bajo el cuerpo del coloso. Tampoco se investigó. En 2025, escaneos satelitales con tecnología de radar de apertura sintética detectaron estructuras subterráneas que se extienden dos kilómetros bajo el plateau de Giza. El Consejo Supremo de Antigüedades de Egipto no emitió ningún comentario.Pero la historia no empieza en el siglo XX. Heródoto describió en el año 450 a.C. pasajes subterráneos que conectaban las grandes pirámides. Cronistas árabes medievales documentaron entradas ocultas en los costados de la Esfinge con mecanismos que solo los sacerdotes iniciados conocían. Y una estela descubierta en 1858 afirma que el faraón Khufu encontró la Esfinge ya enterrada en arena cuando llegó a Giza — lo que significaría que el monumento precede incluso a las pirámides.En este episodio rastreamos el hilo completo. Los datos están. Los testimonios están. Lo que no está es la voluntad institucional de seguirlos hasta donde llevan. Hosted on Acast. See acast.com/privacy for more information.

durée : 00:58:35 - Concordance des temps - par : Jean-Noël Jeanneney - Écrivain de talent, Jacques Benoist-Méchin fut aussi l'un des plus ardents défenseurs de la collaboration avec l'Allemagne nazie. Condamné à mort puis gracié, il incarne l'un des parcours les plus déroutants du XXᵉ siècle. Éric Roussel revient sur ce destin paradoxal. - réalisation : Vincent Abouchar, Jeanne Guérout - invités : Eric Roussel Journaliste et écrivain Vous aimez ce podcast ? Pour écouter tous les épisodes sans limite, rendez-vous sur Radio France

Cette semaine, direction les ruelles pavées de Montmartre à la rencontre de deux peintres de la Buttes. Les deux amis sont contrariés : ils refusent d'être associés à ce qu'ils appellent "la foire aux croûtes" de la place du Tertre. On est en 1965 et déjà, ce quartier, où les plus grands peintres ont séjourné, devient un spot touristique qui perd de son authenticité. Aujourd'hui, ce phénomène a pris une telle ampleur que la place du Tertre est strictement réglementée par la mairie : 300 places sont réservées aux artistes peintres qui doivent passer devant une commission pour être autorisés à s'y installer. Est-ce un moyen d'éviter les croûtes ?***Crédits archive Extrait de l'émission radiophonique "Montmartre sur chevalet" - Reportage : Jacques Février - Première diffusion : 14/01/1965 - France Inter.***Crédits podcast Documentaliste : Anne Brulant - Textes : Lætitia Fourmond, Anouk Valverde - Restauration et mixage : Ian Debeerst - Enregistrement : Laurent Thomas - Voix off : Clara De Antoni - Musique(s) avec l'aimable autorisation d'Universal Production Music France - Chargée de production : Céline Amadori - Assistante de production : Ameline Tarnagda - Cheffe de projet : Lætitia Fourmond - Chargée de projet : Anouk Valverde, Edith Monnier - Responsable éditoriale : Zoé Macheret - Un podcast INA.

ARCHIVO MEXICANO DEL TERROR YO VENDÍ MI NOMBRE, GUADALUPE DUEÑAS Ninguna narración, por extraña que pueda parecer, se crea «de la nada»: todas dependen del contexto en el que vive su autor, aunque este no se refleje de manera directa. El caso de Guadalupe Dueñas es prueba de esto: su obra expresa una visión del mundo mediante los símbolos que le da la imaginación. Formada en una familia numerosa y de rígidos principios religiosos, Dueñas se educó primero en conventos. Allí comenzó a escribir por rebeldía, como una forma de oponerse a una vida que le resultaba asfixiante, y los pocos testimonios que hay sobre su vida sugieren constantemente la misma tensión entre convencionalismos y creatividad individual, entre pasado y presente. Más tarde hizo estudios de literatura en la UNAM, fue discípula de la escritora Emma Godoy y becaria del Centro Mexicano de Escritores. Un amigo de su familia, el sacerdote y crítico literario Alfonso Méndez Plancarte, le había recomendado que se dedicara exclusivamente a la prosa; Dueñas lo hizo así y exceptuando trabajos ocasionales como guionista y articulista toda su obra consiste en maravillosas narraciones breves. Tiene la noche un árbol (1958), se convirtió en poco tiempo en una de las cuentistas mexicanas más destacadas de mediados del siglo XX. Después publicó comparativamente pocos libros: No moriré del todo (1976), Imaginaciones (1977) y Antes del silencio (1991). Una producción de Historias para ser Leídas. La música pertenece a Epidemic Sound con Licencia autorizada para este Podcast. Este podcast se sostiene gracias a tu apoyo y a tus aportaciones, puedes aportar si lo deseas desde 1,99€ 💀 Por todas las horas de diversión: ✨ Gracias por tu escucha y hasta el próximo audio 🚀 🖤 Voz y sonido Olga Paraíso BIO Olga Paraíso: https://instabio.cc/Hleidas Escucha el episodio completo en la app de iVoox, o descubre todo el catálogo de iVoox Originals

Demografia to kwestia złożona, wieloaspektowa, jednak zaskakująco często poprzestaje się na dość powierzchownym jej omówieniu. Przejawia się to w odmienianiu przez wszystkie przypadki tzw. współczynnika dzietności, czyli średniej liczby dzieci przypadającej na jedną kobietę w dowolnie wybranej społeczności lokalnej (miejscowość, jednostka samorządowa, grupa etniczna, naród). Oczywiście artykuł publicystyczny nie jest odpowiednią formą do prowadzenia pogłębionych analiz statystycznych. Chciałbym natomiast zwrócić uwagę na jeden z rzadko (lub wcale?) poruszanych aspektów matematyki demograficznej.

¿Qué se esconde realmente tras las imponentes puertas de bronce de la Ciudad del Vaticano? Esta semana en El Club de los Curiosos nos alejamos de los mitos fáciles para adentrarnos en la realidad pura y dura de la institución más hermética del planeta. Una realidad repleta de intrigas palaciegas, crímenes sin resolver, persecuciones ideológicas y una sorprendente mirada hacia el cosmos. Descendemos a los sótanos de la historia para desvelar los secretos que custodian los 85 kilómetros de estanterías del Archivo Apostólico. Ponemos bajo la lupa uno de los expedientes más espinosos y oscuros del siglo XX: los documentos recientemente desclasificados del polémico pontificado de Pío XII y su vinculación con los nazis durante la Segunda Guerra Mundial. ¿Silencio cómplice, estrategia de supervivencia o diplomacia subterránea? Analizamos los cables reales que cruzaban el eje Berlín-Roma-Vaticano. Además, en este episodio abrimos una serie de melones espectaculares: Sangre en el Vaticano: Reviviremos el terror y el caos del Saco de Roma de 1527, la gran carnicería que puso en jaque al papado bajo las tropas imperiales. El profeta perseguido: La increíble y olvidada historia del monje español que, desde su exilio en Chile, desafió a la Santa Sede al vaticinar el fin inminente de la Iglesia, desatando una auténtica caza de brujas ideológica. Papas en el lado oscuro: Hacemos un repaso sin concesiones por aquellos pontífices cuyas biografías, excesos y crueldades los sitúan, históricamente, muchísimo más cerca del Anticristo que el mismísimo Ozzy Osbourne. La voz de los ángeles, el dolor de los hombres: Nos adentramos en la estremecedora historia de los castrati del Vaticano y el coro de la Capilla Sixtina. Crimen entre uniformes: Analizamos el misterioso y turbio caso del Guardia Suizo asesinado en extrañas circunstancias dentro de los muros vaticanos. ¿Crimen pasional o conspiración interna? Mirando al cosmos: Rompemos esquemas revelando la callada pero constante búsqueda del Vaticano de vida extraterrestre a través de sus astrónomos oficiales. Prepara el café, acomódate los auriculares y déjate atrapar por la rigurosidad y el misterio. Porque cuando el Vaticano cierra sus puertas... empieza la verdadera historia. Programa patrocinado por Truglup, 666 gracias. Visita la nueva web del Club de los Curiosos: www.elclubdeloscuriosos.com Quieres anunciarte en este podcast? Hazlo con advoices.com/podcast/ivoox/614720 Libro Fenómenos Para-anormales: https://www.exlibric.com/libro/fenomenos-para-anormales/ Youtube del Club de los Curiosos: https://www.youtube.com/channel/UC6KgIO7QIVyYNY8LDbVvErA Nuevo Telegram del Club de los Curiosos: https://t.me/elclubdeloscuriosos Hazte #Mecenas del Club para apoyar la lucha de los anormales por el mundo o bien haz una donación por #Bizum indicando tu nombre y la palabra anormal al 688 323 552 Web del Glan Lidel: www.albertoenriquepons.es Libro de Mark Knopfler Málaga 360: https://360malaga.es Web de Lázaro Chico (anversoabogados): https://anversoabogados.com/ Telf:951 438 863 No dejes de dejar comentarios, todos serán leídos y respondidos en el próximo programa, se os quiere. Estamos en Twitch, Instagram, TikTok, Youtube, Facebook y Twitter. Nuestro Mail de Contacto: albertoenriquepons@gmail.com Facebook del Club de los Curiosos: https://www.facebook.com/elclubdeloscuriosos Instagram del Club de los Curiosos: https://www.instagram.com/elclubdeloscuriosos/?hl=es Canal de Gabriel Santana: https://www.youtube.com/@TanaPORELMUNDO/videos WhasApp de nuestra Medium Amanda Solano: 661737356 CENTRO NASHIRA de Amanda Solano: https://centronashira.es/ Canal Cocina con Marisa y Thermomix: https://www.youtube.com/@cocinaconmarisaythermomix6053 Tema Cierre: Walker Texas Tema Cabecera: Makuki Ivoox: https://go.ivoox.com/rf/119655736 El Baúl de Margarita: https://instagram.com/elbauldemargarita8?igshid=MzRlODBiNWFlZA== Ivoox de Narraciones de un Burro: https://www.ivoox.com/podcast-narraciones-burro_sq_f1507763_1.html Podcast de nuestro amigo Francisco Bustamante, el éxtasis de las abejas:https://go.ivoox.com/sq/925346 Web donde escribe Alejandro Cervilla:https://www.motosan.es/ Podcast del Doctor Osorio y sus alumnos: https://go.ivoox.com/sq/2406678 Instagram Postureo en la Cocina de Manu Calatrava: https://www.instagram.com/postureoenlacocina?igsh=YjVnbXpyMWZnMDN5&utm_source=qr Instagram Marta Gonzalez Vallovera: https://www.instagram.com/artealday/?hl=es.Javier Si te gustan los animales visita www.airedelatoscana.com Escucha el episodio completo en la app de iVoox, o descubre todo el catálogo de iVoox Originals

