Podcasts about XX

We've got so many super simple ways to de-clutter your home, and we're going rapid fire today, so get out your pencil!DTT DEFINES: Chiavari chairWe participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.CRUSHES:Kelly's crush is the wonderful YouTube yoga instructor Charlie Follows. Check out Charlie's channel HERE.Anita's crush is this charming pink toaster HERE.Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.Studio.com is having a summer sale - get Kelly's app at 20% off until July 3rd !! Go to studio.com/kelly or simply click HERE.Need help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyBirthday music courtesy https://www.chosic.com/free-musicDI - 9:51 /20:43See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

On this week's show special guest co-host Rob Joyce joins Patrick Gray and James Wilson to discuss the week's cybersecurity news. Rob served as an advisor to Donald Trump during his first term as president and also served at NSA for 34 years. While at the agency, Joyce led Tailored Access Operations (TAO), and later became NSA's Director of Cybersecurity. They cover: The surprisingly well done Fortibleed campaign Stolen Klue OAuth tokens lead to Salesforce data theft OpenAI wants to patch the planet runZero gets acquired by Accenture, congrats HD Moore! Much, much more! This episode is also available on YouTube. Show notes FortiBleed campaign used custom FortiGate sniffer to steal credentials | BleepingComputer FortiBleed: Fortinet device credential compromise expands into broader credential-attack guidance | unit42.paloaltonetworks.com Cybercriminals allegedly hacked tens of thousands of Fortinet firewalls used by major companies all over the world | TechCrunch Security Klue OAuth breach linked to 'Icarus' Salesforce data theft attacks | BleepingComputer Polymarket (@Polymarket) on X | X (formerly Twitter) The Korean telecom giant at the center of Anthropic's Mythos controversy | wrd.cm Beyond Fable: Can a Local LLM Replace Cloud AI for Security Code Reviews - SRLabs Research | SRLabs OpenAI Launches Full-Scale Effort to Patch Open-Source Bugs as It Takes on Anthropic's Mythos | wired.com Sponsored: Trail of Bits and OpenAI patch the planet | Risky Bulletin Intel agencies: Frontier AI models will reshape cybersecurity faster than expected | cyberscoop.com Embedding Forbidden Text in Spyware to Discourage AI Analysis | Schneier on Security A new unpatchable flaw in Apple chips opens the door to an iPhone jailbreak | TechCrunch Security USB worm spreads crypto-stealing malware via Windows shortcut files | BleepingComputer Android verification is coming: Google confirms timeline and supported app stores | Ars Technica California water utility probes breach claim by Iran-linked actor | Cybersecurity Dive Suspected cyberattack triggers false emergency alerts across parts of Brazil | The Record Tesco moving 40,000 server workloads off VMware amid Broadcom's "abusive conduct" | Ars Technica Trump directs federal agencies to protect US data from quantum threats | therecord.media Accenture shells out $4.18B on three companies in big industrial cybersecurity push | cyberscoop.com

Ya está aquí el primer baño fluvial modernista en el río Segura a su paso por Cieza. Las asociaciones participantes buscan recrear la estética y las costumbres de principios del siglo XX, combinando el rigor histórico con actividades lúdicas como un picnic, música de época y juegos tradicionales. Los invitados destacan que la iniciativa pretende recuperar la memoria histórica regional y fomentar la participación ciudadana, enfatizando que cualquier persona puede asistir utilizando vestimentas sencillas que respeten el pudor y el estilo de la época. El propósito es promocionar este encuentro de convivencia que celebra la identidad local y transforma un entorno natural en un escenario de recreación histórica abierto al público.

How can you reclaim your truth and your power?We're diving into Horse Barbie, Geena Rocero's radiant and illuminating memoir that shows her journey from a one-room home in Manila, Philippians, to trans pageants, from the perfume counter at Macy's to New York fashion sets, and from private, suffocating, fear to a TED Talk that reframed transness as power. Along the way, we discuss her father's complicated love, her mother's unwavering belief and reflect on what can spark when the people who matter most tell you there's nothing wrong with you. Geena's story and memoir widens from personal to political: pre-colonial history without gendered pronouns, the costs of documentation that doesn't match your face, and the power of her viral TED Talk to turn shame into strength. We sit with the big questions: Why is femininity seen as a threat? How do entertainment and policy diverge? What changes when a community moves from visibility to rights? By the end, Horse Barbie reads like a manual for courage. It shows how story becomes strategy and how one woman's voice can help many step out of the shadows.If you care about trans rights, immigration, pageant culture, modeling, or the way love can change a life, this conversation brings nuance, warmth, and a clear takeaway: policy matters, family matters, and stories move hearts faster than arguments ever will. If the conversation resonates, subscribe, leave a review, and share this episode with someone who needs a hopeful, human lens on trans life and advocacy.For the extended discussion of this episode (an additional 24 min), subscribe on Apple Podcasts or Patreon.Support the show:Buy us a bookBuy cute merchBuy Horse BarbieOther Links:Evan Hurst substack This episode is produced, recorded, and its content edited by me.Special thanks to my dear friend, Jaime! Xx, AlexConnect with us and suggest a great memoir!Follow us on instagram! @babesinbooklandpod Hosted on Acast. See acast.com/privacy for more information.

Año XX. Núm. 60. La respuesta dirigida a Mons. Heiner Wilmer, presidente de la Conferencia Episcopal Alemana, por parte del Dicasterio para el Culto Divino y la Disciplina de los Sacramentos.

Durante los primeros cincuenta años del siglo XX, el mundo cambió más rápido que en siglos anteriores. Imperios desaparecieron, nuevas potencias surgieron, dos guerras mundiales transformaron el mapa político, la tecnología revolucionó la vida cotidiana y millones de personas fueron testigos de acontecimientos que marcarían para siempre la historia de la humanidad. En este especial de Interesante Historia, recorreremos década por década los eventos, personajes y cambios que definieron el periodo entre 1900 y 1950. Desde los últimos días de los grandes imperios hasta el inicio de la Guerra Fría, descubriremos cómo se construyó el mundo moderno que conocemos hoy. Si no has escuchado los episodios de cada década del Siglo XX cuando se publicaron, prepárate para un viaje por una época de avances extraordinarios, conflictos devastadores y transformaciones que aún influyen en nuestras vidas, en este especial de más de 2 horas de duración. Porque para entender el presente, primero debemos comprender el siglo que lo cambió todo.

Jak wyglądało życie na wsi, gdy o przyszłości często decydowały miejsce urodzenia, płeć i status społeczny? W tym odcinku wideokastu „Polityka o historii” naszą gościnią jest Antonina Tosiek, autorka książki „Przepraszam za brzydkie pismo. Pamiętniki wiejskich kobiet”, opartej na autentycznych wspomnieniach kobiet z polskiej wsi, spisywanych od lat 30. aż po koniec XX w. To nie jest historia jednej bohaterki ani jedna polska wieś lata 50 czy polska wieś lata 70. To wielogłosowa opowieść o kobietach, które próbowały opowiedzieć o swoim życiu własnymi słowami. W ich pamiętnikach wracają marzenia o edukacji, pragnienie samodzielności, ciężka praca i wykluczenie społeczne, ale także poczucie dumy, sprawczości i walka o własne miejsce w świecie. Rozmawiamy o tym, czym było wiejskie życie widziane z perspektywy kobiet. Dlaczego tak wiele z nich musiało rezygnować ze szkoły? Jak wyglądała codzienność w gospodarstwie, relacje rodzinne, małżeństwa i macierzyństwo? Co oznaczał awans społeczny dla dziewczyny ze wsi i dlaczego dla wielu kobiet edukacja pozostawała niespełnionym marzeniem? Jednym z najważniejszych tematów rozmowy jest wstyd: związany z pochodzeniem, biedą, brakiem wykształcenia czy niespełnionymi aspiracjami. Antonina Tosiek pokazuje jednak, że pamiętniki nie są wyłącznie opowieścią o cierpieniu. To także świadectwo kobiecej podmiotowości i prób wyrwania się z narzuconych ról. Rozmawiamy również o tym, jak współcześnie opowiadamy historię polskiej wsi i dlaczego uproszczone wyobrażenia o „chłopkach” często nie mają wiele wspólnego z rzeczywistością. Dzięki tym niezwykłym świadectwom życie na wsi w Polsce przestaje być anonimową historią społeczną, a staje się opowieścią o konkretnych kobietach, ich marzeniach, rozczarowaniach i codziennej walce o godność. 3 rzeczy, których dowiesz się z odcinka: 1. Dlaczego edukacja była dla wielu wiejskich kobiet największym niespełnionym marzeniem? 2. Jak wyglądało życie na polskiej wsi z perspektywy kobiet, a nie podręczników historii? 3. Dlaczego wstyd był jednym z najważniejszych doświadczeń kolejnych pokoleń kobiet ze wsi? Oś czasu: 00:00 - Najciekawsze fragmenty 03:05 - Wstyd kobiet wiejskich: obecny mimo pokoleń 05:15 - Dlaczego wiejskie kobiety nie bały się pisać intymnych pamiętników? 07:20 - Wstyd przed biedą i marzenie o edukacji kobiet 15:50 - Lata 30. XX w. są niereprezentatywne dla całego pokolenia kobiet 23:42 - Zinternalizowana przemoc kobiet w społecznościach wiejskich 29:20 - Wstyd przed medycyną i kaprys rodzenia w szpitalach 37:05 - Jaką rolę w kulturze wstydu odgrywa Kościół? 43:55 - Wstyd zakochania się w innej kobiecie.

La dimensión del fracaso toma forma con las decisiones obligadasQuien no haya vivido el siglo XX posiblemente no sepa muy bien que significaba Cuba fuera de Cuba. Más aún luego de la caída del Muro de Berlín y la disolución de la Unión Soviética. Cuba era la heredera de un sueño. Por supuesto también la portadora de una pesadilla descomunal para muchos. Cuba era el sueño de que sí se podía plantar orgullo frente al poder imperialista. Cuba simbolizaba otro modo de hacer política, de encarnar la valentía, otra forma de hacer cultura, una esperanza de no caer inevitablemente en los indicativos del sistema capitalista voraz e insensible. Un modo de escapar de la fuerza gravitacional del sistema impuesto desde Hollywood, el New York Times, Fox, CNN o las casas de moda europea, de saltar el cerco informativo, de hallar valor más allá del dinero, de no pecar de abundancias superfluas. Cuba era la posibilidad de ser distintoECDQEMSD podcast episodio 6331 Cuba y el Final de un SueñoConducen: El Pirata y El Sr. Lagartija https://canaltrans.comNoticias del Mundo: Bolivia declara estado de excepción - Trump habla de cobrar peajes en Ormúz - La defensa de Sánchez a Zapatero - Banderazo para CFK - La estatua de Rocky y la suerte argentina - Y si el Tri sale campeón? - Ronaldinho regresa.Historias Desintegradas: El nombre de mi hija - Dónde está su esposa? - Cosas de Chihuahua - Sonara y sus cócteles - Niños envueltos - La Traviata de Verdi - Vicuñas limpias - Triste acontecimiento - La muerte de una Cebra - Juzgado laboral - El Volkswagen Escarabajo - Día del Futbolista Argentino - El gol de Maradona a los ingleses, en México 86 - Aros de cebolla y del lemoncelloEn Caso De Que El Mundo Se Desintegre - Podcast no tiene publicidad, sponsors ni organizaciones que aporten para mantenerlo al aire. Solo el sistema cooperativo de los que aportan a través de las suscripciones hacen posible que todo esto siga siendo una realidad. Gracias Dragones Dorados!!NO AI: ECDQEMSD Podcast no utiliza ninguna inteligencia artificial de manera directa para su realización. Diseño, guionado, música, edición y voces son de  nuestra completa intervención humana.

Rodolfo Montero del Palacio, cineasta y director del documental "Sangre, Sudor y alegría" te cuenta el emotivo viaje de varios miembros de la asociación malagueña de hemofilia junto a sus familias haciendo el Camino de Santiago. Sus emocionantes historias humanas, sus dolencias y fuerza de voluntad. Cómo el deporte y los tratamientos actuales han mejorado su calidad de vida. Una evolución alucinante si recordamos que a principios del siglo XX a penas tenían una esperanza de vida de diez años. El documental lo puedes ver gratis en Agroculturatelvision

Lords: Kate Andrew Topics: Movies that are supposed to be good and kind of annoyingly are actually good Making friends when you're old Paris's pneumatic mail system Forgiven, by A. A. Milne https://www.poeticous.com/a-a-milne/forgiven-i-found-a-little-beetle-so-that-beetle-was-his-name Microtopics: The skill to make the noises that are in your head. Writing quests for Hello Kitty Island Adventure. Sanrio's NDA-enforcing snipers. A crocodile that was invented in 1978. Writing quests that force the artists to figure out how a crocodile would wear sunglasses. Writing the dialog tree as the player is clicking on options, like Gromit placing tracks right as the train is about to roll over them. Spending five years shipping the first part of a live service game and then shipping additional parts every few weeks. Trying to rebuild a house while someone is living in it. Lingo 2. Dungeon Gals. The kind of game you can't draw a map of. Teetering on the razor's edge of "I'm a genius" and "I don't understand anything." What does the developer tooling look like for games that have non-euclidean spaces? Duplicated spaces with secret warp volumes. The kind of movie that an Infinite Jest reader will recommend. Terry Cavanagh's game about making tea. Egg Game? Astonishing movie running times. Five minutes of two men intensely looking at each other. An adventure movie about two best friends who hate each other. James Joyce: maybe he's good? A guy who lives in the middle of nowhere who thinks about politics a lot and never talks to anyone. Gerry, starring Ben Affleck and Matt Damon. The art is coming from inside the head. Pro Shots vs. bootlegs. Jukebox musicals. Shawshank Redimension. Shawshank Redemption 2: Shawshank Herdemption. Communicating between cell blocks by flushing the toilet. Loove a.k.a. Flushed a.k.a. Lavatory Lovastory. Mixing your DNA with someone you've never seen. Florida Writing. Gravity Slingshotting around your hobbies to reach friendship. Going to GDC and making a bunch of game dev friends. Plateauing at two digits. Getting hobbies that put you in a room with people of your desired gender. Your co-worker at the call center who's married to the CEO of Twinings. Playing puzzle games on the Internet in front of a chatful of puzzle experts. The protagonist getting stuck on a puzzle and the narrator turning to the audience and saying "chat, help us out with this one." Making friends vs. keeping friends. The friendships that you both care enough about to have maintained. I Love a Thoont. Building a pneumo to relieve congestion on the telegraph system. When the telegraph was invented vs. when pneumatics were invented. Why banks had pneumatic drive thrus rather than the teller just handing you the stuff through the window like a fast food drive thrus. Whimsical coffee preparation. The cost of building a giant tube between the coasts of North America. The Alameda-Weehawken Burrito Tunnel. Inventing a frictionless tube. Preparing burritos to be magnetically fired. Rifled sewers. Plunging into the Lithosphere. A diagram that shows how the burrito gets heated. The Taste of Breaking the Sound Barrier. Getting your poem voice on. Whether A. A. Milne knew about hash tags. Non-Fungible Beetles. Oh great, the same beetle came back! The kind of look that means "it's me, the same beetle!" The XIX and XX centuries. Writing Very Blackly. Why Does Pooh Own a Shotgun? All the talking Winnie the Pooh animals turning out to be aliens, like Starfox. A pneumatic tube, except instead of burritos you're firing cork. Writing your thesis on pop guns and continuing to do post doc research on pop guns. Spud Guns vs. Potato Cannons. A Normal Spud Gun for Normal Children. Seeing that Wikipedia considers Spud Guns low importance and thinking of ways to make Spud Guns more important. Too Many Posts.