Michał Nowak przedstawia sytuację na frontach wojny rosyjsko-ukraińskiej w dniu 10 czerwca 2026 roku.

Molina de Segura conmemora su época dorada como potencia conservera con una serie de actividades que arrancan con una exposición en el Mudem del 13 al 16 de junio. Esta muestra presenta etiquetas, latas antiguas y el corto documental "Molina de Segura: cuando sonaban las sirenas", el cual narra la transformación de la ciudad a finales del siglo XIX y principios del XX a través de las voces de quienes trabajaron en las fábricas, destacando especialmente el papel de las mujeres en esta industria. Entre las curiosidades de la exposición se encuentra la historia de un productor local que registró la imagen de Mickey Mouse para productos alimenticios antes que Disney. La celebración se completará los días 19 y 20 de junio en la Plaza de España con Gastrolata, un festival que combinará la degustación de conservas gourmet con talleres infantiles, showcookings y música en directo para reivindicar la alta gastronomía del producto en lata.

Le mix de Dannic dans 103 Klubb le 06 Juin 2026 de 19H à 20H Tracklist: Mixmasters, Mellizos - My Life Is A Disco (In The Mix) *** Alenx B - House Religion *** Ellison - Set Fire To The Rain x Rain (Hard Mashup) *** Melsen vs. D.O.D, Carla Monroe - What I Feel x Still Sleepless (Melsen Mashup) *** NIIKO x SWAE, KSHMR feat. DEV - Bass Down Low *** Dannic & Dastic - Dim Your Light *** Nicky Romero & Deniz Koyu vs. Post Malone - Tomorrow Comes vs Chemical (SunJay Mashup) *** Dannic - My Mind (VIBR Edit Vincent Brouwer) *** Firebeatz - Pump That Bass *** Afrojack - Polkadots (Reloader 2026 Edit) *** Yves V & VIKTOR - Fit The Tempo *** Daddy's Groove vs. Justin Michael vs. Dada Life - Born To Follow Synthemilk (Dyro Mashup) *** Mike Williams & Phillip Strand x Kesha - All My Life x Die Young (AIRAES Mashup) *** Dannic - Chasing Freedom vs Beauty & The Beat (Dannic Mashup) *** Dannic vs. Delerium vs. The XX vs. Tiësto - Tombo vs Silence vs... (Dimitro Re-Cut Mashup) *** Dannic - I'll Be There For You *** Yohann Warren feat. Scarlett - Think About *** Dannic vs. Calvin Harris & Alesso & Hurts vs. Martin Garrix & Matisse & Sadko - Wake Me Up vs Under Control vs... (SunJay Mashup) *** Skrillex, Ahadadream, Raf Saperra - Bass Dhol *** Tiësto, Olivia Sebastianelli - Don't Lose Your Head *** Swedish House Mafia x Daft Punk, Pejt - Don't You Worry Child x One More Time (AIRAES Mashup)

No episódio, Ana Frazão conversa com Maria de Lourdes Rollemberg Mollo, Professora Titular de Economia da Universidade de Brasília - UnB sobre Economia Política. A entrevistada explica por que a economia é sempre política, os meios pelos quais se tentou despolitizá-la a partir da revolução marginalista e como a Economia Política foi crescentemente recuperando o seu prestígio ao longo do século XX. A professora trata também das principais diferenças entre as visões ortodoxa, marxista e pós-keynesiana na compreensão dos principais assuntos vinculados à Economia Política, como a relação entre economia real e economia monetária, o papel do Estado e da política econômica para o controle da inflação e do desemprego, e os efeitos da financeirização. Na parte final, a professora aborda as discussões mais recentes sobre desenvolvimentismo e políticas econômicas alternativas.

En esta primera parte de la saga sobre Francisco Franco, Leyendas Legendarias explora los orígenes de uno de los dictadores más importantes y polémicos del siglo XX. Lejos de la imagen clásica del líder carismático y dominante, descubrimos a un hombre inseguro, acomplejado, rechazado por su padre y eclipsado por un hermano mucho más exitoso. El episodio recorre su infancia, su paso por la academia militar, la brutal campaña colonial en Marruecos y la construcción del mito de la "Baraka", la supuesta suerte sobrenatural que convenció a muchos de que estaba destinado a gobernar España. También puedes escucharnos en Spotify, Apple Podcasts, Amazon Music o tu app de podcasts favorita. Apóyanos en Patreon:https://www.patreon.com/leyendaspodcast Apóyanos en YouTube:https://www.youtube.com/c/leyendaslegendarias/join Visita nuestra página para ver contenido extra:https://www.leyendaslegendarias.com Síguenos:https://instagram.com/leyendaspodcasthttps://www.tiktok.com/@leyendaspodcasthttps://twitter.com/leyendaspodcasthttps://facebook.com/leyendaspodcast #Podcast #LeyendasLegendarias

Is your home ready for summer? School's out and we're here to help get your home ready for the summer months.Anita is focused on entertaining, and has lots of ideas for your table. You can find the items she mentioned in the episode HERE.Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.We participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.The rattan pagodas are HERE and tole pagodas are HERE.Discover & explore Kelly's Home Design Coach App HERE. Take the quiz and get your personalized plan!Anita's crush is the movie Impromptu about author George Sand (Judy Davis) who wears men's clothing in 1830's and falls in love with composer Frédéric Chopin (Hugh Grant). It's fun and not too serious. HERE. Kelly's crush is the gorgeous book Glorious Gardens: Private Edens of the World's Leading Interior Designers. Get your copy HERE.Need help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 7:03 / 14:29See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Links:Canal jorge Uesu falando do Maik https://www.instagram.com/reels/DZYcFzzqUSI/Curso Cabala e Magia Planetária com Marcos Keller: https://www.sympla.com.br/evento-online/fundamentos-e-pratica-da-cabala-hermetica-e-magia-planetaria/3424981Camisetas Mundo Freak: https://umapenca.com/mundofreak/loja/?srsltid=AfmBOoqsX2AWTRFB-FUyRZnCnRrfrZjDjoCtaZalh2J2cF_lCI4OiqaUNa live de hoje do Mundo Freak, vamos mergulhar na longa e fascinante história do ocultismo ocidental, um conjunto de tradições esotéricas, filosóficas e espirituais que atravessa séculos e ajudou a moldar parte do imaginário do Ocidente.A conversa começa nas origens mais antigas desse pensamento, passando por Alexandria, pelo hermetismo, por Hermes Trismegisto, pelo gnosticismo, pelo neoplatonismo e pelas formas como esse conhecimento sobreviveu, foi codificado e transformado ao longo da Idade Média. A pauta também entra em temas como alquimia, cabala, grimórios medievais e a ideia de que o ocultismo sempre esteve ligado à busca por uma gnose, um conhecimento interior e transformador.Ao longo da live, a gente avança para o Renascimento, quando magia, filosofia e ciência ainda caminhavam lado a lado, e passa por figuras fundamentais como John Dee, além do surgimento de tradições e estruturas como rosacrucianismo, maçonaria, Éliphas Lévi, Helena Blavatsky e a Golden Dawn, que ajudaram a organizar o ocultismo moderno.Na reta final, o papo chega ao século XX com nomes incontornáveis como Aleister Crowley, Jack Parsons e Anton LaVey, mostrando como o ocultismo saiu dos círculos iniciáticos e passou a dialogar com contracultura, mídia, ciência, espiritualidade alternativa e cultura pop.Se você gosta de história do esoterismo, magia cerimonial, sociedades secretas, alquimia, hermetismo, cabala, Crowley, satanismo moderno, mistérios históricos e da relação entre ocultismo e cultura contemporânea, essa live é para você.▶ Assista e participa no chat: qual fase da história do ocultismo mais te intriga, Antiguidade, Renascimento, sociedades secretas ou século XX?As Duas Vidas de Rudolf: https://open.spotify.com/show/3okz3u1rW9O0Lz3fdfmqZiApoie o Mundo Freak: https://apoia.se/confidencialRafael Jacauna Autor (Instagram): https://www.instagram.com/rafaeljacaunaautor/Lynda MD: http://lyndamd.com.brAnuncie com a Paratopia: https://www.instagram.com/paratopiapodcast/Edição: https://www.instagram.com/instadogrmachado/#MundoFreak#LiveMundoFreak#Ocultismo#OcultismoOcidental#Hermetismo#Alquimia#Cabala#AleisterCrowley#SociedadesSecretas#Esoterismo