Surprise release! Clive Staples Lewis was a Christian apologist, Oxford don and spanking enthusiast. The Screwtape Letters is one of his many books, an epistolary novel published serially in the (now-defunct) Anglican weekly newspaper The Guardian during the Second World War. The letters are written by Uncle Screwtape, a demon administrator in the bureaucracy of Hell, and addressed to his young nephew Wormwood, an inexperienced tempter. Screwtape offers Wormwood advice on how to lead his 'patient' away from God, so that when he dies he can serve as food for demons. Hell as a gigantic, noisy bureaucracy in which subordinates are eaten by their superiors? Lovely stuff.Joining us for this episode are friends of the show Kevin and Matt from the wonderful Art of Darkness podcast (and There Will Be Books, Matt's other podcast, also wonderful!) For more info on Lewis' spanking enthusiasms, listen to the Art of Darkness episode on C.S. Lewis (https://artofdarkpod.com/c-s-lewis-a-jack-in-the-wardrobe)!Art of Darkness: https://artofdarkpod.comArt of Darkness on X: X.com/artofdarkpodThere Will Be Books: X.com/TherewillbbooksKevin on X: X.com/kautzmaniaMatt's new book: https://shop.ingramspark.com/b/084?params=az0Zsh8Ol7BjqsK8S6M4XHoJlnrHvYZSQMjh4VUlBZL VERY IMPORTANT INFORMATIONContact: jack.bcfh@gmail.comJack has an upcoming novel called 'Audience Capture', out October 2026 through Bonfire Books!Ko-fi: https://ko-fi.com/bookclubfromhellOur Patreon: www.patreon.com/TheBookClubfromHellJack's Substack: jackbc.substack.comJoin our Discord (the best place to interact with us): discord.gg/ZMtDJ9HscrWatch us on YouTube: https://www.youtube.com/channel/UC0n7r1ZTpsUw5exoYxb4aKA/featuredX: @bookclubhell666Jack on X: @supersquat1Capitalisimo on X: @thecapitalisimoArt by moog

Natalia Ginzburg (Palermo, 1916-Roma, 1991) es una de las voces más singulares de la literatura italiana del siglo XX. Comprometida, combativa, inteligente y brillante. Ha publicado novelas, como 'Léxico familiar', 'Todos nuestros ayeres' o 'Sagitario', teatro, como 'Me casé por alegría' y ensayos, como el maravilloso 'Las pequeñas virtudes'. Publica 'Valentino' en 1957. 

- Tổng Bí thư, Chủ tịch nước yêu cầu rà soát, nhận diện, đánh giá kỹ lưỡng các "nguy cơ" để có phương hướng, giải pháp khắc phục, điều chỉnh kịp thời sau 1 năm vận hành mô hình chính quyền 3 cấp- Lễ trao Giải Báo chí Quốc gia lần thứ XX - năm 2025 diễn ra tối nay  tại thành phố Hải Phòng đánh dấu  chặng đường 20 năm hình thành và phát triển Giải Báo chí Quốc gia.- Thành phố HCM kiên quyết điều chuyển vốn đối với các dự án có tỷ lệ giải ngân thấp, đặc biệt là những đơn vị giải ngân dưới 5% hoặc chưa giải ngân tính đến cuối tháng 6/2026- Hôm nay, Thụy Sĩ trở thành tâm điểm ngoại giao quốc tế khi các phái đoàn Mỹ, Iran, Pakistan và các bên liên quan tập trung tại đây để chuẩn bị cho vòng đàm phán đầu tiên nhằm triển khai Bản ghi nhớ chấm dứt xung đột vừa được ký kết giữa Mỹ và Iran.- Châu Âu oằn mình dưới "vòm nhiệt" cực đoan, nhiệt độ tiến sát mốc 45°C

Nauji įrašai. Pristatome kinų rašytojos Jung Chang šeimos biografiją „Laukinės gulbės. Trys Kinijos dukros“. Vertė Gabrielė Gailiūtė, išleido Lietuvos rašytojų sąjungos leidykla.Tai trijų kartų XX a. Kinijos moterų istorija, kurioje dera memuarams būdingas intymumas, epo kūrybinis užmojis ir panoraminis istorijos liudytojų naratyvas. Pasakodama apie savo močiutę, motiną ir dukrą Jung Chang atskleidžia tragišką ir audringą savo šalies istoriją – nuo kardais ginkluotų karo vadų iki pirmininko Mao, nuo Mandžiūrijos imperijos iki kultūrinės revoliucijos. Knygos ištraukas skaito aktorė Vesta Šumilovaitė.

Esta semana en El Abrazo del Oso nos vamos al París de principios del siglo XX para encontrarnos con un icono inolvidable, Mata Hari. Una mujer con una historia apasionante y trágica, envuelta en un mito que voló a buen seguro mucho más alto que la realidad. Una historia llena de seducción, engaños y prejuicios con un triste final en manos de intereses mucho más mundanos que su figura. De sensual bailarina a enigmática espía en los albores de la Primera Guerra Mundial, acabaría sus días frente a un pelotón de fusilamiento para sellar por siempre su leyenda. El Abrazo del Oso 30x29 Guion: Yolanda Barreno Dirección y Producción: Eduardo Moreno Navarro Accede a más contenidos extra y haz posible la producción de El Abrazo del Oso pinchando en el botón 'apoyar' aquí en iVoox. O pásate por www.patreon.com/elabrazodeloso ¡GRACIAS! www.elabrazodeloso.es www.elaprenditivo.com Historia del Cómic: https://go.ivoox.com/sq/3144300 Sintonía de inicio y cierre: Navegantes del tiempo de José Apolo iVoox: https://go.ivoox.com/sq/3737 www.patreon.com/elabrazodeloso Programa publicado originalmente el 21 de junio de 2026. Camisetas, bolsas, tazas: www.latostadora.com/elabrazodeloso Canal de Telegram para estar informado: https://t.me/+T6RxUKg_xhk0NzE0 Grupo abierto de Telegram para conversar con el equipo y la audiencia: https://t.me/+tBHrUSWNbZswNThk Twitch: https://www.twitch.tv/elabrazodeloso Premium anual en oferta exclusiva para oyentes de El Abrazo del Oso: https://www.ivoox.vip/premium?affiliate-code=54aa1e717bf882368a5fa4f18bf67c3f ¿Quieres patrocinar este podcast?: https://advoices.com/el-abrazo-del-oso-podcast Escucha el episodio completo en la app de iVoox, o descubre todo el catálogo de iVoox Originals

La Houppa est une chanteuse fantaisiste qui a eu son heure de gloire dans l'entre-deux guerre. On est en 1985 et cette parisienne de 85 ans n'a rien perdu de son énergie débordante qui l'a fait connaître. Avec sa gouaille typique des Faubourgs parisiens, elle nous transporte dans les années folles, où elle performait comme artiste de music-hall, un genre très populaire à l'époque. Malgré les mœurs parfois douteuses du milieu, elle a réussi à mener sa carrière tout en restant fidèle à ses principes.*** Crédits archive *** Extrait de l'émission radiophonique "Atout coeur" de Francis Colnot - Première diffusion : 1985 - Radio bleue***Crédits podcast Documentaliste : Anne Brulant - Textes : Lætitia Fourmond, Edith Monnier - Restauration et mixage : Ian Debeerst - Enregistrement : Laurent Thomas - Voix off : Clara De Antoni - Musique(s) avec l'aimable autorisation d'Universal Production Music France - Chargée de production : Céline Amadori - Assistante de production : Ameline Tarnagda - Cheffe de projet : Lætitia Fourmond - Chargée de projet : Anouk Valverde, Edith Monnier - Responsable éditoriale : Zoé Macheret - Un podcast INA.

Nauji įrašai. Pristatome kinų rašytojos Jung Chang šeimos biografiją „Laukinės gulbės. Trys Kinijos dukros“. Vertė Gabrielė Gailiūtė, išleido Lietuvos rašytojų sąjungos leidykla.Tai trijų kartų XX a. Kinijos moterų istorija, kurioje dera memuarams būdingas intymumas, epo kūrybinis užmojis ir panoraminis istorijos liudytojų naratyvas. Pasakodama apie savo močiutę, motiną ir dukrą Jung Chang atskleidžia tragišką ir audringą savo šalies istoriją – nuo kardais ginkluotų karo vadų iki pirmininko Mao, nuo Mandžiūrijos imperijos iki kultūrinės revoliucijos. Knygos ištraukas skaito aktorė Vesta Šumilovaitė.

SER Viajeros se emite desde Villa Ana, una de las muchas villas preciosas que tiene esta localidad, construida a principios del siglo XX, en la playa de La Almadraba, en la conocida como ruta de las Villas. Se trata de un inmueble histórico que originalmente servía como la casa de los caseros de la vecina Villa María. Una villa restaurada por el ayuntamiento de Benicàssim para acercar la lectura a los residentes y turistas, ofreciendo servicio de préstamo y prensa diaria. Emitir SER Viajeros desde primera línea de playa, en una zona conocida a principios del siglo XX como el "Biarritz del Levante, es todo un lujo. Además, recordamos que esta localidad es Premio Ser Viajeros . Nos acompaña la alcaldesa de Benicàssim, Susana Marqués, con la que conocemos cómo se presenta este verano 2026. Desde este ayuntamiento se hace especial hincapié en la accesibilidad de sus playas: accesos y pasarelas adaptadas para las principales zonas de baño de todo el municipio.En los primeros minutos de programa conversamos también con el Director General de Turismo, Israel Martínez, con el que conocemos el compromiso de Turisme Comunitat Valenciana por un modelo más accesible, inclusivo y competitivo. Y todo esto incluye desde la accesibilidad a lo largo de toda la experiencia turística, espacios públicos como playas, cultura, y también gastronomía. Martínez insiste en que también es un modelo de sostenibilidad social, porque se ha puesto el foco en este punto como una cuestión de convivencia, reputación y competitividad de los destinos.

A través del movimiento por los derechos civiles, las movilizaciones estudiantiles, el hippismo y las nuevas luchas por el reconocimiento y la inclusión, analizamos cómo una generación comenzó a cuestionar las certezas heredadas en la posguerra y a ampliar los límites de la participación democrática. Más allá de sus expresiones más conocidas, la contracultura abrió debates fundamentales sobre la igualdad, la libertad, la diversidad, la convivencia y el sentido de vida. Una reflexión histórica sobre cómo las democracias se fortalecen cuando son capaces de incorporar nuevas voces, tramitar el desacuerdo y construir espacios comunes para personas y grupos profundamente diferentes.   Notas del Episodio  Martin Luther King Jr. y la Carta desde la Cárcel de Birmingham Aquí encontrarán uno de los textos más importantes de la historia de los derechos civiles, una carta escrita por Martin Luther King Jr. donde explica por qué la desobediencia civil puede ser una herramienta legítima frente a las leyes injustas.  Henry David Thoreau y la Desobediencia Civil El ensayo inspiró a Gandhi, Martin Luther King Jr. y numerosos movimientos pacifistas alrededor del mundo. Una reflexión sobre la relación entre conciencia, ley y justicia.  Rosa Parks y el Boicot de Autobuses de Montgomery La historia de la protesta que dio inicio al movimiento moderno por los derechos civiles en Estados Unidos y cambió el rumbo de la democracia estadounidense. (Inglés) El Movimiento por los Derechos Civiles en Estados Unidos Una introducción elaborada por Encyclopaedia Britannica sobre el proceso histórico que impulsó la ampliación de derechos y la lucha contra la segregación racial.  Black Power y Stokely Carmichael Una explicación sobre el surgimiento del orgullo afroamericano y la construcción de nuevas identidades políticas dentro del movimiento por los derechos civiles. Berkeley y el Free Speech Movement El movimiento estudiantil que defendió la libertad de expresión y el derecho al disenso dentro de las universidades durante los años sesenta. (Inglés) Rachel Carson y el nacimiento del ambientalismo moderno La historia de una de las autoras más influyentes del siglo XX y de cómo sus ideas ayudaron a construir la conciencia ecológica contemporánea. (Inglés)   Recomendaciones Culturales Película recomendada: Selma (2014) Una reconstrucción cinematográfica de las marchas por el derecho al voto lideradas por Martin Luther King Jr. en Alabama.  ➡ https://mubi.com/en/co/films/selma   Película recomendada: Malcolm X (1992) Para complementar la historia de los derechos civiles desde una perspectiva distinta a la de Martin Luther King Jr.  ➡ https://tv.apple.com/us/movie/malcolm-x/umc.cmc.4tvd4yh39kjpqi67uyd8jvl2i Película recomendada: The Trial of the Chicago 7 (2020) Para entender las protestas contra la Guerra de Vietnam y la dimensión política de la contracultura. ➡ http://netflix.com/co-en/title/81043755 Album recomendado: What's Going On – Marvin Gaye Uno de los álbumes más importantes de la historia de la música popular, dedicado a temas como la guerra, la discriminación y la justicia social. ➡ https://www.youtube.com/watch?v=H-kA3UtBj4M&list=PLnif9Rfb5AdnouIPnWr4DunLkZ7vO23Ef Canción recomendada: The Times They Are A-Changin' -  Bob Dylan (1964) Probablemente la canción que mejor resume el espíritu de cambio que atravesó la década de los sesenta. ➡ https://youtu.be/90WD_ats6eE?si=in-bQYv5TTgULj67    Sigue mis proyectos en otros lugares:  YouTube ➔ youtube.com/@DianaUribefm  Instagram ➔ instagram.com/dianauribe.fm Facebook ➔ facebook.com/dianauribe.fm Sitio web ➔ dianauribe.fm Twitter ➔ x.com/DianaUribefm  LinkedIn ➔ www.linkedin.com/in/diana-uribe    Gracias de nuevo a nuestra comunidad de patreon por apoyar la producción de este episodio. Si quieres unirte, visita www.dianauribe.fm/comunidad

Agradece a este podcast tantas horas de entretenimiento y disfruta de episodios exclusivos como éste. ¡Apóyale en iVoox! El conflicto de Irlanda del Norte, conocido con el eufemismo de “The Troubles”, se cobró cerca de 3.500 vidas entre 1968 y 1998 y marcó a fuego la política británica durante el último tercio del siglo XX. Sus orígenes hay que ir a buscarlos mucho más lejos, a la invasión inglesa del siglo XII y, sobre todo, a las plantaciones del Úlster del siglo XVII, que convirtieron al norte en la única región irlandesa de mayoría protestante. La fractura religiosa se superpuso a la nacional hasta volverse indistinguible con el tiempo. El IRA fue una sucesión de distintos grupos que se sucedieron y que, en ocasiones, se enfrentaron entre sí. Tras la guerra de la independencia y el tratado anglo-irlandés de 1921 que partió la isla, el republicanismo armado quedó relegado a la clandestinidad durante décadas. Los “Troubles” no surgieron de aquel viejo IRA, sino de la discriminación que el régimen unionista del Úlster ejercía contra los católicos en vivienda, empleo y reparto electoral. El movimiento por los derechos civiles de finales de los 60 se encontró con represión policial, algo que agravó el despliegue del Ejército en 1969, los internamientos sin juicio de 1971 y algunos episodios como el Domingo Sangriento de 1972. De todo ello se aprovechó una escisión del IRA que se autodenominó IRA Provisional. Sus miembros abogaban por una guerra larga de desgaste mediante atentados y asesinatos selectivos, simple terrorismo no muy distinto al que los extremistas de aquella época practicaban en otras partes de Europa. Pero el caso de Irlanda del Norte fue peculiar. Allí los partidarios de mantenerse en el Reino Unido formaron organizaciones “lealistas” que eran muy violentas y que, en ocasiones, colaboraban con el Estado. Las huelgas de hambre de 1981 regalaron al IRA una generación de mártires y dieron paso a la estrategia del fusil y la urna, mediante la cual, mientras una parte del movimiento, el Partido Sinn Féin, mantenía la lucha armada, otra participaba en política. Fue ya en los años 90 cuando se empezó a negociar, un proceso que consumió mucho tiempo y que culminó con el Acuerdo de Viernes Santo de 1998 que reconocía una amplia autonomía política para Irlanda del Norte y abría la puerta a la reunificación con la República de Irlanda . El IRA se desarmó años después y el Sinn Féin hoy gobierna el Úlster. Bibliografía: “No digas nada” de Patrick Radden Keefe - https://amzn.to/4oIhZLw “Matar por Irlanda” de Rogelio Alonso - https://amzn.to/43NwGDj “Guerra y paz en Irlanda del Norte” de Jacobo Celnik - https://amzn.to/4xDagSH “The Troubles” de Leon Maher - https://amzn.to/4elfP0N “Irlanda del Norte: historia del conflicto” de Luis Antonio Sierra - https://amzn.to/3QwZcWz Escucha el episodio completo en la app de iVoox, o descubre todo el catálogo de iVoox Originals

Agradece a este podcast tantas horas de entretenimiento y disfruta de episodios exclusivos como éste. ¡Apóyale en iVoox! El conflicto de Irlanda del Norte, conocido con el eufemismo de “The Troubles”, se cobró cerca de 3.500 vidas entre 1968 y 1998 y marcó a fuego la política británica durante el último tercio del siglo XX. Sus orígenes hay que ir a buscarlos mucho más lejos, a la invasión inglesa del siglo XII y, sobre todo, a las plantaciones del Úlster del siglo XVII, que convirtieron al norte en la única región irlandesa de mayoría protestante. La fractura religiosa se superpuso a la nacional hasta volverse indistinguible con el tiempo. El IRA fue una sucesión de distintos grupos que se sucedieron y que, en ocasiones, se enfrentaron entre sí. Tras la guerra de la independencia y el tratado anglo-irlandés de 1921 que partió la isla, el republicanismo armado quedó relegado a la clandestinidad durante décadas. Los “Troubles” no surgieron de aquel viejo IRA, sino de la discriminación que el régimen unionista del Úlster ejercía contra los católicos en vivienda, empleo y reparto electoral. El movimiento por los derechos civiles de finales de los 60 se encontró con represión policial, algo que agravó el despliegue del Ejército en 1969, los internamientos sin juicio de 1971 y algunos episodios como el Domingo Sangriento de 1972. De todo ello se aprovechó una escisión del IRA que se autodenominó IRA Provisional. Sus miembros abogaban por una guerra larga de desgaste mediante atentados y asesinatos selectivos, simple terrorismo no muy distinto al que los extremistas de aquella época practicaban en otras partes de Europa. Pero el caso de Irlanda del Norte fue peculiar. Allí los partidarios de mantenerse en el Reino Unido formaron organizaciones “lealistas” que eran muy violentas y que, en ocasiones, colaboraban con el Estado. Las huelgas de hambre de 1981 regalaron al IRA una generación de mártires y dieron paso a la estrategia del fusil y la urna, mediante la cual, mientras una parte del movimiento, el Partido Sinn Féin, mantenía la lucha armada, otra participaba en política. Fue ya en los años 90 cuando se empezó a negociar, un proceso que consumió mucho tiempo y que culminó con el Acuerdo de Viernes Santo de 1998 que reconocía una amplia autonomía política para Irlanda del Norte y abría la puerta a la reunificación con la República de Irlanda . El IRA se desarmó años después y el Sinn Féin hoy gobierna el Úlster. Bibliografía: “No digas nada” de Patrick Radden Keefe - https://amzn.to/4oIhZLw “Matar por Irlanda” de Rogelio Alonso - https://amzn.to/43NwGDj “Guerra y paz en Irlanda del Norte” de Jacobo Celnik - https://amzn.to/4xDagSH “The Troubles” de Leon Maher - https://amzn.to/4elfP0N “Irlanda del Norte: historia del conflicto” de Luis Antonio Sierra - https://amzn.to/3QwZcWz Escucha el episodio completo en la app de iVoox, o descubre todo el catálogo de iVoox Originals

Hay vidas que parecen demasiado grandes para caber en una sola biografía. Vidas que obligan a preguntarse si realmente una persona puede ser muchas personas al mismo tiempo. Albert Schweitzer fue una de ellas. Médico, filósofo, teólogo, músico, pensador, humanitario y Premio Nobel de la Paz. Pero por encima de todos esos títulos hubo algo que definió cada una de sus decisiones: una búsqueda constante de sentido y una convicción profunda de que el conocimiento solo tiene valor cuando mejora la vida de los demás. Nacido en la Alsacia de finales del siglo XIX, una tierra situada entre dos mundos y marcada por cambios de fronteras e identidades, Schweitzer creció entre iglesias, partituras y preguntas. Desde muy joven destacó por una inteligencia extraordinaria que lo llevó a estudiar teología, filosofía y música hasta convertirse en una figura reconocida dentro de la Europa intelectual de comienzos del siglo XX. Parecía destinado a una vida cómoda entre universidades y auditorios. Sin embargo, cuando ya había alcanzado prestigio académico y reconocimiento internacional, tomó una decisión que desconcertó a quienes lo rodeaban: volver a empezar. Estudió medicina y abandonó la seguridad de Europa para viajar al corazón de África, donde fundó un hospital y dedicó décadas de su existencia al cuidado de miles de enfermos. La historia de Albert Schweitzer no es únicamente la historia de un hombre excepcional. Es también la historia de una idea poderosa y profundamente humana: que toda vida merece respeto. Una idea que él llamó “reverencia por la vida” y que intentó aplicar en cada aspecto de su existencia, desde sus libros hasta sus consultas médicas, desde sus conciertos de órgano hasta sus discursos contra la guerra y el armamento nuclear. Hoy nos acercamos a una figura que desafió las etiquetas y que convirtió su propia vida en una pregunta abierta para todos nosotros: ¿qué hacemos realmente con el talento, el tiempo y las oportunidades que recibimos?

Nauji įrašai. Pristatome kinų rašytojos Jung Chang šeimos biografiją „Laukinės gulbės. Trys Kinijos dukros“. Vertė Gabrielė Gailiūtė, išleido Lietuvos rašytojų sąjungos leidykla.Tai trijų kartų XX a. Kinijos moterų istorija, kurioje dera memuarams būdingas intymumas, epo kūrybinis užmojis ir panoraminis istorijos liudytojų naratyvas. Pasakodama apie savo močiutę, motiną ir dukrą Jung Chang atskleidžia tragišką ir audringą savo šalies istoriją – nuo kardais ginkluotų karo vadų iki pirmininko Mao, nuo Mandžiūrijos imperijos iki kultūrinės revoliucijos. Knygos ištraukas skaito aktorė Vesta Šumilovaitė.

Industrializacja Górnego Śląska wcale nie zaczęła się od węgla kamiennego. W XIX wieku najważniejsze były kopalnie cynku i ołowiu oraz huty żelaza, wszystko to w dużej mierze na potrzeby przemysłu zbrojeniowego. – Pod koniec XIX wieku właściwie prawie całość produkcji cynku i ołowiu europejskiego tutaj się koncentrowała – mówi prof. Ryszard Kaczmarek z Instytutu Historii Uniwersytetu Śląskiego. Przed wami jeden z dwóch odcinków o historii i tożsamości Górnego Śląska.

Año XX. Núm. 58. Pensamos que el soberbio se cree mejor que los demás, pero puede suceder exactamente al revés.

En este episodio analizo el caso de los 60 estudiantes excluidos por recibir un 0 en vascuence en la PAU del País Vasco, la antigua selectividad.

What are you keeping and why? Let's discuss what's in your house, and why letting go feels so hard. It certainly helped us to understand ourselves and our family members. And maybe it will help you sort through your things and make some hard decisions.The things we keep representmemoriesidentityhopes for the futureguiltobligationcomfortand even fearListen to our episode on the art of Swedish Death cleaning HEREWe participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.Check out Kelly's app HERE !Anita's crush is the podcast Dish HEREKelly's crush is the follow up book to The Gentle Art of Swedish Death Cleaning - The Swedish Art of Aging Exuberantly! HERENeed help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 15:33 / 22:53See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

On this week's show Patrick Gray, Adam Boileau and James Wilson discuss the week's cybersecurity news. They cover: Anthropic's Fable 5 and Mythos 5 get nuked by the US government four days after launch “because security” Why “guardrails” won't keep the world safe from your AI doomsday machine The FISA 702 statute expired, but the spying can (probably) continue! NPM v12 delivers some protection against supply chain attacks, but not enough. Microsoft has a series of bugs that prevent Windows Update from … updating Much, much more! This episode is also available on YouTube Show notes Anthropic suspends new AI models after government directive | NBC News Tech Anthropic rankles users with safety-first Fable release | NBC News Tech How a 90-minute White House deadline sparked Silicon Valley's biggest AI fight | washingtonpost.com Pete Hegseth (@PeteHegseth) on X | X (formerly Twitter) David Sacks (@DavidSacks) on X | X (formerly Twitter) DoW CIO Kirsten Davies (@DoWCIODavies) on X | X (formerly Twitter) David Shulman (@DavidShulmanFL) on X | X (formerly Twitter) Controversial FISA spying law expires tonight. The spying will continue. | Ars Technica GitHub announces npm security changes to tackle supply-chain attacks | BleepingComputer Why NPM v12 won't stop supply chain attacks - Risky Business Media | Social Signals Oracle PeopleSoft servers hacked in ShinyHunters data theft attacks | BleepingComputer Microsoft patches Exchange Server zero-day exploited in attacks | BleepingComputer Max severity Ivanti Sentry vulnerability now exploited in attacks | BleepingComputer CISA warns of another cPanel plugin flaw exploited in attacks | BleepingComputer Critical Fortinet FortiSandbox flaws now exploited in attacks | BleepingComputer CISA orders feds to patch actively exploited Ivanti flaw by Sunday | BleepingComputer CISA to require federal agencies to patch some cyber vulnerabilities within 3 days | therecord.media Path traversal flaw in AI dev platform Langflow exploited in attacks | BleepingComputer Microsoft: Some Windows PCs fail to install latest monthly updates | BleepingComputer Microsoft fixes BitLocker recovery bug on Windows Server 2025 | BleepingComputer Microsoft fixes Windows update failures linked to WUSA installer | BleepingComputer New attack turned Microsoft 365 Copilot into 1-click data theft tool | BleepingComputer Over 73,000 French govt employees affected in Tchap messenger breach | BleepingComputer Signal Alums Reveal ‘Encrypted Spaces,' a System for Making Private Collaboration Apps | wired.com FBI disrupts massive AI-powered phishing service using a million URLs | BleepingComputer Cyberattack shuts down major Australian sugar mills, disrupting harvest | The Record Drug Sites Hijacked Spotify's Search Ranking Through Fake Podcasts, Report Finds | wired.com It Is Trivially Easy to Use Reddit to Manipulate AI Search, Research Suggests | 404.feed.press Who Runs the Ransomware Group ‘The Gentlemen?' | krebsonsecurity.com :brdKnife: (@cR0w@infosec.exchange) | Infosec Exchange

El 17 de junio de 1882 nació Ígor Stravinski, fue un compositor y director de orquesta ruso y uno de los músicos más importantes y trascendentales del siglo XX.

What if your burnout isn't just a scheduling problem — what if it's an energy problem? In this powerful episode, we explore why emotional healing is the missing piece in most nurse leader recovery plans. Drawing from Scripture, quantum physics, and the bioresonance technology behind WholeBio Insights, Shan reveals how unresolved emotions are stored in the body as disrupted frequencies — and why you can't think your way out of what your nervous system is holding.   Joined by Aligned leadership coach, Kassandra Hamilton, this episode takes you inside a real burnout story and the emotional work that made recovery possible. If you've tried all the strategies and still feel depleted, this episode will help you understand why — and what to do about it.   What You'll Learn in This Episode This episode is for you if you've been asking why rest isn't enough, why strategies aren't sticking, or why you still feel empty even when things look okay on the outside. Here's what we cover:   Why burnout is fundamentally an energy problem — and what the Bible has always said about it The Hebrew meaning of Shalom and why it's actually a description of energetic wholeness What Proverbs 17:22 reveals about the body-emotion-health connection (that science is just now catching up to) How Mark Virkler's "Prayers that Heal the Heart" framework reframes emotional healing as a clinical necessity, not just spiritual work What quantum physics' Observer Effect has to do with your burnout recovery Why emotion is literally "energy in motion" — and what happens when you suppress it How bioresonance technology (WholeBio Insights™) reads the body's frequencies to reveal emotional disruption before symptoms become diagnosable Kassandra Hamilton's honest account of what burnout looked and felt like as a leader — and what it actually took to heal   About Our Guest: Kassandra Hamilton Leadership & Alignment Coach, Best-Selling Author, Facilitator, Bioenergy & Sound Healer, and quality improvement specialist in the healthcare field.

What I do: I align high achieving women with a life that feels as good on the inside as it does on the outside. 

Think More energy, time freedom, less stress.

Kassandra's links: 
LinkedIn: https://www.linkedin.com/in/kassandra-hamilton-15b8b175/

Facebook: https://www.facebook.com/VancouverIslandHealing

Instagram: https://www.instagram.com/kassandra_hamilton/
Tik tok: https://www.tiktok.com/@coachingwithkassandra?_r=1&_t=ZS-92Iynv6RAoe

Website: www.readysetrealign.ca

Link to BestSeller Book: https://a.co/d/2yWISSu
   Key Takeaways "Burnout is not a scheduling problem. It is an energy problem. And you cannot fix an energy problem with a schedule change."   "Management and healing are not the same thing. You can manage your emotions your whole life and still be running on frequencies of unresolved fear and grief that are quietly depleting you."   "The word emotion is energy in motion. When we suppress emotion, we stop that energy in its tracks — and stored emotional energy creates disruption in the body's field."   Scriptures Referenced "He gives strength to the weary and increases the power of the weak."  — Isaiah 40:29, NIV "A cheerful heart is good medicine, but a crushed spirit dries up the bones."  — Proverbs 17:22, NIV "For as he thinks in his heart, so is he."  — Proverbs 23:7, NKJV "The Spirit of God has made me; the breath of the Almighty gives me life."  — Job 33:4, NIV "We all, who with unveiled faces contemplate the Lord's glory, are being transformed into his image with ever-increasing glory."  — 2 Corinthians 3:18, NIV   Resources & People Mentioned Books & Teachers cwgministries.org Prayers that Heal the Heart by Mark & Patti Virkler — glorywaves.org Charity Virkler Kayembe — quantum physics and Christian spirituality — "Quantum Glory" (framework referenced) — the science of heaven invading earth   Work With Shan: The Wholly Well Intensive To learn more: DM "360" to Shan on Instagram @theshanwright or email: hello@theshanwright.com Connect With Shan Instagram: @theshanwright Website: theshanwright.com Stress Less Community (Storehouse Wellness)    Shalom Shalom,  Xx, Shan  ……CONNECT…… The STAT Protocol: 5 min Emergency Reset Take the Free QUIZ-  Are you in burnout or just stressed??