En esta primera parte de la saga sobre Francisco Franco, Leyendas Legendarias explora los orígenes de uno de los dictadores más importantes y polémicos del siglo XX. Lejos de la imagen clásica del líder carismático y dominante, descubrimos a un hombre inseguro, acomplejado, rechazado por su padre y eclipsado por un hermano mucho más exitoso. El episodio recorre su infancia, su paso por la academia militar, la brutal campaña colonial en Marruecos y la construcción del mito de la "Baraka", la supuesta suerte sobrenatural que convenció a muchos de que estaba destinado a gobernar España. También puedes escucharnos en Spotify, Apple Podcasts, Amazon Music o tu app de podcasts favorita. Apóyanos en Patreon:https://www.patreon.com/leyendaspodcast Apóyanos en YouTube:https://www.youtube.com/c/leyendaslegendarias/join Visita nuestra página para ver contenido extra:https://www.leyendaslegendarias.com Síguenos:https://instagram.com/leyendaspodcasthttps://www.tiktok.com/@leyendaspodcasthttps://twitter.com/leyendaspodcasthttps://facebook.com/leyendaspodcast #Podcast #LeyendasLegendarias

What would it mean to build women's sports around women's bodies? This week, my friend Becca returns to discuss Lauren Fleshman's memoir "Good for a Girl". What starts as a running memoir quickly reveals itself as something far bigger: an investigation into the science of female puberty and athletics, the eating disorder epidemic inside women's collegiate and professional sports, and one woman's relentless fight to change a sport she loves from the inside out. We discuss Lauren's unconventional path to running, the ways puberty has been treated as "the one injury a girl can't come back from," the NCAA's staggering lack of policy around eating disorders in women's athletics, Lauren's famous Nike "Objectify Me" campaign, and the ways female athletes are still being failed by the systems built to protect them.This episode is for every woman who was ever told she was good — for a girl. The runners, the former athletes, the moms of daughters in sports, and anyone who has ever felt their body was working against them instead of for them.If this conversation resonated with you, the best thing you can do is share it with a friend who needs to hear it, and rate and review the show wherever you listen. It makes a huge difference!Purchase "Good for a Girl"Other Links:https://www.milesplit.com/articles/211759/dear-younger-me-lauren-fleshman (Lauren's letter to her younger self)Follow Becca's bookstagram: @bookedwithbecca and her new running account: @run.with.beccaSupport the show:On PatreonBuy us a bookBuy cute merchSubscribe to the Babes in Bookland SubstackConnect with us and suggest a great memoir!Follow us on instagram! @babesinbooklandpod Thank you for listening!Xx, Alex Hosted on Acast. See acast.com/privacy for more information.

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

Carlos Iribarren | Tras un primer capítulo dedicado a los mejores sinfonistas portugueses de los siglos XIX y XX, hoy completamos nuestra serie con la segunda entrega, donde podemos escuchar las obras de 3 autores: el contemporáneo Luis Cipriano y 2 compositores que forman parte del panteón de ilustres de la música orquestal lusa, Fernando Lopes-Graça y Joly Braga Santos. Su música es realmente emotiva y contiene momentos con una tensión dramática que estamos seguros de que te va a conmover. Carlos y Mario comentan las obras y detalles de las vidas de estos 3 autores para que nos acompañes en un nuevo episodio sinfónico de Hoy Toca, el programa de Clásica FM que te quiere sorprender.

** VIDEO EN NUESTRO CANAL DE YOUTUBE **** https://youtube.com/live/Nx-a1odwDbo +++++ Hazte con nuestras camisetas en https://www.bhmshop.app +++++ ¿Sabías que George S. Patton, el mítico general de los tanques, fue hasta finales de los años 30 un apasionado defensor de la caballería a caballo? Diseñó su propio sable modelo 1913, escribió en la Cavalry Journal defendiendo que “si el caballero medieval se adaptó a la pólvora, nosotros podemos adaptarnos al motor”… y sin embargo, nunca fue un converso inmediato a los blindados. En este nuevo programa de Bellumartis Historia Militar, el historiador Fernando del Castillo, autor de "Patton: Soldado en dos guerras mundiales" nos desmonta con rigor y fuentes primarias la cara menos conocida del “Blood and Guts”: su profunda nostalgia por el caballo, sus dudas reales sobre la mecanización total y cómo la campaña de África del Norte (especialmente la batalla de El Guettar) supuso su punto de no retorno. Desde West Point y su experiencia con tanques en la Gran Guerra, pasando por los debates del Ejército estadounidense entre tradición y modernidad, hasta el desierto tunecino donde sus Sherman demostraron que la verdadera caballería del siglo XX era blindada. En este episodio descubrirás: Por qué Patton volvió a la caballería después de mandar tanques en 1918 Sus artículos de los años 30: entre el escepticismo y la visión de futuro La llegada a África tras el desastre de Kasserine: disciplina, audacia y el bautismo de fuego blindado El Guettar: la primera gran victoria americana contra los Panzers alemanes Cómo el espíritu del jinete audaz se trasladó al interior de un tanque Análisis objetivo, sin mitos hollywoodenses, con el rigor histórico que nos caracteriza. Porque entender cómo Patton vivió esa transición tecnológica nos ayuda a comprender mejor los conflictos actuales: movilidad, adaptación y audacia siguen siendo decisivas. Si te gusta la historia militar contada con honestidad, dale like, comenta y activa la campanita. ¿Crees que Patton habría preferido seguir cargando con sable si la tecnología lo hubiera permitido? ¿O el cambio era inevitable? Cuéntamelo abajo SUSCRÍBETE para no perderte ningún programa y únete a nuestra comunidad de apasionados por la historia militar, la geopolítica y los conflictos del mundo. Apóyanos para seguir creando contenido riguroso e independiente: Patreon: https://www.patreon.com/bellumartis PayPal: https://www.paypal.me/bellumartis Bizum: 656 778 825 Síguenos también en redes: Instagram: https://www.instagram.com/bellumartis Twitter / X: https://twitter.com/Bellumartis #Patton #UltimoJinetes #CaballeriaBlindada #SegundaGuerraMundial #HistoriaMilitar #FernandoDelCastillo #WWII #Tanques #ElGuettar #AfricaDelNorte #Bellumartis #GuerraMecanizada #GeneralPatton

Nurse leader, You gave everything today. Now it's time to give it to God. If you're listening to this on your commute home — in your car, on the train, walking to your front door — this episode was made for this exact moment. You just survived a shift that had no business being that hard. You were in charge. The floor was chaos. There was a patient loss, coworker conflict, impossible decisions made in seconds, and nurses running on empty. And somehow, you were the one holding it all together. Your body is exhausted. Your nervous system is still activated. And if you go to sleep without releasing what you absorbed today, you'll carry it into tomorrow — because the body keeps the score. In this Breathe & Be Still episode, we do something simple and sacred: we release it all through guided breath prayer — and receive the peace God promises in return. In this episode, you'll: Move through a 3-minute guided breath prayer rooted in Philippians 4:6-7 Learn why releasing post-shift emotions isn't weakness — it's nervous system wisdom and biblical obedience Discover the difference between true shalom and just "being tired" Stop the rumination loop so your mind and body can actually restore overnight This episode is for you if: You're a nurse, charge nurse, or nurse leader coming off a hard shift You struggle to "turn off" your brain after work You've experienced patient loss, compassion fatigue, or secondary trauma You're a faith-based nurse looking for a spiritual practice that actually fits your life You want to protect your peace without toxic positivity or empty self-care advice Scripture Featured:

* Apoie a Cultura: Chave Pix: 7296e2d1-e34e-4c2e-b4a0-9ac072720b88Imagine viver em um país que acabou de declarar a sua liberdade, mas ser tratada, mesmo depois de morta, como se o seu corpo pertencesse ao Estado. Jacinta Maria de Santana foi uma mulher negra cuja existência foi marcada pela pobreza e pela vulnerabilidade na São Paulo do início do século XX. No entanto, o verdadeiro e estarrecedor impacto de sua história veio após a sua morte: seu corpo foi ilegalmente retido por quase trinta anos pela Faculdade de Direito da USP para servir de objeto de trotes e estudos, tornando-se o estopim para debates profundos sobre dignidade humana, racismo científico e direitos civis no Brasil.Essa história passou décadas escondida nos porões da burocracia e do preconceito, mas que precisa ser contada para que nunca mais se repita. A trajetória de Jacinta Maria de Santana, mesmo sem querer, desafiou uma das instituições mais poderosas do país e ajudou a mudar a lei e os protocolos de como os hospitais e as universidades lidam com corpos não reclamados, garantindo que o respeito e a dignidade não terminem com o último suspiro. Eu sou Tania Barros. Seja Bem-vindo a mais uma história no Canal Loucos por Biografias. Essa é a nossa história de hoje. Se você gostou deixe seu like, faça seu comentário, compartilhe essa biografia com mais pessoas. Vamos incentivar a cultura em nosso pais. Encontro voces na próxima história. Até lá! (Tania Barros)- Contato: e-mail - taniabarros339@gmail.com