What questions should you be asking the people you love while you still can?That's the thread running through this conversation with Dorothy Roberts, whose memoir The Mixed Marriage Projectstarted with a stack of boxes. After her parents passed, Dorothy opened them and found nearly 500 interviews her white father had conducted with interracial couples in Chicago, beginning in 1937, almost two decades before he married her Black mother. Inside were wild parties, a nudist camp, a turn-of-the-century club for mixed couples, and a file labeled number 224 that turned out to be about her.We talk about the interviews her father never published and the book contracts he kept walking away from, why she hid her father's whiteness from her Black classmates at Yale, and the painful stereotypes that surfaced even inside a club devoted to interracial marriage. We get into the big question at the heart of the memoir too: whether love, the everyday intimate kind, can actually dismantle racism. Spoiler, it's complicated.Dorothy Roberts is a scholar, professor, and author of five books on race, gender, and the systems that devalue Black women and mothers. The Mixed Marriage Project is her first memoir, built from the nearly 500 interviews her father left behind. If this conversation moved you, share it with a friend and leave a review and rating.Purchase The Mixed Marriage ProjectSupport the show:On PatreonBuy us a bookBuy cute merchSubscribe to the Babes in Bookland SubstackConnect with us and suggest a great memoir!Follow us on instagram! @babesinbooklandpod Thank you for listening!Xx, Alex Hosted on Acast. See acast.com/privacy for more information.

Ian de la Rosa debuta en el largometraje con Iván & Hadoum, una historia de amor ambientada en los invernaderos de Níjar (Almería) que llega a los cines tras conquistar el Teddy Award del Festival de Berlín y dos Biznagas de Plata en el Festival de Málaga. La película sigue la relación entre Iván, un joven trans, y Hadoum, una chica de origen marroquí, y aborda cuestiones como la identidad, el amor y la búsqueda de un futuro en un entorno marcado por la precariedad laboral y las diferencias culturales.La actualidad cultural pasa también por F. Javier Cárdenas García, ganador del XVIII Premio de Poesía Joven RNE-Fundación Montemadrid por Latitud cero, un poemario que ha destacado por su madurez poética y su capacidad para evocar Ecuador a través de imágenes sugerentes. Además, el Centro KBR de Barcelona dedica una gran retrospectiva a Minor White, una de las figuras fundamentales de la fotografía del siglo XX, con imágenes que se muestran por primera vez en Europa y que recorren algunos de los temas centrales de su obra, como el paisaje, el retrato y el desnudo masculino.El programa se acerca también a la historia del teatro y del cine español con la adaptación de La escopeta nacional, la célebre comedia de Luis García Berlanga y Rafael Azcona, dirigida por Juan Echanove en el Teatro Español de Madrid. Nuestra colaboradora Xaviera Torres recuerda el descubrimiento de los infrarrojos por parte del astrónomo William Herschel, mientras que Enrique Mejías nos lleva hasta el Festival de Bayreuth, el certamen creado por Richard Wagner que celebra este verano su 150 aniversario y sigue siendo una de las grandes citas de la música clásica internacional.Escuchar audio

En Haïti, dix ans après le dernier scrutin, qui avait porté Jovenel Moïse au pouvoir, le gouvernement du Premier ministre Alix Didier Fils-Aimé peine à faire avancer le processus électoral. Le report d'une première échéance prévue fin août 2026, la publication controversée du décret électoral et la nomination d'un directeur général au sein du Conseil électoral provisoire alimentent les tensions. Nous en avons parlé avec Édouard Paultre, du Conseil haïtien des acteurs non étatiques. Dans un contexte d'insécurité persistante, malgré le début des opérations de la Force de répression des gangs, aucun calendrier électoral n'est actuellement fixé. Selon Édouard Paultre, du Conseil haïtien des acteurs non étatiques, la communauté internationale pourrait néanmoins pousser à la tenue d'élections. Reste une autre inconnue majeure : le rôle que pourraient jouer les gangs, dont les ressources financières considérables pourraient leur permettre d'influencer les futures campagnes. À lire aussiHaïti: l'incertitude sur le processus électoral Venezuela : la crise électrique s'aggrave Au Venezuela, les coupures de courant faisaient déjà partie du quotidien dans de nombreuses régions du pays. Mais depuis le 3 janvier 2026, le phénomène s'est aggravé. Des villes jusqu'ici relativement épargnées, comme Valencia, principal centre industriel du pays, subissent désormais jusqu'à douze heures de coupure d'électricité par jour. Ces pénuries affectent non seulement la vie quotidienne des Vénézuéliens, mais pèsent aussi lourdement sur les entreprises. Un coup dur pour un pays qui tente de relancer son économie et de séduire les investisseurs, alors que la crise énergétique vient fragiliser un peu plus la reprise. Reportage d'Alice Campaignolle.   À lire aussiVenezuela: face au manque d'électricité, le gouvernement réduit les horaires des fonctionnaires À Iquitos dans l'Amazonie péruvienne, une prospérité bâtie sur les violences du caoutchouc Au cœur de l'Amazonie péruvienne, la ville d'Iquitos réserve bien des surprises. Ses bâtiments de style Art nouveau contrastent avec les eaux de l'Amazone et la végétation luxuriante qui l'entourent. Un héritage du début du XXè siècle, lorsque la ville a connu un spectaculaire essor économique grâce au commerce du caoutchouc. Mais cette période longtemps célébrée est aujourd'hui de plus en plus remise en question par les jeunes générations des communautés locales, nous explique notre correspondant Martin Chabal. Elles souhaitent faire entendre la voix des peuples autochtones de la région, exploités et victimes de massacres perpétrés par les compagnies du caoutchouc. À écouter aussiUne sixième année de déficit mondial pour le caoutchouc naturel Journal 1ère On annonçait un « arrivage exceptionnel », mais finalement les algues sargasses seraient restées plutôt discrètes ce week-end en Martinique. À écouter aussiPourquoi les sargasses envahissent-elles chaque année les plages des Caraïbes?

Fue una de las figuras más singulares de la cultura española del siglo XX. Hija y nieta de maestras, la vida de Josefina Aldecoa (1926-2011) está marcada por la herencia pedagógica de la Segunda República. Imbuida del espíritu de la Institución Libre de Enseñanza, entendió la enseñanza como una forma de transformación social y la escritura como aliada de la memoria. Conoció de primera mano los sistemas educativos británico y estadounidense. En 1959, en plena dictadura, funda el Colegio Estilo en Madrid, un espacio de libertad, creatividad y pensamiento crítico por el que pasaron hijos de destacados escritores, cineastas, artistas e intelectuales de la época como Antonio Buero Vallejo, Alfonso Sastre, Fernando Vizcaíno Casas, Juan Antonio Bardem, Luis García Berlanga o Antonio López.Este documental sonoro, con guion de Minerva Oso y realización de Samuel Alarcón, recorre la biografía de la autora, desde su infancia en las montañas de León hasta la vinculación con la generación del 50. Fue crucial su relación con el escritor Ignacio Aldecoa, del que tomó el apellido tras su prematura muerte. En obras como 'Historia de una maestra', 'La enredadera' o 'Desde la distancia' plasmó su mirada sobre el contexto social y educativo de la España contemporánea. El programa cuenta con los testimonios de su hija, Susana Aldecoa; los periodistas Juan Cruz y Amelia Castilla; la doctora en literatura Estefanía Cabello; y la historiadora de la educación María del Mar del Pozo. Participan también los antiguos alumnos Teo Planell, Manuela Velasco, Marcos Giralt Torrente, Pablo Mingote y Carlos Saura, y profesoras del Colegio Estilo como Carmen Carbajosa. Además, suena la voz de la propia Josefina, conservada en el Archivo RTVE.Escuchar audio

En 1991, un equipo de geólogos de la Universidad de Boston realizó estudios sísmicos alrededor de la Esfinge y detectó una cámara rectangular de varios metros de profundidad exactamente debajo de sus patas delanteras. Nunca se excavó. En 1987, un equipo japonés de la Universidad de Waseda ya había reportado un túnel orientado de norte a sur corriendo bajo el cuerpo del coloso. Tampoco se investigó. En 2025, escaneos satelitales con tecnología de radar de apertura sintética detectaron estructuras subterráneas que se extienden dos kilómetros bajo el plateau de Giza. El Consejo Supremo de Antigüedades de Egipto no emitió ningún comentario.Pero la historia no empieza en el siglo XX. Heródoto describió en el año 450 a.C. pasajes subterráneos que conectaban las grandes pirámides. Cronistas árabes medievales documentaron entradas ocultas en los costados de la Esfinge con mecanismos que solo los sacerdotes iniciados conocían. Y una estela descubierta en 1858 afirma que el faraón Khufu encontró la Esfinge ya enterrada en arena cuando llegó a Giza — lo que significaría que el monumento precede incluso a las pirámides.En este episodio rastreamos el hilo completo. Los datos están. Los testimonios están. Lo que no está es la voluntad institucional de seguirlos hasta donde llevan. Hosted on Acast. See acast.com/privacy for more information.

durée : 00:58:35 - Concordance des temps - par : Jean-Noël Jeanneney - Écrivain de talent, Jacques Benoist-Méchin fut aussi l'un des plus ardents défenseurs de la collaboration avec l'Allemagne nazie. Condamné à mort puis gracié, il incarne l'un des parcours les plus déroutants du XXᵉ siècle. Éric Roussel revient sur ce destin paradoxal. - réalisation : Vincent Abouchar, Jeanne Guérout - invités : Eric Roussel Journaliste et écrivain Vous aimez ce podcast ? Pour écouter tous les épisodes sans limite, rendez-vous sur Radio France

ARCHIVO MEXICANO DEL TERROR YO VENDÍ MI NOMBRE, GUADALUPE DUEÑAS Ninguna narración, por extraña que pueda parecer, se crea «de la nada»: todas dependen del contexto en el que vive su autor, aunque este no se refleje de manera directa. El caso de Guadalupe Dueñas es prueba de esto: su obra expresa una visión del mundo mediante los símbolos que le da la imaginación. Formada en una familia numerosa y de rígidos principios religiosos, Dueñas se educó primero en conventos. Allí comenzó a escribir por rebeldía, como una forma de oponerse a una vida que le resultaba asfixiante, y los pocos testimonios que hay sobre su vida sugieren constantemente la misma tensión entre convencionalismos y creatividad individual, entre pasado y presente. Más tarde hizo estudios de literatura en la UNAM, fue discípula de la escritora Emma Godoy y becaria del Centro Mexicano de Escritores. Un amigo de su familia, el sacerdote y crítico literario Alfonso Méndez Plancarte, le había recomendado que se dedicara exclusivamente a la prosa; Dueñas lo hizo así y exceptuando trabajos ocasionales como guionista y articulista toda su obra consiste en maravillosas narraciones breves. Tiene la noche un árbol (1958), se convirtió en poco tiempo en una de las cuentistas mexicanas más destacadas de mediados del siglo XX. Después publicó comparativamente pocos libros: No moriré del todo (1976), Imaginaciones (1977) y Antes del silencio (1991). Una producción de Historias para ser Leídas. La música pertenece a Epidemic Sound con Licencia autorizada para este Podcast. Este podcast se sostiene gracias a tu apoyo y a tus aportaciones, puedes aportar si lo deseas desde 1,99€ 💀 Por todas las horas de diversión: ✨ Gracias por tu escucha y hasta el próximo audio 🚀 🖤 Voz y sonido Olga Paraíso BIO Olga Paraíso: https://instabio.cc/Hleidas Escucha el episodio completo en la app de iVoox, o descubre todo el catálogo de iVoox Originals

Le mix de Dannic dans 103 Klubb le 06 Juin 2026 de 19H à 20H Tracklist: Mixmasters, Mellizos - My Life Is A Disco (In The Mix) *** Alenx B - House Religion *** Ellison - Set Fire To The Rain x Rain (Hard Mashup) *** Melsen vs. D.O.D, Carla Monroe - What I Feel x Still Sleepless (Melsen Mashup) *** NIIKO x SWAE, KSHMR feat. DEV - Bass Down Low *** Dannic & Dastic - Dim Your Light *** Nicky Romero & Deniz Koyu vs. Post Malone - Tomorrow Comes vs Chemical (SunJay Mashup) *** Dannic - My Mind (VIBR Edit Vincent Brouwer) *** Firebeatz - Pump That Bass *** Afrojack - Polkadots (Reloader 2026 Edit) *** Yves V & VIKTOR - Fit The Tempo *** Daddy's Groove vs. Justin Michael vs. Dada Life - Born To Follow Synthemilk (Dyro Mashup) *** Mike Williams & Phillip Strand x Kesha - All My Life x Die Young (AIRAES Mashup) *** Dannic - Chasing Freedom vs Beauty & The Beat (Dannic Mashup) *** Dannic vs. Delerium vs. The XX vs. Tiësto - Tombo vs Silence vs... (Dimitro Re-Cut Mashup) *** Dannic - I'll Be There For You *** Yohann Warren feat. Scarlett - Think About *** Dannic vs. Calvin Harris & Alesso & Hurts vs. Martin Garrix & Matisse & Sadko - Wake Me Up vs Under Control vs... (SunJay Mashup) *** Skrillex, Ahadadream, Raf Saperra - Bass Dhol *** Tiësto, Olivia Sebastianelli - Don't Lose Your Head *** Swedish House Mafia x Daft Punk, Pejt - Don't You Worry Child x One More Time (AIRAES Mashup)

En esta primera parte de la saga sobre Francisco Franco, Leyendas Legendarias explora los orígenes de uno de los dictadores más importantes y polémicos del siglo XX. Lejos de la imagen clásica del líder carismático y dominante, descubrimos a un hombre inseguro, acomplejado, rechazado por su padre y eclipsado por un hermano mucho más exitoso. El episodio recorre su infancia, su paso por la academia militar, la brutal campaña colonial en Marruecos y la construcción del mito de la "Baraka", la supuesta suerte sobrenatural que convenció a muchos de que estaba destinado a gobernar España. También puedes escucharnos en Spotify, Apple Podcasts, Amazon Music o tu app de podcasts favorita. Apóyanos en Patreon:https://www.patreon.com/leyendaspodcast Apóyanos en YouTube:https://www.youtube.com/c/leyendaslegendarias/join Visita nuestra página para ver contenido extra:https://www.leyendaslegendarias.com Síguenos:https://instagram.com/leyendaspodcasthttps://www.tiktok.com/@leyendaspodcasthttps://twitter.com/leyendaspodcasthttps://facebook.com/leyendaspodcast #Podcast #LeyendasLegendarias

Is your home ready for summer? School's out and we're here to help get your home ready for the summer months.Anita is focused on entertaining, and has lots of ideas for your table. You can find the items she mentioned in the episode HERE.Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.We participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.The rattan pagodas are HERE and tole pagodas are HERE.Discover & explore Kelly's Home Design Coach App HERE. Take the quiz and get your personalized plan!Anita's crush is the movie Impromptu about author George Sand (Judy Davis) who wears men's clothing in 1830's and falls in love with composer Frédéric Chopin (Hugh Grant). It's fun and not too serious. HERE. Kelly's crush is the gorgeous book Glorious Gardens: Private Edens of the World's Leading Interior Designers. Get your copy HERE.Need help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 7:03 / 14:29See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.

Links:Canal jorge Uesu falando do Maik https://www.instagram.com/reels/DZYcFzzqUSI/Curso Cabala e Magia Planetária com Marcos Keller: https://www.sympla.com.br/evento-online/fundamentos-e-pratica-da-cabala-hermetica-e-magia-planetaria/3424981Camisetas Mundo Freak: https://umapenca.com/mundofreak/loja/?srsltid=AfmBOoqsX2AWTRFB-FUyRZnCnRrfrZjDjoCtaZalh2J2cF_lCI4OiqaUNa live de hoje do Mundo Freak, vamos mergulhar na longa e fascinante história do ocultismo ocidental, um conjunto de tradições esotéricas, filosóficas e espirituais que atravessa séculos e ajudou a moldar parte do imaginário do Ocidente.A conversa começa nas origens mais antigas desse pensamento, passando por Alexandria, pelo hermetismo, por Hermes Trismegisto, pelo gnosticismo, pelo neoplatonismo e pelas formas como esse conhecimento sobreviveu, foi codificado e transformado ao longo da Idade Média. A pauta também entra em temas como alquimia, cabala, grimórios medievais e a ideia de que o ocultismo sempre esteve ligado à busca por uma gnose, um conhecimento interior e transformador.Ao longo da live, a gente avança para o Renascimento, quando magia, filosofia e ciência ainda caminhavam lado a lado, e passa por figuras fundamentais como John Dee, além do surgimento de tradições e estruturas como rosacrucianismo, maçonaria, Éliphas Lévi, Helena Blavatsky e a Golden Dawn, que ajudaram a organizar o ocultismo moderno.Na reta final, o papo chega ao século XX com nomes incontornáveis como Aleister Crowley, Jack Parsons e Anton LaVey, mostrando como o ocultismo saiu dos círculos iniciáticos e passou a dialogar com contracultura, mídia, ciência, espiritualidade alternativa e cultura pop.Se você gosta de história do esoterismo, magia cerimonial, sociedades secretas, alquimia, hermetismo, cabala, Crowley, satanismo moderno, mistérios históricos e da relação entre ocultismo e cultura contemporânea, essa live é para você.▶ Assista e participa no chat: qual fase da história do ocultismo mais te intriga, Antiguidade, Renascimento, sociedades secretas ou século XX?As Duas Vidas de Rudolf: https://open.spotify.com/show/3okz3u1rW9O0Lz3fdfmqZiApoie o Mundo Freak: https://apoia.se/confidencialRafael Jacauna Autor (Instagram): https://www.instagram.com/rafaeljacaunaautor/Lynda MD: http://lyndamd.com.brAnuncie com a Paratopia: https://www.instagram.com/paratopiapodcast/Edição: https://www.instagram.com/instadogrmachado/#MundoFreak#LiveMundoFreak#Ocultismo#OcultismoOcidental#Hermetismo#Alquimia#Cabala#AleisterCrowley#SociedadesSecretas#Esoterismo

En esta primera parte de la saga sobre Francisco Franco, Leyendas Legendarias explora los orígenes de uno de los dictadores más importantes y polémicos del siglo XX. Lejos de la imagen clásica del líder carismático y dominante, descubrimos a un hombre inseguro, acomplejado, rechazado por su padre y eclipsado por un hermano mucho más exitoso. El episodio recorre su infancia, su paso por la academia militar, la brutal campaña colonial en Marruecos y la construcción del mito de la "Baraka", la supuesta suerte sobrenatural que convenció a muchos de que estaba destinado a gobernar España. También puedes escucharnos en Spotify, Apple Podcasts, Amazon Music o tu app de podcasts favorita. Apóyanos en Patreon:https://www.patreon.com/leyendaspodcast Apóyanos en YouTube:https://www.youtube.com/c/leyendaslegendarias/join Visita nuestra página para ver contenido extra:https://www.leyendaslegendarias.com Síguenos:https://instagram.com/leyendaspodcasthttps://www.tiktok.com/@leyendaspodcasthttps://twitter.com/leyendaspodcasthttps://facebook.com/leyendaspodcast #Podcast #LeyendasLegendarias

What would it mean to build women's sports around women's bodies? This week, my friend Becca returns to discuss Lauren Fleshman's memoir "Good for a Girl". What starts as a running memoir quickly reveals itself as something far bigger: an investigation into the science of female puberty and athletics, the eating disorder epidemic inside women's collegiate and professional sports, and one woman's relentless fight to change a sport she loves from the inside out. We discuss Lauren's unconventional path to running, the ways puberty has been treated as "the one injury a girl can't come back from," the NCAA's staggering lack of policy around eating disorders in women's athletics, Lauren's famous Nike "Objectify Me" campaign, and the ways female athletes are still being failed by the systems built to protect them.This episode is for every woman who was ever told she was good — for a girl. The runners, the former athletes, the moms of daughters in sports, and anyone who has ever felt their body was working against them instead of for them.If this conversation resonated with you, the best thing you can do is share it with a friend who needs to hear it, and rate and review the show wherever you listen. It makes a huge difference!Purchase "Good for a Girl"Other Links:https://www.milesplit.com/articles/211759/dear-younger-me-lauren-fleshman (Lauren's letter to her younger self)Follow Becca's bookstagram: @bookedwithbecca and her new running account: @run.with.beccaSupport the show:On PatreonBuy us a bookBuy cute merchSubscribe to the Babes in Bookland SubstackConnect with us and suggest a great memoir!Follow us on instagram! @babesinbooklandpod Thank you for listening!Xx, Alex Hosted on Acast. See acast.com/privacy for more information.