– Kultura chorwacka kształtowała się w obrębie tradycji śródziemnomorskiej. Wszystko, co najważniejsze w kulturze chorwackiej wiąże się z morzem. – mówi Maciej Czerwiński.  Z filologiem, tłumaczem i slawistą rozmawiamy o dziejach Chorwacji, ale także o literaturze, która pomaga zrozumieć ten kraj i jego związek z morzem. Omawiamy między innymi eseje Miroslava Krležy, który zbudował podstawy do wyborów geopolitycznych Jugosławii w XX wieku. Nie pomijamy także wątków translatorskich. Czy warto na nowo tłumaczyć "Most na Drinie" Iwo Andricia? To wybitne dzieło noblisty do dziś jest instrumentalizowane przez Chorwatów, Boszniaków i Serbów. – Istniejący przekład jest świetny, ale pochodzi z poprzedniej epoki. Nowe spolszczenie Andricia ma być misją podobną do wznowienia Buddenbrooków Manna, który niedawno zrealizowano. Tych dwóch autorów ciekawie ze sobą koresponduje - mówił Czerwiński. Maciej Czerwiński to filolog, slawista, tłumacz, krytyk literacki, dyrektor Instytutu Filologii Słowiańskiej UJ. Realizowane w ramach projektu Archipelagos finansowanego przez Unię Europejską. Dofinansowano ze środków Ministra Kultury i Dziedzictwa Narodowego pochodzących z Funduszu Promocji Kultury

En este episodio presentamos el personalismo como una filosofía nacida de la crisis del siglo XX para defender la dignidad irreductible de la persona humana frente al individualismo liberal y los colectivismos. Veremos cómo, para autores como Emmanuel Mounier, la persona no puede reducirse a un objeto ni a una función social: es libre, responsable, creativa, encarnada y esencialmente abierta a la comunidad. Abordaremos también el contexto histórico en que surgió esta corriente, especialmente la crisis de 1929 y la convicción de que el mal moderno era a la vez económico, moral y espiritual. Desde ahí, el personalismo se presenta como una propuesta de renovación humana y comunitaria, una filosofía viva que quiso responder a la crisis de la civilización moderna sin encerrarse en un sistema rígido. ÍNDICE 1. Qué es el personalismo 2. Las características de la persona 3. La persona está encarnada e históricamente situada 4. La persona es constitutivamente comunitaria 5. Mounier y la revista Esprit 6. El contexto histórico en que surgió el personalismo 7. Entre capitalismo deshumanizado y totalitarismos Música de la época: Concerto Grosso (Vaughan Williams) compuesto en 1950. Imagen: El Personalismo. Pulsen un Me Gusta y colaboren a partir de 2,99 €/mes si se lo pueden permitir para asegurar la permanencia del programa ¡Muchas gracias a todos!

Los dos años decisivos que condujeron al colapso repentino de la URSS, contados por sus principales protagonistas. Moscú, 31 de diciembre de 1991: la bandera roja del Kremlin es arriada y sustituida por la tricolor de Rusia, marcando el fin de la Unión Soviética y de sus ideologías. ¿Quién hubiera imaginado que solo dos años después de la caída del Muro de Berlín, los ciudadanos soviéticos derribarían las estatuas de Lenin en el mismo lugar donde nació el comunismo? Entre 1989 y 1991 tuvo lugar una sucesión de acontecimientos impredecibles e inevitables, a través de una sacudida aceleración de la historia, que puso de manifiesto la rivalidad entre dos hombres y su lucha por el poder: Gorbachov, lastrado por los resultados económicos de su perestroika, y Yeltsin, encarnación de las esperanzas del pueblo ruso. Ilustrado con entrevistas a protagonistas clave, incluido el propio Mijaíl Gorbachov, este documental narra, día a día, los dos últimos años decisivos de la URSS y arroja luz sobre las luchas de poder que llevaron al colapso repentino de uno de los imperios más totalitarios del siglo XX. Documentario: The Last Days of the USSR (2010) Dirigido por: Jean-Charles Deniau & Sergey Kostin Producción: ROCHE Producions

Cap

Renée Jolivet est une ancienne modèle qui posait pour le peintre Auguste Renoir. On est en 1957 et Renée a 72 ans. Elle raconte avec admiration et nostalgie sa rencontre avec l'artiste, et comment elle est devenue sa muse alors qu'elle était encore adolescente. Aujourd'hui, difficile de l'écouter sans questionner cette relation. Leur lien, pourtant jamais décrit comme romantique, interroge sur les dynamiques de pouvoir, de consentement, et sur la place des jeunes femmes dans l'art à la fin du XIXe siècle. Jusqu'en juillet 2026 le musée d'Orsay consacre une double exposition à Renoir, l'occasion de revisiter son œuvre à la lumière de ces questions. ***Crédits archive Extrait de l'émission radiophonique "La chasse aux souvenirs". Réalisation : Jacques Guinchard - Production : André Gillois - Première diffusion : 16/02/1957 - Chaîne Parisienne. ***Crédits podcast Documentaliste : Anne Brulant - Textes : Lætitia Fourmond, Anouk Valverde - Restauration et mixage : Ian Debeerst - Enregistrement : Laurent Thomas - Voix off : Clara De Antoni - Musique(s) avec l'aimable autorisation d'Universal Production Music France - Chargée de production : Céline Amadori - Assistante de production : Ameline Tarnagda - Cheffe de projet : Lætitia Fourmond - Chargée de projet : Anouk Valverde, Edith Monnier - Responsable éditoriale : Zoé Macheret - Un podcast INA.

Todo el mundo conoce el extraordinario sentido de la orientación de las palomas mensajeras: aunque te las lleves a centenares de kilómetros de su palomar son capaces de encontrar el camino de vuelta. ¿Cómo hacen para calcular estos viajes? Ésta es una pregunta que la biología está tratando de responder desde mediados del siglo XX. Hoy sabemos que se debe a una combinación de habilidades: observación del sol, uso de olores familiares para encontrar el camino y también percepción del campo magnético terrestre. Las palomas, y también muchos otros animales migratorios, parecen ser capaces de "leer dónde está el norte" gracias al magnetismo de nuestro planeta. Pero, de nuevo: ¿cómo logran hacer tal cosa? Ha de haber algo en su cuerpo que interaccione con el campo magnético y traslade esa información a su mente. A día de hoy, junio de 2026, todavía no tenemos una respuesta definitiva a esa pregunta, pero en el programa de hoy os contamos un descubrimiento reciente: que el sentido magnético podría residir en ciertas células del sistema inmunitario presentes en el hígado de las palomas. Esas células tienen en su interior partículas magnéticas, y parece que sin ellas no son capaces de orientarse. El artículo en que nos hemos basado para este programa es "Homing pigeon navigation relies on superparamagnetic macrophages under overcast conditions", de Clivia Lisowski et al. Podéis encontrarlo aquí: https://www.science.org/doi/10.1126/science.ady2486 Si os interesa este asunto hemos hablado ya un par de veces del sentido magnético de los animales. En el capítulo s09e15 de La Brújula de la Ciencia os hablamos de los otros dos mecanismos que se han propuesto para explicar la magnetorrecepción en aves. Y si vais a nuestro pódcast hermano, Aparici en Órbita, en el episodio s07e24 os hablamos de magnetorrecepción en tortugas marinas. Este programa se emitió originalmente el 1 de junio de 2026. Podéis escuchar el resto de audios de La Brújula en la app de Onda Cero y en su web, ondacero.es