Dr. Deb Muth 00:00:09 Hi there, how are you? Bob Miller 00:00:10 Excellent! Pedaling as fast as humanly possible, but doing okay. Dr. Deb Muth 00:00:14 Good, good. Well, I’m looking forward to our conversation today. This should be amazing. Bob Miller 00:00:20 Yeah, it should be a lot of fun. Dr. Deb Muth 00:00:22 Yeah, anything that’s off-limits for you in, our conversation? Bob Miller 00:00:28 No. Dr. Deb Muth 00:00:29 Okay, anything you want me to make sure we cover for you? Bob Miller 00:00:33 Well, I mean, is it okay if we put a little plug-in for our software? Dr. Deb Muth 00:00:35 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:36 Yeah. Dr. Deb Muth 00:00:37 Absolutely. Bob Miller 00:00:38 Hey, can we… can we do a screen share? Yes, we can. Yeah, because I want to show you some maps, and… Dr. Deb Muth 00:00:43 Okay. Things like that, yeah, so… Perfect. So just let me know when you want to do screen share. Bob Miller 00:00:48 Okay. Dr. Deb Muth 00:00:49 And yeah, feel free to plug your software wherever you want to. Bob Miller 00:00:53 Okay, well, good. Let me pull up a, a slide for that, and give me one second, I just want to shut the door to my office to get the noise down. Dr. Deb Muth 00:01:01 No worries. Bob Miller 00:01:16 And, how should I refer to you? Dr. Debb? Dr. Muth, what do you like? Dr. Deb Muth 00:01:18 Dr. Deb is great, or Deb, either way, I’m pretty informal, so… Bob Miller 00:01:22 Yeah, and… Bob is fine for me. Okay. Yeah. Yeah, there you go. Why people feel like they need this, son. Special name, it’s like, seriously. Dr. Deb Muth 00:01:33 Right? I agree. Bob Miller 00:01:35 When I work with my clients, it’s like, Dr. Millison, just, just bop, just, just bop. Dr. Deb Muth 00:01:41 Yep, that’s how I am, too. Just call me Deb, it’s good. Dr. Deb Muth 00:01:44 They feel a little awkward with that, you know? They’re not used to that, but… Bob Miller 00:01:48 Alright. And you’re a naturopath, medical doctor. Dr. Deb Muth 00:01:52 A nastropathic doctor and a nurse practitioner. Oh, nice. Yeah, so I got the best of both worlds, right? Bob Miller 00:01:58 Yeah, damn. Okay. Alright, so here we go… There we go. Alright, so I got that ready, and then I will do a, I will do a screen share. I think you’re gonna really, appreciate what we’ve come up with. We’ve come up with the concept of, Cellular CPR. Dr. Deb Muth 00:02:23 Oh, nice! Bob Miller 00:02:24 And that is, construct the cell membrane, Protect the cell membrane. And restore it if it’s damaged. Dr. Deb Muth 00:02:32 Love that. Bob Miller 00:02:34 I love that. Yeah, so that’s what we’re focusing on, and then how, You know, we want to get to the point that, you know, most people think of genetics, they think of, like, 23andMe or Ancestry. Dr. Deb Muth 00:02:44 Yeah. Bob Miller 00:02:45 And then you have the professional geneticists who are looking at, you know, odd things that could create a disease. We’re looking at functional genomics. Dr. Deb Muth 00:02:54 Which is so much better. Bob Miller 00:02:56 Yeah. Are you familiar with what we do here, or… Dr. Deb Muth 00:02:58 A little bit, a little bit. So, it’ll be new to me, too, so I’m excited. Bob Miller 00:03:03 And how much time do we have? Dr. Deb Muth 00:03:04 We have an hour, give or take a little bit on either side. Do you have a hard stop anywhere? Bob Miller 00:03:10 No, no, I put a, I moved my clients around, and I don’t have anybody till, 3.30, so we’re good. Okay. Dr. Deb Muth 00:03:16 Perfect. Alright. Bob Miller 00:03:18 It’s like we’re getting started early as well, so… Dr. Deb Muth 00:03:19 Yeah, we’re getting started a little bit early, so that’s good. Bob Miller 00:03:22 Yeah, I just got my office cleaned up, so… Dr. Deb Muth 00:03:23 Okay, good. All right, are you all set to get started? Bob Miller 00:03:28 I’m good to go, my friend. Dr. Deb Muth 00:03:29 I’m gonna just record a little intro and a little bit of a, hook for people, and then we’ll get started. I’ll ask you to kind of tell us a little bit about yourself, and then we’ll just take this conversation wherever it’s supposed to go. Bob Miller 00:03:39 Okay, you got it. Dr. Deb Muth 00:03:40 Alright, sounds good. So what if the reason you’re not healing isn’t your diet, your supplements, or your labs, but it’s actually your genes? Dr. Bob Miller is uncovering how genetic variants, when combined with modern toxins, explain why some of us stay sick no matter what we try. Today, we’re talking genetic pathways, detox blocks, and the new science every wellness warrior needs to know. Welcome back to Let’s Talk Wellness Now, the show where we uncover the root causes of chronic illness, exploring cutting-edge regenerative medicine, and empower you to heal from the inside out. I’m Dr. Deb, your medical detective, and today, our guest, Dr. Bob Miller, is a true pioneer in functional genomics. He’s a board-certified traditional naturopath and the founder of Neutrogenetic Research Institute. And he’s the leading groundbreaking research on how genetic variants influence chronic illness, inflammation, and detoxification. His work has been recognized on international stages, uncovering links between genetic expression and conditions like Lyme disease, mast cell activation, or MCAS, and mitochondrial dysfunction. I’m so excited to talk to Dr. Bob today. He is gonna reveal some things that even I don’t know about, so I’m excited to learn alongside of you guys. So… Dr. Bob, let’s get started. Tell us a little bit about yourself, and kind of how you got on this journey. Bob Miller 00:05:04 Well, that’s, that’s interesting. I was sort of like a mid-career coming to the natural health field, because in my early 30s, I found myself with a severe case of ulcerative colitis. Bob Miller 00:05:15 And I was in the hospital for 21 days. probably within hours of death, pleading to death. And they told me I’ve got one option, and that is cut out the colon and wear a bag. Didn’t sound like a lot of fun. Dr. Deb Muth 00:05:27 Not an option I would want. Bob Miller 00:05:29 So, you know, the medical folks wasn’t real happy with me, but I said, yeah, I’d like to explore some alternative things.Never thinking that I’d get into this field, and then I just, you know, worked with some herbalists and things that I found absolutely fascinating. So, that’s how I got into this around 30 years ago. And, haven’t looked back since, and just having a… having a blast as we now move into how our genetics impacts things. So, that’s what we’re gonna… that’s what we’re gonna talk about today. Dr. Deb Muth 00:05:58 I’m excited to talk about this genetic thing. When you started over 30 years ago, what kind of patience and problems first inspired you to dig deeper into that root cause healing and kind of get into the genetic piece of it? Bob Miller 00:06:10 Sure. Well, you know, as a… now, I’m in a part of the country called Lancaster County, Pennsylvania, where there’s a lot of Amish and Mennonite, and they gravitate towards these things.So, this is their first thing to do, and that doesn’t work, then they’ll go other routes. So, you know, back then, we just saw typical, you know, a little tired, constipation. You know, a little bit of fatigue, arthritis, those kind of things. But things have changed dramatically over the years, as people are now getting more chronically sick. You know, it’s worse than it’s ever been. And what we’re finding is the, the culprits Primarily is mold exposure and Lyme disease. When people get those two together, they’re just… it’s an inflammatory cascade that nobody can seem to unravel. So that’s where we spend a lot of our time. And we’re also spending a lot of time looking at mental health, like ADD, ADHD. And, we give… this year I’ll be speaking at three autism conferences. And we can dig into that a little bit as to why we think we’re seeing such a dramatic increase. And aside from autism, that used to be 1 out of 1,000, now it’s 1 out of 33, or 23. You know, we’re also seeing dramatic increases in ADD, ADHD. People are stressed out. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. And today, I think we’ll have the time to actually go through and show how environmental factors combine with genetics to cause that to happen. So we’ll… we should have a fun visit here today. Dr. Deb Muth 00:07:37 This should be a fun visit. We can cover lots of topics. I am so excited. So, you founded Nutri Genetic Research Institute in 2015. What did you hope to accomplish, and what kind of surprised you in your findings so far about that? Bob Miller 00:07:51 Well, you know, let’s back up at what, you know, genetics is used for. Everybody’s familiar with 23andMe and Ancestry that, you know, tells you where your ancestors came from. Then you have your professional geneticists. I mean, these are people with a degree in genetics. And they’ll look for, you know, very odd sort of things that are prone to relate to a disease. So there are disease-related genetics. Well, in functional, we don’t look at either of those. We look at For example, how you’re breaking down your fats and utilizing them. How you’re recycling your glutathione. How you might be handling your iron. And none of those are disease-causing on their own.And none of those are disease-causing on their own. But when they pile up on you, and then combine that with environmental factors, that’s when things start to go south on us. So, that’s what we’re doing, we’re looking at patterns. And our first foray into this was, we did studies on Lyme disease. And our first foray into this was, we did studies on Lyme disease. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. So, we looked at, like, I think 50 people with Lyme disease. We looked at their genome. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. And we found patterns that were more evident in those with Lyme. Now, this doesn’t… these genetics don’t mean you get Lyme, it just means if you get Lyme, you react worse to it. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. So, as you know, some people get Lyme, they go on a round of antibiotics, and they’re done. Others have a little more struggle, and then others are struggling terribly for years. So there’s an old adage of genetics loads the gun, environment pulls the trigger. Dr. Deb Muth 00:09:14 Yeah, that is so true, and I think when we’re talking about Lyme and mold and things like that, we forget sometimes that our genetics can predispose us to be more sensitive to those things, and if we have genetic pathways where we don’t clear things properly, it’s harder for us to get them out of the body. And then you add on that whole rain barrel effect that we’ve always used as a functional medicine term, right? If the barrel’s half full, you’re okay. If it’s full, and now it’s spilling over, it’s a bigger problem. Have you guys found, too, that some of these environmental things actually are changing the genetics of people, or how they’re processing their own genetics? Bob Miller 00:09:53 Well, let’s go back to, Genetics 101. But we’ll go back a little bit further. So, what an interesting mechanism, what a miracle the body is. Bob Miller 00:10:03 Fats, carbohydrates, proteins, drink water, breathe air, expose the sunlight, and somehow everything gets made. I mean, when you just step back and think about that, it’s like, It’s pretty darn amazing. Dr. Deb Muth 00:10:15 I always tell women, you know, the fact that we get pregnant and we have healthy pregnancies and births is a miracle, because if we had to try to control that, that wouldn’t work so well. Bob Miller 00:10:25 Right. Well, that’s another miracle. These microscopic sperm and egg, human being, 9 months later, it’s like. But even inside of us. We are making our hair, our skin, our nails, our blood vessels, our ATP, our energy, it’s all being created. Well, that gets created by enzymes. So, enzymes take one substance, combine it with something else, and make something new. Then another enzyme comes along and does the same thing. Your DNA is the instructions on how to make the enzymes. So, when we are conceived. If it’s a, if it’s a female, of course, it’s the XX, the two chromosomes. You know, we’ve… everybody’s seen those… the genetics that… Listed pair. So, if it’s a female, the father donated the X enzyme. And the mother has no choice but to give the eggs, so that’s female. If the father donates the Y, you have a male that’s in chromosome number 1. Then 2 through 23 is the rest of the instructions on how to make enzymes. So, what can happen? We can get what are called SNPs, single nucleotide polymorphisms. And SNPs just mean that the instructions to make the enzyme’s not quite as good. So, if one parent gives a SNP on the making of an enzyme, The enzyme’s fine. It works. But, general rule of thumb, It may only work at 70-80% of efficiency. Now, a good analogy is think of an 8-cylinder and a 6-cylinder car. If parents give you good information, that’s like having an 8-cylinder car. If one parent gives you that snip, it’s like having a 6-cylinder car. Now, is a 6-cylinder car a fine car? Sure. It’ll get you from point A to point B, but it’s just going to have the power of an 8-cylinder. Then if both parents give you a SNP on the same enzyme, it may be 30-40%, and that’s like having a 4-cylinder car. Sits in the driveway, looks the same, puts gas in it, everything. But if you’ve got a 4-cylinder car. Probably not a good idea to go cross-country pulling a trailer behind you up and down mountains. Dr. Deb Muth 00:12:29 This is true. Bob Miller 00:12:32 So… We can get an 8-cylinder, 6-cylinder, or 4-cylinder enzyme. Now, if it’s not under a lot of stress, if that 4-cylinder car is just taking you to the bank and the grocery store. It’s just as good as an 8-cylinder car. But if you gotta pull that trailer, and there’s a lot of stress on it, being mountains, it’s gonna struggle. Now, there’s one other little caveat to this, and that is some genetic mutations are gain-of-function. They actually work faster. Now, we have enzymes that do all kinds of things. We have enzymes that make and recycle our antioxidants, but we also have enzymes that make inflammation. No, that’s a good thing, because if we get a virus or bacteria, if you didn’t make inflammation to kill it, well, we’d all die of infection. So, you know, we tend to think of free radicals as bad, antioxidants as good. They both play an important role. But interestingly, some of the major enzymes that make inflammation, they can be overactive. They can be turbocharged. And when they’re stimulated by environmental toxins, they overreact. Bob Miller 00:13:40 And therein lies the problem. When they overreact, we have a problem. Bob Miller 00:13:46 So, if we have genes that overreact when stimulated. And then the enzymes that take care of inflammation are underactive. Then you’re gonna be more inflamed. You know, the majority of people that, you know, come for functional medicine Or naturopathic help, or… Inflammation that they can’t seem to get under control. Dr. Deb Muth 00:14:06 Right. Bob Miller 00:14:07 And we will be, you know, during this hour, we’re going to look at some of the pathways that make that happen. So, what we can do then, we can’t change our genetics. When you’re conceived, that’s the hand you’re dealt. When your life would be over, if someone would take some tissue and measure, it’d be exactly the same as conception. Does it change. Bob Miller 00:14:28 The enzyme’s ability to do its job may be compromised. Because remember I said there’s a, the enzyme takes a cofactor. So an enzyme takes substance A, cofactor, make substance B. Well, if that cofactor’s not there, the enzyme’s not going to work either. So, you could have an 8-cylinder car, and if there’s no gas in it, it’s not going anywhere. So… It’s the strength of the enzyme, it’s the cofactor to do the A to B conversion. And that’s what we’re going to get into. So, many people say, well, where did these SNPs come from? Nobody knows for sure. Sometimes they’re what’s just called de novo, when the sperm and egg go together, the instructions get mixed up a little bit. We do believe a lot of it came from a long time ago, when we were almost wiped out by sexually transmitted diseases. And those STDs were altering the genes when the conception, in other words, when the sperm went into the egg, the STDs were interfering. And causing the problem, so… I often joke, if you want to blame somebody. Blame your great-great-great-great-great-great-great-grandparents for, being a bit promiscuous, so… Dr. Deb Muth 00:15:31 Yeah, for being… having a little too much fun, right? Bob Miller 00:15:35 So, we don’t know for sure, but, you know, there are some that, But most of the SNPs that we get inherit from our parents. So, if you look at a child. And you look at the SNPs. 99.9% of the time, it came from one of the parents. Dr. Deb Muth 00:15:50 In identical twins, do they have the exact same identical makeup? Bob Miller 00:15:54 Yep, Dr. Deb Muth 00:15:56 But not in fraternal twins, correct? Bob Miller 00:15:59 No, no, those could be different, Jeff. Dr. Deb Muth 00:16:00 It could be different because they have different sacs, they’re not sharing that same genetic makeup. Bob Miller 00:16:04 Yeah, so keep in mind, both your mother and your father have, you know, the two And so you get one from one parent, one from another. Dr. Deb Muth 00:16:13 So… Bob Miller 00:16:14 Interesting situation. I had, 3, 3 boys. And, we were looking at an enzyme related to breaking down oxalates. Now, the mother and father each had one SNP, and that’s called heterozygous. Three boys, and they all come together, they’re Amish boys, they’re a lot of fun. And I looked at their genomes, and the one boy didn’t have any SNPs at all. And one had won. And the other one had two. Dr. Deb Muth 00:16:41 Interesting. Bob Miller 00:16:42 So, we don’t quite know how these things get handed off, but with the parents each having one, you could have a child with none, one, or two. So, the one, his ability to break down oxalates, which is fine. The other one was slightly impaired, and the other one was dramatically impaired. So, you can have 3 children, and it all depends what the parents have. Now, if a parent has a homozygous, or 2 copies. And the other parent has nothing. Every child will have one. Okay. If both parents are homozygous, that they both have two, Every child will have two. Dr. Deb Muth 00:17:19 too. Bob Miller 00:17:20 Yes, so that’s the way it works, but, you know, but it’s somewhat rare that both parents are homozygous on an enzyme, but it can happen. Dr. Deb Muth 00:17:27 Do we think that infections today, like Lyme disease or mold exposure, things like that, if the parent, the woman, primarily, I’m thinking, is pregnant, and she actively has these infections. Can those infections affect the genetics, kind of like a past sexual transmission did where we thought back in the day? Bob Miller 00:17:47 Yeah, I… I mean, I’m not that much of a geneticist to answer that for sure, but my thought would be no, that at conception, the pattern’s made. Dr. Deb Muth 00:17:55 Okay. And then that’s… that’s the hand you’re dealt. Bob Miller 00:17:58 Yeah. So, I tell people we have good news and bad news. The good news is we can compensate for the weakness. The bad news is we can compensate for the weakness. Dr. Deb Muth 00:18:09 That is so very true. Bob Miller 00:18:11 Yeah, we can’t, because I often get asked, so we’ll do some things now, and we’ll check my genes again, and they’ll be better. It’s like, nope. Dr. Deb Muth 00:18:18 Oh, – – Bob Miller 00:18:19 You gotta play the hands you’re dealt, so… Dr. Deb Muth 00:18:21 That’s right. Bob Miller 00:18:22 You can test your genetics… if you’re looking at the same enzyme, you can test it every year. It’s not gonna change. It’s like the blueprint. Dr. Deb Muth 00:18:30 It’s good and bad, right? It’s the one test you only have to do once in your lifetime. Bob Miller 00:18:34 No, unless, you know, like, our. Dr. Deb Muth 00:18:36 All the time. Bob Miller 00:18:37 Yeah, now our test looks at, called the Functional Genomic Analysis Test of your genomic Resource. We look at 220,000 steps. Dr. Deb Muth 00:18:46 Wow, that’s a lot. Bob Miller 00:18:47 That’s not all of them. Dr. Deb Muth 00:18:49 Right. Bob Miller 00:18:50 So, maybe in the next year, we’re gonna come out with our third version of the chip. And then, if someone wants to get those new things that weren’t on it, they’d have to repeat. But whatever we measured is gonna stay the same. Dr. Deb Muth 00:19:03 That’s a lot of SNPs to look at. Bob Miller 00:19:05 Keeps us busy. Dr. Deb Muth 00:19:06 But there’s still, but there’s still SNPs that we. Bob Miller 00:19:09 That we’d like to have that we don’t have, so… Bob Miller 00:19:11 We started out with version 1 on our genetic test, then we worked with version 2, and we’re already compiling a list of what version 3 would look like. So if somebody has our version 2, And we’re saying, you know what, it’d be nice if we could see these, well, then you’d repeat, but it won’t change what you already know, so… Dr. Deb Muth 00:19:29 Got it, got it. So, when you started out, and you started looking at the research of Lyme disease and chronic infections, which detox pathways are most important for people who struggle with those conditions? Bob Miller 00:19:43 Okay. You know what might make sense as we do a screen share, and I’ll actually show you the pathway. Does that make sense? Bob Miller 00:19:48 Alright, so… let’s see if I… let me just press the share… Dr. Deb Muth 00:19:52 Yep, you should just be able to press share. Bob Miller 00:19:54 And… number 2. Okay. Are we seeing the screen there? Bob Miller 00:20:01 Okay. Dr. Deb Muth 00:20:02 So, this is a map that we made. Bob Miller 00:20:05 And by the way, this is not… All-inclusive of all the things we look at, but we believe this is a core issue. So, where we’re going to start here, there’s something called the microglia. And the microglia are glial cells. They’re in the brain and the central nervous system. And they’re very interesting little creatures, because most of the time, and this is just a drawing of what they sort of look like. Most of the time, they’re in what’s called the M2 anti-inflammatory mood. What that means, these little guys pick up dirt, debris, Recycle them. Turns on an enzyme called interleukin-10 that’s anti-inflammatory. And just kind of does general housekeeping. And just kind of does general housekeeping. However, when a trigger comes along. However, when a trigger comes along. They… it’s the same glial cell, but it moves over to a very pro-inflammatory enzyme. A pro-inflammatory glial cell. And it triggers these 3 enzymes, Actually, these four. That are pro-inflammatory. Tumor necrosis vector alpha, Interleukin-6. NF Kappa B, Inos. Now, these create inflammation. So you might think, well, why is that good? Well, if you have some foreign invader, virus, bacteria coming in, parasite. If you didn’t have these guys coming to the rescue, you would just die of infection. So, these guys are your friend unless they’re your worst enemy. Because TNFA, and we’ll show you when we actually do a demo account, TNFA can be overactive. So, in other words, it over-responds. Interleukin-6 can be overactive. And if Kappa-B can be overactive. The INOS, and I’ll explain each of these as we go through a demo, can be overactive. Now, what that means is, you’re very good at killing virus and bacteria. But this is where autoimmune disease comes in, and just inflammatory conditions. Now, this is just speculation, but we think what happened is, as you know. Thousands of years ago, we didn’t have refrigeration, we didn’t have sewer, we didn’t have pure water, and we didn’t have antibiotics. So, if you made it to 40, you were an old-timer, because everybody was dying of infection. So, what we believe happened is, by what’s called natural selection, Having these overactive. A thousand years ago was to your advantage. Dr. Deb Muth 00:22:31 Hmm. Bob Miller 00:22:32 But now… We have pure water, we have refrigeration, we have sewers, we have antibiotics. But now we have environmental factors that are stimulating them. Now it’s to our disadvantage. And we’ll talk about that a little bit as it relates to the hemochromatosis genes and maybe the G6PD. Dr. Deb Muth 00:22:48 Yep. Bob Miller 00:22:49 Now, why are we becoming so inflamed? Let’s look at the triggers. Now, one of my, favorite expressions is. I was born all the way back in 1954. Dr. Deb Muth 00:23:01 And it was a different world back then. Bob Miller 00:23:05 These are some of the triggers. And we’ll get into these, but right now, high fructose corn syrup, And the high-fat diet. High fructose corn syrup only came about in 1968. So now we’re being exposed to high fructose corn syrup. Then… we didn’t have these, these viruses like COVID. Dr. Deb Muth 00:23:26 Yeah. Bob Miller 00:23:27 Now, there’s now pretty strong evidence that COVID Was actually, you know, made as a gain of function. It’s debated, and I’m not taking an opinion on it, but there’s some people who believe Lyme disease was also a part of experimentation. Dr. Deb Muth 00:23:40 Go. Bob Miller 00:23:41 Then we have molds, and it appears as though mold is getting stronger. you know, 20 years ago, when I was seeing folks, mold wasn’t on the radar. I would say 7 out of the 10 folks we speak to today have mold problems. Yeah, 20 years ago, we talked more about mold allergy being an issue versus mold toxicity being an issue. Right. So… I know some folks are, you know, speculating what’s happening, but one of the theories out there is that EMF is strengthening mold. I don’t know if you ever heard that theory, and I don’t… Dr. Deb Muth 00:24:13 I have. Bob Miller 00:24:14 I’m not claiming it’s true, but it’s an interesting theory. Then even, you know, your black mold from water-damaged buildings. Then our air pollution is getting worse. We’re getting more toxic metals. Dr. Deb Muth 00:24:26 You know, if we have a… Bob Miller 00:24:27 You know, we’re gonna look back someday and say, what were we thinking, smearing aluminum into our armpits? The, what were we doing putting mercury in our teeth? Then, you know, glyphosate. When I was a kid, there was no glyphosate. So, all of these herbicides and pesticides. Polychlorinated biphenols, And then EMF. So, we love our cell phones, you know, and I think unless you, or in the middle of the desert, or down in a cave, you’re being exposed to EMF somewhere. So, you know, we have our cell phones with us, we have, We have Wi-Fi, the towers are everywhere. And we don’t know long-term, but we may find that this can… this creates some inflammation. And I don’t know if you get any folks, but do you have any folks that have… are they EMF sensitive? Dr. Deb Muth 00:25:16 Oh yeah, we have a whole bunch of them. Bob Miller 00:25:18 Yeah, and then if you have any TBIs, So, plenty of things here. that will stimulate into the microglia, M1. Now, you could say, well. We’re all pretty much exposed to the same thing. Why do some people get hit harder than others? So here’s where we’re gonna start. There’s an enzyme called Nrf2 and RF2. And Nrf2 is the enzyme that senses when there’s inflammation. And turns on hundreds of anti-inflammatory enzymes. We’ll show when we do the demo, you can have genetic weakness on NERF2. And NERF2 inhibits and slows down microglia M1. supports M2. Now, if it’s not complicated enough, there’s an enzyme called KEEP1. And KEEP1 inhibits NRF2. And you can actually have gain of function on keep 1, that makes Keap 1 stronger. So… A lot of the people who land on my doorstep So… A lot of the people who land on my doorstep Both parents gave a mutation on KEEP1, making it overactive. Both parents gave a mutation on KEEP1, making it overactive. Dr. Deb Muth 00:26:31 Hmm. Dr. Deb Muth 00:26:31 Hmm. Bob Miller 00:26:32 Suppressing Nrf2, nerve 2 might be weak. So, nobody’s putting the brakes on, M1. And by the same token, Nerve 2 supports M2. Then there’s a process called mTOR and autophagy. mTOR stands for mammalian tard of rapamycin, the growth of new cells. And then autophagy, taking our dead cells and recycling them. We need a balance between the two of them. If we didn’t have mTOR, the sperm and the egg would never become the baby, the baby would never become the adult, we wouldn’t make new cells. But our cells are constantly, you know, the old cells dying off. Autophagy is where we take that debris from the cell and recycle it, just like a farmer Plows the crop under at the end of the year. The dead plant then becomes the fuel for the spring, your dead cell becomes the fuel for the spring, and that’s autophagy. So we’re gonna look back someday and say, what were we thinking? We give our animals growth hormones so they get fatter faster. Oh my. So, we consume those animals, and inventory runs faster. Now, for anybody who’s, You know, maybe above 40, 45 years old. Think back when you were 12, and what did girls look like? They were primarily flat-chested little girls. Now they look like 16-year-olds. Because environmentally, we’re jacking up mTOR. So, mTOR stimulates microglia M1, suppresses microglia M2. Probably 80% of the folks we visit with. This is the part of the problem. NRF2 is weak. mTOR is strong. Environmental factors come along. And this guy gets carried away. He doesn’t do that burst and move back. Stays here. We’re calling that How environmental factors create a locked-in, pro-inflammatory. and neurotoxic phenotype. In other words, once it starts, it just keeps… Feeding upon itself. Alright, so what happens now when microglia is overactive. it triggers these 3 enzymes, TNFA, N of kappa B, And interleukin-6. Each one of these can have genetics that make them run stronger. Then it stimulates an enzyme called NLRP3, Which makes