Steve Hillage and Miquette Giraudy talk to Kevin Paul about their techno project System 7 and ambient project Mirror System, discussing their studio setup, live performances and creative approach to making music.Chapters00:00 - Introduction00:38 - Current Projects01:20 - System 7 and Mirror System02:20 - Songwriting Process04:27 - Using Gong-Influenced Sounds05:17 - Main Creative Tools06:19 - Learning From DJ Experience06:55 - Collaborative Workflow08:49 - Structuring Albums09:27 - Streamlined Studio Setup11:20 - Limitations and Finishing Tracks13:31 - Live Performances16:58 - 10 Records and Collaborations21:02 - The Future Of System 726:18 - Mirror System for Ambient Music#HelixLT #Logic #ProTools #Ableton #AnimoogZhttp://www.a-wave.comhttps://www.facebook.com/System7pagehttps://www.facebook.com/MirrorsystemSteve Hillage BiogSteve Hillage is an English guitarist, producer and electronic music pioneer best known for his work with the psychedelic prog band Gong, his influential solo albums of the 1970s and the electronic duo System 7. Renowned for his distinctive “glissando” guitar style, Hillage later became a key figure in ambient techno and psychedelic dance music, blending electronic production with live guitar performance alongside longtime creative partner Miquette Giraudy.http://stevehillage.comhttps://www.facebook.com/stevehillagebandhttps://www.instagram.com/stevehillageMiquette BiogMiquette is a French keyboardist, vocalist and composer known for her work with Gong, Steve Hillage and System 7. A major creative force behind System 7's ambient and techno sound, she has collaborated with Hillage since the 1970s, contributing keyboards, vocals, programming and production across a wide range of psychedelic, ambient and electronic music projects.https://www.instagram.com/miqui7777https://www.facebook.com/p/Miquette-Giraudy-100063115306402Kevin Paul BiogKevin Paul started his career as a DJ but quickly found his passion was sound engineering. His first audio job was at Soho Studios in 1991, moving to Konk Studios six months later, where he worked alongside successful producers and engineers such as Bob Clearmountain, Adam Mosley, Pascal Gabriel and Gil Norton, as well as bands such as The Kinks, Galliano, Terrorvision, UFO and Elastica. After working on archiving the Depeche Mode back catalogue in 1994, he was offered an engineering role at Mute Records' in-house studio, which eventually lead to a position as Head Engineer, which gave him access to the entire Mute Records roster. Highlights include mixing Goldfrapp's 'Felt Mountain', David Bowie's 'Hours' and Nick Cave's 'No More Shall We Part'. He also worked in 5:1, mixing Moby's 'Hotel', Goldfrapp's 'Black Cherry' and more for DVD.In 2004 Kevin went freelance and re-mixed the entire Depeche Mode and Nick Cave and the Bad Seeds back catalogues for SACD/DVD. Since 2008, Kevin Paul has been in charge of mixing and remixing performances at the iTunes Festivals in the UK and Germany. He has mixed over 100 artists to date, including Adele, Ed Sheeran, Alicia Keys, Nick Cave & The Bad Seeds, The XX, Calvin Harris, Foo Fighters, Jack White, Linkin Park, Florence & The Machine, Deadmau5, David Guetta, Jessie J., Norah Jones, Oasis, Mumford & Sons, N.E.R.D., Lykke Li, James Blunt, KT Tunstall, Hot Chip, Paul Weller and many more.He continues to record, engineer, produce and mix many projects in music and film, runs the mixing and surround mixing modules for the Masters Degree course at UK's Westminster University and divides his time between London and Berlin. Recent works include the International selling new album by Nick Cave & The Bad Seeds and mixing the latest album of Denmark's “Dúné” with the first single premiering at the Danish Music Awards, plus the latest iTunes Music Festival.http://www.kevin-paul.com/Catch more shows on our other podcast channels: https://www.soundonsound.com/sos-podcasts

Con Carlos López I Nacida en el corazón de la tradición musical india, la tabla emprendió un viaje inesperado hasta encontrarse con uno de los lenguajes más libres y creativos del siglo XX: el Jazz. El diálogo entre sus complejos patrones rítmicos y el espíritu de la improvisación abrió nuevas sendas sonoras que transformaron a músicos y oyentes por igual. Te invitamos a descubrir las historias, los encuentros y las grabaciones que marcaron esta fascinante convergencia cultural. Déjate envolver por el sonido vibrante de la tabla y acompáñanos en un recorrido por las huellas que ha dejado en la constante reinvención del Jazz.

Are you frustrated with your decorating project? Deep breaths and let's talk about how to keep from being overwhelmed with your project. We've got several great tips on how to approach a project in a way that doesn't feel so stressful.Join Kelly on her app Home Design Coach. Click HERE to learn more!Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.We participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.Anita's crush is a movie that used to be played every Christmas, although it's not a Christmas movie. It's Philadelphia Story with Katherine Hepburn, Cary Grant, and Jimmy Stewart. You can find it HERE. Kelly's crush is the yummy & good for you Oatmeal Cups recipe from the NYT. Try it HERE.Need help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 5:23 / 19:04See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

What would change if women stopped being silent about the hardest parts of their lives?This week, I sit down with psychologist, author, and advocate Dr. Jessica Zucker, the woman behind the viral #IHadAMiscarriage movement, to discuss her memoir I Had a Miscarriage and her newest book Normalize It: Upending the Silence, Stigma, and Shame That Shape Women's Lives.TW: Infant loss, pregnancy lossJessica shares the story behind her 16-week miscarriage, how it deepened her already decade-long clinical work in women's reproductive and maternal mental health, and why she felt compelled to bring that conversation into the public sphere. We also dig into why so many women default to self-blame after pregnancy loss, how our culture has taught us to minimize our grief, and why avoiding painful emotions can be far more destructive than actually feeling them. Plus these big questions: Can pleasure and grief coexist? What does it mean to truly honor a loss? And why do the people who most deserve joy so often feel the least entitled to it?Come for the conversation about pregnancy loss and women's shame. Stay for the moment Alex and Jessica unpack why grief avoidance, not grief itself, is what really derails us, and the simple, no-excuse practice Jessica recommends for anyone who doesn't have time to fall apart.Warm, honest, and full of gentle permission slips, this conversation is for anyone who has ever shrunk their pain to make others more comfortable — which is probably all of us.Purchase Jessica's Books:Normalize It!I had a MiscarriageFind Jessica!Dr. Jessica Zucker's website: drjessicazucker.comHer Instagram: @IHadAMiscarriageSupport the show:On PatreonBuy us a bookBuy cute merchSubscribe to the Babes in Bookland SubstackConnect with us and suggest a great memoir!Follow us on instagram! @babesinbooklandpod Thank you for listening!Xx, Alex Hosted on Acast. See acast.com/privacy for more information.

En El Ojo Crítico entrevistamos al escritor argentino Eduardo Sacheri por "Demasiado lejos", una novela histórica sobre la Guerra de las Malvinas de 1982 que aborda el conflicto entre Argentina y Reino Unido desde la mirada de quienes lo vivieron lejos del frente. Una historia coral que retrata la vida cotidiana durante uno de los episodios más significativos de la historia contemporánea argentina.Además, conversamos con Ramón Martín, codirector junto a Julio Martí de Noches del Botánico, el festival que celebra su 10º aniversario en la Universidad Complutense de Madrid y que se ha consolidado como una de las principales citas musicales del verano gracias a una programación ecléctica con artistas nacionales e internacionales.Nuestro colaborador Inko Martín nos adelanta el concierto de este viernes en el Teatro Monumental de Madrid con la Orquesta y Coro RTVE, que interpretará obras de Dvořák, Shostakóvich y Verdi. Y Pedro Torrijos nos descubre el origen real de la popular “Casita de Bad Bunny”, inspirado en los diseños residenciales creados por el arquitecto William Levitt en Levittown (Long Island) en 1947, uno de los desarrollos urbanísticos más influyentes del siglo XX.Escuchar audio

durée : 00:11:42 - par : Emilie Munera, Rodolphe Bruneau-Boulmier - Souvent cantonné à la période baroque et considéré disparu après la Révolution française, le clavecin refleurit dès le premier tiers du XXe siècle et inspire des compositions originales. Justin Taylor nous propose un panorama « personnel » du clavecin au XXe siècle. - réalisation : Pauline Boisaubert Vous aimez ce podcast ? Pour écouter tous les épisodes sans limite, rendez-vous sur Radio France

durée : 00:11:42 - par : Emilie Munera, Rodolphe Bruneau-Boulmier - Souvent cantonné à la période baroque et considéré disparu après la Révolution française, le clavecin refleurit dès le premier tiers du XXe siècle et inspire des compositions originales. Justin Taylor nous propose un panorama « personnel » du clavecin au XXe siècle. - réalisation : Pauline Boisaubert Vous aimez ce podcast ? Pour écouter tous les épisodes sans limite, rendez-vous sur Radio France

It's in the News! The top diabetes stories and headlines happening now. Top stories this week include: Afrezza inhaled Insulin is Approved for Kids, CGM + Ketone Monitor gets European approval, Food Coloring & Diabetes Study, Device Recalls include Omnipod and Dexcom, Beta Bionics shares more about their patch pump, ADA conference info and more! This podcast is not intended as medical advice. If you have those kinds of questions, please contact your health care provider. Announcing Community Commericals! Learn how to get your message on the show here. Learn more about studies and research at Thrivable here Please visit our Sponsors & Partners - they help make the show possible! Omnipod - Simplify Life All about Dexcom  All about VIVI Cap to protect your insulin from extreme temperatures The best way to keep up with Stacey and the show is by signing up for our weekly newsletter: Sign up for our newsletter here Here's where to find us: Facebook (Group) Facebook (Page) Instagram Check out Stacey's books! Learn more about everything at our home page www.diabetes-connections.com  Episode transcripts: Welcome! I'm your host Stacey Simms and this is an In The News episode.. where we bring you the top diabetes stories and headlines happening now. A reminder that you can find the sources and links and a transcript and more info for every story mentioned here in the show notes. ADA starts this week – safe travels to those of you heading to New Orleans. We'll be covering remotely so please follow on social – make sure to Like the FB page or join the group. We've got a wrap up episode planned for this podcast as well as some indepth interviews with the newsmakers from the conference. I will see some of you next week in Chicago. We have a couple of seats left for our Club 1921 dinner on June 10th in Northbrook – this is a FREE dinner for HCPs and patient leaders – all about screening for T1D. More info on the website under the events tab. Okay.. our top story this week: XX Afrezza inhaled insulin is now approved for kids and teens. The FDA okayed MannKind's afrezza for children 6 and older with type 1 and type 2 diabetes. MannKind says its proprietary Technosphere drug delivery platform enables the rapid absorption of insulin into systemic circulation. This follows FDA approval earlier this year for an update that revises recommendations for the starting mealtime dosage when patients switch from subcutaneous mealtime insulin regimens. MannKind also completed enrollment in February for a study evaluating the initiation of Afrezza therapy shortly after type 1 diabetes diagnosis in pediatric patients.   The company said it made Afrezza available for eligible patients for $35 or less per month. Desmond Schatz, professor of pediatrics at the University of Florida College of Medicine, said: "Mealtime insulin can be especially challenging for children because eating and snacking patterns, activity levels, and daily settings like school and sports often vary. With its rapid onset and dosing at the start of a meal, Afrezza may help clinicians better match insulin therapy to how children and families live day to day, while offering a needle-free mealtime option." Lots more to come on this – we're working on a bonus episode with one of the pediatric endos who worked on the clinical trials that led to this approval – hopefully have that out later this week. https://www.massdevice.com/mannkind-fda-approval-inhaled-insulin-children/ XX FDA has agreed to consider a new drug for the treatment of adults with type 1 and chronic kidney disease. Finerenone (fy-near-uh-known) is currently approved in the US for adults with CKD associated with type 2 diabetes and for adults with heart failure with left ventricular ejection fraction of 40% or greater. Chronic kidney disease (CKD) is present in over one-third of adults with diabetes, and because it's such a serious condition, interventions are needed to reduce its incidence and help people live a long and prosperous life. https://www.docwirenews.com/post/fda-grants-priority-review-to-finerenone-snda-for-type-1-diabetes-associated-ckd XX Abbot gets European approval for the world's first dual glucose‑ketone sensing technology for people with diabetes. They're calling this Libre Duo and Libre Duo 10 Day, and it's designed to continuously measure glucose and ketone levels every minute. Abbott plans to begin launching Libre Duo systems in select European countries later this year. Libre Duo delivers up to 15 days of wear and will be offered to adults ages 18 and older. Libre Duo 10 Day offers up to 10 days of wear and is intended for people ages 2 and older. Abbott is also working with leading pump companies to allow automated insulin delivery (AID) systems to connect with the sensors. https://abbott.mediaroom.com/2026-05-27-Abbott-secures-CE-Mark-for-worlds-first-dual-glucose-ketone-sensing-technology-for-people-with-diabetes   XX Huge recall for Omnipod. Insulin says a manufacturing issue through ongoing product monitoring that could result in insulin under-delivery  with specific lots of its Omnipod 5, Dash and Eros pods. Insulet said the scope of this action reaches approximately 7 million pods. This issue is separate from the March recall that affected certain Omnipod 5 lots. According to the Acton, Massachusetts-based company, some of its affected pods may have a small tear in the tubing (cannula) just above the skin. This tear lands between the pod and the point where the cannula enters the body. If this occurs, insulin may leak outside of the device instead of being fully delivered into the body as intended. This may lead to under-delivery of the therapeutic.   Individuals using an affected pod may notice wetness on the skin or pod adhesive or detect the smell of insulin. However, some cases may prove difficult to detect and go unnoticed. Of the approximately 7 million pods included in the action, approximately 60% have been consumed or are expired. The pods affected by the correction represent approximately 8.5% of the 2025 global Omnipod pod prodcution. Insulet says it has sufficient supply to replace affected pods. It expects no disruption to product availability. The company said it has notified the FDA and all other relevant regulatory authorities of its action.   The full list of affected pod lots can be found here. https://www.massdevice.com/insulet-another-omnipod-5-recall-dash-eros/ XX Dexcom is warning that certain scrapped glucose sensors have been stolen and resold. Dexcom said it has not received any reports of severe adverse events associated with the stolen product. One lot of scrapped devices carries a risk of infection for sensors that are not properly sterilized, and another lot had an elevated internal testing failure rate, meaning users would have an increased risk of having no sensor readings available. Dexcom said the affected sensors were stolen during the destruction process and then sold by third parties. The company routinely scraps sensors that do not meet its standards. The sensors are sent to a third-party vendor for destruction and recycling.   Dexcom said it traced sales of the stolen devices to Pharmsource, which is not an authorized Dexcom distributor but supplies some independent pharmacies and U.S. durable medical equipment distributors. Because of this, pharmacies that purchase products from Pharmsource should review their inventory, Dexcom said.   People with sensors from the affected lots should not use those sensors and can call customer support to request replacements. Dexcom has set up a website to help users check if their devices are affected. https://www.medtechdive.com/news/dexcom-warns-of-scrapped-glucose-sensors-being-resold/821139/ XX XX   Beta Bionics plans to debut its first insulin patch pump by the end of the second quarter of 2027, subject to Food and Drug Administration clearance. The device, called Mint, would be compatible with Beta Bionics' interoperable automated glycemic controller, a software that allows for the pump to automatically adjust insulin delivery based on readings from a glucose sensor. Beta Bionics first unveiled the prototype for Mint last year at the American Diabetes Association's Scientific Sessions. The device is expected to have a similar size and wear time, at three days, to Insulet's patch pumps on the market. It would have a 200-unit insulin reservoir.   Mint differs by containing a mix of reusable and disposable components. Beta Bionics plans to make the device exclusively available in the pharmacy channel, building on its existing agreements for its current iLet insulin pump. Beta Bionics is one of several diabetes tech companies developing patch pumps to compete with market leader Insulet. Tandem Diabetes Care and Medtronic spinoff MiniMed have also announced planned patch pumps. Tandem said it plans to file a 510(k) submission this quarter for a tubeless version of its small, durable pump, and Medtronic plans to submit its patch pump to the FDA this fall.   https://www.medtechdive.com/news/beta-bionics-to-launch-its-first-insulin-patch-pump-to-compete-with-insulet/821091/ XX CVS puts Zepbound back on it's coverage list – with it's Caremark PBM. They also added Foundayo, Lilly's obesity pill. CVS had dropped Lilly's Zepound last summer but kept competitor Wegovy. It'll be back at Caremark October first. All three of the nation's largest pharmacy ⁠benefit managers ​now cover Lilly's full obesity medicine portfolio. https://www.reuters.com/legal/litigation/cvs-brings-back-coverage-lillys-obesity-drug-zepbound-2026-05-28/   More to come, including a new benefit from metformin for women, something new from Tidepool, big news for T1D in Austalia and more.. XX A new study suggests that higher long-term exposure to food colouring additives — including both synthetic and natural colourings commonly found in processed foods and beverages — may be associated with an increased risk of developing type 2 diabetes. Researchers analyzed data from more than 108,000 adults in the French NutriNet-Santé cohort between 2009 and 2023, following participants for a median of just over eight years. During that time, 1,131 participants developed type 2 diabetes. The study found that people with the highest intake of total food colouring additives had a 38% higher risk of developing type 2 diabetes compared with non- or low-consumers.   Several specific additives were linked to increased risk, including caramel colouring additives such as total caramel (E150 family), plain caramel (E150a), sulphite ammonia caramel (E150d), and beta-carotene (E160a). Additional associations were observed for curcumin (E100), anthocyanins (E163), paprika extract (E160c), lutein (E161b), and cochineal-derived colourings (E120). "Our findings revealed positive associations between widely consumed food colouring additives and type 2 diabetes incidence," the authors wrote, adding that further research is needed to better understand the mechanisms behind the findings and whether food colouring regulations should be reevaluated. https://www.medscape.com/viewarticle/use-common-food-colours-tied-high-type-2-diabetes-risk-2026a1000hes XX Big news for Australia – their Therapeutic Goods Administration (TGA) approves Tzield. Tzield is now approved in Australia to delay the onset of stage 3 (or clinical) T1D in people aged eight years and older with stage 2 T1D – the early, pre-symptomatic stage of the condition, where changes in blood glucose levels have begun but insulin therapy is not yet required. Breakthrough T1D Australia Chief Executive Officer, Sydney Yovic, said the approval represented a transformational moment for Australians affected by T1D. https://newshub.medianet.com.au/2026/05/landmark-approval-of-tzield-in-australia-ushers-in-a-new-era-of-delay-for-type-1-diabetes/155036/ XX https://www.theatlantic.com/health/2026/05/diabetes-pregnancy/687324/ XX A common diabetes drug may hold great potential to help with aging, even if scientists aren't exactly sure why. According to a study, the drug metformin doesn't just help patients to effectively manage their type 2 diabetes. it may also give older women a better chance of living to 90. Scientists in the US and Germany used data from a long-term US study of postmenopausal women.   Records for a total of 438 people were selected – half of whom took metformin to treat diabetes, and half of whom took a different diabetes drug, sulfonylurea.   While there are some caveats and asterisks to the study, those in the metformin group were calculated to have a 30 percent lower risk of dying before the age of 90 than those in the sulfonylurea group. The study used age 90 as the marker for 'exceptional' longevity. However, scientists aren't yet sure that the drug extends lifespan, especially in humans – which is part of the reason for this study. RCTs could follow further down the line to dig deeper into these results, the researchers suggest. In the meantime, as the global population continues to skew older, studies continue to find ways to keep us healthier for longer and reduce damage to the body as we age. https://www.sciencealert.com/a-common-diabetes-drug-is-linked-with-exceptional-longevity-in-women XX The American Diabetes Association® (ADA) will host the 2026 Scientific Sessions from June 5-8 in New Orleans. The ADA's Scientific Sessions is the world's largest diabetes meeting, convening an expected audience of over 12,000 leading physicians, scientists, researchers, and healthcare professionals from around the globe. The premier diabetes meeting, which is also offered virtually, will feature the latest scientific findings in diabetes and obesity, where leading experts and peers will share findings in research for prevention, care, and cures at the Ernest N. Morial Convention Center. Key themes will include: Advancing obesity and metabolic health: Prevention, early detection, and disease modification: Improving cardiometabolic outcomes: Transforming care through innovation and access: New research will highlight how technology, artificial intelligence, and implementation strategies are reshaping diabetes care—reducing treatment burden, expanding access, and enabling more person-centered care. Advancing beta cell replacement and cure strategies: Fostering innovation: On Saturday, June 6, from 4:30-6:00 p.m., the Innovation Challenge, which debuted in 2023, invites emerging companies to pitch novel ideas to improve the lives of people living with diabetes. A panel of judges, with input from a live audience, determines which contestants will earn a private audience with potential funders. XX Tidepool, the nonprofit leader advancing innovation in diabetes technology, announced that Tidepool+ Direct Connect is now available through the Epic Showroom. Built on SMART on FHIR, Direct Connect brings interactive diabetes device data directly into Epic workflows, helping clinicians use patient data during routine care. "Tidepool has always focused on making diabetes data more accessible and actionable," said Brandon Arbiter, CEO. "We're excited to empower clinicians using Epic with insightful, intuitive patient data that fits directly into their encounter workflow so they can use it to improve care in the moment it matters."   Tidepool+ Direct Connect supports scalable deployment across Epic-enabled health systems. This architecture enables faster, more intuitive rollouts, enhancing Tidepool's existing EHR integration capabilities.   Direct Connect is part of Tidepool's ongoing work to improve how clinicians can use timely and relevant diabetes device data during patient visits to help drive better health outcomes.   The feature is now available in the Connection Hub of the Epic Showroom.   https://www.businesswire.com/news/home/20260527780274/en/Tidepool-Launches-in-Epic-Showroom-to-Bring-Diabetes-Device-Data-into-the-Point-of-Care XX