what are called inflammasomes. Now, guess what inflammasomes can be? Your best friend or your worst enemy? Because they will, if you’ve got, again, a virus or bacteria, or possibly even some bad cells in the body. They will zap them. Well, that’s good. Unless it’s overactive. Unless it’s overactive. And then what it does, through interleukin-1 beta, makes excess glutamate. And then what it does, through interleukin-1 beta, makes excess glutamate. Anxiety, gut inflammation, OCD, ADD, autism. And, you know, glutamate, we’ll talk about that a little bit, but glutamate makes you intelligent, highly motivated go-getter. but can also be excitatory. And then, look what it does. Let’s see, do I have the drawing tool here? Yes, I do. Okay. So, it comes down through here, Makes the glutamate. Comes back up through here. through the ADORA 2A enzyme, Then we’ve got a feedback loop that feeds upon itself. Then, through interleukin-18, we make histamine. and mast cells. And then through histamine receptor site number 1, we come back and spin it. And now you’ve just got this spinning feedback loop. So, the glutamate will make you anxious, the histamine will give you allergies and make you anxious. And you’re allergic to everything, and you’re feeling horrible. Now, it doesn’t end there, Dr. Dad. It then goes on to make something called gast dermins that creates pyroptosis, where it actually starts punching a hole in the cell membrane. And you’re only going to be as healthy as your cells are. Just a little background. You know, we’re made up of trillions of cells, and each one of them has what’s called a lipid bilayer, made from lipids, which comes from fats. And you’re only going to be as healthy as those membranes are. So that’s why we coined an interesting phrase. Cellular CPR. Construct the cell. Protect the cell. And restore the cell membrane. And we believe that’s going to be revolutionary in the functional medicine world. So… It’s not hard to figure out that if you start punching holes in the cell membrane, that’s not a good thing, okay? Bob Miller 00:31:22 Now… There’s an interesting molecule called NAD. Thicotide adenoside dinucleotide. And anybody who’s in the, you know, listening to the health podcasts and things, they’re… They’re, they’re learning about NAD. And I’m going to show you a chart later, all the good things that NAD does, but For the most part, it helps what’s called sirtuins. And sirtuins are quite interesting. If anybody’s looking at longevity. The sirtuins is where they’re looking at.Because sirtuins turn on good things. Turn off bad things. And I’ll show some charts on that later. So for right here, this sirtuin uses NAD, to slow down NF-kappa-B. CERT 2 uses NAD to slow down an ORP3. So, if we’ve got genetic weakness on these, or we don’t have enough NAD, We don’t hold this pathway back. Make sense? Dr. Deb Muth 00:32:24 Yeah, makes perfect sense. Bob Miller 00:32:25 Now, I’ll show this a little bit later. So, people are like, oh, well, I’m gonna start taking some NAD. Dr. Deb Muth 00:32:31 Right. Bob Miller 00:32:32 And there’s functional doctors who give NAD intravenous. It was just this morning, I was talking to a woman who said, Oh my gosh. I went and got intravenous NAD, and it took me a month to recover from that. Dr. Deb Muth 00:32:45 Hmm. Bob Miller 00:32:46 what happens is, and I’ll show this in a little more detail, there’s an enzyme called CD38, that’s stimulated by NF-kappa-B. And it takes NAD, To make intracellular calcium. that stimulates NLRP3 and actually makes things worse. So, if we have this guy upregulated, and I’ll show a chart what does that. taking NAD will make you worse. Again, when I go into the software, I’ll show you that whole pathway, so… I would encourage people, you know, just don’t go out and start taking massive amounts of NAD, you know, stick your toe in the water, see how you do. Because everything you’ve heard about, how good it is, is true, unless this guy says, oh, thank you very much, let me make more inflammation. Now, this might be part of our innate immune system, that if we have some pathogen that’s gonna kill us. By golly, we want that to happen. But if this is happening by environmental factors, Then it’s detrimental. So the immune system that protected us a thousand years ago now might be turning on us because of the environmental factors that we showed earlier. All right. Then there’s an enzyme called PARP that’s NAD-dependent, and that actually repairs strain breaks in your DNA. Now, the next thing that happens… is there’s an enzyme called NADPH oxidase that gets stimulated. and something called INOS. Now, I’m sure most people know about nitric oxide. It’s a gas that dilates your blood vessels. That’s why sometimes they’ll even give people drugs, nitroglycerin, to boost their nitric oxide. That’s why people are doing beetroots and other things to boost their nitric oxide. But there’s an OS3 enzyme that makes the nitric oxide that’s good for blood flow. But there’s an INOS That makes nitric oxide to kill pathogens. probably might be the third or fourth time I’ve said this. That’s a good thing, unless it isn’t. So, if it’s killing some pathogen, great. It was just misfiring. it combines… With superoxide that’s made by this enzyme, and makes something called peroxynitrite, which is one nasty free radical that chews you up and spits you out. So, the NOx enzyme, NADPH oxidase, uses NADPH, To make this free radical called superoxide. If we have time, we’ll get into it. NADPH is what your body needs to recycle your antioxidants.So, I coined the phrase, the NADPH steel. Where the NOX enzyme takes this very important NADPH, And rather than being useful, makes superoxide. Now, again, is that fine if you’ve got some bacteria to kill? Of course. But if it’s just chronically running, it’s just making all this chronic inflammation. Then it makes something called hydrogen peroxide. And we need to clear hydrogen peroxide by 3 enzymes, catalase, thyroid reduction. And glutathione peroxidase. If we have genetic issues on here, or we don’t have the cofactors. There’s something called the Fenton reaction, discovered in 1895 by Dr. Fenton. Where hydrogen peroxide combines with iron to make what are called hydroxyl radicals. And guess what they do? They create lipid peroxides, That damages your cell membranes. Now, again, the body’s pretty darn amazing. We have glutathione, And here’s where your body’s taking glutathione and recycling it. But look who’s needed to recycle it. NADPH. So, if this guy up here is chewing it up, We don’t recycle our glutathione. And then an enzyme called glufon peroxidase 4, Takes this damaged lipid and repairs it. So, here we’ve got this protecting, we want to protect it by not having this happen. But then we also need this guy to do the restoration. So, there’s a lot that can go wrong in here, Dr. Deb. Dr. Deb Muth 00:37:07 There’s a lot that could go wrong. And I can imagine some of my listeners are thinking that lipid peroxidase, is that the same thing as what they’re thinking of when we talk about lipids and cholesterol? Is that the same process that’s happening there? Bob Miller 00:37:22 Well, no, no, the lipids can be used to make cholesterol, but here we’re talking about where they’re going to build the cell membrane. And they’re being… and they’re being, destroyed. If anybody would like to see a visual representation of this, just go on YouTube. And type in, ferrooptosis Animation. cool little video, it’s about 3 minutes long, and it shows the lipids coming over, being oxidized, and now GPX4 fixes them, so… YouTube, Pharaoptosis Animation, cute little video. It’s just that really… Shows vividly what we’re… what we’re talking about here. Now, this is… Dr. Deb Muth 00:37:59 And so this is very common, too. Like, a lot of people do hydrogen peroxide IVs. Dr. Deb Muth 00:38:04 And so, if somebody doesn’t know their genetics, they could have a problem with doing those, just like they could doing the NADHIVs, correct? Bob Miller 00:38:13 Sure, yeah, yeah, yeah. So, I’ve talked to so many, you know, of course, the hydrogen peroxide kills pathogens. I mean, that’s what it does. So… but I’ve spoken to so many people that said. I had one client that said they’ve never been the same after having one hydrogen peroxide infusion. Dr. Deb Muth 00:38:30 Interesting. Bob Miller 00:38:31 Yeah. So… it can be… I see why people use it, because it. Bob Miller 00:38:36 pathogens, But on the other hand. And now’s a good time to speak about… I don’t have it on here, but there’s a, there’s an enzyme called the HFE gene. And that is what causes you to absorb iron. And there’s mutations in it that cause something called hemochromatosis. Were you overabsorb iron? Now, true hemochromatosis is when both parents give you a mutation. But there’s now growing evidence even a heterozygous can cause a little bit more iron absorption, not to the human chromatosis point, but overabsorption. So, if you overabsorb iron, And you have too much hydrogen peroxide that’s not cleared, All kinds of inflammation. Now, what’s happened is sometimes this inflammation Will damage the red blood cells. And some well-meaning doctor says, oh, you need some iron. And they take iron and it makes it worse. So, can’t tell you how many people I’ve said, you’ve got the overabsorption of iron, and they say, well, that can’t be right, because I’m low in iron. Well, that could be because it’s being chewed up here. Dr. Deb Muth 00:39:40 Sure. GPX1 and TXN turn it into, to water. The, catalase turns it into water and oxygen. Dr. Deb Muth 00:39:58 Now, I see a lot of my clients who have mutations or SNPs on that GPX gene, on that glutathione gene. And they really struggle to clear a lot of their toxins. Bob Miller 00:40:12 Sure. Dr. Deb Muth 00:40:14 Yeah, absolutely. Well, GPX4. Bob Miller 00:40:18 is what, repairs, but you can see GPX1 Is what uses glutathione. To turn hydrogen peroxide. So, but it all depends upon having enough glutathione. Dr. Deb Muth 00:40:30 Yeah. Bob Miller 00:40:31 Well, guess who controls making a glutathione? Dr. Deb Muth 00:40:34 Nerf 2. Bob Miller 00:40:37 So, if you have a keep one weakness, or strength to two… I’m sorry, keep one is too strong. Nrf2 is too weak. You don’t make glutathione. So, when a lot of people do that, it’s like, well, I’m gonna take glutathione. Dr. Deb Muth 00:40:51 Right. Bob Miller 00:40:52 And some do great, and some do poorly. You know, because… and I’ll show this on one of the other charts. You can see here that the, The glutathione has to be recycled. And if we don’t recycle it, it actually turns into superoxide free radical. So… NADPH are the cofactors, For taking the oxidi… here’s oxidized glutathione, here’s reduced. So, this is a good glutathione. After it does its job, you can see it becomes oxidized.We need to recycle it. Well, if we have weakness on the enzyme that does that, or a weakness in Nrf2, or not enough NADPH. The oxidized glutathione never gets recycled. So, I’ve talked to a lot of people who said, oh, glutathione made me so sick, and say, well. Dr. Deb Muth 00:41:43 Yeah. Bob Miller 00:41:44 You need it, but you need to recycle it. Dr. Deb Muth 00:41:46 Can you speak for just a brief moment, too, about MTHFR? That is a very popular gene, it’s all over social media as the major gene, but can you speak to a little bit about that, and how that fits into this whole process of things? Because it is just such a small piece. Dr. Deb Muth 00:42:04 understanding genetics. Bob Miller 00:42:06 Yeah, to be honest, it drives me nuts. Dr. Deb Muth 00:42:08 Me too. Bob Miller 00:42:11 Alright, so… You know, there are people on social media I won’t say what I think, I’ll be kind. But… But the, And, you know, they might mean well. But they talk about, if you have MTHFR and COMT and PEMT, that’s… oh my goodness, that’s horrible, and we’ll fix that for you, and you’ll be fine. Bob Miller 00:42:36 it just irritates me to no end. And it really could get anybody who’s doing this legitimately in trouble. I mean, I’m afraid someday, you know, there might be some cracking down on this kind of nonsense. Now, to answer your question about MTHFR. Dr. Deb Muth 00:42:51 I mean, it really is, but I’ll tell you what, why don’t we hold that thought until I go to another map and I can actually… Okay. Bob Miller 00:42:56 But the real… the cliff notes is the MTHFR puts a methyl group on your folate, which is needed, but it has gotten way, way, way too much attention. And people learn they have MTHFR, and they start taking a multivitamin with methylfolate, then they take a B vitamin with methylfolate. Dr. Deb Muth 00:43:13 And they’re pushing it too hard. Bob Miller 00:43:15 Yeah. So I can’t tell you how many people I’ve helped by saying, stop it. Dr. Deb Muth 00:43:20 Yeah, take less of it. Bob Miller 00:43:21 Take less of it, yeah. So, yeah. Yeah, there’s a… If somebody, say, ranked the enzymes at their level of importance, MTHFR might be 40 or 50 on a scale of 100, you know. Keep one Nerf two. big deals. Dr. Deb Muth 00:43:40 deals. Bob Miller 00:43:41 NQO1 that I didn’t even talk about yet, NQO1, takes your, NA… your NAD goes into NADH, To make electrons for the electron transport chain. you need NQ01 to bring that back. If that’s not working, and I’ll show you on the NAD map how disastrous that can be. Now, the next piece is here, and I think You know, if you talk to any school teachers and say, if you’ve taught for more than 10 years, how are the kids today? Every one of them says, more ADD, ADHD, more autism. Just look at human beings, we’ve never been so agitated. You know, everybody, and it might be a social media thing, but people take a position on something, and if anybody doesn’t share that position, they view them as the enemy. Dr. Deb Muth 00:44:29 And it’s kind of scary what’s happening to us. Bob Miller 00:44:33 So, we can’t agree to disagree anymore. We see anybody who has a differing opinion as the enemy. And, you know, there was… there’s people that didn’t have Christmas dinners together, because they had political differences, like… Dr. Deb Muth 00:44:44 Excuse me. Bob Miller 00:44:45 can’t you put your political differences aside to have Christmas together, you know? Dr. Deb Muth 00:44:49 Right? Bob Miller 00:44:50 become that, you know, no matter what your position is, and I’m not saying anyone’s right or wrong, I’m just saying. You know, in the old days, they used to say that the Republicans and Democrats in Congress would argue policy and then go have dinner together. And now everybody’s all up in arms, angry. Dr. Deb Muth 00:45:05 Yeah. Bob Miller 00:45:06 So… There’s likely multiple reasons for that. But let me show you one of them. That, you know, to what degree this is… very important, we don’t know, but I think We’re beginning to believe this is very important. So, there’s something… there’s a neurotransmitter called GABA. And God buys the don’t worry, relax, be happy. Chill. Okay. Dr. Deb Muth 00:45:31 Nobody has enough of that anymore. Bob Miller 00:45:33 Well, yeah, you’ll be surprised what I’m gonna show you. So, let me see if I can find a, Let me see if I can find the right slide here. Let me look for it here. So, there’s something called a GABA receptor site. And here you can see… This is a neuron, and this is where you, The neuron normally is excitatory. However, there’s normally low chloride in the neuron. Dr. Deb Muth 00:46:09 Hmm. Bob Miller 00:46:10 So, GABA itself is neither relaxing. For excitatory, all GABA does, it opens up what’s called a chloride channel. And then chloride, which has a negative charge, will flow into the neuron. Follow me there? Dr. Deb Muth 00:46:26 Yep. Bob Miller 00:46:27 And as it does, it changes this from a positive charge to a negative charge, And it’s relaxing. and inhibitory. Dr. Deb Muth 00:46:34 Hmm. Bob Miller 00:46:36 Now, on the other hand, there’s enzymes called NKCC1, That will push chloride in. and KCC2 that will bring chlor… oops and bring chloride out. And then there’s a sodium channel. And, sodium has a positive charge. And glutamate will push that in. So, as long as this is happening. And GABA says, receptor sites, open, chloride goes in, Chill. However, If NKCC1 Pushes extra chloride in. KCC2 doesn’t pull it out. and GABA hits the receptor site, the GABA comes flowing out, Sodium comes in, And now it’s excitatory. So Gabba didn’t change. GABA just opened the receptor site, that’s all it does. Dr. Deb Muth 00:47:33 Yeah. Bob Miller 00:47:34 But it’s the chloride balance that’s going to determine whether this is relaxing or not. Now, these are the things that go along with when they lose that KCC2 or gain NKCC1. Pain and sensitivity, burning electrical, neuropathic pain. Normal touch hurts. Sound and light sensitivity. Tinnitus can flare. Headaches and migraines. Seizure tendency. Body jolts. Spasticity, cramps, stiffness, startle reflex. Trouble falling asleep, non-restorative sleep. Anxiety, stress, reactivity, that’s what we have now. Hyperarousal, panic-like surges, irritability, racing thoughts. Brain fog, slowed processing, working memory slip-ups. Mental fatigue. Episodes of racing hearts, sweaty palms, guts on edge. Those are all the things that happen when this GABA switch occurs. Now, here’s what happens, and this is what I’m going to be presenting at an autism conference. When you have a newborn, they need that NKCC dominant to develop. By early childhood, it should… or, sorry, early adulthood. we should move over to the KCC dominant, that’s the taking the chloride out. Nice-looking 25-year-old boys, functioning very well. However, when we get microglia M1 upregulated. Because of environmental toxins, processed foods, Tylenol, aluminum. they stay in NKCC1 dominant, and there’s ADD, ADHD, Autism, the whole spectrum. because… They’ve not moved over to the… They’ve not moved over to the KCC2. And again, this is caused by… Environmental factors. Stimulating the microglia. And then, interleukin-1, interleukin-18 weakens KCC2, interleukin-1 beta, Strengthens NKCC1. high chloride. We open up the chloride channel, In Rebell Excitatory. So, I think when, When the pediatricians get ahold of this, they’re going to be very excited to know that This could be why we’re seeing such a rise, and not just autism, but ADD, ADHD, anxiety, the whole shit mess. Dr. Deb Muth 00:49:58 thing. Bob Miller 00:49:59 Yeah, so… and you can see NF-kappa-B stimulates that. These stimulate it, and I think that’s why everyone’s getting so anxious. Now, there’s a little bit more to it, and we’ll get into this when we look at some of the maps, but… The, the glutamate, Which is excitatory. will stimulate the NMDA receptor, make more glutamate, And glutamate will inhibit KCC2. And then we also need an astrocyte To, take both ammonia And glutamate, and… Turn them back into glutamine. And I’m going to talk to you a little bit about arachidenic acid, and if we have too much arachidenic acid. or TNFA is upregulated, that doesn’t happen. Ammonia goes up, and there may be multiple reasons for this, but this is a reason why some of the autistic kids do flapping. Dr. Deb Muth 00:50:49 Hmm. Bob Miller 00:50:50 Because they’re not clearing their ammonia. And you can tell if somebody has high ammonia by… they get that old person smell, you know. Dr. Deb Muth 00:51:00 Yup. Bob Miller 00:51:01 your vehicle cycle’s not taking out the, the ammonia. Now, last pathway here. There’s growing interest in mast cell activation. So, back here, we talked about peroxynitride. And that will stimulate mast cells, and those are white blood cells that are your best friend, unless they’re your worst enemy. Then it’ll make histamine. And there’s enzymes called histidine decarboxylase that’ll make more. Dr. Deb Muth 00:51:28 I’m sure everybody’s heard of DAO, the enzyme that degrades histamine. Yep. Bob Miller 00:51:31 We can have genetic weakness, we don’t make that. There’s an enzyme called histamine and methyltransferase, That, That breaks down the histamine. Then if we don’t do that, it’ll get stuck in the histamine receptor site. And then it’ll make something called, renin. Which will cause angiotensinogen to turn into angiotensin. One, that turns into angiotensin II,And that’s where people make aldosterone, where they’ll get the, The swollen ankles and high blood pressure. But interestingly, there’s an enzyme called ACE2, that takes this guy and turns it into angiotensin 1-7, Which is anti-inflammatory and also inhibits… TNFA. Now, you can have weakness on ACE2, But… and anybody’s saying, that sounds familiar? Dr. Deb Muth 00:52:25 That’s where COVID comes in, using ACE2. Bob Miller 00:52:28 And now we just found there’s literature that if you get COVID long enough, it can actually make ACE2 not be able to work as well. So look what it does. It comes down here, stimulates the NADPH oxidase, More superoxide. More peroxynitrite. And we’re on a cycle here. We’ve actually named this the Home Cycle Hypothesis, the proposed feed-forward loop. That just keeps feeding on itself. All being caused by… Primarily, The environmental factors. But hitting those who have genetic weakness the hardest. That’s why. Dr. Deb Muth 00:53:08 To the people. Bob Miller 00:53:09 Don’t live in a moldy house. One person is sick as can be, and the other person says, well, you must be imagining things, because I don’t feel anything. Dr. Deb Muth Yeah. Same thing with long haul, right? Two people can both get sick, one gets sick and never seems to recover, and somebody else gets sick, and they have absolutely no problems with it at all. Bob Miller 00:53:30 Sure. Well, think about it, if you get COVID, and ACE2 is weak, and some of this other stuff is going on. This thing just starts feeding upon itself. Dr. Deb Muth 00:53:38 Keep creating more inflammation, more complications, nothing’s calming down. Bob Miller 00:53:43 Yeah. Now, you, you ask about, MTHFR. So, this is the, this is the, the software called Functional Genomic Analysis. There’s a demo report we have. So, let’s talk a little bit about, MTHFR. So, we actually have a map called a methylation map. Now, what happens is, when you do your saliva test, you, you know, you spit, you put some saliva. in a collection kit, goes to a lab, takes out the DNA data, sends it to the computer, and now you can actually see it visually. Okay. So, it’s gonna take a second for this, data to load up, it’s, and each of these Circles, each of these ovals, is an enzyme. And the data gets loaded up to see where it is. So, until it gets loaded up here, I didn’t preload this. There it goes. So… The primary thing about methylation is There’s a nasty substance called homocysteine that, if it’s too high, can really be detrimental. The body takes methylfolate, and combines with methyl B12, To bring this back up to methionine. And then through the MAT genes, we make SAMI, S-adml methionine. Which is involved in so many processes. Then after it does its thing, it turns back into homocysteine. And this thing needs to keep spinning around. That’s why, you know, it’s a good idea to keep homocysteine at, do you have a number that you’d like? 7, 8? What do you like for a number? Dr. Deb Muth 00:55:24 Yeah, I like mine below 7. Bob Miller 00:55:26 Yeah. So if the homocysteine goes too high. It, caused all kinds of problems. So, here’s where you ask about the MTHFR. So, here you can see on this individual. I click on MTHFR, and you can see it comes up here, here’s the C677. And you can see here where it says, variants. I’ll… I’ll draw in case somebody’s having a hard time seeing that. So, you can see there’s nothing in there. That means there’s no genetic mutations. If one parent would have given a mutation, there’d be a 1. If both parents did, there’d be a 2. Now, here’s why Yes, methylation is important, I’m not saying it isn’t important, but look at this MTHFRC677. In my software. Only 42.5% of the population does not have a mutation. 44.7% have won. 12.9 have 2. So, this isn’t some rare, oh my god, I’m gonna die… Kind of thing, yeah. Dr. Deb Muth 00:56:27 Right. Bob Miller 00:56:28 So, And then what happens is that, and again, I’m not dismissing methylation, I… we could do a whole show on methylation. Bob Miller 00:56:36 get it. But I think that what people are doing is they’re, they’re learning about MTHFR, they get it measured, they panic. They start taking massive amounts of methylfolate, which many times is to their detriment. Dr. Deb Muth 00:56:50 Well, it’s… and isn’t it true, too, with MTHFR, like, you have to also look at MTR, MTRR, and the more we stack up of those, the more complicated than MTHFR can be. It’s not… it’s not as simple as just saying MTHFR 677 versus 1298. It’s more complex than that, kind of like what you’ve already shown with some of the other things. There’s more to it than just that one little sliver. Bob Miller 00:57:17 Oh, sure, well, let’s take a look. So, remember I said there’s a cofactor? One of the cofactors is called FAD. Just a Bob Miller observation, that’s all. But when people have trouble with their riboflavin and they don’t have enough FAD, They’re doing much worse than people who have just a C677. So, right here, you could have perfect C677th. And if you don’t have the cofactor, it’s not gonna work, okay? Dr. Deb Muth 00:57:48 And as you said, there’s an MTR enzyme. Bob Miller 00:57:51 that takes methylfolate and methyl B12, to spin it around. So, here on this individual. here’s your… here’s your B vitamins, or I’m sorry, your B12s. There’s an enzyme called TCN1 that takes it from the stomach into the blood. Then there’s other enzymes that take it from the blood into the tissue. And if you’re having trouble here. Well, then you’re not going to have this working, so… Even if you don’t have MTHFR, And you have MTR, like this, no, I’m sorry, this person doesn’t. But they have the MTRR, and then they don’t have enough B12, this isn’t gonna work, aside from that. And then there’s a middle pathway. And then there’s enzymes called the MAT1. they take the methionine to the salmon. If that’s not working, we stick… we get stuck in methionine. So, it’s, it’s not just an MTHFR. And then, one of the things that people forget about. is through these CBS enzymes and CTH, We make cysteine, which is needed to make glutathione. The master antioxidant. So, it really is that… I call it the, The 3D chess game played underwater. Dr. Deb Muth 00:59:07 It really is. I mean, I see people who have CVS, COMT, glutathione, MGHFR genes. And some of them function just fine. Like, they have Like, I look at this person and I’m like, oh my gosh, I don’t know how they’re functioning because they’re double mutated on so many pathways, but yet they don’t have a lot of symptoms, they don’t have a lot of complications. Somehow their body has figured out a way to adapt to what it has so it can stay alive and it can function at a high functioning level. Bob Miller 00:59:36 Yeah, and they may be, you know, eating right? Yeah. Staying out of a moldy house. reducing stress. So, it’s diet, it’s stress, it’s genetics, environmental factors. So, yeah, we can’t just say somebody’s gonna be good or somebody’s gonna be bad. You know, some people get scared, oh, I got all these, it’s like, well… Bob Miller 00:59:56 Are you living in a moldy house? You know, and if you live in a moldy house and your glucuronidation pathway doesn’t do well, or if you’re, you know, a smoker, or you’re constantly eating junk food, I mean, all. Bob Miller 01:00:07 things come together. Although, you know, when we focus on genetics, we’re well aware that this is just a piece of it. You know, you could have identical twins, Genetically, and if one… Is exposed to mold and smokes and drinks and stressed out. They’re gonna be a whole lot sicker than their sibling. Bob Miller 01:00:28 Yep. Dr. Deb Muth 01:00:29 Yeah, it’s that concept of taking twins, and one gets raced with one family, and one gets raced with another family, and they don’t have the same… problems that… that each other have, you know? It’s a very unique situation, we don’t think about that enough. Bob Miller 01:00:44 Alright, so again, genetics loads the gun, environment pulls the trigger. So, if you’ve got a loaded gun, but you don’t have the triggers, you’re okay. Dr. Deb Muth 01:00:53 Yeah. Bob Miller 01:00:54 Yeah. So, remember I said I was going to talk about NAD? So, here’s NAD, and what it does, it turns into NADH. And what NADH does, it, Comes down this pathway, what’s called the electron transport chain. And that makes your ATP, that’s your energy. So, if this wasn’t working, we wouldn’t be alive, because we wouldn’t have energy. So it donates an electron, that’s why it’s called electron transport chain. So, we need NAD, To make this, to make the energy. But remember I said that NQ01, this would probably be, like, on my top 10 list of… Bob Miller 01:01:36 Much more important than MTHFR. This one takes NADH back to NAD. If we’re stuck over here, We’re low in this NAD+, But what happens is, NQO1 also provides CoQ10. And CoQ10 Is what’s needed for the electron transport chain to flow. So if we get too many electrons up here. And they don’t turn them into energy. They make a nasty free radical called superoxide. Okay. Now, NAD plus also makes NADPH, And that is needed. Remember I said we need to recycle our antioxidants. So, if we have a problem with FAD from riboflavin. Yeah, we don’t have enough NADPH, Glutathione’s not getting recycled, and you’re gonna be inflamed. And you take glutathione, you’ll feel worse. There’s another enzyme called thimoredoxin. Same thing, needs NADPH and FAD. And same way with your nitric oxide, there’s an enzyme called NOS3, That makes the nitric oxide that dilates your blood vessels. And if we don’t have enough NADPH or fat, You’re gonna make superoxide. Rather than nitric oxide. Now, remember

Nurse leader, You gave everything today. Now it's time to give it to God. If you're listening to this on your commute home — in your car, on the train, walking to your front door — this episode was made for this exact moment. You just survived a shift that had no business being that hard. You were in charge. The floor was chaos. There was a patient loss, coworker conflict, impossible decisions made in seconds, and nurses running on empty. And somehow, you were the one holding it all together. Your body is exhausted. Your nervous system is still activated. And if you go to sleep without releasing what you absorbed today, you'll carry it into tomorrow — because the body keeps the score. In this Breathe & Be Still episode, we do something simple and sacred: we release it all through guided breath prayer — and receive the peace God promises in return. In this episode, you'll: Move through a 3-minute guided breath prayer rooted in Philippians 4:6-7 Learn why releasing post-shift emotions isn't weakness — it's nervous system wisdom and biblical obedience Discover the difference between true shalom and just "being tired" Stop the rumination loop so your mind and body can actually restore overnight This episode is for you if: You're a nurse, charge nurse, or nurse leader coming off a hard shift You struggle to "turn off" your brain after work You've experienced patient loss, compassion fatigue, or secondary trauma You're a faith-based nurse looking for a spiritual practice that actually fits your life You want to protect your peace without toxic positivity or empty self-care advice Scripture Featured:

Are you frustrated with your decorating project? Deep breaths and let's talk about how to keep from being overwhelmed with your project. We've got several great tips on how to approach a project in a way that doesn't feel so stressful.Join Kelly on her app Home Design Coach. Click HERE to learn more!Looking for amazing decor on Amazon? Anita has updated her favorite picks HERE.We participate in the affiliate program with Amazon and other retailers. We may receive a small fee for qualified purchases at no extra cost to you.Anita's crush is a movie that used to be played every Christmas, although it's not a Christmas movie. It's Philadelphia Story with Katherine Hepburn, Cary Grant, and Jimmy Stewart. You can find it HERE. Kelly's crush is the yummy & good for you Oatmeal Cups recipe from the NYT. Try it HERE.Need help with your home? We'd love to help! We do personalized consults, and we'll offer advice specific to your room that typically includes room layout ideas, suggestions for what the room needs, and how to pull the room together. We'll also help you to decide what isn't working for you. We work with any budget, large or small. Find out more HEREAre you subscribed to the podcast? Don't need to search for us each Wednesday let us come right to your door ...er...device. Subscribe wherever you listen to your podcasts. Just hit the SUBSCRIBE button & we'll show up!XX,Anita & KellyDI - 5:23 / 19:04See Privacy Policy at https://art19.com/privacy and California Privacy Notice at https://art19.com/privacy#do-not-sell-my-info.