If you have emotions, this episode is for you...

Will and Jeremiah discuss the upcoming Primavera Sound Porto music festival.

En El ojo crítico conversamos con Juan Mayorga sobre El jardín quemado, la obra que dirige y firma como dramaturgo en el Teatro de La Abadía hasta el 12 de julio. Una propuesta que reflexiona sobre la memoria histórica, la dificultad de juzgar el pasado y el papel de la imaginación y el teatro como refugio frente a la dureza de la guerra y sus consecuencias. Además, Xaviera Torres nos explica cómo los rayos X aplicados al arte permiten descubrir dibujos ocultos, correcciones y secretos bajo la superficie de los cuadros.También hablamos con Enrique Mejías sobre Pilar Lorengar, una de las grandes sopranos europeas del siglo XX, figura clave de la lírica española. Y repasamos los momentos más destacados de los Premios Max de las Artes Escénicas, una de las principales citas del teatro y la creación escénica en España.Escuchar audio

This week, Brett is back for a bonus episode and we are getting into "Famesick", Lena Dunham's new memoir about illness, ambition, Hollywood, and whether any of it was worth it. Brett isn't just any reader here — she worked closely with Lena during the Girls era at United Talent Agency, which means this conversation comes with some serious behind-the-scenes context that you are not going to get anywhere else.We break down what the book actually delivers versus what it promises: the health journey, the name dropping, the rise-to-fame years, the rehab chapters, the wildest dedication page we have ever read, and the moment with Barbara Walters that has been making the rounds for good reason. We also get into what the memoir conspicuously leaves out — her friendship with Taylor Swift, the wording of the November 17th statement — and why those omissions say just as much as what she chose to include.The big question we keep coming back to: is "Famesick" a genuine reckoning, or is it a very calculated, self-serving reframe of a career and a public image? If you loved Girls, hated Girls, or have ever had a complicated relationship with a woman who is clearly brilliant... but also kind of a lot — this episode is for you.Thank you so much for supporting the show!Xx, Alex Hosted on Acast. See acast.com/privacy for more information.

„Bez opowieści człowiek ledwo by wiedział, na jakim świecie się znajduje” – to zdanie z powieści bohatera dzisiejszego odcinka Woldemar miał siedem lat, kiedy wypowiedział życzenie, że nigdy nie chce dorosnąć, że na zawsze chce pozostać w niewinnym świecie dzieciństwa.Tylko że dzieciństwo Woldemara nie było ani beztroskie, ani naiwne. Rozmyślanie o przemijaniu stopniowo stawało się jego obsesją i kiedy zorientował się, czym jest sekunda, mimowolnie ją stracił.Woldemar to tytułowy bohater powieści mołdawskiego pisarza Olega Serebriana. To historia chłopca, który dorastał w latach 70. XX wieku w Północnej Bukowinie, w mieście Czerniowce, które na przełomie XIX i XX stulecia przechodziło z rąk Cesarstwa Austriackiego w posiadanie Rumunii, by latem 1940 roku zostać przyłączonym do Związku Radzieckiego.„Woldemar” to opowieść o dzieciństwie, które jest naznaczone ciężarem historii i polityki, ale także ogromnym niepokojem egzystencjalnym. Bo przecież jako dzieci po raz pierwszy uświadamiamy sobie rzecz być może najważniejszą – śmiertelność.„Powiedz, Woldemarze, dlaczego tak bardzo różnisz się od nas wszystkich? Co sprawiło, że taki jesteś?” – pytali go wszyscy.Z okazji Dnia Dziecka w „Raporcie o książkach” zapraszam na opowieść o 7-letnim chłopcu, który żył na styku światów i czasów. Wychowywany przez trzy kobiety, z których każda reprezentowała inny kawałek popękanego krajobrazu Europy Środkowej przełomu XIX i XX wieku.Prowadzenie: Agata KasprolewiczGość: Oleg SerebrianKsiążka: „Woldemar” / Oleg Serebrian / przekład: Radosława Janowska-Lascar / Wydawnictwo Amaltea---------------------------------------------Raport o stanie świata to audycja, która istnieje dzięki naszym Patronom, dołącz się do zbiórki ➡️ ⁠https://patronite.pl/DariuszRosiak⁠Subskrybuj newsletter Raportu o stanie świata ⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠⁠➡️ ⁠https://dariuszrosiak.substack.com⁠Koszulki i kubki Raportu ➡️ ⁠https://patronite-sklep.pl/kolekcja/raport-o-stanie-swiata/⁠ [Autopromocja]

Tres figuras de primer nivel internacional visitaron la Argentina: Lady Di, Albert Einstein y (el más grande) don Ramón Valdez. Este Sábado te contamos los pormenores de sus visitas en el siglo XX. Encontra este y mucho más contenido todos los sábados a las 13hs por www.fm913.com.ar o en Spotify

Après neuf ans d'hésitation, les députés et sénateurs français ont fini, le 7 mai 2026, par voter à l'unanimité une loi-cadre sur la restitution des objets pillés pendant la colonisation. C'est une victoire pour les anciennes colonies françaises comme le Bénin. Mais le retour en Afrique de ces biens culturels ne risque-t-il pas d'être stoppé l'année prochaine s'il y a un changement de majorité en France ? C'est l'une des questions que Christophe Boisbouvier a posées à l'historienne franco-béninoise Marie-Cécile Zinsou, qui préside la Fondation Zinsou à Ouidah, près de Cotonou. RFI : Avec tous ces lieux de mémoire, avec tous ces musées, quelles sont les ambitions du Bénin de Romuald Wadagni ? Marie-Cécile Zinsou : Alors écoutez, le Bénin s'est engagé depuis quelques années dans une préservation de son patrimoine et dans une nouvelle politique qui est totalement axée à la fois sur son histoire et sur l'avenir. Donc, je crois que le nouveau président va s'inscrire dans le chemin qu'il avait tracé aux côtés de Patrice Talon, notre précédent président, ces dernières années. On est un peu dans la continuité, c'est ça ? Il me semble qu'on est effectivement dans la continuité je pense, le président Wadagni était notre ministre des Finances pendant les dix dernières années. Donc, il me semble qu'on est dans quelque chose qui ressemble à une continuité. À l'origine de ce renouveau mémoriel, il y a l'action initiale du ministre Aurélien Agbenonci, il y a dix ans déjà, quand il a réclamé à la France de François Hollande la restitution des trésors royaux du Bénin. Est-ce que la loi-cadre votée par la France répond à vos attentes ? Ce qui est absolument fondamental, c'est ce que le courrier du Bénin a déclenché en France. Et la réponse qu'Emmanuel Macron y a apportée à Ouagadougou en 2017. Puisqu'on peut vraiment dater le début du processus de restitution à partir de ce discours. Donc aujourd'hui, la loi-cadre, après neuf ans d'attente, arrive pour donner une forme à ces restitutions et pour éviter les lois d'exception qui avaient eu lieu jusqu'alors pour à la fois le Bénin, le Sénégal et la Côte d'Ivoire. Oui, c'est-à-dire que jusqu'à présent il fallait une loi par restitution ? Oui, il fallait des lois spécifiques. Et la loi-cadre, elle va être efficace très rapidement puisque le gouvernement français est motivé et l'a fait savoir. Les parlementaires ont voté à l'unanimité cette loi, ce qui montre bien que la représentation nationale française est tout aussi concernée par les questions de restitution. Et il y a de nombreuses demandes en cours du côté du continent. Donc, c'est une loi qui, avec un gouvernement et une représentation nationale française motivés, peut être extrêmement efficace. Est-ce qu'il y a des failles malgré tout dans cette loi ? Alors cette loi, elle propose qu'il y ait des commissions bi-nationales, donc entre l'État demandeur et l'État français pour examiner le cas des biens qui ont été pillés illicitement. Evidemment, si la France est motivée, elle met en place une commission qui regarde avec intérêt, objectivité et bienveillance les demandes de restitutions. Si le prochain gouvernement était moins motivé par les restitutions, ces commissions pourraient être nettement plus dures et pourraient bloquer certains processus. Je pense que c'est pour ça que c'est très important qu'il y ait des premières restitutions assez rapidement, parce que, après, ce sera un phénomène inarrêtable. Et le président français l'a dit à Nairobi, a priori, c'est une loi irréversible. On ne reviendra pas sur les restitutions. Il faut créer une dynamique ? Il faut créer une dynamique et il faut créer des précédents pendant cette année où toutes les étoiles semblent alignées. Quels sont les pays développés les plus réticents à restituer les œuvres qui ont été pillées au 19ᵉ et au XXᵉ siècle ? Alors chaque pays est complexe. En Europe, le plus réticent est peut-être celui qui a le plus restitué, et les gens ne s'en rendent pas forcément compte. Mais aujourd'hui, on peut dire que le Royaume-Uni est celui qui se protège le plus. Notamment parce que les Britanniques sont face à des demandes de restitutions européennes, et notamment des Grecs qui réclament les frises du Parthénon, qui sont au British Museum. Et donc, à la fois, le Royaume-Uni refuse de légiférer et bloque une partie des demandes. Et en même temps, c'est le Royaume-Uni qui a restitué plus de 70 biens déjà à travers ses musées non nationaux, donc à travers ses musées universitaires et ses musées régionaux. Donc, le Royaume-Uni est peut-être celui qui, dans la loi, est le moins ambitieux, mais qui restitue le plus. Alors qu'il y a des pays comme la Belgique qui ont restitué officiellement, massivement, puisqu'il y a plus de 1 200 objets qui sont déjà concernés par la loi belge, et en même temps, il y a un seul masque qui a fait le voyage de retour au Congo. Est-ce que les différentes institutions béninoises qui ont été créées ces derniers mois, notamment le Comité scientifique national auquel vous appartenez… Est-ce que tout cela peut encourager les pays qui détiennent des biens culturels et qui ne veulent pas s'en séparer ? Est-ce que cela peut les encourager à les restituer à des pays demandeurs comme le vôtre ? Il me semble que l'exemple du Bénin a été important, notamment avec la première restitution, avec la première loi d'exception de la France, quand les 26 objets du Trésor royal d'Abomey sont revenus, le Bénin a fait une démonstration assez éclatante de ce que pouvait être le retour du patrimoine et le partage à tous de ces collections. Donc, je pense que ça a aussi permis de faire sauter des verrous qui étaient bien souvent des préjugés sur une base raciste, ou sur juste une base de se dire que l'Afrique n'était pas intéressée à son patrimoine, que les Africains n'allaient pas au musée. Ça, c'était des propos qu'on entendait dans la société française. Tout à coup, à partir du moment où on a fait la démonstration que les gens se sont passionnés pour cette exposition, qu'elle a été ouverte à tous en grand, je crois que les gens ont réalisé ce que c'était, en fait, que d'être privé de son patrimoine depuis des dizaines ou des centaines d'années, et d'y avoir enfin un accès. Donc, je crois qu'évidemment certains pays, qui prenaient la question très à la légère, ont vu la preuve devant leurs yeux que c'étaient des questions fondamentales et qu'on ne pouvait pas rester sur des préjugés souvent racistes qui étaient prédominants.

Robert Bremner: Duke of Perth's Reel, Capt. Ross's Reel, Short Apron, Hoptoun House, Lady Hariot Hope's Reel, The Highlandman Kiss'd His Mother, Miss Murray's Reel, Drummore's Rant, He Hirpl'd till her, Had the Lass till I winn at her, Cadgers of the Cannongate, Jeremy Kingsbury Sets: Colonel MacBain's Fancy, Brenda Stubbert's Reel, The Gravel Walk, Highlandman Kissed His Mother & Jenny Sutton From Bannocks of Barley Meal. Paddy Cary, Jigg Poltage, Ryan's Rant from Pay the Pipemaker. Dark-eyed one of the Night, Lady Seaforth, Dark Girl of the White Feet, Lady Mary Mackay, Mary Gray, Sweet Molly From Rowly Powly. J. Johnson: The Lads of Boot, William Ross: Miss Victoria Ross Iain MacInnes: Miss Victoria Ross, Lady Doll Sinclair, A'Chubhag (The Cuckoo), McFarlane's from Album Tryst Anselm Lingnau: (Traditional Tune Archive) Lady Susan Stewart's Reel John Walsh: Susan Stewart's Reel, Big thank you to Iain MacInnes for his blessing to include his track from Tryst. Tryst was published by Greentrax Label: https://greentrax.com/product/iain-macinnes-tryst-cd/ But is available on most streaming platforms as well. +X+ Cover Art is a Receipt from Robert Bremner's Shop in London courtesy of the British Museum: https://www.britishmuseum.org/collection/image/1544287001 +X+X+ I played tracks from Pay the Pipemaker: https://jeremykingsbury.bandcamp.com/album/pay-the-pipemaker Bannocks of Barley Meal: https://jeremykingsbury.bandcamp.com/album/bannocks-of-barley-meal and Rowly Powly: https://jeremykingsbury.bandcamp.com/album/rowly-powly +X+X+ Nearly all of the tunes this week come from Robert Bremner's 1757(ish) publication: A Collection of Scots Reels or Country Dances https://digital.nls.uk/special-collections-of-printed-music/archive/105002262 +X+X+ 1750: Lads of Boot From J Johnson's Country Dances (Via Traditional Tune Archive) https://tunearch.org/wiki/Lads_of_Boot +X+X+ 1869: Miss Victoria Ross: from William Ross's Collection of Pipe Music: https://ceolsean.net/content/WRoss/WRoss_TOC.html +X+X+ Susan Stewart's Reel From Traditional Tune Archive: https://tunearch.org/wiki/Lady_Susan_Stewart%27s_Reel +X+X+ 1758 (I've also seen 1760): Lady Susan Stewart's Reel from John Walsh's Caledonian Country Dances Vol 2 part IV https://archive.org/details/walsh4caledonian/ +X+X+ For may Hihland Man Kissed His Mother Episode Listen here: https://www.wetootwaag.com/s5e28 +X+X+ FIN Here are some ways you can support the show: You can support the Podcast by joining the Patreon page at https://www.patreon.com/wetootwaag You can also take a minute to leave a review of the podcast if you listen on Itunes! Tell your piping and history friends about the podcast! Checkout my Merch Store on Bagpipeswag: https://www.bagpipeswag.com/wetootwaag You can also support me by Buying my Albums on Bandcamp: https://jeremykingsbury.bandcamp.com/ You can now buy physical CDs of my albums using this Kunaki link: https://kunaki.com/msales.asp?PublisherId=166528&pp=1 You can just send me an email at wetootwaag@gmail.com letting me know what you thought of the episode! Listener mail keeps me going! Finally I have some other support options here: https://www.wetootwaag.com/support Thanks! Listen on Itunes/Apple Podcasts: https://podcasts.apple.com/us/podcast/wetootwaags-bagpipe-and-history-podcast/id129776677 Listen on Spotify: https://open.spotify.com/show/5QxzqrSm0pu6v8y8pLsv5j?si=QLiG0L1pT1eu7B5_FDmgGA

Are the hot colors predictions for 2026 really a blaze? Today we take a look at what paint brands & industry experts declared for 2026 to see if any of those colors have made it onto your walls and into your homes. Check out this funny SNL skit on Farrow and Ball paints HERE.Create a home you will LOVE along with Kelly on her app - Home Design Coach. Check out HERE !Need help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HERELooking for amazing decor on Amazon? Anita has updated her favorite picks HERE.Are you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 6:22See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Style - Do you have it? Want to know how to get it? Today we are sharing the secrets to developing a signature style.Our DTT living room challenge is step FIVE. Create a list of what you'll keep, what you'll give away and what you are going to sell.Kelly's style icon to know is India Hicks. See India's vibe, homes and style in these places:Her site HEREHer IG HEREHer books HERE, HERE, and HEREAnita thinks Paula Sutton has a very unique and joyful personal styleHer book HEREHer IG HEREHer youtube channel HEREDTT defines pearlcore. Listen & learn!We participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.Anita's crush is Paula Deen's beautiful living room on YouTube HEREKelly's crush is The Great Grow Along a 10 day virtual garden event March 11-20. Register for FREE for the live events HERE or purchase a ticket to enjoy for six months.Stephanie Rose of Garden Therapy will be one of the 40 speakers at the event.Listen to Kelly's interview with Stephanie in this episode of DTT HERENeed help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HERELooking for amazing decor on Amazon? Anita has updated her favorite picks HERE.Are you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 9:43 / 22:24See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.